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Psychology and Neuroscience : Publications since January 2017

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%% Adcock, Rachel A.   
@article{fds319596,
   Author = {Scult, MA and Knodt, AR and Hanson, JL and Ryoo, M and Adcock, RA and Hariri, AR and Strauman, TJ},
   Title = {Individual differences in regulatory focus predict neural
             response to reward.},
   Journal = {Social Neuroscience},
   Volume = {12},
   Number = {4},
   Pages = {419-429},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1080/17470919.2016.1178170},
   Abstract = {Although goal pursuit is related to both functioning of the
             brain's reward circuits and psychological factors, the
             literatures surrounding these concepts have often been
             separate. Here, we use the psychological construct of
             regulatory focus to investigate individual differences in
             neural response to reward. Regulatory focus theory proposes
             two motivational orientations for personal goal pursuit: (1)
             promotion, associated with sensitivity to potential gain,
             and (2) prevention, associated with sensitivity to potential
             loss. The monetary incentive delay task was used to
             manipulate reward circuit function, along with instructional
             framing corresponding to promotion and prevention in a
             within-subject design. We observed that the more promotion
             oriented an individual was, the lower their ventral striatum
             response to gain cues. Follow-up analyses revealed that
             greater promotion orientation was associated with decreased
             ventral striatum response even to no-value cues, suggesting
             that promotion orientation may be associated with relatively
             hypoactive reward system function. The findings are also
             likely to represent an interaction between the cognitive and
             motivational characteristics of the promotion system with
             the task demands. Prevention orientation did not correlate
             with ventral striatum response to gain cues, supporting the
             discriminant validity of regulatory focus theory. The
             results highlight a dynamic association between individual
             differences in self-regulation and reward system
             function.},
   Doi = {10.1080/17470919.2016.1178170},
   Key = {fds319596}
}

@article{fds310030,
   Author = {Murty, VP and Ballard, IC and Adcock, RA},
   Title = {Hippocampus and Prefrontal Cortex Predict Distinct
             Timescales of Activation in the Human Ventral Tegmental
             Area.},
   Journal = {Cerebral Cortex},
   Volume = {27},
   Number = {2},
   Pages = {1660-1669},
   Year = {2017},
   Month = {February},
   ISSN = {1047-3211},
   url = {http://dx.doi.org/10.1093/cercor/bhw005},
   Abstract = {The mesolimbic dopamine system contributes to a remarkable
             variety of behaviors at multiple timescales. Midbrain
             neurons have fast and slow signaling components, and
             specific afferent systems, such as the hippocampus (HPC) and
             prefrontal cortex (PFC), have been demonstrated to drive
             these components in anesthetized animals. Whether these
             interactions exist during behavior, however, is unknown. To
             address this question, we developed a novel analysis of
             human functional magnetic resonance imaging data that fits
             models of network excitation and inhibition on ventral
             tegmental area (VTA) activation. We show that specific
             afferent systems predict distinct temporal components of
             midbrain VTA signal. We found that PFC, but not HPC,
             positively predicted transient, event-evoked VTA activation.
             In contrast, HPC, but not PFC, positively predicted slow
             shifts in VTA baseline variability. Thus, unique functional
             contributions of afferent systems to VTA physiology are
             detectable at the network level in behaving humans. The
             findings support models of dopamine function in which
             dissociable neural circuits support different aspects of
             motivated behavior via active regulation of tonic and phasic
             signals.},
   Doi = {10.1093/cercor/bhw005},
   Key = {fds310030}
}


%% Appelbaum, Lawrence G.   
@article{fds332131,
   Author = {Burris, K and Vittetoe, K and Ramger, B and Suresh, S and Tokdar, ST and Reiter, JP and Appelbaum, LG},
   Title = {Sensorimotor abilities predict on-field performance in
             professional baseball.},
   Journal = {Scientific Reports},
   Volume = {8},
   Number = {1},
   Pages = {116},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1038/s41598-017-18565-7},
   Abstract = {Baseball players must be able to see and react in an
             instant, yet it is hotly debated whether superior
             performance is associated with superior sensorimotor
             abilities. In this study, we compare sensorimotor abilities,
             measured through 8 psychomotor tasks comprising the Nike
             Sensory Station assessment battery, and game statistics in a
             sample of 252 professional baseball players to evaluate the
             links between sensorimotor skills and on-field performance.
             For this purpose, we develop a series of Bayesian
             hierarchical latent variable models enabling us to compare
             statistics across professional baseball leagues. Within this
             framework, we find that sensorimotor abilities are
             significant predictors of on-base percentage, walk rate and
             strikeout rate, accounting for age, position, and league. We
             find no such relationship for either slugging percentage or
             fielder-independent pitching. The pattern of results
             suggests performance contributions from both visual-sensory
             and visual-motor abilities and indicates that sensorimotor
             screenings may be useful for player scouting.},
   Doi = {10.1038/s41598-017-18565-7},
   Key = {fds332131}
}

@article{fds332130,
   Author = {Rao, HM and Khanna, R and Zielinski, DJ and Lu, Y and Clements, JM and Potter, ND and Sommer, MA and Kopper, R and Appelbaum,
             LG},
   Title = {Sensorimotor Learning during a Marksmanship Task in
             Immersive Virtual Reality.},
   Journal = {Frontiers in Psychology},
   Volume = {9},
   Number = {58},
   Pages = {58},
   Publisher = {Frontiers Media},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fpsyg.2018.00058},
   Abstract = {Sensorimotor learning refers to improvements that occur
             through practice in the performance of sensory-guided motor
             behaviors. Leveraging novel technical capabilities of an
             immersive virtual environment, we probed the component
             kinematic processes that mediate sensorimotor learning.
             Twenty naïve subjects performed a simulated marksmanship
             task modeled after Olympic Trap Shooting standards. We
             measured movement kinematics and shooting performance as
             participants practiced 350 trials while receiving
             trial-by-trial feedback about shooting success.
             Spatiotemporal analysis of motion tracking elucidated the
             ballistic and refinement phases of hand movements. We found
             systematic changes in movement kinematics that accompanied
             improvements in shot accuracy during training, though
             reaction and response times did not change over blocks. In
             particular, we observed longer, slower, and more precise
             ballistic movements that replaced effort spent on
             corrections and refinement. Collectively, these results
             leverage developments in immersive virtual reality
             technology to quantify and compare the kinematics of
             movement during early learning of full-body sensorimotor
             orienting.},
   Doi = {10.3389/fpsyg.2018.00058},
   Key = {fds332130}
}

@article{fds324820,
   Author = {Klemish, D and Ramger, B and Vittetoe, K and Reiter, JP and Tokdar, ST and Appelbaum, LG},
   Title = {Visual abilities distinguish pitchers from hitters in
             professional baseball.},
   Journal = {Journal of Sports Sciences},
   Volume = {36},
   Number = {2},
   Pages = {171-179},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1080/02640414.2017.1288296},
   Abstract = {This study aimed to evaluate the possibility that
             differences in sensorimotor abilities exist between hitters
             and pitchers in a large cohort of baseball players of
             varying levels of experience. Secondary data analysis was
             performed on 9 sensorimotor tasks comprising the Nike
             Sensory Station assessment battery. Bayesian hierarchical
             regression modelling was applied to test for differences
             between pitchers and hitters in data from 566 baseball
             players (112 high school, 85 college, 369 professional)
             collected at 20 testing centres. Explanatory variables
             including height, handedness, eye dominance, concussion
             history, and player position were modelled along with age
             curves using basis regression splines. Regression analyses
             revealed better performance for hitters relative to pitchers
             at the professional level in the visual clarity and depth
             perception tasks, but these differences did not exist at the
             high school or college levels. No significant differences
             were observed in the other 7 measures of sensorimotor
             capabilities included in the test battery, and no systematic
             biases were found between the testing centres. These
             findings, indicating that professional-level hitters have
             better visual acuity and depth perception than
             professional-level pitchers, affirm the notion that highly
             experienced athletes have differing perceptual skills.
             Findings are discussed in relation to deliberate practice
             theory.},
   Doi = {10.1080/02640414.2017.1288296},
   Key = {fds324820}
}

@misc{fds330473,
   Author = {Jack, J and Appelbaum, LG and Beam, E and Moody, J and Huettel,
             SA},
   Title = {Mapping rhetorical topologies in cognitive
             neuroscience},
   Pages = {125-150},
   Booktitle = {Topologies as Techniques for a Post-Critical
             Rhetoric},
   Year = {2017},
   Month = {January},
   ISBN = {9783319512686},
   url = {http://dx.doi.org/10.1007/978-3-319-51268-6_7},
   Abstract = {© The Author(s) 2017. Many tools that neuroscientists use
             to trace the complex topography of the human brain draw on
             the neuroscience literature to yield “metanalyses” or
             “syntheses of data.” These approaches conflate
             rhetorical connections in the literature with physical
             connections in the brain. By contrast, the model presented
             in this chapter seeks not a topography of the brain but a
             topology of neuroscience. A social network analysis of
             titles and abstracts for cognitive neuroscience articles
             yields a topology of brain regions and functions. This map
             can help researchers identify underresearched areas (e.g.,
             the thalamus) or areas that are oversaturated (e.g., the
             amygdala). The map also helps researchers identify
             subdisciplines, such as “neuroeconomics,” that have not
             yet integrated with the broader field–“islands” where
             rhetorical work could yield benefits.},
   Doi = {10.1007/978-3-319-51268-6_7},
   Key = {fds330473}
}


%% Asher, Steven R.   
@article{fds332880,
   Author = {McDonald, KL and Asher, SR},
   Title = {Pacifists and Revenge-Seekers in Response to Unambiguous
             Peer Provocation.},
   Journal = {Journal of Youth and Adolescence},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s10964-017-0767-4},
   Abstract = {In order to better understand why some children retaliate
             when they feel provoked and others do not, the present study
             identified "pacifistically-oriented" children who made
             negative interpretations in response to unambiguous
             provocations, yet did not endorse revenge goals, and
             compared them to "revenge-seeking" children who also made
             negative interpretations but did endorse revenge goals.
             Groups were identified based on seventh graders'
             (N = 367; 54.77% male; 22.89% racial/ethnic minority)
             responses to hypothetical situations in which a peer
             excluded and insulted them. Comparing these groups revealed
             that Pacifists endorsed relationship-maintaining goals and
             emotion regulation goals more highly than Revenge-Seekers.
             Revenge-Seekers reported more anger and endorsed beliefs
             about negative reciprocity and aggression being legitimate
             more highly than Pacifists. Additionally, Revenge-Seekers
             were more disrespect sensitive than were Pacifists, based on
             a measure of vigilance for signs of disrespect and
             expectations that others would disrespect them. Together
             these findings point to social-cognitive and emotion-related
             processes that may inhibit revenge-seeking in unambiguous
             provocation situations, even when children interpret the
             peer's behavior quite negatively.},
   Doi = {10.1007/s10964-017-0767-4},
   Key = {fds332880}
}

@article{fds322939,
   Author = {Rose, AJ and Asher, SR},
   Title = {The Social Tasks of Friendship: Do Boys and Girls Excel in
             Different Tasks?},
   Journal = {Child Development Perspectives},
   Volume = {11},
   Number = {1},
   Pages = {3-8},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1111/cdep.12214},
   Doi = {10.1111/cdep.12214},
   Key = {fds322939}
}


%% Bejjani, Christina   
@article{fds333711,
   Author = {DiMenichi, BC and Lempert, KM and Bejjani, C and Tricomi,
             E},
   Title = {Writing about past failures attenuates cortisol responses
             and sustained attention deficits following psychosocial
             stress},
   Journal = {Frontiers in Behavioral Neuroscience},
   Volume = {12},
   Number = {45},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.3389/fnbeh.2018.00045},
   Doi = {10.3389/fnbeh.2018.00045},
   Key = {fds333711}
}

@article{fds332947,
   Author = {Bejjani, C and Zhang, Z and Egner, T},
   Title = {Control by association: Transfer of implicitly primed
             attentional states across linked stimuli.},
   Journal = {Psychonomic Bulletin and Review},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.3758/s13423-018-1445-6},
   Abstract = {Although cognitive control has traditionally been viewed in
             opposition to associative learning, recent studies show that
             people can learn to link particular stimuli with specific
             cognitive control states (e.g., high attentional
             selectivity). Here, we tested whether such learned
             stimulus-control associations can transfer across
             paired-associates. In the Stimulus-Stimulus (S-S)
             Association phase, specific face or house images repeatedly
             preceded the presentation of particular scene stimuli,
             creating paired face/house-scene associates in memory. The
             Stimulus-Control (S-C) Association phase then associated
             these scenes with different attentional control states by
             probabilistically biasing specific scenes to mostly precede
             either congruent or incongruent trials in a Stroop task.
             Finally, in the Stimulus-Control Transfer (S-CT) phase, the
             faces and houses from the S-S phase preceded Stroop trials
             but were not predictive of congruency, testing whether
             stimulus-control associations would transfer from scenes to
             their associated face/house stimuli. In Experiments 1 and 3,
             we found that learned implicit stimulus-control associations
             could transfer across closely linked cues, and in Experiment
             2, we showed that this transfer depended on the memory
             associations formed in the S-S phase. While this form of
             transfer learning has previously been demonstrated for
             stimulus-reward associations, the present study provides the
             first evidence for the associative transfer of
             stimulus-control associations across arbitrarily linked
             stimuli. This work demonstrates how people can learn to
             implicitly adapt their processing strategies in a flexible
             context-dependent manner and establishes a novel learning
             mechanism supporting the generalization of cognitive
             control.},
   Doi = {10.3758/s13423-018-1445-6},
   Key = {fds332947}
}


%% Belsky, Daniel W   
@article{fds333731,
   Author = {Wertz, J and Caspi, A and Belsky, DW and Beckley, AL and Arseneault, L and Barnes, JC and Corcoran, DL and Hogan, S and Houts, RM and Morgan, N and Odgers, CL and Prinz, JA and Sugden, K and Williams, BS and Poulton, R and Moffitt, TE},
   Title = {Genetics and Crime: Integrating New Genomic Discoveries Into
             Psychological Research About Antisocial Behavior.},
   Journal = {Psychological Science},
   Pages = {956797617744542},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/0956797617744542},
   Abstract = {Drawing on psychological and sociological theories of crime
             causation, we tested the hypothesis that genetic risk for
             low educational attainment (assessed via a genome-wide
             polygenic score) is associated with criminal offending. We
             further tested hypotheses of how polygenic risk relates to
             the development of antisocial behavior from childhood
             through adulthood. Across the Dunedin and Environmental Risk
             (E-Risk) birth cohorts of individuals growing up 20 years
             and 20,000 kilometers apart, education polygenic scores
             predicted risk of a criminal record with modest effects.
             Polygenic risk manifested during primary schooling in lower
             cognitive abilities, lower self-control, academic
             difficulties, and truancy, and it was associated with a
             life-course-persistent pattern of antisocial behavior that
             onsets in childhood and persists into adulthood. Crime is
             central in the nature-nurture debate, and findings reported
             here demonstrate how molecular-genetic discoveries can be
             incorporated into established theories of antisocial
             behavior. They also suggest that improving school
             experiences might prevent genetic influences on crime from
             unfolding.},
   Doi = {10.1177/0956797617744542},
   Key = {fds333731}
}

@article{fds332056,
   Author = {Domingue, BW and Belsky, DW and Fletcher, JM and Conley, D and Boardman,
             JD and Harris, KM},
   Title = {The social genome of friends and schoolmates in the National
             Longitudinal Study of Adolescent to Adult
             Health.},
   Journal = {Proceedings of the National Academy of Sciences of
             USA},
   Volume = {115},
   Number = {4},
   Pages = {702-707},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1073/pnas.1711803115},
   Abstract = {Humans tend to form social relationships with others who
             resemble them. Whether this sorting of like with like arises
             from historical patterns of migration, meso-level social
             structures in modern society, or individual-level selection
             of similar peers remains unsettled. Recent research has
             evaluated the possibility that unobserved genotypes may play
             an important role in the creation of homophilous
             relationships. We extend this work by using data from 5,500
             adolescents from the National Longitudinal Study of
             Adolescent to Adult Health (Add Health) to examine genetic
             similarities among pairs of friends. Although there is some
             evidence that friends have correlated genotypes, both at the
             whole-genome level as well as at trait-associated loci (via
             polygenic scores), further analysis suggests that meso-level
             forces, such as school assignment, are a principal source of
             genetic similarity between friends. We also observe apparent
             social-genetic effects in which polygenic scores of an
             individual's friends and schoolmates predict the
             individual's own educational attainment. In contrast, an
             individual's height is unassociated with the height genetics
             of peers.},
   Doi = {10.1073/pnas.1711803115},
   Key = {fds332056}
}

@article{fds327153,
   Author = {Belsky, DW and Huffman, KM and Pieper, CF and Shalev, I and Kraus,
             WE},
   Title = {Change in the Rate of Biological Aging in Response to
             Caloric Restriction: CALERIE Biobank Analysis.},
   Journal = {Journals of Gerontology: Series A},
   Volume = {73},
   Number = {1},
   Pages = {4-10},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1093/gerona/glx096},
   Abstract = {Biological aging measures have been proposed as proxies for
             extension of healthy life span in trials of geroprotective
             therapies that aim to slow aging. Several methods to measure
             biological aging show promise but it is not known if these
             methods are sensitive to changes caused by geroprotective
             therapy. We conducted analysis of two proposed methods to
             quantify biological aging using data from a recently
             concluded trial of an established geroprotector, caloric
             restriction. We obtained data from the National Institute on
             Aging CALERIE randomized trial through its public-access
             biobank (https://calerie.duke.edu/). The CALERIE trial
             randomized N = 220 nonobese adults to 25% caloric
             restriction (n = 145; 11.7% caloric restriction was
             achieved, on average) or to maintain current diet (n = 75)
             for 2 years. We analyzed biomarker data collected at
             baseline, 12-, and 24-month follow-up assessments. We
             applied published biomarker algorithms to these data to
             calculate two biological age measures, Klemera-Doubal Method
             Biological Age and homeostatic dysregulation.
             Intent-to-treat analysis using mixed-effects growth models
             of within-person change over time tested if caloric
             restriction slowed increase in measures of biological aging
             across follow-up. Analyses of both measures indicated
             caloric restriction slowed biological aging. Weight loss did
             not account for the observed effects. Results suggest future
             directions for testing of geroprotective therapies in
             humans.},
   Doi = {10.1093/gerona/glx096},
   Key = {fds327153}
}

@article{fds331420,
   Author = {Hastings, WJ and Shalev, I and Belsky, DW},
   Title = {Translating measures of biological aging to test
             effectiveness of geroprotective interventions: What can we
             learn from research on telomeres?},
   Journal = {Frontiers in Genetics},
   Volume = {8},
   Number = {NOV},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.3389/fgene.2017.00164},
   Abstract = {© 2017 Hastings, Shalev and Belsky. Intervention studies in
             animals suggest molecular changes underlying age-related
             disease and disability can be slowed or reversed. To speed
             translation of these so-called "geroprotective" therapies to
             prevent age-related disease and disability in humans,
             biomarkers are needed that can track changes in the rate of
             human aging over the course of intervention trials.
             Algorithm methods that measure biological processes of aging
             from combinations of DNA methylation marks or clinical
             biomarkers show promise. To identify next steps for
             establishing utility of these algorithm-based measures of
             biological aging for geroprotector trials, we considered the
             history a candidate biomarker of aging that has received
             substantial research attention, telomere length. Although
             telomere length possesses compelling biology to recommend it
             as a biomarker of aging, mixed research findings have
             impeded clinical and epidemiologic translation. Strengths of
             telomeres that should be established for algorithm
             biomarkers of aging are correlation with chronological age
             across the lifespan, prediction of disease, disability, and
             early death, and responsiveness to risk and protective
             exposures. Key challenges in telomere research that
             algorithm biomarkers of aging must address are measurement
             precision and reliability, establishing links between
             longitudinal rates of change across repeated measurements
             and aging outcomes, and clarity over whether the biomarker
             is a causal mechanism of aging. These strengths and
             challenges suggest a research agenda to advance translation
             of algorithm-based aging biomarkers: establish validity in
             young-adult and midlife individuals; test responsiveness to
             exposures that shorten or extend healthy lifespan; and
             conduct repeated-measures longitudinal studies to test
             differential rates of change.},
   Doi = {10.3389/fgene.2017.00164},
   Key = {fds331420}
}

@article{fds330421,
   Author = {Schaefer, JD and Scult, MA and Caspi, A and Arseneault, L and Belsky,
             DW and Hariri, AR and Harrington, H and Houts, R and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Is low cognitive functioning a predictor or consequence of
             major depressive disorder? A test in two longitudinal birth
             cohorts.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s095457941700164x},
   Abstract = {Cognitive impairment has been identified as an important
             aspect of major depressive disorder (MDD). We tested two
             theories regarding the association between MDD and cognitive
             functioning using data from longitudinal cohort studies. One
             theory, the cognitive reserve hypothesis, suggests that
             higher cognitive ability in childhood decreases risk of
             later MDD. The second, the scarring hypothesis, instead
             suggests that MDD leads to persistent cognitive deficits
             following disorder onset. We tested both theories in the
             Dunedin Study, a population-representative cohort followed
             from birth to midlife and assessed repeatedly for both
             cognitive functioning and psychopathology. We also used data
             from the Environmental Risk Longitudinal Twin Study to test
             whether childhood cognitive functioning predicts future MDD
             risk independent of family-wide and genetic risk using a
             discordant twin design. Contrary to both hypotheses, we
             found that childhood cognitive functioning did not predict
             future risk of MDD, nor did study members with a past
             history of MDD show evidence of greater cognitive decline
             unless MDD was accompanied by other comorbid psychiatric
             conditions. Our results thus suggest that low cognitive
             functioning is related to comorbidity, but is neither an
             antecedent nor an enduring consequence of MDD. Future
             research may benefit from considering cognitive deficits
             that occur during depressive episodes from a transdiagnostic
             perspective.},
   Doi = {10.1017/s095457941700164x},
   Key = {fds330421}
}

@article{fds328924,
   Author = {Domingue, BW and Liu, H and Okbay, A and Belsky, DW},
   Title = {Genetic Heterogeneity in Depressive Symptoms Following the
             Death of a Spouse: Polygenic Score Analysis of the U.S.
             Health and Retirement Study.},
   Journal = {American Journal of Psychiatry},
   Volume = {174},
   Number = {10},
   Pages = {963-970},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.16111209},
   Abstract = {Experience of stressful life events is associated with risk
             of depression. Yet many exposed individuals do not become
             depressed. A controversial hypothesis is that genetic
             factors influence vulnerability to depression following
             stress. This hypothesis is often tested with a
             "diathesis-stress" model, in which genes confer excess
             vulnerability. The authors tested an alternative formulation
             of this model: genes may buffer against depressogenic
             effects of life stress.The hypothesized genetic buffer was
             measured using a polygenic score derived from a published
             genome-wide association study of subjective well-being. The
             authors tested whether married older adults who had higher
             polygenic scores were less vulnerable to depressive symptoms
             following the death of their spouse compared with
             age-matched peers who had also lost their spouse and who had
             lower polygenic scores. Data were analyzed from 8,588
             non-Hispanic white adults in the Health and Retirement Study
             (HRS), a population-representative longitudinal study of
             older adults in the United States.HRS adults with higher
             well-being polygenic scores experienced fewer depressive
             symptoms during follow-up. Those who survived the death of
             their spouses (N=1,647) experienced a sharp increase in
             depressive symptoms following the death and returned toward
             baseline over the following 2 years. Having a higher
             well-being polygenic score buffered against increased
             depressive symptoms following a spouse's death.The effects
             were small, and the clinical relevance is uncertain,
             although polygenic score analyses may provide clues to
             behavioral pathways that can serve as therapeutic targets.
             Future studies of gene-environment interplay in depression
             may benefit from focus on genetics discovered for putative
             protective factors.},
   Doi = {10.1176/appi.ajp.2017.16111209},
   Key = {fds328924}
}

@article{fds328923,
   Author = {Belsky, DW and Snyder-Mackler, N},
   Title = {Invited Commentary: Integrating Genomics and Social
             Epidemiology-Analysis of Late-Life Low Socioeconomic Status
             and the Conserved Transcriptional Response to
             Adversity.},
   Journal = {American Journal of Epidemiology},
   Volume = {186},
   Number = {5},
   Pages = {510-513},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1093/aje/kwx145},
   Abstract = {Socially disadvantaged children face increased morbidity and
             mortality as they age. Understanding mechanisms through
             which social disadvantage becomes biologically embedded and
             devising measurements that can track this embedding are
             critical priorities for research to address social gradients
             in health. The analysis by Levine et al. (Am J Epidemiol.
             2017;186(5):503-509) of genome-wide gene expression in a
             subsample of US Health and Retirement Study participants
             suggests important new directions for the field.
             Specifically, findings suggest promise in integrating gene
             expression data into population studies and provide further
             evidence for the conserved transcriptional response to
             adversity as a marker of biological embedding of social
             disadvantage. The study also highlights methodological
             issues related to the analysis of gene expression data and
             social gradients in health and a need to examine the
             conserved transcriptional response to adversity alongside
             other proposed measurements of biological embedding. Looking
             to the future, advances in genome science are opening new
             opportunities for sociogenomic epidemiology.},
   Doi = {10.1093/aje/kwx145},
   Key = {fds328923}
}

@article{fds326223,
   Author = {Belsky, DW and Caspi, A and Cohen, HJ and Kraus, WE and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Impact of early personal-history characteristics on the Pace
             of Aging: implications for clinical trials of therapies to
             slow aging and extend healthspan.},
   Journal = {Aging Cell},
   Volume = {16},
   Number = {4},
   Pages = {644-651},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/acel.12591},
   Abstract = {Therapies to extend healthspan are poised to move from
             laboratory animal models to human clinical trials.
             Translation from mouse to human will entail challenges,
             among them the multifactorial heterogeneity of human aging.
             To inform clinical trials about this heterogeneity, we
             report how humans' pace of biological aging relates to
             personal-history characteristics. Because geroprotective
             therapies must be delivered by midlife to prevent
             age-related disease onset, we studied young-adult members of
             the Dunedin Study 1972-73 birth cohort (n = 954). Cohort
             members' Pace of Aging was measured as coordinated decline
             in the integrity of multiple organ systems, by quantifying
             rate of decline across repeated measurements of 18
             biomarkers assayed when cohort members were ages 26, 32, and
             38 years. The childhood personal-history characteristics
             studied were known predictors of age-related disease and
             mortality, and were measured prospectively during childhood.
             Personal-history characteristics of familial longevity,
             childhood social class, adverse childhood experiences, and
             childhood health, intelligence, and self-control all
             predicted differences in cohort members' adulthood Pace of
             Aging. Accumulation of more personal-history risks predicted
             faster Pace of Aging. Because trials of anti-aging therapies
             will need to ascertain personal histories retrospectively,
             we replicated results using cohort members' retrospective
             personal-history reports made in adulthood. Because many
             trials recruit participants from clinical settings, we
             replicated results in the cohort subset who had recent
             health system contact according to electronic medical
             records. Quick, inexpensive measures of trial participants'
             early personal histories can enable clinical trials to study
             who volunteers for trials, who adheres to treatment, and who
             responds to anti-aging therapies.},
   Doi = {10.1111/acel.12591},
   Key = {fds326223}
}

@article{fds327274,
   Author = {Domingue, BW and Belsky, DW and Harrati, A and Conley, D and Weir, DR and Boardman, JD},
   Title = {Mortality selection in a genetic sample and implications for
             association studies},
   Journal = {International Journal of Epidemiology},
   Volume = {46},
   Number = {4},
   Pages = {1285-1294},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1093/ije/dyx041},
   Doi = {10.1093/ije/dyx041},
   Key = {fds327274}
}

@article{fds327272,
   Author = {Belsky, DW},
   Title = {Translating Polygenic Analysis for Prevention},
   Journal = {Circulation: Cardiovascular Genetics},
   Volume = {10},
   Number = {3},
   Pages = {e001798-e001798},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1161/CIRCGENETICS.117.001798},
   Doi = {10.1161/CIRCGENETICS.117.001798},
   Key = {fds327272}
}

@article{fds327273,
   Author = {Richmond-Rakerd, LS and Belsky, DW},
   Title = {Swedish Register Analysis of Divorce and Alcohol Use
             Disorder Highlights Social Relationships as a Target for
             Preventive Psychiatry and Genetic Research.},
   Journal = {American Journal of Psychiatry},
   Volume = {174},
   Number = {5},
   Pages = {411-413},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.17020170},
   Doi = {10.1176/appi.ajp.2017.17020170},
   Key = {fds327273}
}

@article{fds328283,
   Author = {Domingue, BW and Belsky, DW},
   Title = {The social genome: Current findings and implications for the
             study of human genetics.},
   Journal = {PLoS genetics},
   Volume = {13},
   Number = {3},
   Pages = {e1006615},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1371/journal.pgen.1006615},
   Doi = {10.1371/journal.pgen.1006615},
   Key = {fds328283}
}

@article{fds325853,
   Author = {Reuben, A and Caspi, A and Belsky, DW and Broadbent, J and Harrington,
             H and Sugden, K and Houts, RM and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Cognitive
             Function and Socioeconomic Status at Age 38 Years and With
             IQ Change and Socioeconomic Mobility Between Childhood and
             Adulthood.},
   Journal = {JAMA : the journal of the American Medical
             Association},
   Volume = {317},
   Number = {12},
   Pages = {1244-1251},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1001/jama.2017.1712},
   Abstract = {Many children in the United States and around the world are
             exposed to lead, a developmental neurotoxin. The long-term
             cognitive and socioeconomic consequences of lead exposure
             are uncertain.To test the hypothesis that childhood lead
             exposure is associated with cognitive function and
             socioeconomic status in adulthood and with changes in IQ and
             socioeconomic mobility between childhood and midlife.A
             prospective cohort study based on a population-representative
             1972-1973 birth cohort from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (until December 2012).Childhood
             lead exposure ascertained as blood lead levels measured at
             age 11 years. High blood lead levels were observed among
             children from all socioeconomic status levels in this
             cohort.The IQ (primary outcome) and indexes of Verbal
             Comprehension, Perceptual Reasoning, Working Memory, and
             Processing Speed (secondary outcomes) were assessed at age
             38 years using the Wechsler Adult Intelligence Scale-IV
             (WAIS-IV; IQ range, 40-160). Socioeconomic status (primary
             outcome) was assessed at age 38 years using the New Zealand
             Socioeconomic Index-2006 (NZSEI-06; range, 10 [lowest]-90
             [highest]).Of 1037 original participants, 1007 were alive at
             age 38 years, of whom 565 (56%) had been lead tested at age
             11 years (54% male; 93% white). Mean (SD) blood lead level
             at age 11 years was 10.99 (4.63) µg/dL. Among blood-tested
             participants included at age 38 years, mean WAIS-IV score
             was 101.16 (14.82) and mean NZSEI-06 score was 49.75
             (17.12). After adjusting for maternal IQ, childhood IQ, and
             childhood socioeconomic status, each 5-µg/dL higher level
             of blood lead in childhood was associated with a 1.61-point
             lower score (95% CI, -2.48 to -0.74) in adult IQ, a
             2.07-point lower score (95% CI, -3.14 to -1.01) in
             perceptual reasoning, and a 1.26-point lower score (95% CI,
             -2.38 to -0.14) in working memory. Associations of childhood
             blood lead level with deficits in verbal comprehension and
             processing speed were not statistically significant. After
             adjusting for confounders, each 5-µg/dL higher level of
             blood lead in childhood was associated with a 1.79-unit
             lower score (95% CI, -3.17 to -0.40) in socioeconomic
             status. An association between greater blood lead levels and
             a decline in IQ and socioeconomic status from childhood to
             adulthood was observed with 40% of the association with
             downward mobility mediated by cognitive decline from
             childhood.In this cohort born in New Zealand in 1972-1973,
             childhood lead exposure was associated with lower cognitive
             function and socioeconomic status at age 38 years and with
             declines in IQ and with downward social mobility. Childhood
             lead exposure may have long-term ramifications.},
   Doi = {10.1001/jama.2017.1712},
   Key = {fds325853}
}

@article{fds328103,
   Author = {Moffitt, TE and Belsky, DW and Danese, A and Poulton, R and Caspi,
             A},
   Title = {The Longitudinal Study of Aging in Human Young Adults:
             Knowledge Gaps and Research Agenda.},
   Journal = {Journals of Gerontology: Series A},
   Volume = {72},
   Number = {2},
   Pages = {210-215},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1093/gerona/glw191},
   Abstract = {To prevent onset of age-related diseases and physical and
             cognitive decline, interventions to slow human aging and
             extend health span must eventually be applied to people
             while they are still young and healthy. Yet most human aging
             research examines older adults, many with chronic disease,
             and little is known about aging in healthy young humans.This
             article explains how this knowledge gap is a barrier to
             extending health span and puts forward the case that
             geroscience should invest in researching the pace of aging
             in young adults. As one illustrative example, we describe an
             initial effort to study the pace of aging in a young-adult
             birth cohort by using repeated waves of biomarkers collected
             across the third and fourth decades to quantify the pace of
             coordinated physiological deterioration across multiple
             organ systems (eg, pulmonary, periodontal, cardiovascular,
             renal, hepatic, metabolic, and immune function).Findings
             provided proof of principle that it is possible to quantify
             individual variation in the pace of aging in young adults
             still free of age-related diseases.This article articulates
             research needs to improve longitudinal measurement of the
             pace of aging in young people, to pinpoint factors that slow
             or speed the pace of aging, to compare pace of aging against
             genomic clocks, to explain slow-aging young adults, and to
             apply pace of aging in preventive clinical trials of
             antiaging therapies. This article puts forward a research
             agenda to fill the knowledge gap concerning lifelong causes
             of aging.},
   Doi = {10.1093/gerona/glw191},
   Key = {fds328103}
}

@article{fds324457,
   Author = {Schaefer, JD and Caspi, A and Belsky, DW and Harrington, H and Houts, R and Horwood, LJ and Hussong, A and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Enduring mental health: Prevalence and prediction.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {2},
   Pages = {212-224},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1037/abn0000232},
   Abstract = {We review epidemiological evidence indicating that most
             people will develop a diagnosable mental disorder,
             suggesting that only a minority experience enduring mental
             health. This minority has received little empirical study,
             leaving the prevalence and predictors of enduring mental
             health unknown. We turn to the population-representative
             Dunedin cohort, followed from birth to midlife, to compare
             people never-diagnosed with mental disorder (N = 171; 17%
             prevalence) to those diagnosed at 1-2 study waves, the
             cohort mode (N = 409). Surprisingly, compared to this modal
             group, never-diagnosed Study members were not born into
             unusually well-to-do families, nor did their enduring mental
             health follow markedly sound physical health, or unusually
             high intelligence. Instead, they tended to have an
             advantageous temperament/personality style, and negligible
             family history of mental disorder. As adults, they report
             superior educational and occupational attainment, greater
             life satisfaction, and higher-quality relationships. Our
             findings draw attention to "enduring mental health" as a
             revealing psychological phenotype and suggest it deserves
             further study. (PsycINFO Database Record},
   Doi = {10.1037/abn0000232},
   Key = {fds324457}
}

@article{fds324523,
   Author = {Moskalev, A and Anisimov, V and Aliper, A and Artemov, A and Asadullah,
             K and Belsky, D and Baranova, A and de Grey, A and Dixit, VD and Debonneuil, E and Dobrovolskaya, E and Fedichev, P and Fedintsev, A and Fraifeld, V and Franceschi, C and Freer, R and Fülöp, T and Feige, J and Gems, D and Gladyshev, V and Gorbunova, V and Irincheeva, I and Jager,
             S and Jazwinski, SM and Kaeberlein, M and Kennedy, B and Khaltourina, D and Kovalchuk, I and Kovalchuk, O and Kozin, S and Kulminski, A and Lashmanova, E and Lezhnina, K and Liu, G-H and Longo, V and Mamoshina,
             P and Maslov, A and Pedro de Magalhaes and J et al.},
   Title = {A review of the biomedical innovations for healthy
             longevity.},
   Journal = {Aging},
   Volume = {9},
   Number = {1},
   Pages = {7-25},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.18632/aging.101163},
   Doi = {10.18632/aging.101163},
   Key = {fds324523}
}


%% Bennett, Gary G.   
@article{fds332978,
   Author = {Pagoto, SL and Bennett, GG},
   Title = {Healthy Lifestyle for All Through Patient Care and
             Policy.},
   Journal = {JAMA Internal Medicine},
   Volume = {178},
   Number = {1},
   Pages = {152-153},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1001/jamainternmed.2017.6797},
   Doi = {10.1001/jamainternmed.2017.6797},
   Key = {fds332978}
}

@article{fds332939,
   Author = {Armstrong, S and Mendelsohn, A and Bennett, G and Taveras, EM and Kimberg, A and Kemper, AR},
   Title = {Texting Motivational Interviewing: A Randomized Controlled
             Trial of Motivational Interviewing Text Messages Designed to
             Augment Childhood Obesity Treatment},
   Journal = {Childhood Obesity},
   Volume = {14},
   Number = {1},
   Pages = {4-10},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1089/chi.2017.0089},
   Doi = {10.1089/chi.2017.0089},
   Key = {fds332939}
}

@article{fds329767,
   Author = {Bennett, GG and Shelton, RC},
   Title = {Extending Our Reach for Greater Impact.},
   Journal = {Health Education & Behavior},
   Volume = {44},
   Number = {6},
   Pages = {835-838},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1177/1090198117736354},
   Doi = {10.1177/1090198117736354},
   Key = {fds329767}
}

@article{fds330845,
   Author = {Buscemi, J and Bennett, GG and Gorin, SS and Pagoto, SL and Sallis, JF and Wilson, DK and Fitzgibbon, ML},
   Title = {A 6-year update of the health policy and advocacy priorities
             of the Society of Behavioral Medicine.},
   Journal = {Translational Behavioral Medicine},
   Volume = {7},
   Number = {4},
   Pages = {903-911},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1007/s13142-017-0507-z},
   Abstract = {Government policy affects virtually every topic of interest
             to health behavior researchers, from research funding to
             reimbursement for clinical services to application of
             evidence to impact health outcomes. This paper provides a
             6-year update on the expansion of Society of Behavioral
             Medicine's (SBM) public policy and advocacy agenda and
             proposed future directions. SBM's Health Policy Council is
             responsible for ensuring coordination of the policy-related
             activities of the Health Policy Committee (HPC), the Civic
             and Public Engagement Committee (CPEC), and the Scientific
             and Professional Liaison Council (SPLC). These committees
             and councils have written letters to Congress, signed onto
             advocacy letters with hundreds of organizations, and
             developed and disseminated 15 health policy briefs, the
             majority of which have been presented to legislative
             staffers on Capitol Hill. With the assistance of the SPLC,
             SBM has collaborated on policy efforts with like-minded
             organizations to increase the impact of the Society's policy
             work. Moving forward, SBM plans to continue to increase
             efforts to disseminate policy work more broadly and develop
             long-term relationships with Congressional staffers. SBM
             leadership realizes that to remain relevant, demonstrate
             impact, and advance the role of behavioral medicine, we must
             advance a policy agenda that reflects our mission of better
             health through behavior change.},
   Doi = {10.1007/s13142-017-0507-z},
   Key = {fds330845}
}

@article{fds329768,
   Author = {Greaney, ML and Askew, S and Wallington, SF and Foley, PB and Quintiliani, LM and Bennett, GG},
   Title = {The effect of a weight gain prevention intervention on
             moderate-vigorous physical activity among black women: the
             Shape Program.},
   Journal = {International Journal of Behavioral Nutrition and Physical
             Activity},
   Volume = {14},
   Number = {1},
   Pages = {139},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1186/s12966-017-0596-6},
   Abstract = {Rates of physical inactivity are high among Black women
             living in the United States with overweight or obesity,
             especially those living in the rural South. This study was
             conducted to determine if an efficacious weight gain
             prevention intervention increased moderate-vigorous physical
             activity (MVPA).The Shape Program, a weight gain prevention
             intervention implemented in community health centers in
             rural North Carolina, was designed for socioeconomically
             disadvantaged Black women with overweight or obesity. MVPA
             was measured using accelerometers, and summarized into 1-
             and 10-min bouts. We employed analyses of covariance
             (ANCOVA) to assess the relationship between changes in MVPA
             over 12 months, calculated as a change score, and
             intervention assignment (intervention versus usual
             care).Participants completing both baseline and 12-month
             accelerometer assessments (n = 121) had a mean age of 36.1
             (SD = 5.43) years and a mean body mass index of
             30.24 kg/m2 (SD = 2.60). At baseline, 38% met the
             physical activity recommendation (150 min of MVPA/week)
             when assessed using 10-min bouts, and 76% met the
             recommendation when assessed using 1-min bouts. There were
             no significant differences in change in MVPA participation
             among participants randomized to the intervention from
             baseline to 12-months using 1-min bouts (adjusted
             intervention mean [95% CI]: 20.50 [-109.09 to 150.10] vs.
             adjusted usual care mean [95% CI]: -80.04 [-209.21 to
             49.13], P = .29), or 10-min bouts (adjusted intervention
             mean [95% CI]: 7.39 [-83.57 to 98.35] vs. adjusted usual
             care mean [95% CI]: -17.26 [-107.93 to 73.40],
             P = .70).Although prior research determined that the Shape
             intervention promoted weight gain prevention, MVPA did not
             increase significantly among intervention participants from
             baseline to 12 months. The classification of bouts had a
             marked effect on the prevalence estimates of those meeting
             physical activity recommendations. More research is needed
             to understand how to promote increased MVPA in weight gain
             prevention interventions.This study is registered at
             www.clinicaltrials.gov database (No. NCT00938535.
             Retrospectively Registered 7/10/2009).},
   Doi = {10.1186/s12966-017-0596-6},
   Key = {fds329768}
}

@article{fds324092,
   Author = {Winkler, MR and Moore, ED and Bennett, GG and Armstrong, SC and Brandon,
             DH},
   Title = {Parent-adolescent influences on everyday dietary practices:
             Perceptions of adolescent females with obesity and their
             mothers.},
   Journal = {Maternal and Child Nutrition},
   Volume = {13},
   Number = {4},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1111/mcn.12416},
   Abstract = {Parents demonstrate an important influence on adolescent
             obesity and dietary behavior; yet, family-based obesity
             interventions continue to exhibit limited success among
             adolescents. To further inform family-based approaches for
             adolescent obesity treatment, we examined the perceptions of
             adolescent females with obesity and their mothers of the
             influences experienced within the parent-adolescent
             relationship that affect everyday dietary practices. We
             conducted six focus group interviews (three adolescent
             female and three mother) among 15 adolescent (12-17 years
             old) females with obesity and 12 of their mothers. Content
             analysis techniques were used to analyze the transcribed
             interviews. Adolescent females with obesity discussed a
             diverse set of parental influences (controlling, supporting
             and cultivating, overlooking and tempting, acquiescing,
             providing, attending, and not providing and avoiding) on
             their daily dietary practices. Among mother focus groups,
             mothers discussed specific intentional and unintentional
             types of influences from children that affected the food and
             drink they consumed, prepared, and acquired. Findings
             provide a fuller view of the varied social influences on
             everyday dietary practices within the parent-adolescent
             relationship. They indicate the importance of examining both
             parent-to-child and child-to-parent influences and begin to
             illuminate the value of attending to the social
             circumstances surrounding dietary behaviors to strengthen
             family-based obesity treatment approaches.},
   Doi = {10.1111/mcn.12416},
   Key = {fds324092}
}

@article{fds326827,
   Author = {Proeschold-Bell, RJ and Turner, EL and Bennett, GG and Yao, J and Li,
             X-F and Eagle, DE and Meyer, RA and Williams, RB and Swift, RY and Moore,
             HE and Kolkin, MA and Weisner, CC and Rugani, KM and Hough, HJ and Williams, VP and Toole, DC},
   Title = {A 2-Year Holistic Health and Stress Intervention: Results of
             an RCT in Clergy.},
   Journal = {American Journal of Preventive Medicine},
   Volume = {53},
   Number = {3},
   Pages = {290-299},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.amepre.2017.04.009},
   Abstract = {This study sought to determine the effect of a 2-year,
             multicomponent health intervention (Spirited Life) targeting
             metabolic syndrome and stress simultaneously.An RCT using a
             three-cohort multiple baseline design was conducted in
             2010-2014.Participants were United Methodist clergy in North
             Carolina, U.S., in 2010, invited based on occupational
             status. Of invited 1,745 clergy, 1,114 consented, provided
             baseline data, and were randomly assigned to immediate
             intervention (n=395), 1-year waitlist (n=283), or 2-year
             waitlist (n=436) cohorts for a 48-month trial duration.The
             2-year intervention consisted of personal goal setting and
             encouragement to engage in monthly health coaching, an
             online weight loss intervention, a small grant, and three
             workshops delivering stress management and theological
             content supporting healthy behaviors. Participants were not
             blinded to intervention.Trial outcomes were metabolic
             syndrome (primary) and self-reported stress and depressive
             symptoms (secondary). Intervention effects were estimated in
             2016 in an intention-to-treat framework using generalized
             estimating equations with adjustment for baseline level of
             the outcome and follow-up time points. Log-link Poisson
             generalized estimating equations with robust SEs was used to
             estimate prevalence ratios (PRs) for binary outcomes; mean
             differences were used for continuous/score outcomes.Baseline
             prevalence of metabolic syndrome was 50.9% and depression
             was 11.4%. The 12-month intervention effect showed a benefit
             for metabolic syndrome (PR=0.86, 95% CI=0.79, 0.94,
             p<0.001). This benefit was sustained at 24 months of
             intervention (PR=0.88; 95% CI=0.78, 1.00, p=0.04). There was
             no significant effect on depression or stress scores.The
             Spirited Life intervention improved metabolic syndrome
             prevalence in a population of U.S. Christian clergy and
             sustained improvements during 24 months of intervention.
             These findings offer support for long-duration behavior
             change interventions and population-level interventions that
             allow participants to set their own health goals.This study
             is registered at www.clinicaltrials.gov NCT01564719.},
   Doi = {10.1016/j.amepre.2017.04.009},
   Key = {fds326827}
}

@article{fds327016,
   Author = {Herring, SJ and Cruice, JF and Bennett, GG and Darden, N and Wallen, JJ and Rose, MZ and Davey, A and Foster, GD},
   Title = {Intervening during and after pregnancy to prevent weight
             retention among African American women.},
   Journal = {Preventive Medicine Reports},
   Volume = {7},
   Pages = {119-123},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.pmedr.2017.05.015},
   Abstract = {Efforts to prevent postpartum weight retention in extant
             clinical trials of African American women have proven
             exceedingly challenging. The primary purpose of this pilot
             study was to determine whether a behavioral intervention
             implemented in early pregnancy through 6 months postpartum
             could increase the proportion of African American women who
             were at or below their early pregnancy weights by 6 months
             postpartum. We additionally evaluated whether mothers'
             postpartum weight loss could be maintained at 12 months
             postpartum. Participants were 66 socioeconomically
             disadvantaged African American women (36% overweight, 64%
             obese) randomly assigned to a behavioral intervention or
             usual care group. The intervention, implemented from early
             pregnancy to 6 months postpartum, promoted weight control
             through: (1) empirically supported behavior change goals;
             (2) interactive self-monitoring text messages; (3) weekly to
             monthly health coach calls; and (4) skills training and
             support through Facebook. In modified intent-to-treat
             analyses, participants assigned to the intervention were
             significantly more likely to be at or below their early
             pregnancy weights by 6 months postpartum compared to usual
             care (56% vs. 29%, p = 0.04). At 12 months postpartum,
             the maternal weight difference between intervention and
             usual care groups was not maintained (41% vs. 38%
             respectively at or below early pregnancy weights,
             p = 0.83). Findings suggest that a combined pregnancy and
             postpartum weight control intervention improves 6 month
             weight outcomes in socioeconomically disadvantaged African
             American women with obesity. Longer interventions may be
             needed to overcome late postpartum weight gain among this
             high risk group. Clinical trial registration number:
             ClinicalTrials.gov identifier NCT01530776.},
   Doi = {10.1016/j.pmedr.2017.05.015},
   Key = {fds327016}
}

@article{fds324090,
   Author = {Steinberg, DM and Christy, J and Batch, BC and Askew, S and Moore, RH and Parker, P and Bennett, GG},
   Title = {Preventing Weight Gain Improves Sleep Quality Among Black
             Women: Results from a RCT.},
   Journal = {Annals of Behavioral Medicine},
   Volume = {51},
   Number = {4},
   Pages = {555-566},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1007/s12160-017-9879-z},
   Abstract = {Obesity and poor sleep are highly prevalent among Black
             women.We examined whether a weight gain prevention
             intervention improved sleep among Black women.We conducted a
             randomized trial comparing a 12-month weight gain prevention
             intervention that included self-monitoring through mobile
             technologies and phone coaching to usual care in community
             health centers. We measured sleep using the Medical Outcomes
             Study Sleep Scale at baseline, 12 months, and 18 months.
             The scale examines quantity of sleep, sleep disturbance,
             sleep adequacy, daytime somnolence, snoring, shortness of
             breath, and global sleep problems (sleep problem indices I
             and II).Participants (n = 184) were on average 35.4 years
             and obese (BMI 30.2 kg/m2); 74% made <$30,000/year. At
             baseline, average sleep duration was 6.4 (1.5) hours.
             Controlling for weight change and sleep medication, the
             intervention group reported greater improvements in sleep
             disturbance [-8.35 (-16.24, -0.45)] and sleep problems at
             12 months: sleep problem index I [-8.35 (-16.24, -0.45)];
             sleep problem index II [-8.35 (-16.24, -0.45)]. However,
             these findings did not persist at 18 months.Preventing
             weight gain may afford clinical benefit on improving sleep
             quality.The trial was registered with the ClinicalTrials.gov
             database (NCT00938535).},
   Doi = {10.1007/s12160-017-9879-z},
   Key = {fds324090}
}

@article{fds324093,
   Author = {Greaney, ML and Askew, S and Foley, P and Wallington, SF and Bennett,
             GG},
   Title = {Linking patients with community resources: use of a free
             YMCA membership among low-income black women.},
   Journal = {Translational Behavioral Medicine},
   Volume = {7},
   Number = {2},
   Pages = {341-348},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s13142-016-0431-7},
   Abstract = {Given the increasing interest in expanding obesity
             prevention efforts to cover community-based programs, we
             examined whether individuals would access a YMCA for
             physical activity promotion. We provided a no-cost 12-month
             YMCA membership to socioeconomically disadvantaged black
             women who were randomized to the intervention arm of a
             weight gain prevention trial (n = 91). Analyses examined
             associations of membership activation and use with baseline
             psychosocial, contextual, health-related, and
             sociodemographic factors. Many participants (70.3 %)
             activated their memberships; however, use was low (42.2 %
             had no subsequent visits, 46.9 % had one to ten visits).
             There were no predictors of membership activation, but
             individuals living below/borderline the federal poverty line
             were more likely to use the center (1+ visits), as were
             those who met physical activity guidelines at baseline. More
             comprehensive and intensive interventions may be necessary
             to promote use of community resources-even when provided
             free-among high-risk populations of women with obesity that
             live in rural areas of the USA.},
   Doi = {10.1007/s13142-016-0431-7},
   Key = {fds324093}
}

@article{fds328845,
   Author = {Steinberg, D and Bennett, GG and Svetkey, L},
   Title = {The DASH Diet, 20 Years Later.},
   Journal = {JAMA : the journal of the American Medical
             Association},
   Volume = {317},
   Number = {15},
   Pages = {1529-1530},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1001/jama.2017.1628},
   Doi = {10.1001/jama.2017.1628},
   Key = {fds328845}
}

@article{fds324091,
   Author = {Benjamin Neelon and SE and Østbye, T and Bennett, GG and Kravitz, RM and Clancy, SM and Stroo, M and Iversen, E and Hoyo, C},
   Title = {Cohort profile for the Nurture Observational Study examining
             associations of multiple caregivers on infant growth in the
             Southeastern USA.},
   Journal = {BMJ Open},
   Volume = {7},
   Number = {2},
   Pages = {e013939},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1136/bmjopen-2016-013939},
   Abstract = {Childcare has been associated with obesity in children in
             cross-sectional and longitudinal studies, although some
             observed no association. Few studies have focused on care
             during infancy, a period when children may be especially
             vulnerable.The Nurture Study is an observational birth
             cohort designed to assess longitudinal associations of
             childcare and the presence of multiple caregivers on infant
             adiposity and weight trajectories throughout the first year
             of life. We examine as potential mediators feeding, physical
             activity, sleep and stress. We completed recruitment in
             2015. Of the 860 women who enrolled during pregnancy, 799
             delivered a single live infant who met our inclusion
             criteria. Of those, 666 mothers (77.4%) agreed to
             participate in the study for themselves and their
             infants.Among the 666 women in the study, 472 (71%)
             identified as black, 127 (19%) as white, 7 (1%) as Asian or
             Asian American, 6 (1%) as Native American and 49 (7%) as
             other race or more than one race; 43 (7%) identified as
             Hispanic/Latina. Just under half (48%) had a high school
             diploma or less, 61% had household incomes <$20 000/year
             and 59% were married or living with a partner. The mean (SD)
             infant gestational age was 41.28 weeks (2.29) and birth
             weight for gestational age z-score was -0.31 (0.93). Just
             under half (49%) of infants were females, 69% received some
             human milk and 40% were exclusively breast fed at hospital
             discharge. Data collection began in 2013, is currently
             underway, and is scheduled to conclude in late 2016.Results
             will help assess the magnitude of associations between
             childcare in infancy and subsequent obesity. Findings will
             also inform intervention and policy efforts to improve
             childcare environments and help prevent obesity in settings
             where many infants spend time.Clinicaltrials.gov,
             NCT01788644.},
   Doi = {10.1136/bmjopen-2016-013939},
   Key = {fds324091}
}

@article{fds315310,
   Author = {Winkler, MR and Bennett, GG and Brandon, DH},
   Title = {Factors related to obesity and overweight among Black
             adolescent girls in the United States.},
   Journal = {Women & Health},
   Volume = {57},
   Number = {2},
   Pages = {208-248},
   Year = {2017},
   Month = {February},
   ISSN = {0363-0242},
   url = {http://dx.doi.org/10.1080/03630242.2016.1159267},
   Abstract = {In the United States, Black adolescents have the highest
             prevalence of pediatric obesity and overweight among girls.
             While Black girls are disproportionately affected, the
             reasons for this health disparity remain unclear. The
             authors conducted a systematic review to investigate the
             factors related to obesity and overweight among Black
             adolescent girls. The authors searched four databases for
             relevant English-language publications using all publication
             years through 2015. Fifty-one studies met the inclusion
             criteria and were used for this review. Using a
             configuration approach to synthesis, three categories were
             identified, paralleling the bioecological theory of human
             development: (1) individual, (2) interpersonal, and (3)
             community and societal factors. A description of each
             factor's association with obesity among Black adolescent
             girls is presented. From this review, the authors identified
             a diverse and vast set of individual, interpersonal, and
             community and societal factors explored for their
             relationship with obesity and overweight. Given the
             insufficient repetition and limited significant findings
             among most factors, the authors believe that multiple gaps
             in knowledge exist across all categories regarding the
             factors related to obesity and overweight among Black
             adolescent girls. To improve the quality of research in this
             area, suggested research directions and methodological
             recommendations are provided.},
   Doi = {10.1080/03630242.2016.1159267},
   Key = {fds315310}
}


%% Bergelson, Elika   
@article{fds333673,
   Author = {Amatuni, A and He, E and Bergelson, E},
   Title = {Preserved Structure Across Vector Space Representations.},
   Journal = {CoRR},
   Volume = {abs/1802.00840},
   Year = {2018},
   Key = {fds333673}
}

@article{fds330846,
   Author = {Bergelson, E and Aslin, RN},
   Title = {Nature and origins of the lexicon in 6-mo-olds.},
   Journal = {Proceedings of the National Academy of Sciences of
             USA},
   Volume = {114},
   Number = {49},
   Pages = {12916-12921},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1073/pnas.1712966114},
   Abstract = {Recent research reported the surprising finding that even
             6-mo-olds understand common nouns [Bergelson E, Swingley D
             (2012) Proc Natl Acad Sci USA 109:3253-3258]. However, is
             their early lexicon structured and acquired like older
             learners? We test 6-mo-olds for a hallmark of the mature
             lexicon: cross-word relations. We also examine whether
             properties of the home environment that have been linked
             with lexical knowledge in older children are detectable in
             the initial stage of comprehension. We use a new dataset,
             which includes in-lab comprehension and home measures from
             the same infants. We find evidence for cross-word structure:
             On seeing two images of common nouns, infants looked
             significantly more at named target images when the
             competitor images were semantically unrelated (e.g., milk
             and foot) than when they were related (e.g., milk and
             juice), just as older learners do. We further find initial
             evidence for home-lab links: common noun "copresence" (i.e.,
             whether words' referents were present and attended to in
             home recordings) correlated with in-lab comprehension. These
             findings suggest that, even in neophyte word learners,
             cross-word relations are formed early and the home learning
             environment measurably helps shape the lexicon from the
             outset.},
   Doi = {10.1073/pnas.1712966114},
   Key = {fds330846}
}

@article{fds325486,
   Author = {Frank, MC and Bergelson, E and Bergmann, C and Cristia, A and Floccia,
             C and Gervain, J and Hamlin, JK and Hannon, EE and Kline, M and Levelt, C and Lew-Williams, C and Nazzi, T and Panneton, R and Rabagliati, H and Soderstrom, M and Sullivan, J and Waxman, S and Yurovsky,
             D},
   Title = {A Collaborative Approach to Infant Research: Promoting
             Reproducibility, Best Practices, and Theory-Building},
   Journal = {Infancy},
   Volume = {22},
   Number = {4},
   Pages = {421-435},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1111/infa.12182},
   Abstract = {© 2017 International Congress of Infant Studies (ICIS).The
             ideal of scientific progress is that we accumulate
             measurements and integrate these into theory, but recent
             discussion of replicability issues has cast doubt on whether
             psychological research conforms to this model. Developmental
             research-especially with infant participants-also has
             discipline-specific replicability challenges, including
             small samples and limited measurement methods. Inspired by
             collaborative replication efforts in cognitive and social
             psychology, we describe a proposal for assessing and
             promoting replicability in infancy research: large-scale,
             multi-laboratory replication efforts aiming for a more
             precise understanding of key developmental phenomena. The
             ManyBabies project, our instantiation of this proposal, will
             not only help us estimate how robust and replicable these
             phenomena are, but also gain new theoretical insights into
             how they vary across ages, linguistic communities, and
             measurement methods. This project has the potential for a
             variety of positive outcomes, including less-biased
             estimates of theoretically important effects, estimates of
             variability that can be used for later study planning, and a
             series of best-practices blueprints for future infancy
             research.},
   Doi = {10.1111/infa.12182},
   Key = {fds325486}
}

@article{fds327239,
   Author = {Bergelson, E and Swingley, D},
   Title = {Young Infants' Word Comprehension Given An Unfamiliar Talker
             or Altered Pronunciations.},
   Journal = {Child Development},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1111/cdev.12888},
   Abstract = {To understand spoken words, listeners must appropriately
             interpret co-occurring talker characteristics and speech
             sound content. This ability was tested in 6- to
             14-months-olds by measuring their looking to named food and
             body part images. In the new talker condition (n = 90),
             pictures were named by an unfamiliar voice; in the
             mispronunciation condition (n = 98), infants' mothers
             "mispronounced" the words (e.g., nazz for nose). Six- to
             7-month-olds fixated target images above chance across
             conditions, understanding novel talkers, and mothers'
             phonologically deviant speech equally. Eleven- to
             14-months-olds also understood new talkers, but performed
             poorly with mispronounced speech, indicating sensitivity to
             phonological deviation. Between these ages, performance was
             mixed. These findings highlight the changing roles of
             acoustic and phonetic variability in early word
             comprehension, as infants learn which variations alter
             meaning.},
   Doi = {10.1111/cdev.12888},
   Key = {fds327239}
}

@article{fds327381,
   Author = {Bergelson, E and Aslin, R},
   Title = {Semantic Specificity in One-Year-Olds' Word
             Comprehension.},
   Journal = {Language Learning and Development},
   Volume = {13},
   Number = {4},
   Pages = {481-501},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1080/15475441.2017.1324308},
   Abstract = {The present study investigated infants' knowledge about
             familiar nouns. Infants (n = 46, 12-20-month-olds) saw
             two-image displays of familiar objects, or one familiar and
             one novel object. Infants heard either a matching word (e.g.
             "foot' when seeing foot and juice), a related word (e.g.
             "sock" when seeing foot and juice) or a nonce word (e.g.
             "fep" when seeing a novel object and dog). Across the whole
             sample, infants reliably fixated the referent on matching
             and nonce trials. On the critical related trials we found
             increasingly less looking to the incorrect (but related)
             image with age. These results suggest that one-year-olds
             look at familiar objects both when they hear them labeled
             and when they hear related labels, to similar degrees, but
             over the second year increasingly rely on semantic fit. We
             suggest that infants' initial semantic representations are
             imprecise, and continue to sharpen over the second postnatal
             year.},
   Doi = {10.1080/15475441.2017.1324308},
   Key = {fds327381}
}


%% Bettman, James R.   
@article{fds323857,
   Author = {Escalas, JE and Bettman, JR},
   Title = {Connecting With Celebrities: How Consumers Appropriate
             Celebrity Meanings for a Sense of Belonging},
   Journal = {Journal of Advertising},
   Volume = {46},
   Number = {2},
   Pages = {297-308},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1080/00913367.2016.1274925},
   Doi = {10.1080/00913367.2016.1274925},
   Key = {fds323857}
}


%% Bilbo, Staci D.   
@article{fds326607,
   Author = {Bolton, JL and Marinero, S and Hassanzadeh, T and Natesan, D and Le, D and Belliveau, C and Mason, SN and Auten, RL and Bilbo,
             SD},
   Title = {Gestational exposure to air pollution alters cortical
             volume, microglial morphology, and microglia-neuron
             interactions in a sex-specific manner},
   Journal = {Frontiers in Synaptic Neuroscience},
   Volume = {9},
   Number = {MAY},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.3389/fnsyn.2017.00010},
   Abstract = {© 2017 Bolton, Marinero, Hassanzadeh, Natesan, Le,
             Belliveau, Mason, Auten and Bilbo.Microglia are the resident
             immune cells of the brain, important for normal neural
             development in addition to host defense in response to
             inflammatory stimuli. Air pollution is one of the most
             pervasive and harmful environmental toxicants in the modern
             world, and several large scale epidemiological studies have
             recently linked prenatal air pollution exposure with an
             increased risk of neurodevelopmental disorders such as
             autism spectrum disorder (ASD). Diesel exhaust particles
             (DEP) are a primary toxic component of air pollution, and
             markedly activate microglia in vitro and in vivo in adult
             rodents. We have demonstrated that prenatal exposure to DEP
             in mice, i.e., to the pregnant dams throughout gestation,
             results in a persistent vulnerability to behavioral deficits
             in adult offspring, especially in males, which is intriguing
             given the greater incidence of ASD in males to females
             (4:1). Moreover, there is a striking upregulation of
             toll-like receptor (TLR) 4 gene expression within the brains
             of the same mice, and this expression is primarily in
             microglia. Here we explored the impact of gestational
             exposure to DEP or vehicle on microglial morphology in the
             developing brains of male and female mice. DEP exposure
             increased inflammatory cytokine protein and altered the
             morphology of microglia, consistent with activation or a
             delay in maturation, only within the embryonic brains of
             male mice; and these effects were dependent on TLR4. DEP
             exposure also increased cortical volume at embryonic day
             (E)18, which switched to decreased volume by post-natal day
             (P)30 in males, suggesting an impact on the developing
             neural stem cell niche. Consistent with this hypothesis, we
             found increased microglial-neuronal interactions in male
             offspring that received DEP compared to all other groups.
             Taken together, these data suggest a mechanism by which
             prenatal exposure to environmental toxins may affect
             microglial development and long-term function, and thereby
             contribute to the risk of neurodevelopmental
             disorders.},
   Doi = {10.3389/fnsyn.2017.00010},
   Key = {fds326607}
}

@article{fds326608,
   Author = {Bilbo, SD},
   Title = {Sex differences in microglial appetites during development:
             Inferences and implications.},
   Journal = {Brain, Behavior, and Immunity},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.bbi.2017.05.010},
   Doi = {10.1016/j.bbi.2017.05.010},
   Key = {fds326608}
}

@article{fds326354,
   Author = {Parker, W and Hornik, CD and Bilbo, S and Holzknecht, ZE and Gentry, L and Rao, R and Lin, SS and Herbert, MR and Nevison, CD},
   Title = {The role of oxidative stress, inflammation and acetaminophen
             exposure from birth to early childhood in the induction of
             autism.},
   Journal = {The Journal of international medical research},
   Volume = {45},
   Number = {2},
   Pages = {407-438},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1177/0300060517693423},
   Abstract = {The wide range of factors associated with the induction of
             autism is invariably linked with either inflammation or
             oxidative stress, and sometimes both. The use of
             acetaminophen in babies and young children may be much more
             strongly associated with autism than its use during
             pregnancy, perhaps because of well-known deficiencies in the
             metabolic breakdown of pharmaceuticals during early
             development. Thus, one explanation for the increased
             prevalence of autism is that increased exposure to
             acetaminophen, exacerbated by inflammation and oxidative
             stress, is neurotoxic in babies and small children. This
             view mandates extreme urgency in probing the long-term
             effects of acetaminophen use in babies and the possibility
             that many cases of infantile autism may actually be induced
             by acetaminophen exposure shortly after birth.},
   Doi = {10.1177/0300060517693423},
   Key = {fds326354}
}

@article{fds325332,
   Author = {Kopec, AM and Rivera, PD and Lacagnina, MJ and Hanamsagar, R and Bilbo,
             SD},
   Title = {Optimized solubilization of TRIzol-precipitated protein
             permits Western blotting analysis to maximize data available
             from brain tissue.},
   Journal = {Journal of Neuroscience Methods},
   Volume = {280},
   Pages = {64-76},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.jneumeth.2017.02.002},
   Abstract = {Techniques simultaneously assessing multiple levels of
             molecular processing are appealing because molecular
             signaling underlying complex neural phenomena occurs at
             complementary levels. The TRIzol method isolates RNA and
             DNA, but protein retrieval is difficult due to inefficient
             solubilization of precipitated protein pellets.We optimized
             a buffer for the efficient solubilization of protein from
             TRIzol-precipitated brain tissue for Western blotting
             analysis, which was also more effective at directly
             homogenizing brain tissue than RIPA buffer.Protein yield
             during solubilization, in addition to protein yield via
             direct homogenization, is increased by optimizing
             concentrations of chemicals in a standard lysis buffer.
             Effective incubation parameters for both total protein yield
             and the analysis of post-translational modifications is
             remarkably flexible. Importantly, different neural cell
             types and protein classes are represented in solubilized
             protein samples. Moreover, we used dissociated mouse brain
             tissue to isolate microglia from other cell types and
             successfully resolved cell type-specific proteins from these
             small and difficult to attain samples.Solubilization buffers
             to date have been comprised primarily of SDS or urea; the
             data herein demonstrate that components common to lysis
             buffers can also enhance protein solubilization both after
             direct homogenization and after precipitation.This method is
             suitable for assessing gene and protein expression from a
             single brain sample, allowing for a more comprehensive
             evaluation of neural phenomena while minimizing the number
             of subjects.},
   Doi = {10.1016/j.jneumeth.2017.02.002},
   Key = {fds325332}
}

@article{fds321831,
   Author = {Lacagnina, MJ and Rivera, PD and Bilbo, SD},
   Title = {Glial and Neuroimmune Mechanisms as Critical Modulators of
             Drug Use and Abuse.},
   Journal = {Neuropsychopharmacology (Nature)},
   Volume = {42},
   Number = {1},
   Pages = {156-177},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1038/npp.2016.121},
   Abstract = {Drugs of abuse cause persistent alterations in synaptic
             plasticity that may underlie addiction behaviors. Evidence
             suggests glial cells have an essential and underappreciated
             role in the development and maintenance of drug abuse by
             influencing neuronal and synaptic functions in multifaceted
             ways. Microglia and astrocytes perform critical functions in
             synapse formation and refinement in the developing brain,
             and there is growing evidence that disruptions in glial
             function may be implicated in numerous neurological
             disorders throughout the lifespan. Linking evidence of
             function in health and under pathological conditions, this
             review will outline the glial and neuroimmune mechanisms
             that may contribute to drug-abuse liability, exploring
             evidence from opioids, alcohol, and psychostimulants. Drugs
             of abuse can activate microglia and astrocytes through
             signaling at innate immune receptors, which in turn
             influence neuronal function not only through secretion of
             soluble factors (eg, cytokines and chemokines) but also
             potentially through direct remodeling of the synapses. In
             sum, this review will argue that neural-glial interactions
             represent an important avenue for advancing our
             understanding of substance abuse disorders.},
   Doi = {10.1038/npp.2016.121},
   Key = {fds321831}
}


%% Blumenthal, James A.   
@article{fds331180,
   Author = {Smith, PJ and Snyder, LD and Palmer, SM and Hoffman, BM and Stonerock,
             GL and Ingle, KK and Saulino, CK and Blumenthal, JA},
   Title = {Depression, social support, and clinical outcomes following
             lung transplantation: a single-center cohort
             study.},
   Journal = {Transplant International},
   Volume = {31},
   Number = {5},
   Pages = {495-502},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1111/tri.13094},
   Abstract = {Depressive symptoms are common among lung transplant
             candidates and have been associated with poorer clinical
             outcomes in some studies. Previous studies have been plagued
             by methodologic problems, including small sample sizes, few
             clinical events, and uncontrolled confounders, particularly
             perioperative complications. In addition, few studies have
             examined social support as a potential protective factor. We
             therefore examined the association between pretransplant
             depressive symptoms, social support, and mortality in a
             large sample of lung transplant recipients. As a secondary
             aim, we also examined the associations between psychosocial
             factors, perioperative outcomes [indexed by hospital length
             of stay (LOS)], and mortality. We hypothesized that
             depression would be associated with longer LOS and that the
             association between depression, social support, and
             mortality would be moderated by LOS. Participants included
             lung transplant recipients, transplanted at Duke University
             Medical Center from January 2009 to December 2014.
             Depressive symptoms were evaluated using the Beck Depression
             Inventory (BDI-II) and social support using the Perceived
             Social Support Scale (PSSS). Medical risk factors included
             forced vital capacity (FVC), partial pressure of carbon
             dioxide (PCO2 ), donor age, acute rejection, and transplant
             type. Functional status was assessed using six-minute walk
             distance (6MWD). We also controlled for demographic factors,
             including age, gender, and native disease. Transplant
             hospitalization LOS was examined as a marker of
             perioperative clinical outcomes. Participants included 273
             lung recipients (174 restrictive, 67 obstructive, 26 cystic
             fibrosis, and six "other"). Pretransplant depressive
             symptoms were common, with 56 participants (21%) exhibiting
             clinically elevated levels (BDI-II ≥ 14). Greater
             depressive symptoms were associated with longer LOS
             [adjusted b = 0.20 (2 days per 7-point higher BDI-II score),
             P < 0.01]. LOS moderated the associations between depressive
             symptoms (P = 0.019), social support (P < 0.001), and
             mortality, such that greater depressive symptoms and lower
             social support were associated with greater mortality only
             among individuals with longer LOS. For individuals with LOS
             ≥ 1 month, clinically elevated depressive symptoms (BDI-II
             ≥ 14) were associated with a threefold increased risk of
             mortality (HR = 2.97). Greater pretransplant depressive
             symptoms and lower social support may be associated with
             greater mortality among a subset of individuals with worse
             perioperative outcomes.},
   Doi = {10.1111/tri.13094},
   Key = {fds331180}
}

@article{fds333684,
   Author = {Blumenthal, JA and Sherwood, A and Smith, PJ and Hinderliter,
             A},
   Title = {The Role of Salt Reduction in the Management of
             Hypertension},
   Journal = {JACC - Journal of the American College of
             Cardiology},
   Volume = {71},
   Number = {14},
   Pages = {1597-1598},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jacc.2018.01.071},
   Doi = {10.1016/j.jacc.2018.01.071},
   Key = {fds333684}
}

@article{fds330458,
   Author = {Smith, PJ and Blumenthal, JA and Hoffman, BM and Davis, RD and Palmer,
             SM},
   Title = {Postoperative cognitive dysfunction and mortality following
             lung transplantation.},
   Journal = {American Journal of Transplantation},
   Volume = {18},
   Number = {3},
   Pages = {696-703},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1111/ajt.14570},
   Abstract = {Preliminary evidence suggests that postoperative cognitive
             dysfunction (POCD) is common after lung transplantation. The
             impact of POCD on clinical outcomes has yet to be studied.
             The association between POCD and longer-term survival was
             therefore examined in a pilot study of posttransplantation
             survivors. Forty-nine participants from a prior randomized
             clinical trial underwent a neurocognitive assessment battery
             pretransplantation and 6 months posttransplantation,
             including assessments of the domains of Executive Function
             (Trail Making Test, Stroop, Digit Span), Processing Speed
             (Ruff 2 and 7 Test, Digit Symbol Substitution Test), and
             Verbal Memory (Verbal Paired Associates, Logical Memory,
             Animal Naming, and Controlled Oral Word Association Test).
             During a 13-year follow-up, 33 (67%) participants died.
             Greater neurocognition was associated with longer survival
             (hazard ratio [HR] = 0.49 [0.25-0.96], P = .039), and
             this association was strongest on tests assessing Processing
             Speed (HR = 0.58 [0.36-0.95], P = .03) and Executive
             Function (HR = 0.52 [0.28-0.97], P = .040). In addition,
             unadjusted analyses suggested an association between greater
             Memory performance and lower risk of CLAD (HR = 0.54
             [0.29-1.00], P = .050). Declines in Executive Function
             tended to be predictive of worse survival. These preliminary
             findings suggest that postoperative neurocognition is
             predictive of subsequent mortality among lung transplant
             recipients. Further research is needed to confirm these
             findings in a larger sample and to examine mechanisms
             responsible for this relationship.},
   Doi = {10.1111/ajt.14570},
   Key = {fds330458}
}

@article{fds329487,
   Author = {Sherwood, A and Hill, LK and Blumenthal, JA and Hinderliter,
             AL},
   Title = {Circadian hemodynamics in men and women with high blood
             pressure: dipper vs. nondipper and racial
             differences.},
   Journal = {Journal of Hypertension},
   Volume = {36},
   Number = {2},
   Pages = {250-258},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1097/hjh.0000000000001533},
   Abstract = {The 'nondipping' pattern of circadian blood pressure (BP)
             variation is an established independent predictor of adverse
             cardiovascular outcomes. Although this phenomenon has been
             widely studied, its underlying circadian hemodynamics of
             cardiac output and systemic vascular resistance (SVR) have
             not been well characterized. We evaluated the hypothesis
             that BP nondipping would be associated with a blunted
             night-time reduction in SVR in a biracial sample of 140 (63
             African-American and 77 white) men and women with elevated
             clinic BP (130-159/85-99 mmHg).Twenty-four-hour ambulatory
             hemodynamics were assessed using standard ambulatory BP
             monitoring coupled with synchronized ambulatory impedance
             cardiography. Using the criterion of less than 10% dip in
             SBP, there were 51 nondippers (SBP dip = 7.3 ± 2.6%)
             and 89 dippers (SBP dip = 15.5 ± 3.4%). There was
             minimal change in cardiac output from daytime to night-time
             in both dippers and nondippers. However, SVR decreased from
             daytime to night-time, but nondippers compared with dippers
             exhibited a significantly attenuated decrease in SVR from
             daytime to night-time (7.8 vs. 16.1%, P < 0.001).
             Relative to their white counterparts, African-Americans also
             exhibited blunted SBP dipping (10.9 vs. 14.6%,
             P < 0.001) as well as an attenuated decrease in SVR
             (10.8 vs. 15.6%, P < 0.001).Overall, these findings
             indicate that blunted night-time BP dipping is associated
             with impairment of the systemic vasodilation that is
             characteristic of the night-time sleep period and is
             especially prominent among African-Americans. In the context
             of high BP, these findings suggest that nondipping may be a
             manifestation, or marker, of more advanced vascular
             disease.},
   Doi = {10.1097/hjh.0000000000001533},
   Key = {fds329487}
}

@article{fds331882,
   Author = {Hill, LK and Sherwood, A and McNeilly, M and Anderson, NB and Blumenthal, JA and Hinderliter, AL},
   Title = {Impact of Racial Discrimination and Hostility on Adrenergic
             Receptor Responsiveness in African American
             Adults.},
   Journal = {Psychosomatic Medicine},
   Volume = {80},
   Number = {2},
   Pages = {208-215},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1097/psy.0000000000000547},
   Abstract = {Racial discrimination is increasingly recognized as a
             contributor to increased cardiovascular disease (CVD) risk
             among African Americans. Previous research has shown
             significant overlap between racial discrimination and
             hostility, an established predictor of CVD risk including
             alterations in adrenergic receptor functioning. The present
             study examined the associations of racial discrimination and
             hostility with adrenergic receptor responsiveness.In a
             sample (N = 57) of young to middle-aged African American
             adults (51% female) with normal and mildly elevated blood
             pressure, a standardized isoproterenol sensitivity test
             (CD25) was used to evaluate β-AR responsiveness, whereas
             the dose of phenylephrine required to increase mean arterial
             pressure by 25 mm Hg (PD25) was used to assess α1-AR
             responsiveness. Racial discrimination was measured using the
             Perceived Racism Scale and hostility was assessed using the
             Cook-Medley Hostility Scale.In hierarchical regression
             models, greater racial discrimination, but not hostility,
             emerged as a significant predictor of decreased
             β-adrenergic receptor responsiveness (β = .38, p = .004).
             However, moderation analysis revealed that the association
             between racial discrimination and blunted β-adrenergic
             receptor responsiveness was strongest among those with
             higher hostility (β = .49, 95% confidence interval =
             .17-.82, p = .004). In addition, hostility, but not racial
             discrimination, significantly predicted α1-AR
             responsiveness.These findings suggest racial discrimination
             was associated with blunted β-adrenergic receptor
             responsiveness, providing further evidence of the potential
             contribution of racial discrimination to increased CVD risk
             among African Americans. The adverse effects of
             discrimination on cardiovascular health may be enhanced in
             individuals with higher levels of hostility.},
   Doi = {10.1097/psy.0000000000000547},
   Key = {fds331882}
}

@article{fds332217,
   Author = {Smith, PJ and Sherwood, A and Blumenthal, JA},
   Title = {Effects of aerobic exercise on white matter
             hyperintensities: An exploratory analysis.},
   Journal = {General Hospital Psychiatry},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.genhosppsych.2018.01.007},
   Doi = {10.1016/j.genhosppsych.2018.01.007},
   Key = {fds332217}
}

@article{fds330457,
   Author = {Farquhar, JM and Smith, PJ and Snyder, L and Gray, AL and Reynolds, JM and Blumenthal, JA},
   Title = {Patterns and predictors of pain following lung
             transplantation.},
   Journal = {General Hospital Psychiatry},
   Volume = {50},
   Pages = {125-130},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.genhosppsych.2017.11.007},
   Abstract = {Our objective was to examine variability in pain levels
             following lung transplantation, and examine individual
             biopsychosocial factors influencing changes in pain.We
             performed a retrospective study of a cohort of 150 patients
             transplanted and discharged from Duke University Hospital
             between January 2015 and September 2016. During
             hospitalization and at clinic visits up to two months after
             discharge, subjective pain ratings were obtained using a
             0-10 Numeric Rating Scale. Psychiatric diagnoses of anxiety
             and depression and Center for Epidemiological Studies -
             Depression (CES-D) scores collected after hospital discharge
             were examined as predictors of post-surgery pain. Medical
             and surgical variables were examined as covariates.During
             hospitalization, pain ratings decreased over time (p<0.001).
             Predictors of higher pain levels included pre-transplant
             history of depression (p=0.001) and anxiety (p=0.04),
             bilateral lung transplant (p=0.03), and lower six-minute
             walk distance (p=0.02). Two months after discharge, 18% of
             patients reported continued pain and 34% remained on opioid
             pain medications. Two months after discharge, more frequent
             post-operative complications predicted higher pain levels in
             a univariate analysis (p=0.02) although this relationship
             was attenuated after adjustment for depression. In a
             multivariate analysis, elevated CES-D scores (p=0.002), and
             greater opioid use (p=0.031) predicted higher pain levels
             2-months post-discharge.We conclude that patients with
             psychiatric comorbidities may be at risk for greater pain,
             and may require additional strategies for more effective
             pain management.},
   Doi = {10.1016/j.genhosppsych.2017.11.007},
   Key = {fds330457}
}

@article{fds330347,
   Author = {Nieuwsma, JA and Williams, JW and Namdari, N and Washam, JB and Raitz,
             G and Blumenthal, JA and Jiang, W and Yapa, R and McBroom, AJ and Lallinger, K and Schmidt, R and Kosinski, AS and Sanders,
             GD},
   Title = {Diagnostic Accuracy of Screening Tests and Treatment for
             Post-Acute Coronary Syndrome Depression: A Systematic
             Review.},
   Journal = {Annals of internal medicine},
   Volume = {167},
   Number = {10},
   Pages = {725-735},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.7326/m17-1811},
   Abstract = {Patients who have had an acute coronary syndrome (ACS) event
             have an increased risk for depression.To evaluate the
             diagnostic accuracy of depression screening instruments and
             to compare safety and effectiveness of depression treatments
             in adults within 3 months of an ACS event.MEDLINE, EMBASE,
             PsycINFO, CINAHL, and Cochrane Database of Systematic
             Reviews from January 2003 to August 2017, and a manual
             search of citations from key primary and review
             articles.English-language studies of post-ACS patients that
             evaluated the diagnostic accuracy of depression screening
             tools or compared the safety and effectiveness of a broad
             range of pharmacologic and nonpharmacologic depression
             treatments.2 investigators independently screened each
             article for inclusion; abstracted the data; and rated the
             quality, applicability, and strength of evidence.Evidence
             from 6 of the 10 included studies showed that a range of
             depression screening instruments produces acceptable levels
             of diagnostic sensitivity, specificity, and negative
             predictive values (70% to 100%) but low positive predictive
             values (below 50%). The Beck Depression Inventory-II was the
             most studied tool. A large study found that a combination of
             cognitive behavioral therapy (CBT) and antidepressant
             medication improved depression symptoms, mental
             health-related function, and overall life satisfaction more
             than usual care.Few studies, no evaluation of the influence
             of screening on clinical outcomes, and no studies addressing
             several clinical interventions of interest.Depression
             screening instruments produce diagnostic accuracy metrics
             that are similar in post-ACS patients and other clinical
             populations. Depression interventions have an uncertain
             effect on cardiovascular outcomes, but CBT combined with
             antidepressant medication produces modest improvement in
             psychosocial outcomes.Agency for Healthcare Research and
             Quality (PROSPERO: CRD42016047032).},
   Doi = {10.7326/m17-1811},
   Key = {fds330347}
}

@article{fds331883,
   Author = {Smith, PJ and Sherwood, A and Mabe, S and Watkins, L and Hinderliter, A and Blumenthal, JA},
   Title = {Physical activity and psychosocial function following
             cardiac rehabilitation: One-year follow-up of the ENHANCED
             study.},
   Journal = {General Hospital Psychiatry},
   Volume = {49},
   Pages = {32-36},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.genhosppsych.2017.05.001},
   Abstract = {To examine the long-term association between physical
             activity (PA) and psychosocial functioning following
             completion of cardiac rehabilitation (CR) among participants
             in the ENHANCED study.ENHANCED was a 3-month clinical trial
             examining standard CR with and without stress management
             training (SMT). Participants completed the Leisure-Time
             Exercise Questionnaire (LTEQ), Beck Depression Inventory-II
             (BDI-II), and the Spielberger State-Trait Anxiety Inventory
             (STAI) at baseline, 3- and 12-months after randomization.
             Prospective associations between physical activity and
             psychosocial function, as well as treatment group
             differences, were examined using repeated measures mixed
             modeling.One hundred nineteen participants were available at
             12month follow-up. During the 12-month follow-up, PA
             remained higher compared to baseline (P<0.001), with the
             majority of participants reporting that they engaged in PA
             on a regular basis. Depressive and anxious symptoms
             continued to remain lower than baseline (Ps<0.001) Higher
             levels of PA at 12-month follow-up were associated with
             lower depressive (P=0.032) and anxious (P=0.003)
             symptoms.Higher physical activity levels following CR are
             associated with lower depressive and anxious symptoms.
             Encouraging patients to sustain higher levels of PA may
             promote both cardiovascular and mental health.},
   Doi = {10.1016/j.genhosppsych.2017.05.001},
   Key = {fds331883}
}

@article{fds330037,
   Author = {Tyson, CC and Smith, PJ and Sherwood, A and Mabe, S and Hinderliter, AL and Blumenthal, JA},
   Title = {Association between normal or mildly reduced kidney
             function, cardiovascular risk and biomarkers for
             atherosclerosis: results from the ENCORE
             trial.},
   Journal = {CKJ: Clinical Kidney Journal},
   Volume = {10},
   Number = {5},
   Pages = {666-671},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1093/ckj/sfx025},
   Abstract = {Moderate-to-severe kidney dysfunction is associated with
             atherosclerotic cardiovascular disease (ASCVD). Gradations
             of normal or mildly reduced kidney function may also
             associate with ASCVD risk.We conducted a secondary analysis
             using baseline data from the Exercise and Nutritional
             Interventions for Cardiovascular Health (ENCORE) trial.
             Participants were sedentary, overweight and obese adults
             with unmedicated pre-hypertension or Stage I hypertension
             and an estimated glomerular filtration rate (eGFR) ≥60
             mL/min/1.73 m2. The Pooled Cohorts Equations were used to
             estimate a 10-year risk for first ASCVD event. Carotid
             artery intima-media thickness (IMT) and brachial artery
             flow-mediated dilation (FMD) were measured to assess
             subclinical atherosclerosis and vascular endothelial
             function, respectively. Using linear regression, we examined
             the association between eGFR and ASCVD risk, IMT and
             FMD.Participants (N = 139) were predominantly women
             (65%), white (60%), with a mean age of 52.0 ± 9.6 years
             and mean eGFR of 89.1 ± 15.0 mL/min/1.73 m2. Lower
             eGFR of 15 mL/min/1.73 m2 was associated with higher
             ASCVD risk [b = -2.7% (95% confidence interval: -3.7,
             -1.8%), P < 0.001], higher IMT [b = 0.05 mm (0.03,
             0.08 mm), P < 0.001] and lower FMD [b = -0.87%
             (-1.64, -0.11%), P = 0.026]. Compared with
             eGFR ≥90 mL/min/1.73 m2, those with eGFR
             60-89 mL/min/1.73 m2 had higher mean ASCVD risk (7.6%
             versus 2.7%; P < 0.001), greater mean IMT (0.74 mm
             versus 0.66 mm; P < 0.001) and lower mean FMD (2.0%
             versus 3.7%; P = 0.026). After controlling for CVD risk
             factors, the association between eGFR and IMT remained
             significant (P < 0.001), and eGFR and FMD trended toward
             significance (P = 0.08).Among overweight and obese
             adults with unmedicated high blood pressure and
             eGFR ≥60 mL/min/1.73 m2, lower eGFR is associated
             with a greater 10-year risk for first ASCVD event, higher
             IMT and relatively impaired FMD.},
   Doi = {10.1093/ckj/sfx025},
   Key = {fds330037}
}

@article{fds327848,
   Author = {Sherwood, A and Hill, LK and Blumenthal, JA and Adams, KF and Paine, NJ and Koch, GG and O'Connor, CM and Johnson, KS and Hinderliter,
             AL},
   Title = {Blood pressure reactivity to psychological stress is
             associated with clinical outcomes in patients with heart
             failure.},
   Journal = {American Heart Journal},
   Volume = {191},
   Pages = {82-90},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.ahj.2017.07.003},
   Abstract = {Cardiovascular (CV) reactivity to psychological stress has
             been implicated in the development and exacerbation of
             cardiovascular disease (CVD). Although high CV reactivity
             traditionally is thought to convey greater risk of CVD, the
             relationship between reactivity and clinical outcomes is
             inconsistent and may depend on the patient population under
             investigation. The present study examined CV reactivity in
             patients with heart failure (HF) and its potential
             association with long-term clinical outcomes.One hundred
             ninety-nine outpatients diagnosed with HF, with ejection
             fraction ≤40%, underwent an evaluation of blood pressure
             (BP) and heart rate reactivity to a laboratory-based
             simulated public-speaking stressor. Cox proportional hazards
             regression models were used to examine the prospective
             association between BP and heart rate reactivity on a
             combined end point of death or CV hospitalization over a
             5-year median follow-up period.Both systolic blood pressure
             (SBP) and diastolic blood pressure (DBP) reactivity,
             quantified as continuous variables, were inversely related
             to risk of death or CV hospitalization (Ps < .01) after
             controlling for established risk factors, including HF
             disease severity and etiology. In similar models, heart rate
             reactivity was unrelated to outcome (P = .12). In models
             with tertiles of reactivity, high SBP reactivity, compared
             with intermediate SBP reactivity, was associated with lower
             risk (hazard ratio [HR] = .498, 95% CI .335-.742, P =.001),
             whereas low SBP reactivity did not differ from intermediate
             reactivity. For DBP, high reactivity was marginally
             associated with lower risk compared with intermediate DBP
             reactivity (HR = .767, 95% CI .515-1.14, P =.193), whereas
             low DBP reactivity was associated with greater risk (HR =
             1.49, 95% CI 1.027-2.155, P =.0359). No relationship of
             heart rate reactivity to outcome was identified.For HF
             patients with reduced ejection fraction, a robust increase
             in BP evoked by a laboratory-based psychological challenge
             was associated with lower risk for adverse CVD events and
             may be a novel and unique marker of left ventricular
             systolic reserve that is accompanied by a more favorable
             long-term prognosis.},
   Doi = {10.1016/j.ahj.2017.07.003},
   Key = {fds327848}
}

@article{fds330038,
   Author = {Hayano, J and Yasuma, F and Watanabe, E and Carney, RM and Stein, PK and Blumenthal, JA and Arsenos, P and Gatzoulis, KA and Takahashi, H and Ishii, H and Kiyono, K and Yamamoto, Y and Yoshida, Y and Yuda, E and Kodama, I},
   Title = {Blunted cyclic variation of heart rate predicts mortality
             risk in post-myocardial infarction, end-stage renal disease,
             and chronic heart failure patients.},
   Journal = {Europace (Elsevier)},
   Volume = {19},
   Number = {8},
   Pages = {1392-1400},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1093/europace/euw222},
   Abstract = {Cyclic variation of heart rate (CVHR) associated with
             sleep-disordered breathing is thought to reflect cardiac
             autonomic responses to apnoeic/hypoxic stress. We examined
             whether blunted CVHR observed in ambulatory ECG could
             predict the mortality risk.CVHR in night-time Holter ECG was
             detected by an automated algorithm, and the prognostic
             relationships of the frequency (FCV) and amplitude (ACV) of
             CVHR were examined in 717 patients after myocardial
             infarction (post-MI 1, 6% mortality, median follow-up 25
             months). The predictive power was prospectively validated in
             three independent cohorts: a second group of 220 post-MI
             patients (post-MI 2, 25.5% mortality, follow-up 45 months);
             299 patients with end-stage renal disease on chronic
             haemodialysis (ESRD, 28.1% mortality, follow-up 85 months);
             and 100 patients with chronic heart failure (CHF, 35%
             mortality, follow-up 38 months). Although CVHR was observed
             in ≥96% of the patients in all cohorts, FCV did not
             predict mortality in any cohort. In contrast, decreased ACV
             was a powerful predictor of mortality in the post-MI 1
             cohort (hazard ratio [95% CI] per 1 ln [ms] decrement, 2.9
             [2.2-3.7], P < 0.001). This prognostic relationship was
             validated in the post-MI 2 (1.8 [1.4-2.2], P < 0.001), ESRD
             (1.5 [1.3-1.8], P < 0.001), and CHF (1.4 [1.1-1.8], P =
             0.02) cohorts. The prognostic value of ACV was independent
             of age, gender, diabetes, β-blocker therapy, left
             ventricular ejection fraction, sleep-time mean R-R interval,
             and FCV.Blunted CVHR detected by decreased ACV in a
             night-time Holter ECG predicts increased mortality risk in
             post-MI, ESRD, and CHF patients.},
   Doi = {10.1093/europace/euw222},
   Key = {fds330038}
}

@article{fds326740,
   Author = {Tussing-Humphreys, L and Lamar, M and Blumenthal, JA and Babyak, M and Fantuzzi, G and Blumstein, L and Schiffer, L and Fitzgibbon,
             ML},
   Title = {Building research in diet and cognition: The BRIDGE
             randomized controlled trial.},
   Journal = {Contemporary Clinical Trials},
   Volume = {59},
   Pages = {87-97},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.cct.2017.06.003},
   Abstract = {Obesity has been linked to cognitive impairment, cognitive
             decline and dementia. Given that 38.5% of U.S. adults
             60years and older are obese and these numbers are rapidly
             increasing, strategies to decouple obesity from cognitive
             decline are needed. Innovative lifestyle strategies that may
             postpone the onset of subclinical symptoms or even arrest
             the transition to overt dementia in at-risk individuals are
             critically needed. Poor diet is central to the development
             of obesity and diet may affect cognition. Adherence to a
             Mediterranean Diet (MedDiet) is associated with reduced risk
             of cognitive impairment and dementia. Furthermore, weight
             loss through caloric restriction improves cognitive
             function. This paper describes the Building Research in Diet
             and CoGnition (BRIDGE) study, a randomized trial examining
             the effect of the MedDiet, with and without weight loss, on
             cognitive functioning in obese older adults. Obese (BMI≥30
             and ≤50kg/m2) older adults (≥55years) (n=180) will be
             randomized in a 2:2:1 allocation scheme to: Typical Diet
             Control; MedDiet alone, without weight loss; or MedDiet
             lifestyle intervention to promote weight loss and weight
             loss maintenance. Both MedDiet intervention groups will meet
             for one individual session and 27 group sessions over an
             8-month period. Individuals in the control group will not
             receive instruction on changing lifestyle habits. Outcomes
             will be assessed at baseline, 8 and 14months. The primary
             outcome is cognitive functioning; secondary outcomes will
             include changes in body weight, diet, cardiovascular,
             metabolic, and inflammatory biomarkers.},
   Doi = {10.1016/j.cct.2017.06.003},
   Key = {fds326740}
}

@article{fds326855,
   Author = {Blumenthal, JA and Smith, PJ and Mabe, S and Hinderliter, A and Welsh-Bohmer, K and Browndyke, JN and Lin, P-H and Kraus, W and Doraiswamy, PM and Burke, J and Sherwood, A},
   Title = {Lifestyle and Neurocognition in Older Adults With
             Cardiovascular Risk Factors and Cognitive
             Impairment.},
   Journal = {Psychosomatic Medicine},
   Volume = {79},
   Number = {6},
   Pages = {719-727},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1097/psy.0000000000000474},
   Abstract = {The aim of the study was to determine the relationship of
             lifestyle factors and neurocognitive functioning in older
             adults with vascular risk factors and cognitive impairment,
             no dementia (CIND).One hundred sixty adults (M [SD] = 65.4
             [6.8] years) with CIND completed neurocognitive assessments
             of executive function, processing speed, and memory.
             Objective measures of physical activity using accelerometry,
             aerobic capacity determined by exercise testing, and dietary
             habits quantified by the Food Frequency Questionnaire and
             4-Day Food Diary to assess adherence to the Mediterranean
             and Dietary Approaches to Stop Hypertension (DASH) diets
             were obtained to assess direct effects with neurocognition.
             Potential indirect associations of high-sensitivity
             C-reactive protein and the Framingham Stroke Risk Profile
             also were examined.Greater aerobic capacity (β = 0.24) and
             daily physical activity (β = 0.15) were associated with
             better executive functioning/processing speed and verbal
             memory (βs = 0.24; 0.16). Adherence to the DASH diet was
             associated with better verbal memory (β = 0.17). Greater
             high-sensitivity C-reactive protein (βs = -0.14; -0.21) and
             Framingham Stroke Risk Profile (β = -0.18; -0.18) were
             associated with poorer executive functioning/processing
             speed and verbal memory. Greater stroke risk partially
             mediated the association of aerobic capacity with executive
             functioning/processing speed, and verbal memory and greater
             inflammation partially mediated the association of physical
             activity and aerobic fitness, with verbal memory.Higher
             levels of physical activity, aerobic fitness, and adherence
             to the DASH diet are associated with better neurocognitive
             performance in adults with CIND. These findings suggest that
             the adoption of healthy lifestyle habits could reduce the
             risk of neurocognitive decline in vulnerable older
             adults.NCT01573546.},
   Doi = {10.1097/psy.0000000000000474},
   Key = {fds326855}
}

@article{fds326632,
   Author = {Hill, LK and Watkins, LL and Hinderliter, AL and Blumenthal, JA and Sherwood, A},
   Title = {Racial differences in the association between heart rate
             variability and left ventricular mass.},
   Journal = {Experimental Physiology},
   Volume = {102},
   Number = {7},
   Pages = {764-772},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1113/ep086228},
   Abstract = {What is the central question of this study? Decreased heart
             rate variability (HRV) is associated with increased
             cardiovascular disease (CVD) risk, including greater left
             ventricular mass (LVM). Despite their enhanced CVD risk
             profile, African Americans have been shown to exhibit higher
             HRV, relative to Whites; however, it is unclear whether this
             pattern extends to the association between HRV and LVM. What
             is the main finding and its importance? Using ECG and
             echocardiographic data, HRV was positively associated with
             LVM in a non-clinical sample of African Americans. These
             findings suggest that current assumptions regarding the
             meaning of higher HRV might not be universal, which might
             have implications for HRV as a risk marker among African
             Americans. Increased left ventricular mass (LVM) is an early
             precursor of target organ damage attributable to
             hypertension. Diminished parasympathetic cardiac control has
             been linked to both hypertension onset and left ventricular
             impairment; however, emerging evidence suggests that this
             pattern might be different in African Americans. The present
             study sought to determine whether race impacts the
             relationship between parasympathetic cardiac control and
             LVM. The LVM was assessed via echocardiography in a sample
             (n = 148) of African American and White adults (mean
             age 33.20 ± 5.71 years) with normal or mildly elevated
             blood pressure. Parasympathetic cardiac control was assessed
             by a measure of high-frequency heart rate variability
             (HF-HRV) determined from ECG recordings during 5 min of
             rest. In regression analysis, greater HF-HRV was associated
             with greater LVM among African Americans (P = 0.002) but
             was not related to LVM in Whites (P = 0.919). These are
             the first data to demonstrate that race moderates the
             relationship between HRV and LVM and further suggest that
             race might be an important factor in the association between
             parasympathetic cardiac control and other cardiovascular
             disease risk factors.},
   Doi = {10.1113/ep086228},
   Key = {fds326632}
}

