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Psychology and Neuroscience : Publications since January 2017

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%% Adcock, Rachel A.   
@article{fds319596,
   Author = {Scult, MA and Knodt, AR and Hanson, JL and Ryoo, M and Adcock, RA and Hariri, AR and Strauman, TJ},
   Title = {Individual differences in regulatory focus predict neural
             response to reward.},
   Journal = {Social Neuroscience},
   Volume = {12},
   Number = {4},
   Pages = {419-429},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1080/17470919.2016.1178170},
   Abstract = {Although goal pursuit is related to both functioning of the
             brain's reward circuits and psychological factors, the
             literatures surrounding these concepts have often been
             separate. Here, we use the psychological construct of
             regulatory focus to investigate individual differences in
             neural response to reward. Regulatory focus theory proposes
             two motivational orientations for personal goal pursuit: (1)
             promotion, associated with sensitivity to potential gain,
             and (2) prevention, associated with sensitivity to potential
             loss. The monetary incentive delay task was used to
             manipulate reward circuit function, along with instructional
             framing corresponding to promotion and prevention in a
             within-subject design. We observed that the more promotion
             oriented an individual was, the lower their ventral striatum
             response to gain cues. Follow-up analyses revealed that
             greater promotion orientation was associated with decreased
             ventral striatum response even to no-value cues, suggesting
             that promotion orientation may be associated with relatively
             hypoactive reward system function. The findings are also
             likely to represent an interaction between the cognitive and
             motivational characteristics of the promotion system with
             the task demands. Prevention orientation did not correlate
             with ventral striatum response to gain cues, supporting the
             discriminant validity of regulatory focus theory. The
             results highlight a dynamic association between individual
             differences in self-regulation and reward system
             function.},
   Doi = {10.1080/17470919.2016.1178170},
   Key = {fds319596}
}

@article{fds310030,
   Author = {Murty, VP and Ballard, IC and Adcock, RA},
   Title = {Hippocampus and Prefrontal Cortex Predict Distinct
             Timescales of Activation in the Human Ventral Tegmental
             Area.},
   Journal = {Cerebral Cortex (New York, N.Y. : 1991)},
   Volume = {27},
   Number = {2},
   Pages = {1660-1669},
   Year = {2017},
   Month = {February},
   ISSN = {1047-3211},
   url = {http://dx.doi.org/10.1093/cercor/bhw005},
   Abstract = {The mesolimbic dopamine system contributes to a remarkable
             variety of behaviors at multiple timescales. Midbrain
             neurons have fast and slow signaling components, and
             specific afferent systems, such as the hippocampus (HPC) and
             prefrontal cortex (PFC), have been demonstrated to drive
             these components in anesthetized animals. Whether these
             interactions exist during behavior, however, is unknown. To
             address this question, we developed a novel analysis of
             human functional magnetic resonance imaging data that fits
             models of network excitation and inhibition on ventral
             tegmental area (VTA) activation. We show that specific
             afferent systems predict distinct temporal components of
             midbrain VTA signal. We found that PFC, but not HPC,
             positively predicted transient, event-evoked VTA activation.
             In contrast, HPC, but not PFC, positively predicted slow
             shifts in VTA baseline variability. Thus, unique functional
             contributions of afferent systems to VTA physiology are
             detectable at the network level in behaving humans. The
             findings support models of dopamine function in which
             dissociable neural circuits support different aspects of
             motivated behavior via active regulation of tonic and phasic
             signals.},
   Doi = {10.1093/cercor/bhw005},
   Key = {fds310030}
}


%% Appelbaum, Lawrence G.   
@article{fds336042,
   Author = {Teel, EF and Marshall, SW and Appelbaum, LG and Battaglini, CL and Carneiro, KA and Guskiewicz, KM and Register-Mihalik, JK and Mihalik,
             JP},
   Title = {A Randomized Controlled Trial Investigating the Feasibility
             and Adherence to an Aerobic Training Program in Healthy
             Individuals.},
   Journal = {Journal of Sport Rehabilitation},
   Pages = {1-22},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1123/jsr.2018-0007},
   Abstract = {Concussion management is moving from passive rest strategies
             to active interventions, including aerobic exercise therapy.
             Little information is available regarding the feasibility
             and adherence of these programs.To determine whether an
             aerobic exercise training program, intended for
             rehabilitation in persons with concussion, is feasible.
             Healthy non-concussed subjects were studied in this Phase I
             trial.Phase 1 parallel-group randomized controlled trial in
             a sample of healthy (non-concussed), recreationally active
             university students.Laboratory.40 healthy, university
             students.Participants were equally randomized into acute
             concussion therapy intervention (ACTIVE) training or
             non-training groups. All participants completed maximal
             cardiopulmonary exercise tests (CPETs) on a stationary cycle
             ergometer at two test sessions approximately 14 days apart.
             During this 2-week study period, ACTIVE training
             participants completed six 30-minute cycling sessions,
             progressing from 60-80% of the participant's individualized
             maximal oxygen consumption (VO2max). A subset of
             participants (NACTIVE=12, Nnon-training=11) wore physical
             activity monitors throughout the 2-week study period.Study
             protocol and randomization effectiveness, exercise safety
             and adherence, and progressive intensity of the ACTIVE
             training procedures.No adverse events occurred during any
             exercise sessions. Twelve ACTIVE training participants (60%)
             completed all training sessions and every participant
             completed at least four sessions. Heart rate increased
             throughout the training period (p<0.001), but symptom
             changes and training adherence remained stable despite the
             progressively increasing workload. ACTIVE training
             participants completed approximately 30 additional minutes
             of physical activity on training sessions days, although not
             statistically significant (p=0.20).University aged students
             were adherent to the ACTIVE training protocol. Future
             research should investigate the safety and feasibility of
             aerobic training programs in acutely concussed individuals
             to determine their appropriateness as a clinical
             rehabilitation strategy.},
   Doi = {10.1123/jsr.2018-0007},
   Key = {fds336042}
}

@article{fds332131,
   Author = {Burris, K and Vittetoe, K and Ramger, B and Suresh, S and Tokdar, ST and Reiter, JP and Appelbaum, LG},
   Title = {Sensorimotor abilities predict on-field performance in
             professional baseball.},
   Journal = {Scientific Reports},
   Volume = {8},
   Number = {1},
   Pages = {116},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1038/s41598-017-18565-7},
   Abstract = {Baseball players must be able to see and react in an
             instant, yet it is hotly debated whether superior
             performance is associated with superior sensorimotor
             abilities. In this study, we compare sensorimotor abilities,
             measured through 8 psychomotor tasks comprising the Nike
             Sensory Station assessment battery, and game statistics in a
             sample of 252 professional baseball players to evaluate the
             links between sensorimotor skills and on-field performance.
             For this purpose, we develop a series of Bayesian
             hierarchical latent variable models enabling us to compare
             statistics across professional baseball leagues. Within this
             framework, we find that sensorimotor abilities are
             significant predictors of on-base percentage, walk rate and
             strikeout rate, accounting for age, position, and league. We
             find no such relationship for either slugging percentage or
             fielder-independent pitching. The pattern of results
             suggests performance contributions from both visual-sensory
             and visual-motor abilities and indicates that sensorimotor
             screenings may be useful for player scouting.},
   Doi = {10.1038/s41598-017-18565-7},
   Key = {fds332131}
}

@article{fds336043,
   Author = {Appelbaum, LG and Erickson, G},
   Title = {Sports vision training: A review of the state-of-the-art in
             digital training techniques},
   Journal = {International Review of Sport and Exercise
             Psychology},
   Volume = {11},
   Number = {1},
   Pages = {160-189},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1080/1750984X.2016.1266376},
   Doi = {10.1080/1750984X.2016.1266376},
   Key = {fds336043}
}

@article{fds332130,
   Author = {Rao, HM and Khanna, R and Zielinski, DJ and Lu, Y and Clements, JM and Potter, ND and Sommer, MA and Kopper, R and Appelbaum,
             LG},
   Title = {Sensorimotor Learning during a Marksmanship Task in
             Immersive Virtual Reality.},
   Journal = {Frontiers in Psychology},
   Volume = {9},
   Number = {58},
   Pages = {58},
   Publisher = {Frontiers Media},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fpsyg.2018.00058},
   Abstract = {Sensorimotor learning refers to improvements that occur
             through practice in the performance of sensory-guided motor
             behaviors. Leveraging novel technical capabilities of an
             immersive virtual environment, we probed the component
             kinematic processes that mediate sensorimotor learning.
             Twenty naïve subjects performed a simulated marksmanship
             task modeled after Olympic Trap Shooting standards. We
             measured movement kinematics and shooting performance as
             participants practiced 350 trials while receiving
             trial-by-trial feedback about shooting success.
             Spatiotemporal analysis of motion tracking elucidated the
             ballistic and refinement phases of hand movements. We found
             systematic changes in movement kinematics that accompanied
             improvements in shot accuracy during training, though
             reaction and response times did not change over blocks. In
             particular, we observed longer, slower, and more precise
             ballistic movements that replaced effort spent on
             corrections and refinement. Collectively, these results
             leverage developments in immersive virtual reality
             technology to quantify and compare the kinematics of
             movement during early learning of full-body sensorimotor
             orienting.},
   Doi = {10.3389/fpsyg.2018.00058},
   Key = {fds332130}
}

@article{fds324820,
   Author = {Klemish, D and Ramger, B and Vittetoe, K and Reiter, JP and Tokdar, ST and Appelbaum, LG},
   Title = {Visual abilities distinguish pitchers from hitters in
             professional baseball.},
   Journal = {Journal of Sports Sciences},
   Volume = {36},
   Number = {2},
   Pages = {171-179},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1080/02640414.2017.1288296},
   Abstract = {This study aimed to evaluate the possibility that
             differences in sensorimotor abilities exist between hitters
             and pitchers in a large cohort of baseball players of
             varying levels of experience. Secondary data analysis was
             performed on 9 sensorimotor tasks comprising the Nike
             Sensory Station assessment battery. Bayesian hierarchical
             regression modelling was applied to test for differences
             between pitchers and hitters in data from 566 baseball
             players (112 high school, 85 college, 369 professional)
             collected at 20 testing centres. Explanatory variables
             including height, handedness, eye dominance, concussion
             history, and player position were modelled along with age
             curves using basis regression splines. Regression analyses
             revealed better performance for hitters relative to pitchers
             at the professional level in the visual clarity and depth
             perception tasks, but these differences did not exist at the
             high school or college levels. No significant differences
             were observed in the other 7 measures of sensorimotor
             capabilities included in the test battery, and no systematic
             biases were found between the testing centres. These
             findings, indicating that professional-level hitters have
             better visual acuity and depth perception than
             professional-level pitchers, affirm the notion that highly
             experienced athletes have differing perceptual skills.
             Findings are discussed in relation to deliberate practice
             theory.},
   Doi = {10.1080/02640414.2017.1288296},
   Key = {fds324820}
}

@misc{fds330473,
   Author = {Jack, J and Appelbaum, LG and Beam, E and Moody, J and Huettel,
             SA},
   Title = {Mapping rhetorical topologies in cognitive
             neuroscience},
   Pages = {125-150},
   Booktitle = {Topologies as Techniques for a Post-Critical
             Rhetoric},
   Year = {2017},
   Month = {January},
   ISBN = {9783319512686},
   url = {http://dx.doi.org/10.1007/978-3-319-51268-6_7},
   Abstract = {© The Author(s) 2017. Many tools that neuroscientists use
             to trace the complex topography of the human brain draw on
             the neuroscience literature to yield “metanalyses” or
             “syntheses of data.” These approaches conflate
             rhetorical connections in the literature with physical
             connections in the brain. By contrast, the model presented
             in this chapter seeks not a topography of the brain but a
             topology of neuroscience. A social network analysis of
             titles and abstracts for cognitive neuroscience articles
             yields a topology of brain regions and functions. This map
             can help researchers identify underresearched areas (e.g.,
             the thalamus) or areas that are oversaturated (e.g., the
             amygdala). The map also helps researchers identify
             subdisciplines, such as “neuroeconomics,” that have not
             yet integrated with the broader field–“islands” where
             rhetorical work could yield benefits.},
   Doi = {10.1007/978-3-319-51268-6_7},
   Key = {fds330473}
}

@article{fds320202,
   Author = {Devyatko, D and Appelbaum, LG and Mitroff, SR},
   Title = {A Common Mechanism for Perceptual Reversals in
             Motion-Induced Blindness, the Troxler Effect, and Perceptual
             Filling-In.},
   Journal = {Perception},
   Volume = {46},
   Number = {1},
   Pages = {50-77},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1177/0301006616672577},
   Abstract = {Several striking visual phenomena involve a physically
             present stimulus that alternates between being perceived and
             being "invisible." For example, motion-induced blindness,
             the Troxler effect, and perceptual filling-in all consist of
             subjective alternations where an item repeatedly changes
             from being seen to unseen. In the present study, we explored
             whether these three specific visual phenomena share any
             commonalities in their alternation rates and patterns to
             better understand the mechanisms of each. Data from 69
             individuals revealed moderate to strong correlations across
             the three phenomena for the number of perceptual
             disappearances and the accumulated duration of the
             disappearances. Importantly, these effects were not
             correlated with eye movement patterns (saccades) assessed
             through eye tracking, differences in motion sensitivity as
             indexed by dot coherence and speed perception thresholds, or
             simple reaction time abilities. Principal component analyses
             revealed a single component that explained 67% of the
             variance for the number of perceptual reversals and 60% for
             the accumulated duration of the disappearances. The temporal
             dynamics of illusory disappearances was also compared for
             each phenomenon, and normalized durations of disappearances
             were well fit by a gamma distribution with similar shape
             parameters for each phenomenon, suggesting that they may be
             driven by a single oscillatory mechanism.},
   Doi = {10.1177/0301006616672577},
   Key = {fds320202}
}


%% Asher, Steven R.   
@article{fds332880,
   Author = {McDonald, KL and Asher, SR},
   Title = {Pacifists and Revenge-Seekers in Response to Unambiguous
             Peer Provocation.},
   Journal = {Journal of Youth and Adolescence},
   Volume = {47},
   Number = {9},
   Pages = {1907-1925},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1007/s10964-017-0767-4},
   Abstract = {In order to better understand why some children retaliate
             when they feel provoked and others do not, the present study
             identified "pacifistically-oriented" children who made
             negative interpretations in response to unambiguous
             provocations, yet did not endorse revenge goals, and
             compared them to "revenge-seeking" children who also made
             negative interpretations but did endorse revenge goals.
             Groups were identified based on seventh graders'
             (N = 367; 54.77% male; 22.89% racial/ethnic minority)
             responses to hypothetical situations in which a peer
             excluded and insulted them. Comparing these groups revealed
             that Pacifists endorsed relationship-maintaining goals and
             emotion regulation goals more highly than Revenge-Seekers.
             Revenge-Seekers reported more anger and endorsed beliefs
             about negative reciprocity and aggression being legitimate
             more highly than Pacifists. Additionally, Revenge-Seekers
             were more disrespect sensitive than were Pacifists, based on
             a measure of vigilance for signs of disrespect and
             expectations that others would disrespect them. Together
             these findings point to social-cognitive and emotion-related
             processes that may inhibit revenge-seeking in unambiguous
             provocation situations, even when children interpret the
             peer's behavior quite negatively.},
   Doi = {10.1007/s10964-017-0767-4},
   Key = {fds332880}
}

@article{fds322939,
   Author = {Rose, AJ and Asher, SR},
   Title = {The Social Tasks of Friendship: Do Boys and Girls Excel in
             Different Tasks?},
   Journal = {Child Development Perspectives},
   Volume = {11},
   Number = {1},
   Pages = {3-8},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1111/cdep.12214},
   Doi = {10.1111/cdep.12214},
   Key = {fds322939}
}


%% Bejjani, Christina   
@article{fds332947,
   Author = {Bejjani, C and Zhang, Z and Egner, T},
   Title = {Control by association: Transfer of implicitly primed
             attentional states across linked stimuli.},
   Journal = {Psychonomic Bulletin & Review},
   Volume = {25},
   Number = {2},
   Pages = {617-626},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.3758/s13423-018-1445-6},
   Abstract = {Although cognitive control has traditionally been viewed in
             opposition to associative learning, recent studies show that
             people can learn to link particular stimuli with specific
             cognitive control states (e.g., high attentional
             selectivity). Here, we tested whether such learned
             stimulus-control associations can transfer across
             paired-associates. In the Stimulus-Stimulus (S-S)
             Association phase, specific face or house images repeatedly
             preceded the presentation of particular scene stimuli,
             creating paired face/house-scene associates in memory. The
             Stimulus-Control (S-C) Association phase then associated
             these scenes with different attentional control states by
             probabilistically biasing specific scenes to mostly precede
             either congruent or incongruent trials in a Stroop task.
             Finally, in the Stimulus-Control Transfer (S-CT) phase, the
             faces and houses from the S-S phase preceded Stroop trials
             but were not predictive of congruency, testing whether
             stimulus-control associations would transfer from scenes to
             their associated face/house stimuli. In Experiments 1 and 3,
             we found that learned implicit stimulus-control associations
             could transfer across closely linked cues, and in Experiment
             2, we showed that this transfer depended on the memory
             associations formed in the S-S phase. While this form of
             transfer learning has previously been demonstrated for
             stimulus-reward associations, the present study provides the
             first evidence for the associative transfer of
             stimulus-control associations across arbitrarily linked
             stimuli. This work demonstrates how people can learn to
             implicitly adapt their processing strategies in a flexible
             context-dependent manner and establishes a novel learning
             mechanism supporting the generalization of cognitive
             control.},
   Doi = {10.3758/s13423-018-1445-6},
   Key = {fds332947}
}

@article{fds333711,
   Author = {DiMenichi, BC and Lempert, KM and Bejjani, C and Tricomi,
             E},
   Title = {Writing about past failures attenuates cortisol responses
             and sustained attention deficits following psychosocial
             stress},
   Journal = {Frontiers in Behavioral Neuroscience},
   Volume = {12},
   Number = {45},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.3389/fnbeh.2018.00045},
   Doi = {10.3389/fnbeh.2018.00045},
   Key = {fds333711}
}


%% Belsky, Daniel W   
@article{fds335815,
   Author = {Gaydosh, L and Belsky, DW and Domingue, BW and Boardman, JD and Harris,
             KM},
   Title = {Father Absence and Accelerated Reproductive Development in
             Non-Hispanic White Women in the United States},
   Journal = {Demography},
   Volume = {55},
   Number = {4},
   Pages = {1245-1267},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1007/s13524-018-0696-1},
   Doi = {10.1007/s13524-018-0696-1},
   Key = {fds335815}
}

@article{fds335816,
   Author = {Belsky, DW and Domingue, BW and Wedow, R and Arseneault, L and Boardman,
             JD and Caspi, A and Conley, D and Fletcher, JM and Freese, J and Herd, P and Moffitt, TE and Poulton, R and Sicinski, K and Wertz, J and Harris,
             KM},
   Title = {Genetic analysis of social-class mobility in five
             longitudinal studies.},
   Journal = {Proceedings of the National Academy of Sciences of the
             United States of America},
   Volume = {115},
   Number = {31},
   Pages = {E7275-E7284},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1073/pnas.1801238115},
   Abstract = {A summary genetic measure, called a "polygenic score,"
             derived from a genome-wide association study (GWAS) of
             education can modestly predict a person's educational and
             economic success. This prediction could signal a biological
             mechanism: Education-linked genetics could encode
             characteristics that help people get ahead in life.
             Alternatively, prediction could reflect social history:
             People from well-off families might stay well-off for social
             reasons, and these families might also look alike
             genetically. A key test to distinguish biological mechanism
             from social history is if people with higher education
             polygenic scores tend to climb the social ladder beyond
             their parents' position. Upward mobility would indicate
             education-linked genetics encodes characteristics that
             foster success. We tested if education-linked polygenic
             scores predicted social mobility in >20,000 individuals in
             five longitudinal studies in the United States, Britain, and
             New Zealand. Participants with higher polygenic scores
             achieved more education and career success and accumulated
             more wealth. However, they also tended to come from
             better-off families. In the key test, participants with
             higher polygenic scores tended to be upwardly mobile
             compared with their parents. Moreover, in sibling-difference
             analysis, the sibling with the higher polygenic score was
             more upwardly mobile. Thus, education GWAS discoveries are
             not mere correlates of privilege; they influence social
             mobility within a life. Additional analyses revealed that a
             mother's polygenic score predicted her child's attainment
             over and above the child's own polygenic score, suggesting
             parents' genetics can also affect their children's
             attainment through environmental pathways. Education GWAS
             discoveries affect socioeconomic attainment through
             influence on individuals' family-of-origin environments and
             their social mobility.},
   Doi = {10.1073/pnas.1801238115},
   Key = {fds335816}
}

@article{fds335817,
   Author = {Belsky, DW},
   Title = {LIFE-COURSE LONGITUDINAL STUDIES ARE NEEDED TO ADVANCE
             INTEGRATION OF GENOMICS AND SOCIAL EPIDEMIOLOGY},
   Journal = {American Journal of Epidemiology},
   Volume = {187},
   Number = {6},
   Pages = {1337-1338},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1093/aje/kwy060},
   Doi = {10.1093/aje/kwy060},
   Key = {fds335817}
}

@article{fds335818,
   Author = {Belsky, DW and Moffitt, TE and Cohen, AA and Corcoran, DL and Levine,
             ME and Prinz, JA and Schaefer, J and Sugden, K and Williams, B and Poulton,
             R and Caspi, A},
   Title = {Eleven Telomere, Epigenetic Clock, and Biomarker-Composite
             Quantifications of Biological Aging: Do They Measure the
             Same Thing?},
   Journal = {American Journal of Epidemiology},
   Volume = {187},
   Number = {6},
   Pages = {1220-1230},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1093/aje/kwx346},
   Abstract = {The geroscience hypothesis posits that therapies to slow
             biological processes of aging can prevent disease and extend
             healthy years of life. To test such "geroprotective"
             therapies in humans, outcome measures are needed that can
             assess extension of disease-free life span. This need has
             spurred development of different methods to quantify
             biological aging. But different methods have not been
             systematically compared in the same humans. We implemented 7
             methods to quantify biological aging using repeated-measures
             physiological and genomic data in 964 middle-aged humans in
             the Dunedin Study (New Zealand; persons born 1972-1973). We
             studied 11 measures in total: telomere-length and erosion, 3
             epigenetic-clocks and their ticking rates, and 3
             biomarker-composites. Contrary to expectation, we found low
             agreement between different measures of biological aging. We
             next compared associations between biological aging measures
             and outcomes that geroprotective therapies seek to modify:
             physical functioning, cognitive decline, and subjective
             signs of aging, including aged facial appearance. The
             71-cytosine-phosphate-guanine epigenetic clock and biomarker
             composites were consistently related to these aging-related
             outcomes. However, effect sizes were modest. Results
             suggested that various proposed approaches to quantifying
             biological aging may not measure the same aspects of the
             aging process. Further systematic evaluation and refinement
             of measures of biological aging is needed to furnish
             outcomes for geroprotector trials.},
   Doi = {10.1093/aje/kwx346},
   Key = {fds335818}
}

@article{fds335819,
   Author = {Marsman, D and Belsky, DW and Gregori, D and Johnson, MA and Low Dog and T and Meydani, S and Pigat, S and Sadana, R and Shao, A and Griffiths,
             JC},
   Title = {Healthy ageing: the natural consequences of good
             nutrition—a conference report},
   Journal = {European Journal of Nutrition},
   Volume = {57},
   Number = {S2},
   Pages = {15-34},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s00394-018-1723-0},
   Abstract = {© 2018 The Author(s) Many countries are witnessing a marked
             increase in longevity and with this increased lifespan and
             the desire for healthy ageing, many, however, suffer from
             the opposite including mental and physical deterioration,
             lost productivity and quality of life, and increased medical
             costs. While adequate nutrition is fundamental for good
             health, it remains unclear what impact various dietary
             interventions may have on prolonging good quality of life.
             Studies which span age, geography and income all suggest
             that access to quality foods, host immunity and response to
             inflammation/infections, impaired senses (i.e., sight,
             taste, smell) or mobility are all factors which can limit
             intake or increase the body’s need for specific
             micronutrients. New clinical studies of healthy ageing are
             needed and quantitative biomarkers are an essential
             component, particularly tools which can measure improvements
             in physiological integrity throughout life, thought to be a
             primary contributor to a long and productive life (a healthy
             “lifespan”). A framework for progress has recently been
             proposed in a WHO report which takes a broad,
             person-centered focus on healthy ageing, emphasizing the
             need to better understand an individual’s intrinsic
             capacity, their functional abilities at various life stages,
             and the impact by mental, and physical health, and the
             environments they inhabit.},
   Doi = {10.1007/s00394-018-1723-0},
   Key = {fds335819}
}

@article{fds335821,
   Author = {Marzi, SJ and Sugden, K and Arseneault, L and Belsky, DW and Burrage, J and Corcoran, DL and Danese, A and Fisher, HL and Hannon, E and Moffitt, TE and Odgers, CL and Pariante, C and Poulton, R and Williams, BS and Wong,
             CCY and Mill, J and Caspi, A},
   Title = {Analysis of DNA Methylation in Young People: Limited
             Evidence for an Association Between Victimization Stress and
             Epigenetic Variation in Blood.},
   Journal = {The American Journal of Psychiatry},
   Volume = {175},
   Number = {6},
   Pages = {517-529},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.17060693},
   Abstract = {DNA methylation has been proposed as an epigenetic mechanism
             by which early-life experiences become "embedded" in the
             genome and alter transcriptional processes to compromise
             health. The authors sought to investigate whether early-life
             victimization stress is associated with genome-wide DNA
             methylation.The authors tested the hypothesis that
             victimization is associated with DNA methylation in the
             Environmental Risk (E-Risk) Longitudinal Study, a nationally
             representative 1994-1995 birth cohort of 2,232 twins born in
             England and Wales and assessed at ages 5, 7, 10, 12, and 18
             years. Multiple forms of victimization were ascertained in
             childhood and adolescence (including physical, sexual, and
             emotional abuse; neglect; exposure to intimate-partner
             violence; bullying; cyber-victimization; and
             crime).Epigenome-wide analyses of polyvictimization across
             childhood and adolescence revealed few significant
             associations with DNA methylation in peripheral blood at age
             18, but these analyses were confounded by tobacco smoking
             and/or did not survive co-twin control tests. Secondary
             analyses of specific forms of victimization revealed sparse
             associations with DNA methylation that did not replicate
             across different operationalizations of the same putative
             victimization experience. Hypothesis-driven analyses of six
             candidate genes in the stress response (NR3C1, FKBP5, BDNF,
             AVP, CRHR1, SLC6A4) did not reveal predicted associations
             with DNA methylation in probes annotated to these
             genes.Findings from this epidemiological analysis of the
             epigenetic effects of early-life stress do not support the
             hypothesis of robust changes in DNA methylation in
             victimized young people. We need to come to terms with the
             possibility that epigenetic epidemiology is not yet well
             matched to experimental, nonhuman models in uncovering the
             biological embedding of stress.},
   Doi = {10.1176/appi.ajp.2017.17060693},
   Key = {fds335821}
}

@article{fds335820,
   Author = {Rasmussen, LJH and Moffitt, TE and Eugen-Olsen, J and Belsky, DW and Danese, A and Harrington, H and Houts, RM and Poulton, R and Sugden, K and Williams, B and Caspi, A},
   Title = {Cumulative childhood risk is associated with a new measure
             of chronic inflammation in adulthood.},
   Journal = {Journal of Child Psychology and Psychiatry, and Allied
             Disciplines},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1111/jcpp.12928},
   Abstract = {Childhood risk factors are associated with elevated
             inflammatory biomarkers in adulthood, but it is unknown
             whether these risk factors are associated with increased
             adult levels of the chronic inflammation marker soluble
             urokinase plasminogen activator receptor (suPAR). We aimed
             to test the hypothesis that childhood exposure to risk
             factors for adult disease is associated with elevated suPAR
             in adulthood and to compare suPAR with the oft-reported
             inflammatory biomarker C-reactive protein (CRP).Prospective
             study of a population-representative 1972-1973 birth cohort;
             the Dunedin Multidisciplinary Health and Development Study
             observed participants to age 38 years. Main childhood
             predictors were poor health, socioeconomic disadvantage,
             adverse childhood experiences (ACEs), low IQ, and poor
             self-control. Main adult outcomes were adulthood
             inflammation measured as suPAR and high-sensitivity CRP
             (hsCRP).Participants with available plasma samples at age 38
             were included (N = 837, 50.5% male). suPAR (mean
             2.40 ng/ml; SD 0.91) was positively correlated with hsCRP
             (r 0.15, p < .001). After controlling for sex, body mass
             index (BMI), and smoking, children who experienced more
             ACEs, lower IQ, or had poorer self-control showed elevated
             adult suPAR. When the five childhood risks were aggregated
             into a Cumulative Childhood Risk index, and controlling for
             sex, BMI, and smoking, Cumulative Childhood Risk was
             associated with higher suPAR (b 0.10; SE 0.03; p = .002).
             Cumulative Childhood Risk predicted elevated suPAR, after
             controlling for hsCRP (b 0.18; SE 0.03; p < .001).Exposure
             to more childhood risk factors was associated with higher
             suPAR levels, independent of CRP. suPAR is a useful addition
             to studies connecting childhood risk to adult inflammatory
             burden.},
   Doi = {10.1111/jcpp.12928},
   Key = {fds335820}
}

@article{fds333731,
   Author = {Wertz, J and Caspi, A and Belsky, DW and Beckley, AL and Arseneault, L and Barnes, JC and Corcoran, DL and Hogan, S and Houts, RM and Morgan, N and Odgers, CL and Prinz, JA and Sugden, K and Williams, BS and Poulton, R and Moffitt, TE},
   Title = {Genetics and Crime: Integrating New Genomic Discoveries Into
             Psychological Research About Antisocial Behavior.},
   Journal = {Psychological Science},
   Volume = {29},
   Number = {5},
   Pages = {791-803},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1177/0956797617744542},
   Abstract = {Drawing on psychological and sociological theories of crime
             causation, we tested the hypothesis that genetic risk for
             low educational attainment (assessed via a genome-wide
             polygenic score) is associated with criminal offending. We
             further tested hypotheses of how polygenic risk relates to
             the development of antisocial behavior from childhood
             through adulthood. Across the Dunedin and Environmental Risk
             (E-Risk) birth cohorts of individuals growing up 20 years
             and 20,000 kilometers apart, education polygenic scores
             predicted risk of a criminal record with modest effects.
             Polygenic risk manifested during primary schooling in lower
             cognitive abilities, lower self-control, academic
             difficulties, and truancy, and it was associated with a
             life-course-persistent pattern of antisocial behavior that
             onsets in childhood and persists into adulthood. Crime is
             central in the nature-nurture debate, and findings reported
             here demonstrate how molecular-genetic discoveries can be
             incorporated into established theories of antisocial
             behavior. They also suggest that improving school
             experiences might prevent genetic influences on crime from
             unfolding.},
   Doi = {10.1177/0956797617744542},
   Key = {fds333731}
}

@article{fds332056,
   Author = {Domingue, BW and Belsky, DW and Fletcher, JM and Conley, D and Boardman,
             JD and Harris, KM},
   Title = {The social genome of friends and schoolmates in the National
             Longitudinal Study of Adolescent to Adult
             Health.},
   Journal = {Proceedings of the National Academy of Sciences of the
             United States of America},
   Volume = {115},
   Number = {4},
   Pages = {702-707},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1073/pnas.1711803115},
   Abstract = {Humans tend to form social relationships with others who
             resemble them. Whether this sorting of like with like arises
             from historical patterns of migration, meso-level social
             structures in modern society, or individual-level selection
             of similar peers remains unsettled. Recent research has
             evaluated the possibility that unobserved genotypes may play
             an important role in the creation of homophilous
             relationships. We extend this work by using data from 5,500
             adolescents from the National Longitudinal Study of
             Adolescent to Adult Health (Add Health) to examine genetic
             similarities among pairs of friends. Although there is some
             evidence that friends have correlated genotypes, both at the
             whole-genome level as well as at trait-associated loci (via
             polygenic scores), further analysis suggests that meso-level
             forces, such as school assignment, are a principal source of
             genetic similarity between friends. We also observe apparent
             social-genetic effects in which polygenic scores of an
             individual's friends and schoolmates predict the
             individual's own educational attainment. In contrast, an
             individual's height is unassociated with the height genetics
             of peers.},
   Doi = {10.1073/pnas.1711803115},
   Key = {fds332056}
}

@article{fds327153,
   Author = {Belsky, DW and Huffman, KM and Pieper, CF and Shalev, I and Kraus,
             WE},
   Title = {Change in the Rate of Biological Aging in Response to
             Caloric Restriction: CALERIE Biobank Analysis.},
   Journal = {The Journals of Gerontology. Series A, Biological Sciences
             and Medical Sciences},
   Volume = {73},
   Number = {1},
   Pages = {4-10},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1093/gerona/glx096},
   Abstract = {Biological aging measures have been proposed as proxies for
             extension of healthy life span in trials of geroprotective
             therapies that aim to slow aging. Several methods to measure
             biological aging show promise but it is not known if these
             methods are sensitive to changes caused by geroprotective
             therapy. We conducted analysis of two proposed methods to
             quantify biological aging using data from a recently
             concluded trial of an established geroprotector, caloric
             restriction. We obtained data from the National Institute on
             Aging CALERIE randomized trial through its public-access
             biobank (https://calerie.duke.edu/). The CALERIE trial
             randomized N = 220 nonobese adults to 25% caloric
             restriction (n = 145; 11.7% caloric restriction was
             achieved, on average) or to maintain current diet (n = 75)
             for 2 years. We analyzed biomarker data collected at
             baseline, 12-, and 24-month follow-up assessments. We
             applied published biomarker algorithms to these data to
             calculate two biological age measures, Klemera-Doubal Method
             Biological Age and homeostatic dysregulation.
             Intent-to-treat analysis using mixed-effects growth models
             of within-person change over time tested if caloric
             restriction slowed increase in measures of biological aging
             across follow-up. Analyses of both measures indicated
             caloric restriction slowed biological aging. Weight loss did
             not account for the observed effects. Results suggest future
             directions for testing of geroprotective therapies in
             humans.},
   Doi = {10.1093/gerona/glx096},
   Key = {fds327153}
}

@article{fds331420,
   Author = {Hastings, WJ and Shalev, I and Belsky, DW},
   Title = {Translating measures of biological aging to test
             effectiveness of geroprotective interventions: What can we
             learn from research on telomeres?},
   Journal = {Frontiers in Genetics},
   Volume = {8},
   Number = {NOV},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.3389/fgene.2017.00164},
   Abstract = {© 2017 Hastings, Shalev and Belsky. Intervention studies in
             animals suggest molecular changes underlying age-related
             disease and disability can be slowed or reversed. To speed
             translation of these so-called "geroprotective" therapies to
             prevent age-related disease and disability in humans,
             biomarkers are needed that can track changes in the rate of
             human aging over the course of intervention trials.
             Algorithm methods that measure biological processes of aging
             from combinations of DNA methylation marks or clinical
             biomarkers show promise. To identify next steps for
             establishing utility of these algorithm-based measures of
             biological aging for geroprotector trials, we considered the
             history a candidate biomarker of aging that has received
             substantial research attention, telomere length. Although
             telomere length possesses compelling biology to recommend it
             as a biomarker of aging, mixed research findings have
             impeded clinical and epidemiologic translation. Strengths of
             telomeres that should be established for algorithm
             biomarkers of aging are correlation with chronological age
             across the lifespan, prediction of disease, disability, and
             early death, and responsiveness to risk and protective
             exposures. Key challenges in telomere research that
             algorithm biomarkers of aging must address are measurement
             precision and reliability, establishing links between
             longitudinal rates of change across repeated measurements
             and aging outcomes, and clarity over whether the biomarker
             is a causal mechanism of aging. These strengths and
             challenges suggest a research agenda to advance translation
             of algorithm-based aging biomarkers: establish validity in
             young-adult and midlife individuals; test responsiveness to
             exposures that shorten or extend healthy lifespan; and
             conduct repeated-measures longitudinal studies to test
             differential rates of change.},
   Doi = {10.3389/fgene.2017.00164},
   Key = {fds331420}
}

@article{fds330421,
   Author = {Schaefer, JD and Scult, MA and Caspi, A and Arseneault, L and Belsky,
             DW and Hariri, AR and Harrington, H and Houts, R and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Is low cognitive functioning a predictor or consequence of
             major depressive disorder? A test in two longitudinal birth
             cohorts.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s095457941700164x},
   Abstract = {Cognitive impairment has been identified as an important
             aspect of major depressive disorder (MDD). We tested two
             theories regarding the association between MDD and cognitive
             functioning using data from longitudinal cohort studies. One
             theory, the cognitive reserve hypothesis, suggests that
             higher cognitive ability in childhood decreases risk of
             later MDD. The second, the scarring hypothesis, instead
             suggests that MDD leads to persistent cognitive deficits
             following disorder onset. We tested both theories in the
             Dunedin Study, a population-representative cohort followed
             from birth to midlife and assessed repeatedly for both
             cognitive functioning and psychopathology. We also used data
             from the Environmental Risk Longitudinal Twin Study to test
             whether childhood cognitive functioning predicts future MDD
             risk independent of family-wide and genetic risk using a
             discordant twin design. Contrary to both hypotheses, we
             found that childhood cognitive functioning did not predict
             future risk of MDD, nor did study members with a past
             history of MDD show evidence of greater cognitive decline
             unless MDD was accompanied by other comorbid psychiatric
             conditions. Our results thus suggest that low cognitive
             functioning is related to comorbidity, but is neither an
             antecedent nor an enduring consequence of MDD. Future
             research may benefit from considering cognitive deficits
             that occur during depressive episodes from a transdiagnostic
             perspective.},
   Doi = {10.1017/s095457941700164x},
   Key = {fds330421}
}

@article{fds328924,
   Author = {Domingue, BW and Liu, H and Okbay, A and Belsky, DW},
   Title = {Genetic Heterogeneity in Depressive Symptoms Following the
             Death of a Spouse: Polygenic Score Analysis of the U.S.
             Health and Retirement Study.},
   Journal = {The American Journal of Psychiatry},
   Volume = {174},
   Number = {10},
   Pages = {963-970},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.16111209},
   Abstract = {Experience of stressful life events is associated with risk
             of depression. Yet many exposed individuals do not become
             depressed. A controversial hypothesis is that genetic
             factors influence vulnerability to depression following
             stress. This hypothesis is often tested with a
             "diathesis-stress" model, in which genes confer excess
             vulnerability. The authors tested an alternative formulation
             of this model: genes may buffer against depressogenic
             effects of life stress.The hypothesized genetic buffer was
             measured using a polygenic score derived from a published
             genome-wide association study of subjective well-being. The
             authors tested whether married older adults who had higher
             polygenic scores were less vulnerable to depressive symptoms
             following the death of their spouse compared with
             age-matched peers who had also lost their spouse and who had
             lower polygenic scores. Data were analyzed from 8,588
             non-Hispanic white adults in the Health and Retirement Study
             (HRS), a population-representative longitudinal study of
             older adults in the United States.HRS adults with higher
             well-being polygenic scores experienced fewer depressive
             symptoms during follow-up. Those who survived the death of
             their spouses (N=1,647) experienced a sharp increase in
             depressive symptoms following the death and returned toward
             baseline over the following 2 years. Having a higher
             well-being polygenic score buffered against increased
             depressive symptoms following a spouse's death.The effects
             were small, and the clinical relevance is uncertain,
             although polygenic score analyses may provide clues to
             behavioral pathways that can serve as therapeutic targets.
             Future studies of gene-environment interplay in depression
             may benefit from focus on genetics discovered for putative
             protective factors.},
   Doi = {10.1176/appi.ajp.2017.16111209},
   Key = {fds328924}
}

@article{fds328923,
   Author = {Belsky, DW and Snyder-Mackler, N},
   Title = {Invited Commentary: Integrating Genomics and Social
             Epidemiology-Analysis of Late-Life Low Socioeconomic Status
             and the Conserved Transcriptional Response to
             Adversity.},
   Journal = {American Journal of Epidemiology},
   Volume = {186},
   Number = {5},
   Pages = {510-513},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1093/aje/kwx145},
   Abstract = {Socially disadvantaged children face increased morbidity and
             mortality as they age. Understanding mechanisms through
             which social disadvantage becomes biologically embedded and
             devising measurements that can track this embedding are
             critical priorities for research to address social gradients
             in health. The analysis by Levine et al. (Am J Epidemiol.
             2017;186(5):503-509) of genome-wide gene expression in a
             subsample of US Health and Retirement Study participants
             suggests important new directions for the field.
             Specifically, findings suggest promise in integrating gene
             expression data into population studies and provide further
             evidence for the conserved transcriptional response to
             adversity as a marker of biological embedding of social
             disadvantage. The study also highlights methodological
             issues related to the analysis of gene expression data and
             social gradients in health and a need to examine the
             conserved transcriptional response to adversity alongside
             other proposed measurements of biological embedding. Looking
             to the future, advances in genome science are opening new
             opportunities for sociogenomic epidemiology.},
   Doi = {10.1093/aje/kwx145},
   Key = {fds328923}
}

@article{fds326223,
   Author = {Belsky, DW and Caspi, A and Cohen, HJ and Kraus, WE and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Impact of early personal-history characteristics on the Pace
             of Aging: implications for clinical trials of therapies to
             slow aging and extend healthspan.},
   Journal = {Aging Cell},
   Volume = {16},
   Number = {4},
   Pages = {644-651},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/acel.12591},
   Abstract = {Therapies to extend healthspan are poised to move from
             laboratory animal models to human clinical trials.
             Translation from mouse to human will entail challenges,
             among them the multifactorial heterogeneity of human aging.
             To inform clinical trials about this heterogeneity, we
             report how humans' pace of biological aging relates to
             personal-history characteristics. Because geroprotective
             therapies must be delivered by midlife to prevent
             age-related disease onset, we studied young-adult members of
             the Dunedin Study 1972-73 birth cohort (n = 954). Cohort
             members' Pace of Aging was measured as coordinated decline
             in the integrity of multiple organ systems, by quantifying
             rate of decline across repeated measurements of 18
             biomarkers assayed when cohort members were ages 26, 32, and
             38 years. The childhood personal-history characteristics
             studied were known predictors of age-related disease and
             mortality, and were measured prospectively during childhood.
             Personal-history characteristics of familial longevity,
             childhood social class, adverse childhood experiences, and
             childhood health, intelligence, and self-control all
             predicted differences in cohort members' adulthood Pace of
             Aging. Accumulation of more personal-history risks predicted
             faster Pace of Aging. Because trials of anti-aging therapies
             will need to ascertain personal histories retrospectively,
             we replicated results using cohort members' retrospective
             personal-history reports made in adulthood. Because many
             trials recruit participants from clinical settings, we
             replicated results in the cohort subset who had recent
             health system contact according to electronic medical
             records. Quick, inexpensive measures of trial participants'
             early personal histories can enable clinical trials to study
             who volunteers for trials, who adheres to treatment, and who
             responds to anti-aging therapies.},
   Doi = {10.1111/acel.12591},
   Key = {fds326223}
}

@article{fds327274,
   Author = {Domingue, BW and Belsky, DW and Harrati, A and Conley, D and Weir, DR and Boardman, JD},
   Title = {Mortality selection in a genetic sample and implications for
             association studies.},
   Journal = {International Journal of Epidemiology},
   Volume = {46},
   Number = {4},
   Pages = {1285-1294},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1093/ije/dyx041},
   Abstract = {Mortality selection occurs when a non-random subset of a
             population of interest has died before data collection and
             is unobserved in the data. Mortality selection is of general
             concern in the social and health sciences, but has received
             little attention in genetic epidemiology. We tested the
             hypothesis that mortality selection may bias genetic
             association estimates, using data from the US-based Health
             and Retirement Study (HRS).We tested mortality selection
             into the HRS genetic database by comparing HRS respondents
             who survive until genetic data collection in 2006 with those
             who do not. We next modelled mortality selection on
             demographic, health and social characteristics to calculate
             mortality selection probability weights. We analysed
             polygenic score associations with several traits before and
             after applying inverse-probability weighting to account for
             mortality selection. We tested simple associations and
             time-varying genetic associations (i.e. gene-by-cohort
             interactions).We observed mortality selection into the HRS
             genetic database on demographic, health and social
             characteristics. Correction for mortality selection using
             inverse probability weighting methods did not change simple
             association estimates. However, using these methods did
             change estimates of gene-by-cohort interaction effects.
             Correction for mortality selection changed gene-by-cohort
             interaction estimates in the opposite direction from
             increased mortality selection based on analysis of HRS
             respondents surviving through 2012.Mortality selection may
             bias estimates of gene-by-cohort interaction effects.
             Analyses of HRS data can adjust for mortality selection
             associated with observables by including probability
             weights. Mortality selection is a potential confounder of
             genetic association studies, but the magnitude of
             confounding varies by trait.},
   Doi = {10.1093/ije/dyx041},
   Key = {fds327274}
}

@article{fds327272,
   Author = {Belsky, DW},
   Title = {Translating Polygenic Analysis for Prevention},
   Journal = {Circulation Cardiovascular Genetics},
   Volume = {10},
   Number = {3},
   Pages = {e001798-e001798},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1161/CIRCGENETICS.117.001798},
   Doi = {10.1161/CIRCGENETICS.117.001798},
   Key = {fds327272}
}

@article{fds327273,
   Author = {Richmond-Rakerd, LS and Belsky, DW},
   Title = {Swedish Register Analysis of Divorce and Alcohol Use
             Disorder Highlights Social Relationships as a Target for
             Preventive Psychiatry and Genetic Research.},
   Journal = {The American Journal of Psychiatry},
   Volume = {174},
   Number = {5},
   Pages = {411-413},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.17020170},
   Doi = {10.1176/appi.ajp.2017.17020170},
   Key = {fds327273}
}

@article{fds328283,
   Author = {Domingue, BW and Belsky, DW},
   Title = {The social genome: Current findings and implications for the
             study of human genetics.},
   Journal = {Plos Genetics},
   Volume = {13},
   Number = {3},
   Pages = {e1006615},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1371/journal.pgen.1006615},
   Doi = {10.1371/journal.pgen.1006615},
   Key = {fds328283}
}

@article{fds325853,
   Author = {Reuben, A and Caspi, A and Belsky, DW and Broadbent, J and Harrington,
             H and Sugden, K and Houts, RM and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Cognitive
             Function and Socioeconomic Status at Age 38 Years and With
             IQ Change and Socioeconomic Mobility Between Childhood and
             Adulthood.},
   Journal = {Jama},
   Volume = {317},
   Number = {12},
   Pages = {1244-1251},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1001/jama.2017.1712},
   Abstract = {Many children in the United States and around the world are
             exposed to lead, a developmental neurotoxin. The long-term
             cognitive and socioeconomic consequences of lead exposure
             are uncertain.To test the hypothesis that childhood lead
             exposure is associated with cognitive function and
             socioeconomic status in adulthood and with changes in IQ and
             socioeconomic mobility between childhood and midlife.A
             prospective cohort study based on a population-representative
             1972-1973 birth cohort from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (until December 2012).Childhood
             lead exposure ascertained as blood lead levels measured at
             age 11 years. High blood lead levels were observed among
             children from all socioeconomic status levels in this
             cohort.The IQ (primary outcome) and indexes of Verbal
             Comprehension, Perceptual Reasoning, Working Memory, and
             Processing Speed (secondary outcomes) were assessed at age
             38 years using the Wechsler Adult Intelligence Scale-IV
             (WAIS-IV; IQ range, 40-160). Socioeconomic status (primary
             outcome) was assessed at age 38 years using the New Zealand
             Socioeconomic Index-2006 (NZSEI-06; range, 10 [lowest]-90
             [highest]).Of 1037 original participants, 1007 were alive at
             age 38 years, of whom 565 (56%) had been lead tested at age
             11 years (54% male; 93% white). Mean (SD) blood lead level
             at age 11 years was 10.99 (4.63) µg/dL. Among blood-tested
             participants included at age 38 years, mean WAIS-IV score
             was 101.16 (14.82) and mean NZSEI-06 score was 49.75
             (17.12). After adjusting for maternal IQ, childhood IQ, and
             childhood socioeconomic status, each 5-µg/dL higher level
             of blood lead in childhood was associated with a 1.61-point
             lower score (95% CI, -2.48 to -0.74) in adult IQ, a
             2.07-point lower score (95% CI, -3.14 to -1.01) in
             perceptual reasoning, and a 1.26-point lower score (95% CI,
             -2.38 to -0.14) in working memory. Associations of childhood
             blood lead level with deficits in verbal comprehension and
             processing speed were not statistically significant. After
             adjusting for confounders, each 5-µg/dL higher level of
             blood lead in childhood was associated with a 1.79-unit
             lower score (95% CI, -3.17 to -0.40) in socioeconomic
             status. An association between greater blood lead levels and
             a decline in IQ and socioeconomic status from childhood to
             adulthood was observed with 40% of the association with
             downward mobility mediated by cognitive decline from
             childhood.In this cohort born in New Zealand in 1972-1973,
             childhood lead exposure was associated with lower cognitive
             function and socioeconomic status at age 38 years and with
             declines in IQ and with downward social mobility. Childhood
             lead exposure may have long-term ramifications.},
   Doi = {10.1001/jama.2017.1712},
   Key = {fds325853}
}

@article{fds328103,
   Author = {Moffitt, TE and Belsky, DW and Danese, A and Poulton, R and Caspi,
             A},
   Title = {The Longitudinal Study of Aging in Human Young Adults:
             Knowledge Gaps and Research Agenda.},
   Journal = {The Journals of Gerontology. Series A, Biological Sciences
             and Medical Sciences},
   Volume = {72},
   Number = {2},
   Pages = {210-215},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1093/gerona/glw191},
   Abstract = {To prevent onset of age-related diseases and physical and
             cognitive decline, interventions to slow human aging and
             extend health span must eventually be applied to people
             while they are still young and healthy. Yet most human aging
             research examines older adults, many with chronic disease,
             and little is known about aging in healthy young humans.This
             article explains how this knowledge gap is a barrier to
             extending health span and puts forward the case that
             geroscience should invest in researching the pace of aging
             in young adults. As one illustrative example, we describe an
             initial effort to study the pace of aging in a young-adult
             birth cohort by using repeated waves of biomarkers collected
             across the third and fourth decades to quantify the pace of
             coordinated physiological deterioration across multiple
             organ systems (eg, pulmonary, periodontal, cardiovascular,
             renal, hepatic, metabolic, and immune function).Findings
             provided proof of principle that it is possible to quantify
             individual variation in the pace of aging in young adults
             still free of age-related diseases.This article articulates
             research needs to improve longitudinal measurement of the
             pace of aging in young people, to pinpoint factors that slow
             or speed the pace of aging, to compare pace of aging against
             genomic clocks, to explain slow-aging young adults, and to
             apply pace of aging in preventive clinical trials of
             antiaging therapies. This article puts forward a research
             agenda to fill the knowledge gap concerning lifelong causes
             of aging.},
   Doi = {10.1093/gerona/glw191},
   Key = {fds328103}
}

@article{fds324457,
   Author = {Schaefer, JD and Caspi, A and Belsky, DW and Harrington, H and Houts, R and Horwood, LJ and Hussong, A and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Enduring mental health: Prevalence and prediction.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {2},
   Pages = {212-224},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1037/abn0000232},
   Abstract = {We review epidemiological evidence indicating that most
             people will develop a diagnosable mental disorder,
             suggesting that only a minority experience enduring mental
             health. This minority has received little empirical study,
             leaving the prevalence and predictors of enduring mental
             health unknown. We turn to the population-representative
             Dunedin cohort, followed from birth to midlife, to compare
             people never-diagnosed with mental disorder (N = 171; 17%
             prevalence) to those diagnosed at 1-2 study waves, the
             cohort mode (N = 409). Surprisingly, compared to this modal
             group, never-diagnosed Study members were not born into
             unusually well-to-do families, nor did their enduring mental
             health follow markedly sound physical health, or unusually
             high intelligence. Instead, they tended to have an
             advantageous temperament/personality style, and negligible
             family history of mental disorder. As adults, they report
             superior educational and occupational attainment, greater
             life satisfaction, and higher-quality relationships. Our
             findings draw attention to "enduring mental health" as a
             revealing psychological phenotype and suggest it deserves
             further study. (PsycINFO Database Record},
   Doi = {10.1037/abn0000232},
   Key = {fds324457}
}

@article{fds324523,
   Author = {Moskalev, A and Anisimov, V and Aliper, A and Artemov, A and Asadullah,
             K and Belsky, D and Baranova, A and de Grey, A and Dixit, VD and Debonneuil, E and Dobrovolskaya, E and Fedichev, P and Fedintsev, A and Fraifeld, V and Franceschi, C and Freer, R and Fülöp, T and Feige, J and Gems, D and Gladyshev, V and Gorbunova, V and Irincheeva, I and Jager,
             S and Jazwinski, SM and Kaeberlein, M and Kennedy, B and Khaltourina, D and Kovalchuk, I and Kovalchuk, O and Kozin, S and Kulminski, A and Lashmanova, E and Lezhnina, K and Liu, G-H and Longo, V and Mamoshina,
             P and Maslov, A and Pedro de Magalhaes and J and Mitchell, J and Mitnitski,
             A and Nikolsky, Y and Ozerov, I and Pasyukova, E and Peregudova, D and Popov, V and Proshkina, E and Putin, E and Rogaev, E and Rogina, B and Schastnaya, J and Seluanov, A and Shaposhnikov, M and Simm, A and Skulachev, V and Skulachev, M and Solovev, I and Spindler, S and Stefanova, N and Suh, Y and Swick, A and Tower, J and Gudkov, AV and Vijg,
             J and Voronkov, A and West, M and Wagner, W and Yashin, A and Zemskaya, N and Zhumadilov, Z and Zhavoronkov, A},
   Title = {A review of the biomedical innovations for healthy
             longevity.},
   Journal = {Aging},
   Volume = {9},
   Number = {1},
   Pages = {7-25},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.18632/aging.101163},
   Doi = {10.18632/aging.101163},
   Key = {fds324523}
}


%% Bennett, Gary G.   
@article{fds337999,
   Author = {Hopkins, CM and Bennett, GG},
   Title = {Weight-Related Terms Differentially Affect Self-Efficacy and
             Perception of Obesity.},
   Journal = {Obesity (Silver Spring, Md.)},
   Volume = {26},
   Number = {9},
   Pages = {1405-1411},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1002/oby.22255},
   Abstract = {Little work has explored the effect of weight-related terms
             on treatment initiation; only one study has investigated
             weight-related terms and the psychological constructs
             associated with treatment uptake. The present study examines
             the effects of four common weight-related terms on treatment
             initiation and the moderating effect of weight bias
             internalization.Adult participants with overweight and
             obesity (n = 436) were recruited online and asked to
             read three vignettes describing clinical encounters; the
             weight-related term (i.e., "weight," "BMI," "obesity," or
             "fat") was varied randomly. Participants then reported
             self-efficacy, cognitive and emotional illness beliefs about
             obesity (i.e., illness perception), and interest in a weight
             loss program.The term "obesity" resulted in the greatest
             self-efficacy and perceived control over obesity. "Fat"
             resulted in the least illness coherence (i.e., understanding
             of obesity). Weight bias internalization did not moderate
             the effect of term on self-efficacy, nor did it moderate
             illness perception. No differences in weight loss program
             enrollment were observed.Use of the term "obesity" may
             promote patients' perceived control and self-efficacy. Use
             of "fat" should be avoided. Results suggest that, despite
             patient and clinician preference for euphemistic weight
             terms, use of clinical language such as "obesity" may
             perform better in provider intervention.},
   Doi = {10.1002/oby.22255},
   Key = {fds337999}
}

@article{fds335636,
   Author = {McVay, MA and Yancy, WS and Bennett, GG and Jung, S-H and Voils,
             CI},
   Title = {Perceived barriers and facilitators of initiation of
             behavioral weight loss interventions among adults with
             obesity: a qualitative study.},
   Journal = {Bmc Public Health},
   Volume = {18},
   Number = {1},
   Pages = {854},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1186/s12889-018-5795-9},
   Abstract = {Evidence-based behavioral weight loss interventions are
             under-utilized. To inform efforts to increase uptake of
             these interventions, it is important to understand the
             perspectives of adults with obesity regarding barriers and
             facilitators of weight loss intervention initiation.We
             conducted a qualitative study in adults with obesity who had
             recently attempted weight loss either with assistance from
             an evidence-based behavioral intervention (intervention
             initiators) or without use of a formal intervention
             (intervention non-initiators). We recruited primary care
             patients, members of a commercial weight loss program, and
             members of a Veterans Affairs weight loss program.
             Intervention initiators and non-initiators were interviewed
             separately using a semi-structured interview guide that
             asked participants about barriers and facilitators of weight
             loss intervention initiation. Conversations were
             audio-recorded and transcribed. Data were analyzed with
             qualitative content analysis. Two researchers used open
             coding to generate the code book on a subset of transcripts
             and a single researcher coded remaining transcripts. Codes
             were combined into subthemes, which were combined in to
             higher order themes. Intervention initiators and
             non-initiators were compared.We conducted three focus groups
             with participants who had initiated interventions
             (n = 26) and three focus groups (n = 24) and 8
             individual interviews with participants who had not
             initiated interventions. Intervention initiators and
             non-initiators were, respectively, 65% and 37.5% white, 62%
             and 63% female, mean age of 55 and 54 years old, and mean
             BMI of 34 kg/m2. Three themes were identified. One theme
             was practical factors, with subthemes of reasonable cost and
             scheduling compatibility. A second theme was anticipated
             effectiveness of intervention, with subthemes of
             intervention content addressing individual needs; social
             aspects influencing effectiveness; and evaluating evidence
             of effectiveness. A third theme was anticipated pleasantness
             of intervention, with subthemes of social aspects
             influencing enjoyment; anticipated dietary and tracking
             prescriptions; and identity and self-reliance factors.
             Different perspectives were identified from intervention
             initiators and non-initiators.Strategies to engage
             individuals in evidence-based weight loss interventions can
             be developed using these results. Strategies could target
             individuals' perceived barriers and benefits to initiating
             interventions, or could focus on refining interventions to
             appeal to more individuals.},
   Doi = {10.1186/s12889-018-5795-9},
   Key = {fds335636}
}

@article{fds335637,
   Author = {Ayazi, M and Johnson, KT and Merritt, MM and Di Paolo and MR and Edwards,
             CL and Koenig, HG and Bennett, GG and Whitfield, KA and Barker,
             CS},
   Title = {Religiosity, Education, John Henryism Active Coping, and
             Cardiovascular Responses to Anger Recall for African
             American Men},
   Journal = {Journal of Black Psychology},
   Volume = {44},
   Number = {4},
   Pages = {295-321},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1177/0095798418765859},
   Doi = {10.1177/0095798418765859},
   Key = {fds335637}
}

@article{fds337340,
   Author = {Azar, KMJ and Bennett, GG and Nolting, LA and Rosas, LG and Burke, LE and Ma, J},
   Title = {A framework for examining the function of digital health
             technologies for weight management.},
   Journal = {Translational Behavioral Medicine},
   Volume = {8},
   Number = {2},
   Pages = {280-294},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1093/tbm/ibx050},
   Abstract = {Research is rapidly extending its focus to develop and
             evaluate weight management interventions that incorporate
             eHealth technologies. Comparative effectiveness of eHealth
             interventions is partly limited by the extensive
             heterogeneity in intervention design, variation in use of
             eHealth tools, and expanding development of novel tools to
             promote weight management. We closely examined,
             characterized, and categorized the use and function of
             eHealth tools across a wide range of eHealth interventions
             for weight management in order to first create a novel
             schematic framework for eHealth interventions and, second,
             to evaluate eHealth interventions using this framework. We
             examined 49 randomized controlled trials from two systematic
             reviews evaluating the effectiveness of eHealth
             interventions for weight loss. Further characterization of
             each intervention identified common use and function of
             eHealth tools represented within interventions and thus
             important to include in the proposed framework. This
             resulted in six descriptive domains. We then categorized
             each eHealth intervention within the context of the newly
             developed framework. Last, we examined efficacious
             interventions in the context of the framework. Twenty-five
             randomized controlled trials reported significantly more
             weight loss between the intervention group utilizing
             eHealth, compared to a non-eHealth control intervention
             and/or within an eHealth intervention group. Of these 25
             interventions, 15 (60%) used automated feedback (Domain 1),
             13 (52%) used non-eHealth tailored feedback by a health care
             provider (Domain 5), and 8 (32%) used tailored feedback from
             a health care professional through an electronic channel
             (Domain 2). The proposed schematic framework offers an
             alternative and novel approach for comparing across
             interventions and informing the development and evaluation
             of eHealth interventions.},
   Doi = {10.1093/tbm/ibx050},
   Key = {fds337340}
}

@article{fds332978,
   Author = {Pagoto, SL and Bennett, GG},
   Title = {Healthy Lifestyle for All Through Patient Care and
             Policy.},
   Journal = {Jama Internal Medicine},
   Volume = {178},
   Number = {1},
   Pages = {152-153},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1001/jamainternmed.2017.6797},
   Doi = {10.1001/jamainternmed.2017.6797},
   Key = {fds332978}
}

@article{fds332939,
   Author = {Armstrong, S and Mendelsohn, A and Bennett, G and Taveras, EM and Kimberg, A and Kemper, AR},
   Title = {Texting Motivational Interviewing: A Randomized Controlled
             Trial of Motivational Interviewing Text Messages Designed to
             Augment Childhood Obesity Treatment},
   Journal = {Childhood Obesity},
   Volume = {14},
   Number = {1},
   Pages = {4-10},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1089/chi.2017.0089},
   Doi = {10.1089/chi.2017.0089},
   Key = {fds332939}
}

@article{fds329767,
   Author = {Bennett, GG and Shelton, RC},
   Title = {Extending Our Reach for Greater Impact.},
   Journal = {Health Education & Behavior : the Official Publication of
             the Society for Public Health Education},
   Volume = {44},
   Number = {6},
   Pages = {835-838},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1177/1090198117736354},
   Doi = {10.1177/1090198117736354},
   Key = {fds329767}
}

@article{fds330845,
   Author = {Buscemi, J and Bennett, GG and Gorin, SS and Pagoto, SL and Sallis, JF and Wilson, DK and Fitzgibbon, ML},
   Title = {A 6-year update of the health policy and advocacy priorities
             of the Society of Behavioral Medicine.},
   Journal = {Translational Behavioral Medicine},
   Volume = {7},
   Number = {4},
   Pages = {903-911},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1007/s13142-017-0507-z},
   Abstract = {Government policy affects virtually every topic of interest
             to health behavior researchers, from research funding to
             reimbursement for clinical services to application of
             evidence to impact health outcomes. This paper provides a
             6-year update on the expansion of Society of Behavioral
             Medicine's (SBM) public policy and advocacy agenda and
             proposed future directions. SBM's Health Policy Council is
             responsible for ensuring coordination of the policy-related
             activities of the Health Policy Committee (HPC), the Civic
             and Public Engagement Committee (CPEC), and the Scientific
             and Professional Liaison Council (SPLC). These committees
             and councils have written letters to Congress, signed onto
             advocacy letters with hundreds of organizations, and
             developed and disseminated 15 health policy briefs, the
             majority of which have been presented to legislative
             staffers on Capitol Hill. With the assistance of the SPLC,
             SBM has collaborated on policy efforts with like-minded
             organizations to increase the impact of the Society's policy
             work. Moving forward, SBM plans to continue to increase
             efforts to disseminate policy work more broadly and develop
             long-term relationships with Congressional staffers. SBM
             leadership realizes that to remain relevant, demonstrate
             impact, and advance the role of behavioral medicine, we must
             advance a policy agenda that reflects our mission of better
             health through behavior change.},
   Doi = {10.1007/s13142-017-0507-z},
   Key = {fds330845}
}

@article{fds329768,
   Author = {Greaney, ML and Askew, S and Wallington, SF and Foley, PB and Quintiliani, LM and Bennett, GG},
   Title = {The effect of a weight gain prevention intervention on
             moderate-vigorous physical activity among black women: the
             Shape Program.},
   Journal = {The International Journal of Behavioral Nutrition and
             Physical Activity},
   Volume = {14},
   Number = {1},
   Pages = {139},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1186/s12966-017-0596-6},
   Abstract = {Rates of physical inactivity are high among Black women
             living in the United States with overweight or obesity,
             especially those living in the rural South. This study was
             conducted to determine if an efficacious weight gain
             prevention intervention increased moderate-vigorous physical
             activity (MVPA).The Shape Program, a weight gain prevention
             intervention implemented in community health centers in
             rural North Carolina, was designed for socioeconomically
             disadvantaged Black women with overweight or obesity. MVPA
             was measured using accelerometers, and summarized into 1-
             and 10-min bouts. We employed analyses of covariance
             (ANCOVA) to assess the relationship between changes in MVPA
             over 12 months, calculated as a change score, and
             intervention assignment (intervention versus usual
             care).Participants completing both baseline and 12-month
             accelerometer assessments (n = 121) had a mean age of 36.1
             (SD = 5.43) years and a mean body mass index of
             30.24 kg/m2 (SD = 2.60). At baseline, 38% met the
             physical activity recommendation (150 min of MVPA/week)
             when assessed using 10-min bouts, and 76% met the
             recommendation when assessed using 1-min bouts. There were
             no significant differences in change in MVPA participation
             among participants randomized to the intervention from
             baseline to 12-months using 1-min bouts (adjusted
             intervention mean [95% CI]: 20.50 [-109.09 to 150.10] vs.
             adjusted usual care mean [95% CI]: -80.04 [-209.21 to
             49.13], P = .29), or 10-min bouts (adjusted intervention
             mean [95% CI]: 7.39 [-83.57 to 98.35] vs. adjusted usual
             care mean [95% CI]: -17.26 [-107.93 to 73.40],
             P = .70).Although prior research determined that the Shape
             intervention promoted weight gain prevention, MVPA did not
             increase significantly among intervention participants from
             baseline to 12 months. The classification of bouts had a
             marked effect on the prevalence estimates of those meeting
             physical activity recommendations. More research is needed
             to understand how to promote increased MVPA in weight gain
             prevention interventions.This study is registered at
             www.clinicaltrials.gov database (No. NCT00938535.
             Retrospectively Registered 7/10/2009).},
   Doi = {10.1186/s12966-017-0596-6},
   Key = {fds329768}
}

@article{fds324092,
   Author = {Winkler, MR and Moore, ED and Bennett, GG and Armstrong, SC and Brandon,
             DH},
   Title = {Parent-adolescent influences on everyday dietary practices:
             Perceptions of adolescent females with obesity and their
             mothers.},
   Journal = {Maternal & Child Nutrition},
   Volume = {13},
   Number = {4},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1111/mcn.12416},
   Abstract = {Parents demonstrate an important influence on adolescent
             obesity and dietary behavior; yet, family-based obesity
             interventions continue to exhibit limited success among
             adolescents. To further inform family-based approaches for
             adolescent obesity treatment, we examined the perceptions of
             adolescent females with obesity and their mothers of the
             influences experienced within the parent-adolescent
             relationship that affect everyday dietary practices. We
             conducted six focus group interviews (three adolescent
             female and three mother) among 15 adolescent (12-17 years
             old) females with obesity and 12 of their mothers. Content
             analysis techniques were used to analyze the transcribed
             interviews. Adolescent females with obesity discussed a
             diverse set of parental influences (controlling, supporting
             and cultivating, overlooking and tempting, acquiescing,
             providing, attending, and not providing and avoiding) on
             their daily dietary practices. Among mother focus groups,
             mothers discussed specific intentional and unintentional
             types of influences from children that affected the food and
             drink they consumed, prepared, and acquired. Findings
             provide a fuller view of the varied social influences on
             everyday dietary practices within the parent-adolescent
             relationship. They indicate the importance of examining both
             parent-to-child and child-to-parent influences and begin to
             illuminate the value of attending to the social
             circumstances surrounding dietary behaviors to strengthen
             family-based obesity treatment approaches.},
   Doi = {10.1111/mcn.12416},
   Key = {fds324092}
}

@article{fds326827,
   Author = {Proeschold-Bell, RJ and Turner, EL and Bennett, GG and Yao, J and Li,
             X-F and Eagle, DE and Meyer, RA and Williams, RB and Swift, RY and Moore,
             HE and Kolkin, MA and Weisner, CC and Rugani, KM and Hough, HJ and Williams, VP and Toole, DC},
   Title = {A 2-Year Holistic Health and Stress Intervention: Results of
             an RCT in Clergy.},
   Journal = {American Journal of Preventive Medicine},
   Volume = {53},
   Number = {3},
   Pages = {290-299},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.amepre.2017.04.009},
   Abstract = {This study sought to determine the effect of a 2-year,
             multicomponent health intervention (Spirited Life) targeting
             metabolic syndrome and stress simultaneously.An RCT using a
             three-cohort multiple baseline design was conducted in
             2010-2014.Participants were United Methodist clergy in North
             Carolina, U.S., in 2010, invited based on occupational
             status. Of invited 1,745 clergy, 1,114 consented, provided
             baseline data, and were randomly assigned to immediate
             intervention (n=395), 1-year waitlist (n=283), or 2-year
             waitlist (n=436) cohorts for a 48-month trial duration.The
             2-year intervention consisted of personal goal setting and
             encouragement to engage in monthly health coaching, an
             online weight loss intervention, a small grant, and three
             workshops delivering stress management and theological
             content supporting healthy behaviors. Participants were not
             blinded to intervention.Trial outcomes were metabolic
             syndrome (primary) and self-reported stress and depressive
             symptoms (secondary). Intervention effects were estimated in
             2016 in an intention-to-treat framework using generalized
             estimating equations with adjustment for baseline level of
             the outcome and follow-up time points. Log-link Poisson
             generalized estimating equations with robust SEs was used to
             estimate prevalence ratios (PRs) for binary outcomes; mean
             differences were used for continuous/score outcomes.Baseline
             prevalence of metabolic syndrome was 50.9% and depression
             was 11.4%. The 12-month intervention effect showed a benefit
             for metabolic syndrome (PR=0.86, 95% CI=0.79, 0.94,
             p<0.001). This benefit was sustained at 24 months of
             intervention (PR=0.88; 95% CI=0.78, 1.00, p=0.04). There was
             no significant effect on depression or stress scores.The
             Spirited Life intervention improved metabolic syndrome
             prevalence in a population of U.S. Christian clergy and
             sustained improvements during 24 months of intervention.
             These findings offer support for long-duration behavior
             change interventions and population-level interventions that
             allow participants to set their own health goals.This study
             is registered at www.clinicaltrials.gov NCT01564719.},
   Doi = {10.1016/j.amepre.2017.04.009},
   Key = {fds326827}
}

@article{fds327016,
   Author = {Herring, SJ and Cruice, JF and Bennett, GG and Darden, N and Wallen, JJ and Rose, MZ and Davey, A and Foster, GD},
   Title = {Intervening during and after pregnancy to prevent weight
             retention among African American women.},
   Journal = {Preventive Medicine Reports},
   Volume = {7},
   Pages = {119-123},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.pmedr.2017.05.015},
   Abstract = {Efforts to prevent postpartum weight retention in extant
             clinical trials of African American women have proven
             exceedingly challenging. The primary purpose of this pilot
             study was to determine whether a behavioral intervention
             implemented in early pregnancy through 6 months postpartum
             could increase the proportion of African American women who
             were at or below their early pregnancy weights by 6 months
             postpartum. We additionally evaluated whether mothers'
             postpartum weight loss could be maintained at 12 months
             postpartum. Participants were 66 socioeconomically
             disadvantaged African American women (36% overweight, 64%
             obese) randomly assigned to a behavioral intervention or
             usual care group. The intervention, implemented from early
             pregnancy to 6 months postpartum, promoted weight control
             through: (1) empirically supported behavior change goals;
             (2) interactive self-monitoring text messages; (3) weekly to
             monthly health coach calls; and (4) skills training and
             support through Facebook. In modified intent-to-treat
             analyses, participants assigned to the intervention were
             significantly more likely to be at or below their early
             pregnancy weights by 6 months postpartum compared to usual
             care (56% vs. 29%, p = 0.04). At 12 months postpartum,
             the maternal weight difference between intervention and
             usual care groups was not maintained (41% vs. 38%
             respectively at or below early pregnancy weights,
             p = 0.83). Findings suggest that a combined pregnancy and
             postpartum weight control intervention improves 6 month
             weight outcomes in socioeconomically disadvantaged African
             American women with obesity. Longer interventions may be
             needed to overcome late postpartum weight gain among this
             high risk group. Clinical trial registration number:
             ClinicalTrials.gov identifier NCT01530776.},
   Doi = {10.1016/j.pmedr.2017.05.015},
   Key = {fds327016}
}

@article{fds324090,
   Author = {Steinberg, DM and Christy, J and Batch, BC and Askew, S and Moore, RH and Parker, P and Bennett, GG},
   Title = {Preventing Weight Gain Improves Sleep Quality Among Black
             Women: Results from a RCT.},
   Journal = {Annals of Behavioral Medicine},
   Volume = {51},
   Number = {4},
   Pages = {555-566},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1007/s12160-017-9879-z},
   Abstract = {Obesity and poor sleep are highly prevalent among Black
             women.We examined whether a weight gain prevention
             intervention improved sleep among Black women.We conducted a
             randomized trial comparing a 12-month weight gain prevention
             intervention that included self-monitoring through mobile
             technologies and phone coaching to usual care in community
             health centers. We measured sleep using the Medical Outcomes
             Study Sleep Scale at baseline, 12 months, and 18 months.
             The scale examines quantity of sleep, sleep disturbance,
             sleep adequacy, daytime somnolence, snoring, shortness of
             breath, and global sleep problems (sleep problem indices I
             and II).Participants (n = 184) were on average 35.4 years
             and obese (BMI 30.2 kg/m2); 74% made <$30,000/year. At
             baseline, average sleep duration was 6.4 (1.5) hours.
             Controlling for weight change and sleep medication, the
             intervention group reported greater improvements in sleep
             disturbance [-8.35 (-16.24, -0.45)] and sleep problems at
             12 months: sleep problem index I [-8.35 (-16.24, -0.45)];
             sleep problem index II [-8.35 (-16.24, -0.45)]. However,
             these findings did not persist at 18 months.Preventing
             weight gain may afford clinical benefit on improving sleep
             quality.The trial was registered with the ClinicalTrials.gov
             database (NCT00938535).},
   Doi = {10.1007/s12160-017-9879-z},
   Key = {fds324090}
}

@article{fds324093,
   Author = {Greaney, ML and Askew, S and Foley, P and Wallington, SF and Bennett,
             GG},
   Title = {Linking patients with community resources: use of a free
             YMCA membership among low-income black women.},
   Journal = {Translational Behavioral Medicine},
   Volume = {7},
   Number = {2},
   Pages = {341-348},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s13142-016-0431-7},
   Abstract = {Given the increasing interest in expanding obesity
             prevention efforts to cover community-based programs, we
             examined whether individuals would access a YMCA for
             physical activity promotion. We provided a no-cost 12-month
             YMCA membership to socioeconomically disadvantaged black
             women who were randomized to the intervention arm of a
             weight gain prevention trial (n = 91). Analyses examined
             associations of membership activation and use with baseline
             psychosocial, contextual, health-related, and
             sociodemographic factors. Many participants (70.3 %)
             activated their memberships; however, use was low (42.2 %
             had no subsequent visits, 46.9 % had one to ten visits).
             There were no predictors of membership activation, but
             individuals living below/borderline the federal poverty line
             were more likely to use the center (1+ visits), as were
             those who met physical activity guidelines at baseline. More
             comprehensive and intensive interventions may be necessary
             to promote use of community resources-even when provided
             free-among high-risk populations of women with obesity that
             live in rural areas of the USA.},
   Doi = {10.1007/s13142-016-0431-7},
   Key = {fds324093}
}

@article{fds328845,
   Author = {Steinberg, D and Bennett, GG and Svetkey, L},
   Title = {The DASH Diet, 20 Years Later.},
   Journal = {Jama},
   Volume = {317},
   Number = {15},
   Pages = {1529-1530},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1001/jama.2017.1628},
   Doi = {10.1001/jama.2017.1628},
   Key = {fds328845}
}

@article{fds324091,
   Author = {Benjamin Neelon and SE and Østbye, T and Bennett, GG and Kravitz, RM and Clancy, SM and Stroo, M and Iversen, E and Hoyo, C},
   Title = {Cohort profile for the Nurture Observational Study examining
             associations of multiple caregivers on infant growth in the
             Southeastern USA.},
   Journal = {Bmj Open},
   Volume = {7},
   Number = {2},
   Pages = {e013939},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1136/bmjopen-2016-013939},
   Abstract = {Childcare has been associated with obesity in children in
             cross-sectional and longitudinal studies, although some
             observed no association. Few studies have focused on care
             during infancy, a period when children may be especially
             vulnerable.The Nurture Study is an observational birth
             cohort designed to assess longitudinal associations of
             childcare and the presence of multiple caregivers on infant
             adiposity and weight trajectories throughout the first year
             of life. We examine as potential mediators feeding, physical
             activity, sleep and stress. We completed recruitment in
             2015. Of the 860 women who enrolled during pregnancy, 799
             delivered a single live infant who met our inclusion
             criteria. Of those, 666 mothers (77.4%) agreed to
             participate in the study for themselves and their
             infants.Among the 666 women in the study, 472 (71%)
             identified as black, 127 (19%) as white, 7 (1%) as Asian or
             Asian American, 6 (1%) as Native American and 49 (7%) as
             other race or more than one race; 43 (7%) identified as
             Hispanic/Latina. Just under half (48%) had a high school
             diploma or less, 61% had household incomes <$20 000/year
             and 59% were married or living with a partner. The mean (SD)
             infant gestational age was 41.28 weeks (2.29) and birth
             weight for gestational age z-score was -0.31 (0.93). Just
             under half (49%) of infants were females, 69% received some
             human milk and 40% were exclusively breast fed at hospital
             discharge. Data collection began in 2013, is currently
             underway, and is scheduled to conclude in late 2016.Results
             will help assess the magnitude of associations between
             childcare in infancy and subsequent obesity. Findings will
             also inform intervention and policy efforts to improve
             childcare environments and help prevent obesity in settings
             where many infants spend time.Clinicaltrials.gov,
             NCT01788644.},
   Doi = {10.1136/bmjopen-2016-013939},
   Key = {fds324091}
}

@article{fds315310,
   Author = {Winkler, MR and Bennett, GG and Brandon, DH},
   Title = {Factors related to obesity and overweight among Black
             adolescent girls in the United States.},
   Journal = {Women & Health},
   Volume = {57},
   Number = {2},
   Pages = {208-248},
   Year = {2017},
   Month = {February},
   ISSN = {0363-0242},
   url = {http://dx.doi.org/10.1080/03630242.2016.1159267},
   Abstract = {In the United States, Black adolescents have the highest
             prevalence of pediatric obesity and overweight among girls.
             While Black girls are disproportionately affected, the
             reasons for this health disparity remain unclear. The
             authors conducted a systematic review to investigate the
             factors related to obesity and overweight among Black
             adolescent girls. The authors searched four databases for
             relevant English-language publications using all publication
             years through 2015. Fifty-one studies met the inclusion
             criteria and were used for this review. Using a
             configuration approach to synthesis, three categories were
             identified, paralleling the bioecological theory of human
             development: (1) individual, (2) interpersonal, and (3)
             community and societal factors. A description of each
             factor's association with obesity among Black adolescent
             girls is presented. From this review, the authors identified
             a diverse and vast set of individual, interpersonal, and
             community and societal factors explored for their
             relationship with obesity and overweight. Given the
             insufficient repetition and limited significant findings
             among most factors, the authors believe that multiple gaps
             in knowledge exist across all categories regarding the
             factors related to obesity and overweight among Black
             adolescent girls. To improve the quality of research in this
             area, suggested research directions and methodological
             recommendations are provided.},
   Doi = {10.1080/03630242.2016.1159267},
   Key = {fds315310}
}


%% Bergelson, Elika   
@article{fds337129,
   Author = {Bergelson, E and Amatuni, A and Dailey, S and Koorathota, S and Tor,
             S},
   Title = {Day by day, hour by hour: Naturalistic language input to
             infants.},
   Journal = {Developmental Science},
   Pages = {e12715},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1111/desc.12715},
   Abstract = {Measurements of infants' quotidian experiences provide
             critical information about early development. However, the
             role of sampling methods in providing these measurements is
             rarely examined. Here we directly compare language input
             from hour-long video-recordings and daylong audio-recordings
             within the same group of 44 infants at 6 and 7 months. We
             compared 12 measures of language quantity and lexical
             diversity, talker variability, utterance-type, and object
             presence, finding moderate correlations across
             recording-types. However, video-recordings generally
             featured far denser noun input across these measures
             compared to the daylong audio-recordings, more akin to
             'peak' audio hours (though not as high in talkers and
             word-types). Although audio-recordings captured ~10 times
             more awake-time than videos, the noun input in them was only
             2-4 times greater. Notably, whether we compared videos to
             daylong audio-recordings or peak audio times, videos
             featured relatively fewer declaratives and more questions;
             furthermore, the most common video-recorded nouns were less
             consistent across families than the top audio-recording
             nouns were. Thus, hour-long videos and daylong
             audio-recordings revealed fairly divergent pictures of the
             language infants hear and learn from in their daily lives.
             We suggest that short video-recordings provide a dense and
             somewhat different sample of infants' language experiences,
             rather than a typical one, and should be used cautiously for
             extrapolation about common words, talkers, utterance-types,
             and contexts at larger timescales. If theories of language
             development are to be held accountable to 'facts on the
             ground' from observational data, greater care is needed to
             unpack the ramifications of sampling methods of early
             language input.},
   Doi = {10.1111/desc.12715},
   Key = {fds337129}
}

@article{fds333673,
   Author = {Amatuni, A and He, E and Bergelson, E},
   Title = {Preserved Structure Across Vector Space Representations.},
   Journal = {CoRR},
   Volume = {abs/1802.00840},
   Year = {2018},
   Key = {fds333673}
}

@article{fds330846,
   Author = {Bergelson, E and Aslin, RN},
   Title = {Nature and origins of the lexicon in 6-mo-olds.},
   Journal = {Proceedings of the National Academy of Sciences of the
             United States of America},
   Volume = {114},
   Number = {49},
   Pages = {12916-12921},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1073/pnas.1712966114},
   Abstract = {Recent research reported the surprising finding that even
             6-mo-olds understand common nouns [Bergelson E, Swingley D
             (2012) Proc Natl Acad Sci USA 109:3253-3258]. However, is
             their early lexicon structured and acquired like older
             learners? We test 6-mo-olds for a hallmark of the mature
             lexicon: cross-word relations. We also examine whether
             properties of the home environment that have been linked
             with lexical knowledge in older children are detectable in
             the initial stage of comprehension. We use a new dataset,
             which includes in-lab comprehension and home measures from
             the same infants. We find evidence for cross-word structure:
             On seeing two images of common nouns, infants looked
             significantly more at named target images when the
             competitor images were semantically unrelated (e.g., milk
             and foot) than when they were related (e.g., milk and
             juice), just as older learners do. We further find initial
             evidence for home-lab links: common noun "copresence" (i.e.,
             whether words' referents were present and attended to in
             home recordings) correlated with in-lab comprehension. These
             findings suggest that, even in neophyte word learners,
             cross-word relations are formed early and the home learning
             environment measurably helps shape the lexicon from the
             outset.},
   Doi = {10.1073/pnas.1712966114},
   Key = {fds330846}
}

@article{fds325486,
   Author = {Frank, MC and Bergelson, E and Bergmann, C and Cristia, A and Floccia,
             C and Gervain, J and Hamlin, JK and Hannon, EE and Kline, M and Levelt, C and Lew-Williams, C and Nazzi, T and Panneton, R and Rabagliati, H and Soderstrom, M and Sullivan, J and Waxman, S and Yurovsky,
             D},
   Title = {A Collaborative Approach to Infant Research: Promoting
             Reproducibility, Best Practices, and Theory-Building},
   Journal = {Infancy},
   Volume = {22},
   Number = {4},
   Pages = {421-435},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1111/infa.12182},
   Abstract = {© 2017 International Congress of Infant Studies (ICIS).The
             ideal of scientific progress is that we accumulate
             measurements and integrate these into theory, but recent
             discussion of replicability issues has cast doubt on whether
             psychological research conforms to this model. Developmental
             research-especially with infant participants-also has
             discipline-specific replicability challenges, including
             small samples and limited measurement methods. Inspired by
             collaborative replication efforts in cognitive and social
             psychology, we describe a proposal for assessing and
             promoting replicability in infancy research: large-scale,
             multi-laboratory replication efforts aiming for a more
             precise understanding of key developmental phenomena. The
             ManyBabies project, our instantiation of this proposal, will
             not only help us estimate how robust and replicable these
             phenomena are, but also gain new theoretical insights into
             how they vary across ages, linguistic communities, and
             measurement methods. This project has the potential for a
             variety of positive outcomes, including less-biased
             estimates of theoretically important effects, estimates of
             variability that can be used for later study planning, and a
             series of best-practices blueprints for future infancy
             research.},
   Doi = {10.1111/infa.12182},
   Key = {fds325486}
}

@article{fds327239,
   Author = {Bergelson, E and Swingley, D},
   Title = {Young Infants' Word Comprehension Given An Unfamiliar Talker
             or Altered Pronunciations.},
   Journal = {Child Development},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1111/cdev.12888},
   Abstract = {To understand spoken words, listeners must appropriately
             interpret co-occurring talker characteristics and speech
             sound content. This ability was tested in 6- to
             14-months-olds by measuring their looking to named food and
             body part images. In the new talker condition (n = 90),
             pictures were named by an unfamiliar voice; in the
             mispronunciation condition (n = 98), infants' mothers
             "mispronounced" the words (e.g., nazz for nose). Six- to
             7-month-olds fixated target images above chance across
             conditions, understanding novel talkers, and mothers'
             phonologically deviant speech equally. Eleven- to
             14-months-olds also understood new talkers, but performed
             poorly with mispronounced speech, indicating sensitivity to
             phonological deviation. Between these ages, performance was
             mixed. These findings highlight the changing roles of
             acoustic and phonetic variability in early word
             comprehension, as infants learn which variations alter
             meaning.},
   Doi = {10.1111/cdev.12888},
   Key = {fds327239}
}

@article{fds327381,
   Author = {Bergelson, E and Aslin, R},
   Title = {Semantic Specificity in One-Year-Olds' Word
             Comprehension.},
   Journal = {Language Learning and Development},
   Volume = {13},
   Number = {4},
   Pages = {481-501},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1080/15475441.2017.1324308},
   Abstract = {The present study investigated infants' knowledge about
             familiar nouns. Infants (n = 46, 12-20-month-olds) saw
             two-image displays of familiar objects, or one familiar and
             one novel object. Infants heard either a matching word (e.g.
             "foot' when seeing foot and juice), a related word (e.g.
             "sock" when seeing foot and juice) or a nonce word (e.g.
             "fep" when seeing a novel object and dog). Across the whole
             sample, infants reliably fixated the referent on matching
             and nonce trials. On the critical related trials we found
             increasingly less looking to the incorrect (but related)
             image with age. These results suggest that one-year-olds
             look at familiar objects both when they hear them labeled
             and when they hear related labels, to similar degrees, but
             over the second year increasingly rely on semantic fit. We
             suggest that infants' initial semantic representations are
             imprecise, and continue to sharpen over the second postnatal
             year.},
   Doi = {10.1080/15475441.2017.1324308},
   Key = {fds327381}
}


%% Bettman, James R.   
@article{fds323857,
   Author = {Escalas, JE and Bettman, JR},
   Title = {Connecting With Celebrities: How Consumers Appropriate
             Celebrity Meanings for a Sense of Belonging},
   Journal = {Journal of Advertising},
   Volume = {46},
   Number = {2},
   Pages = {297-308},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1080/00913367.2016.1274925},
   Doi = {10.1080/00913367.2016.1274925},
   Key = {fds323857}
}


%% Bilbo, Staci D.   
@article{fds326607,
   Author = {Bolton, JL and Marinero, S and Hassanzadeh, T and Natesan, D and Le, D and Belliveau, C and Mason, SN and Auten, RL and Bilbo,
             SD},
   Title = {Gestational exposure to air pollution alters cortical
             volume, microglial morphology, and microglia-neuron
             interactions in a sex-specific manner},
   Journal = {Frontiers in Synaptic Neuroscience},
   Volume = {9},
   Number = {MAY},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.3389/fnsyn.2017.00010},
   Abstract = {© 2017 Bolton, Marinero, Hassanzadeh, Natesan, Le,
             Belliveau, Mason, Auten and Bilbo.Microglia are the resident
             immune cells of the brain, important for normal neural
             development in addition to host defense in response to
             inflammatory stimuli. Air pollution is one of the most
             pervasive and harmful environmental toxicants in the modern
             world, and several large scale epidemiological studies have
             recently linked prenatal air pollution exposure with an
             increased risk of neurodevelopmental disorders such as
             autism spectrum disorder (ASD). Diesel exhaust particles
             (DEP) are a primary toxic component of air pollution, and
             markedly activate microglia in vitro and in vivo in adult
             rodents. We have demonstrated that prenatal exposure to DEP
             in mice, i.e., to the pregnant dams throughout gestation,
             results in a persistent vulnerability to behavioral deficits
             in adult offspring, especially in males, which is intriguing
             given the greater incidence of ASD in males to females
             (4:1). Moreover, there is a striking upregulation of
             toll-like receptor (TLR) 4 gene expression within the brains
             of the same mice, and this expression is primarily in
             microglia. Here we explored the impact of gestational
             exposure to DEP or vehicle on microglial morphology in the
             developing brains of male and female mice. DEP exposure
             increased inflammatory cytokine protein and altered the
             morphology of microglia, consistent with activation or a
             delay in maturation, only within the embryonic brains of
             male mice; and these effects were dependent on TLR4. DEP
             exposure also increased cortical volume at embryonic day
             (E)18, which switched to decreased volume by post-natal day
             (P)30 in males, suggesting an impact on the developing
             neural stem cell niche. Consistent with this hypothesis, we
             found increased microglial-neuronal interactions in male
             offspring that received DEP compared to all other groups.
             Taken together, these data suggest a mechanism by which
             prenatal exposure to environmental toxins may affect
             microglial development and long-term function, and thereby
             contribute to the risk of neurodevelopmental
             disorders.},
   Doi = {10.3389/fnsyn.2017.00010},
   Key = {fds326607}
}

@article{fds326608,
   Author = {Bilbo, SD},
   Title = {Sex differences in microglial appetites during development:
             Inferences and implications.},
   Journal = {Brain, Behavior, and Immunity},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.bbi.2017.05.010},
   Doi = {10.1016/j.bbi.2017.05.010},
   Key = {fds326608}
}

@article{fds326354,
   Author = {Parker, W and Hornik, CD and Bilbo, S and Holzknecht, ZE and Gentry, L and Rao, R and Lin, SS and Herbert, MR and Nevison, CD},
   Title = {The role of oxidative stress, inflammation and acetaminophen
             exposure from birth to early childhood in the induction of
             autism.},
   Journal = {The Journal of international medical research},
   Volume = {45},
   Number = {2},
   Pages = {407-438},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1177/0300060517693423},
   Abstract = {The wide range of factors associated with the induction of
             autism is invariably linked with either inflammation or
             oxidative stress, and sometimes both. The use of
             acetaminophen in babies and young children may be much more
             strongly associated with autism than its use during
             pregnancy, perhaps because of well-known deficiencies in the
             metabolic breakdown of pharmaceuticals during early
             development. Thus, one explanation for the increased
             prevalence of autism is that increased exposure to
             acetaminophen, exacerbated by inflammation and oxidative
             stress, is neurotoxic in babies and small children. This
             view mandates extreme urgency in probing the long-term
             effects of acetaminophen use in babies and the possibility
             that many cases of infantile autism may actually be induced
             by acetaminophen exposure shortly after birth.},
   Doi = {10.1177/0300060517693423},
   Key = {fds326354}
}

@article{fds325332,
   Author = {Kopec, AM and Rivera, PD and Lacagnina, MJ and Hanamsagar, R and Bilbo,
             SD},
   Title = {Optimized solubilization of TRIzol-precipitated protein
             permits Western blotting analysis to maximize data available
             from brain tissue.},
   Journal = {Journal of Neuroscience Methods},
   Volume = {280},
   Pages = {64-76},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.jneumeth.2017.02.002},
   Abstract = {Techniques simultaneously assessing multiple levels of
             molecular processing are appealing because molecular
             signaling underlying complex neural phenomena occurs at
             complementary levels. The TRIzol method isolates RNA and
             DNA, but protein retrieval is difficult due to inefficient
             solubilization of precipitated protein pellets.We optimized
             a buffer for the efficient solubilization of protein from
             TRIzol-precipitated brain tissue for Western blotting
             analysis, which was also more effective at directly
             homogenizing brain tissue than RIPA buffer.Protein yield
             during solubilization, in addition to protein yield via
             direct homogenization, is increased by optimizing
             concentrations of chemicals in a standard lysis buffer.
             Effective incubation parameters for both total protein yield
             and the analysis of post-translational modifications is
             remarkably flexible. Importantly, different neural cell
             types and protein classes are represented in solubilized
             protein samples. Moreover, we used dissociated mouse brain
             tissue to isolate microglia from other cell types and
             successfully resolved cell type-specific proteins from these
             small and difficult to attain samples.Solubilization buffers
             to date have been comprised primarily of SDS or urea; the
             data herein demonstrate that components common to lysis
             buffers can also enhance protein solubilization both after
             direct homogenization and after precipitation.This method is
             suitable for assessing gene and protein expression from a
             single brain sample, allowing for a more comprehensive
             evaluation of neural phenomena while minimizing the number
             of subjects.},
   Doi = {10.1016/j.jneumeth.2017.02.002},
   Key = {fds325332}
}

@article{fds321831,
   Author = {Lacagnina, MJ and Rivera, PD and Bilbo, SD},
   Title = {Glial and Neuroimmune Mechanisms as Critical Modulators of
             Drug Use and Abuse.},
   Journal = {Neuropsychopharmacology (Nature)},
   Volume = {42},
   Number = {1},
   Pages = {156-177},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1038/npp.2016.121},
   Abstract = {Drugs of abuse cause persistent alterations in synaptic
             plasticity that may underlie addiction behaviors. Evidence
             suggests glial cells have an essential and underappreciated
             role in the development and maintenance of drug abuse by
             influencing neuronal and synaptic functions in multifaceted
             ways. Microglia and astrocytes perform critical functions in
             synapse formation and refinement in the developing brain,
             and there is growing evidence that disruptions in glial
             function may be implicated in numerous neurological
             disorders throughout the lifespan. Linking evidence of
             function in health and under pathological conditions, this
             review will outline the glial and neuroimmune mechanisms
             that may contribute to drug-abuse liability, exploring
             evidence from opioids, alcohol, and psychostimulants. Drugs
             of abuse can activate microglia and astrocytes through
             signaling at innate immune receptors, which in turn
             influence neuronal function not only through secretion of
             soluble factors (eg, cytokines and chemokines) but also
             potentially through direct remodeling of the synapses. In
             sum, this review will argue that neural-glial interactions
             represent an important avenue for advancing our
             understanding of substance abuse disorders.},
   Doi = {10.1038/npp.2016.121},
   Key = {fds321831}
}