@article{fds325510,
   Author = {Sherwood, A and Hill, LK and Blumenthal, JA and Johnson, KS and Hinderliter, AL},
   Title = {Race and sex differences in cardiovascular α-adrenergic and
             β-adrenergic receptor responsiveness in men and women with
             high blood pressure.},
   Journal = {Journal of Hypertension},
   Volume = {35},
   Number = {5},
   Pages = {975-981},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1097/hjh.0000000000001266},
   Abstract = {Hypertension is associated with unfavorable changes in
             adrenergic receptor responsiveness, but the relationship of
             race and sex to adrenergic receptor responsiveness in the
             development of cardiovascular disease is unclear. This study
             examined α-adrenergic and ß-adrenergic receptor
             responsiveness in African-American and white men and women
             with untreated high blood pressure (BP) (HBP) and with
             normal BP.The study sample comprised 161 African-American
             and white men and women in the age range 25-45 years.
             Isoproterenol, a nonselective ß-adrenergic receptor
             agonist, was administered intravenously to determine the
             bolus dose required to increase heart rate by 25 bpm, an
             index of β-adrenergic receptor responsiveness. Similarly,
             phenylephrine, an α1-adrenergic receptor agonist, was
             administered to determine the bolus dose required to
             increase BP by 25 mmHg, an index of vascular
             α1-adrenergic receptor responsiveness. HBP (P < 0.01),
             male sex (P = 0.04), and higher BMI (P < 0.01) were
             all associated with reduced β-adrenergic receptor
             responsiveness, with a similar trend observed for
             African-American race (P = 0.07). Conversely,
             α1-adrenergic receptor responsiveness was increased in
             association with HBP (P < 0.01), female sex
             (P < 0.01), and African-American race (P < 0.01).In
             the early stages of hypertension, cardiovascular
             β-adrenergic receptors demonstrate blunted responsiveness,
             whereas conversely α1-adrenergic receptors exhibit
             increased responsiveness. This pattern of receptor changes
             is especially evident in men and African-Americans, is
             exacerbated by obesity, and may contribute to the
             development of cardiovascular disease.},
   Doi = {10.1097/hjh.0000000000001266},
   Key = {fds325510}
}

@article{fds329488,
   Author = {Hill, LK and Sherwood, A and Blumenthal, JA and Hinderliter,
             AL},
   Title = {SYNERGISTIC IMPACT OF PERCEIVED DISCRIMINATION AND HOSTILITY
             ON ADRENERGIC RESPONSIVITY},
   Journal = {Psychosomatic Medicine},
   Volume = {79},
   Number = {4},
   Pages = {A85-A85},
   Year = {2017},
   Month = {May},
   Key = {fds329488}
}

@article{fds321706,
   Author = {Stonerock, GL and Blumenthal, JA},
   Title = {Role of Counseling to Promote Adherence in Healthy Lifestyle
             Medicine: Strategies to Improve Exercise Adherence and
             Enhance Physical Activity.},
   Journal = {Progress in Cardiovascular Diseases},
   Volume = {59},
   Number = {5},
   Pages = {455-462},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.pcad.2016.09.003},
   Abstract = {Although healthy lifestyles (HL) offer a number of health
             benefits, nonadherence to recommended lifestyle changes
             remains a frequent and difficult obstacle to realizing these
             benefits. Behavioral counseling can improve adherence to an
             HL. However, individuals' motivation for change and
             resistance to altering unhealthy habits must be considered
             when developing an effective approach to counseling. In the
             present article, we review psychological, behavioral, and
             environmental factors that may promote adherence and
             contribute to nonadherence. We discuss two established
             models for counseling, motivational interviewing and the
             transtheoretical model of behavior change, and provide an
             example of how these approaches can be used to counsel
             patients to exercise and increase their levels of physical
             activity.},
   Doi = {10.1016/j.pcad.2016.09.003},
   Key = {fds321706}
}

@article{fds323315,
   Author = {Smith, PJ and Blumenthal, JA and Snyder, LD and Mathew, JP and Durheim,
             MT and Hoffman, BM and Rivelli, SK and Palmer, SM},
   Title = {Depressive symptoms and early mortality following lung
             transplantation: A pilot study.},
   Journal = {Clinical Transplantation},
   Volume = {31},
   Number = {2},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1111/ctr.12874},
   Abstract = {Impaired psychological function is common among lung
             transplant candidates and may affect clinical outcomes
             following transplantation. Although numerous studies have
             examined the relationship between pretransplant depression,
             quality of life (QoL), and post-transplant outcomes, few
             have examined the relationship between depression and QoL
             shortly following transplantation and subsequent clinical
             outcomes. We therefore examined the association between
             depression, QoL, and short-term mortality in a consecutive
             series of lung transplant recipients.Depression (Patient
             Health Questionnaire-9; Hospital Anxiety and Depression
             Scale; Centers for Epidemiologic Studies Depression Scale)
             and QoL (UCSD Shortness of Breath Questionnaire; Pulmonary
             Quality of Life Scale) were assessed prior to
             transplantation (median 0.9 months [IQR=1.6]) and again
             approximately 2 weeks following transplantation
             (median=0.5 months [IQR=0.5]), in a series of 66 patients
             transplanted between March 2013 and April 2014. The
             association between psychiatric diagnoses from participants'
             comprehensive pretransplant assessment and mortality also
             was examined. Cox proportional hazards models were used to
             examine the association between depression, QoL, and
             mortality.During a median follow-up of 2.8 years (range
             0.4-3.3), 21 patients died (32%). Greater depressive
             symptoms assessed shortly after transplant were associated
             with subsequent mortality (HR=2.17 [1.01, 4.67], P=.048),
             and this relationship persisted after controlling for
             primary graft dysfunction, duration of transplant
             hospitalization, and gender. In contrast, neither
             pretransplant depression, history of depression, nor QoL was
             associated with mortality.Greater post-transplant depressive
             symptoms are independently associated with mortality among
             lung transplant recipients.},
   Doi = {10.1111/ctr.12874},
   Key = {fds323315}
}

@article{fds329489,
   Author = {Sherwood, A and Smith, PJ and Hinderliter, AL and Georgiades, A and Blumenthal, JA},
   Title = {Effects of exercise and stress management training on
             nighttime blood pressure dipping in patients with coronary
             heart disease: A randomized, controlled trial.},
   Journal = {American Heart Journal},
   Volume = {183},
   Pages = {85-90},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.ahj.2016.10.011},
   Abstract = {Blunted nighttime blood pressure (BP) dipping is prognostic
             of cardiovascular morbidity and mortality. Patients with
             coronary heart disease (CHD) are often characterized by a
             blunted nighttime BP dipping pattern. The present study
             compared the effects of 2 behavioral intervention programs,
             aerobic exercise (EX) and stress management (SM) training,
             with a usual care (UC) control group on BP dipping in a
             sample of CHD patients.This was a secondary analysis of a
             randomized, controlled trial with allocation concealment and
             blinded outcome assessment in 134 patients with stable CHD
             and exercise-induced myocardial ischemia. Nighttime BP
             dipping was assessed by 24-hour ambulatory BP monitoring, at
             prerandomization baseline and after 16 weeks of one of the
             following treatments: usual medical care; UC plus supervised
             aerobic EX for 35 minutes, 3 times per week; UC plus weekly
             1.5-hour sessions of SM training.The EX and SM groups
             exhibited greater improvements in systolic BP dipping
             (P=.052) and diastolic BP dipping (P=.031) compared with UC.
             Postintervention systolic BP percent-dipping means were
             12.9% (SE=1.5) for SM, 11.1% (SE=1.4) for EX, and 8.6%
             (SE=1.4) for UC. Postintervention diastolic BP
             percent-dipping means were 13.3% (SE=1.9) for SM, 14.1%
             (SE=1.8) for EX, and 8.8% (1.8) for UC.For patients with
             stable CHD, EX or SM training resulted in improved nighttime
             BP dipping compared with usual medical care. These favorable
             effects of healthy lifestyle modifications may help reduce
             the risk of adverse clinical events.},
   Doi = {10.1016/j.ahj.2016.10.011},
   Key = {fds329489}
}

@article{fds329490,
   Author = {Sherwood, A and Blumenthal, JA and Koch, GG and Hoffman, BM and Watkins,
             LL and Smith, PJ and O'Connor, CM and Adams, KF and Rogers, JG and Sueta,
             C and Chang, PP and Johnson, KS and Schwartz, J and Hinderliter,
             AL},
   Title = {Effects of Coping Skills Training on Quality of Life,
             Disease Biomarkers, and Clinical Outcomes in Patients With
             Heart Failure: A Randomized Clinical Trial.},
   Journal = {Circulation: Heart Failure},
   Volume = {10},
   Number = {1},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1161/circheartfailure.116.003410},
   Abstract = {Heart failure (HF) is a chronic disease that compromises
             patients' quality of life (QoL). Interventions designed to
             reduce distress and improve disease self-management are
             needed. We evaluated the efficacy of a telephone-based
             coping skills training (CST) intervention.This randomized
             clinical trial involved 180 HF outpatients with reduced
             ejection fraction. Participants ranged in age from 29 to 87
             years (mean=58 years); 27% were women, and 47% were
             nonwhite. Participants were randomized to either a CST
             intervention or heart failure education, both delivered over
             16 weeks. The primary outcomes were (1) postintervention
             effects on QoL and HF disease biomarkers (both with
             α=0.01), and (2) a composite measure of time to death or
             first hospitalization (with α=0.03) over a median follow-up
             period of 3 years. CST resulted in greater improvements in
             QoL compared with heart failure education (P<0.01),
             including the Kansas City Cardiomyopathy Questionnaire
             (P=0.009), depressive symptoms (P=0.027), and the 6-minute
             walk test (P=0.012). However, it did not differentially
             improve HF disease biomarkers or reduce risk of all-cause
             hospitalizations or death (hazard ratio=0.84 [95% confidence
             interval, 0.59-1.12]). Interestingly, exploratory analyses
             showed that participants randomized to CST experienced a
             reduction in the composite end point of worsening HF
             hospitalization or death during the 3-year follow-up period
             (hazard ratio=0.65 [95% confidence interval, 0.44-0.98];
             P=0.040).CST improved QoL in patients with HF. Monitoring
             and improving QoL is emerging as an important aspect of the
             clinical management of HF that can reduce disease burden and
             may help improve clinical outcomes in this vulnerable
             patient population.URL: https://www.clinicaltrials.gov.
             Unique identifier: NCT00873418.},
   Doi = {10.1161/circheartfailure.116.003410},
   Key = {fds329490}
}


%% Bonner, Melanie J.   
@article{fds319600,
   Author = {Allen, TM and Anderson, LM and Rothman, JA and Bonner,
             MJ},
   Title = {[Formula: see text]Executive functioning and health-related
             quality of life in pediatric sickle cell
             disease.},
   Journal = {Child Neuropsychology},
   Volume = {23},
   Number = {8},
   Pages = {889-906},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1080/09297049.2016.1205011},
   Abstract = {Research consistently indicates that children with sickle
             cell disease (SCD) face multiple risk factors for
             neurocognitive impairment. Despite this, no empirical
             research to date has examined the impact of neurocognitive
             functioning on quality of life for this pediatric group.
             Thus, the current study aims to examine the relationship
             between executive functioning and quality of life in a
             sample of children with SCD and further explore psychosocial
             and family/caregiver resources as moderators of this
             relationship. A total of 45 children with SCD aged 8 to 16
             years and their caregivers completed measures of quality of
             life, behavioral ratings of executive functioning, and
             psychosocial functioning. Hierarchical linear regression
             models were utilized to determine the impact of executive
             functioning on quality of life and further test the
             interaction effects of proposed moderating variables.
             Controlling for age, pain, and socioeconomic status (SES),
             executive functioning was found to significantly predict
             child- and parent-reported quality of life among youth with
             SCD. Psychosocial resources of the primary caregiver or
             family was not found to moderate the relationship between
             executive functioning and quality of life. These results
             provide the first empirical evidence that lower executive
             skills negatively predict quality of life for children with
             SCD, supporting clinical and research efforts which aim to
             establish efficacious interventions that target cognitive
             decrements within this pediatric population.},
   Doi = {10.1080/09297049.2016.1205011},
   Key = {fds319600}
}

@article{fds330040,
   Author = {Schreiber, JE and Palmer, SL and Conklin, HM and Mabbott, DJ and Swain,
             MA and Bonner, MJ and Chapieski, ML and Huang, L and Zhang, H and Gajjar,
             A},
   Title = {Posterior fossa syndrome and long-term neuropsychological
             outcomes among children treated for medulloblastoma on a
             multi-institutional, prospective study.},
   Journal = {Neuro-Oncology},
   Volume = {19},
   Number = {12},
   Pages = {1673-1682},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1093/neuonc/nox135},
   Abstract = {Patients treated for medulloblastoma who experience
             posterior fossa syndrome (PFS) demonstrate increased risk
             for neurocognitive impairment at one year post diagnosis.
             The aim of the study was to examine longitudinal
             trajectories of neuropsychological outcomes in patients who
             experienced PFS compared with patients who did
             not.Participants were 36 patients (22 males) who experienced
             PFS and 36 comparison patients (21 males) who were matched
             on age at diagnosis and treatment exposure but did not
             experience PFS. All patients underwent serial evaluation of
             neurocognitive functioning spanning 1 to 5 years post
             diagnosis.The PFS group demonstrated lower estimated mean
             scores at 1, 3, and 5 years post diagnosis on measures of
             general intellectual ability, processing speed, broad
             attention, working memory, and spatial relations compared
             with the non-PFS group. The PFS group exhibited estimated
             mean scores that were at least one standard deviation below
             the mean for intellectual ability, processing speed, and
             broad attention across all time points and for working
             memory by 5 years post diagnosis. Processing speed was
             stable over time. Attention and working memory declined over
             time. Despite some change over time, caregiver ratings of
             executive function and behavior problem symptoms remained
             within the average range.Compared with patients who do not
             experience PFS, patients who experience PFS exhibit greater
             neurocognitive impairment, show little recovery over time,
             and decline further in some domains. Findings highlight the
             particularly high risk for long-term neurocognitive problems
             in patients who experience PFS and the need for close
             follow-up and intervention.},
   Doi = {10.1093/neuonc/nox135},
   Key = {fds330040}
}

@article{fds330041,
   Author = {Gallentine, WB and Shinnar, S and Hesdorffer, DC and Epstein, L and Nordli, DR and Lewis, DV and Frank, LM and Seinfeld, S and Shinnar, RC and Cornett, K and Liu, B and Moshé, SL and Sun, S and FEBSTAT Investigator
             Team},
   Title = {Plasma cytokines associated with febrile status epilepticus
             in children: A potential biomarker for acute hippocampal
             injury.},
   Journal = {Epilepsia},
   Volume = {58},
   Number = {6},
   Pages = {1102-1111},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1111/epi.13750},
   Abstract = {Our aim was to explore the association between plasma
             cytokines and febrile status epilepticus (FSE) in children,
             as well as their potential as biomarkers of acute
             hippocampal injury.Analysis was performed on residual
             samples of children with FSE (n = 33) as part of the
             Consequences of Prolonged Febrile Seizures in Childhood
             study (FEBSTAT) and compared to children with fever
             (n = 17). Magnetic resonance imaging (MRI) was obtained as
             part of FEBSTAT within 72 h of FSE. Cytokine levels and
             ratios of antiinflammatory versus proinflammatory cytokines
             in children with and without hippocampal T2 hyperintensity
             were assessed as biomarkers of acute hippocampal injury
             after FSE.Levels of interleukin (IL)-8 and epidermal growth
             factor (EGF) were significantly elevated after FSE in
             comparison to controls. IL-1β levels trended higher and
             IL-1RA trended lower following FSE, but did not reach
             statistical significance. Children with FSE were found to
             have significantly lower ratios of IL-1RA/IL-1β and
             IL-1RA/IL-8. Specific levels of any one individual cytokine
             were not associated with FSE. However, lower ratios of
             IL-1RA/IL-1β, IL-1RA/1L-6, and IL-1RA/ IL-8 were all
             associated with FSE. IL-6 and IL-8 levels were significantly
             higher and ratios of IL-1RA/IL-6 and IL-1RA/IL-8 were
             significantly lower in children with T2 hippocampal
             hyperintensity on MRI after FSE in comparison to those
             without hippocampal signal abnormalities. Neither individual
             cytokine levels nor ratios of IL-1RA/IL-1β or IL-1RA/IL-8
             were predictive of MRI changes. However, a lower ratio of
             IL-1RA/IL-6 was strongly predictive (odds ratio [OR] 21.5,
             95% confidence interval [CI] 1.17-393) of hippocampal T2
             hyperintensity after FSE.Our data support involvement of the
             IL-1 cytokine system, IL-6, and IL-8 in FSE in children. The
             identification of the IL-1RA/IL-6 ratio as a potential
             biomarker of acute hippocampal injury following FSE is the
             most significant finding. If replicated in another study,
             the IL-1RA/IL-6 ratio could represent a serologic biomarker
             that offers rapid identification of patients at risk for
             ultimately developing mesial temporal lobe epilepsy
             (MTLE).},
   Doi = {10.1111/epi.13750},
   Key = {fds330041}
}

@article{fds316069,
   Author = {Willard, VW and Allen, TM and Hardy, KK and Bonner,
             MJ},
   Title = {Social functioning in survivors of pediatric brain tumors:
             Contribution of neurocognitive and social-cognitive
             skills},
   Journal = {Children's Health Care},
   Volume = {46},
   Number = {2},
   Pages = {181-195},
   Year = {2017},
   Month = {April},
   ISSN = {0273-9615},
   url = {http://dx.doi.org/10.1080/02739615.2015.1124769},
   Doi = {10.1080/02739615.2015.1124769},
   Key = {fds316069}
}

@article{fds324821,
   Author = {Shashi, V and Pena, LDM and Kim, K and Burton, B and Hempel, M and Schoch,
             K and Walkiewicz, M and McLaughlin, HM and Cho, M and Stong, N and Hickey,
             SE and Shuss, CM and Undiagnosed Diseases Network, and Freemark, MS and Bellet, JS and Keels, MA and Bonner, MJ and El-Dairi, M and Butler, M and Kranz, PG and Stumpel, CTRM and Klinkenberg, S and Oberndorff, K and Alawi, M and Santer, R and Petrovski, S and Kuismin, O and Korpi-Heikkilä, S and Pietilainen, O and Aarno, P and Kurki, MI and Hoischen, A and Need, AC and Goldstein, DB and Kortüm,
             F},
   Title = {De Novo Truncating Variants in ASXL2 Are Associated with a
             Unique and Recognizable Clinical Phenotype.},
   Journal = {The American Journal of Human Genetics},
   Volume = {100},
   Number = {1},
   Pages = {179},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.ajhg.2016.12.004},
   Doi = {10.1016/j.ajhg.2016.12.004},
   Key = {fds324821}
}


%% Brownell, Kelly D.   
@article{fds331326,
   Author = {Bragg, MA and Roberto, CA and Harris, JL and Brownell, KD and Elbel,
             B},
   Title = {Marketing Food and Beverages to Youth Through
             Sports},
   Journal = {Journal of Adolescent Health},
   Volume = {62},
   Number = {1},
   Pages = {5-13},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jadohealth.2017.06.016},
   Doi = {10.1016/j.jadohealth.2017.06.016},
   Key = {fds331326}
}

@article{fds331325,
   Author = {Cooksey-Stowers, K and Schwartz, MB and Brownell,
             KD},
   Title = {Food swamps predict obesity rates better than food deserts
             in the United States},
   Journal = {International journal of environmental research and public
             health},
   Volume = {14},
   Number = {11},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.3390/ijerph14111366},
   Abstract = {© 2017 by the authors. Licensee MDPI, Basel, Switzerland.
             This paper investigates the effect of food environments,
             characterized as food swamps, on adult obesity rates. Food
             swamps have been described as areas with a high-density of
             establishments selling high-calorie fast food and junk food,
             relative to healthier food options. This study examines
             multiple ways of categorizing food environments as food
             swamps and food deserts, including alternate versions of the
             Retail Food Environment Index. We merged food outlet,
             sociodemographic and obesity data from the United States
             Department of Agriculture (USDA) Food Environment Atlas, the
             American Community Survey, and a commercial street reference
             dataset. We employed an instrumental variables (IV) strategy
             to correct for the endogeneity of food environments (i.e.,
             that individuals self-select into neighborhoods and may
             consider food availability in their decision). Our results
             suggest that the presence of a food swamp is a stronger
             predictor of obesity rates than the absence of full-service
             grocery stores. We found, even after controlling for food
             desert effects, food swamps have a positive, statistically
             significant effect on adult obesity rates. All three food
             swamp measures indicated the same positive association, but
             reflected different magnitudes of the food swamp effect on
             rates of adult obesity (p values ranged from 0.00 to 0.16).
             Our adjustment for reverse causality, using an IV approach,
             revealed a stronger effect of food swamps than would have
             been obtained by naïve ordinary least squares (OLS)
             estimates. The food swamp effect was stronger in counties
             with greater income inequality (p < 0.05) and where
             residents are less mobile (p < 0.01). Based on these
             findings, local government policies such as zoning laws
             simultaneously restricting access to unhealthy food outlets
             and incentivizing healthy food retailers to locate in
             underserved neighborhoods warrant consideration as
             strategies to increase health equity.},
   Doi = {10.3390/ijerph14111366},
   Key = {fds331325}
}

@article{fds324189,
   Author = {Roberto, CA and Brownell, KD},
   Title = {Strategic science for eating disorders research and policy
             impact.},
   Journal = {International Journal of Eating Disorders},
   Volume = {50},
   Number = {3},
   Pages = {312-314},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1002/eat.22678},
   Abstract = {Scientific research often fails to have relevance and impact
             because scientists do not engage policy makers and
             influencers in the process of identifying information needs
             and generating high priority questions. To address this
             scholarship-policy gap, we have developed a model of
             Strategic Science. This research approach involves working
             with policy makers and influencers to craft research
             questions that will answer important and timely
             policy-related questions. The goal is to create tighter
             links between research and policy and ensure findings are
             communicated efficiently to change agents best positioned to
             apply the research to policy debates. In this article, we
             lay out a model for Strategic Science and describe how this
             approach may help advance policy research and action for
             eating disorders.},
   Doi = {10.1002/eat.22678},
   Key = {fds324189}
}

@article{fds326083,
   Author = {Bleich, SN and Rimm, EB and Brownell, KD},
   Title = {U.S. Nutrition Assistance, 2018 - Modifying SNAP to Promote
             Population Health.},
   Journal = {The New England journal of medicine},
   Volume = {376},
   Number = {13},
   Pages = {1205-1207},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1056/nejmp1613222},
   Doi = {10.1056/nejmp1613222},
   Key = {fds326083}
}


%% Cabeza, Roberto   
@article{fds332826,
   Author = {Wing, EA and Iyengar, V and Hess, TM and LaBar, KS and Huettel, SA and Cabeza, R},
   Title = {Neural mechanisms underlying subsequent memory for personal
             beliefs:An fMRI study.},
   Journal = {Cognitive, Affective, & Behaviorial Neuroscience},
   Volume = {18},
   Number = {2},
   Pages = {216-231},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.3758/s13415-018-0563-y},
   Abstract = {Many fMRI studies have examined the neural mechanisms
             supporting emotional memory for stimuli that generate
             emotion rather automatically (e.g., a picture of a dangerous
             animal or of appetizing food). However, far fewer studies
             have examined how memory is influenced by emotion related to
             social and political issues (e.g., a proposal for large
             changes in taxation policy), which clearly vary across
             individuals. In order to investigate the neural substrates
             of affective and mnemonic processes associated with personal
             opinions, we employed an fMRI task wherein participants
             rated the intensity of agreement/disagreement to
             sociopolitical belief statements paired with neural face
             pictures. Following the rating phase, participants performed
             an associative recognition test in which they distinguished
             identical versus recombined face-statement pairs. The study
             yielded three main findings: behaviorally, the intensity of
             agreement ratings was linked to greater subjective emotional
             arousal as well as enhanced high-confidence subsequent
             memory. Neurally, statements that elicited strong (vs. weak)
             agreement or disagreement were associated with greater
             activation of the amygdala. Finally, a subsequent memory
             analysis showed that the behavioral memory advantage for
             statements generating stronger ratings was dependent on the
             medial prefrontal cortex (mPFC). Together, these results
             both underscore consistencies in neural systems supporting
             emotional arousal and suggest a modulation of
             arousal-related encoding mechanisms when emotion is
             contingent on referencing personal beliefs.},
   Doi = {10.3758/s13415-018-0563-y},
   Key = {fds332826}
}

@article{fds327833,
   Author = {Monge, ZA and Wing, EA and Stokes, J and Cabeza, R},
   Title = {Search and recovery of autobiographical and laboratory
             memories: Shared and distinct neural components.},
   Journal = {Neuropsychologia},
   Volume = {110},
   Pages = {44-54},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2017.07.030},
   Abstract = {Functional neuroimaging evidence suggests that there are
             differences in the neural correlates of episodic memory for
             laboratory stimuli (laboratory memory) and for events from
             one's own life (autobiographical memory). However, this
             evidence is scarce and often confounded with differences in
             memory testing procedures. Here, we directly compared the
             neural mechanisms underlying the search and recovery of
             autobiographical and laboratory memories while minimizing
             testing differences. Before scanning, participants completed
             a laboratory memory encoding task in which they studied
             four-word "chains" spread across three word pairs. During
             scanning, participants completed a laboratory memory
             retrieval task, in which they recalled the word chains, and
             an autobiographical memory retrieval task, in which they
             recalled specific personal events associated with word cues.
             Importantly, response times were similar in the two tasks,
             allowing for a direct comparison of the activation time
             courses. We found that during memory search (searching for
             the memory target), similar brain regions were activated
             during both the autobiographical and laboratory tasks,
             whereas during memory recovery (accessing the memory traces;
             i.e., ecphory), clear differences emerged: regions of the
             default mode network (DMN) were activated greater during
             autobiographical than laboratory memory, whereas the
             bilateral superior parietal lobules were activated greater
             during laboratory than autobiographical memory. Also,
             multivariate functional connectivity analyses revealed that
             regardless of memory stage, the DMN and ventral attention
             network exhibited a more integrated topology in the
             functional network underlying autobiographical (vs.
             laboratory) memory retrieval, whereas the fronto-parietal
             task control network exhibited a more integrated topology in
             the functional network underlying laboratory (vs.
             autobiographical) memory retrieval. These findings further
             characterize the shared and distinct neural components
             underlying autobiographical and laboratory memories, and
             suggest that differences in autobiographical vs. laboratory
             memory brain activation previously reported in the
             literature reflect memory recovery rather than search
             differences.},
   Doi = {10.1016/j.neuropsychologia.2017.07.030},
   Key = {fds327833}
}

@article{fds333712,
   Author = {Fandakova, Y and Sander, MC and Grandy, TH and Cabeza, R and Werkle-Bergner, M and Shing, YL},
   Title = {Age differences in false memory: The importance of retrieval
             monitoring processes and their modulation by memory
             quality.},
   Journal = {Psychology and Aging},
   Volume = {33},
   Number = {1},
   Pages = {119-133},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1037/pag0000212},
   Abstract = {Older adults are more likely than younger adults to falsely
             recall past episodes that occurred differently or not at
             all. We examined whether older adults' propensity for false
             associative memory is related to declines in postretrieval
             monitoring processes and their modulation with varying
             memory representations. Younger (N = 20) and older adults (N
             = 32) studied and relearned unrelated scene-word pairs,
             followed by a final cued recall that was used to distribute
             the pairs for an associative recognition test 24 hours
             later. This procedure allowed individualized formation of
             rearranged pairs that were made up of elements of pairs that
             were correctly recalled in the final cued recall
             ("high-quality" pairs), and of pairs that were not correctly
             recalled ("low-quality" pairs). Both age groups falsely
             recognized more low-quality than high-quality rearranged
             pairs, with a less pronounced reduction in false alarms to
             high-quality pairs in older adults. In younger adults,
             cingulo-opercular activity was enhanced for false alarms and
             for low-quality correct rejections, consistent with its role
             in postretrieval monitoring. Older adults did not show such
             modulated recruitment, suggesting deficits in their
             selective engagement of monitoring processes given
             variability in the fidelity of memory representations. There
             were no age differences in hippocampal activity, which was
             higher for high-quality than low-quality correct rejections
             in both age groups. These results demonstrate that the
             engagement of cingulo-opercular monitoring mechanisms varies
             with memory representation quality and contributes to
             age-related deficits in false associative memory. (PsycINFO
             Database Record},
   Doi = {10.1037/pag0000212},
   Key = {fds333712}
}

@article{fds330539,
   Author = {Browndyke, JN and Berger, M and Smith, PJ and Harshbarger, TB and Monge,
             ZA and Panchal, V and Bisanar, TL and Glower, DD and Alexander, JH and Cabeza, R and Welsh-Bohmer, K and Newman, MF and Mathew, JP and Duke
             Neurologic Outcomes Research Group (NORG)},
   Title = {Task-related changes in degree centrality and local
             coherence of the posterior cingulate cortex after major
             cardiac surgery in older adults.},
   Journal = {Human Brain Mapping},
   Volume = {39},
   Number = {2},
   Pages = {985-1003},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1002/hbm.23898},
   Abstract = {Older adults often display postoperative cognitive decline
             (POCD) after surgery, yet it is unclear to what extent
             functional connectivity (FC) alterations may underlie these
             deficits. We examined for postoperative voxel-wise FC
             changes in response to increased working memory load demands
             in cardiac surgery patients and nonsurgical controls.Older
             cardiac surgery patients (n = 25) completed a verbal
             N-back working memory task during MRI scanning and cognitive
             testing before and 6 weeks after surgery; nonsurgical
             controls with cardiac disease (n = 26) underwent these
             assessments at identical time intervals. We measured
             postoperative changes in degree centrality, the number of
             edges attached to a brain node, and local coherence, the
             temporal homogeneity of regional functional correlations,
             using voxel-wise graph theory-based FC metrics. Group ×
             time differences were evaluated in these FC metrics
             associated with increased N-back working memory load
             (2-back > 1-back), using a two-stage partitioned
             variance, mixed ANCOVA.Cardiac surgery patients demonstrated
             postoperative working memory load-related degree centrality
             increases in the left dorsal posterior cingulate cortex
             (dPCC; p < .001, cluster p-FWE < .05). The dPCC also
             showed a postoperative increase in working memory
             load-associated local coherence (p < .001, cluster
             p-FWE < .05). dPCC degree centrality and local coherence
             increases were inversely associated with global cognitive
             change in surgery patients (p < .01), but not in
             controls.Cardiac surgery patients showed postoperative
             increases in working memory load-associated degree
             centrality and local coherence of the dPCC that were
             inversely associated with postoperative global cognitive
             outcomes and independent of perioperative cerebrovascular
             damage.},
   Doi = {10.1002/hbm.23898},
   Key = {fds330539}
}

@article{fds333223,
   Author = {Davis, SW and Wing, EA and Cabeza, R},
   Title = {Contributions of the ventral parietal cortex to declarative
             memory.},
   Journal = {Handbook of clinical neurology / edited by P.J. Vinken and
             G.W. Bruyn},
   Volume = {151},
   Pages = {525-553},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/b978-0-444-63622-5.00027-9},
   Abstract = {Our understanding of the role that ventral parietal cortex
             (VPC) plays in declarative memory processes has changed
             dramatically over the last two decades. The goal of this
             chapter is to provide a concise overview data concerning VPC
             involvement in episodic memory (EM), and to connect this
             data to several key theories of VPC function. We review
             evidence from five methodological domains in cognitive
             neuroscience: neuropsychological lesion evidence, univariate
             activation studies, multivoxel pattern analyses, functional
             connectivity studies, and brain stimulation experiments. We
             discuss how the body of empirical work bears on putative
             mnemonic functions of VPC related to attention and stimulus
             representation, and detail the strengths and weaknesses of
             related theories. Lastly, we identify several broad
             conceptual questions raised by recent investigations, and
             outline directions for future research.},
   Doi = {10.1016/b978-0-444-63622-5.00027-9},
   Key = {fds333223}
}

@article{fds329045,
   Author = {Davis, SW and Luber, B and Murphy, DLK and Lisanby, SH and Cabeza,
             R},
   Title = {Frequency-specific neuromodulation of local and distant
             connectivity in aging and episodic memory
             function.},
   Journal = {Human Brain Mapping},
   Volume = {38},
   Number = {12},
   Pages = {5987-6004},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1002/hbm.23803},
   Abstract = {A growing literature has focused on the brain's ability to
             augment processing in local regions by recruiting distant
             communities of neurons in response to neural decline or
             insult. In particular, both younger and older adult
             populations recruit bilateral prefrontal cortex (PFC) as a
             means of compensating for increasing neural effort to
             maintain successful cognitive function. However, it remains
             unclear how local changes in neural activity affect the
             recruitment of this adaptive mechanism. To address this
             problem, we combined graph theoretical measures from
             functional MRI with diffusion weighted imaging and
             repetitive transcranial magnetic stimulation (rTMS) to
             resolve a central hypothesis: how do aged brains flexibly
             adapt to local changes in cortical activity? Specifically,
             we applied neuromodulation to increase or decrease local
             activity in a cortical region supporting successful memory
             encoding (left dorsolateral PFC or DLPFC) using 5 or 1 Hz
             rTMS, respectively. We then assessed a region's local
             within-module degree, or the distributed between-module
             degree (BMD) between distant cortical communities. We
             predicted that (1) local stimulation-related deficits may be
             counteracted by boosting BMD between bilateral PFC, and that
             this effect should be (2) positively correlated with
             structural connectivity. Both predictions were confirmed; 5
             Hz rTMS increased local success-related activity and local
             increases in PFC connectivity, while 1 Hz rTMS decreases
             local activity and triggered a more distributed pattern of
             bilateral PFC connectivity to compensate for this local
             inhibitory effect. These results provide an integrated,
             causal explanation for the network interactions associated
             with successful memory encoding in older adults. Hum Brain
             Mapp 38:5987-6004, 2017. © 2017 Wiley Periodicals,
             Inc.},
   Doi = {10.1002/hbm.23803},
   Key = {fds329045}
}

@misc{fds333678,
   Author = {Doshi, A and Cabeza, R and Berger, M},
   Title = {Geriatric anesthesia: Age-dependent changes in the central
             and peripheral nervous systems},
   Pages = {145-160},
   Booktitle = {Geriatric Anesthesiology: Third Edition},
   Year = {2017},
   Month = {October},
   ISBN = {9783319668789},
   url = {http://dx.doi.org/10.1007/978-3-319-66878-9_10},
   Doi = {10.1007/978-3-319-66878-9_10},
   Key = {fds333678}
}

@article{fds326734,
   Author = {Brashier, NM and Umanath, S and Cabeza, R and Marsh,
             EJ},
   Title = {Competing cues: Older adults rely on knowledge in the face
             of fluency.},
   Journal = {Psychology and Aging},
   Volume = {32},
   Number = {4},
   Pages = {331-337},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1037/pag0000156},
   Abstract = {Consumers regularly encounter repeated false claims in
             political and marketing campaigns, but very little empirical
             work addresses their impact among older adults. Repeated
             statements feel easier to process, and thus more truthful,
             than new ones (i.e., illusory truth). When judging truth,
             older adults' accumulated general knowledge may offset this
             perception of fluency. In two experiments, participants read
             statements that contradicted information stored in memory; a
             post-experimental knowledge check confirmed what individual
             participants knew. Unlike young adults, older adults
             exhibited illusory truth only when they lacked knowledge
             about claims. This interaction between knowledge and fluency
             extends dual-process theories of aging. (PsycINFO Database
             Record},
   Doi = {10.1037/pag0000156},
   Key = {fds326734}
}

@article{fds323839,
   Author = {Geib, BR and Stanley, ML and Dennis, NA and Woldorff, MG and Cabeza,
             R},
   Title = {From hippocampus to whole-brain: The role of integrative
             processing in episodic memory retrieval.},
   Journal = {Human Brain Mapping},
   Volume = {38},
   Number = {4},
   Pages = {2242-2259},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1002/hbm.23518},
   Abstract = {Multivariate functional connectivity analyses of
             neuroimaging data have revealed the importance of complex,
             distributed interactions between disparate yet
             interdependent brain regions. Recent work has shown that
             topological properties of functional brain networks are
             associated with individual and group differences in
             cognitive performance, including in episodic memory. After
             constructing functional whole-brain networks derived from an
             event-related fMRI study of memory retrieval, we examined
             differences in functional brain network architecture between
             forgotten and remembered words. This study yielded three
             main findings. First, graph theory analyses showed that
             successfully remembering compared to forgetting was
             associated with significant changes in the connectivity
             profile of the left hippocampus and a corresponding increase
             in efficient communication with the rest of the brain.
             Second, bivariate functional connectivity analyses indicated
             stronger interactions between the left hippocampus and a
             retrieval assembly for remembered versus forgotten items.
             This assembly included the left precuneus, left caudate,
             bilateral supramarginal gyrus, and the bilateral
             dorsolateral superior frontal gyrus. Integrative properties
             of the retrieval assembly were greater for remembered than
             forgotten items. Third, whole-brain modularity analyses
             revealed that successful memory retrieval was marginally
             significantly associated with a less segregated modular
             architecture in the network. The magnitude of the decreases
             in modularity between remembered and forgotten conditions
             was related to memory performance. These findings indicate
             that increases in integrative properties at the nodal,
             retrieval assembly, and whole-brain topological levels
             facilitate memory retrieval, while also underscoring the
             potential of multivariate brain connectivity approaches for
             providing valuable new insights into the neural bases of
             memory processes. Hum Brain Mapp 38:2242-2259, 2017. © 2017
             Wiley Periodicals, Inc.},
   Doi = {10.1002/hbm.23518},
   Key = {fds323839}
}

@article{fds320544,
   Author = {Davis, SW and Stanley, ML and Moscovitch, M and Cabeza,
             R},
   Title = {Resting-state networks do not determine cognitive function
             networks: a commentary on Campbell and Schacter
             (2016).},
   Journal = {Language Cognition and Neuroscience},
   Volume = {32},
   Number = {6},
   Pages = {669-673},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1080/23273798.2016.1252847},
   Doi = {10.1080/23273798.2016.1252847},
   Key = {fds320544}
}

@article{fds332754,
   Author = {Geib, BR and Stanley, ML and Wing, EA and Laurienti, PJ and Cabeza,
             R},
   Title = {Hippocampal Contributions to the Large-Scale Episodic Memory
             Network Predict Vivid Visual Memories.},
   Journal = {Cerebral Cortex},
   Volume = {27},
   Number = {1},
   Pages = {680-693},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1093/cercor/bhv272},
   Abstract = {A common approach in memory research is to isolate the
             function(s) of individual brain regions, such as the
             hippocampus, without addressing how those regions interact
             with the larger network. To investigate the properties of
             the hippocampus embedded within large-scale networks, we
             used functional magnetic resonance imaging and graph theory
             to characterize complex hippocampal interactions during the
             active retrieval of vivid versus dim visual memories. The
             study yielded 4 main findings. First, the right hippocampus
             displayed greater communication efficiency with the network
             (shorter path length) and became a more convergent structure
             for information integration (higher centrality measures) for
             vivid than dim memories. Second, vivid minus dim differences
             in our graph theory measures of interest were greater in
             magnitude for the right hippocampus than for any other
             region in the 90-region network. Moreover, the right
             hippocampus significantly reorganized its set of direct
             connections from dim to vivid memory retrieval. Finally,
             beyond the hippocampus, communication throughout the
             whole-brain network was more efficient (shorter global path
             length) for vivid than dim memories. In sum, our findings
             illustrate how multivariate network analyses can be used to
             investigate the roles of specific regions within the
             large-scale network, while also accounting for global
             network changes.},
   Doi = {10.1093/cercor/bhv272},
   Key = {fds332754}
}


%% Caspi, Avshalom   
@article{fds326205,
   Author = {Romer, AL and Knodt, AR and Houts, R and Brigidi, BD and Moffitt, TE and Caspi, A and Hariri, AR},
   Title = {Structural alterations within cerebellar circuitry are
             associated with general liability for common mental
             disorders.},
   Journal = {Molecular Psychiatry},
   Volume = {23},
   Number = {4},
   Pages = {1084-1090},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1038/mp.2017.57},
   Abstract = {Accumulating mental-health research encourages a shift in
             focus toward transdiagnostic dimensional features that are
             shared across categorical disorders. In support of this
             shift, recent studies have identified a general liability
             factor for psychopathology-sometimes called the 'p factor'-
             that underlies shared risk for a wide range of mental
             disorders. Identifying neural correlates of this general
             liability would substantiate its importance in
             characterizing the shared origins of mental disorders and
             help us begin to understand the mechanisms through which the
             'p factor' contributes to risk. Here we believe we first
             replicate the 'p factor' using cross-sectional data from a
             volunteer sample of 1246 university students, and then using
             high-resolution multimodal structural neuroimaging, we
             demonstrate that individuals with higher 'p factor' scores
             show reduced structural integrity of white matter pathways,
             as indexed by lower fractional anisotropy values, uniquely
             within the pons. Whole-brain analyses further revealed that
             higher 'p factor' scores are associated with reduced gray
             matter volume in the occipital lobe and left cerebellar
             lobule VIIb, which is functionally connected with prefrontal
             regions supporting cognitive control. Consistent with the
             preponderance of cerebellar afferents within the pons, we
             observed a significant positive correlation between the
             white matter integrity of the pons and cerebellar gray
             matter volume associated with higher 'p factor' scores. The
             results of our analyses provide initial evidence that
             structural alterations in corticocerebellar circuitry
             supporting core functions related to the basic integration,
             coordination and monitoring of information may contribute to
             a general liability for common mental disorders.},
   Doi = {10.1038/mp.2017.57},
   Key = {fds326205}
}

@article{fds333704,
   Author = {Wertz, J and Caspi, A and Belsky, DW and Beckley, AL and Arseneault, L and Barnes, JC and Corcoran, DL and Hogan, S and Houts, RM and Morgan, N and Odgers, CL and Prinz, JA and Sugden, K and Williams, BS and Poulton, R and Moffitt, TE},
   Title = {Genetics and Crime: Integrating New Genomic Discoveries Into
             Psychological Research About Antisocial Behavior.},
   Journal = {Psychological Science},
   Pages = {956797617744542},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/0956797617744542},
   Abstract = {Drawing on psychological and sociological theories of crime
             causation, we tested the hypothesis that genetic risk for
             low educational attainment (assessed via a genome-wide
             polygenic score) is associated with criminal offending. We
             further tested hypotheses of how polygenic risk relates to
             the development of antisocial behavior from childhood
             through adulthood. Across the Dunedin and Environmental Risk
             (E-Risk) birth cohorts of individuals growing up 20 years
             and 20,000 kilometers apart, education polygenic scores
             predicted risk of a criminal record with modest effects.
             Polygenic risk manifested during primary schooling in lower
             cognitive abilities, lower self-control, academic
             difficulties, and truancy, and it was associated with a
             life-course-persistent pattern of antisocial behavior that
             onsets in childhood and persists into adulthood. Crime is
             central in the nature-nurture debate, and findings reported
             here demonstrate how molecular-genetic discoveries can be
             incorporated into established theories of antisocial
             behavior. They also suggest that improving school
             experiences might prevent genetic influences on crime from
             unfolding.},
   Doi = {10.1177/0956797617744542},
   Key = {fds333704}
}

@article{fds328904,
   Author = {Meier, MH and Caspi, A and Danese, A and Fisher, HL and Houts, R and Arseneault, L and Moffitt, TE},
   Title = {Associations between adolescent cannabis use and
             neuropsychological decline: a longitudinal co-twin control
             study.},
   Journal = {Addiction},
   Volume = {113},
   Number = {2},
   Pages = {257-265},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1111/add.13946},
   Abstract = {This study tested whether adolescents who used cannabis or
             met criteria for cannabis dependence showed
             neuropsychological impairment prior to cannabis initiation
             and neuropsychological decline from before to after cannabis
             initiation.A longitudinal co-twin control study.Participants
             were 1989 twins from the Environmental Risk (E-Risk)
             Longitudinal Twin Study, a nationally representative birth
             cohort of twins born in England and Wales from 1994 to
             1995.Frequency of cannabis use and cannabis dependence were
             assessed at age 18. Intelligence quotient (IQ) was obtained
             at ages 5, 12 and 18. Executive functions were assessed at
             age 18.Compared with adolescents who did not use cannabis,
             adolescents who used cannabis had lower IQ in childhood
             prior to cannabis initiation and lower IQ at age 18, but
             there was little evidence that cannabis use was associated
             with IQ decline from ages 12-18. For example, adolescents
             with cannabis dependence had age 12 and age 18 IQ scores
             that were 5.61 (t = -3.11, P = 0.002) and 7.34 IQ points
             (t = -5.27, P < 0.001) lower than adolescents without
             cannabis dependence, but adolescents with cannabis
             dependence did not show greater IQ decline from age 12-18
             (t = -1.27, P = 0.20). Moreover, adolescents who used
             cannabis had poorer executive functions at age 18 than
             adolescents who did not use cannabis, but these associations
             were generally not apparent within twin pairs. For example,
             twins who used cannabis more frequently than their co-twin
             performed similarly to their co-twin on five of six
             executive function tests (Ps > 0.10). The one exception
             was that twins who used cannabis more frequently than their
             co-twin performed worse on one working memory test (Spatial
             Span reversed; β = -0.07, P = 0.036).Short-term
             cannabis use in adolescence does not appear to cause IQ
             decline or impair executive functions, even when cannabis
             use reaches the level of dependence. Family background
             factors explain why adolescent cannabis users perform worse
             on IQ and executive function tests.},
   Doi = {10.1111/add.13946},
   Key = {fds328904}
}

@article{fds331469,
   Author = {Newbury, J and Arseneault, L and Caspi, A and Moffitt, TE and Odgers,
             CL and Fisher, HL},
   Title = {Cumulative Effects of Neighborhood Social Adversity and
             Personal Crime Victimization on Adolescent Psychotic
             Experiences.},
   Journal = {Schizophrenia Bulletin},
   Volume = {44},
   Number = {2},
   Pages = {348-358},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/schbul/sbx060},
   Abstract = {Little is known about the impact of urbanicity, adverse
             neighborhood conditions and violent crime victimization on
             the emergence of adolescent psychotic experiences.Participants
             were from the Environmental Risk (E-Risk) Longitudinal Twin
             Study, a nationally-representative cohort of 2232 British
             twins who were interviewed about adolescent psychotic
             experiences at age 18. Urbanicity, neighborhood
             characteristics, and personal victimization by violent crime
             were measured during childhood and adolescence via geocoded
             census data, surveys of over 5000 immediate neighbors of the
             E-Risk participants, and interviews with participants
             themselves.Adolescents raised in urban vs rural
             neighborhoods were significantly more likely to have
             psychotic experiences (OR = 1.67, 95% CI = 1.21-2.30, P =
             .002). This association remained significant after
             considering potential confounders including family
             socioeconomic status, family psychiatric history, and
             adolescent substance problems (OR = 1.43, 95% CI =
             1.01-2.03, P = .042), but became nonsignificant after
             considering adverse social conditions in urban neighborhoods
             such as low social cohesion and high neighborhood disorder
             (OR = 1.35, 95% CI = 0.94-1.92, P = .102). The combined
             association of adverse neighborhood social conditions and
             personal crime victimization with adolescent psychotic
             experiences (adjusted OR = 4.86, 95% CI = 3.28-7.20, P <
             .001) was substantially greater than for either exposure
             alone, highlighting a potential interaction between
             neighborhood conditions and crime victimization (interaction
             contrast ratio = 1.81, 95% CI = -0.03 to 3.65) that was
             significant at the P = .054 level.Cumulative effects of
             adverse neighborhood social conditions and personal
             victimization by violent crime during upbringing partly
             explain why adolescents in urban settings are more likely to
             report psychotic experiences. Early intervention efforts for
             psychosis could be targeted towards victimized youth living
             in urban and socially adverse neighborhoods.},
   Doi = {10.1093/schbul/sbx060},
   Key = {fds331469}
}

@article{fds332385,
   Author = {Beckley, AL and Caspi, A and Broadbent, J and Harrington, H and Houts,
             RM and Poulton, R and Ramrakha, S and Reuben, A and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Criminal
             Offending.},
   Journal = {JAMA Pediatrics},
   Volume = {172},
   Number = {2},
   Pages = {166-173},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1001/jamapediatrics.2017.4005},
   Abstract = {Lead is a neurotoxin with well-documented effects on health.
             Research suggests that lead may be associated with criminal
             behavior. This association is difficult to disentangle from
             low socioeconomic status, a factor in both lead exposure and
             criminal offending.To test the hypothesis that a higher
             childhood blood lead level (BLL) is associated with greater
             risk of criminal conviction, recidivism (repeat conviction),
             conviction for violent offenses, and variety of
             self-reported criminal offending in a setting where BLL was
             not associated with low socioeconomic status.A total of 553
             individuals participated in a prospective study based on a
             population-representative cohort born between April 1, 1972,
             and March 31, 1973, from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (December 2012). Statistical
             analysis was performed from November 10, 2016, to September
             5, 2017.Blood lead level measured at age 11 years.Official
             criminal conviction cumulative to age 38 years (data
             collected in 2013), single conviction or recidivism,
             conviction for nonviolent or violent crime, and
             self-reported variety of crime types at ages 15, 18, 21, 26,
             32, and 38 years.Participants included 553 individuals (255
             female and 298 male participants) who had their blood tested
             for lead at age 11 years. The mean (SD) BLL at age 11 years
             was 11.01 (4.62) μg/dL. A total of 154 participants (27.8%)
             had a criminal conviction, 86 (15.6%) had recidivated, and
             53 (9.6%) had a violent offense conviction. Variety scores
             for self-reported offending ranged from 0 to 10 offense
             types at each assessment; higher numbers indicated greater
             crime involvement. Self-reported offending followed the
             well-established age-crime curve (ie, the mean [SD] variety
             of self-reported offending increased from 1.99 [2.82] at age
             15 years to its peak of 4.24 [3.15] at age 18 years and 4.22
             [3.02] at age 21 years and declined thereafter to 1.10
             [1.59] at age 38 years). Blood lead level was a poor
             discriminator between no conviction and conviction (area
             under the curve, 0.58). Overall, associations between BLL
             and conviction outcomes were weak. The estimated effect of
             BLL was lower for recidivism than for single convictions and
             lower for violent offending than for nonviolent offending.
             Sex-adjusted associations between BLL reached statistical
             significance for only 1 of the 6 self-reported offending
             outcomes at age 15 years (r = 0.10; 95% CI, 0.01-0.18;
             P = .02).This study overcomes past limitations of
             studies of BLL and crime by studying the association in a
             place and time where the correlation was not confounded by
             childhood socioeconomic status. Findings failed to support a
             dose-response association between BLL and consequential
             criminal offending.},
   Doi = {10.1001/jamapediatrics.2017.4005},
   Key = {fds332385}
}

@article{fds329541,
   Author = {Baldwin, JR and Arseneault, L and Caspi, A and Fisher, HL and Moffitt,
             TE and Odgers, CL and Pariante, C and Ambler, A and Dove, R and Kepa, A and Matthews, T and Menard, A and Sugden, K and Williams, B and Danese,
             A},
   Title = {Childhood victimization and inflammation in young adulthood:
             A genetically sensitive cohort study.},
   Journal = {Brain, Behavior, and Immunity},
   Volume = {67},
   Pages = {211-217},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.bbi.2017.08.025},
   Abstract = {Childhood victimization is an important risk factor for
             later immune-related disorders. Previous evidence has
             demonstrated that childhood victimization is associated with
             elevated levels of inflammation biomarkers measured decades
             after exposure. However, it is unclear whether this
             association is (1) already detectable in young people, (2)
             different in males and females, and (3) confounded by
             genetic liability to inflammation. Here we sought to address
             these questions.Participants were 2232 children followed
             from birth to age 18years as part of the Environmental Risk
             (E-Risk) Longitudinal Twin Study. Childhood victimization
             was measured prospectively from birth to age 12years.
             Inflammation was measured through C-reactive protein (CRP)
             levels in dried blood spots at age 18years. Latent genetic
             liability for high inflammation levels was assessed through
             a twin-based method.Greater exposure to childhood
             victimization was associated with higher CRP levels at age
             18 (serum-equivalent means were 0.65 in non-victimized Study
             members, 0.74 in those exposed to one victimization type,
             and 0.81 in those exposed to poly-victimization; p=0.018).
             However, this association was driven by a significant
             association in females (serum-equivalent means were 0.75 in
             non-victimized females, 0.87 in those exposed to one type of
             victimization, and 1.19 in those exposed to
             poly-victimization; p=0.010), while no significant
             association was observed in males (p=0.19). Victimized
             females showed elevated CRP levels independent of latent
             genetic influence, as well as childhood socioeconomic
             status, and waist-hip ratio and body temperature at the time
             of CRP assessment.Childhood victimization is associated with
             elevated CRP levels in young women, independent of latent
             genetic influences and other key risk factors. These results
             strengthen causal inference about the effects of childhood
             victimization on inflammation levels in females by
             accounting for potential genetic confounding.},
   Doi = {10.1016/j.bbi.2017.08.025},
   Key = {fds329541}
}

@article{fds329982,
   Author = {Newbury, JB and Arseneault, L and Moffitt, TE and Caspi, A and Danese,
             A and Baldwin, JR and Fisher, HL},
   Title = {Measuring childhood maltreatment to predict early-adult
             psychopathology: Comparison of prospective informant-reports
             and retrospective self-reports.},
   Journal = {Journal of Psychiatric Research},
   Volume = {96},
   Pages = {57-64},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jpsychires.2017.09.020},
   Abstract = {Both prospective informant-reports and retrospective
             self-reports may be used to measure childhood maltreatment,
             though both methods entail potential limitations such as
             underestimation and memory biases. The validity and utility
             of standard measures of childhood maltreatment requires
             clarification in order to inform the design of future
             studies investigating the mental health consequences of
             maltreatment. The present study assessed agreement between
             prospective informant-reports and retrospective self-reports
             of childhood maltreatment, as well as the comparative
             utility of both reports for predicting a range of
             psychiatric problems at age 18. Data were obtained from the
             Environmental Risk (E-Risk) Longitudinal Twin Study, a
             nationally-representative birth cohort of 2232 children
             followed to 18 years of age (with 93% retention). Childhood
             maltreatment was assessed in two ways: (i) prospective
             informant-reports from caregivers, researchers, and
             clinicians when children were aged 5, 7, 10 and 12; and (ii)
             retrospective self-reports of maltreatment experiences
             occurring up to age 12, obtained at age 18 using the
             Childhood Trauma Questionnaire. Participants were privately
             interviewed at age 18 concerning several psychiatric
             problems including depression, anxiety, self-injury,
             alcohol/cannabis dependence, and conduct disorder. There was
             only slight to fair agreement between prospective and
             retrospective reports of childhood maltreatment (all
             Kappa's ≤ 0.31). Both prospective and retrospective
             reports of maltreatment were associated with age-18
             psychiatric problems, though the strongest associations were
             found when maltreatment was retrospectively self-reported.
             These findings indicate that prospective and retrospective
             reports of childhood maltreatment capture largely
             non-overlapping groups of individuals. Young adults who
             recall being maltreated have a particularly elevated risk
             for psychopathology.},
   Doi = {10.1016/j.jpsychires.2017.09.020},
   Key = {fds329982}
}

@article{fds332035,
   Author = {Wertz, J and Agnew-Blais, J and Caspi, A and Danese, A and Fisher, HL and Goldman-Mellor, S and Moffitt, TE and Arseneault,
             L},
   Title = {From Childhood Conduct Problems to Poor Functioning at Age
             18 Years: Examining Explanations in a Longitudinal Cohort
             Study.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {57},
   Number = {1},
   Pages = {54-60.e4},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.09.437},
   Abstract = {Childhood conduct problems are associated with poor
             functioning in early adulthood. We tested a series of
             hypotheses to understand the mechanisms underlying this
             association.We used data from the Environmental Risk
             (E-Risk) Longitudinal Twin Study, a birth cohort of 2,232
             twins born in England and Wales in 1994 and 1995, followed
             up to age 18 years with 93% retention. Severe conduct
             problems in childhood were assessed at ages 5, 7, and 10
             years using parent and teacher reports. Poor functioning at
             age 18 years, including cautions and convictions, daily
             cigarette smoking, heavy drinking, and psychosocial
             difficulties, was measured through interviews with
             participants and official crime record searches.Participants
             18 years old with versus without a childhood history of
             severe conduct problems had greater rates of each poor
             functional outcome, and they were more likely to experience
             multiple poor outcomes. This association was partly
             accounted for by concurrent psychopathology in early
             adulthood, as well as by early familial risk factors, both
             genetic and environmental. Childhood conduct problems,
             however, continued to predict poor outcomes at age 18 years
             after accounting for these explanations.Children with severe
             conduct problems display poor functioning at age 18 years
             because of concurrent problems in early adulthood and
             familial risk factors originating in childhood. However,
             conduct problems also exert a lasting effect on young
             people's lives independent of these factors, pointing to
             early conduct problems as a target for early interventions
             aimed at preventing poor functional outcomes.},
   Doi = {10.1016/j.jaac.2017.09.437},
   Key = {fds332035}
}

@article{fds331398,
   Author = {Rivenbark, JG and Odgers, CL and Caspi, A and Harrington, H and Hogan,
             S and Houts, RM and Poulton, R and Moffitt, TE},
   Title = {The high societal costs of childhood conduct problems:
             evidence from administrative records up to age 38 in a
             longitudinal birth cohort.},
   Journal = {The Journal of Child Psychology and Psychiatry and Allied
             Disciplines},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1111/jcpp.12850},
   Abstract = {Children with conduct problems that persist into adulthood
             are at increased risk for future behavioral, health, and
             social problems. However, the longer term public service
             usage among these children has not been fully documented. To
             aid public health and intervention planning, adult service
             usage across criminal justice, health care, and social
             welfare domains is compared among all individuals from a
             representative cohort who followed different conduct problem
             trajectories from childhood into adulthood.Participants are
             from the Dunedin Multidisciplinary Health and Development
             Study, a prospective, representative cohort of consecutive
             births (N = 1,037) from April 1972 to March 1973 in
             Dunedin, New Zealand. Regression analyses were used to
             compare levels of public service usage up to age 38,
             gathered via administrative and electronic medical records,
             between participants who displayed distinct subtypes of
             childhood conduct problems (low, childhood-limited,
             adolescent-onset, and life-course persistent).Children
             exhibiting life-course persistent conduct problems used
             significantly more services as adults than those with low
             levels of childhood conduct problems. Although this group
             comprised only 9.0% of the population, they accounted for
             53.3% of all convictions, 15.7% of emergency department
             visits, 20.5% of prescription fills, 13.1% of injury claims,
             and 24.7% of welfare benefit months. Half of this group
             (50.0%) also accrued high service use across all three
             domains of criminal justice, health, and social welfare
             services, as compared to only 11.3% of those with low
             conduct problems (OR = 7.27, 95% CI = 4.42-12.0).Conduct
             problems in childhood signal high future costs in terms of
             service utilization across multiple sectors. Future
             evaluations of interventions aimed at conduct problems
             should also track potential reductions in health burden and
             service usage that stretch into midlife.},
   Doi = {10.1111/jcpp.12850},
   Key = {fds331398}
}

@article{fds331468,
   Author = {Newbury, JB and Arseneault, L and Caspi, A and Moffitt, TE and Odgers,
             CL and Baldwin, JR and Zavos, HMS and Fisher, HL},
   Title = {In the eye of the beholder: Perceptions of neighborhood
             adversity and psychotic experiences in adolescence.},
   Journal = {Development and Psychopathology},
   Volume = {29},
   Number = {5},
   Pages = {1823-1837},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1017/s0954579417001420},
   Abstract = {Adolescent psychotic experiences increase risk for
             schizophrenia and other severe psychopathology in adulthood.
             Converging evidence implicates urban and adverse
             neighborhood conditions in the etiology of adolescent
             psychotic experiences, but the role of young people's
             personal perceptions of disorder (i.e., physical and social
             signs of threat) in their neighborhood is unknown. This was
             examined using data from the Environmental Risk Longitudinal
             Twin Study, a nationally representative birth cohort of
             2,232 British twins. Participants were interviewed at age 18
             about psychotic phenomena and perceptions of disorder in the
             neighborhood. Multilevel, longitudinal, and genetically
             sensitive analyses investigated the association between
             perceptions of neighborhood disorder and adolescent
             psychotic experiences. Adolescents who perceived higher
             levels of neighborhood disorder were significantly more
             likely to have psychotic experiences, even after accounting
             for objectively/independently measured levels of crime and
             disorder, neighborhood- and family-level socioeconomic
             status, family psychiatric history, adolescent substance and
             mood problems, and childhood psychotic symptoms: odds ratio
             = 1.62, 95% confidence interval [1.27, 2.05], p < .001. The
             phenotypic overlap between adolescent psychotic experiences
             and perceptions of neighborhood disorder was explained by
             overlapping common environmental influences, rC = .88, 95%
             confidence interval [0.26, 1.00]. Findings suggest that
             early psychological interventions to prevent adolescent
             psychotic experiences should explore the role of young
             people's (potentially modifiable) perceptions of threatening
             neighborhood conditions.},
   Doi = {10.1017/s0954579417001420},
   Key = {fds331468}
}

@article{fds330402,
   Author = {Schaefer, JD and Scult, MA and Caspi, A and Arseneault, L and Belsky,
             DW and Hariri, AR and Harrington, H and Houts, R and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Is low cognitive functioning a predictor or consequence of
             major depressive disorder? A test in two longitudinal birth
             cohorts.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s095457941700164x},
   Abstract = {Cognitive impairment has been identified as an important
             aspect of major depressive disorder (MDD). We tested two
             theories regarding the association between MDD and cognitive
             functioning using data from longitudinal cohort studies. One
             theory, the cognitive reserve hypothesis, suggests that
             higher cognitive ability in childhood decreases risk of
             later MDD. The second, the scarring hypothesis, instead
             suggests that MDD leads to persistent cognitive deficits
             following disorder onset. We tested both theories in the
             Dunedin Study, a population-representative cohort followed
             from birth to midlife and assessed repeatedly for both
             cognitive functioning and psychopathology. We also used data
             from the Environmental Risk Longitudinal Twin Study to test
             whether childhood cognitive functioning predicts future MDD
             risk independent of family-wide and genetic risk using a
             discordant twin design. Contrary to both hypotheses, we
             found that childhood cognitive functioning did not predict
             future risk of MDD, nor did study members with a past
             history of MDD show evidence of greater cognitive decline
             unless MDD was accompanied by other comorbid psychiatric
             conditions. Our results thus suggest that low cognitive
             functioning is related to comorbidity, but is neither an
             antecedent nor an enduring consequence of MDD. Future
             research may benefit from considering cognitive deficits
             that occur during depressive episodes from a transdiagnostic
             perspective.},
   Doi = {10.1017/s095457941700164x},
   Key = {fds330402}
}

@article{fds328903,
   Author = {Williams, MJA and Milne, BJ and Ambler, A and Theodore, R and Ramrakha,
             S and Caspi, A and Moffitt, TE and Poulton, R},
   Title = {Childhood body mass index and endothelial dysfunction
             evaluated by peripheral arterial tonometry in early
             midlife.},
   Journal = {International Journal of Obesity},
   Volume = {41},
   Number = {9},
   Pages = {1355-1360},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1038/ijo.2017.108},
   Abstract = {Endothelial dysfunction predicts mortality but it is unknown
             whether childhood obesity predicts adult endothelial
             dysfunction. The aim of this study was to determine whether
             anthropometric indices of body fat in childhood, adolescence
             and early midlife are associated with endothelial
             dysfunction in early midlife.Participants belonged to a
             representative birth cohort of 1037 individuals born in
             Dunedin, New Zealand in 1972 and 1973 and followed to age 38
             years, with 95% retention (the Dunedin Multidisciplinary
             Health and Development Study). We assessed anthropometric
             indices of obesity at ages 3, 5, 7, 9, 11, 13, 15, 18, 21,
             26, 32 and 38 years. We tested associations between
             endothelial function assessed by peripheral arterial
             tonometry (PAT) at age 38 and; age 38 cardiovascular risk
             factors; age 3 body mass index (BMI); and four BMI
             trajectory groups from childhood to early midlife.Early
             midlife endothelial dysfunction was associated with BMI,
             large waist circumference, low high-density lipoprotein
             cholesterol, low cardiorespiratory fitness and increased
             high-sensitivity C-reactive protein. After adjustment for
             sex and childhood socioeconomic status, 3-year-olds with BMI
             1 s.d. above the mean had Framingham-reactive hyperemia
             index (F-RHI) ratios that were 0.10 below those with normal
             BMI (β=-0.10, 95% confidence interval (CI) -0.17 to -0.03,
             P=0.007) at age 38. Cohort members in the 'overweight',
             'obese' and 'morbidly obese' trajectories had F-RHI ratios
             that were 0.08 (β=-0.08, 95% CI -0.14 to -0.03, P=0.003),
             0.13 (β=-0.13, 95% CI -0.21 to -0.06, P<0.001) and 0.17
             (β=-0.17, 95% CI -0.33 to -0.01, P=0.033), respectively,
             below age-peers in the 'normal' trajectory.Childhood BMI and
             the trajectories of BMI from childhood to early midlife
             predict endothelial dysfunction evaluated by PAT in early
             midlife.},
   Doi = {10.1038/ijo.2017.108},
   Key = {fds328903}
}

@article{fds328625,
   Author = {Matthews, T and Danese, A and Gregory, AM and Caspi, A and Moffitt, TE and Arseneault, L},
   Title = {Sleeping with one eye open: loneliness and sleep quality in
             young adults},
   Journal = {Psychological Medicine},
   Volume = {47},
   Number = {12},
   Pages = {2177-2186},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1017/S0033291717000629},
   Doi = {10.1017/S0033291717000629},
   Key = {fds328625}
}

@article{fds327017,
   Author = {Suppli, NP and Bukh, JD and Moffitt, TE and Caspi, A and Johansen, C and Tjønneland, A and Kessing, LV and Dalton, SO},
   Title = {Genetic variants in 5-HTTLPR, BDNF, HTR1A, COMT, and FKBP5
             and risk for treated depression after cancer
             diagnosis.},
   Journal = {Depression and Anxiety},
   Volume = {34},
   Number = {9},
   Pages = {845-855},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1002/da.22660},
   Abstract = {The role of gene-environment interactions in the
             pathogenesis of depression is unclear. Previous studies
             addressed vulnerability for depression after childhood
             adversity and stressful life events among carriers of
             numerous specific genetic variants; however, the importance
             of individual genetic variants, the environmental exposures
             with which they interact, and the magnitude of the risk
             conveyed by these interactions remain elusive.We included
             7,320 people with a first primary cancer identified in the
             prospective Diet, Cancer and Health study in an exposed-only
             cohort study. The mean age of the individuals was 68 years
             (5th, 95th percentiles: 58, 78) at cancer diagnosis. Using
             Cox regression models and cumulative incidence plots, we
             analyzed the associations between genetic variants in
             5-HTTLPR, BDNF, HTR1A, COMT, and FKBP5 and use of
             antidepressants as well as hospital contact for depression
             after diagnosis of cancer.Overall, we observed no
             statistically significant associations, with nonsignificant
             hazard ratio estimates for use of antidepressants of
             0.95-1.07.This study of elderly people indicates that it is
             unlikely that the investigated genetic variants are
             clinically relevantly associated with depression after
             diagnosis of cancer. The mechanisms for gene-environment
             interactions in younger individuals are probably different,
             and we advise caution in extrapolating our results to early
             life stress. However, conclusion from the present study
             might be generalizable to elderly persons exposed to other
             stressful life events.},
   Doi = {10.1002/da.22660},
   Key = {fds327017}
}

@article{fds326204,
   Author = {Belsky, DW and Caspi, A and Cohen, HJ and Kraus, WE and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Impact of early personal-history characteristics on the Pace
             of Aging: implications for clinical trials of therapies to
             slow aging and extend healthspan.},
   Journal = {Aging Cell},
   Volume = {16},
   Number = {4},
   Pages = {644-651},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/acel.12591},
   Abstract = {Therapies to extend healthspan are poised to move from
             laboratory animal models to human clinical trials.
             Translation from mouse to human will entail challenges,
             among them the multifactorial heterogeneity of human aging.
             To inform clinical trials about this heterogeneity, we
             report how humans' pace of biological aging relates to
             personal-history characteristics. Because geroprotective
             therapies must be delivered by midlife to prevent
             age-related disease onset, we studied young-adult members of
             the Dunedin Study 1972-73 birth cohort (n = 954). Cohort
             members' Pace of Aging was measured as coordinated decline
             in the integrity of multiple organ systems, by quantifying
             rate of decline across repeated measurements of 18
             biomarkers assayed when cohort members were ages 26, 32, and
             38 years. The childhood personal-history characteristics
             studied were known predictors of age-related disease and
             mortality, and were measured prospectively during childhood.
             Personal-history characteristics of familial longevity,
             childhood social class, adverse childhood experiences, and
             childhood health, intelligence, and self-control all
             predicted differences in cohort members' adulthood Pace of
             Aging. Accumulation of more personal-history risks predicted
             faster Pace of Aging. Because trials of anti-aging therapies
             will need to ascertain personal histories retrospectively,
             we replicated results using cohort members' retrospective
             personal-history reports made in adulthood. Because many
             trials recruit participants from clinical settings, we
             replicated results in the cohort subset who had recent
             health system contact according to electronic medical
             records. Quick, inexpensive measures of trial participants'
             early personal histories can enable clinical trials to study
             who volunteers for trials, who adheres to treatment, and who
             responds to anti-aging therapies.},
   Doi = {10.1111/acel.12591},
   Key = {fds326204}
}

@article{fds325848,
   Author = {Kotov, R and Krueger, RF and Watson, D and Achenbach, TM and Althoff,
             RR and Bagby, RM and Brown, TA and Carpenter, WT and Caspi, A and Clark,
             LA and Eaton, NR and Forbes, MK and Forbush, KT and Goldberg, D and Hasin,
             D and Hyman, SE and Ivanova, MY and Lynam, DR and Markon, K and Miller, JD and Moffitt, TE and Morey, LC and Mullins-Sweatt, SN and Ormel, J and Patrick, CJ and Regier, DA and Rescorla, L and Ruggero, CJ and Samuel,
             DB and Sellbom, M and Simms, LJ and Skodol, AE and Slade, T and South, SC and Tackett, JL and Waldman, ID and Waszczuk, MA and Widiger, TA and Wright,
             AGC and Zimmerman, M},
   Title = {The Hierarchical Taxonomy of Psychopathology (HiTOP): A
             dimensional alternative to traditional nosologies.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {4},
   Pages = {454-477},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1037/abn0000258},
   Abstract = {The reliability and validity of traditional taxonomies are
             limited by arbitrary boundaries between psychopathology and
             normality, often unclear boundaries between disorders,
             frequent disorder co-occurrence, heterogeneity within
             disorders, and diagnostic instability. These taxonomies went
             beyond evidence available on the structure of
             psychopathology and were shaped by a variety of other
             considerations, which may explain the aforementioned
             shortcomings. The Hierarchical Taxonomy Of Psychopathology
             (HiTOP) model has emerged as a research effort to address
             these problems. It constructs psychopathological syndromes
             and their components/subtypes based on the observed
             covariation of symptoms, grouping related symptoms together
             and thus reducing heterogeneity. It also combines
             co-occurring syndromes into spectra, thereby mapping out
             comorbidity. Moreover, it characterizes these phenomena
             dimensionally, which addresses boundary problems and
             diagnostic instability. Here, we review the development of
             the HiTOP and the relevant evidence. The new classification
             already covers most forms of psychopathology. Dimensional
             measures have been developed to assess many of the
             identified components, syndromes, and spectra. Several
             domains of this model are ready for clinical and research
             applications. The HiTOP promises to improve research and
             clinical practice by addressing the aforementioned
             shortcomings of traditional nosologies. It also provides an
             effective way to summarize and convey information on risk
             factors, etiology, pathophysiology, phenomenology, illness
             course, and treatment response. This can greatly improve the
             utility of the diagnosis of mental disorders. The new
             classification remains a work in progress. However, it is
             developing rapidly and is poised to advance mental health
             research and care significantly as the relevant science
             matures. (PsycINFO Database Record},
   Doi = {10.1037/abn0000258},
   Key = {fds325848}
}

@article{fds324429,
   Author = {Danese, A and Moffitt, TE and Arseneault, L and Bleiberg, BA and Dinardo, PB and Gandelman, SB and Houts, R and Ambler, A and Fisher, HL and Poulton, R and Caspi, A},
   Title = {The Origins of Cognitive Deficits in Victimized Children:
             Implications for Neuroscientists and Clinicians.},
   Journal = {American Journal of Psychiatry},
   Volume = {174},
   Number = {4},
   Pages = {349-361},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1176/appi.ajp.2016.16030333},
   Abstract = {Individuals reporting a history of childhood violence
             victimization have impaired brain function. However, the
             clinical significance, reproducibility, and causality of
             these findings are disputed. The authors used data from two
             large cohort studies to address these research questions
             directly.The authors tested the association between
             prospectively collected measures of childhood violence
             victimization and cognitive functions in childhood,
             adolescence, and adulthood among 2,232 members of the U.K.
             E-Risk Study and 1,037 members of the New Zealand Dunedin
             Study who were followed up from birth until ages 18 and 38
             years, respectively. Multiple measures of victimization and
             cognition were used, and comparisons were made of cognitive
             scores for twins discordant for victimization.Individuals
             exposed to childhood victimization had pervasive impairments
             in clinically relevant cognitive functions, including
             general intelligence, executive function, processing speed,
             memory, perceptual reasoning, and verbal comprehension in
             adolescence and adulthood. However, the observed cognitive
             deficits in victimized individuals were largely explained by
             cognitive deficits that predated childhood victimization and
             by confounding genetic and environmental risks.Findings from
             two population-representative birth cohorts totaling more
             than 3,000 individuals and born 20 years and 20,000 km apart
             suggest that the association between childhood violence
             victimization and later cognition is largely noncausal, in
             contrast to conventional interpretations. These findings
             support the adoption of a more circumspect approach to
             causal inference in the neuroscience of stress. Clinically,
             cognitive deficits should be conceptualized as individual
             risk factors for victimization as well as potential
             complicating features during treatment.},
   Doi = {10.1176/appi.ajp.2016.16030333},
   Key = {fds324429}
}

@article{fds325847,
   Author = {Reuben, A and Caspi, A and Belsky, DW and Broadbent, J and Harrington,
             H and Sugden, K and Houts, RM and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Cognitive
             Function and Socioeconomic Status at Age 38 Years and With
             IQ Change and Socioeconomic Mobility Between Childhood and
             Adulthood.},
   Journal = {JAMA : the journal of the American Medical
             Association},
   Volume = {317},
   Number = {12},
   Pages = {1244-1251},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1001/jama.2017.1712},
   Abstract = {Many children in the United States and around the world are
             exposed to lead, a developmental neurotoxin. The long-term
             cognitive and socioeconomic consequences of lead exposure
             are uncertain.To test the hypothesis that childhood lead
             exposure is associated with cognitive function and
             socioeconomic status in adulthood and with changes in IQ and
             socioeconomic mobility between childhood and midlife.A
             prospective cohort study based on a population-representative
             1972-1973 birth cohort from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (until December 2012).Childhood
             lead exposure ascertained as blood lead levels measured at
             age 11 years. High blood lead levels were observed among
             children from all socioeconomic status levels in this
             cohort.The IQ (primary outcome) and indexes of Verbal
             Comprehension, Perceptual Reasoning, Working Memory, and
             Processing Speed (secondary outcomes) were assessed at age
             38 years using the Wechsler Adult Intelligence Scale-IV
             (WAIS-IV; IQ range, 40-160). Socioeconomic status (primary
             outcome) was assessed at age 38 years using the New Zealand
             Socioeconomic Index-2006 (NZSEI-06; range, 10 [lowest]-90
             [highest]).Of 1037 original participants, 1007 were alive at
             age 38 years, of whom 565 (56%) had been lead tested at age
             11 years (54% male; 93% white). Mean (SD) blood lead level
             at age 11 years was 10.99 (4.63) µg/dL. Among blood-tested
             participants included at age 38 years, mean WAIS-IV score
             was 101.16 (14.82) and mean NZSEI-06 score was 49.75
             (17.12). After adjusting for maternal IQ, childhood IQ, and
             childhood socioeconomic status, each 5-µg/dL higher level
             of blood lead in childhood was associated with a 1.61-point
             lower score (95% CI, -2.48 to -0.74) in adult IQ, a
             2.07-point lower score (95% CI, -3.14 to -1.01) in
             perceptual reasoning, and a 1.26-point lower score (95% CI,
             -2.38 to -0.14) in working memory. Associations of childhood
             blood lead level with deficits in verbal comprehension and
             processing speed were not statistically significant. After
             adjusting for confounders, each 5-µg/dL higher level of
             blood lead in childhood was associated with a 1.79-unit
             lower score (95% CI, -3.17 to -0.40) in socioeconomic
             status. An association between greater blood lead levels and
             a decline in IQ and socioeconomic status from childhood to
             adulthood was observed with 40% of the association with
             downward mobility mediated by cognitive decline from
             childhood.In this cohort born in New Zealand in 1972-1973,
             childhood lead exposure was associated with lower cognitive
             function and socioeconomic status at age 38 years and with
             declines in IQ and with downward social mobility. Childhood
             lead exposure may have long-term ramifications.},
   Doi = {10.1001/jama.2017.1712},
   Key = {fds325847}
}

@article{fds321660,
   Author = {Schaefer, JD and Caspi, A and Belsky, DW and Harrington, H and Houts, R and Horwood, LJ and Hussong, A and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Enduring mental health: Prevalence and prediction.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {2},
   Pages = {212-224},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1037/abn0000232},
   Abstract = {We review epidemiological evidence indicating that most
             people will develop a diagnosable mental disorder,
             suggesting that only a minority experience enduring mental
             health. This minority has received little empirical study,
             leaving the prevalence and predictors of enduring mental
             health unknown. We turn to the population-representative
             Dunedin cohort, followed from birth to midlife, to compare
             people never-diagnosed with mental disorder (N = 171; 17%
             prevalence) to those diagnosed at 1-2 study waves, the
             cohort mode (N = 409). Surprisingly, compared to this modal
             group, never-diagnosed Study members were not born into
             unusually well-to-do families, nor did their enduring mental
             health follow markedly sound physical health, or unusually
             high intelligence. Instead, they tended to have an
             advantageous temperament/personality style, and negligible
             family history of mental disorder. As adults, they report
             superior educational and occupational attainment, greater
             life satisfaction, and higher-quality relationships. Our
             findings draw attention to "enduring mental health" as a
             revealing psychological phenotype and suggest it deserves
             further study. (PsycINFO Database Record},
   Doi = {10.1037/abn0000232},
   Key = {fds321660}
}

@article{fds328127,
   Author = {Moffitt, TE and Belsky, DW and Danese, A and Poulton, R and Caspi,
             A},
   Title = {The Longitudinal Study of Aging in Human Young Adults:
             Knowledge Gaps and Research Agenda.},
   Journal = {Journals of Gerontology: Series A},
   Volume = {72},
   Number = {2},
   Pages = {210-215},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1093/gerona/glw191},
   Abstract = {To prevent onset of age-related diseases and physical and
             cognitive decline, interventions to slow human aging and
             extend health span must eventually be applied to people
             while they are still young and healthy. Yet most human aging
             research examines older adults, many with chronic disease,
             and little is known about aging in healthy young humans.This
             article explains how this knowledge gap is a barrier to
             extending health span and puts forward the case that
             geroscience should invest in researching the pace of aging
             in young adults. As one illustrative example, we describe an
             initial effort to study the pace of aging in a young-adult
             birth cohort by using repeated waves of biomarkers collected
             across the third and fourth decades to quantify the pace of
             coordinated physiological deterioration across multiple
             organ systems (eg, pulmonary, periodontal, cardiovascular,
             renal, hepatic, metabolic, and immune function).Findings
             provided proof of principle that it is possible to quantify
             individual variation in the pace of aging in young adults
             still free of age-related diseases.This article articulates
             research needs to improve longitudinal measurement of the
             pace of aging in young people, to pinpoint factors that slow
             or speed the pace of aging, to compare pace of aging against
             genomic clocks, to explain slow-aging young adults, and to
             apply pace of aging in preventive clinical trials of
             antiaging therapies. This article puts forward a research
             agenda to fill the knowledge gap concerning lifelong causes
             of aging.},
   Doi = {10.1093/gerona/glw191},
   Key = {fds328127}
}


%% Castrellon, Jaime J.   
@article{fds333912,
   Author = {Dang, LC and Samanez-Larkin, GR and Castrellon, JJ and Perkins, SF and Cowan, RL and Zald, DH},
   Title = {Individual differences in dopamine D2 receptor availability
             correlate with reward valuation.},
   Journal = {Cognitive, Affective, & Behaviorial Neuroscience},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.3758/s13415-018-0601-9},
   Abstract = {Reward valuation, which underlies all value-based
             decision-making, has been associated with dopamine function
             in many studies of nonhuman animals, but there is relatively
             less direct evidence for an association in humans. Here, we
             measured dopamine D2 receptor (DRD2) availability in vivo in
             humans to examine relations between individual differences
             in dopamine receptor availability and neural activity
             associated with a measure of reward valuation, expected
             value (i.e., the product of reward magnitude and the
             probability of obtaining the reward). Fourteen healthy adult
             subjects underwent PET with [18F]fallypride, a radiotracer
             with strong affinity for DRD2, and fMRI (on a separate day)
             while performing a reward valuation task. [18F]fallypride
             binding potential, reflecting DRD2 availability, in the
             midbrain correlated positively with neural activity
             associated with expected value, specifically in the left
             ventral striatum/caudate. The present results provide in
             vivo evidence from humans showing midbrain dopamine
             characteristics are associated with reward
             valuation.},
   Doi = {10.3758/s13415-018-0601-9},
   Key = {fds333912}
}

@article{fds333913,
   Author = {Seaman, KL and Brooks, N and Karrer, TM and Castrellon, JJ and Perkins,
             SF and Dang, L and Hsu, M and Zald, DH and Samanez-Larkin,
             GR},
   Title = {Subjective Value Representations during Effort, Probability,
             and Time Discounting across Adulthood.},
   Journal = {Social Cognitive and Affective Neuroscience},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1093/scan/nsy021},
   Abstract = {Every day, humans make countless decisions which require the
             integration of information about potential benefits (i.e.,
             rewards) with other decision features (i.e., effort
             required, probability of an outcome, or time delays). Here
             we examine the overlap and dissociation of behavioral
             preferences and neural representations of subjective value
             in the context of three different decision features
             (physical effort, probability, time delays) in a healthy
             adult life-span sample. While undergoing functional
             neuroimaging, participants (N = 75) made
             incentive-compatible choices between a smaller monetary
             reward with lower physical effort, higher probability, or a
             shorter time delay, versus a larger monetary reward with
             higher physical effort, lower probability, or a longer time
             delay. Behavioral preferences were estimated from observed
             choices and subjective values were computed using individual
             hyperbolic discount functions. We found that discount rates
             were uncorrelated across tasks. In spite of this apparent
             behavioral dissociation between preferences, we found
             overlapping subjective value-related activity in the medial
             prefrontal cortex across all three tasks. We found no
             consistent evidence for age differences in either
             preferences or the neural representations of subjective
             value across adulthood. These results suggest that while the
             tolerance of decision features is behaviorally dissociable,
             subjective value signals share a common representation
             across adulthood.},
   Doi = {10.1093/scan/nsy021},
   Key = {fds333913}
}

@article{fds330888,
   Author = {Dang, LC and Samanez-Larkin, GR and Smith, CT and Castrellon, JJ and Perkins, SF and Cowan, RL and Claassen, DO and Zald,
             DH},
   Title = {FTO affects food cravings and interacts with age to
             influence age-related decline in food cravings.},
   Journal = {Physiology & Behavior},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.physbeh.2017.12.013},
   Abstract = {The fat mass and obesity associated gene (FTO) was the first
             gene identified by genome-wide association studies to
             correlate with higher body mass index (BMI) and increased
             odds of obesity. FTO remains the locus with the largest and
             most replicated effect on body weight, but the mechanism
             whereby FTO affects body weight and the development of
             obesity is not fully understood. Here we tested whether FTO
             is associated with differences in food cravings and a key
             aspect of dopamine function that has been hypothesized to
             influence food reward mechanisms. Moreover, as food cravings
             and dopamine function are known to decline with age, we
             explored effects of age on relations between FTO and food
             cravings and dopamine function. Seven-eight healthy subjects
             between 22 and 83years old completed the Food Cravings
             Questionnaire and underwent genotyping for FTO rs9939609,
             the first FTO single nucleotide polymorphism associated with
             obesity. Compared to TT homozygotes, individuals carrying
             the obesity-susceptible A allele had higher total food
             cravings, which correlated with higher BMI. Additionally,
             food cravings declined with age, but this age effect
             differed across variants of FTO rs9939609: while TT
             homozygotes showed the typical age-related decline in food
             cravings, there was no such decline among A carriers. All
             subjects were scanned with [18F]fallypride PET to assess a
             recent proposal that at the neurochemical level FTO alters
             dopamine D2-like receptor (DRD2) function to influence food
             reward related mechanisms. However, we observed no evidence
             of FTO effects on DRD2 availability.},
   Doi = {10.1016/j.physbeh.2017.12.013},
   Key = {fds330888}
}

@article{fds328773,
   Author = {Dang, LC and Samanez-Larkin, GR and Castrellon, JJ and Perkins, SF and Cowan, RL and Newhouse, PA and Zald, DH},
   Title = {Spontaneous Eye Blink Rate (EBR) Is Uncorrelated with
             Dopamine D2 Receptor Availability and Unmodulated by
             Dopamine Agonism in Healthy Adults.},
   Journal = {eNeuro},
   Volume = {4},
   Number = {5},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1523/eneuro.0211-17.2017},
   Abstract = {Spontaneous eye blink rate (EBR) has been proposed as a
             noninvasive, inexpensive marker of dopamine functioning.
             Support for a relation between EBR and dopamine function
             comes from observations that EBR is altered in populations
             with dopamine dysfunction and EBR changes under a
             dopaminergic manipulation. However, the evidence across the
             literature is inconsistent and incomplete. A direct
             correlation between EBR and dopamine function has so far
             been observed only in nonhuman animals. Given significant
             interest in using EBR as a proxy for dopamine function, this
             study aimed to verify a direct association in healthy, human
             adults. Here we measured EBR in healthy human subjects whose
             dopamine D2 receptor (DRD2) availability was assessed with
             positron emission tomography (PET)-[18F]fallypride to
             examine the predictive power of EBR for DRD2 availability.
             Effects of the dopamine agonist bromocriptine on EBR also
             were examined to determine the responsiveness of EBR to
             dopaminergic stimulation and, in light of the hypothesized
             inverted-U profile of dopamine effects, the role of DRD2
             availability in EBR responsivity to bromocriptine. Results
             from 20 subjects (age 33.6 ± 7.6 years, 9F) showed no
             relation between EBR and DRD2 availability. EBR also was not
             responsive to dopaminergic stimulation by bromocriptine, and
             individual differences in DRD2 availability did not modulate
             EBR responsivity to bromocriptine. Given that EBR is
             hypothesized to be particularly sensitive to DRD2 function,
             these findings suggest caution in using EBR as a proxy for
             dopamine function in healthy humans.},
   Doi = {10.1523/eneuro.0211-17.2017},
   Key = {fds328773}
}

@article{fds327928,
   Author = {Barber, SJ and Castrellon, JJ and Opitz, P and Mather,
             M},
   Title = {Younger and older adults' collaborative recall of shared and
             unshared emotional pictures.},
   Journal = {Memory & Cognition},
   Volume = {45},
   Number = {5},
   Pages = {716-730},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.3758/s13421-017-0694-3},
   Abstract = {Although a group of people working together recalls more
             items than any one individual, they recall fewer unique
             items than the same number of people working apart whose
             responses are combined. This is known as collaborative
             inhibition, and it is a robust effect that occurs for both
             younger and older adults. However, almost all previous
             studies documenting collaborative inhibition have used
             stimuli that were neutral in emotional valence, low in
             arousal, and studied by all group members. In the current
             experiments, we tested the impact of picture-stimuli
             valence, picture-stimuli arousal, and information
             distribution in modulating the magnitude of collaborative
             inhibition. We included both younger and older adults
             because there are age differences in how people remember
             emotional pictures that could modulate any effects of
             emotion on collaborative inhibition. Results revealed that
             when information was shared (i.e., studied by all group
             members), there were robust collaborative inhibition effects
             for both neutral and emotional stimuli for both younger and
             older adults. However, when information was unshared (i.e.,
             studied by only a single group member), these effects were
             attenuated. Together, these results provide mixed support
             for the retrieval strategy disruption account of
             collaborative inhibition. Supporting the retrieval strategy
             disruption account, unshared study information was less
             susceptible to collaborative inhibition than shared study
             information. Contradicting the retrieval strategy disruption
             account, emotional valence and arousal did not modulate the
             magnitude of collaborative inhibition despite the fact that
             participants clustered the emotional, but not neutral,
             information together in memory.},
   Doi = {10.3758/s13421-017-0694-3},
   Key = {fds327928}
}

@article{fds327929,
   Author = {Dang, LC and Castrellon, JJ and Perkins, SF and Le, NT and Cowan, RL and Zald, DH and Samanez-Larkin, GR},
   Title = {Reduced effects of age on dopamine D2 receptor levels in
             physically active adults.},
   Journal = {NeuroImage},
   Volume = {148},
   Pages = {123-129},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2017.01.018},
   Abstract = {Physical activity has been shown to ameliorate dopaminergic
             degeneration in non-human animal models. However, the
             effects of regular physical activity on normal age-related
             changes in dopamine function in humans are unknown. Here we
             present cross-sectional data from forty-four healthy human
             subjects between 23 and 80 years old, showing that typical
             age-related dopamine D2 receptor loss, assessed with PET
             [18F]fallypride, was significantly reduced in physically
             active adults compared to less active adults.},
   Doi = {10.1016/j.neuroimage.2017.01.018},
   Key = {fds327929}
}


%% Chartrand, Tanya L.   
@article{fds332782,
   Title = {Erratum},
   Journal = {Journal of Consumer Research},
   Volume = {44},
   Number = {5},
   Pages = {1174-1174},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/jcr/ucx095},
   Doi = {10.1093/jcr/ucx095},
   Key = {fds332782}
}

@article{fds327053,
   Author = {Leander, NP and Chartrand, TL},
   Title = {On thwarted goals and displaced aggression: A compensatory
             competence model},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {72},
   Pages = {88-100},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.04.010},
   Doi = {10.1016/j.jesp.2017.04.010},
   Key = {fds327053}
}

@article{fds332783,
   Author = {Brick, DJ and Fitzsimons, GM and Chartrand, TL and Fitzsimons,
             GJ},
   Title = {Coke vs. Pepsi: Brand Compatibility, Relationship Power, and
             Life Satisfaction},
   Journal = {Journal of Consumer Research},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1093/jcr/ucx079},
   Doi = {10.1093/jcr/ucx079},
   Key = {fds332783}
}

@article{fds327054,
   Author = {Leander, NP and Kay, AC and Chartrand, TL and Payne,
             BK},
   Title = {An Affect Misattribution Pathway to Perceptions of Intrinsic
             Reward},
   Journal = {Social Cognition},
   Volume = {35},
   Number = {2},
   Pages = {163-180},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1521/soco.2017.35.2.163},
   Abstract = {© 2017 Guilford Publications, Inc. Intrinsic rewards are
             typically thought to stem from an activity's inherent
             properties and not from separable rewards one receives from
             it. Yet, people may not consciously notice or remember all
             the subtle external rewards that correspond with an activity
             and may misattribute some directly to the activity itself.
             We propose that perceptions of intrinsic reward can often be
             byproducts of misattributed causal inference, and present
             some initial evidence that perceptions of intrinsic reward
             can in fact increase when words pertaining to an activity
             are subtly paired with pleasant context cues. Importantly,
             these effects follow classic boundary conditions of both
             misattribution and intrinsic motivation, insofar as they
             were extinguished when participants could make a proper
             source attribution and/or when the activity became
             associated with a blatant external reward. We further
             propose a distinction can be made between authentically
             "intrinsic" rewards and the illusion of intrinsic rewards
             caused by misattributed positive affect.},
   Doi = {10.1521/soco.2017.35.2.163},
   Key = {fds327054}
}

@article{fds323533,
   Author = {Duffy, KA and Harris, LT and Chartrand, TL and Stanton,
             SJ},
   Title = {Women recovering from social rejection: The effect of the
             person and the situation on a hormonal mechanism of
             affiliation.},
   Journal = {Psychoneuroendocrinology},
   Volume = {76},
   Pages = {174-182},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2016.11.017},
   Abstract = {Rejection can motivate either affiliation or withdrawal. In
             order to study how personality and situational variables
             influence whether women will be motivated to affiliate
             versus withdraw, we manipulate social feedback (rejection
             vs. acceptance) and opportunity for face-to-face interaction
             (blocked vs. face-to-face) and measure the individual
             difference variables rejection sensitivity and social
             anxiety. We test how these variables affect endogenous
             progesterone and cortisol concentrations, which are presumed
             to signal motivational responses to rejection. We find that
             three-way interactions involving social feedback,
             opportunity for face-to-face interactions, and either social
             anxiety or rejection sensitivity significantly predict
             progesterone change, but not cortisol change. Both
             interactions are driven by sharp progesterone decreases for
             women high in social anxiety/rejection sensitivity who have
             been rejected and who have no opportunity to reaffiliate in
             a face-to-face interaction. This progesterone change may be
             a physiological marker of motivation for social avoidance
             following rejection for women who cannot reaffiliate and who
             are particularly socially anxious or sensitive to
             rejection.},
   Doi = {10.1016/j.psyneuen.2016.11.017},
   Key = {fds323533}
}


%% Cherenack, Emily M.   
@article{fds331648,
   Author = {Sikkema, KJ and Mulawa, MI and Robertson, C and Watt, MH and Ciya, N and Stein, DJ and Cherenack, EM and Choi, KW and Kombora, M and Joska,
             JA},
   Title = {Improving AIDS Care After Trauma (ImpACT): Pilot Outcomes of
             a Coping intervention Among HIV-Infected Women with Sexual
             Trauma in South Africa.},
   Journal = {AIDS and Behavior},
   Volume = {22},
   Number = {3},
   Pages = {1039-1052},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1007/s10461-017-2013-1},
   Abstract = {Improving AIDS Care after Trauma (ImpACT), a coping
             intervention for HIV-infected women with sexual abuse
             histories, was evaluated for feasibility and potential
             efficacy in a public clinic in Cape Town, South Africa.
             Sixty-four participants were enrolled prior to starting
             antiretroviral therapy (ART). After completing baseline
             assessments, participants were randomly assigned to standard
             of care (SoC: three adherence counseling sessions) or ImpACT
             (SoC plus four individual and three group sessions).
             Participants completed assessments at 3 months (after
             individual sessions) and 6 months post-baseline. In
             exploratory analysis of primary outcomes, ImpACT
             participants, compared to SoC, reported greater reductions
             in avoidance and arousal symptoms of PTSD and greater
             increases in ART adherence motivation at 3 months.
             Clinically significant decreases in overall PTSD symptoms
             were also demonstrated at 3 months. These effects continued
             as trends at the 6-month assessment, in addition to
             increases in social/spiritual coping. In analysis of
             secondary outcomes, high levels of non-adherence to ART and
             poor care engagement were evident at 6 months, with no
             differences between study arms. A trauma-focused,
             culturally-adapted individual intervention delivered by a
             non-specialist in the HIV care setting is feasible and
             acceptable. Preliminary findings suggest ImpACT has
             potential to reduce PTSD symptoms and increase ART adherence
             motivation, but a more intensive intervention may be needed
             to improve and maintain care engagement among this
             population.ClinicalTrials.gov NCT02223390.},
   Doi = {10.1007/s10461-017-2013-1},
   Key = {fds331648}
}

@article{fds332042,
   Author = {Cherenack, EM and Sikkema, KJ and Watt, MH and Hansen, NB and Wilson,
             PA},
   Title = {Avoidant Coping Mediates the Relationship Between
             Self-Efficacy for HIV Disclosure and Depression Symptoms
             Among Men Who Have Sex with Men Newly Diagnosed with
             HIV.},
   Journal = {AIDS and Behavior},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s10461-018-2036-2},
   Abstract = {HIV diagnosis presents a critical opportunity to reduce
             secondary transmission, improve engagement in care, and
             enhance overall well-being. To develop relevant
             interventions, research is needed on the psychosocial
             experiences of newly diagnosed individuals. This study
             examined avoidant coping, self-efficacy for HIV disclosure
             decisions, and depression among 92 newly diagnosed men who
             have sex with men who reported recent sexual risk behavior.
             It was hypothesized that avoidant coping would mediate the
             relationship between self-efficacy and depression.
             Cross-sectional surveys were collected from participants
             3 months after HIV diagnosis. To test for mediation,
             multiple linear regressions were conducted while controlling
             for HIV disclosure to sexual partners. Self-efficacy for HIV
             disclosure decisions showed a negative linear relationship
             to depression symptoms, and 99% of this relationship was
             mediated by avoidant coping. The index of mediation of
             self-efficacy on depression indicated a small-to-medium
             effect. Higher self-efficacy was related to less avoidant
             coping, and less avoidant coping was related to decreased
             depression symptoms, all else held constant. These findings
             highlight the role of avoidant coping in explaining the
             relationship between self-efficacy for HIV disclosure
             decisions and depression.},
   Doi = {10.1007/s10461-018-2036-2},
   Key = {fds332042}
}