%% Blumenthal, James A.   
@article{fds336044,
   Author = {Li, S and Blumenthal, JA and Shi, C and Millican, D and Li, X and Du, X and Patel, A and Gao, P and Delong, E and Maulik, PK and Gao, R and Yu, X and Wu,
             Y},
   Title = {I-CARE randomized clinical trial integrating depression and
             acute coronary syndrome care in low-resource hospitals in
             China: Design and rationale.},
   Journal = {American Heart Journal},
   Volume = {202},
   Pages = {109-115},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.ahj.2018.05.004},
   Abstract = {Depression and acute coronary syndromes (ACS) are both
             common public health challenges. Patients with ACS often
             develop depression, which in turn adversely affects
             prognosis. Low-cost, sustainable, and effective service
             models that integrate depression care into the management of
             ACS patients to reduce depression and improve ACS outcomes
             are critically needed. Integrating Depression Care in ACS
             patients in Low Resource Hospitals in China (I-CARE) is a
             multicenter, randomized controlled trial to evaluate the
             efficacy of an 11-month integrated care (IC) intervention
             compared to usual care (UC) in management of ACS patients.
             Four thousand inpatients will be recruited and then
             randomized in a 1:1 ratio to an IC intervention consisting
             of nurse-led risk factor management, group-based counseling
             supplemented by individual problem-solving therapy, and
             antidepressant medications as needed, or to UC. The primary
             outcomes are depression symptoms measured by the Patient
             Health Questionnaire-9 at 6 and 12 months. Secondary
             endpoints include anxiety measured by the Generalized
             Anxiety Disorder-7; quality of life measured by the EQ-5D at
             6 and 12 months; and major adverse events including the
             combined end point of all-cause death, suicide attempts,
             nonfatal myocardial infarction, nonfatal stroke, and
             all-cause rehospitalization at yearly intervals for a median
             follow-up of 2 years. Analyses of the cost-effectiveness
             and cost-utility of IC also will be performed. I-CARE trial
             will be the largest study to test the effectiveness of an
             integrated care model on depression and cardiovascular
             outcomes among ACS patients in resource-limited clinical
             settings.},
   Doi = {10.1016/j.ahj.2018.05.004},
   Key = {fds336044}
}

@article{fds336045,
   Author = {Farquhar, JM and Stonerock, GL and Blumenthal,
             JA},
   Title = {Treatment of Anxiety in Patients With Coronary Heart
             Disease: A Systematic Review.},
   Journal = {Psychosomatics},
   Volume = {59},
   Number = {4},
   Pages = {318-332},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.psym.2018.03.008},
   Abstract = {Anxiety is common in patients with coronary heart disease
             (CHD) and is associated with an increased risk for adverse
             outcomes. There has been a relative paucity of studies
             concerning treatment of anxiety in patients with CHD.We
             conducted a systematic review to organize and assess
             research into the treatment of anxiety in patients with
             CHD.We searched CCTR/CENTRAL, MEDLINE, EMBASE, PsycINFO, and
             CINAHL for randomized clinical trials conducted before
             October 2016 that measured anxiety before and after an
             intervention for patients with CHD.A total of 475 articles
             were subjected to full text review, yielding 112
             publications that met inclusion criteria plus an additional
             7 studies from reference lists and published reviews,
             yielding 119 studies. Sample size, country of origin, study
             quality, and demographics varied widely among studies. Most
             studies were conducted with nonanxious patients. The
             Hospital Anxiety and Depression Scale and State-Trait
             Anxiety Inventory were the most frequently used instruments
             to assess anxiety. Interventions included pharmacological,
             counseling, relaxation-based, educational, or "alternative"
             therapies. Forty (33% of total) studies reported that the
             interventions reduced anxiety; treatment efficacy varied by
             study and type of intervention. Elevated anxiety was an
             inclusion criterion in only 4 studies, with inconsistent
             results.Although there have been a number of randomized
             clinical trials of patients with CHD that assessed anxiety,
             in most cases anxiety was a secondary outcome, and only
             one-third found that symptoms of anxiety were reduced with
             treatment. Future studies need to target anxious patients
             and evaluate the effects of treatment on anxiety and
             relevant clinical endpoints.},
   Doi = {10.1016/j.psym.2018.03.008},
   Key = {fds336045}
}

@article{fds332217,
   Author = {Smith, PJ and Sherwood, A and Blumenthal, JA},
   Title = {Effects of aerobic exercise on white matter
             hyperintensities: An exploratory analysis.},
   Journal = {General Hospital Psychiatry},
   Volume = {53},
   Pages = {84-85},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.genhosppsych.2018.01.007},
   Doi = {10.1016/j.genhosppsych.2018.01.007},
   Key = {fds332217}
}

@article{fds337029,
   Author = {Smith, PJ and Blumenthal, JA and Hinderliter, AL and Watkins, LL and Hoffman, BM and Sherwood, A},
   Title = {Microvascular Endothelial Function and Neurocognition Among
             Adults With Major Depressive Disorder.},
   Journal = {American Journal of Geriatric Psychiatry},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.jagp.2018.06.011},
   Abstract = {Cardiovascular risk factors (CVRFs) and endothelial
             dysfunction have been associated independently with poorer
             neurocognition in middle-aged adults, particularly on tests
             of frontal lobe function. However, to our knowledge, no
             studies have examined markers of microvascular dysfunction
             on neurocognition or the potential interaction between
             macro- and microvascular biomarkers on neurocognition in
             middle-aged and older adults with major depressive disorder
             (MDD).Participants included 202 adults with MDD who were not
             receiving mental health treatment. Microvascular endothelial
             function was assessed using a noninvasive marker of forearm
             reactive hyperemia velocity while macrovascular endothelial
             function was assessed using flow-mediated dilation (FMD) of
             the brachial artery. CVRFs were assessed using the
             Framingham Stroke Risk Profile and fasting lipid levels. A
             standardized neurocognitive assessment battery was used to
             assess three cognitive domains: executive function, working
             memory, and verbal memory.Greater microvascular dysfunction
             was associated with poorer neurocognition across all three
             domains. Microvascular function continued to predict verbal
             memory performance after accounting for background factors
             and CVRFs. Macro- and microvascular function interacted to
             predict working memory performance (F = 4.511, 178,
             p = 0.035), with a similar nonsignificant association
             for executive function (F = 2.731, 178, p = 0.095),
             with moderate associations observed between microvascular
             function and neurocognition in the presence of preserved FMD
             (r61 = 0.40, p = 0.001), but not when FMD was
             impaired (r63 = -0.05, p = 0.675).Greater
             microvascular dysfunction is associated with poorer
             neurocognition among middle-aged and older adults. This
             association was strongest in participants with preserved
             macrovascular function.},
   Doi = {10.1016/j.jagp.2018.06.011},
   Key = {fds337029}
}

@article{fds336046,
   Author = {Zhu, Y and Blumenthal, JA and Shi, C and Jiang, R and Patel, A and Zhang,
             A and Yu, X and Gao, R and Wu, Y},
   Title = {Sedentary Behavior and the Risk of Depression in Patients
             With Acute Coronary Syndromes.},
   Journal = {The American Journal of Cardiology},
   Volume = {121},
   Number = {12},
   Pages = {1456-1460},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.amjcard.2018.02.031},
   Abstract = {Although there is good evidence that sedentary behavior is
             associated with poor health outcomes in healthy persons and
             patients with cardiovascular disease, the mental health
             consequences of sedentary behavior have not been widely
             studied. In this report, we conducted a cross-sectional
             analysis to examine the relation of self-reported sedentary
             behavior and depression in a sample of 4,043 hospitalized
             men and women with acute coronary syndrome enrolled in a
             randomized clinical trial in rural China. Sedentary behavior
             was assessed by self-report, and depression was assessed
             with the Patient Health Questionnaire-9 (PHQ-9); a subset of
             1,209 patients also completed the Beck Depression
             Inventory-II. Results revealed that greater sedentary
             behavior was associated with higher levels of depressive
             symptoms measured by both the PHQ-9 (p <0.001) and the
             Beck Depression Inventory-II (p <0.001). Compared with
             patients who reported that they were seldom sedentary,
             patients reporting that they were frequently sedentary were
             4.7 times (odds ratio 4.73, 95% confidence interval 2.71 to
             8.24) more likely to be clinically depressed defined as
             PHQ-9 scores ≥10 after adjusting for demographic factors,
             lifestyle behaviors, clinical characteristics, and
             in-hospital treatments. In conclusion, greater sedentary
             behavior is significantly related to greater depression in
             Chinese patients with acute coronary syndrome, independent
             of physical activity. These findings suggest that strategies
             to reduce sedentary behavior may improve medical outcomes
             and reduce risk for depression.},
   Doi = {10.1016/j.amjcard.2018.02.031},
   Key = {fds336046}
}

@article{fds336047,
   Author = {Farquhar, JM and Stonerock, GL and Blumenthal,
             JA},
   Title = {When Something is Better Than Nothing: Response to Tully and
             Cosh.},
   Journal = {Psychosomatics},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.psym.2018.05.001},
   Doi = {10.1016/j.psym.2018.05.001},
   Key = {fds336047}
}

@article{fds331180,
   Author = {Smith, PJ and Snyder, LD and Palmer, SM and Hoffman, BM and Stonerock,
             GL and Ingle, KK and Saulino, CK and Blumenthal, JA},
   Title = {Depression, social support, and clinical outcomes following
             lung transplantation: a single-center cohort
             study.},
   Journal = {Transplant International : Official Journal of the European
             Society for Organ Transplantation},
   Volume = {31},
   Number = {5},
   Pages = {495-502},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1111/tri.13094},
   Abstract = {Depressive symptoms are common among lung transplant
             candidates and have been associated with poorer clinical
             outcomes in some studies. Previous studies have been plagued
             by methodologic problems, including small sample sizes, few
             clinical events, and uncontrolled confounders, particularly
             perioperative complications. In addition, few studies have
             examined social support as a potential protective factor. We
             therefore examined the association between pretransplant
             depressive symptoms, social support, and mortality in a
             large sample of lung transplant recipients. As a secondary
             aim, we also examined the associations between psychosocial
             factors, perioperative outcomes [indexed by hospital length
             of stay (LOS)], and mortality. We hypothesized that
             depression would be associated with longer LOS and that the
             association between depression, social support, and
             mortality would be moderated by LOS. Participants included
             lung transplant recipients, transplanted at Duke University
             Medical Center from January 2009 to December 2014.
             Depressive symptoms were evaluated using the Beck Depression
             Inventory (BDI-II) and social support using the Perceived
             Social Support Scale (PSSS). Medical risk factors included
             forced vital capacity (FVC), partial pressure of carbon
             dioxide (PCO2 ), donor age, acute rejection, and transplant
             type. Functional status was assessed using six-minute walk
             distance (6MWD). We also controlled for demographic factors,
             including age, gender, and native disease. Transplant
             hospitalization LOS was examined as a marker of
             perioperative clinical outcomes. Participants included 273
             lung recipients (174 restrictive, 67 obstructive, 26 cystic
             fibrosis, and six "other"). Pretransplant depressive
             symptoms were common, with 56 participants (21%) exhibiting
             clinically elevated levels (BDI-II ≥ 14). Greater
             depressive symptoms were associated with longer LOS
             [adjusted b = 0.20 (2 days per 7-point higher BDI-II score),
             P < 0.01]. LOS moderated the associations between depressive
             symptoms (P = 0.019), social support (P < 0.001), and
             mortality, such that greater depressive symptoms and lower
             social support were associated with greater mortality only
             among individuals with longer LOS. For individuals with LOS
             ≥ 1 month, clinically elevated depressive symptoms (BDI-II
             ≥ 14) were associated with a threefold increased risk of
             mortality (HR = 2.97). Greater pretransplant depressive
             symptoms and lower social support may be associated with
             greater mortality among a subset of individuals with worse
             perioperative outcomes.},
   Doi = {10.1111/tri.13094},
   Key = {fds331180}
}

@article{fds333684,
   Author = {Blumenthal, JA and Sherwood, A and Smith, PJ and Hinderliter,
             A},
   Title = {The Role of Salt Reduction in the Management of
             Hypertension},
   Journal = {Journal of the American College of Cardiology},
   Volume = {71},
   Number = {14},
   Pages = {1597-1598},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jacc.2018.01.071},
   Doi = {10.1016/j.jacc.2018.01.071},
   Key = {fds333684}
}

@article{fds336050,
   Author = {Smith, PJ and Stonerock, GL and Ingle, KK and Saulino, CK and Hoffman,
             B and Wasserman, B and Blumenthal, JA and Palmer, SM and Klapper, JA and Hartwig, MG and Esposito, VR and Snyder, LD},
   Title = {Neurological Sequelae and Clinical Outcomes After Lung
             Transplantation.},
   Journal = {Transplantation Direct},
   Volume = {4},
   Number = {4},
   Pages = {e353},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1097/TXD.0000000000000766},
   Abstract = {Neurological complications are common after lung
             transplantation. However, no large cohort studies have
             examined the incidence, predictors, and clinical
             significance of neurological events sustained by lung
             transplant recipients.We conducted a retrospective cohort
             analysis of a consecutive series of lung transplant
             recipients, transplanted at Duke University Medical Center
             between May 2014 and February 2017 (n = 276). Early
             neurological complications (ie, occurring during the first
             week after transplant) were documented by transplant mental
             health specialists and included delirium, ischemic injury,
             and posterior reversible encephalopathy syndrome. Analyses
             accounted for age, native disease, sex, type of transplant,
             lung allocation score, and primary graft dysfunction. The
             objectives of the study were to characterize the prevalence
             and predictors of early neurological sequelae (NSE),
             occurring during the first week posttransplant, and the
             association between NSE and subsequent clinical outcomes,
             including length of stay and mortality.Neurological sequelae
             were common, occurring in 123 (45%) patients. Fifty-seven
             patients died over a follow-up interval of 2.1 years. The
             most common NSE were postoperative delirium (n = 110 [40%])
             and posterior reversible encephalopathy syndrome (n = 12
             [4%]), followed by stroke/transient ischemic attack and
             neurotoxicity. Higher lung allocation score was the
             strongest predictor of delirium. The presence of a NSE was
             associated with longer length of hospital stay (32 days vs
             17 days, P < 0.001) and greater mortality (hazard ratio,
             1.90; 95% confidence interval, 1.09-3.32], P = 0.024), with
             the greatest mortality risk occurring approximately 2 years
             after transplantation.Neurological events are relatively
             common after lung transplantation and associated with
             adverse clinical outcomes.},
   Doi = {10.1097/TXD.0000000000000766},
   Key = {fds336050}
}

@article{fds336048,
   Author = {Chu, MC and Smith, PJ and Stonerock, GL and Blumenthal,
             JA},
   Title = {DEPRESSION AND MEDICATION NONADHERENCE FOLLOWING LUNG
             TRANSPLANTATION},
   Journal = {Annals of Behavioral Medicine},
   Volume = {52},
   Pages = {S560-S560},
   Publisher = {OXFORD UNIV PRESS INC},
   Year = {2018},
   Month = {April},
   Key = {fds336048}
}

@article{fds336049,
   Author = {Smith, PJ and Blumenthal, JA and Hinderliter, A and Sherwood,
             A},
   Title = {MICROVASCULAR FUNCTION AND COGNITIVE PERFORMANCE AMONG
             INDIVIDUALS WITH MAJOR DEPRESSION},
   Journal = {Psychosomatic Medicine},
   Volume = {80},
   Number = {3},
   Pages = {A123-A124},
   Publisher = {LIPPINCOTT WILLIAMS & WILKINS},
   Year = {2018},
   Month = {April},
   Key = {fds336049}
}

@article{fds330458,
   Author = {Smith, PJ and Blumenthal, JA and Hoffman, BM and Davis, RD and Palmer,
             SM},
   Title = {Postoperative cognitive dysfunction and mortality following
             lung transplantation.},
   Journal = {American Journal of Transplantation : Official Journal of
             the American Society of Transplantation and the American
             Society of Transplant Surgeons},
   Volume = {18},
   Number = {3},
   Pages = {696-703},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1111/ajt.14570},
   Abstract = {Preliminary evidence suggests that postoperative cognitive
             dysfunction (POCD) is common after lung transplantation. The
             impact of POCD on clinical outcomes has yet to be studied.
             The association between POCD and longer-term survival was
             therefore examined in a pilot study of posttransplantation
             survivors. Forty-nine participants from a prior randomized
             clinical trial underwent a neurocognitive assessment battery
             pretransplantation and 6 months posttransplantation,
             including assessments of the domains of Executive Function
             (Trail Making Test, Stroop, Digit Span), Processing Speed
             (Ruff 2 and 7 Test, Digit Symbol Substitution Test), and
             Verbal Memory (Verbal Paired Associates, Logical Memory,
             Animal Naming, and Controlled Oral Word Association Test).
             During a 13-year follow-up, 33 (67%) participants died.
             Greater neurocognition was associated with longer survival
             (hazard ratio [HR] = 0.49 [0.25-0.96], P = .039), and
             this association was strongest on tests assessing Processing
             Speed (HR = 0.58 [0.36-0.95], P = .03) and Executive
             Function (HR = 0.52 [0.28-0.97], P = .040). In addition,
             unadjusted analyses suggested an association between greater
             Memory performance and lower risk of CLAD (HR = 0.54
             [0.29-1.00], P = .050). Declines in Executive Function
             tended to be predictive of worse survival. These preliminary
             findings suggest that postoperative neurocognition is
             predictive of subsequent mortality among lung transplant
             recipients. Further research is needed to confirm these
             findings in a larger sample and to examine mechanisms
             responsible for this relationship.},
   Doi = {10.1111/ajt.14570},
   Key = {fds330458}
}

@article{fds329487,
   Author = {Sherwood, A and Hill, LK and Blumenthal, JA and Hinderliter,
             AL},
   Title = {Circadian hemodynamics in men and women with high blood
             pressure: dipper vs. nondipper and racial
             differences.},
   Journal = {Journal of Hypertension},
   Volume = {36},
   Number = {2},
   Pages = {250-258},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1097/hjh.0000000000001533},
   Abstract = {The 'nondipping' pattern of circadian blood pressure (BP)
             variation is an established independent predictor of adverse
             cardiovascular outcomes. Although this phenomenon has been
             widely studied, its underlying circadian hemodynamics of
             cardiac output and systemic vascular resistance (SVR) have
             not been well characterized. We evaluated the hypothesis
             that BP nondipping would be associated with a blunted
             night-time reduction in SVR in a biracial sample of 140 (63
             African-American and 77 white) men and women with elevated
             clinic BP (130-159/85-99 mmHg).Twenty-four-hour ambulatory
             hemodynamics were assessed using standard ambulatory BP
             monitoring coupled with synchronized ambulatory impedance
             cardiography. Using the criterion of less than 10% dip in
             SBP, there were 51 nondippers (SBP dip = 7.3 ± 2.6%)
             and 89 dippers (SBP dip = 15.5 ± 3.4%). There was
             minimal change in cardiac output from daytime to night-time
             in both dippers and nondippers. However, SVR decreased from
             daytime to night-time, but nondippers compared with dippers
             exhibited a significantly attenuated decrease in SVR from
             daytime to night-time (7.8 vs. 16.1%, P < 0.001).
             Relative to their white counterparts, African-Americans also
             exhibited blunted SBP dipping (10.9 vs. 14.6%,
             P < 0.001) as well as an attenuated decrease in SVR
             (10.8 vs. 15.6%, P < 0.001).Overall, these findings
             indicate that blunted night-time BP dipping is associated
             with impairment of the systemic vasodilation that is
             characteristic of the night-time sleep period and is
             especially prominent among African-Americans. In the context
             of high BP, these findings suggest that nondipping may be a
             manifestation, or marker, of more advanced vascular
             disease.},
   Doi = {10.1097/hjh.0000000000001533},
   Key = {fds329487}
}

@article{fds336051,
   Author = {Blumenthal, JA and Sherwood, A},
   Title = {Missing ingredients for a lifestyle recipe to treat
             hypertension.},
   Journal = {Journal of Thoracic Disease},
   Volume = {10},
   Number = {2},
   Pages = {653-656},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.21037/jtd.2018.01.06},
   Doi = {10.21037/jtd.2018.01.06},
   Key = {fds336051}
}

@article{fds331882,
   Author = {Hill, LK and Sherwood, A and McNeilly, M and Anderson, NB and Blumenthal, JA and Hinderliter, AL},
   Title = {Impact of Racial Discrimination and Hostility on Adrenergic
             Receptor Responsiveness in African American
             Adults.},
   Journal = {Psychosomatic Medicine},
   Volume = {80},
   Number = {2},
   Pages = {208-215},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1097/psy.0000000000000547},
   Abstract = {Racial discrimination is increasingly recognized as a
             contributor to increased cardiovascular disease (CVD) risk
             among African Americans. Previous research has shown
             significant overlap between racial discrimination and
             hostility, an established predictor of CVD risk including
             alterations in adrenergic receptor functioning. The present
             study examined the associations of racial discrimination and
             hostility with adrenergic receptor responsiveness.In a
             sample (N = 57) of young to middle-aged African American
             adults (51% female) with normal and mildly elevated blood
             pressure, a standardized isoproterenol sensitivity test
             (CD25) was used to evaluate β-AR responsiveness, whereas
             the dose of phenylephrine required to increase mean arterial
             pressure by 25 mm Hg (PD25) was used to assess α1-AR
             responsiveness. Racial discrimination was measured using the
             Perceived Racism Scale and hostility was assessed using the
             Cook-Medley Hostility Scale.In hierarchical regression
             models, greater racial discrimination, but not hostility,
             emerged as a significant predictor of decreased
             β-adrenergic receptor responsiveness (β = .38, p = .004).
             However, moderation analysis revealed that the association
             between racial discrimination and blunted β-adrenergic
             receptor responsiveness was strongest among those with
             higher hostility (β = .49, 95% confidence interval =
             .17-.82, p = .004). In addition, hostility, but not racial
             discrimination, significantly predicted α1-AR
             responsiveness.These findings suggest racial discrimination
             was associated with blunted β-adrenergic receptor
             responsiveness, providing further evidence of the potential
             contribution of racial discrimination to increased CVD risk
             among African Americans. The adverse effects of
             discrimination on cardiovascular health may be enhanced in
             individuals with higher levels of hostility.},
   Doi = {10.1097/psy.0000000000000547},
   Key = {fds331882}
}

@article{fds330457,
   Author = {Farquhar, JM and Smith, PJ and Snyder, L and Gray, AL and Reynolds, JM and Blumenthal, JA},
   Title = {Patterns and predictors of pain following lung
             transplantation.},
   Journal = {General Hospital Psychiatry},
   Volume = {50},
   Pages = {125-130},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.genhosppsych.2017.11.007},
   Abstract = {Our objective was to examine variability in pain levels
             following lung transplantation, and examine individual
             biopsychosocial factors influencing changes in pain.We
             performed a retrospective study of a cohort of 150 patients
             transplanted and discharged from Duke University Hospital
             between January 2015 and September 2016. During
             hospitalization and at clinic visits up to two months after
             discharge, subjective pain ratings were obtained using a
             0-10 Numeric Rating Scale. Psychiatric diagnoses of anxiety
             and depression and Center for Epidemiological Studies -
             Depression (CES-D) scores collected after hospital discharge
             were examined as predictors of post-surgery pain. Medical
             and surgical variables were examined as covariates.During
             hospitalization, pain ratings decreased over time (p<0.001).
             Predictors of higher pain levels included pre-transplant
             history of depression (p=0.001) and anxiety (p=0.04),
             bilateral lung transplant (p=0.03), and lower six-minute
             walk distance (p=0.02). Two months after discharge, 18% of
             patients reported continued pain and 34% remained on opioid
             pain medications. Two months after discharge, more frequent
             post-operative complications predicted higher pain levels in
             a univariate analysis (p=0.02) although this relationship
             was attenuated after adjustment for depression. In a
             multivariate analysis, elevated CES-D scores (p=0.002), and
             greater opioid use (p=0.031) predicted higher pain levels
             2-months post-discharge.We conclude that patients with
             psychiatric comorbidities may be at risk for greater pain,
             and may require additional strategies for more effective
             pain management.},
   Doi = {10.1016/j.genhosppsych.2017.11.007},
   Key = {fds330457}
}

@article{fds330347,
   Author = {Nieuwsma, JA and Williams, JW and Namdari, N and Washam, JB and Raitz,
             G and Blumenthal, JA and Jiang, W and Yapa, R and McBroom, AJ and Lallinger, K and Schmidt, R and Kosinski, AS and Sanders,
             GD},
   Title = {Diagnostic Accuracy of Screening Tests and Treatment for
             Post-Acute Coronary Syndrome Depression: A Systematic
             Review.},
   Journal = {Annals of Internal Medicine},
   Volume = {167},
   Number = {10},
   Pages = {725-735},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.7326/m17-1811},
   Abstract = {Patients who have had an acute coronary syndrome (ACS) event
             have an increased risk for depression.To evaluate the
             diagnostic accuracy of depression screening instruments and
             to compare safety and effectiveness of depression treatments
             in adults within 3 months of an ACS event.MEDLINE, EMBASE,
             PsycINFO, CINAHL, and Cochrane Database of Systematic
             Reviews from January 2003 to August 2017, and a manual
             search of citations from key primary and review
             articles.English-language studies of post-ACS patients that
             evaluated the diagnostic accuracy of depression screening
             tools or compared the safety and effectiveness of a broad
             range of pharmacologic and nonpharmacologic depression
             treatments.2 investigators independently screened each
             article for inclusion; abstracted the data; and rated the
             quality, applicability, and strength of evidence.Evidence
             from 6 of the 10 included studies showed that a range of
             depression screening instruments produces acceptable levels
             of diagnostic sensitivity, specificity, and negative
             predictive values (70% to 100%) but low positive predictive
             values (below 50%). The Beck Depression Inventory-II was the
             most studied tool. A large study found that a combination of
             cognitive behavioral therapy (CBT) and antidepressant
             medication improved depression symptoms, mental
             health-related function, and overall life satisfaction more
             than usual care.Few studies, no evaluation of the influence
             of screening on clinical outcomes, and no studies addressing
             several clinical interventions of interest.Depression
             screening instruments produce diagnostic accuracy metrics
             that are similar in post-ACS patients and other clinical
             populations. Depression interventions have an uncertain
             effect on cardiovascular outcomes, but CBT combined with
             antidepressant medication produces modest improvement in
             psychosocial outcomes.Agency for Healthcare Research and
             Quality (PROSPERO: CRD42016047032).},
   Doi = {10.7326/m17-1811},
   Key = {fds330347}
}

@article{fds331883,
   Author = {Smith, PJ and Sherwood, A and Mabe, S and Watkins, L and Hinderliter, A and Blumenthal, JA},
   Title = {Physical activity and psychosocial function following
             cardiac rehabilitation: One-year follow-up of the ENHANCED
             study.},
   Journal = {General Hospital Psychiatry},
   Volume = {49},
   Pages = {32-36},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.genhosppsych.2017.05.001},
   Abstract = {To examine the long-term association between physical
             activity (PA) and psychosocial functioning following
             completion of cardiac rehabilitation (CR) among participants
             in the ENHANCED study.ENHANCED was a 3-month clinical trial
             examining standard CR with and without stress management
             training (SMT). Participants completed the Leisure-Time
             Exercise Questionnaire (LTEQ), Beck Depression Inventory-II
             (BDI-II), and the Spielberger State-Trait Anxiety Inventory
             (STAI) at baseline, 3- and 12-months after randomization.
             Prospective associations between physical activity and
             psychosocial function, as well as treatment group
             differences, were examined using repeated measures mixed
             modeling.One hundred nineteen participants were available at
             12month follow-up. During the 12-month follow-up, PA
             remained higher compared to baseline (P<0.001), with the
             majority of participants reporting that they engaged in PA
             on a regular basis. Depressive and anxious symptoms
             continued to remain lower than baseline (Ps<0.001) Higher
             levels of PA at 12-month follow-up were associated with
             lower depressive (P=0.032) and anxious (P=0.003)
             symptoms.Higher physical activity levels following CR are
             associated with lower depressive and anxious symptoms.
             Encouraging patients to sustain higher levels of PA may
             promote both cardiovascular and mental health.},
   Doi = {10.1016/j.genhosppsych.2017.05.001},
   Key = {fds331883}
}

@article{fds330037,
   Author = {Tyson, CC and Smith, PJ and Sherwood, A and Mabe, S and Hinderliter, AL and Blumenthal, JA},
   Title = {Association between normal or mildly reduced kidney
             function, cardiovascular risk and biomarkers for
             atherosclerosis: results from the ENCORE
             trial.},
   Journal = {Clinical Kidney Journal},
   Volume = {10},
   Number = {5},
   Pages = {666-671},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1093/ckj/sfx025},
   Abstract = {Moderate-to-severe kidney dysfunction is associated with
             atherosclerotic cardiovascular disease (ASCVD). Gradations
             of normal or mildly reduced kidney function may also
             associate with ASCVD risk.We conducted a secondary analysis
             using baseline data from the Exercise and Nutritional
             Interventions for Cardiovascular Health (ENCORE) trial.
             Participants were sedentary, overweight and obese adults
             with unmedicated pre-hypertension or Stage I hypertension
             and an estimated glomerular filtration rate (eGFR) ≥60
             mL/min/1.73 m2. The Pooled Cohorts Equations were used to
             estimate a 10-year risk for first ASCVD event. Carotid
             artery intima-media thickness (IMT) and brachial artery
             flow-mediated dilation (FMD) were measured to assess
             subclinical atherosclerosis and vascular endothelial
             function, respectively. Using linear regression, we examined
             the association between eGFR and ASCVD risk, IMT and
             FMD.Participants (N = 139) were predominantly women
             (65%), white (60%), with a mean age of 52.0 ± 9.6 years
             and mean eGFR of 89.1 ± 15.0 mL/min/1.73 m2. Lower
             eGFR of 15 mL/min/1.73 m2 was associated with higher
             ASCVD risk [b = -2.7% (95% confidence interval: -3.7,
             -1.8%), P < 0.001], higher IMT [b = 0.05 mm (0.03,
             0.08 mm), P < 0.001] and lower FMD [b = -0.87%
             (-1.64, -0.11%), P = 0.026]. Compared with
             eGFR ≥90 mL/min/1.73 m2, those with eGFR
             60-89 mL/min/1.73 m2 had higher mean ASCVD risk (7.6%
             versus 2.7%; P < 0.001), greater mean IMT (0.74 mm
             versus 0.66 mm; P < 0.001) and lower mean FMD (2.0%
             versus 3.7%; P = 0.026). After controlling for CVD risk
             factors, the association between eGFR and IMT remained
             significant (P < 0.001), and eGFR and FMD trended toward
             significance (P = 0.08).Among overweight and obese
             adults with unmedicated high blood pressure and
             eGFR ≥60 mL/min/1.73 m2, lower eGFR is associated
             with a greater 10-year risk for first ASCVD event, higher
             IMT and relatively impaired FMD.},
   Doi = {10.1093/ckj/sfx025},
   Key = {fds330037}
}

@article{fds327848,
   Author = {Sherwood, A and Hill, LK and Blumenthal, JA and Adams, KF and Paine, NJ and Koch, GG and O'Connor, CM and Johnson, KS and Hinderliter,
             AL},
   Title = {Blood pressure reactivity to psychological stress is
             associated with clinical outcomes in patients with heart
             failure.},
   Journal = {American Heart Journal},
   Volume = {191},
   Pages = {82-90},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.ahj.2017.07.003},
   Abstract = {Cardiovascular (CV) reactivity to psychological stress has
             been implicated in the development and exacerbation of
             cardiovascular disease (CVD). Although high CV reactivity
             traditionally is thought to convey greater risk of CVD, the
             relationship between reactivity and clinical outcomes is
             inconsistent and may depend on the patient population under
             investigation. The present study examined CV reactivity in
             patients with heart failure (HF) and its potential
             association with long-term clinical outcomes.One hundred
             ninety-nine outpatients diagnosed with HF, with ejection
             fraction ≤40%, underwent an evaluation of blood pressure
             (BP) and heart rate reactivity to a laboratory-based
             simulated public-speaking stressor. Cox proportional hazards
             regression models were used to examine the prospective
             association between BP and heart rate reactivity on a
             combined end point of death or CV hospitalization over a
             5-year median follow-up period.Both systolic blood pressure
             (SBP) and diastolic blood pressure (DBP) reactivity,
             quantified as continuous variables, were inversely related
             to risk of death or CV hospitalization (Ps < .01) after
             controlling for established risk factors, including HF
             disease severity and etiology. In similar models, heart rate
             reactivity was unrelated to outcome (P = .12). In models
             with tertiles of reactivity, high SBP reactivity, compared
             with intermediate SBP reactivity, was associated with lower
             risk (hazard ratio [HR] = .498, 95% CI .335-.742, P =.001),
             whereas low SBP reactivity did not differ from intermediate
             reactivity. For DBP, high reactivity was marginally
             associated with lower risk compared with intermediate DBP
             reactivity (HR = .767, 95% CI .515-1.14, P =.193), whereas
             low DBP reactivity was associated with greater risk (HR =
             1.49, 95% CI 1.027-2.155, P =.0359). No relationship of
             heart rate reactivity to outcome was identified.For HF
             patients with reduced ejection fraction, a robust increase
             in BP evoked by a laboratory-based psychological challenge
             was associated with lower risk for adverse CVD events and
             may be a novel and unique marker of left ventricular
             systolic reserve that is accompanied by a more favorable
             long-term prognosis.},
   Doi = {10.1016/j.ahj.2017.07.003},
   Key = {fds327848}
}

@article{fds330038,
   Author = {Hayano, J and Yasuma, F and Watanabe, E and Carney, RM and Stein, PK and Blumenthal, JA and Arsenos, P and Gatzoulis, KA and Takahashi, H and Ishii, H and Kiyono, K and Yamamoto, Y and Yoshida, Y and Yuda, E and Kodama, I},
   Title = {Blunted cyclic variation of heart rate predicts mortality
             risk in post-myocardial infarction, end-stage renal disease,
             and chronic heart failure patients.},
   Journal = {Europace},
   Volume = {19},
   Number = {8},
   Pages = {1392-1400},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1093/europace/euw222},
   Abstract = {Cyclic variation of heart rate (CVHR) associated with
             sleep-disordered breathing is thought to reflect cardiac
             autonomic responses to apnoeic/hypoxic stress. We examined
             whether blunted CVHR observed in ambulatory ECG could
             predict the mortality risk.CVHR in night-time Holter ECG was
             detected by an automated algorithm, and the prognostic
             relationships of the frequency (FCV) and amplitude (ACV) of
             CVHR were examined in 717 patients after myocardial
             infarction (post-MI 1, 6% mortality, median follow-up 25
             months). The predictive power was prospectively validated in
             three independent cohorts: a second group of 220 post-MI
             patients (post-MI 2, 25.5% mortality, follow-up 45 months);
             299 patients with end-stage renal disease on chronic
             haemodialysis (ESRD, 28.1% mortality, follow-up 85 months);
             and 100 patients with chronic heart failure (CHF, 35%
             mortality, follow-up 38 months). Although CVHR was observed
             in ≥96% of the patients in all cohorts, FCV did not
             predict mortality in any cohort. In contrast, decreased ACV
             was a powerful predictor of mortality in the post-MI 1
             cohort (hazard ratio [95% CI] per 1 ln [ms] decrement, 2.9
             [2.2-3.7], P < 0.001). This prognostic relationship was
             validated in the post-MI 2 (1.8 [1.4-2.2], P < 0.001), ESRD
             (1.5 [1.3-1.8], P < 0.001), and CHF (1.4 [1.1-1.8], P =
             0.02) cohorts. The prognostic value of ACV was independent
             of age, gender, diabetes, β-blocker therapy, left
             ventricular ejection fraction, sleep-time mean R-R interval,
             and FCV.Blunted CVHR detected by decreased ACV in a
             night-time Holter ECG predicts increased mortality risk in
             post-MI, ESRD, and CHF patients.},
   Doi = {10.1093/europace/euw222},
   Key = {fds330038}
}

@article{fds326740,
   Author = {Tussing-Humphreys, L and Lamar, M and Blumenthal, JA and Babyak, M and Fantuzzi, G and Blumstein, L and Schiffer, L and Fitzgibbon,
             ML},
   Title = {Building research in diet and cognition: The BRIDGE
             randomized controlled trial.},
   Journal = {Contemporary Clinical Trials},
   Volume = {59},
   Pages = {87-97},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.cct.2017.06.003},
   Abstract = {Obesity has been linked to cognitive impairment, cognitive
             decline and dementia. Given that 38.5% of U.S. adults
             60years and older are obese and these numbers are rapidly
             increasing, strategies to decouple obesity from cognitive
             decline are needed. Innovative lifestyle strategies that may
             postpone the onset of subclinical symptoms or even arrest
             the transition to overt dementia in at-risk individuals are
             critically needed. Poor diet is central to the development
             of obesity and diet may affect cognition. Adherence to a
             Mediterranean Diet (MedDiet) is associated with reduced risk
             of cognitive impairment and dementia. Furthermore, weight
             loss through caloric restriction improves cognitive
             function. This paper describes the Building Research in Diet
             and CoGnition (BRIDGE) study, a randomized trial examining
             the effect of the MedDiet, with and without weight loss, on
             cognitive functioning in obese older adults. Obese (BMI≥30
             and ≤50kg/m2) older adults (≥55years) (n=180) will be
             randomized in a 2:2:1 allocation scheme to: Typical Diet
             Control; MedDiet alone, without weight loss; or MedDiet
             lifestyle intervention to promote weight loss and weight
             loss maintenance. Both MedDiet intervention groups will meet
             for one individual session and 27 group sessions over an
             8-month period. Individuals in the control group will not
             receive instruction on changing lifestyle habits. Outcomes
             will be assessed at baseline, 8 and 14months. The primary
             outcome is cognitive functioning; secondary outcomes will
             include changes in body weight, diet, cardiovascular,
             metabolic, and inflammatory biomarkers.},
   Doi = {10.1016/j.cct.2017.06.003},
   Key = {fds326740}
}

@article{fds326855,
   Author = {Blumenthal, JA and Smith, PJ and Mabe, S and Hinderliter, A and Welsh-Bohmer, K and Browndyke, JN and Lin, P-H and Kraus, W and Doraiswamy, PM and Burke, J and Sherwood, A},
   Title = {Lifestyle and Neurocognition in Older Adults With
             Cardiovascular Risk Factors and Cognitive
             Impairment.},
   Journal = {Psychosomatic Medicine},
   Volume = {79},
   Number = {6},
   Pages = {719-727},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1097/PSY.0000000000000474},
   Abstract = {The aim of the study was to determine the relationship of
             lifestyle factors and neurocognitive functioning in older
             adults with vascular risk factors and cognitive impairment,
             no dementia (CIND).One hundred sixty adults (M [SD] = 65.4
             [6.8] years) with CIND completed neurocognitive assessments
             of executive function, processing speed, and memory.
             Objective measures of physical activity using accelerometry,
             aerobic capacity determined by exercise testing, and dietary
             habits quantified by the Food Frequency Questionnaire and
             4-Day Food Diary to assess adherence to the Mediterranean
             and Dietary Approaches to Stop Hypertension (DASH) diets
             were obtained to assess direct effects with neurocognition.
             Potential indirect associations of high-sensitivity
             C-reactive protein and the Framingham Stroke Risk Profile
             also were examined.Greater aerobic capacity (β = 0.24) and
             daily physical activity (β = 0.15) were associated with
             better executive functioning/processing speed and verbal
             memory (βs = 0.24; 0.16). Adherence to the DASH diet was
             associated with better verbal memory (β = 0.17). Greater
             high-sensitivity C-reactive protein (βs = -0.14; -0.21) and
             Framingham Stroke Risk Profile (β = -0.18; -0.18) were
             associated with poorer executive functioning/processing
             speed and verbal memory. Greater stroke risk partially
             mediated the association of aerobic capacity with executive
             functioning/processing speed, and verbal memory and greater
             inflammation partially mediated the association of physical
             activity and aerobic fitness, with verbal memory.Higher
             levels of physical activity, aerobic fitness, and adherence
             to the DASH diet are associated with better neurocognitive
             performance in adults with CIND. These findings suggest that
             the adoption of healthy lifestyle habits could reduce the
             risk of neurocognitive decline in vulnerable older
             adults.NCT01573546.},
   Doi = {10.1097/PSY.0000000000000474},
   Key = {fds326855}
}

@article{fds326632,
   Author = {Hill, LK and Watkins, LL and Hinderliter, AL and Blumenthal, JA and Sherwood, A},
   Title = {Racial differences in the association between heart rate
             variability and left ventricular mass.},
   Journal = {Experimental Physiology},
   Volume = {102},
   Number = {7},
   Pages = {764-772},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1113/ep086228},
   Abstract = {What is the central question of this study? Decreased heart
             rate variability (HRV) is associated with increased
             cardiovascular disease (CVD) risk, including greater left
             ventricular mass (LVM). Despite their enhanced CVD risk
             profile, African Americans have been shown to exhibit higher
             HRV, relative to Whites; however, it is unclear whether this
             pattern extends to the association between HRV and LVM. What
             is the main finding and its importance? Using ECG and
             echocardiographic data, HRV was positively associated with
             LVM in a non-clinical sample of African Americans. These
             findings suggest that current assumptions regarding the
             meaning of higher HRV might not be universal, which might
             have implications for HRV as a risk marker among African
             Americans. Increased left ventricular mass (LVM) is an early
             precursor of target organ damage attributable to
             hypertension. Diminished parasympathetic cardiac control has
             been linked to both hypertension onset and left ventricular
             impairment; however, emerging evidence suggests that this
             pattern might be different in African Americans. The present
             study sought to determine whether race impacts the
             relationship between parasympathetic cardiac control and
             LVM. The LVM was assessed via echocardiography in a sample
             (n = 148) of African American and White adults (mean
             age 33.20 ± 5.71 years) with normal or mildly elevated
             blood pressure. Parasympathetic cardiac control was assessed
             by a measure of high-frequency heart rate variability
             (HF-HRV) determined from ECG recordings during 5 min of
             rest. In regression analysis, greater HF-HRV was associated
             with greater LVM among African Americans (P = 0.002) but
             was not related to LVM in Whites (P = 0.919). These are
             the first data to demonstrate that race moderates the
             relationship between HRV and LVM and further suggest that
             race might be an important factor in the association between
             parasympathetic cardiac control and other cardiovascular
             disease risk factors.},
   Doi = {10.1113/ep086228},
   Key = {fds326632}
}

@article{fds325510,
   Author = {Sherwood, A and Hill, LK and Blumenthal, JA and Johnson, KS and Hinderliter, AL},
   Title = {Race and sex differences in cardiovascular α-adrenergic and
             β-adrenergic receptor responsiveness in men and women with
             high blood pressure.},
   Journal = {Journal of Hypertension},
   Volume = {35},
   Number = {5},
   Pages = {975-981},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1097/HJH.0000000000001266},
   Abstract = {Hypertension is associated with unfavorable changes in
             adrenergic receptor responsiveness, but the relationship of
             race and sex to adrenergic receptor responsiveness in the
             development of cardiovascular disease is unclear. This study
             examined α-adrenergic and ß-adrenergic receptor
             responsiveness in African-American and white men and women
             with untreated high blood pressure (BP) (HBP) and with
             normal BP.The study sample comprised 161 African-American
             and white men and women in the age range 25-45 years.
             Isoproterenol, a nonselective ß-adrenergic receptor
             agonist, was administered intravenously to determine the
             bolus dose required to increase heart rate by 25 bpm, an
             index of β-adrenergic receptor responsiveness. Similarly,
             phenylephrine, an α1-adrenergic receptor agonist, was
             administered to determine the bolus dose required to
             increase BP by 25 mmHg, an index of vascular
             α1-adrenergic receptor responsiveness. HBP (P < 0.01),
             male sex (P = 0.04), and higher BMI (P < 0.01) were
             all associated with reduced β-adrenergic receptor
             responsiveness, with a similar trend observed for
             African-American race (P = 0.07). Conversely,
             α1-adrenergic receptor responsiveness was increased in
             association with HBP (P < 0.01), female sex
             (P < 0.01), and African-American race (P < 0.01).In
             the early stages of hypertension, cardiovascular
             β-adrenergic receptors demonstrate blunted responsiveness,
             whereas conversely α1-adrenergic receptors exhibit
             increased responsiveness. This pattern of receptor changes
             is especially evident in men and African-Americans, is
             exacerbated by obesity, and may contribute to the
             development of cardiovascular disease.},
   Doi = {10.1097/HJH.0000000000001266},
   Key = {fds325510}
}

@article{fds329488,
   Author = {Hill, LK and Sherwood, A and Blumenthal, JA and Hinderliter,
             AL},
   Title = {SYNERGISTIC IMPACT OF PERCEIVED DISCRIMINATION AND HOSTILITY
             ON ADRENERGIC RESPONSIVITY},
   Journal = {Psychosomatic Medicine},
   Volume = {79},
   Number = {4},
   Pages = {A85-A85},
   Year = {2017},
   Month = {May},
   Key = {fds329488}
}

@article{fds321706,
   Author = {Stonerock, GL and Blumenthal, JA},
   Title = {Role of Counseling to Promote Adherence in Healthy Lifestyle
             Medicine: Strategies to Improve Exercise Adherence and
             Enhance Physical Activity.},
   Journal = {Progress in Cardiovascular Diseases},
   Volume = {59},
   Number = {5},
   Pages = {455-462},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.pcad.2016.09.003},
   Abstract = {Although healthy lifestyles (HL) offer a number of health
             benefits, nonadherence to recommended lifestyle changes
             remains a frequent and difficult obstacle to realizing these
             benefits. Behavioral counseling can improve adherence to an
             HL. However, individuals' motivation for change and
             resistance to altering unhealthy habits must be considered
             when developing an effective approach to counseling. In the
             present article, we review psychological, behavioral, and
             environmental factors that may promote adherence and
             contribute to nonadherence. We discuss two established
             models for counseling, motivational interviewing and the
             transtheoretical model of behavior change, and provide an
             example of how these approaches can be used to counsel
             patients to exercise and increase their levels of physical
             activity.},
   Doi = {10.1016/j.pcad.2016.09.003},
   Key = {fds321706}
}

@article{fds323315,
   Author = {Smith, PJ and Blumenthal, JA and Snyder, LD and Mathew, JP and Durheim,
             MT and Hoffman, BM and Rivelli, SK and Palmer, SM},
   Title = {Depressive symptoms and early mortality following lung
             transplantation: A pilot study.},
   Journal = {Clinical Transplantation},
   Volume = {31},
   Number = {2},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1111/ctr.12874},
   Abstract = {Impaired psychological function is common among lung
             transplant candidates and may affect clinical outcomes
             following transplantation. Although numerous studies have
             examined the relationship between pretransplant depression,
             quality of life (QoL), and post-transplant outcomes, few
             have examined the relationship between depression and QoL
             shortly following transplantation and subsequent clinical
             outcomes. We therefore examined the association between
             depression, QoL, and short-term mortality in a consecutive
             series of lung transplant recipients.Depression (Patient
             Health Questionnaire-9; Hospital Anxiety and Depression
             Scale; Centers for Epidemiologic Studies Depression Scale)
             and QoL (UCSD Shortness of Breath Questionnaire; Pulmonary
             Quality of Life Scale) were assessed prior to
             transplantation (median 0.9 months [IQR=1.6]) and again
             approximately 2 weeks following transplantation
             (median=0.5 months [IQR=0.5]), in a series of 66 patients
             transplanted between March 2013 and April 2014. The
             association between psychiatric diagnoses from participants'
             comprehensive pretransplant assessment and mortality also
             was examined. Cox proportional hazards models were used to
             examine the association between depression, QoL, and
             mortality.During a median follow-up of 2.8 years (range
             0.4-3.3), 21 patients died (32%). Greater depressive
             symptoms assessed shortly after transplant were associated
             with subsequent mortality (HR=2.17 [1.01, 4.67], P=.048),
             and this relationship persisted after controlling for
             primary graft dysfunction, duration of transplant
             hospitalization, and gender. In contrast, neither
             pretransplant depression, history of depression, nor QoL was
             associated with mortality.Greater post-transplant depressive
             symptoms are independently associated with mortality among
             lung transplant recipients.},
   Doi = {10.1111/ctr.12874},
   Key = {fds323315}
}