@article{fds330403,
   Author = {Wilson, PA and Cherenack, EM and Jadwin-Cakmak, L and Harper, GW and Adolescent Medicine Trials Network for HIV/AIDS
             Interventions},
   Title = {Selection and Evaluation of Media for Behavioral Health
             Interventions Employing Critical Media Analysis.},
   Journal = {Health Promotion Practice},
   Volume = {19},
   Number = {1},
   Pages = {145-156},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1177/1524839917711384},
   Abstract = {Although a growing number of psychosocial health promotion
             interventions use the critical analysis of media to
             facilitate behavior change, no specific guidelines exist to
             assist researchers and practitioners in the selection and
             evaluation of culturally relevant media stimuli for
             intervention development. Mobilizing Our Voices for
             Empowerment is a critical consciousness-based health
             enhancement intervention for HIV-positive Black young
             gay/bisexual men that employs the critical analysis of
             popular media. In the process of developing and testing this
             intervention, feedback on media stimuli was collected from
             youth advisory board members (n = 8), focus group
             participants (n = 19), intervention participants (n = 40),
             and intervention facilitators (n = 6). A thematic analysis
             of qualitative data resulted in the identification of four
             key attributes of media stimuli and participants' responses
             to media stimuli that are important to consider when
             selecting and evaluating media stimuli for use in behavioral
             health interventions employing the critical analysis of
             media: comprehension, relevance, emotionality, and action.
             These four attributes are defined and presented as a
             framework for evaluating media, and adaptable tools are
             provided based on this framework to guide researchers and
             practitioners in the selection and evaluation of media for
             similar interventions.},
   Doi = {10.1177/1524839917711384},
   Key = {fds330403}
}


%% Compton, Scott N.   
@article{fds328085,
   Author = {Selles, RR and Franklin, M and Sapyta, J and Compton, SN and Tommet, D and Jones, RN and Garcia, A and Freeman, J},
   Title = {Children's and Parents' Ability to Tolerate Child Distress:
             Impact on Cognitive Behavioral Therapy for Pediatric
             Obsessive Compulsive Disorder.},
   Journal = {Child Psychiatry & Human Development},
   Volume = {49},
   Number = {2},
   Pages = {308-316},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s10578-017-0748-6},
   Abstract = {The present study explored the concept of tolerance for
             child distress in 46 children (ages 5-8), along with their
             mothers and fathers, who received family-based CBT for OCD.
             The study sought to describe baseline tolerance, changes in
             tolerance with treatment, and the predictive impact of
             tolerance on symptom improvement. Tolerance was rated by
             clinicians on a single item and the CY-BOCS was used to
             measure OCD severity. Descriptive results suggested that all
             participants had some difficulty tolerating the child's
             distress at baseline while paired t tests indicated large
             improvements were made over treatment (d = 1.2-2.0).
             Fathers' initial tolerance was significantly related to
             symptom improvement in a multivariate regression as were
             fathers' and children's changes in distress tolerance over
             the course of treatment. Overall, results provide support
             for examining tolerance of child distress including its
             predictive impact and potential as a supplemental
             intervention target.},
   Doi = {10.1007/s10578-017-0748-6},
   Key = {fds328085}
}

@article{fds327202,
   Author = {Hale, AE and Ginsburg, GS and Chan, G and Kendall, PC and McCracken, JT and Sakolsky, D and Birmaher, B and Compton, SN and Albano, AM and Walkup,
             JT},
   Title = {Mediators of Treatment Outcomes for Anxious Children and
             Adolescents: The Role of Somatic Symptoms.},
   Journal = {Journal of Clinical Child & Adolescent Psychology},
   Volume = {47},
   Number = {1},
   Pages = {94-104},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1080/15374416.2017.1280804},
   Abstract = {Cognitive behavioral therapy (CBT) and selective serotonin
             reuptake inhibitors are effective treatments for pediatric
             anxiety disorders. However, the mechanisms of these
             treatments are unknown. Previous research indicated that
             somatic symptoms are reduced following treatment, but it is
             unclear if their reductions are merely a consequence of
             treatment gains. This study examined reductions in somatic
             symptoms as a potential mediator of the relationship between
             treatment and anxiety outcomes. Participants were 488
             anxious youth ages 7-17 (M = 10.7), 50.4% male, 78.9%
             Caucasian, enrolled in Child/Adolescent Anxiety Multimodal
             Study, a large randomized control trial comparing 12-week
             treatments of CBT, sertraline, a combination of CBT and
             sertraline, and a pill placebo. Causal mediation models were
             tested in R using data from baseline, 8-, and 12-week
             evaluations. Somatic symptoms were assessed using the
             Panic/Somatic subscale from the Screen for Child Anxiety
             Related Emotional Disorders. Youth outcomes were assessed
             using the Pediatric Anxiety Rating Scale and Children's
             Global Assessment Scale. Reductions in somatic symptoms
             mediated improvement in anxiety symptoms and global
             functioning for those in the sertraline-only condition based
             on parent report. Conditions involving CBT and data based on
             child reported somatic symptoms did not show a mediation
             effect. Findings indicate that reductions in somatic
             symptoms may be a mediator of improvements for treatments
             including pharmacotherapy and not CBT. Although the overall
             efficacy of sertraline and CBT for anxiety may be similar,
             the treatments appear to function via different
             mechanisms.},
   Doi = {10.1080/15374416.2017.1280804},
   Key = {fds327202}
}

@article{fds330474,
   Author = {Peris, TS and Caporino, NE and O'Rourke, S and Kendall, PC and Walkup,
             JT and Albano, AM and Bergman, RL and McCracken, JT and Birmaher, B and Ginsburg, GS and Sakolsky, D and Piacentini, J and Compton,
             SN},
   Title = {Therapist-Reported Features of Exposure Tasks That Predict
             Differential Treatment Outcomes for Youth With
             Anxiety.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {12},
   Pages = {1043-1052},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.10.001},
   Abstract = {Exposure tasks are recognized widely as a key component of
             cognitive-behavioral therapy (CBT) for child and adolescent
             anxiety. However, little research has examined specific
             exposure characteristics that predict outcomes for youth
             with anxiety and that may guide its application in
             therapy.This study draws on a sample of 279 children and
             adolescents (48.4% male; 79.6% white) with a principal
             anxiety disorder who received 14 sessions of CBT, either
             alone or in combination with medication, through the
             Child/adolescent Anxiety Multimodal treatment Study (CAMS).
             The present study examines therapist-reported quantity,
             difficulty level, compliance, and mastery of exposure tasks
             as they related to CBT response (i.e., Clinical Global
             Impressions-Improvement ratings). Secondary treatment
             outcomes included reduction in anxiety symptom severity on
             the Pediatric Anxiety Rating Scale, global impairment
             measured via the Children's Global Assessment Scale, and
             parent-report of anxiety-specific functional impairment on
             the Child Anxiety Impairment Scale.Regression analyses
             indicated a dose-response relationship between
             therapist-reported quantity of exposure and independent
             evaluations of treatment outcome, with more time devoted to
             exposure linked to better outcomes. Similarly, greater time
             spent on more difficult (rather than mild or moderate)
             exposure tasks predicted better outcomes, as did therapist
             ratings of child compliance and mastery.The present findings
             highlight the importance of challenging children and
             adolescents with difficult exposure tasks and of
             collaborating to ensure compliance and mastery.},
   Doi = {10.1016/j.jaac.2017.10.001},
   Key = {fds330474}
}

@article{fds330044,
   Author = {Højgaard, DR and Hybel, KA and Ivarsson, T and Skarphedinsson, G and Becker Nissen and J and Weidle, B and Melin, K and Torp, NC and Valderhaug,
             R and Dahl, K and Mortensen, EL and Compton, S and Jensen, S and Lenhard,
             F and Thomsen, PH},
   Title = {One-Year Outcome for Responders of Cognitive-Behavioral
             Therapy for Pediatric Obsessive-Compulsive
             Disorder},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {940-947.e1},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.09.002},
   Doi = {10.1016/j.jaac.2017.09.002},
   Key = {fds330044}
}

@article{fds323540,
   Author = {Caporino, NE and Read, KL and Shiffrin, N and Settipani, C and Kendall,
             PC and Compton, SN and Sherrill, J and Piacentini, J and Walkup, J and Ginsburg, G and Keeton, C and Birmaher, B and Sakolsky, D and Gosch, E and Albano, AM},
   Title = {Sleep-Related Problems and the Effects of Anxiety Treatment
             in Children and Adolescents.},
   Journal = {Journal of Clinical Child & Adolescent Psychology},
   Volume = {46},
   Number = {5},
   Pages = {675-685},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1080/15374416.2015.1063429},
   Abstract = {This study examined (a) demographic and clinical
             characteristics associated with sleep-related problems
             (SRPs) among youth with anxiety disorders, and (b) the
             impact of anxiety treatment: cognitive-behavioral therapy
             (CBT; Coping Cat), medication (sertraline), their
             combination, and pill placebo on SRPs. Youth (N = 488,
             ages 7-17, 50% female, 79% White) with a principal diagnosis
             of generalized anxiety disorder, separation anxiety
             disorder, or social phobia participated. SRPs were reported
             by parents and youth. Findings differed by informant and by
             type of SRP, with evidence that SRPs are associated with
             age, anxiety severity, externalizing problems, functional
             impairment, and family burden at pretreatment. Anxiety
             treatment reduced SRPs; effect sizes were small to medium.
             Reductions in parent-reported separation-related sleep
             difficulties were significantly greater in active treatment
             than in the placebo condition, with the greatest reductions
             reported by parents of youth whose active treatment was
             multimodal or included sertraline. Youth whose anxiety
             treatment involved CBT reported significantly greater
             decreases in dysregulated sleep (e.g., sleeplessness). Both
             CBT for anxiety and sertraline appear to be somewhat
             effective in reducing SRPs, and multimodal treatment may be
             preferable depending on the symptom presentation. To inform
             practice, future research should examine a broad range of
             SRPs, incorporate objective measures of sleep, and evaluate
             the impact of behavioral strategies that directly target
             SRPs in youth with anxiety disorders.},
   Doi = {10.1080/15374416.2015.1063429},
   Key = {fds323540}
}

@article{fds327204,
   Author = {Hoff, AL and Kendall, PC and Langley, A and Ginsburg, G and Keeton, C and Compton, S and Sherrill, J and Walkup, J and Birmaher, B and Albano, AM and Suveg, C and Piacentini, J},
   Title = {Developmental Differences in Functioning in Youth With
             Social Phobia.},
   Journal = {Journal of Clinical Child & Adolescent Psychology},
   Volume = {46},
   Number = {5},
   Pages = {686-694},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1080/15374416.2015.1079779},
   Abstract = {Social phobia (SoP) in youth may manifest differently across
             development as parent involvement in their social lives
             changes and social and academic expectations increase. This
             cross-sectional study investigated whether self-reported and
             parent-reported functioning in youth with SoP changes with
             age in social, academic, and home/family domains. Baseline
             anxiety impairment data from 488 treatment-seeking
             anxiety-disordered youth (ages 7-17, N = 400 with a SoP
             diagnosis) and their parents were gathered using the Child
             Anxiety Impact Scale and were analyzed using generalized
             estimating equations. According to youth with SoP and their
             parents, overall difficulties, social difficulties, and
             academic difficulties increased with age, even when
             controlling for SoP severity. These effects significantly
             differed for youth with anxiety disorders other than SoP.
             Adolescents may avoid social situations as parental
             involvement in their social lives decreases, and their
             withdrawn behavior may result in increasing difficulty in
             the social domain. Their avoidance of class participation
             and oral presentations may increasingly impact their
             academic performance as school becomes more demanding.
             Implications are discussed for the early detection and
             intervention of SoP to prevent increased impairment over the
             course of development.},
   Doi = {10.1080/15374416.2015.1079779},
   Key = {fds327204}
}

@article{fds327200,
   Author = {Strawn, JR and Dobson, ET and Mills, JA and Cornwall, GJ and Sakolsky,
             D and Birmaher, B and Compton, SN and Piacentini, J and McCracken, JT and Ginsburg, GS and Kendall, PC and Walkup, JT and Albano, AM and Rynn,
             MA},
   Title = {Placebo Response in Pediatric Anxiety Disorders: Results
             from the Child/Adolescent Anxiety Multimodal
             Study.},
   Journal = {Journal of Child and Adolescent Psychopharmacology},
   Volume = {27},
   Number = {6},
   Pages = {501-508},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1089/cap.2016.0198},
   Abstract = {The aim of this study is to identify predictors of pill
             placebo response and to characterize the temporal course of
             pill placebo response in anxious youth.Data from
             placebo-treated patients (N = 76) in the
             Child/Adolescent Anxiety Multimodal Study (CAMS), a
             multisite, randomized controlled trial that examined the
             efficacy of cognitive-behavioral therapy, sertraline, their
             combination, and placebo for the treatment of separation,
             generalized, and social anxiety disorders, were evaluated.
             Multiple linear regression models identified features
             associated with placebo response and models were confirmed
             with leave-one-out cross-validation. The likelihood of
             improvement in patients receiving pill placebo-over
             time-relative to improvement associated with active
             treatment was determined using probabilistic Bayesian
             analyses.Based on a categorical definition of response
             (Clinical Global Impressions-Improvement Scale score ≤2),
             nonresponders (n = 48), and pill placebo responders
             (n = 18) did not differ in age (p = 0.217), sex
             (p = 0.980), race (p = 0.743), or primary diagnosis
             (all ps > 0.659). In terms of change in anxiety symptoms,
             separation anxiety disorder and treatment expectation were
             associated with the degree of pill placebo response. Greater
             probability of placebo-related anxiety symptom improvement
             was observed early in the course of treatment (baseline to
             week 4, p < 0.0001). No significant change in the
             probability of placebo-related improvement was observed
             after week 4 (weeks 4-8, p = 0.07; weeks 8-12,
             p = 0.85), whereas the probability of improvement, in
             general, significantly increased week over week with active
             treatment.Pill placebo-related improvement occurs early in
             the course of treatment and both clinical factors and
             expectation predict this improvement. Additionally,
             probabilistic approaches may refine our understanding and
             prediction of pill placebo response.},
   Doi = {10.1089/cap.2016.0198},
   Key = {fds327200}
}

@article{fds326112,
   Author = {Gonzalez, A and Rozenman, M and Langley, AK and Kendall, PC and Ginsburg, GS and Compton, S and Walkup, JT and Birmaher, B and Albano,
             AM and Piacentini, J},
   Title = {Social Interpretation Bias in Children and Adolescents with
             Anxiety Disorders: Psychometric Examination of the
             Self-report of Ambiguous Social Situations for Youth (SASSY)
             Scale.},
   Journal = {Child & Youth Care Forum},
   Volume = {46},
   Number = {3},
   Pages = {395-412},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s10566-016-9381-y},
   Abstract = {Anxiety disorders are among the most common mental health
             problems in youth, and faulty interpretation bias has been
             positively linked to anxiety severity, even within
             anxiety-disordered youth. Quick, reliable assessment of
             interpretation bias may be useful in identifying youth with
             certain types of anxiety or assessing changes on cognitive
             bias during intervention.This study examined the factor
             structure, reliability, and validity of the Self-report of
             Ambiguous Social Situations for Youth (SASSY) scale, a
             self-report measure developed to assess interpretation bias
             in youth.Participants (N=488, age 7 to 17) met diagnostic
             criteria for Social Phobia, Generalized Anxiety Disorder,
             and/or Separation Anxiety Disorder. An exploratory factor
             analysis was performed on baseline data from youth
             participating in a large randomized clinical
             trial.Exploratory factor analysis yielded two factors
             (Accusation/Blame, Social Rejection). The SASSY full scale
             and Social Rejection factor demonstrated adequate internal
             consistency, convergent validity with social anxiety, and
             discriminant validity as evidenced by non-significant
             correlations with measures of non-social anxiety. Further,
             the SASSY Social Rejection factor accurately distinguished
             children and adolescents with Social Phobia from those with
             other anxiety disorders, supporting its criterion validity,
             and revealed sensitivity to changes with treatment. Given
             the relevance to youth with social phobia, pre- and
             post-intervention data were examined for youth social phobia
             to test sensitivity to treatment effects; results suggested
             that SASSY scores reduced for treatment responders.Findings
             suggest the potential utility of the SASSY Social Rejection
             factor as a quick, reliable, and efficient way of assessing
             interpretation bias in anxious youth, particularly as
             related to social concerns, in research and clinical
             settings.},
   Doi = {10.1007/s10566-016-9381-y},
   Key = {fds326112}
}

@article{fds326330,
   Author = {Lee, P and Zehgeer, A and Ginsburg, GS and McCracken, J and Keeton, C and Kendall, PC and Birmaher, B and Sakolsky, D and Walkup, J and Peris, T and Albano, AM and Compton, S},
   Title = {Child and Adolescent Adherence With Cognitive Behavioral
             Therapy for Anxiety: Predictors and Associations With
             Outcomes.},
   Journal = {Journal of Clinical Child & Adolescent Psychology},
   Pages = {1-12},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1080/15374416.2017.1310046},
   Abstract = {Cognitive behavioral therapy (CBT) for anxiety disorders is
             effective, but nonadherence with treatment may reduce the
             benefits of CBT. This study examined (a) four baseline
             domains (i.e., demographic, youth clinical characteristics,
             therapy related, family/parent factors) as predictors of
             youth adherence with treatment and (b) the associations
             between youth adherence and treatment outcomes. Data were
             from 279 youth (7-17 years of age, 51.6% female; 79.6%
             White, 9% African American), with Diagnostic and Statistical
             Manual of Mental Disorders (4th ed., text rev.) diagnoses of
             separation anxiety disorder, generalized anxiety disorder,
             and/or social phobia, who participated in CBT in the
             Child/Adolescent Anxiety Multimodal Study. Adherence was
             defined in three ways (session attendance, therapist-rated
             compliance, and homework completion). Multiple regressions
             revealed several significant predictors of youth adherence
             with CBT, but predictors varied according to the definition
             of adherence. The most robust predictors of greater
             adherence were living with both parents and fewer youth
             comorbid externalizing disorders. With respect to outcomes,
             therapist ratings of higher youth compliance with CBT
             predicted several indices of favorable outcome: lower
             anxiety severity, higher global functioning, and treatment
             responder status after 12 weeks of CBT. Number of sessions
             attended and homework completion did not predict treatment
             outcomes. Findings provide information about risks for youth
             nonadherence, which can inform treatment and highlight the
             importance of youth compliance with participating in therapy
             activities, rather than just attending sessions or
             completing homework assignments.},
   Doi = {10.1080/15374416.2017.1310046},
   Key = {fds326330}
}

@article{fds327201,
   Author = {Alexander, JR and Houghton, DC and Twohig, MP and Franklin, ME and Saunders, SM and Neal-Barnett, AM and Compton, SN and Woods,
             DW},
   Title = {Clarifying the Relationship between Trichotillomania and
             Anxiety.},
   Journal = {Journal of Obsessive-Compulsive and Related
             Disorders},
   Volume = {13},
   Pages = {30-34},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jocrd.2017.02.004},
   Abstract = {Although research has consistently linked unidimensional
             anxiety with Trichotillomania (TTM) severity, the
             relationships between TTM severity and anxiety dimensions
             (i.e., cognitive and somatic anxiety) are unknown. This
             knowledge gap limits current TTM conceptualization and
             treatment. The current study examined these relationships
             with data collected from ninety-one adults who participated
             in a randomized clinical trial for TTM treatment. To examine
             whether the Beck Anxiety Inventory (BAI; Beck, Epstein,
             Brown, & Steer, 1988) could be used to measure
             multidimensional anxiety in TTM samples, we conducted a
             factor analysis. Results showed four emergent factors,
             including a cognitive factor and three somatic factors
             (neurophysiological, autonomic, and panic). Based on prior
             research, it was hypothesized that TTM severity would be
             related to the cognitive anxiety dimension and that
             psychological inflexibility would mediate the association.
             Hypotheses were not made regarding the relationship between
             TTM severity and somatic anxiety. Regression analyses
             indicated that only cognitive dimensions of anxiety
             predicted TTM severity and that psychological inflexibility
             mediated this relationship. Implications for the
             conceptualization and treatment of TTM are
             discussed.},
   Doi = {10.1016/j.jocrd.2017.02.004},
   Key = {fds327201}
}

@article{fds327203,
   Author = {Strawn, JR and Compton, SN and Robertson, B and Albano, AM and Hamdani,
             M and Rynn, MA},
   Title = {Extended Release Guanfacine in Pediatric Anxiety Disorders:
             A Pilot, Randomized, Placebo-Controlled Trial.},
   Journal = {Journal of Child and Adolescent Psychopharmacology},
   Volume = {27},
   Number = {1},
   Pages = {29-37},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1089/cap.2016.0132},
   Abstract = {This is a feasibility study evaluating the safety,
             tolerability, and potential anxiolytic efficacy of the α2
             agonist guanfacine extended-release (GXR) in children and
             adolescents with generalized anxiety disorder (GAD),
             separation anxiety disorder (SAD), or social phobia/social
             anxiety disorder.Youth aged 6-17 years with a primary
             diagnosis of GAD, SAD, and/or social anxiety disorder were
             treated with flexibly dosed GXR (1-6 mg daily, n = 62)
             or placebo (n = 21) for 12 weeks. The primary aim of
             this study was to determine the safety and tolerability of
             GXR in youth with anxiety disorders, which involved the
             analysis of treatment-emergent adverse events (TEAEs), the
             emergence of suicidal ideation and behaviors, vital signs,
             and electrocardiographic/laboratory parameters. Exploratory
             efficacy measures included dimensional anxiety scales
             (Pediatric Anxiety Rating Scale [PARS] and Screen for Child
             Anxiety Related Emotional Disorders [SCARED]), as well as
             the Clinical Global Impression-Improvement (CGI-I) scale. As
             this was an exploratory study, no inferential statistical
             analyses were performed.GXR was safe and well tolerated.
             Treatment-related mean ± standard deviation changes in
             heart rate (GXR: 1.8 ± 12 beats per minute [bpm]
             decrease; placebo: 0.5 ± 11 bpm decrease), systolic
             blood pressure (GXR: 2.3 ± 11 mm Hg decrease;
             placebo: 1.7 ± 11 mm Hg decrease), or diastolic blood
             pressure (GXR: 1.3 ± 9 mm Hg decrease; placebo:
             0.9 ± 7 mm Hg increase) were similar between
             treatment groups. TEAEs, including headache,
             somnolence/fatigue, abdominal pain, and dizziness, were
             consistent with the known safety profile of GXR. No
             differences were observed between treatment groups for PARS
             and SCARED scores, although at endpoint, a higher proportion
             of subjects receiving GXR versus placebo demonstrated CGI-I
             scores ≤2 (54.2% vs. 31.6%), as rated by the clinician
             investigator.GXR was well tolerated in pediatric subjects
             with GAD, SAD, and/or social anxiety disorder.
             ClinicalTrials.gov Identifier: NCT01470469.},
   Doi = {10.1089/cap.2016.0132},
   Key = {fds327203}
}


%% Cooper, Harris M.   
@article{fds332043,
   Author = {Appelbaum, M and Cooper, H and Kline, RB and Mayo-Wilson, E and Nezu,
             AM and Rao, SM},
   Title = {Journal article reporting standards for quantitative
             research in psychology: The APA Publications and
             Communications Board task force report.},
   Journal = {American Psychologist},
   Volume = {73},
   Number = {1},
   Pages = {3-25},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1037/amp0000191},
   Abstract = {Following a review of extant reporting standards for
             scientific publication, and reviewing 10 years of experience
             since publication of the first set of reporting standards by
             the American Psychological Association (APA; APA
             Publications and Communications Board Working Group on
             Journal Article Reporting Standards, 2008), the APA Working
             Group on Quantitative Research Reporting Standards
             recommended some modifications to the original standards.
             Examples of modifications include division of hypotheses,
             analyses, and conclusions into 3 groupings (primary,
             secondary, and exploratory) and some changes to the section
             on meta-analysis. Several new modules are included that
             report standards for observational studies, clinical trials,
             longitudinal studies, replication studies, and N-of-1
             studies. In addition, standards for analytic methods with
             unique characteristics and output (structural equation
             modeling and Bayesian analysis) are included. These
             proposals were accepted by the Publications and
             Communications Board of APA and supersede the standards
             included in the 6th edition of the Publication Manual of the
             American Psychological Association (APA, 2010). (PsycINFO
             Database Record},
   Doi = {10.1037/amp0000191},
   Key = {fds332043}
}

@article{fds324856,
   Author = {Sanchez, CE and Atkinson, KM and Koenka, AC and Moshontz, H and Cooper,
             H},
   Title = {Self-grading and peer-grading for formative and summative
             assessments in 3rd through 12th grade classrooms: A
             meta-analysis},
   Journal = {Journal of Educational Psychology},
   Volume = {109},
   Number = {8},
   Pages = {1049-1066},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1037/edu0000190},
   Abstract = {© 2017 American Psychological Association. The "assessment
             for learning" movement in education has increased attention
             to self-grading and peer-grading practices in primary and
             secondary schools. This research synthesis examined several
             questions pertaining to the use of self-grading and
             peer-grading in conjunction with criterion-referenced
             testing in 3rd- through 12th-grade-level classrooms. We
             investigated (a) the effects of students' participation in
             grading on subsequent test performance, (b) the difference
             between grades when assigned by students or teachers, and
             (c) the correlation between grades assigned by students and
             teachers. Students who engaged in self-grading performed
             better (g = .34) on subsequent tests than did students who
             did not. Moderator analyses suggested that the benefits of
             self-grading were estimated to be greater when the study
             controlled for group differences through random assignment.
             Students who engaged in peergrading performed better on
             subsequent tests than did students who did not (g = .29). On
             average, students did not grade themselves or peers
             significantly differently than teachers (self-grades, g =
             .04; peer-grades, g = .04) and showed moderate correlation
             (self-grading, r = .67; peer-grading, r = .68) with teacher
             grades. Further, other moderator analyses and examination of
             studies suggested that self- and peer-grading practices can
             be implemented to positive effect in primary and secondary
             schools with the use of rubrics and training for students in
             a formative assessment environment. However, because of a
             limited number of studies, these mediating variables need
             more research to allow more conclusive findings.},
   Doi = {10.1037/edu0000190},
   Key = {fds324856}
}

@book{fds326524,
   Author = {Cooper, H},
   Title = {Research Synthesis and Meta-Analysis: A Step-by-Step
             Approach, 5th Edition},
   Volume = {2},
   Pages = {384 pages},
   Publisher = {Sage Publications, Inc.},
   Year = {2017},
   ISBN = {1483331156},
   Abstract = {The Fifth Edition of Harris Cooper's bestselling Research
             Synthesis and Meta-Analysis: A Step-by-Step Approach offers
             practical advice on how to conduct a synthesis of research
             in the social, behavioral, and health sciences. The book is
             written in plain language with four running examples drawn
             from psychology, education, and health science. With ample
             coverage of literature searching and the technical aspects
             of meta-analysis, this one-of-a-kind book applies the basic
             principles of sound data gathering to the task of producing
             a comprehensive assessment of existing research.},
   Key = {fds326524}
}


%% Costanzo, Philip R.   
@article{fds326697,
   Author = {Golonka, MM and Peairs, KF and Malone, PS and Grimes, CL and Costanzo,
             PR},
   Title = {Natural Peer Leaders as Substance Use Prevention Agents: the
             Teens' Life Choice Project.},
   Journal = {Prevention Science},
   Volume = {18},
   Number = {5},
   Pages = {555-566},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1007/s11121-017-0790-4},
   Abstract = {In adolescent social groups, natural peer leaders have been
             found to engage in more frequent experimentation with
             substance use and to possess disproportionate power to
             affect the behavior and social choices of their associated
             peer followers. In the current exploratory study, we used
             sociometrics and social cognitive mapping to identify
             natural leaders of cliques in a seventh grade population and
             invited the leaders to develop anti-drug presentations for
             an audience of younger peers. The program employed
             social-psychological approaches directed at having leaders
             proceed from extrinsic inducements to intrinsic
             identification with their persuasive products in the context
             of the group intervention process. The goals of the
             intervention were to induce substance resistant
             self-persuasion in the leaders and to produce a spread of
             this resistance effect to their peer followers. To test the
             intervention, we compared the substance use behaviors of the
             selected leaders and their peers to a control cohort. The
             study found preliminary support that the intervention
             produced changes in the substance use behavior among the
             leaders who participated in the intervention, but did not
             detect a spread to non-leader peers in the short term. This
             descriptive study speaks to the plausibility of employing
             self-persuasion paradigms to bring about change in high-risk
             behaviors among highly central adolescents. In addition, it
             highlights the viability of applying social psychological
             principles to prevention work and calls for more research in
             this area.},
   Doi = {10.1007/s11121-017-0790-4},
   Key = {fds326697}
}

@article{fds331136,
   Author = {Rothenberg, WA and Hussong, AM and Langley, HA and Egerton, GA and Halberstadt, AG and Coffman, JL and Mokrova, I and Costanzo,
             PR},
   Title = {Grateful parents raising grateful children: Niche selection
             and the socialization of child gratitude.},
   Journal = {Applied Developmental Science},
   Volume = {21},
   Number = {2},
   Pages = {106-120},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1080/10888691.2016.1175945},
   Abstract = {Given that children's exposure to gratitude-related
             activities may be one way that parents can socialize
             gratitude in their children, we examined whether parents'
             niche selection (i.e., tendency to choose perceived
             gratitude-inducing activities for their children) mediates
             the association between parents' reports of their own and
             their children's gratitude. Parent-child dyads (N =101;
             children aged 6-9; 52% girls; 80% Caucasian; 85% mothers)
             participated in a laboratory visit and parents also
             completed a seven-day online diary regarding children's
             gratitude. Decomposing specific indirect effects within a
             structural equation model, we found that parents high in
             gratitude were more likely to set goals to use niche
             selection as a gratitude socialization strategy, and thereby
             more likely to place their children in gratitude-related
             activities. Placement in these activities, in turn, was
             associated with more frequent expression of gratitude in
             children. We describe future directions for research on
             parents' role in socializing gratitude in their
             children.},
   Doi = {10.1080/10888691.2016.1175945},
   Key = {fds331136}
}


%% Costello, Elizabeth J.   
@article{fds333242,
   Author = {Akee, R and Copeland, W and Costello, EJ and Simeonova,
             E},
   Title = {How Does Household Income Affect Child Personality Traits
             and Behaviors?},
   Journal = {American Economic Review},
   Volume = {108},
   Number = {3},
   Pages = {775-827},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1257/aer.20160133},
   Abstract = {We examine the effects of a quasi-experimental unconditional
             household income transfer on child emotional and behavioral
             health and personality traits. Using longitudinal data, we
             find that there are large beneficial effects on children's
             emotional and behavioral health and personality traits
             during adolescence. We find evidence that these effects are
             most pronounced for children who start out with the lowest
             initial endowments. The income intervention also results in
             improvements in parental relationships which we interpret as
             a potential mechanism behind our findings.},
   Doi = {10.1257/aer.20160133},
   Key = {fds333242}
}

@article{fds329142,
   Author = {Hill, S and Shanahan, L and Costello, EJ and Copeland,
             W},
   Title = {Predicting Persistent, Limited, and Delayed Problematic
             Cannabis Use in Early Adulthood: Findings From a
             Longitudinal Study.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {966-974.e4},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.08.012},
   Abstract = {To identify risk profiles associated with patterns of
             problematic cannabis use in early adulthood.Data came from
             1,229 participants in the Great Smoky Mountains Study, a
             prospective 20-year cohort study from 1993 to 2015 that is
             representative of western North Carolina with yearly
             assessments conducted from ages 9 and 16 years, and
             assessments at ages 19, 21, 26, and 30 years. Patterns of
             problematic cannabis use (i.e., DSM-5 cannabis use disorder
             or daily use) in early adulthood included the following:
             nonproblematic use in late adolescence (ages 19-21) and
             early adulthood (ages 26-30); limited problematic use in
             late adolescence only; persistent problematic use in late
             adolescence and early adulthood; and delayed problematic use
             in early adulthood only. Multinominal logistic regression
             models examined pairwise associations between these patterns
             and risk factors in childhood/early adolescence (ages 9-16)
             and late adolescence (ages 19-21). Risk factors included
             psychiatric disorders (e.g., anxiety, depressive), other
             substance use (smoking, alcohol, illicit drugs), and
             challenging social factors (e.g., low socioeconomic status,
             family functioning, peers). Sex and race/ethnicity (white,
             African American, American Indian) interactions were
             tested.The persistent pattern (6.7% of sample) was
             characterized by more anxiety disorders across development
             and more DSM-5 CUD symptoms during late adolescence compared
             to the limited pattern (13.3%), which, in turn, had more
             childhood family instability and dysfunction. The delayed
             pattern (3.7%) was characterized by more externalizing
             disorders, maltreatment, and peer bullying in childhood
             compared to those in nonproblematic users. There were no
             significant interactions of sex or race/ethnicity.Problematic
             cannabis use patterns during early adulthood have
             distinctive risk profiles, which may be useful in tailoring
             targeted interventions.},
   Doi = {10.1016/j.jaac.2017.08.012},
   Key = {fds329142}
}

@article{fds329547,
   Author = {Copeland, WE and Goldston, DB and Costello, EJ},
   Title = {Adult Associations of Childhood Suicidal Thoughts and
             Behaviors: A Prospective, Longitudinal Analysis.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {958-965.e4},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.08.015},
   Abstract = {Suicidal thoughts and behavior (STBs) have their peak period
             of onset in adolescence, but little is known about how such
             behavior is associated with later functioning. The aim of
             this study is to test whether childhood STBs are related to
             adult psychiatric, suicidal, and functional outcomes.This is
             a prospective, population-based community study of 1,420
             participants assessed with structured interviews up to 7
             times in childhood/adolescence (ages 9-16 years; 6,674
             observations) for STBs including passive and active
             ideation, plans, and attempts. Participants were then
             assessed 4 times in young adulthood (ages 19, 21, 24, and 30
             years; 4,556 observations of 1,273 participants) for
             psychiatric diagnoses, STBs, and functional outcomes.By age
             16 years, 7.0% of participants had reported some type of
             STBs, with 3.9% reporting an attempt. Both ideation only and
             suicide attempts were associated with higher levels of
             anxiety disorders and STBs in adulthood, as well as poor
             functioning across financial, health, risky/illegal, and
             social domains. These observed effects generally were
             attenuated after adjusting for other psychiatric and
             psychosocial factors that predict childhood STBs
             (particularly maltreatment, depression, and disruptive
             behavior disorders). The exception was adult suicidal
             behavior, which was predicted by both childhood ideation and
             attempts, even in the fully adjusted model. Children and
             adolescents with STBs were more likely to have had a
             disrupted transition to adulthood.Childhood STBs are a
             marker for a multitude of poor psychiatric and functional
             outcomes in adulthood, but these effects are largely
             accounted for by other factors. In contrast, childhood STBs
             are a robust risk factor for adult suicidal thoughts and
             behavior.},
   Doi = {10.1016/j.jaac.2017.08.015},
   Key = {fds329547}
}

@article{fds326255,
   Author = {Clark, SL and McClay, JL and Adkins, DE and Kumar, G and Aberg, KA and Nerella, S and Xie, L and Collins, AL and Crowley, JJ and Quackenbush,
             CR and Hilliard, CE and Shabalin, AA and Vrieze, SI and Peterson, RE and Copeland, WE and Silberg, JL and McGue, M and Maes, H and Iacono, WG and Sullivan, PF and Costello, EJ and van den Oord,
             EJ},
   Title = {Deep Sequencing of 71 Candidate Genes to Characterize
             Variation Associated with Alcohol Dependence.},
   Journal = {Alcoholism: Clinical and Experimental Research},
   Volume = {41},
   Number = {4},
   Pages = {711-718},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1111/acer.13352},
   Abstract = {Previous genomewide association studies (GWASs) have
             identified a number of putative risk loci for alcohol
             dependence (AD). However, only a few loci have replicated
             and these replicated variants only explain a small
             proportion of AD risk. Using an innovative approach, the
             goal of this study was to generate hypotheses about
             potentially causal variants for AD that can be explored
             further through functional studies.We employed targeted
             capture of 71 candidate loci and flanking regions followed
             by next-generation deep sequencing (mean coverage 78X) in
             806 European Americans. Regions included in our targeted
             capture library were genes identified through published GWAS
             of alcohol, all human alcohol and aldehyde dehydrogenases,
             reward system genes including dopaminergic and opioid
             receptors, prioritized candidate genes based on previous
             associations, and genes involved in the absorption,
             distribution, metabolism, and excretion of drugs. We
             performed single-locus tests to determine if any single
             variant was associated with AD symptom count. Sets of
             variants that overlapped with biologically meaningful
             annotations were tested for association in aggregate.No
             single, common variant was significantly associated with AD
             in our study. We did, however, find evidence for association
             with several variant sets. Two variant sets were significant
             at the q-value <0.10 level: a genic enhancer for ADHFE1
             (p = 1.47 × 10-5 ; q = 0.019), an alcohol
             dehydrogenase, and ADORA1 (p = 5.29 × 10-5 ;
             q = 0.035), an adenosine receptor that belongs to a
             G-protein-coupled receptor gene family.To our knowledge,
             this is the first sequencing study of AD to examine variants
             in entire genes, including flanking and regulatory regions.
             We found that in addition to protein coding variant sets,
             regulatory variant sets may play a role in AD. From these
             findings, we have generated initial functional hypotheses
             about how these sets may influence AD.},
   Doi = {10.1111/acer.13352},
   Key = {fds326255}
}

@article{fds323141,
   Author = {Copeland, WE and Hill, S and Costello, EJ and Shanahan,
             L},
   Title = {Cannabis Use and Disorder From Childhood to Adulthood in a
             Longitudinal Community Sample With American
             Indians.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {2},
   Pages = {124-132.e2},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.jaac.2016.11.006},
   Abstract = {Recent changes in DSM criteria require new documentation of
             the prevalence and developmental sequences of cannabis use
             disorder (CUD). The goal of this study was to investigate
             the early course of DSM-5 CUD and its overlap with DSM-IV
             and consumption constructs in a community-representative
             sample of American Indians.Data came from the prospective,
             longitudinal, population-based Great Smoky Mountains Study
             in North Carolina (N = 1,420, including 349 American
             Indians). Cannabis use and disorder were assessed during
             yearly interviews from 9 to 16 years of age and at 19, 21,
             26, and 30 years of age (up to 11 assessments per
             participant from 1993 through 2015).By 30 years of age,
             approximately 70% of participants had used cannabis, 34% had
             used cannabis daily, and 18% had met criteria for DSM-5 CUD.
             Approximately 1 in 4 cannabis users met criteria for CUD at
             some point. Those who met criteria initiated use more than 2
             years previously (at 13.3 years old) compared with other
             users. Despite higher risks from increased poverty, American
             Indians' patterns of use were similar to those of the rest
             of the sample. Concordance between DSM-5 CUD and DSM-IV
             abuse or dependence was substantial but was even higher
             between DSM-5 CUD and daily use.It was common to have used
             cannabis daily or to have met criteria for DSM-5 CUD by
             adulthood. DSM-5 CUD was an improvement over DSM-IV
             diagnostic constructs by raising the threshold for
             diagnosis.},
   Doi = {10.1016/j.jaac.2016.11.006},
   Key = {fds323141}
}


%% Curry, John F.   
@article{fds328869,
   Author = {Meyer, AE and Curry, JF},
   Title = {Pathways from anxiety to stressful events: An expansion of
             the stress generation hypothesis.},
   Journal = {Clinical Psychology Review},
   Volume = {57},
   Pages = {93-116},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.cpr.2017.08.003},
   Doi = {10.1016/j.cpr.2017.08.003},
   Key = {fds328869}
}

@article{fds327307,
   Author = {Shepherd-Banigan, M and Kelley, ML and Katon, JG and Curry, JF and Goldstein, KM and Brancu, M and Wagner, HR and Fecteau, TE and VA
             Mid-Atlantic MIRECC Women Veterans Workgroup, and VA
             Mid-Atlantic MIRECC Workgroup, and Van Houtven,
             CH},
   Title = {Paternal history of mental illness associated with
             posttraumatic stress disorder among veterans.},
   Journal = {Psychiatry Research},
   Volume = {256},
   Pages = {461-468},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.psychres.2017.06.053},
   Abstract = {This study examined the association between parent and
             family reported history of non-PTSD mental illness (MI),
             PTSD specifically, and substance use problems, and
             participant clinical diagnosis of PTSD. Participants were
             drawn from the US Department of Veterans Affairs
             Mid-Atlantic Mental Illness Research, Education and Clinical
             Center (MIRECC) Post-Deployment Mental Health (PDMH) study
             (n = 3191), an ongoing multi-site cohort study of US
             Afghanistan and Iraq conflict era veterans. Participants who
             recalled a father history of PTSD had a 26-percentage point
             higher likelihood of meeting criteria for PTSD; while
             participants reporting any family history of PTSD had a
             15-percentage point higher probability of endorsing symptoms
             consistent with PTSD. Mother history of substance use
             problems was associated with Veteran current PTSD, but
             results were sensitive to model specification. Current PTSD
             was not associated with family/parent history of non-PTSD
             mental illness, mother history of PTSD, or family/father
             history of substance use problems. Family history of PTSD
             may increase PTSD risk among veterans exposed to trauma,
             particularly when a father history is reported. Knowledge of
             family history could improve clinical decision-making for
             trauma-exposed individuals and allow for more effective
             targeting of programs and clinical services.},
   Doi = {10.1016/j.psychres.2017.06.053},
   Key = {fds327307}
}


%% Dawson, Geraldine   
@article{fds333739,
   Author = {Campbell, K and Carpenter, KL and Hashemi, J and Espinosa, S and Marsan,
             S and Borg, JS and Chang, Z and Qiu, Q and Vermeer, S and Adler, E and Tepper,
             M and Egger, HL and Baker, JP and Sapiro, G and Dawson,
             G},
   Title = {Computer vision analysis captures atypical attention in
             toddlers with autism.},
   Journal = {Autism},
   Pages = {1362361318766247},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/1362361318766247},
   Abstract = {To demonstrate the capability of computer vision analysis to
             detect atypical orienting and attention behaviors in
             toddlers with autism spectrum disorder. One hundered and
             four toddlers of 16-31 months old (mean = 22)
             participated in this study. Twenty-two of the toddlers had
             autism spectrum disorder and 82 had typical development or
             developmental delay. Toddlers watched video stimuli on a
             tablet while the built-in camera recorded their head
             movement. Computer vision analysis measured participants'
             attention and orienting in response to name calls.
             Reliability of the computer vision analysis algorithm was
             tested against a human rater. Differences in behavior were
             analyzed between the autism spectrum disorder group and the
             comparison group. Reliability between computer vision
             analysis and human coding for orienting to name was
             excellent (intra-class coefficient 0.84, 95% confidence
             interval 0.67-0.91). Only 8% of toddlers with autism
             spectrum disorder oriented to name calling on >1 trial,
             compared to 63% of toddlers in the comparison group
             (p = 0.002). Mean latency to orient was significantly
             longer for toddlers with autism spectrum disorder (2.02 vs
             1.06 s, p = 0.04). Sensitivity for autism spectrum
             disorder of atypical orienting was 96% and specificity was
             38%. Older toddlers with autism spectrum disorder showed
             less attention to the videos overall (p = 0.03).
             Automated coding offers a reliable, quantitative method for
             detecting atypical social orienting and reduced sustained
             attention in toddlers with autism spectrum
             disorder.},
   Doi = {10.1177/1362361318766247},
   Key = {fds333739}
}

@article{fds331234,
   Author = {Murias, M and Major, S and Davlantis, K and Franz, L and Harris, A and Rardin, B and Sabatos-DeVito, M and Dawson, G},
   Title = {Validation of eye-tracking measures of social attention as a
             potential biomarker for autism clinical trials.},
   Journal = {Autism Research},
   Volume = {11},
   Number = {1},
   Pages = {166-174},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1002/aur.1894},
   Abstract = {Social communication impairments are a core feature of
             autism spectrum disorder (ASD), and this class of symptoms
             is a target for treatments for the disorder. Measures of
             social attention, assessed via eye-gaze tracking (EGT), have
             been proposed as an early efficacy biomarker for clinical
             trials targeting social communication skills. EGT measures
             have been shown to differentiate children with ASD from
             typical children; however, there is less known about their
             relationships with social communication outcome measures
             that are typically used in ASD clinical trials. In the
             present study, an EGT task involving viewing a videotape of
             an actor making bids for a child's attention was evaluated
             in 25 children with ASD aged 24-72 months. Children's
             attention to the actor during the dyadic bid condition
             measured via EGT was found to be strongly associated with
             five well-validated caregiver-reported outcome measures that
             are commonly used to assess social communication in clinical
             trials. These results highlight the convergent validity of
             EGT measures of social attention in relation to
             caregiver-reported clinical measures. EGT holds promise as a
             non-invasive, quantitative, and objective biomarker that is
             associated with social communication abilities in children
             with ASD. Autism Res 2018, 11: 166-174. © 2017
             International Society for Autism Research, Wiley
             Periodicals, Inc.Eye-gaze tracking (EGT), an automated tool
             that tracks eye-gaze patterns, might help measure outcomes
             in clinical trials investigating interventions to treat
             autism spectrum disorders. In this study, an EGT task was
             evaluated in children with ASD, who watched a video with an
             actor talking directly to them. Patterns of eye-gaze were
             associated with caregiver-reported measures of social
             communication that are used in clinical trials. We show EGT
             may be a promising objective tool measuring
             outcomes.},
   Doi = {10.1002/aur.1894},
   Key = {fds331234}
}

@article{fds326271,
   Author = {Jones, EJH and Dawson, G and Webb, SJ},
   Title = {Sensory hypersensitivity predicts enhanced attention capture
             by faces in the early development of ASD.},
   Journal = {Developmental Cognitive Neuroscience},
   Volume = {29},
   Pages = {11-20},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.dcn.2017.04.001},
   Abstract = {Sensory sensitivity is prevalent among young children with
             ASD, but its relation to social communication impairment is
             unclear. Recently, increased sensory hypersensitivity has
             been linked to greater activity of the neural salience
             network (Green et al., 2016). Increased neural sensitivity
             to stimuli, especially social stimuli, could provide greater
             opportunity for social learning and improved outcomes.
             Consistent with this framework, in Experiment 1 we found
             that parent report of greater sensory hypersensitivity at 2
             years in toddlers with ASD (N=27) was predictive of
             increased neural responsiveness to social stimuli (larger
             amplitude event-related potential/ERP responses to faces at
             P1, P400 and Nc) at 4 years, and this in turn was related to
             parent report of increased social approach at 4 years. In
             Experiment 2, parent report of increased perceptual
             sensitivity at 6 months in infants at low and high familial
             risk for ASD (N=35) predicted larger ERP P1 amplitude to
             faces at 18 months. Increased sensory hypersensitivity in
             early development thus predicted greater attention capture
             by faces in later development, and this related to more
             optimal social behavioral development. Sensory
             hypersensitivity may index a child's ability to benefit from
             supportive environments during development. Early sensory
             symptoms may not always be developmentally problematic for
             individuals with ASD.},
   Doi = {10.1016/j.dcn.2017.04.001},
   Key = {fds326271}
}

@article{fds329121,
   Author = {St John and T and Dawson, G and Estes, A},
   Title = {Brief Report: Executive Function as a Predictor of Academic
             Achievement in School-Aged Children with
             ASD.},
   Journal = {Journal of Autism and Developmental Disorders},
   Volume = {48},
   Number = {1},
   Pages = {276-283},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s10803-017-3296-9},
   Abstract = {The contributions of Executive Function (EF) to academic
             achievement in children with Autism Spectrum Disorder (ASD)
             are not well understood. Academic achievement and its
             association with EF is described in 32, 9-year-old children
             with ASD. EF at age 6 and 9, and academic achievement at age
             9 were assessed as part of a larger longitudinal study.
             Better performance on a Spatial Reversal task but not
             A-not-B with Invisible Displacement at age 6 was associated
             with better math achievement at age 9. No relationship was
             found between these EF measures at age 6 and reading or
             spelling achievement at age 9. Future studies are needed to
             explore whether improving early EF skills can increase math
             achievement in children with ASD.},
   Doi = {10.1007/s10803-017-3296-9},
   Key = {fds329121}
}

@article{fds327659,
   Title = {Meta-analysis of GWAS of over 16,000 individuals with autism
             spectrum disorder highlights a novel locus at 10q24.32 and a
             significant overlap with schizophrenia},
   Journal = {Molecular Autism},
   Volume = {8},
   Number = {1},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1186/s13229-017-0137-9},
   Doi = {10.1186/s13229-017-0137-9},
   Key = {fds327659}
}

@article{fds326793,
   Author = {Bangerter, A and Ness, S and Aman, MG and Esbensen, AJ and Goodwin, MS and Dawson, G and Hendren, R and Leventhal, B and Khan, A and Opler, M and Harris, A and Pandina, G},
   Title = {Autism Behavior Inventory: A Novel Tool for Assessing Core
             and Associated Symptoms of Autism Spectrum
             Disorder.},
   Journal = {Journal of Child and Adolescent Psychopharmacology},
   Volume = {27},
   Number = {9},
   Pages = {814-822},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1089/cap.2017.0018},
   Abstract = {Autism Behavior Inventory (ABI) is a new measure for
             assessing changes in core and associated symptoms of autism
             spectrum disorder (ASD) in participants (ages: 3
             years-adulthood) diagnosed with ASD. It is a web-based tool
             with five domains (two ASD core domains: social
             communication, restrictive and repetitive behaviors; three
             associated domains: mental health, self-regulation, and
             challenging behavior). This study describes design,
             development, and initial psychometric properties of the
             ABI.ABI items were generated following review of existing
             measures and inputs from expert clinicians. Initial ABI
             scale contained 161 items that were reduced to fit a factor
             analytic model, retaining items of adequate reliability. Two
             versions of the scale, ABI-full (ABI-F; 93 items) and
             ABI-short version (ABI-S; 36 items), were developed and
             evaluated for psychometric properties, including validity
             comparisons with commonly used measures. Both scales were
             administered to parents and healthcare professionals (HCPs)
             involved with study participants.Test-retest reliability
             (intraclass correlation coefficient [ICC] = 0.79) for
             parent ratings on ABI was robust and compared favorably to
             existing scales. Test-retest correlations for HCP ratings
             were generally lower versus parent ratings. ABI core domains
             and comparison measures strongly correlated
             (r ≥ 0.70), demonstrating good concurrent
             validity.Overall, ABI demonstrates promise as a tool for
             measuring change in core symptoms of autism in ASD clinical
             studies, with further validation required.},
   Doi = {10.1089/cap.2017.0018},
   Key = {fds326793}
}

@misc{fds332364,
   Author = {Carpenter, K and Major, S and Tallman, C and Chen, L and Franz, L and Sun,
             J and Kurtzberg, J and Song, A and Dawson, G},
   Title = {Phase I Open Label Trial of Autologous Cord Blood for
             Treatment of Autism: Correlations Between Behavioral
             Improvement and Increased White Matter Connectivity in
             Children With Autism},
   Journal = {Neuropsychopharmacology (Nature)},
   Volume = {43},
   Pages = {S495-S496},
   Year = {2017},
   Month = {November},
   Key = {fds332364}
}

@misc{fds331661,
   Author = {Pandina, G and Manyakov, NV and Bangerter, A and Lewin, D and Jagannatha, S and Boice, M and Skalkin, A and Dawson, G and Goodwin, MS and Hendren, R and Leventhal, B and Shic, F and Ness,
             S},
   Title = {Prospective, observational cohort study of JAKE (TM), an
             autism knowledge},
   Journal = {European Neuropsychopharmacology},
   Volume = {27},
   Pages = {S1118-S1118},
   Year = {2017},
   Month = {October},
   Key = {fds331661}
}

@article{fds329122,
   Author = {Cidav, Z and Munson, J and Estes, A and Dawson, G and Rogers, S and Mandell, D},
   Title = {Cost Offset Associated With Early Start Denver Model for
             Children With Autism.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {9},
   Pages = {777-783},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.06.007},
   Abstract = {To determine the effect of the Early Start Denver Model
             (ESDM) for treatment of young children with autism on health
             care service use and costs.We used data from a randomized
             trial that tested the efficacy of the ESDM, which is based
             on developmental and applied behavioral analytic principles
             and delivered by trained therapists and parents, for 2
             years. Parents were interviewed about their children's
             service use every 6 months from the onset of the
             intervention to follow-up (age 6 years). The sample for this
             study consisted of 39 children with autism who participated
             in the original randomized trial at age 18 to 30 months, and
             were also assessed at age 6 years. Of this sample, 21
             children were in the ESDM group, and 18 children were in the
             community care (COM) group. Reported services were
             categorized and costed by applying unit hourly costs.
             Annualized service use and costs during the intervention and
             post intervention for the two study arms were
             compared.During the intervention, children who received the
             ESDM had average annualized total health-related costs that
             were higher by about $14,000 than those of children who
             received community-based treatment. The higher cost of ESDM
             was partially offset during the intervention period because
             children in the ESDM group used less applied behavior
             analysis (ABA)/early intensive behavioral intervention
             (EIBI) and speech therapy services than children in the
             comparison group. In the postintervention period, compared
             with children who had earlier received treatment as usual in
             community settings, children in the ESDM group used less
             ABA/EIBI, occupational/physical therapy, and speech therapy
             services, resulting in significant cost savings in the
             amount of about $19,000 per year per child.Costs associated
             with ESDM treatment were fully offset within a few years
             after the intervention because of reductions in other
             service use and associated costs.Early Characteristics of
             Autism; http://clinicaltrials.gov/; NCT0009415.},
   Doi = {10.1016/j.jaac.2017.06.007},
   Key = {fds329122}
}

@article{fds327660,
   Author = {Weiner, DJ and Wigdor, EM and Ripke, S and Walters, RK and Kosmicki, JA and Grove, J and Samocha, KE and Goldstein, JI and Okbay, A and Bybjerg-Grauholm, J and Werge, T and Hougaard, DM and Taylor, J and iPSYCH-Broad Autism Group, and Psychiatric Genomics Consortium
             Autism Group, and Skuse, D and Devlin, B and Anney, R and Sanders, SJ and Bishop, S and Mortensen, PB and Børglum, AD and Smith, GD and Daly, MJ and Robinson, EB},
   Title = {Polygenic transmission disequilibrium confirms that common
             and rare variation act additively to create risk for autism
             spectrum disorders.},
   Journal = {Nature Genetics},
   Volume = {49},
   Number = {7},
   Pages = {978-985},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1038/ng.3863},
   Abstract = {Autism spectrum disorder (ASD) risk is influenced by common
             polygenic and de novo variation. We aimed to clarify the
             influence of polygenic risk for ASD and to identify
             subgroups of ASD cases, including those with strongly acting
             de novo variants, in which polygenic risk is relevant. Using
             a novel approach called the polygenic transmission
             disequilibrium test and data from 6,454 families with a
             child with ASD, we show that polygenic risk for ASD,
             schizophrenia, and greater educational attainment is
             over-transmitted to children with ASD. These findings hold
             independent of proband IQ. We find that polygenic variation
             contributes additively to risk in ASD cases who carry a
             strongly acting de novo variant. Lastly, we show that
             elements of polygenic risk are independent and differ in
             their relationship with phenotype. These results confirm
             that the genetic influences on ASD are additive and suggest
             that they create risk through at least partially distinct
             etiologic pathways.},
   Doi = {10.1038/ng.3863},
   Key = {fds327660}
}

@article{fds326792,
   Author = {Dawson, G},
   Title = {Questions Remain Regarding the Effectiveness of Many
             Commonly Used Autism Treatments.},
   Journal = {Pediatrics},
   Volume = {139},
   Number = {6},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1542/peds.2017-0730},
   Doi = {10.1542/peds.2017-0730},
   Key = {fds326792}
}

@article{fds325854,
   Author = {Jones, EJH and Dawson, G and Kelly, J and Estes, A and Jane Webb,
             S},
   Title = {Parent-delivered early intervention in infants at risk for
             ASD: Effects on electrophysiological and habituation
             measures of social attention.},
   Journal = {Autism Research},
   Volume = {10},
   Number = {5},
   Pages = {961-972},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1002/aur.1754},
   Abstract = {Prospective longitudinal studies of infants with older
             siblings with autism spectrum disorder (ASD) have indicated
             that differences in the neurocognitive systems underlying
             social attention may emerge prior to the child meeting ASD
             diagnostic criteria. Thus, targeting social attention with
             early intervention might have the potential to alter
             developmental trajectories for infants at high risk for ASD.
             Electrophysiological and habituation measures of social
             attention were collected at 6, 12, and 18 months in a group
             of high-risk infant siblings of children with ASD
             (N = 33). Between 9 and 11 months of age, infant
             siblings received a parent-delivered intervention, promoting
             first relationships (PFR), (n = 19) or on-going
             assessment without intervention (n = 14). PFR has been
             previously shown to increase parental responsivity to infant
             social communicative cues and infant contingent responding.
             Compared to infants who only received assessment and
             monitoring, infants who received the intervention showed
             improvements in neurocognitive metrics of social attention,
             as reflected in a greater reduction in habituation times to
             face versus object stimuli between 6 and 12 months,
             maintained at 18 months; a greater increase in frontal EEG
             theta power between 6 and 12 months; and a more comparable
             P400 response to faces and objects at 12 months. The
             high-risk infants who received the intervention showed a
             pattern of responses that appeared closer to the normative
             responses of two groups of age-matched low-risk control
             participants. Though replication is necessary, these results
             suggest that early parent-mediated intervention has the
             potential to impact the brain systems underpinning social
             attention in infants at familial risk for ASD. Autism Res
             2017, 10: 961-972. © 2017 International Society for Autism
             Research, Wiley Periodicals, Inc.},
   Doi = {10.1002/aur.1754},
   Key = {fds325854}
}

@article{fds326349,
   Author = {Dawson, G and Sun, JM and Davlantis, KS and Murias, M and Franz, L and Troy, J and Simmons, R and Sabatos-DeVito, M and Durham, R and Kurtzberg, J},
   Title = {Autologous Cord Blood Infusions Are Safe and Feasible in
             Young Children with Autism Spectrum Disorder: Results of a
             Single-Center Phase I Open-Label Trial.},
   Journal = {Stem cells translational medicine},
   Volume = {6},
   Number = {5},
   Pages = {1332-1339},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1002/sctm.16-0474},
   Abstract = {Despite advances in early diagnosis and behavioral
             therapies, more effective treatments for children with
             autism spectrum disorder (ASD) are needed. We hypothesized
             that umbilical cord blood-derived cell therapies may have
             potential in alleviating ASD symptoms by modulating
             inflammatory processes in the brain. Accordingly, we
             conducted a phase I, open-label trial to assess the safety
             and feasibility of a single intravenous infusion of
             autologous umbilical cord blood, as well as sensitivity to
             change in several ASD assessment tools, to determine
             suitable endpoints for future trials. Twenty-five children,
             median age 4.6 years (range 2.26-5.97), with a confirmed
             diagnosis of ASD and a qualified banked autologous umbilical
             cord blood unit, were enrolled. Children were evaluated with
             a battery of behavioral and functional tests immediately
             prior to cord blood infusion (baseline) and 6 and 12 months
             later. Assessment of adverse events across the 12-month
             period indicated that the treatment was safe and well
             tolerated. Significant improvements in children's behavior
             were observed on parent-report measures of social
             communication skills and autism symptoms, clinician ratings
             of overall autism symptom severity and degree of
             improvement, standardized measures of expressive vocabulary,
             and objective eye-tracking measures of children's attention
             to social stimuli, indicating that these measures may be
             useful endpoints in future studies. Behavioral improvements
             were observed during the first 6 months after infusion and
             were greater in children with higher baseline nonverbal
             intelligence quotients. These data will serve as the basis
             for future studies to determine the efficacy of umbilical
             cord blood infusions in children with ASD. Stem Cells
             Translational Medicine 2017;6:1332-1339.},
   Doi = {10.1002/sctm.16-0474},
   Key = {fds326349}
}

@article{fds324049,
   Author = {Campbell, K and Carpenter, KLH and Espinosa, S and Hashemi, J and Qiu,
             Q and Tepper, M and Calderbank, R and Sapiro, G and Egger, HL and Baker,
             JP and Dawson, G},
   Title = {Use of a Digital Modified Checklist for Autism in Toddlers -
             Revised with Follow-up to Improve Quality of Screening for
             Autism.},
   Journal = {The Journal of Pediatrics},
   Volume = {183},
   Pages = {133-139.e1},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jpeds.2017.01.021},
   Abstract = {To assess changes in quality of care for children at risk
             for autism spectrum disorders (ASD) due to process
             improvement and implementation of a digital screening
             form.The process of screening for ASD was studied in an
             academic primary care pediatrics clinic before and after
             implementation of a digital version of the Modified
             Checklist for Autism in Toddlers - Revised with Follow-up
             with automated risk assessment. Quality metrics included
             accuracy of documentation of screening results and
             appropriate action for positive screens (secondary screening
             or referral). Participating physicians completed pre- and
             postintervention surveys to measure changes in attitudes
             toward feasibility and value of screening for ASD. Evidence
             of change was evaluated with statistical process control
             charts and χ2 tests.Accurate documentation in the
             electronic health record of screening results increased from
             54% to 92% (38% increase, 95% CI 14%-64%) and appropriate
             action for children screening positive increased from 25% to
             85% (60% increase, 95% CI 35%-85%). A total of 90% of
             participating physicians agreed that the transition to a
             digital screening form improved their clinical assessment of
             autism risk.Implementation of a tablet-based digital version
             of the Modified Checklist for Autism in Toddlers - Revised
             with Follow-up led to improved quality of care for children
             at risk for ASD and increased acceptability of screening for
             ASD. Continued efforts towards improving the process of
             screening for ASD could facilitate rapid, early diagnosis of
             ASD and advance the accuracy of studies of the impact of
             screening.},
   Doi = {10.1016/j.jpeds.2017.01.021},
   Key = {fds324049}
}

@article{fds329542,
   Author = {Ness, SL and Manyakov, NV and Bangerter, A and Lewin, D and Jagannatha,
             S and Boice, M and Skalkin, A and Dawson, G and Janvier, YM and Goodwin,
             MS and Hendren, R and Leventhal, B and Shic, F and Cioccia, W and Pandina,
             G},
   Title = {JAKE® Multimodal Data Capture System: Insights from an
             Observational Study of Autism Spectrum Disorder.},
   Journal = {Frontiers in Neuroscience},
   Volume = {11},
   Pages = {517},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fnins.2017.00517},
   Abstract = {Objective: To test usability and optimize the Janssen Autism
             Knowledge Engine (JAKE®) system's components, biosensors,
             and procedures used for objective measurement of core and
             associated symptoms of autism spectrum disorder (ASD) in
             clinical trials. Methods: A prospective, observational study
             of 29 children and adolescents with ASD using the JAKE
             system was conducted at three sites in the United States.
             This study was designed to establish the feasibility of the
             JAKE system and to learn practical aspects of its
             implementation. In addition to information collected by web
             and mobile components, wearable biosensor data were
             collected both continuously in natural settings and
             periodically during a battery of experimental tasks
             administered in laboratory settings. This study is
             registered at clinicaltrials.gov, NCT02299700. Results:
             Feedback collected throughout the study allowed future
             refinements to be planned for all components of the system.
             The Autism Behavior Inventory (ABI), a parent-reported
             measure of ASD core and associated symptoms, performed well.
             Among biosensors studied, the eye-tracker, sleep monitor,
             and electrocardiogram were shown to capture high quality
             data, whereas wireless electroencephalography was difficult
             to use due to its form factor. On an exit survey, the
             majority of parents rated their overall reaction to JAKE as
             positive/very positive. No significant device-related events
             were reported in the study. Conclusion: The results of this
             study, with the described changes, demonstrate that the JAKE
             system is a viable, useful, and safe platform for use in
             clinical trials of ASD, justifying larger validation and
             deployment studies of the optimized system.},
   Doi = {10.3389/fnins.2017.00517},
   Key = {fds329542}
}

@article{fds333740,
   Author = {Li, Y and Murias, M and Major, S and Dawson, G and Dzirasa, K and Carin, L and Carlson, DE},
   Title = {Targeting EEG/LFP Synchrony with Neural Nets.},
   Journal = {NIPS},
   Pages = {4623-4633},
   Editor = {Guyon, I and Luxburg, UV and Bengio, S and Wallach, HM and Fergus, R and Vishwanathan, SVN and Garnett, R},
   Year = {2017},
   Key = {fds333740}
}


%% De Bellis, Michael D.   
@article{fds333815,
   Author = {Pfefferbaum, A and Kwon, D and Brumback, T and Thompson, WK and Cummins,
             K and Tapert, SF and Brown, SA and Colrain, IM and Baker, FC and Prouty, D and De Bellis, MD and Clark, DB and Nagel, BJ and Chu, W and Park, SH and Pohl,
             KM and Sullivan, EV},
   Title = {Altered Brain Developmental Trajectories in Adolescents
             After Initiating Drinking.},
   Journal = {American Journal of Psychiatry},
   Volume = {175},
   Number = {4},
   Pages = {370-380},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.17040469},
   Abstract = {The authors sought evidence for altered adolescent brain
             growth trajectory associated with moderate and heavy alcohol
             use in a large national, multisite, prospective study of
             adolescents before and after initiation of appreciable
             alcohol use.This study examined 483 adolescents (ages 12-21)
             before initiation of drinking and 1 and 2 years later. At
             the 2-year assessment, 356 participants continued to meet
             the study's no/low alcohol consumption entry criteria, 65
             had initiated moderate drinking, and 62 had initiated heavy
             drinking. MRI was used to quantify regional cortical and
             white matter volumes. Percent change per year (slopes) in
             adolescents who continued to meet no/low criteria served as
             developmental control trajectories against which to compare
             those who initiated moderate or heavy drinking.In no/low
             drinkers, gray matter volume declined throughout adolescence
             and slowed in many regions in later adolescence.
             Complementing gray matter declines, white matter regions
             grew at faster rates at younger ages and slowed toward young
             adulthood. Youths who initiated heavy drinking exhibited an
             accelerated frontal cortical gray matter trajectory,
             divergent from the norm. Although significant effects on
             trajectories were not observed in moderate drinkers, their
             intermediate position between no/low and heavy drinkers
             suggests a dose effect. Neither marijuana co-use nor
             baseline volumes contributed significantly to the alcohol
             effect.Initiation of drinking during adolescence, with or
             without marijuana co-use, disordered normal brain growth
             trajectories. Factors possibly contributing to abnormal
             cortical volume trajectories include peak consumption in the
             past year and family history of alcoholism.},
   Doi = {10.1176/appi.ajp.2017.17040469},
   Key = {fds333815}
}

@article{fds331438,
   Author = {Nooner, KB and Hooper, SR and De Bellis, MD},
   Title = {An Examination of Sex Differences on Neurocognitive
             Functioning and Behavior Problems in Maltreated
             Youth.},
   Journal = {Psychological Trauma: Theory, Research, Practice, and
             Policy},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1037/tra0000356},
   Abstract = {In the developmental traumatology model, the biological
             construct of sex is considered a moderator that may
             negatively influence child maltreatment sequelae including
             those pertaining to neurocognitive function.This study
             examined sex-differences in neurocognitive function and
             behavior problems in maltreated boys (n = 42), maltreated
             girls (n = 56) versus nonmaltreated boys (n = 45) and girls
             (n = 59). Maltreated boys were hypothesized to have poorer
             neurocognitive functioning than maltreated girls, and
             nonmaltreated boys and girls, in all neurocognitive domains,
             particularly pertaining to executive function and attention.
             We also examined correlations between cognitive function and
             parent report of child behavior problems for maltreated and
             nonmaltreated children.Maltreated boys performed more poorly
             on measures of intelligence, attention, language, memory,
             executive function, and academic achievement in both reading
             and math than nonmaltreated boys. Maltreated boys did not
             perform more poorly on these cognitive measures or
             behavioral measures than maltreated girls, except for one
             memory measure. Maltreated girls performed more poorly on
             measures of intelligence, language, memory, executive
             function, and academic achievement than nonmaltreated girls.
             Maltreated girls with better visual-spatial skills had more
             internalizing and externalizing problems. Effect sizes for
             these sex differences ranged from small to large.Both
             maltreated boys and girls showed poorer cognitive function
             than their nonmaltreated sex-matched controls. Maltreated
             girls had subtle sparing of attention and short-term memory
             (STM). Understanding sex differences in neurocognitive
             functioning may have implications for designing large
             population studies of maltreated youth. (PsycINFO Database
             Record},
   Doi = {10.1037/tra0000356},
   Key = {fds331438}
}

@misc{fds332915,
   Author = {Tervo-Clemmens, B and Quach, A and Luna, B and Foran, W and Chung, T and De
             Bellis, MD and Clark, DB},
   Title = {Neural correlates of rewarded response inhibition in youth
             at risk for problematic alcohol use},
   Journal = {Frontiers in Behavioral Neuroscience},
   Volume = {11},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.3389/fnbeh.2017.00205},
   Abstract = {© 2017 Tervo-Clemmens, Quach, Luna, Foran, Chung, De Bellis
             and Clark. Risk for substance use disorder (SUD) is
             associated with poor response inhibition and heightened
             reward sensitivity. During adolescence, incentives improve
             performance on response inhibition tasks and increase
             recruitment of cortical control areas (Geier et al., 2010)
             associated with SUD (Chung et al., 2011). However, it is
             unknown whether incentives moderate the relationship between
             response inhibition and trait-level psychopathology and
             personality features of substance use risk. We examined
             these associations in the current project using a rewarded
             antisaccade (AS) task (Geier et al., 2010) in youth at risk
             for substance use. Participants were 116 adolescents and
             young adults (ages 12–21) from the University of
             Pittsburgh site of the National Consortium on Adolescent
             Neurodevelopment and Alcohol [NCANDA] study, with
             neuroimaging data collected at baseline and 1 year follow up
             visits. Building upon previous work using this task in
             normative developmental samples (Geier et al., 2010) and
             adolescents with SUD (Chung et al., 2011), we examined both
             trial-wise BOLD responses and those associated with
             individual task-epochs (cue presentation, response
             preparation, and response) and associated them with multiple
             substance use risk factors (externalizing and internalizing
             psychopathology, family history of substance use, and trait
             impulsivity). Results showed that externalizing
             psychopathology and high levels of trait impulsivity
             (positive urgency, SUPPS-P) were associated with general
             decreases in antisaccade performance. Accompanying this main
             effect of poor performance, positive urgency was associated
             with reduced recruitment of the frontal eye fields (FEF) and
             inferior frontal gyrus (IFG) in both a priori regions of
             interest and at the voxelwise level. Consistent with
             previous work, monetary incentive improved antisaccade
             behavioral performance and was associated with increased
             activation in the striatum and cortical control areas.
             However, incentives did not moderate the association between
             response inhibition behavioral performance and any
             trait-level psychopathology and personality factor of
             substance use risk. Reward interactions were observed for
             BOLD responses at the task-epoch level, however, they were
             inconsistent across substance use risk types. The results
             from this study may suggest poor response inhibition and
             heightened reward sensitivity are not overlapping
             neurocognitive features of substance use risk.
             Alternatively, more subtle, common longitudinal processes
             might jointly explain reward sensitivity and response
             inhibition deficits in substance use risk.},
   Doi = {10.3389/fnbeh.2017.00205},
   Key = {fds332915}
}

@article{fds330475,
   Author = {Sullivan, EV and Lane, B and Kwon, D and Meloy, MJ and Tapert, SF and Brown, SA and Colrain, IM and Baker, FC and De Bellis, MD and Clark, DB and Nagel, BJ and Pohl, KM and Pfefferbaum, A},
   Title = {Structural brain anomalies in healthy adolescents in the
             NCANDA cohort: relation to neuropsychological test
             performance, sex, and ethnicity.},
   Journal = {Brain Imaging and Behavior},
   Volume = {11},
   Number = {5},
   Pages = {1302-1315},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1007/s11682-016-9634-2},
   Abstract = {Structural MRI of volunteers deemed "normal" following
             clinical interview provides a window into normal brain
             developmental morphology but also reveals unexpected
             dysmorphology, commonly known as "incidental findings."
             Although unanticipated, these anatomical findings raise
             questions regarding possible treatment that could even
             ultimately require neurosurgical intervention, which itself
             carries significant risk but may not be indicated if the
             anomaly is nonprogressive or of no functional consequence.
             Neuroradiological readings of 833 structural MRI from the
             National Consortium on Alcohol and NeuroDevelopment in
             Adolescence (NCANDA) cohort found an 11.8 % incidence of
             brain structural anomalies, represented proportionately
             across the five collection sites and ethnic groups.
             Anomalies included 26 mega cisterna magna, 15 subarachnoid
             cysts, 12 pineal cysts, 12 white matter dysmorphologies, 5
             tonsillar ectopias, 5 prominent perivascular spaces, 5 gray
             matter heterotopias, 4 pituitary masses, 4 excessively large
             or asymmetrical ventricles, 4 cavum septum pellucidum, 3
             developmental venous anomalies, 1 exceptionally large
             midsagittal vein, and single cases requiring clinical
             followup: cranio-cervical junction stenosis, parietal
             cortical mass, and Chiari I malformation. A case of possible
             demyelinating disorder (e.g., neuromyelitis optica or
             multiple sclerosis) newly emerged at the 1-year NCANDA
             followup, requiring clinical referral. Comparing test
             performance of the 98 anomalous cases with 619 anomaly-free
             no-to-low alcohol consuming adolescents revealed
             significantly lower scores on speed measures of attention
             and motor functions; these differences were not attributed
             to any one anomaly subgroup. Further, we devised an
             automated approach for quantifying posterior fossa CSF
             volumes for detection of mega cisterna magna, which
             represented 26.5 % of clinically identified anomalies.
             Automated quantification fit a Gaussian distribution with a
             rightward skew. Using a 3SD cut-off, quantification
             identified 22 of the 26 clinically-identified cases,
             indicating that cases with percent of CSF in the
             posterior-inferior-middle aspect of the posterior fossa
             ≥3SD merit further review, and support complementing
             clinical readings with objective quantitative analysis.
             Discovery of asymptomatic brain structural anomalies, even
             when no clinical action is indicated, can be disconcerting
             to the individual and responsible family members, raising a
             disclosure dilemma: refrain from relating the incidental
             findings to avoid unnecessary alarm or anxiety; or
             alternatively, relate the neuroradiological findings as
             "normal variants" to the study volunteers and family,
             thereby equipping them with knowledge for the future should
             they have the occasion for a brain scan following an illness
             or accident that the incidental findings predated the later
             event.},
   Doi = {10.1007/s11682-016-9634-2},
   Key = {fds330475}
}

@article{fds326501,
   Author = {Hasler, BP and Franzen, PL and de Zambotti, M and Prouty, D and Brown,
             SA and Tapert, SF and Pfefferbaum, A and Pohl, KM and Sullivan, EV and De
             Bellis, MD and Nagel, BJ and Baker, FC and Colrain, IM and Clark,
             DB},
   Title = {Eveningness and Later Sleep Timing Are Associated with
             Greater Risk for Alcohol and Marijuana Use in Adolescence:
             Initial Findings from the National Consortium on Alcohol and
             Neurodevelopment in Adolescence Study.},
   Journal = {Alcoholism: Clinical and Experimental Research},
   Volume = {41},
   Number = {6},
   Pages = {1154-1165},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1111/acer.13401},
   Abstract = {Abundant cross-sectional evidence links eveningness (a
             preference for later sleep-wake timing) and increased
             alcohol and drug use among adolescents and young adults.
             However, longitudinal studies are needed to examine whether
             eveningness is a risk factor for subsequent alcohol and drug
             use, particularly during adolescence, which is marked by
             parallel peaks in eveningness and risk for the onset of
             alcohol use disorders. This study examined whether
             eveningness and other sleep characteristics were associated
             with concurrent or subsequent substance involvement in a
             longitudinal study of adolescents.Participants were 729
             adolescents (368 females; age 12 to 21 years) in the
             National Consortium on Alcohol and Neurodevelopment in
             Adolescence study. Associations between the sleep variables
             (circadian preference, sleep quality, daytime sleepiness,
             sleep timing, and sleep duration) and 3 categorical
             substance variables (at-risk alcohol use, alcohol bingeing,
             and past-year marijuana use [y/n]) were examined using
             ordinal and logistic regression with baseline age, sex,
             race, ethnicity, socioeconomic status, and psychiatric
             problems as covariates.At baseline, greater eveningness was
             associated with greater at-risk alcohol use, greater
             bingeing, and past-year use of marijuana. Later weekday and
             weekend bedtimes, but not weekday or weekend sleep duration,
             showed similar associations across the 3 substance outcomes
             at baseline. Greater baseline eveningness was also
             prospectively associated with greater bingeing and past-year
             use of marijuana at the 1-year follow-up, after covarying
             for baseline bingeing and marijuana use. Later baseline
             weekday and weekend bedtimes, and shorter baseline weekday
             sleep duration, were similarly associated with greater
             bingeing and past-year use of marijuana at the 1-year
             follow-up after covarying for baseline values.Findings
             suggest that eveningness and sleep timing may be under
             recognized risk factors and future areas of intervention for
             adolescent involvement in alcohol and marijuana that should
             be considered along with other previously identified sleep
             factors such as insomnia and insufficient
             sleep.},
   Doi = {10.1111/acer.13401},
   Key = {fds326501}
}

@article{fds325128,
   Author = {Sullivan, EV and Brumback, T and Tapert, SF and Prouty, D and Fama, R and Thompson, WK and Brown, SA and Cummins, K and Colrain, IM and Baker, FC and Clark, DB and Chung, T and De Bellis, MD and Hooper, SR and Nagel, BJ and Nichols, BN and Chu, W and Kwon, D and Pohl, KM and Pfefferbaum,
             A},
   Title = {Effects of prior testing lasting a full year in NCANDA
             adolescents: Contributions from age, sex, socioeconomic
             status, ethnicity, site, family history of alcohol or drug
             abuse, and baseline performance.},
   Journal = {Developmental Cognitive Neuroscience},
   Volume = {24},
   Pages = {72-83},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.dcn.2017.01.003},
   Abstract = {Longitudinal study provides a robust method for tracking
             developmental trajectories. Yet inherent problems of
             retesting pose challenges in distinguishing biological
             developmental change from prior testing experience. We
             examined factors potentially influencing change scores on 16
             neuropsychological test composites over 1year in 568
             adolescents in the National Consortium on Alcohol and
             NeuroDevelopment in Adolescence (NCANDA) project. The
             twice-minus-once-tested method revealed that performance
             gain was mainly attributable to testing experience
             (practice) with little contribution from predicted
             developmental effects. Group mean practice slopes for 13
             composites indicated that 60% to ∼100% variance was
             attributable to test experience; General Ability accuracy
             showed the least practice effect (29%). Lower baseline
             performance, especially in younger participants, was a
             strong predictor of greater gain. Contributions from age,
             sex, ethnicity, examination site, socioeconomic status, or
             family history of alcohol/substance abuse were nil to small,
             even where statistically significant. Recognizing that a
             substantial proportion of change in longitudinal testing,
             even over 1-year, is attributable to testing experience
             indicates caution against assuming that performance gain
             observed during periods of maturation necessarily reflects
             development. Estimates of testing experience, a form of
             learning, may be a relevant metric for detecting interim
             influences, such as alcohol use or traumatic episodes, on
             behavior.},
   Doi = {10.1016/j.dcn.2017.01.003},
   Key = {fds325128}
}

@article{fds330476,
   Author = {Stave, EA and De Bellis, MD and Hooper, SR and Woolley, DP and Chang,
             SK and Chen, SD},
   Title = {Dimensions of Attention Associated With the Microstructure
             of Corona Radiata White Matter.},
   Journal = {Journal of Child Neurology},
   Volume = {32},
   Number = {5},
   Pages = {458-466},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1177/0883073816685652},
   Abstract = {Mirsky proposed a model of attention that included these
             dimensions: focus/execute, sustain, stabilize, encode, and
             shift. The neural correlates of these dimensions were
             investigated within corona radiata subregions in healthy
             youth. Diffusion tensor imaging and neuropsychological
             assessments were conducted in 79 healthy, right-handed youth
             aged 4-17 years. Diffusion tensor imaging maps were analyzed
             using standardized parcellation methods. Partial Pearson
             correlations between neuropsychological standardized scores,
             representing these attention dimensions, and diffusion
             tensor imaging measures of corona radiata subregions were
             calculated after adjusting for gender and IQ. Significant
             correlations were found between the focus/execute, sustain,
             stabilize, and shift dimensions and imaging metrics in
             hypothesized corona radiata subregions. Results suggest that
             greater microstructural white matter integrity of the corona
             radiata is partly associated with attention across 4
             attention dimensions. Findings suggest that white matter
             microstructure of the corona radiata is a neural correlate
             of several, but not all, attention dimensions.},
   Doi = {10.1177/0883073816685652},
   Key = {fds330476}
}

@article{fds326856,
   Author = {Sege, RD and Amaya-Jackson, L and AMERICAN ACADEMY OF PEDIATRICS
             Committee on Child Abuse and Neglect, Council on Foster
             Care and Adoption and Kinship Care and AMERICAN ACADEMY OF CHILD
             AND ADOLESCENT PSYCHIATRY Committee on Child Maltreatment and Violence and NATIONAL CENTER FOR CHILD TRAUMATIC
             STRESS},
   Title = {Clinical Considerations Related to the Behavioral
             Manifestations of Child Maltreatment.},
   Journal = {Pediatrics},
   Volume = {139},
   Number = {4},
   Pages = {e20170100-e20170100},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1542/peds.2017-0100},
   Abstract = {Children who have suffered early abuse or neglect may later
             present with significant health and behavior problems that
             may persist long after the abusive or neglectful environment
             has been remediated. Neurobiological research suggests that
             early maltreatment may result in an altered psychological
             and physiologic response to stressful stimuli, a response
             that deleteriously affects the child's subsequent
             development. Pediatricians can assist caregivers by helping
             them recognize the abused or neglected child's emotional and
             behavioral responses associated with child maltreatment and
             guide them in the use of positive parenting strategies,
             referring the children and families to evidence-based
             therapeutic treatment and mobilizing available community
             resources.},
   Doi = {10.1542/peds.2017-0100},
   Key = {fds326856}
}

@article{fds332916,
   Author = {Clark, DB and Chung, T and Martin, CS and Hasler, BP and Fitzgerald, DH and Luna, B and Brown, SA and Tapert, SF and Brumback, T and Cummins, K and Pfefferbaum, A and Sullivan, EV and Pohl, KM and Colrain, IM and Baker,
             FC and De Bellis, MD and Nooner, KB and Nagel, BJ},
   Title = {Adolescent Executive Dysfunction in Daily Life:
             Relationships to Risks, Brain Structure and Substance
             Use.},
   Journal = {Frontiers in Behavioral Neuroscience},
   Volume = {11},
   Pages = {223},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fnbeh.2017.00223},
   Abstract = {During adolescence, problems reflecting cognitive,
             behavioral and affective dysregulation, such as inattention
             and emotional dyscontrol, have been observed to be
             associated with substance use disorder (SUD) risks and
             outcomes. Prior studies have typically been with small
             samples, and have typically not included comprehensive
             measurement of executive dysfunction domains. The
             relationships of executive dysfunction in daily life with
             performance based testing of cognitive skills and structural
             brain characteristics, thought to be the basis for executive
             functioning, have not been definitively determined. The aims
             of this study were to determine the relationships between
             executive dysfunction in daily life, measured by the
             Behavior Rating Inventory of Executive Function (BRIEF),
             cognitive skills and structural brain characteristics, and
             SUD risks, including a global SUD risk indicator, sleep
             quality, and risky alcohol and cannabis use. In addition to
             bivariate relationships, multivariate models were tested.
             The subjects (n = 817; ages 12 through 21) were participants
             in the National Consortium on Alcohol and Neurodevelopment
             in Adolescence (NCANDA) study. The results indicated that
             executive dysfunction was significantly related to SUD
             risks, poor sleep quality, risky alcohol use and cannabis
             use, and was not significantly related to cognitive skills
             or structural brain characteristics. In multivariate models,
             the relationship between poor sleep quality and risky
             substance use was mediated by executive dysfunction. While
             these cross-sectional relationships need to be further
             examined in longitudinal analyses, the results suggest that
             poor sleep quality and executive dysfunction may be viable
             preventive intervention targets to reduce adolescent
             substance use.},
   Doi = {10.3389/fnbeh.2017.00223},
   Key = {fds332916}
}


%% De Brigard, Felipe   
@article{fds332864,
   Author = {Murray, S and Murray, ED and Stewart, G and Sinnott-Armstrong, W and de
             Brigard, F},
   Title = {Responsibility for forgetting},
   Journal = {Philosophical Studies},
   Pages = {1-25},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1007/s11098-018-1053-3},
   Abstract = {© 2018 Springer Science+Business Media B.V., part of
             Springer Nature In this paper, we focus on whether and to
             what extent we judge that people are responsible for the
             consequences of their forgetfulness. We ran a series of
             behavioral studies to measure judgments of responsibility
             for the consequences of forgetfulness. Our results show that
             we are disposed to hold others responsible for some of their
             forgetfulness. The level of stress that the forgetful agent
             is under modulates judgments of responsibility, though the
             level of care that the agent exhibits toward performing the
             forgotten action does not. We argue that this result has
             important implications for a long-running debate about the
             nature of responsible agency.},
   Doi = {10.1007/s11098-018-1053-3},
   Key = {fds332864}
}

@article{fds329948,
   Author = {De Brigard and F and Parikh, N and Stewart, GW and Szpunar, KK and Schacter, DL},
   Title = {Neural activity associated with repetitive simulation of
             episodic counterfactual thoughts},
   Journal = {Neuropsychologia},
   Volume = {106},
   Pages = {123-132},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2017.09.022},
   Doi = {10.1016/j.neuropsychologia.2017.09.022},
   Key = {fds329948}
}

@article{fds329104,
   Author = {Stanley, ML and Dougherty, AM and Yang, BW and Henne, P and De Brigard,
             F},
   Title = {Reasons Probably Won't Change Your Mind: The Role of Reasons
             in Revising Moral Decisions},
   Journal = {Journal of Experimental Psychology: General},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1037/xge0000368},
   Abstract = {© 2017 APA, all rights reserved). Although many
             philosophers argue that making and revising moral decisions
             ought to be a matter of deliberating over reasons, the
             extent to which the consideration of reasons informs
             people's moral decisions and prompts them to change their
             decisions remains unclear. Here, after making an initial
             decision in 2-option moral dilemmas, participants examined
             reasons for only the option initially chosen (affirming
             reasons), reasons for only the option not initially chosen
             (opposing reasons), or reasons for both options. Although
             participants were more likely to change their initial
             decisions when presented with only opposing reasons compared
             with only affirming reasons, these effect sizes were
             consistently small. After evaluating reasons, participants
             were significantly more likely not to change their initial
             decisions than to change them, regardless of the set of
             reasons they considered. The initial decision accounted for
             most of the variance in predicting the final decision,
             whereas the reasons evaluated accounted for a relatively
             small proportion of the variance in predicting the final
             decision. This resistance to changing moral decisions is at
             least partly attributable to a biased, motivated evaluation
             of the available reasons: participants rated the reasons
             supporting their initial decisions more favorably than the
             reasons opposing their initial decisions, regardless of the
             reported strategy used to make the initial decision.
             Overall, our results suggest that the consideration of
             reasons rarely induces people to change their initial
             decisions in moral dilemmas. (PsycINFO Database
             Record},
   Doi = {10.1037/xge0000368},
   Key = {fds329104}
}

@article{fds329949,
   Author = {De Brigard and F},
   Title = {Memory and imagination},
   Pages = {127-140},
   Booktitle = {The Routledge Handbook of Philosophy of Memory},
   Year = {2017},
   Month = {June},
   ISBN = {9781315687315},
   url = {http://dx.doi.org/10.4324/9781315687315},
   Doi = {10.4324/9781315687315},
   Key = {fds329949}
}

@article{fds326194,
   Author = {Stanley, ML and Henne, P and Iyengar, V and Sinnott-Armstrong, W and De
             Brigard, F},
   Title = {I'm not the person I used to be: The self and
             autobiographical memories of immoral actions.},
   Journal = {Journal of Experimental Psychology: General},
   Volume = {146},
   Number = {6},
   Pages = {884-895},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1037/xge0000317},
   Abstract = {People maintain a positive identity in at least two ways:
             They evaluate themselves more favorably than other people,
             and they judge themselves to be better now than they were in
             the past. Both strategies rely on autobiographical memories.
             The authors investigate the role of autobiographical
             memories of lying and emotional harm in maintaining a
             positive identity. For memories of lying to or emotionally
             harming others, participants judge their own actions as less
             morally wrong and less negative than those in which other
             people lied to or emotionally harmed them. Furthermore,
             people judge those actions that happened further in the past
             to be more morally wrong than those that happened more
             recently. Finally, for periods of the past when they
             believed that they were very different people than they are
             now, participants judge their actions to be more morally
             wrong and more negative than those actions from periods of
             their pasts when they believed that they were very similar
             to who they are now. The authors discuss these findings in
             relation to theories about the function of autobiographical
             memory and moral cognition in constructing and perceiving
             the self over time. (PsycINFO Database Record},
   Doi = {10.1037/xge0000317},
   Key = {fds326194}
}

@article{fds327002,
   Author = {De Brigard and F},
   Title = {Cognitive systems and the changing brain},
   Journal = {Philosophical Explorations},
   Volume = {20},
   Number = {2},
   Pages = {224-241},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1080/13869795.2017.1312503},
   Doi = {10.1080/13869795.2017.1312503},
   Key = {fds327002}
}

@article{fds327003,
   Author = {De Brigard and F and Rodriguez, DC and Montañés,
             P},
   Title = {Exploring the experience of episodic past, future, and
             counterfactual thinking in younger and older adults: A study
             of a Colombian sample},
   Journal = {Consciousness and Cognition},
   Volume = {51},
   Pages = {258-267},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.concog.2017.04.007},
   Doi = {10.1016/j.concog.2017.04.007},
   Key = {fds327003}
}

@article{fds318357,
   Author = {Henne, P and Pinillos, Á and De Brigard and F},
   Title = {Cause by Omission and Norm: Not Watering
             Plants},
   Journal = {Australasian Journal of Philosophy},
   Volume = {95},
   Number = {2},
   Pages = {270-283},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1080/00048402.2016.1182567},
   Doi = {10.1080/00048402.2016.1182567},
   Key = {fds318357}
}

@article{fds323231,
   Author = {Stanley, ML and Parikh, N and Stewart, GW and De Brigard,
             F},
   Title = {Emotional intensity in episodic autobiographical memory and
             counterfactual thinking},
   Journal = {Consciousness and Cognition},
   Volume = {48},
   Pages = {283-291},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.concog.2016.12.013},
   Abstract = {© 2016Episodic counterfactual thoughts—imagined
             alternative ways in which personal past events might have
             occurred—are frequently accompanied by intense emotions.
             Here, participants recollected positive and negative
             autobiographical memories and then generated better and
             worse episodic counterfactual events from those memories.
             Our results suggest that the projected emotional intensity
             during the simulated remembered/imagined event is
             significantly higher than but typically positively related
             to the emotional intensity while remembering/imagining the
             event. Furthermore, repeatedly simulating counterfactual
             events heightened the emotional intensity felt while
             simulating the counterfactual event. Finally, for both the
             emotional intensity accompanying the experience of
             remembering/imagining and the projected emotional intensity
             during the simulated remembered/imagined event, the
             emotional intensity of negative memories was greater than
             the emotional intensity of upward counterfactuals generated
             from them but lower than the emotional intensity of downward
             counterfactuals generated from them. These findings are
             discussed in relation to clinical work and functional
             theories of counterfactual thinking.},
   Doi = {10.1016/j.concog.2016.12.013},
   Key = {fds323231}
}

@article{fds323662,
   Author = {De Brigard and F and Brady, TF and Ruzic, L and Schacter,
             DL},
   Title = {Tracking the emergence of memories: A category-learning
             paradigm to explore schema-driven recognition.},
   Journal = {Memory & Cognition},
   Volume = {45},
   Number = {1},
   Pages = {105-120},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.3758/s13421-016-0643-6},
   Abstract = {Previous research has shown that prior knowledge structures
             or schemas affect recognition memory. However, since the
             acquisition of schemas occurs over prolonged periods of
             time, few paradigms allow the direct manipulation of schema
             acquisition to study their effect on memory performance.
             Recently, a number of parallelisms in recognition memory
             between studies involving schemas and studies involving
             category learning have been identified. The current paper
             capitalizes on these findings and offers a novel
             experimental paradigm that allows manipulation of category
             learning between individuals to study the effects of schema
             acquisition on recognition. First, participants learn to
             categorize computer-generated items whose category-inclusion
             criteria differ between participants. Next, participants
             study items that belong to either the learned category, the
             non-learned category, both, or neither. Finally,
             participants receive a recognition test that includes old
             and new items, either from the learned, the non-learned, or
             neither category. Using variations on this paradigm, four
             experiments were conducted. The results from the first three
             studies suggest that learning a category increases hit rates
             for old category-consistent items and false alarm rates for
             new category-consistent lures. Absent the category learning,
             no such effects are evident, even when participants are
             exposed to the same learning trials as those who learned the
             categories. The results from the fourth experiment suggest
             that, at least for false alarm rates, the effects of
             category learning are not solely attributable to frequency
             of occurrence of category-consistent items during learning.
             Implications for recognition memory as well as advantages of
             the proposed paradigm are discussed.},
   Doi = {10.3758/s13421-016-0643-6},
   Key = {fds323662}
}

@article{fds331533,
   Author = {De Brigard and F},
   Title = {Responsibility and the relevance of alternative future
             possibilities},
   Journal = {Teoria},
   Volume = {37},
   Number = {2},
   Pages = {25-35},
   Year = {2017},
   Month = {January},
   Abstract = {In the past decade, philosophical and psychological research
             on people's beliefs about free will and responsibility has
             skyrocketed. For the most part, these vignette-based studies
             have exclusively focused on participants' judgments of the
             causal history of the events leading up to an agent's action
             and considerations about what the agent could have done
             differently in the past. However, recent evidence suggests
             that, when judging whether or not an individual is
             responsible for a certain action - even in concrete,
             emotionally laden and fully deterministic scenarios -
             considerations about alternative future possibilities may
             become relevant. This paper reviews this evidence and
             suggests a way of interpreting the nature of these effects
             as well as some consequences for experimental philosophy and
             psychology of free will and responsibility going
             forward.},
   Key = {fds331533}
}


%% Dodge, Kenneth A.   
@article{fds333726,
   Author = {Duell, N and Icenogle, G and Silva, K and Chein, J and Steinberg, L and Banich, MT and Di Guinta and L and Dodge, KA and Fanti, KA and Lansford,
             JE and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Tapanya, S and Uribe Tirado and LM and Alampay, LP and Al-Hassan, SM and Takash, HMS and Bacchini, D and Chang, L and Chaudhary,
             N},
   Title = {A cross-sectional examination of response inhibition and
             working memory on the Stroop task},
   Journal = {Cognitive Development},
   Volume = {47},
   Pages = {19-31},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.cogdev.2018.02.003},
   Abstract = {© 2018 Elsevier Inc. The authors examined the association
             between working memory and response inhibition on the Stroop
             task using a cross-sectional, international sample of 5099
             individuals (49.3% male) ages 10–30 (M = 17.04 years; SD =
             5.9). Response inhibition was measured using a Stroop task
             that included “equal” and “unequal” blocks, during
             which the relative frequency of neutral and incongruent
             trials was manipulated. Competing stimuli in incongruent
             trials evinced inhibitory functioning, and having a lower
             proportion of incongruent trials (as in unequal blocks)
             placed higher demands on working memory. Results for
             accuracy indicated that age and working memory were
             independently associated with response inhibition. Age
             differences in response inhibition followed a curvilinear
             trajectory, with performance improving into early adulthood.
             Response inhibition was greatest among individuals with high
             working memory. For response time, age uniquely predicted
             response inhibition in unequal blocks. In equal blocks, age
             differences in response inhibition varied as a function of
             working memory, with age differences being least pronounced
             among individuals with high working memory. The implications
             of considering the association between response inhibition
             and working memory in the context of development are
             discussed.},
   Doi = {10.1016/j.cogdev.2018.02.003},
   Key = {fds333726}
}

@article{fds329395,
   Author = {Duell, N and Steinberg, L and Icenogle, G and Chein, J and Chaudhary, N and Di Giunta and L and Dodge, KA and Fanti, KA and Lansford, JE and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Tapanya, S and Uribe
             Tirado, LM and Alampay, LP and Al-Hassan, SM and Takash, HMS and Bacchini, D and Chang, L},
   Title = {Age Patterns in Risk Taking Across the World.},
   Journal = {Journal of Youth and Adolescence},
   Volume = {47},
   Number = {5},
   Pages = {1052-1072},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1007/s10964-017-0752-y},
   Abstract = {Epidemiological data indicate that risk behaviors are among
             the leading causes of adolescent morbidity and mortality
             worldwide. Consistent with this, laboratory-based studies of
             age differences in risk behavior allude to a peak in
             adolescence, suggesting that adolescents demonstrate a
             heightened propensity, or inherent inclination, to take
             risks. Unlike epidemiological reports, studies of risk
             taking propensity have been limited to Western samples,
             leaving questions about the extent to which heightened risk
             taking propensity is an inherent or culturally constructed
             aspect of adolescence. In the present study, age patterns in
             risk-taking propensity (using two laboratory tasks: the
             Stoplight and the BART) and real-world risk taking (using
             self-reports of health and antisocial risk taking) were
             examined in a sample of 5227 individuals (50.7% female) ages
             10-30 (M = 17.05 years, SD = 5.91) from 11 Western
             and non-Western countries (China, Colombia, Cyprus, India,
             Italy, Jordan, Kenya, the Philippines, Sweden, Thailand, and
             the US). Two hypotheses were tested: (1) risk taking follows
             an inverted-U pattern across age groups, peaking earlier on
             measures of risk taking propensity than on measures of
             real-world risk taking, and (2) age patterns in risk taking
             propensity are more consistent across countries than age
             patterns in real-world risk taking. Overall, risk taking
             followed the hypothesized inverted-U pattern across age
             groups, with health risk taking evincing the latest peak.
             Age patterns in risk taking propensity were more consistent
             across countries than age patterns in real-world risk
             taking. Results suggest that although the association
             between age and risk taking is sensitive to measurement and
             culture, around the world, risk taking is generally highest
             among late adolescents.},
   Doi = {10.1007/s10964-017-0752-y},
   Key = {fds329395}
}

@article{fds332672,
   Author = {Crowley, DM and Dodge, KA and Barnett, WS and Corso, P and Duffy, S and Graham, P and Greenberg, M and Haskins, R and Hill, L and Jones, DE and Karoly, LA and Kuklinski, MR and Plotnick, R},
   Title = {Standards of Evidence for Conducting and Reporting Economic
             Evaluations in Prevention Science.},
   Journal = {Prevention Science},
   Volume = {19},
   Number = {3},
   Pages = {366-390},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s11121-017-0858-1},
   Abstract = {Over a decade ago, the Society for Prevention Research
             endorsed the first standards of evidence for research in
             preventive interventions. The growing recognition of the
             need to use limited resources to make sound investments in
             prevention led the Board of Directors to charge a new task
             force to set standards for research in analysis of the
             economic impact of preventive interventions. This article
             reports the findings of this group's deliberations, proposes
             standards for economic analyses, and identifies
             opportunities for future prevention science. Through
             examples, policymakers' need and use of economic analysis
             are described. Standards are proposed for framing economic
             analysis, estimating costs of prevention programs,
             estimating benefits of prevention programs, implementing
             summary metrics, handling uncertainty in estimates, and
             reporting findings. Topics for research in economic analysis
             are identified. The SPR Board of Directors endorses the
             "Standards of Evidence for Conducting and Reporting Economic
             Evaluations in Prevention Science."},
   Doi = {10.1007/s11121-017-0858-1},
   Key = {fds332672}
}

@article{fds323764,
   Author = {Steinberg, L and Icenogle, G and Shulman, EP and Breiner, K and Chein,
             J and Bacchini, D and Chang, L and Chaudhary, N and Giunta, LD and Dodge,
             KA and Fanti, KA and Lansford, JE and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Tapanya, S and Tirado,
             LMU and Alampay, LP and Al-Hassan, SM and Takash,
             HMS},
   Title = {Around the world, adolescence is a time of heightened
             sensation seeking and immature self-regulation.},
   Journal = {Developmental Science},
   Volume = {21},
   Number = {2},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1111/desc.12532},
   Abstract = {The dual systems model of adolescent risk-taking portrays
             the period as one characterized by a combination of
             heightened sensation seeking and still-maturing
             self-regulation, but most tests of this model have been
             conducted in the United States or Western Europe. In the
             present study, these propositions are tested in an
             international sample of more than 5000 individuals between
             ages 10 and 30 years from 11 countries in Africa, Asia,
             Europe and the Americas, using a multi-method test battery
             that includes both self-report and performance-based
             measures of both constructs. Consistent with the dual
             systems model, sensation seeking increased between
             preadolescence and late adolescence, peaked at age 19, and
             declined thereafter, whereas self-regulation increased
             steadily from preadolescence into young adulthood, reaching
             a plateau between ages 23 and 26. Although there were some
             variations in the magnitude of the observed age trends, the
             developmental patterns were largely similar across
             countries.},
   Doi = {10.1111/desc.12532},
   Key = {fds323764}
}

@article{fds330823,
   Author = {Petersen, IT and Lindhiem, O and LeBeau, B and Bates, JE and Pettit, GS and Lansford, JE and Dodge, KA},
   Title = {Development of internalizing problems from adolescence to
             emerging adulthood: Accounting for heterotypic continuity
             with vertical scaling.},
   Journal = {Developmental Psychology},
   Volume = {54},
   Number = {3},
   Pages = {586-599},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1037/dev0000449},
   Abstract = {Manifestations of internalizing problems, such as specific
             symptoms of anxiety and depression, can change across
             development, even if individuals show strong continuity in
             rank-order levels of internalizing problems. This
             illustrates the concept of heterotypic continuity, and
             raises the question of whether common measures might be
             construct-valid for one age but not another. This study
             examines mean-level changes in internalizing problems across
             a long span of development at the same time as accounting
             for heterotypic continuity by using age-appropriate,
             changing measures. Internalizing problems from age 14-24
             were studied longitudinally in a community sample (N = 585),
             using Achenbach's Youth Self-Report (YSR) and Young Adult
             Self-Report (YASR). Heterotypic continuity was evaluated
             with an item response theory (IRT) approach to vertical
             scaling, linking different measures over time to be on the
             same scale, as well as with a Thurstone scaling approach.
             With vertical scaling, internalizing problems peaked in
             mid-to-late adolescence and showed a group-level decrease
             from adolescence to early adulthood, a change that would not
             have been seen with the approach of using only age-common
             items. Individuals' trajectories were sometimes different
             than would have been seen with the common-items approach.
             Findings support the importance of considering heterotypic
             continuity when examining development and vertical scaling
             to account for heterotypic continuity with changing
             measures. (PsycINFO Database Record},
   Doi = {10.1037/dev0000449},
   Key = {fds330823}
}

@article{fds333727,
   Author = {Olson, SL and Davis-Kean, P and Chen, M and Lansford, JE and Bates, JE and Pettit, GS and Dodge, KA},
   Title = {Mapping the Growth of Heterogeneous Forms of Externalizing
             Problem Behavior Between Early Childhood and
             Adolescence:A Comparison of Parent and Teacher
             Ratings.},
   Journal = {Journal of Abnormal Child Psychology},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1007/s10802-018-0407-9},
   Abstract = {We compared long-term growth patterns in teachers' and
             mothers' ratings of Overt Aggression, Covert Aggression,
             Oppositional Defiance, Impulsivity/inattention, and Emotion
             Dysregulation across developmental periods spanning
             kindergarten through grade 8 (ages 5 to 13 years). We also
             determined whether salient background characteristics and
             measures of child temperament and parenting risk
             differentially predicted growth in discrete categories of
             child externalizing symptoms across development.
             Participants were 549 kindergarten-age children (51% male;
             83% European American; 17% African American) whose problem
             behaviors were rated by teachers and parents each successive
             year of development through 8th grade. Latent growth curve
             analyses were performed for each component scale,
             contrasting with an overall index of externalizing, in a
             piecewise fashion encompassing two periods of development:
             K-1and grades 1-8. Our findings showed that there were
             meaningful differences and similarities between informants
             in their levels of concern about specific forms of
             externalizing problems, patterns of change in problem
             behavior reports across development, and in the extent to
             which their ratings of specific problems were associated
             with distal and proximal covariates. Thus, these data
             provided novel information about issues that have received
             scant empirical attention and have important implications
             for understanding the development and prevention of
             children's long-term externalizing problems.},
   Doi = {10.1007/s10802-018-0407-9},
   Key = {fds333727}
}

@article{fds330822,
   Author = {Lansford, JE and Godwin, J and Al-Hassan, SM and Bacchini, D and Bornstein, MH and Chang, L and Chen, B-B and Deater-Deckard, K and Di
             Giunta, L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Alampay,
             LP and Uribe Tirado and LM and Zelli, A},
   Title = {Longitudinal associations between parenting and youth
             adjustment in twelve cultural groups: Cultural normativeness
             of parenting as a moderator.},
   Journal = {Developmental Psychology},
   Volume = {54},
   Number = {2},
   Pages = {362-377},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1037/dev0000416},
   Abstract = {To examine whether the cultural normativeness of parents'
             beliefs and behaviors moderates the links between those
             beliefs and behaviors and youths' adjustment, mothers,
             fathers, and children (N = 1,298 families) from 12 cultural
             groups in 9 countries (China, Colombia, Italy, Jordan,
             Kenya, Philippines, Sweden, Thailand, and the United States)
             were interviewed when children were, on average, 10 years
             old and again when children were 12 years old. Multilevel
             models examined 5 aspects of parenting (expectations
             regarding family obligations, monitoring, psychological
             control, behavioral control, warmth/affection) in relation
             to 5 aspects of youth adjustment (social competence,
             prosocial behavior, academic achievement, externalizing
             behavior, internalizing behavior). Interactions between
             family level and culture-level predictors were tested to
             examine whether cultural normativeness of parenting
             behaviors moderated the link between those behaviors and
             children's adjustment. More evidence was found for within-
             than between-culture differences in parenting predictors of
             youth adjustment. In 7 of the 8 instances in which cultural
             normativeness was found to moderate the link between
             parenting and youth adjustment, the link between a
             particular parenting behavior and youth adjustment was
             magnified in cultural contexts in which the parenting
             behavior was more normative. (PsycINFO Database
             Record},
   Doi = {10.1037/dev0000416},
   Key = {fds330822}
}

@article{fds333728,
   Author = {Miller, AB and Sheridan, MA and Hanson, JL and McLaughlin, KA and Bates,
             JE and Lansford, JE and Pettit, GS and Dodge, KA},
   Title = {Dimensions of deprivation and threat, psychopathology, and
             potential mediators: A multi-year longitudinal
             analysis.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {127},
   Number = {2},
   Pages = {160-170},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1037/abn0000331},
   Abstract = {Prior research demonstrates a link between exposure to
             childhood adversity and psychopathology later in
             development. However, work on mechanisms linking adversity
             to psychopathology fails to account for specificity in these
             pathways across different types of adversity. Here, we test
             a conceptual model that distinguishes deprivation and threat
             as distinct forms of childhood adversity with different
             pathways to psychopathology. Deprivation involves an absence
             of inputs from the environment, such as cognitive and social
             stimulation, that influence psychopathology by altering
             cognitive development, such as verbal abilities. Threat
             includes experiences involving harm or threat of harm that
             increase risk for psychopathology through disruptions in
             social-emotional processing. We test the prediction that
             deprivation, but not threat, increases risk for
             psychopathology through altered verbal abilities. Data were
             drawn from the Child Development Project (N = 585), which
             followed children for over a decade. We analyze data from
             assessment points at age 5, 6, 14, and 17 years. Mothers
             completed interviews at age 5 and 6 on exposure to threat
             and deprivation experiences. Youth verbal abilities were
             assessed at age 14. At age 17, mothers reported on child
             psychopathology. A path analysis model tested longitudinal
             paths to internalizing and externalizing problems from
             experiences of deprivation and threat. Consistent with
             predictions, deprivation was associated with risk for
             externalizing problems via effects on verbal abilities at
             age 14. Threat was associated longitudinally with both
             internalizing and externalizing problems, but these effects
             were not mediated by verbal abilities. Results suggest that
             unique developmental mechanisms link different forms of
             adversity with psychopathology. (PsycINFO Database
             Record},
   Doi = {10.1037/abn0000331},
   Key = {fds333728}
}

@article{fds326573,
   Author = {Wang, FL and Chassin, L and Bates, JE and Dick, D and Lansford, JE and Pettit, GS and Dodge, KA},
   Title = {Serotonin functioning and adolescents' alcohol use: A
             genetically informed study examining mechanisms of
             risk.},
   Journal = {Development and Psychopathology},
   Volume = {30},
   Number = {1},
   Pages = {213-233},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1017/s095457941700058x},
   Abstract = {The current study used data from two longitudinal samples to
             test whether self-regulation, depressive symptoms, and
             aggression/antisociality were mediators in the relation
             between a polygenic score indexing serotonin (5-HT)
             functioning and alcohol use in adolescence. The results from
             an independent genome-wide association study of
             5-hydroxyindoleacetic acid in the cerebrospinal fluid were
             used to create 5-HT polygenic risk scores. Adolescents
             and/or parents reported on adolescents' self-regulation
             (Time 1), depressive symptoms (Time 2), aggression/antisociality
             (Time 2), and alcohol use (Time 3). The results showed that
             5-HT polygenic risk did not predict self-regulation.
             However, adolescents with higher levels of 5-HT polygenic
             risk showed greater depression and aggression/antisociality.
             Adolescents' aggression/antisociality mediated the relation
             between 5-HT polygenic risk and later alcohol use. Deficits
             in self-regulation also predicted depression and
             aggression/antisociality, and indirectly predicted alcohol
             use through aggression/antisociality. Pathways to alcohol
             use were especially salient for males from families with low
             parental education in one of the two samples. The results
             provide insights into the longitudinal mechanisms underlying
             the relation between 5-HT functioning and alcohol use (i.e.,
             earlier aggression/antisociality). There was no evidence
             that genetically based variation in 5-HT functioning
             predisposed individuals to deficits in self-regulation.
             Genetically based variation in 5-HT functioning and
             self-regulation might be separate, transdiagnostic risk
             factors for several types of psychopathology.},
   Doi = {10.1017/s095457941700058x},
   Key = {fds326573}
}

@article{fds330213,
   Author = {Schwartz, D and Lansford, JE and Dodge, KA and Pettit, GS and Bates,
             JE},
   Title = {Peer Victimization during Middle Childhood as a Marker of
             Attenuated Risk for Adult Arrest.},
   Journal = {Journal of Abnormal Child Psychology},
   Volume = {46},
   Number = {1},
   Pages = {57-65},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s10802-017-0354-x},
   Abstract = {This longitudinal investigation examined interactions
             between aggression and peer victimization during middle
             childhood in the prediction of arrest through the adult
             years for 388 (198 boys, 190 girls) study participants. As
             part of an ongoing multisite study (i.e., Child Development
             Project), peer victimization and aggression were assessed
             via a peer nomination inventory in middle childhood, and
             juvenile and adult arrest histories were assessed via a
             self-report questionnaire as well as review of court
             records. Early aggression was linked to later arrest but
             only for those youths who were rarely victimized by peers.
             Although past investigators have viewed youths who are both
             aggressive and victimized as a high-risk subgroup, our
             findings suggest that the psychological and behavioral
             attributes of these children may mitigate trajectories
             toward antisocial problems.},
   Doi = {10.1007/s10802-017-0354-x},
   Key = {fds330213}
}

@article{fds332381,
   Author = {Zheng, Y and Albert, D and McMahon, RJ and Dodge, K and Dick,
             D},
   Title = {Glucocorticoid Receptor (NR3C1) Gene Polymorphism Moderate
             Intervention Effects on the Developmental Trajectory of
             African-American Adolescent Alcohol Abuse},
   Journal = {Prevention Science},
   Volume = {19},
   Number = {1},
   Pages = {79-89},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s11121-016-0726-4},
   Doi = {10.1007/s11121-016-0726-4},
   Key = {fds332381}
}

@article{fds327155,
   Author = {Deater-Deckard, K and Godwin, J and Lansford, JE and Bacchini, D and Bombi, AS and Bornstein, MH and Chang, L and Di Giunta and L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring,
             E and Steinberg, L and Tapanya, S and Alampay, LP and Uribe Tirado and LM and Zelli, A and Al-Hassan, SM},
   Title = {Within- and between-person and group variance in behavior
             and beliefs in cross-cultural longitudinal
             data.},
   Journal = {Journal of Adolescence},
   Volume = {62},
   Pages = {207-217},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.adolescence.2017.06.002},
   Abstract = {This study grapples with what it means to be part of a
             cultural group, from a statistical modeling perspective. The
             method we present compares within- and between-cultural
             group variability, in behaviors in families. We demonstrate
             the method using a cross-cultural study of adolescent
             development and parenting, involving three biennial waves of
             longitudinal data from 1296 eight-year-olds and their
             parents (multiple cultures in nine countries). Family
             members completed surveys about parental negativity and
             positivity, child academic and social-emotional adjustment,
             and attitudes about parenting and adolescent behavior.
             Variance estimates were computed at the cultural group,
             person, and within-person level using multilevel models. Of
             the longitudinally consistent variance, most was within and
             not between cultural groups-although there was a wide range
             of between-group differences. This approach to quantifying
             cultural group variability may prove valuable when applied
             to quantitative studies of acculturation.},
   Doi = {10.1016/j.adolescence.2017.06.002},
   Key = {fds327155}
}

@article{fds327289,
   Author = {Berlin, LJ and Martoccio, TL and Appleyard Carmody and K and Goodman,
             WB and O'Donnell, K and Williams, J and Murphy, RA and Dodge,
             KA},
   Title = {Can typical US home visits affect infant attachment?
             Preliminary findings from a randomized trial of Healthy
             Families Durham.},
   Journal = {Attachment & Human Development},
   Volume = {19},
   Number = {6},
   Pages = {559-579},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1080/14616734.2017.1339359},
   Abstract = {US government-funded early home visiting services are
             expanding significantly. The most widely implemented home
             visiting models target at-risk new mothers and their
             infants. Such home visiting programs typically aim to
             support infant-parent relationships; yet, such programs'
             effects on infant attachment quality per se are as yet
             untested. Given these programs' aims, and the crucial role
             of early attachments in human development, it is important
             to understand attachment processes in home visited families.
             The current, preliminary study examined 94 high-risk
             mother-infant dyads participating in a randomized evaluation
             of the Healthy Families Durham (HFD) home visiting program.
             We tested (a) infant attachment security and disorganization
             as predictors of toddler behavior problems and (b) program
             effects on attachment security and disorganization. We found
             that (a) infant attachment disorganization (but not
             security) predicted toddler behavior problems and (b)
             participation in HFD did not significantly affect infant
             attachment security or disorganization. Findings are
             discussed in terms of the potential for attachment-specific
             interventions to enhance the typical array of home visiting
             services.},
   Doi = {10.1080/14616734.2017.1339359},
   Key = {fds327289}
}

@article{fds330211,
   Author = {Lansford, JE and Godwin, J and Bornstein, MH and Chang, L and Deater-Deckard, K and Di Giunta and L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Alampay, LP and Uribe Tirado and LM and Al-Hassan, SM and Bacchini, D},
   Title = {Reward sensitivity, impulse control, and social cognition as
             mediators of the link between childhood family adversity and
             externalizing behavior in eight countries.},
   Journal = {Development and Psychopathology},
   Volume = {29},
   Number = {5},
   Pages = {1675-1688},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1017/s0954579417001328},
   Abstract = {Using data from 1,177 families in eight countries (Colombia,
             Italy, Jordan, Kenya, the Philippines, Sweden, Thailand, and
             the United States), we tested a conceptual model of direct
             effects of childhood family adversity on subsequent
             externalizing behaviors as well as indirect effects through
             psychological mediators. When children were 9 years old,
             mothers and fathers reported on financial difficulties and
             their use of corporal punishment, and children reported
             perceptions of their parents' rejection. When children were
             10 years old, they completed a computerized battery of tasks
             assessing reward sensitivity and impulse control and
             responded to questions about hypothetical social
             provocations to assess their hostile attributions and
             proclivity for aggressive responding. When children were 12
             years old, they reported on their externalizing behavior.
             Multigroup structural equation models revealed that across
             all eight countries, childhood family adversity had direct
             effects on externalizing behaviors 3 years later, and
             childhood family adversity had indirect effects on
             externalizing behavior through psychological mediators. The
             findings suggest ways in which family-level adversity poses
             risk for children's subsequent development of problems at
             psychological and behavioral levels, situated within diverse
             cultural contexts.},
   Doi = {10.1017/s0954579417001328},
   Key = {fds330211}
}

@article{fds330210,
   Author = {Sorensen, LC and Cook, PJ and Dodge, KA},
   Title = {From Parents to Peers: Trajectories in Sources of Academic
             Influence Grades 4 to 8},
   Journal = {Educational Evaluation and Policy Analysis},
   Volume = {39},
   Number = {4},
   Pages = {697-711},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.3102/0162373717708335},
   Doi = {10.3102/0162373717708335},
   Key = {fds330210}
}

@article{fds330212,
   Author = {McQuillan, ME and Kultur, EC and Bates, JE and O'Reilly, LM and Dodge,
             KA and Lansford, JE and Pettit, GS},
   Title = {Dysregulation in children: Origins and implications from age
             5 to age 28.},
   Journal = {Development and Psychopathology},
   Pages = {1-19},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s0954579417001572},
   Abstract = {Research shows that childhood dysregulation is associated
             with later psychiatric disorders. It does not yet resolve
             discrepancies in the operationalization of dysregulation. It
             is also far from settled on the origins and implications of
             individual differences in dysregulation. This study tested
             several operational definitions of dysregulation using
             Achenbach attention, anxious/depressed, and aggression
             subscales. Individual growth curves of dysregulation were
             computed, and predictors of growth differences were
             considered. The study also compared the predictive utility
             of the dysregulation indexes to standard externalizing and
             internalizing indexes. Dysregulation was indexed annually
             for 24 years in a community sample (n = 585). Hierarchical
             linear models considered changes in dysregulation in
             relation to possible influences from parenting, family
             stress, child temperament, language, and peer relations. In
             a test of the meaning of dysregulation, it was related to
             functional and psychiatric outcomes in adulthood.
             Dysregulation predictions were further compared to those of
             the more standard internalizing and externalizing indexes.
             Growth curve analyses showed strong stability of
             dysregulation. Initial levels of dysregulation were
             predicted by temperamental resistance to control, and change
             in dysregulation was predicted by poor language ability and
             peer relations. Dysregulation and externalizing problems
             were associated with negative adult outcomes to a similar
             extent.},
   Doi = {10.1017/s0954579417001572},
   Key = {fds330212}
}

@article{fds330175,
   Author = {Cook, PJ and Dodge, KA and Gifford, EJ and Schulting,
             AB},
   Title = {A new program to prevent primary school absenteeism: Results
             of a pilot study in five schools},
   Journal = {Children and Youth Services Review},
   Volume = {82},
   Pages = {262-270},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.childyouth.2017.09.017},
   Doi = {10.1016/j.childyouth.2017.09.017},
   Key = {fds330175}
}

@article{fds327351,
   Author = {Goodnight, JA and Bates, JE and Holtzworth-Munroe, A and Pettit, GS and Ballard, RH and Iskander, JM and Swanson, A and Dodge, KA and Lansford,
             JE},
   Title = {Dispositional, demographic, and social predictors of
             trajectories of intimate partner aggression in early
             adulthood.},
   Journal = {Journal of Consulting and Clinical Psychology},
   Volume = {85},
   Number = {10},
   Pages = {950-965},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1037/ccp0000226},
   Abstract = {From a developmental systems perspective, the origins of
             maladjusted behavior are multifaceted, interdependent, and
             may differ at different points in development. Personality
             traits influence developmental outcomes, as do socialization
             environments, but the influence of personality depends on
             the socialization environment, and the influence of the
             socialization environment varies according to personality.
             The present study takes a developmental systems approach to
             investigate pathways through which dispositional traits in
             childhood might act in concert with peer and parental
             socialization contexts to predict trajectories of intimate
             partner aggression (IPA) during emerging adulthood.The study
             included 466 participants (49% male, 81% European American,
             15% African American) from a longitudinal study of social
             development. Measures of demographics, temperament,
             personality, parent-child relations, romantic relationships,
             peer relationships, and IPA were administered between 5 and
             23 years of age. The study used latent growth curve analysis
             to predict variations in trajectories of IPA during early
             adulthood.Numerous variables predicted risk for the
             perpetration of IPA, but different factors were associated
             at the end of adolescence (e.g., psychopathic traits) than
             with changes across early adulthood (e.g., friend
             antisociality). Males and individuals with a history of
             resistance to control temperament showed enhanced
             susceptibility to social risk factors, such as exposure to
             antisocial peers and poor parent-adolescent
             relations.Consistent with a developmental systems
             perspective, multiple factors, including personality traits
             in early childhood and aspects of the social environment in
             adolescence, predict trajectories of IPA during early
             adulthood through additive, mediated, and moderated
             pathways. Knowledge of these risk factors and for whom they
             are most influential could help inform efforts to prevent
             the emergence and persistence of IPA. (PsycINFO Database
             Record},
   Doi = {10.1037/ccp0000226},
   Key = {fds327351}
}

@article{fds322250,
   Author = {Icenogle, G and Steinberg, L and Olino, TM and Shulman, EP and Chein, J and Alampay, LP and Al-Hassan, SM and Takash, HMS and Bacchini, D and Chang,
             L and Chaudhary, N and Di Giunta and L and Dodge, KA and Fanti, KA and Lansford, JE and Malone, PS and Oburu, P and Pastorelli, C and Skinner,
             AT and Sorbring, E and Tapanya, S and Uribe Tirado,
             LM},
   Title = {Puberty Predicts Approach But Not Avoidance on the Iowa
             Gambling Task in a Multinational Sample.},
   Journal = {Child Development},
   Volume = {88},
   Number = {5},
   Pages = {1598-1614},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1111/cdev.12655},
   Abstract = {According to the dual systems model of adolescent risk
             taking, sensation seeking and impulse control follow
             different developmental trajectories across adolescence and
             are governed by two different brain systems. The authors
             tested whether different underlying processes also drive age
             differences in reward approach and cost avoidance. Using a
             modified Iowa Gambling Task in a multinational,
             cross-sectional sample of 3,234 adolescents (ages 9-17;
             M = 12.87, SD = 2.36), pubertal maturation, but not age,
             predicted reward approach, mediated through higher sensation
             seeking. In contrast, age, but not pubertal maturation,
             predicted increased cost avoidance, mediated through greater
             impulse control. These findings add to evidence that
             adolescent behavior is best understood as the product of two
             interacting, but independently developing, brain
             systems.},
   Doi = {10.1111/cdev.12655},
   Key = {fds322250}
}

@article{fds325210,
   Author = {Bornstein, MH and Putnick, DL and Lansford, JE and Al-Hassan, SM and Bacchini, D and Bombi, AS and Chang, L and Deater-Deckard, K and Di
             Giunta, L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Tirado,
             LMU and Zelli, A and Alampay, LP},
   Title = {'Mixed blessings': parental religiousness, parenting, and
             child adjustment in global perspective.},
   Journal = {The Journal of Child Psychology and Psychiatry and Allied
             Disciplines},
   Volume = {58},
   Number = {8},
   Pages = {880-892},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/jcpp.12705},
   Abstract = {Most studies of the effects of parental religiousness on
             parenting and child development focus on a particular
             religion or cultural group, which limits generalizations
             that can be made about the effects of parental religiousness
             on family life.We assessed the associations among parental
             religiousness, parenting, and children's adjustment in a
             3-year longitudinal investigation of 1,198 families from
             nine countries. We included four religions (Catholicism,
             Protestantism, Buddhism, and Islam) plus unaffiliated
             parents, two positive (efficacy and warmth) and two negative
             (control and rejection) parenting practices, and two
             positive (social competence and school performance) and two
             negative (internalizing and externalizing) child outcomes.
             Parents and children were informants.Greater parent
             religiousness had both positive and negative associations
             with parenting and child adjustment. Greater parent
             religiousness when children were age 8 was associated with
             higher parental efficacy at age 9 and, in turn, children's
             better social competence and school performance and fewer
             child internalizing and externalizing problems at age 10.
             However, greater parent religiousness at age 8 was also
             associated with more parental control at age 9, which in
             turn was associated with more child internalizing and
             externalizing problems at age 10. Parental warmth and
             rejection had inconsistent relations with parental
             religiousness and child outcomes depending on the informant.
             With a few exceptions, similar patterns of results held for
             all four religions and the unaffiliated, nine sites, mothers
             and fathers, girls and boys, and controlling for demographic
             covariates.Parents and children agree that parental
             religiousness is associated with more controlling parenting
             and, in turn, increased child problem behaviors. However,
             children see religiousness as related to parental rejection,
             whereas parents see religiousness as related to parental
             efficacy and warmth, which have different associations with
             child functioning. Studying both parent and child views of
             religiousness and parenting are important to understand the
             effects of parental religiousness on parents and
             children.},
   Doi = {10.1111/jcpp.12705},
   Key = {fds325210}
}

@article{fds327154,
   Author = {Alampay, LP and Godwin, J and Lansford, JE and Bombi, AS and Bornstein,
             MH and Chang, L and Deater-Deckard, K and Di Giunta and L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring,
             E and Tapanya, S and Uribe Tirado and LM and Zelli, A and Al-Hassan, S and Bacchini, D},
   Title = {Severity and Justness Do Not Moderate the Relation Between
             Corporal Punishment and Negative Child Outcomes: A
             Multicultural and Longitudinal Study.},
   Journal = {International Journal of Behavioral Development},
   Volume = {41},
   Number = {4},
   Pages = {491-502},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1177/0165025417697852},
   Abstract = {There is strong evidence of a positive association between
             corporal punishment and negative child outcomes, but
             previous studies have suggested that the manner in which
             parents implement corporal punishment moderates the effects
             of its use. This study investigated whether severity and
             justness in the use of corporal punishment moderate the
             associations between frequency of corporal punishment and
             child externalizing and internalizing behaviors. This
             question was examined using a multicultural sample from
             eight countries and two waves of data collected one year
             apart. Interviews were conducted with 998 children aged 7-10
             years, and their mothers and fathers, from China, Colombia,
             Italy, Jordan, Kenya, Philippines, Thailand, and the United
             States. Mothers and fathers responded to questions on the
             frequency, severity, and justness of their use of corporal
             punishment; they also reported on the externalizing and
             internalizing behavior of their child. Children reported on
             their aggression. Multigroup path models revealed that
             across cultural groups, and as reported by mothers and
             fathers, there is a positive relation between the frequency
             of corporal punishment and externalizing child behaviors.
             Mother-reported severity and father-reported justness were
             associated with child-reported aggression. Neither severity
             nor justness moderated the relation between frequency of
             corporal punishment and child problem behavior. The null
             result suggests that more use of corporal punishment is
             harmful to children regardless of how it is implemented, but
             requires further substantiation as the study is unable to
             definitively conclude that there is no true interaction
             effect.},
   Doi = {10.1177/0165025417697852},
   Key = {fds327154}
}

@article{fds332382,
   Author = {Saint-Eloi Cadely and H and Pittman, JF and Pettit, GS and Lansford, JE and Bates, JE and Dodge, KA and Holtzworth-Munroe,
             A},
   Title = {Classes of Intimate Partner Violence From Late Adolescence
             to Young Adulthood.},
   Journal = {Journal of Interpersonal Violence},
   Pages = {886260517715601},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1177/0886260517715601},
   Abstract = {Researchers do not agree on how intimate partner violence
             (IPV) emerges and changes from adolescence to young
             adulthood. This may be because change in these behaviors
             varies across individuals. The present study uses a
             longitudinal, person-centered approach to examine whether
             there are multiple classes or patterns of change in the
             perpetration of IPV during the transitional period from
             adolescence (age 18) to young adulthood (age 25) using data
             collected annually from a community sample of 484
             participants. Latent class analysis was the analytic
             approach used. Results revealed three patterns for
             psychological IPV (Little-to-None, Minor/Increasing, and
             Extensive/Increasing) and two patterns for physical IPV
             (Little-to-None and Extensive). Patterns varied greatly in
             number of representatives, although they were more balanced
             in size for psychological than physical IPV. Variations in
             IPV behaviors were also revealed across classes, although as
             expected in a community sample, minor forms of IPV were more
             common than severe forms. In addition, classes differed in
             demographic and relationship status variables. These
             findings suggest that IPV may occur in multiple distinct
             patterns as opposed to one average pattern across a
             population. This suggests that interventions for IPV may
             need to be geared to differences in patterns to enhance
             their efficacy.},
   Doi = {10.1177/0886260517715601},
   Key = {fds332382}
}

@article{fds315899,
   Author = {Di Giunta and L and Iselin, A-MR and Eisenberg, N and Pastorelli, C and Gerbino, M and Lansford, JE and Dodge, KA and Caprara, GV and Bacchini,
             D and Uribe Tirado and LM and Thartori, E},
   Title = {Measurement Invariance and Convergent Validity of Anger and
             Sadness Self-Regulation Among Youth From Six Cultural
             Groups.},
   Journal = {Assessment},
   Volume = {24},
   Number = {4},
   Pages = {484-502},
   Year = {2017},
   Month = {June},
   ISSN = {1073-1911},
   url = {http://dx.doi.org/10.1177/1073191115615214},
   Abstract = {The present study examined measurement invariance and
             convergent validity of a novel vignette-based measure of
             emotion-specific self-regulation that simultaneously
             assesses attributional bias, emotion-regulation, and
             self-efficacy beliefs about emotion regulation. Participants
             included 541 youth-mother dyads from three countries (Italy,
             the United States, and Colombia) and six ethnic/cultural
             groups. Participants were 12.62 years old ( SD = 0.69). In
             response to vignettes involving ambiguous peer interactions,
             children reported their hostile/depressive attribution bias,
             self-efficacy beliefs about anger and sadness regulation,
             and anger/sadness regulation strategies (i.e., dysregulated
             expression and rumination). Across the six cultural groups,
             anger and sadness self-regulation subscales had full metric
             and partial scalar invariance for a one-factor model, with
             some exceptions. We found support for both a four- and
             three-factor oblique model (dysregulated expression and
             rumination loaded on a second-order factor) for both anger
             and sadness. Anger subscales were related to externalizing
             problems, while sadness subscales were related to
             internalizing symptoms.},
   Doi = {10.1177/1073191115615214},
   Key = {fds315899}
}

@article{fds320116,
   Author = {Dodge, KA and Bai, Y and Ladd, HF and Muschkin, CG},
   Title = {Impact of North Carolina's Early Childhood Programs and
             Policies on Educational Outcomes in Elementary
             School.},
   Journal = {Child Development},
   Volume = {88},
   Number = {3},
   Pages = {996-1014},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1111/cdev.12645},
   Abstract = {North Carolina's Smart Start and More at Four (MAF) early
             childhood programs were evaluated through the end of
             elementary school (age 11) by estimating the impact of state
             funding allocations to programs in each of 100 counties
             across 13 consecutive years on outcomes for all children in
             each county-year group (n = 1,004,571; 49% female; 61%
             non-Latinx White, 30% African American, 4% Latinx, 5%
             other). Student-level regression models with county and year
             fixed effects indicated significant positive impacts of each
             program on reading and math test scores and reductions in
             special education and grade retention in each grade. Effect
             sizes grew or held steady across years. Positive effects
             held for both high- and low-poverty families, suggesting
             spillover of effects to nonparticipating
             peers.},
   Doi = {10.1111/cdev.12645},
   Key = {fds320116}
}

@article{fds324191,
   Author = {Calvert, SL and Appelbaum, M and Dodge, KA and Graham, S and Nagayama
             Hall, GC and Hamby, S and Fasig-Caldwell, LG and Citkowicz, M and Galloway, DP and Hedges, LV},
   Title = {The American Psychological Association Task Force assessment
             of violent video games: Science in the service of public
             interest.},
   Journal = {American Psychologist},
   Volume = {72},
   Number = {2},
   Pages = {126-143},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1037/a0040413},
   Abstract = {A task force of experts was convened by the American
             Psychological Association (APA) to update the knowledge and
             policy about the impact of violent video game use on
             potential adverse outcomes. This APA Task Force on Media
             Violence examined the existing literature, including the
             meta-analyses in the field, since the last APA report on
             media violence in 2005. Because the most recent
             meta-analyses were published in 2010 and reflected work
             through 2009, the task force conducted a search of the
             published studies from 2009-2013. These recently published
             articles were scored and assessed by a systematic
             evidentiary review, followed by a meta-analysis of the high
             utility studies, as documented in the evidentiary review.
             Consistent with the literature that we reviewed, we found
             that violent video game exposure was associated with: an
             increased composite aggression score; increased aggressive
             behavior; increased aggressive cognitions; increased
             aggressive affect, increased desensitization, and decreased
             empathy; and increased physiological arousal. The size of
             the effects was similar to that in prior meta-analyses,
             suggesting a stable result. Our task force concluded that
             violent video game use is a risk factor for adverse
             outcomes, but found insufficient studies to examine any
             potential link between violent video game use and
             delinquency or criminal behavior. Our technical report is
             the basis of this article. (PsycINFO Database
             Record},
   Doi = {10.1037/a0040413},
   Key = {fds324191}
}


%% Duffy, Korrina A   
@article{fds323898,
   Author = {Duffy, KA and Harris, LT and Chartrand, TL and Stanton,
             SJ},
   Title = {Women recovering from social rejection: The effect of the
             person and the situation on a hormonal mechanism of
             affiliation.},
   Journal = {Psychoneuroendocrinology},
   Volume = {76},
   Pages = {174-182},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2016.11.017},
   Abstract = {Rejection can motivate either affiliation or withdrawal. In
             order to study how personality and situational variables
             influence whether women will be motivated to affiliate
             versus withdraw, we manipulate social feedback (rejection
             vs. acceptance) and opportunity for face-to-face interaction
             (blocked vs. face-to-face) and measure the individual
             difference variables rejection sensitivity and social
             anxiety. We test how these variables affect endogenous
             progesterone and cortisol concentrations, which are presumed
             to signal motivational responses to rejection. We find that
             three-way interactions involving social feedback,
             opportunity for face-to-face interactions, and either social
             anxiety or rejection sensitivity significantly predict
             progesterone change, but not cortisol change. Both
             interactions are driven by sharp progesterone decreases for
             women high in social anxiety/rejection sensitivity who have
             been rejected and who have no opportunity to reaffiliate in
             a face-to-face interaction. This progesterone change may be
             a physiological marker of motivation for social avoidance
             following rejection for women who cannot reaffiliate and who
             are particularly socially anxious or sensitive to
             rejection.},
   Doi = {10.1016/j.psyneuen.2016.11.017},
   Key = {fds323898}
}


%% Egger, Helen L.   
@article{fds328293,
   Author = {Zucker, N and Mauro, C and Craske, M and Wagner, HR and Datta, N and Hopkins, H and Caldwell, K and Kiridly, A and Marsan, S and Maslow, G and Mayer, E and Egger, H},
   Title = {Acceptance-based interoceptive exposure for young children
             with functional abdominal pain.},
   Journal = {Behaviour Research and Therapy},
   Volume = {97},
   Pages = {200-212},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.brat.2017.07.009},
   Abstract = {Functional abdominal pain (FAP) is a common childhood
             somatic complaint that contributes to impairment in daily
             functioning (e.g., school absences) and increases risk for
             chronic pain and psychiatric illness. Cognitive behavioral
             treatments for FAP target primarily older children
             (9 + years) and employ strategies to reduce a focus on
             pain. The experience of pain may be an opportunity to teach
             viscerally hypersensitive children to interpret the function
             of a variety of bodily signals (including those of hunger,
             emotions) thereby reducing fear of bodily sensations and
             facilitating emotion awareness and self-regulation. We
             designed and tested an interoceptive exposure treatment for
             younger children (5-9 years) with FAP. Assessments included
             diagnostic interviews, 14 days of daily pain monitoring, and
             questionnaires. Treatment involved 10 weekly appointments.
             Using cartoon characters to represent bodily sensations
             (e.g., Gassy Gus), children were trained to be "FBI agents"
             - Feeling and Body Investigators - who investigated
             sensations through exercises that provoked somatic
             experience. 24 parent-child dyads are reported. Pain
             (experience, distress, and interference) and negative affect
             demonstrated clinically meaningful and statistically
             significant change with effect sizes ranging from 0.48 to 71
             for pain and from 0.38 to 0.61 for pain distress, total
             pain: X2 (1, n = 24) = 13.14, p < 0.0003. An
             intervention that helps children adopt a curious stance and
             focus on somatic symptoms reduces pain and may help lessen
             somatic fear generally.NCT02075437.},
   Doi = {10.1016/j.brat.2017.07.009},
   Key = {fds328293}
}

@article{fds323858,
   Author = {Campbell, K and Carpenter, KLH and Espinosa, S and Hashemi, J and Qiu,
             Q and Tepper, M and Calderbank, R and Sapiro, G and Egger, HL and Baker,
             JP and Dawson, G},
   Title = {Use of a Digital Modified Checklist for Autism in Toddlers -
             Revised with Follow-up to Improve Quality of Screening for
             Autism.},
   Journal = {The Journal of Pediatrics},
   Volume = {183},
   Pages = {133-139.e1},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jpeds.2017.01.021},
   Abstract = {To assess changes in quality of care for children at risk
             for autism spectrum disorders (ASD) due to process
             improvement and implementation of a digital screening
             form.The process of screening for ASD was studied in an
             academic primary care pediatrics clinic before and after
             implementation of a digital version of the Modified
             Checklist for Autism in Toddlers - Revised with Follow-up
             with automated risk assessment. Quality metrics included
             accuracy of documentation of screening results and
             appropriate action for positive screens (secondary screening
             or referral). Participating physicians completed pre- and
             postintervention surveys to measure changes in attitudes
             toward feasibility and value of screening for ASD. Evidence
             of change was evaluated with statistical process control
             charts and χ2 tests.Accurate documentation in the
             electronic health record of screening results increased from
             54% to 92% (38% increase, 95% CI 14%-64%) and appropriate
             action for children screening positive increased from 25% to
             85% (60% increase, 95% CI 35%-85%). A total of 90% of
             participating physicians agreed that the transition to a
             digital screening form improved their clinical assessment of
             autism risk.Implementation of a tablet-based digital version
             of the Modified Checklist for Autism in Toddlers - Revised
             with Follow-up led to improved quality of care for children
             at risk for ASD and increased acceptability of screening for
             ASD. Continued efforts towards improving the process of
             screening for ASD could facilitate rapid, early diagnosis of
             ASD and advance the accuracy of studies of the impact of
             screening.},
   Doi = {10.1016/j.jpeds.2017.01.021},
   Key = {fds323858}
}


%% Egner, Tobias   
@article{fds332979,
   Author = {Bejjani, C and Zhang, Z and Egner, T},
   Title = {Control by association: Transfer of implicitly primed
             attentional states across linked stimuli.},
   Journal = {Psychonomic Bulletin and Review},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.3758/s13423-018-1445-6},
   Abstract = {Although cognitive control has traditionally been viewed in
             opposition to associative learning, recent studies show that
             people can learn to link particular stimuli with specific
             cognitive control states (e.g., high attentional
             selectivity). Here, we tested whether such learned
             stimulus-control associations can transfer across
             paired-associates. In the Stimulus-Stimulus (S-S)
             Association phase, specific face or house images repeatedly
             preceded the presentation of particular scene stimuli,
             creating paired face/house-scene associates in memory. The
             Stimulus-Control (S-C) Association phase then associated
             these scenes with different attentional control states by
             probabilistically biasing specific scenes to mostly precede
             either congruent or incongruent trials in a Stroop task.
             Finally, in the Stimulus-Control Transfer (S-CT) phase, the
             faces and houses from the S-S phase preceded Stroop trials
             but were not predictive of congruency, testing whether
             stimulus-control associations would transfer from scenes to
             their associated face/house stimuli. In Experiments 1 and 3,
             we found that learned implicit stimulus-control associations
             could transfer across closely linked cues, and in Experiment
             2, we showed that this transfer depended on the memory
             associations formed in the S-S phase. While this form of
             transfer learning has previously been demonstrated for
             stimulus-reward associations, the present study provides the
             first evidence for the associative transfer of
             stimulus-control associations across arbitrarily linked
             stimuli. This work demonstrates how people can learn to
             implicitly adapt their processing strategies in a flexible
             context-dependent manner and establishes a novel learning
             mechanism supporting the generalization of cognitive
             control.},
   Doi = {10.3758/s13423-018-1445-6},
   Key = {fds332979}
}

@article{fds331649,
   Author = {Muhle-Karbe, PS and Jiang, J and Egner, T},
   Title = {Causal Evidence for Learning-Dependent Frontal Lobe
             Contributions to Cognitive Control.},
   Journal = {The Journal of neuroscience : the official journal of the
             Society for Neuroscience},
   Volume = {38},
   Number = {4},
   Pages = {962-973},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1523/jneurosci.1467-17.2017},
   Abstract = {The lateral prefrontal cortex (LPFC) plays a central role in
             the prioritization of sensory input based on task relevance.
             Such top-down control of perception is of fundamental
             importance in goal-directed behavior, but can also be costly
             when deployed excessively, necessitating a mechanism that
             regulates control engagement to align it with changing
             environmental demands. We have recently introduced the
             "flexible control model" (FCM), which explains this
             regulation as resulting from a self-adjusting
             reinforcement-learning mechanism that infers latent
             statistical structure in dynamic task environments to
             predict forthcoming states. From this perspective,
             LPFC-based control is engaged as a function of anticipated
             cognitive demand, a notion for which we previously obtained
             correlative neuroimaging evidence. Here, we put this
             hypothesis to a rigorous, causal test by combining the FCM
             with a transcranial magnetic stimulation (TMS) intervention
             that transiently perturbed the LPFC. Human participants
             (male and female) completed a nonstationary version of the
             Stroop task with dynamically changing probabilities of
             conflict between task-relevant and task-irrelevant stimulus
             features. TMS was given on each trial before stimulus onset
             either over the LPFC or over a control site. In the control
             condition, we observed adaptive performance fluctuations
             consistent with demand predictions that were inferred from
             recent and remote trial history and effectively captured by
             our model. Critically, TMS over the LPFC eliminated these
             fluctuations while leaving basic cognitive and motor
             functions intact. These results provide causal evidence for
             a learning-based account of cognitive control and delineate
             the nature of the signals that regulate top-down biases over
             stimulus processing.SIGNIFICANCE STATEMENT A core function
             of the human prefrontal cortex is to control the signal flow
             in sensory brain regions to prioritize processing of
             task-relevant information. Abundant work suggests that such
             control is flexibly recruited to accommodate dynamically
             changing environmental demands, yet the nature of the
             signals that serve to engage control remains unknown. Here,
             we combined computational modeling with noninvasive brain
             stimulation to show that changes in control engagement are
             captured by a self-adjusting reinforcement-learning
             mechanism that tracks changing environmental statistics to
             predict forthcoming processing demands and that transient
             perturbation of the prefrontal cortex abolishes these
             adjustments. These findings delineate the learning signals
             that underpin adaptive engagement of prefrontal control
             functions and provide causal evidence for their relevance in
             behavioral control.},
   Doi = {10.1523/jneurosci.1467-17.2017},
   Key = {fds331649}
}

@article{fds328427,
   Author = {Kiyonaga, A and Dowd, EW and Egner, T},
   Title = {Neural Representation of Working Memory Content Is Modulated
             by Visual Attentional Demand.},
   Journal = {Journal of Cognitive Neuroscience},
   Volume = {29},
   Number = {12},
   Pages = {2011-2024},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1162/jocn_a_01174},
   Abstract = {Recent theories assert that visual working memory (WM)
             relies on the same attentional resources and sensory
             substrates as visual attention to external stimuli.
             Behavioral studies have observed competitive tradeoffs
             between internal (i.e., WM) and external (i.e., visual)
             attentional demands, and neuroimaging studies have revealed
             representations of WM content as distributed patterns of
             activity within the same cortical regions engaged by
             perception of that content. Although a key function of WM is
             to protect memoranda from competing input, it remains
             unknown how neural representations of WM content are
             impacted by incoming sensory stimuli and concurrent
             attentional demands. Here, we investigated how neural
             evidence for WM information is affected when attention is
             occupied by visual search-at varying levels of
             difficulty-during the delay interval of a WM match-to-sample
             task. Behavioral and fMRI analyses suggested that WM
             maintenance was impacted by the difficulty of a concurrent
             visual task. Critically, multivariate classification
             analyses of category-specific ventral visual areas revealed
             a reduction in decodable WM-related information when
             attention was diverted to a visual search task, especially
             when the search was more difficult. This study suggests that
             the amount of available attention during WM maintenance
             influences the detection of sensory WM representations.},
   Doi = {10.1162/jocn_a_01174},
   Key = {fds328427}
}

@article{fds326206,
   Author = {Chiu, Y-C and Egner, T},
   Title = {Cueing cognitive flexibility: Item-specific learning of
             switch readiness.},
   Journal = {Journal of Experimental Psychology: Human Perception and
             Performance},
   Volume = {43},
   Number = {12},
   Pages = {1950-1960},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1037/xhp0000420},
   Abstract = {The rich behavioral repertoire of the human species derives
             from our ability to flexibly reconfigure processing
             strategies (task sets) in response to changing requirements.
             This updating of task sets is effortful, as reflected by
             longer response times when switching a task than repeating
             it (switch costs). However, some recent data suggest that
             switch costs can be reduced by cueing switch readiness
             bottom-up, by associating particular stimuli with frequent
             switch requirements. This type of "stimulus-control (S-C)
             learning" would be highly adaptive, as it combines the speed
             of automatic (bottom-up) processing with the flexibility and
             generalizability of controlled (top-down) processing.
             However, it is unclear whether S-C learning of switch
             readiness is truly possible, and what the underlying
             mechanisms are. Here we address these questions by pairing
             specific stimuli with a need to update task-sets either
             frequently or rarely. In all 3 experiments, we observe
             robust item-specific switch probability (ISSP) effects as
             revealed by smaller switch costs for frequent switch items
             than for rare switch items. By including a neutral
             condition, we also show that the ISSP effect is primarily
             driven by S-C learning reducing switch costs in frequent
             switch items. Furthermore, by employing 3 tasks in
             Experiment 3, we establish that the ISSP effect reflects an
             enhancement of general switch readiness, rather than of the
             readiness to switch to a specific alternate task. These
             results firmly establish that switch readiness is malleable
             by item-specific S-C learning processes, documenting that a
             generalizable state of cognitive flexibility can be primed
             by a bottom-up stimulus. (PsycINFO Database
             Record},
   Doi = {10.1037/xhp0000420},
   Key = {fds326206}
}

@article{fds331138,
   Author = {Whitehead, PS and Egner, T},
   Title = {Cognitive Control Over Prospective Task-set
             Interference},
   Journal = {Journal of Experimental Psychology: Human Perception and
             Performance},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1037/xhp0000493},
   Abstract = {© 2017 APA, all rights reserved). Recent studies have
             demonstrated that maintaining task-sets in working memory
             (WM) for prospective implementation can interfere with
             performance on an intervening task when the same stimulus
             requires incompatible responses in the ongoing versus the
             prospective task. This prospective task-set interference
             effect has previously been conceptualized as an obligatory
             process, resulting from instruction-based reflexivity (IBR).
             However, the extent to which strategic control can be
             exerted over interference in ongoing behavior from
             prospective task-sets held in WM has heretofore not been
             tested directly. To probe for strategic control over this
             effect, the authors conducted 3 experiments using a common
             inducer-diagnostic task design that manipulated the
             proportion compatibility of trials in the ongoing task. They
             hypothesized that if prospective task-set interference were
             malleable by control, participants would suppress the
             influence of the prospective set on ongoing processing when
             incompatible trials are frequent. Consistent with this
             prediction, the results show that prospective task-set
             interference is subject to modulation by strategic control
             such that the magnitude of interference is reduced,
             eliminated, or reversed in the presence of frequent
             incompatible trials. Thus, the influence on ongoing behavior
             of a prospective task-set held in WM is not obligatory, but
             subject to strategic control. (PsycINFO Database
             Record},
   Doi = {10.1037/xhp0000493},
   Key = {fds331138}
}

@article{fds329983,
   Author = {Qiao, L and Zhang, L and Chen, A and Egner, T},
   Title = {Dynamic Trial-by-Trial Recoding of Task-Set Representations
             in the Frontoparietal Cortex Mediates Behavioral
             Flexibility.},
   Journal = {The Journal of neuroscience : the official journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {45},
   Pages = {11037-11050},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1523/jneurosci.0935-17.2017},
   Abstract = {Cognitive flexibility forms the core of the extraordinary
             ability of humans to adapt, but the precise neural
             mechanisms underlying our ability to nimbly shift between
             task sets remain poorly understood. Recent functional
             magnetic resonance imaging (fMRI) studies employing
             multivoxel pattern analysis (MVPA) have shown that a
             currently relevant task set can be decoded from activity
             patterns in the frontoparietal cortex, but whether these
             regions support the dynamic transformation of task sets from
             trial to trial is not clear. Here, we combined a cued
             task-switching protocol with human (both sexes) fMRI, and
             harnessed representational similarity analysis (RSA) to
             facilitate a novel assessment of trial-by-trial changes in
             neural task-set representations. We first used MVPA to
             define task-sensitive frontoparietal and visual regions and
             found that neural task-set representations on switch trials
             are less stably encoded than on repeat trials. We then
             exploited RSA to show that the neural representational
             pattern dissimilarity across consecutive trials is greater
             for switch trials than for repeat trials, and that the
             degree of this pattern dissimilarity predicts behavior.
             Moreover, the overall neural pattern of representational
             dissimilarities followed from the assumption that repeating
             sets, compared with switching sets, results in stronger
             neural task representations. Finally, when moving from cue
             to target phase within a trial, pattern dissimilarities
             tracked the transformation from previous-trial task
             representations to the currently relevant set. These results
             provide neural evidence for the longstanding assumptions of
             an effortful task-set reconfiguration process hampered by
             task-set inertia, and they demonstrate that frontoparietal
             and stimulus processing regions support "dynamic adaptive
             coding," flexibly representing changing task sets in a
             trial-by-trial fashion.SIGNIFICANCE STATEMENT Humans can
             fluently switch between different tasks, reflecting an
             ability to dynamically configure "task sets," rule
             representations that link stimuli to appropriate responses.
             Recent studies show that neural signals in frontal and
             parietal brain regions can tell us which of two tasks a
             person is currently performing. However, it is not known
             whether these regions are also involved in dynamically
             reconfiguring task-set representations when switching
             between tasks. Here we measured human brain activity during
             task switching and tracked the similarity of neural task-set
             representations from trial to trial. We show that frontal
             and parietal brain regions flexibly recode changing task
             sets in a trial-by-trial fashion, and that task-set
             similarity over consecutive trials predicts
             behavior.},
   Doi = {10.1523/jneurosci.0935-17.2017},
   Key = {fds329983}
}

@article{fds328321,
   Author = {Oh-Descher, H and Beck, JM and Ferrari, S and Sommer, MA and Egner,
             T},
   Title = {Probabilistic inference under time pressure leads to a
             cortical-to-subcortical shift in decision evidence
             integration.},
   Journal = {NeuroImage},
   Volume = {162},
   Pages = {138-150},
   Publisher = {Elsevier},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2017.08.069},
   Abstract = {Real-life decision-making often involves combining multiple
             probabilistic sources of information under finite time and
             cognitive resources. To mitigate these pressures, people
             "satisfice", foregoing a full evaluation of all available
             evidence to focus on a subset of cues that allow for fast
             and "good-enough" decisions. Although this form of
             decision-making likely mediates many of our everyday
             choices, very little is known about the way in which the
             neural encoding of cue information changes when we satisfice
             under time pressure. Here, we combined human functional
             magnetic resonance imaging (fMRI) with a probabilistic
             classification task to characterize neural substrates of
             multi-cue decision-making under low (1500 ms) and high
             (500 ms) time pressure. Using variational Bayesian
             inference, we analyzed participants' choices to track and
             quantify cue usage under each experimental condition, which
             was then applied to model the fMRI data. Under low time
             pressure, participants performed near-optimally,
             appropriately integrating all available cues to guide
             choices. Both cortical (prefrontal and parietal cortex) and
             subcortical (hippocampal and striatal) regions encoded
             individual cue weights, and activity linearly tracked
             trial-by-trial variations in the amount of evidence and
             decision uncertainty. Under increased time pressure,
             participants adaptively shifted to using a satisficing
             strategy by discounting the least informative cue in their
             decision process. This strategic change in decision-making
             was associated with an increased involvement of the
             dopaminergic midbrain, striatum, thalamus, and cerebellum in
             representing and integrating cue values. We conclude that
             satisficing the probabilistic inference process under time
             pressure leads to a cortical-to-subcortical shift in the
             neural drivers of decisions.},
   Doi = {10.1016/j.neuroimage.2017.08.069},
   Key = {fds328321}
}

@article{fds333713,
   Author = {Oh-Descher, H and Beck, J and Ferrari, S and Sommer, MA and Egner,
             T},
   Title = {Probabilistic inference under time pressure leads to a
             cortical-to-subcortical shift in decision evidence
             integration},
   Publisher = {Elsevier},
   Year = {2017},
   Month = {August},
   Abstract = {Real-life decision-making often involves combining multiple
             probabilistic sources of information under finite time and
             cognitive resources. To mitigate these pressures, people
             “satisfice”, foregoing a full evaluation of all
             available evidence to focus on a subset of cues that allow
             for fast and “good-enough” decisions. Although this form
             of decision-making likely mediates many of our everyday
             choices, very little is known about the way in which the
             neural encoding of cue information changes when we satisfice
             under time pressure. Here, we combined human functional
             magnetic resonance imaging (fMRI) with a probabilistic
             classification task to characterize neural substrates of
             multi-cue decision-making under low (1500 ms) and high (500
             ms) time pressure. Using variational Bayesian inference, we
             analyzed participants’ choices to track and quantify cue
             usage under each experimental condition, which was then
             applied to model the fMRI data. Under low time pressure,
             participants performed near-optimally, appropriately
             integrating all available cues to guide choices. Both
             cortical (prefrontal and parietal cortex) and subcortical
             (hippocampal and striatal) regions encoded individual cue
             weights, and activity linearly tracked trial-by-trial
             variations in amount of evidence and decision uncertainty.
             Under increased time pressure, participants adaptively
             shifted to using a satisficing strategy by discounting the
             least informative cue in their decision process. This
             strategic change in decision-making was associated with an
             increased involvement of the dopaminergic midbrain,
             striatum, thalamus, and cerebellum in representing and
             integrating cue values. We conclude that satisficing the
             probabilistic inference process under time pressure leads to
             a cortical-to-subcortical shift in the neural drivers of
             decisions.},
   Key = {fds333713}
}

@article{fds328117,
   Author = {Korb, FM and Jiang, J and King, JA and Egner, T},
   Title = {Hierarchically Organized Medial Frontal Cortex-Basal Ganglia
             Loops Selectively Control Task- and Response-Selection.},
   Journal = {The Journal of neuroscience : the official journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {33},
   Pages = {7893-7905},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1523/jneurosci.3289-16.2017},
   Abstract = {Adaptive behavior requires context-sensitive configuration
             of task-sets that specify time-varying stimulus-response
             mappings. Intriguingly, response time costs associated with
             changing task-sets and motor responses are known to be
             strongly interactive: switch costs at the task level are
             small in the presence of a response-switch but large when
             accompanied by a response-repetition, and vice versa for
             response-switch costs. The reasons behind this well known
             interdependence between task- and response-level control
             processes are currently not well understood. Here, we
             formalized and tested a model assuming a hierarchical
             organization of superordinate task-set and subordinate
             response-set selection processes to account for this effect.
             The model was found to successfully explain the full range
             of behavioral task- and response-switch costs across first
             and second order trial transitions. Using functional
             magnetic resonance imaging (fMRI) in healthy humans, we then
             characterized the neural circuitry mediating these effects.
             We found that presupplementary motor area (preSMA) activity
             tracked task-set control costs, SMA activity tracked
             response-set control costs, and basal ganglia (BG) activity
             mirrored the interaction between task- and response-set
             regulation processes that characterized participants'
             response times. A subsequent fMRI-guided transcranial
             magnetic stimulation experiment confirmed dissociable roles
             of the preSMA and SMA in determining response costs.
             Together, these data provide evidence for a hierarchical
             organization of posterior medial frontal cortex and its
             interaction with the BG, where a superordinate preSMA-BG
             loop establishes task-set selection, which imposes a
             (unidirectional) constraint on a subordinate SMA-BG loop
             that determines response-selection, resulting in the
             characteristic interdependence in task- and response-switch
             costs in behavior.SIGNIFICANCE STATEMENT The ability to use
             context-sensitive task-sets to guide our responses is
             central to human adaptive behavior. Task and response
             selection are strongly interactive: it is more difficult to
             repeat a response in the context of a changing task-set, and
             vice versa. However, the neurocognitive architecture giving
             rise to this interdependence is currently not understood.
             Here we use modeling, neuroimaging, and noninvasive
             neurostimulation to show that this phenomenon derives from a
             hierarchical organization of posterior medial frontal cortex
             and its interaction with the basal ganglia, where a more
             anterior corticostriatal loop establishes task-set
             selection, which constrains a more posterior loop
             responsible for response-selection. These data provide a
             neural explanation for a key behavioral signature of human
             cognitive control.},
   Doi = {10.1523/jneurosci.3289-16.2017},
   Key = {fds328117}
}

@article{fds324879,
   Author = {Dowd, EW and Pearson, JM and Egner, T},
   Title = {Decoding working memory content from attentional
             biases.},
   Journal = {Psychonomic Bulletin and Review},
   Volume = {24},
   Number = {4},
   Pages = {1252-1260},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.3758/s13423-016-1204-5},
   Abstract = {What we are currently thinking influences where we attend.
             The finding that active maintenance of visual items in
             working memory (WM) biases attention toward memory-matching
             objects-even when WM content is irrelevant for attentional
             goals-suggests a tight link between WM and attention. To
             test whether this link is reliable enough to infer specific
             WM content from measures of attentional bias, we applied
             multivariate pattern classification techniques to response
             times from an unrelated visual search task during a WM
             delay. Single-trial WM content was successfully decoded from
             incidental attentional bias within an individual,
             highlighting the specificity and reliability of the
             WM-attention link. Furthermore, classifiers trained on a
             group of individuals predicted WM content in another,
             completely independent individual-implying a shared
             cognitive mechanism of memory-driven attentional bias. The
             existence of such classifiers demonstrates that memory-based
             attentional bias is both a robust and generalizable probe of
             WM.},
   Doi = {10.3758/s13423-016-1204-5},
   Key = {fds324879}
}

@book{fds328428,
   Author = {Egner, T},
   Title = {The Wiley Handbook of Cognitive Control},
   Pages = {656 pages},
   Publisher = {Wiley-Blackwell},
   Year = {2017},
   Month = {March},
   ISBN = {1118920546},
   Abstract = {Covering basic theory, new research, and intersections with
             adjacent fields, this is the first comprehensive reference
             work on cognitive control – our ability to use internal
             goals to guide thought and behavior.},
   Key = {fds328428}
}

@article{fds318684,
   Author = {Braem, S and King, JA and Korb, FM and Krebs, RM and Notebaert, W and Egner, T},
   Title = {The Role of Anterior Cingulate Cortex in the Affective
             Evaluation of Conflict.},
   Journal = {Journal of Cognitive Neuroscience},
   Volume = {29},
   Number = {1},
   Pages = {137-149},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1162/jocn_a_01023},
   Abstract = {An influential theory of ACC function argues that this brain
             region plays a crucial role in the affective evaluation of
             performance monitoring and control demands. Specifically,
             control-demanding processes such as response conflict are
             thought to be registered as aversive signals by ACC, which
             in turn triggers processing adjustments to support avoidance
             learning. In support of conflict being treated as an
             aversive event, recent behavioral studies demonstrated that
             incongruent (i.e., conflict inducing), relative to
             congruent, stimuli can speed up subsequent negative,
             relative to positive, affective picture processing. Here, we
             used fMRI to investigate directly whether ACC activity in
             response to negative versus positive pictures is modulated
             by preceding control demands, consisting of conflict and
             task-switching conditions. The results show that negative,
             relative to positive, pictures elicited higher ACC
             activation after congruent, relative to incongruent, trials,
             suggesting that ACC's response to negative (positive)
             pictures was indeed affectively primed by incongruent
             (congruent) trials. Interestingly, this pattern of results
             was observed on task repetitions but disappeared on task
             alternations. This study supports the proposal that conflict
             induces negative affect and is the first to show that this
             affective signal is reflected in ACC activation.},
   Doi = {10.1162/jocn_a_01023},
   Key = {fds318684}
}

@article{fds323840,
   Author = {Chiu, Y-C and Jiang, J and Egner, T},
   Title = {The Caudate Nucleus Mediates Learning of Stimulus-Control
             State Associations.},
   Journal = {The Journal of neuroscience : the official journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {4},
   Pages = {1028-1038},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1523/jneurosci.0778-16.2016},
   Abstract = {A longstanding dichotomy in cognitive psychology and
             neuroscience pits controlled, top-down driven behavior
             against associative, bottom-up driven behavior, where
             cognitive control processes allow us to override
             well-learned stimulus-response (S-R) associations. By
             contrast, some previous studies have raised the intriguing
             possibility of an integration between associative and
             controlled processing in the form of stimulus-control state
             (S-C) associations, the learned linkage of specific stimuli
             to particular control states, such as high attentional
             selectivity. The neural machinery mediating S-C learning
             remains poorly understood, however. Here, we combined human
             functional magnetic resonance imaging (fMRI) with a
             previously developed Stroop protocol that allowed us to
             dissociate reductions in Stroop interference based on S-R
             learning from those based on S-C learning. We modeled
             subjects' acquisition of S-C and S-R associations using an
             associative learning model and then used trial-by-trial S-C
             and S-R prediction error (PE) estimates in model-based
             behavioral and fMRI analyses. We found that PE estimates
             derived from S-C and S-R associations accounted for the
             reductions in behavioral Stroop interference effects in the
             S-C and S-R learning conditions, respectively. Moreover,
             model-based fMRI analyses identified the caudate nucleus as
             the key structure involved in selectively updating
             stimulus-control state associations. Complementary analyses
             also revealed a greater reliance on parietal cortex when
             using the learned S-R versus S-C associations to minimize
             Stroop interference. These results support the emerging view
             that generalizable control states can become associated with
             specific bottom-up cues, and they place the caudate nucleus
             of the dorsal striatum at the center of the neural
             stimulus-control learning machinery.Previous behavioral
             studies have demonstrated that control states, for instance,
             heightened attentional selectivity, can become directly
             associated with, and subsequently retrieved by, particular
             stimuli, thus breaking down the traditional dichotomy
             between top-down and bottom-up driven behavior. However, the
             neural mechanisms underlying this type of stimulus-control
             learning remain poorly understood. We therefore combined
             noninvasive human neuroimaging with a task that allowed us
             to dissociate the acquisition of stimulus-control
             associations from that of stimulus-response associations.
             The results revealed the caudate nucleus as the key brain
             structure involved in selectively driving stimulus-control
             learning. These data represent the first identification of
             the neural mechanisms of stimulus-specific control
             associations, and they significantly extend current
             conceptions of the type of learning processes mediated by
             the caudate.},
   Doi = {10.1523/jneurosci.0778-16.2016},
   Key = {fds323840}
}

@article{fds323485,
   Author = {Mansouri, FA and Egner, T and Buckley, MJ},
   Title = {Monitoring Demands for Executive Control: Shared Functions
             between Human and Nonhuman Primates.},
   Journal = {Trends in Neurosciences},
   Volume = {40},
   Number = {1},
   Pages = {15-27},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.tins.2016.11.001},
   Abstract = {Fifteen years ago, an influential model proposed that the
             human dorsal anterior cingulate cortex (dACC) detects
             conflict and induces adaptive control of behavior. Over the
             years support for this model has been mixed, in particular
             due to divergent findings in human versus nonhuman primates.
             We here review recent findings that suggest greater
             commonalities across species. These include equivalent
             behavioral consequences of conflict and similar neuronal
             signals in the dACC, but also a common failure of dACC
             lesions to reliably abolish conflict-driven behavior. We
             conclude that conflict might be one among many drivers of
             adjustments in executive control and that the ACC might be
             just one component of overlapping distributed systems
             involved in context-dependent learning and behavioral
             control.},
   Doi = {10.1016/j.tins.2016.11.001},
   Key = {fds323485}
}


%% Ehrensaft, Miriam K   
@article{fds329143,
   Author = {Ehrensaft, MK and Westfall, HK and Niolon, PH and Lopez, T and Kamboukos, D and Huang, K-Y and Brotman, LM},
   Title = {Can a Parenting Intervention to Prevent Early Conduct
             Problems Interrupt Girls' Risk for Intimate Partner Violence
             10 Years Later?},
   Journal = {Prevention Science},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1007/s11121-017-0831-z},
   Abstract = {This study tests whether a parenting intervention for
             families of preschoolers at risk for conduct problems can
             prevent later risk for intimate partner violence (IPV).
             Ninety-nine preschoolers at familial risk for conduct
             problems were randomly assigned to intervention or control
             conditions. Ten years later, 45 preschoolers and 43 of their
             siblings completed an assessment of their romantic
             relationships, including measures of physical and
             psychological IPV. The study focuses on the 54 females,
             including targets (n = 27) and siblings (n = 27) who
             participated in a 10-year follow-up (M age = 16.5,
             SD = 5.2, range = 10-28). Using an intent-to-treat (ITT)
             design, multivariate regressions suggest that females from
             families randomly assigned to intervention in early
             childhood scored lower than those in the control condition
             on perceptions of dating violence as normative, beliefs
             about IPV prevalence, exposure to IPV in their own peer
             group, and expected sanction behaviors for IPV perpetration
             and victimization. Findings suggest that early parenting
             intervention may reduce association of high-risk females
             with aggressive peers and partners in adolescence.},
   Doi = {10.1007/s11121-017-0831-z},
   Key = {fds329143}
}

@article{fds319604,
   Author = {Ehrensaft, MK and Knous-Westfall, H and Cohen,
             P},
   Title = {Long-term influence of intimate partner violence and
             parenting practices on offspring trauma symptoms.},
   Journal = {Psychology of Violence},
   Volume = {7},
   Number = {2},
   Pages = {296-305},
   Year = {2017},
   url = {http://dx.doi.org/10.1037/a0040168},
   Doi = {10.1037/a0040168},
   Key = {fds319604}
}


%% Fairbank, John A.   
@article{fds331314,
   Author = {Martindale, SL and Farrell-Carnahan, LV and Ulmer, CS and Kimbrel,
             NA and McDonald, SD and Rowland, JA and VA Mid-Atlantic MIRECC
             Registry Workgroup},
   Title = {Sleep quality in returning veterans: The influence of mild
             traumatic brain injury.},
   Journal = {Rehabilitation Psychology},
   Volume = {62},
   Number = {4},
   Pages = {563-570},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1037/rep0000159},
   Abstract = {Sleep disturbance is a key behavioral health concern among
             Iraq and Afghanistan era veterans and is a frequent
             complaint among veterans with a history of mild traumatic
             brain injury (mTBI). Currently, it is unclear whether sleep
             disturbance is a core sequelae of mTBI or if it may be
             related to other behavioral health conditions that are
             commonly present in postdeployment veterans. The purpose of
             this study was to determine if history of mTBI is associated
             with poor sleep quality beyond combat exposure and
             behavioral health concerns, including posttraumatic stress
             disorder (PTSD), mood disorders, anxiety disorders, and
             substance use disorders.Participants included 527 veterans
             who deployed after September 11, 2001, in support of the
             wars in Iraq and Afghanistan. Participants completed the
             Structured Clinical Interview for DSM-IV Disorders, Combat
             Exposure Scale, a structured clinical TBI interview, and the
             Pittsburgh Sleep Quality Index.Deployment-related mTBI was
             associated with poor sleep quality independent of combat
             exposure, PTSD, mood disorders, anxiety disorders, and
             substance use disorders (B = 1.84, p = .001) an average of 6
             years after the injury event. No interaction effects between
             deployment-related mTBI and combat exposure, PTSD, mood
             disorders, anxiety disorders, or substance use disorders on
             sleep quality were detected.Veterans with a history of mTBI
             sustained during deployment reported significantly poorer
             sleep quality than veterans without history of mTBI, even
             when considering combat exposure and behavioral health
             issues. Clinicians should consider mTBI as a potential
             contributor to sleep problems, even years after an injury
             event. (PsycINFO Database Record},
   Doi = {10.1037/rep0000159},
   Key = {fds331314}
}

@article{fds331181,
   Author = {Naylor, JC and Ryan Wagner and H and Brancu, M and Shepherd-Banigan, M and Elbogen, E and Kelley, M and Fecteau, T and Goldstein, K and Kimbrel,
             NA and Marx, CE and VA Mid-Atlantic MIRECC Work Group, and VA
             Mid-Atlantic MIRECC Women Veterans Work Group, and Strauss,
             JL},
   Title = {Self-Reported Pain in Male and Female Iraq/Afghanistan-Era
             Veterans: Associations with Psychiatric Symptoms and
             Functioning.},
   Journal = {Pain Medicine},
   Volume = {18},
   Number = {9},
   Pages = {1658-1667},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1093/pm/pnw308},
   Abstract = {To examine pain symptoms and co-occurring psychiatric and
             functional indices in male and female Iraq/Afghanistan-era
             veterans.Self-reported data collection and interviews of
             Iraq/Afghanistan-era veterans who participated in a
             multisite study of postdeployment mental health.Veterans
             were enrolled at one of four participating VA sites.Two
             thousand five hundred eighty-seven male and 662 female
             Iraq/Afghanistan-era veterans.Nonparametric Wilcoxon rank
             tests examined differences in pain scores between male and
             female veterans. Chi-square tests assessed differences
             between male and female veterans in the proportion of
             respondents endorsing moderate to high levels of pain vs no
             pain. Multilevel regression analyses evaluated the effect of
             pain on a variety of psychiatric and functional
             measures.Compared with males, female veterans reported
             significantly higher mean levels of headache ( P
              < 0.0001), muscle soreness ( P  < 0.008), and total
             pain ( P  < 0.0001), and were more likely to report the
             highest levels of headache ( P  < 0.0001) and muscle
             soreness ( P  < 0.0039). The presence of pain symptoms
             in Iraq/Afghanistan-era veterans was positively associated
             with psychiatric comorbidity and negatively associated with
             psychosocial functioning. There were no observed gender
             differences in psychiatric and functional indices when
             levels of pain were equated.Although female
             Iraq/Afghanistan-era veterans reported higher levels of pain
             than male veterans overall, male and female veterans
             experienced similar levels of psychiatric and functional
             problems at equivalent levels of reported pain. These
             findings suggest that pain-associated psychological and
             functional impacts are comparable and consequential for both
             male and female veterans.},
   Doi = {10.1093/pm/pnw308},
   Key = {fds331181}
}

@article{fds327056,
   Author = {Brancu, M and Wagner, HR and Morey, RA and Beckham, JC and Calhoun, PS and Tupler, LA and Marx, CE and Taber, KH and Hurley, RA and Rowland, J and McDonald, SD and Hoerle, JM and Moore, SD and Kudler, HS and Weiner, RD and VA Mid-Atlantic MIRECC Workgroup, and Fairbank,
             JA},
   Title = {The Post-Deployment Mental Health (PDMH) study and
             repository: A multi-site study of US Afghanistan and Iraq
             era veterans.},
   Journal = {International Journal of Methods in Psychiatric
             Research},
   Volume = {26},
   Number = {3},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1002/mpr.1570},
   Abstract = {The United States (US) Department of Veterans Affairs (VA)
             Mid-Atlantic Mental Illness Research, Education, and
             Clinical Center (MIRECC) Post-Deployment Mental Health
             (PDMH) multi-site study examines post-deployment mental
             health in US military Afghanistan/Iraq-era veterans. The
             study includes the comprehensive behavioral health
             characterization of over 3600 study participants and the
             genetic, metabolomic, neurocognitive, and neuroimaging data
             for many of the participants. The study design also
             incorporates an infrastructure for a data repository to
             re-contact participants for follow-up studies. The
             overwhelming majority (94%) of participants consented to be
             re-contacted for future studies, and our recently completed
             feasibility study indicates that 73-83% of these
             participants could be reached successfully for enrollment
             into longitudinal follow-up investigations. Longitudinal
             concurrent cohort follow-up studies will be conducted (5-10+
             years post-baseline) to examine predictors of illness
             chronicity, resilience, recovery, functional outcome, and
             other variables, and will include neuroimaging,
             genetic/epigenetic, serum biomarker, and neurocognitive
             studies, among others. To date, the PDMH study has generated
             more than 35 publications from the baseline data and the
             repository has been leveraged in over 20 publications from
             follow-up studies drawing from this cohort. Limitations that
             may affect data collection for a longitudinal follow-up
             study are also presented.},
   Doi = {10.1002/mpr.1570},
   Key = {fds327056}
}

@article{fds326331,
   Author = {McMaster, HS and LeardMann, CA and Speigle, S and Dillman, DA and Millennium Cohort Family Study Team},
   Title = {An experimental comparison of web-push vs. paper-only survey
             procedures for conducting an in-depth health survey of
             military spouses.},
   Journal = {BMC Medical Research Methodology},
   Volume = {17},
   Number = {1},
   Pages = {73},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1186/s12874-017-0337-1},
   Abstract = {Previous research has found that a "web-push" approach to
             data collection, which involves contacting people by mail to
             request an Internet survey response while withholding a
             paper response option until later in the contact process,
             consistently achieves lower response rates than a
             "paper-only" approach, whereby all respondents are contacted
             and requested to respond by mail.An experiment was designed,
             as part of the Millennium Cohort Family Study, to compare
             response rates, sample representativeness, and cost between
             a web-push and a paper-only approach; each approach
             comprised 3 stages of mail contacts. The invited sample
             (n = 4,935) consisted of spouses married to U.S. Service
             members, who had been serving in the military between 2 and
             5 years as of October, 2011.The web-push methodology
             produced a significantly higher response rate, 32.8%
             compared to 27.8%. Each of the 3 stages of postal contact
             significantly contributed to response for both treatments
             with 87.1% of the web-push responses received over the
             Internet. The per-respondent cost of the paper-only
             treatment was almost 40% higher than the web-push treatment
             group. Analyses revealed no meaningfully significant
             differences between treatment groups in representation.These
             results provide evidence that a web-push methodology is more
             effective and less expensive than a paper-only approach
             among young military spouses, perhaps due to their heavy
             reliance on the internet, and we suggest that this approach
             may be more effective with the general population as they
             become more uniformly internet savvy.},
   Doi = {10.1186/s12874-017-0337-1},
   Key = {fds326331}
}

@article{fds326857,
   Author = {Sege, RD and Amaya-Jackson, L and AMERICAN ACADEMY OF PEDIATRICS
             Committee on Child Abuse and Neglect, Council on Foster
             Care and Adoption and Kinship Care and AMERICAN ACADEMY OF CHILD
             AND ADOLESCENT PSYCHIATRY Committee on Child Maltreatment and Violence and NATIONAL CENTER FOR CHILD TRAUMATIC
             STRESS},
   Title = {Clinical Considerations Related to the Behavioral
             Manifestations of Child Maltreatment.},
   Journal = {Pediatrics},
   Volume = {139},
   Number = {4},
   Pages = {e20170100-e20170100},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1542/peds.2017-0100},
   Abstract = {Children who have suffered early abuse or neglect may later
             present with significant health and behavior problems that
             may persist long after the abusive or neglectful environment
             has been remediated. Neurobiological research suggests that
             early maltreatment may result in an altered psychological
             and physiologic response to stressful stimuli, a response
             that deleteriously affects the child's subsequent
             development. Pediatricians can assist caregivers by helping
             them recognize the abused or neglected child's emotional and
             behavioral responses associated with child maltreatment and
             guide them in the use of positive parenting strategies,
             referring the children and families to evidence-based
             therapeutic treatment and mobilizing available community
             resources.},
   Doi = {10.1542/peds.2017-0100},
   Key = {fds326857}
}


%% Fitzsimons, Gavan J.   
@article{fds332784,
   Title = {Erratum},
   Journal = {Journal of Consumer Research},
   Volume = {44},
   Number = {5},
   Pages = {1174-1174},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/jcr/ucx095},
   Doi = {10.1093/jcr/ucx095},
   Key = {fds332784}
}

@article{fds333551,
   Author = {Acikalin, MY and Watson, KK and Fitzsimons, GJ and Platt,
             ML},
   Title = {Rhesus macaques form preferences for brand logos through sex
             and social status based advertising.},
   Journal = {PloS one},
   Volume = {13},
   Number = {2},
   Pages = {e0193055},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1371/journal.pone.0193055},
   Abstract = {Like humans, monkeys value information about sex and status,
             inviting the hypothesis that our susceptibility to these
             factors in advertising arises from shared, ancestral
             biological mechanisms that prioritize social information. To
             test this idea, we asked whether rhesus macaques (Macaca
             mulatta) show choice behavior that is similar to humans in
             response to sex and social status in advertising. Our
             results show that monkeys form preferences for brand logos
             repeatedly paired with images of macaque genitals and high
             status monkeys. Moreover, monkeys sustain preferences for
             these brand logos even though choosing them provided no
             tangible rewards, a finding that cannot be explained by a
             decision mechanism operating solely on material outcomes.
             Together, our results endorse the hypothesis that the power
             of sex and status in advertising emerges from the
             spontaneous engagement of shared, ancestral neural circuits
             that prioritize information useful for navigating the social
             environment. Finally, our results show that simple
             associative conditioning is sufficient to explain the
             formation of preferences for brand logos paired with sexual
             or status-based images.},
   Doi = {10.1371/journal.pone.0193055},
   Key = {fds333551}
}

@article{fds329373,
   Author = {Wu, F and Samper, A and Morales, AC and Fitzsimons,
             GJ},
   Title = {It’s Too Pretty to Use! When and How Enhanced Product
             Aesthetics Discourage Usage and Lower Consumption
             Enjoyment},
   Journal = {Journal of Consumer Research},
   Volume = {44},
   Number = {3},
   Pages = {651-672},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1093/jcr/ucx057},
   Doi = {10.1093/jcr/ucx057},
   Key = {fds329373}
}

@article{fds324822,
   Author = {Zemack-Rugar, Y and Moore, SG and Fitzsimons, GJ},
   Title = {Just do it! Why committed consumers react negatively to
             assertive ads},
   Journal = {Journal of Consumer Psychology},
   Volume = {27},
   Number = {3},
   Pages = {287-301},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.jcps.2017.01.002},
   Doi = {10.1016/j.jcps.2017.01.002},
   Key = {fds324822}
}

@article{fds332785,
   Author = {Brick, DJ and Fitzsimons, GM and Chartrand, TL and Fitzsimons,
             GJ},
   Title = {Coke vs. Pepsi: Brand Compatibility, Relationship Power, and
             Life Satisfaction},
   Journal = {Journal of Consumer Research},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1093/jcr/ucx079},
   Doi = {10.1093/jcr/ucx079},
   Key = {fds332785}
}

@article{fds322756,
   Author = {Brick, DJ and Fitzsimons, GJ},
   Title = {Oppositional brand choice: Using brands to respond to
             relationship frustration},
   Journal = {Journal of Consumer Psychology},
   Volume = {27},
   Number = {2},
   Pages = {257-263},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jcps.2016.10.002},
   Doi = {10.1016/j.jcps.2016.10.002},
   Key = {fds322756}
}


%% Fitzsimons, Gráinne M   
@article{fds332786,
   Author = {Brick, DJ and Fitzsimons, GM and Chartrand, TL and Fitzsimons,
             GJ},
   Title = {Coke vs. Pepsi: Brand Compatibility, Relationship Power, and
             Life Satisfaction},
   Journal = {Journal of Consumer Research},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1093/jcr/ucx079},
   Doi = {10.1093/jcr/ucx079},
   Key = {fds332786}
}

@article{fds332998,
   Author = {Briskin, JL and Kopetz, CE and Fitzsimons, GM and Slatcher,
             RB},
   Title = {For Better or for Worse? Outsourcing Self-Regulation and
             Goal Pursuit},
   Journal = {Social Psychological and Personality Science},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1177/1948550617736112},
   Abstract = {© 2017, The Author(s) 2017. In recent years, there has been
             increasing interest in how close relationships can impact
             personal goal pursuit. Extensive research on social support
             has shown that support often facilitates goal pursuit.
             However, Fitzsimons and Finkel found that perceived partner
             support may actually undermine motivation and decrease goal
             pursuit intentions. In this article, we report three
             well-powered studies (N = 850) that investigated the
             conditions under which romantic partners may bolster or
             undermine goal pursuit. In contrast with the original
             Fitzsimons and Finkel’s findings, the results of these
             studies consistently showed that perceived partner support
             bolsters goal pursuit intentions by increasing goal
             commitment. Implications for successful goal pursuit in the
             context of relationships are discussed.},
   Doi = {10.1177/1948550617736112},
   Key = {fds332998}
}

@article{fds330732,
   Author = {Green, PI and Finkel, EJ and Fitzsimons, GM and Gino,
             F},
   Title = {The energizing nature of work engagement: Toward a new
             need-based theory of work motivation},
   Journal = {Research in Organizational Behavior},
   Volume = {37},
   Pages = {1-18},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.riob.2017.10.007},
   Abstract = {© 2017 Elsevier Ltd We present theory suggesting that
             experiences at work that meet employees’ expectations of
             need fulfillment drive work engagement. Employees have needs
             (e.g., a desire to be authentic) and they also have
             expectations for how their job or their organization will
             fulfill them. We argue that experiences at work that confirm
             employees’ need fulfillment expectations yield a positive
             emotional state that is energizing, and that this energy is
             manifested in employees’ behaviors at work. Our theorizing
             draws on a review of the work engagement literature, in
             which we identify three core characteristics of work
             engagement: (a) a positive emotional state that (b) yields a
             feeling of energy and (c) leads to positive work-oriented
             behaviors. These key themes provide the foundation for
             further theorizing suggesting that interactions at work
             confirm or disconfirm employees’ need fulfillment
             expectations, leading to different levels of engagement. We
             extend our theorizing to argue that confirmation, or
             disconfirmation, of different need expectations will yield
             emotional experience of varying magnitudes, with
             confirmation of approach-oriented need expectations exerting
             stronger effects than the confirmation of avoidance-oriented
             need expectations. We close with a review suggesting that
             organizational contextual features influence the expression
             of these needs, sustaining or undermining the positive
             emotional experiences that fuel work engagement.},
   Doi = {10.1016/j.riob.2017.10.007},
   Key = {fds330732}
}


%% Flanagan, Owen   
@article{fds329381,
   Author = {Gyal, P and Flanagan, O},
   Title = {The role of pain in buddhism: The conquest of
             suffering},
   Pages = {288-296},
   Booktitle = {The Routledge Handbook of Philosophy of Pain},
   Year = {2017},
   Month = {June},
   ISBN = {9781315742205},
   url = {http://dx.doi.org/10.4324/9781315742205},
   Doi = {10.4324/9781315742205},
   Key = {fds329381}
}

@article{fds327006,
   Author = {Flanagan, O},
   Title = {Addiction Doesn’t Exist, But it is Bad for
             You},
   Journal = {Neuroethics},
   Volume = {10},
   Number = {1},
   Pages = {91-98},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s12152-016-9298-z},
   Doi = {10.1007/s12152-016-9298-z},
   Key = {fds327006}
}


%% Fuemmeler, Bernard F.   
@article{fds330367,
   Author = {Sanchez, CE and Barry, C and Sabhlok, A and Russell, K and Majors, A and Kollins, SH and Fuemmeler, BF},
   Title = {Maternal pre-pregnancy obesity and child neurodevelopmental
             outcomes: a meta-analysis.},
   Journal = {Obesity Reviews},
   Volume = {19},
   Number = {4},
   Pages = {464-484},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1111/obr.12643},
   Abstract = {This review examined evidence of the association between
             maternal pre-pregnancy overweight/obesity status and child
             neurodevelopmental outcomes. PubMed and PsycINFO databases
             were systematically searched for empirical studies published
             before April 2017 using keywords related to prenatal obesity
             and children's neurodevelopment. Of 1483 identified papers,
             41 were included in the systematic review, and 32 articles
             representing 36 cohorts were included in the meta-analysis.
             Findings indicated that compared with children of normal
             weight mothers, children whose mothers were overweight or
             obese prior to pregnancy were at increased risk for
             compromised neurodevelopmental outcomes (overweight:
             OR = 1.17, 95% CI [1.11, 1.24], I2  = 65.51; obese:
             OR = 1.51; 95% CI [1.35, 1.69], I2  = 79.63).
             Pre-pregnancy obesity increased the risk of attention
             deficit-hyperactivity disorder (OR = 1.62; 95% CI [1.23,
             2.14], I2  = 70.15), autism spectrum disorder
             (OR = 1.36; 95% CI [1.08, 1.70], I2  = 60.52),
             developmental delay (OR = 1.58; 95% CI [1.39, 1.79], I2
              = 75.77) and emotional/behavioural problems (OR = 1.42;
             95% CI [1.26, 1.59], I2  = 87.74). Given the current
             obesity prevalence among young adults and women of
             childbearing age, this association between maternal obesity
             during pregnancy and atypical child neurodevelopment
             represents a potentially high public health
             burden.},
   Doi = {10.1111/obr.12643},
   Key = {fds330367}
}

@article{fds333243,
   Author = {Cates, JR and Fuemmeler, BF and Diehl, SJ and Stockton, LL and Porter,
             J and Ihekweazu, C and Gurbani, AS and Coyne-Beasley,
             T},
   Title = {Developing a Serious Videogame for Preteens to Motivate HPV
             Vaccination Decision Making: Land of Secret
             Gardens.},
   Journal = {Games for health journal},
   Volume = {7},
   Number = {1},
   Pages = {51-66},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1089/g4h.2017.0002},
   Abstract = {Vaccination against human papillomavirus (HPV) is routinely
             recommended for ages 11-12, yet in 2016 only 49.5% of women
             and 37.5% of men had completed the three-dose series in the
             United States. Offering information and cues to action
             through a serious videogame for preteens may foster HPV
             vaccination awareness, information seeking, and
             communication.An iterative process was used to develop an
             interactive videogame, Land of Secret Gardens. Three focus
             groups were conducted with 16 boys and girls, ages 11-12,
             for input on game design, acceptability, and functioning.
             Two parallel focus groups explored parents' (n = 9)
             perspectives on the game concept. Three researchers
             identified key themes.Preteens wanted a game that is both
             entertaining and instructional. Some parents were skeptical
             that games could be motivational. A back-story about a
             secret garden was developed as a metaphor for a preteen's
             body and keeping it healthy. The goal is to plant a lush
             secret garden and protect the seedlings by treating them
             with a potion when they sprout to keep them healthy as they
             mature. Points to buy seeds and create the potion are earned
             by playing mini-games. Throughout play, players are exposed
             to messaging about HPV and the benefits of the vaccine. Both
             boys and girls liked the garden concept and getting facts
             about HPV. Parents were encouraged to discuss the game with
             their preteens.Within a larger communication strategy,
             serious games could be useful for engaging preteens in
             health decision making about HPV vaccination.},
   Doi = {10.1089/g4h.2017.0002},
   Key = {fds333243}
}

@article{fds331558,
   Author = {Schechter, JC and Fuemmeler, BF and Hoyo, C and Murphy, SK and Zhang,
             JJ and Kollins, SH},
   Title = {Impact of Smoking Ban on Passive Smoke Exposure in Pregnant
             Non-Smokers in the Southeastern United States.},
   Journal = {International journal of environmental research and public
             health},
   Volume = {15},
   Number = {1},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.3390/ijerph15010083},
   Abstract = {Prenatal passive smoke exposure raises risk for negative
             birth outcomes. Legislation regulating public smoking has
             been shown to impact exposure levels, though fewer studies
             involving pregnant women have been conducted within the U.S.
             where bans are inconsistent across regions. This study
             examined the effect of a ban enacted in the southeastern
             U.S. on pregnant women's cotinine levels. Additional
             analyses compared self-reported exposure to cotinine and
             identified characteristics associated with passive exposure.
             Pregnant women (N = 851) were recruited prospectively
             between 2005 and 2011 in North Carolina. Sociodemographic
             and health data were collected via surveys; maternal blood
             samples were assayed for cotinine. Among non-active smokers
             who provided self-report data regarding passive exposure (N
             = 503), 20% were inconsistent with corresponding cotinine.
             Among all non-smokers (N = 668), being unmarried, African
             American, and less educated were each associated with
             greater passive exposure. Controlling for covariates, mean
             cotinine was higher prior to the ban compared to after, F(1,
             640) = 24.65, p < 0.001. Results suggest that banning
             smoking in public spaces may reduce passive smoke exposure
             for non-smoking pregnant women. These data are some of the
             first to examine the impact of legislation on passive smoke
             exposure in pregnant women within the U.S. using a biomarker
             and can inform policy in regions lacking comprehensive
             smoke-free legislation.},
   Doi = {10.3390/ijerph15010083},
   Key = {fds331558}
}

@article{fds333723,
   Author = {Van Voorhees and EE and Dennis, PA and Elbogen, EB and Fuemmeler, B and Neal, LC and Calhoun, PS and Beckham, JC},
   Title = {Characterizing anger-related affect in individuals with
             posttraumatic stress disorder using ecological momentary
             assessment.},
   Journal = {Psychiatry Research},
   Volume = {261},
   Pages = {274-280},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.psychres.2017.12.080},
   Abstract = {This study employed secondary analyses of existing
             ecological momentary assessment (EMA) data to characterize
             hostile and irritable affect in the day-to-day experience of
             52 smokers with, and 65 smokers without, posttraumatic
             stress disorder (PTSD). EMA monitoring occurred over a mean
             of 8.2 days, and participants responded to an average of 2.8
             random prompts/day. Analyses included Wilcoxon rank sum
             tests of group differences, and path analyses of
             cross-lagged multilevel models. Participants with PTSD
             endorsed a significantly higher proportion of total EMA
             entries indicating hostile affect and irritable affect than
             did individuals without PTSD. Cross-lagged analyses
             indicated that over a period of hours, PTSD symptoms
             significantly predicted subsequent hostile and irritable
             affect, but hostile and irritable affect did not predict
             subsequent PTSD symptoms. Findings suggest that day-to-day
             exposure to PTSD-related trauma cues may contribute to
             chronically elevated levels of anger-related affect. Such
             heightened affective arousal may, in turn, underlie an
             increased risk for verbal or physical aggression, as well as
             other health and quality-of-life related impairments
             associated with PTSD. Clinical implications include
             conceptualizing anger treatment in the broader context of
             trauma history and symptoms, and specifically targeting
             physiological arousal and maladaptive hostile cognitions
             triggered by trauma reminders in patients with
             PTSD.},
   Doi = {10.1016/j.psychres.2017.12.080},
   Key = {fds333723}
}

@article{fds331494,
   Author = {Chawla, D and Fuemmeler, B and Benjamin-Neelon, SE and Hoyo, C and Murphy, S and Daniels, JL},
   Title = {Early prenatal vitamin D concentrations and social-emotional
             development in infants.},
   Journal = {Journal of Maternal-Fetal and Neonatal Medicine
             (Informa)},
   Pages = {1-8},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1080/14767058.2017.1408065},
   Abstract = {Many pregnant women in the United States have suboptimal
             vitamin D, but the impact on infant development is unclear.
             Moreover, no pregnancy-specific vitamin D recommendations
             have been widely accepted.Given the ubiquitous expression of
             vitamin D receptors in the brain, we investigated the
             association between early prenatal plasma 25-hydroxyvitamin
             D (25(OH)D) concentrations and children's social and
             emotional development in the Newborn Epigenetic Study, a
             prospective study of pregnancies from 2009 to 2011 in
             Durham, North Carolina.We measured 25(OH)D concentrations in
             first or second trimester plasma samples and categorized
             25(OH)D concentrations into quartiles. Covariates were
             derived from maternal questionnaires. Mothers completed the
             Infant Toddler Social-Emotional Development Assessment when
             children were 12-24 months of age. We used multivariable
             linear regression to evaluate associations between 25(OH)D
             and specific behavior scores, adjusted for season of blood
             draw, maternal age, education, parity, smoking, marital
             status, prepregnancy BMI, and infant gender. We investigated
             effect-measure modification by race/ethnicity.Of the 218
             mother-infant pairs with complete data, Black mothers had
             much lower 25(OH)D concentrations as compared to White and
             Hispanic mothers. After adjustment, lower prenatal 25(OH)D
             was associated with slightly higher (less favorable)
             Internalizing scores among White children, but lower (more
             favorable) Internalizing scores among Black and Hispanic
             children. Lower prenatal 25(OH)D also appears to be
             associated with higher (less favorable) dysregulation
             scores, though only among White and Hispanic children.Though
             imprecise, preliminary results warrant further investigation
             regarding a role for prenatal vitamin D on children's early
             social and emotional development.},
   Doi = {10.1080/14767058.2017.1408065},
   Key = {fds331494}
}

@article{fds329030,
   Author = {McCullough, LE and Miller, EE and Calderwood, LE and Shivappa, N and Steck, SE and Forman, MR and A Mendez and M and Maguire, R and Fuemmeler,
             BF and Kollins, SH and D Bilbo and S and Huang, Z and Murtha, AP and Murphy,
             SK and Hébert, JR and Hoyo, C},
   Title = {Maternal inflammatory diet and adverse pregnancy outcomes:
             Circulating cytokines and genomic imprinting as potential
             regulators?},
   Journal = {Epigenetics : official journal of the DNA Methylation
             Society},
   Volume = {12},
   Number = {8},
   Pages = {688-697},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1080/15592294.2017.1347241},
   Abstract = {Excessive inflammation during pregnancy alters homeostatic
             mechanisms of the developing fetus and has been linked to
             adverse pregnancy outcomes. An anti-inflammatory diet could
             be a promising avenue to combat the pro-inflammatory state
             of pregnancy, particularly in obese women, but we lack
             mechanistic data linking this dietary pattern during
             pregnancy to inflammation and birth outcomes. In an
             ethnically diverse cohort of 1057 mother-child pairs, we
             estimated the relationships between dietary inflammatory
             potential [measured via the energy-adjusted dietary
             inflammatory index (E-DII™)] and birth outcomes overall,
             as well as by offspring sex and maternal pre-pregnancy body
             mass index (BMI). In a subset of women, we also explored
             associations between E-DII, circulating cytokines (n = 105),
             and offspring methylation (n = 338) as potential modulators
             of these relationships using linear regression. Adjusted
             regression models revealed that women with pro-inflammatory
             diets had elevated rates of preterm birth among female
             offspring [β = -0.22, standard error (SE) = 0.07, P<0.01],
             but not male offspring (β=0.09, SE = 0.06, P<0.12)
             (Pinteraction = 0.003). Similarly, we observed
             pro-inflammatory diets were associated with higher rates of
             caesarean delivery among obese women (β = 0.17, SE = 0.08,
             P = 0.03), but not among women with BMI <25 kg/m2
             (Pinteraction = 0.02). We observed consistent inverse
             associations between maternal inflammatory cytokine
             concentrations (IL-12, IL-17, IL-4, IL-6, and TNFα) and
             lower methylation at the MEG3 regulatory sequence (P<0.05);
             however, results did not support the link between maternal
             E-DII and circulating cytokines. We replicate work by others
             on the association between maternal inflammatory diet and
             adverse pregnancy outcomes and provide the first empirical
             evidence supporting the inverse association between
             circulating cytokine concentrations and offspring
             methylation.},
   Doi = {10.1080/15592294.2017.1347241},
   Key = {fds329030}
}

@article{fds326256,
   Author = {Fuemmeler, BF and Behrman, P and Taylor, M and Sokol, R and Rothman, E and Jacobson, LT and Wischenka, D and Tercyak, KP},
   Title = {Child and family health in the era of prevention: new
             opportunities and challenges.},
   Journal = {Journal of Behavioral Medicine},
   Volume = {40},
   Number = {1},
   Pages = {159-174},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1007/s10865-016-9791-1},
   Abstract = {To maintain positive health outcomes over the life course,
             prevention efforts should begin early in childhood. Two
             research domains that significantly impact the trajectory of
             health over the life course are childhood obesity and early
             trauma and violence. Prevention strategies addressing
             multiple levels of influence are being adopted in these
             fields. Childhood obesity prevention efforts no longer focus
             solely on individuals, but embrace multiple ecological
             levels, such as family, school, and community. Similarly,
             research on early trauma and violence has broadened to
             consider risk and protective factors across domains of
             influence. Although we have improved our understanding and
             prevention of these two issues, gaps remain in research,
             practice, and policy. The purpose of this review is to relay
             relevant findings that could enhance prevention strategies.
             We describe early life and multilevel risk factors relevant
             to these areas of research. We also provide recommendations
             for future efforts to better ensure good health for
             generations to come.},
   Doi = {10.1007/s10865-016-9791-1},
   Key = {fds326256}
}