@article{fds329489,
   Author = {Sherwood, A and Smith, PJ and Hinderliter, AL and Georgiades, A and Blumenthal, JA},
   Title = {Effects of exercise and stress management training on
             nighttime blood pressure dipping in patients with coronary
             heart disease: A randomized, controlled trial.},
   Journal = {American Heart Journal},
   Volume = {183},
   Pages = {85-90},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.ahj.2016.10.011},
   Abstract = {Blunted nighttime blood pressure (BP) dipping is prognostic
             of cardiovascular morbidity and mortality. Patients with
             coronary heart disease (CHD) are often characterized by a
             blunted nighttime BP dipping pattern. The present study
             compared the effects of 2 behavioral intervention programs,
             aerobic exercise (EX) and stress management (SM) training,
             with a usual care (UC) control group on BP dipping in a
             sample of CHD patients.This was a secondary analysis of a
             randomized, controlled trial with allocation concealment and
             blinded outcome assessment in 134 patients with stable CHD
             and exercise-induced myocardial ischemia. Nighttime BP
             dipping was assessed by 24-hour ambulatory BP monitoring, at
             prerandomization baseline and after 16 weeks of one of the
             following treatments: usual medical care; UC plus supervised
             aerobic EX for 35 minutes, 3 times per week; UC plus weekly
             1.5-hour sessions of SM training.The EX and SM groups
             exhibited greater improvements in systolic BP dipping
             (P=.052) and diastolic BP dipping (P=.031) compared with UC.
             Postintervention systolic BP percent-dipping means were
             12.9% (SE=1.5) for SM, 11.1% (SE=1.4) for EX, and 8.6%
             (SE=1.4) for UC. Postintervention diastolic BP
             percent-dipping means were 13.3% (SE=1.9) for SM, 14.1%
             (SE=1.8) for EX, and 8.8% (1.8) for UC.For patients with
             stable CHD, EX or SM training resulted in improved nighttime
             BP dipping compared with usual medical care. These favorable
             effects of healthy lifestyle modifications may help reduce
             the risk of adverse clinical events.},
   Doi = {10.1016/j.ahj.2016.10.011},
   Key = {fds329489}
}

@article{fds329490,
   Author = {Sherwood, A and Blumenthal, JA and Koch, GG and Hoffman, BM and Watkins,
             LL and Smith, PJ and O'Connor, CM and Adams, KF and Rogers, JG and Sueta,
             C and Chang, PP and Johnson, KS and Schwartz, J and Hinderliter,
             AL},
   Title = {Effects of Coping Skills Training on Quality of Life,
             Disease Biomarkers, and Clinical Outcomes in Patients With
             Heart Failure: A Randomized Clinical Trial.},
   Journal = {Circulation. Heart Failure},
   Volume = {10},
   Number = {1},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1161/CIRCHEARTFAILURE.116.003410},
   Abstract = {Heart failure (HF) is a chronic disease that compromises
             patients' quality of life (QoL). Interventions designed to
             reduce distress and improve disease self-management are
             needed. We evaluated the efficacy of a telephone-based
             coping skills training (CST) intervention.This randomized
             clinical trial involved 180 HF outpatients with reduced
             ejection fraction. Participants ranged in age from 29 to 87
             years (mean=58 years); 27% were women, and 47% were
             nonwhite. Participants were randomized to either a CST
             intervention or heart failure education, both delivered over
             16 weeks. The primary outcomes were (1) postintervention
             effects on QoL and HF disease biomarkers (both with
             α=0.01), and (2) a composite measure of time to death or
             first hospitalization (with α=0.03) over a median follow-up
             period of 3 years. CST resulted in greater improvements in
             QoL compared with heart failure education (P<0.01),
             including the Kansas City Cardiomyopathy Questionnaire
             (P=0.009), depressive symptoms (P=0.027), and the 6-minute
             walk test (P=0.012). However, it did not differentially
             improve HF disease biomarkers or reduce risk of all-cause
             hospitalizations or death (hazard ratio=0.84 [95% confidence
             interval, 0.59-1.12]). Interestingly, exploratory analyses
             showed that participants randomized to CST experienced a
             reduction in the composite end point of worsening HF
             hospitalization or death during the 3-year follow-up period
             (hazard ratio=0.65 [95% confidence interval, 0.44-0.98];
             P=0.040).CST improved QoL in patients with HF. Monitoring
             and improving QoL is emerging as an important aspect of the
             clinical management of HF that can reduce disease burden and
             may help improve clinical outcomes in this vulnerable
             patient population.URL: https://www.clinicaltrials.gov.
             Unique identifier: NCT00873418.},
   Doi = {10.1161/CIRCHEARTFAILURE.116.003410},
   Key = {fds329490}
}


%% Bonner, Melanie J.   
@article{fds336054,
   Author = {Anderson, LM and Leonard, S and Jonassaint, J and Lunyera, J and Bonner,
             M and Shah, N},
   Title = {Mobile health intervention for youth with sickle cell
             disease: Impact on adherence, disease knowledge, and quality
             of life},
   Journal = {Pediatric Blood & Cancer},
   Volume = {65},
   Number = {8},
   Pages = {e27081-e27081},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1002/pbc.27081},
   Doi = {10.1002/pbc.27081},
   Key = {fds336054}
}

@article{fds319600,
   Author = {Allen, TM and Anderson, LM and Rothman, JA and Bonner,
             MJ},
   Title = {[Formula: see text]Executive functioning and health-related
             quality of life in pediatric sickle cell
             disease.},
   Journal = {Child Neuropsychology : a Journal on Normal and Abnormal
             Development in Childhood and Adolescence},
   Volume = {23},
   Number = {8},
   Pages = {889-906},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1080/09297049.2016.1205011},
   Abstract = {Research consistently indicates that children with sickle
             cell disease (SCD) face multiple risk factors for
             neurocognitive impairment. Despite this, no empirical
             research to date has examined the impact of neurocognitive
             functioning on quality of life for this pediatric group.
             Thus, the current study aims to examine the relationship
             between executive functioning and quality of life in a
             sample of children with SCD and further explore psychosocial
             and family/caregiver resources as moderators of this
             relationship. A total of 45 children with SCD aged 8 to 16
             years and their caregivers completed measures of quality of
             life, behavioral ratings of executive functioning, and
             psychosocial functioning. Hierarchical linear regression
             models were utilized to determine the impact of executive
             functioning on quality of life and further test the
             interaction effects of proposed moderating variables.
             Controlling for age, pain, and socioeconomic status (SES),
             executive functioning was found to significantly predict
             child- and parent-reported quality of life among youth with
             SCD. Psychosocial resources of the primary caregiver or
             family was not found to moderate the relationship between
             executive functioning and quality of life. These results
             provide the first empirical evidence that lower executive
             skills negatively predict quality of life for children with
             SCD, supporting clinical and research efforts which aim to
             establish efficacious interventions that target cognitive
             decrements within this pediatric population.},
   Doi = {10.1080/09297049.2016.1205011},
   Key = {fds319600}
}

@article{fds330040,
   Author = {Schreiber, JE and Palmer, SL and Conklin, HM and Mabbott, DJ and Swain,
             MA and Bonner, MJ and Chapieski, ML and Huang, L and Zhang, H and Gajjar,
             A},
   Title = {Posterior fossa syndrome and long-term neuropsychological
             outcomes among children treated for medulloblastoma on a
             multi-institutional, prospective study.},
   Journal = {Neuro Oncology},
   Volume = {19},
   Number = {12},
   Pages = {1673-1682},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1093/neuonc/nox135},
   Abstract = {Patients treated for medulloblastoma who experience
             posterior fossa syndrome (PFS) demonstrate increased risk
             for neurocognitive impairment at one year post diagnosis.
             The aim of the study was to examine longitudinal
             trajectories of neuropsychological outcomes in patients who
             experienced PFS compared with patients who did
             not.Participants were 36 patients (22 males) who experienced
             PFS and 36 comparison patients (21 males) who were matched
             on age at diagnosis and treatment exposure but did not
             experience PFS. All patients underwent serial evaluation of
             neurocognitive functioning spanning 1 to 5 years post
             diagnosis.The PFS group demonstrated lower estimated mean
             scores at 1, 3, and 5 years post diagnosis on measures of
             general intellectual ability, processing speed, broad
             attention, working memory, and spatial relations compared
             with the non-PFS group. The PFS group exhibited estimated
             mean scores that were at least one standard deviation below
             the mean for intellectual ability, processing speed, and
             broad attention across all time points and for working
             memory by 5 years post diagnosis. Processing speed was
             stable over time. Attention and working memory declined over
             time. Despite some change over time, caregiver ratings of
             executive function and behavior problem symptoms remained
             within the average range.Compared with patients who do not
             experience PFS, patients who experience PFS exhibit greater
             neurocognitive impairment, show little recovery over time,
             and decline further in some domains. Findings highlight the
             particularly high risk for long-term neurocognitive problems
             in patients who experience PFS and the need for close
             follow-up and intervention.},
   Doi = {10.1093/neuonc/nox135},
   Key = {fds330040}
}

@article{fds330041,
   Author = {Gallentine, WB and Shinnar, S and Hesdorffer, DC and Epstein, L and Nordli, DR and Lewis, DV and Frank, LM and Seinfeld, S and Shinnar, RC and Cornett, K and Liu, B and Moshé, SL and Sun, S and FEBSTAT Investigator
             Team},
   Title = {Plasma cytokines associated with febrile status epilepticus
             in children: A potential biomarker for acute hippocampal
             injury.},
   Journal = {Epilepsia},
   Volume = {58},
   Number = {6},
   Pages = {1102-1111},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1111/epi.13750},
   Abstract = {Our aim was to explore the association between plasma
             cytokines and febrile status epilepticus (FSE) in children,
             as well as their potential as biomarkers of acute
             hippocampal injury.Analysis was performed on residual
             samples of children with FSE (n = 33) as part of the
             Consequences of Prolonged Febrile Seizures in Childhood
             study (FEBSTAT) and compared to children with fever
             (n = 17). Magnetic resonance imaging (MRI) was obtained as
             part of FEBSTAT within 72 h of FSE. Cytokine levels and
             ratios of antiinflammatory versus proinflammatory cytokines
             in children with and without hippocampal T2 hyperintensity
             were assessed as biomarkers of acute hippocampal injury
             after FSE.Levels of interleukin (IL)-8 and epidermal growth
             factor (EGF) were significantly elevated after FSE in
             comparison to controls. IL-1β levels trended higher and
             IL-1RA trended lower following FSE, but did not reach
             statistical significance. Children with FSE were found to
             have significantly lower ratios of IL-1RA/IL-1β and
             IL-1RA/IL-8. Specific levels of any one individual cytokine
             were not associated with FSE. However, lower ratios of
             IL-1RA/IL-1β, IL-1RA/1L-6, and IL-1RA/ IL-8 were all
             associated with FSE. IL-6 and IL-8 levels were significantly
             higher and ratios of IL-1RA/IL-6 and IL-1RA/IL-8 were
             significantly lower in children with T2 hippocampal
             hyperintensity on MRI after FSE in comparison to those
             without hippocampal signal abnormalities. Neither individual
             cytokine levels nor ratios of IL-1RA/IL-1β or IL-1RA/IL-8
             were predictive of MRI changes. However, a lower ratio of
             IL-1RA/IL-6 was strongly predictive (odds ratio [OR] 21.5,
             95% confidence interval [CI] 1.17-393) of hippocampal T2
             hyperintensity after FSE.Our data support involvement of the
             IL-1 cytokine system, IL-6, and IL-8 in FSE in children. The
             identification of the IL-1RA/IL-6 ratio as a potential
             biomarker of acute hippocampal injury following FSE is the
             most significant finding. If replicated in another study,
             the IL-1RA/IL-6 ratio could represent a serologic biomarker
             that offers rapid identification of patients at risk for
             ultimately developing mesial temporal lobe epilepsy
             (MTLE).},
   Doi = {10.1111/epi.13750},
   Key = {fds330041}
}

@article{fds316069,
   Author = {Willard, VW and Allen, TM and Hardy, KK and Bonner,
             MJ},
   Title = {Social functioning in survivors of pediatric brain tumors:
             Contribution of neurocognitive and social-cognitive
             skills},
   Journal = {Children's Health Care},
   Volume = {46},
   Number = {2},
   Pages = {181-195},
   Year = {2017},
   Month = {April},
   ISSN = {0273-9615},
   url = {http://dx.doi.org/10.1080/02739615.2015.1124769},
   Doi = {10.1080/02739615.2015.1124769},
   Key = {fds316069}
}

@article{fds324821,
   Author = {Shashi, V and Pena, LDM and Kim, K and Burton, B and Hempel, M and Schoch,
             K and Walkiewicz, M and McLaughlin, HM and Cho, M and Stong, N and Hickey,
             SE and Shuss, CM and Undiagnosed Diseases Network, and Freemark, MS and Bellet, JS and Keels, MA and Bonner, MJ and El-Dairi, M and Butler, M and Kranz, PG and Stumpel, CTRM and Klinkenberg, S and Oberndorff, K and Alawi, M and Santer, R and Petrovski, S and Kuismin, O and Korpi-Heikkilä, S and Pietilainen, O and Aarno, P and Kurki, MI and Hoischen, A and Need, AC and Goldstein, DB and Kortüm,
             F},
   Title = {De Novo Truncating Variants in ASXL2 Are Associated with a
             Unique and Recognizable Clinical Phenotype.},
   Journal = {American Journal of Human Genetics},
   Volume = {100},
   Number = {1},
   Pages = {179},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.ajhg.2016.12.004},
   Doi = {10.1016/j.ajhg.2016.12.004},
   Key = {fds324821}
}


%% Brownell, Kelly D.   
@article{fds335162,
   Author = {Tomaselli, G and Roach, WH and Piña, IL and Oster, ME and Dietz, WH and Horton, K and Borden, WB and Brownell, K and Gibbons, RJ and Otten, JJ and Lee, CS and Hill, C and Heidenreich, PA and Siscovick, DS and Whitsel,
             LP},
   Title = {Government continues to have an important role in promoting
             cardiovascular health.},
   Journal = {American Heart Journal},
   Volume = {198},
   Pages = {160-165},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.ahj.2017.11.002},
   Doi = {10.1016/j.ahj.2017.11.002},
   Key = {fds335162}
}

@article{fds335161,
   Author = {Bragg, MA and Miller, AN and Roberto, CA and Sam, R and Sarda, V and Harris, JL and Brownell, KD},
   Title = {Sports Sponsorships of Food and Nonalcoholic
             Beverages},
   Journal = {Pediatrics},
   Volume = {141},
   Number = {4},
   Pages = {e20172822-e20172822},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1542/peds.2017-2822},
   Abstract = {© 2018 by the American Academy of Pediatrics. BACKGROUND:
             Food and nonalcoholic beverage companies spend millions of
             dollars on professional sports sponsorships, yet this form
             of marketing is understudied. These sponsorships are
             valuable marketing tools but prompt concerns when unhealthy
             products are associated with popular sports organizations,
             especially those viewed by youth. METHODS: This descriptive
             study used Nielsen audience data to select 10 sports
             organizations with the most 2-17 year old viewers of 2015
             televised events. Sponsors of these organizations were
             identified and assigned to product categories. We identified
             advertisements promoting food and/or nonalcoholic beverage
             sponsorships on television, YouTube, and sports organization
             Web sites from 2006 to 2016, and the number of YouTube
             advertisement views. The nutritional quality of advertised
             products was assessed. RESULTS: Youth watched telecasts
             associated with these sports organizations over 412 million
             times. These organizations had 44 food and/or nonalcoholic
             beverage sponsors (18.8% of sponsors), second to automotive
             sponsors (n = 46). The National Football League had the most
             food and/or nonalcoholic beverage sponsors (n = 10),
             followed by the National Hockey League (n = 7) and Little
             League (n = 7). We identified 273 advertisements that
             featured food and/or nonalcoholic beverage products 328
             times and product logos 83 times (some advertisements showed
             multiple products). Seventy-six percent (n = 132) of foods
             had unhealthy nutrition scores, and 52.4% (n = 111) of
             nonalcoholic beverages were sugarsweetened. YouTube
             sponsorship advertisements totaled 195.6 million views.
             CONCLUSIONS: Sports sponsorships are commonly used to market
             unhealthy food and nonalcoholic beverages, exposing millions
             of consumers to these advertisements.},
   Doi = {10.1542/peds.2017-2822},
   Key = {fds335161}
}

@article{fds331326,
   Author = {Bragg, MA and Roberto, CA and Harris, JL and Brownell, KD and Elbel,
             B},
   Title = {Marketing Food and Beverages to Youth Through
             Sports},
   Journal = {Journal of Adolescent Health},
   Volume = {62},
   Number = {1},
   Pages = {5-13},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jadohealth.2017.06.016},
   Doi = {10.1016/j.jadohealth.2017.06.016},
   Key = {fds331326}
}

@article{fds331325,
   Author = {Cooksey-Stowers, K and Schwartz, MB and Brownell,
             KD},
   Title = {Food Swamps Predict Obesity Rates Better Than Food Deserts
             in the United States.},
   Journal = {International Journal of Environmental Research and Public
             Health},
   Volume = {14},
   Number = {11},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.3390/ijerph14111366},
   Abstract = {This paper investigates the effect of food environments,
             characterized as food swamps, on adult obesity rates. Food
             swamps have been described as areas with a high-density of
             establishments selling high-calorie fast food and junk food,
             relative to healthier food options. This study examines
             multiple ways of categorizing food environments as food
             swamps and food deserts, including alternate versions of the
             Retail Food Environment Index. We merged food outlet,
             sociodemographic and obesity data from the United States
             Department of Agriculture (USDA) Food Environment Atlas, the
             American Community Survey, and a commercial street reference
             dataset. We employed an instrumental variables (IV) strategy
             to correct for the endogeneity of food environments (i.e.,
             that individuals self-select into neighborhoods and may
             consider food availability in their decision). Our results
             suggest that the presence of a food swamp is a stronger
             predictor of obesity rates than the absence of full-service
             grocery stores. We found, even after controlling for food
             desert effects, food swamps have a positive, statistically
             significant effect on adult obesity rates. All three food
             swamp measures indicated the same positive association, but
             reflected different magnitudes of the food swamp effect on
             rates of adult obesity (p values ranged from 0.00 to 0.16).
             Our adjustment for reverse causality, using an IV approach,
             revealed a stronger effect of food swamps than would have
             been obtained by naïve ordinary least squares (OLS)
             estimates. The food swamp effect was stronger in counties
             with greater income inequality (p < 0.05) and where
             residents are less mobile (p < 0.01). Based on these
             findings, local government policies such as zoning laws
             simultaneously restricting access to unhealthy food outlets
             and incentivizing healthy food retailers to locate in
             underserved neighborhoods warrant consideration as
             strategies to increase health equity.},
   Doi = {10.3390/ijerph14111366},
   Key = {fds331325}
}

@article{fds324189,
   Author = {Roberto, CA and Brownell, KD},
   Title = {Strategic science for eating disorders research and policy
             impact.},
   Journal = {The International Journal of Eating Disorders},
   Volume = {50},
   Number = {3},
   Pages = {312-314},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1002/eat.22678},
   Abstract = {Scientific research often fails to have relevance and impact
             because scientists do not engage policy makers and
             influencers in the process of identifying information needs
             and generating high priority questions. To address this
             scholarship-policy gap, we have developed a model of
             Strategic Science. This research approach involves working
             with policy makers and influencers to craft research
             questions that will answer important and timely
             policy-related questions. The goal is to create tighter
             links between research and policy and ensure findings are
             communicated efficiently to change agents best positioned to
             apply the research to policy debates. In this article, we
             lay out a model for Strategic Science and describe how this
             approach may help advance policy research and action for
             eating disorders.},
   Doi = {10.1002/eat.22678},
   Key = {fds324189}
}

@article{fds326083,
   Author = {Bleich, SN and Rimm, EB and Brownell, KD},
   Title = {U.S. Nutrition Assistance, 2018 - Modifying SNAP to Promote
             Population Health.},
   Journal = {The New England Journal of Medicine},
   Volume = {376},
   Number = {13},
   Pages = {1205-1207},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1056/nejmp1613222},
   Doi = {10.1056/nejmp1613222},
   Key = {fds326083}
}


%% Cabeza, Roberto   
@article{fds338000,
   Author = {Cabeza, R and Stanley, ML and Moscovitch, M},
   Title = {Process-Specific Alliances (PSAs) in Cognitive
             Neuroscience.},
   Journal = {Trends in Cognitive Sciences},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.tics.2018.08.005},
   Abstract = {Most cognitive neuroscience theories have focused on the
             functions of individual brain regions, but cognitive
             abilities depend also on functional interactions among
             multiple regions. Many recent studies on these interactions
             have examined large-scale, resting-state networks, but these
             networks are difficult to link to theories about specific
             cognitive processes. Cognitive theories are easier to link
             to the mini-networks we call process specific alliances
             (PSAs). A PSA is a small team of brain regions that rapidly
             assemble to mediate a cognitive process in response to task
             demands but quickly disassemble when the process is no
             longer needed. We compare PSAs to resting-state networks and
             to other connectivity-based, task-related networks, and we
             characterize the advantages and disadvantages of each type
             of network.},
   Doi = {10.1016/j.tics.2018.08.005},
   Key = {fds338000}
}

@article{fds337615,
   Author = {Wang, W-C and Wing, EA and Murphy, DLK and Luber, BM and Lisanby, SH and Cabeza, R and Davis, SW},
   Title = {Excitatory TMS modulates memory representations.},
   Journal = {Cognitive Neuroscience},
   Pages = {1-16},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1080/17588928.2018.1512482},
   Abstract = {Brain stimulation technologies have seen increasing
             application in basic science investigations, specifically
             toward the goal of improving memory function. However,
             proposals concerning the neural mechanisms underlying
             cognitive enhancement often rely on simplified notions of
             excitation. As a result, most applications examining the
             effects of transcranial magnetic stimulation (TMS) on
             functional neuroimaging measures have been limited to
             univariate analyses of brain activity. We present here
             analyses using representational similarity analysis (RSA)
             and encoding-retrieval similarity (ERS) analysis to quantify
             the effect of TMS on memory representations. To test whether
             an increase in local excitability in PFC can have measurable
             influences on upstream representations in earlier temporal
             memory regions, we compared 1 and 5Hz stimulation to the
             left dorsolateral PFC (DLPFC). We found that 5Hz rTMS,
             relative to 1Hz, had multiple effects on neural
             representations: 1) greater representational similarity
             during both encoding and retrieval in ventral stream
             regions, 2) greater ERS in the hippocampus, and, critically,
             3) increasing ERS in MTL was correlated with increasing
             univariate activity in DLPFC, and greater functional
             connectivity for hits than misses between these regions.
             These results provide the first evidence of rTMS modulating
             semantic representations and strengthen the idea that rTMS
             may affect the reinstatement of previously experienced
             events in upstream regions.},
   Doi = {10.1080/17588928.2018.1512482},
   Key = {fds337615}
}

@article{fds335638,
   Author = {Monge, ZA and Stanley, ML and Geib, BR and Davis, SW and Cabeza,
             R},
   Title = {Functional networks underlying item and source memory:
             shared and distinct network components and age-related
             differences.},
   Journal = {Neurobiology of Aging},
   Volume = {69},
   Pages = {140-150},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.neurobiolaging.2018.05.016},
   Abstract = {Although the medial temporal lobes (MTLs) are critical for
             both item memory (IM) and source memory (SM), the lateral
             prefrontal cortex and posterior parietal cortex play a
             greater role during SM than IM. It is unclear, however, how
             these differences translate into shared and distinct IM
             versus SM network components and how these network
             components vary with age. Within a sample of younger adults
             (YAs; n = 15, Mage = 19.5 years) and older adults (OAs;
             n = 40, Mage = 68.6 years), we investigated the
             functional networks underlying IM and SM. Before functional
             MRI scanning, participants encoded nouns while making either
             pleasantness or size judgments. During functional MRI
             scanning, participants completed IM and SM retrieval tasks.
             We found that MTL nodes were similarly interconnected among
             each other during both IM and SM (shared network components)
             but maintained more intermodule connections during SM
             (distinct network components). Also, during SM, OAs
             (compared to YAs) had MTL nodes with more widespread
             connections. These findings provide a novel viewpoint on
             neural mechanism differences underlying IM versus SM in YAs
             and OAs.},
   Doi = {10.1016/j.neurobiolaging.2018.05.016},
   Key = {fds335638}
}

@article{fds337616,
   Author = {Lighthall, NR and Pearson, JM and Huettel, SA and Cabeza,
             R},
   Title = {Feedback-based learning in aging: Contributions and
             trajectories of change in striatal and hippocampal
             systems.},
   Journal = {The Journal of Neuroscience : the Official Journal of the
             Society for Neuroscience},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1523/jneurosci.0769-18.2018},
   Abstract = {The striatum supports learning from immediate feedback by
             coding prediction errors (PEs), while the hippocampus plays
             a parallel role in learning from delayed feedback. Both
             regions show evidence of decline in human aging, but
             behavioral research suggests greater decline in hippocampal
             versus striatal functions. The present study included male
             and female humans and used functional magnetic resonance
             imaging (fMRI) to examine younger and older adults' brain
             activation patterns during a learning task with choice
             feedback presented immediately or after a brief delay.
             Participants then completed a surprise memory task that
             tested their recognition of trial-unique feedback stimuli,
             followed by post-learning assessments of cue preference,
             outcome probability awareness, and willingness-to-pay. The
             study yielded three main findings. First, behavioral
             measures indicated similar rates of learning in younger and
             older adults across conditions, but post-learning measures
             indicated impairment in older adults' ability to
             subsequently apply learning to discriminate between cues.
             Second, PE signals in the striatum were greater for
             immediate versus delayed feedback in both age groups, but PE
             signals in the hippocampus were greater for delayed versus
             immediate feedback only in younger adults. Third, unlike
             younger adults, older adults failed to exhibit enhanced
             episodic memory for outcome stimuli in the delayed-feedback
             condition. Taken together, these findings indicate that
             hippocampal circuits supporting learning and memory decline
             more than striatal circuits in healthy aging, which suggests
             that declines in hippocampal learning signals may be an
             important predictor of deficits in learning-dependent
             economic decisions among older adults.SIGNIFICANCE
             STATEMENTThe hippocampus and striatum play distinct and
             critical roles in learning. Substantial research suggests
             that age-related decline in learning supported by the
             hippocampus outpaces decline in learning supported by the
             striatum; however, such inferences have been drawn by
             comparing performance in tasks with fundamentally different
             structures. The present study overcomes this obstacle by
             implementing a single fMRI-learning paradigm with a subtle
             variation in feedback timing to examine differential age
             effects on memory supported by the hippocampus and striatum.
             Our results provide converging behavioral and brain imaging
             evidence showing that hippocampal circuits supporting
             learning and memory decline more than striatal circuits in
             healthy aging, and that declines in hippocampal learning
             signals may predict early deficits in learning-dependent
             decisions among older adults.},
   Doi = {10.1523/jneurosci.0769-18.2018},
   Key = {fds337616}
}

@article{fds337617,
   Author = {Martinikorena, I and Cabeza, R and Villanueva, A and Urtasun, I and Larumbe, A},
   Title = {Fast and robust ellipse detection algorithm for head-mounted
             eye tracking systems},
   Journal = {Machine Vision and Applications},
   Volume = {29},
   Number = {5},
   Pages = {845-860},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1007/s00138-018-0940-0},
   Doi = {10.1007/s00138-018-0940-0},
   Key = {fds337617}
}

@article{fds335639,
   Author = {Wang, W-C and Brashier, NM and Wing, EA and Marsh, EJ and Cabeza,
             R},
   Title = {Knowledge supports memory retrieval through familiarity, not
             recollection.},
   Journal = {Neuropsychologia},
   Volume = {113},
   Pages = {14-21},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2018.01.019},
   Abstract = {Semantic memory, or general knowledge of the world, guides
             learning and supports the formation and retrieval of new
             episodic memories. Behavioral evidence suggests that this
             knowledge effect is supported by recollection-a more
             controlled form of memory retrieval generally accompanied by
             contextual details-to a greater degree than familiarity-a
             more automatic form of memory retrieval generally absent of
             contextual details. In the current study, we used functional
             magnetic resonance imaging (fMRI) to investigate the role
             that regions associated with recollection and familiarity
             play in retrieving recent instances of known (e.g., The
             Summer Olympic Games are held four years apart) and unknown
             (e.g., A flaky deposit found in port bottles is beeswing)
             statements. Our results revealed a surprising pattern:
             Episodic retrieval of known statements recruited regions
             associated with familiarity, but not recollection. Instead,
             retrieval of unknown statements recruited regions associated
             with recollection. These data, in combination with quicker
             reaction times for the retrieval of known than unknown
             statements, suggest that known statements can be
             successfully retrieved on the basis of familiarity, whereas
             unknown statements were retrieved on the basis of
             recollection. Our results provide insight into how knowledge
             influences episodic retrieval and demonstrate the role of
             neuroimaging in providing insights into cognitive processes
             in the absence of explicit behavioral responses.},
   Doi = {10.1016/j.neuropsychologia.2018.01.019},
   Key = {fds335639}
}

@article{fds332826,
   Author = {Wing, EA and Iyengar, V and Hess, TM and LaBar, KS and Huettel, SA and Cabeza, R},
   Title = {Neural mechanisms underlying subsequent memory for personal
             beliefs:An fMRI study.},
   Journal = {Cognitive, Affective & Behavioral Neuroscience},
   Volume = {18},
   Number = {2},
   Pages = {216-231},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.3758/s13415-018-0563-y},
   Abstract = {Many fMRI studies have examined the neural mechanisms
             supporting emotional memory for stimuli that generate
             emotion rather automatically (e.g., a picture of a dangerous
             animal or of appetizing food). However, far fewer studies
             have examined how memory is influenced by emotion related to
             social and political issues (e.g., a proposal for large
             changes in taxation policy), which clearly vary across
             individuals. In order to investigate the neural substrates
             of affective and mnemonic processes associated with personal
             opinions, we employed an fMRI task wherein participants
             rated the intensity of agreement/disagreement to
             sociopolitical belief statements paired with neural face
             pictures. Following the rating phase, participants performed
             an associative recognition test in which they distinguished
             identical versus recombined face-statement pairs. The study
             yielded three main findings: behaviorally, the intensity of
             agreement ratings was linked to greater subjective emotional
             arousal as well as enhanced high-confidence subsequent
             memory. Neurally, statements that elicited strong (vs. weak)
             agreement or disagreement were associated with greater
             activation of the amygdala. Finally, a subsequent memory
             analysis showed that the behavioral memory advantage for
             statements generating stronger ratings was dependent on the
             medial prefrontal cortex (mPFC). Together, these results
             both underscore consistencies in neural systems supporting
             emotional arousal and suggest a modulation of
             arousal-related encoding mechanisms when emotion is
             contingent on referencing personal beliefs.},
   Doi = {10.3758/s13415-018-0563-y},
   Key = {fds332826}
}

@article{fds327833,
   Author = {Monge, ZA and Wing, EA and Stokes, J and Cabeza, R},
   Title = {Search and recovery of autobiographical and laboratory
             memories: Shared and distinct neural components.},
   Journal = {Neuropsychologia},
   Volume = {110},
   Pages = {44-54},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2017.07.030},
   Abstract = {Functional neuroimaging evidence suggests that there are
             differences in the neural correlates of episodic memory for
             laboratory stimuli (laboratory memory) and for events from
             one's own life (autobiographical memory). However, this
             evidence is scarce and often confounded with differences in
             memory testing procedures. Here, we directly compared the
             neural mechanisms underlying the search and recovery of
             autobiographical and laboratory memories while minimizing
             testing differences. Before scanning, participants completed
             a laboratory memory encoding task in which they studied
             four-word "chains" spread across three word pairs. During
             scanning, participants completed a laboratory memory
             retrieval task, in which they recalled the word chains, and
             an autobiographical memory retrieval task, in which they
             recalled specific personal events associated with word cues.
             Importantly, response times were similar in the two tasks,
             allowing for a direct comparison of the activation time
             courses. We found that during memory search (searching for
             the memory target), similar brain regions were activated
             during both the autobiographical and laboratory tasks,
             whereas during memory recovery (accessing the memory traces;
             i.e., ecphory), clear differences emerged: regions of the
             default mode network (DMN) were activated greater during
             autobiographical than laboratory memory, whereas the
             bilateral superior parietal lobules were activated greater
             during laboratory than autobiographical memory. Also,
             multivariate functional connectivity analyses revealed that
             regardless of memory stage, the DMN and ventral attention
             network exhibited a more integrated topology in the
             functional network underlying autobiographical (vs.
             laboratory) memory retrieval, whereas the fronto-parietal
             task control network exhibited a more integrated topology in
             the functional network underlying laboratory (vs.
             autobiographical) memory retrieval. These findings further
             characterize the shared and distinct neural components
             underlying autobiographical and laboratory memories, and
             suggest that differences in autobiographical vs. laboratory
             memory brain activation previously reported in the
             literature reflect memory recovery rather than search
             differences.},
   Doi = {10.1016/j.neuropsychologia.2017.07.030},
   Key = {fds327833}
}

@article{fds333712,
   Author = {Fandakova, Y and Sander, MC and Grandy, TH and Cabeza, R and Werkle-Bergner, M and Shing, YL},
   Title = {Age differences in false memory: The importance of retrieval
             monitoring processes and their modulation by memory
             quality.},
   Journal = {Psychology and Aging},
   Volume = {33},
   Number = {1},
   Pages = {119-133},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1037/pag0000212},
   Abstract = {Older adults are more likely than younger adults to falsely
             recall past episodes that occurred differently or not at
             all. We examined whether older adults' propensity for false
             associative memory is related to declines in postretrieval
             monitoring processes and their modulation with varying
             memory representations. Younger (N = 20) and older adults (N
             = 32) studied and relearned unrelated scene-word pairs,
             followed by a final cued recall that was used to distribute
             the pairs for an associative recognition test 24 hours
             later. This procedure allowed individualized formation of
             rearranged pairs that were made up of elements of pairs that
             were correctly recalled in the final cued recall
             ("high-quality" pairs), and of pairs that were not correctly
             recalled ("low-quality" pairs). Both age groups falsely
             recognized more low-quality than high-quality rearranged
             pairs, with a less pronounced reduction in false alarms to
             high-quality pairs in older adults. In younger adults,
             cingulo-opercular activity was enhanced for false alarms and
             for low-quality correct rejections, consistent with its role
             in postretrieval monitoring. Older adults did not show such
             modulated recruitment, suggesting deficits in their
             selective engagement of monitoring processes given
             variability in the fidelity of memory representations. There
             were no age differences in hippocampal activity, which was
             higher for high-quality than low-quality correct rejections
             in both age groups. These results demonstrate that the
             engagement of cingulo-opercular monitoring mechanisms varies
             with memory representation quality and contributes to
             age-related deficits in false associative memory. (PsycINFO
             Database Record},
   Doi = {10.1037/pag0000212},
   Key = {fds333712}
}

@article{fds330539,
   Author = {Browndyke, JN and Berger, M and Smith, PJ and Harshbarger, TB and Monge,
             ZA and Panchal, V and Bisanar, TL and Glower, DD and Alexander, JH and Cabeza, R and Welsh-Bohmer, K and Newman, MF and Mathew, JP and Duke
             Neurologic Outcomes Research Group (NORG)},
   Title = {Task-related changes in degree centrality and local
             coherence of the posterior cingulate cortex after major
             cardiac surgery in older adults.},
   Journal = {Human Brain Mapping},
   Volume = {39},
   Number = {2},
   Pages = {985-1003},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1002/hbm.23898},
   Abstract = {Older adults often display postoperative cognitive decline
             (POCD) after surgery, yet it is unclear to what extent
             functional connectivity (FC) alterations may underlie these
             deficits. We examined for postoperative voxel-wise FC
             changes in response to increased working memory load demands
             in cardiac surgery patients and nonsurgical controls.Older
             cardiac surgery patients (n = 25) completed a verbal
             N-back working memory task during MRI scanning and cognitive
             testing before and 6 weeks after surgery; nonsurgical
             controls with cardiac disease (n = 26) underwent these
             assessments at identical time intervals. We measured
             postoperative changes in degree centrality, the number of
             edges attached to a brain node, and local coherence, the
             temporal homogeneity of regional functional correlations,
             using voxel-wise graph theory-based FC metrics. Group ×
             time differences were evaluated in these FC metrics
             associated with increased N-back working memory load
             (2-back > 1-back), using a two-stage partitioned
             variance, mixed ANCOVA.Cardiac surgery patients demonstrated
             postoperative working memory load-related degree centrality
             increases in the left dorsal posterior cingulate cortex
             (dPCC; p < .001, cluster p-FWE < .05). The dPCC also
             showed a postoperative increase in working memory
             load-associated local coherence (p < .001, cluster
             p-FWE < .05). dPCC degree centrality and local coherence
             increases were inversely associated with global cognitive
             change in surgery patients (p < .01), but not in
             controls.Cardiac surgery patients showed postoperative
             increases in working memory load-associated degree
             centrality and local coherence of the dPCC that were
             inversely associated with postoperative global cognitive
             outcomes and independent of perioperative cerebrovascular
             damage.},
   Doi = {10.1002/hbm.23898},
   Key = {fds330539}
}

@article{fds333223,
   Author = {Davis, SW and Wing, EA and Cabeza, R},
   Title = {Contributions of the ventral parietal cortex to declarative
             memory.},
   Journal = {Handbook of Clinical Neurology},
   Volume = {151},
   Pages = {525-553},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/b978-0-444-63622-5.00027-9},
   Abstract = {Our understanding of the role that ventral parietal cortex
             (VPC) plays in declarative memory processes has changed
             dramatically over the last two decades. The goal of this
             chapter is to provide a concise overview data concerning VPC
             involvement in episodic memory (EM), and to connect this
             data to several key theories of VPC function. We review
             evidence from five methodological domains in cognitive
             neuroscience: neuropsychological lesion evidence, univariate
             activation studies, multivoxel pattern analyses, functional
             connectivity studies, and brain stimulation experiments. We
             discuss how the body of empirical work bears on putative
             mnemonic functions of VPC related to attention and stimulus
             representation, and detail the strengths and weaknesses of
             related theories. Lastly, we identify several broad
             conceptual questions raised by recent investigations, and
             outline directions for future research.},
   Doi = {10.1016/b978-0-444-63622-5.00027-9},
   Key = {fds333223}
}

@article{fds329045,
   Author = {Davis, SW and Luber, B and Murphy, DLK and Lisanby, SH and Cabeza,
             R},
   Title = {Frequency-specific neuromodulation of local and distant
             connectivity in aging and episodic memory
             function.},
   Journal = {Human Brain Mapping},
   Volume = {38},
   Number = {12},
   Pages = {5987-6004},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1002/hbm.23803},
   Abstract = {A growing literature has focused on the brain's ability to
             augment processing in local regions by recruiting distant
             communities of neurons in response to neural decline or
             insult. In particular, both younger and older adult
             populations recruit bilateral prefrontal cortex (PFC) as a
             means of compensating for increasing neural effort to
             maintain successful cognitive function. However, it remains
             unclear how local changes in neural activity affect the
             recruitment of this adaptive mechanism. To address this
             problem, we combined graph theoretical measures from
             functional MRI with diffusion weighted imaging and
             repetitive transcranial magnetic stimulation (rTMS) to
             resolve a central hypothesis: how do aged brains flexibly
             adapt to local changes in cortical activity? Specifically,
             we applied neuromodulation to increase or decrease local
             activity in a cortical region supporting successful memory
             encoding (left dorsolateral PFC or DLPFC) using 5 or 1 Hz
             rTMS, respectively. We then assessed a region's local
             within-module degree, or the distributed between-module
             degree (BMD) between distant cortical communities. We
             predicted that (1) local stimulation-related deficits may be
             counteracted by boosting BMD between bilateral PFC, and that
             this effect should be (2) positively correlated with
             structural connectivity. Both predictions were confirmed; 5
             Hz rTMS increased local success-related activity and local
             increases in PFC connectivity, while 1 Hz rTMS decreases
             local activity and triggered a more distributed pattern of
             bilateral PFC connectivity to compensate for this local
             inhibitory effect. These results provide an integrated,
             causal explanation for the network interactions associated
             with successful memory encoding in older adults. Hum Brain
             Mapp 38:5987-6004, 2017. © 2017 Wiley Periodicals,
             Inc.},
   Doi = {10.1002/hbm.23803},
   Key = {fds329045}
}

@misc{fds333678,
   Author = {Doshi, A and Cabeza, R and Berger, M},
   Title = {Geriatric anesthesia: Age-dependent changes in the central
             and peripheral nervous systems},
   Pages = {145-160},
   Booktitle = {Geriatric Anesthesiology: Third Edition},
   Year = {2017},
   Month = {October},
   ISBN = {9783319668789},
   url = {http://dx.doi.org/10.1007/978-3-319-66878-9_10},
   Doi = {10.1007/978-3-319-66878-9_10},
   Key = {fds333678}
}

@article{fds326734,
   Author = {Brashier, NM and Umanath, S and Cabeza, R and Marsh,
             EJ},
   Title = {Competing cues: Older adults rely on knowledge in the face
             of fluency.},
   Journal = {Psychology and Aging},
   Volume = {32},
   Number = {4},
   Pages = {331-337},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1037/pag0000156},
   Abstract = {Consumers regularly encounter repeated false claims in
             political and marketing campaigns, but very little empirical
             work addresses their impact among older adults. Repeated
             statements feel easier to process, and thus more truthful,
             than new ones (i.e., illusory truth). When judging truth,
             older adults' accumulated general knowledge may offset this
             perception of fluency. In two experiments, participants read
             statements that contradicted information stored in memory; a
             post-experimental knowledge check confirmed what individual
             participants knew. Unlike young adults, older adults
             exhibited illusory truth only when they lacked knowledge
             about claims. This interaction between knowledge and fluency
             extends dual-process theories of aging. (PsycINFO Database
             Record},
   Doi = {10.1037/pag0000156},
   Key = {fds326734}
}

@article{fds323839,
   Author = {Geib, BR and Stanley, ML and Dennis, NA and Woldorff, MG and Cabeza,
             R},
   Title = {From hippocampus to whole-brain: The role of integrative
             processing in episodic memory retrieval.},
   Journal = {Human Brain Mapping},
   Volume = {38},
   Number = {4},
   Pages = {2242-2259},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1002/hbm.23518},
   Abstract = {Multivariate functional connectivity analyses of
             neuroimaging data have revealed the importance of complex,
             distributed interactions between disparate yet
             interdependent brain regions. Recent work has shown that
             topological properties of functional brain networks are
             associated with individual and group differences in
             cognitive performance, including in episodic memory. After
             constructing functional whole-brain networks derived from an
             event-related fMRI study of memory retrieval, we examined
             differences in functional brain network architecture between
             forgotten and remembered words. This study yielded three
             main findings. First, graph theory analyses showed that
             successfully remembering compared to forgetting was
             associated with significant changes in the connectivity
             profile of the left hippocampus and a corresponding increase
             in efficient communication with the rest of the brain.
             Second, bivariate functional connectivity analyses indicated
             stronger interactions between the left hippocampus and a
             retrieval assembly for remembered versus forgotten items.
             This assembly included the left precuneus, left caudate,
             bilateral supramarginal gyrus, and the bilateral
             dorsolateral superior frontal gyrus. Integrative properties
             of the retrieval assembly were greater for remembered than
             forgotten items. Third, whole-brain modularity analyses
             revealed that successful memory retrieval was marginally
             significantly associated with a less segregated modular
             architecture in the network. The magnitude of the decreases
             in modularity between remembered and forgotten conditions
             was related to memory performance. These findings indicate
             that increases in integrative properties at the nodal,
             retrieval assembly, and whole-brain topological levels
             facilitate memory retrieval, while also underscoring the
             potential of multivariate brain connectivity approaches for
             providing valuable new insights into the neural bases of
             memory processes. Hum Brain Mapp 38:2242-2259, 2017. © 2017
             Wiley Periodicals, Inc.},
   Doi = {10.1002/hbm.23518},
   Key = {fds323839}
}

@article{fds320544,
   Author = {Davis, SW and Stanley, ML and Moscovitch, M and Cabeza,
             R},
   Title = {Resting-state networks do not determine cognitive function
             networks: a commentary on Campbell and Schacter
             (2016).},
   Journal = {Language, Cognition and Neuroscience},
   Volume = {32},
   Number = {6},
   Pages = {669-673},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1080/23273798.2016.1252847},
   Doi = {10.1080/23273798.2016.1252847},
   Key = {fds320544}
}

@article{fds332754,
   Author = {Geib, BR and Stanley, ML and Wing, EA and Laurienti, PJ and Cabeza,
             R},
   Title = {Hippocampal Contributions to the Large-Scale Episodic Memory
             Network Predict Vivid Visual Memories.},
   Journal = {Cerebral Cortex (New York, N.Y. : 1991)},
   Volume = {27},
   Number = {1},
   Pages = {680-693},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1093/cercor/bhv272},
   Abstract = {A common approach in memory research is to isolate the
             function(s) of individual brain regions, such as the
             hippocampus, without addressing how those regions interact
             with the larger network. To investigate the properties of
             the hippocampus embedded within large-scale networks, we
             used functional magnetic resonance imaging and graph theory
             to characterize complex hippocampal interactions during the
             active retrieval of vivid versus dim visual memories. The
             study yielded 4 main findings. First, the right hippocampus
             displayed greater communication efficiency with the network
             (shorter path length) and became a more convergent structure
             for information integration (higher centrality measures) for
             vivid than dim memories. Second, vivid minus dim differences
             in our graph theory measures of interest were greater in
             magnitude for the right hippocampus than for any other
             region in the 90-region network. Moreover, the right
             hippocampus significantly reorganized its set of direct
             connections from dim to vivid memory retrieval. Finally,
             beyond the hippocampus, communication throughout the
             whole-brain network was more efficient (shorter global path
             length) for vivid than dim memories. In sum, our findings
             illustrate how multivariate network analyses can be used to
             investigate the roles of specific regions within the
             large-scale network, while also accounting for global
             network changes.},
   Doi = {10.1093/cercor/bhv272},
   Key = {fds332754}
}


%% Caruso, Valeria   
@article{fds335640,
   Author = {Caruso, VC and Mohl, JT and Glynn, C and Lee, J and Willett, SM and Zaman,
             A and Ebihara, AF and Estrada, R and Freiwald, WA and Tokdar, ST and Groh,
             JM},
   Title = {Single neurons may encode simultaneous stimuli by switching
             between activity patterns.},
   Journal = {Nature Communications},
   Volume = {9},
   Number = {1},
   Pages = {2715},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1038/s41467-018-05121-8},
   Abstract = {How the brain preserves information about multiple
             simultaneous items is poorly understood. We report that
             single neurons can represent multiple stimuli by
             interleaving signals across time. We record single units in
             an auditory region, the inferior colliculus, while monkeys
             localize 1 or 2 simultaneous sounds. During dual-sound
             trials, we find that some neurons fluctuate between firing
             rates observed for each single sound, either on a
             whole-trial or on a sub-trial timescale. These fluctuations
             are correlated in pairs of neurons, can be predicted by the
             state of local field potentials prior to sound onset, and,
             in one monkey, can predict which sound will be reported
             first. We find corroborating evidence of fluctuating
             activity patterns in a separate dataset involving responses
             of inferotemporal cortex neurons to multiple visual stimuli.
             Alternation between activity patterns corresponding to each
             of multiple items may therefore be a general strategy to
             enhance the brain processing capacity, potentially linking
             such disparate phenomena as variable neural firing, neural
             oscillations, and limits in attentional/memory
             capacity.},
   Doi = {10.1038/s41467-018-05121-8},
   Key = {fds335640}
}