@article{fds72465,
   Title = {Fuemmeler BF, Agurs-Collins T, McClernon FJ, Kollins SH,
             Kail ME, Bergen AW, Ashley-Koch AE.  Genes implicated in
             serotonergic and dopamine functioning interact with gender
             to predict BMI categories: Findings from a nationally
             representative sample of young adults.  Obesity (in
             press) },
   Key = {fds72465}
}

@article{fds72466,
   Title = {McClernon FJ, Fuemmeler BF, Kollins SH, Kail ME, Ashley-Koch
             AE. Interactions between genotype and retrospective ADHD
             symptoms predict lifetime smoking risk in a sample of young
             adults. Nicotine & Tobacco Research (in press)
               },
   Key = {fds72466}
}

@article{fds202522,
   Title = {Fuemmeler, B. F., Yang, C., Costanzo, P., Hoyle, R.H.,
             Siegler, I.C., Williams, R.B., Ostbye, T. (in press).
             Parenting styles and BMI trajectories from adolescence into
             adulthood: A longitudinal study of the Add Health cohort.
             Health Psychology},
   Key = {fds202522}
}

@article{fds202523,
   Title = {Ranby, K.W., Boynton, M.H., Kollins, S. H., McClernon, J.,
             Yang, C., & Fuemmeler, B.F. (in press). Understanding the
             phenotypic structure of ADHD in a US population-based
             sample. Journal of Clinical Child and Adolescent Psychology.
              },
   Key = {fds202523}
}

@article{fds211227,
   Title = {Fuemmeler, B.F., Lovelady, C.A., Zucker, N.L., Ostbye, T.
             (in press). Parental obesity moderates relationship between
             childhood appetitive traits and weight. Obesity},
   Key = {fds211227}
}

@article{fds211230,
   Title = {Fuemmeler, B.F., Pendzich, M.K., Clark, K., Rossoff, P.,
             Blatt, J., Lovelady , C., Demark-Wahnefried, W. (in press).
             Changes in weight, body composition, diet, and physical
             activity during the first year of treatment for childhood
             acute leukemia and lymphoma. Journal of Pediatric Hematology
             and Oncology },
   Key = {fds211230}
}

@article{fds215514,
   Title = {Fuemmeler, B.F., Lovelady, C.A., Zucker, N.L., Ostbye, T.
             (in press). Parental obesity moderates relationship between
             childhood eating behaviors and weight. Obesity },
   Key = {fds215514}
}


%% Gaither, Sarah E.   
@article{fds330902,
   Author = {Dukes, KN and Gaither, SE},
   Title = {Black Racial Stereotypes and Victim Blaming: Implications
             for Media Coverage and Criminal Proceedings in Cases of
             Police Violence against Racial and Ethnic
             Minorities},
   Journal = {Journal of Social Issues},
   Volume = {73},
   Number = {4},
   Pages = {789-807},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1111/josi.12248},
   Abstract = {© 2017 The Society for the Psychological Study of Social
             Issues Posthumous stereotypical media portrayals of Michael
             Brown and other racial and ethnic minority victims of police
             violence have sparked questions about the influence of
             racial stereotypes on public opinions about their deaths and
             criminal proceedings for their killers. However, few studies
             have empirically investigated how the specific type of
             information released about a victim impacts opinions
             surrounding such incidents. Participants (N = 453) read
             about an altercation that resulted in a shooting death where
             the race of the victim and shooter (Black vs. White) was
             randomly assigned. Participants learned either negative,
             Black male stereotypic or positive, Black male
             counterstereotypic information about the victim. Next,
             participants appraised levels of fault and blame, sympathy
             and empathy for the victim and shooter, and indictment
             recommendations for the shooter. Findings suggest that the
             type of information released about a victim can
             significantly sway attitudes toward the victim and the
             shooter. Implications for media portrayals of racial/ethnic
             minority victims of police violence and its impact on
             criminal sentencing are discussed.},
   Doi = {10.1111/josi.12248},
   Key = {fds330902}
}

@article{fds330903,
   Author = {Gomez, EM and Young, DM and Preston, AG and Wilton, LS and Gaither, SE and Kaiser, CR},
   Title = {Loss and loyalty: Change in political and gender identity
             among Clinton supporters after the 2016 U.S. presidential
             election},
   Journal = {Self and Identity},
   Pages = {1-23},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1080/15298868.2017.1391873},
   Doi = {10.1080/15298868.2017.1391873},
   Key = {fds330903}
}

@article{fds332755,
   Author = {Gaither, SE and Apfelbaum, EP and Birnbaum, HJ and Babbitt, LG and Sommers, SR},
   Title = {Mere Membership in Racially Diverse Groups Reduces
             Conformity},
   Journal = {Social Psychological and Personality Science},
   Pages = {194855061770801-194855061770801},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1177/1948550617708013},
   Doi = {10.1177/1948550617708013},
   Key = {fds332755}
}


%% Gassman-Pines, Anna   
@article{fds328959,
   Author = {Schenck-Fontaine, A and Gassman-Pines, A and Hill,
             Z},
   Title = {Use of Informal Safety Nets during the Supplemental
             Nutrition Assistance Program Benefit Cycle: How Poor
             Families Cope with Within-Month Economic
             Instability},
   Journal = {Social Service Review},
   Volume = {91},
   Number = {3},
   Pages = {456-487},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1086/694091},
   Doi = {10.1086/694091},
   Key = {fds328959}
}

@article{fds326690,
   Author = {Ananat, EO and Gassman-Pines, A and Francis, DV and Gibson-Davis,
             CM},
   Title = {Linking job loss, inequality, mental health, and
             education.},
   Journal = {Science},
   Volume = {356},
   Number = {6343},
   Pages = {1127-1128},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1126/science.aam5347},
   Doi = {10.1126/science.aam5347},
   Key = {fds326690}
}

@article{fds326510,
   Author = {Schenck-Fontaine, A and Gassman-Pines, A and Gibson-Davis, CM and Ananat, EO},
   Title = {Local Job Losses and Child Maltreatment: The Importance of
             Community Context},
   Journal = {Social Service Review},
   Volume = {91},
   Number = {2},
   Pages = {233-263},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1086/692075},
   Abstract = {© 2017 by The University of Chicago. All rights reserved.A
             growing body of literature suggests that economic downturns
             predict an increase in child maltreatment. However, to
             inform policies and practices to prevent and intervene in
             child maltreatment, it is necessary to identify how, when,
             and under what conditions community-level economic
             conditions affect child maltreatment. In this study, we use
             North Carolina administrative data from 2006 to 2011 on
             child maltreatment reports and job losses to distinguish
             effects on maltreatment frequency from effects on severity,
             identify the timing of these effects, and test whether
             community characteristics moderate these effects. To isolate
             effects of unanticipated job losses and to control for
             potential confounding factors, we use a fixed effects
             regression approach. We find that, though job losses did not
             affect the frequency of reports, job losses increased the
             share of reports that were relatively severe. This effect
             endured for 9 months following job losses and was only
             evident in economically disadvantaged communities.},
   Doi = {10.1086/692075},
   Key = {fds326510}
}

@article{fds321769,
   Author = {Gassman-Pines, A and Gibson-Davis, CM and Vernot, C and Butler, M and Hall, N and Taylor, L and Eastwood, K and Zhang, X},
   Title = {They Should Say “I Don't”: Norms About Midpregnancy
             Marriage and Job Loss},
   Journal = {Journal of Marriage and Family},
   Volume = {79},
   Number = {2},
   Pages = {405-418},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1111/jomf.12379},
   Doi = {10.1111/jomf.12379},
   Key = {fds321769}
}

@article{fds333175,
   Author = {Gassman-Pines, A and Skinner, AT},
   Title = {Psychological Acculturation and Parenting Behaviors in
             Mexican-Immigrant Families},
   Journal = {Journal of Family Issues},
   Pages = {0192513X1668700-0192513X1668700},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1177/0192513X16687001},
   Doi = {10.1177/0192513X16687001},
   Key = {fds333175}
}


%% George, Linda K.   
@article{fds256055,
   Author = {Saltz, CC and Eisenberg, M and Fillenbaum, G and George,
             LK},
   Title = {Functional Status and Service Use Among Community-based
             Spinal Cord-injured Male Veterans12},
   Journal = {NeuroRehabilitation},
   Volume = {1},
   Number = {4},
   Pages = {25-35},
   Editor = {Frank, RG},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.3233/NRE-1991-1405},
   Doi = {10.3233/NRE-1991-1405},
   Key = {fds256055}
}


%% Gibson-Davis, Christina M.   
@article{fds326123,
   Author = {Rackin, HM and Gibson-Davis, CM},
   Title = {Low-Income Childless Young Adults' Marriage and Fertility
             Frameworks},
   Journal = {Journal of Marriage and Family},
   Volume = {79},
   Number = {4},
   Pages = {1096-1110},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/jomf.12405},
   Abstract = {© National Council on Family Relations, 2017.The authors
             investigated how low-income young adults without children
             understand marriage and fertility. Data come from the
             Becoming Partners and Parents Study (N=69), a qualitative
             study of African American adults aged 18 to 22 in a mid-size
             southern city. This is the first study to analyze young,
             low-income, childless, and unmarried Black respondents'
             frameworks (i.e., internal understandings of the world) of
             marriage and fertility. In contrast to research conducted on
             parents, this research on childless adults indicated a
             narrative in which there were close connections between
             marriage and fertility and an economic bar adhered to both
             marriage and childbearing. Respondents also believed that
             childbearing was meaningful and provided purpose, but that
             it was morally questionable if the parent was not
             financially stable. The results suggest that prior findings
             related to meanings of family formation and childbearing for
             low-income parents may not extend to those without
             children.},
   Doi = {10.1111/jomf.12405},
   Key = {fds326123}
}

@article{fds324192,
   Author = {Holbein, JB and Hillygus, DS and Gibson-Davis, CM and Hill, D and Lenard, MA},
   Title = {The Development of Students’ Engagement in School,
             Community, and Democracy},
   Year = {2017},
   Month = {June},
   Key = {fds324192}
}

@article{fds326472,
   Author = {Schenck-Fontaine, A and Gassman-Pines, A and Gibson-Davis, CM and Ananat, EO},
   Title = {Local Job Losses and Child Maltreatment: The Importance of
             Community Context},
   Journal = {Social Service Review},
   Volume = {91},
   Number = {2},
   Pages = {233-263},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1086/692075},
   Abstract = {© 2017 by The University of Chicago. All rights reserved.A
             growing body of literature suggests that economic downturns
             predict an increase in child maltreatment. However, to
             inform policies and practices to prevent and intervene in
             child maltreatment, it is necessary to identify how, when,
             and under what conditions community-level economic
             conditions affect child maltreatment. In this study, we use
             North Carolina administrative data from 2006 to 2011 on
             child maltreatment reports and job losses to distinguish
             effects on maltreatment frequency from effects on severity,
             identify the timing of these effects, and test whether
             community characteristics moderate these effects. To isolate
             effects of unanticipated job losses and to control for
             potential confounding factors, we use a fixed effects
             regression approach. We find that, though job losses did not
             affect the frequency of reports, job losses increased the
             share of reports that were relatively severe. This effect
             endured for 9 months following job losses and was only
             evident in economically disadvantaged communities.},
   Doi = {10.1086/692075},
   Key = {fds326472}
}

@article{fds326877,
   Author = {Ananat, EO and Gassman-Pines, A and Francis, DV and Gibson-Davis,
             CM},
   Title = {Linking job loss, inequality, mental health, and
             education.},
   Journal = {Science},
   Volume = {356},
   Number = {6343},
   Pages = {1127-1128},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1126/science.aam5347},
   Doi = {10.1126/science.aam5347},
   Key = {fds326877}
}

@article{fds320823,
   Author = {Gassman-Pines, A and Gibson-Davis, CM and Vernot, C and Butler, M and Hall, N and Taylor, L and Eastwood, K and Zhang, X},
   Title = {They Should Say “I Don't”: Norms About Midpregnancy
             Marriage and Job Loss},
   Journal = {Journal of Marriage and Family},
   Volume = {79},
   Number = {2},
   Pages = {405-418},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1111/jomf.12379},
   Doi = {10.1111/jomf.12379},
   Key = {fds320823}
}


%% Giusto, Ali   
@article{fds326207,
   Author = {Giusto, AM and Green, EP and Puffer, ES},
   Title = {Associations Between Fathers' and Sons' Sexual Risk in Rural
             Kenya: The Potential for Intergenerational
             Transmission.},
   Journal = {Journal of Adolescent Health},
   Volume = {61},
   Number = {2},
   Pages = {219-225},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.jadohealth.2017.02.007},
   Abstract = {Despite high rates of HIV in areas of Sub-Saharan Africa and
             men's role in driving the epidemic, little is known about
             whether or how sexual risk-both behaviors and beliefs-may be
             passed down through generations of males. This study
             examined associations between sexual risk behaviors and
             sex-related beliefs of adolescent males and those of their
             male caregivers in Kenya, as well as the potential
             moderating effects of parenting characteristics and
             father-son relationship quality.Cross-sectional linear
             regression analysis was applied to baseline data from a
             trial of a family- and church-based intervention for
             families in rural Kenya that followed a stepped-wedge
             cluster randomized design. Our subsample consisted of 79
             male caregiver and son (aged 10-16 years) dyads.Results
             demonstrated a direct relationship between fathers' and
             sons' sex-related beliefs that was not moderated by
             parenting or quality of father-son relationship.
             Parenting/relationship characteristics did moderate the
             relationship between fathers' and sons' sexual behavior; if
             fathers did not engage in high-risk sex and exhibited more
             positive parenting/higher relationship quality, their sons
             were less likely to be sexually active. Among fathers having
             high-risk sex, parenting was unrelated to sons' behavior
             except at very high levels of positive parenting/relationship
             quality; at these levels, sons were actually more likely to
             have had sex.Findings support recommendations to include
             male caregivers in youth HIV prevention efforts, potentially
             by targeting fathers' parenting strategies and their
             individual risk.},
   Doi = {10.1016/j.jadohealth.2017.02.007},
   Key = {fds326207}
}


%% Gold, Deborah T.   
@article{fds326232,
   Author = {Kerr, C and Bottomley, C and Shingler, S and Giangregorio, L and de
             Freitas, HM and Patel, C and Randall, S and Gold,
             DT},
   Title = {The importance of physical function to people with
             osteoporosis.},
   Journal = {Osteoporosis International},
   Volume = {28},
   Number = {5},
   Pages = {1597-1607},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1007/s00198-017-3911-9},
   Abstract = {There is increasing need to understand patient outcomes in
             osteoporosis. This article discusses that fracture in
             osteoporosis can lead to a cycle of impairment, driven by
             complex psychosocial factors, having a profound impact on
             physical function/activity which accumulates over time. More
             information is required on how treatments impact physical
             function.There is increasing need to understand
             patient-centred outcomes in osteoporosis (OP) clinical
             research and management. This multi-method paper provides
             insight on the effect of OP on patients' physical function
             and everyday activity.Data were collected from three
             sources: (1) targeted literature review on OP and physical
             function, conducted in MEDLINE, Embase and PsycINFO; (2)
             secondary thematic analysis of transcripts from patient
             interviews, conducted to develop a patient-reported outcome
             instrument. Transcripts were re-coded to focus on OP impact
             on daily activities and physical function for those with and
             without fracture history; and (3) discussions of the
             literature review and secondary qualitative analysis results
             with three clinical experts to review and interpret the
             importance and implications of the findings.Results suggest
             that OP, particularly with fracture, can have profound
             impacts on physical function/activity. These impacts
             accumulate over time through a cycle of impairment, as
             fracture leads to longer term detriments in physical
             function, including loss of muscle, activity avoidance and
             reduced physical capacity, which in turn leads to greater
             risk of fracture and potential for further physical
             restrictions. The cycle of impairment is complex, as other
             physical, psychosocial and treatment-related factors, such
             as comorbidities, fears and beliefs about physical activity
             and fracture risk influence physical function and everyday
             activity.More information on how treatments impact physical
             function would benefit healthcare professionals and persons
             with OP in making treatment decisions and improving
             treatment compliance/persistence, as these impacts may be
             more salient to patients than fracture incidence.},
   Doi = {10.1007/s00198-017-3911-9},
   Key = {fds326232}
}

@article{fds326233,
   Author = {Diez-Perez, A and Naylor, KE and Abrahamsen, B and Agnusdei, D and Brandi, ML and Cooper, C and Dennison, E and Eriksen, EF and Gold, DT and Guañabens, N and Hadji, P and Hiligsmann, M and Horne, R and Josse, R and Kanis, JA and Obermayer-Pietsch, B and Prieto-Alhambra, D and Reginster, J-Y and Rizzoli, R and Silverman, S and Zillikens, MC and Eastell, R and Adherence Working Group of the International
             Osteoporosis Foundation and the European Calcified Tissue
             Society},
   Title = {International Osteoporosis Foundation and European Calcified
             Tissue Society Working Group. Recommendations for the
             screening of adherence to oral bisphosphonates.},
   Journal = {Osteoporosis International},
   Volume = {28},
   Number = {3},
   Pages = {767-774},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1007/s00198-017-3906-6},
   Abstract = {Adherence to oral bisphosphonates is low. A screening
             strategy is proposed based on the response of biochemical
             markers of bone turnover after 3 months of therapy. If no
             change is observed, the clinician should reassess the
             adherence to the treatment and also other potential issues
             with the drug.Low adherence to oral bisphosphonates is a
             common problem that jeopardizes the efficacy of treatment of
             osteoporosis. No clear screening strategy for the assessment
             of compliance is widely accepted in these patients.The
             International Osteoporosis Foundation and the European
             Calcified Tissue Society have convened a working group to
             propose a screening strategy to detect a lack of adherence
             to these drugs. The question to answer was whether the bone
             turnover markers (BTMs) PINP and CTX can be used to identify
             low adherence in patients with postmenopausal osteoporosis
             initiating oral bisphosphonates for osteoporosis. The
             findings of the TRIO study specifically address this
             question and were used as the basis for testing the
             hypothesis.Based on the findings of the TRIO study,
             specifically addressing this question, the working group
             recommends measuring PINP and CTX at baseline and 3 months
             after starting therapy to check for a decrease above the
             least significant change (decrease of more than 38% for PINP
             and 56% for CTX). Detection rate for the measurement of PINP
             is 84%, for CTX 87% and, if variation in at least one is
             considered when measuring both, the level of detection is
             94.5%.If a significant decrease is observed, the treatment
             can continue, but if no decrease occurs, the clinician
             should reassess to identify problems with the treatment,
             mainly low adherence.},
   Doi = {10.1007/s00198-017-3906-6},
   Key = {fds326233}
}


%% Goldston, David B.   
@article{fds330045,
   Author = {Copeland, WE and Goldston, DB and Costello, EJ},
   Title = {Adult Associations of Childhood Suicidal Thoughts and
             Behaviors: A Prospective, Longitudinal Analysis.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {958-965.e4},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.08.015},
   Abstract = {Suicidal thoughts and behavior (STBs) have their peak period
             of onset in adolescence, but little is known about how such
             behavior is associated with later functioning. The aim of
             this study is to test whether childhood STBs are related to
             adult psychiatric, suicidal, and functional outcomes.This is
             a prospective, population-based community study of 1,420
             participants assessed with structured interviews up to 7
             times in childhood/adolescence (ages 9-16 years; 6,674
             observations) for STBs including passive and active
             ideation, plans, and attempts. Participants were then
             assessed 4 times in young adulthood (ages 19, 21, 24, and 30
             years; 4,556 observations of 1,273 participants) for
             psychiatric diagnoses, STBs, and functional outcomes.By age
             16 years, 7.0% of participants had reported some type of
             STBs, with 3.9% reporting an attempt. Both ideation only and
             suicide attempts were associated with higher levels of
             anxiety disorders and STBs in adulthood, as well as poor
             functioning across financial, health, risky/illegal, and
             social domains. These observed effects generally were
             attenuated after adjusting for other psychiatric and
             psychosocial factors that predict childhood STBs
             (particularly maltreatment, depression, and disruptive
             behavior disorders). The exception was adult suicidal
             behavior, which was predicted by both childhood ideation and
             attempts, even in the fully adjusted model. Children and
             adolescents with STBs were more likely to have had a
             disrupted transition to adulthood.Childhood STBs are a
             marker for a multitude of poor psychiatric and functional
             outcomes in adulthood, but these effects are largely
             accounted for by other factors. In contrast, childhood STBs
             are a robust risk factor for adult suicidal thoughts and
             behavior.},
   Doi = {10.1016/j.jaac.2017.08.015},
   Key = {fds330045}
}

@article{fds322758,
   Author = {Daniel, SS and Goldston, DB and Erkanli, A and Heilbron, N and Franklin,
             JC},
   Title = {Prospective Study of Major Loss Life Events and Risk for
             Suicidal Thoughts and Behaviors Among Adolescents and Young
             Adults.},
   Journal = {Suicide and Life-Threatening Behavior},
   Volume = {47},
   Number = {4},
   Pages = {436-449},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/sltb.12305},
   Abstract = {This prospective, naturalistic study examined the
             association between major loss life experiences, other
             psychiatric risk factors (depression, hopelessness, and
             anxiety), and suicidal thoughts and behaviors (STBs) among
             adolescents followed through young adulthood for up to
             14 years. Major loss life events were related to subsequent
             increases in STBs. Major loss life events were primarily
             related to increases in suicide ideation in the presence of
             lower levels of other risk factors. There was a
             bidirectional relationship between major losses and other
             risk factors. Implications for the association between loss
             experiences, other risk factors, and future STBs are
             discussed.},
   Doi = {10.1111/sltb.12305},
   Key = {fds322758}
}


%% Grimes, Christina   
@article{fds326698,
   Author = {Golonka, MM and Peairs, KF and Malone, PS and Grimes, CL and Costanzo,
             PR},
   Title = {Natural Peer Leaders as Substance Use Prevention Agents: the
             Teens' Life Choice Project.},
   Journal = {Prevention Science},
   Volume = {18},
   Number = {5},
   Pages = {555-566},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1007/s11121-017-0790-4},
   Abstract = {In adolescent social groups, natural peer leaders have been
             found to engage in more frequent experimentation with
             substance use and to possess disproportionate power to
             affect the behavior and social choices of their associated
             peer followers. In the current exploratory study, we used
             sociometrics and social cognitive mapping to identify
             natural leaders of cliques in a seventh grade population and
             invited the leaders to develop anti-drug presentations for
             an audience of younger peers. The program employed
             social-psychological approaches directed at having leaders
             proceed from extrinsic inducements to intrinsic
             identification with their persuasive products in the context
             of the group intervention process. The goals of the
             intervention were to induce substance resistant
             self-persuasion in the leaders and to produce a spread of
             this resistance effect to their peer followers. To test the
             intervention, we compared the substance use behaviors of the
             selected leaders and their peers to a control cohort. The
             study found preliminary support that the intervention
             produced changes in the substance use behavior among the
             leaders who participated in the intervention, but did not
             detect a spread to non-leader peers in the short term. This
             descriptive study speaks to the plausibility of employing
             self-persuasion paradigms to bring about change in high-risk
             behaviors among highly central adolescents. In addition, it
             highlights the viability of applying social psychological
             principles to prevention work and calls for more research in
             this area.},
   Doi = {10.1007/s11121-017-0790-4},
   Key = {fds326698}
}


%% Groh, Jennifer M.   
@article{fds332221,
   Author = {Caruso, VC and Pages, DS and Sommer, MA and Groh,
             JM},
   Title = {Beyond the labeled line: variation in visual reference
             frames from intraparietal cortex to frontal eye fields and
             the superior colliculus.},
   Journal = {Journal of neurophysiology},
   Volume = {119},
   Number = {4},
   Pages = {1411-1421},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1152/jn.00584.2017},
   Abstract = {We accurately perceive the visual scene despite moving our
             eyes ~3 times per second, an ability that requires
             incorporation of eye position and retinal information. In
             this study, we assessed how this neural computation unfolds
             across three interconnected structures: frontal eye fields
             (FEF), intraparietal cortex (LIP/MIP), and the superior
             colliculus (SC). Single-unit activity was assessed in
             head-restrained monkeys performing visually guided saccades
             from different initial fixations. As previously shown, the
             receptive fields of most LIP/MIP neurons shifted to novel
             positions on the retina for each eye position, and these
             locations were not clearly related to each other in either
             eye- or head-centered coordinates (defined as hybrid
             coordinates). In contrast, the receptive fields of most SC
             neurons were stable in eye-centered coordinates. In FEF,
             visual signals were intermediate between those patterns:
             around 60% were eye-centered, whereas the remainder showed
             changes in receptive field location, boundaries, or
             responsiveness that rendered the response patterns hybrid or
             occasionally head-centered. These results suggest that FEF
             may act as a transitional step in an evolution of
             coordinates between LIP/MIP and SC. The persistence across
             cortical areas of mixed representations that do not provide
             unequivocal location labels in a consistent reference frame
             has implications for how these representations must be read
             out. NEW & NOTEWORTHY How we perceive the world as stable
             using mobile retinas is poorly understood. We compared the
             stability of visual receptive fields across different
             fixation positions in three visuomotor regions. Irregular
             changes in receptive field position were ubiquitous in
             intraparietal cortex, evident but less common in the frontal
             eye fields, and negligible in the superior colliculus (SC),
             where receptive fields shifted reliably across fixations.
             Only the SC provides a stable labeled-line code for stimuli
             across saccades.},
   Doi = {10.1152/jn.00584.2017},
   Key = {fds332221}
}

@article{fds332360,
   Author = {Gruters, KG and Murphy, DLK and Jenson, CD and Smith, DW and Shera, CA and Groh, JM},
   Title = {The eardrums move when the eyes move: A multisensory effect
             on the mechanics of hearing.},
   Journal = {Proceedings of the National Academy of Sciences of
             USA},
   Volume = {115},
   Number = {6},
   Pages = {E1309-E1318},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1073/pnas.1717948115},
   Abstract = {Interactions between sensory pathways such as the visual and
             auditory systems are known to occur in the brain, but where
             they first occur is uncertain. Here, we show a multimodal
             interaction evident at the eardrum. Ear canal microphone
             measurements in humans (n = 19 ears in 16 subjects) and
             monkeys (n = 5 ears in three subjects) performing a saccadic
             eye movement task to visual targets indicated that the
             eardrum moves in conjunction with the eye movement. The
             eardrum motion was oscillatory and began as early as 10 ms
             before saccade onset in humans or with saccade onset in
             monkeys. These eardrum movements, which we dub eye
             movement-related eardrum oscillations (EMREOs), occurred in
             the absence of a sound stimulus. The amplitude and phase of
             the EMREOs depended on the direction and horizontal
             amplitude of the saccade. They lasted throughout the saccade
             and well into subsequent periods of steady fixation. We
             discuss the possibility that the mechanisms underlying
             EMREOs create eye movement-related binaural cues that may
             aid the brain in evaluating the relationship between visual
             and auditory stimulus locations as the eyes
             move.},
   Doi = {10.1073/pnas.1717948115},
   Key = {fds332360}
}


%% Hariri, Ahmad   
@article{fds332756,
   Author = {Nikolova, YS and Misquitta, KA and Rocco, BR and Prevot, TD and Knodt,
             AR and Ellegood, J and Voineskos, AN and Lerch, JP and Hariri, AR and Sibille, E and Banasr, M},
   Title = {Shifting priorities: Highly conserved behavioral and brain
             network adaptations to chronic stress across
             species},
   Journal = {Translational Psychiatry},
   Volume = {8},
   Number = {1},
   Year = {2018},
   Month = {December},
   url = {http://dx.doi.org/10.1038/s41398-017-0083-5},
   Abstract = {© 2018 The Author(s). Parallel clinical and preclinical
             research have begun to illuminate the biological basis of
             stress-related disorders, including major depression, but
             translational bridges informing discrete mechanistic targets
             for intervention are missing. To address this critical need,
             we used structural MRI in a mouse model and in a large human
             sample to examine stress effects on brain structure that may
             be conserved across species. Specifically, we focused on a
             previously unexplored approach, whole-brain structural
             covariance, as it reflects synchronized changes in
             neuroanatomy, potentially due to mutual trophic influences
             or shared plasticity across regions. Using the unpredictable
             chronic mild stress (UCMS) paradigm in mouse we first
             demonstrate that UCMS-induced elevated behavioral
             emotionality correlates with increased size of the amygdala
             and other corticolimbic regions. We further identify focal
             increases in the amygdala's 'hubness' (degree and strength)
             set against the background of a global stress-related loss
             of network clustering and modularity. These macroscopic
             changes are supported on the molecular level by increased
             postsynaptic density-95 protein in the amygdala, consistent
             with stress-induced plastic changes and synaptic
             strengthening. Finally, we provide clinical evidence that
             strikingly similar structural network reorganization
             patterns exist in young adults reporting high childhood
             trauma and increased mood symptoms. Collectively, we provide
             initial translational evidence for a conserved
             stress-related increase in amygdala-centered structural
             synchrony, as measured by enhanced structural covariance,
             which is paralleled by a decrease in global structural
             synchrony. This putative trade-off reflected in increased
             amygdala-centered plastic changes at the expense of global
             structural dedifferentiation may represent a mechanistic
             pathway for depression and related psychopathology.},
   Doi = {10.1038/s41398-017-0083-5},
   Key = {fds332756}
}

@article{fds326208,
   Author = {Romer, AL and Knodt, AR and Houts, R and Brigidi, BD and Moffitt, TE and Caspi, A and Hariri, AR},
   Title = {Structural alterations within cerebellar circuitry are
             associated with general liability for common mental
             disorders.},
   Journal = {Molecular Psychiatry},
   Volume = {23},
   Number = {4},
   Pages = {1084-1090},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1038/mp.2017.57},
   Abstract = {Accumulating mental-health research encourages a shift in
             focus toward transdiagnostic dimensional features that are
             shared across categorical disorders. In support of this
             shift, recent studies have identified a general liability
             factor for psychopathology-sometimes called the 'p factor'-
             that underlies shared risk for a wide range of mental
             disorders. Identifying neural correlates of this general
             liability would substantiate its importance in
             characterizing the shared origins of mental disorders and
             help us begin to understand the mechanisms through which the
             'p factor' contributes to risk. Here we believe we first
             replicate the 'p factor' using cross-sectional data from a
             volunteer sample of 1246 university students, and then using
             high-resolution multimodal structural neuroimaging, we
             demonstrate that individuals with higher 'p factor' scores
             show reduced structural integrity of white matter pathways,
             as indexed by lower fractional anisotropy values, uniquely
             within the pons. Whole-brain analyses further revealed that
             higher 'p factor' scores are associated with reduced gray
             matter volume in the occipital lobe and left cerebellar
             lobule VIIb, which is functionally connected with prefrontal
             regions supporting cognitive control. Consistent with the
             preponderance of cerebellar afferents within the pons, we
             observed a significant positive correlation between the
             white matter integrity of the pons and cerebellar gray
             matter volume associated with higher 'p factor' scores. The
             results of our analyses provide initial evidence that
             structural alterations in corticocerebellar circuitry
             supporting core functions related to the basic integration,
             coordination and monitoring of information may contribute to
             a general liability for common mental disorders.},
   Doi = {10.1038/mp.2017.57},
   Key = {fds326208}
}

@article{fds332044,
   Author = {Scult, MA and Hariri, AR},
   Title = {A BRIEF INTRODUCTION TO THE NEUROGENETICS OF
             COGNITION-EMOTION INTERACTIONS.},
   Journal = {Current Opinion in Behavioral Sciences},
   Volume = {19},
   Pages = {50-54},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.cobeha.2017.09.014},
   Abstract = {Neuroscience research has demonstrated that cognition,
             emotion, and their dynamic interactions emerge from complex
             and flexible patterns of activity across distributed neural
             circuits. A parallel branch of research in genetics has
             begun to identify common variation in the human DNA sequence
             (i.e., genome) that may shape individual differences in
             cognition-emotion interactions by altering molecular and
             cellular pathways that modulate the activity of these neural
             circuits. Here we provide a brief introduction to such
             neurogenetics research and how it may usefully inform our
             understanding of the biological mechanisms through which
             dynamic cognition-emotion interactions emerge and,
             subsequently, help shape normal and abnormal
             behavior.},
   Doi = {10.1016/j.cobeha.2017.09.014},
   Key = {fds332044}
}

@article{fds331564,
   Author = {Swartz, JR and Knodt, AR and Radtke, SR and Hariri,
             AR},
   Title = {Post-secondary maternal education buffers against neural
             risk for psychological vulnerability to future life
             stress},
   Journal = {Neuropsychologia},
   Volume = {109},
   Pages = {134-139},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2017.12.019},
   Abstract = {© 2017 Elsevier Ltd We have previously reported that
             threat-related amygdala activity measured during a baseline
             fMRI scan predicts the experience of depression and anxiety
             associated with stressful life events years later. Here, we
             examine whether two broad measures of childhood
             environmental enrichment, namely parental educational
             achievement and subjective parental socioeconomic status,
             buffer against the effects of amygdala activity on future
             vulnerability to stress. Analyses of data available from 579
             young adults revealed that maternal, but not paternal,
             educational achievement moderates the association between
             amygdala activity, recent life stress, and changes in mood
             and anxiety symptoms, even when controlling for
             participants’ current subjective socioeconomic status.
             Specifically, only participants reporting lower maternal
             educational achievement exhibited our previously observed
             interaction between amygdala activity and future life stress
             predicting increases in depression and anxiety. These
             results suggest that higher maternal educational achievement
             may help buffer stress sensitivity associated with
             heightened threat-related amygdala activity.},
   Doi = {10.1016/j.neuropsychologia.2017.12.019},
   Key = {fds331564}
}

@article{fds332980,
   Author = {Hanson, JL and Knodt, AR and Brigidi, BD and Hariri,
             AR},
   Title = {Heightened connectivity between the ventral striatum and
             medial prefrontal cortex as a biomarker for stress-related
             psychopathology: understanding interactive effects of early
             and more recent stress},
   Journal = {Psychological Medicine},
   Pages = {1-9},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1017/S0033291717003348},
   Abstract = {Copyright © Cambridge University Press 2017 Background: The
             experience of childhood maltreatment is a significant risk
             factor for the development of depression. This risk is
             particularly heightened after exposure to additional, more
             contemporaneous stress. While behavioral evidence exists for
             this relation, little is known about biological correlates
             of these stress interactions. Identifying such correlates
             may provide biomarkers of risk for later depression.
             Methods: Here, we leverage behavioral, experiential, and
             neuroimaging data from the Duke Neurogenetics Study to
             identify potential biomarkers of stress exposure. Based on
             the past research, we were specifically interested in
             reward-related connectivity and the interaction of early and
             more recent stress. We examined psychophysiological
             interactions between the ventral striatum and other brain
             regions in relation to these stress variables, as well as
             measures of internalizing symptomatology (n = 926,
             participant age range = 18–22 years of age). Results: We
             found relatively increased reward-related functional
             connectivity between the left ventral striatum and the
             medial prefrontal cortex in individuals exposed to greater
             levels of childhood maltreatment who also experienced
             greater levels of recent life stress (β = 0.199, p <
             0.005). This pattern of functional connectivity was further
             associated with elevated symptoms of depression (β = 0.089,
             p = 0.006). Furthermore, using a moderated mediation
             framework, we demonstrate that this functional connectivity
             provides a biological link between cumulative stress
             exposure and internalizing symptomatology. Conclusions:
             These findings suggest a novel biomarker linking cumulative
             stress exposure with the later experience of depressive
             symptoms. Our results are discussed in the context of past
             research examining stress exposure in relation to
             depression.},
   Doi = {10.1017/S0033291717003348},
   Key = {fds332980}
}

@article{fds329769,
   Author = {Dotterer, HL and Waller, R and Neumann, CS and Shaw, DS and Forbes, EE and Hariri, AR and Hyde, LW},
   Title = {Examining the Factor Structure of the Self-Report of
             Psychopathy Short-Form Across Four Young Adult
             Samples},
   Journal = {Assessment},
   Volume = {24},
   Number = {8},
   Pages = {1062-1079},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1177/1073191116640355},
   Doi = {10.1177/1073191116640355},
   Key = {fds329769}
}

@article{fds329035,
   Author = {Kim, MJ and Avinun, R and Knodt, AR and Radtke, SR and Hariri,
             AR},
   Title = {Neurogenetic plasticity and sex influence the link between
             corticolimbic structural connectivity and trait
             anxiety},
   Journal = {Scientific Reports},
   Volume = {7},
   Number = {1},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1038/s41598-017-11497-2},
   Doi = {10.1038/s41598-017-11497-2},
   Key = {fds329035}
}

@article{fds330404,
   Author = {Schaefer, JD and Scult, MA and Caspi, A and Arseneault, L and Belsky,
             DW and Hariri, AR and Harrington, H and Houts, R and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Is low cognitive functioning a predictor or consequence of
             major depressive disorder? A test in two longitudinal birth
             cohorts.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s095457941700164x},
   Abstract = {Cognitive impairment has been identified as an important
             aspect of major depressive disorder (MDD). We tested two
             theories regarding the association between MDD and cognitive
             functioning using data from longitudinal cohort studies. One
             theory, the cognitive reserve hypothesis, suggests that
             higher cognitive ability in childhood decreases risk of
             later MDD. The second, the scarring hypothesis, instead
             suggests that MDD leads to persistent cognitive deficits
             following disorder onset. We tested both theories in the
             Dunedin Study, a population-representative cohort followed
             from birth to midlife and assessed repeatedly for both
             cognitive functioning and psychopathology. We also used data
             from the Environmental Risk Longitudinal Twin Study to test
             whether childhood cognitive functioning predicts future MDD
             risk independent of family-wide and genetic risk using a
             discordant twin design. Contrary to both hypotheses, we
             found that childhood cognitive functioning did not predict
             future risk of MDD, nor did study members with a past
             history of MDD show evidence of greater cognitive decline
             unless MDD was accompanied by other comorbid psychiatric
             conditions. Our results thus suggest that low cognitive
             functioning is related to comorbidity, but is neither an
             antecedent nor an enduring consequence of MDD. Future
             research may benefit from considering cognitive deficits
             that occur during depressive episodes from a transdiagnostic
             perspective.},
   Doi = {10.1017/s095457941700164x},
   Key = {fds330404}
}

@article{fds330405,
   Author = {Lam, M and Trampush, JW and Yu, J and Knowles, E and Davies, G and Liewald,
             DC and Starr, JM and Djurovic, S and Melle, I and Sundet, K and Christoforou, A and Reinvang, I and DeRosse, P and Lundervold, AJ and Steen, VM and Espeseth, T and Räikkönen, K and Widen, E and Palotie,
             A and Eriksson, JG and Giegling, I and Konte, B and Roussos, P and Giakoumaki, S and Burdick, KE and Payton, A and Ollier, W and Chiba-Falek, O and Attix, DK and Need, AC and Cirulli, ET and Voineskos,
             AN and Stefanis, NC and Avramopoulos, D and Hatzimanolis, A and Arking,
             DE and Smyrnis, N and Bilder, RM and Freimer, NA and Cannon, TD et
             al.},
   Title = {Large-Scale Cognitive GWAS Meta-Analysis Reveals
             Tissue-Specific Neural Expression and Potential Nootropic
             Drug Targets.},
   Journal = {Cell Reports},
   Volume = {21},
   Number = {9},
   Pages = {2597-2613},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.celrep.2017.11.028},
   Abstract = {Here, we present a large (n = 107,207) genome-wide
             association study (GWAS) of general cognitive ability ("g"),
             further enhanced by combining results with a large-scale
             GWAS of educational attainment. We identified 70 independent
             genomic loci associated with general cognitive ability.
             Results showed significant enrichment for genes causing
             Mendelian disorders with an intellectual disability
             phenotype. Competitive pathway analysis implicated the
             biological processes of neurogenesis and synaptic
             regulation, as well as the gene targets of two pharmacologic
             agents: cinnarizine, a T-type calcium channel blocker, and
             LY97241, a potassium channel inhibitor. Transcriptome-wide
             and epigenome-wide analysis revealed that the implicated
             loci were enriched for genes expressed across all brain
             regions (most strongly in the cerebellum). Enrichment was
             exclusive to genes expressed in neurons but not
             oligodendrocytes or astrocytes. Finally, we report genetic
             correlations between cognitive ability and disparate
             phenotypes including psychiatric disorders, several
             autoimmune disorders, longevity, and maternal age at first
             birth.},
   Doi = {10.1016/j.celrep.2017.11.028},
   Key = {fds330405}
}

@article{fds332981,
   Author = {Lissek, T and Adams, M and Adelman, J and Ahissar, E and Akaaboune, M and Akil, H and al'Absi, M and Arain, F and Arango-Lasprilla, JC and Atasoy,
             D and Avila, J and Badawi, A and Bading, H and Baig, AM and Baleriola, J and Belmonte, C and Bertocchi, I and Betz, H and Blakemore, C and Blanke, O and Boehm-Sturm, P and Bonhoeffer, T and Bonifazi, P and Brose, N and Campolongo, P and Celikel, T and Chang, CC and Chang, T-Y and Citri, A and Cline, HT and Cortes, JM and Cullen, K and Dean, K and Delgado-Garcia,
             JM and Desroches, M and Disterhoft, JF and Dowling, JE and Draguhn, A and El-Khamisy, SF et al.},
   Title = {Building Bridges through Science.},
   Journal = {Neuron},
   Volume = {96},
   Number = {4},
   Pages = {730-735},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.neuron.2017.09.028},
   Abstract = {Science is ideally suited to connect people from different
             cultures and thereby foster mutual understanding. To promote
             international life science collaboration, we have launched
             "The Science Bridge" initiative. Our current project focuses
             on partnership between Western and Middle Eastern
             neuroscience communities.},
   Doi = {10.1016/j.neuron.2017.09.028},
   Key = {fds332981}
}

@article{fds328846,
   Author = {Gard, AM and Waller, R and Shaw, DS and Forbes, EE and Hariri, AR and Hyde,
             LW},
   Title = {The Long Reach of Early Adversity: Parenting, Stress, and
             Neural Pathways to Antisocial Behavior in
             Adulthood},
   Journal = {Biological psychiatry : cognitive neuroscience and
             neuroimaging},
   Volume = {2},
   Number = {7},
   Pages = {582-590},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.bpsc.2017.06.005},
   Doi = {10.1016/j.bpsc.2017.06.005},
   Key = {fds328846}
}

@article{fds329468,
   Author = {Avinun, R and Nevo, A and Knodt, AR and Elliott, ML and Radtke, SR and Brigidi, BD and Hariri, AR},
   Title = {Reward-Related Ventral Striatum Activity Buffers against the
             Experience of Depressive Symptoms Associated with Sleep
             Disturbances.},
   Journal = {The Journal of neuroscience : the official journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {40},
   Pages = {9724-9729},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1523/jneurosci.1734-17.2017},
   Abstract = {Sleep disturbances represent one risk factor for depression.
             Reward-related brain function, particularly the activity of
             the ventral striatum (VS), has been identified as a
             potential buffer against stress-related depression. We were
             therefore interested in testing whether reward-related VS
             activity would moderate the effect of sleep disturbances on
             depression in a large cohort of young adults. Data were
             available from 1129 university students (mean age 19.71 ±
             1.25 years; 637 women) who completed a reward-related
             functional MRI task to assay VS activity and provided
             self-reports of sleep using the Pittsburgh Sleep Quality
             Index and symptoms of depression using a summation of the
             General Distress/Depression and Anhedonic Depression
             subscales of the Mood and Anxiety Symptoms
             Questionnaire-short form. Analyses revealed that as VS
             activity increased the association between sleep
             disturbances and depressive symptoms decreased. The
             interaction between sleep disturbances and VS activity was
             robust to the inclusion of sex, age, race/ethnicity, past or
             present clinical disorder, early and recent life stress, and
             anxiety symptoms, as well as the interactions between VS
             activity and early or recent life stress as covariates. We
             provide initial evidence that high reward-related VS
             activity may buffer against depressive symptoms associated
             with poor sleep. Our analyses help advance an emerging
             literature supporting the importance of individual
             differences in reward-related brain function as a potential
             biomarker of relative risk for depression.SIGNIFICANCE
             STATEMENT Sleep disturbances are a common risk factor for
             depression. An emerging literature suggests that
             reward-related activity of the ventral striatum (VS), a
             brain region critical for motivation and goal-directed
             behavior, may buffer against the effect of negative
             experiences on the development of depression. Using data
             from a large sample of 1129 university students we
             demonstrate that as reward-related VS activity increases,
             the link between sleep disturbances and depression
             decreases. This finding contributes to accumulating research
             demonstrating that reward-related brain function may be a
             useful biomarker of relative risk for depression in the
             context of negative experiences.},
   Doi = {10.1523/jneurosci.1734-17.2017},
   Key = {fds329468}
}

@article{fds331565,
   Author = {Baranger, DAA and Margolis, S and Hariri, AR and Bogdan,
             R},
   Title = {An earlier time of scan is associated with greater
             threat-related amygdala reactivity},
   Journal = {Social Cognitive and Affective Neuroscience},
   Volume = {12},
   Number = {8},
   Pages = {1272-1283},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1093/scan/nsx057},
   Doi = {10.1093/scan/nsx057},
   Key = {fds331565}
}

@article{fds325037,
   Author = {Bogdan, R and Salmeron, BJ and Carey, CE and Agrawal, A and Calhoun, VD and Garavan, H and Hariri, AR and Heinz, A and Hill, MN and Holmes, A and Kalin, NH and Goldman, D},
   Title = {Imaging Genetics and Genomics in Psychiatry: A Critical
             Review of Progress and Potential},
   Journal = {Biological Psychiatry},
   Volume = {82},
   Number = {3},
   Pages = {165-175},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.biopsych.2016.12.030},
   Doi = {10.1016/j.biopsych.2016.12.030},
   Key = {fds325037}
}

@article{fds315805,
   Author = {Scult, MA and Knodt, AR and Hanson, JL and Ryoo, M and Adcock, RA and Hariri, AR and Strauman, TJ},
   Title = {Individual differences in regulatory focus predict neural
             response to reward.},
   Journal = {Social Neuroscience},
   Volume = {12},
   Number = {4},
   Pages = {419-429},
   Year = {2017},
   Month = {August},
   ISSN = {1747-0919},
   url = {http://dx.doi.org/10.1080/17470919.2016.1178170},
   Abstract = {Although goal pursuit is related to both functioning of the
             brain's reward circuits and psychological factors, the
             literatures surrounding these concepts have often been
             separate. Here, we use the psychological construct of
             regulatory focus to investigate individual differences in
             neural response to reward. Regulatory focus theory proposes
             two motivational orientations for personal goal pursuit: (1)
             promotion, associated with sensitivity to potential gain,
             and (2) prevention, associated with sensitivity to potential
             loss. The monetary incentive delay task was used to
             manipulate reward circuit function, along with instructional
             framing corresponding to promotion and prevention in a
             within-subject design. We observed that the more promotion
             oriented an individual was, the lower their ventral striatum
             response to gain cues. Follow-up analyses revealed that
             greater promotion orientation was associated with decreased
             ventral striatum response even to no-value cues, suggesting
             that promotion orientation may be associated with relatively
             hypoactive reward system function. The findings are also
             likely to represent an interaction between the cognitive and
             motivational characteristics of the promotion system with
             the task demands. Prevention orientation did not correlate
             with ventral striatum response to gain cues, supporting the
             discriminant validity of regulatory focus theory. The
             results highlight a dynamic association between individual
             differences in self-regulation and reward system
             function.},
   Doi = {10.1080/17470919.2016.1178170},
   Key = {fds315805}
}

@article{fds325971,
   Author = {Di Iorio and CR and Carey, CE and Michalski, LJ and Corral-Frias, NS and Conley, ED and Hariri, AR and Bogdan, R},
   Title = {Hypothalamic-pituitary-adrenal axis genetic variation and
             early stress moderates amygdala function.},
   Journal = {Psychoneuroendocrinology},
   Volume = {80},
   Pages = {170-178},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2017.03.016},
   Abstract = {Early life stress may precipitate psychopathology, at least
             in part, by influencing amygdala function. Converging
             evidence across species suggests that links between
             childhood stress and amygdala function may be dependent upon
             hypothalamic-pituitary-adrenal (HPA) axis function. Using
             data from college-attending non-Hispanic European-Americans
             (n=308) who completed the Duke Neurogenetics Study, we
             examined whether early life stress (ELS) and HPA axis
             genetic variation interact to predict threat-related
             amygdala function as well as psychopathology symptoms. A
             biologically-informed multilocus profile score (BIMPS)
             captured HPA axis genetic variation (FKBP5 rs1360780, CRHR1
             rs110402; NR3C2 rs5522/rs4635799) previously associated with
             its function (higher BIMPS are reflective of higher HPA axis
             activity). BOLD fMRI data were acquired while participants
             completed an emotional face matching task. ELS and
             depression and anxiety symptoms were measured using the
             childhood trauma questionnaire and the mood and anxiety
             symptom questionnaire, respectively. The interaction between
             HPA axis BIMPS and ELS was associated with right amygdala
             reactivity to threat-related stimuli, after accounting for
             multiple testing (empirical-p=0.016). Among individuals with
             higher BIMPS (i.e., the upper 21.4%), ELS was positively
             coupled with threat-related amygdala reactivity, which was
             absent among those with average or low BIMPS. Further,
             higher BIMPS were associated with greater self-reported
             anxious arousal, though there was no evidence that amygdala
             function mediated this relationship. Polygenic variation
             linked to HPA axis function may moderate the effects of
             early life stress on threat-related amygdala function and
             confer risk for anxiety symptomatology. However, what, if
             any, neural mechanisms may mediate the relationship between
             HPA axis BIMPS and anxiety symptomatology remains
             unclear.},
   Doi = {10.1016/j.psyneuen.2017.03.016},
   Key = {fds325971}
}

@article{fds325036,
   Author = {Dotterer, HL and Hyde, LW and Swartz, JR and Hariri, AR and Williamson,
             DE},
   Title = {Amygdala reactivity predicts adolescent antisocial behavior
             but not callous-unemotional traits.},
   Journal = {Developmental Cognitive Neuroscience},
   Volume = {24},
   Pages = {84-92},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.dcn.2017.02.008},
   Abstract = {Recent neuroimaging studies have suggested divergent
             relationships between antisocial behavior (AB) and
             callous-unemotional (CU) traits and amygdala reactivity to
             fearful and angry facial expressions in adolescents.
             However, little work has examined if these findings extend
             to dimensional measures of behavior in ethnically diverse,
             non-clinical samples, or if participant sex, ethnicity,
             pubertal stage, and age moderate associations. We examined
             links between amygdala reactivity and dimensions of AB and
             CU traits in 220 Hispanic and non-Hispanic Caucasian
             adolescents (age 11-15; 49.5% female; 38.2% Hispanic), half
             of whom had a family history for depression and thus were at
             relatively elevated risk for late starting, emotionally
             dysregulated AB. We found that AB was significantly related
             to increased right amygdala reactivity to angry facial
             expressions independent of sex, ethnicity, pubertal stage,
             age, and familial risk status for depression. CU traits were
             not related to fear- or anger-related amygdala reactivity.
             The present study further demonstrates that AB is related to
             increased amygdala reactivity to interpersonal threat cues
             in adolescents, and that this relationship generalizes
             across sex, ethnicity, pubertal stage, age, and familial
             risk status for depression.},
   Doi = {10.1016/j.dcn.2017.02.008},
   Key = {fds325036}
}

@article{fds325035,
   Author = {Swartz, JR and Prather, AA and Hariri, AR},
   Title = {Threat-related amygdala activity is associated with
             peripheral CRP concentrations in men but not
             women.},
   Journal = {Psychoneuroendocrinology},
   Volume = {78},
   Pages = {93-96},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2017.01.024},
   Abstract = {Increased levels of peripheral inflammatory markers,
             including C-Reactive Protein (CRP), are associated with
             increased risk for depression, anxiety, and suicidality. The
             brain mechanisms that may underlie the association between
             peripheral inflammation and internalizing problems remain to
             be determined. The present study examines associations
             between peripheral CRP concentrations and threat-related
             amygdala activity, a neural biomarker of depression and
             anxiety risk, in a sample of 172 young adult undergraduate
             students. Participants underwent functional MRI scanning
             while performing an emotional face matching task to obtain a
             measure of threat-related amygdala activity to angry and
             fearful faces; CRP concentrations were assayed from dried
             blood spots. Results indicated a significant interaction
             between CRP and sex: in men, but not women, higher CRP was
             associated with higher threat-related amygdala activity.
             These results add to the literature finding associations
             between systemic levels of inflammation and brain function
             and suggest that threat-related amygdala activity may serve
             as a potential pathway through which heightened chronic
             inflammation may increase risk for mood and anxiety
             problems.},
   Doi = {10.1016/j.psyneuen.2017.01.024},
   Key = {fds325035}
}

@article{fds323253,
   Author = {Carey, CE and Knodt, AR and Conley, ED and Hariri, AR and Bogdan,
             R},
   Title = {Reward-Related Ventral Striatum Activity Links Polygenic
             Risk for Attention-Deficit/Hyperactivity Disorder to
             Problematic Alcohol Use in Young Adulthood},
   Journal = {Biological psychiatry : cognitive neuroscience and
             neuroimaging},
   Volume = {2},
   Number = {2},
   Pages = {180-187},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.bpsc.2016.10.003},
   Doi = {10.1016/j.bpsc.2016.10.003},
   Key = {fds323253}
}

@article{fds323689,
   Author = {Trampush, JW and Yang, MLZ and Yu, J and Knowles, E and Davies, G and Liewald, DC and Starr, JM and Djurovic, S and Melle, I and Sundet, K and Christoforou, A and Reinvang, I and DeRosse, P and Lundervold, AJ and Steen, VM and Espeseth, T and Räikkönen, K and Widen, E and Palotie,
             A and Eriksson, JG and Giegling, I and Konte, B and Roussos, P and Giakoumaki, S and Burdick, KE and Payton, A and Ollier, W and Horan, M and Chiba-Falek, O and Attix, DK and Need, AC and Cirulli, ET and Voineskos,
             AN and Stefanis, NC and Avramopoulos, D and Hatzimanolis, A and Arking,
             DE and Smyrnis, N and Bilder, RM et al.},
   Title = {GWAS meta-analysis reveals novel loci and genetic correlates
             for general cognitive function: a report from the COGENT
             consortium.},
   Journal = {Molecular Psychiatry},
   Volume = {22},
   Number = {3},
   Pages = {336-345},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1038/mp.2016.244},
   Abstract = {The complex nature of human cognition has resulted in
             cognitive genomics lagging behind many other fields in terms
             of gene discovery using genome-wide association study (GWAS)
             methods. In an attempt to overcome these barriers, the
             current study utilized GWAS meta-analysis to examine the
             association of common genetic variation (~8M
             single-nucleotide polymorphisms (SNP) with minor allele
             frequency ⩾1%) to general cognitive function in a sample
             of 35 298 healthy individuals of European ancestry across
             24 cohorts in the Cognitive Genomics Consortium (COGENT). In
             addition, we utilized individual SNP lookups and polygenic
             score analyses to identify genetic overlap with other
             relevant neurobehavioral phenotypes. Our primary GWAS
             meta-analysis identified two novel SNP loci (top SNPs:
             rs76114856 in the CENPO gene on chromosome 2 and rs6669072
             near LOC105378853 on chromosome 1) associated with cognitive
             performance at the genome-wide significance level (P<5 ×
             10-8). Gene-based analysis identified an additional three
             Bonferroni-corrected significant loci at chromosomes
             17q21.31, 17p13.1 and 1p13.3. Altogether, common variation
             across the genome resulted in a conservatively estimated SNP
             heritability of 21.5% (s.e.=0.01%) for general cognitive
             function. Integration with prior GWAS of cognitive
             performance and educational attainment yielded several
             additional significant loci. Finally, we found robust
             polygenic correlations between cognitive performance and
             educational attainment, several psychiatric disorders, birth
             length/weight and smoking behavior, as well as a novel
             genetic association to the personality trait of openness.
             These data provide new insight into the genetics of
             neurocognitive function with relevance to understanding the
             pathophysiology of neuropsychiatric illness.},
   Doi = {10.1038/mp.2016.244},
   Key = {fds323689}
}

@article{fds330406,
   Author = {Swartz, JR and Waller, R and Bogdan, R and Knodt, AR and Sabhlok, A and Hyde, LW and Hariri, AR},
   Title = {A Common Polymorphism in a Williams Syndrome Gene Predicts
             Amygdala Reactivity and Extraversion in Healthy
             Adults.},
   Journal = {Biological Psychiatry},
   Volume = {81},
   Number = {3},
   Pages = {203-210},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.biopsych.2015.12.007},
   Abstract = {Williams syndrome (WS), a genetic disorder resulting from
             hemizygous microdeletion of chromosome 7q11.23, has emerged
             as a model for identifying the genetic architecture of
             socioemotional behavior. Common polymorphisms in GTF2I,
             which is found within the WS microdeletion, have been
             associated with reduced social anxiety in the general
             population. Identifying neural phenotypes affected by these
             polymorphisms would help advance our understanding not only
             of this specific genetic association but also of the broader
             neurogenetic mechanisms of variability in socioemotional
             behavior.Through an ongoing parent protocol, the Duke
             Neurogenetics Study, we measured threat-related amygdala
             reactivity to fearful and angry facial expressions using
             functional magnetic resonance imaging, assessed trait
             personality using the Revised NEO Personality Inventory, and
             imputed GTF2I rs13227433 from saliva-derived DNA using
             custom Illumina arrays. Participants included 808
             non-Hispanic Caucasian, African American, and Asian
             university students.The GTF2I rs13227433 AA genotype,
             previously associated with lower social anxiety, predicted
             decreased threat-related amygdala reactivity. An indirect
             effect of GTF2I genotype on the warmth facet of extraversion
             was mediated by decreased threat-related amygdala reactivity
             in women but not men.A common polymorphism in the WS gene
             GTF2I associated with reduced social anxiety predicts
             decreased threat-related amygdala reactivity, which mediates
             an association between genotype and increased warmth in
             women. These results are consistent with reduced
             threat-related amygdala reactivity in WS and suggest that
             common variation in GTF2I contributes to broader variability
             in socioemotional brain function and behavior, with
             implications for understanding the neurogenetic bases of WS
             as well as social anxiety.},
   Doi = {10.1016/j.biopsych.2015.12.007},
   Key = {fds330406}
}

@article{fds322497,
   Author = {Swartz, JR and Knodt, AR and Radtke, SR and Hariri,
             AR},
   Title = {Peering into the brain to predict behavior: Peer-reported,
             but not self-reported, conscientiousness links
             threat-related amygdala activity to future problem
             drinking.},
   Journal = {NeuroImage},
   Volume = {146},
   Pages = {894-903},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2016.10.003},
   Abstract = {Personality traits such as conscientiousness as
             self-reported by individuals can help predict a range of
             outcomes, from job performance to longevity. Asking others
             to rate the personality of their acquaintances often
             provides even better predictive power than using
             self-report. Here, we examine whether peer-reported
             personality can provide a better link between brain
             function, namely threat-related amygdala activity, and
             future health-related behavior, namely problem drinking,
             than self-reported personality. Using data from a sample of
             377 young adult university students who were rated on five
             personality traits by peers, we find that higher
             threat-related amygdala activity to fearful facial
             expressions is associated with higher peer-reported, but not
             self-reported, conscientiousness. Moreover, higher
             peer-reported, but not self-reported, conscientiousness
             predicts lower future problem drinking more than one year
             later, an effect specific to men. Remarkably, relatively
             higher amygdala activity has an indirect effect on future
             drinking behavior in men, linked by peer-reported
             conscientiousness to lower future problem drinking. Our
             results provide initial evidence that the perceived
             conscientiousness of an individual by their peers uniquely
             reflects variability in a core neural mechanism supporting
             threat responsiveness. These novel patterns further suggest
             that incorporating peer-reported measures of personality
             into individual differences research can reveal novel
             predictive pathways of risk and protection for problem
             behaviors.},
   Doi = {10.1016/j.neuroimage.2016.10.003},
   Key = {fds322497}
}

@article{fds316880,
   Author = {Swartz, JR and Hariri, AR and Williamson, DE},
   Title = {An epigenetic mechanism links socioeconomic status to
             changes in depression-related brain function in high-risk
             adolescents.},
   Journal = {Molecular Psychiatry},
   Volume = {22},
   Number = {2},
   Pages = {209-214},
   Year = {2017},
   Month = {February},
   ISSN = {1359-4184},
   url = {http://dx.doi.org/10.1038/mp.2016.82},
   Abstract = {Identifying biological mechanisms through which the
             experience of adversity emerges as individual risk for
             mental illness is an important step toward developing
             strategies for personalized treatment and, ultimately,
             prevention. Preclinical studies have identified epigenetic
             modification of gene expression as one such mechanism.
             Recent clinical studies have suggested that epigenetic
             modification, particularly methylation of gene regulatory
             regions, also acts to shape human brain function associated
             with risk for mental illness. However, it is not yet clear
             whether differential gene methylation as a function of
             adversity contributes to the emergence of individual risk
             for mental illness. Using prospective longitudinal
             epigenetic, neuroimaging and behavioral data from 132
             adolescents, we demonstrate that changes in gene methylation
             associated with lower socioeconomic status (SES) predict
             changes in risk-related brain function. Specifically, we
             find that lower SES during adolescence is associated with an
             increase in methylation of the proximal promoter of the
             serotonin transporter gene, which predicts greater increases
             in threat-related amygdala reactivity. We subsequently
             demonstrate that greater increases in amygdala reactivity
             moderate the association between a positive family history
             for depression and the later manifestation of depressive
             symptoms. These initial results suggest a specific
             biological mechanism through which adversity contributes to
             altered brain function, which in turn moderates the
             emergence of general liability as individual risk for mental
             illness. If replicated, this prospective pathway may
             represent a novel target biomarker for intervention and
             prevention among high-risk individuals.},
   Doi = {10.1038/mp.2016.82},
   Key = {fds316880}
}

@article{fds318715,
   Author = {Swartz, JR and Prather, AA and Di Iorio and CR and Bogdan, R and Hariri,
             AR},
   Title = {A Functional Interleukin-18 Haplotype Predicts Depression
             and Anxiety through Increased Threat-Related Amygdala
             Reactivity in Women but Not Men.},
   Journal = {Neuropsychopharmacology (Nature)},
   Volume = {42},
   Number = {2},
   Pages = {419-426},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1038/npp.2016.129},
   Abstract = {Common functional polymorphisms in the gene encoding
             interleukin-18 (IL18), a cytokine belonging to the IL-1
             superfamily that can induce synthesis of several other
             cytokines, have been associated with major depressive
             episodes following the experience of stressful life events.
             The neural mechanisms underlying these associations remain
             unexamined. Here we use an imaging genetics strategy to
             examine the effects of risk-related IL18 haplotypes
             comprising rs187238 and rs1946518 on threat-related amygdala
             reactivity and, through an indirect effect, stress-related
             symptoms of depression and anxiety in 448 non-Hispanic
             Caucasian university students. Analyses indicated that women
             but not men possessing an IL18 haplotype comprising both
             risk-related alleles evidenced increased threat-related left
             centromedial amygdala reactivity relative to other haplotype
             groups. Moreover, in women only, increased threat-related
             left centromedial amygdala reactivity predicted increased
             symptoms of depression and anxiety in individuals also
             reporting higher levels of life stress. Path analyses
             revealed a significant indirect effect of IL18 risk
             haplotype on symptoms of depression and anxiety through
             increased threat-related amygdala reactivity. These results
             suggest that a common functional IL18 haplotype associated
             with heightened proinflammatory responses confers
             susceptibility to stress-related depression and anxiety
             through effects on threat-related amygdala function, a risk
             pathway specific to women. If replicated, these patterns can
             inform the search for personalized interventions targeting
             neurobiological pathways of risk associated with
             inflammation.},
   Doi = {10.1038/npp.2016.129},
   Key = {fds318715}
}

@article{fds323776,
   Author = {Scult, MA and Knodt, AR and Swartz, JR and Brigidi, BD and Hariri,
             AR},
   Title = {Thinking and Feeling: Individual Differences in Habitual
             Emotion Regulation and Stress-Related Mood are Associated
             with Prefrontal Executive Control.},
   Journal = {Clinical Psychological Science},
   Volume = {5},
   Number = {1},
   Pages = {150-157},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1177/2167702616654688},
   Abstract = {Calculating math problems from memory may seem unrelated to
             everyday processing of emotions, but they have more in
             common than one might think. Prior research highlights the
             importance of the dorsolateral prefrontal cortex (dlPFC) in
             executive control, intentional emotion regulation, and
             experience of dysfunctional mood and anxiety. While it has
             been hypothesized that emotion regulation may be related to
             'cold' (ie. not emotion-related) executive control, this
             assertion has not been tested. We address this gap by
             providing evidence that greater dlPFC activity during 'cold'
             executive control is associated with increased use of
             cognitive reappraisal to regulate emotions in everyday life.
             We then demonstrate that in the presence of increased life
             stress, increased dlPFC activity is associated with lower
             mood and anxiety symptoms and clinical diagnoses.
             Collectively, our results encourage ongoing efforts to
             understand prefrontal executive control as a possible
             intervention target for improving emotion regulation in mood
             and anxiety disorders.},
   Doi = {10.1177/2167702616654688},
   Key = {fds323776}
}

@article{fds318712,
   Author = {Scult, MA and Paulli, AR and Mazure, ES and Moffitt, TE and Hariri, AR and Strauman, TJ},
   Title = {The association between cognitive function and subsequent
             depression: a systematic review and meta-analysis.},
   Journal = {Psychological Medicine},
   Volume = {47},
   Number = {1},
   Pages = {1-17},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1017/s0033291716002075},
   Abstract = {Despite a growing interest in understanding the cognitive
             deficits associated with major depressive disorder (MDD), it
             is largely unknown whether such deficits exist before
             disorder onset or how they might influence the severity of
             subsequent illness. The purpose of the present study was to
             conduct a systematic review and meta-analysis of
             longitudinal datasets to determine whether cognitive
             function acts as a predictor of later MDD diagnosis or
             change in depression symptoms. Eligible studies included
             longitudinal designs with baseline measures of cognitive
             functioning, and later unipolar MDD diagnosis or symptom
             assessment. The systematic review identified 29
             publications, representing 34 unique samples, and 121 749
             participants, that met the inclusion/exclusion criteria.
             Quantitative meta-analysis demonstrated that higher
             cognitive function was associated with decreased levels of
             subsequent depression (r = -0.088, 95% confidence interval.
             -0.121 to -0.054, p < 0.001). However, sensitivity analyses
             revealed that this association is likely driven by
             concurrent depression symptoms at the time of cognitive
             assessment. Our review and meta-analysis indicate that the
             association between lower cognitive function and later
             depression is confounded by the presence of contemporaneous
             depression symptoms at the time of cognitive assessment.
             Thus, cognitive deficits predicting MDD likely represent
             deleterious effects of subclinical depression symptoms on
             performance rather than premorbid risk factors for
             disorder.},
   Doi = {10.1017/s0033291716002075},
   Key = {fds318712}
}

@article{fds331566,
   Author = {Avinun, R and Nevo, A and Knodt, AR and Elliott, ML and Hariri,
             AR},
   Title = {Replication in Imaging Genetics: The Case of Threat-Related
             Amygdala Reactivity},
   Journal = {Biological Psychiatry},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.biopsych.2017.11.010},
   Abstract = {© 2017 Society of Biological Psychiatry. Background: Low
             replication rates are a concern in most, if not all,
             scientific disciplines. In psychiatric genetics
             specifically, targeting intermediate brain phenotypes, which
             are more closely associated with putative genetic effects,
             was touted as a strategy leading to increased power and
             replicability. In the current study, we attempted to
             replicate previously published associations between single
             nucleotide polymorphisms and threat-related amygdala
             reactivity, which represents a robust brain phenotype not
             only implicated in the pathophysiology of multiple
             disorders, but also used as a biomarker of future risk.
             Methods: We conducted a literature search for published
             associations between single nucleotide polymorphisms and
             threat-related amygdala reactivity and found 37 unique
             findings. Our replication sample consisted of 1117 young
             adult volunteers (629 women, mean age 19.72 ± 1.25 years)
             for whom both genetic and functional magnetic resonance
             imaging data were available. Results: Of the 37 unique
             associations identified, only three replicated as previously
             reported. When exploratory analyses were conducted with
             different model parameters compared to the original
             findings, significant associations were identified for 28
             additional studies: eight of these were for a different
             contrast/laterality; five for a different gender and/or
             race/ethnicity; and 15 in the opposite direction and for a
             different contrast, laterality, gender, and/or
             race/ethnicity. No significant associations, regardless of
             model parameters, were detected for six studies. Notably,
             none of the significant associations survived correction for
             multiple comparisons. Conclusions: We discuss these patterns
             of poor replication with regard to the general strategy of
             targeting intermediate brain phenotypes in genetic
             association studies and the growing importance of advancing
             the replicability of imaging genetics findings.},
   Doi = {10.1016/j.biopsych.2017.11.010},
   Key = {fds331566}
}


%% Hoyle, Rick   
@article{fds332881,
   Author = {Arco-Tirado, JL and Fernández-Martín, FD and Hoyle,
             RH},
   Title = {Development and validation of a Spanish version of the
             Grit-S Scale},
   Journal = {Frontiers in Psychology},
   Volume = {9},
   Number = {FEB},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.3389/fpsyg.2018.00096},
   Abstract = {© 2018 Arco-Tirado, Fernández-Martín and Hoyle. This
             paper describes the development and initial validation of a
             Spanish version of the Short Grit (Grit-S) Scale. The Grit-S
             Scale was adapted and translated into Spanish using the
             Translation, Review, Adjudication, Pre-testing, and
             Documentation model and responses to a preliminary set of
             items from a large sample of university students (N =
             1,129). The resultant measure was validated using data from
             a large stratified random sample of young adults (N =
             1,826). Initial validation involved evaluating the internal
             consistency of the adapted scale and its subscales and
             comparing the factor structure of the adapted version to
             that of the original scale. The results were comparable to
             results from similar analyses of the English version of the
             scale. Although the internal consistency of the subscales
             was low, the internal consistency of the full scale was
             well-within the acceptable range. A two-factor model offered
             an acceptable account of the data; however, when a single
             correlated error involving two highly similar items was
             included, a single factor model fit the data very well. The
             results support the use of overall scores from the Spanish
             Grit-S Scale in future research.},
   Doi = {10.3389/fpsyg.2018.00096},
   Key = {fds332881}
}

@article{fds327866,
   Author = {Piontak, JR and Russell, MA and Danese, A and Copeland, WE and Hoyle,
             RH and Odgers, CL},
   Title = {Violence exposure and adolescents' same-day obesogenic
             behaviors: New findings and a replication.},
   Journal = {Social Science & Medicine},
   Volume = {189},
   Pages = {145-151},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.socscimed.2017.07.004},
   Abstract = {To test whether exposure to violence is associated with
             same-day increases in obesogenic behaviors among young
             adolescents, including unhealthy food and beverage
             consumption, poor quality sleep, and lack of physical
             activity.Young at-risk adolescents between 12 and 15 years
             of age were recruited via telephone screening from
             low-income neighborhoods. Adolescents and their parents
             completed in-person assessments, followed by Ecological
             Momentary Assessment (EMA) delivered to 151 adolescents'
             mobile phones three times a day for 30 days (4329 person
             days). Three obesogenic behaviors - unhealthy food
             consumption, poor sleep quality, and lack of physical
             activity - and violence exposure were assessed daily.
             Adolescents' body mass index (BMI) was assessed prior to the
             EMA and 18 months later. A replication was performed among
             395 adolescents from a population-representative sample
             (with 5276 EMA person days).On days that at-risk adolescents
             were exposed versus not exposed to violence, they were more
             likely to consume unhealthy foods and beverages (b = 0.12,
             p = 0.01), report feeling tired the next morning
             (OR = 1.58, p < 0.01), and to be active (OR = 1.61,
             p < 0.01). At-risk adolescents who reported higher
             consumption of soda and caffeinated beverages during the
             30-day EMA were more likely to experience increases in BMI
             in later adolescence. Findings related to sleep and activity
             were supported in the population-based replication sample;
             however, no significant same-day associations were found
             between violence exposure and unhealthy dietary
             consumption.This study provides evidence that exposure to
             violence is associated with same-day unhealthy dietary
             consumption among at-risk adolescents and next-day tiredness
             related to sleep quality among adolescents from both at-risk
             and normative populations. Findings also point to unhealthy
             soda consumption during early adolescence as an important
             predictor of weight gain among at-risk adolescents.},
   Doi = {10.1016/j.socscimed.2017.07.004},
   Key = {fds327866}
}

@article{fds326828,
   Author = {Leary, MR and Diebels, KJ and Davisson, EK and Jongman-Sereno, KP and Isherwood, JC and Raimi, KT and Deffler, SA and Hoyle,
             RH},
   Title = {Cognitive and Interpersonal Features of Intellectual
             Humility.},
   Journal = {Personality and Social Psychology Bulletin},
   Volume = {43},
   Number = {6},
   Pages = {793-813},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1177/0146167217697695},
   Abstract = {Four studies examined intellectual humility-the degree to
             which people recognize that their beliefs might be wrong.
             Using a new Intellectual Humility (IH) Scale, Study 1 showed
             that intellectual humility was associated with variables
             related to openness, curiosity, tolerance of ambiguity, and
             low dogmatism. Study 2 revealed that participants high in
             intellectual humility were less certain that their beliefs
             about religion were correct and judged people less on the
             basis of their religious opinions. In Study 3, participants
             high in intellectual humility were less inclined to think
             that politicians who changed their attitudes were
             "flip-flopping," and Study 4 showed that people high in
             intellectual humility were more attuned to the strength of
             persuasive arguments than those who were low. In addition to
             extending our understanding of intellectual humility, this
             research demonstrates that the IH Scale is a valid measure
             of the degree to which people recognize that their beliefs
             are fallible.},
   Doi = {10.1177/0146167217697695},
   Key = {fds326828}
}


%% Huettel, Scott   
@article{fds332798,
   Author = {Wing, EA and Iyengar, V and Hess, TM and LaBar, KS and Huettel, SA and Cabeza, R},
   Title = {Neural mechanisms underlying subsequent memory for personal
             beliefs:An fMRI study.},
   Journal = {Cognitive, Affective, & Behaviorial Neuroscience},
   Volume = {18},
   Number = {2},
   Pages = {216-231},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.3758/s13415-018-0563-y},
   Abstract = {Many fMRI studies have examined the neural mechanisms
             supporting emotional memory for stimuli that generate
             emotion rather automatically (e.g., a picture of a dangerous
             animal or of appetizing food). However, far fewer studies
             have examined how memory is influenced by emotion related to
             social and political issues (e.g., a proposal for large
             changes in taxation policy), which clearly vary across
             individuals. In order to investigate the neural substrates
             of affective and mnemonic processes associated with personal
             opinions, we employed an fMRI task wherein participants
             rated the intensity of agreement/disagreement to
             sociopolitical belief statements paired with neural face
             pictures. Following the rating phase, participants performed
             an associative recognition test in which they distinguished
             identical versus recombined face-statement pairs. The study
             yielded three main findings: behaviorally, the intensity of
             agreement ratings was linked to greater subjective emotional
             arousal as well as enhanced high-confidence subsequent
             memory. Neurally, statements that elicited strong (vs. weak)
             agreement or disagreement were associated with greater
             activation of the amygdala. Finally, a subsequent memory
             analysis showed that the behavioral memory advantage for
             statements generating stronger ratings was dependent on the
             medial prefrontal cortex (mPFC). Together, these results
             both underscore consistencies in neural systems supporting
             emotional arousal and suggest a modulation of
             arousal-related encoding mechanisms when emotion is
             contingent on referencing personal beliefs.},
   Doi = {10.3758/s13415-018-0563-y},
   Key = {fds332798}
}

@article{fds327384,
   Author = {Meade, CS and Addicott, M and Hobkirk, AL and Towe, SL and Chen, N-K and Sridharan, S and Huettel, SA},
   Title = {Cocaine and HIV are independently associated with neural
             activation in response to gain and loss valuation during
             economic risky choice.},
   Journal = {Addiction Biology},
   Volume = {23},
   Number = {2},
   Pages = {796-809},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1111/adb.12529},
   Abstract = {Stimulant abuse is disproportionately common in HIV-positive
             persons. Both HIV and stimulants are independently
             associated with deficits in reward-based decision making,
             but their interactive and/or additive effects are poorly
             understood despite their prevalent co-morbidity. Here, we
             examined the effects of cocaine dependence and HIV infection
             in 69 adults who underwent functional magnetic resonance
             imaging while completing an economic loss aversion task. We
             identified two neural networks that correlated with the
             evaluation of the favorable characteristics of the gamble
             (i.e. higher gains/lower losses: ventromedial prefrontal
             cortex, anterior cingulate, anterior and posterior precuneus
             and visual cortex) versus unfavorable characteristics of the
             gamble (i.e. lower gains/higher losses: dorsal prefrontal,
             lateral orbitofrontal, posterior parietal cortex, anterior
             insula and dorsal caudate). Behaviorally, cocaine and HIV
             had additive effects on loss aversion scores, with
             HIV-positive cocaine users being the least loss averse.
             Cocaine users had greater activation in brain regions that
             tracked the favorability of gamble characteristics (i.e.
             increased activation to gains, but decreased activation to
             losses). In contrast, HIV infection was independently
             associated with lesser activation in regions that tracked
             the unfavorability of gamble characteristics. These results
             suggest that cocaine is associated with an overactive
             reward-seeking system, while HIV is associated with an
             underactive cognitive control system. Together, these
             alterations may leave HIV-positive cocaine users
             particularly vulnerable to making unfavorable decisions when
             outcomes are uncertain.},
   Doi = {10.1111/adb.12529},
   Key = {fds327384}
}

@article{fds330849,
   Author = {Kwak, Y and Huettel, S},
   Title = {The order of information processing alters economic
             gain-loss framing effects.},
   Journal = {Acta Psychologica},
   Volume = {182},
   Pages = {46-54},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.actpsy.2017.11.013},
   Abstract = {Adaptive decision making requires analysis of available
             information during the process of choice. In many decisions
             that information is presented visually - which means that
             variations in visual properties (e.g., salience, complexity)
             can potentially influence the process of choice. In the
             current study, we demonstrate that variation in the
             left-right positioning of risky and safe decision options
             can influence the canonical gain-loss framing effect. Two
             experiments were conducted using an economic framing task in
             which participants chose between gambles and certain
             outcomes. The first experiment demonstrated that the
             magnitude of the gain-loss framing effect was greater when
             the certain option signaling the current frame was presented
             on the left side of the visual display. Eye-tracking data
             during task performance showed a left-gaze bias for initial
             fixations, suggesting that the option presented on the left
             side was processed first. Combination of eye-tracking and
             choice data revealed that there was a significant effect of
             direction of first gaze (i.e. left vs. right) as well as an
             interaction between gaze direction and identity of the first
             fixated information (i.e. certain vs. gamble) regardless of
             frame. A second experiment presented the gamble and certain
             options in a random order, with a temporal delay between
             their presentations. We found that the magnitude of
             gain-loss framing was larger when the certain option was
             presented first, regardless of left and right positioning,
             only in individuals with lower risk-taking tendencies. The
             effect of presentation order on framing was not present in
             high risk-takers. These results suggest that the sequence of
             visual information processing as well as their left-right
             positioning can bias choices by changing the impact of the
             presented information during risky decision
             making.},
   Doi = {10.1016/j.actpsy.2017.11.013},
   Key = {fds330849}
}

@article{fds329283,
   Author = {Stanton, SJ and Sinnott-Armstrong, W and Huettel,
             SA},
   Title = {Neuromarketing: Ethical Implications of its Use and
             Potential Misuse},
   Journal = {Journal of Business Ethics},
   Volume = {144},
   Number = {4},
   Pages = {799-811},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1007/s10551-016-3059-0},
   Doi = {10.1007/s10551-016-3059-0},
   Key = {fds329283}
}

@article{fds327383,
   Author = {Meade, CS and Hobkirk, AL and Towe, SL and Chen, N-K and Bell, RP and Huettel, SA},
   Title = {Cocaine dependence modulates the effect of HIV infection on
             brain activation during intertemporal decision
             making.},
   Journal = {Drug and Alcohol Dependence},
   Volume = {178},
   Pages = {443-451},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.drugalcdep.2017.05.043},
   Abstract = {Both HIV infection and chronic cocaine use alter the neural
             circuitry of decision making, but the interactive effects of
             these commonly comorbid conditions have not been adequately
             examined. This study tested how cocaine moderates
             HIV-related neural activation during an intertemporal
             decision-making task.The sample included 73 participants who
             differed on cocaine and HIV status (18 COC+/HIV+, 19
             COC+/HIV-, 19 COC-/HIV+, 17 COC-/HIV-). Participants made
             choices between smaller, sooner and larger, delayed rewards
             while undergoing functional MRI. Choices varied in
             difficulty based on subjective value: hard (equivalently
             valued), easy (disparately valued), and control choices. A
             mixed-effects model controlling for education and smoking
             identified main and interactive effects of HIV and COC
             during hard relative to easy choices (difficulty
             contrast).COC+ status was associated with lower activation
             in bilateral frontal gyri and right insular and posterior
             parietal cortices. HIV+ status was associated with higher
             activation in the visual cortex, but lower activation in
             bilateral prefrontal cortices and cerebellum and left
             posterior parietal cortex. COC moderated the effects of HIV
             in several clusters centered in the bilateral prefrontal
             cortices and cerebellum. In post-hoc analyses, there were
             significant effects of HIV status on activation for COC+,
             but not COC-, participants; interaction effects remained
             after controlling for polysubstance use.Cocaine use may
             diminish the compensatory neural activation often seen among
             HIV+ samples during decision making. Our results highlight
             the importance of examining the neuropsychiatric effects of
             comorbid medical conditions to identify potential neural
             targets for cognitive remediation interventions.},
   Doi = {10.1016/j.drugalcdep.2017.05.043},
   Key = {fds327383}
}

@article{fds330179,
   Author = {Kelly, M and Ngo, L and Chituc, V and Huettel, S and Sinnott-Armstrong,
             W},
   Title = {Moral conformity in online interactions: rational
             justifications increase influence of peer opinions on moral
             judgments},
   Journal = {Social Influence},
   Volume = {12},
   Number = {2-3},
   Pages = {57-68},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1080/15534510.2017.1323007},
   Doi = {10.1080/15534510.2017.1323007},
   Key = {fds330179}
}

@article{fds325992,
   Author = {Li, R and Roberts, RC and Huettel, SA and Brannon,
             EM},
   Title = {Five-year-olds do not show ambiguity aversion in a risk and
             ambiguity task with physical objects.},
   Journal = {Journal of Experimental Child Psychology},
   Volume = {159},
   Pages = {319-326},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.jecp.2017.02.013},
   Abstract = {Ambiguity aversion arises when a decision maker prefers
             risky gambles with known probabilities over equivalent
             ambiguous gambles with unknown probabilities. This
             phenomenon has been consistently observed in adults across a
             large body of empirical work. Evaluating ambiguity aversion
             in young children, however, has posed methodological
             challenges because probabilistic representations appropriate
             for adults might not be understood by young children. Here,
             we established a novel method for representing risk and
             ambiguity with physical objects that overcomes previous
             methodological limitations and allows us to measure
             ambiguity aversion in young children. We found that
             individual 5-year-olds exhibited consistent choice
             preferences and, as a group, exhibited no ambiguity aversion
             in a task that evokes ambiguity aversion in adults. Across
             individuals, 5-year-olds exhibited greater variance in
             ambiguity preferences compared with adults tested under
             similar conditions. This suggests that ambiguity aversion is
             absent during early childhood and emerges over the course of
             development.},
   Doi = {10.1016/j.jecp.2017.02.013},
   Key = {fds325992}
}

@article{fds324858,
   Author = {Cordero, DM and Towe, SL and Chen, N-K and Robertson, KR and Madden, DJ and Huettel, SA and Meade, CS},
   Title = {Cocaine dependence does not contribute substantially to
             white matter abnormalities in HIV infection.},
   Journal = {Journal of NeuroVirology},
   Volume = {23},
   Number = {3},
   Pages = {441-450},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s13365-017-0512-5},
   Abstract = {This study investigated the association of HIV infection and
             cocaine dependence with cerebral white matter integrity
             using diffusion tensor imaging (DTI). One hundred
             thirty-five participants stratified by HIV and cocaine
             status (26 HIV+/COC+, 37 HIV+/COC-, 37 HIV-/COC+, and 35
             HIV-/COC-) completed a comprehensive substance abuse
             assessment, neuropsychological testing, and MRI with DTI.
             Among HIV+ participants, all were receiving HIV care and 46%
             had an AIDS diagnosis. All COC+ participants were current
             users and met criteria for cocaine use disorder. We used
             tract-based spatial statistics (TBSS) to assess the relation
             of HIV and cocaine to fractional anisotropy (FA) and mean
             diffusivity (MD). In whole-brain analyses, HIV+ participants
             had significantly reduced FA and increased MD compared to
             HIV- participants. The relation of HIV and FA was widespread
             throughout the brain, whereas the HIV-related MD effects
             were restricted to the corpus callosum and thalamus. There
             were no significant cocaine or HIV-by-cocaine effects. These
             DTI metrics correlated significantly with duration of HIV
             disease, nadir CD4+ cell count, and AIDS diagnosis, as well
             as some measures of neuropsychological functioning. These
             results suggest that HIV is related to white matter
             integrity throughout the brain, and that HIV-related effects
             are more pronounced with increasing duration of infection
             and greater immune compromise. We found no evidence for
             independent effects of cocaine dependence on white matter
             integrity, and cocaine dependence did not appear to
             exacerbate the effects of HIV.},
   Doi = {10.1007/s13365-017-0512-5},
   Key = {fds324858}
}

@article{fds325991,
   Author = {Li, R and Smith, DV and Clithero, JA and Venkatraman, V and Carter, RM and Huettel, SA},
   Title = {Reason's Enemy Is Not Emotion: Engagement of Cognitive
             Control Networks Explains Biases in Gain/Loss
             Framing.},
   Journal = {The Journal of neuroscience : the official journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {13},
   Pages = {3588-3598},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1523/jneurosci.3486-16.2017},
   Abstract = {In the classic gain/loss framing effect, describing a gamble
             as a potential gain or loss biases people to make
             risk-averse or risk-seeking decisions, respectively. The
             canonical explanation for this effect is that frames
             differentially modulate emotional processes, which in turn
             leads to irrational choice behavior. Here, we evaluate the
             source of framing biases by integrating functional magnetic
             resonance imaging data from 143 human participants
             performing a gain/loss framing task with meta-analytic data
             from >8000 neuroimaging studies. We found that activation
             during choices consistent with the framing effect were most
             correlated with activation associated with the resting or
             default brain, while activation during choices inconsistent
             with the framing effect was most correlated with the
             task-engaged brain. Our findings argue against the common
             interpretation of gain/loss framing as a competition between
             emotion and control. Instead, our study indicates that this
             effect results from differential cognitive engagement across
             decision frames.SIGNIFICANCE STATEMENT The biases frequently
             exhibited by human decision makers have often been
             attributed to the presence of emotion. Using a large fMRI
             sample and analysis of whole-brain networks defined with the
             meta-analytic tool Neurosynth, we find that neural activity
             during frame-biased decisions was more significantly
             associated with default behaviors (and the absence of
             executive control) than with emotion. These findings point
             to a role for neuroscience in shaping long-standing
             psychological theories in decision science.},
   Doi = {10.1523/jneurosci.3486-16.2017},
   Key = {fds325991}
}

@misc{fds330540,
   Author = {Jack, J and Appelbaum, LG and Beam, E and Moody, J and Huettel,
             SA},
   Title = {Mapping rhetorical topologies in cognitive
             neuroscience},
   Pages = {125-150},
   Booktitle = {Topologies as Techniques for a Post-Critical
             Rhetoric},
   Year = {2017},
   Month = {January},
   ISBN = {9783319512686},
   url = {http://dx.doi.org/10.1007/978-3-319-51268-6_7},
   Abstract = {© The Author(s) 2017. Many tools that neuroscientists use
             to trace the complex topography of the human brain draw on
             the neuroscience literature to yield “metanalyses” or
             “syntheses of data.” These approaches conflate
             rhetorical connections in the literature with physical
             connections in the brain. By contrast, the model presented
             in this chapter seeks not a topography of the brain but a
             topology of neuroscience. A social network analysis of
             titles and abstracts for cognitive neuroscience articles
             yields a topology of brain regions and functions. This map
             can help researchers identify underresearched areas (e.g.,
             the thalamus) or areas that are oversaturated (e.g., the
             amygdala). The map also helps researchers identify
             subdisciplines, such as “neuroeconomics,” that have not
             yet integrated with the broader field–“islands” where
             rhetorical work could yield benefits.},
   Doi = {10.1007/978-3-319-51268-6_7},
   Key = {fds330540}
}

@article{fds329566,
   Author = {Utevsky, AV and Smith, DV and Young, JS and Huettel,
             SA},
   Title = {Large-Scale Network Coupling with the Fusiform Cortex
             Facilitates Future Social Motivation},
   Journal = {eNeuro},
   Volume = {4},
   Number = {5},
   Pages = {ENEURO.0084-17.2017-ENEURO.0084-17.2017},
   Year = {2017},
   url = {http://dx.doi.org/10.1523/ENEURO.0084-17.2017},
   Doi = {10.1523/ENEURO.0084-17.2017},
   Key = {fds329566}
}


%% Kay, Aaron C.   
@article{fds333552,
   Author = {Proudfoot, D and Kay, AC},
   Title = {How perceptions of one's organization can affect perceptions
             of the self: Membership in a stable organization can sustain
             individuals' sense of control},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {76},
   Pages = {104-115},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.jesp.2018.01.004},
   Abstract = {© 2018 Elsevier Inc. Building on contemporary perspectives
             regarding the role that group identification can play in
             sustaining control motives, we propose that being a member
             of a stable organization—one experienced as predictable
             and consistent rather than changing and in flux—can
             maintain individuals' sense of control. Four studies test
             this prediction. We observe that higher social
             identification as an organizational member (as compared to
             lower identification) is associated with an increased
             generalized sense of personal efficacy in life specifically
             when one's organization is experienced as relatively stable
             (Study 1 and Study 2). Further, the perceived stability of
             one's organization moderates the extent to which those who
             recently experienced a threat to personal control—and are
             thereby motivated to reestablish feelings of control—seek
             increased social identification as an organizational member
             (Study 3 and Study 4). Results suggest that membership in a
             stable organization can provide a psychological buffer
             against threats to personal control encountered in daily
             life outside work. Contributions to understanding the ways
             in which people maintain feelings of personal control in the
             social world are discussed.},
   Doi = {10.1016/j.jesp.2018.01.004},
   Key = {fds333552}
}

@article{fds332999,
   Author = {Landau, MJ and Khenfer, J and Keefer, LA and Swanson, TJ and Kay,
             AC},
   Title = {When and why does belief in a controlling God strengthen
             goal commitment?},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {75},
   Pages = {71-82},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.11.012},
   Abstract = {© 2017 Elsevier Inc. The perception that God controls one's
             life can bolster motivation to pursue personal goals, but it
             can also have no impact and even squelch motivation. To
             better understand how religious beliefs impact
             self-regulation, the current research built on Compensatory
             Control Theory's claim that perceiving the environment as
             predictable (vs. unpredictable) strengthens commitment to
             long-term goals. Perceiving God's intervention as following
             an understandable logic, which implies a predictable
             environment, increased self-reported and behavioral
             commitment to save money (Studies 1–3), excel academically
             (Study 4), and improve physical health (Study 5). In
             contrast, perceiving God as intervening in mysterious ways,
             which implies that worldly affairs are under control yet
             unpredictable, did not increase goal commitment. Exploratory
             mediational analyses focused on self-efficacy, response
             efficacy, and confidence in God's control. A meta-analysis
             (Study 6) yielded a reliable effect whereby belief in divine
             control supports goal pursuit specifically when it signals
             the predictability of one's environment.},
   Doi = {10.1016/j.jesp.2017.11.012},
   Key = {fds332999}
}

@article{fds333000,
   Author = {Tang, S and King, M and Kay, AC},
   Title = {Fate as a motivated (and de-motivating) belief: Evidence for
             a link from task importance to belief in fate to
             effort},
   Journal = {Organizational Behavior and Human Decision
             Processes},
   Volume = {144},
   Pages = {74-84},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.obhdp.2017.08.003},
   Doi = {10.1016/j.obhdp.2017.08.003},
   Key = {fds333000}
}

@article{fds329823,
   Author = {Khenfer, J and Laurin, K and Tafani, E and Roux, E and Kay,
             AC},
   Title = {Interventionist external agents make specific advice less
             demotivating},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {73},
   Pages = {189-196},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.07.003},
   Doi = {10.1016/j.jesp.2017.07.003},
   Key = {fds329823}
}

@article{fds333001,
   Author = {Fath, S and Proudfoot, D and Kay, AC},
   Title = {Effective to a fault: Organizational structure predicts
             attitudes toward minority organizations},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {73},
   Pages = {290-297},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.10.003},
   Doi = {10.1016/j.jesp.2017.10.003},
   Key = {fds333001}
}

@article{fds323320,
   Author = {Shepherd, S and Eibach, RP and Kay, AC},
   Title = {“One Nation Under God”: The System-Justifying Function
             of Symbolically Aligning God and Government},
   Journal = {Political Psychology},
   Volume = {38},
   Number = {5},
   Pages = {703-720},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1111/pops.12353},
   Abstract = {© 2016 International Society of Political Psychology. Do
             references to God in political discourse increase confidence
             in the U.S. sociopolitical system? Using a system
             justification framework (Jost & Banaji, ), five studies
             provide evidence that, (1) increasingly governments
             symbolically associate the nation with God when public
             confidence in the social system may be threatened and (2)
             associating the nation with God serves a system-justifying
             function by increasing public confidence in the system. In
             an analysis of U.S. presidential speeches, presidents were
             more likely to symbolically associate the nation with God
             during threatening times (Study 1). Among religious
             individuals, referencing God in political rhetoric increased
             the perceived trustworthiness of politicians, compared to
             patriotic secular rhetoric (Study 2) or simply priming the
             concept of God (Study 3). These effects were also unique to
             politicians from one's own sociopolitical system (Study 4).
             Finally, believing God has a plan for the United States
             attenuates the deleterious effect that perceptions of
             national decline have on system confidence (Study 5).
             Implications for the system-justifying function of religion
             are discussed.},
   Doi = {10.1111/pops.12353},
   Key = {fds323320}
}

@article{fds328086,
   Author = {Ma, A and Landau, MJ and Narayanan, J and Kay, AC},
   Title = {Thought-control difficulty motivates structure
             seeking.},
   Journal = {Journal of Experimental Psychology: General},
   Volume = {146},
   Number = {8},
   Pages = {1067-1072},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1037/xge0000282},
   Abstract = {Struggling to control one's mind can change how the world
             appears. In prior studies testing the compensatory control
             theory, reduced control over the external environment
             motivated the search for perceptual patterns and other forms
             of structured knowledge, even in remote domains. Going
             further, the current studies test whether difficulty
             controlling thoughts similarly predicts structure seeking.
             As hypothesized, thought-control difficulty positively
             predicted perceptions of causal connections between remote
             events (Study 1a) and nonexistent objects in visual noise
             (Study 1b). This effect was mediated by aversive arousal
             (Study 2) and caused specifically by thought-control
             difficulty as distinct from general difficulty (Study 3).
             Study 4 replicated the effect with a sample of meditators
             learning to control their thoughts, showing that
             thought-control difficulty was a powerful predictor of
             structure seeking. These findings reveal a novel form of
             motivated perception. (PsycINFO Database
             Record},
   Doi = {10.1037/xge0000282},
   Key = {fds328086}
}

@article{fds326633,
   Author = {Ma, A and Kay, AC},
   Title = {Compensatory control and ambiguity intolerance},
   Journal = {Organizational Behavior and Human Decision
             Processes},
   Volume = {140},
   Pages = {46-61},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.obhdp.2017.04.001},
   Doi = {10.1016/j.obhdp.2017.04.001},
   Key = {fds326633}
}

@article{fds327057,
   Author = {Leander, NP and Kay, AC and Chartrand, TL and Payne,
             BK},
   Title = {An Affect Misattribution Pathway to Perceptions of Intrinsic
             Reward},
   Journal = {Social Cognition},
   Volume = {35},
   Number = {2},
   Pages = {163-180},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1521/soco.2017.35.2.163},
   Abstract = {© 2017 Guilford Publications, Inc. Intrinsic rewards are
             typically thought to stem from an activity's inherent
             properties and not from separable rewards one receives from
             it. Yet, people may not consciously notice or remember all
             the subtle external rewards that correspond with an activity
             and may misattribute some directly to the activity itself.
             We propose that perceptions of intrinsic reward can often be
             byproducts of misattributed causal inference, and present
             some initial evidence that perceptions of intrinsic reward
             can in fact increase when words pertaining to an activity
             are subtly paired with pleasant context cues. Importantly,
             these effects follow classic boundary conditions of both
             misattribution and intrinsic motivation, insofar as they
             were extinguished when participants could make a proper
             source attribution and/or when the activity became
             associated with a blatant external reward. We further
             propose a distinction can be made between authentically
             "intrinsic" rewards and the illusion of intrinsic rewards
             caused by misattributed positive affect.},
   Doi = {10.1521/soco.2017.35.2.163},
   Key = {fds327057}
}

@article{fds326064,
   Author = {Laurin, K and Kay, AC},
   Title = {The Motivational Underpinnings of Belief in
             God},
   Journal = {Advances in experimental social psychology},
   Volume = {56},
   Pages = {201-257},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/bs.aesp.2017.02.004},
   Abstract = {© 2017 Elsevier Inc. Beliefs in powerful Gods are prevalent
             across time and across societies. In this chapter, we
             explore the motivated underpinnings of this phenomenon.
             After describing two popular theories that help account for
             some of this prevalence—one focused on byproducts of
             normal human cognition and the other focused on the cultural
             benefit conferred by shared belief in powerful Gods—we
             propose that a third perspective may be needed to fully
             explain why so many people believe: that believing in God is
             one mechanism through which people fulfill their need to
             perceive the world as structured, orderly, and nonrandom. We
             then describe a model that outlines the causes and
             consequences of perceptions of structure, and leverage this
             model to organize the evidence connecting belief in God to
             people's need for structure. We then note the ways in which
             belief in a powerful God, though not the only form of belief
             that can satisfy the need for structure, may hold an
             advantage over most alternatives. Finally, we conclude by
             discussing the implications of this perspective for
             understanding the ongoing evolution of religious
             belief.},
   Doi = {10.1016/bs.aesp.2017.02.004},
   Key = {fds326064}
}