%% Caspi, Avshalom   
@article{fds336491,
   Author = {Crush, E and Arseneault, L and Moffitt, TE and Danese, A and Caspi, A and Jaffee, SR and Matthews, T and Fisher, HL},
   Title = {Protective factors for psychotic experiences amongst
             adolescents exposed to multiple forms of
             victimization.},
   Journal = {Journal of Psychiatric Research},
   Volume = {104},
   Pages = {32-38},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.jpsychires.2018.06.011},
   Abstract = {Experiencing multiple types of victimization
             (poly-victimization) during adolescence is associated with
             the onset of psychotic experiences (such as hearing voices,
             having visions, or being extremely paranoid). However, many
             poly-victimized adolescents will not develop such
             subclinical phenomena and the factors that protect them are
             unknown. This study investigated whether individual, family,
             or community-level characteristics were associated with an
             absence of psychotic experiences amongst poly-victimized
             adolescents. Participants were from the Environmental Risk
             (E-Risk) Longitudinal Twin Study, a nationally-representative
             cohort of 2232 UK-born twins. Exposure to seven different
             types of victimization between ages 12-18 was ascertained
             using a modified version of the Juvenile Victimization
             Questionnaire at age 18. Adolescents were also interviewed
             about psychotic experiences at age 18. Protective factors
             were measured at ages 12 and 18. We found that exposure to
             poly-victimization during adolescence was associated with
             age-18 psychotic experiences (OR = 4.62, 95% CI
             3.59-5.94, P < 0.001), but more than a third of the
             poly-victimized adolescents reported having no psychotic
             experiences (40.1%). Greater social support was found to be
             protective against adolescent psychotic experiences even
             amongst those exposed to poly-victimization. Engaging in
             physical activity and greater neighborhood social cohesion
             were also associated with a reduced likelihood of age-18
             psychotic experiences in the whole sample, with
             non-significant trends in the poly-victimized group.
             Increasing social support and promoting physical activity
             appear to be important areas for future research into the
             development of preventive interventions targeting adolescent
             psychotic experiences. This adds further weight to calls to
             increase the promotion of these factors on a public health
             scale.},
   Doi = {10.1016/j.jpsychires.2018.06.011},
   Key = {fds336491}
}

@article{fds336496,
   Author = {Caspi, A and Moffitt, TE},
   Title = {All for One and One for All: Mental Disorders in One
             Dimension.},
   Journal = {The American Journal of Psychiatry},
   Volume = {175},
   Number = {9},
   Pages = {831-844},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1176/appi.ajp.2018.17121383},
   Abstract = {In both child and adult psychiatry, empirical evidence has
             now accrued to suggest that a single dimension is able to
             measure a person's liability to mental disorder, comorbidity
             among disorders, persistence of disorders over time, and
             severity of symptoms. This single dimension of general
             psychopathology has been termed "p," because it conceptually
             parallels a dimension already familiar to behavioral
             scientists and clinicians: the "g" factor of general
             intelligence. As the g dimension reflects low to high mental
             ability, the p dimension represents low to high
             psychopathology severity, with thought disorder at the
             extreme. The dimension of p unites all disorders. It
             influences present/absent status on hundreds of psychiatric
             symptoms, which modern nosological systems typically
             aggregate into dozens of distinct diagnoses, which in turn
             aggregate into three overarching domains, namely, the
             externalizing, internalizing, and psychotic experience
             domains, which finally aggregate into one dimension of
             psychopathology from low to high: p. Studies show that the
             higher a person scores on p, the worse that person fares on
             measures of family history of psychiatric illness, brain
             function, childhood developmental history, and adult life
             impairment. A dimension of p may help account for ubiquitous
             nonspecificity in psychiatry: multiple disorders share the
             same risk factors and biomarkers and often respond to the
             same therapies. Here, the authors summarize the history of
             the unidimensional idea, review modern research into p,
             demystify statistical models, articulate some implications
             of p for prevention and clinical practice, and outline a
             transdiagnostic research agenda. [AJP AT 175: Remembering
             Our Past As We Envision Our Future October 1910: A Study of
             Association in Insanity Grace Helen Kent and A.J. Rosanoff:
             "No sharp distinction can be drawn between mental health and
             mental disease; a large collection of material shows a
             gradual and not an abrupt transition from the normal state
             to pathological states."(Am J Psychiatry 1910; 67(2):317-390
             )].},
   Doi = {10.1176/appi.ajp.2018.17121383},
   Key = {fds336496}
}

@article{fds337341,
   Author = {Belsky, DW and Domingue, BW and Wedow, R and Arseneault, L and Boardman,
             JD and Caspi, A and Conley, D and Fletcher, JM and Freese, J and Herd, P and Moffitt, TE and Poulton, R and Sicinski, K and Wertz, J and Harris,
             KM},
   Title = {Genetic analysis of social-class mobility in five
             longitudinal studies.},
   Journal = {Proceedings of the National Academy of Sciences of the
             United States of America},
   Volume = {115},
   Number = {31},
   Pages = {E7275-E7284},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1073/pnas.1801238115},
   Abstract = {A summary genetic measure, called a "polygenic score,"
             derived from a genome-wide association study (GWAS) of
             education can modestly predict a person's educational and
             economic success. This prediction could signal a biological
             mechanism: Education-linked genetics could encode
             characteristics that help people get ahead in life.
             Alternatively, prediction could reflect social history:
             People from well-off families might stay well-off for social
             reasons, and these families might also look alike
             genetically. A key test to distinguish biological mechanism
             from social history is if people with higher education
             polygenic scores tend to climb the social ladder beyond
             their parents' position. Upward mobility would indicate
             education-linked genetics encodes characteristics that
             foster success. We tested if education-linked polygenic
             scores predicted social mobility in >20,000 individuals in
             five longitudinal studies in the United States, Britain, and
             New Zealand. Participants with higher polygenic scores
             achieved more education and career success and accumulated
             more wealth. However, they also tended to come from
             better-off families. In the key test, participants with
             higher polygenic scores tended to be upwardly mobile
             compared with their parents. Moreover, in sibling-difference
             analysis, the sibling with the higher polygenic score was
             more upwardly mobile. Thus, education GWAS discoveries are
             not mere correlates of privilege; they influence social
             mobility within a life. Additional analyses revealed that a
             mother's polygenic score predicted her child's attainment
             over and above the child's own polygenic score, suggesting
             parents' genetics can also affect their children's
             attainment through environmental pathways. Education GWAS
             discoveries affect socioeconomic attainment through
             influence on individuals' family-of-origin environments and
             their social mobility.},
   Doi = {10.1073/pnas.1801238115},
   Key = {fds337341}
}

@article{fds336492,
   Author = {Belsky, DW and Moffitt, TE and Cohen, AA and Corcoran, DL and Levine,
             ME and Prinz, JA and Schaefer, J and Sugden, K and Williams, B and Poulton,
             R and Caspi, A},
   Title = {Eleven Telomere, Epigenetic Clock, and Biomarker-Composite
             Quantifications of Biological Aging: Do They Measure the
             Same Thing?},
   Journal = {American Journal of Epidemiology},
   Volume = {187},
   Number = {6},
   Pages = {1220-1230},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1093/aje/kwx346},
   Abstract = {The geroscience hypothesis posits that therapies to slow
             biological processes of aging can prevent disease and extend
             healthy years of life. To test such "geroprotective"
             therapies in humans, outcome measures are needed that can
             assess extension of disease-free life span. This need has
             spurred development of different methods to quantify
             biological aging. But different methods have not been
             systematically compared in the same humans. We implemented 7
             methods to quantify biological aging using repeated-measures
             physiological and genomic data in 964 middle-aged humans in
             the Dunedin Study (New Zealand; persons born 1972-1973). We
             studied 11 measures in total: telomere-length and erosion, 3
             epigenetic-clocks and their ticking rates, and 3
             biomarker-composites. Contrary to expectation, we found low
             agreement between different measures of biological aging. We
             next compared associations between biological aging measures
             and outcomes that geroprotective therapies seek to modify:
             physical functioning, cognitive decline, and subjective
             signs of aging, including aged facial appearance. The
             71-cytosine-phosphate-guanine epigenetic clock and biomarker
             composites were consistently related to these aging-related
             outcomes. However, effect sizes were modest. Results
             suggested that various proposed approaches to quantifying
             biological aging may not measure the same aspects of the
             aging process. Further systematic evaluation and refinement
             of measures of biological aging is needed to furnish
             outcomes for geroprotector trials.},
   Doi = {10.1093/aje/kwx346},
   Key = {fds336492}
}

@article{fds336500,
   Author = {Marzi, SJ and Sugden, K and Arseneault, L and Belsky, DW and Burrage, J and Corcoran, DL and Danese, A and Fisher, HL and Hannon, E and Moffitt, TE and Odgers, CL and Pariante, C and Poulton, R and Williams, BS and Wong,
             CCY and Mill, J and Caspi, A},
   Title = {Analysis of DNA Methylation in Young People: Limited
             Evidence for an Association Between Victimization Stress and
             Epigenetic Variation in Blood.},
   Journal = {The American Journal of Psychiatry},
   Volume = {175},
   Number = {6},
   Pages = {517-529},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.17060693},
   Abstract = {DNA methylation has been proposed as an epigenetic mechanism
             by which early-life experiences become "embedded" in the
             genome and alter transcriptional processes to compromise
             health. The authors sought to investigate whether early-life
             victimization stress is associated with genome-wide DNA
             methylation.The authors tested the hypothesis that
             victimization is associated with DNA methylation in the
             Environmental Risk (E-Risk) Longitudinal Study, a nationally
             representative 1994-1995 birth cohort of 2,232 twins born in
             England and Wales and assessed at ages 5, 7, 10, 12, and 18
             years. Multiple forms of victimization were ascertained in
             childhood and adolescence (including physical, sexual, and
             emotional abuse; neglect; exposure to intimate-partner
             violence; bullying; cyber-victimization; and
             crime).Epigenome-wide analyses of polyvictimization across
             childhood and adolescence revealed few significant
             associations with DNA methylation in peripheral blood at age
             18, but these analyses were confounded by tobacco smoking
             and/or did not survive co-twin control tests. Secondary
             analyses of specific forms of victimization revealed sparse
             associations with DNA methylation that did not replicate
             across different operationalizations of the same putative
             victimization experience. Hypothesis-driven analyses of six
             candidate genes in the stress response (NR3C1, FKBP5, BDNF,
             AVP, CRHR1, SLC6A4) did not reveal predicted associations
             with DNA methylation in probes annotated to these
             genes.Findings from this epidemiological analysis of the
             epigenetic effects of early-life stress do not support the
             hypothesis of robust changes in DNA methylation in
             victimized young people. We need to come to terms with the
             possibility that epigenetic epidemiology is not yet well
             matched to experimental, nonhuman models in uncovering the
             biological embedding of stress.},
   Doi = {10.1176/appi.ajp.2017.17060693},
   Key = {fds336500}
}

@article{fds336493,
   Author = {Rasmussen, LJH and Moffitt, TE and Eugen-Olsen, J and Belsky, DW and Danese, A and Harrington, H and Houts, RM and Poulton, R and Sugden, K and Williams, B and Caspi, A},
   Title = {Cumulative childhood risk is associated with a new measure
             of chronic inflammation in adulthood.},
   Journal = {Journal of Child Psychology and Psychiatry, and Allied
             Disciplines},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1111/jcpp.12928},
   Abstract = {Childhood risk factors are associated with elevated
             inflammatory biomarkers in adulthood, but it is unknown
             whether these risk factors are associated with increased
             adult levels of the chronic inflammation marker soluble
             urokinase plasminogen activator receptor (suPAR). We aimed
             to test the hypothesis that childhood exposure to risk
             factors for adult disease is associated with elevated suPAR
             in adulthood and to compare suPAR with the oft-reported
             inflammatory biomarker C-reactive protein (CRP).Prospective
             study of a population-representative 1972-1973 birth cohort;
             the Dunedin Multidisciplinary Health and Development Study
             observed participants to age 38 years. Main childhood
             predictors were poor health, socioeconomic disadvantage,
             adverse childhood experiences (ACEs), low IQ, and poor
             self-control. Main adult outcomes were adulthood
             inflammation measured as suPAR and high-sensitivity CRP
             (hsCRP).Participants with available plasma samples at age 38
             were included (N = 837, 50.5% male). suPAR (mean
             2.40 ng/ml; SD 0.91) was positively correlated with hsCRP
             (r 0.15, p < .001). After controlling for sex, body mass
             index (BMI), and smoking, children who experienced more
             ACEs, lower IQ, or had poorer self-control showed elevated
             adult suPAR. When the five childhood risks were aggregated
             into a Cumulative Childhood Risk index, and controlling for
             sex, BMI, and smoking, Cumulative Childhood Risk was
             associated with higher suPAR (b 0.10; SE 0.03; p = .002).
             Cumulative Childhood Risk predicted elevated suPAR, after
             controlling for hsCRP (b 0.18; SE 0.03; p < .001).Exposure
             to more childhood risk factors was associated with higher
             suPAR levels, independent of CRP. suPAR is a useful addition
             to studies connecting childhood risk to adult inflammatory
             burden.},
   Doi = {10.1111/jcpp.12928},
   Key = {fds336493}
}

@article{fds336494,
   Author = {Schaefer, JD and Moffitt, TE and Arseneault, L and Danese, A and Fisher,
             HL and Houts, R and Sheridan, MA and Wertz, J and Caspi,
             A},
   Title = {Adolescent Victimization and Early-Adult Psychopathology:
             Approaching Causal Inference Using a Longitudinal Twin Study
             to Rule Out Noncausal Explanations.},
   Journal = {Clinical Psychological Science : a Journal of the
             Association for Psychological Science},
   Volume = {6},
   Number = {3},
   Pages = {352-371},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1177/2167702617741381},
   Abstract = {Adolescence is the peak age for both victimization and
             mental disorder onset. Previous research has reported
             associations between victimization exposure and many
             psychiatric conditions. However, causality remains
             controversial. Within the Environmental Risk Longitudinal
             Twin Study, we tested whether seven types of adolescent
             victimization increased risk of multiple psychiatric
             conditions and approached causal inference by systematically
             ruling out noncausal explanations. Longitudinal
             within-individual analyses showed that victimization was
             followed by increased mental health problems over a
             childhood baseline of emotional/behavioral problems.
             Discordant-twin analyses showed that victimization increased
             risk of mental health problems independent of family
             background and genetic risk. Both childhood and adolescent
             victimization made unique contributions to risk.
             Victimization predicted heightened generalized liability
             (the "p factor") to multiple psychiatric spectra, including
             internalizing, externalizing, and thought disorders. Results
             recommend violence reduction and identification and
             treatment of adolescent victims to reduce psychiatric
             burden.},
   Doi = {10.1177/2167702617741381},
   Key = {fds336494}
}

@article{fds333704,
   Author = {Wertz, J and Caspi, A and Belsky, DW and Beckley, AL and Arseneault, L and Barnes, JC and Corcoran, DL and Hogan, S and Houts, RM and Morgan, N and Odgers, CL and Prinz, JA and Sugden, K and Williams, BS and Poulton, R and Moffitt, TE},
   Title = {Genetics and Crime: Integrating New Genomic Discoveries Into
             Psychological Research About Antisocial Behavior.},
   Journal = {Psychological Science},
   Volume = {29},
   Number = {5},
   Pages = {791-803},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1177/0956797617744542},
   Abstract = {Drawing on psychological and sociological theories of crime
             causation, we tested the hypothesis that genetic risk for
             low educational attainment (assessed via a genome-wide
             polygenic score) is associated with criminal offending. We
             further tested hypotheses of how polygenic risk relates to
             the development of antisocial behavior from childhood
             through adulthood. Across the Dunedin and Environmental Risk
             (E-Risk) birth cohorts of individuals growing up 20 years
             and 20,000 kilometers apart, education polygenic scores
             predicted risk of a criminal record with modest effects.
             Polygenic risk manifested during primary schooling in lower
             cognitive abilities, lower self-control, academic
             difficulties, and truancy, and it was associated with a
             life-course-persistent pattern of antisocial behavior that
             onsets in childhood and persists into adulthood. Crime is
             central in the nature-nurture debate, and findings reported
             here demonstrate how molecular-genetic discoveries can be
             incorporated into established theories of antisocial
             behavior. They also suggest that improving school
             experiences might prevent genetic influences on crime from
             unfolding.},
   Doi = {10.1177/0956797617744542},
   Key = {fds333704}
}

@article{fds336495,
   Author = {Matthews, T and Danese, A and Caspi, A and Fisher, HL and Goldman-Mellor, S and Kepa, A and Moffitt, TE and Odgers, CL and Arseneault, L},
   Title = {Lonely young adults in modern Britain: findings from an
             epidemiological cohort study.},
   Journal = {Psychological Medicine},
   Pages = {1-10},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1017/s0033291718000788},
   Abstract = {The aim of this study was to build a detailed, integrative
             profile of the correlates of young adults' feelings of
             loneliness, in terms of their current health and functioning
             and their childhood experiences and circumstances.Data were
             drawn from the Environmental Risk Longitudinal Twin Study, a
             birth cohort of 2232 individuals born in England and Wales
             in 1994 and 1995. Loneliness was measured when participants
             were aged 18. Regression analyses were used to test
             concurrent associations between loneliness and health and
             functioning in young adulthood. Longitudinal analyses were
             conducted to examine childhood factors associated with young
             adult loneliness.Lonelier young adults were more likely to
             experience mental health problems, to engage in physical
             health risk behaviours, and to use more negative strategies
             to cope with stress. They were less confident in their
             employment prospects and were more likely to be out of work.
             Lonelier young adults were, as children, more likely to have
             had mental health difficulties and to have experienced
             bullying and social isolation. Loneliness was evenly
             distributed across genders and socioeconomic
             backgrounds.Young adults' experience of loneliness co-occurs
             with a diverse range of problems, with potential
             implications for health in later life. The findings
             underscore the importance of early intervention to prevent
             lonely young adults from being trapped in loneliness as they
             age.},
   Doi = {10.1017/s0033291718000788},
   Key = {fds336495}
}

@article{fds326205,
   Author = {Romer, AL and Knodt, AR and Houts, R and Brigidi, BD and Moffitt, TE and Caspi, A and Hariri, AR},
   Title = {Structural alterations within cerebellar circuitry are
             associated with general liability for common mental
             disorders.},
   Journal = {Molecular Psychiatry},
   Volume = {23},
   Number = {4},
   Pages = {1084-1090},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1038/mp.2017.57},
   Abstract = {Accumulating mental-health research encourages a shift in
             focus toward transdiagnostic dimensional features that are
             shared across categorical disorders. In support of this
             shift, recent studies have identified a general liability
             factor for psychopathology-sometimes called the 'p factor'-
             that underlies shared risk for a wide range of mental
             disorders. Identifying neural correlates of this general
             liability would substantiate its importance in
             characterizing the shared origins of mental disorders and
             help us begin to understand the mechanisms through which the
             'p factor' contributes to risk. Here we believe we first
             replicate the 'p factor' using cross-sectional data from a
             volunteer sample of 1246 university students, and then using
             high-resolution multimodal structural neuroimaging, we
             demonstrate that individuals with higher 'p factor' scores
             show reduced structural integrity of white matter pathways,
             as indexed by lower fractional anisotropy values, uniquely
             within the pons. Whole-brain analyses further revealed that
             higher 'p factor' scores are associated with reduced gray
             matter volume in the occipital lobe and left cerebellar
             lobule VIIb, which is functionally connected with prefrontal
             regions supporting cognitive control. Consistent with the
             preponderance of cerebellar afferents within the pons, we
             observed a significant positive correlation between the
             white matter integrity of the pons and cerebellar gray
             matter volume associated with higher 'p factor' scores. The
             results of our analyses provide initial evidence that
             structural alterations in corticocerebellar circuitry
             supporting core functions related to the basic integration,
             coordination and monitoring of information may contribute to
             a general liability for common mental disorders.},
   Doi = {10.1038/mp.2017.57},
   Key = {fds326205}
}

@article{fds336499,
   Author = {Beckley, AL and Caspi, A and Arseneault, L and Barnes, JC and Fisher,
             HL and Harrington, H and Houts, R and Morgan, N and Odgers, CL and Wertz,
             J and Moffitt, TE},
   Title = {The Developmental Nature of the Victim-Offender
             Overlap.},
   Volume = {4},
   Number = {1},
   Pages = {24-49},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1007/s40865-017-0068-3},
   Abstract = {It is well-established that victims and offenders are often
             the same people, a phenomenon known as the victim-offender
             overlap, but the developmental nature of this overlap
             remains uncertain. In this study, we drew from a
             developmental theoretical framework to test effects of
             genetics, individual characteristics, and
             routine-activity-based risks. Drawing from developmental
             literature, we additionally tested the effect of an
             accumulation of adverse childhood experiences (ACEs).Data
             came from the Environmental Risk (E-Risk) Study, a
             representative UK birth cohort of 2232 twins born in
             1994-1995 and followed to age 18 (with 93% retention). Crime
             victimization and offending were assessed through
             self-reports at age 18 (but findings replicated using crime
             records). We used the classical twin study method to
             decompose variance in the victim-offender overlap into
             genetic and environmental components. We used logistic
             regression to test the effects of childhood risk factors.In
             contrast to past twin studies, we found that environment (as
             well as genes) contributed to the victim-offender overlap.
             Our logistic regression results showed that childhood low
             self-control and childhood antisocial behavior nearly
             doubled the odds of becoming a victim-offender, compared to
             a victim-only or an offender-only. Each additional ACE
             increased the odds of becoming a victim-offender, compared
             to a victim-only or an offender-only, by approximately 12%,
             pointing to the importance of cumulative childhood
             adversity.This study showed that the victim-offender overlap
             is, at least partially, developmental in nature and
             predictable from personal childhood characteristics and an
             accumulation of many adverse childhood experiences.},
   Doi = {10.1007/s40865-017-0068-3},
   Key = {fds336499}
}

@article{fds328904,
   Author = {Meier, MH and Caspi, A and Danese, A and Fisher, HL and Houts, R and Arseneault, L and Moffitt, TE},
   Title = {Associations between adolescent cannabis use and
             neuropsychological decline: a longitudinal co-twin control
             study.},
   Journal = {Addiction (Abingdon, England)},
   Volume = {113},
   Number = {2},
   Pages = {257-265},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1111/add.13946},
   Abstract = {This study tested whether adolescents who used cannabis or
             met criteria for cannabis dependence showed
             neuropsychological impairment prior to cannabis initiation
             and neuropsychological decline from before to after cannabis
             initiation.A longitudinal co-twin control study.Participants
             were 1989 twins from the Environmental Risk (E-Risk)
             Longitudinal Twin Study, a nationally representative birth
             cohort of twins born in England and Wales from 1994 to
             1995.Frequency of cannabis use and cannabis dependence were
             assessed at age 18. Intelligence quotient (IQ) was obtained
             at ages 5, 12 and 18. Executive functions were assessed at
             age 18.Compared with adolescents who did not use cannabis,
             adolescents who used cannabis had lower IQ in childhood
             prior to cannabis initiation and lower IQ at age 18, but
             there was little evidence that cannabis use was associated
             with IQ decline from ages 12-18. For example, adolescents
             with cannabis dependence had age 12 and age 18 IQ scores
             that were 5.61 (t = -3.11, P = 0.002) and 7.34 IQ points
             (t = -5.27, P < 0.001) lower than adolescents without
             cannabis dependence, but adolescents with cannabis
             dependence did not show greater IQ decline from age 12-18
             (t = -1.27, P = 0.20). Moreover, adolescents who used
             cannabis had poorer executive functions at age 18 than
             adolescents who did not use cannabis, but these associations
             were generally not apparent within twin pairs. For example,
             twins who used cannabis more frequently than their co-twin
             performed similarly to their co-twin on five of six
             executive function tests (Ps > 0.10). The one exception
             was that twins who used cannabis more frequently than their
             co-twin performed worse on one working memory test (Spatial
             Span reversed; β = -0.07, P = 0.036).Short-term
             cannabis use in adolescence does not appear to cause IQ
             decline or impair executive functions, even when cannabis
             use reaches the level of dependence. Family background
             factors explain why adolescent cannabis users perform worse
             on IQ and executive function tests.},
   Doi = {10.1111/add.13946},
   Key = {fds328904}
}

@article{fds331469,
   Author = {Newbury, J and Arseneault, L and Caspi, A and Moffitt, TE and Odgers,
             CL and Fisher, HL},
   Title = {Cumulative Effects of Neighborhood Social Adversity and
             Personal Crime Victimization on Adolescent Psychotic
             Experiences.},
   Journal = {Schizophrenia Bulletin},
   Volume = {44},
   Number = {2},
   Pages = {348-358},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/schbul/sbx060},
   Abstract = {Little is known about the impact of urbanicity, adverse
             neighborhood conditions and violent crime victimization on
             the emergence of adolescent psychotic experiences.Participants
             were from the Environmental Risk (E-Risk) Longitudinal Twin
             Study, a nationally-representative cohort of 2232 British
             twins who were interviewed about adolescent psychotic
             experiences at age 18. Urbanicity, neighborhood
             characteristics, and personal victimization by violent crime
             were measured during childhood and adolescence via geocoded
             census data, surveys of over 5000 immediate neighbors of the
             E-Risk participants, and interviews with participants
             themselves.Adolescents raised in urban vs rural
             neighborhoods were significantly more likely to have
             psychotic experiences (OR = 1.67, 95% CI = 1.21-2.30, P =
             .002). This association remained significant after
             considering potential confounders including family
             socioeconomic status, family psychiatric history, and
             adolescent substance problems (OR = 1.43, 95% CI =
             1.01-2.03, P = .042), but became nonsignificant after
             considering adverse social conditions in urban neighborhoods
             such as low social cohesion and high neighborhood disorder
             (OR = 1.35, 95% CI = 0.94-1.92, P = .102). The combined
             association of adverse neighborhood social conditions and
             personal crime victimization with adolescent psychotic
             experiences (adjusted OR = 4.86, 95% CI = 3.28-7.20, P <
             .001) was substantially greater than for either exposure
             alone, highlighting a potential interaction between
             neighborhood conditions and crime victimization (interaction
             contrast ratio = 1.81, 95% CI = -0.03 to 3.65) that was
             significant at the P = .054 level.Cumulative effects of
             adverse neighborhood social conditions and personal
             victimization by violent crime during upbringing partly
             explain why adolescents in urban settings are more likely to
             report psychotic experiences. Early intervention efforts for
             psychosis could be targeted towards victimized youth living
             in urban and socially adverse neighborhoods.},
   Doi = {10.1093/schbul/sbx060},
   Key = {fds331469}
}

@article{fds332385,
   Author = {Beckley, AL and Caspi, A and Broadbent, J and Harrington, H and Houts,
             RM and Poulton, R and Ramrakha, S and Reuben, A and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Criminal
             Offending.},
   Journal = {Jama Pediatrics},
   Volume = {172},
   Number = {2},
   Pages = {166-173},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1001/jamapediatrics.2017.4005},
   Abstract = {Lead is a neurotoxin with well-documented effects on health.
             Research suggests that lead may be associated with criminal
             behavior. This association is difficult to disentangle from
             low socioeconomic status, a factor in both lead exposure and
             criminal offending.To test the hypothesis that a higher
             childhood blood lead level (BLL) is associated with greater
             risk of criminal conviction, recidivism (repeat conviction),
             conviction for violent offenses, and variety of
             self-reported criminal offending in a setting where BLL was
             not associated with low socioeconomic status.A total of 553
             individuals participated in a prospective study based on a
             population-representative cohort born between April 1, 1972,
             and March 31, 1973, from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (December 2012). Statistical
             analysis was performed from November 10, 2016, to September
             5, 2017.Blood lead level measured at age 11 years.Official
             criminal conviction cumulative to age 38 years (data
             collected in 2013), single conviction or recidivism,
             conviction for nonviolent or violent crime, and
             self-reported variety of crime types at ages 15, 18, 21, 26,
             32, and 38 years.Participants included 553 individuals (255
             female and 298 male participants) who had their blood tested
             for lead at age 11 years. The mean (SD) BLL at age 11 years
             was 11.01 (4.62) μg/dL. A total of 154 participants (27.8%)
             had a criminal conviction, 86 (15.6%) had recidivated, and
             53 (9.6%) had a violent offense conviction. Variety scores
             for self-reported offending ranged from 0 to 10 offense
             types at each assessment; higher numbers indicated greater
             crime involvement. Self-reported offending followed the
             well-established age-crime curve (ie, the mean [SD] variety
             of self-reported offending increased from 1.99 [2.82] at age
             15 years to its peak of 4.24 [3.15] at age 18 years and 4.22
             [3.02] at age 21 years and declined thereafter to 1.10
             [1.59] at age 38 years). Blood lead level was a poor
             discriminator between no conviction and conviction (area
             under the curve, 0.58). Overall, associations between BLL
             and conviction outcomes were weak. The estimated effect of
             BLL was lower for recidivism than for single convictions and
             lower for violent offending than for nonviolent offending.
             Sex-adjusted associations between BLL reached statistical
             significance for only 1 of the 6 self-reported offending
             outcomes at age 15 years (r = 0.10; 95% CI, 0.01-0.18;
             P = .02).This study overcomes past limitations of
             studies of BLL and crime by studying the association in a
             place and time where the correlation was not confounded by
             childhood socioeconomic status. Findings failed to support a
             dose-response association between BLL and consequential
             criminal offending.},
   Doi = {10.1001/jamapediatrics.2017.4005},
   Key = {fds332385}
}

@article{fds329541,
   Author = {Baldwin, JR and Arseneault, L and Caspi, A and Fisher, HL and Moffitt,
             TE and Odgers, CL and Pariante, C and Ambler, A and Dove, R and Kepa, A and Matthews, T and Menard, A and Sugden, K and Williams, B and Danese,
             A},
   Title = {Childhood victimization and inflammation in young adulthood:
             A genetically sensitive cohort study.},
   Journal = {Brain, Behavior, and Immunity},
   Volume = {67},
   Pages = {211-217},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.bbi.2017.08.025},
   Abstract = {Childhood victimization is an important risk factor for
             later immune-related disorders. Previous evidence has
             demonstrated that childhood victimization is associated with
             elevated levels of inflammation biomarkers measured decades
             after exposure. However, it is unclear whether this
             association is (1) already detectable in young people, (2)
             different in males and females, and (3) confounded by
             genetic liability to inflammation. Here we sought to address
             these questions.Participants were 2232 children followed
             from birth to age 18years as part of the Environmental Risk
             (E-Risk) Longitudinal Twin Study. Childhood victimization
             was measured prospectively from birth to age 12years.
             Inflammation was measured through C-reactive protein (CRP)
             levels in dried blood spots at age 18years. Latent genetic
             liability for high inflammation levels was assessed through
             a twin-based method.Greater exposure to childhood
             victimization was associated with higher CRP levels at age
             18 (serum-equivalent means were 0.65 in non-victimized Study
             members, 0.74 in those exposed to one victimization type,
             and 0.81 in those exposed to poly-victimization; p=0.018).
             However, this association was driven by a significant
             association in females (serum-equivalent means were 0.75 in
             non-victimized females, 0.87 in those exposed to one type of
             victimization, and 1.19 in those exposed to
             poly-victimization; p=0.010), while no significant
             association was observed in males (p=0.19). Victimized
             females showed elevated CRP levels independent of latent
             genetic influence, as well as childhood socioeconomic
             status, and waist-hip ratio and body temperature at the time
             of CRP assessment.Childhood victimization is associated with
             elevated CRP levels in young women, independent of latent
             genetic influences and other key risk factors. These results
             strengthen causal inference about the effects of childhood
             victimization on inflammation levels in females by
             accounting for potential genetic confounding.},
   Doi = {10.1016/j.bbi.2017.08.025},
   Key = {fds329541}
}

@article{fds329982,
   Author = {Newbury, JB and Arseneault, L and Moffitt, TE and Caspi, A and Danese,
             A and Baldwin, JR and Fisher, HL},
   Title = {Measuring childhood maltreatment to predict early-adult
             psychopathology: Comparison of prospective informant-reports
             and retrospective self-reports.},
   Journal = {Journal of Psychiatric Research},
   Volume = {96},
   Pages = {57-64},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jpsychires.2017.09.020},
   Abstract = {Both prospective informant-reports and retrospective
             self-reports may be used to measure childhood maltreatment,
             though both methods entail potential limitations such as
             underestimation and memory biases. The validity and utility
             of standard measures of childhood maltreatment requires
             clarification in order to inform the design of future
             studies investigating the mental health consequences of
             maltreatment. The present study assessed agreement between
             prospective informant-reports and retrospective self-reports
             of childhood maltreatment, as well as the comparative
             utility of both reports for predicting a range of
             psychiatric problems at age 18. Data were obtained from the
             Environmental Risk (E-Risk) Longitudinal Twin Study, a
             nationally-representative birth cohort of 2232 children
             followed to 18 years of age (with 93% retention). Childhood
             maltreatment was assessed in two ways: (i) prospective
             informant-reports from caregivers, researchers, and
             clinicians when children were aged 5, 7, 10 and 12; and (ii)
             retrospective self-reports of maltreatment experiences
             occurring up to age 12, obtained at age 18 using the
             Childhood Trauma Questionnaire. Participants were privately
             interviewed at age 18 concerning several psychiatric
             problems including depression, anxiety, self-injury,
             alcohol/cannabis dependence, and conduct disorder. There was
             only slight to fair agreement between prospective and
             retrospective reports of childhood maltreatment (all
             Kappa's ≤ 0.31). Both prospective and retrospective
             reports of maltreatment were associated with age-18
             psychiatric problems, though the strongest associations were
             found when maltreatment was retrospectively self-reported.
             These findings indicate that prospective and retrospective
             reports of childhood maltreatment capture largely
             non-overlapping groups of individuals. Young adults who
             recall being maltreated have a particularly elevated risk
             for psychopathology.},
   Doi = {10.1016/j.jpsychires.2017.09.020},
   Key = {fds329982}
}

@article{fds332035,
   Author = {Wertz, J and Agnew-Blais, J and Caspi, A and Danese, A and Fisher, HL and Goldman-Mellor, S and Moffitt, TE and Arseneault,
             L},
   Title = {From Childhood Conduct Problems to Poor Functioning at Age
             18 Years: Examining Explanations in a Longitudinal Cohort
             Study.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {57},
   Number = {1},
   Pages = {54-60.e4},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.09.437},
   Abstract = {Childhood conduct problems are associated with poor
             functioning in early adulthood. We tested a series of
             hypotheses to understand the mechanisms underlying this
             association.We used data from the Environmental Risk
             (E-Risk) Longitudinal Twin Study, a birth cohort of 2,232
             twins born in England and Wales in 1994 and 1995, followed
             up to age 18 years with 93% retention. Severe conduct
             problems in childhood were assessed at ages 5, 7, and 10
             years using parent and teacher reports. Poor functioning at
             age 18 years, including cautions and convictions, daily
             cigarette smoking, heavy drinking, and psychosocial
             difficulties, was measured through interviews with
             participants and official crime record searches.Participants
             18 years old with versus without a childhood history of
             severe conduct problems had greater rates of each poor
             functional outcome, and they were more likely to experience
             multiple poor outcomes. This association was partly
             accounted for by concurrent psychopathology in early
             adulthood, as well as by early familial risk factors, both
             genetic and environmental. Childhood conduct problems,
             however, continued to predict poor outcomes at age 18 years
             after accounting for these explanations.Children with severe
             conduct problems display poor functioning at age 18 years
             because of concurrent problems in early adulthood and
             familial risk factors originating in childhood. However,
             conduct problems also exert a lasting effect on young
             people's lives independent of these factors, pointing to
             early conduct problems as a target for early interventions
             aimed at preventing poor functional outcomes.},
   Doi = {10.1016/j.jaac.2017.09.437},
   Key = {fds332035}
}

@article{fds331398,
   Author = {Rivenbark, JG and Odgers, CL and Caspi, A and Harrington, H and Hogan,
             S and Houts, RM and Poulton, R and Moffitt, TE},
   Title = {The high societal costs of childhood conduct problems:
             evidence from administrative records up to age 38 in a
             longitudinal birth cohort.},
   Journal = {Journal of Child Psychology and Psychiatry, and Allied
             Disciplines},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1111/jcpp.12850},
   Abstract = {Children with conduct problems that persist into adulthood
             are at increased risk for future behavioral, health, and
             social problems. However, the longer term public service
             usage among these children has not been fully documented. To
             aid public health and intervention planning, adult service
             usage across criminal justice, health care, and social
             welfare domains is compared among all individuals from a
             representative cohort who followed different conduct problem
             trajectories from childhood into adulthood.Participants are
             from the Dunedin Multidisciplinary Health and Development
             Study, a prospective, representative cohort of consecutive
             births (N = 1,037) from April 1972 to March 1973 in
             Dunedin, New Zealand. Regression analyses were used to
             compare levels of public service usage up to age 38,
             gathered via administrative and electronic medical records,
             between participants who displayed distinct subtypes of
             childhood conduct problems (low, childhood-limited,
             adolescent-onset, and life-course persistent).Children
             exhibiting life-course persistent conduct problems used
             significantly more services as adults than those with low
             levels of childhood conduct problems. Although this group
             comprised only 9.0% of the population, they accounted for
             53.3% of all convictions, 15.7% of emergency department
             visits, 20.5% of prescription fills, 13.1% of injury claims,
             and 24.7% of welfare benefit months. Half of this group
             (50.0%) also accrued high service use across all three
             domains of criminal justice, health, and social welfare
             services, as compared to only 11.3% of those with low
             conduct problems (OR = 7.27, 95% CI = 4.42-12.0).Conduct
             problems in childhood signal high future costs in terms of
             service utilization across multiple sectors. Future
             evaluations of interventions aimed at conduct problems
             should also track potential reductions in health burden and
             service usage that stretch into midlife.},
   Doi = {10.1111/jcpp.12850},
   Key = {fds331398}
}

@article{fds331468,
   Author = {Newbury, JB and Arseneault, L and Caspi, A and Moffitt, TE and Odgers,
             CL and Baldwin, JR and Zavos, HMS and Fisher, HL},
   Title = {In the eye of the beholder: Perceptions of neighborhood
             adversity and psychotic experiences in adolescence.},
   Journal = {Development and Psychopathology},
   Volume = {29},
   Number = {5},
   Pages = {1823-1837},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1017/s0954579417001420},
   Abstract = {Adolescent psychotic experiences increase risk for
             schizophrenia and other severe psychopathology in adulthood.
             Converging evidence implicates urban and adverse
             neighborhood conditions in the etiology of adolescent
             psychotic experiences, but the role of young people's
             personal perceptions of disorder (i.e., physical and social
             signs of threat) in their neighborhood is unknown. This was
             examined using data from the Environmental Risk Longitudinal
             Twin Study, a nationally representative birth cohort of
             2,232 British twins. Participants were interviewed at age 18
             about psychotic phenomena and perceptions of disorder in the
             neighborhood. Multilevel, longitudinal, and genetically
             sensitive analyses investigated the association between
             perceptions of neighborhood disorder and adolescent
             psychotic experiences. Adolescents who perceived higher
             levels of neighborhood disorder were significantly more
             likely to have psychotic experiences, even after accounting
             for objectively/independently measured levels of crime and
             disorder, neighborhood- and family-level socioeconomic
             status, family psychiatric history, adolescent substance and
             mood problems, and childhood psychotic symptoms: odds ratio
             = 1.62, 95% confidence interval [1.27, 2.05], p < .001. The
             phenotypic overlap between adolescent psychotic experiences
             and perceptions of neighborhood disorder was explained by
             overlapping common environmental influences, rC = .88, 95%
             confidence interval [0.26, 1.00]. Findings suggest that
             early psychological interventions to prevent adolescent
             psychotic experiences should explore the role of young
             people's (potentially modifiable) perceptions of threatening
             neighborhood conditions.},
   Doi = {10.1017/s0954579417001420},
   Key = {fds331468}
}

@article{fds330402,
   Author = {Schaefer, JD and Scult, MA and Caspi, A and Arseneault, L and Belsky,
             DW and Hariri, AR and Harrington, H and Houts, R and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Is low cognitive functioning a predictor or consequence of
             major depressive disorder? A test in two longitudinal birth
             cohorts.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s095457941700164x},
   Abstract = {Cognitive impairment has been identified as an important
             aspect of major depressive disorder (MDD). We tested two
             theories regarding the association between MDD and cognitive
             functioning using data from longitudinal cohort studies. One
             theory, the cognitive reserve hypothesis, suggests that
             higher cognitive ability in childhood decreases risk of
             later MDD. The second, the scarring hypothesis, instead
             suggests that MDD leads to persistent cognitive deficits
             following disorder onset. We tested both theories in the
             Dunedin Study, a population-representative cohort followed
             from birth to midlife and assessed repeatedly for both
             cognitive functioning and psychopathology. We also used data
             from the Environmental Risk Longitudinal Twin Study to test
             whether childhood cognitive functioning predicts future MDD
             risk independent of family-wide and genetic risk using a
             discordant twin design. Contrary to both hypotheses, we
             found that childhood cognitive functioning did not predict
             future risk of MDD, nor did study members with a past
             history of MDD show evidence of greater cognitive decline
             unless MDD was accompanied by other comorbid psychiatric
             conditions. Our results thus suggest that low cognitive
             functioning is related to comorbidity, but is neither an
             antecedent nor an enduring consequence of MDD. Future
             research may benefit from considering cognitive deficits
             that occur during depressive episodes from a transdiagnostic
             perspective.},
   Doi = {10.1017/s095457941700164x},
   Key = {fds330402}
}

@article{fds328903,
   Author = {Williams, MJA and Milne, BJ and Ambler, A and Theodore, R and Ramrakha,
             S and Caspi, A and Moffitt, TE and Poulton, R},
   Title = {Childhood body mass index and endothelial dysfunction
             evaluated by peripheral arterial tonometry in early
             midlife.},
   Journal = {International Journal of Obesity (2005)},
   Volume = {41},
   Number = {9},
   Pages = {1355-1360},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1038/ijo.2017.108},
   Abstract = {Endothelial dysfunction predicts mortality but it is unknown
             whether childhood obesity predicts adult endothelial
             dysfunction. The aim of this study was to determine whether
             anthropometric indices of body fat in childhood, adolescence
             and early midlife are associated with endothelial
             dysfunction in early midlife.Participants belonged to a
             representative birth cohort of 1037 individuals born in
             Dunedin, New Zealand in 1972 and 1973 and followed to age 38
             years, with 95% retention (the Dunedin Multidisciplinary
             Health and Development Study). We assessed anthropometric
             indices of obesity at ages 3, 5, 7, 9, 11, 13, 15, 18, 21,
             26, 32 and 38 years. We tested associations between
             endothelial function assessed by peripheral arterial
             tonometry (PAT) at age 38 and; age 38 cardiovascular risk
             factors; age 3 body mass index (BMI); and four BMI
             trajectory groups from childhood to early midlife.Early
             midlife endothelial dysfunction was associated with BMI,
             large waist circumference, low high-density lipoprotein
             cholesterol, low cardiorespiratory fitness and increased
             high-sensitivity C-reactive protein. After adjustment for
             sex and childhood socioeconomic status, 3-year-olds with BMI
             1 s.d. above the mean had Framingham-reactive hyperemia
             index (F-RHI) ratios that were 0.10 below those with normal
             BMI (β=-0.10, 95% confidence interval (CI) -0.17 to -0.03,
             P=0.007) at age 38. Cohort members in the 'overweight',
             'obese' and 'morbidly obese' trajectories had F-RHI ratios
             that were 0.08 (β=-0.08, 95% CI -0.14 to -0.03, P=0.003),
             0.13 (β=-0.13, 95% CI -0.21 to -0.06, P<0.001) and 0.17
             (β=-0.17, 95% CI -0.33 to -0.01, P=0.033), respectively,
             below age-peers in the 'normal' trajectory.Childhood BMI and
             the trajectories of BMI from childhood to early midlife
             predict endothelial dysfunction evaluated by PAT in early
             midlife.},
   Doi = {10.1038/ijo.2017.108},
   Key = {fds328903}
}

@article{fds328625,
   Author = {Matthews, T and Danese, A and Gregory, AM and Caspi, A and Moffitt, TE and Arseneault, L},
   Title = {Sleeping with one eye open: loneliness and sleep quality in
             young adults.},
   Journal = {Psychological Medicine},
   Volume = {47},
   Number = {12},
   Pages = {2177-2186},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1017/S0033291717000629},
   Abstract = {Feelings of loneliness are common among young adults, and
             are hypothesized to impair the quality of sleep. In the
             present study, we tested associations between loneliness and
             sleep quality in a nationally representative sample of young
             adults. Further, based on the hypothesis that sleep problems
             in lonely individuals are driven by increased vigilance for
             threat, we tested whether past exposure to violence
             exacerbated this association.Data were drawn from the
             Environmental Risk (E-Risk) Longitudinal Twin Study, a birth
             cohort of 2232 twins born in England and Wales in 1994 and
             1995. We measured loneliness using items from the UCLA
             Loneliness Scale, and sleep quality using the Pittsburgh
             Sleep Quality Index. We controlled for covariates including
             social isolation, psychopathology, employment status and
             being a parent of an infant. We examined twin differences to
             control for unmeasured genetic and family environment
             factors.Feelings of loneliness were associated with worse
             overall sleep quality. Loneliness was associated
             specifically with subjective sleep quality and daytime
             dysfunction. These associations were robust to controls for
             covariates. Among monozygotic twins, within-twin pair
             differences in loneliness were significantly associated with
             within-pair differences in sleep quality, indicating an
             association independent of unmeasured familial influences.
             The association between loneliness and sleep quality was
             exacerbated among individuals exposed to violence
             victimization in adolescence or maltreatment in
             childhood.Loneliness is robustly associated with poorer
             sleep quality in young people, underscoring the importance
             of early interventions to mitigate the long-term outcomes of
             loneliness. Special care should be directed towards
             individuals who have experienced victimization.},
   Doi = {10.1017/S0033291717000629},
   Key = {fds328625}
}

@article{fds327017,
   Author = {Suppli, NP and Bukh, JD and Moffitt, TE and Caspi, A and Johansen, C and Tjønneland, A and Kessing, LV and Dalton, SO},
   Title = {Genetic variants in 5-HTTLPR, BDNF, HTR1A, COMT, and FKBP5
             and risk for treated depression after cancer
             diagnosis.},
   Journal = {Depression and Anxiety},
   Volume = {34},
   Number = {9},
   Pages = {845-855},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1002/da.22660},
   Abstract = {The role of gene-environment interactions in the
             pathogenesis of depression is unclear. Previous studies
             addressed vulnerability for depression after childhood
             adversity and stressful life events among carriers of
             numerous specific genetic variants; however, the importance
             of individual genetic variants, the environmental exposures
             with which they interact, and the magnitude of the risk
             conveyed by these interactions remain elusive.We included
             7,320 people with a first primary cancer identified in the
             prospective Diet, Cancer and Health study in an exposed-only
             cohort study. The mean age of the individuals was 68 years
             (5th, 95th percentiles: 58, 78) at cancer diagnosis. Using
             Cox regression models and cumulative incidence plots, we
             analyzed the associations between genetic variants in
             5-HTTLPR, BDNF, HTR1A, COMT, and FKBP5 and use of
             antidepressants as well as hospital contact for depression
             after diagnosis of cancer.Overall, we observed no
             statistically significant associations, with nonsignificant
             hazard ratio estimates for use of antidepressants of
             0.95-1.07.This study of elderly people indicates that it is
             unlikely that the investigated genetic variants are
             clinically relevantly associated with depression after
             diagnosis of cancer. The mechanisms for gene-environment
             interactions in younger individuals are probably different,
             and we advise caution in extrapolating our results to early
             life stress. However, conclusion from the present study
             might be generalizable to elderly persons exposed to other
             stressful life events.},
   Doi = {10.1002/da.22660},
   Key = {fds327017}
}

@article{fds326204,
   Author = {Belsky, DW and Caspi, A and Cohen, HJ and Kraus, WE and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Impact of early personal-history characteristics on the Pace
             of Aging: implications for clinical trials of therapies to
             slow aging and extend healthspan.},
   Journal = {Aging Cell},
   Volume = {16},
   Number = {4},
   Pages = {644-651},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/acel.12591},
   Abstract = {Therapies to extend healthspan are poised to move from
             laboratory animal models to human clinical trials.
             Translation from mouse to human will entail challenges,
             among them the multifactorial heterogeneity of human aging.
             To inform clinical trials about this heterogeneity, we
             report how humans' pace of biological aging relates to
             personal-history characteristics. Because geroprotective
             therapies must be delivered by midlife to prevent
             age-related disease onset, we studied young-adult members of
             the Dunedin Study 1972-73 birth cohort (n = 954). Cohort
             members' Pace of Aging was measured as coordinated decline
             in the integrity of multiple organ systems, by quantifying
             rate of decline across repeated measurements of 18
             biomarkers assayed when cohort members were ages 26, 32, and
             38 years. The childhood personal-history characteristics
             studied were known predictors of age-related disease and
             mortality, and were measured prospectively during childhood.
             Personal-history characteristics of familial longevity,
             childhood social class, adverse childhood experiences, and
             childhood health, intelligence, and self-control all
             predicted differences in cohort members' adulthood Pace of
             Aging. Accumulation of more personal-history risks predicted
             faster Pace of Aging. Because trials of anti-aging therapies
             will need to ascertain personal histories retrospectively,
             we replicated results using cohort members' retrospective
             personal-history reports made in adulthood. Because many
             trials recruit participants from clinical settings, we
             replicated results in the cohort subset who had recent
             health system contact according to electronic medical
             records. Quick, inexpensive measures of trial participants'
             early personal histories can enable clinical trials to study
             who volunteers for trials, who adheres to treatment, and who
             responds to anti-aging therapies.},
   Doi = {10.1111/acel.12591},
   Key = {fds326204}
}

@article{fds325848,
   Author = {Kotov, R and Krueger, RF and Watson, D and Achenbach, TM and Althoff,
             RR and Bagby, RM and Brown, TA and Carpenter, WT and Caspi, A and Clark,
             LA and Eaton, NR and Forbes, MK and Forbush, KT and Goldberg, D and Hasin,
             D and Hyman, SE and Ivanova, MY and Lynam, DR and Markon, K and Miller, JD and Moffitt, TE and Morey, LC and Mullins-Sweatt, SN and Ormel, J and Patrick, CJ and Regier, DA and Rescorla, L and Ruggero, CJ and Samuel,
             DB and Sellbom, M and Simms, LJ and Skodol, AE and Slade, T and South, SC and Tackett, JL and Waldman, ID and Waszczuk, MA and Widiger, TA and Wright,
             AGC and Zimmerman, M},
   Title = {The Hierarchical Taxonomy of Psychopathology (HiTOP): A
             dimensional alternative to traditional nosologies.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {4},
   Pages = {454-477},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1037/abn0000258},
   Abstract = {The reliability and validity of traditional taxonomies are
             limited by arbitrary boundaries between psychopathology and
             normality, often unclear boundaries between disorders,
             frequent disorder co-occurrence, heterogeneity within
             disorders, and diagnostic instability. These taxonomies went
             beyond evidence available on the structure of
             psychopathology and were shaped by a variety of other
             considerations, which may explain the aforementioned
             shortcomings. The Hierarchical Taxonomy Of Psychopathology
             (HiTOP) model has emerged as a research effort to address
             these problems. It constructs psychopathological syndromes
             and their components/subtypes based on the observed
             covariation of symptoms, grouping related symptoms together
             and thus reducing heterogeneity. It also combines
             co-occurring syndromes into spectra, thereby mapping out
             comorbidity. Moreover, it characterizes these phenomena
             dimensionally, which addresses boundary problems and
             diagnostic instability. Here, we review the development of
             the HiTOP and the relevant evidence. The new classification
             already covers most forms of psychopathology. Dimensional
             measures have been developed to assess many of the
             identified components, syndromes, and spectra. Several
             domains of this model are ready for clinical and research
             applications. The HiTOP promises to improve research and
             clinical practice by addressing the aforementioned
             shortcomings of traditional nosologies. It also provides an
             effective way to summarize and convey information on risk
             factors, etiology, pathophysiology, phenomenology, illness
             course, and treatment response. This can greatly improve the
             utility of the diagnosis of mental disorders. The new
             classification remains a work in progress. However, it is
             developing rapidly and is poised to advance mental health
             research and care significantly as the relevant science
             matures. (PsycINFO Database Record},
   Doi = {10.1037/abn0000258},
   Key = {fds325848}
}

@article{fds324429,
   Author = {Danese, A and Moffitt, TE and Arseneault, L and Bleiberg, BA and Dinardo, PB and Gandelman, SB and Houts, R and Ambler, A and Fisher, HL and Poulton, R and Caspi, A},
   Title = {The Origins of Cognitive Deficits in Victimized Children:
             Implications for Neuroscientists and Clinicians.},
   Journal = {The American Journal of Psychiatry},
   Volume = {174},
   Number = {4},
   Pages = {349-361},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1176/appi.ajp.2016.16030333},
   Abstract = {Individuals reporting a history of childhood violence
             victimization have impaired brain function. However, the
             clinical significance, reproducibility, and causality of
             these findings are disputed. The authors used data from two
             large cohort studies to address these research questions
             directly.The authors tested the association between
             prospectively collected measures of childhood violence
             victimization and cognitive functions in childhood,
             adolescence, and adulthood among 2,232 members of the U.K.
             E-Risk Study and 1,037 members of the New Zealand Dunedin
             Study who were followed up from birth until ages 18 and 38
             years, respectively. Multiple measures of victimization and
             cognition were used, and comparisons were made of cognitive
             scores for twins discordant for victimization.Individuals
             exposed to childhood victimization had pervasive impairments
             in clinically relevant cognitive functions, including
             general intelligence, executive function, processing speed,
             memory, perceptual reasoning, and verbal comprehension in
             adolescence and adulthood. However, the observed cognitive
             deficits in victimized individuals were largely explained by
             cognitive deficits that predated childhood victimization and
             by confounding genetic and environmental risks.Findings from
             two population-representative birth cohorts totaling more
             than 3,000 individuals and born 20 years and 20,000 km apart
             suggest that the association between childhood violence
             victimization and later cognition is largely noncausal, in
             contrast to conventional interpretations. These findings
             support the adoption of a more circumspect approach to
             causal inference in the neuroscience of stress. Clinically,
             cognitive deficits should be conceptualized as individual
             risk factors for victimization as well as potential
             complicating features during treatment.},
   Doi = {10.1176/appi.ajp.2016.16030333},
   Key = {fds324429}
}

@article{fds325847,
   Author = {Reuben, A and Caspi, A and Belsky, DW and Broadbent, J and Harrington,
             H and Sugden, K and Houts, RM and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Association of Childhood Blood Lead Levels With Cognitive
             Function and Socioeconomic Status at Age 38 Years and With
             IQ Change and Socioeconomic Mobility Between Childhood and
             Adulthood.},
   Journal = {Jama},
   Volume = {317},
   Number = {12},
   Pages = {1244-1251},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1001/jama.2017.1712},
   Abstract = {Many children in the United States and around the world are
             exposed to lead, a developmental neurotoxin. The long-term
             cognitive and socioeconomic consequences of lead exposure
             are uncertain.To test the hypothesis that childhood lead
             exposure is associated with cognitive function and
             socioeconomic status in adulthood and with changes in IQ and
             socioeconomic mobility between childhood and midlife.A
             prospective cohort study based on a population-representative
             1972-1973 birth cohort from New Zealand; the Dunedin
             Multidisciplinary Health and Development Study observed
             participants to age 38 years (until December 2012).Childhood
             lead exposure ascertained as blood lead levels measured at
             age 11 years. High blood lead levels were observed among
             children from all socioeconomic status levels in this
             cohort.The IQ (primary outcome) and indexes of Verbal
             Comprehension, Perceptual Reasoning, Working Memory, and
             Processing Speed (secondary outcomes) were assessed at age
             38 years using the Wechsler Adult Intelligence Scale-IV
             (WAIS-IV; IQ range, 40-160). Socioeconomic status (primary
             outcome) was assessed at age 38 years using the New Zealand
             Socioeconomic Index-2006 (NZSEI-06; range, 10 [lowest]-90
             [highest]).Of 1037 original participants, 1007 were alive at
             age 38 years, of whom 565 (56%) had been lead tested at age
             11 years (54% male; 93% white). Mean (SD) blood lead level
             at age 11 years was 10.99 (4.63) µg/dL. Among blood-tested
             participants included at age 38 years, mean WAIS-IV score
             was 101.16 (14.82) and mean NZSEI-06 score was 49.75
             (17.12). After adjusting for maternal IQ, childhood IQ, and
             childhood socioeconomic status, each 5-µg/dL higher level
             of blood lead in childhood was associated with a 1.61-point
             lower score (95% CI, -2.48 to -0.74) in adult IQ, a
             2.07-point lower score (95% CI, -3.14 to -1.01) in
             perceptual reasoning, and a 1.26-point lower score (95% CI,
             -2.38 to -0.14) in working memory. Associations of childhood
             blood lead level with deficits in verbal comprehension and
             processing speed were not statistically significant. After
             adjusting for confounders, each 5-µg/dL higher level of
             blood lead in childhood was associated with a 1.79-unit
             lower score (95% CI, -3.17 to -0.40) in socioeconomic
             status. An association between greater blood lead levels and
             a decline in IQ and socioeconomic status from childhood to
             adulthood was observed with 40% of the association with
             downward mobility mediated by cognitive decline from
             childhood.In this cohort born in New Zealand in 1972-1973,
             childhood lead exposure was associated with lower cognitive
             function and socioeconomic status at age 38 years and with
             declines in IQ and with downward social mobility. Childhood
             lead exposure may have long-term ramifications.},
   Doi = {10.1001/jama.2017.1712},
   Key = {fds325847}
}

@article{fds321660,
   Author = {Schaefer, JD and Caspi, A and Belsky, DW and Harrington, H and Houts, R and Horwood, LJ and Hussong, A and Ramrakha, S and Poulton, R and Moffitt,
             TE},
   Title = {Enduring mental health: Prevalence and prediction.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {126},
   Number = {2},
   Pages = {212-224},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1037/abn0000232},
   Abstract = {We review epidemiological evidence indicating that most
             people will develop a diagnosable mental disorder,
             suggesting that only a minority experience enduring mental
             health. This minority has received little empirical study,
             leaving the prevalence and predictors of enduring mental
             health unknown. We turn to the population-representative
             Dunedin cohort, followed from birth to midlife, to compare
             people never-diagnosed with mental disorder (N = 171; 17%
             prevalence) to those diagnosed at 1-2 study waves, the
             cohort mode (N = 409). Surprisingly, compared to this modal
             group, never-diagnosed Study members were not born into
             unusually well-to-do families, nor did their enduring mental
             health follow markedly sound physical health, or unusually
             high intelligence. Instead, they tended to have an
             advantageous temperament/personality style, and negligible
             family history of mental disorder. As adults, they report
             superior educational and occupational attainment, greater
             life satisfaction, and higher-quality relationships. Our
             findings draw attention to "enduring mental health" as a
             revealing psychological phenotype and suggest it deserves
             further study. (PsycINFO Database Record},
   Doi = {10.1037/abn0000232},
   Key = {fds321660}
}

@article{fds328127,
   Author = {Moffitt, TE and Belsky, DW and Danese, A and Poulton, R and Caspi,
             A},
   Title = {The Longitudinal Study of Aging in Human Young Adults:
             Knowledge Gaps and Research Agenda.},
   Journal = {The Journals of Gerontology. Series A, Biological Sciences
             and Medical Sciences},
   Volume = {72},
   Number = {2},
   Pages = {210-215},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1093/gerona/glw191},
   Abstract = {To prevent onset of age-related diseases and physical and
             cognitive decline, interventions to slow human aging and
             extend health span must eventually be applied to people
             while they are still young and healthy. Yet most human aging
             research examines older adults, many with chronic disease,
             and little is known about aging in healthy young humans.This
             article explains how this knowledge gap is a barrier to
             extending health span and puts forward the case that
             geroscience should invest in researching the pace of aging
             in young adults. As one illustrative example, we describe an
             initial effort to study the pace of aging in a young-adult
             birth cohort by using repeated waves of biomarkers collected
             across the third and fourth decades to quantify the pace of
             coordinated physiological deterioration across multiple
             organ systems (eg, pulmonary, periodontal, cardiovascular,
             renal, hepatic, metabolic, and immune function).Findings
             provided proof of principle that it is possible to quantify
             individual variation in the pace of aging in young adults
             still free of age-related diseases.This article articulates
             research needs to improve longitudinal measurement of the
             pace of aging in young people, to pinpoint factors that slow
             or speed the pace of aging, to compare pace of aging against
             genomic clocks, to explain slow-aging young adults, and to
             apply pace of aging in preventive clinical trials of
             antiaging therapies. This article puts forward a research
             agenda to fill the knowledge gap concerning lifelong causes
             of aging.},
   Doi = {10.1093/gerona/glw191},
   Key = {fds328127}
}


%% Castrellon, Jaime J.   
@article{fds330888,
   Author = {Dang, LC and Samanez-Larkin, GR and Smith, CT and Castrellon, JJ and Perkins, SF and Cowan, RL and Claassen, DO and Zald,
             DH},
   Title = {FTO affects food cravings and interacts with age to
             influence age-related decline in food cravings.},
   Journal = {Physiology & Behavior},
   Volume = {192},
   Pages = {188-193},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.physbeh.2017.12.013},
   Abstract = {The fat mass and obesity associated gene (FTO) was the first
             gene identified by genome-wide association studies to
             correlate with higher body mass index (BMI) and increased
             odds of obesity. FTO remains the locus with the largest and
             most replicated effect on body weight, but the mechanism
             whereby FTO affects body weight and the development of
             obesity is not fully understood. Here we tested whether FTO
             is associated with differences in food cravings and a key
             aspect of dopamine function that has been hypothesized to
             influence food reward mechanisms. Moreover, as food cravings
             and dopamine function are known to decline with age, we
             explored effects of age on relations between FTO and food
             cravings and dopamine function. Seven-eight healthy subjects
             between 22 and 83years old completed the Food Cravings
             Questionnaire and underwent genotyping for FTO rs9939609,
             the first FTO single nucleotide polymorphism associated with
             obesity. Compared to TT homozygotes, individuals carrying
             the obesity-susceptible A allele had higher total food
             cravings, which correlated with higher BMI. Additionally,
             food cravings declined with age, but this age effect
             differed across variants of FTO rs9939609: while TT
             homozygotes showed the typical age-related decline in food
             cravings, there was no such decline among A carriers. All
             subjects were scanned with [18F]fallypride PET to assess a
             recent proposal that at the neurochemical level FTO alters
             dopamine D2-like receptor (DRD2) function to influence food
             reward related mechanisms. However, we observed no evidence
             of FTO effects on DRD2 availability.},
   Doi = {10.1016/j.physbeh.2017.12.013},
   Key = {fds330888}
}

@article{fds333912,
   Author = {Dang, LC and Samanez-Larkin, GR and Castrellon, JJ and Perkins, SF and Cowan, RL and Zald, DH},
   Title = {Individual differences in dopamine D2 receptor availability
             correlate with reward valuation.},
   Journal = {Cognitive, Affective & Behavioral Neuroscience},
   Volume = {18},
   Number = {4},
   Pages = {739-747},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.3758/s13415-018-0601-9},
   Abstract = {Reward valuation, which underlies all value-based
             decision-making, has been associated with dopamine function
             in many studies of nonhuman animals, but there is relatively
             less direct evidence for an association in humans. Here, we
             measured dopamine D2 receptor (DRD2) availability in vivo in
             humans to examine relations between individual differences
             in dopamine receptor availability and neural activity
             associated with a measure of reward valuation, expected
             value (i.e., the product of reward magnitude and the
             probability of obtaining the reward). Fourteen healthy adult
             subjects underwent PET with [18F]fallypride, a radiotracer
             with strong affinity for DRD2, and fMRI (on a separate day)
             while performing a reward valuation task. [18F]fallypride
             binding potential, reflecting DRD2 availability, in the
             midbrain correlated positively with neural activity
             associated with expected value, specifically in the left
             ventral striatum/caudate. The present results provide in
             vivo evidence from humans showing midbrain dopamine
             characteristics are associated with reward
             valuation.},
   Doi = {10.3758/s13415-018-0601-9},
   Key = {fds333912}
}

@article{fds333913,
   Author = {Seaman, KL and Brooks, N and Karrer, TM and Castrellon, JJ and Perkins,
             SF and Dang, L and Hsu, M and Zald, DH and Samanez-Larkin,
             GR},
   Title = {Subjective Value Representations during Effort, Probability,
             and Time Discounting across Adulthood.},
   Journal = {Social Cognitive and Affective Neuroscience},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1093/scan/nsy021},
   Abstract = {Every day, humans make countless decisions which require the
             integration of information about potential benefits (i.e.,
             rewards) with other decision features (i.e., effort
             required, probability of an outcome, or time delays). Here
             we examine the overlap and dissociation of behavioral
             preferences and neural representations of subjective value
             in the context of three different decision features
             (physical effort, probability, time delays) in a healthy
             adult life-span sample. While undergoing functional
             neuroimaging, participants (N = 75) made
             incentive-compatible choices between a smaller monetary
             reward with lower physical effort, higher probability, or a
             shorter time delay, versus a larger monetary reward with
             higher physical effort, lower probability, or a longer time
             delay. Behavioral preferences were estimated from observed
             choices and subjective values were computed using individual
             hyperbolic discount functions. We found that discount rates
             were uncorrelated across tasks. In spite of this apparent
             behavioral dissociation between preferences, we found
             overlapping subjective value-related activity in the medial
             prefrontal cortex across all three tasks. We found no
             consistent evidence for age differences in either
             preferences or the neural representations of subjective
             value across adulthood. These results suggest that while the
             tolerance of decision features is behaviorally dissociable,
             subjective value signals share a common representation
             across adulthood.},
   Doi = {10.1093/scan/nsy021},
   Key = {fds333913}
}

@article{fds328773,
   Author = {Dang, LC and Samanez-Larkin, GR and Castrellon, JJ and Perkins, SF and Cowan, RL and Newhouse, PA and Zald, DH},
   Title = {Spontaneous Eye Blink Rate (EBR) Is Uncorrelated with
             Dopamine D2 Receptor Availability and Unmodulated by
             Dopamine Agonism in Healthy Adults.},
   Journal = {Eneuro},
   Volume = {4},
   Number = {5},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1523/ENEURO.0211-17.2017},
   Abstract = {Spontaneous eye blink rate (EBR) has been proposed as a
             noninvasive, inexpensive marker of dopamine functioning.
             Support for a relation between EBR and dopamine function
             comes from observations that EBR is altered in populations
             with dopamine dysfunction and EBR changes under a
             dopaminergic manipulation. However, the evidence across the
             literature is inconsistent and incomplete. A direct
             correlation between EBR and dopamine function has so far
             been observed only in nonhuman animals. Given significant
             interest in using EBR as a proxy for dopamine function, this
             study aimed to verify a direct association in healthy, human
             adults. Here we measured EBR in healthy human subjects whose
             dopamine D2 receptor (DRD2) availability was assessed with
             positron emission tomography (PET)-[18F]fallypride to
             examine the predictive power of EBR for DRD2 availability.
             Effects of the dopamine agonist bromocriptine on EBR also
             were examined to determine the responsiveness of EBR to
             dopaminergic stimulation and, in light of the hypothesized
             inverted-U profile of dopamine effects, the role of DRD2
             availability in EBR responsivity to bromocriptine. Results
             from 20 subjects (age 33.6 ± 7.6 years, 9F) showed no
             relation between EBR and DRD2 availability. EBR also was not
             responsive to dopaminergic stimulation by bromocriptine, and
             individual differences in DRD2 availability did not modulate
             EBR responsivity to bromocriptine. Given that EBR is
             hypothesized to be particularly sensitive to DRD2 function,
             these findings suggest caution in using EBR as a proxy for
             dopamine function in healthy humans.},
   Doi = {10.1523/ENEURO.0211-17.2017},
   Key = {fds328773}
}

@article{fds327928,
   Author = {Barber, SJ and Castrellon, JJ and Opitz, P and Mather,
             M},
   Title = {Younger and older adults' collaborative recall of shared and
             unshared emotional pictures.},
   Journal = {Memory & Cognition},
   Volume = {45},
   Number = {5},
   Pages = {716-730},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.3758/s13421-017-0694-3},
   Abstract = {Although a group of people working together recalls more
             items than any one individual, they recall fewer unique
             items than the same number of people working apart whose
             responses are combined. This is known as collaborative
             inhibition, and it is a robust effect that occurs for both
             younger and older adults. However, almost all previous
             studies documenting collaborative inhibition have used
             stimuli that were neutral in emotional valence, low in
             arousal, and studied by all group members. In the current
             experiments, we tested the impact of picture-stimuli
             valence, picture-stimuli arousal, and information
             distribution in modulating the magnitude of collaborative
             inhibition. We included both younger and older adults
             because there are age differences in how people remember
             emotional pictures that could modulate any effects of
             emotion on collaborative inhibition. Results revealed that
             when information was shared (i.e., studied by all group
             members), there were robust collaborative inhibition effects
             for both neutral and emotional stimuli for both younger and
             older adults. However, when information was unshared (i.e.,
             studied by only a single group member), these effects were
             attenuated. Together, these results provide mixed support
             for the retrieval strategy disruption account of
             collaborative inhibition. Supporting the retrieval strategy
             disruption account, unshared study information was less
             susceptible to collaborative inhibition than shared study
             information. Contradicting the retrieval strategy disruption
             account, emotional valence and arousal did not modulate the
             magnitude of collaborative inhibition despite the fact that
             participants clustered the emotional, but not neutral,
             information together in memory.},
   Doi = {10.3758/s13421-017-0694-3},
   Key = {fds327928}
}

@article{fds327929,
   Author = {Dang, LC and Castrellon, JJ and Perkins, SF and Le, NT and Cowan, RL and Zald, DH and Samanez-Larkin, GR},
   Title = {Reduced effects of age on dopamine D2 receptor levels in
             physically active adults.},
   Journal = {Neuroimage},
   Volume = {148},
   Pages = {123-129},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2017.01.018},
   Abstract = {Physical activity has been shown to ameliorate dopaminergic
             degeneration in non-human animal models. However, the
             effects of regular physical activity on normal age-related
             changes in dopamine function in humans are unknown. Here we
             present cross-sectional data from forty-four healthy human
             subjects between 23 and 80 years old, showing that typical
             age-related dopamine D2 receptor loss, assessed with PET
             [18F]fallypride, was significantly reduced in physically
             active adults compared to less active adults.},
   Doi = {10.1016/j.neuroimage.2017.01.018},
   Key = {fds327929}
}


%% Chartrand, Tanya L.   
@article{fds336936,
   Author = {Rim, S and Min, KE and Liu, PJ and Chartrand, TL and Trope,
             Y},
   Title = {The Gift of Psychological Closeness: How Feasible Versus
             Desirable Gifts Reduce Psychological Distance to the
             Giver.},
   Journal = {Personality & Social Psychology Bulletin},
   Pages = {146167218784899},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1177/0146167218784899},
   Abstract = {Gift-giving is a common form of social exchange but little
             research has examined how different gift types affect the
             psychological distance between giver and recipient. We
             examined how two types of gifts influence recipients'
             perceived psychological distance to the giver. Specifically,
             we compared desirable gifts focused on the quality of the
             gift with feasible gifts focused on the gift's practicality
             or ease of use. We found that feasible (vs. desirable) gifts
             led recipients to feel psychologically closer to givers
             (Studies 1-4). Further clarifying the process by which
             receiving a desirable versus feasible gift affects perceived
             distance, when recipients were told that the giver focused
             on the gift's practicality or ease of use (vs. the gift's
             overall quality), while holding the specific features of the
             gifts constant, they felt closer to the gift-giver (Study
             5). These results shed light on how different gifts can
             influence interpersonal relationships.},
   Doi = {10.1177/0146167218784899},
   Key = {fds336936}
}

@article{fds332782,
   Title = {Erratum},
   Journal = {Journal of Consumer Research},
   Volume = {44},
   Number = {5},
   Pages = {1174-1174},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/jcr/ucx095},
   Doi = {10.1093/jcr/ucx095},
   Key = {fds332782}
}

@article{fds336937,
   Author = {Duffy, KA and Helzer, EG and Hoyle, RH and Fukukura Helzer and J and Chartrand, TL},
   Title = {Pessimistic expectations and poorer experiences: The role of
             (low) extraversion in anticipated and experienced enjoyment
             of social interaction.},
   Journal = {Plos One},
   Volume = {13},
   Number = {7},
   Pages = {e0199146},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1371/journal.pone.0199146},
   Abstract = {Given research suggesting that social interactions are
             beneficial, it is unclear why individuals lower in
             extraversion engage less in social interactions. In this
             study, we test whether individuals lower in extraversion
             reap fewer hedonic rewards from social interactions and
             explore social psychological processes that explain their
             experiences. Before participants socialized, we measured
             extraversion, state positive affect, cognitive capacity, and
             expectations about the social interactions. After
             participants socialized with one another, we measured state
             positive affect and cognitive capacity again as well as fear
             of negative evaluation and belief in limited cognitive
             capacity. Participants also rated the social skillfulness of
             each interaction partner. We found that less extraverted
             individuals expect to feel worse after socializing. However,
             all but those extremely low in extraversion (17% of sample)
             actually experience an increase in positive affect after
             socializing. Surprisingly, those low in extraversion did not
             show reduced cognitive capacity after socializing. Although
             they are more likely to believe that cognitive capacity is
             limited and to be fearful of negative evaluation, these
             characteristics did not explain the social experience of
             those low in extraversion.},
   Doi = {10.1371/journal.pone.0199146},
   Key = {fds336937}
}

@article{fds327053,
   Author = {Leander, NP and Chartrand, TL},
   Title = {On thwarted goals and displaced aggression: A compensatory
             competence model},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {72},
   Pages = {88-100},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.04.010},
   Doi = {10.1016/j.jesp.2017.04.010},
   Key = {fds327053}
}

@article{fds332783,
   Author = {Brick, DJ and Fitzsimons, GM and Chartrand, TL and Fitzsimons,
             GJ},
   Title = {Coke vs. Pepsi: Brand Compatibility, Relationship Power, and
             Life Satisfaction},
   Journal = {Journal of Consumer Research},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1093/jcr/ucx079},
   Doi = {10.1093/jcr/ucx079},
   Key = {fds332783}
}

@article{fds327054,
   Author = {Leander, NP and Kay, AC and Chartrand, TL and Payne,
             BK},
   Title = {An Affect Misattribution Pathway to Perceptions of Intrinsic
             Reward},
   Journal = {Social Cognition},
   Volume = {35},
   Number = {2},
   Pages = {163-180},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1521/soco.2017.35.2.163},
   Abstract = {© 2017 Guilford Publications, Inc. Intrinsic rewards are
             typically thought to stem from an activity's inherent
             properties and not from separable rewards one receives from
             it. Yet, people may not consciously notice or remember all
             the subtle external rewards that correspond with an activity
             and may misattribute some directly to the activity itself.
             We propose that perceptions of intrinsic reward can often be
             byproducts of misattributed causal inference, and present
             some initial evidence that perceptions of intrinsic reward
             can in fact increase when words pertaining to an activity
             are subtly paired with pleasant context cues. Importantly,
             these effects follow classic boundary conditions of both
             misattribution and intrinsic motivation, insofar as they
             were extinguished when participants could make a proper
             source attribution and/or when the activity became
             associated with a blatant external reward. We further
             propose a distinction can be made between authentically
             "intrinsic" rewards and the illusion of intrinsic rewards
             caused by misattributed positive affect.},
   Doi = {10.1521/soco.2017.35.2.163},
   Key = {fds327054}
}

@article{fds323533,
   Author = {Duffy, KA and Harris, LT and Chartrand, TL and Stanton,
             SJ},
   Title = {Women recovering from social rejection: The effect of the
             person and the situation on a hormonal mechanism of
             affiliation.},
   Journal = {Psychoneuroendocrinology},
   Volume = {76},
   Pages = {174-182},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2016.11.017},
   Abstract = {Rejection can motivate either affiliation or withdrawal. In
             order to study how personality and situational variables
             influence whether women will be motivated to affiliate
             versus withdraw, we manipulate social feedback (rejection
             vs. acceptance) and opportunity for face-to-face interaction
             (blocked vs. face-to-face) and measure the individual
             difference variables rejection sensitivity and social
             anxiety. We test how these variables affect endogenous
             progesterone and cortisol concentrations, which are presumed
             to signal motivational responses to rejection. We find that
             three-way interactions involving social feedback,
             opportunity for face-to-face interactions, and either social
             anxiety or rejection sensitivity significantly predict
             progesterone change, but not cortisol change. Both
             interactions are driven by sharp progesterone decreases for
             women high in social anxiety/rejection sensitivity who have
             been rejected and who have no opportunity to reaffiliate in
             a face-to-face interaction. This progesterone change may be
             a physiological marker of motivation for social avoidance
             following rejection for women who cannot reaffiliate and who
             are particularly socially anxious or sensitive to
             rejection.},
   Doi = {10.1016/j.psyneuen.2016.11.017},
   Key = {fds323533}
}


%% Cherenack, Emily M.   
@article{fds331648,
   Author = {Sikkema, KJ and Mulawa, MI and Robertson, C and Watt, MH and Ciya, N and Stein, DJ and Cherenack, EM and Choi, KW and Kombora, M and Joska,
             JA},
   Title = {Improving AIDS Care After Trauma (ImpACT): Pilot Outcomes of
             a Coping intervention Among HIV-Infected Women with Sexual
             Trauma in South Africa.},
   Journal = {Aids and Behavior},
   Volume = {22},
   Number = {3},
   Pages = {1039-1052},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1007/s10461-017-2013-1},
   Abstract = {Improving AIDS Care after Trauma (ImpACT), a coping
             intervention for HIV-infected women with sexual abuse
             histories, was evaluated for feasibility and potential
             efficacy in a public clinic in Cape Town, South Africa.
             Sixty-four participants were enrolled prior to starting
             antiretroviral therapy (ART). After completing baseline
             assessments, participants were randomly assigned to standard
             of care (SoC: three adherence counseling sessions) or ImpACT
             (SoC plus four individual and three group sessions).
             Participants completed assessments at 3 months (after
             individual sessions) and 6 months post-baseline. In
             exploratory analysis of primary outcomes, ImpACT
             participants, compared to SoC, reported greater reductions
             in avoidance and arousal symptoms of PTSD and greater
             increases in ART adherence motivation at 3 months.
             Clinically significant decreases in overall PTSD symptoms
             were also demonstrated at 3 months. These effects continued
             as trends at the 6-month assessment, in addition to
             increases in social/spiritual coping. In analysis of
             secondary outcomes, high levels of non-adherence to ART and
             poor care engagement were evident at 6 months, with no
             differences between study arms. A trauma-focused,
             culturally-adapted individual intervention delivered by a
             non-specialist in the HIV care setting is feasible and
             acceptable. Preliminary findings suggest ImpACT has
             potential to reduce PTSD symptoms and increase ART adherence
             motivation, but a more intensive intervention may be needed
             to improve and maintain care engagement among this
             population.ClinicalTrials.gov NCT02223390.},
   Doi = {10.1007/s10461-017-2013-1},
   Key = {fds331648}
}

@article{fds335641,
   Author = {Carrico, AW and Cherenack, EM and Roach, ME and Riley, ED and Oni, O and Dilworth, SE and Shoptaw, S and Hunt, P and Roy, S and Pallikkuth, S and Pahwa, S},
   Title = {Substance-associated elevations in monocyte activation among
             methamphetamine users with treated HIV infection.},
   Journal = {Aids},
   Volume = {32},
   Number = {6},
   Pages = {767-771},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1097/qad.0000000000001751},
   Abstract = {Microbial translocation and monocyte activation predict
             mortality in treated HIV. We examined whether substance use
             independently contributes to these pathophysiologic
             processes.Cross-sectional study at baseline for a randomized
             controlled trial.HIV-positive, methamphetamine-using MSM
             with undetectable HIV viral load (less than 40 copies/ml)
             were enrolled. We examined if plasma biomarkers of monocyte
             activation and intestinal barrier integrity were associated
             with the following: reactive urine toxicology results (Tox+)
             for stimulants (i.e., methamphetamine or cocaine) and
             substance use severity measured by the Addiction Severity
             Index. Multiple linear regression models adjusted for age,
             antiretroviral therapy regimen, CD4 T-cell count,
             interleukin-6, and alcohol use severity.The sample of 84
             virally suppressed MSM had a median CD4 T-cell count of
             645 cells/μl. Those who were Tox+ for stimulants
             displayed higher soluble CD14 (sCD14) levels (2087 versus
             1801 ng/ml; P = 0.009), and this difference remained
             significant after adjusting for covariates (standardized
             beta = 0.23, P = 0.026). Greater substance use
             severity was also independently associated with higher sCD14
             after adjusting for covariates (standardized
             beta = 0.29, P = 0.013). Being Tox+ for stimulants
             and substance use severity were not associated with soluble
             CD163 (sCD163) or intestinal fatty acid binding protein
             (iFABP) levels (P > 0.05).Monocyte activation is one
             plausible mechanism by which stimulant use may increase
             clinical HIV progression.},
   Doi = {10.1097/qad.0000000000001751},
   Key = {fds335641}
}

@article{fds332042,
   Author = {Cherenack, EM and Sikkema, KJ and Watt, MH and Hansen, NB and Wilson,
             PA},
   Title = {Avoidant Coping Mediates the Relationship Between
             Self-Efficacy for HIV Disclosure and Depression Symptoms
             Among Men Who Have Sex with Men Newly Diagnosed with
             HIV.},
   Journal = {Aids and Behavior},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s10461-018-2036-2},
   Abstract = {HIV diagnosis presents a critical opportunity to reduce
             secondary transmission, improve engagement in care, and
             enhance overall well-being. To develop relevant
             interventions, research is needed on the psychosocial
             experiences of newly diagnosed individuals. This study
             examined avoidant coping, self-efficacy for HIV disclosure
             decisions, and depression among 92 newly diagnosed men who
             have sex with men who reported recent sexual risk behavior.
             It was hypothesized that avoidant coping would mediate the
             relationship between self-efficacy and depression.
             Cross-sectional surveys were collected from participants
             3 months after HIV diagnosis. To test for mediation,
             multiple linear regressions were conducted while controlling
             for HIV disclosure to sexual partners. Self-efficacy for HIV
             disclosure decisions showed a negative linear relationship
             to depression symptoms, and 99% of this relationship was
             mediated by avoidant coping. The index of mediation of
             self-efficacy on depression indicated a small-to-medium
             effect. Higher self-efficacy was related to less avoidant
             coping, and less avoidant coping was related to decreased
             depression symptoms, all else held constant. These findings
             highlight the role of avoidant coping in explaining the
             relationship between self-efficacy for HIV disclosure
             decisions and depression.},
   Doi = {10.1007/s10461-018-2036-2},
   Key = {fds332042}
}

@article{fds330403,
   Author = {Wilson, PA and Cherenack, EM and Jadwin-Cakmak, L and Harper, GW and Adolescent Medicine Trials Network for HIV/AIDS
             Interventions},
   Title = {Selection and Evaluation of Media for Behavioral Health
             Interventions Employing Critical Media Analysis.},
   Journal = {Health Promotion Practice},
   Volume = {19},
   Number = {1},
   Pages = {145-156},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1177/1524839917711384},
   Abstract = {Although a growing number of psychosocial health promotion
             interventions use the critical analysis of media to
             facilitate behavior change, no specific guidelines exist to
             assist researchers and practitioners in the selection and
             evaluation of culturally relevant media stimuli for
             intervention development. Mobilizing Our Voices for
             Empowerment is a critical consciousness-based health
             enhancement intervention for HIV-positive Black young
             gay/bisexual men that employs the critical analysis of
             popular media. In the process of developing and testing this
             intervention, feedback on media stimuli was collected from
             youth advisory board members (n = 8), focus group
             participants (n = 19), intervention participants (n = 40),
             and intervention facilitators (n = 6). A thematic analysis
             of qualitative data resulted in the identification of four
             key attributes of media stimuli and participants' responses
             to media stimuli that are important to consider when
             selecting and evaluating media stimuli for use in behavioral
             health interventions employing the critical analysis of
             media: comprehension, relevance, emotionality, and action.
             These four attributes are defined and presented as a
             framework for evaluating media, and adaptable tools are
             provided based on this framework to guide researchers and
             practitioners in the selection and evaluation of media for
             similar interventions.},
   Doi = {10.1177/1524839917711384},
   Key = {fds330403}
}


%% Cogan, Gregory B   
@article{fds334778,
   Author = {Cogan, GB and Iyer, A and Melloni, L and Thesen, T and Friedman, D and Doyle, W and Devinsky, O and Pesaran, B},
   Title = {Manipulating stored phonological input during verbal working
             memory.},
   Journal = {Nature Neuroscience},
   Volume = {20},
   Number = {2},
   Pages = {279-286},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1038/nn.4459},
   Abstract = {Verbal working memory (vWM) involves storing and
             manipulating information in phonological sensory input. An
             influential theory of vWM proposes that manipulation is
             carried out by a central executive while storage is
             performed by two interacting systems: a phonological input
             buffer that captures sound-based information and an
             articulatory rehearsal system that controls speech motor
             output. Whether, when and how neural activity in the brain
             encodes these components remains unknown. Here we read out
             the contents of vWM from neural activity in human subjects
             as they manipulated stored speech sounds. As predicted, we
             identified storage systems that contained both phonological
             sensory and articulatory motor representations.
             Unexpectedly, however, we found that manipulation did not
             involve a single central executive but rather involved two
             systems with distinct contributions to successful
             manipulation. We propose, therefore, that multiple
             subsystems comprise the central executive needed to
             manipulate stored phonological input for articulatory motor
             output in vWM.},
   Doi = {10.1038/nn.4459},
   Key = {fds334778}
}


%% Coie, John D.   
@article{fds334888,
   Author = {Zheng, Y and Albert, D and McMahon, RJ and Dodge, K and Dick, D and Conduct
             Problems Prevention Research Group},
   Title = {Glucocorticoid Receptor (NR3C1) Gene Polymorphism Moderate
             Intervention Effects on the Developmental Trajectory of
             African-American Adolescent Alcohol Abuse.},
   Journal = {Prevention Science : the Official Journal of the Society for
             Prevention Research},
   Volume = {19},
   Number = {1},
   Pages = {79-89},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s11121-016-0726-4},
   Abstract = {Accumulative evidence from recent genotype × intervention
             studies suggests that individuals carrying susceptible
             genotypes benefit more from intervention and provides one
             avenue to identify subgroups that respond differentially to
             intervention. This study examined the moderation by
             glucocorticoid receptor (NR3C1) gene variants of
             intervention effects on the developmental trajectories of
             alcohol abuse through adolescence. Participants were
             randomized into Fast Track intervention and control groups
             self-reported past-year alcohol abuse annually from grade 7
             through 2 years post-high school and provided genotype data
             at age 21 (69% males; European Americans [EAs] = 270,
             African-Americans [AAs] = 282). Latent growth curve
             models were fit to examine developmental trajectories of
             alcohol abuse. The interactions of 10 single nucleotide
             polymorphisms (SNPs) in NR3C1 with intervention were
             examined separately. Both EAs and AAs showed significant
             increases in past-year alcohol abuse with substantial
             inter-individual differences in rates of linear growth. AAs
             showed lower general levels and slower rates of linear
             growth than EAs. Adjusting for multiple tests, one NR3C1 SNP
             (rs12655166) significantly moderated intervention effects on
             the developmental trajectories of alcohol abuse among AAs.
             Intervention effects on the rates of linear growth were
             stronger among AAs carrying minor alleles than those not
             carrying minor alleles. The findings highlight the
             importance of taking a developmental perspective on
             adolescent alcohol use and have implications for future
             intervention design and evaluation by identifying subgroups
             that could disproportionally benefit from
             intervention.},
   Doi = {10.1007/s11121-016-0726-4},
   Key = {fds334888}
}


%% Coleman, Jessica N.   
@article{fds334111,
   Author = {O'Cleirigh, C and Perry, NS and Taylor, SW and Coleman, JN and Costa,
             PT and Mayer, KH and Safren, SA},
   Title = {Personality Traits and Adaptive HIV Disease Management:
             Relationships with Engagement in Care and Condomless Anal
             Intercourse Among Highly Sexually Active Sexual Minority Men
             Living with HIV.},
   Journal = {LGBT health},
   Volume = {5},
   Number = {4},
   Pages = {257-263},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1089/lgbt.2016.0065},
   Abstract = {The purpose of this study was to identify systematic
             relationships between personality domains and engagement in
             HIV care and secondary HIV prevention among sexual minority
             men living with HIV.This cross-sectional study examined the
             relationships between general personality traits of the
             Five-Factor Model of personality (e.g., Neuroticism and
             Conscientiousness) and engagement in medical care and
             condomless anal intercourse among a sample of highly
             sexually active sexual minority men living with HIV
             (N = 60).Conscientiousness (B = -0.01,
             P < 0.05), Openness (B = -0.03, P < 0.05), and
             Extraversion (B = -0.03, P < 0.001) were each
             associated with engaging in fewer episodes of condomless
             anal intercourse and Conscientiousness alone was
             significantly related to having fewer sexual partners
             (B = -0.04, P < 0.001). Conscientiousness (odds
             ratio [OR] = 1.07, confidence interval [CI]: 1.01-1.13)
             and Extraversion (OR = 1.13, CI: 1.04-1.22) were both
             associated significantly with prevention service use.
             Conscientiousness alone was related to engagement in HIV
             medical case management (B = -0.11, P < 0.05),
             whereas both Conscientiousness (B = 0.41,
             P < 0.0001) and Neuroticism (B = -0.64,
             P < 0.001) were associated with perceived health.
             Furthermore, compared with the normative sample for the
             NEO-Personality Inventory-Revised, men in our sample scored
             significantly higher on Neuroticism and significantly lower
             on Conscientiousness (Ps < 0.05).These findings suggest
             that enduring individual differences may account, in part,
             for some of the high levels of condomless anal intercourse
             reported by this group, as well as engagement in and use of
             prevention services. We suggest strategies for engaging this
             group in secondary HIV prevention programs and
             initiatives.},
   Doi = {10.1089/lgbt.2016.0065},
   Key = {fds334111}
}

@article{fds334112,
   Author = {Shaffer, KM and Jacobs, JM and Coleman, JN and Temel, JS and Rosand, J and Greer, JA and Vranceanu, A-M},
   Title = {Anxiety and Depressive Symptoms Among Two Seriously
             Medically Ill Populations and Their Family Caregivers: A
             Comparison and Clinical Implications},
   Journal = {Neurocritical Care},
   Volume = {27},
   Number = {2},
   Pages = {180-186},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1007/s12028-016-0358-3},
   Doi = {10.1007/s12028-016-0358-3},
   Key = {fds334112}
}

@article{fds334113,
   Author = {Ashaba, S and Kaida, A and Coleman, JN and Burns, BF and Dunkley, E and O’Neil, K and Kastner, J and Sanyu, N and Akatukwasa, C and Bangsberg,
             DR and Matthews, LT and Psaros, C},
   Title = {Psychosocial challenges facing women living with HIV during
             the perinatal period in rural Uganda},
   Journal = {Plos One},
   Volume = {12},
   Number = {5},
   Pages = {e0176256-e0176256},
   Editor = {Mazza, M},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1371/journal.pone.0176256},
   Doi = {10.1371/journal.pone.0176256},
   Key = {fds334113}
}


%% Compton, Scott N.   
@article{fds337698,
   Author = {Swan, AJ and Kendall, PC and Olino, T and Ginsburg, G and Keeton, C and Compton, S and Piacentini, J and Peris, T and Sakolsky, D and Birmaher,
             B and Albano, AM},
   Title = {Results from the Child/Adolescent Anxiety Multimodal
             longitudinal study (CAMELS): Functional outcomes},
   Journal = {Journal of Consulting and Clinical Psychology},
   Volume = {86},
   Number = {9},
   Pages = {738-750},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1037/ccp0000334},
   Abstract = {© 2018 American Psychological Association. Objective: To
             report functional outcomes from the multisite
             Child/Adolescent Anxiety Multimodal Extended Long-term Study
             (CAMELS), which examined the impact of youth anxiety
             treatment (cognitive-behavioral therapy [CBT], coping cat;
             Sertraline, SRT; COMB [CBT + SRT]; pill placebo) on (a)
             global and (b) domain-specific functioning assessed an
             average of 3.1 times, 3- to 12-years postrandomization
             (first assessment = mean 6.5 years postrandomization).
             Method: Three-hundred and 19 of 488 families from the
             Child/Adolescent Anxiety Multimodal Study (CAMS; Walkup et
             al., 2008) participated. Growth curve modeling examined the
             impact of treatment condition and acute treatment outcomes
             (i.e., response, remission) on global functioning, global
             and domain-specific impairment, and life satisfaction across
             follow-up visits. Logistic regressions explored the impact
             of treatment remission and condition on low frequency events
             (arrests/convictions) and education. Results: Treatment
             responders and remitters demonstrated better global
             functioning, decreased overall impairment, and increased
             life satisfaction at follow-up. Treatment remission, but not
             response, predicted decreased domain-specific impairment
             (social relationships, self-care/independence, academic
             functioning), and maintenance of increased life satisfaction
             across follow-ups. Participants in the CBT condition,
             compared with pill placebo, demonstrated improved
             trajectories pertaining to life satisfaction, overall
             impairment, and impairment in academic functioning.
             Randomization to CBT or COMB treatment was associated with
             increasing employment rates. Trajectories for participants
             randomized to SRT was not significantly different from
             placebo. Treatment outcome and condition did not predict
             legal outcomes, school/ work variables, or family life.
             Conclusion: Positive early intervention outcomes are
             associated with improved overall functioning, life
             satisfaction, and functioning within specific domains 6.5
             years posttreatment. Treatment type differentially predicted
             trajectories of functioning. Findings support the positive
             impact of pediatric anxiety treatment into adolescence and
             early adulthood.},
   Doi = {10.1037/ccp0000334},
   Key = {fds337698}
}

@article{fds337699,
   Author = {Villabø, MA and Narayanan, M and Compton, SN and Kendall, PC and Neumer, S-P},
   Title = {Cognitive-behavioral therapy for youth anxiety: An
             effectiveness evaluation in community practice.},
   Journal = {Journal of Consulting and Clinical Psychology},
   Volume = {86},
   Number = {9},
   Pages = {751-764},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1037/ccp0000326},
   Abstract = {To compare the effectiveness of individual
             cognitive-behavioral therapy (ICBT) and group CBT (GCBT) for
             referred children with anxiety disorders within community
             mental health clinics.Children (N = 165; ages 7-13 years)
             referred to 5 clinics in Norway because of primary
             separation anxiety disorder (SAD), social anxiety disorder
             (SOC), or generalized anxiety disorder (GAD) based on
             Diagnostic and Statistical Manual of Mental Disorders (4th
             ed., text rev.) criteria participated in a randomized
             clinical trial. Participants were randomized to ICBT, GCBT,
             or wait list (WL). WL participants were randomized to 1 of
             the 2 active treatment conditions following the wait period.
             Primary outcome was loss of principal anxiety disorder over
             12 weeks and 2-year follow-up.Both ICBT and GCBT were
             superior to WL on all outcomes. In the intent-to-treat
             analysis, 52% in ICBT, 65% in GCBT, and 14% in WL were
             treatment responders. Planned pairwise comparisons found no
             significant differences between ICBT and GCBT. GCBT was
             superior to ICBT for children diagnosed with SOC.
             Improvement continued during 2-year follow-up with no
             significant between-groups differences.Among anxiety
             disordered children, both individual and group CBT can be
             effectively delivered in community clinics. Response rates
             were similar to those reported in efficacy trials. Although
             GCBT was more effective than ICBT for children with SOC
             following treatment, both treatments were comparable at
             2-year follow-up. Dropout rates were lower in GCBT than in
             ICBT, suggesting that GCBT may be better tolerated. Response
             rates continued to improve over the follow-up period, with
             low rates of relapse. (PsycINFO Database
             Record},
   Doi = {10.1037/ccp0000326},
   Key = {fds337699}
}