%% Keefe, Francis J.   
@article{fds330575,
   Author = {O'Sullivan, ML and Shelby, RA and Dorfman, CS and Kelleher, SA and Fisher, HM and Rowe Nichols and KA and Keefe, FJ and Sung, AD and Somers,
             TJ},
   Title = {The effect of pre-transplant pain and chronic disease
             self-efficacy on quality of life domains in the year
             following hematopoietic stem cell transplantation.},
   Journal = {Supportive Care in Cancer},
   Volume = {26},
   Number = {4},
   Pages = {1243-1252},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s00520-017-3947-6},
   Abstract = {Pain is common for hematopoietic stem cell transplant (HSCT)
             patients and may be experienced pre-transplant, acutely
             post-transplant, and for months or years following
             transplant. HSCT patients with persistent pain may be at
             risk for poor quality of life following transplant; however,
             the impact of pre-transplant pain on quality of life
             post-transplant is not well understood. Self-efficacy for
             chronic disease management is associated with quality of
             life among cancer patients and may impact quality of life
             for HSCT patients. The primary aim was to examine the effect
             of pre-transplant pain and self-efficacy on quality of life
             domains in the year following transplant.One hundred
             sixty-six HSCT patients completed questionnaires providing
             information on pain, self-efficacy, and quality of life
             prior to transplant, at discharge, and 3-, 6-, and 12-months
             post-transplant as part of a longitudinal, observational
             study. Linear mixed modeling examined the trajectories of
             these variables and the effect of pre-transplant pain and
             self-efficacy on post-transplant quality of life.Pain and
             social and emotional quality of life remained stable in the
             year following transplant while self-efficacy and physical
             and functional quality of life improved. Pre-transplant pain
             was significantly related to lower physical well-being
             post-transplant. Lower pre-transplant self-efficacy was
             related to lower quality of life across all domains
             post-transplant.Above and beyond the effect of
             pre-transplant pain, self-efficacy for managing chronic
             disease is important in understanding quality of life
             following transplant. Identifying patients with pain and/or
             low self-efficacy pre-transplant may allow for early
             intervention with self-management strategies.},
   Doi = {10.1007/s00520-017-3947-6},
   Key = {fds330575}
}

@article{fds330868,
   Author = {Rini, C and Vu, MB and Lerner, H and Bloom, C and Carda-Auten, J and Wood,
             WA and Basch, EM and Voorhees, PM and Reeder-Hayes, KE and Keefe,
             FJ},
   Title = {A qualitative study of patient and provider perspectives on
             using web-based pain coping skills training to treat
             persistent cancer pain.},
   Journal = {Palliative & Supportive Care},
   Volume = {16},
   Number = {2},
   Pages = {155-169},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1017/s1478951517000086},
   Abstract = {Persistent pain is common and inadequately treated in cancer
             patients. Behavioral pain interventions are a recommended
             part of multimodal pain treatments, but they are underused
             in clinical care due to barriers such as a lack of the
             resources needed to deliver them in person and difficulties
             coordinating their use with clinical care. Pain coping
             skills training (PCST) is an evidence-based behavioral pain
             intervention traditionally delivered in person. Delivering
             this training via the web would increase access to it by
             addressing barriers that currently limit its use. We
             conducted a patient pilot study of an 8-week web-based PCST
             program to determine the acceptability of this approach to
             patients and the program features needed to meet their
             needs. Focus groups with healthcare providers identified
             strategies for coordinating the use of web-based PCST in
             clinical care.Participants included 7 adults with bone pain
             due to multiple myeloma or metastasized breast or prostate
             cancer and 12 healthcare providers (4 physicians and 8
             advanced practice providers) who treat cancer-related bone
             pain. Patients completed web-based PCST at home and then
             took part in an in-depth qualitative interview. Providers
             attended focus groups led by a trained moderator.
             Qualitative analyses identified themes in the patient and
             provider data.Patients reported strongly favorable responses
             to web-based PCST and described emotional and physical
             benefits. They offered suggestions for adapting the approach
             to better fit their needs and to overcome barriers to
             completion. Focus groups indicated a need to familiarize
             healthcare providers with PCST and to address concerns about
             overburdening patients. Providers would recommend the
             program to patients they felt could benefit. They suggested
             applying a broad definition of cancer pain and having
             various types of providers help coordinate program its use
             with clinical care.Web-based PCST was acceptable to patients
             and providers. Our findings suggest that patients could
             benefit from this approach, especially if patient and
             provider barriers are addressed.},
   Doi = {10.1017/s1478951517000086},
   Key = {fds330868}
}

@article{fds333734,
   Author = {DeBar, L and Benes, L and Bonifay, A and Deyo, RA and Elder, CR and Keefe,
             FJ and Leo, MC and McMullen, C and Mayhew, M and Owen-Smith, A and Smith,
             DH and Trinacty, CM and Vollmer, WM},
   Title = {Interdisciplinary team-based care for patients with chronic
             pain on long-term opioid treatment in primary care (PPACT) -
             Protocol for a pragmatic cluster randomized
             trial.},
   Journal = {Contemporary Clinical Trials},
   Volume = {67},
   Pages = {91-99},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.cct.2018.02.015},
   Abstract = {Chronic pain is one of the most common, disabling, and
             expensive public health problems in the United States.
             Interdisciplinary pain management treatments that employ
             behavioral approaches have been successful in helping
             patients with chronic pain reduce symptoms and regain
             functioning. However, most patients lack access to such
             treatments. We are conducting a pragmatic clinical trial to
             test the hypothesis that patients who receive an
             interdisciplinary biopsychosocial intervention, the Pain
             Program for Active Coping and Training (PPACT), at their
             primary care clinic will have a greater reduction in pain
             impact in the year following than patients receiving usual
             care.This is an effectiveness-implementation hybrid
             pragmatic clinical trial in which we randomize clusters of
             primary care providers and their patients with chronic pain
             who are on long-term opioid therapy to 1) receive an
             interdisciplinary behavioral intervention in conjunction
             with their current health care or 2) continue with current
             health care services. Our primary outcome is pain impact (a
             composite of pain intensity and pain-related interference)
             measured using the PEG, a validated three-item assessment.
             Secondary outcomes include pain-related disability, patient
             satisfaction, opioids dispensed and health care utilization.
             An economic evaluation assesses the resources and costs
             necessary to deliver the intervention and its
             cost-effectiveness compared with usual care. A formative
             evaluation employs mixed methods to understand the context
             for implementation in the participating health care
             systems.This trial will inform the feasibility of
             implementing interdisciplinary behavioral approaches to pain
             management in the primary care setting, potentially
             providing a more effective, safer, and more satisfactory
             alternative to opioid-based chronic pain treatment. Clinical
             Trials Registration Number: NCT02113592.},
   Doi = {10.1016/j.cct.2018.02.015},
   Key = {fds333734}
}

@article{fds332333,
   Author = {Lumley, MA and Anderson, T and Ankawi, B and Goldman, G and Perri, LM and Bianco, JA and Keefe, FJ},
   Title = {The working alliance and Clinician-assisted Emotional
             Disclosure for rheumatoid arthritis.},
   Journal = {Journal of Psychosomatic Research},
   Volume = {104},
   Pages = {9-15},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jpsychores.2017.11.004},
   Abstract = {The working alliance predicts improvement following general
             psychotherapy, but how it operates in brief interventions
             conducted with medically ill patients is unknown. Also, the
             role of the working alliance may differ in emotion-focused
             versus educational interventions.We report secondary
             analyses of a randomized clinical trial (Keefe et al.) [35],
             in which patients with rheumatoid arthritis (RA) received
             four nurse-provided sessions of either a) Clinician-assisted
             Emotional Disclosure (CAED), which emphasized the
             disclosure, expression, and processing of emotions related
             to stressful events; or b) Arthritis Education (AE), which
             provided basic education about RA. The Working Alliance
             Inventory was completed by both patient and nurse after each
             session. Patients were evaluated on multiple health measures
             at baseline and 1, 3, and 12months post-treatment.Analyses
             compared the alliance between interventions and related the
             alliance to outcomes within interventions. Patients in CAED
             reported a lower alliance than patients in AE.
             Interestingly, in CAED, lower alliance ratings predicted
             better outcomes (improved functioning, lower pain behaviors,
             lower inflammation, lower daily stress), whereas in AE, the
             working alliance was largely not predictive of
             outcomes.Having nurses encourage emotional disclosure among
             patients with RA reduced the patients' working alliance, but
             a lower alliance nonetheless predicted better patient
             outcomes, perhaps reflecting successful engagement in an
             intervention that is emotionally and relationally
             challenging. The level and predictive validity of the
             working alliance likely depends on patient, provider, and
             intervention factors, and further study of the working
             alliance in psychosocial interventions in the medical
             context is needed.},
   Doi = {10.1016/j.jpsychores.2017.11.004},
   Key = {fds332333}
}

@article{fds331439,
   Author = {Porter, LS and Keefe, FJ},
   Title = {Couple-based communication interventions for cancer: moving
             beyond a 'one size fits all' approach.},
   Journal = {Acta Oncologica (Informa)},
   Pages = {1-2},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1080/0284186x.2017.1400687},
   Doi = {10.1080/0284186x.2017.1400687},
   Key = {fds331439}
}

@article{fds333735,
   Author = {Gerhart, JI and Burns, JW and Bruehl, S and Smith, DA and Post, KM and Porter, LS and Schuster, E and Buvanendran, A and Fras, AM and Keefe,
             FJ},
   Title = {Variability in negative emotions among individuals with
             chronic low back pain: relationships with pain and
             function.},
   Journal = {PAIN},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1097/j.pain.0000000000001102},
   Abstract = {Chronic pain is associated with elevated negative emotions,
             and resources needed to adaptively regulate these emotions
             can be depleted during prolonged pain. Studies of links
             between pain, function, and negative emotions in people with
             chronic pain, however, have focused almost exclusively on
             relationships among mean levels of these factors. Indexes
             that may reflect aspects of emotion regulation have
             typically not been analyzed. We propose that 1 index of
             emotion regulation is variability in emotion over time as
             opposed to average emotion over time. The sample was 105
             people with chronic low back pain and 105 of their pain-free
             spouses. They completed electronic diary measures 5x/d for
             14 consecutive days, producing 70 observations per person
             from which we derived estimates of within-subject variance
             in negative emotions. Location-scale models were used to
             simultaneously model predictors of both mean level and
             variance in patient negative emotions over time. Patients
             reported significantly more variability in negative emotions
             compared to their spouses. Patients who reported higher
             average levels of pain, pain interference, and downtime
             reported significantly higher levels of variability in
             negative emotions. Spouse-observed pain and pain behaviors
             were also associated with greater variability in patients'
             negative emotions. Test of the inverse associations between
             negative emotion level and variability in pain and function
             were significant but weaker in magnitude. These findings
             support the notion that chronic pain may erode negative
             emotion regulation resources, to the potential detriment of
             intra- and inter-personal function.},
   Doi = {10.1097/j.pain.0000000000001102},
   Key = {fds333735}
}

@article{fds333736,
   Author = {Burns, JW and Post, KM and Smith, DA and Porter, LS and Buvanendran, A and Fras, AM and Keefe, FJ},
   Title = {Spouse criticism and hostility during marital interaction:
             effects on pain intensity and behaviors among individuals
             with chronic low back pain.},
   Journal = {PAIN},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1097/j.pain.0000000000001037},
   Abstract = {Individuals with chronic pain may experience negative
             responses from spouse, family, and friends. Responses such
             as overt criticism and hostility may be associated with
             worsening pain and function for chronic pain sufferers. We
             used a laboratory procedure to evaluate whether variability
             in spouse criticism/hostility exhibited toward chronic low
             back pain (CLBP) patients during a conflictual discussion
             predicted variability in patient pain and function during a
             subsequent pain-induction task. Chronic low back pain
             patients (n = 71) and their spouses (n = 71) participated in
             a 10-minute discussion followed by the patient undergoing a
             10-minute structured pain behavior task (SPBT). Spouse
             criticism/hostility perceived by patients and patient Beck
             Depression Inventory-II (BDI) scores correlated
             significantly and positively with pain intensity during the
             SPBT, whereas perceived spouse hostility, patient BDI
             scores, and spouse trait hostility correlated significantly
             and positively with observed pain behaviors during the SPBT.
             Spouse criticism/hostility coded by raters from video
             recordings interacted significantly with patient BDI scores,
             such that observed spouse criticism/hostility was related
             significantly and positively with pain behaviors only for
             patients with high BDI scores. Patient sex interacted
             significantly with observed spouse criticism/hostility, such
             that observed spouse criticism/hostility was related
             significantly and positively with pain behaviors only for
             female patients. Results support the hypothesis that spouse
             criticism and hostility-actually expressed or perceived-may
             worsen CLBP patient symptoms. Further, women patients and
             patients high in depressive symptoms appeared most
             vulnerable to spouse criticism/hostility. Thus, negative
             marital communication patterns may be appropriate targets
             for intervention, especially among these 2 at risk
             groups.},
   Doi = {10.1097/j.pain.0000000000001037},
   Key = {fds333736}
}

@article{fds330576,
   Author = {Lefebvre, JC and Jensen, MP and Waters, SJ and Molton, IR and Keefe, FJ and Caldwell, DS},
   Title = {The development and assessment of the Worry About Pain
             Questionnaire.},
   Journal = {European Journal of Pain},
   Volume = {21},
   Number = {7},
   Pages = {1154-1164},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1002/ejp.1015},
   Abstract = {Worry can be conceptualized as a cognitive-affective
             automatic process initiated in order to address uncertainty
             and potential personal inadequacies that could result in
             negative outcomes. The purpose of the current study was to
             develop a measure of pain-specific worry - the Worry About
             Pain Questionnaire (WAPQ).In study 1, responses of 335
             pain-free participants were used to complete an item
             analysis and exploratory factors analysis to develop and
             assess the internal structure of the WAPQ. Study 2 included
             224 pain-free participants who completed the WAPQ in order
             to confirm its factor structure, and to examine its relation
             to the experience of acute experimental pain. In study 3,
             137 individuals with persistent pain were asked to complete
             the WAPQ as well as measures of pain and depressive
             symptoms.The resulting 15-item measure assesses
             uncertainties and potential negative outcomes related to the
             experience of pain. The results of the exploratory and
             confirmatory factor analyses showed a two-factor structure.
             Across all studies, the WAPQ was found to be related to
             measures of pain in clinical and non-clinical samples, acute
             experimental pain stimuli, as well as pain anxiety, pain
             catastrophizing, fear of pain, rumination and depressive
             symptomatology.The results suggest that the WAPQ is a
             reliable and valid measure for the assessment of worry about
             pain that can be used to understand how pain-specific
             worries are related to the experience and impact of pain
             across different populations.Worry has been assessed in pain
             populations using measures that assess worry in general. The
             current study shows a relationship between pain-specific
             worry and the experience of pain. Further, worry about pain
             is related to but not synonymous with pain
             catastrophizing.},
   Doi = {10.1002/ejp.1015},
   Key = {fds330576}
}

@article{fds319635,
   Author = {Porter, LS and Keefe, FJ and Baucom, DH and Olsen, M and Zafar, SY and Uronis, H},
   Title = {A randomized pilot trial of a videoconference couples
             communication intervention for advanced GI
             cancer.},
   Journal = {Psycho-Oncology},
   Volume = {26},
   Number = {7},
   Pages = {1027-1035},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1002/pon.4121},
   Abstract = {This study aims to test the feasibility and preliminary
             efficacy of a couple-based communication intervention for
             advanced GI cancer delivered via videoconference.Thirty-two
             couples were randomly assigned to either couples
             communication skills training (CCST) or an education
             comparison intervention, both delivered via videoconference.
             Participation was limited to couples who reported
             communication difficulties at screening. Patients and
             partners completed measures of relationship functioning and
             individual functioning at baseline and post-intervention.Eighty-eight
             percent of randomized dyads completed all six sessions and
             reported high levels of satisfaction with the intervention.
             Between-group effect sizes suggested that the CCST
             intervention led to improvements in relationship
             satisfaction for patients and partners and to improvements
             in intimacy and communication for patients.A couples-based
             communication intervention delivered via videoconference is
             feasible and acceptable in the context of advanced cancer.
             Preliminary findings suggest that the intervention shows
             promise in contributing to enhanced relationship
             functioning. Copyright © 2016 John Wiley & Sons,
             Ltd.},
   Doi = {10.1002/pon.4121},
   Key = {fds319635}
}

@article{fds330867,
   Author = {Parmelee, PA and Cox, BS and DeCaro, JA and Keefe, FJ and Smith,
             DM},
   Title = {Racial/ethnic differences in sleep quality among older
             adults with osteoarthritis.},
   Journal = {Sleep health},
   Volume = {3},
   Number = {3},
   Pages = {163-169},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.sleh.2017.03.010},
   Abstract = {To examine racial/ethnic differences in sleep quality and
             the pain-sleep association among older adults with
             osteoarthritis of the knee.Baseline interview followed by a
             7-day microlongitudinal study using accelerometry and
             self-reports.Participants were community residents in
             western Alabama and Long Island, NY.Ninety-six African
             Americans (AAs) and 128 non-Hispanic whites (NHWs) with
             physician-diagnosed knee osteoarthritis, recruited from a
             variety of clinical and community settings.Self-reports
             yielded demographics, body mass index, physical health
             problems, and depressive symptoms. Sleep quality was
             measured for 3 to 7 nights using wrist-worn accelerometers;
             pain was self-reported daily over the same period.With
             demographics and health controlled, AAs displayed poorer
             sleep efficiency, greater time awake after sleep onset and
             sleep fragmentation, and marginally more awakenings during
             the night, but no differences in total sleep time. AAs also
             showed greater night-to-night variability in number of
             awakenings and sleep fragmentation, and marginally greater
             variability in total sleep time and sleep efficiency. Sleep
             quality was not associated with pain either the day before
             sleep or the day after. Average daily pain interacted with
             race, whereas AAs displayed no effect of pain on sleep
             efficiency, NHWs exhibited better sleep efficiency at higher
             levels of average pain.These data corroborate previous
             studies documenting poorer sleep among AAs vs NHWs. The
             findings of greater night-to-night variability in sleep
             among AAs, as well as a negative association of pain with
             sleep quality among NHWs, are unique. Further study is
             needed to elucidate these findings.},
   Doi = {10.1016/j.sleh.2017.03.010},
   Key = {fds330867}
}

@article{fds330577,
   Author = {Gerhart, JI and Burns, JW and Post, KM and Smith, DA and Porter, LS and Burgess, HJ and Schuster, E and Buvanendran, A and Fras, AM and Keefe,
             FJ},
   Title = {Relationships Between Sleep Quality and Pain-Related Factors
             for People with Chronic Low Back Pain: Tests of Reciprocal
             and Time of Day Effects.},
   Journal = {Annals of Behavioral Medicine},
   Volume = {51},
   Number = {3},
   Pages = {365-375},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s12160-016-9860-2},
   Abstract = {Poor sleep quality among people with chronic low back pain
             appears to be related to worse pain, affect, poor physical
             function, and pain catastrophizing. The causal direction
             between poor sleep and pain remains an open question,
             however, as does whether sleep quality exerts effects on low
             back pain differently across the course of the day.This
             daily diary study examined lagged temporal associations
             between prior night sleep quality and subsequent day pain,
             affect, physical function and pain catastrophizing, the
             reverse lagged temporal associations between prior day
             pain-related factors and subsequent night sleep quality, and
             whether the time of day during which an assessment was made
             moderated these temporal associations.Chronic low back pain
             patients (n = 105) completed structured electronic diary
             assessments five times per day for 14 days. Items included
             patient ratings of their pain, affect, physical function,
             and pain catastrophizing.Collapsed across all observations,
             poorer sleep quality was significantly related to higher
             pain ratings, higher negative affect, lower positive affect,
             poorer physical function, and higher pain catastrophizing.
             Lagged analyses averaged across the day revealed that poorer
             prior night sleep quality significantly predicted greater
             next day patient ratings of pain, and poorer physical
             function and higher pain catastrophizing. Prior poorer night
             sleep quality significantly predicted greater reports of
             pain, and poorer physical function, and higher pain
             catastrophizing, especially during the early part of the
             day. Sleep quality × time of day interactions showed that
             poor sleepers reported high pain, and negative mood and low
             function uniformly across the day, whereas good sleepers
             reported relatively good mornings, but showed pain, affect
             and function levels comparable to poor sleepers by the end
             of the day. Analyses of the reverse causal pathway were
             mostly nonsignificant.Sleep quality appears related not only
             to pain intensity but also to a wide range of patient mood
             and function factors. A good night's sleep also appears to
             offer only temporary respite, suggesting that comprehensive
             interventions for chronic low back pain not only should
             include attention to sleep problems but also focus on
             problems with pain appraisals and coping.},
   Doi = {10.1007/s12160-016-9860-2},
   Key = {fds330577}
}

@article{fds331440,
   Title = {Internet-Delivered Exercise and Pain-Coping Skills Training
             for Chronic Knee Pain},
   Journal = {Annals of internal medicine},
   Volume = {166},
   Number = {7},
   Pages = {I-13-I-13},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.7326/P17-9031},
   Doi = {10.7326/P17-9031},
   Key = {fds331440}
}

@article{fds333023,
   Author = {Keefe, FJ and Sommer, C},
   Title = {An introduction to the Biennial Review of
             Pain.},
   Journal = {PAIN},
   Volume = {158 Suppl 1},
   Pages = {S1},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1097/j.pain.0000000000000877},
   Doi = {10.1097/j.pain.0000000000000877},
   Key = {fds333023}
}

@article{fds333024,
   Author = {Bennell, KL and Nelligan, R and Dobson, F and Rini, C and Keefe, F and Kasza, J and French, S and Bryant, C and Dalwood, A and Abbott, JH and Hinman, RS},
   Title = {Effectiveness of an Internet-Delivered Exercise and
             Pain-Coping Skills Training Intervention for Persons With
             Chronic Knee Pain: A Randomized Trial.},
   Journal = {Annals of internal medicine},
   Volume = {166},
   Number = {7},
   Pages = {453-462},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.7326/m16-1714},
   Abstract = {Effective, accessible biopsychosocial treatments are needed
             to manage chronic knee pain on a population level.To
             evaluate the effectiveness of Internet-delivered,
             physiotherapist-prescribed home exercise and pain-coping
             skills training (PCST).Pragmatic parallel-group randomized,
             controlled trial. (Australian New Zealand Clinical Trials
             Registry: ACTRN12614000243617).Community (Australia).148
             persons aged 50 years or older with chronic knee pain.The
             intervention was delivered via the Internet and included
             educational material, 7 videoconferencing (Skype
             [Microsoft]) sessions with a physiotherapist for home
             exercise, and a PCST program over 3 months. The control was
             Internet-based educational material.Primary outcomes were
             pain during walking (11-point numerical rating scale) and
             physical function (Western Ontario and McMaster Universities
             Osteoarthritis Index) at 3 months. Secondary outcomes were
             knee pain, quality of life, global change (overall, pain,
             and functional status), arthritis self-efficacy, coping, and
             pain catastrophizing. Outcomes were also measured at 9
             months.Of participants enrolled, 139 (94%) completed primary
             outcome measures at 3 months and 133 (90%) completed
             secondary outcome measures at 9 months; multiple imputation
             was used for missing data. The intervention group reported
             significantly more improvement in pain (mean difference, 1.6
             units [95% CI, 0.9 to 2.3 units]) and physical function
             (mean difference, 9.3 units [CI, 5.9 to 12.7 units]) than
             the control group at 3 months, and improvements were
             sustained at 9 months (mean differences, 1.1 units [CI, 0.4
             to 1.8 units] and 7.0 units [CI, 3.4 to 10.5 units],
             respectively). Intervention participants showed
             significantly more improvement in most secondary outcomes
             than control participants. At both time points,
             significantly more intervention participants reported global
             improvements.Participants were unblinded.For persons with
             chronic knee pain, Internet-delivered, physiotherapist-prescribed
             exercise and PCST provide clinically meaningful improvements
             in pain and function that are sustained for at least 6
             months.National Health and Medical Research
             Council.},
   Doi = {10.7326/m16-1714},
   Key = {fds333024}
}

@article{fds330906,
   Author = {Edmond, SN and Shelby, RA and Keefe, FJ and Fisher, HM and Schmidt, JE and Soo, MS and Skinner, CS and Ahrendt, GM and Manculich, J and Sumkin, JH and Zuley, ML and Bovbjerg, DH},
   Title = {Persistent Breast Pain Among Women With Histories of
             Breast-conserving Surgery for Breast Cancer Compared With
             Women Without Histories of Breast Surgery or
             Cancer.},
   Journal = {Clinical Journal of Pain},
   Volume = {33},
   Number = {1},
   Pages = {51-56},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1097/ajp.0000000000000377},
   Abstract = {This study compared persistent breast pain among women who
             received breast-conserving surgery for breast cancer and
             women without a history of breast cancer.Breast cancer
             survivors (n=200) were recruited at their first postsurgical
             surveillance mammogram (6 to 15 mo postsurgery). Women
             without a breast cancer history (n=150) were recruited at
             the time of a routine screening mammogram. All women
             completed measures of breast pain, pain interference with
             daily activities and intimacy, worry about breast pain,
             anxiety symptoms, and depression symptoms. Demographic and
             medical information were also collected.Persistent breast
             pain (duration ≥6 mo) was reported by 46.5% of breast
             cancer survivors and 12.7% of women without a breast cancer
             history (P<0.05). Breast cancer survivors also had
             significantly higher rates of clinically significant
             persistent breast pain (pain intensity score ≥3/10), as
             well as higher average breast pain intensity and
             unpleasantness scores. Breast cancer survivors with
             persistent breast pain had significantly higher levels of
             depressive symptoms, as well as pain worry and interference,
             compared with survivors without persistent breast pain or
             women without a breast cancer history. Anxiety symptoms were
             significantly higher in breast cancer survivors with
             persistent breast pain compared with women without a breast
             cancer history.Results indicate that persistent breast pain
             negatively impacts women with a history of breast-conserving
             cancer surgery compared with women without that history.
             Strategies to ameliorate persistent breast pain and to
             improve adjustment among women with persistent breast pain
             should be explored for incorporation into standard care for
             breast cancer survivors.},
   Doi = {10.1097/ajp.0000000000000377},
   Key = {fds330906}
}


%% Keefe, Richard S.   
@article{fds332787,
   Author = {Xavier, RM and Dungan, JR and Keefe, RSE and Vorderstrasse,
             A},
   Title = {Polygenic signal for symptom dimensions and cognitive
             performance in patients with chronic schizophrenia.},
   Journal = {Schizophrenia research. Cognition},
   Volume = {12},
   Pages = {11-19},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.scog.2018.01.001},
   Abstract = {Genetic etiology of psychopathology symptoms and cognitive
             performance in schizophrenia is supported by candidate gene
             and polygenic risk score (PRS) association studies. Such
             associations are reported to be dependent on several factors
             - sample characteristics, illness phase, illness severity
             etc. We aimed to examine if schizophrenia PRS predicted
             psychopathology symptoms and cognitive performance in
             patients with chronic schizophrenia. We also examined if
             schizophrenia associated autosomal loci were associated with
             specific symptoms or cognitive domains. Case-only analysis
             using data from the Clinical Antipsychotics Trials of
             Intervention Effectiveness-Schizophrenia trials
             (n = 730). PRS was constructed using Psychiatric
             Genomics Consortium (PGC) leave one out genome wide
             association analysis as the discovery data set. For
             candidate region analysis, we selected 105-schizophrenia
             associated autosomal loci from the PGC study. We found a
             significant effect of PRS on positive symptoms at
             p-threshold (PT ) of 0.5 (R2 = 0.007, p = 0.029,
             empirical p = 0.029) and negative symptoms at PT of
             1e-07 (R2 = 0.005, p = 0.047, empirical
             p = 0.048). For models that additionally controlled for
             neurocognition, best fit PRS predicted positive (p-threshold
             0.01, R2 = 0.007, p = 0.013, empirical
             p = 0.167) and negative symptoms (p-threshold 0.1,
             R2 = 0.012, p = 0.004, empirical p = 0.329). No
             associations were seen for overall neurocognitive and social
             cognitive performance tests. Post-hoc analyses revealed that
             PRS predicted working memory and vigilance performance but
             did not survive correction. No candidate regions that
             survived multiple testing corrections were associated with
             either symptoms or cognitive performance. Our findings point
             to potentially distinct pathogenic mechanisms for
             schizophrenia symptoms.},
   Doi = {10.1016/j.scog.2018.01.001},
   Key = {fds332787}
}

@article{fds327061,
   Author = {Kendler, KS and Ohlsson, H and Keefe, RSE and Sundquist, K and Sundquist, J},
   Title = {The joint impact of cognitive performance in adolescence and
             familial cognitive aptitude on risk for major psychiatric
             disorders: a delineation of four potential pathways to
             illness.},
   Journal = {Molecular Psychiatry},
   Volume = {23},
   Number = {4},
   Pages = {1076-1083},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1038/mp.2017.78},
   Abstract = {How do joint measures of premorbid cognitive ability and
             familial cognitive aptitude (FCA) reflect risk for a
             diversity of psychiatric and substance use disorders? To
             address this question, we examined, using Cox models, the
             predictive effects of school achievement (SA) measured at
             age 16 and FCA-assessed from SA in siblings and cousins, and
             educational attainment in parents-on risk for 12 major
             psychiatric syndromes in 1 140 608 Swedes born
             1972-1990. Four developmental patterns emerged. In the
             first, risk was predicted jointly by low levels of SA and
             high levels of FCA-that is a level of SA lower than would be
             predicted from the FCA. This pattern was strongest in autism
             spectrum disorders and schizophrenia, and weakest in bipolar
             illness. In these disorders, a pathologic process seems to
             have caused cognitive functioning to fall substantially
             short of familial potential. In the second pattern, seen in
             the internalizing conditions of major depression and anxiety
             disorders, risk was associated with low SA but was unrelated
             to FCA. Externalizing disorders-drug abuse and alcohol use
             disorders-demonstrated the third pattern, in which risk was
             predicted jointly by low SA and low FCA. The fourth pattern,
             seen in eating disorders, was directly opposite of that
             observed in externalizing disorders with risk associated
             with high SA and high FCA. When measured together,
             adolescent cognitive ability and FCA identified four
             developmental patterns leading to diverse psychiatric
             disorders. The value of cognitive assessments in psychiatric
             research can be substantially increased by also evaluating
             familial cognitive potential.},
   Doi = {10.1038/mp.2017.78},
   Key = {fds327061}
}

@article{fds327210,
   Author = {Xavier, RM and Pan, W and Dungan, JR and Keefe, RSE and Vorderstrasse,
             A},
   Title = {Unraveling interrelationships among psychopathology
             symptoms, cognitive domains and insight dimensions in
             chronic schizophrenia.},
   Journal = {Schizophrenia Research},
   Volume = {193},
   Pages = {83-90},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.schres.2017.07.002},
   Abstract = {Insight in schizophrenia is long known to have a complex
             relationship with psychopathology symptoms and cognition.
             However, very few studies have examined models that explain
             these interrelationships.In a large sample derived from the
             NIMH Clinical Antipsychotic Trials of Intervention
             Effectiveness (CATIE) schizophrenia trial (N=1391), we
             interrogated these interrelationships for potential causal
             pathways using structural equation modeling. Using the NIMH
             consensus model, latent variables were constructed for
             psychopathology symptom dimensions, including positive,
             negative, disorganized, excited and depressed from the
             Positive and Negative Syndrome Scale (PANSS) items.
             Neurocognitive variables were created from five predefined
             domains of working memory, verbal memory, reasoning,
             vigilance and processing speed. Illness insight and
             treatment insight were tested using latent variables
             constructed from the Illness and Treatment Attitude
             Questionnaire (ITAQ).Disorganized symptoms had the strongest
             effect on insight. Illness insight mediated the relationship
             of positive, depressed, and disorganized symptoms with
             treatment insight. Neurocognition mediated the relationship
             between disorganized and treatment insight and depressed
             symptoms and treatment insight. There was no effect of
             negative symptoms on either illness insight or treatment
             insight. Taken together, our results indicate overlapping
             and unique relational paths for illness and treatment
             insight dimensions, which could suggest differences in
             causal mechanisms and potential interventions to improve
             insight.},
   Doi = {10.1016/j.schres.2017.07.002},
   Key = {fds327210}
}

@article{fds326634,
   Author = {Hochberger, WC and Combs, T and Reilly, JL and Bishop, JR and Keefe,
             RSE and Clementz, BA and Keshavan, MS and Pearlson, GD and Tamminga, CA and Hill, SK and Sweeney, JA},
   Title = {Deviation from expected cognitive ability across psychotic
             disorders.},
   Journal = {Schizophrenia Research},
   Volume = {192},
   Pages = {300-307},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.schres.2017.05.019},
   Abstract = {Patients with schizophrenia show a deficit in cognitive
             ability compared to estimated premorbid and familial
             intellectual abilities. However, the degree to which this
             pattern holds across psychotic disorders and is familial is
             unclear. The present study examined deviation from expected
             cognitive level in schizophrenia, schizoaffective disorder,
             and psychotic bipolar disorder probands and their
             first-degree relatives. Using a norm-based regression
             approach, parental education and WRAT-IV Reading scores
             (both significant predictors of cognitive level in the
             healthy control group) were used to predict global
             neuropsychological function as measured by the composite
             score from the Brief Assessment of Cognition in
             Schizophrenia (BACS) test in probands and relatives. When
             compared to healthy control group, psychotic probands showed
             a significant gap between observed and predicted BACS
             composite scores and a greater likelihood of robust
             cognitive decline. This effect was not seen in unaffected
             relatives. While BACS and WRAT-IV Reading scores were
             themselves highly familial, the decline in cognitive
             function from expectation had lower estimates of
             familiality. Thus, illness-related factors such as
             epigenetic, treatment, or pathophysiological factors may be
             important causes of illness related decline in cognitive
             abilities across psychotic disorders. This is consistent
             with the markedly greater level of cognitive impairment seen
             in affected individuals compared to their unaffected family
             members.},
   Doi = {10.1016/j.schres.2017.05.019},
   Key = {fds326634}
}

@article{fds326113,
   Author = {Eum, S and Hill, SK and Rubin, LH and Carnahan, RM and Reilly, JL and Ivleva, EI and Keedy, SK and Tamminga, CA and Pearlson, GD and Clementz,
             BA and Gershon, ES and Keshavan, MS and Keefe, RSE and Sweeney, JA and Bishop, JR},
   Title = {Cognitive burden of anticholinergic medications in psychotic
             disorders.},
   Journal = {Schizophrenia Research},
   Volume = {190},
   Pages = {129-135},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.schres.2017.03.034},
   Abstract = {Patients with psychotic disorders are often treated with
             numerous medications, many of which have anticholinergic
             activity. We assessed cognition in relation to the
             cumulative anticholinergic burden of multiple drugs included
             in treatment regimens of participants from the
             Bipolar-Schizophrenia Network on Intermediate Phenotypes
             (B-SNIP) study.Clinically stable participants with
             schizophrenia (n=206), schizoaffective disorder (n=131), and
             psychotic bipolar disorder (n=146) were examined.
             Anticholinergic properties of all scheduled drugs were
             quantified using the Anticholinergic Drug Scale (ADS). ADS
             scores were summed across individual drugs to create a total
             ADS burden score for each participant and examined in
             relation to the Brief Assessment of Cognition in
             Schizophrenia (BACS).Anticholinergic burden aggregated
             across all medications was inversely related to cognitive
             performance starting at ADS scores of 4 in participants with
             schizophrenia. Those with ADS scores ≥4 had lower
             composite BACS scores compared to those with ADS<4
             (p=0.004). Among BACS subtests, Verbal Memory was the most
             adversely affected by high anticholinergic burden. Despite
             similar anticholinergic burden scores across groups, a
             significant threshold effect of anticholinergic burden was
             not detected in schizoaffective or psychotic bipolar
             disorder.We identified an adverse effect threshold of
             anticholinergic burden on cognition in clinically stable
             participants with schizophrenia. This relationship was not
             identified in affective psychoses. Examination of other
             medications, doses, and clinical measures did not account
             for these findings. Patients with schizophrenia may have
             increased cognitive susceptibility to anticholinergic
             medications and the aggregate effects of one's medication
             regimen may be important to consider in clinical
             practice.},
   Doi = {10.1016/j.schres.2017.03.034},
   Key = {fds326113}
}

@article{fds330046,
   Author = {Ang, MS and Abdul Rashid and NA and Lam, M and Rapisarda, A and Kraus, M and Keefe, RSE and Lee, J},
   Title = {The Impact of Medication Anticholinergic Burden on Cognitive
             Performance in People With Schizophrenia.},
   Journal = {Journal of Clinical Psychopharmacology},
   Volume = {37},
   Number = {6},
   Pages = {651-656},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1097/jcp.0000000000000790},
   Abstract = {Cognitive deficits are prevalent in people with
             schizophrenia and associated with functional impairments. In
             addition to antipsychotics, pharmacotherapy in schizophrenia
             often includes other psychotropics, and some of these agents
             possess anticholinergic properties, which may impair
             cognition. The objective of this study was to explore the
             association between medication anticholinergic burden and
             cognition in schizophrenia.Seven hundred five individuals
             with schizophrenia completed a neuropsychological battery
             comprising Judgment of Line Orientation Test, Wechsler
             Abbreviated Scale of Intelligence Matrix Reasoning,
             Continuous Performance Test-Identical Pairs Version, and the
             Brief Assessment of Cognition in Schizophrenia. Cognitive g
             and 3 cognitive factor scores that include executive
             function, memory/fluency, and speed of processing/vigilance,
             which were derived from a previously published analysis,
             were entered as cognitive variables. Anticholinergic burden
             was computed using 2 anticholinergic scales: Anticholinergic
             Burden Scale and Anticholinergic Drug Scale. Duration and
             severity of illness, antipsychotic dose, smoking status,
             age, and sex were included as covariates.Anticholinergic
             burden was associated with poorer cognitive performance in
             cognitive g, all 3 cognitive domains and most cognitive
             tasks in multivariate analyses. The associations were
             statistically significant, but the effect sizes were small
             (for Anticholinergic Burden Scale, Cohen f = 0.008; for
             Anticholinergic Drug Scale, Cohen f = 0.017).Although our
             results showed a statistically significant association
             between medications with anticholinergic properties and
             cognition in people with schizophrenia, the impact is of
             doubtful or minimal clinical significance.},
   Doi = {10.1097/jcp.0000000000000790},
   Key = {fds330046}
}

@article{fds332788,
   Author = {Khan, A and Liharska, L and Harvey, PD and Atkins, A and Ulshen, D and Keefe, RSE},
   Title = {Negative Symptom Dimensions of the Positive and Negative
             Syndrome Scale Across Geographical Regions: Implications for
             Social, Linguistic, and Cultural Consistency.},
   Journal = {Innovations in Clinical Neuroscience},
   Volume = {14},
   Number = {11-12},
   Pages = {30-40},
   Year = {2017},
   Month = {December},
   Abstract = {Objective: Recognizing the discrete dimensions that underlie
             negative symptoms in schizophrenia and how these dimensions
             are understood across localities might result in better
             understanding and treatment of these symptoms. To this end,
             the objectives of this study were to 1) identify the
             Positive and Negative Syndrome Scale negative symptom
             dimensions of expressive deficits and experiential deficits
             and 2) analyze performance on these dimensions over 15
             geographical regions to determine whether the items defining
             them manifest similar reliability across these regions.
             Design: Data were obtained for the baseline Positive and
             Negative Syndrome Scale visits of 6,889 subjects across 15
             geographical regions. Using confirmatory factor analysis, we
             examined whether a two-factor negative symptom structure
             that is found in schizophrenia (experiential deficits and
             expressive deficits) would be replicated in our sample, and
             using differential item functioning, we tested the degree to
             which specific items from each negative symptom subfactor
             performed across geographical regions in comparison with the
             United States. Results: The two-factor negative symptom
             solution was replicated in this sample. Most geographical
             regions showed moderate-to-large differential item
             functioning for Positive and Negative Syndrome Scale
             expressive deficit items, especially N3 Poor Rapport, as
             compared with Positive and Negative Syndrome Scale
             experiential deficit items, showing that these items might
             be interpreted or scored differently in different regions.
             Across countries, except for India, the differential item
             functioning values did not favor raters in the United
             States. Conclusion: These results suggest that the Positive
             and Negative Syndrome Scale negative symptom factor can be
             better represented by a two-factor model than by a
             single-factor model. Additionally, the results show
             significant differences in responses to items representing
             the Positive and Negative Syndrome Scale expressive factors,
             but not the experiential factors, across regions. This could
             be due to a lack of equivalence between the original and
             translated versions, cultural differences with the
             interpretation of items, dissimilarities in rater training,
             or diversity in the understanding of scoring anchors.
             Knowing which items are challenging for raters across
             regions can help to guide Positive and Negative Syndrome
             Scale training and improve the results of international
             clinical trials aimed at negative symptoms.},
   Key = {fds332788}
}

@article{fds332789,
   Author = {Harvey, PD and Khan, A and Keefe, RSE},
   Title = {Using the Positive and Negative Syndrome Scale (PANSS) to
             Define Different Domains of Negative Symptoms: Prediction of
             Everyday Functioning by Impairments in Emotional Expression
             and Emotional Experience.},
   Journal = {Innovations in Clinical Neuroscience},
   Volume = {14},
   Number = {11-12},
   Pages = {18-22},
   Year = {2017},
   Month = {December},
   Abstract = {Background: Reduced emotional experience and expression are
             two domains of negative symptoms. The authors assessed these
             two domains of negative symptoms using previously developed
             Positive and Negative Syndrome Scale (PANSS) factors. Using
             an existing dataset, the authors predicted three different
             elements of everyday functioning (social, vocational, and
             everyday activities) with these two factors, as well as with
             performance on measures of functional capacity. Methods: A
             large (n=630) sample of people with schizophrenia was used
             as the data source of this study. Using regression analyses,
             the authors predicted the three different aspects of
             everyday functioning, first with just the two Positive and
             Negative Syndrome Scale factors and then with a global
             negative symptom factor. Finally, we added neurocognitive
             performance and functional capacity as predictors. Results:
             The Positive and Negative Syndrome Scale reduced emotional
             experience factor accounted for 21 percent of the variance
             in everyday social functioning, while reduced emotional
             expression accounted for no variance. The total Positive and
             Negative Syndrome Scale negative symptom factor accounted
             for less variance (19%) than the reduced experience factor
             alone. The Positive and Negative Syndrome Scale expression
             factor accounted for, at most, one percent of the variance
             in any of the functional outcomes, with or without the
             addition of other predictors. Implications: Reduced
             emotional experience measured with the Positive and Negative
             Syndrome Scale, often referred to as "avolition and
             anhedonia," specifically predicted impairments in social
             outcomes. Further, reduced experience predicted social
             impairments better than emotional expression or the total
             Positive and Negative Syndrome Scale negative symptom
             factor. In this cross-sectional study, reduced emotional
             experience was specifically related with social outcomes,
             accounting for essentially no variance in work or everyday
             activities, and being the sole meaningful predictor of
             impairment in social outcomes.},
   Key = {fds332789}
}

@article{fds327060,
   Author = {Georgiades, A and Davis, VG and Atkins, AS and Khan, A and Walker, TW and Loebel, A and Haig, G and Hilt, DC and Dunayevich, E and Umbricht, D and Sand, M and Keefe, RSE},
   Title = {Psychometric characteristics of the MATRICS Consensus
             Cognitive Battery in a large pooled cohort of stable
             schizophrenia patients.},
   Journal = {Schizophrenia Research},
   Volume = {190},
   Pages = {172-179},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.schres.2017.03.040},
   Abstract = {The MATRICS Consensus Cognitive Battery (MCCB) was developed
             to assess cognitive treatment effects in schizophrenia
             clinical trials, and is considered the FDA gold standard
             outcome measure for that purpose. The aim of the present
             study was to establish pre-treatment psychometric
             characteristics of the MCCB in a large pooled sample. The
             dataset included 2616 stable schizophrenia patients enrolled
             in 15 different clinical trials between 2007 and 2016 within
             the United States (94%) and Canada (6%). The MCCB was
             administered twice prior to the initiation of treatment in
             1908 patients. Test-retest reliability and practice effects
             of the cognitive composite score, the neurocognitive
             composite score, which excludes the domain Social Cognition,
             and the subtests/domains were examined using Intra-Class
             Correlations (ICC) and Cohen's d. Simulated regression
             models explored which domains explained the greatest portion
             of variance in composite scores. Test-retest reliability was
             high (ICC=0.88) for both composite scores. Practice effects
             were small for the cognitive (d=0.15) and neurocognitive
             (d=0.17) composites. Simulated bootstrap regression analyses
             revealed that 3 of the 7 domains explained 86% of the
             variance for both composite scores. The domains that entered
             most frequently in the top 3 positions of the regression
             models were Speed of Processing, Working Memory, and Visual
             Learning. Findings provide definitive psychometric
             characteristics and a benchmark comparison for clinical
             trials using the MCCB. The test-retest reliability of the
             MCCB composite scores is considered excellent and the
             learning effects are small, fulfilling two of the key
             criteria for outcome measures in cognition clinical
             trials.},
   Doi = {10.1016/j.schres.2017.03.040},
   Key = {fds327060}
}

@article{fds329482,
   Author = {Xavier, RM and Vorderstrasse, A and Keefe, RSE and Dungan,
             JR},
   Title = {Genetic correlates of insight in schizophrenia.},
   Journal = {Schizophrenia Research},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.schres.2017.10.021},
   Abstract = {Insight in schizophrenia is clinically important as it is
             associated with several adverse outcomes. Genetic
             contributions to insight are unknown. We examined genetic
             contributions to insight by investigating if polygenic risk
             scores (PRS) and candidate regions were associated with
             insight.Schizophrenia case-only analysis of the Clinical
             Antipsychotics Trials of Intervention Effectiveness trial.
             Schizophrenia PRS was constructed using Psychiatric Genomics
             Consortium (PGC) leave-one out GWAS as discovery data set.
             For candidate regions, we selected 105 schizophrenia-associated
             autosomal loci and 11 schizophrenia-related oligodendrocyte
             genes. We used regressions to examine PRS associations and
             set-based testing for candidate analysis.We examined data
             from 730 subjects. Best-fit PRS at p-threshold of 1e-07 was
             associated with total insight (R(2)=0.005, P=0.05, empirical
             P=0.054) and treatment insight (R(2)=0.005, P=0.048,
             empirical P=0.048). For models that controlled for
             neurocognition, PRS significantly predicted treatment
             insight but at higher p-thresholds (0.1 to 0.5) but did not
             survive correction. Patients with highest polygenic burden
             had 5.9 times increased risk for poor insight compared to
             patients with lowest burden. PRS explained 3.2% (P=0.002,
             empirical P=0.011) of variance in poor insight. Set-based
             analyses identified two variants associated with poor
             insight- rs320703, an intergenic variant (within-set
             P=6e-04, FDR P=0.046) and rs1479165 in SOX2-OT (within-set
             P=9e-04, FDR P=0.046).To the best of our knowledge, this is
             the first study examining genetic basis of insight. We
             provide evidence for genetic contributions to impaired
             insight. Relevance of findings and necessity for replication
             are discussed.},
   Doi = {10.1016/j.schres.2017.10.021},
   Key = {fds329482}
}

@article{fds329375,
   Author = {Blokland, GAM and Del Re and EC and Mesholam-Gately, RI and Jovicich, J and Trampush, JW and Keshavan, MS and DeLisi, LE and Walters, JTR and Turner, JA and Malhotra, AK and Lencz, T and Shenton, ME and Voineskos,
             AN and Rujescu, D and Giegling, I and Kahn, RS and Roffman, JL and Holt,
             DJ and Ehrlich, S and Kikinis, Z and Dazzan, P and Murray, RM and Di Forti,
             M and Lee, J and Sim, K and Lam, M and Wolthusen, RPF and de Zwarte, SMC and Walton, E and Cosgrove, D and Kelly, S and Maleki, N and Osiecki, L and Picchioni, MM and Bramon, E and Russo, M and David, AS and Mondelli, V and Reinders, AATS and Falcone, MA et al.},
   Title = {The Genetics of Endophenotypes of Neurofunction to
             Understand Schizophrenia (GENUS) consortium: A collaborative
             cognitive and neuroimaging genetics project.},
   Journal = {Schizophrenia Research},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.schres.2017.09.024},
   Abstract = {Schizophrenia has a large genetic component, and the
             pathways from genes to illness manifestation are beginning
             to be identified. The Genetics of Endophenotypes of
             Neurofunction to Understand Schizophrenia (GENUS) Consortium
             aims to clarify the role of genetic variation in brain
             abnormalities underlying schizophrenia. This article
             describes the GENUS Consortium sample collection.We
             identified existing samples collected for schizophrenia
             studies consisting of patients, controls, and/or individuals
             at familial high-risk (FHR) for schizophrenia. Samples had
             single nucleotide polymorphism (SNP) array data or genomic
             DNA, clinical and demographic data, and neuropsychological
             and/or brain magnetic resonance imaging (MRI) data. Data
             were subjected to quality control procedures at a central
             site.Sixteen research groups contributed data from 5199
             psychosis patients, 4877 controls, and 725 FHR individuals.
             All participants have relevant demographic data and all
             patients have relevant clinical data. The sex ratio is 56.5%
             male and 43.5% female. Significant differences exist between
             diagnostic groups for premorbid and current IQ (both
             p<1×10-10). Data from a diversity of neuropsychological
             tests are available for 92% of participants, and 30% have
             structural MRI scans (half also have diffusion-weighted MRI
             scans). SNP data are available for 76% of participants. The
             ancestry composition is 70% European, 20% East Asian, 7%
             African, and 3% other.The Consortium is investigating the
             genetic contribution to brain phenotypes in a schizophrenia
             sample collection of >10,000 participants. The breadth of
             data across clinical, genetic, neuropsychological, and MRI
             modalities provides an important opportunity for elucidating
             the genetic basis of neural processes underlying
             schizophrenia.},
   Doi = {10.1016/j.schres.2017.09.024},
   Key = {fds329375}
}

@article{fds326635,
   Author = {Lam, M and Wang, M and Huang, W and Eng, GK and Rapisarda, A and Kraus, M and Kang, S and Keefe, RSE and Lee, J},
   Title = {Establishing the Brief Assessment of Cognition - Short
             form.},
   Journal = {Journal of Psychiatric Research},
   Volume = {93},
   Pages = {1-11},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.jpsychires.2017.05.006},
   Abstract = {The study aims to identify and validate a parsimonious
             subset of tests in the commonly used Brief Assessment of
             Cognition in Schizophrenia (BACS) that allows the evaluation
             of global cognitive ability. Several permutations of
             subtests from the BACS were examined to identify the best
             subset of tests to compose the short form measure. The Brief
             Assessment of Cognition-Short Form (BAC-SF) was evaluated
             for convergent validity in healthy and psychiatric samples
             (N = 3718). Verbal Memory, Digit Sequencing, and Symbol
             Coding subtests were found to best summarize the variance of
             composite scores in both Asian and US Norming samples
             (r = 0.91) indicating that BAC-SF is an appropriate
             approximation of cognitive deficits. Test re-test
             reliability of the BAC-SF was adequate (Intraclass
             Correlation Coefficient (ICC) = 0.73) and showed
             sufficient separation between healthy controls and
             schizophrenia (Average Predictive Accuracy = 79.9%;
             replication = 76.5%). Findings indicate that the BAC-SF an
             could be used as a cognitive screener for large-scale
             clinical and epidemiological studies. The short form does
             not replace the need for comprehensive neuropsychological
             batteries purposed for detailed neuropsychological and
             clinical investigation of cognitive function. Further
             replication of the construct might be necessary in other
             clinical populations.},
   Doi = {10.1016/j.jpsychires.2017.05.006},
   Key = {fds326635}
}

@article{fds330047,
   Author = {Cella, M and Stahl, D and Morris, S and Keefe, RSE and Bell, MD and Wykes,
             T},
   Title = {Effects of cognitive remediation on negative symptoms
             dimensions: exploring the role of working
             memory.},
   Journal = {Psychological Medicine},
   Pages = {1-9},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1017/s0033291717000757},
   Abstract = {Recent theories suggest that poor working memory (WM) may be
             the cognitive underpinning of negative symptoms in people
             with schizophrenia. In this study, we first explore the
             effect of cognitive remediation (CR) on two clusters of
             negative symptoms (i.e. expressive and social amotivation),
             and then assess the relevance of WM gains as a possible
             mediator of symptom improvement.Data were accessed for 309
             people with schizophrenia from the NIMH Database of
             Cognitive Training and Remediation Studies and a separate
             study. Approximately half the participants received CR and
             the rest were allocated to a control condition. All
             participants were assessed before and after therapy and at
             follow-up. Expressive negative symptoms and social
             amotivation symptoms scores were calculated from the
             Positive and Negative Syndrome Scale. WM was assessed with
             digit span and letter-number span tests.Participants who
             received CR had a significant improvement in WM scores (d =
             0.27) compared with those in the control condition.
             Improvements in social amotivation levels approached
             statistical significance (d = -0.19), but change in
             expressive negative symptoms did not differ between groups.
             WM change did not mediate the effect of CR on social
             amotivation.The results suggest that a course of CR may
             benefit behavioural negative symptoms. Despite hypotheses
             linking memory problems with negative symptoms, the current
             findings do not support the role of this cognitive domain as
             a significant mediator. The results indicate that WM
             improves independently from negative symptoms
             reduction.},
   Doi = {10.1017/s0033291717000757},
   Key = {fds330047}
}

@article{fds327058,
   Author = {Keefe, RSE and Davis, VG and Harvey, PD and Atkins, AS and Haig, GM and Hagino, O and Marder, S and Hilt, DC and Umbricht,
             D},
   Title = {Placebo Response and Practice Effects in Schizophrenia
             Cognition Trials.},
   Journal = {JAMA Psychiatry},
   Volume = {74},
   Number = {8},
   Pages = {807-814},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1001/jamapsychiatry.2017.1574},
   Abstract = {Patients' previous experience with performance-based
             cognitive tests in clinical trials for cognitive impairment
             associated with schizophrenia can create practice-related
             improvements. Placebo-controlled trials for cognitive
             impairment associated with schizophrenia are at risk for
             these practice effects, which can be difficult to
             distinguish from placebo effects.To conduct a systematic
             evaluation of the magnitude of practice effects on the
             Measurement and Treatment Research to Improve Cognition in
             Schizophrenia Consensus Cognitive Battery (MCCB) in
             cognitive impairment associated with schizophrenia and to
             examine which demographic, clinical, and cognitive
             characteristics were associated with improvement in placebo
             conditions.A blinded review was conducted of data from 813
             patients with schizophrenia who were treated with placebo in
             12 randomized placebo-controlled clinical trials conducted
             mostly in outpatient clinics in North America, Europe, Asia,
             and Latin America from February 22, 2007, to March 1, 2014.
             A total of 779 patients provided data for the primary
             outcome measure at baseline and at least 1 follow-up. Seven
             trials had prebaseline assessments wherein the patients knew
             that they were not receiving treatment, allowing a
             comparison of practice and placebo effects in the same
             patients.Placebo compared with various experimental drug
             treatments.Composite score on the MCCB.Of the 813 patients
             in the study (260 women and 553 men; mean [SD] age, 41.2
             [11.5] years), the mean MCCB composite score at baseline was
             22.8 points below the normative mean, and the mean (SEM)
             total change in the MCCB during receipt of placebo was 1.8
             (0.2) T-score points (95% CI, 1.40-2.18), equivalent to a
             change of 0.18 SD. Practice effects in the 7 studies in
             which there was a prebaseline assessment were essentially
             identical to the postbaseline placebo changes. Baseline
             factors associated with greater improvements in the MCCB
             during receipt of placebo included more depression/anxiety
             (F1,438 = 5.41; P = .02), more motivation
             (F1,272 = 4.63; P = .03), and less improvement from
             screening to baseline (F1,421 = 59.32;
             P < .001).Placebo effects were minimal and associated
             with the number of postbaseline assessments and several
             patient characteristics. Given that the patients performed
             2.28 SDs below normative standards on average at baseline, a
             mean placebo-associated improvement of less than 0.2 SD
             provides evidence that ceiling effects do not occur in these
             trials. These minimal changes in the MCCB could not be
             responsible for effective active treatments failing to
             separate from placebo.},
   Doi = {10.1001/jamapsychiatry.2017.1574},
   Key = {fds327058}
}

@article{fds324823,
   Author = {Mazhari, S and Ghafaree-Nejad, AR and Soleymani-Zade, S and Keefe,
             RSE},
   Title = {Validation of the Persian version of the Schizophrenia
             Cognition Rating Scale (SCoRS) in patients with
             schizophrenia.},
   Journal = {Asian Journal of Psychiatry},
   Volume = {27},
   Pages = {12-15},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.ajp.2017.02.007},
   Abstract = {The Schizophrenia Cognition Rating Scale (SCoRS) is an
             interview-based assessment of cognition that involves
             interviews with patients and informants. The SCoRS has shown
             good reliability, validity, and sensitivity to cognitive
             impairment in schizophrenia, with the advantage of brief
             administration and scoring time. The present study aimed to
             test the concurrent validity of the Persian version of the
             SCoRS. A group of 35 patients with schizophrenia and a group
             of 35 healthy controls received the Persian-SCoRS in the
             first session, and a standardized performance-based
             cognitive battery, the Brief Assessment of Cognition in
             Schizophrenia (BACS), in the second session.Our results
             indicated that the Persian version of the SCoRS was
             sensitive to cognitive impairment in the patients. The
             Persian SCoRS global rating was significantly associated
             with the composite score generated from the Persian version
             of the BACS and predicted functional outcomes as measured by
             Global Assessment of Functioning (GAF) and World Health
             Organization Quality of Life (WHO QOL). A Persian version of
             the SCoRS, an interview based measure of cognition that
             included informants, is related to cognitive performance and
             global functioning.},
   Doi = {10.1016/j.ajp.2017.02.007},
   Key = {fds324823}
}

@article{fds327059,
   Author = {Keefe, RSE and Kahn, RS},
   Title = {Cognitive Decline and Disrupted Cognitive Trajectory in
             Schizophrenia.},
   Journal = {JAMA Psychiatry},
   Volume = {74},
   Number = {5},
   Pages = {535-536},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1001/jamapsychiatry.2017.0312},
   Doi = {10.1001/jamapsychiatry.2017.0312},
   Key = {fds327059}
}

@article{fds323546,
   Author = {Atkins, AS and Tseng, T and Vaughan, A and Twamley, EW and Harvey, P and Patterson, T and Narasimhan, M and Keefe, RSE},
   Title = {Validation of the tablet-administered Brief Assessment of
             Cognition (BAC App).},
   Journal = {Schizophrenia Research},
   Volume = {181},
   Pages = {100-106},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.schres.2016.10.010},
   Abstract = {Computerized tests benefit from automated scoring procedures
             and standardized administration instructions. These methods
             can reduce the potential for rater error. However,
             especially in patients with severe mental illnesses, the
             equivalency of traditional and tablet-based tests cannot be
             assumed. The Brief Assessment of Cognition in Schizophrenia
             (BACS) is a pen-and-paper cognitive assessment tool that has
             been used in hundreds of research studies and clinical
             trials, and has normative data available for generating age-
             and gender-corrected standardized scores. A tablet-based
             version of the BACS called the BAC App has been developed.
             This study compared performance on the BACS and the BAC App
             in patients with schizophrenia and healthy controls. Test
             equivalency was assessed, and the applicability of
             paper-based normative data was evaluated. Results
             demonstrated the distributions of standardized composite
             scores for the tablet-based BAC App and the pen-and-paper
             BACS were indistinguishable, and the between-methods mean
             differences were not statistically significant. The
             discrimination between patients and controls was similarly
             robust. The between-methods correlations for individual
             measures in patients were r>0.70 for most subtests. When
             data from the Token Motor Test was omitted, the
             between-methods correlation of composite scores was r=0.88
             (df=48; p<0.001) in healthy controls and r=0.89 (df=46;
             p<0.001) in patients, consistent with the test-retest
             reliability of each measure. Taken together, results
             indicate that the tablet-based BAC App generates results
             consistent with the traditional pen-and-paper BACS, and
             support the notion that the BAC App is appropriate for use
             in clinical trials and clinical practice.},
   Doi = {10.1016/j.schres.2016.10.010},
   Key = {fds323546}
}

@article{fds327062,
   Author = {Reilly, JL and Hill, SK and Gold, JM and Keefe, RSE and Clementz, BA and Gershon, E and Keshavan, MS and Pearlson, G and Tamminga, CA and Sweeney, JA},
   Title = {Impaired Context Processing is Attributable to Global
             Neuropsychological Impairment in Schizophrenia and Psychotic
             Bipolar Disorder.},
   Journal = {Schizophrenia Bulletin},
   Volume = {43},
   Number = {2},
   Pages = {397-406},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1093/schbul/sbw081},
   Abstract = {Context processing may reflect a specific cognitive
             impairment in schizophrenia. Whether impaired context
             processing is observed across psychotic disorders or among
             relatives of affected individuals, and whether it is a
             deficit that is independent from the generalized
             neuropsychological deficits seen in psychotic disorders, are
             less established.Schizophrenia, schizoaffective, and
             psychotic bipolar probands (n = 660), their first-degree
             relatives (n = 741), and healthy individuals (n = 308)
             studied by the Bipolar-Schizophrenia Network on Intermediate
             Phenotypes consortium performed an expectancy task requiring
             use of contextual information to overcome a pre-potent
             response. Sensitivity for target detection and false alarm
             rates on trials requiring inhibition or goal maintenance
             were measured.Proband groups and relatives with psychosis
             spectrum personality traits demonstrated reduced target
             sensitivity and elevated false alarm rates. False alarm rate
             was higher under inhibition vs goal maintenance conditions
             although this difference was attenuated in schizophrenia and
             schizoaffective proband groups. After accounting for global
             neuropsychological impairment, as reflected by the composite
             score from the Brief Assessment of Cognition in
             Schizophrenia neuropsychological battery, deficits in
             schizophrenia and bipolar proband groups were no longer
             significant. Performance measures were moderately
             familial.Reduced target detection, but not a specific
             deficit in context processing, is observed across psychotic
             disorders. Impairments in both goal maintenance and response
             inhibition appear to contribute comparably to deficits in
             schizophrenia and schizoaffective disorder, whereas greater
             difficulty with response inhibition underlies deficits in
             bipolar disorder. Yet, these deficits are not independent
             from the generalized neurocognitive impairment observed in
             schizophrenia and psychotic bipolar disorder.},
   Doi = {10.1093/schbul/sbw081},
   Key = {fds327062}
}

@article{fds325352,
   Author = {Addington, J and Liu, L and Perkins, DO and Carrion, RE and Keefe, RSE and Woods, SW},
   Title = {The Role of Cognition and Social Functioning as Predictors
             in the Transition to Psychosis for Youth With Attenuated
             Psychotic Symptoms.},
   Journal = {Schizophrenia Bulletin},
   Volume = {43},
   Number = {1},
   Pages = {57-63},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1093/schbul/sbw152},
   Abstract = {In the literature, there have been several attempts to
             develop prediction models for youth who are at clinical high
             risk (CHR) of developing psychosis. Although there are no
             specific clinical or demographic variables that seem to
             consistently predict the later transition to psychosis in
             those CHR youth, in addition to attenuated psychotic
             symptoms, the most commonly occuring predictors tend to be
             poor social functioning and certain cognitive tasks.
             Unfortunately, there has been little attempt to replicate
             alogorithms. A recently published article by Cornblatt et al
             suggested that, for individuals with attentuated psychotic
             symptoms (APS), disorganized communication, suspiciousness,
             verbal memory, and a decline in social functioning were the
             best predictors of later transition to psychosis (the RAP
             model). The purpose of this article was to first test the
             prediction model of Cornblatt et al with a new sample of
             individuals with APS from the PREDICT study. The RAP model
             was not the best fit for the PREDICT data. However, using
             other variables from PREDICT, it was demonstrated that
             unusual thought content, disorganized communication,
             baseline social functioning, verbal fluency, and memory,
             processing speed and age were predictors of later transition
             to psychosis in the PREDICT sample. Although the predictors
             were different in these 2 models, both supported that
             disorganized communication, poor social functioning, and
             verbal memory, were good candidates as predictors for later
             conversion to psychosis.},
   Doi = {10.1093/schbul/sbw152},
   Key = {fds325352}
}


%% Keeling, Lori A.   
@article{fds326672,
   Author = {Spiridigliozzi, GA and Keeling, LA and Stefanescu, M and Li, C and Austin, S and Kishnani, PS},
   Title = {Cognitive and academic outcomes in long-term survivors of
             infantile-onset Pompe disease: A longitudinal
             follow-up.},
   Journal = {Molecular Genetics and Metabolism},
   Volume = {121},
   Number = {2},
   Pages = {127-137},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.ymgme.2017.04.014},
   Abstract = {This study examines the long-term cognitive and academic
             outcomes of 11 individuals with infantile onset Pompe
             disease (IOPD) (median age=11years, 1month, range=5years,
             6months through 17years of age) treated with enzyme
             replacement therapy from an early age. All participants (7
             males, 4 females) were administered individual intelligence
             tests (Wechsler or Leiter scales or both), a measure of
             their academic skill levels (Woodcock-Johnson Tests of
             Achievement), and a screening measure of visual-motor
             integration ability (Beery-Buktenica). Consistent with our
             earlier findings, median IQ scores for the entire group on
             the Wechsler (median=84) and Leiter (median=92) scales
             continue to fall at the lower end of the average range
             compared to same-aged peers. The median scores for the group
             on a measure of visual-motor integration (median=76), visual
             perception (median=74) and motor coordination (median=60)
             were below average. Two distinct subgroups emerged based on
             participants' average or below average performance on the
             majority of academic subtests. Those participants with below
             average academic skills (n=6) demonstrated average nonverbal
             cognitive abilities on the Leiter, but had weaknesses in
             speech and language skills and greater medical involvement.
             Their profiles were more consistent with a learning
             disability diagnosis than an intellectual disability. Two of
             these participants showed a significant decline (15 and 23
             points, respectively) on repeated Wechsler scales, but one
             continued to earn average scores on the Leiter scales where
             the verbal and motor demands are minimal. Participants with
             average academic skills (n=5) demonstrated average cognitive
             abilities (verbal and nonverbal) on the Wechsler scales and
             less medical involvement. Their speech and language skills
             appeared to be more intact. However, both groups earned
             below average median scores on the Beery-Buktenica motor
             coordination task. This study highlights the importance of
             using appropriate tests to capture both verbal and nonverbal
             abilities, considering each individual's motor skills,
             speech and language abilities, hearing status and native
             language. This will allow for a more accurate assessment of
             whether there is a learning disability or an intellectual
             disability. Long-term outcomes may be related to the
             stability of an individual's expressive and/or receptive
             language abilities over time. Changes in the speech and
             language domain may account for the decline in IQ observed
             in some IOPD long-term survivors, reflecting a learning
             disability rather than a decline in overall cognition or an
             intellectual disability. These observations, in conjunction
             with neuroimaging, will further our understanding of the
             neurocognitive profile of long-term IOPD
             survivors.},
   Doi = {10.1016/j.ymgme.2017.04.014},
   Key = {fds326672}
}


%% Kim, Namsoo   
@article{fds333930,
   Author = {Bey, AL and Wang, X and Yan, H and Kim, N and Passman, RL and Yang, Y and Cao,
             X and Towers, AJ and Hulbert, SW and Duffney, LJ and Gaidis, E and Rodriguiz, RM and Wetsel, WC and Yin, HH and Jiang,
             Y-H},
   Title = {Brain region-specific disruption of Shank3 in mice reveals a
             dissociation for cortical and striatal circuits in
             autism-related behaviors.},
   Journal = {Translational Psychiatry},
   Volume = {8},
   Number = {1},
   Pages = {94},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1038/s41398-018-0142-6},
   Abstract = {We previously reported a new line of Shank3 mutant mice
             which led to a complete loss of Shank3 by deleting exons
             4-22 (Δe4-22) globally. Δe4-22 mice display robust
             ASD-like behaviors including impaired social interaction and
             communication, increased stereotypical behavior and
             excessive grooming, and a profound deficit in instrumental
             learning. However, the anatomical and neural circuitry
             underlying these behaviors are unknown. We generated mice
             with Shank3 selectively deleted in forebrain, striatum, and
             striatal D1 and D2 cells. These mice were used to
             interrogate the circuit/brain-region and cell-type specific
             role of Shank3 in the expression of autism-related
             behaviors. Whole-cell patch recording and biochemical
             analyses were used to study the synaptic function and
             molecular changes in specific brain regions. We found
             perseverative exploratory behaviors in mice with deletion of
             Shank3 in striatal inhibitory neurons. Conversely,
             self-grooming induced lesions were observed in mice with
             deletion of Shank3 in excitatory neurons of forebrain.
             However, social, communicative, and instrumental learning
             behaviors were largely unaffected in these mice, unlike what
             is seen in global Δe4-22 mice. We discovered unique
             patterns of change for the biochemical and
             electrophysiological findings in respective brain regions
             that reflect the complex nature of transcriptional
             regulation of Shank3. Reductions in Homer1b/c and membrane
             hyper-excitability were observed in striatal loss of Shank3.
             By comparison, Shank3 deletion in hippocampal neurons
             resulted in increased NMDAR-currents and GluN2B-containing
             NMDARs. These results together suggest that Shank3 may
             differentially regulate neural circuits that control
             behavior. Our study supports a dissociation of Shank3
             functions in cortical and striatal neurons in ASD-related
             behaviors, and it illustrates the complexity of neural
             circuit mechanisms underlying these behaviors.},
   Doi = {10.1038/s41398-018-0142-6},
   Key = {fds333930}
}

@article{fds333931,
   Author = {Toda, K and Lusk, NA and Watson, GDR and Kim, N and Lu, D and Li, HE and Meck,
             WH and Yin, HH},
   Title = {Nigrotectal Stimulation Stops Interval Timing in
             Mice.},
   Journal = {Current Biology},
   Volume = {27},
   Number = {24},
   Pages = {3763-3770.e3},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.cub.2017.11.003},
   Abstract = {Considerable evidence implicates the basal ganglia in
             interval timing, yet the underlying mechanisms remain poorly
             understood. Using a novel behavioral task, we demonstrate
             that head-fixed mice can be trained to show the key features
             of timing behavior within a few sessions. Single-trial
             analysis of licking behavior reveals stepping dynamics with
             variable onset times, which is responsible for the canonical
             Gaussian distribution of timing behavior. Moreover, the
             duration of licking bouts decreased as mice became sated,
             showing a strong motivational modulation of licking bout
             initiation and termination. Using optogenetics, we examined
             the role of the basal ganglia output in interval timing. We
             stimulated a pathway important for licking behavior, the
             GABAergic output projections from the substantia nigra pars
             reticulata to the deep layers of the superior colliculus. We
             found that stimulation of this pathway not only cancelled
             licking but also delayed the initiation of anticipatory
             licking for the next interval in a frequency-dependent
             manner. By combining quantitative behavioral analysis with
             optogenetics in the head-fixed setup, we established a new
             approach for studying the neural basis of interval
             timing.},
   Doi = {10.1016/j.cub.2017.11.003},
   Key = {fds333931}
}

@article{fds333932,
   Author = {O'Hare, JK and Li, H and Kim, N and Gaidis, E and Ade, K and Beck, J and Yin,
             H and Calakos, N},
   Title = {Striatal fast-spiking interneurons selectively modulate
             circuit output and are required for habitual
             behavior.},
   Journal = {eLife},
   Volume = {6},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.7554/elife.26231},
   Abstract = {Habit formation is a behavioral adaptation that automates
             routine actions. Habitual behavior correlates with broad
             reconfigurations of dorsolateral striatal (DLS) circuit
             properties that increase gain and shift pathway timing. The
             mechanism(s) for these circuit adaptations are unknown and
             could be responsible for habitual behavior. Here we find
             that a single class of interneuron, fast-spiking
             interneurons (FSIs), modulates all of these habit-predictive
             properties. Consistent with a role in habits, FSIs are more
             excitable in habitual mice compared to goal-directed and
             acute chemogenetic inhibition of FSIs in DLS prevents the
             expression of habitual lever pressing. In vivo recordings
             further reveal a previously unappreciated selective
             modulation of SPNs based on their firing patterns; FSIs
             inhibit most SPNs but paradoxically promote the activity of
             a subset displaying high fractions of gamma-frequency
             spiking. These results establish a microcircuit mechanism
             for habits and provide a new example of how interneurons
             mediate experience-dependent behavior.},
   Doi = {10.7554/elife.26231},
   Key = {fds333932}
}


%% Kollins, Scott H.   
@article{fds330274,
   Author = {Sanchez, CE and Barry, C and Sabhlok, A and Russell, K and Majors, A and Kollins, SH and Fuemmeler, BF},
   Title = {Maternal pre-pregnancy obesity and child neurodevelopmental
             outcomes: a meta-analysis.},
   Journal = {Obesity Reviews},
   Volume = {19},
   Number = {4},
   Pages = {464-484},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1111/obr.12643},
   Abstract = {This review examined evidence of the association between
             maternal pre-pregnancy overweight/obesity status and child
             neurodevelopmental outcomes. PubMed and PsycINFO databases
             were systematically searched for empirical studies published
             before April 2017 using keywords related to prenatal obesity
             and children's neurodevelopment. Of 1483 identified papers,
             41 were included in the systematic review, and 32 articles
             representing 36 cohorts were included in the meta-analysis.
             Findings indicated that compared with children of normal
             weight mothers, children whose mothers were overweight or
             obese prior to pregnancy were at increased risk for
             compromised neurodevelopmental outcomes (overweight:
             OR = 1.17, 95% CI [1.11, 1.24], I2  = 65.51; obese:
             OR = 1.51; 95% CI [1.35, 1.69], I2  = 79.63).
             Pre-pregnancy obesity increased the risk of attention
             deficit-hyperactivity disorder (OR = 1.62; 95% CI [1.23,
             2.14], I2  = 70.15), autism spectrum disorder
             (OR = 1.36; 95% CI [1.08, 1.70], I2  = 60.52),
             developmental delay (OR = 1.58; 95% CI [1.39, 1.79], I2
              = 75.77) and emotional/behavioural problems (OR = 1.42;
             95% CI [1.26, 1.59], I2  = 87.74). Given the current
             obesity prevalence among young adults and women of
             childbearing age, this association between maternal obesity
             during pregnancy and atypical child neurodevelopment
             represents a potentially high public health
             burden.},
   Doi = {10.1111/obr.12643},
   Key = {fds330274}
}

@article{fds329549,
   Author = {Lee, C-T and McClernon, FJ and Kollins, SH and Fuemmeler,
             BF},
   Title = {Childhood ADHD Symptoms and Future Illicit Drug Use: The
             Role of Adolescent Cigarette Use.},
   Journal = {Journal of Pediatric Psychology},
   Volume = {43},
   Number = {2},
   Pages = {162-171},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1093/jpepsy/jsx098},
   Abstract = {The aim of this study is to understand how early cigarette
             use might predict subsequent illicit drug use, especially
             among individuals with attention-deficit hyperactivity
             disorder (ADHD) symptoms during childhood.Data were drawn
             from the National Longitudinal Study of Adolescent Health
             (Waves I-IV). The analysis sample involves participants who
             had not used illicit drugs at Wave I, with no missing
             responses for studied predictors (N = 7,332).Smoking
             status at Wave I (ever regular vs. never regular) and
             childhood ADHD symptoms predicted subsequent illicit drug
             use at Waves II to IV. No interaction effect of smoking
             status at Wave I and childhood ADHD symptoms was found.
             However, an indirect effect from childhood ADHD symptoms on
             illicit drug use was identified, through smoking status at
             Wave I. Similar results were observed for predicting illicit
             drug dependence.The findings support the notion that smoking
             status during early adolescence may mediate the association
             between childhood ADHD symptoms and risk of later adult drug
             use. Interventions to prevent smoking among adolescents may
             be particularly effective at decreasing subsequent drug use,
             especially among children with ADHD symptoms.},
   Doi = {10.1093/jpepsy/jsx098},
   Key = {fds329549}
}

@article{fds329548,
   Author = {Sweitzer, MM and Kollins, SH and Kozink, RV and Hallyburton, M and English, J and Addicott, MA and Oliver, JA and McClernon,
             FJ},
   Title = {ADHD, Smoking Withdrawal, and Inhibitory Control: Results of
             a Neuroimaging Study with Methylphenidate
             Challenge.},
   Journal = {Neuropsychopharmacology (Nature)},
   Volume = {43},
   Number = {4},
   Pages = {851-858},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1038/npp.2017.248},
   Abstract = {Smoking withdrawal negatively impacts inhibitory control,
             and these effects are greater for smokers with preexisting
             attention problems, such as attention deficit/hyperactivity
             disorder (ADHD). The current study preliminarily evaluated
             changes in inhibitory control-related behavior and brain
             activation during smoking withdrawal among smokers with
             ADHD. Moreover, we investigated the role of catecholamine
             transmission in these changes by examining the effects of
             40 mg methylphenidate (MPH) administration. Adult daily
             smokers with (n=17) and without (n=20) ADHD completed fMRI
             scanning under each of three conditions: (a) smoking as
             usual+placebo; (b) 24 h smoking abstinence+placebo and (c)
             24 h smoking abstinence+MPH. Scan order was randomized and
             counterbalanced. Participants completed a modified Go/No-Go
             task to assess both sustained and transient inhibitory
             control. Voxelwise analysis of task-related BOLD signal
             revealed a significant group-by-abstinence interaction in
             occipital/parietal cortex during sustained inhibition, with
             greater abstinence-induced decreases in activation observed
             among ADHD smokers compared with non-ADHD smokers. Changes
             in behavioral performance during abstinence were associated
             with changes in activation in regions of occipital and
             parietal cortex and bilateral insula during sustained
             inhibition in both groups. MPH administration improved
             behavioral performance and increased sustained inhibitory
             control-related activation for both groups. During transient
             inhibition, MPH increased prefrontal activation for both
             groups and increased striatal activation only among ADHD
             smokers. These preliminary findings suggest that
             abstinence-induced changes in catecholamine transmission in
             visual attention areas (eg, occipital and superior parietal
             cortex) may be associated with inhibitory control deficits
             and contribute to smoking vulnerability among individuals
             with ADHD.},
   Doi = {10.1038/npp.2017.248},
   Key = {fds329548}
}

@article{fds332067,
   Author = {Davis, NO and Bower, J and Kollins, SH},
   Title = {Proof-of-concept study of an at-home, engaging, digital
             intervention for pediatric ADHD.},
   Journal = {PloS one},
   Volume = {13},
   Number = {1},
   Pages = {e0189749},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1371/journal.pone.0189749},
   Abstract = {Pharmacological and behavioral therapies have limited impact
             on the distinct neurocognitive impairments associated with
             ADHD, and existing cognitive training programs have shown
             limited efficacy. This proof-of-concept study assessed
             treatment acceptability and explored outcomes for a novel
             digital treatment targeting cognitive processes implicated
             in ADHD.Participants included 40 children with ADHD and 40
             children without ADHD. Following psychiatric screening, ADHD
             ratings, and baseline neuropsychological measures,
             participants completed 28-days of at-home treatment.
             Neuropsychological assessment was repeated at end-of-study
             along with treatment satisfaction measures.Eighty-four
             percent of treatment sessions were completed and ratings
             showed strong intervention appeal. Significant improvements
             were observed on a computerized attention task for the ADHD
             group and a highly impaired ADHD High Severity subgroup.
             There was no change for the non-ADHD group. Spatial working
             memory also improved for the ADHD group and the ADHD High
             Severity subgroup.Findings provide preliminary support that
             this treatment may improve attention, working memory, and
             inhibition in children with ADHD. Future research requires
             larger-scale randomized controlled trials that also evaluate
             treatment impact on functional impairments.ClinicalTrials.gov
             NCT01943539.},
   Doi = {10.1371/journal.pone.0189749},
   Key = {fds332067}
}

@article{fds331559,
   Author = {Schechter, JC and Fuemmeler, BF and Hoyo, C and Murphy, SK and Zhang,
             JJ and Kollins, SH},
   Title = {Impact of Smoking Ban on Passive Smoke Exposure in Pregnant
             Non-Smokers in the Southeastern United States.},
   Journal = {International journal of environmental research and public
             health},
   Volume = {15},
   Number = {1},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.3390/ijerph15010083},
   Abstract = {Prenatal passive smoke exposure raises risk for negative
             birth outcomes. Legislation regulating public smoking has
             been shown to impact exposure levels, though fewer studies
             involving pregnant women have been conducted within the U.S.
             where bans are inconsistent across regions. This study
             examined the effect of a ban enacted in the southeastern
             U.S. on pregnant women's cotinine levels. Additional
             analyses compared self-reported exposure to cotinine and
             identified characteristics associated with passive exposure.
             Pregnant women (N = 851) were recruited prospectively
             between 2005 and 2011 in North Carolina. Sociodemographic
             and health data were collected via surveys; maternal blood
             samples were assayed for cotinine. Among non-active smokers
             who provided self-report data regarding passive exposure (N
             = 503), 20% were inconsistent with corresponding cotinine.
             Among all non-smokers (N = 668), being unmarried, African
             American, and less educated were each associated with
             greater passive exposure. Controlling for covariates, mean
             cotinine was higher prior to the ban compared to after, F(1,
             640) = 24.65, p < 0.001. Results suggest that banning
             smoking in public spaces may reduce passive smoke exposure
             for non-smoking pregnant women. These data are some of the
             first to examine the impact of legislation on passive smoke
             exposure in pregnant women within the U.S. using a biomarker
             and can inform policy in regions lacking comprehensive
             smoke-free legislation.},
   Doi = {10.3390/ijerph15010083},
   Key = {fds331559}
}

@article{fds311593,
   Author = {Mitchell, JT and McIntyre, EM and English, JS and Dennis, MF and Beckham, JC and Kollins, SH},
   Title = {A Pilot Trial of Mindfulness Meditation Training for ADHD in
             Adulthood: Impact on Core Symptoms, Executive Functioning,
             and Emotion Dysregulation.},
   Journal = {Journal of Attention Disorders},
   Volume = {21},
   Number = {13},
   Pages = {1105-1120},
   Year = {2017},
   Month = {November},
   url = {http://www.ncbi.nlm.nih.gov/pubmed/24305060},
   Abstract = {Mindfulness meditation training is garnering increasing
             empirical interest as an intervention for ADHD in adulthood,
             although no studies of mindfulness as a standalone treatment
             have included a sample composed entirely of adults with ADHD
             or a comparison group. The aim of this study was to assess
             the feasibility, acceptability, and preliminary efficacy of
             mindfulness meditation for ADHD, executive functioning (EF),
             and emotion dysregulation symptoms in an adult ADHD
             sample.Adults with ADHD were stratified by ADHD medication
             status and otherwise randomized into an 8-week group-based
             mindfulness treatment ( n = 11) or waitlist group ( n =
             9).Treatment feasibility and acceptability were positive. In
             addition, self-reported ADHD and EF symptoms (assessed in
             the laboratory and ecological momentary assessment),
             clinician ratings of ADHD and EF symptoms, and self-reported
             emotion dysregulation improved for the treatment group
             relative to the waitlist group over time with large effect
             sizes. Improvement was not observed for EF tasks.Findings
             support preliminary treatment efficacy, though require
             larger trials.},
   Doi = {10.1177/1087054713513328},
   Key = {fds311593}
}

@misc{fds329393,
   Author = {Murphy, SK and Schrott, R and Visco, Z and Huang, Z and Grenier, C and Mitchell, J and Schechter, J and Lucas, J and Levin, ED and Price, T and Kollins, SH},
   Title = {Environment and Gametic Epigenetic Reprogramming.},
   Journal = {Environmental and Molecular Mutagenesis},
   Volume = {58},
   Pages = {S28-S28},
   Year = {2017},
   Month = {September},
   Key = {fds329393}
}

@article{fds327694,
   Author = {McCullough, LE and Miller, EE and Calderwood, LE and Shivappa, N and Steck, SE and Forman, MR and A Mendez and M and Maguire, R and Fuemmeler,
             BF and Kollins, SH and D Bilbo and S and Huang, Z and Murtha, AP and Murphy,
             SK and Hébert, JR and Hoyo, C},
   Title = {Maternal inflammatory diet and adverse pregnancy outcomes:
             Circulating cytokines and genomic imprinting as potential
             regulators?},
   Journal = {Epigenetics : official journal of the DNA Methylation
             Society},
   Volume = {12},
   Number = {8},
   Pages = {688-697},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1080/15592294.2017.1347241},
   Abstract = {Excessive inflammation during pregnancy alters homeostatic
             mechanisms of the developing fetus and has been linked to
             adverse pregnancy outcomes. An anti-inflammatory diet could
             be a promising avenue to combat the pro-inflammatory state
             of pregnancy, particularly in obese women, but we lack
             mechanistic data linking this dietary pattern during
             pregnancy to inflammation and birth outcomes. In an
             ethnically diverse cohort of 1057 mother-child pairs, we
             estimated the relationships between dietary inflammatory
             potential [measured via the energy-adjusted dietary
             inflammatory index (E-DII™)] and birth outcomes overall,
             as well as by offspring sex and maternal pre-pregnancy body
             mass index (BMI). In a subset of women, we also explored
             associations between E-DII, circulating cytokines (n = 105),
             and offspring methylation (n = 338) as potential modulators
             of these relationships using linear regression. Adjusted
             regression models revealed that women with pro-inflammatory
             diets had elevated rates of preterm birth among female
             offspring [β = -0.22, standard error (SE) = 0.07, P<0.01],
             but not male offspring (β=0.09, SE = 0.06, P<0.12)
             (Pinteraction = 0.003). Similarly, we observed
             pro-inflammatory diets were associated with higher rates of
             caesarean delivery among obese women (β = 0.17, SE = 0.08,
             P = 0.03), but not among women with BMI <25 kg/m2
             (Pinteraction = 0.02). We observed consistent inverse
             associations between maternal inflammatory cytokine
             concentrations (IL-12, IL-17, IL-4, IL-6, and TNFα) and
             lower methylation at the MEG3 regulatory sequence (P<0.05);
             however, results did not support the link between maternal
             E-DII and circulating cytokines. We replicate work by others
             on the association between maternal inflammatory diet and
             adverse pregnancy outcomes and provide the first empirical
             evidence supporting the inverse association between
             circulating cytokine concentrations and offspring
             methylation.},
   Doi = {10.1080/15592294.2017.1347241},
   Key = {fds327694}
}

@article{fds324863,
   Author = {Schechter, JC and Kollins, SH},
   Title = {Prenatal Smoke Exposure and ADHD: Advancing the
             Field.},
   Journal = {Pediatrics},
   Volume = {139},
   Number = {2},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1542/peds.2016-3481},
   Doi = {10.1542/peds.2016-3481},
   Key = {fds324863}
}

@article{fds319636,
   Author = {Childress, AC and Kollins, SH and Cutler, AJ and Marraffino, A and Sikes, CR},
   Title = {Efficacy, Safety, and Tolerability of an Extended-Release
             Orally Disintegrating Methylphenidate Tablet in Children
             6-12 Years of Age with Attention-Deficit/Hyperactivity
             Disorder in the Laboratory Classroom Setting.},
   Journal = {Journal of Child and Adolescent Psychopharmacology},
   Volume = {27},
   Number = {1},
   Pages = {66-74},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1089/cap.2016.0002},
   Abstract = {Methylphenidate extended-release orally disintegrating
             tablets (MPH XR-ODTs) represent a new technology for MPH
             delivery. ODTs disintegrate in the mouth without water and
             provide a pharmacokinetic profile that is consistent with
             once-daily dosing. This study sought to determine the
             efficacy, safety, and tolerability of this novel MPH XR-ODT
             formulation in school-age children with attention-deficit/hyperactivity
             disorder (ADHD) in a laboratory classroom setting.Children
             aged 6-12 years with ADHD (n = 87) were enrolled in this
             randomized, multicenter, double-blind, placebo-controlled,
             parallel, laboratory classroom study. The MPH XR-ODT dose
             was titrated to an optimized dose during a 4-week open-label
             period and maintained on that dose for 1 week. Participants
             (n = 85) were then randomized to receive their optimized
             dose of MPH XR-ODT or placebo once daily for 1 week (double
             blind), culminating in a laboratory classroom testing day.
             Efficacy was evaluated using the Swanson, Kotkin, Agler,
             M-Flynn, and Pelham (SKAMP) Attention, Deportment, and
             Combined scores along with Permanent Product Measure of
             Performance (PERMP; Attempted and Correct) assessments.
             Onset and duration of drug action were also evaluated as key
             secondary endpoints. Safety assessments included adverse
             events (AEs), physical examinations, electrocardiograms
             (ECGs), and the Columbia Suicide Severity Rating Scale
             (C-SSRS).The average SKAMP-Combined score on the classroom
             study day was significantly better for the MPH XR-ODT group
             (n = 43) than for the placebo group (n = 39;
             p < 0.0001). The effect was evident at 1 hour and lasted
             through 12 hours postdose. The average SKAMP-Attention,
             SKAMP-Deportment, PERMP-A, and PERMP-C scores were
             indicative of significantly greater ADHD symptom control for
             the MPH XR-ODT group. The most common AEs reported were
             decreased appetite, upper abdominal pain, headache,
             insomnia, upper respiratory tract infection, affect
             lability, irritability, cough, and vomiting.MPH XR-ODT was
             effective and well tolerated for the treatment of children
             with ADHD in a laboratory classroom setting. Clinical Trial
             Registry: NCT01835548 ( ClinicalTrials.gov
             ).},
   Doi = {10.1089/cap.2016.0002},
   Key = {fds319636}
}

@article{fds327695,
   Author = {Mitchell, JT and Weisner, TS and Jensen, PS and Murray, DW and Molina,
             BSG and Arnold, EL and Hechtman, L and Swanson, JM and Hinshaw, SP and Victor, EC and Kollins, SH and Wells, KC and Belendiuk, KA and Blonde,
             A and Nguyen, C and Ambriz, L and Nguyen, JL},
   Title = {How Substance Users With ADHD Perceive the Relationship
             Between Substance Use and Emotional Functioning.},
   Journal = {Journal of Attention Disorders},
   Pages = {1087054716685842},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1177/1087054716685842},
   Abstract = {Although substance use (SU) is elevated in ADHD and both are
             associated with disrupted emotional functioning, little is
             known about how emotions and SU interact in ADHD. We used a
             mixed qualitative-quantitative approach to explore this
             relationship.Narrative comments were coded for 67 persistent
             (50 ADHD, 17 local normative comparison group [LNCG]) and 25
             desistent (20 ADHD, 5 LNCG) substance users from the
             Multimodal Treatment Study of Children with ADHD (MTA) adult
             follow-up (21.7-26.7 years-old).SU persisters perceived SU
             positively affects emotional states and positive emotional
             effects outweigh negative effects. No ADHD group effects
             emerged. Qualitative analysis identified perceptions that
             cannabis enhanced positive mood for ADHD and LNCG SU
             persisters, and improved negative mood and ADHD for ADHD SU
             persisters.Perceptions about SU broadly and mood do not
             differentiate ADHD and non-ADHD SU persisters. However,
             perceptions that cannabis is therapeutic may inform
             ADHD-related risk for cannabis use.},
   Doi = {10.1177/1087054716685842},
   Key = {fds327695}
}


%% LaBar, Kevin S   
@article{fds332658,
   Author = {Wing, EA and Iyengar, V and Hess, TM and LaBar, KS and Huettel, SA and Cabeza, R},
   Title = {Neural mechanisms underlying subsequent memory for personal
             beliefs:An fMRI study.},
   Journal = {Cognitive, Affective, & Behaviorial Neuroscience},
   Volume = {18},
   Number = {2},
   Pages = {216-231},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.3758/s13415-018-0563-y},
   Abstract = {Many fMRI studies have examined the neural mechanisms
             supporting emotional memory for stimuli that generate
             emotion rather automatically (e.g., a picture of a dangerous
             animal or of appetizing food). However, far fewer studies
             have examined how memory is influenced by emotion related to
             social and political issues (e.g., a proposal for large
             changes in taxation policy), which clearly vary across
             individuals. In order to investigate the neural substrates
             of affective and mnemonic processes associated with personal
             opinions, we employed an fMRI task wherein participants
             rated the intensity of agreement/disagreement to
             sociopolitical belief statements paired with neural face
             pictures. Following the rating phase, participants performed
             an associative recognition test in which they distinguished
             identical versus recombined face-statement pairs. The study
             yielded three main findings: behaviorally, the intensity of
             agreement ratings was linked to greater subjective emotional
             arousal as well as enhanced high-confidence subsequent
             memory. Neurally, statements that elicited strong (vs. weak)
             agreement or disagreement were associated with greater
             activation of the amygdala. Finally, a subsequent memory
             analysis showed that the behavioral memory advantage for
             statements generating stronger ratings was dependent on the
             medial prefrontal cortex (mPFC). Together, these results
             both underscore consistencies in neural systems supporting
             emotional arousal and suggest a modulation of
             arousal-related encoding mechanisms when emotion is
             contingent on referencing personal beliefs.},
   Doi = {10.3758/s13415-018-0563-y},
   Key = {fds332658}
}

@article{fds330541,
   Author = {LaBar, KS},
   Title = {Advances in neuroscience.},
   Journal = {Science Advances},
   Volume = {3},
   Number = {11},
   Pages = {eaar2953},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1126/sciadv.aar2953},
   Doi = {10.1126/sciadv.aar2953},
   Key = {fds330541}
}

@article{fds327386,
   Author = {Zucker, NL and Kragel, PA and Wagner, HR and Keeling, L and Mayer, E and Wang, J and Kang, MS and Merwin, R and Simmons, WK and LaBar,
             KS},
   Title = {The Clinical Significance of Posterior Insular Volume in
             Adolescent Anorexia Nervosa.},
   Journal = {Psychosomatic Medicine},
   Volume = {79},
   Number = {9},
   Pages = {1025-1035},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1097/psy.0000000000000510},
   Abstract = {The diagnostic criterion disturbance in the experience of
             the body remains a poorly understood and persistent feature
             of anorexia nervosa (AN). Increased sophistication in
             understanding the structure of the insular cortex-a neural
             structure that receives and integrates visceral sensations
             with action and meaning-may elucidate the nature of this
             disturbance. We explored age, weight status, illness
             severity, and self-reported body dissatisfaction
             associations with insular cortex volume.Structural magnetic
             resonance imaging data were collected from 21 adolescents
             with a history of AN and 20 age-, sex-, and body mass
             index-matched controls. Insular cortical volumes (bilateral
             anterior and posterior regions) were identified using manual
             tracing.Volumes of the right posterior insula demonstrated
             the following: (a) a significant age by clinical status
             interaction (β = -0.018 [0.008]; t = 2.32, p = .02) and (b)
             larger volumes were associated with longer duration of
             illness (r = 0.48, p < .04). In contrast, smaller volumes of
             the right anterior insula were associated with longer
             duration of illness (r = -0.50, p < .03). The associations
             of insular volume with body dissatisfaction were of moderate
             effect size and also of opposite direction, but a
             statistical trend in right posterior (r = 0.40, p < .10 in
             right posterior; r = -0.49, p < .04 in right anterior).In
             this exploratory study, findings of atypical structure of
             the right posterior insular cortex point to the importance
             of future work investigating the role of visceral afferent
             signaling in understanding disturbance in body experience in
             AN.},
   Doi = {10.1097/psy.0000000000000510},
   Key = {fds327386}
}

@article{fds318723,
   Author = {Li, D and Zucker, NL and Kragel, PA and Covington, VE and LaBar,
             KS},
   Title = {Adolescent development of insula-dependent interoceptive
             regulation.},
   Journal = {Developmental Science},
   Volume = {20},
   Number = {5},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1111/desc.12438},
   Abstract = {Adolescence is hypothesized to be a critical period for the
             maturation of self-regulatory capacities, including those
             that depend on interoceptive sensitivity, but the neural
             basis of interoceptive regulation in adolescence is unknown.
             We used functional magnetic resonance imaging and
             psychophysiology to study interoceptive regulation in
             healthy adolescent females. Participants regulated their gut
             activities in response to a virtual roller coaster by deep
             breathing aided by visually monitoring their online
             electrogastrogram (EGG) activity through a virtual
             thermometer (i.e. gut biofeedback), or without biofeedback.
             Analyses focused on the insula, given its putative role in
             interoception. The bilateral posterior insula showed
             increased activation in the no-biofeedback compared to
             biofeedback condition, suggesting that the participants
             relied more on interoceptive input when exteroceptive
             feedback was unavailable. The bilateral dorsal anterior
             insula showed activation linearly associated with age during
             both induction and regulation, and its activation during
             regulation correlated positively with change of EGG in the
             tachygastria frequency band from induction to regulation.
             Induction-related activation in the bilateral ventral
             anterior insula was nonlinearly associated with age and
             peaked at mid-adolescence. These results implicate different
             developmental trajectories of distinct sub-regions of the
             insula in interoceptive processes, with implications for
             competing neurobiological theories of female adolescent
             development.},
   Doi = {10.1111/desc.12438},
   Key = {fds318723}
}


%% Larrick, Richard P.   
@article{fds333265,
   Author = {Schaerer, M and Tost, LP and Huang, L and Gino, F and Larrick,
             R},
   Title = {Advice Giving: A Subtle Pathway to Power.},
   Journal = {Personality and Social Psychology Bulletin},
   Pages = {146167217746341},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1177/0146167217746341},
   Abstract = {We propose that interpersonal behaviors can activate
             feelings of power, and we examine this idea in the context
             of advice giving. Specifically, we show (a) that advice
             giving is an interpersonal behavior that enhances
             individuals' sense of power and (b) that those who seek
             power are motivated to engage in advice giving. Four
             studies, including two experiments ( N = 290, N = 188), an
             organization-based field study ( N = 94), and a negotiation
             simulation ( N = 124), demonstrate that giving advice
             enhances the adviser's sense of power because it gives the
             adviser perceived influence over others' actions. Two of our
             studies further demonstrate that people with a high tendency
             to seek power are more likely to give advice than those with
             a low tendency. This research establishes advice giving as a
             subtle route to a sense of power, shows that the desire to
             feel powerful motivates advice giving, and highlights the
             dynamic interplay between power and advice.},
   Doi = {10.1177/0146167217746341},
   Key = {fds333265}
}