@article{fds336055,
   Author = {Ginsburg, GS and Becker-Haimes, EM and Keeton, C and Kendall, PC and Iyengar, S and Sakolsky, D and Albano, AM and Peris, T and Compton, SN and Piacentini, J},
   Title = {Results From the Child/Adolescent Anxiety
             Multimodal Extended Long-Term Study (CAMELS): Primary
             Anxiety Outcomes.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {57},
   Number = {7},
   Pages = {471-480},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.jaac.2018.03.017},
   Abstract = {To report anxiety outcomes from the multisite
             Child/Adolescent Anxiety Multimodal Extended Long-term Study
             (CAMELS). Rates of stable anxiety remission (defined
             rigorously as the absence of all DSM-IV TR anxiety disorders
             across all follow-up years) and predictors of anxiety
             remission across a 4-year period, beginning 4 to 12 years
             after randomization to 12 weeks of medication,
             cognitive-behavioral therapy (CBT), their combination, or
             pill placebo were examined. Examined predictors of remission
             included acute treatment response, treatment assignment,
             baseline child and family variables, and interim negative
             life events.Data were from 319 youths (age range 10.9-25.2
             years; mean age 17.12 years) originally diagnosed with
             separation, social, and/or generalized anxiety disorders and
             enrolled in the multi-site Child/Adolescent Anxiety
             Multimodal Study (CAMS). Participants were assessed annually
             by independent evaluators using the age-appropriate version
             of the Anxiety Disorders Interview Schedule and completed
             questionnaires (eg, about family functioning, life events,
             and mental health service use).Almost 22% of youth were in
             stable remission, 30% were chronically ill, and 48% were
             relapsers. Acute treatment responders were less likely to be
             in the chronically ill group (odds ratio = 2.73; confidence
             interval = 1.14-6.54; p < .02); treatment type was not
             associated with remission status across the follow-up.
             Several variables (eg, male gender) predicted stable
             remission from anxiety disorders.Findings suggest that acute
             positive response to anxiety treatment may reduce risk for
             chronic anxiety disability; identified predictors can help
             tailor treatments to youth at greatest risk for chronic
             illness.Child and Adolescent Anxiety Disorders (CAMS).
             http://clinicaltrials.gov/; NCT00052078.},
   Doi = {10.1016/j.jaac.2018.03.017},
   Key = {fds336055}
}

@article{fds336056,
   Author = {Kiff, CJ and Ernestus, S and Gonzalez, A and Kendall, PC and Albano, AM and Compton, SN and Birmaher, B and Ginsburg, GS and Rynn, M and Walkup, JT and McCracken, J and Piacentini, J},
   Title = {The Interplay of Familial and Individual Risk in Predicting
             Clinical Improvements in Pediatric Anxiety
             Disorders.},
   Journal = {Journal of Clinical Child and Adolescent Psychology : the
             Official Journal for the Society of Clinical Child and
             Adolescent Psychology, American Psychological Association,
             Division 53},
   Pages = {1-13},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1080/15374416.2018.1460848},
   Abstract = {Bioecological models of developmental psychopathology
             underscore the role of familial experiences of adversity and
             children's individual-level characteristics in heightening
             risk for pediatric anxiety through direct, combined, and
             interactive effects. To date, much of the existing research
             dedicated to pediatric anxiety disorders has largely been
             examined in bioecological models of diathesis-stress using
             community samples. This study extends our understanding of
             children's differential responsiveness to familial adversity
             by examining the diathesis-stress interaction of cumulative
             risk and children's individual-level vulnerabilities
             (negative affectivity and coping efficacy) within a
             clinic-referred treatment study for pediatric anxiety
             disorders. A cumulative risk index assessing exposure to
             familial adversity (e.g., socioeconomic status [SES], parent
             psychiatric illness) and self-reported measures of
             children's negative affectivity and coping efficacy were
             each measured at the intake of a randomized controlled
             clinical trial for the treatment of pediatric anxiety
             disorders (N = 488; 7-17 years of age). Trajectories of
             interviewer-rated anxiety symptoms were assessed across 12
             weeks of treatment at baseline, 4 weeks, 8 weeks, and
             12 weeks. Consistent with models of temperamental risk for
             mental health problems, negative affectivity predicted
             higher anxiety symptoms at intake. A significant
             diathesis-stress interaction between cumulative risk and
             coping efficacy emerged, as high risk and perceptions of
             lower coping efficacy attenuated declines in anxiety across
             12 weeks. These patterns did not differ across treatment
             conditions. The results indicate that for youth experiencing
             high levels of stress, additional treatment efforts
             targeting familial stressors and coping efficacy may be
             important in maximizing treatment outcomes.},
   Doi = {10.1080/15374416.2018.1460848},
   Key = {fds336056}
}

@article{fds336057,
   Author = {Bushnell, GA and Brookhart, MA and Gaynes, BN and Compton, SN and Dusetzina, SB and Stürmer, T},
   Title = {Examining Parental Medication Adherence as a Predictor of
             Child Medication Adherence in Pediatric Anxiety
             Disorders.},
   Journal = {Medical Care},
   Volume = {56},
   Number = {6},
   Pages = {510-519},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1097/mlr.0000000000000911},
   Abstract = {Selective serotonin reuptake inhibitors (SSRIs) are the
             recommended first-line pharmacotherapy for pediatric anxiety
             disorders but adherence remains difficult to predict.To
             estimate SSRI adherence in children with anxiety disorders
             and determine if prior parental medication adherence is
             predictive of child high SSRI adherence.We identified
             children (3-17 y) initiating SSRI treatment after an
             anxiety disorder diagnosis in a commercial claims database
             (2005-2014). We evaluated parent SSRI, statin, and
             antihypertensive adherence [6-mo proportion days covered
             (PDC), high adherence=PDC≥0.80] in the year before child
             SSRI initiation. We estimated risk differences (RD) of child
             high SSRI adherence (6-mo PDC) stratified by parent
             adherence and multivariable risk ratios using modified
             Poisson regression. We estimated change in c-statistic and
             risk reclassification when adding parent-level covariates
             with child-level covariates to predict child adherence.In
             70,979 children with an anxiety disorder (59%=female,
             14=median age), the mean 6-month SSRI PDC was 0.72, with
             variation by anxiety disorder. Overall 64% of children had
             high adherence if their parent had high SSRI adherence
             versus 53% of children with parents with low SSRI adherence
             (RD, 12%; multivariable risk ratios, 1.17; 95% confidence
             interval, 1.14-1.20). Findings were similar for parent
             statin (RD=10%) and antihypertensive adherence (RD=8%) and
             when stratified by child age and parent sex. There was minor
             improvement in risk reclassification and the c-statistic
             after adding parent adherence and parent-level
             covariates.Parental medication adherence could help
             providers identify children at risk of nonadherence to
             inform the treatment decision, reduce unnecessary medication
             switches, and lead to broader effective interventions.},
   Doi = {10.1097/mlr.0000000000000911},
   Key = {fds336057}
}

@article{fds336058,
   Author = {Palitz, SA and Caporino, NE and McGuire, JF and Piacentini, J and Albano, AM and Birmaher, B and Walkup, JT and Compton, SN and Ginsburg,
             GS and Kendall, PC},
   Title = {Defining Treatment Response and Remission in Youth Anxiety:
             A Signal Detection Analysis With the Multidimensional
             Anxiety Scale for Children.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {57},
   Number = {6},
   Pages = {418-427},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.jaac.2018.03.013},
   Abstract = {To determine the percent reduction cutoffs on the
             Multidimensional Anxiety Scale for Children (MASC) that
             optimally predict treatment response and remission in youth
             with anxiety disorders.Youths and their parents completed
             the MASC-C/P before and after treatment, and the Anxiety
             Disorders Interview Schedule for DSM-IV-Child and Parent
             Versions (ADIS-IV-C/P) and the Clinical Global
             Impression-Improvement Scale (CGI-I) were administered by
             independent evaluators. Treatment response and remission
             were defined by post-treatment ratings on the CGI-I and the
             ADIS-IV-C/P, respectively. Quality receiver operating
             characteristic methods determined the optimal cutoff on the
             MASC-P for predicting overall remission (loss of all study
             entry diagnoses) and optimal percent reductions on the
             MASC-P for predicting treatment response and remission of
             separation anxiety, social anxiety, and generalized
             anxiety.A post-treatment raw score of 42 optimally predicted
             remission. A reduction of 35% on the total MASC-P predicted
             treatment response. A reduction of 30% on the Separation
             Anxiety/Panic subscale of the MASC-P predicted separation
             anxiety remission. A reduction of 35% on the Social Anxiety
             subscale of the MASC-P predicted social anxiety remission.
             The MASC did not evidence a cutoff for remission of
             generalized anxiety disorder.MASC cutoffs can facilitate
             comparison across studies and guide practice, aiding
             clinicians in assessing progress and informing treatment
             plans.},
   Doi = {10.1016/j.jaac.2018.03.013},
   Key = {fds336058}
}

@article{fds328085,
   Author = {Selles, RR and Franklin, M and Sapyta, J and Compton, SN and Tommet, D and Jones, RN and Garcia, A and Freeman, J},
   Title = {Children's and Parents' Ability to Tolerate Child Distress:
             Impact on Cognitive Behavioral Therapy for Pediatric
             Obsessive Compulsive Disorder.},
   Journal = {Child Psychiatry and Human Development},
   Volume = {49},
   Number = {2},
   Pages = {308-316},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s10578-017-0748-6},
   Abstract = {The present study explored the concept of tolerance for
             child distress in 46 children (ages 5-8), along with their
             mothers and fathers, who received family-based CBT for OCD.
             The study sought to describe baseline tolerance, changes in
             tolerance with treatment, and the predictive impact of
             tolerance on symptom improvement. Tolerance was rated by
             clinicians on a single item and the CY-BOCS was used to
             measure OCD severity. Descriptive results suggested that all
             participants had some difficulty tolerating the child's
             distress at baseline while paired t tests indicated large
             improvements were made over treatment (d = 1.2-2.0).
             Fathers' initial tolerance was significantly related to
             symptom improvement in a multivariate regression as were
             fathers' and children's changes in distress tolerance over
             the course of treatment. Overall, results provide support
             for examining tolerance of child distress including its
             predictive impact and potential as a supplemental
             intervention target.},
   Doi = {10.1007/s10578-017-0748-6},
   Key = {fds328085}
}

@article{fds336059,
   Author = {Bushnell, GA and Compton, SN and Dusetzina, SB and Gaynes, BN and Brookhart, MA and Walkup, JT and Rynn, MA and Stürmer,
             T},
   Title = {Treating Pediatric Anxiety: Initial Use of SSRIs and Other
             Antianxiety Prescription Medications.},
   Journal = {The Journal of Clinical Psychiatry},
   Volume = {79},
   Number = {1},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.4088/jcp.16m11415},
   Abstract = {Multiple pharmacotherapies for treating anxiety disorders
             exist, including selective serotonin reuptake inhibitors
             (SSRIs), the recommended first-line pharmacotherapy for
             pediatric anxiety. We sought to describe initial antianxiety
             medication use in children and estimate how long antianxiety
             medications were continued.In a large commercial claims
             database, we identified children (3-17 years) initiating
             prescription antianxiety medication from 2004 to 2014 with a
             recent anxiety diagnosis (ICD-9-CM = 293.84, 300.0x, 300.2x,
             300.3x, 309.21, 309.81, 313.23). We estimated the proportion
             of children initiating each medication class across the
             study period and used multivariable regression to evaluate
             factors associated with initiation with an SSRI. We
             evaluated treatment length for each initial medication
             class.Of 84,500 children initiating antianxiety medication,
             70% initiated with an SSRI (63% [95% CI, 62%-63%] SSRI
             alone, 7% [95% CI, 7%-7%] SSRI + another antianxiety
             medication). Non-SSRI medications initiated included
             benzodiazepines (8%), non-SSRI antidepressants (7%),
             hydroxyzine (4%), and atypical antipsychotics (3%). Anxiety
             disorder, age, provider type, and comorbid diagnoses were
             associated with initial medication class. The proportion of
             children refilling their initial medication ranged from 19%
             (95% CI, 18%-20%) of hydroxyzine initiators and 25% (95% CI,
             24%-26%) of benzodiazepine initiators to 81% (95% CI,
             80%-81%) of SSRI initiators. Over half (55%, 95% CI,
             55%-56%) of SSRI initiators continued SSRI treatment for 6
             months.SSRIs are the most commonly used first-line
             medication for pediatric anxiety disorders, with about half
             of SSRI initiators continuing treatment for 6 months. Still,
             a third began therapy on a non-SSRI medication, for which
             there is limited evidence of effectiveness for pediatric
             anxiety, and a notable proportion of children initiated with
             2 antianxiety medication classes.},
   Doi = {10.4088/jcp.16m11415},
   Key = {fds336059}
}

@article{fds327202,
   Author = {Hale, AE and Ginsburg, GS and Chan, G and Kendall, PC and McCracken, JT and Sakolsky, D and Birmaher, B and Compton, SN and Albano, AM and Walkup,
             JT},
   Title = {Mediators of Treatment Outcomes for Anxious Children and
             Adolescents: The Role of Somatic Symptoms.},
   Journal = {Journal of Clinical Child and Adolescent Psychology : the
             Official Journal for the Society of Clinical Child and
             Adolescent Psychology, American Psychological Association,
             Division 53},
   Volume = {47},
   Number = {1},
   Pages = {94-104},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1080/15374416.2017.1280804},
   Abstract = {Cognitive behavioral therapy (CBT) and selective serotonin
             reuptake inhibitors are effective treatments for pediatric
             anxiety disorders. However, the mechanisms of these
             treatments are unknown. Previous research indicated that
             somatic symptoms are reduced following treatment, but it is
             unclear if their reductions are merely a consequence of
             treatment gains. This study examined reductions in somatic
             symptoms as a potential mediator of the relationship between
             treatment and anxiety outcomes. Participants were 488
             anxious youth ages 7-17 (M = 10.7), 50.4% male, 78.9%
             Caucasian, enrolled in Child/Adolescent Anxiety Multimodal
             Study, a large randomized control trial comparing 12-week
             treatments of CBT, sertraline, a combination of CBT and
             sertraline, and a pill placebo. Causal mediation models were
             tested in R using data from baseline, 8-, and 12-week
             evaluations. Somatic symptoms were assessed using the
             Panic/Somatic subscale from the Screen for Child Anxiety
             Related Emotional Disorders. Youth outcomes were assessed
             using the Pediatric Anxiety Rating Scale and Children's
             Global Assessment Scale. Reductions in somatic symptoms
             mediated improvement in anxiety symptoms and global
             functioning for those in the sertraline-only condition based
             on parent report. Conditions involving CBT and data based on
             child reported somatic symptoms did not show a mediation
             effect. Findings indicate that reductions in somatic
             symptoms may be a mediator of improvements for treatments
             including pharmacotherapy and not CBT. Although the overall
             efficacy of sertraline and CBT for anxiety may be similar,
             the treatments appear to function via different
             mechanisms.},
   Doi = {10.1080/15374416.2017.1280804},
   Key = {fds327202}
}

@article{fds330474,
   Author = {Peris, TS and Caporino, NE and O'Rourke, S and Kendall, PC and Walkup,
             JT and Albano, AM and Bergman, RL and McCracken, JT and Birmaher, B and Ginsburg, GS and Sakolsky, D and Piacentini, J and Compton,
             SN},
   Title = {Therapist-Reported Features of Exposure Tasks That Predict
             Differential Treatment Outcomes for Youth With
             Anxiety.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {12},
   Pages = {1043-1052},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.10.001},
   Abstract = {Exposure tasks are recognized widely as a key component of
             cognitive-behavioral therapy (CBT) for child and adolescent
             anxiety. However, little research has examined specific
             exposure characteristics that predict outcomes for youth
             with anxiety and that may guide its application in
             therapy.This study draws on a sample of 279 children and
             adolescents (48.4% male; 79.6% white) with a principal
             anxiety disorder who received 14 sessions of CBT, either
             alone or in combination with medication, through the
             Child/adolescent Anxiety Multimodal treatment Study (CAMS).
             The present study examines therapist-reported quantity,
             difficulty level, compliance, and mastery of exposure tasks
             as they related to CBT response (i.e., Clinical Global
             Impressions-Improvement ratings). Secondary treatment
             outcomes included reduction in anxiety symptom severity on
             the Pediatric Anxiety Rating Scale, global impairment
             measured via the Children's Global Assessment Scale, and
             parent-report of anxiety-specific functional impairment on
             the Child Anxiety Impairment Scale.Regression analyses
             indicated a dose-response relationship between
             therapist-reported quantity of exposure and independent
             evaluations of treatment outcome, with more time devoted to
             exposure linked to better outcomes. Similarly, greater time
             spent on more difficult (rather than mild or moderate)
             exposure tasks predicted better outcomes, as did therapist
             ratings of child compliance and mastery.The present findings
             highlight the importance of challenging children and
             adolescents with difficult exposure tasks and of
             collaborating to ensure compliance and mastery.},
   Doi = {10.1016/j.jaac.2017.10.001},
   Key = {fds330474}
}

@article{fds330044,
   Author = {Højgaard, DRMA and Hybel, KA and Ivarsson, T and Skarphedinsson, G and Becker Nissen and J and Weidle, B and Melin, K and Torp, NC and Valderhaug,
             R and Dahl, K and Mortensen, EL and Compton, S and Jensen, S and Lenhard,
             F and Thomsen, PH},
   Title = {One-Year Outcome for Responders of Cognitive-Behavioral
             Therapy for Pediatric Obsessive-Compulsive
             Disorder.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {940-947.e1},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.09.002},
   Abstract = {This study describes 1-year treatment outcomes from a large
             sample of cognitive-behavioral therapy (CBT) responders,
             investigates age as a possible moderator of these treatment
             outcomes, and evaluates clinical relapse at the 1-year
             follow-up.This study is the planned follow-up to the Nordic
             Long-term OCD [obsessive-compulsive disorder] Treatment
             Study (NordLOTS), which included 177 children and
             adolescents who were rated as treatment responders following
             CBT for OCD. Participants were assessed with the Children's
             Yale-Brown Obsessive-Compulsive Scale (CY-BOCS) at 6- and
             12-month follow-up. Treatment response and remission were
             defined as CY-BOCS total scores ≤15 and ≤10,
             respectively. Linear mixed-effects models were used to
             analyze all outcomes.At 1 year, a total of 155 children and
             adolescents (87.6%) were available for follow-up assessment,
             with 142 of these (91.6%) rated below a total score
             of ≤15 on the CY-BOCS. At 1-year follow-up, 121 (78.1%)
             were in remission. On average, CY-BOCS total scores dropped
             by 1.72 points during the first year after terminating
             treatment (p = .001). A total of 28 participants (15.8%)
             relapsed (CY-BOCS ≥ 16) at either the 6- or 12-month
             assessment; only 2 patients required additional CBT.Results
             suggest that manualized CBT in a community setting for
             pediatric OCD has durable effects for those who respond to
             an initial course of treatment; children and adolescents who
             respond to such treatment can be expected to maintain their
             treatment gains for at least 1 year following acute care.
             Clinical trial registration information- Nordic Long-term
             Obsessive-Compulsive Disorder (OCD) Treatment Study;
             www.controlled-trials.com; ISRCTN66385119.},
   Doi = {10.1016/j.jaac.2017.09.002},
   Key = {fds330044}
}

@article{fds336060,
   Author = {Pistorello, J and Jobes, DA and Compton, SN and Locey, NS and Walloch,
             JC and Gallop, R and Au, JS and Noose, SK and Young, M and Johnson, J and Dickens, Y and Chatham, P and Jeffcoat, T and Dalto, G and Goswami,
             S},
   Title = {Developing Adaptive Treatment Strategies to Address Suicidal
             Risk in College Students: A Pilot Sequential, Multiple
             Assignment, Randomized Trial (SMART).},
   Journal = {Archives of Suicide Research : Official Journal of the
             International Academy for Suicide Research},
   Volume = {22},
   Number = {4},
   Pages = {644-664},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1080/13811118.2017.1392915},
   Abstract = {This pilot study investigated the potential to utilize
             adaptive treatment strategies for treating moderate to
             severe suicidal risk among college students. This article
             will describe the unique study design and report on
             feasibility and acceptability findings. A 2-stage Sequential
             Multiple Assignment Randomized Trial (SMART) was conducted:
             In Stage 1, 62 suicidal college students were randomized to
             either a suicide-focused or a treatment-as-usual condition
             (4-8 weeks). Those deemed insufficient responders were
             re-randomized to one of two Stage 2 interventions-both
             suicide-focused but one comprehensive and multimodal and the
             other flexible and theoretically agnostic (4-16 additional
             weeks). Recruitment rates were high, treatment dropout
             levels were lower than expected for the setting, study
             dropouts were rare, and counselors were able to deliver
             suicide-focused approaches with fidelity. Treatment
             satisfaction was high among clients and moderately high
             among counselors. Findings from this pilot show that a SMART
             is highly feasible and acceptable to suicidal college
             students, counselors, and campuses.},
   Doi = {10.1080/13811118.2017.1392915},
   Key = {fds336060}
}

@article{fds323540,
   Author = {Caporino, NE and Read, KL and Shiffrin, N and Settipani, C and Kendall,
             PC and Compton, SN and Sherrill, J and Piacentini, J and Walkup, J and Ginsburg, G and Keeton, C and Birmaher, B and Sakolsky, D and Gosch, E and Albano, AM},
   Title = {Sleep-Related Problems and the Effects of Anxiety Treatment
             in Children and Adolescents.},
   Journal = {Journal of Clinical Child and Adolescent Psychology : the
             Official Journal for the Society of Clinical Child and
             Adolescent Psychology, American Psychological Association,
             Division 53},
   Volume = {46},
   Number = {5},
   Pages = {675-685},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1080/15374416.2015.1063429},
   Abstract = {This study examined (a) demographic and clinical
             characteristics associated with sleep-related problems
             (SRPs) among youth with anxiety disorders, and (b) the
             impact of anxiety treatment: cognitive-behavioral therapy
             (CBT; Coping Cat), medication (sertraline), their
             combination, and pill placebo on SRPs. Youth (N = 488,
             ages 7-17, 50% female, 79% White) with a principal diagnosis
             of generalized anxiety disorder, separation anxiety
             disorder, or social phobia participated. SRPs were reported
             by parents and youth. Findings differed by informant and by
             type of SRP, with evidence that SRPs are associated with
             age, anxiety severity, externalizing problems, functional
             impairment, and family burden at pretreatment. Anxiety
             treatment reduced SRPs; effect sizes were small to medium.
             Reductions in parent-reported separation-related sleep
             difficulties were significantly greater in active treatment
             than in the placebo condition, with the greatest reductions
             reported by parents of youth whose active treatment was
             multimodal or included sertraline. Youth whose anxiety
             treatment involved CBT reported significantly greater
             decreases in dysregulated sleep (e.g., sleeplessness). Both
             CBT for anxiety and sertraline appear to be somewhat
             effective in reducing SRPs, and multimodal treatment may be
             preferable depending on the symptom presentation. To inform
             practice, future research should examine a broad range of
             SRPs, incorporate objective measures of sleep, and evaluate
             the impact of behavioral strategies that directly target
             SRPs in youth with anxiety disorders.},
   Doi = {10.1080/15374416.2015.1063429},
   Key = {fds323540}
}

@article{fds327204,
   Author = {Hoff, AL and Kendall, PC and Langley, A and Ginsburg, G and Keeton, C and Compton, S and Sherrill, J and Walkup, J and Birmaher, B and Albano, AM and Suveg, C and Piacentini, J},
   Title = {Developmental Differences in Functioning in Youth With
             Social Phobia.},
   Journal = {Journal of Clinical Child and Adolescent Psychology : the
             Official Journal for the Society of Clinical Child and
             Adolescent Psychology, American Psychological Association,
             Division 53},
   Volume = {46},
   Number = {5},
   Pages = {686-694},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1080/15374416.2015.1079779},
   Abstract = {Social phobia (SoP) in youth may manifest differently across
             development as parent involvement in their social lives
             changes and social and academic expectations increase. This
             cross-sectional study investigated whether self-reported and
             parent-reported functioning in youth with SoP changes with
             age in social, academic, and home/family domains. Baseline
             anxiety impairment data from 488 treatment-seeking
             anxiety-disordered youth (ages 7-17, N = 400 with a SoP
             diagnosis) and their parents were gathered using the Child
             Anxiety Impact Scale and were analyzed using generalized
             estimating equations. According to youth with SoP and their
             parents, overall difficulties, social difficulties, and
             academic difficulties increased with age, even when
             controlling for SoP severity. These effects significantly
             differed for youth with anxiety disorders other than SoP.
             Adolescents may avoid social situations as parental
             involvement in their social lives decreases, and their
             withdrawn behavior may result in increasing difficulty in
             the social domain. Their avoidance of class participation
             and oral presentations may increasingly impact their
             academic performance as school becomes more demanding.
             Implications are discussed for the early detection and
             intervention of SoP to prevent increased impairment over the
             course of development.},
   Doi = {10.1080/15374416.2015.1079779},
   Key = {fds327204}
}

@article{fds327200,
   Author = {Strawn, JR and Dobson, ET and Mills, JA and Cornwall, GJ and Sakolsky,
             D and Birmaher, B and Compton, SN and Piacentini, J and McCracken, JT and Ginsburg, GS and Kendall, PC and Walkup, JT and Albano, AM and Rynn,
             MA},
   Title = {Placebo Response in Pediatric Anxiety Disorders: Results
             from the Child/Adolescent Anxiety Multimodal
             Study.},
   Journal = {Journal of Child and Adolescent Psychopharmacology},
   Volume = {27},
   Number = {6},
   Pages = {501-508},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1089/cap.2016.0198},
   Abstract = {The aim of this study is to identify predictors of pill
             placebo response and to characterize the temporal course of
             pill placebo response in anxious youth.Data from
             placebo-treated patients (N = 76) in the
             Child/Adolescent Anxiety Multimodal Study (CAMS), a
             multisite, randomized controlled trial that examined the
             efficacy of cognitive-behavioral therapy, sertraline, their
             combination, and placebo for the treatment of separation,
             generalized, and social anxiety disorders, were evaluated.
             Multiple linear regression models identified features
             associated with placebo response and models were confirmed
             with leave-one-out cross-validation. The likelihood of
             improvement in patients receiving pill placebo-over
             time-relative to improvement associated with active
             treatment was determined using probabilistic Bayesian
             analyses.Based on a categorical definition of response
             (Clinical Global Impressions-Improvement Scale score ≤2),
             nonresponders (n = 48), and pill placebo responders
             (n = 18) did not differ in age (p = 0.217), sex
             (p = 0.980), race (p = 0.743), or primary diagnosis
             (all ps > 0.659). In terms of change in anxiety symptoms,
             separation anxiety disorder and treatment expectation were
             associated with the degree of pill placebo response. Greater
             probability of placebo-related anxiety symptom improvement
             was observed early in the course of treatment (baseline to
             week 4, p < 0.0001). No significant change in the
             probability of placebo-related improvement was observed
             after week 4 (weeks 4-8, p = 0.07; weeks 8-12,
             p = 0.85), whereas the probability of improvement, in
             general, significantly increased week over week with active
             treatment.Pill placebo-related improvement occurs early in
             the course of treatment and both clinical factors and
             expectation predict this improvement. Additionally,
             probabilistic approaches may refine our understanding and
             prediction of pill placebo response.},
   Doi = {10.1089/cap.2016.0198},
   Key = {fds327200}
}

@article{fds326112,
   Author = {Gonzalez, A and Rozenman, M and Langley, AK and Kendall, PC and Ginsburg, GS and Compton, S and Walkup, JT and Birmaher, B and Albano,
             AM and Piacentini, J},
   Title = {Social Interpretation Bias in Children and Adolescents with
             Anxiety Disorders: Psychometric Examination of the
             Self-report of Ambiguous Social Situations for Youth (SASSY)
             Scale.},
   Journal = {Child & Youth Care Forum},
   Volume = {46},
   Number = {3},
   Pages = {395-412},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s10566-016-9381-y},
   Abstract = {Anxiety disorders are among the most common mental health
             problems in youth, and faulty interpretation bias has been
             positively linked to anxiety severity, even within
             anxiety-disordered youth. Quick, reliable assessment of
             interpretation bias may be useful in identifying youth with
             certain types of anxiety or assessing changes on cognitive
             bias during intervention.This study examined the factor
             structure, reliability, and validity of the Self-report of
             Ambiguous Social Situations for Youth (SASSY) scale, a
             self-report measure developed to assess interpretation bias
             in youth.Participants (N=488, age 7 to 17) met diagnostic
             criteria for Social Phobia, Generalized Anxiety Disorder,
             and/or Separation Anxiety Disorder. An exploratory factor
             analysis was performed on baseline data from youth
             participating in a large randomized clinical
             trial.Exploratory factor analysis yielded two factors
             (Accusation/Blame, Social Rejection). The SASSY full scale
             and Social Rejection factor demonstrated adequate internal
             consistency, convergent validity with social anxiety, and
             discriminant validity as evidenced by non-significant
             correlations with measures of non-social anxiety. Further,
             the SASSY Social Rejection factor accurately distinguished
             children and adolescents with Social Phobia from those with
             other anxiety disorders, supporting its criterion validity,
             and revealed sensitivity to changes with treatment. Given
             the relevance to youth with social phobia, pre- and
             post-intervention data were examined for youth social phobia
             to test sensitivity to treatment effects; results suggested
             that SASSY scores reduced for treatment responders.Findings
             suggest the potential utility of the SASSY Social Rejection
             factor as a quick, reliable, and efficient way of assessing
             interpretation bias in anxious youth, particularly as
             related to social concerns, in research and clinical
             settings.},
   Doi = {10.1007/s10566-016-9381-y},
   Key = {fds326112}
}

@article{fds326330,
   Author = {Lee, P and Zehgeer, A and Ginsburg, GS and McCracken, J and Keeton, C and Kendall, PC and Birmaher, B and Sakolsky, D and Walkup, J and Peris, T and Albano, AM and Compton, S},
   Title = {Child and Adolescent Adherence With Cognitive Behavioral
             Therapy for Anxiety: Predictors and Associations With
             Outcomes.},
   Journal = {Journal of Clinical Child and Adolescent Psychology : the
             Official Journal for the Society of Clinical Child and
             Adolescent Psychology, American Psychological Association,
             Division 53},
   Pages = {1-12},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1080/15374416.2017.1310046},
   Abstract = {Cognitive behavioral therapy (CBT) for anxiety disorders is
             effective, but nonadherence with treatment may reduce the
             benefits of CBT. This study examined (a) four baseline
             domains (i.e., demographic, youth clinical characteristics,
             therapy related, family/parent factors) as predictors of
             youth adherence with treatment and (b) the associations
             between youth adherence and treatment outcomes. Data were
             from 279 youth (7-17 years of age, 51.6% female; 79.6%
             White, 9% African American), with Diagnostic and Statistical
             Manual of Mental Disorders (4th ed., text rev.) diagnoses of
             separation anxiety disorder, generalized anxiety disorder,
             and/or social phobia, who participated in CBT in the
             Child/Adolescent Anxiety Multimodal Study. Adherence was
             defined in three ways (session attendance, therapist-rated
             compliance, and homework completion). Multiple regressions
             revealed several significant predictors of youth adherence
             with CBT, but predictors varied according to the definition
             of adherence. The most robust predictors of greater
             adherence were living with both parents and fewer youth
             comorbid externalizing disorders. With respect to outcomes,
             therapist ratings of higher youth compliance with CBT
             predicted several indices of favorable outcome: lower
             anxiety severity, higher global functioning, and treatment
             responder status after 12 weeks of CBT. Number of sessions
             attended and homework completion did not predict treatment
             outcomes. Findings provide information about risks for youth
             nonadherence, which can inform treatment and highlight the
             importance of youth compliance with participating in therapy
             activities, rather than just attending sessions or
             completing homework assignments.},
   Doi = {10.1080/15374416.2017.1310046},
   Key = {fds326330}
}

@article{fds327201,
   Author = {Alexander, JR and Houghton, DC and Twohig, MP and Franklin, ME and Saunders, SM and Neal-Barnett, AM and Compton, SN and Woods,
             DW},
   Title = {Clarifying the Relationship between Trichotillomania and
             Anxiety.},
   Journal = {Journal of Obsessive Compulsive and Related
             Disorders},
   Volume = {13},
   Pages = {30-34},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jocrd.2017.02.004},
   Abstract = {Although research has consistently linked unidimensional
             anxiety with Trichotillomania (TTM) severity, the
             relationships between TTM severity and anxiety dimensions
             (i.e., cognitive and somatic anxiety) are unknown. This
             knowledge gap limits current TTM conceptualization and
             treatment. The current study examined these relationships
             with data collected from ninety-one adults who participated
             in a randomized clinical trial for TTM treatment. To examine
             whether the Beck Anxiety Inventory (BAI; Beck, Epstein,
             Brown, & Steer, 1988) could be used to measure
             multidimensional anxiety in TTM samples, we conducted a
             factor analysis. Results showed four emergent factors,
             including a cognitive factor and three somatic factors
             (neurophysiological, autonomic, and panic). Based on prior
             research, it was hypothesized that TTM severity would be
             related to the cognitive anxiety dimension and that
             psychological inflexibility would mediate the association.
             Hypotheses were not made regarding the relationship between
             TTM severity and somatic anxiety. Regression analyses
             indicated that only cognitive dimensions of anxiety
             predicted TTM severity and that psychological inflexibility
             mediated this relationship. Implications for the
             conceptualization and treatment of TTM are
             discussed.},
   Doi = {10.1016/j.jocrd.2017.02.004},
   Key = {fds327201}
}

@article{fds327203,
   Author = {Strawn, JR and Compton, SN and Robertson, B and Albano, AM and Hamdani,
             M and Rynn, MA},
   Title = {Extended Release Guanfacine in Pediatric Anxiety Disorders:
             A Pilot, Randomized, Placebo-Controlled Trial.},
   Journal = {Journal of Child and Adolescent Psychopharmacology},
   Volume = {27},
   Number = {1},
   Pages = {29-37},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1089/cap.2016.0132},
   Abstract = {This is a feasibility study evaluating the safety,
             tolerability, and potential anxiolytic efficacy of the α2
             agonist guanfacine extended-release (GXR) in children and
             adolescents with generalized anxiety disorder (GAD),
             separation anxiety disorder (SAD), or social phobia/social
             anxiety disorder.Youth aged 6-17 years with a primary
             diagnosis of GAD, SAD, and/or social anxiety disorder were
             treated with flexibly dosed GXR (1-6 mg daily, n = 62)
             or placebo (n = 21) for 12 weeks. The primary aim of
             this study was to determine the safety and tolerability of
             GXR in youth with anxiety disorders, which involved the
             analysis of treatment-emergent adverse events (TEAEs), the
             emergence of suicidal ideation and behaviors, vital signs,
             and electrocardiographic/laboratory parameters. Exploratory
             efficacy measures included dimensional anxiety scales
             (Pediatric Anxiety Rating Scale [PARS] and Screen for Child
             Anxiety Related Emotional Disorders [SCARED]), as well as
             the Clinical Global Impression-Improvement (CGI-I) scale. As
             this was an exploratory study, no inferential statistical
             analyses were performed.GXR was safe and well tolerated.
             Treatment-related mean ± standard deviation changes in
             heart rate (GXR: 1.8 ± 12 beats per minute [bpm]
             decrease; placebo: 0.5 ± 11 bpm decrease), systolic
             blood pressure (GXR: 2.3 ± 11 mm Hg decrease;
             placebo: 1.7 ± 11 mm Hg decrease), or diastolic blood
             pressure (GXR: 1.3 ± 9 mm Hg decrease; placebo:
             0.9 ± 7 mm Hg increase) were similar between
             treatment groups. TEAEs, including headache,
             somnolence/fatigue, abdominal pain, and dizziness, were
             consistent with the known safety profile of GXR. No
             differences were observed between treatment groups for PARS
             and SCARED scores, although at endpoint, a higher proportion
             of subjects receiving GXR versus placebo demonstrated CGI-I
             scores ≤2 (54.2% vs. 31.6%), as rated by the clinician
             investigator.GXR was well tolerated in pediatric subjects
             with GAD, SAD, and/or social anxiety disorder.
             ClinicalTrials.gov Identifier: NCT01470469.},
   Doi = {10.1089/cap.2016.0132},
   Key = {fds327203}
}


%% Cooper, Harris M.   
@article{fds332043,
   Author = {Appelbaum, M and Cooper, H and Kline, RB and Mayo-Wilson, E and Nezu,
             AM and Rao, SM},
   Title = {Journal article reporting standards for quantitative
             research in psychology: The APA Publications and
             Communications Board task force report.},
   Journal = {American Psychologist},
   Volume = {73},
   Number = {1},
   Pages = {3-25},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1037/amp0000191},
   Abstract = {Following a review of extant reporting standards for
             scientific publication, and reviewing 10 years of experience
             since publication of the first set of reporting standards by
             the American Psychological Association (APA; APA
             Publications and Communications Board Working Group on
             Journal Article Reporting Standards, 2008), the APA Working
             Group on Quantitative Research Reporting Standards
             recommended some modifications to the original standards.
             Examples of modifications include division of hypotheses,
             analyses, and conclusions into 3 groupings (primary,
             secondary, and exploratory) and some changes to the section
             on meta-analysis. Several new modules are included that
             report standards for observational studies, clinical trials,
             longitudinal studies, replication studies, and N-of-1
             studies. In addition, standards for analytic methods with
             unique characteristics and output (structural equation
             modeling and Bayesian analysis) are included. These
             proposals were accepted by the Publications and
             Communications Board of APA and supersede the standards
             included in the 6th edition of the Publication Manual of the
             American Psychological Association (APA, 2010). (PsycINFO
             Database Record},
   Doi = {10.1037/amp0000191},
   Key = {fds332043}
}

@article{fds324856,
   Author = {Sanchez, CE and Atkinson, KM and Koenka, AC and Moshontz, H and Cooper,
             H},
   Title = {Self-grading and peer-grading for formative and summative
             assessments in 3rd through 12th grade classrooms: A
             meta-analysis},
   Journal = {Journal of Educational Psychology},
   Volume = {109},
   Number = {8},
   Pages = {1049-1066},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1037/edu0000190},
   Abstract = {© 2017 American Psychological Association. The "assessment
             for learning" movement in education has increased attention
             to self-grading and peer-grading practices in primary and
             secondary schools. This research synthesis examined several
             questions pertaining to the use of self-grading and
             peer-grading in conjunction with criterion-referenced
             testing in 3rd- through 12th-grade-level classrooms. We
             investigated (a) the effects of students' participation in
             grading on subsequent test performance, (b) the difference
             between grades when assigned by students or teachers, and
             (c) the correlation between grades assigned by students and
             teachers. Students who engaged in self-grading performed
             better (g = .34) on subsequent tests than did students who
             did not. Moderator analyses suggested that the benefits of
             self-grading were estimated to be greater when the study
             controlled for group differences through random assignment.
             Students who engaged in peergrading performed better on
             subsequent tests than did students who did not (g = .29). On
             average, students did not grade themselves or peers
             significantly differently than teachers (self-grades, g =
             .04; peer-grades, g = .04) and showed moderate correlation
             (self-grading, r = .67; peer-grading, r = .68) with teacher
             grades. Further, other moderator analyses and examination of
             studies suggested that self- and peer-grading practices can
             be implemented to positive effect in primary and secondary
             schools with the use of rubrics and training for students in
             a formative assessment environment. However, because of a
             limited number of studies, these mediating variables need
             more research to allow more conclusive findings.},
   Doi = {10.1037/edu0000190},
   Key = {fds324856}
}

@book{fds326524,
   Author = {Cooper, H},
   Title = {Research Synthesis and Meta-Analysis: A Step-by-Step
             Approach, 5th Edition},
   Volume = {2},
   Pages = {384 pages},
   Publisher = {SAGE PUBLICATIONS INC},
   Year = {2017},
   ISBN = {1483331156},
   Abstract = {The Fifth Edition of Harris Cooper's bestselling Research
             Synthesis and Meta-Analysis: A Step-by-Step Approach offers
             practical advice on how to conduct a synthesis of research
             in the social, behavioral, and health sciences. The book is
             written in plain language with four running examples drawn
             from psychology, education, and health science. With ample
             coverage of literature searching and the technical aspects
             of meta-analysis, this one-of-a-kind book applies the basic
             principles of sound data gathering to the task of producing
             a comprehensive assessment of existing research.},
   Key = {fds326524}
}


%% Costanzo, Philip R.   
@article{fds335642,
   Author = {Hussong, AM and Langley, HA and Rothenberg, WA and Coffman, JL and Halberstadt, AG and Costanzo, PR and Mokrova, I},
   Title = {Raising grateful children one day at a time},
   Journal = {Applied Developmental Science},
   Pages = {1-14},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1080/10888691.2018.1441713},
   Doi = {10.1080/10888691.2018.1441713},
   Key = {fds335642}
}

@article{fds337726,
   Author = {Hussong, AM and Langley, HA and Thomas, TE and Coffman, JL and Halberstadt, AG and Costanzo, PR and Rothenberg,
             WA},
   Title = {Measuring gratitude in children},
   Journal = {The Journal of Positive Psychology},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1080/17439760.2018.1497692},
   Abstract = {© 2018, © 2018 Informa UK Limited, trading as Taylor &
             Francis Group. Gratitude is a rich socioemotional construct
             that emerges over development beginning in early childhood.
             Existing measures of children’s gratitude as a trait or
             behavior may be limited because they do not capture
             different aspects of gratitude moments (i.e. awareness,
             thoughts, feelings, and actions) and the way that these
             facets appear in children. The current study evaluates a
             battery of new measures assessing children’s gratitude to
             address these limitations. Parent-child dyads (N = 101;
             children aged 6–9) completed a lab-based assessment
             followed by a 7-day online parental diary and 18-month
             follow-up survey. In addition to newly developed measures of
             children’s gratitude, the battery included indicators of
             convergent, concurrent, divergent, and predictive validity.
             Results demonstrate the complexity of gratitude as a
             construct and the relative benefits and limits of various
             assessment modalities. Implications for the measurement of
             children’s gratitude and suggestions for future research
             on the development of gratitude are discussed.},
   Doi = {10.1080/17439760.2018.1497692},
   Key = {fds337726}
}

@article{fds326697,
   Author = {Golonka, MM and Peairs, KF and Malone, PS and Grimes, CL and Costanzo,
             PR},
   Title = {Natural Peer Leaders as Substance Use Prevention Agents: the
             Teens' Life Choice Project.},
   Journal = {Prevention Science : the Official Journal of the Society for
             Prevention Research},
   Volume = {18},
   Number = {5},
   Pages = {555-566},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1007/s11121-017-0790-4},
   Abstract = {In adolescent social groups, natural peer leaders have been
             found to engage in more frequent experimentation with
             substance use and to possess disproportionate power to
             affect the behavior and social choices of their associated
             peer followers. In the current exploratory study, we used
             sociometrics and social cognitive mapping to identify
             natural leaders of cliques in a seventh grade population and
             invited the leaders to develop anti-drug presentations for
             an audience of younger peers. The program employed
             social-psychological approaches directed at having leaders
             proceed from extrinsic inducements to intrinsic
             identification with their persuasive products in the context
             of the group intervention process. The goals of the
             intervention were to induce substance resistant
             self-persuasion in the leaders and to produce a spread of
             this resistance effect to their peer followers. To test the
             intervention, we compared the substance use behaviors of the
             selected leaders and their peers to a control cohort. The
             study found preliminary support that the intervention
             produced changes in the substance use behavior among the
             leaders who participated in the intervention, but did not
             detect a spread to non-leader peers in the short term. This
             descriptive study speaks to the plausibility of employing
             self-persuasion paradigms to bring about change in high-risk
             behaviors among highly central adolescents. In addition, it
             highlights the viability of applying social psychological
             principles to prevention work and calls for more research in
             this area.},
   Doi = {10.1007/s11121-017-0790-4},
   Key = {fds326697}
}

@misc{fds335643,
   Author = {Hussong, AM and Langley, HA and Coffman, JL and Halberstadt, AG and Costanzo, PR},
   Title = {Parent socialization of children’s gratitude},
   Pages = {199-219},
   Booktitle = {Developing Gratitude in Children and Adolescents},
   Year = {2017},
   Month = {January},
   ISBN = {9781316863121},
   url = {http://dx.doi.org/10.1017/9781316863121.010},
   Abstract = {© Cambridge University Press 2018. Parent Socialization of
             Children’s Gratitude Thank-You Note I wanted small pierced
             earrings (gold), You gave me slippers (gray). My mother said
             that she would scold Unless I wrote to say How much I like
             them. Not much. -Judith Viorst The desire to cultivate
             gratitude in ourselves and others dates back centuries, as
             is evident in the early writings of Aristotle on virtues
             (Thomson, 1955), although our understanding of what
             gratitude means continues to evolve through ongoing
             scholarly debate and societal discourse (Kapp, 2013; Reiser,
             2014). One of the voices in this debate comes from social
             psychologists affiliated with the Positive Psychology
             movement who spearheaded research that has shaped our
             understanding of gratitude in adults. These researchers
             differentially adopt the view of gratitude as a life
             orientation (Wood, Froh, & Geraghty, 2010); a character,
             virtue, or personality trait (Froh, Sefick, & Emmons, 2008);
             and a mood or emotional state (McCullough, Emmons, & Tsang,
             2002). Research based on this view shows that adults and
             adolescents who more strongly endorse gratitude traits also
             report greater life satisfaction, better health outcomes,
             and more successful relationships (see Algoe, Haidt, &
             Gable, 2008; Bausert et al., Chapter 7, this volume; Emmons
             & McCullough, 2003; Froh, Kashdan, Ozimkowski, & Miller,
             2009; Wood et al., 2010). Similar research with children has
             lagged behind that with adults, although this volume is one
             of a handful of recent works demonstrating growing interest
             in this topic. As with other areas of research, the
             psychological study of gratitude has largely followed a
             downward extension model with the goal of uncovering how
             early in childhood scientists can replicate findings from
             adult samples. This approach to understanding gratitude in
             children aligns with that from the classic descriptive focus
             of developmental psychology that seeks to identify at what
             ages a given competency emerges. More recent approaches to
             understanding development, such as the developmental science
             framework (Cairns & Elder, 2001), eschew age difference
             findings as an end goal in favor of understanding how a
             given competency emerges over ontogeny and what form it
             takes within the system of influences from which that
             competency might arise. To meet this aim, a developmental
             science approach must squarely tackle the issues of what
             gratitude is at its core, how it changes with ontogeny, and
             how we best capture its elements as they emerge first in a
             nascent and then in a mature form.},
   Doi = {10.1017/9781316863121.010},
   Key = {fds335643}
}

@article{fds331136,
   Author = {Rothenberg, WA and Hussong, AM and Langley, HA and Egerton, GA and Halberstadt, AG and Coffman, JL and Mokrova, I and Costanzo,
             PR},
   Title = {Grateful parents raising grateful children: Niche selection
             and the socialization of child gratitude.},
   Journal = {Applied Developmental Science},
   Volume = {21},
   Number = {2},
   Pages = {106-120},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1080/10888691.2016.1175945},
   Abstract = {Given that children's exposure to gratitude-related
             activities may be one way that parents can socialize
             gratitude in their children, we examined whether parents'
             niche selection (i.e., tendency to choose perceived
             gratitude-inducing activities for their children) mediates
             the association between parents' reports of their own and
             their children's gratitude. Parent-child dyads (N =101;
             children aged 6-9; 52% girls; 80% Caucasian; 85% mothers)
             participated in a laboratory visit and parents also
             completed a seven-day online diary regarding children's
             gratitude. Decomposing specific indirect effects within a
             structural equation model, we found that parents high in
             gratitude were more likely to set goals to use niche
             selection as a gratitude socialization strategy, and thereby
             more likely to place their children in gratitude-related
             activities. Placement in these activities, in turn, was
             associated with more frequent expression of gratitude in
             children. We describe future directions for research on
             parents' role in socializing gratitude in their
             children.},
   Doi = {10.1080/10888691.2016.1175945},
   Key = {fds331136}
}