@article{fds330876,
   Author = {Kay, MB and Proudfoot, D and Larrick, RP},
   Title = {There's No Team in I: How Observers Perceive Individual
             Creativity in a Team Setting},
   Journal = {Journal of Applied Psychology},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1037/apl0000270},
   Abstract = {© 2017 APA, all rights reserved). Creativity is highly
             valued in organizations as an important source of
             innovation. As most creative projects require the efforts of
             groups of individuals working together, it is important to
             understand how creativity is perceived for team products,
             including how observers attribute creative ability to focal
             actors who worked as part of a creative team. Evidence from
             three experiments suggests that observers commit the
             fundamental attribution error-systematically discounting the
             contribution of the group when assessing the creative
             ability of a single group representative, particularly when
             the group itself is not visually salient. In a pilot study,
             we found that, in the context of the design team at Apple, a
             target group member visually depicted alone is perceived to
             have greater personal creative ability than when he is
             visually depicted with his team. In Study 1, using a sample
             of managers, we conceptually replicated this finding and
             further observed that, when shown alone, a target member of
             a group that produced a creative product is perceived to be
             as creative as an individual described as working alone on
             the same output. In Study 2, we replicated the findings of
             Study 1 and also observed that a target group member
             depicted alone, rather than with his team, is also
             attributed less creative ability for uncreative group
             output. Findings are discussed in light of how
             overattribution of individual creative ability can harm
             organizations in the long run.(PsycINFO Database
             Record},
   Doi = {10.1037/apl0000270},
   Key = {fds330876}
}

@article{fds328721,
   Author = {Rader, CA and Larrick, RP and Soll, JB},
   Title = {Advice as a form of social influence: Informational motives
             and the consequences for accuracy},
   Journal = {Social and Personality Psychology Compass},
   Volume = {11},
   Number = {8},
   Pages = {e12329-e12329},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/spc3.12329},
   Doi = {10.1111/spc3.12329},
   Key = {fds328721}
}

@article{fds325129,
   Author = {Tang, S and Morewedge, CK and Larrick, RP and Klein,
             JG},
   Title = {Disloyalty aversion: Greater reluctance to bet against close
             others than the self},
   Journal = {Organizational Behavior and Human Decision
             Processes},
   Volume = {140},
   Pages = {1-13},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.obhdp.2017.02.001},
   Doi = {10.1016/j.obhdp.2017.02.001},
   Key = {fds325129}
}

@article{fds327118,
   Author = {Aribarg, A and Burson, KA and Larrick, RP},
   Title = {Tipping the Scale: The Role of Discriminability in Conjoint
             Analysis},
   Journal = {Journal of Marketing Research},
   Volume = {54},
   Number = {2},
   Pages = {279-292},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1509/jmr.14.0659},
   Doi = {10.1509/jmr.14.0659},
   Key = {fds327118}
}

@article{fds333266,
   Author = {Ungemach, C and Camilleri, AR and Johnson, EJ and Larrick, RP and Weber,
             EU},
   Title = {Translated Attributes as Choice Architecture: Aligning
             Objectives and Choices Through Decision Signposts},
   Year = {2017},
   Month = {February},
   Key = {fds333266}
}

@article{fds331495,
   Author = {Tong, J and Feiler, D and Larrick, R},
   Title = {A Behavioral Remedy for the Censorship Bias},
   Journal = {Production and Operations Management},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1111/poms.12823},
   Abstract = {© 2017 Production and Operations Management Society.
             Existing evidence suggests that managers exhibit a
             censorship bias: demand beliefs tend to be biased low when
             lost sales from stockouts are unobservable (censored demand)
             compared to when they are observable (uncensored demand). We
             develop a non-constraining, easily implementable behavioral
             debias technique to help mitigate this tendency in demand
             forecasting and inventory decision-making settings. The
             debiasing technique has individuals record estimates of
             demand outcomes (REDO): participants explicitly record a
             self-generated estimate of every demand realization,
             allowing them to record a different value than the number of
             sales in periods with stockouts. In doing so, they construct
             a more representative sample of demand realizations (that
             differs from the sales sample). In three laboratory
             experiments with MBA and undergraduate students, this remedy
             significantly reduces downward bias in demand beliefs under
             censorship and leads to higher inventory order
             decisions.},
   Doi = {10.1111/poms.12823},
   Key = {fds331495}
}

@article{fds326636,
   Author = {De Langhe and B and Puntoni, S and Larrick, R},
   Title = {LINEAR THINKING IN A NONLINEAR WORLD},
   Journal = {Harvard Business Review},
   Volume = {95},
   Number = {3},
   Pages = {130-139},
   Year = {2017},
   Key = {fds326636}
}


%% Leary, Mark R.   
@misc{fds332882,
   Author = {Leary, MR},
   Title = {Self-awareness, hypo-egoicism, and psychological
             well-being},
   Pages = {392-408},
   Booktitle = {Subjective Well-Being and Life Satisfaction},
   Year = {2017},
   Month = {December},
   ISBN = {9781351231879},
   url = {http://dx.doi.org/10.4324/9781351231879},
   Doi = {10.4324/9781351231879},
   Key = {fds332882}
}

@misc{fds332883,
   Author = {Leary, MR and Bolino, MC},
   Title = {An actor-perceiver model of impression management in
             organizations},
   Pages = {253-272},
   Booktitle = {The Self at Work: Fundamental Theory and
             Research},
   Year = {2017},
   Month = {December},
   ISBN = {9781315626543},
   url = {http://dx.doi.org/10.4324/9781315626543},
   Doi = {10.4324/9781315626543},
   Key = {fds332883}
}

@article{fds331404,
   Author = {Shumway, M and Mangurian, C and Carraher, N and Momenzadeh, A and Leary,
             M and Lee, EK and Dilley, JW},
   Title = {Increasing HIV Testing in Inpatient Psychiatry},
   Journal = {Psychosomatics},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.psym.2017.10.007},
   Doi = {10.1016/j.psym.2017.10.007},
   Key = {fds331404}
}

@article{fds326829,
   Author = {Leary, MR and Diebels, KJ and Davisson, EK and Jongman-Sereno, KP and Isherwood, JC and Raimi, KT and Deffler, SA and Hoyle,
             RH},
   Title = {Cognitive and Interpersonal Features of Intellectual
             Humility.},
   Journal = {Personality and Social Psychology Bulletin},
   Volume = {43},
   Number = {6},
   Pages = {793-813},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1177/0146167217697695},
   Abstract = {Four studies examined intellectual humility-the degree to
             which people recognize that their beliefs might be wrong.
             Using a new Intellectual Humility (IH) Scale, Study 1 showed
             that intellectual humility was associated with variables
             related to openness, curiosity, tolerance of ambiguity, and
             low dogmatism. Study 2 revealed that participants high in
             intellectual humility were less certain that their beliefs
             about religion were correct and judged people less on the
             basis of their religious opinions. In Study 3, participants
             high in intellectual humility were less inclined to think
             that politicians who changed their attitudes were
             "flip-flopping," and Study 4 showed that people high in
             intellectual humility were more attuned to the strength of
             persuasive arguments than those who were low. In addition to
             extending our understanding of intellectual humility, this
             research demonstrates that the IH Scale is a valid measure
             of the degree to which people recognize that their beliefs
             are fallible.},
   Doi = {10.1177/0146167217697695},
   Key = {fds326829}
}


%% Levin, Edward D.   
@article{fds333553,
   Author = {Oliveri, AN and Ortiz, E and Levin, ED},
   Title = {Developmental exposure to an organophosphate flame retardant
             alters later behavioral responses to dopamine antagonism in
             zebrafish larvae.},
   Journal = {Neurotoxicology and Teratology},
   Volume = {67},
   Pages = {25-30},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.ntt.2018.03.002},
   Abstract = {Human exposure to organophosphate flame retardants (OPFRs)
             is widespread, including pregnant women and young children
             with whom developmental neurotoxic risk is a concern. Given
             similarities of OPFRs to organophosphate (OP) pesticides,
             research into the possible neurotoxic impacts of
             developmental OPFR exposure has been growing. Building upon
             research implicating exposure to OP pesticides in
             dopaminergic (DA) dysfunction, we exposed developing
             zebrafish to the OPFR tris(1,3-dichloroisopropyl) phosphate
             (TDCIPP), during the first 5 days following fertilization.
             On day 6, larvae were challenged with acute administration
             of dopamine D1 and D2 receptor antagonists and then tested
             in a light-dark locomotor assay. We found that both
             developmental TDCIPP exposure and acute dopamine D1 and D2
             antagonism decreased locomotor activity separately. The OPFR
             and DA effects were not additive; rather, TDCIPP blunted
             further D1 and D2 antagonist-induced decreases in activity.
             Our results suggest that TDCIPP exposure may be disrupting
             dopamine signaling. These findings support further research
             on the effects of OPFR exposure on the normal
             neurodevelopment of DA systems, whether these results might
             persist into adulthood, and whether they interact with OPFR
             effects on other neurotransmitter systems in producing the
             developmental neurobehavioral toxicity.},
   Doi = {10.1016/j.ntt.2018.03.002},
   Key = {fds333553}
}

@article{fds332218,
   Author = {Levin, ED and Wells, C and Slade, S and Rezvani, AH},
   Title = {Mutually augmenting interactions of dextromethorphan and
             sazetidine-A for reducing nicotine self-administration in
             rats.},
   Journal = {Pharmacology Biochemistry and Behavior},
   Volume = {166},
   Pages = {42-47},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.pbb.2018.01.005},
   Abstract = {A variety of nicotinic drug treatments have been found to
             decrease nicotine self-administration. However, interactions
             of drugs affecting different nicotinic receptor subtypes
             have not been much investigated. This study investigated the
             interactions between dextromethorphan, which blocks
             nicotinic α3β2 receptors as well as a variety of other
             receptors with sazetidine-A which is a potent and selective
             α4β2 nicotinic receptor partial agonist with desensitizing
             properties. This interaction was compared with
             dextromethorphan combination treatment with mecamylamine,
             which is a nonspecific nicotinic channel blocker.
             Co-administration of dextromethorphan (either 0.5 or
             5 mg/kg) and lower dose of sazetidine-A (0.3 mg/kg)
             caused a significant reduction in nicotine SA. With regard
             to food-motivated responding, 3 mg/kg of sazetidine-A
             given alone caused a significant decrease in food intake.
             However, the lower 0.3 mg/kg sazetidine-A dose did not
             significantly affect food-motivated responding even when
             given in combination with the higher 5 mg/kg
             dextromethorphan dose which itself caused a significant
             decrease in food motivated responding. Interestingly, this
             higher dextromethorphan dose significantly attenuated the
             decrease in food motivated responding caused by 3 mg/kg of
             sazetidine-A. Locomotor activity was increased by the lower
             0.3 mg/kg sazetidine-A dose and decreased by the 5 mg/kg
             dextromethorphan dose. Mecamylamine at the doses (0.1 and
             1 mg/kg) did not affect nicotine SA, but at 1 mg/kg
             significantly decreased food-motivated responding. None of
             the mecamylamine doses augmented the effect of
             dextromethorphan in reducing nicotine self-administration.
             These studies showed that the combination of
             dextromethorphan and sazetidine-A had mutually potentiating
             effects, which could provide a better efficacy for promoting
             smoking cessation, however the strength of the interactions
             was fairly modest.},
   Doi = {10.1016/j.pbb.2018.01.005},
   Key = {fds332218}
}

@article{fds332068,
   Author = {Rezvani, AH and Tizabi, Y and Slade, S and Getachew, B and Levin,
             ED},
   Title = {Sub-anesthetic doses of ketamine attenuate nicotine
             self-administration in rats.},
   Journal = {Neuroscience Letters},
   Volume = {668},
   Pages = {98-102},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.neulet.2018.01.022},
   Abstract = {Smoking cessation strategies are of prime medical
             importance. Despite availability of various pharmacological
             agents in combating addiction to nicotine, more effective
             medications are needed. Based on recent findings, the
             glutamatergic system in the brain may provide novel targets.
             Here, we evaluated the effects of acute administration of
             sub-anesthetic doses of ketamine, an NMDA receptor
             antagonist, in both male and female Sprague-Dawley rats
             trained to self-administer nicotine. Animals were injected
             subcutaneously with 5, 7.5 and 10 mg/kg ketamine or saline
             and the effects on the number of intravenous nicotine
             infusions during a 45 min session was measured. Ketamine
             treatment significantly reduced nicotine self-administration
             in a dose-dependent manner. Moreover, a differential
             sensitivity between the sexes was observed as male rats
             responded to a lower dose of ketamine and with higher
             magnitude of effect than female rats. It is concluded that
             glutamatergic receptor manipulations may offer a novel and
             potentially sex-dependent intervention in nicotine
             addiction.},
   Doi = {10.1016/j.neulet.2018.01.022},
   Key = {fds332068}
}

@article{fds333002,
   Author = {Massarsky, A and Abdel, A and Glazer, L and Levin, ED and Di Giulio,
             RT},
   Title = {Neurobehavioral effects of 1,2-propanediol in zebrafish
             (Danio rerio).},
   Journal = {NeuroToxicology},
   Volume = {65},
   Pages = {111-124},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.neuro.2018.02.007},
   Abstract = {The use of electronic cigarettes (e-cigarettes) is
             increasing despite insufficient information concerning their
             long-term effects, including the effects of maternal
             e-cigarette use on pre- and postnatal development. Our
             previous study demonstrated that developmental exposure to
             1,2-propanediol (a principal component of e-cigarette
             liquid) affected early development of zebrafish, causing
             reduced growth, deformities, and hyperactive swimming
             behavior in larvae. The current study extends assessment of
             the developmental toxicity of 1,2-propanediol by examining
             additional long-term behavioral effects. We demonstrate that
             embryonic/larval exposure of zebrafish to 1,2-propanediol
             (0.625% or 1.25%) not only affected behavioral parameters in
             the larvae, but also caused persisting behavioral effects in
             adults after early developmental exposure. Additional
             parameters, including neural and vascular development in
             larvae, stress response in adults, and concentration of
             neurotransmitters dopamine and serotonin in adult brain were
             examined, in order to explain the behavioral differences.
             These additional assessments did not find 1,2-propanediol
             exposure to significantly affect Tg(Neurog1:GFP) or the
             transcript abundance of neural genes (Neurog1, Ascl1a,
             Elavl3, and Lef1). Vascular development was not found to be
             affected by 1,2-propanediol exposure, as inferred from
             experiments with Tg(Flk1:eGFP) zebrafish; however,
             transcript abundance of vascular genes (Flk1, Vegf, Tie-2,
             and Angpt1) was decreased. No statistically significant
             changes were noted for plasma cortisol or brain
             neurotransmitters in adult fish. Lastly, analysis of gene
             transcripts involved with 1,2-propanediol metabolism (Adh5,
             Aldh2.1, and Ldha) showed an increase in Adh5 transcript.
             This is the first study to demonstrate that developmental
             exposure to 1,2-propanediol has long-term neurobehavioral
             consequences in adult zebrafish, showing that e-cigarettes
             contain substances potentially harmful to
             neurodevelopment.},
   Doi = {10.1016/j.neuro.2018.02.007},
   Key = {fds333002}
}

@article{fds332917,
   Author = {Pitt, JA and Kozal, JS and Jayasundara, N and Massarsky, A and Trevisan,
             R and Geitner, N and Wiesner, M and Levin, ED and Di Giulio,
             RT},
   Title = {Uptake, tissue distribution, and toxicity of polystyrene
             nanoparticles in developing zebrafish (Danio
             rerio).},
   Journal = {Aquatic Toxicology},
   Volume = {194},
   Pages = {185-194},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.aquatox.2017.11.017},
   Abstract = {Plastic pollution is a critical environmental concern and
             comprises the majority of anthropogenic debris in the ocean,
             including macro, micro, and likely nanoscale (less than
             100nm in at least one dimension) plastic particles. While
             the toxicity of macroplastics and microplastics is
             relatively well studied, the toxicity of nanoplastics is
             largely uncharacterized. Here, fluorescent polystyrene
             nanoparticles (PS NPs) were used to investigate the
             potential toxicity of nanoplastics in developing zebrafish
             (Danio rerio), as well as to characterize the uptake and
             distribution of the particles within embryos and larvae.
             Zebrafish embryos at 6h post-fertilization (hpf) were
             exposed to PS NPs (0.1, 1, or 10ppm) until 120 hpf. Our
             results demonstrate that PS NPs accumulated in the yolk sac
             as early as 24 hpf and migrated to the gastrointestinal
             tract, gallbladder, liver, pancreas, heart, and brain
             throughout development (48-120 hpf). Accumulation of PS NPs
             decreased during the depuration phase (120-168 hpf) in all
             organs, but at a slower rate in the pancreas and
             gastrointestinal tract. Notably, exposure to PS NPs did not
             induce significant mortality, deformities, or changes to
             mitochondrial bioenergetics, but did decrease the heart
             rate. Lastly, exposure to PS NPs altered larval behavior as
             evidenced by swimming hypoactivity in exposed larvae. Taken
             together, these data suggest that at least some nanoplastics
             can penetrate the chorion of developing zebrafish,
             accumulate in the tissues, and affect physiology and
             behavior, potentially affecting organismal fitness in
             contaminated aquatic ecosystems.},
   Doi = {10.1016/j.aquatox.2017.11.017},
   Key = {fds332917}
}

@article{fds330580,
   Author = {Lacagnina, MJ and Kopec, AM and Cox, SS and Hanamsagar, R and Wells, C and Slade, S and Grace, PM and Watkins, LR and Levin, ED and Bilbo,
             SD},
   Title = {Opioid Self-Administration is Attenuated by Early-Life
             Experience and Gene Therapy for Anti-Inflammatory IL-10 in
             the Nucleus Accumbens of Male Rats.},
   Journal = {Neuropsychopharmacology (Nature)},
   Volume = {42},
   Number = {11},
   Pages = {2128-2140},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1038/npp.2017.82},
   Abstract = {Early-life conditions can contribute to the propensity for
             developing neuropsychiatric disease, including substance
             abuse disorders. However, the long-lasting mechanisms that
             shape risk or resilience for drug addiction remain unclear.
             Previous work has shown that a neonatal handling procedure
             in rats (which promotes enriched maternal care) attenuates
             morphine conditioning, reduces morphine-induced glial
             activation, and increases microglial expression of the
             anti-inflammatory cytokine interleukin-10 (IL-10). We thus
             hypothesized that anti-inflammatory signaling may underlie
             the effects of early-life experience on later-life opioid
             drug-taking. Here we demonstrate that neonatal handling
             attenuates intravenous self-administration of the opioid
             remifentanil in a drug-concentration-dependent manner.
             Transcriptional profiling of the nucleus accumbens (NAc)
             from handled rats following repeated exposure to
             remifentanil reveals a suppression of pro-inflammatory
             cytokine and chemokine gene expression, consistent with an
             anti-inflammatory phenotype. To determine if
             anti-inflammatory signaling alters drug-taking behavior, we
             administered intracranial injections of plasmid DNA encoding
             IL-10 (pDNA-IL-10) into the NAc of non-handled rats. We
             discovered that pDNA-IL-10 treatment reduces remifentanil
             self-administration in a drug-concentration-dependent
             manner, similar to the effect of handling. In contrast,
             neither handling nor pDNA-IL-10 treatment alters
             self-administration of food or sucrose rewards. These
             collective observations suggest that neuroimmune signaling
             mechanisms in the NAc are shaped by early-life experience
             and may modify motivated behaviors for opioid drugs.
             Moreover, manipulation of the IL-10 signaling pathway
             represents a novel approach for influencing opioid
             reinforcement.},
   Doi = {10.1038/npp.2017.82},
   Key = {fds330580}
}

@article{fds329144,
   Author = {Glazer, L and Wells, CN and Drastal, M and Odamah, K-A and Galat, RE and Behl, M and Levin, ED},
   Title = {Developmental exposure to low concentrations of two
             brominated flame retardants, BDE-47 and BDE-99, causes
             life-long behavioral alterations in zebrafish.},
   Journal = {NeuroToxicology},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.neuro.2017.09.007},
   Abstract = {Polybrominated diphenyl ethers (PBDEs) were widely used as
             flame retardants until the early 2000s, mainly in home
             furnishings and electronics. The persistence of PBDEs in the
             environment leads to continued ubiquitous exposure to low
             levels, with infants and children experiencing higher
             exposures than adults. Accumulating evidence suggest that
             low-level exposures during early life stages can affect
             brain development and lead to long-term behavioral
             impairments. We investigated the effects of zebrafish
             exposure to low doses of the two prominent PBDEs;
             2,2',4,4',5,-Pentabromodiphenyl ether (BDE-99) and
             2,2',4,4',-Tetrabromodiphenyl ether (BDE-47), during
             embryo-development on short- and long-term behavioral
             endpoints. We included the organophosphate pesticide
             chlorpyrifos (CPF) due to its well documented neurotoxicity
             across species from zebrafish to humans.Zebrafish embryos
             were exposed to the following individual treatments; 0.1%
             DMSO (vehicle control); 0.3μM CPF; 0.01, 0.03, 0.1, 0.3μM
             BDE-47; 0.003, 0.03, 0.3, 1, 3, 10, 20μM BDE-99 from 5
             until 120h post fertilization (hpf). Low exposure levels
             were determined as those not causing immediate overt
             toxicity, and behavior assays were conducted in the
             low-level range. At 144 hpf the larvae were tested for
             locomotor activity. At approximately 6 months of age adult
             zebrafish were tested in a behavioral battery including
             assays for anxiety-related behavior, sensorimotor response
             and habituation, social interaction, and predator
             avoidance.In the short-term, larval locomotor activity was
             reduced in larvae treated with 0.3μM CPF and 0.1μM BDE-47.
             BDE-99 treatment caused non-monotonic dose effects, with
             0.3μM causing hyperactivity and 1μM or higher causing
             hypoactivity. In the long-term, adult anxiety-related
             behavior was reduced in all treatments as measured in both
             the novel tank dive test and tap test.We show that exposure
             of zebrafish embryos to low concentrations of the brominated
             flame retardants BDE-47 and BDE-99, and the organophosphate
             pesticide CPF, caused both short- and long-term behavioral
             impairments. Interestingly, we also found that at very low
             exposure concentrations, where there were no visible effects
             on larval activity, adult behavior was still strongly
             affected.},
   Doi = {10.1016/j.neuro.2017.09.007},
   Key = {fds329144}
}

@article{fds327308,
   Author = {Rezvani, AH and Slade, S and Wells, C and Yenugonda, VM and Liu, Y and Brown, ML and Xiao, Y and Kellar, KJ and Levin, ED},
   Title = {Differential efficacies of the nicotinic α4β2
             desensitizing agents in reducing nicotine
             self-administration in female rats.},
   Journal = {Psychopharmacology},
   Volume = {234},
   Number = {17},
   Pages = {2517-2523},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1007/s00213-017-4641-6},
   Abstract = {Desensitization of neuronal nicotinic acetylcholine
             receptors holds promise as an effective treatment of tobacco
             addiction. Previously, we found that sazetidine-A (Saz-A),
             which selectively desensitizes α4β2 nicotinic receptors,
             significantly decreased intravenous (IV) nicotine
             self-administration (SA) in rats with an effective dose of
             3 mg/kg in acute and repeated injection studies. We also
             found that chronic infusions of Saz-A at doses of 2 and
             6 mg/kg/day significantly reduced nicotine SA in rats. In
             continuing studies, we have characterized other Saz-A
             analogs, YL-2-203 and VMY-2-95, to determine their
             efficacies in reducing nicotine SA in rats.Young adult
             female Sprague-Dawley rats were fitted with IV catheters and
             were trained for nicotine SA (0.03 mg/kg/infusion) on a
             fixed ratio 1 schedule for ten sessions. The same rats were
             also implanted subcutaneously with osmotic minipumps to
             continually deliver 2 or 6 mg/kg body weight YL-2-203,
             VMY-2-95, or saline for four consecutive weeks.Chronic
             administration of VMY-2-95 at doses of 2 and 6 mg/kg/day
             caused significant (p < 0.01) decreases in nicotine SA
             over the 2 weeks of continued nicotine SA and for the
             1-week period of resumed access after a week of enforced
             abstinence, whereas chronic administration of YL-2-203 at
             the same doses was not found to be effective.These studies,
             together with our previous studies of Saz-A, revealed a
             spectrum of efficacies for these α4β2 nicotinic receptor
             desensitizing agents and provide a path forward for the most
             effective compounds to be further developed as possible aids
             to smoking cessation.},
   Doi = {10.1007/s00213-017-4641-6},
   Key = {fds327308}
}

@article{fds330581,
   Author = {Massarsky, A and Abdel, A and Glazer, L and Levin, ED and Di Giulio,
             RT},
   Title = {Exposure to 1,2-Propanediol Impacts Early Development of
             Zebrafish (Danio rerio) and Induces Hyperactivity.},
   Journal = {Zebrafish},
   Volume = {14},
   Number = {3},
   Pages = {216-222},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1089/zeb.2016.1400},
   Abstract = {The use of electronic cigarettes (e-cigarettes) is
             increasing as an alternative to tobacco burning cigarettes;
             however, their safety remains to be fully determined. The
             long-term effects of e-cigarettes are unknown, including the
             effects of maternal e-cigarette use on pre- and postnatal
             development. Additional research on the safety of
             e-cigarettes is needed. Especially useful would be
             information from high- and moderate-throughput economic
             model systems. This study investigates the effects of
             1,2-propanediol, which was identified as the main component
             of e-cigarette liquid, on early development of zebrafish (an
             in vivo high-throughput model system that was recently
             proposed for the study of tobacco cigarette and e-cigarette
             toxicity). Zebrafish embryos were exposed to 1.25% or 2.5%
             1,2-propanediol from 6 to 72 h post-fertilization (hpf).
             We show that exposure to 1,2-propanediol did not
             significantly affect mortality. Hatching success was
             significantly lower in 2.5% 1,2-propanediol-exposed embryos
             at 48 hpf, but at 72 hpf no significant differences were
             noted. Moreover, exposure to 1,2-propanediol reduced growth
             and increased the incidence of string heart, pericardial
             edema, and yolk sac edema. Most importantly, developmental
             exposure to 1.25% 1,2-propanediol caused hyperactive
             swimming behavior in larvae. This study demonstrates that
             1,2-propanediol has adverse impacts on early development in
             zebrafish.},
   Doi = {10.1089/zeb.2016.1400},
   Key = {fds330581}
}

@article{fds324065,
   Author = {Hall, BJ and Abreu-Villaça, Y and Cauley, M and Junaid, S and White, H and Kiany, A and Levin, ED},
   Title = {The ventral hippocampal muscarinic cholinergic system plays
             a key role in sexual dimorphisms of spatial working memory
             in rats.},
   Journal = {Neuropharmacology},
   Volume = {117},
   Pages = {106-113},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.neuropharm.2017.01.019},
   Abstract = {Sex differences in cognitive processing and function have
             been documented in human and animal studies. Females have
             been found to perform better than males on non-spatial
             memory tasks, while males tend to outperform females on
             spatial memory tasks. The neural mechanisms underlying these
             sexual dimorphisms are unclear. However, it is known that
             the cholinergic system is critically involved in memory
             processes, and there are notable differences between males
             and females in cholinergic system function and receptor
             expression. In particular, there are sex differences in the
             processing of information in the frontal cortex and
             hippocampus. In this study, we examined the roles of
             muscarinic and nicotinic acetylcholine receptors in the
             medial frontal cortex (MfC) and ventral hippocampus (VH) on
             spatial working memory in male and female rats. Local
             infusions of scopolamine (SCOP) and mecamylamine (MEC) (10,
             20, 50 μg/side) were used to antagonize these receptors in
             each respective brain region during performance in the
             16-arm radial arm maze. Infusions of SCOP into the VH caused
             a significant increase in memory errors in female rats, but
             had no significant effect on males, while infusions of MEC
             into the VH had no effect on either sex. Infusions of both
             SCOP (50 μg/side) and MEC (20 μg/side) into the MfC caused
             working memory impairments in both sexes. These results show
             that muscarinic acetylcholine receptors in the VH are
             differentially vulnerable to spatial memory impairments in
             females. Ventral hippocampal muscarinic acetylcholine
             receptors may play a key role in male-female differences in
             spatial memory.},
   Doi = {10.1016/j.neuropharm.2017.01.019},
   Key = {fds324065}
}

@article{fds330582,
   Author = {van Thriel, C and Levin, E and Lein, P and Costa, LG and Westerink,
             RHS},
   Title = {Neural mechanisms of functional impairment across the
             lifespan: The 15th Biennial Meeting of the International
             Neurotoxicology Association and 39th Annual Meeting of the
             Neurobehavioral Teratology Society.},
   Journal = {NeuroToxicology},
   Volume = {59},
   Pages = {131-132},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.neuro.2017.03.003},
   Doi = {10.1016/j.neuro.2017.03.003},
   Key = {fds330582}
}

@article{fds324066,
   Author = {Abreu-Villaça, Y and Levin, ED},
   Title = {Developmental neurotoxicity of succeeding generations of
             insecticides.},
   Journal = {Environment International},
   Volume = {99},
   Pages = {55-77},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.envint.2016.11.019},
   Abstract = {Insecticides are by design toxic. They must be toxic to
             effectively kill target species of insects. Unfortunately,
             they also have off-target toxic effects that can harm other
             species, including humans. Developmental neurotoxicity is
             one of the most prominent off-target toxic risks of
             insecticides. Over the past seven decades several classes of
             insecticides have been developed, each with their own
             mechanisms of effect and toxic side effects. This review
             covers the developmental neurotoxicity of the succeeding
             generations of insecticides including organochlorines,
             organophosphates, pyrethroids, carbamates and
             neonicotinoids. The goal of new insecticide development is
             to more effectively kill target species with fewer toxic
             side effects on non-target species. From the experience with
             the developmental neurotoxicity caused by the generations of
             insecticides developed in the past advice is offered how to
             proceed with future insecticide development to decrease
             neurotoxic risk.},
   Doi = {10.1016/j.envint.2016.11.019},
   Key = {fds324066}
}

@article{fds330184,
   Author = {Macaulay, LJ and Chernick, M and Chen, A and Hinton, DE and Bailey, JM and Kullman, SW and Levin, ED and Stapleton, HM},
   Title = {Exposure to a PBDE/OH-BDE mixture alters juvenile zebrafish
             (Danio rerio) development.},
   Journal = {Environmental Toxicology & Chemistry},
   Volume = {36},
   Number = {1},
   Pages = {36-48},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1002/etc.3535},
   Abstract = {Polybrominated diphenyl ethers (PBDEs) and their metabolites
             (e.g., hydroxylated BDEs [OH-BDEs]) are contaminants
             frequently detected together in human tissues and are
             structurally similar to thyroid hormones. Thyroid hormones
             partially mediate metamorphic transitions between life
             stages in zebrafish, making this a critical developmental
             window that may be vulnerable to chemicals disrupting
             thyroid signaling. In the present study, zebrafish were
             exposed to 6-OH-BDE-47 (30 nM; 15 μg/L) alone, or to a
             low-dose (30 μg/L) or high-dose (600 μg/L) mixture of
             PentaBDEs, 6-OH-BDE-47 (0.5-6 μg/L), and
             2,4,6-tribromophenol (5-100 μg/L) during juvenile
             development (9-23 d postfertilization) and evaluated for
             developmental endpoints mediated by thyroid hormone
             signaling. Fish were sampled at 3 time points and examined
             for developmental and skeletal morphology, apical thyroid
             and skeletal gene markers, and modifications in swimming
             behavior (as adults). Exposure to the high-dose mixture
             resulted in >85% mortality within 1 wk of exposure,
             despite being below reported acute toxicity thresholds for
             individual congeners. The low-dose mixture and 6-OH-BDE-47
             groups exhibited reductions in body length and delayed
             maturation, specifically relating to swim bladder, fin, and
             pigmentation development. Reduced skeletal ossification was
             also observed in 6-OH-BDE-47-treated fish. Assessment of
             thyroid and osteochondral gene regulatory networks
             demonstrated significantly increased expression of genes
             that regulate skeletal development and thyroid hormones.
             Overall, these results indicate that exposures to
             PBDE/OH-BDE mixtures adversely impact zebrafish maturation
             during metamorphosis. Environ Toxicol Chem 2017;36:36-48. ©
             2016 SETAC.},
   Doi = {10.1002/etc.3535},
   Key = {fds330184}
}

@article{fds330275,
   Author = {Slotkin, TA and Stadler, A and Skavicus, S and Card, J and Ruff, J and Levin, ED and Seidler, FJ},
   Title = {Is There a Critical Period for the Developmental
             Neurotoxicity of Low-Level Tobacco Smoke
             Exposure?},
   Journal = {Toxicological Sciences (Elsevier)},
   Volume = {155},
   Number = {1},
   Pages = {75-84},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1093/toxsci/kfw180},
   Abstract = {Secondhand tobacco smoke exposure in pregnancy increases the
             risk of neurodevelopmental disorders. We evaluated in rats
             whether there is a critical period during which tobacco
             smoke extract (TSE) affects the development of acetylcholine
             and serotonin systems, prominent targets for adverse effects
             of nicotine and tobacco smoke. We simulated secondhand smoke
             exposure by administering TSE so as to produce nicotine
             concentrations one-tenth those in active smoking, with 3
             distinct, 10-day windows: premating, early gestation or late
             gestation. We conducted longitudinal evaluations in multiple
             brain regions, starting in early adolescence (postnatal day
             30) and continued to full adulthood (day 150). TSE exposure
             in any of the 3 windows impaired presynaptic cholinergic
             activity, exacerbated by a decrement in nicotinic
             cholinergic receptor concentrations. Although the adverse
             effects were seen for all 3 treatment windows, there was a
             distinct progression, with lowest sensitivity for premating
             exposure and higher sensitivity for gestational exposures.
             Serotonin receptors were also reduced by TSE exposure with
             the same profile: little effect with premating exposure,
             intermediate effect with early gestational exposure and
             large effect with late gestational exposure. As serotonergic
             circuits can offset the neurobehavioral impact of
             cholinergic deficits, these receptor changes were
             maladaptive. Thus, there is no single 'critical period' for
             effects of low-level tobacco smoke but there is differential
             sensitivity dependent upon the developmental stage at the
             time of exposure. Our findings reinforce the need to avoid
             secondhand smoke exposure not only during pregnancy, but
             also in the period prior to conception, or generally for
             women of childbearing age.},
   Doi = {10.1093/toxsci/kfw180},
   Key = {fds330275}
}

@article{fds330161,
   Author = {Rezvani, AH and Levin, ED and Cauley, M and Getachew, B and Tizabi,
             Y},
   Title = {Ketamine Differentially Attenuates Alcohol Intake in Male
             Versus Female Alcohol Preferring (P) Rats.},
   Journal = {Journal of drug and alcohol research},
   Volume = {6},
   Pages = {1-6},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.4303/jdar/236030},
   Abstract = {Although various pharmacological tools in combating
             addiction to alcohol are available, their efficacy is
             limited. Hence, there is a critical need for development of
             more effective medications. Recent advances in the field
             have identified the glutamatergic system as a potential
             novel target for intervention in addictive behaviors.Hence,
             we evaluated the effects of acute administration of low
             (subanesthetic) doses of ketamine, an NMDA receptor
             antagonist, on alcohol intake and alcohol preference in both
             male and female rats.Adult alcohol preferring (P) rats were
             exposed to two-bottle choice (ethanol 10% and water) for at
             least three weeks following a nine-day training period and
             the effects of various doses of ketamine (5 mg/kg, 7.5
             mg/kg, and 10 mg/kg, injected subcutaneously, SC) on
             consumption of alcohol over various time periods during a 24
             h interval were measured.Our results indicate that ketamine
             treatment significantly reduced both alcohol intake and
             preference in a time- and dose-dependent manner in both
             sexes. Moreover, a differential sensitivity between the
             sexes was observed. Thus, although alcohol intake was higher
             in males, female rats responded much more strongly to the
             highest dose of ketamine than males in the initial time
             periods.It is concluded that glutamatergic receptor
             manipulations may be of therapeutic potential in addiction
             to alcohol and that different sexes may respond
             differentially to such treatments.},
   Doi = {10.4303/jdar/236030},
   Key = {fds330161}
}


%% Li, Leon Y.   
@article{fds328429,
   Author = {Li, L and Rizzo, MT and Burkholder, AR and Killen,
             M},
   Title = {Theory of mind and resource allocation in the context of
             hidden inequality},
   Journal = {Cognitive Development},
   Volume = {43},
   Pages = {25-36},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.cogdev.2017.02.001},
   Doi = {10.1016/j.cogdev.2017.02.001},
   Key = {fds328429}
}

@article{fds328430,
   Author = {Li, L and Slevc, LR},
   Title = {Of Papers and Pens: Polysemes and Homophones in Lexical
             (mis)Selection},
   Journal = {Cognitive Science - A Multidisciplinary Journal},
   Volume = {41},
   Pages = {1532-1548},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1111/cogs.12402},
   Doi = {10.1111/cogs.12402},
   Key = {fds328430}
}


%% Lisanby, Sarah H.   
@article{fds330881,
   Author = {McCall, WV and Lisanby, SH and Rosenquist, PB and Dooley, M and Husain,
             MM and Knapp, RG and Petrides, G and Rudorfer, MV and Young, RC and McClintock, SM and Mueller, M and Prudic, J and Greenberg, RM and Weiner, RD and Bailine, SH and Youssef, NA and McCloud, L and Kellner,
             CH and CORE/PRIDE Work Group},
   Title = {Effects of continuation electroconvulsive therapy on quality
             of life in elderly depressed patients: A randomized clinical
             trial.},
   Journal = {Journal of Psychiatric Research},
   Volume = {97},
   Pages = {65-69},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.jpsychires.2017.11.001},
   Abstract = {We examined whether electroconvulsive therapy (ECT) plus
             medications ("STABLE + PHARM" group) had superior HRQOL
             compared with medications alone ("PHARM" group) as
             continuation strategy after successful acute right
             unilateral ECT for major depressive disorder (MDD). We
             hypothesized that scores from the Medical Outcomes Study
             Short Form-36 (SF-36) would be higher during continuation
             treatment in the "STABLE + PHARM" group versus the "PHARM"
             group. The overall study design was called "Prolonging
             Remission in Depressed Elderly" (PRIDE). Remitters to the
             acute course of ECT were re-consented to enter a 6 month RCT
             of "STABLE + PHARM" versus "PHARM". Measures of depressive
             symptoms and cognitive function were completed by blind
             raters; SF-36 measurements were patient self-report every 4
             weeks. Participants were 120 patients >60 years old.
             Patients with dementia, schizophrenia, bipolar disorder, or
             substance abuse were excluded. The "PHARM" group received
             venlafaxine and lithium. The "STABLE + PHARM" received the
             same medications, plus 4 weekly outpatient ECT sessions,
             with additional ECT session as needed. Participants were
             mostly female (61.7%) with a mean age of 70.5 ± 7.2
             years. "STABLE + PHARM" patients received 4.5 ± 2.5 ECT
             sessions during Phase 2. "STABLE + PHARM" group had 7
             point advantage (3.5-10.4, 95% CI) for Physical Component
             Score of SF-36 (P < 0.0001), and 8.2 point advantage
             (4.2-12.2, 95% CI) for Mental Component Score
             (P < 0.0001). Additional ECT resulted in overall net
             health benefit. Consideration should be given to
             administration of additional ECT to prevent relapse during
             the continuation phase of treatment of MDD. CLINICAL
             TRIALS.GOV: NCT01028508.},
   Doi = {10.1016/j.jpsychires.2017.11.001},
   Key = {fds330881}
}

@article{fds330049,
   Author = {Loo, CK and Husain, MM and McDonald, WM and Aaronson, S and O'Reardon,
             JP and Alonzo, A and Weickert, CS and Martin, DM and McClintock, SM and Mohan, A and Lisanby, SH and International Consortium of Research
             in tDCS (ICRT)},
   Title = {International randomized-controlled trial of transcranial
             Direct Current Stimulation in depression.},
   Journal = {Brain Stimulation},
   Volume = {11},
   Number = {1},
   Pages = {125-133},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.brs.2017.10.011},
   Abstract = {Evidence suggests that transcranial Direct Current
             Stimulation (tDCS) has antidepressant effects in unipolar
             depression, but there is limited information for patients
             with bipolar depression. Additionally, prior research
             suggests that brain derived neurotrophic factor (BDNF)
             Val66Met genotype may moderate response to tDCS.To examine
             tDCS efficacy in unipolar and bipolar depression and assess
             if BDNF genotype is associated with antidepressant response
             to tDCS.130 participants diagnosed with a major depressive
             episode were randomized to receive active (2.5 milliamps
             (mA), 30 min) or sham (0.034 mA and two 60-second current
             ramps up to 1 and 0.5 mA) tDCS to the left prefrontal
             cortex, administered in 20 sessions over 4 weeks, in a
             double-blinded, international multisite study. Mixed effects
             repeated measures analyses assessed change in mood and
             neuropsychological scores in participants with at least one
             post-baseline rating in the unipolar (N = 84) and bipolar
             (N = 36) samples.Mood improved significantly over the
             4-week treatment period in both unipolar (p = 0.001) and
             bipolar groups (p < 0.001). Among participants with
             unipolar depression, there were more remitters in the sham
             treatment group (p = 0.03). There was no difference
             between active and sham stimulation in the bipolar sample.
             BDNF genotype was unrelated to antidepressant
             outcome.Overall, this study found no antidepressant
             difference between active and sham stimulation for unipolar
             or bipolar depression. However, the possibility that the low
             current delivered in the sham tDCS condition was
             biologically active cannot be discounted. Moreover, BDNF
             genotype did not moderate antidepressant
             outcome.www.clinicaltrials.gov, NCT01562184.},
   Doi = {10.1016/j.brs.2017.10.011},
   Key = {fds330049}
}

@article{fds332918,
   Author = {Bikson, M and Brunoni, AR and Charvet, LE and Clark, VP and Cohen, LG and Deng, Z-D and Dmochowski, J and Edwards, DJ and Frohlich, F and Kappenman, ES and Lim, KO and Loo, C and Mantovani, A and McMullen, DP and Parra, LC and Pearson, M and Richardson, JD and Rumsey, JM and Sehatpour, P and Sommers, D and Unal, G and Wassermann, EM and Woods,
             AJ and Lisanby, SH},
   Title = {Rigor and reproducibility in research with transcranial
             electrical stimulation: An NIMH-sponsored
             workshop.},
   Journal = {Brain Stimulation},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.brs.2017.12.008},
   Abstract = {Neuropsychiatric disorders are a leading source of
             disability and require novel treatments that target
             mechanisms of disease. As such disorders are thought to
             result from aberrant neuronal circuit activity,
             neuromodulation approaches are of increasing interest given
             their potential for manipulating circuits directly. Low
             intensity transcranial electrical stimulation (tES) with
             direct currents (transcranial direct current stimulation,
             tDCS) or alternating currents (transcranial alternating
             current stimulation, tACS) represent novel, safe,
             well-tolerated, and relatively inexpensive putative
             treatment modalities.This report seeks to promote the
             science, technology and effective clinical applications of
             these modalities, identify research challenges, and suggest
             approaches for addressing these needs in order to achieve
             rigorous, reproducible findings that can advance clinical
             treatment.The National Institute of Mental Health (NIMH)
             convened a workshop in September 2016 that brought together
             experts in basic and human neuroscience, electrical
             stimulation biophysics and devices, and clinical trial
             methods to examine the physiological mechanisms underlying
             tDCS/tACS, technologies and technical strategies for
             optimizing stimulation protocols, and the state of the
             science with respect to therapeutic applications and trial
             designs.Advances in understanding mechanisms, methodological
             and technological improvements (e.g., electronics,
             computational models to facilitate proper dosing), and
             improved clinical trial designs are poised to advance
             rigorous, reproducible therapeutic applications of these
             techniques. A number of challenges were identified and
             meeting participants made recommendations made to address
             them.These recommendations align with requirements in NIMH
             funding opportunity announcements to, among other needs,
             define dosimetry, demonstrate dose/response relationships,
             implement rigorous blinded trial designs, employ
             computational modeling, and demonstrate target engagement
             when testing stimulation-based interventions for the
             treatment of mental disorders.},
   Doi = {10.1016/j.brs.2017.12.008},
   Key = {fds332918}
}

@article{fds329063,
   Author = {Davis, SW and Luber, B and Murphy, DLK and Lisanby, SH and Cabeza,
             R},
   Title = {Frequency-specific neuromodulation of local and distant
             connectivity in aging and episodic memory
             function.},
   Journal = {Human Brain Mapping},
   Volume = {38},
   Number = {12},
   Pages = {5987-6004},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1002/hbm.23803},
   Abstract = {A growing literature has focused on the brain's ability to
             augment processing in local regions by recruiting distant
             communities of neurons in response to neural decline or
             insult. In particular, both younger and older adult
             populations recruit bilateral prefrontal cortex (PFC) as a
             means of compensating for increasing neural effort to
             maintain successful cognitive function. However, it remains
             unclear how local changes in neural activity affect the
             recruitment of this adaptive mechanism. To address this
             problem, we combined graph theoretical measures from
             functional MRI with diffusion weighted imaging and
             repetitive transcranial magnetic stimulation (rTMS) to
             resolve a central hypothesis: how do aged brains flexibly
             adapt to local changes in cortical activity? Specifically,
             we applied neuromodulation to increase or decrease local
             activity in a cortical region supporting successful memory
             encoding (left dorsolateral PFC or DLPFC) using 5 or 1 Hz
             rTMS, respectively. We then assessed a region's local
             within-module degree, or the distributed between-module
             degree (BMD) between distant cortical communities. We
             predicted that (1) local stimulation-related deficits may be
             counteracted by boosting BMD between bilateral PFC, and that
             this effect should be (2) positively correlated with
             structural connectivity. Both predictions were confirmed; 5
             Hz rTMS increased local success-related activity and local
             increases in PFC connectivity, while 1 Hz rTMS decreases
             local activity and triggered a more distributed pattern of
             bilateral PFC connectivity to compensate for this local
             inhibitory effect. These results provide an integrated,
             causal explanation for the network interactions associated
             with successful memory encoding in older adults. Hum Brain
             Mapp 38:5987-6004, 2017. © 2017 Wiley Periodicals,
             Inc.},
   Doi = {10.1002/hbm.23803},
   Key = {fds329063}
}

@article{fds330048,
   Author = {Holtzheimer, PE and Husain, MM and Lisanby, SH and Taylor, SF and Whitworth, LA and McClintock, S and Slavin, KV and Berman, J and McKhann, GM and Patil, PG and Rittberg, BR and Abosch, A and Pandurangi,
             AK and Holloway, KL and Lam, RW and Honey, CR and Neimat, JS and Henderson,
             JM and DeBattista, C and Rothschild, AJ and Pilitsis, JG and Espinoza,
             RT and Petrides, G and Mogilner, AY and Matthews, K and Peichel, D and Gross, RE and Hamani, C and Lozano, AM and Mayberg,
             HS},
   Title = {Subcallosal cingulate deep brain stimulation for
             treatment-resistant depression: a multisite, randomised,
             sham-controlled trial.},
   Journal = {The Lancet. Psychiatry},
   Volume = {4},
   Number = {11},
   Pages = {839-849},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/s2215-0366(17)30371-1},
   Abstract = {Deep brain stimulation (DBS) of the subcallosal cingulate
             white matter has shown promise as an intervention for
             patients with chronic, unremitting depression. To test the
             safety and efficacy of DBS for treatment-resistant
             depression, a prospective, randomised, sham-controlled trial
             was conducted.Participants with treatment-resistant
             depression were implanted with a DBS system targeting
             bilateral subcallosal cingulate white matter and randomised
             to 6 months of active or sham DBS, followed by 6 months of
             open-label subcallosal cingulate DBS. Randomisation was
             computer generated with a block size of three at each site
             before the site started the study. The primary outcome was
             frequency of response (defined as a 40% or greater reduction
             in depression severity from baseline) averaged over months
             4-6 of the double-blind phase. A futility analysis was
             performed when approximately half of the proposed sample
             received DBS implantation and completed the double-blind
             phase. At the conclusion of the 12-month study, a subset of
             patients were followed up for up to 24 months. The study is
             registered at ClinicalTrials.gov, number NCT00617162.Before
             the futility analysis, 90 participants were randomly
             assigned to active (n=60) or sham (n=30) stimulation between
             April 10, 2008, and Nov 21, 2012. Both groups showed
             improvement, but there was no statistically significant
             difference in response during the double-blind,
             sham-controlled phase (12 [20%] patients in the stimulation
             group vs five [17%] patients in the control group). 28
             patients experienced 40 serious adverse events; eight of
             these (in seven patients) were deemed to be related to the
             study device or surgery.This study confirmed the safety and
             feasibility of subcallosal cingulate DBS as a treatment for
             treatment-resistant depression but did not show
             statistically significant antidepressant efficacy in a
             6-month double-blind, sham-controlled trial. Future studies
             are needed to investigate factors such as clinical features
             or electrode placement that might improve efficacy.Abbott
             (previously St Jude Medical).},
   Doi = {10.1016/s2215-0366(17)30371-1},
   Key = {fds330048}
}

@article{fds324470,
   Author = {McCall, WV and Lisanby, SH and Rosenquist, PB and Dooley, M and Husain,
             MM and Knapp, RG and Petrides, G and Rudorfer, MV and Young, RC and McClintock, SM and Mueller, M and Prudic, J and Greenberg, RM and Weiner, RD and Bailine, SH and Riley, MA and McCloud, L and Kellner, CH and CORE/PRIDE Work Group},
   Title = {Effects of a Course of Right Unilateral Ultrabrief Pulse
             Electroconvulsive Therapy Combined With Venlafaxine on
             Insomnia Symptoms in Elderly Depressed Patients.},
   Journal = {Journal of Clinical Psychiatry},
   Volume = {32},
   Number = {3},
   Pages = {219-220},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.4088/jcp.16m11089},
   Abstract = {Antidepressant medications have a variety of effects on
             sleep, apart from their antidepressant effects. It is
             unknown whether electroconvulsive therapy (ECT) has effects
             on perceived sleep in depressed patients. This secondary
             analysis examines the effects of ECT on perceived sleep,
             separate from its antidepressant effects.Elderly patients
             with major depressive disorder, as defined by DSM-IV,
             received open-label high-dose, right unilateral ultrabrief
             pulse ECT, combined with venlafaxine, as part of
             participating in phase 1 of the National Institute of Mental
             Health-supported study Prolonging Remission in Depressed
             Elderly (PRIDE). Phase 1 of PRIDE participant enrollment
             period extended from February 2009 to August 2014.
             Depression severity was measured with the Hamilton
             Depression Rating Scale-24 item (HDRS₂₄), and measures
             of insomnia severity were extracted from the HDRS₂₄.
             Participants were characterized at baseline as either
             "high-insomnia" or "low-insomnia" subtypes, based upon the
             sum of the 3 HDRS₂₄ sleep items as either 4-6 or 0-3,
             respectively. Insomnia scores were followed during ECT and
             were adjusted for the sum of all the HDRS₂₄ non-sleep
             items. Generalized linear models were used for longitudinal
             analysis of insomnia scores.Two hundred forty patients
             participated, with 48.3% in the high-insomnia and 51.7% in
             the low-insomnia group. Although there was a reduction in
             the insomnia scores in the high-insomnia group, only 12.4%
             of them experienced remission of insomnia after a course of
             ECT, despite an increase in utilization of sleep aids across
             the course of ECT, from 8.6% to 23.2%. The degree of
             improvement in insomnia symptoms paralleled the degree of
             improvement in non-insomnia symptoms. A "low" amount of
             improvement on the sum of the HDRS non-insomnia items
             (HDRS-sleep) was accompanied by a "low" amount of
             improvement in insomnia scores (change of -1.6 ± 1.2, P <
             .0001), while a "high" amount of improvement on the sum of
             the HDRS non-insomnia items was accompanied by a "higher"
             amount of improvement in insomnia scores (change of -3.1 ±
             1.6, P < .0001). After adjustment for non-insomnia symptoms,
             there was no change in insomnia in the low-insomnia group.We
             found that ECT, combined with venlafaxine, has a modest
             anti-insomnia effect that is linked to its antidepressant
             effect. Most patients will have some degree of residual
             insomnia after ECT, and will require some consideration of
             whether additional, targeted assessment and treatment of
             insomnia is warranted.ClinicalTrials.gov Identifier:
             NCT01028508.},
   Doi = {10.4088/jcp.16m11089},
   Key = {fds324470}
}

@article{fds330050,
   Author = {Zhi-De Deng, and Lisanby, SH},
   Title = {Electric field characteristics of low-field synchronized
             transcranial magnetic stimulation (sTMS).},
   Journal = {Annual International Conference of the IEEE Engineering in
             Medicine and Biology - Proceedings},
   Volume = {2017},
   Pages = {1445-1448},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1109/embc.2017.8037106},
   Abstract = {Low-field synchronized trancranial magnetic stimulation
             (sTMS) was hypothesized to have significant therapeutic
             effects in patients with major depressive disorder by
             entrainment of neural oscillations. The sTMS device is
             comprised of neodymium magnets mounted over multiple brain
             regions and set to rotate at the patients alpha frequency.
             We characterized the electric field strength and
             distribution of sTMS using the finite element method. We
             found that the maximum induced electric field strength on
             the surfaces of the head and cortex are approximately
             0.06Vm-1 and 0.02Vm-1, respectively. These field strengths
             are an order of magnitude lower than that delivered by
             transcranial current stimulation.},
   Doi = {10.1109/embc.2017.8037106},
   Key = {fds330050}
}

@article{fds328089,
   Author = {Lisanby, SH},
   Title = {Noninvasive Brain Stimulation for Depression - The Devil Is
             in the Dosing.},
   Journal = {The New England journal of medicine},
   Volume = {376},
   Number = {26},
   Pages = {2593-2594},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1056/nejme1702492},
   Doi = {10.1056/nejme1702492},
   Key = {fds328089}
}

@article{fds323757,
   Author = {Lee, WH and Lisanby, SH and Laine, AF and Peterchev,
             AV},
   Title = {Minimum Electric Field Exposure for Seizure Induction with
             Electroconvulsive Therapy and Magnetic Seizure
             Therapy.},
   Journal = {Neuropsychopharmacology (Nature)},
   Volume = {42},
   Number = {6},
   Pages = {1192-1200},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1038/npp.2016.276},
   Abstract = {Lowering and individualizing the current amplitude in
             electroconvulsive therapy (ECT) has been proposed as a means
             to produce stimulation closer to the neural activation
             threshold and more focal seizure induction, which could
             potentially reduce cognitive side effects. However, the
             effect of current amplitude on the electric field (E-field)
             in the brain has not been previously linked to the current
             amplitude threshold for seizure induction. We coupled
             MRI-based E-field models with amplitude titrations of motor
             threshold (MT) and seizure threshold (ST) in four nonhuman
             primates (NHPs) to determine the strength, distribution, and
             focality of stimulation in the brain for four ECT electrode
             configurations (bilateral, bifrontal, right-unilateral, and
             frontomedial) and magnetic seizure therapy (MST) with cap
             coil on vertex. At the amplitude-titrated ST, the stimulated
             brain subvolume (23-63%) was significantly less than for
             conventional ECT with high, fixed current (94-99%). The
             focality of amplitude-titrated right-unilateral ECT (25%)
             was comparable to cap coil MST (23%), demonstrating that ECT
             with a low current amplitude and focal electrode placement
             can induce seizures with E-field as focal as MST, although
             these electrode and coil configurations affect differently
             specific brain regions. Individualizing the current
             amplitude reduced interindividual variation in the
             stimulation focality by 40-53% for ECT and 26% for MST,
             supporting amplitude individualization as a means of dosing
             especially for ECT. There was an overall significant
             correlation between the measured amplitude-titrated ST and
             the prediction of the E-field models, supporting a potential
             role of these models in dosing of ECT and MST. These
             findings may guide the development of seizure therapy dosing
             paradigms with improved risk/benefit ratio.},
   Doi = {10.1038/npp.2016.276},
   Key = {fds323757}
}

@article{fds326503,
   Author = {Radman, T and Lisanby, SH},
   Title = {New directions in the rational design of electrical and
             magnetic seizure therapies: individualized Low Amplitude
             Seizure Therapy (iLAST) and Magnetic Seizure Therapy
             (MST).},
   Journal = {International Review of Psychiatry (Informa)},
   Volume = {29},
   Number = {2},
   Pages = {63-78},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1080/09540261.2017.1304898},
   Abstract = {Electroconvulsive therapy remains a key treatment option for
             severe cases of depression, but undesirable side-effects
             continue to limit its use. Innovations in the design of
             novel seizure therapies seek to improve its risk benefit
             ratio through enhanced control of the focality of
             stimulation. The design of seizure therapies with increased
             spatial precision is motivated by avoiding stimulation of
             deep brain structures implicated in memory retention,
             including the hippocampus. The development of two
             innovations in seizure therapy-individualized low-amplitude
             seizure therapy (iLAST) and magnetic seizure therapy (MST),
             are detailed. iLAST is a method of seizure titration
             involving reducing current spread in the brain by titrating
             current amplitude from the traditional fixed amplitudes.
             MST, which can be used in conjunction with iLAST dosing
             methods, involves the use of magnetic stimulation to reduce
             shunting and spreading of current by the scalp occurring
             during electrical stimulation. Evidence is presented on the
             rationale for increasing the focality of ECT in hopes of
             preserving its effectiveness, while reducing cognitive
             side-effects. Finally, the value of electric field and
             neural modelling is illustrated to explain observed clinical
             effects of modifications to ECT technique, and their utility
             in the rational design of the next generation of seizure
             therapies.},
   Doi = {10.1080/09540261.2017.1304898},
   Key = {fds326503}
}

@article{fds326065,
   Author = {Kellner, CH and Knapp, RG and Petrides, G and McCall, WV and Young, RC and Husain, MM and Lisanby, SH and CORE/PRIDE Work
             Group},
   Title = {A Step Toward Optimizing Treatment Schedules for
             Continuation ECT: Response to Rasmussen.},
   Journal = {American Journal of Psychiatry},
   Volume = {174},
   Number = {4},
   Pages = {397-398},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.16111322r},
   Doi = {10.1176/appi.ajp.2017.16111322r},
   Key = {fds326065}
}

@article{fds323725,
   Author = {Luber, BM and Davis, S and Bernhardt, E and Neacsiu, A and Kwapil, L and Lisanby, SH and Strauman, TJ},
   Title = {Using neuroimaging to individualize TMS treatment for
             depression: Toward a new paradigm for imaging-guided
             intervention.},
   Journal = {NeuroImage},
   Volume = {148},
   Pages = {1-7},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2016.12.083},
   Abstract = {The standard clinical technique for using repetitive
             transcranial magnetic stimulation (rTMS) for major
             depressive disorder (MDD) is associated with limited
             efficacy to date. Such limited efficacy may be due to
             reliance on scalp-based targeting rather than
             state-of-the-science methods which incorporate fMRI-guided
             neuronavigation based on a specific model of neurocircuit
             dysfunction. In this review, we examine such a specific
             model drawn from regulatory focus theory, which postulates
             two brain/behavior systems, the promotion and prevention
             systems, underlying goal pursuit. Individual differences in
             these systems have been shown to predict vulnerability to
             MDD as well as to comorbid generalized anxiety disorder
             (GAD). Activation of an individual's promotion or prevention
             goals via priming leads to motivational and affective
             responses modulated by the individual's appraisal of their
             progress in attaining the goal. In addition, priming
             promotion vs. prevention goals induces discriminable
             patterns of brain activation that are sensitive to the
             effects of depression and anxiety: MDD is associated with
             promotion system failure, anhedonic/dysphoric symptoms, and
             hypoactivation in specific regions in left prefrontal
             cortex, whereas GAD is associated with prevention system
             failure, hypervigilant/agitated symptoms, and
             hyperactivation in right prefrontal cortex (PFC). These left
             and right PFC locations can be directly targeted in an
             individualized manner for TMS. Additionally, this
             individually targeted rTMS can be integrated with cognitive
             interventions designed to activate the neural circuitry
             associated with promotion vs. prevention, thus allowing the
             neuroplasticity induced by the rTMS to benefit the systems
             likely to be involved in remediating depression. Targeted
             engagement of cortical systems involved in emotion
             regulation using individualized fMRI guidance may help
             increase the efficacy of rTMS in depression.},
   Doi = {10.1016/j.neuroimage.2016.12.083},
   Key = {fds323725}
}

@article{fds325353,
   Author = {Thome, A and Marrone, DF and Ellmore, TM and Chawla, MK and Lipa, P and Ramirez-Amaya, V and Lisanby, SH and McNaughton, BL and Barnes,
             CA},
   Title = {Evidence for an Evolutionarily Conserved Memory Coding
             Scheme in the Mammalian Hippocampus.},
   Journal = {The Journal of neuroscience : the official journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {10},
   Pages = {2795-2801},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1523/jneurosci.3057-16.2017},
   Abstract = {Decades of research identify the hippocampal formation as
             central to memory storage and recall. Events are stored via
             distributed population codes, the parameters of which (e.g.,
             sparsity and overlap) determine both storage capacity and
             fidelity. However, it remains unclear whether the parameters
             governing information storage are similar between species.
             Because episodic memories are rooted in the space in which
             they are experienced, the hippocampal response to navigation
             is often used as a proxy to study memory. Critically, recent
             studies in rodents that mimic the conditions typical of
             navigation studies in humans and nonhuman primates (i.e.,
             virtual reality) show that reduced sensory input alters
             hippocampal representations of space. The goal of this study
             was to quantify this effect and determine whether there are
             commonalities in information storage across species. Using
             functional molecular imaging, we observe that navigation in
             virtual environments elicits activity in fewer CA1 neurons
             relative to real-world conditions. Conversely, comparable
             neuronal activity is observed in hippocampus region CA3 and
             the dentate gyrus under both conditions. Surprisingly, we
             also find evidence that the absolute number of neurons used
             to represent an experience is relatively stable between
             nonhuman primates and rodents. We propose that this
             convergence reflects an optimal ensemble size for episodic
             memories.SIGNIFICANCE STATEMENT One primary factor
             constraining memory capacity is the sparsity of the engram,
             the proportion of neurons that encode a single experience.
             Investigating sparsity in humans is hampered by the lack of
             single-cell resolution and differences in behavioral
             protocols. Sparsity can be quantified in freely moving
             rodents, but extrapolating these data to humans assumes that
             information storage is comparable across species and is
             robust to restraint-induced reduction in sensory input.
             Here, we test these assumptions and show that species
             differences in brain size build memory capacity without
             altering the structure of the data being stored.
             Furthermore, sparsity in most of the hippocampus is
             resilient to reduced sensory information. This information
             is vital to integrating animal data with human imaging
             navigation studies.},
   Doi = {10.1523/jneurosci.3057-16.2017},
   Key = {fds325353}
}

@article{fds320774,
   Author = {McCall, WV and Lisanby, SH and Rosenquist, PB and Dooley, M and Husain,
             MM and Knapp, RG and Petrides, G and Rudorfer, MV and Young, RC and McClintock, SM and Mueller, M and Prudic, J and Greenberg, RM and Weiner, RD and Bailine, SH and Riley, MA and McCloud, L and Kellner, CH and CORE/PRIDE Work Group},
   Title = {Effects of a right unilateral ultrabrief pulse
             electroconvulsive therapy course on health related quality
             of life in elderly depressed patients.},
   Journal = {Journal of Affective Disorders},
   Volume = {209},
   Pages = {39-45},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.jad.2016.11.003},
   Abstract = {Patients with Major Depressive Disorder (MDD) referred for
             electroconvulsive therapy (ECT) have poorer Health Related
             Quality of Life (HRQOL), compared with other patients with
             MDD, but ECT is associated with significant and durable
             improvement in HRQOL. However, no prior research has focused
             exclusively on elderly patients with MDD receiving ECT.HRQOL
             data from 240 depressed patients over the age of 60 was
             measured with the Medical Outcomes Study Short Form 36
             (SF-36). The SF-36 was measured before and after a course of
             acute ECT. Predictors of change in HRQOL scores were
             identified by generalized linear modeling.At baseline,
             participants showed very poor HRQOL. After treatment with
             ECT, the full sample showed marked and significant
             improvement across all SF-36 measures, with the largest
             gains seen in dimensions of mental health. Across all
             participants, the Physical Component Summary (PCS) score
             improved by 2.1 standardized points (95% CI, 0.61,3.56),
             while the Mental Component Summary (MCS) score improved by
             12.5 points (95% CI, 7.2,10.8) Compared with non-remitters,
             remitters showed a trend toward greater improvement in the
             PCS summary score of 2.7 points (95%CI, -0.45, 5.9), while
             the improvement in the MCS summary score was significantly
             greater (8.5 points, 95% CI, 4.6,12.3) in the remitters than
             non-remitters. Post-ECT SF-36 measurements were consistently
             and positively related to baseline scores and
             remitter/non-remitter status or change in depression
             severity from baseline. Objective measures of cognitive
             function had no significant relationships to changes in
             SF-36 scores.This study's limitations include that it was an
             open label study with no comparison group, and
             generalizability is limited to elderly patients.ECT
             providers and elderly patients with MDD treated with ECT can
             be confident that ECT will result in improved HRQOL in the
             short-term. Attaining remission is a key factor in the
             improvement of HRQOL. Acute changes in select cognitive
             functions were outweighed by improvement in depressive
             symptoms in determining the short term HRQOL of the
             participants treated with ECT.},
   Doi = {10.1016/j.jad.2016.11.003},
   Key = {fds320774}
}

@article{fds322765,
   Author = {Peterchev, AV and Luber, B and Westin, GG and Lisanby,
             SH},
   Title = {Pulse Width Affects Scalp Sensation of Transcranial Magnetic
             Stimulation.},
   Journal = {Brain Stimulation},
   Volume = {10},
   Number = {1},
   Pages = {99-105},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.brs.2016.09.007},
   Abstract = {Scalp sensation and pain comprise the most common side
             effect of transcranial magnetic stimulation (TMS), which can
             reduce tolerability and complicate experimental blinding.We
             explored whether changing the width of single TMS pulses
             affects the quality and tolerability of the resultant
             somatic sensation.Using a controllable pulse parameter TMS
             device with a figure-8 coil, single monophasic magnetic
             pulses inducing electric field with initial phase width of
             30, 60, and 120 µs were delivered in 23 healthy
             volunteers. Resting motor threshold of the right first
             dorsal interosseus was determined for each pulse width, as
             reported previously. Subsequently, pulses were delivered
             over the left dorsolateral prefrontal cortex at each of the
             three pulse widths at two amplitudes (100% and 120% of the
             pulse-width-specific motor threshold), with 20 repetitions
             per condition delivered in random order. After each pulse,
             subjects rated 0-to-10 visual analog scales for Discomfort,
             Sharpness, and Strength of the sensation.Briefer TMS pulses
             with amplitude normalized to the motor threshold were
             perceived as slightly more uncomfortable than longer pulses
             (with an average 0.89 point increase on the Discomfort scale
             for pulse width of 30 µs compared to 120 µs). The
             sensation of the briefer pulses was felt to be substantially
             sharper (2.95 points increase for 30 µs compared to
             120 µs pulse width), but not stronger than longer pulses.
             As expected, higher amplitude pulses increased the perceived
             discomfort and strength, and, to a lesser degree the
             perceived sharpness.Our findings contradict a previously
             published hypothesis that briefer TMS pulses are more
             tolerable. We discovered that the opposite is true, which
             merits further study as a means of enhancing tolerability in
             the context of repetitive TMS.},
   Doi = {10.1016/j.brs.2016.09.007},
   Key = {fds322765}
}

@article{fds320196,
   Author = {Taylor, SF and Bhati, MT and Dubin, MJ and Hawkins, JM and Lisanby, SH and Morales, O and Reti, IM and Sampson, S and Short, EB and Spino, C and Watcharotone, K and Wright, J},
   Title = {A naturalistic, multi-site study of repetitive transcranial
             magnetic stimulation therapy for depression.},
   Journal = {Journal of Affective Disorders},
   Volume = {208},
   Pages = {284-290},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jad.2016.08.049},
   Abstract = {Repetitive transcranial magnetic stimulation (rTMS) was
             approved in 2008 in the United States, and there are
             relatively few studies describing its use in regular
             clinical practice since approval.From April 2011 to October
             2014, ten sites within the National Network of Depression
             Centers (NNDC) provided data on 62 evaluable patients with a
             depressive episode. Treatment was determined
             naturalistically. Response was assessed by the Quick
             Inventory of Depressive Symptoms, Self-Report (QIDS-SR) as
             the primary outcome, and the Patient Health Questionnaire-9
             (PHQ-9) and the clinician-rated Clinical Global Impression
             (CGI) as secondary depression measures.Enrolled patients
             exhibited significant treatment resistance, with 70.2%
             reporting more than 4 prior depressive episodes. Most
             patients received treatment with standard parameters (10Hz
             over the left dorsolateral prefrontal cortex), although
             22.6% of the patients received 1 or 5Hz stimulation at some
             point. Over 6 weeks of treatment, response and remission
             rates were 29.4% and 5.9%, respectively, for the QIDS-SR;
             39.2% and 15.7%, respectively, for the PHQ-9; and 50.9% and
             17.9%, respectively, for the CGI. Moderator analyses
             revealed no effect of prior depressive episodes, history of
             ECT or gender, although early life stress predicted a better
             response to rTMS therapy.The study was an open-label,
             registry trial, with relatively coarse clinical data,
             reflecting practice only in academic, depression-specialty
             centers. Because of the relatively small size and
             heterogeneity of the sample, type 2 errors are possible and
             positive findings are in need of replication.rTMS
             demonstrates effectiveness in clinical practice within the
             NNDC, although remission rates appear slightly lower in
             comparison with other recent naturalistic
             studies.},
   Doi = {10.1016/j.jad.2016.08.049},
   Key = {fds320196}
}

@article{fds319643,
   Author = {Kuo, H-C and Ferre, CL and Carmel, JB and Gowatsky, JL and Stanford, AD and Rowny, SB and Lisanby, SH and Gordon, AM and Friel,
             KM},
   Title = {Using diffusion tensor imaging to identify corticospinal
             tract projection patterns in children with unilateral
             spastic cerebral palsy.},
   Journal = {Developmental Medicine & Child Neurology},
   Volume = {59},
   Number = {1},
   Pages = {65-71},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1111/dmcn.13192},
   Abstract = {To determine whether diffusion tensor imaging (DTI) can be
             an independent assessment for identifying the corticospinal
             tract (CST) projecting from the more-affected motor cortex
             in children with unilateral spastic cerebral palsy
             (CP).Twenty children with unilateral spastic CP participated
             in this study (16 males, four females; mean age 9y 2mo
             [standard deviation (SD) 3y 2mo], Manual Ability
             Classification System [MACS] level I-III). We used DTI
             tractography to reconstruct the CST projecting from the
             more-affected motor cortex. We mapped the motor
             representation of the more-affected hand by stimulating the
             more- and the less-affected motor cortex measured with
             single-pulse transcranial magnetic stimulation (TMS). We
             then verified the presence or absence of the contralateral
             CST by comparing the TMS map and DTI tractography. Fisher's
             exact test was used to determine the association between
             findings of TMS and DTI.DTI tractography successfully
             identified the CST controlling the more-affected hand
             (sensitivity=82%, specificity=78%).Contralateral CST
             projecting from the lesioned motor cortex assessed by DTI is
             consistent with findings of TMS mapping. Since CST
             connectivity may be predictive of response to certain upper
             extremity treatments, DTI-identified CST connectivity may
             potentially be valuable for determining such connectivity
             where TMS is unavailable or inadvisable for children with
             seizures.},
   Doi = {10.1111/dmcn.13192},
   Key = {fds319643}
}


%% Madden, David J.   
@article{fds331315,
   Author = {Zhuang, J and Madden, DJ and Duong-Fernandez, X and Chen, N-K and Cousins, SW and Potter, GG and Diaz, MT and Whitson,
             HE},
   Title = {Language processing in age-related macular degeneration
             associated with unique functional connectivity signatures in
             the right hemisphere.},
   Journal = {Neurobiology of Aging},
   Volume = {63},
   Pages = {65-74},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.neurobiolaging.2017.11.003},
   Abstract = {Age-related macular degeneration (AMD) is a retinal disease
             associated with significant vision loss among older adults.
             Previous large-scale behavioral studies indicate that people
             with AMD are at increased risk of cognitive deficits in
             language processing, particularly in verbal fluency tasks.
             The neural underpinnings of any relationship between AMD and
             higher cognitive functions, such as language processing,
             remain unclear. This study aims to address this issue using
             independent component analysis of spontaneous brain activity
             at rest. In 2 components associated with visual processing,
             we observed weaker functional connectivity in the primary
             visual cortex and lateral occipital cortex in AMD patients
             compared with healthy controls, indicating that AMD might
             lead to differences in the neural representation of vision.
             In a component related to language processing, we found that
             increasing connectivity within the right inferior frontal
             gyrus was associated with better verbal fluency performance
             across all older adults, and the verbal fluency effect was
             greater in AMD patients than controls in both right inferior
             frontal gyrus and right posterior temporal regions. As the
             behavioral performance of our patients is as good as that of
             controls, these findings suggest that preservation of verbal
             fluency performance in AMD patients might be achieved
             through higher contribution from right hemisphere regions in
             bilateral language networks. If that is the case, there may
             be an opportunity to promote cognitive resilience among
             seniors with AMD or other forms of late-life vision
             loss.},
   Doi = {10.1016/j.neurobiolaging.2017.11.003},
   Key = {fds331315}
}

@article{fds326350,
   Author = {Monge, ZA and Geib, BR and Siciliano, RE and Packard, LE and Tallman,
             CW and Madden, DJ},
   Title = {Functional modular architecture underlying attentional
             control in aging.},
   Journal = {NeuroImage},
   Volume = {155},
   Pages = {257-270},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2017.05.002},
   Abstract = {Previous research suggests that age-related differences in
             attention reflect the interaction of top-down and bottom-up
             processes, but the cognitive and neural mechanisms
             underlying this interaction remain an active area of
             research. Here, within a sample of community-dwelling adults
             19-78 years of age, we used diffusion reaction time (RT)
             modeling and multivariate functional connectivity to
             investigate the behavioral components and whole-brain
             functional networks, respectively, underlying bottom-up and
             top-down attentional processes during conjunction visual
             search. During functional MRI scanning, participants
             completed a conjunction visual search task in which each
             display contained one item that was larger than the other
             items (i.e., a size singleton) but was not informative
             regarding target identity. This design allowed us to examine
             in the RT components and functional network measures the
             influence of (a) additional bottom-up guidance when the
             target served as the size singleton, relative to when the
             distractor served as the size singleton (i.e., size
             singleton effect) and (b) top-down processes during target
             detection (i.e., target detection effect; target present vs.
             absent trials). We found that the size singleton effect
             (i.e., increased bottom-up guidance) was associated with RT
             components related to decision and nondecision processes,
             but these effects did not vary with age. Also, a modularity
             analysis revealed that frontoparietal module connectivity
             was important for both the size singleton and target
             detection effects, but this module became central to the
             networks through different mechanisms for each effect.
             Lastly, participants 42 years of age and older, in service
             of the target detection effect, relied more on
             between-frontoparietal module connections. Our results
             further elucidate mechanisms through which frontoparietal
             regions support attentional control and how these mechanisms
             vary in relation to adult age.},
   Doi = {10.1016/j.neuroimage.2017.05.002},
   Key = {fds326350}
}

@article{fds325978,
   Author = {Madden, DJ and Parks, EL and Tallman, CW and Boylan, MA and Hoagey, DA and Cocjin, SB and Packard, LE and Johnson, MA and Chou, Y-H and Potter, GG and Chen, N-K and Siciliano, RE and Monge, ZA and Honig, JA and Diaz,
             MT},
   Title = {Sources of disconnection in neurocognitive aging: cerebral
             white-matter integrity, resting-state functional
             connectivity, and white-matter hyperintensity
             volume.},
   Journal = {Neurobiology of Aging},
   Volume = {54},
   Pages = {199-213},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.neurobiolaging.2017.01.027},
   Abstract = {Age-related decline in fluid cognition can be characterized
             as a disconnection among specific brain structures, leading
             to a decline in functional efficiency. The potential sources
             of disconnection, however, are unclear. We investigated
             imaging measures of cerebral white-matter integrity,
             resting-state functional connectivity, and white-matter
             hyperintensity volume as mediators of the relation between
             age and fluid cognition, in 145 healthy, community-dwelling
             adults 19-79 years of age. At a general level of analysis,
             with a single composite measure of fluid cognition and
             single measures of each of the 3 imaging modalities, age
             exhibited an independent influence on the cognitive and
             imaging measures, and the imaging variables did not mediate
             the age-cognition relation. At a more specific level of
             analysis, resting-state functional connectivity of
             sensorimotor networks was a significant mediator of the
             age-related decline in executive function. These findings
             suggest that different levels of analysis lead to different
             models of neurocognitive disconnection, and that
             resting-state functional connectivity, in particular, may
             contribute to age-related decline in executive
             function.},
   Doi = {10.1016/j.neurobiolaging.2017.01.027},
   Key = {fds325978}
}

@article{fds325365,
   Author = {Cordero, DM and Towe, SL and Chen, N-K and Robertson, KR and Madden, DJ and Huettel, SA and Meade, CS},
   Title = {Cocaine dependence does not contribute substantially to
             white matter abnormalities in HIV infection.},
   Journal = {Journal of NeuroVirology},
   Volume = {23},
   Number = {3},
   Pages = {441-450},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s13365-017-0512-5},
   Abstract = {This study investigated the association of HIV infection and
             cocaine dependence with cerebral white matter integrity
             using diffusion tensor imaging (DTI). One hundred
             thirty-five participants stratified by HIV and cocaine
             status (26 HIV+/COC+, 37 HIV+/COC-, 37 HIV-/COC+, and 35
             HIV-/COC-) completed a comprehensive substance abuse
             assessment, neuropsychological testing, and MRI with DTI.
             Among HIV+ participants, all were receiving HIV care and 46%
             had an AIDS diagnosis. All COC+ participants were current
             users and met criteria for cocaine use disorder. We used
             tract-based spatial statistics (TBSS) to assess the relation
             of HIV and cocaine to fractional anisotropy (FA) and mean
             diffusivity (MD). In whole-brain analyses, HIV+ participants
             had significantly reduced FA and increased MD compared to
             HIV- participants. The relation of HIV and FA was widespread
             throughout the brain, whereas the HIV-related MD effects
             were restricted to the corpus callosum and thalamus. There
             were no significant cocaine or HIV-by-cocaine effects. These
             DTI metrics correlated significantly with duration of HIV
             disease, nadir CD4+ cell count, and AIDS diagnosis, as well
             as some measures of neuropsychological functioning. These
             results suggest that HIV is related to white matter
             integrity throughout the brain, and that HIV-related effects
             are more pronounced with increasing duration of infection
             and greater immune compromise. We found no evidence for
             independent effects of cocaine dependence on white matter
             integrity, and cocaine dependence did not appear to
             exacerbate the effects of HIV.},
   Doi = {10.1007/s13365-017-0512-5},
   Key = {fds325365}
}

@article{fds325785,
   Author = {Siciliano, RE and Madden, DJ and Tallman, CW and Boylan, MA and Kirste,
             I and Monge, ZA and Packard, LE and Potter, GG and Wang,
             L},
   Title = {Task difficulty modulates brain activation in the emotional
             oddball task.},
   Journal = {Brain Research},
   Volume = {1664},
   Pages = {74-86},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.brainres.2017.03.028},
   Abstract = {Previous functional magnetic resonance imaging (fMRI)
             studies have reported that task-irrelevant, emotionally
             salient events can disrupt target discrimination,
             particularly when attentional demands are low, while others
             demonstrate alterations in the distracting effects of
             emotion in behavior and neural activation in the context of
             attention-demanding tasks. We used fMRI, in conjunction with
             an emotional oddball task, at different levels of target
             discrimination difficulty, to investigate the effects of
             emotional distractors on the detection of subsequent
             targets. In addition, we distinguished different behavioral
             components of target detection representing decisional,
             nondecisional, and response criterion processes. Results
             indicated that increasing target discrimination difficulty
             led to increased time required for both the decisional and
             nondecisional components of the detection response, as well
             as to increased target-related neural activation in
             frontoparietal regions. The emotional distractors were
             associated with activation in ventral occipital and frontal
             regions and dorsal frontal regions, but this activation was
             attenuated with increased difficulty. Emotional distraction
             did not alter the behavioral measures of target detection,
             but did lead to increased target-related frontoparietal
             activation for targets following emotional images as
             compared to those following neutral images. This latter
             effect varied with target discrimination difficulty, with an
             increased influence of the emotional distractors on
             subsequent target-related frontoparietal activation in the
             more difficult discrimination condition. This influence of
             emotional distraction was in addition associated
             specifically with the decisional component of target
             detection. These findings indicate that emotion-cognition
             interactions, in the emotional oddball task, vary depending
             on the difficulty of the target discrimination and the
             associated limitations on processing resources.},
   Doi = {10.1016/j.brainres.2017.03.028},
   Key = {fds325785}
}

@article{fds323600,
   Author = {Madden, DJ and Parks, EL and Tallman, CW and Boylan, MA and Hoagey, DA and Cocjin, SB and Johnson, MA and Chou, Y-H and Potter, GG and Chen, N-K and Packard, LE and Siciliano, RE and Monge, ZA and Diaz,
             MT},
   Title = {Frontoparietal activation during visual conjunction search:
             Effects of bottom-up guidance and adult age.},
   Journal = {Human Brain Mapping},
   Volume = {38},
   Number = {4},
   Pages = {2128-2149},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1002/hbm.23509},
   Abstract = {We conducted functional magnetic resonance imaging (fMRI)
             with a visual search paradigm to test the hypothesis that
             aging is associated with increased frontoparietal
             involvement in both target detection and bottom-up
             attentional guidance (featural salience). Participants were
             68 healthy adults, distributed continuously across 19 to 78
             years of age. Frontoparietal regions of interest (ROIs) were
             defined from resting-state scans obtained prior to
             task-related fMRI. The search target was defined by a
             conjunction of color and orientation. Each display contained
             one item that was larger than the others (i.e., a size
             singleton) but was not informative regarding target
             identity. Analyses of search reaction time (RT) indicated
             that bottom-up attentional guidance from the size singleton
             (when coincident with the target) was relatively constant as
             a function of age. Frontoparietal fMRI activation related to
             target detection was constant as a function of age, as was
             the reduction in activation associated with salient targets.
             However, for individuals 35 years of age and older,
             engagement of the left frontal eye field (FEF) in bottom-up
             guidance was more prominent than for younger individuals.
             Further, the age-related differences in left FEF activation
             were a consequence of decreasing resting-state functional
             connectivity in visual sensory regions. These findings
             indicate that age-related compensatory effects may be
             expressed in the relation between activation and behavior,
             rather than in the magnitude of activation, and that
             relevant changes in the activation-RT relation may begin at
             a relatively early point in adulthood. Hum Brain Mapp
             38:2128-2149, 2017. © 2017 Wiley Periodicals,
             Inc.},
   Doi = {10.1002/hbm.23509},
   Key = {fds323600}
}

@article{fds323599,
   Author = {Chou, Y-H and Sundman, M and Whitson, HE and Gaur, P and Chu, M-L and Weingarten, CP and Madden, DJ and Wang, L and Kirste, I and Joliot, M and Diaz, MT and Li, Y-J and Song, AW and Chen, N-K},
   Title = {Maintenance and Representation of Mind Wandering during
             Resting-State fMRI.},
   Journal = {Scientific Reports},
   Volume = {7},
   Pages = {40722},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1038/srep40722},
   Abstract = {Major advances in resting-state functional magnetic
             resonance imaging (fMRI) techniques in the last two decades
             have provided a tool to better understand the functional
             organization of the brain both in health and illness.
             Despite such developments, characterizing regulation and
             cerebral representation of mind wandering, which occurs
             unavoidably during resting-state fMRI scans and may induce
             variability of the acquired data, remains a work in
             progress. Here, we demonstrate that a decrease or decoupling
             in functional connectivity involving the caudate nucleus,
             insula, medial prefrontal cortex and other domain-specific
             regions was associated with more sustained mind wandering in
             particular thought domains during resting-state fMRI.
             Importantly, our findings suggest that temporal and
             between-subject variations in functional connectivity of
             above-mentioned regions might be linked with the continuity
             of mind wandering. Our study not only provides a preliminary
             framework for characterizing the maintenance and cerebral
             representation of different types of mind wandering, but
             also highlights the importance of taking mind wandering into
             consideration when studying brain organization with
             resting-state fMRI in the future.},
   Doi = {10.1038/srep40722},
   Key = {fds323599}
}


%% March, John S.   
@article{fds138619,
   Title = {March, J., Parker, J., Sullivan, K., Stallings, P., Conners,
             C. (in press), The Multidimensional Anxiety Scale for
             Children (MASC): Factor structure, reliability and validity.
             J Am Acad Child Adolesc Psychiatry},
   Key = {fds138619}
}

@article{fds138635,
   Title = {March, J., Amaya-Jackson, L., Costanzo, P., Terry, R. (in
             press), Post-traumatic stress in children and adolescents
             after an industrial fire. Journal of the American Academy of
             Child and Adolescent Psychiatry},
   Key = {fds138635}
}


%% Marsan, Samuel   
@article{fds333602,
   Author = {Campbell, K and Carpenter, KL and Hashemi, J and Espinosa, S and Marsan,
             S and Borg, JS and Chang, Z and Qiu, Q and Vermeer, S and Adler, E and Tepper,
             M and Egger, HL and Baker, JP and Sapiro, G and Dawson,
             G},
   Title = {Computer vision analysis captures atypical attention in
             toddlers with autism.},
   Journal = {Autism},
   Pages = {1362361318766247},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/1362361318766247},
   Abstract = {To demonstrate the capability of computer vision analysis to
             detect atypical orienting and attention behaviors in
             toddlers with autism spectrum disorder. One hundered and
             four toddlers of 16-31 months old (mean = 22)
             participated in this study. Twenty-two of the toddlers had
             autism spectrum disorder and 82 had typical development or
             developmental delay. Toddlers watched video stimuli on a
             tablet while the built-in camera recorded their head
             movement. Computer vision analysis measured participants'
             attention and orienting in response to name calls.
             Reliability of the computer vision analysis algorithm was
             tested against a human rater. Differences in behavior were
             analyzed between the autism spectrum disorder group and the
             comparison group. Reliability between computer vision
             analysis and human coding for orienting to name was
             excellent (intra-class coefficient 0.84, 95% confidence
             interval 0.67-0.91). Only 8% of toddlers with autism
             spectrum disorder oriented to name calling on >1 trial,
             compared to 63% of toddlers in the comparison group
             (p = 0.002). Mean latency to orient was significantly
             longer for toddlers with autism spectrum disorder (2.02 vs
             1.06 s, p = 0.04). Sensitivity for autism spectrum
             disorder of atypical orienting was 96% and specificity was
             38%. Older toddlers with autism spectrum disorder showed
             less attention to the videos overall (p = 0.03).
             Automated coding offers a reliable, quantitative method for
             detecting atypical social orienting and reduced sustained
             attention in toddlers with autism spectrum
             disorder.},
   Doi = {10.1177/1362361318766247},
   Key = {fds333602}
}

@article{fds333603,
   Author = {Zucker, N and Mauro, C and Craske, M and Wagner, HR and Datta, N and Hopkins, H and Caldwell, K and Kiridly, A and Marsan, S and Maslow, G and Mayer, E and Egger, H},
   Title = {Acceptance-based interoceptive exposure for young children
             with functional abdominal pain.},
   Journal = {Behaviour Research and Therapy},
   Volume = {97},
   Pages = {200-212},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.brat.2017.07.009},
   Abstract = {Functional abdominal pain (FAP) is a common childhood
             somatic complaint that contributes to impairment in daily
             functioning (e.g., school absences) and increases risk for
             chronic pain and psychiatric illness. Cognitive behavioral
             treatments for FAP target primarily older children
             (9 + years) and employ strategies to reduce a focus on
             pain. The experience of pain may be an opportunity to teach
             viscerally hypersensitive children to interpret the function
             of a variety of bodily signals (including those of hunger,
             emotions) thereby reducing fear of bodily sensations and
             facilitating emotion awareness and self-regulation. We
             designed and tested an interoceptive exposure treatment for
             younger children (5-9 years) with FAP. Assessments included
             diagnostic interviews, 14 days of daily pain monitoring, and
             questionnaires. Treatment involved 10 weekly appointments.
             Using cartoon characters to represent bodily sensations
             (e.g., Gassy Gus), children were trained to be "FBI agents"
             - Feeling and Body Investigators - who investigated
             sensations through exercises that provoked somatic
             experience. 24 parent-child dyads are reported. Pain
             (experience, distress, and interference) and negative affect
             demonstrated clinically meaningful and statistically
             significant change with effect sizes ranging from 0.48 to 71
             for pain and from 0.38 to 0.61 for pain distress, total
             pain: X2 (1, n = 24) = 13.14, p < 0.0003. An
             intervention that helps children adopt a curious stance and
             focus on somatic symptoms reduces pain and may help lessen
             somatic fear generally.NCT02075437.},
   Doi = {10.1016/j.brat.2017.07.009},
   Key = {fds333603}
}


%% Marsh, Elizabeth J.   
@article{fds333724,
   Author = {Wang, W-C and Brashier, NM and Wing, EA and Marsh, EJ and Cabeza,
             R},
   Title = {Knowledge supports memory retrieval through familiarity, not
             recollection.},
   Journal = {Neuropsychologia},
   Volume = {113},
   Pages = {14-21},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2018.01.019},
   Abstract = {Semantic memory, or general knowledge of the world, guides
             learning and supports the formation and retrieval of new
             episodic memories. Behavioral evidence suggests that this
             knowledge effect is supported by recollection-a more
             controlled form of memory retrieval generally accompanied by
             contextual details-to a greater degree than familiarity-a
             more automatic form of memory retrieval generally absent of
             contextual details. In the current study, we used functional
             magnetic resonance imaging (fMRI) to investigate the role
             that regions associated with recollection and familiarity
             play in retrieving recent instances of known (e.g., The
             Summer Olympic Games are held four years apart) and unknown
             (e.g., A flaky deposit found in port bottles is beeswing)
             statements. Our results revealed a surprising pattern:
             Episodic retrieval of known statements recruited regions
             associated with familiarity, but not recollection. Instead,
             retrieval of unknown statements recruited regions associated
             with recollection. These data, in combination with quicker
             reaction times for the retrieval of known than unknown
             statements, suggest that known statements can be
             successfully retrieved on the basis of familiarity, whereas
             unknown statements were retrieved on the basis of
             recollection. Our results provide insight into how knowledge
             influences episodic retrieval and demonstrate the role of
             neuroimaging in providing insights into cognitive processes
             in the absence of explicit behavioral responses.},
   Doi = {10.1016/j.neuropsychologia.2018.01.019},
   Key = {fds333724}
}

@article{fds332045,
   Author = {Butler, AC and Black-Maier, AC and Raley, ND and Marsh,
             EJ},
   Title = {Retrieving and applying knowledge to different examples
             promotes transfer of learning.},
   Journal = {Journal of Experimental Psychology: Applied},
   Volume = {23},
   Number = {4},
   Pages = {433-446},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1037/xap0000142},
   Abstract = {Introducing variability during learning often facilitates
             transfer to new contexts (i.e., generalization). The goal of
             the present study was to explore the concept of variability
             in an area of research where its effects have received
             little attention: learning through retrieval practice. In
             four experiments, we investigated whether retrieval practice
             with different examples of a concept promotes greater
             transfer than repeated retrieval practice with the same
             example. Participants watched video clips from a lecture
             about geological science and answered application questions
             about concepts: either the same question three times or
             three different questions. Experiments 3 and 4 also included
             conditions that involved repeatedly studying the information
             in the application questions (either the same example or
             three different examples). Two days later, participants took
             a final test with new application questions. All four
             experiments showed that variability during retrieval
             practice produced superior transfer of knowledge to new
             examples. Experiments 3 and 4 also showed a testing effect
             and a benefit from studying different examples. Overall,
             these findings suggest that repeatedly retrieving and
             applying knowledge to different examples is a powerful
             method for acquiring knowledge that will transfer to a
             variety of new contexts. (PsycINFO Database
             Record},
   Doi = {10.1037/xap0000142},
   Key = {fds332045}
}

@article{fds331410,
   Author = {Marsh, EJ and Yang, BW},
   Title = {A Call to Think Broadly about Information
             Literacy},
   Journal = {Journal of Applied Research in Memory and
             Cognition},
   Volume = {6},
   Number = {4},
   Pages = {401-404},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.jarmac.2017.09.012},
   Doi = {10.1016/j.jarmac.2017.09.012},
   Key = {fds331410}
}

@article{fds332982,
   Author = {Marsh, EJ},
   Title = {Family Matters: Measuring Impact Through One's Academic
             Descendants.},
   Journal = {Perspectives on Psychological Science},
   Volume = {12},
   Number = {6},
   Pages = {1130-1132},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1177/1745691617719759},
   Abstract = {Scientific contributions take many forms, not all of which
             result in fame or are captured in traditional metrics of
             success (e.g., h factor). My focus is on one of the most
             lasting and important contributions a scientist can make:
             training scientists who go on to train scientists, who in
             turn train more scientists, etc. Academic genealogies
             provide many examples of scientists whose names might not be
             recognizable today but who trained psychologists that went
             on to publish very influential work. Of course success
             results from a combination of many factors (including but
             not limited to the student's abilities and motivation, luck,
             institutional resources, mentoring, etc.), but the field
             should find more ways to acknowledge the role that mentoring
             does play.},
   Doi = {10.1177/1745691617719759},
   Key = {fds332982}
}

@misc{fds333503,
   Author = {Marsh, EJ and Arnold, KM},
   Title = {Retelling experiences and writing essays how: Storytelling
             reflects and changes memory},
   Pages = {137-155},
   Booktitle = {Representations in Mind and World: Essays Inspired by
             Barbara Tversky},
   Year = {2017},
   Month = {July},
   ISBN = {9781315169781},
   url = {http://dx.doi.org/10.4324/9781315169781},
   Doi = {10.4324/9781315169781},
   Key = {fds333503}
}

@article{fds326719,
   Author = {Brashier, NM and Umanath, S and Cabeza, R and Marsh,
             EJ},
   Title = {Competing cues: Older adults rely on knowledge in the face
             of fluency.},
   Journal = {Psychology and Aging},
   Volume = {32},
   Number = {4},
   Pages = {331-337},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1037/pag0000156},
   Abstract = {Consumers regularly encounter repeated false claims in
             political and marketing campaigns, but very little empirical
             work addresses their impact among older adults. Repeated
             statements feel easier to process, and thus more truthful,
             than new ones (i.e., illusory truth). When judging truth,
             older adults' accumulated general knowledge may offset this
             perception of fluency. In two experiments, participants read
             statements that contradicted information stored in memory; a
             post-experimental knowledge check confirmed what individual
             participants knew. Unlike young adults, older adults
             exhibited illusory truth only when they lacked knowledge
             about claims. This interaction between knowledge and fluency
             extends dual-process theories of aging. (PsycINFO Database
             Record},
   Doi = {10.1037/pag0000156},
   Key = {fds326719}
}

@article{fds326720,
   Author = {Arnold, KM and Umanath, S and Thio, K and Reilly, WB and McDaniel, MA and Marsh, EJ},
   Title = {Understanding the cognitive processes involved in writing to
             learn.},
   Journal = {Journal of Experimental Psychology: Applied},
   Volume = {23},
   Number = {2},
   Pages = {115-127},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1037/xap0000119},
   Abstract = {Writing is often used as a tool for learning. However,
             empirical support for the benefits of writing-to-learn is
             mixed, likely because the literature conflates diverse
             activities (e.g., summaries, term papers) under the single
             umbrella of writing-to-learn. Following recent trends in the
             writing-to-learn literature, the authors focus on the
             underlying cognitive processes. They draw on the largely
             independent writing-to-learn and cognitive psychology
             learning literatures to identify important cognitive
             processes. The current experiment examines learning from 3
             writing tasks (and 1 nonwriting control), with an emphasis
             on whether or not the tasks engaged retrieval. Tasks that
             engaged retrieval (essay writing and free recall) led to
             better final test performance than those that did not (note
             taking and highlighting). Individual differences in
             structure building (the ability to construct mental
             representations of narratives; Gernsbacher, Varner, & Faust,
             1990) modified this effect; skilled structure builders
             benefited more from essay writing and free recall than did
             less skilled structure builders. Further, more essay-like
             responses led to better performance, implicating the
             importance of additional cognitive processes such as
             reorganization and elaboration. The results highlight how
             both task instructions and individual differences affect the
             cognitive processes involved when writing-to-learn, with
             consequences for the effectiveness of the learning strategy.
             (PsycINFO Database Record},
   Doi = {10.1037/xap0000119},
   Key = {fds326720}
}

@article{fds322500,
   Author = {Cantor, AD and Marsh, EJ},
   Title = {Expertise effects in the Moses illusion: detecting
             contradictions with stored knowledge.},
   Journal = {Memory},
   Volume = {25},
   Number = {2},
   Pages = {220-230},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1080/09658211.2016.1152377},
   Abstract = {People frequently miss contradictions with stored knowledge;
             for example, readers often fail to notice any problem with a
             reference to the Atlantic as the largest ocean. Critically,
             such effects occur even though participants later
             demonstrate knowing the Pacific is the largest ocean (the
             Moses Illusion) [Erickson, T. D., & Mattson,
             M. E. (1981). From words to meaning: A semantic illusion.
             Journal of Verbal Learning & Verbal Behavior, 20, 540-551].
             We investigated whether such oversights disappear when
             erroneous references contradict information in one's expert
             domain, material which likely has been encountered many
             times and is particularly well-known. Biology and history
             graduate students monitored for errors while answering
             biology and history questions containing erroneous
             presuppositions ("In what US state were the forty-niners
             searching for oil?"). Expertise helped: participants were
             less susceptible to the illusion and less likely to later
             reproduce errors in their expert domain. However, expertise
             did not eliminate the illusion, even when errors were bolded
             and underlined, meaning that it was unlikely that people
             simply skipped over errors. The results support claims that
             people often use heuristics to judge truth, as opposed to
             directly retrieving information from memory, likely because
             such heuristics are adaptive and often lead to the correct
             answer. Even experts sometimes use such shortcuts,
             suggesting that overlearned and accessible knowledge does
             not guarantee retrieval of that information.},
   Doi = {10.1080/09658211.2016.1152377},
   Key = {fds322500}
}


%% Mauro, Christian F.   
@article{fds333644,
   Author = {Allen, TM and Wren, AA and Anderson, LM and Sabholk, A and Mauro,
             CF},
   Title = {Group CBT-yoga protocol targeting pain-related and
             internalizing symptoms in youth},
   Journal = {Clinical Practice in Pediatric Psychology},
   Volume = {6},
   Number = {1},
   Pages = {7-18},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1037/cpp0000206},
   Abstract = {© 2018 American Psychological Association. There is a high
             prevalence of pain-related somatic symptoms among children
             with internalizing disorders. Despite the documented
             comorbidity between somatic and internalizing symptoms in
             youth, there are limited empirically supported interventions
             that simultaneously target these issues. In light of this,
             the current pilot study investigated the feasibility and
             preliminary efficacy of a novel 12-week manualized
             group-based CBT-yoga protocol, which aimed to target
             co-occurring physical and internalizing symptoms in a sample
             of youth. Twenty-eight children between the ages of 10 and
             12 years old who had clinically relevant internalizing
             symptoms and pain-related somatic symptoms were enrolled in
             the 12-week intervention protocol. Attendance rates and
             parent and child responses on social validity scales
             provided measures of feasibility. Preliminary efficacy was
             examined through a series of paired sample t tests,
             measuring changes in internalizing and somatic symptoms, and
             pain-related coping following the intervention. Results
             suggest strong levels of childand parent-rated feasibility.
             Preliminary data also reveal improvements in internalizing
             and somatic symptoms following the intervention, in addition
             to improvements in pain-related coping. These findings
             provide initial support for the feasibility and acceptance
             of an integrative group-based intervention combining
             cognitive-behavioral strategies with yogic
             practices.},
   Doi = {10.1037/cpp0000206},
   Key = {fds333644}
}

@article{fds328294,
   Author = {Zucker, N and Mauro, C and Craske, M and Wagner, HR and Datta, N and Hopkins, H and Caldwell, K and Kiridly, A and Marsan, S and Maslow, G and Mayer, E and Egger, H},
   Title = {Acceptance-based interoceptive exposure for young children
             with functional abdominal pain.},
   Journal = {Behaviour Research and Therapy},
   Volume = {97},
   Pages = {200-212},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.brat.2017.07.009},
   Abstract = {Functional abdominal pain (FAP) is a common childhood
             somatic complaint that contributes to impairment in daily
             functioning (e.g., school absences) and increases risk for
             chronic pain and psychiatric illness. Cognitive behavioral
             treatments for FAP target primarily older children
             (9 + years) and employ strategies to reduce a focus on
             pain. The experience of pain may be an opportunity to teach
             viscerally hypersensitive children to interpret the function
             of a variety of bodily signals (including those of hunger,
             emotions) thereby reducing fear of bodily sensations and
             facilitating emotion awareness and self-regulation. We
             designed and tested an interoceptive exposure treatment for
             younger children (5-9 years) with FAP. Assessments included
             diagnostic interviews, 14 days of daily pain monitoring, and
             questionnaires. Treatment involved 10 weekly appointments.
             Using cartoon characters to represent bodily sensations
             (e.g., Gassy Gus), children were trained to be "FBI agents"
             - Feeling and Body Investigators - who investigated
             sensations through exercises that provoked somatic
             experience. 24 parent-child dyads are reported. Pain
             (experience, distress, and interference) and negative affect
             demonstrated clinically meaningful and statistically
             significant change with effect sizes ranging from 0.48 to 71
             for pain and from 0.38 to 0.61 for pain distress, total
             pain: X2 (1, n = 24) = 13.14, p < 0.0003. An
             intervention that helps children adopt a curious stance and
             focus on somatic symptoms reduces pain and may help lessen
             somatic fear generally.NCT02075437.},
   Doi = {10.1016/j.brat.2017.07.009},
   Key = {fds328294}
}


%% Mazuka, Reiko   
@article{fds329036,
   Author = {Akimoto, Y and Takahashi, H and Gunji, A and Kaneko, Y and Asano, M and Matsuo, J and Ota, M and Kunugi, H and Hanakawa, T and Mazuka, R and Kamio,
             Y},
   Title = {Alpha band event-related desynchronization underlying social
             situational context processing during irony comprehension: A
             magnetoencephalography source localization
             study.},
   Journal = {Brain and Language},
   Volume = {175},
   Pages = {42-46},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.bandl.2017.09.002},
   Abstract = {Irony comprehension requires integration of social
             contextual information. Previous studies have investigated
             temporal aspects of irony processing and its neural
             substrates using psychological/electroencephalogram or
             functional magnetic resonance imaging methods, but have not
             clarified the temporospatial neural mechanisms of irony
             comprehension. Therefore, we used magnetoencephalography to
             investigate the neural generators of alpha-band (8-13Hz)
             event-related desynchronization (ERD) occurring from 600 to
             900ms following the onset of a critical sentence at which
             social situational contexts activated ironic representation.
             We found that the right anterior temporal lobe, which is
             involved in processing social knowledge and evaluating
             others' intentions, exhibited stronger alpha ERD following
             an ironic statement than following a literal statement. We
             also found that alpha power in the left anterior temporal
             lobe correlated with the participants' communication
             abilities. These results elucidate the temporospatial neural
             mechanisms of language comprehension in social contexts,
             including non-literal processing.},
   Doi = {10.1016/j.bandl.2017.09.002},
   Key = {fds329036}
}

@article{fds325710,
   Author = {Hirose, Y and Mazuka, R},
   Title = {Exploiting Pitch Accent Information in Compound Processing:
             A Comparison between Adults and 6- to 7-Year-Old
             Children},
   Journal = {Language Learning and Development},
   Volume = {13},
   Number = {4},
   Pages = {375-394},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1080/15475441.2017.1292141},
   Doi = {10.1080/15475441.2017.1292141},
   Key = {fds325710}
}

@article{fds326609,
   Author = {Miyazawa, K and Shinya, T and Martin, A and Kikuchi, H and Mazuka,
             R},
   Title = {Vowels in infant-directed speech: More breathy and more
             variable, but not clearer.},
   Journal = {Cognition},
   Volume = {166},
   Pages = {84-93},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.cognition.2017.05.003},
   Abstract = {Infant-directed speech (IDS) is known to differ from
             adult-directed speech (ADS) in a number of ways, and it has
             often been argued that some of these IDS properties
             facilitate infants' acquisition of language. An influential
             study in support of this view is Kuhl et al. (1997), which
             found that vowels in IDS are produced with expanded first
             and second formants (F1/F2) on average, indicating that the
             vowels are acoustically further apart in IDS than in ADS.
             These results have been interpreted to mean that the way
             vowels are produced in IDS makes infants' task of learning
             vowel categories easier. The present paper revisits this
             interpretation by means of a thorough analysis of IDS vowels
             using a large-scale corpus of Japanese natural utterances.
             We will show that the expansion of F1/F2 values does occur
             in spontaneous IDS even when the vowels' prosodic position,
             lexical pitch accent, and lexical bias are accounted for.
             When IDS vowels are compared to carefully read speech (CS)
             by the same mothers, however, larger variability among IDS
             vowel tokens means that the acoustic distances among vowels
             are farther apart only in CS, but not in IDS when compared
             to ADS. Finally, we will show that IDS vowels are
             significantly more breathy than ADS or CS vowels. Taken
             together, our results demonstrate that even though expansion
             of formant values occurs in spontaneous IDS, this expansion
             cannot be interpreted as an indication that the acoustic
             distances among vowels are farther apart, as is the case in
             CS. Instead, we found that IDS vowels are characterized by
             breathy voice, which has been associated with the
             communication of emotional affect.},
   Doi = {10.1016/j.cognition.2017.05.003},
   Key = {fds326609}
}

@article{fds332175,
   Author = {Mazuka, R and Bernard, M and Cristia, A and Dupoux, E and Ludusan,
             B},
   Title = {The role of prosody and speech register in word
             segmentation: A computational modelling perspective},
   Journal = {ACL 2017 - 55th Annual Meeting of the Association for
             Computational Linguistics, Proceedings of the Conference
             (Long Papers)},
   Volume = {2},
   Pages = {178-183},
   Year = {2017},
   Month = {January},
   ISBN = {9781945626760},
   url = {http://dx.doi.org/10.18653/v1/P17-2028},
   Abstract = {© 2017 Association for Computational Linguistics. This
             study explores the role of speech register and prosody for
             the task of word segmentation. Since these two factors are
             thought to play an important role in early language
             acquisition, we aim to quantify their contribution for this
             task. We study a Japanese corpus containing both infant- and
             adult-directed speech and we apply four different word
             segmentation models, with and without knowledge of prosodic
             boundaries. The results showed that the difference between
             registers is smaller than previously reported and that
             prosodic boundary information helps more adult- than
             infant-directed speech.},
   Doi = {10.18653/v1/P17-2028},
   Key = {fds332175}
}

@article{fds332177,
   Author = {Ota, M and Yamane, N and Mazuka, R},
   Title = {The Effects of Lexical Pitch Accent on Infant Word
             Recognition in Japanese.},
   Journal = {Frontiers in Psychology},
   Volume = {8},
   Pages = {2354},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fpsyg.2017.02354},
   Abstract = {Learners of lexical tone languages (e.g., Mandarin) develop
             sensitivity to tonal contrasts and recognize pitch-matched,
             but not pitch-mismatched, familiar words by 11 months.
             Learners of non-tone languages (e.g., English) also show a
             tendency to treat pitch patterns as lexically contrastive up
             to about 18 months. In this study, we examined if this
             early-developing capacity to lexically encode pitch
             variations enables infants to acquire a pitch accent system,
             in which pitch-based lexical contrasts are obscured by the
             interaction of lexical and non-lexical (i.e., intonational)
             features. Eighteen 17-month-olds learning Tokyo Japanese
             were tested on their recognition of familiar words with the
             expected pitch or the lexically opposite pitch pattern. In
             early trials, infants were faster in shifting their eyegaze
             from the distractor object to the target object than in
             shifting from the target to distractor in the pitch-matched
             condition. In later trials, however, infants showed faster
             distractor-to-target than target-to-distractor shifts in
             both the pitch-matched and pitch-mismatched conditions. We
             interpret these results to mean that, in a pitch-accent
             system, the ability to use pitch variations to recognize
             words is still in a nascent state at 17 months.},
   Doi = {10.3389/fpsyg.2017.02354},
   Key = {fds332177}
}

@article{fds321661,
   Author = {Hayashi, A and Mazuka, R},
   Title = {Emergence of Japanese infants' prosodic preferences in
             infant-directed vocabulary.},
   Journal = {Developmental Psychology},
   Volume = {53},
   Number = {1},
   Pages = {28-37},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1037/dev0000259},
   Abstract = {The article examines the role of infant-directed vocabulary
             (IDV) in infants language acquisition, specifically
             addressing the question of whether IDV forms that are not
             prominent in adult language may nonetheless be useful to the
             process of acquisition. Japanese IDV offers a good test
             case, as IDV characteristically takes a bisyllabic
             H(eavy)-L(ight) form that is rare in adult speech. In 5
             experiments using the Headturn Preference Procedure (HPP),
             8- to 10-month-old Japanese infants, but not 4- to
             6-month-olds, were found to show a preference for bisyllabic
             H-L words over other types of words. These results
             demonstrate (a) that infants may develop a preference for a
             dominant prosodic form based on infant-directed speech, even
             when it is not a prominent characteristic of adult language;
             and perhaps more importantly, and (b) that infant-directed
             speech may provide a boost for a feature that could be
             useful for infants' acquisition of language even when it not
             prominent in adult language. (PsycINFO Database
             Record},
   Doi = {10.1037/dev0000259},
   Key = {fds321661}
}

@article{fds332176,
   Author = {Sugiura, L and Toyota, T and Matsuba-Kurita, H and Iwayama, Y and Mazuka, R and Yoshikawa, T and Hagiwara, H},
   Title = {Age-Dependent Effects of Catechol-O-Methyltransferase (COMT)
             Gene Val158Met Polymorphism on Language Function in
             Developing Children.},
   Journal = {Cerebral Cortex},
   Volume = {27},
   Number = {1},
   Pages = {104-116},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1093/cercor/bhw371},
   Abstract = {The genetic basis controlling language development remains
             elusive. Previous studies of the catechol-O-methyltransferase
             (COMT) Val158Met genotype and cognition have focused on
             prefrontally guided executive functions involving dopamine.
             However, COMT may further influence posterior cortical
             regions implicated in language perception. We investigated
             whether COMT influences language ability and cortical
             language processing involving the posterior language regions
             in 246 children aged 6-10 years. We assessed language
             ability using a language test and cortical responses
             recorded during language processing using a word repetition
             task and functional near-infrared spectroscopy. The COMT
             genotype had significant effects on language performance and
             processing. Importantly, Met carriers outperformed Val
             homozygotes in language ability during the early elementary
             school years (6-8 years), whereas Val homozygotes exhibited
             significant language development during the later elementary
             school years. Both genotype groups exhibited equal language
             performance at approximately 10 years of age. Val
             homozygotes exhibited significantly less cortical activation
             compared with Met carriers during word processing,
             particularly at older ages. These findings regarding
             dopamine transmission efficacy may be explained by a
             hypothetical inverted U-shaped curve. Our findings indicate
             that the effects of the COMT genotype on language ability
             and cortical language processing may change in a narrow age
             window of 6-10 years.},
   Doi = {10.1093/cercor/bhw371},
   Key = {fds332176}
}


%% Meade, Christina S.   
@article{fds327328,
   Author = {Meade, CS and Addicott, M and Hobkirk, AL and Towe, SL and Chen, N-K and Sridharan, S and Huettel, SA},
   Title = {Cocaine and HIV are independently associated with neural
             activation in response to gain and loss valuation during
             economic risky choice.},
   Journal = {Addiction Biology},
   Volume = {23},
   Number = {2},
   Pages = {796-809},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1111/adb.12529},
   Abstract = {Stimulant abuse is disproportionately common in HIV-positive
             persons. Both HIV and stimulants are independently
             associated with deficits in reward-based decision making,
             but their interactive and/or additive effects are poorly
             understood despite their prevalent co-morbidity. Here, we
             examined the effects of cocaine dependence and HIV infection
             in 69 adults who underwent functional magnetic resonance
             imaging while completing an economic loss aversion task. We
             identified two neural networks that correlated with the
             evaluation of the favorable characteristics of the gamble
             (i.e. higher gains/lower losses: ventromedial prefrontal
             cortex, anterior cingulate, anterior and posterior precuneus
             and visual cortex) versus unfavorable characteristics of the
             gamble (i.e. lower gains/higher losses: dorsal prefrontal,
             lateral orbitofrontal, posterior parietal cortex, anterior
             insula and dorsal caudate). Behaviorally, cocaine and HIV
             had additive effects on loss aversion scores, with
             HIV-positive cocaine users being the least loss averse.
             Cocaine users had greater activation in brain regions that
             tracked the favorability of gamble characteristics (i.e.
             increased activation to gains, but decreased activation to
             losses). In contrast, HIV infection was independently
             associated with lesser activation in regions that tracked
             the unfavorability of gamble characteristics. These results
             suggest that cocaine is associated with an overactive
             reward-seeking system, while HIV is associated with an
             underactive cognitive control system. Together, these
             alterations may leave HIV-positive cocaine users
             particularly vulnerable to making unfavorable decisions when
             outcomes are uncertain.},
   Doi = {10.1111/adb.12529},
   Key = {fds327328}
}

@article{fds328929,
   Author = {Skalski, LM and Towe, SL and Sikkema, KJ and Meade,
             CS},
   Title = {Memory Impairment in HIV-Infected Individuals with Early and
             Late Initiation of Regular Marijuana Use.},
   Journal = {AIDS and Behavior},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1007/s10461-017-1898-z},
   Abstract = {Marijuana use is disproportionately prevalent among
             HIV-infected individuals. The strongest neurocognitive
             effect of marijuana use is impairment in the domain of
             memory. Memory impairment is also high among HIV-infected
             persons. The present study examined 69 HIV-infected
             individuals who were stratified by age of regular marijuana
             initiation to investigate how marijuana use impacts
             neurocognitive functioning. A comprehensive battery assessed
             substance use and neurocognitive functioning. Findings
             indicated early onset marijuana users (regular use prior to
             age 18), compared to non-marijuana users and late onset
             marijuana users (regular use at age 18 or later), were over
             8 times more likely to have learning impairment and nearly 4
             times more likely to have memory impairment. A similar
             pattern of early onset marijuana users performing worse in
             learning emerged when examining domain deficit scores. The
             potential for early onset of regular marijuana use to
             exacerbate already high levels of memory impairment among
             HIV-infected persons has important clinical implications,
             including increased potential for medication non-adherence
             and difficulty with independent living.},
   Doi = {10.1007/s10461-017-1898-z},
   Key = {fds328929}
}

@article{fds327327,
   Author = {Meade, CS and Hobkirk, AL and Towe, SL and Chen, N-K and Bell, RP and Huettel, SA},
   Title = {Cocaine dependence modulates the effect of HIV infection on
             brain activation during intertemporal decision
             making.},
   Journal = {Drug and Alcohol Dependence},
   Volume = {178},
   Pages = {443-451},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.drugalcdep.2017.05.043},
   Abstract = {Both HIV infection and chronic cocaine use alter the neural
             circuitry of decision making, but the interactive effects of
             these commonly comorbid conditions have not been adequately
             examined. This study tested how cocaine moderates
             HIV-related neural activation during an intertemporal
             decision-making task.The sample included 73 participants who
             differed on cocaine and HIV status (18 COC+/HIV+, 19
             COC+/HIV-, 19 COC-/HIV+, 17 COC-/HIV-). Participants made
             choices between smaller, sooner and larger, delayed rewards
             while undergoing functional MRI. Choices varied in
             difficulty based on subjective value: hard (equivalently
             valued), easy (disparately valued), and control choices. A
             mixed-effects model controlling for education and smoking
             identified main and interactive effects of HIV and COC
             during hard relative to easy choices (difficulty
             contrast).COC+ status was associated with lower activation
             in bilateral frontal gyri and right insular and posterior
             parietal cortices. HIV+ status was associated with higher
             activation in the visual cortex, but lower activation in
             bilateral prefrontal cortices and cerebellum and left
             posterior parietal cortex. COC moderated the effects of HIV
             in several clusters centered in the bilateral prefrontal
             cortices and cerebellum. In post-hoc analyses, there were
             significant effects of HIV status on activation for COC+,
             but not COC-, participants; interaction effects remained
             after controlling for polysubstance use.Cocaine use may
             diminish the compensatory neural activation often seen among
             HIV+ samples during decision making. Our results highlight
             the importance of examining the neuropsychiatric effects of
             comorbid medical conditions to identify potential neural
             targets for cognitive remediation interventions.},
   Doi = {10.1016/j.drugalcdep.2017.05.043},
   Key = {fds327327}
}

@article{fds322766,
   Author = {Berg, MK and Hobkirk, AL and Joska, JA and Meade,
             CS},
   Title = {The role of substance use coping in the relation between
             childhood sexual abuse and depression among methamphetamine
             users in South Africa.},
   Journal = {Psychological Trauma: Theory, Research, Practice, and
             Policy},
   Volume = {9},
   Number = {4},
   Pages = {493-499},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1037/tra0000207},
   Abstract = {Childhood sexual abuse (CSA) is a critical global health
             issue associated with poor psychosocial outcomes.
             Individuals with CSA histories are at risk for drug use,
             which is a growing problem in the Western Cape of South
             Africa. The present study of methamphetamine users in this
             region examined whether substance use coping, a contextually
             relevant type of avoidance-based coping, mediates the
             relation between CSA and depressive symptoms.Participants
             included 161 men and 108 women seeking treatment for
             methamphetamine use. Participants completed a
             computer-assisted survey and a face-to-face interview with
             clinic staff to evaluate history of CSA, current substance
             use severity and coping, and current depressive
             symptoms.Nearly a third of participants reported a history
             of CSA, and the average methamphetamine use severity score
             exceeded the threshold of high risk. A history of CSA was
             significantly associated with higher substance use coping
             and more depression symptoms. Substance use coping was a
             significant mediator of the association between CSA and
             depression symptoms.In this study of high-risk
             methamphetamine users, substance use coping emerged as a
             common means of managing stress, especially for those with a
             history of CSA, which was further linked to depressive
             symptoms. These findings underscore the potential benefit of
             integrating coping interventions and mental health treatment
             into substance abuse treatment programs, particularly for
             those with a history of childhood abuse and violence.
             (PsycINFO Database Record},
   Doi = {10.1037/tra0000207},
   Key = {fds322766}
}

@article{fds327119,
   Author = {Towe, SL and Patel, P and Meade, CS},
   Title = {The Acceptability and Potential Utility of Cognitive
             Training to Improve Working Memory in Persons Living With
             HIV: A Preliminary Randomized Trial.},
   Journal = {Journal of the Association of Nurses in AIDS
             Care},
   Volume = {28},
   Number = {4},
   Pages = {633-643},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.jana.2017.03.007},
   Abstract = {HIV-associated neurocognitive impairments that impact daily
             function persist in the era of effective antiretroviral
             therapy. Cognitive training, a promising low-cost
             intervention, has been shown to improve neurocognitive
             functioning in some clinical populations. We tested the
             feasibility, acceptability, and preliminary effects of
             computerized cognitive training to improve working memory in
             persons living with HIV infection (PLWH) and working memory
             impairment. In this randomized clinical trial, we assigned
             21 adult PLWH to either an experimental cognitive training
             intervention or an attention-matched control training
             intervention. Participants completed 12 training sessions
             across 10 weeks with assessments at baseline and
             post-training. Session attendance was excellent and
             participants rated the program positively. Participants in
             the experimental arm demonstrated improved working memory
             function over time; participants in the control arm showed
             no change. Our results suggest that cognitive training may
             be a promising intervention for working memory impairment in
             PLWH and should be evaluated further.},
   Doi = {10.1016/j.jana.2017.03.007},
   Key = {fds327119}
}

@article{fds324504,
   Author = {Cordero, DM and Towe, SL and Chen, N-K and Robertson, KR and Madden, DJ and Huettel, SA and Meade, CS},
   Title = {Cocaine dependence does not contribute substantially to
             white matter abnormalities in HIV infection.},
   Journal = {Journal of NeuroVirology},
   Volume = {23},
   Number = {3},
   Pages = {441-450},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s13365-017-0512-5},
   Abstract = {This study investigated the association of HIV infection and
             cocaine dependence with cerebral white matter integrity
             using diffusion tensor imaging (DTI). One hundred
             thirty-five participants stratified by HIV and cocaine
             status (26 HIV+/COC+, 37 HIV+/COC-, 37 HIV-/COC+, and 35
             HIV-/COC-) completed a comprehensive substance abuse
             assessment, neuropsychological testing, and MRI with DTI.
             Among HIV+ participants, all were receiving HIV care and 46%
             had an AIDS diagnosis. All COC+ participants were current
             users and met criteria for cocaine use disorder. We used
             tract-based spatial statistics (TBSS) to assess the relation
             of HIV and cocaine to fractional anisotropy (FA) and mean
             diffusivity (MD). In whole-brain analyses, HIV+ participants
             had significantly reduced FA and increased MD compared to
             HIV- participants. The relation of HIV and FA was widespread
             throughout the brain, whereas the HIV-related MD effects
             were restricted to the corpus callosum and thalamus. There
             were no significant cocaine or HIV-by-cocaine effects. These
             DTI metrics correlated significantly with duration of HIV
             disease, nadir CD4+ cell count, and AIDS diagnosis, as well
             as some measures of neuropsychological functioning. These
             results suggest that HIV is related to white matter
             integrity throughout the brain, and that HIV-related effects
             are more pronounced with increasing duration of infection
             and greater immune compromise. We found no evidence for
             independent effects of cocaine dependence on white matter
             integrity, and cocaine dependence did not appear to
             exacerbate the effects of HIV.},
   Doi = {10.1007/s13365-017-0512-5},
   Key = {fds324504}
}

@article{fds324505,
   Author = {Lion, RR and Watt, MH and Wechsberg, WM and Meade,
             CS},
   Title = {Gender and Sex Trading Among Active Methamphetamine Users in
             Cape Town, South Africa.},
   Journal = {Substance Use & Misuse (Informa)},
   Volume = {52},
   Number = {6},
   Pages = {773-784},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1080/10826084.2016.1264964},
   Abstract = {South Africa has experienced a tremendous rise in
             methamphetamine use since the year 2000. Sex trading is a
             global phenomenon that has been observed in active drug
             users and has been associated with risks for HIV infection
             and violence.This paper describes and examines the
             correlates of sex trading among active methamphetamine users
             in Cape Town, South Africa.Through peer referral, 360 (201
             male; 159 female) active methamphetamine users were
             recruited in a peri-urban township. Demographics, sex
             trading, drug use, trauma, and mental health were assessed
             by a structured clinical interview and computer survey.
             Logistic regression models were used to examine predictors
             of sex trading for men and women.In the past 3 months, 40%
             of men and 33% of women endorsed trading sex for
             methamphetamine or money. Among these, they reported trading
             with same sex partners (33%), high rates of inconsistent
             condom use (73%), and incidences of physical (23%) and
             sexual (27%) assault when sex trading. Increased drug use
             severity was correlated with sex trading. Women with
             experiences of violence and trauma were also more likely to
             trade sex. Conclusions/importance: The results stress a need
             for linkage to drug treatment, as addiction may be fueling
             sex trading. Targeted interventions geared towards safe sex
             practices may reduce risky sexual behaviors. Women need
             interventions that are attuned to their specific
             vulnerabilities. More research is needed to explore the
             experiences of men who have sex with men given their
             particularly high rates of sex trading behavior.},
   Doi = {10.1080/10826084.2016.1264964},
   Key = {fds324505}
}

@article{fds322767,
   Author = {Hobkirk, AL and Towe, SL and Patel, P and Meade, CS},
   Title = {Food Insecurity Is Associated with Cognitive Deficits Among
             HIV-Positive, But Not HIV-Negative, Individuals in a United
             States Sample.},
   Journal = {AIDS and Behavior},
   Volume = {21},
   Number = {3},
   Pages = {783-791},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1007/s10461-016-1514-7},
   Abstract = {People living with HIV/AIDS (PLWHA) in the United States
             (US) have disproportionately high rates of food insecurity
             (FI). In the general population, FI has been associated with
             cognitive impairment among older adults and may exacerbate
             HIV-associated neurocognitive disorders. The current study
             assessed the effects of FI and HIV infection on the
             neuropsychological performance of 61 HIV-positive and 36
             HIV-negative adults in the US. While the main effects were
             minimal, the interactive effects revealed that FI was
             related to deficits in speed of information processing,
             learning, memory, motor function, and overall cognitive
             impairment for the HIV-positive group, but not the
             HIV-negative group. The interactive effects remained after
             controlling for relevant sociodemographic characteristics.
             Although bidirectional associations cannot be ruled out in a
             cross-sectional study, the results suggest that FI may
             contribute to cognitive impairment among HIV-positive adults
             in the US. Given the high rates of socioeconomic
             disadvantage among PLWHA in the US, addressing FI as part of
             routine clinical care may be warranted.},
   Doi = {10.1007/s10461-016-1514-7},
   Key = {fds322767}
}

@article{fds319647,
   Author = {Watt, MH and Guidera, KE and Hobkirk, AL and Skinner, D and Meade,
             CS},
   Title = {Intimate partner violence among men and women who use
             methamphetamine: A mixed-methods study in South
             Africa.},
   Journal = {Drug and Alcohol Review},
   Volume = {36},
   Number = {1},
   Pages = {97-106},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1111/dar.12420},
   Abstract = {The prevalence of methamphetamine use has risen dramatically
             in parts of South Africa. Globally, methamphetamine has been
             linked to intimate partner violence (IPV) and other forms of
             aggression. The aim of this mixed-methods study was to
             examine the experiences of physical IPV and its contextual
             factors among methamphetamine users in an urban community in
             Cape Town, South Africa.Active methamphetamine users were
             recruited using respondent driven sampling. All participants
             (n = 360) completed structured surveys, and a subset
             (n = 30) completed in-depth interviews with discussions
             of personal IPV experiences. Quantitative data were examined
             separately by gender, and regression models were used to
             identify factors that were associated with physical IPV
             victimisation and perpetration. Qualitative data were
             analysed to provide contextual understanding.In the past
             3 months, 47% of women and 31% of men reported being a
             victim of IPV, and 30% of women and 28% men reported being a
             perpetrator of IPV. Victimisation and perpetration were
             highly correlated, and both were significantly associated
             with histories of other traumas. Although the survey data
             suggests gender equivalence in IPV, the qualitative data
             provides a more nuanced context, with female victimisation
             by male partners being particularly frequent and intense. In
             narratives, IPV was a product of male aggression while using
             methamphetamine, norms around sex trading and gender-based
             attitudes endorsing violence against women.Addiction to
             methamphetamine creates heightened risks of IPV, especially
             among those with previous traumas. The findings emphasise
             the importance of identifying and addressing IPV among
             methamphetamine users in South Africa. [Watt MH, Guidera KE,
             Hobkirk AL, Skinner D, Meade CS. Intimate partner violence
             among men and women who use methamphetamine: A mixed-methods
             study in South Africa. Drug Alcohol Rev 2017;36:97-106].},
   Doi = {10.1111/dar.12420},
   Key = {fds319647}
}

@article{fds319649,
   Author = {Skalski, LM and Towe, SL and Sikkema, KJ and Meade,
             CS},
   Title = {The Impact of Marijuana Use on Memory in HIV-Infected
             Patients: A Comprehensive Review of the HIV and Marijuana
             Literatures.},
   Journal = {Current Drug Abuse Reviews},
   Volume = {9},
   Number = {2},
   Pages = {126-141},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.2174/1874473709666160502124503},
   Abstract = {The most robust neurocognitive effect of marijuana use is
             memory impairment. Memory deficits are also high among
             persons living with HIV/AIDS, and marijuana is the most
             commonly used drug in this population. Yet research
             examining neurocognitive outcomes resulting from
             co-occurring marijuana and HIV is limited.The primary
             objectives of this comprehensive review are to: (1) examine
             the literature on memory functioning in HIV-infected
             individuals; (2) examine the literature on memory
             functioning in marijuana users; (3) synthesize findings and
             propose a theoretical framework to guide future
             research.PubMed was searched for English publications
             2000-2013. Twenty-two studies met inclusion criteria in the
             HIV literature, and 23 studies in the marijuana
             literature.Among HIV-infected individuals, memory deficits
             with medium to large effect sizes were observed. Marijuana
             users also demonstrated memory problems, but results were
             less consistent due to the diversity of samples.A
             compensatory hypothesis, based on the cognitive aging
             literature, is proposed to provide a framework to explore
             the interaction between marijuana and HIV. There is some
             evidence that individuals infected with HIV recruit
             additional brain regions during memory tasks to compensate
             for HIV-related declines in neurocognitive functioning.
             Marijuana is associated with disturbance in similar brain
             systems, and thus it is hypothesized that the added neural
             strain of marijuana can exhaust neural resources, resulting
             in pronounced memory impairment. It will be important to
             test this hypothesis empirically, and future research
             priorities are discussed.},
   Doi = {10.2174/1874473709666160502124503},
   Key = {fds319649}
}

@article{fds312880,
   Author = {Hatch-Maillette, MA and Beadnell, B and Campbell, ANC and Meade, CS and Tross, S and Calsyn, DA},
   Title = {Heterosexual Anal Sex Among Men and Women in Substance Abuse
             Treatment: Secondary Analysis of Two Gender-Specific
             HIV-Prevention Trials.},
   Journal = {Journal of Sex Research},
   Volume = {54},
   Number = {1},
   Pages = {33-41},
   Year = {2017},
   Month = {January},
   ISSN = {0022-4499},
   url = {http://dx.doi.org/10.1080/00224499.2015.1118426},
   Abstract = {Receptive anal sex has high human immunodeficiency virus
             (HIV) transmission risk, and heterosexual substance-abusing
             individuals report higher anal sex rates compared to their
             counterparts in the general population. This secondary
             analysis evaluated the effectiveness of two gender-specific,
             evidence-based HIV-prevention interventions (Real Men Are
             Safe, or REMAS, for men; Safer Sex Skill Building, or SSSB,
             for women) against an HIV education (HIV-Ed) control
             condition on decreasing unprotected heterosexual anal sex
             (HAS) among substance abuse treatment-seeking men
             (n = 171) and women (n = 105). Two variables, engagement
             in any HAS and engagement in unprotected HAS, were assessed
             at baseline and three months postintervention. Compared to
             the control group, women in the gender-specific intervention
             did not differ on rates of any HAS at follow-up but
             significantly decreased their rates of unprotected HAS. Men
             in both the gender-specific and the control interventions
             reported less HAS and unprotected HAS at three-month
             follow-up compared to baseline, with no treatment condition
             effect. The mechanism of action for SSSB compared to REMAS
             in decreasing unprotected HAS is unclear. More attention to
             HAS in HIV-prevention interventions for heterosexual men and
             women in substance abuse treatment is warranted.},
   Doi = {10.1080/00224499.2015.1118426},
   Key = {fds312880}
}


%% Meck, Warren H.   
@article{fds330542,
   Author = {Toda, K and Lusk, NA and Watson, GDR and Kim, N and Lu, D and Li, HE and Meck,
             WH and Yin, HH},
   Title = {Nigrotectal Stimulation Stops Interval Timing in
             Mice.},
   Journal = {Current Biology},
   Volume = {27},
   Number = {24},
   Pages = {3763-3770.e3},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.cub.2017.11.003},
   Abstract = {Considerable evidence implicates the basal ganglia in
             interval timing, yet the underlying mechanisms remain poorly
             understood. Using a novel behavioral task, we demonstrate
             that head-fixed mice can be trained to show the key features
             of timing behavior within a few sessions. Single-trial
             analysis of licking behavior reveals stepping dynamics with
             variable onset times, which is responsible for the canonical
             Gaussian distribution of timing behavior. Moreover, the
             duration of licking bouts decreased as mice became sated,
             showing a strong motivational modulation of licking bout
             initiation and termination. Using optogenetics, we examined
             the role of the basal ganglia output in interval timing. We
             stimulated a pathway important for licking behavior, the
             GABAergic output projections from the substantia nigra pars
             reticulata to the deep layers of the superior colliculus. We
             found that stimulation of this pathway not only cancelled
             licking but also delayed the initiation of anticipatory
             licking for the next interval in a frequency-dependent
             manner. By combining quantitative behavioral analysis with
             optogenetics in the head-fixed setup, we established a new
             approach for studying the neural basis of interval
             timing.},
   Doi = {10.1016/j.cub.2017.11.003},
   Key = {fds330542}
}

@article{fds329170,
   Author = {Teki, S and Gu, B-M and Meck, WH},
   Title = {The persistence of memory: how the brain encodes time in
             memory},
   Journal = {Current Opinion in Behavioral Sciences},
   Volume = {17},
   Pages = {178-185},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.cobeha.2017.09.003},
   Abstract = {© 2017 Elsevier Ltd Time and memory are inextricably
             linked, but it is far from clear how event durations and
             temporal sequences are encoded in memory. In this review, we
             focus on resource allocation models of working memory which
             suggest that memory resources can be flexibly distributed
             amongst several items such that the precision of working
             memory decreases with the number of items to be encoded.
             This type of model is consistent with human performance in
             working memory tasks based on visual, auditory as well as
             temporal stimulus patterns. At the neural-network level, we
             focus on excitatory–inhibitory oscillatory processes that
             are able to encode both interval timing and working memory
             in a coupled excitatory–inhibitory network. This
             modification of the striatal beat-frequency model of
             interval timing shows how memories for multiple time
             intervals are represented by neural oscillations and can
             also be used to explain the mechanisms of resource
             allocation in working memory.},
   Doi = {10.1016/j.cobeha.2017.09.003},
   Key = {fds329170}
}


%% Meyer, Allison E.   
@article{fds328847,
   Author = {Meyer, AE and Curry, JF},
   Title = {Pathways from anxiety to stressful events: An expansion of
             the stress generation hypothesis.},
   Journal = {Clinical Psychology Review},
   Volume = {57},
   Pages = {93-116},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.cpr.2017.08.003},
   Doi = {10.1016/j.cpr.2017.08.003},
   Key = {fds328847}
}


%% Mitroff, Stephen   
@article{fds332884,
   Author = {Mitroff, SR and Ericson, JM and Sharpe, B},
   Title = {Predicting Airport Screening Officers’ Visual Search
             Competency With a Rapid Assessment},
   Journal = {Human Factors},
   Volume = {60},
   Number = {2},
   Pages = {201-211},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/0018720817743886},
   Abstract = {© 2017, © 2017, Human Factors and Ergonomics Society.
             Objective: The study’s objective was to assess a new
             personnel selection and assessment tool for aviation
             security screeners. A mobile app was modified to create a
             tool, and the question was whether it could predict
             professional screeners’ on-job performance. Background: A
             variety of professions (airport security, radiology, the
             military, etc.) rely on visual search performance—being
             able to detect targets. Given the importance of such
             professions, it is necessary to maximize performance, and
             one means to do so is to select individuals who excel at
             visual search. A critical question is whether it is possible
             to predict search competency within a professional search
             environment. Method: Professional searchers from the USA
             Transportation Security Administration (TSA) completed a
             rapid assessment on a tablet-based X-ray simulator (XRAY
             Screener, derived from the mobile technology app Airport
             Scanner; Kedlin Company). The assessment contained 72 trials
             that were simulated X-ray images of bags. Participants
             searched for prohibited items and tapped on them with their
             finger. Results: Performance on the assessment significantly
             related to on-job performance measures for the TSA officers
             such that those who were better XRAY Screener performers
             were both more accurate and faster at the actual airport
             checkpoint. Conclusion: XRAY Screener successfully predicted
             on-job performance for professional aviation security
             officers. While questions remain about the underlying
             cognitive mechanisms, this quick assessment was found to
             significantly predict on-job success for a task that relies
             on visual search performance. Application: It may be
             possible to quickly assess an individual’s visual search
             competency, which could help organizations select new hires
             and assess their current workforce.},
   Doi = {10.1177/0018720817743886},
   Key = {fds332884}
}

@article{fds329322,
   Author = {Mitroff, SR and Sharpe, B},
   Title = {Using big data to solve real problems through academic and
             industry partnerships},
   Journal = {Current Opinion in Behavioral Sciences},
   Volume = {18},
   Pages = {91-96},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.cobeha.2017.09.013},
   Abstract = {© 2017 Elsevier Ltd Big data has revolutionized a number of
             industries as it provides a powerful tool for asking and
             answering questions in novel ways. Academic researchers can
             join this trend and use immense and complex datasets to
             explore previously intractable questions. Yet, accessing and
             analyzing big data can be difficult. The goal of this
             chapter is to outline various benefits and challenges of
             using big data for academic purposes, and to provide
             thoughts on how to succeed. The primary suggestion is for
             academics to collaborate with appropriate industry partners
             to simultaneously achieve both theoretical and practical
             advances.},
   Doi = {10.1016/j.cobeha.2017.09.013},
   Key = {fds329322}
}

@article{fds327186,
   Author = {Biggs, AT and Clark, K and Mitroff, SR},
   Title = {Who should be searching? Differences in personality can
             affect visual search accuracy},
   Journal = {Personality and Individual Differences},
   Volume = {116},
   Pages = {353-358},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.paid.2017.04.045},
   Doi = {10.1016/j.paid.2017.04.045},
   Key = {fds327186}
}

@article{fds331411,
   Author = {Ericson, JM and Kravitz, DJ and Mitroff, SR},
   Title = {Visual Search: You Are Who You Are (+ A Learning
             Curve)},
   Journal = {Perception},
   Pages = {030100661772109-030100661772109},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1177/0301006617721091},
   Doi = {10.1177/0301006617721091},
   Key = {fds331411}
}

@article{fds323691,
   Author = {Chang, BP and Cain, D and Mitroff, SR},
   Title = {Emergency department crowding associated with differences in
             CXR interpretations between emergency physicians and
             radiologists.},
   Journal = {American Journal of Emergency Medicine},
   Volume = {35},
   Number = {5},
   Pages = {793-794},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.ajem.2016.12.067},
   Doi = {10.1016/j.ajem.2016.12.067},
   Key = {fds323691}
}


%% Moffitt, Terrie E.   
@article{fds326211,
   Author = {Romer, AL and Knodt, AR and Houts, R and Brigidi, BD and Moffitt, TE and Caspi, A and Hariri, AR},
   Title = {Structural alterations within cerebellar circuitry are
             associated with general liability for common mental
             disorders.},
   Journal = {Molecular Psychiatry},
   Volume = {23},
   Number = {4},
   Pages = {1084-1090},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1038/mp.2017.57},
   Abstract = {Accumulating mental-health research encourages a shift in
             focus toward transdiagnostic dimensional features that are
             shared across categorical disorders. In support of this
             shift, recent studies have identified a general liability
             factor for psychopathology-sometimes called the 'p factor'-
             that underlies shared risk for a wide range of mental
             disorders. Identifying neural correlates of this general
             liability would substantiate its importance in
             characterizing the shared origins of mental disorders and
             help us begin to understand the mechanisms through which the
             'p factor' contributes to risk. Here we believe we first
             replicate the 'p factor' using cross-sectional data from a
             volunteer sample of 1246 university students, and then using
             high-resolution multimodal structural neuroimaging, we
             demonstrate that individuals with higher 'p factor' scores
             show reduced structural integrity of white matter pathways,
             as indexed by lower fractional anisotropy values, uniquely
             within the pons. Whole-brain analyses further revealed that
             higher 'p factor' scores are associated with reduced gray
             matter volume in the occipital lobe and left cerebellar
             lobule VIIb, which is functionally connected with prefrontal
             regions supporting cognitive control. Consistent with the
             preponderance of cerebellar afferents within the pons, we
             observed a significant positive correlation between the
             white matter integrity of the pons and cerebellar gray
             matter volume associated with higher 'p factor' scores. The
             results of our analyses provide initial evidence that
             structural alterations in corticocerebellar circuitry
             supporting core functions related to the basic integration,
             coordination and monitoring of information may contribute to
             a general liability for common mental disorders.},
   Doi = {10.1038/mp.2017.57},
   Key = {fds326211}
}

@article{fds333592,
   Author = {Choi, KW and Houts, R and Arseneault, L and Pariante, C and Sikkema, KJ and Moffitt, TE},
   Title = {Maternal depression in the intergenerational transmission of
             childhood maltreatment and its sequelae: Testing postpartum
             effects in a longitudinal birth cohort.},
   Journal = {Development and Psychopathology},
   Pages = {1-14},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1017/s0954579418000032},
   Abstract = {Mothers who have experienced childhood maltreatment are more
             likely to have children also exposed to maltreatment, a
             phenomenon known as intergenerational transmission. Factors
             in the perinatal period may contribute uniquely to this
             transmission, but timing effects have not been ascertained.
             Using structural equation modeling with 1,016 mothers and
             their 2,032 children in the Environmental Risk Longitudinal
             Twin Study, we tested the mediating role of postpartum
             depression between maternal childhood maltreatment and a
             cascade of negative child outcomes, specifically child
             exposure to maltreatment, internalizing symptoms, and
             externalizing symptoms: (a) adjusting for later maternal
             depression, (b) comparing across sex differences, and (c)
             examining the relative role of maltreatment subtypes.
             Mothers who had been maltreated as children, especially
             those who had experienced emotional or sexual abuse, were at
             increased risk for postpartum depression. In turn,
             postpartum depression predicted children's exposure to
             maltreatment, followed by emotional and behavioral problems.
             Indirect effects from maternal childhood maltreatment to
             child outcomes were robust across child sex and supported
             significant mediation through postpartum depression;
             however, this appeared to be carried by mothers' depression
             beyond the postpartum period. Identifying and treating
             postpartum depression, and preventing its recurrence, may
             help interrupt the intergenerational transmission of
             maltreatment and its sequelae.},
   Doi = {10.1017/s0954579418000032},
   Key = {fds333592}
}

@article{fds333594,
   Author = {Beckley, AL and Caspi, A and Arseneault, L and Barnes, JC and Fisher,
             HL and Harrington, H and Houts, R and Morgan, N and Odgers, CL and Wertz,
             J and Moffitt, TE},
   Title = {The Developmental Nature of the Victim-Offender
             Overlap.},
   Volume = {4},
   Number = {1},
   Pages = {24-49},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1007/s40865-017-0068-3},
   Abstract = {It is well-established that victims and offenders are often
             the same people, a phenomenon known as the victim-offender
             overlap, but the developmental nature of this overlap
             remains uncertain. In this study, we drew from a
             developmental theoretical framework to test effects of
             genetics, individual characteristics, and
             routine-activity-based risks. Drawing from developmental
             literature, we additionally tested the effect of an
             accumulation of adverse childhood experiences (ACEs).Data
             came from the Environmental Risk (E-Risk) Study, a
             representative UK birth cohort of 2232 twins born in
             1994-1995 and followed to age 18 (with 93% retention). Crime
             victimization and offending were assessed through
             self-reports at age 18 (but findings replicated using crime
             records). We used the classical twin study method to
             decompose variance in the victim-offender overlap into
             genetic and environmental components. We used logistic
             regression to test the effects of childhood risk factors.In
             contrast to past twin studies, we found that environment (as
             well as genes) contributed to the victim-offender overlap.
             Our logistic regression results showed that childhood low
             self-control and childhood antisocial behavior nearly
             doubled the odds of becoming a victim-offender, compared to
             a victim-only or an offender-only. Each additional ACE
             increased the odds of becoming a victim-offender, compared
             to a victim-only or an offender-only, by approximately 12%,
             pointing to the importance of cumulative childhood
             adversity.This study showed that the victim-offender overlap
             is, at least partially, developmental in nature and
             predictable from personal childhood characteristics and an
             accumulation of many adverse childhood experiences.},
   Doi = {10.1007/s40865-017-0068-3},
   Key = {fds333594}
}

@article{fds333593,
   Author = {Wertz, J and Caspi, A and Belsky, DW and Beckley, AL and Arseneault, L and Barnes, JC and Corcoran, DL and Hogan, S and Houts, RM and Morgan, N and Odgers, CL and Prinz, JA and Sugden, K and Williams, BS and Poulton, R and Moffitt, TE},
   Title = {Genetics and Crime: Integrating New Genomic Discoveries Into
             Psychological Research About Antisocial Behavior.},
   Journal = {Psychological Science},
   Pages = {956797617744542},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/0956797617744542},
   Abstract = {Drawing on psychological and sociological theories of crime
             causation, we tested the hypothesis that genetic risk for
             low educational attainment (assessed via a genome-wide
             polygenic score) is associated with criminal offending. We
             further tested hypotheses of how polygenic risk relates to
             the development of antisocial behavior from childhood
             through adulthood. Across the Dunedin and Environmental Risk
             (E-Risk) birth cohorts of individuals growing up 20 years
             and 20,000 kilometers apart, education polygenic scores
             predicted risk of a criminal record with modest effects.
             Polygenic risk manifested during primary schooling in lower
             cognitive abilities, lower self-control, academic
             difficulties, and truancy, and it was associated with a
             life-course-persistent pattern of antisocial behavior that
             onsets in childhood and persists into adulthood. Crime is
             central in the nature-nurture debate, and findings reported
             here demonstrate how molecular-genetic discoveries can be
             incorporated into established theories of antisocial
             behavior. They also suggest that improving school
             experiences might prevent genetic influences on crime from
             unfolding.},
   Doi = {10.1177/0956797617744542},
   Key = {fds333593}
}

@article{fds333009,
   Author = {Moffitt, TE},
   Title = {Male antisocial behaviour in adolescence and
             beyond},
   Journal = {Nature Human Behaviour},
   Volume = {2},
   Number = {3},
   Pages = {177-186},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1038/s41562-018-0309-4},
   Abstract = {© 2018 The Publisher. Male antisocial behaviour is
             concentrated in the adolescent period of the life course, as
             documented by the curve of crime over age. This article
             reviews recent evidence regarding the hypothesis that the
             age-crime curve conceals two groups with different causes.
             Life-course-persistent males show extreme, pervasive,
             persistent antisocial behaviour from early childhood to
             adulthood. They are hypothesized to be rare, with
             pathological risk factors and poor life outcomes. In
             contrast, adolescence-limited males show similar levels of
             antisocial behaviour but primarily during the adolescent
             stage of development. They are hypothesized to be common and
             normative, whereas abstainers from offending are rare. This
             Review recaps the 25-year history of the developmental
             taxonomy of antisocial behaviour, concluding that it is
             standing the test of time in research, and making an impact
             on policy in early-years prevention and juvenile justice.
             Research is needed into how the taxonomy relates to
             neuroscience, health, genetics and changes in modern crime,
             including digital crime.},
   Doi = {10.1038/s41562-018-0309-4},
   Key = {fds333009}
}

@article{fds329193,
   Author = {Newbury, J and Arseneault, L and Caspi, A and Moffitt, TE and Odgers,
             CL and Fisher, HL},
   Title = {Cumulative Effects of Neighborhood Social Adversity and
             Personal Crime Victimization on Adolescent Psychotic
             Experiences.},
   Journal = {Schizophrenia Bulletin},
   Volume = {44},
   Number = {2},
   Pages = {348-358},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/schbul/sbx060},
   Abstract = {Little is known about the impact of urbanicity, adverse
             neighborhood conditions and violent crime victimization on
             the emergence of adolescent psychotic experiences.Participants
             were from the Environmental Risk (E-Risk) Longitudinal Twin
             Study, a nationally-representative cohort of 2232 British
             twins who were interviewed about adolescent psychotic
             experiences at age 18. Urbanicity, neighborhood
             characteristics, and personal victimization by violent crime
             were measured during childhood and adolescence via geocoded
             census data, surveys of over 5000 immediate neighbors of the
             E-Risk participants, and interviews with participants
             themselves.Adolescents raised in urban vs rural
             neighborhoods were significantly more likely to have
             psychotic experiences (OR = 1.67, 95% CI = 1.21-2.30, P =
             .002). This association remained significant after
             considering potential confounders including family
             socioeconomic status, family psychiatric history, and
             adolescent substance problems (OR = 1.43, 95% CI =
             1.01-2.03, P = .042), but became nonsignificant after
             considering adverse social conditions in urban neighborhoods
             such as low social cohesion and high neighborhood disorder
             (OR = 1.35, 95% CI = 0.94-1.92, P = .102). The combined
             association of adverse neighborhood social conditions and
             personal crime victimization with adolescent psychotic
             experiences (adjusted OR = 4.86, 95% CI = 3.28-7.20, P <
             .001) was substantially greater than for either exposure
             alone, highlighting a potential interaction between
             neighborhood conditions and crime victimization (interaction
             contrast ratio = 1.81, 95% CI = -0.03 to 3.65) that was
             significant at the P = .054 level.Cumulative effects of
             adverse neighborhood social conditions and personal
             victimization by violent crime during upbringing partly
             explain why adolescents in urban settings are more likely to
             report psychotic experiences. Early intervention efforts for
             psychosis could be targeted towards victimized youth living
             in urban and socially adverse neighborhoods.},
   Doi = {10.1093/schbul/sbx060},
   Key = {fds329193}
}

@article{fds328906,
   Author = {Meier, MH and Caspi, A and Danese, A and Fisher, HL and Houts, R and Arseneault, L and Moffitt, TE},
   Title = {Associations between adolescent cannabis use and
             neuropsychological decline: a longitudinal co-twin control
             study.},
   Journal = {Addiction},
   Volume = {113},
   Number = {2},
   Pages = {257-265},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1111/add.13946},
   Abstract = {This study tested whether adolescents who used cannabis or
             met criteria for cannabis dependence showed
             neuropsychological impairment prior to cannabis initiation
             and neuropsychological decline from before to after cannabis
             initiation.A longitudinal co-twin control study.Participants
             were 1989 twins from the Environmental Risk (E-Risk)
             Longitudinal Twin Study, a nationally representative birth
             cohort of twins born in England and Wales from 1994 to
             1995.Frequency of cannabis use and cannabis dependence were
             assessed at age 18. Intelligence quotient (IQ) was obtained
             at ages 5, 12 and 18. Executive functions were assessed at
             age 18.Compared with adolescents who did not use cannabis,
             adolescents who used cannabis had lower IQ in childhood
             prior to cannabis initiation and lower IQ at age 18, but
             there was little evidence that cannabis use was associated
             with IQ decline from ages 12-18. For example, adolescents
             with cannabis dependence had age 12 and age 18 IQ scores
             that were 5.61 (t = -3.11, P = 0.002) and 7.34 IQ points
             (t = -5.27, P < 0.001) lower than adolescents without
             cannabis dependence, but adolescents with cannabis
             dependence did not show greater IQ decline from age 12-18
             (t = -1.27, P = 0.20). Moreover, adolescents who used
             cannabis had poorer executive functions at age 18 than
             adolescents who did not use cannabis, but these associations
             were generally not apparent within twin pairs. For example,
             twins who used cannabis more frequently than their co-twin
             performed similarly to their co-twin on five of six
             executive function tests (Ps > 0.10). The one exception
             was that twins who used cannabis more frequently than their
             co-twin performed worse on one working memory test (Spatial
             Span reversed; β = -0.07, P = 0.036).Short-term
             cannabis use in adolescence does not appear to cause IQ
             decline or impair executive functions, even when cannabis
             use reaches the level of dependence. Family background
             factors explain why adolescent cannabis users perform worse
             on IQ and executive function tests.},
   Doi = {10.1111/add.13946},
   Key = {fds328906}
}

@article{fds332179,
   Author = {Beckley, AL and Caspi, A and Broadbent, J and Harrington, H and Houts,
             RM and Poulton, R and Ramrakha, S and Reuben, A and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Criminal
             Offending.},
   Journal = {JAMA Pediatrics},
   Volume = {172},
   Number = {2},
   Pages = {166-173},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1001/jamapediatrics.2017.4005},
   Abstract = {Lead is a neurotoxin with well-documented effects on health.
             Research suggests that lead may be associated with criminal
             behavior. This association is difficult to disentangle from
             low socioeconomic status, a factor in both lead exposure and
             criminal offending.To test the hypothesis that a higher
             childhood blood lead level (BLL) is associated with greater
             risk of criminal conviction, recidivism (repeat conviction),
             conviction for violent offenses, and variety of
             self-reported criminal offending in a setting where BLL was
             not associated with low socioeconomic status.A total of 553
             individuals participated in a prospective study based on a
             population-representative cohort born between April 1, 1972,
             and March 31, 1973, from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (December 2012). Statistical
             analysis was performed from November 10, 2016, to September
             5, 2017.Blood lead level measured at age 11 years.Official
             criminal conviction cumulative to age 38 years (data
             collected in 2013), single conviction or recidivism,
             conviction for nonviolent or violent crime, and
             self-reported variety of crime types at ages 15, 18, 21, 26,
             32, and 38 years.Participants included 553 individuals (255
             female and 298 male participants) who had their blood tested
             for lead at age 11 years. The mean (SD) BLL at age 11 years
             was 11.01 (4.62) μg/dL. A total of 154 participants (27.8%)
             had a criminal conviction, 86 (15.6%) had recidivated, and
             53 (9.6%) had a violent offense conviction. Variety scores
             for self-reported offending ranged from 0 to 10 offense
             types at each assessment; higher numbers indicated greater
             crime involvement. Self-reported offending followed the
             well-established age-crime curve (ie, the mean [SD] variety
             of self-reported offending increased from 1.99 [2.82] at age
             15 years to its peak of 4.24 [3.15] at age 18 years and 4.22
             [3.02] at age 21 years and declined thereafter to 1.10
             [1.59] at age 38 years). Blood lead level was a poor
             discriminator between no conviction and conviction (area
             under the curve, 0.58). Overall, associations between BLL
             and conviction outcomes were weak. The estimated effect of
             BLL was lower for recidivism than for single convictions and
             lower for violent offending than for nonviolent offending.
             Sex-adjusted associations between BLL reached statistical
             significance for only 1 of the 6 self-reported offending
             outcomes at age 15 years (r = 0.10; 95% CI, 0.01-0.18;
             P = .02).This study overcomes past limitations of
             studies of BLL and crime by studying the association in a
             place and time where the correlation was not confounded by
             childhood socioeconomic status. Findings failed to support a
             dose-response association between BLL and consequential
             criminal offending.},
   Doi = {10.1001/jamapediatrics.2017.4005},
   Key = {fds332179}
}

@article{fds332178,
   Author = {Marzi, SJ and Sugden, K and Arseneault, L and Belsky, DW and Burrage, J and Corcoran, DL and Danese, A and Fisher, HL and Hannon, E and Moffitt, TE and Odgers, CL and Pariante, C and Poulton, R and Williams, BS and Wong,
             CCY and Mill, J and Caspi, A},
   Title = {Analysis of DNA Methylation in Young People: Limited
             Evidence for an Association Between Victimization Stress and
             Epigenetic Variation in Blood.},
   Journal = {American Journal of Psychiatry},
   Pages = {appiajp201717060693},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.17060693},
   Abstract = {DNA methylation has been proposed as an epigenetic mechanism
             by which early-life experiences become "embedded" in the
             genome and alter transcriptional processes to compromise
             health. The authors sought to investigate whether early-life
             victimization stress is associated with genome-wide DNA
             methylation.The authors tested the hypothesis that
             victimization is associated with DNA methylation in the
             Environmental Risk (E-Risk) Longitudinal Study, a nationally
             representative 1994-1995 birth cohort of 2,232 twins born in
             England and Wales and assessed at ages 5, 7, 10, 12, and 18
             years. Multiple forms of victimization were ascertained in
             childhood and adolescence (including physical, sexual, and
             emotional abuse; neglect; exposure to intimate-partner
             violence; bullying; cyber-victimization; and
             crime).Epigenome-wide analyses of polyvictimization across
             childhood and adolescence revealed few significant
             associations with DNA methylation in peripheral blood at age
             18, but these analyses were confounded by tobacco smoking
             and/or did not survive co-twin control tests. Secondary
             analyses of specific forms of victimization revealed sparse
             associations with DNA methylation that did not replicate
             across different operationalizations of the same putative
             victimization experience. Hypothesis-driven analyses of six
             candidate genes in the stress response (NR3C1, FKBP5, BDNF,
             AVP, CRHR1, SLC6A4) did not reveal predicted associations
             with DNA methylation in probes annotated to these
             genes.Findings from this epidemiological analysis of the
             epigenetic effects of early-life stress do not support the
             hypothesis of robust changes in DNA methylation in
             victimized young people. We need to come to terms with the
             possibility that epigenetic epidemiology is not yet well
             matched to experimental, nonhuman models in uncovering the
             biological embedding of stress.},
   Doi = {10.1176/appi.ajp.2017.17060693},
   Key = {fds332178}
}

@article{fds329770,
   Author = {Newbury, JB and Arseneault, L and Moffitt, TE and Caspi, A and Danese,
             A and Baldwin, JR and Fisher, HL},
   Title = {Measuring childhood maltreatment to predict early-adult
             psychopathology: Comparison of prospective informant-reports
             and retrospective self-reports.},
   Journal = {Journal of Psychiatric Research},
   Volume = {96},
   Pages = {57-64},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jpsychires.2017.09.020},
   Abstract = {Both prospective informant-reports and retrospective
             self-reports may be used to measure childhood maltreatment,
             though both methods entail potential limitations such as
             underestimation and memory biases. The validity and utility
             of standard measures of childhood maltreatment requires
             clarification in order to inform the design of future
             studies investigating the mental health consequences of
             maltreatment. The present study assessed agreement between
             prospective informant-reports and retrospective self-reports
             of childhood maltreatment, as well as the comparative
             utility of both reports for predicting a range of
             psychiatric problems at age 18. Data were obtained from the
             Environmental Risk (E-Risk) Longitudinal Twin Study, a
             nationally-representative birth cohort of 2232 children
             followed to 18 years of age (with 93% retention). Childhood
             maltreatment was assessed in two ways: (i) prospective
             informant-reports from caregivers, researchers, and
             clinicians when children were aged 5, 7, 10 and 12; and (ii)
             retrospective self-reports of maltreatment experiences
             occurring up to age 12, obtained at age 18 using the
             Childhood Trauma Questionnaire. Participants were privately
             interviewed at age 18 concerning several psychiatric
             problems including depression, anxiety, self-injury,
             alcohol/cannabis dependence, and conduct disorder. There was
             only slight to fair agreement between prospective and
             retrospective reports of childhood maltreatment (all
             Kappa's ≤ 0.31). Both prospective and retrospective
             reports of maltreatment were associated with age-18
             psychiatric problems, though the strongest associations were
             found when maltreatment was retrospectively self-reported.
             These findings indicate that prospective and retrospective
             reports of childhood maltreatment capture largely
             non-overlapping groups of individuals. Young adults who
             recall being maltreated have a particularly elevated risk
             for psychopathology.},
   Doi = {10.1016/j.jpsychires.2017.09.020},
   Key = {fds329770}
}

@article{fds329192,
   Author = {Baldwin, JR and Arseneault, L and Caspi, A and Fisher, HL and Moffitt,
             TE and Odgers, CL and Pariante, C and Ambler, A and Dove, R and Kepa, A and Matthews, T and Menard, A and Sugden, K and Williams, B and Danese,
             A},
   Title = {Childhood victimization and inflammation in young adulthood:
             A genetically sensitive cohort study.},
   Journal = {Brain, Behavior, and Immunity},
   Volume = {67},
   Pages = {211-217},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.bbi.2017.08.025},
   Abstract = {Childhood victimization is an important risk factor for
             later immune-related disorders. Previous evidence has
             demonstrated that childhood victimization is associated with
             elevated levels of inflammation biomarkers measured decades
             after exposure. However, it is unclear whether this
             association is (1) already detectable in young people, (2)
             different in males and females, and (3) confounded by
             genetic liability to inflammation. Here we sought to address
             these questions.Participants were 2232 children followed
             from birth to age 18years as part of the Environmental Risk
             (E-Risk) Longitudinal Twin Study. Childhood victimization
             was measured prospectively from birth to age 12years.
             Inflammation was measured through C-reactive protein (CRP)
             levels in dried blood spots at age 18years. Latent genetic
             liability for high inflammation levels was assessed through
             a twin-based method.Greater exposure to childhood
             victimization was associated with higher CRP levels at age
             18 (serum-equivalent means were 0.65 in non-victimized Study
             members, 0.74 in those exposed to one victimization type,
             and 0.81 in those exposed to poly-victimization; p=0.018).
             However, this association was driven by a significant
             association in females (serum-equivalent means were 0.75 in
             non-victimized females, 0.87 in those exposed to one type of
             victimization, and 1.19 in those exposed to
             poly-victimization; p=0.010), while no significant
             association was observed in males (p=0.19). Victimized
             females showed elevated CRP levels independent of latent
             genetic influence, as well as childhood socioeconomic
             status, and waist-hip ratio and body temperature at the time
             of CRP assessment.Childhood victimization is associated with
             elevated CRP levels in young women, independent of latent
             genetic influences and other key risk factors. These results
             strengthen causal inference about the effects of childhood
             victimization on inflammation levels in females by
             accounting for potential genetic confounding.},
   Doi = {10.1016/j.bbi.2017.08.025},
   Key = {fds329192}
}

@article{fds332046,
   Author = {Wertz, J and Agnew-Blais, J and Caspi, A and Danese, A and Fisher, HL and Goldman-Mellor, S and Moffitt, TE and Arseneault,
             L},
   Title = {From Childhood Conduct Problems to Poor Functioning at Age
             18 Years: Examining Explanations in a Longitudinal Cohort
             Study.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {57},
   Number = {1},
   Pages = {54-60.e4},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.09.437},
   Abstract = {Childhood conduct problems are associated with poor
             functioning in early adulthood. We tested a series of
             hypotheses to understand the mechanisms underlying this
             association.We used data from the Environmental Risk
             (E-Risk) Longitudinal Twin Study, a birth cohort of 2,232
             twins born in England and Wales in 1994 and 1995, followed
             up to age 18 years with 93% retention. Severe conduct
             problems in childhood were assessed at ages 5, 7, and 10
             years using parent and teacher reports. Poor functioning at
             age 18 years, including cautions and convictions, daily
             cigarette smoking, heavy drinking, and psychosocial
             difficulties, was measured through interviews with
             participants and official crime record searches.Participants
             18 years old with versus without a childhood history of
             severe conduct problems had greater rates of each poor
             functional outcome, and they were more likely to experience
             multiple poor outcomes. This association was partly
             accounted for by concurrent psychopathology in early
             adulthood, as well as by early familial risk factors, both
             genetic and environmental. Childhood conduct problems,
             however, continued to predict poor outcomes at age 18 years
             after accounting for these explanations.Children with severe
             conduct problems display poor functioning at age 18 years
             because of concurrent problems in early adulthood and
             familial risk factors originating in childhood. However,
             conduct problems also exert a lasting effect on young
             people's lives independent of these factors, pointing to
             early conduct problems as a target for early interventions
             aimed at preventing poor functional outcomes.},
   Doi = {10.1016/j.jaac.2017.09.437},
   Key = {fds332046}
}

@article{fds331412,
   Author = {Rivenbark, JG and Odgers, CL and Caspi, A and Harrington, H and Hogan,
             S and Houts, RM and Poulton, R and Moffitt, TE},
   Title = {The high societal costs of childhood conduct problems:
             evidence from administrative records up to age 38 in a
             longitudinal birth cohort.},
   Journal = {The Journal of Child Psychology and Psychiatry and Allied
             Disciplines},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1111/jcpp.12850},
   Abstract = {Children with conduct problems that persist into adulthood
             are at increased risk for future behavioral, health, and
             social problems. However, the longer term public service
             usage among these children has not been fully documented. To
             aid public health and intervention planning, adult service
             usage across criminal justice, health care, and social
             welfare domains is compared among all individuals from a
             representative cohort who followed different conduct problem
             trajectories from childhood into adulthood.Participants are
             from the Dunedin Multidisciplinary Health and Development
             Study, a prospective, representative cohort of consecutive
             births (N = 1,037) from April 1972 to March 1973 in
             Dunedin, New Zealand. Regression analyses were used to
             compare levels of public service usage up to age 38,
             gathered via administrative and electronic medical records,
             between participants who displayed distinct subtypes of
             childhood conduct problems (low, childhood-limited,
             adolescent-onset, and life-course persistent).Children
             exhibiting life-course persistent conduct problems used
             significantly more services as adults than those with low
             levels of childhood conduct problems. Although this group
             comprised only 9.0% of the population, they accounted for
             53.3% of all convictions, 15.7% of emergency department
             visits, 20.5% of prescription fills, 13.1% of injury claims,
             and 24.7% of welfare benefit months. Half of this group
             (50.0%) also accrued high service use across all three
             domains of criminal justice, health, and social welfare
             services, as compared to only 11.3% of those with low
             conduct problems (OR = 7.27, 95% CI = 4.42-12.0).Conduct
             problems in childhood signal high future costs in terms of
             service utilization across multiple sectors. Future
             evaluations of interventions aimed at conduct problems
             should also track potential reductions in health burden and
             service usage that stretch into midlife.},
   Doi = {10.1111/jcpp.12850},
   Key = {fds331412}
}

@article{fds330410,
   Author = {Newbury, JB and Arseneault, L and Caspi, A and Moffitt, TE and Odgers,
             CL and Baldwin, JR and Zavos, HMS and Fisher, HL},
   Title = {In the eye of the beholder: Perceptions of neighborhood
             adversity and psychotic experiences in adolescence.},
   Journal = {Development and Psychopathology},
   Volume = {29},
   Number = {5},
   Pages = {1823-1837},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1017/s0954579417001420},
   Abstract = {Adolescent psychotic experiences increase risk for
             schizophrenia and other severe psychopathology in adulthood.
             Converging evidence implicates urban and adverse
             neighborhood conditions in the etiology of adolescent
             psychotic experiences, but the role of young people's
             personal perceptions of disorder (i.e., physical and social
             signs of threat) in their neighborhood is unknown. This was
             examined using data from the Environmental Risk Longitudinal
             Twin Study, a nationally representative birth cohort of
             2,232 British twins. Participants were interviewed at age 18
             about psychotic phenomena and perceptions of disorder in the
             neighborhood. Multilevel, longitudinal, and genetically
             sensitive analyses investigated the association between
             perceptions of neighborhood disorder and adolescent
             psychotic experiences. Adolescents who perceived higher
             levels of neighborhood disorder were significantly more
             likely to have psychotic experiences, even after accounting
             for objectively/independently measured levels of crime and
             disorder, neighborhood- and family-level socioeconomic
             status, family psychiatric history, adolescent substance and
             mood problems, and childhood psychotic symptoms: odds ratio
             = 1.62, 95% confidence interval [1.27, 2.05], p < .001. The
             phenotypic overlap between adolescent psychotic experiences
             and perceptions of neighborhood disorder was explained by
             overlapping common environmental influences, rC = .88, 95%
             confidence interval [0.26, 1.00]. Findings suggest that
             early psychological interventions to prevent adolescent
             psychotic experiences should explore the role of young
             people's (potentially modifiable) perceptions of threatening
             neighborhood conditions.},
   Doi = {10.1017/s0954579417001420},
   Key = {fds330410}
}

@article{fds330411,
   Author = {Schaefer, JD and Scult, MA and Caspi, A and Arseneault, L and Belsky,
             DW and Hariri, AR and Harrington, H and Houts, R and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Is low cognitive functioning a predictor or consequence of
             major depressive disorder? A test in two longitudinal birth
             cohorts.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s095457941700164x},
   Abstract = {Cognitive impairment has been identified as an important
             aspect of major depressive disorder (MDD). We tested two
             theories regarding the association between MDD and cognitive
             functioning using data from longitudinal cohort studies. One
             theory, the cognitive reserve hypothesis, suggests that
             higher cognitive ability in childhood decreases risk of
             later MDD. The second, the scarring hypothesis, instead
             suggests that MDD leads to persistent cognitive deficits
             following disorder onset. We tested both theories in the
             Dunedin Study, a population-representative cohort followed
             from birth to midlife and assessed repeatedly for both
             cognitive functioning and psychopathology. We also used data
             from the Environmental Risk Longitudinal Twin Study to test
             whether childhood cognitive functioning predicts future MDD
             risk independent of family-wide and genetic risk using a
             discordant twin design. Contrary to both hypotheses, we
             found that childhood cognitive functioning did not predict
             future risk of MDD, nor did study members with a past
             history of MDD show evidence of greater cognitive decline
             unless MDD was accompanied by other comorbid psychiatric
             conditions. Our results thus suggest that low cognitive
             functioning is related to comorbidity, but is neither an
             antecedent nor an enduring consequence of MDD. Future
             research may benefit from considering cognitive deficits
             that occur during depressive episodes from a transdiagnostic
             perspective.},
   Doi = {10.1017/s095457941700164x},
   Key = {fds330411}
}

@article{fds328626,
   Author = {Matthews, T and Danese, A and Gregory, AM and Caspi, A and Moffitt, TE and Arseneault, L},
   Title = {Sleeping with one eye open: loneliness and sleep quality in
             young adults},
   Journal = {Psychological Medicine},
   Volume = {47},
   Number = {12},
   Pages = {2177-2186},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1017/S0033291717000629},
   Doi = {10.1017/S0033291717000629},
   Key = {fds328626}
}

@article{fds328905,
   Author = {Williams, MJA and Milne, BJ and Ambler, A and Theodore, R and Ramrakha,
             S and Caspi, A and Moffitt, TE and Poulton, R},
   Title = {Childhood body mass index and endothelial dysfunction
             evaluated by peripheral arterial tonometry in early
             midlife.},
   Journal = {International Journal of Obesity},
   Volume = {41},
   Number = {9},
   Pages = {1355-1360},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1038/ijo.2017.108},
   Abstract = {Endothelial dysfunction predicts mortality but it is unknown
             whether childhood obesity predicts adult endothelial
             dysfunction. The aim of this study was to determine whether
             anthropometric indices of body fat in childhood, adolescence
             and early midlife are associated with endothelial
             dysfunction in early midlife.Participants belonged to a
             representative birth cohort of 1037 individuals born in
             Dunedin, New Zealand in 1972 and 1973 and followed to age 38
             years, with 95% retention (the Dunedin Multidisciplinary
             Health and Development Study). We assessed anthropometric
             indices of obesity at ages 3, 5, 7, 9, 11, 13, 15, 18, 21,
             26, 32 and 38 years. We tested associations between
             endothelial function assessed by peripheral arterial
             tonometry (PAT) at age 38 and; age 38 cardiovascular risk
             factors; age 3 body mass index (BMI); and four BMI
             trajectory groups from childhood to early midlife.Early
             midlife endothelial dysfunction was associated with BMI,
             large waist circumference, low high-density lipoprotein
             cholesterol, low cardiorespiratory fitness and increased
             high-sensitivity C-reactive protein. After adjustment for
             sex and childhood socioeconomic status, 3-year-olds with BMI
             1 s.d. above the mean had Framingham-reactive hyperemia
             index (F-RHI) ratios that were 0.10 below those with normal
             BMI (β=-0.10, 95% confidence interval (CI) -0.17 to -0.03,
             P=0.007) at age 38. Cohort members in the 'overweight',
             'obese' and 'morbidly obese' trajectories had F-RHI ratios
             that were 0.08 (β=-0.08, 95% CI -0.14 to -0.03, P=0.003),
             0.13 (β=-0.13, 95% CI -0.21 to -0.06, P<0.001) and 0.17
             (β=-0.17, 95% CI -0.33 to -0.01, P=0.033), respectively,
             below age-peers in the 'normal' trajectory.Childhood BMI and
             the trajectories of BMI from childhood to early midlife
             predict endothelial dysfunction evaluated by PAT in early
             midlife.},
   Doi = {10.1038/ijo.2017.108},
   Key = {fds328905}
}

@article{fds327018,
   Author = {Suppli, NP and Bukh, JD and Moffitt, TE and Caspi, A and Johansen, C and Tjønneland, A and Kessing, LV and Dalton, SO},
   Title = {Genetic variants in 5-HTTLPR, BDNF, HTR1A, COMT, and FKBP5
             and risk for treated depression after cancer
             diagnosis.},
   Journal = {Depression and Anxiety},
   Volume = {34},
   Number = {9},
   Pages = {845-855},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1002/da.22660},
   Abstract = {The role of gene-environment interactions in the
             pathogenesis of depression is unclear. Previous studies
             addressed vulnerability for depression after childhood
             adversity and stressful life events among carriers of
             numerous specific genetic variants; however, the importance
             of individual genetic variants, the environmental exposures
             with which they interact, and the magnitude of the risk
             conveyed by these interactions remain elusive.We included
             7,320 people with a first primary cancer identified in the
             prospective Diet, Cancer and Health study in an exposed-only
             cohort study. The mean age of the individuals was 68 years
             (5th, 95th percentiles: 58, 78) at cancer diagnosis. Using
             Cox regression models and cumulative incidence plots, we
             analyzed the associations between genetic variants in
             5-HTTLPR, BDNF, HTR1A, COMT, and FKBP5 and use of
             antidepressants as well as hospital contact for depression
             after diagnosis of cancer.Overall, we observed no
             statistically significant associations, with nonsignificant
             hazard ratio estimates for use of antidepressants of
             0.95-1.07.This study of elderly people indicates that it is
             unlikely that the investigated genetic variants are
             clinically relevantly associated with depression after
             diagnosis of cancer. The mechanisms for gene-environment
             interactions in younger individuals are probably different,
             and we advise caution in extrapolating our results to early
             life stress. However, conclusion from the present study
             might be generalizable to elderly persons exposed to other
             stressful life events.},
   Doi = {10.1002/da.22660},
   Key = {fds327018}
}

@article{fds321669,
   Author = {Erskine, HE and Baxter, AJ and Patton, G and Moffitt, TE and Patel, V and Whiteford, HA and Scott, JG},
   Title = {The global coverage of prevalence data for mental disorders
             in children and adolescents.},
   Journal = {Epidemiology and Psychiatric Sciences},
   Volume = {26},
   Number = {4},
   Pages = {395-402},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1017/s2045796015001158},
   Abstract = {Children and adolescents make up almost a quarter of the
             world's population with 85% living in low- and middle-income
             countries (LMICs). Globally, mental (and substance use)
             disorders are the leading cause of disability in young
             people; however, the representativeness or 'coverage' of the
             prevalence data is unknown. Coverage refers to the
             proportion of the target population (ages 5-17 years)
             represented by the available data.Prevalence data for
             conduct disorder (CD), attention-deficit/hyperactivity
             disorder (ADHD), autism spectrum disorders (ASDs), eating
             disorders (EDs), depression, and anxiety disorders were
             sourced from systematic reviews conducted for the Global
             Burden of Disease Study 2010 (GBD 2010) and 2013 (GBD 2013).
             For each study, the location proportion was multiplied by
             the age proportion to give study coverage. Location
             proportion was calculated by dividing the total study
             location population by the total study location population.
             Age proportion was calculated by dividing the population of
             the country aged within the age range of the study sample by
             the population of the country aged within the age range of
             the study sample. If a study only sampled one sex, study
             coverage was halved. Coverage across studies was then summed
             for each country to give coverage by country. This method
             was repeated at the region and global level, and separately
             for GBD 2013 and GBD 2010.Mean global coverage of prevalence
             data for mental disorders in ages 5-17 years was 6.7% (CD:
             5.0%, ADHD: 5.5%, ASDs: 16.1%, EDs: 4.4%, depression: 6.2%,
             anxiety: 3.2%). Of 187 countries, 124 had no data for any
             disorder. Many LMICs were poorly represented in the
             available prevalence data, for example, no region in
             sub-Saharan Africa had more than 2% coverage for any
             disorder. While coverage increased between GBD 2010 and GBD
             2013, this differed greatly between disorders and few new
             countries provided data.The global coverage of prevalence
             data for mental disorders in children and adolescents is
             limited. Practical methodology must be developed and
             epidemiological surveys funded to provide representative
             prevalence estimates so as to inform appropriate resource
             allocation and support policies that address mental health
             needs of children and adolescents.},
   Doi = {10.1017/s2045796015001158},
   Key = {fds321669}
}

@article{fds326210,
   Author = {Belsky, DW and Caspi, A and Cohen, HJ and Kraus, WE and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Impact of early personal-history characteristics on the Pace
             of Aging: implications for clinical trials of therapies to
             slow aging and extend healthspan.},
   Journal = {Aging Cell},
   Volume = {16},
   Number = {4},
   Pages = {644-651},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/acel.12591},
   Abstract = {Therapies to extend healthspan are poised to move from
             laboratory animal models to human clinical trials.
             Translation from mouse to human will entail challenges,
             among them the multifactorial heterogeneity of human aging.
             To inform clinical trials about this heterogeneity, we
             report how humans' pace of biological aging relates to
             personal-history characteristics. Because geroprotective
             therapies must be delivered by midlife to prevent
             age-related disease onset, we studied young-adult members of
             the Dunedin Study 1972-73 birth cohort (n = 954). Cohort
             members' Pace of Aging was measured as coordinated decline
             in the integrity of multiple organ systems, by quantifying
             rate of decline across repeated measurements of 18
             biomarkers assayed when cohort members were ages 26, 32, and
             38 years. The childhood personal-history characteristics
             studied were known predictors of age-related disease and
             mortality, and were measured prospectively during childhood.
             Personal-history characteristics of familial longevity,
             childhood social class, adverse childhood experiences, and
             childhood health, intelligence, and self-control all
             predicted differences in cohort members' adulthood Pace of
             Aging. Accumulation of more personal-history risks predicted
             faster Pace of Aging. Because trials of anti-aging therapies
             will need to ascertain personal histories retrospectively,
             we replicated results using cohort members' retrospective
             personal-history reports made in adulthood. Because many
             trials recruit participants from clinical settings, we
             replicated results in the cohort subset who had recent
             health system contact according to electronic medical
             records. Quick, inexpensive measures of trial participants'
             early personal histories can enable clinical trials to study
             who volunteers for trials, who adheres to treatment, and who
             responds to anti-aging therapies.},
   Doi = {10.1111/acel.12591},
   Key = {fds326210}
}

@article{fds325850,
   Author = {Kotov, R and Krueger, RF and Watson, D and Achenbach, TM and Althoff,
             RR and Bagby, RM and Brown, TA and Carpenter, WT and Caspi, A and Clark,
             LA and Eaton, NR and Forbes, MK and Forbush, KT and Goldberg, D and Hasin,
             D and Hyman, SE and Ivanova, MY and Lynam, DR and Markon, K and Miller, JD and Moffitt, TE and Morey, LC and Mullins-Sweatt, SN and Ormel, J and Patrick, CJ and Regier, DA and Rescorla, L and Ruggero, CJ and Samuel,
             DB and Sellbom, M and Simms, LJ and Skodol, AE and Slade, T and South, SC and Tackett, JL and Waldman, ID and Waszczuk, MA and Widiger, TA and Wright,
             AGC and Zimmerman, M},
   Title = {The Hierarchical Taxonomy of Psychopathology (HiTOP): A
             dimensional alternative to traditional nosologies.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {4},
   Pages = {454-477},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1037/abn0000258},
   Abstract = {The reliability and validity of traditional taxonomies are
             limited by arbitrary boundaries between psychopathology and
             normality, often unclear boundaries between disorders,
             frequent disorder co-occurrence, heterogeneity within
             disorders, and diagnostic instability. These taxonomies went
             beyond evidence available on the structure of
             psychopathology and were shaped by a variety of other
             considerations, which may explain the aforementioned
             shortcomings. The Hierarchical Taxonomy Of Psychopathology
             (HiTOP) model has emerged as a research effort to address
             these problems. It constructs psychopathological syndromes
             and their components/subtypes based on the observed
             covariation of symptoms, grouping related symptoms together
             and thus reducing heterogeneity. It also combines
             co-occurring syndromes into spectra, thereby mapping out
             comorbidity. Moreover, it characterizes these phenomena
             dimensionally, which addresses boundary problems and
             diagnostic instability. Here, we review the development of
             the HiTOP and the relevant evidence. The new classification
             already covers most forms of psychopathology. Dimensional
             measures have been developed to assess many of the
             identified components, syndromes, and spectra. Several
             domains of this model are ready for clinical and research
             applications. The HiTOP promises to improve research and
             clinical practice by addressing the aforementioned
             shortcomings of traditional nosologies. It also provides an
             effective way to summarize and convey information on risk
             factors, etiology, pathophysiology, phenomenology, illness
             course, and treatment response. This can greatly improve the
             utility of the diagnosis of mental disorders. The new
             classification remains a work in progress. However, it is
             developing rapidly and is poised to advance mental health
             research and care significantly as the relevant science
             matures. (PsycINFO Database Record},
   Doi = {10.1037/abn0000258},
   Key = {fds325850}
}

@article{fds325487,
   Author = {Arseneault, L and Agnew-Blais, J and Moffitt, TE},
   Title = {Child vs Adult Onset of Attention-Deficit/Hyperactivity
             Disorder-Reply.},
   Journal = {JAMA Psychiatry},
   Volume = {74},
   Number = {4},
   Pages = {422-423},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1001/jamapsychiatry.2016.2781},
   Doi = {10.1001/jamapsychiatry.2016.2781},
   Key = {fds325487}
}

@article{fds324440,
   Author = {Danese, A and Moffitt, TE and Arseneault, L and Bleiberg, BA and Dinardo, PB and Gandelman, SB and Houts, R and Ambler, A and Fisher, HL and Poulton, R and Caspi, A},
   Title = {The Origins of Cognitive Deficits in Victimized Children:
             Implications for Neuroscientists and Clinicians.},
   Journal = {American Journal of Psychiatry},
   Volume = {174},
   Number = {4},
   Pages = {349-361},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1176/appi.ajp.2016.16030333},
   Abstract = {Individuals reporting a history of childhood violence
             victimization have impaired brain function. However, the
             clinical significance, reproducibility, and causality of
             these findings are disputed. The authors used data from two
             large cohort studies to address these research questions
             directly.The authors tested the association between
             prospectively collected measures of childhood violence
             victimization and cognitive functions in childhood,
             adolescence, and adulthood among 2,232 members of the U.K.
             E-Risk Study and 1,037 members of the New Zealand Dunedin
             Study who were followed up from birth until ages 18 and 38
             years, respectively. Multiple measures of victimization and
             cognition were used, and comparisons were made of cognitive
             scores for twins discordant for victimization.Individuals
             exposed to childhood victimization had pervasive impairments
             in clinically relevant cognitive functions, including
             general intelligence, executive function, processing speed,
             memory, perceptual reasoning, and verbal comprehension in
             adolescence and adulthood. However, the observed cognitive
             deficits in victimized individuals were largely explained by
             cognitive deficits that predated childhood victimization and
             by confounding genetic and environmental risks.Findings from
             two population-representative birth cohorts totaling more
             than 3,000 individuals and born 20 years and 20,000 km apart
             suggest that the association between childhood violence
             victimization and later cognition is largely noncausal, in
             contrast to conventional interpretations. These findings
             support the adoption of a more circumspect approach to
             causal inference in the neuroscience of stress. Clinically,
             cognitive deficits should be conceptualized as individual
             risk factors for victimization as well as potential
             complicating features during treatment.},
   Doi = {10.1176/appi.ajp.2016.16030333},
   Key = {fds324440}
}

@article{fds325849,
   Author = {Reuben, A and Caspi, A and Belsky, DW and Broadbent, J and Harrington,
             H and Sugden, K and Houts, RM and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Cognitive
             Function and Socioeconomic Status at Age 38 Years and With
             IQ Change and Socioeconomic Mobility Between Childhood and
             Adulthood.},
   Journal = {JAMA : the journal of the American Medical
             Association},
   Volume = {317},
   Number = {12},
   Pages = {1244-1251},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1001/jama.2017.1712},
   Abstract = {Many children in the United States and around the world are
             exposed to lead, a developmental neurotoxin. The long-term
             cognitive and socioeconomic consequences of lead exposure
             are uncertain.To test the hypothesis that childhood lead
             exposure is associated with cognitive function and
             socioeconomic status in adulthood and with changes in IQ and
             socioeconomic mobility between childhood and midlife.A
             prospective cohort study based on a population-representative
             1972-1973 birth cohort from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (until December 2012).Childhood
             lead exposure ascertained as blood lead levels measured at
             age 11 years. High blood lead levels were observed among
             children from all socioeconomic status levels in this
             cohort.The IQ (primary outcome) and indexes of Verbal
             Comprehension, Perceptual Reasoning, Working Memory, and
             Processing Speed (secondary outcomes) were assessed at age
             38 years using the Wechsler Adult Intelligence Scale-IV
             (WAIS-IV; IQ range, 40-160). Socioeconomic status (primary
             outcome) was assessed at age 38 years using the New Zealand
             Socioeconomic Index-2006 (NZSEI-06; range, 10 [lowest]-90
             [highest]).Of 1037 original participants, 1007 were alive at
             age 38 years, of whom 565 (56%) had been lead tested at age
             11 years (54% male; 93% white). Mean (SD) blood lead level
             at age 11 years was 10.99 (4.63) µg/dL. Among blood-tested
             participants included at age 38 years, mean WAIS-IV score
             was 101.16 (14.82) and mean NZSEI-06 score was 49.75
             (17.12). After adjusting for maternal IQ, childhood IQ, and
             childhood socioeconomic status, each 5-µg/dL higher level
             of blood lead in childhood was associated with a 1.61-point
             lower score (95% CI, -2.48 to -0.74) in adult IQ, a
             2.07-point lower score (95% CI, -3.14 to -1.01) in
             perceptual reasoning, and a 1.26-point lower score (95% CI,
             -2.38 to -0.14) in working memory. Associations of childhood
             blood lead level with deficits in verbal comprehension and
             processing speed were not statistically significant. After
             adjusting for confounders, each 5-µg/dL higher level of
             blood lead in childhood was associated with a 1.79-unit
             lower score (95% CI, -3.17 to -0.40) in socioeconomic
             status. An association between greater blood lead levels and
             a decline in IQ and socioeconomic status from childhood to
             adulthood was observed with 40% of the association with
             downward mobility mediated by cognitive decline from
             childhood.In this cohort born in New Zealand in 1972-1973,
             childhood lead exposure was associated with lower cognitive
             function and socioeconomic status at age 38 years and with
             declines in IQ and with downward social mobility. Childhood
             lead exposure may have long-term ramifications.},
   Doi = {10.1001/jama.2017.1712},
   Key = {fds325849}
}

@article{fds321664,
   Author = {Gregory, AM and Agnew-Blais, JC and Matthews, T and Moffitt, TE and Arseneault, L},
   Title = {ADHD and Sleep Quality: Longitudinal Analyses From Childhood
             to Early Adulthood in a Twin Cohort.},
   Journal = {Journal of Clinical Child & Adolescent Psychology},
   Volume = {46},
   Number = {2},
   Pages = {284-294},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1080/15374416.2016.1183499},
   Abstract = {Attention-deficit/hyperactivity disorder (ADHD) is
             associated with poor sleep quality, but there is more to
             learn about the longitudinal association and aetiology of
             this association. We investigated the following: (a) Is
             there an association between childhood ADHD and poor sleep
             quality in young adulthood? (b) Is this driven by the
             long-term effects of childhood ADHD or concurrent
             associations with ADHD in young adulthood? (c) To what
             extent do genetic and environmental influences explain the
             overlap between symptoms of ADHD and poor sleep quality?
             Participants were from the Environmental Risk Longitudinal
             Twin Study of 2,232 twin children born in the United Kingdom
             in 1994-1995. We ascertained ADHD diagnoses at ages 5, 7,
             10, 12, and 18. We assessed sleep quality using the
             Pittsburgh Sleep Quality Index at age 18. We used regression
             models to examine longitudinal associations and bivariate
             twin modelling to test genetic and environmental influences.
             Children with ADHD had poorer sleep quality in young
             adulthood, but only if their ADHD persisted. Adults with
             ADHD had more sleep problems than those without ADHD, over
             and above psychiatric comorbidity and maternal insomnia.
             ADHD and sleep problems in young adulthood were associated
             because of genetic (55%) and nonshared environmental
             influences (45%). Should ADHD remit, children with ADHD do
             not appear to have an increased risk of later sleep
             problems. Good quality sleep is important for multiple areas
             of functioning, and a better understanding of why adults
             with ADHD have poorer sleep quality will further the goal of
             improving treatments.},
   Doi = {10.1080/15374416.2016.1183499},
   Key = {fds321664}
}

@article{fds324773,
   Author = {Moffitt, TE and Belsky, DW and Danese, A and Poulton, R and Caspi,
             A},
   Title = {The Longitudinal Study of Aging in Human Young Adults:
             Knowledge Gaps and Research Agenda.},
   Journal = {Journals of Gerontology: Series A},
   Volume = {72},
   Number = {2},
   Pages = {210-215},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1093/gerona/glw191},
   Abstract = {To prevent onset of age-related diseases and physical and
             cognitive decline, interventions to slow human aging and
             extend health span must eventually be applied to people
             while they are still young and healthy. Yet most human aging
             research examines older adults, many with chronic disease,
             and little is known about aging in healthy young humans.This
             article explains how this knowledge gap is a barrier to
             extending health span and puts forward the case that
             geroscience should invest in researching the pace of aging
             in young adults. As one illustrative example, we describe an
             initial effort to study the pace of aging in a young-adult
             birth cohort by using repeated waves of biomarkers collected
             across the third and fourth decades to quantify the pace of
             coordinated physiological deterioration across multiple
             organ systems (eg, pulmonary, periodontal, cardiovascular,
             renal, hepatic, metabolic, and immune function).Findings
             provided proof of principle that it is possible to quantify
             individual variation in the pace of aging in young adults
             still free of age-related diseases.This article articulates
             research needs to improve longitudinal measurement of the
             pace of aging in young people, to pinpoint factors that slow
             or speed the pace of aging, to compare pace of aging against
             genomic clocks, to explain slow-aging young adults, and to
             apply pace of aging in preventive clinical trials of
             antiaging therapies. This article puts forward a research
             agenda to fill the knowledge gap concerning lifelong causes
             of aging.},
   Doi = {10.1093/gerona/glw191},
   Key = {fds324773}
}

@article{fds321672,
   Author = {Schaefer, JD and Caspi, A and Belsky, DW and Harrington, H and Houts, R and Horwood, LJ and Hussong, A and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Enduring mental health: Prevalence and prediction.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {2},
   Pages = {212-224},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1037/abn0000232},
   Abstract = {We review epidemiological evidence indicating that most
             people will develop a diagnosable mental disorder,
             suggesting that only a minority experience enduring mental
             health. This minority has received little empirical study,
             leaving the prevalence and predictors of enduring mental
             health unknown. We turn to the population-representative
             Dunedin cohort, followed from birth to midlife, to compare
             people never-diagnosed with mental disorder (N = 171; 17%
             prevalence) to those diagnosed at 1-2 study waves, the
             cohort mode (N = 409). Surprisingly, compared to this modal
             group, never-diagnosed Study members were not born into
             unusually well-to-do families, nor did their enduring mental
             health follow markedly sound physical health, or unusually
             high intelligence. Instead, they tended to have an
             advantageous temperament/personality style, and negligible
             family history of mental disorder. As adults, they report
             superior educational and occupational attainment, greater
             life satisfaction, and higher-quality relationships. Our
             findings draw attention to "enduring mental health" as a
             revealing psychological phenotype and suggest it deserves
             further study. (PsycINFO Database Record},
   Doi = {10.1037/abn0000232},
   Key = {fds321672}
}

@article{fds320829,
   Author = {Scult, MA and Paulli, AR and Mazure, ES and Moffitt, TE and Hariri, AR and Strauman, TJ},
   Title = {The association between cognitive function and subsequent
             depression: a systematic review and meta-analysis.},
   Journal = {Psychological Medicine},
   Volume = {47},
   Number = {1},
   Pages = {1-17},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1017/s0033291716002075},
   Abstract = {Despite a growing interest in understanding the cognitive
             deficits associated with major depressive disorder (MDD), it
             is largely unknown whether such deficits exist before
             disorder onset or how they might influence the severity of
             subsequent illness. The purpose of the present study was to
             conduct a systematic review and meta-analysis of
             longitudinal datasets to determine whether cognitive
             function acts as a predictor of later MDD diagnosis or
             change in depression symptoms. Eligible studies included
             longitudinal designs with baseline measures of cognitive
             functioning, and later unipolar MDD diagnosis or symptom
             assessment. The systematic review identified 29
             publications, representing 34 unique samples, and 121 749
             participants, that met the inclusion/exclusion criteria.
             Quantitative meta-analysis demonstrated that higher
             cognitive function was associated with decreased levels of
             subsequent depression (r = -0.088, 95% confidence interval.
             -0.121 to -0.054, p < 0.001). However, sensitivity analyses
             revealed that this association is likely driven by
             concurrent depression symptoms at the time of cognitive
             assessment. Our review and meta-analysis indicate that the
             association between lower cognitive function and later
             depression is confounded by the presence of contemporaneous
             depression symptoms at the time of cognitive assessment.
             Thus, cognitive deficits predicting MDD likely represent
             deleterious effects of subclinical depression symptoms on
             performance rather than premorbid risk factors for
             disorder.},
   Doi = {10.1017/s0033291716002075},
   Key = {fds320829}
}


%% Monge, Zachary A.   
@article{fds327867,
   Author = {Monge, ZA and Wing, EA and Stokes, J and Cabeza, R},
   Title = {Search and recovery of autobiographical and laboratory
             memories: Shared and distinct neural components.},
   Journal = {Neuropsychologia},
   Volume = {110},
   Pages = {44-54},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2017.07.030},
   Abstract = {Functional neuroimaging evidence suggests that there are
             differences in the neural correlates of episodic memory for
             laboratory stimuli (laboratory memory) and for events from
             one's own life (autobiographical memory). However, this
             evidence is scarce and often confounded with differences in
             memory testing procedures. Here, we directly compared the
             neural mechanisms underlying the search and recovery of
             autobiographical and laboratory memories while minimizing
             testing differences. Before scanning, participants completed
             a laboratory memory encoding task in which they studied
             four-word "chains" spread across three word pairs. During
             scanning, participants completed a laboratory memory
             retrieval task, in which they recalled the word chains, and
             an autobiographical memory retrieval task, in which they
             recalled specific personal events associated with word cues.
             Importantly, response times were similar in the two tasks,
             allowing for a direct comparison of the activation time
             courses. We found that during memory search (searching for
             the memory target), similar brain regions were activated
             during both the autobiographical and laboratory tasks,
             whereas during memory recovery (accessing the memory traces;
             i.e., ecphory), clear differences emerged: regions of the
             default mode network (DMN) were activated greater during
             autobiographical than laboratory memory, whereas the
             bilateral superior parietal lobules were activated greater
             during laboratory than autobiographical memory. Also,
             multivariate functional connectivity analyses revealed that
             regardless of memory stage, the DMN and ventral attention
             network exhibited a more integrated topology in the
             functional network underlying autobiographical (vs.
             laboratory) memory retrieval, whereas the fronto-parietal
             task control network exhibited a more integrated topology in
             the functional network underlying laboratory (vs.
             autobiographical) memory retrieval. These findings further
             characterize the shared and distinct neural components
             underlying autobiographical and laboratory memories, and
             suggest that differences in autobiographical vs. laboratory
             memory brain activation previously reported in the
             literature reflect memory recovery rather than search
             differences.},
   Doi = {10.1016/j.neuropsychologia.2017.07.030},
   Key = {fds327867}
}

@article{fds328109,
   Author = {Regier, PS and Monge, ZA and Franklin, TR and Wetherill, RR and Teitelman, A and Jagannathan, K and Suh, JJ and Wang, Z and Young, KA and Gawrysiak, M and Langleben, DD and Kampman, KM and O'Brien, CP and Childress, AR},
   Title = {Emotional, physical and sexual abuse are associated with a
             heightened limbic response to cocaine cues},
   Journal = {Addiction Biology},
   Volume = {22},
   Number = {6},
   Pages = {1768-1777},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1111/adb.12445},
   Doi = {10.1111/adb.12445},
   Key = {fds328109}
}

@article{fds327868,
   Author = {Monge, ZA and Geib, BR and Siciliano, RE and Packard, LE and Tallman,
             CW and Madden, DJ},
   Title = {Functional modular architecture underlying attentional
             control in aging.},
   Journal = {NeuroImage},
   Volume = {155},
   Pages = {257-270},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2017.05.002},
   Abstract = {Previous research suggests that age-related differences in
             attention reflect the interaction of top-down and bottom-up
             processes, but the cognitive and neural mechanisms
             underlying this interaction remain an active area of
             research. Here, within a sample of community-dwelling adults
             19-78 years of age, we used diffusion reaction time (RT)
             modeling and multivariate functional connectivity to
             investigate the behavioral components and whole-brain
             functional networks, respectively, underlying bottom-up and
             top-down attentional processes during conjunction visual
             search. During functional MRI scanning, participants
             completed a conjunction visual search task in which each
             display contained one item that was larger than the other
             items (i.e., a size singleton) but was not informative
             regarding target identity. This design allowed us to examine
             in the RT components and functional network measures the
             influence of (a) additional bottom-up guidance when the
             target served as the size singleton, relative to when the
             distractor served as the size singleton (i.e., size
             singleton effect) and (b) top-down processes during target
             detection (i.e., target detection effect; target present vs.
             absent trials). We found that the size sin