%% Costello, Elizabeth J.   
@article{fds337359,
   Author = {Eg, J and Bilenberg, N and Costello, EJ and Wesselhoeft,
             R},
   Title = {Self- and parent-reported depressive symptoms rated by the
             mood and feelings questionnaire.},
   Journal = {Psychiatry Research},
   Volume = {268},
   Pages = {419-425},
   Year = {2018},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.psychres.2018.07.016},
   Abstract = {The Mood and Feelings Questionnaire (MFQ) was developed to
             measure depressive symptoms in children and adolescents. It
             includes a self-report and a parent-report part. This study
             set out to test the psychometric properties of the MFQ in a
             Danish population of children and adolescents.The study
             included a population-based sample of n = 992
             individuals aged 9-17 years and n = 703 parents from
             five schools. The internal consistencies of both MFQ
             versions were excellent with high alpha coefficients. With
             few exceptions, correlation between items and the total
             score was moderate to high. Vegetative symptoms were among
             the lowest correlating items while cognitive symptoms were
             among the highest. Girls reported more depressive symptoms
             than boys, and reports from offspring indicated more
             depressive symptoms than reports from parents. There was no
             difference in depressive symptoms by respondents aged 9 to
             11 compared to respondents aged 12 to 17 in schools where
             all pupils participated. However, in schools where pupils
             participated by choice, an increase in depressive symptoms
             by age was found. This study suggests that MFQ is reliable
             for evaluating depressive symptoms in a population of
             children and adolescents. Furthermore, it is of clinical
             relevance that parents tend to underreport depressive
             symptoms of their offspring.},
   Doi = {10.1016/j.psychres.2018.07.016},
   Key = {fds337359}
}

@article{fds333242,
   Author = {Akee, R and Copeland, W and Costello, EJ and Simeonova,
             E},
   Title = {How Does Household Income Affect Child Personality Traits
             and Behaviors?},
   Journal = {American Economic Review},
   Volume = {108},
   Number = {3},
   Pages = {775-827},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1257/aer.20160133},
   Abstract = {We examine the effects of a quasi-experimental unconditional
             household income transfer on child emotional and behavioral
             health and personality traits. Using longitudinal data, we
             find that there are large beneficial effects on children's
             emotional and behavioral health and personality traits
             during adolescence. We find evidence that these effects are
             most pronounced for children who start out with the lowest
             initial endowments. The income intervention also results in
             improvements in parental relationships which we interpret as
             a potential mechanism behind our findings.},
   Doi = {10.1257/aer.20160133},
   Key = {fds333242}
}

@article{fds329142,
   Author = {Hill, S and Shanahan, L and Costello, EJ and Copeland,
             W},
   Title = {Predicting Persistent, Limited, and Delayed Problematic
             Cannabis Use in Early Adulthood: Findings From a
             Longitudinal Study.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {966-974.e4},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.08.012},
   Abstract = {To identify risk profiles associated with patterns of
             problematic cannabis use in early adulthood.Data came from
             1,229 participants in the Great Smoky Mountains Study, a
             prospective 20-year cohort study from 1993 to 2015 that is
             representative of western North Carolina with yearly
             assessments conducted from ages 9 and 16 years, and
             assessments at ages 19, 21, 26, and 30 years. Patterns of
             problematic cannabis use (i.e., DSM-5 cannabis use disorder
             or daily use) in early adulthood included the following:
             nonproblematic use in late adolescence (ages 19-21) and
             early adulthood (ages 26-30); limited problematic use in
             late adolescence only; persistent problematic use in late
             adolescence and early adulthood; and delayed problematic use
             in early adulthood only. Multinominal logistic regression
             models examined pairwise associations between these patterns
             and risk factors in childhood/early adolescence (ages 9-16)
             and late adolescence (ages 19-21). Risk factors included
             psychiatric disorders (e.g., anxiety, depressive), other
             substance use (smoking, alcohol, illicit drugs), and
             challenging social factors (e.g., low socioeconomic status,
             family functioning, peers). Sex and race/ethnicity (white,
             African American, American Indian) interactions were
             tested.The persistent pattern (6.7% of sample) was
             characterized by more anxiety disorders across development
             and more DSM-5 CUD symptoms during late adolescence compared
             to the limited pattern (13.3%), which, in turn, had more
             childhood family instability and dysfunction. The delayed
             pattern (3.7%) was characterized by more externalizing
             disorders, maltreatment, and peer bullying in childhood
             compared to those in nonproblematic users. There were no
             significant interactions of sex or race/ethnicity.Problematic
             cannabis use patterns during early adulthood have
             distinctive risk profiles, which may be useful in tailoring
             targeted interventions.},
   Doi = {10.1016/j.jaac.2017.08.012},
   Key = {fds329142}
}

@article{fds329547,
   Author = {Copeland, WE and Goldston, DB and Costello, EJ},
   Title = {Adult Associations of Childhood Suicidal Thoughts and
             Behaviors: A Prospective, Longitudinal Analysis.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {958-965.e4},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.08.015},
   Abstract = {Suicidal thoughts and behavior (STBs) have their peak period
             of onset in adolescence, but little is known about how such
             behavior is associated with later functioning. The aim of
             this study is to test whether childhood STBs are related to
             adult psychiatric, suicidal, and functional outcomes.This is
             a prospective, population-based community study of 1,420
             participants assessed with structured interviews up to 7
             times in childhood/adolescence (ages 9-16 years; 6,674
             observations) for STBs including passive and active
             ideation, plans, and attempts. Participants were then
             assessed 4 times in young adulthood (ages 19, 21, 24, and 30
             years; 4,556 observations of 1,273 participants) for
             psychiatric diagnoses, STBs, and functional outcomes.By age
             16 years, 7.0% of participants had reported some type of
             STBs, with 3.9% reporting an attempt. Both ideation only and
             suicide attempts were associated with higher levels of
             anxiety disorders and STBs in adulthood, as well as poor
             functioning across financial, health, risky/illegal, and
             social domains. These observed effects generally were
             attenuated after adjusting for other psychiatric and
             psychosocial factors that predict childhood STBs
             (particularly maltreatment, depression, and disruptive
             behavior disorders). The exception was adult suicidal
             behavior, which was predicted by both childhood ideation and
             attempts, even in the fully adjusted model. Children and
             adolescents with STBs were more likely to have had a
             disrupted transition to adulthood.Childhood STBs are a
             marker for a multitude of poor psychiatric and functional
             outcomes in adulthood, but these effects are largely
             accounted for by other factors. In contrast, childhood STBs
             are a robust risk factor for adult suicidal thoughts and
             behavior.},
   Doi = {10.1016/j.jaac.2017.08.015},
   Key = {fds329547}
}

@article{fds326255,
   Author = {Clark, SL and McClay, JL and Adkins, DE and Kumar, G and Aberg, KA and Nerella, S and Xie, L and Collins, AL and Crowley, JJ and Quackenbush,
             CR and Hilliard, CE and Shabalin, AA and Vrieze, SI and Peterson, RE and Copeland, WE and Silberg, JL and McGue, M and Maes, H and Iacono, WG and Sullivan, PF and Costello, EJ and van den Oord,
             EJ},
   Title = {Deep Sequencing of 71 Candidate Genes to Characterize
             Variation Associated with Alcohol Dependence.},
   Journal = {Alcoholism, Clinical and Experimental Research},
   Volume = {41},
   Number = {4},
   Pages = {711-718},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1111/acer.13352},
   Abstract = {Previous genomewide association studies (GWASs) have
             identified a number of putative risk loci for alcohol
             dependence (AD). However, only a few loci have replicated
             and these replicated variants only explain a small
             proportion of AD risk. Using an innovative approach, the
             goal of this study was to generate hypotheses about
             potentially causal variants for AD that can be explored
             further through functional studies.We employed targeted
             capture of 71 candidate loci and flanking regions followed
             by next-generation deep sequencing (mean coverage 78X) in
             806 European Americans. Regions included in our targeted
             capture library were genes identified through published GWAS
             of alcohol, all human alcohol and aldehyde dehydrogenases,
             reward system genes including dopaminergic and opioid
             receptors, prioritized candidate genes based on previous
             associations, and genes involved in the absorption,
             distribution, metabolism, and excretion of drugs. We
             performed single-locus tests to determine if any single
             variant was associated with AD symptom count. Sets of
             variants that overlapped with biologically meaningful
             annotations were tested for association in aggregate.No
             single, common variant was significantly associated with AD
             in our study. We did, however, find evidence for association
             with several variant sets. Two variant sets were significant
             at the q-value <0.10 level: a genic enhancer for ADHFE1
             (p = 1.47 × 10-5 ; q = 0.019), an alcohol
             dehydrogenase, and ADORA1 (p = 5.29 × 10-5 ;
             q = 0.035), an adenosine receptor that belongs to a
             G-protein-coupled receptor gene family.To our knowledge,
             this is the first sequencing study of AD to examine variants
             in entire genes, including flanking and regulatory regions.
             We found that in addition to protein coding variant sets,
             regulatory variant sets may play a role in AD. From these
             findings, we have generated initial functional hypotheses
             about how these sets may influence AD.},
   Doi = {10.1111/acer.13352},
   Key = {fds326255}
}

@article{fds323141,
   Author = {Copeland, WE and Hill, S and Costello, EJ and Shanahan,
             L},
   Title = {Cannabis Use and Disorder From Childhood to Adulthood in a
             Longitudinal Community Sample With American
             Indians.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {2},
   Pages = {124-132.e2},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.jaac.2016.11.006},
   Abstract = {Recent changes in DSM criteria require new documentation of
             the prevalence and developmental sequences of cannabis use
             disorder (CUD). The goal of this study was to investigate
             the early course of DSM-5 CUD and its overlap with DSM-IV
             and consumption constructs in a community-representative
             sample of American Indians.Data came from the prospective,
             longitudinal, population-based Great Smoky Mountains Study
             in North Carolina (N = 1,420, including 349 American
             Indians). Cannabis use and disorder were assessed during
             yearly interviews from 9 to 16 years of age and at 19, 21,
             26, and 30 years of age (up to 11 assessments per
             participant from 1993 through 2015).By 30 years of age,
             approximately 70% of participants had used cannabis, 34% had
             used cannabis daily, and 18% had met criteria for DSM-5 CUD.
             Approximately 1 in 4 cannabis users met criteria for CUD at
             some point. Those who met criteria initiated use more than 2
             years previously (at 13.3 years old) compared with other
             users. Despite higher risks from increased poverty, American
             Indians' patterns of use were similar to those of the rest
             of the sample. Concordance between DSM-5 CUD and DSM-IV
             abuse or dependence was substantial but was even higher
             between DSM-5 CUD and daily use.It was common to have used
             cannabis daily or to have met criteria for DSM-5 CUD by
             adulthood. DSM-5 CUD was an improvement over DSM-IV
             diagnostic constructs by raising the threshold for
             diagnosis.},
   Doi = {10.1016/j.jaac.2016.11.006},
   Key = {fds323141}
}


%% Curry, John F.   
@article{fds337454,
   Author = {Donoho, CJ and LeardMann, C and O'Malley, CA and Walter, KH and Riviere,
             LA and Curry, JF and Adler, AB},
   Title = {Depression among military spouses: Demographic, military,
             and service member psychological health risk
             factors.},
   Journal = {Depression and Anxiety},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1002/da.22820},
   Abstract = {More than a decade of war has strained service members and
             their families and the psychological health of military
             spouses is a concern. This study uses data from the largest
             study of military families in the United States to examine
             the demographic, military-specific, and service member
             mental health correlates of probable diagnosis of major
             depressive disorder (MDD) among military spouses.Data were
             from service member-spouse dyads from all branches of the
             U.S. military. Demographic and military-specific factors
             were assessed using administrative personnel records and
             survey data.Of the 9,038 spouses, 4.9% had a probable
             diagnosis of MDD. In unadjusted models, spouses of service
             members who deployed and experiencecd combat-related events,
             were enlisted, had a probable posttraumatic stress disorder
             (PTSD) diagnosis, or screened positive for alcohol misuse
             were more likely to screen positive for MDD. In adjusted
             models, only spouses married to enlisted service members or
             those with PTSD had increased risk for MDD. Other
             demographic and military-related factors associated with MDD
             in spouses included less educational attainment,
             unemployment, having four or more children, and having prior
             military service (although not currently serving in the
             military) in the adjusted models.Findings characterize
             demographic, military, and service member psychological
             health factors that are associated with depression among
             military spouses. These findings imply that deployment alone
             may not negatively affect military spouses, but rather it
             may be the mental health impact on the service member,
             especially PTSD that increases the odds for MDD among
             military spouses.},
   Doi = {10.1002/da.22820},
   Key = {fds337454}
}

@article{fds336065,
   Author = {Curry, JF and Kiser, LJ and Fernandez, PE and Elliott, AV and Dowling,
             LM},
   Title = {Development and initial piloting of a measure of
             post-deployment parenting reintegration experiences.},
   Journal = {Professional Psychology: Research and Practice},
   Volume = {49},
   Number = {2},
   Pages = {159-166},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1037/pro0000185},
   Doi = {10.1037/pro0000185},
   Key = {fds336065}
}

@article{fds328869,
   Author = {Meyer, AE and Curry, JF},
   Title = {Pathways from anxiety to stressful events: An expansion of
             the stress generation hypothesis.},
   Journal = {Clinical Psychology Review},
   Volume = {57},
   Pages = {93-116},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.cpr.2017.08.003},
   Doi = {10.1016/j.cpr.2017.08.003},
   Key = {fds328869}
}

@article{fds327307,
   Author = {Shepherd-Banigan, M and Kelley, ML and Katon, JG and Curry, JF and Goldstein, KM and Brancu, M and Wagner, HR and Fecteau, TE and VA
             Mid-Atlantic MIRECC Women Veterans Workgroup, and VA
             Mid-Atlantic MIRECC Workgroup, and Van Houtven,
             CH},
   Title = {Paternal history of mental illness associated with
             posttraumatic stress disorder among veterans.},
   Journal = {Psychiatry Research},
   Volume = {256},
   Pages = {461-468},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.psychres.2017.06.053},
   Abstract = {This study examined the association between parent and
             family reported history of non-PTSD mental illness (MI),
             PTSD specifically, and substance use problems, and
             participant clinical diagnosis of PTSD. Participants were
             drawn from the US Department of Veterans Affairs
             Mid-Atlantic Mental Illness Research, Education and Clinical
             Center (MIRECC) Post-Deployment Mental Health (PDMH) study
             (n = 3191), an ongoing multi-site cohort study of US
             Afghanistan and Iraq conflict era veterans. Participants who
             recalled a father history of PTSD had a 26-percentage point
             higher likelihood of meeting criteria for PTSD; while
             participants reporting any family history of PTSD had a
             15-percentage point higher probability of endorsing symptoms
             consistent with PTSD. Mother history of substance use
             problems was associated with Veteran current PTSD, but
             results were sensitive to model specification. Current PTSD
             was not associated with family/parent history of non-PTSD
             mental illness, mother history of PTSD, or family/father
             history of substance use problems. Family history of PTSD
             may increase PTSD risk among veterans exposed to trauma,
             particularly when a father history is reported. Knowledge of
             family history could improve clinical decision-making for
             trauma-exposed individuals and allow for more effective
             targeting of programs and clinical services.},
   Doi = {10.1016/j.psychres.2017.06.053},
   Key = {fds327307}
}


%% Dailey, Shannon   
@article{fds337152,
   Author = {Bergelson, E and Amatuni, A and Dailey, S and Koorathota, S and Tor,
             S},
   Title = {Day by day, hour by hour: Naturalistic language input to
             infants.},
   Journal = {Developmental Science},
   Pages = {e12715},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1111/desc.12715},
   Abstract = {Measurements of infants' quotidian experiences provide
             critical information about early development. However, the
             role of sampling methods in providing these measurements is
             rarely examined. Here we directly compare language input
             from hour-long video-recordings and daylong audio-recordings
             within the same group of 44 infants at 6 and 7 months. We
             compared 12 measures of language quantity and lexical
             diversity, talker variability, utterance-type, and object
             presence, finding moderate correlations across
             recording-types. However, video-recordings generally
             featured far denser noun input across these measures
             compared to the daylong audio-recordings, more akin to
             'peak' audio hours (though not as high in talkers and
             word-types). Although audio-recordings captured ~10 times
             more awake-time than videos, the noun input in them was only
             2-4 times greater. Notably, whether we compared videos to
             daylong audio-recordings or peak audio times, videos
             featured relatively fewer declaratives and more questions;
             furthermore, the most common video-recorded nouns were less
             consistent across families than the top audio-recording
             nouns were. Thus, hour-long videos and daylong
             audio-recordings revealed fairly divergent pictures of the
             language infants hear and learn from in their daily lives.
             We suggest that short video-recordings provide a dense and
             somewhat different sample of infants' language experiences,
             rather than a typical one, and should be used cautiously for
             extrapolation about common words, talkers, utterance-types,
             and contexts at larger timescales. If theories of language
             development are to be held accountable to 'facts on the
             ground' from observational data, greater care is needed to
             unpack the ramifications of sampling methods of early
             language input.},
   Doi = {10.1111/desc.12715},
   Key = {fds337152}
}

@article{fds337153,
   Author = {Hunt, M and Al-Braiki, F and Dailey, S and Russell, R and Simon,
             K},
   Title = {Mindfulness Training, Yoga, or Both? Dismantling the Active
             Components of a Mindfulness-Based Stress Reduction
             Intervention},
   Journal = {Mindfulness},
   Volume = {9},
   Number = {2},
   Pages = {512-520},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s12671-017-0793-z},
   Doi = {10.1007/s12671-017-0793-z},
   Key = {fds337153}
}


%% Dawson, Geraldine   
@article{fds338008,
   Author = {Hashemi, J and Dawson, G and Carpenter, KLH and Campbell, K and Qiu, Q and Espinosa, S and Marsan, S and Baker, JP and Egger, HL and Sapiro,
             G},
   Title = {Computer Vision Analysis for Quantification of Autism Risk
             Behaviors},
   Journal = {Ieee Transactions on Affective Computing},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1109/TAFFC.2018.2868196},
   Abstract = {IEEE Observational behavior analysis plays a key role for
             the discovery and evaluation of risk markers for many
             neurodevelopmental disorders. Research on autism spectrum
             disorder (ASD) suggests that behavioral risk markers can be
             observed at 12 months of age, with diagnosis possible at 18
             months. To date, studies and evaluations involving
             observational analysis tend to rely heavily on clinical
             practitioners and specialists who have undergone intensive
             training to be able to reliably administer carefully
             designed behavioral-eliciting tasks, code the resulting
             behaviors, and interpret them. These methods are therefore
             extremely expensive, time-intensive, and are not easily
             scalable for large or longitudinal observational analysis.
             We developed a self-contained, closed-loop, mobile
             application with movie stimuli designed to engage the
             child&#x0027;s attention and elicit specific behavioral and
             social responses, which are recorded with the mobile
             device&#x0027;s camera and analyzed via computer vision
             algorithms. Here, in addition to presenting this paradigm,
             we validate the system to measure engagement, name-call, and
             emotional responses of toddlers with and without ASD who
             were presented with the application. Additionally, we
             demonstrate how the proposed framework can further risk
             marker research with fine-grained quantification of
             behaviors. The results suggest these objective and automatic
             methods can be considered to aid behavioral
             analysis.},
   Doi = {10.1109/TAFFC.2018.2868196},
   Key = {fds338008}
}

@article{fds337473,
   Author = {Murias, M and Major, S and Compton, S and Buttinger, J and Sun, JM and Kurtzberg, J and Dawson, G},
   Title = {Electrophysiological Biomarkers Predict Clinical Improvement
             in an Open-Label Trial Assessing Efficacy of Autologous
             Umbilical Cord Blood for Treatment of Autism.},
   Journal = {Stem Cells Translational Medicine},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1002/sctm.18-0090},
   Abstract = {This study was a phase I, single-center, and open-label
             trial of a single intravenous infusion of autologous
             umbilical cord blood in young children with autism spectrum
             disorder (ASD). Twenty-five children between the ages of 2
             and 6 with a confirmed diagnosis of ASD and a qualified
             banked autologous umbilical cord blood unit were enrolled.
             Safety results and clinical outcomes measured at 6 and 12
             months post-infusion have been previously published. The
             purpose of the present analysis was to explore whether
             measures of electroencephalography (EEG) theta, alpha, and
             beta power showed evidence of change after treatment and
             whether baseline EEG characteristics were predictive of
             clinical improvement. The primary endpoint was the
             parent-reported Vineland adaptive behavior scales-II
             socialization subscale score, collected at baseline, 6- and
             12-month visits. In addition, the expressive one word
             picture vocabulary test 4 and the clinical global
             impression-improvement scale were administered.
             Electrophysiological recordings were taken during viewing of
             dynamic social and nonsocial stimuli at 6 and 12 months
             post-treatment. Significant changes in EEG spectral
             characteristics were found by 12 months post-infusion, which
             were characterized by increased alpha and beta power and
             decreased EEG theta power. Furthermore, higher baseline
             posterior EEG beta power was associated with a greater
             degree of improvement in social communication symptoms,
             highlighting the potential for an EEG biomarker to predict
             variation in outcome. Taken together, the results suggest
             that EEG measures may be useful endpoints for future ASD
             clinical trials.},
   Doi = {10.1002/sctm.18-0090},
   Key = {fds337473}
}

@article{fds335827,
   Author = {Frazier, TW and Dawson, G and Murray, D and Shih, A and Sachs, JS and Geiger, A},
   Title = {Brief Report: A Survey of Autism Research Priorities Across
             a Diverse Community of Stakeholders.},
   Journal = {Journal of Autism and Developmental Disorders},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s10803-018-3642-6},
   Abstract = {Inclusion of stakeholder voices in the allocation of
             research funding can increase the relevance of results and
             improve community engagement in research. We describe the
             results of an online survey that gathered input from
             community stakeholders regarding autism research priorities.
             A demographically diverse sample of respondents
             (N = 6004; 79.1% female; 72.5% ages 30-59; 86.4% USA)
             completed the survey. Results indicated a preference for
             applied relative to basic science topics, though both basic
             and applied science areas were rated as important.
             Respondents gave their highest ratings to research focused
             on co-occurring conditions, health and well-being, adult
             transition, and lifespan issues. These results can guide
             decision-making by public and private funders when
             developing science funding priorities and engaging in
             science dissemination activities.},
   Doi = {10.1007/s10803-018-3642-6},
   Key = {fds335827}
}

@article{fds335828,
   Author = {Anttila, V and Bulik-Sullivan, B and Finucane, HK and Walters, RK and Bras, J and Duncan, L and Escott-Price, V and Falcone, GJ and Gormley,
             P and Malik, R and Patsopoulos, NA and Ripke, S and Wei, Z and Yu, D and Lee,
             PH and Turley, P and Grenier-Boley, B and Chouraki, V and Kamatani, Y and Berr, C and Letenneur, L and Hannequin, D and Amouyel, P and Boland, A and Deleuze, J-F and Duron, E and Vardarajan, BN and Reitz, C and Goate, AM and Huentelman, MJ and Kamboh, MI and Larson, EB and Rogaeva, E and St
             George-Hyslop, P and Hakonarson, H and Kukull, WA and Farrer, LA and Barnes, LL and Beach, TG and Demirci, FY and Head, E and Hulette, CM and Jicha, GA and Kauwe, JSK and Kaye, JA and Leverenz, JB and Levey, AI and Lieberman, AP and Pankratz, VS and Poon, WW and Quinn, JF and Saykin,
             AJ and Schneider, LS and Smith, AG and Sonnen, JA and Stern, RA and Van
             Deerlin, VM and Van Eldik and LJ and Harold, D and Russo, G and Rubinsztein, DC and Bayer, A and Tsolaki, M and Proitsi, P and Fox, NC and Hampel, H and Owen, MJ and Mead, S and Passmore, P and Morgan, K and Nöthen, MM and Rossor, M and Lupton, MK and Hoffmann, P and Kornhuber,
             J and Lawlor, B and McQuillin, A and Al-Chalabi, A and Bis, JC and Ruiz, A and Boada, M and Seshadri, S and Beiser, A and Rice, K and van der Lee, SJ and De Jager and PL and Geschwind, DH and Riemenschneider, M and Riedel-Heller, S and Rotter, JI and Ransmayr, G and Hyman, BT and Cruchaga, C and Alegret, M and Winsvold, B and Palta, P and Farh, K-H and Cuenca-Leon, E and Furlotte, N and Kurth, T and Ligthart, L and Terwindt, GM and Freilinger, T and Ran, C and Gordon, SD and Borck, G and Adams, HHH and Lehtimäki, T and Wedenoja, J and Buring, JE and Schürks, M and Hrafnsdottir, M and Hottenga, J-J and Penninx, B and Artto, V and Kaunisto, M and Vepsäläinen, S and Martin, NG and Montgomery, GW and Kurki, MI and Hämäläinen, E and Huang, H and Huang, J and Sandor, C and Webber, C and Muller-Myhsok, B and Schreiber,
             S and Salomaa, V and Loehrer, E and Göbel, H and Macaya, A and Pozo-Rosich, P and Hansen, T and Werge, T and Kaprio, J and Metspalu, A and Kubisch, C and Ferrari, MD and Belin, AC and van den Maagdenberg,
             AMJM and Zwart, J-A and Boomsma, D and Eriksson, N and Olesen, J and Chasman, DI and Nyholt, DR and Avbersek, A and Baum, L and Berkovic, S and Bradfield, J and Buono, R and Catarino, CB and Cossette, P and De
             Jonghe, P and Depondt, C and Dlugos, D and Ferraro, TN and French, J and Hjalgrim, H and Jamnadas-Khoda, J and Kälviäinen, R and Kunz, WS and Lerche, H and Leu, C and Lindhout, D and Lo, W and Lowenstein, D and McCormack, M and Møller, RS and Molloy, A and Ng, P-W and Oliver, K and Privitera, M and Radtke, R and Ruppert, A-K and Sander, T and Schachter,
             S and Schankin, C and Scheffer, I and Schoch, S and Sisodiya, SM and Smith,
             P and Sperling, M and Striano, P and Surges, R and Thomas, GN and Visscher,
             F and Whelan, CD and Zara, F and Heinzen, EL and Marson, A and Becker, F and Stroink, H and Zimprich, F and Gasser, T and Gibbs, R and Heutink, P and Martinez, M and Morris, HR and Sharma, M and Ryten, M and Mok, KY and Pulit, S and Bevan, S and Holliday, E and Attia, J and Battey, T and Boncoraglio, G and Thijs, V and Chen, W-M and Mitchell, B and Rothwell,
             P and Sharma, P and Sudlow, C and Vicente, A and Markus, H and Kourkoulis,
             C and Pera, J and Raffeld, M and Silliman, S and Boraska Perica and V and Thornton, LM and Huckins, LM and William Rayner and N and Lewis, CM and Gratacos, M and Rybakowski, F and Keski-Rahkonen, A and Raevuori, A and Hudson, JI and Reichborn-Kjennerud, T and Monteleone, P and Karwautz,
             A and Mannik, K and Baker, JH and O'Toole, JK and Trace, SE and Davis, OSP and Helder, SG and Ehrlich, S and Herpertz-Dahlmann, B and Danner, UN and van Elburg, AA and Clementi, M and Forzan, M and Docampo, E and Lissowska, J and Hauser, J and Tortorella, A and Maj, M and Gonidakis,
             F and Tziouvas, K and Papezova, H and Yilmaz, Z and Wagner, G and Cohen-Woods, S and Herms, S and Julià, A and Rabionet, R and Dick, DM and Ripatti, S and Andreassen, OA and Espeseth, T and Lundervold, AJ and Steen, VM and Pinto, D and Scherer, SW and Aschauer, H and Schosser, A and Alfredsson, L and Padyukov, L and Halmi, KA and Mitchell, J and Strober,
             M and Bergen, AW and Kaye, W and Szatkiewicz, JP and Cormand, B and Ramos-Quiroga, JA and Sánchez-Mora, C and Ribasés, M and Casas, M and Hervas, A and Arranz, MJ and Haavik, J and Zayats, T and Johansson, S and Williams, N and Dempfle, A and Rothenberger, A and Kuntsi, J and Oades,
             RD and Banaschewski, T and Franke, B and Buitelaar, JK and Arias
             Vasquez, A and Doyle, AE and Reif, A and Lesch, K-P and Freitag, C and Rivero, O and Palmason, H and Romanos, M and Langley, K and Rietschel,
             M and Witt, SH and Dalsgaard, S and Børglum, AD and Waldman, I and Wilmot,
             B and Molly, N and Bau, CHD and Crosbie, J and Schachar, R and Loo, SK and McGough, JJ and Grevet, EH and Medland, SE and Robinson, E and Weiss,
             LA and Bacchelli, E and Bailey, A and Bal, V and Battaglia, A and Betancur,
             C and Bolton, P and Cantor, R and Celestino-Soper, P and Dawson, G and De
             Rubeis, S and Duque, F and Green, A and Klauck, SM and Leboyer, M and Levitt, P and Maestrini, E and Mane, S and De-Luca, DM and Parr, J and Regan, R and Reichenberg, A and Sandin, S and Vorstman, J and Wassink,
             T and Wijsman, E and Cook, E and Santangelo, S and Delorme, R and Rogé, B and Magalhaes, T and Arking, D and Schulze, TG and Thompson, RC and Strohmaier, J and Matthews, K and Melle, I and Morris, D and Blackwood,
             D and McIntosh, A and Bergen, SE and Schalling, M and Jamain, S and Maaser,
             A and Fischer, SB and Reinbold, CS and Fullerton, JM and Guzman-Parra,
             J and Mayoral, F and Schofield, PR and Cichon, S and Mühleisen, TW and Degenhardt, F and Schumacher, J and Bauer, M and Mitchell, PB and Gershon, ES and Rice, J and Potash, JB and Zandi, PP and Craddock, N and Ferrier, IN and Alda, M and Rouleau, GA and Turecki, G and Ophoff, R and Pato, C and Anjorin, A and Stahl, E and Leber, M and Czerski, PM and Cruceanu, C and Jones, IR and Posthuma, D and Andlauer, TFM and Forstner, AJ and Streit, F and Baune, BT and Air, T and Sinnamon, G and Wray, NR and MacIntyre, DJ and Porteous, D and Homuth, G and Rivera, M and Grove, J and Middeldorp, CM and Hickie, I and Pergadia, M and Mehta, D and Smit, JH and Jansen, R and de Geus, E and Dunn, E and Li, QS and Nauck, M and Schoevers, RA and Beekman, AT and Knowles, JA and Viktorin, A and Arnold, P and Barr, CL and Bedoya-Berrio, G and Bienvenu, OJ and Brentani, H and Burton, C and Camarena, B and Cappi, C and Cath, D and Cavallini, M and Cusi, D and Darrow, S and Denys, D and Derks, EM and Dietrich, A and Fernandez, T and Figee, M and Freimer, N and Gerber, G and Grados, M and Greenberg, E and Hanna, GL and Hartmann, A and Hirschtritt, ME and Hoekstra, PJ and Huang, A and Huyser, C and Illmann,
             C and Jenike, M and Kuperman, S and Leventhal, B and Lochner, C and Lyon,
             GJ and Macciardi, F and Madruga-Garrido, M and Malaty, IA and Maras, A and McGrath, L and Miguel, EC and Mir, P and Nestadt, G and Nicolini, H and Okun, MS and Pakstis, A and Paschou, P and Piacentini, J and Pittenger,
             C and Plessen, K and Ramensky, V and Ramos, EM and Reus, V and Richter, MA and Riddle, MA and Robertson, MM and Roessner, V and Rosário, M and Samuels, JF and Sandor, P and Stein, DJ and Tsetsos, F and Van
             Nieuwerburgh, F and Weatherall, S and Wendland, JR and Wolanczyk, T and Worbe, Y and Zai, G and Goes, FS and McLaughlin, N and Nestadt, PS and Grabe, H-J and Depienne, C and Konkashbaev, A and Lanzagorta, N and Valencia-Duarte, A and Bramon, E and Buccola, N and Cahn, W and Cairns,
             M and Chong, SA and Cohen, D},
   Title = {Analysis of shared heritability in common disorders of the
             brain.},
   Journal = {Science (New York, N.Y.)},
   Volume = {360},
   Number = {6395},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1126/science.aap8757},
   Abstract = {Disorders of the brain can exhibit considerable
             epidemiological comorbidity and often share symptoms,
             provoking debate about their etiologic overlap. We
             quantified the genetic sharing of 25 brain disorders from
             genome-wide association studies of 265,218 patients and
             784,643 control participants and assessed their relationship
             to 17 phenotypes from 1,191,588 individuals. Psychiatric
             disorders share common variant risk, whereas neurological
             disorders appear more distinct from one another and from the
             psychiatric disorders. We also identified significant
             sharing between disorders and a number of brain phenotypes,
             including cognitive measures. Further, we conducted
             simulations to explore how statistical power, diagnostic
             misclassification, and phenotypic heterogeneity affect
             genetic correlations. These results highlight the importance
             of common genetic variation as a risk factor for brain
             disorders and the value of heritability-based methods in
             understanding their etiology.},
   Doi = {10.1126/science.aap8757},
   Key = {fds335828}
}

@misc{fds335829,
   Author = {Kurtzberg, J and Sun, J and Prasad, VK and Meadows, N and Rudisill, A and Noldner, P and Parrott, R and Lillich, M and Cheatham, L and Lew, J and Page, K and Filiano, AJ and Dawson, G},
   Title = {MANUFACTURING OF ALLOGENEIC HUMAN CORD TISSUE MESENCHYMAL
             STROMAL CELLS: INITIAL RESULTS OF A PHASE I CLINICAL TRIAL
             IN CHILDREN WITH AUTISM SPECTRUM DISORDER},
   Journal = {Cytotherapy},
   Volume = {20},
   Number = {5},
   Pages = {S44-S44},
   Publisher = {ELSEVIER SCI LTD},
   Year = {2018},
   Month = {May},
   Key = {fds335829}
}

@article{fds333739,
   Author = {Campbell, K and Carpenter, KL and Hashemi, J and Espinosa, S and Marsan,
             S and Borg, JS and Chang, Z and Qiu, Q and Vermeer, S and Adler, E and Tepper,
             M and Egger, HL and Baker, JP and Sapiro, G and Dawson,
             G},
   Title = {Computer vision analysis captures atypical attention in
             toddlers with autism.},
   Journal = {Autism},
   Pages = {1362361318766247},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/1362361318766247},
   Abstract = {To demonstrate the capability of computer vision analysis to
             detect atypical orienting and attention behaviors in
             toddlers with autism spectrum disorder. One hundered and
             four toddlers of 16-31 months old (mean = 22)
             participated in this study. Twenty-two of the toddlers had
             autism spectrum disorder and 82 had typical development or
             developmental delay. Toddlers watched video stimuli on a
             tablet while the built-in camera recorded their head
             movement. Computer vision analysis measured participants'
             attention and orienting in response to name calls.
             Reliability of the computer vision analysis algorithm was
             tested against a human rater. Differences in behavior were
             analyzed between the autism spectrum disorder group and the
             comparison group. Reliability between computer vision
             analysis and human coding for orienting to name was
             excellent (intra-class coefficient 0.84, 95% confidence
             interval 0.67-0.91). Only 8% of toddlers with autism
             spectrum disorder oriented to name calling on >1 trial,
             compared to 63% of toddlers in the comparison group
             (p = 0.002). Mean latency to orient was significantly
             longer for toddlers with autism spectrum disorder (2.02 vs
             1.06 s, p = 0.04). Sensitivity for autism spectrum
             disorder of atypical orienting was 96% and specificity was
             38%. Older toddlers with autism spectrum disorder showed
             less attention to the videos overall (p = 0.03).
             Automated coding offers a reliable, quantitative method for
             detecting atypical social orienting and reduced sustained
             attention in toddlers with autism spectrum
             disorder.},
   Doi = {10.1177/1362361318766247},
   Key = {fds333739}
}

@article{fds331234,
   Author = {Murias, M and Major, S and Davlantis, K and Franz, L and Harris, A and Rardin, B and Sabatos-DeVito, M and Dawson, G},
   Title = {Validation of eye-tracking measures of social attention as a
             potential biomarker for autism clinical trials.},
   Journal = {Autism Research},
   Volume = {11},
   Number = {1},
   Pages = {166-174},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1002/aur.1894},
   Abstract = {Social communication impairments are a core feature of
             autism spectrum disorder (ASD), and this class of symptoms
             is a target for treatments for the disorder. Measures of
             social attention, assessed via eye-gaze tracking (EGT), have
             been proposed as an early efficacy biomarker for clinical
             trials targeting social communication skills. EGT measures
             have been shown to differentiate children with ASD from
             typical children; however, there is less known about their
             relationships with social communication outcome measures
             that are typically used in ASD clinical trials. In the
             present study, an EGT task involving viewing a videotape of
             an actor making bids for a child's attention was evaluated
             in 25 children with ASD aged 24-72 months. Children's
             attention to the actor during the dyadic bid condition
             measured via EGT was found to be strongly associated with
             five well-validated caregiver-reported outcome measures that
             are commonly used to assess social communication in clinical
             trials. These results highlight the convergent validity of
             EGT measures of social attention in relation to
             caregiver-reported clinical measures. EGT holds promise as a
             non-invasive, quantitative, and objective biomarker that is
             associated with social communication abilities in children
             with ASD. Autism Res 2018, 11: 166-174. © 2017
             International Society for Autism Research, Wiley
             Periodicals, Inc.Eye-gaze tracking (EGT), an automated tool
             that tracks eye-gaze patterns, might help measure outcomes
             in clinical trials investigating interventions to treat
             autism spectrum disorders. In this study, an EGT task was
             evaluated in children with ASD, who watched a video with an
             actor talking directly to them. Patterns of eye-gaze were
             associated with caregiver-reported measures of social
             communication that are used in clinical trials. We show EGT
             may be a promising objective tool measuring
             outcomes.},
   Doi = {10.1002/aur.1894},
   Key = {fds331234}
}

@article{fds326271,
   Author = {Jones, EJH and Dawson, G and Webb, SJ},
   Title = {Sensory hypersensitivity predicts enhanced attention capture
             by faces in the early development of ASD.},
   Journal = {Developmental Cognitive Neuroscience},
   Volume = {29},
   Pages = {11-20},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.dcn.2017.04.001},
   Abstract = {Sensory sensitivity is prevalent among young children with
             ASD, but its relation to social communication impairment is
             unclear. Recently, increased sensory hypersensitivity has
             been linked to greater activity of the neural salience
             network (Green et al., 2016). Increased neural sensitivity
             to stimuli, especially social stimuli, could provide greater
             opportunity for social learning and improved outcomes.
             Consistent with this framework, in Experiment 1 we found
             that parent report of greater sensory hypersensitivity at 2
             years in toddlers with ASD (N=27) was predictive of
             increased neural responsiveness to social stimuli (larger
             amplitude event-related potential/ERP responses to faces at
             P1, P400 and Nc) at 4 years, and this in turn was related to
             parent report of increased social approach at 4 years. In
             Experiment 2, parent report of increased perceptual
             sensitivity at 6 months in infants at low and high familial
             risk for ASD (N=35) predicted larger ERP P1 amplitude to
             faces at 18 months. Increased sensory hypersensitivity in
             early development thus predicted greater attention capture
             by faces in later development, and this related to more
             optimal social behavioral development. Sensory
             hypersensitivity may index a child's ability to benefit from
             supportive environments during development. Early sensory
             symptoms may not always be developmentally problematic for
             individuals with ASD.},
   Doi = {10.1016/j.dcn.2017.04.001},
   Key = {fds326271}
}

@article{fds329121,
   Author = {St John and T and Dawson, G and Estes, A},
   Title = {Brief Report: Executive Function as a Predictor of Academic
             Achievement in School-Aged Children with
             ASD.},
   Journal = {Journal of Autism and Developmental Disorders},
   Volume = {48},
   Number = {1},
   Pages = {276-283},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s10803-017-3296-9},
   Abstract = {The contributions of Executive Function (EF) to academic
             achievement in children with Autism Spectrum Disorder (ASD)
             are not well understood. Academic achievement and its
             association with EF is described in 32, 9-year-old children
             with ASD. EF at age 6 and 9, and academic achievement at age
             9 were assessed as part of a larger longitudinal study.
             Better performance on a Spatial Reversal task but not
             A-not-B with Invisible Displacement at age 6 was associated
             with better math achievement at age 9. No relationship was
             found between these EF measures at age 6 and reading or
             spelling achievement at age 9. Future studies are needed to
             explore whether improving early EF skills can increase math
             achievement in children with ASD.},
   Doi = {10.1007/s10803-017-3296-9},
   Key = {fds329121}
}

@article{fds327659,
   Title = {Meta-analysis of GWAS of over 16,000 individuals with autism
             spectrum disorder highlights a novel locus at 10q24.32 and a
             significant overlap with schizophrenia},
   Journal = {Molecular Autism},
   Volume = {8},
   Number = {1},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1186/s13229-017-0137-9},
   Doi = {10.1186/s13229-017-0137-9},
   Key = {fds327659}
}

@article{fds326793,
   Author = {Bangerter, A and Ness, S and Aman, MG and Esbensen, AJ and Goodwin, MS and Dawson, G and Hendren, R and Leventhal, B and Khan, A and Opler, M and Harris, A and Pandina, G},
   Title = {Autism Behavior Inventory: A Novel Tool for Assessing Core
             and Associated Symptoms of Autism Spectrum
             Disorder.},
   Journal = {Journal of Child and Adolescent Psychopharmacology},
   Volume = {27},
   Number = {9},
   Pages = {814-822},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1089/cap.2017.0018},
   Abstract = {Autism Behavior Inventory (ABI) is a new measure for
             assessing changes in core and associated symptoms of autism
             spectrum disorder (ASD) in participants (ages: 3
             years-adulthood) diagnosed with ASD. It is a web-based tool
             with five domains (two ASD core domains: social
             communication, restrictive and repetitive behaviors; three
             associated domains: mental health, self-regulation, and
             challenging behavior). This study describes design,
             development, and initial psychometric properties of the
             ABI.ABI items were generated following review of existing
             measures and inputs from expert clinicians. Initial ABI
             scale contained 161 items that were reduced to fit a factor
             analytic model, retaining items of adequate reliability. Two
             versions of the scale, ABI-full (ABI-F; 93 items) and
             ABI-short version (ABI-S; 36 items), were developed and
             evaluated for psychometric properties, including validity
             comparisons with commonly used measures. Both scales were
             administered to parents and healthcare professionals (HCPs)
             involved with study participants.Test-retest reliability
             (intraclass correlation coefficient [ICC] = 0.79) for
             parent ratings on ABI was robust and compared favorably to
             existing scales. Test-retest correlations for HCP ratings
             were generally lower versus parent ratings. ABI core domains
             and comparison measures strongly correlated
             (r ≥ 0.70), demonstrating good concurrent
             validity.Overall, ABI demonstrates promise as a tool for
             measuring change in core symptoms of autism in ASD clinical
             studies, with further validation required.},
   Doi = {10.1089/cap.2017.0018},
   Key = {fds326793}
}

@misc{fds332364,
   Author = {Carpenter, K and Major, S and Tallman, C and Chen, L and Franz, L and Sun,
             J and Kurtzberg, J and Song, A and Dawson, G},
   Title = {Phase I Open Label Trial of Autologous Cord Blood for
             Treatment of Autism: Correlations Between Behavioral
             Improvement and Increased White Matter Connectivity in
             Children With Autism},
   Journal = {Neuropsychopharmacology},
   Volume = {43},
   Pages = {S495-S496},
   Year = {2017},
   Month = {November},
   Key = {fds332364}
}

@misc{fds331661,
   Author = {Pandina, G and Manyakov, NV and Bangerter, A and Lewin, D and Jagannatha, S and Boice, M and Skalkin, A and Dawson, G and Goodwin, MS and Hendren, R and Leventhal, B and Shic, F and Ness,
             S},
   Title = {Prospective, observational cohort study of JAKE (TM), an
             autism knowledge},
   Journal = {European Neuropsychopharmacology},
   Volume = {27},
   Pages = {S1118-S1118},
   Year = {2017},
   Month = {October},
   Key = {fds331661}
}

@article{fds329122,
   Author = {Cidav, Z and Munson, J and Estes, A and Dawson, G and Rogers, S and Mandell, D},
   Title = {Cost Offset Associated With Early Start Denver Model for
             Children With Autism.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {9},
   Pages = {777-783},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.06.007},
   Abstract = {To determine the effect of the Early Start Denver Model
             (ESDM) for treatment of young children with autism on health
             care service use and costs.We used data from a randomized
             trial that tested the efficacy of the ESDM, which is based
             on developmental and applied behavioral analytic principles
             and delivered by trained therapists and parents, for 2
             years. Parents were interviewed about their children's
             service use every 6 months from the onset of the
             intervention to follow-up (age 6 years). The sample for this
             study consisted of 39 children with autism who participated
             in the original randomized trial at age 18 to 30 months, and
             were also assessed at age 6 years. Of this sample, 21
             children were in the ESDM group, and 18 children were in the
             community care (COM) group. Reported services were
             categorized and costed by applying unit hourly costs.
             Annualized service use and costs during the intervention and
             post intervention for the two study arms were
             compared.During the intervention, children who received the
             ESDM had average annualized total health-related costs that
             were higher by about $14,000 than those of children who
             received community-based treatment. The higher cost of ESDM
             was partially offset during the intervention period because
             children in the ESDM group used less applied behavior
             analysis (ABA)/early intensive behavioral intervention
             (EIBI) and speech therapy services than children in the
             comparison group. In the postintervention period, compared
             with children who had earlier received treatment as usual in
             community settings, children in the ESDM group used less
             ABA/EIBI, occupational/physical therapy, and speech therapy
             services, resulting in significant cost savings in the
             amount of about $19,000 per year per child.Costs associated
             with ESDM treatment were fully offset within a few years
             after the intervention because of reductions in other
             service use and associated costs.Early Characteristics of
             Autism; http://clinicaltrials.gov/; NCT0009415.},
   Doi = {10.1016/j.jaac.2017.06.007},
   Key = {fds329122}
}

@article{fds327660,
   Author = {Weiner, DJ and Wigdor, EM and Ripke, S and Walters, RK and Kosmicki, JA and Grove, J and Samocha, KE and Goldstein, JI and Okbay, A and Bybjerg-Grauholm, J and Werge, T and Hougaard, DM and Taylor, J and iPSYCH-Broad Autism Group, and Psychiatric Genomics Consortium
             Autism Group, and Skuse, D and Devlin, B and Anney, R and Sanders, SJ and Bishop, S and Mortensen, PB and Børglum, AD and Smith, GD and Daly, MJ and Robinson, EB},
   Title = {Polygenic transmission disequilibrium confirms that common
             and rare variation act additively to create risk for autism
             spectrum disorders.},
   Journal = {Nature Genetics},
   Volume = {49},
   Number = {7},
   Pages = {978-985},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1038/ng.3863},
   Abstract = {Autism spectrum disorder (ASD) risk is influenced by common
             polygenic and de novo variation. We aimed to clarify the
             influence of polygenic risk for ASD and to identify
             subgroups of ASD cases, including those with strongly acting
             de novo variants, in which polygenic risk is relevant. Using
             a novel approach called the polygenic transmission
             disequilibrium test and data from 6,454 families with a
             child with ASD, we show that polygenic risk for ASD,
             schizophrenia, and greater educational attainment is
             over-transmitted to children with ASD. These findings hold
             independent of proband IQ. We find that polygenic variation
             contributes additively to risk in ASD cases who carry a
             strongly acting de novo variant. Lastly, we show that
             elements of polygenic risk are independent and differ in
             their relationship with phenotype. These results confirm
             that the genetic influences on ASD are additive and suggest
             that they create risk through at least partially distinct
             etiologic pathways.},
   Doi = {10.1038/ng.3863},
   Key = {fds327660}
}

@article{fds326792,
   Author = {Dawson, G},
   Title = {Questions Remain Regarding the Effectiveness of Many
             Commonly Used Autism Treatments.},
   Journal = {Pediatrics},
   Volume = {139},
   Number = {6},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1542/peds.2017-0730},
   Doi = {10.1542/peds.2017-0730},
   Key = {fds326792}
}

@article{fds325854,
   Author = {Jones, EJH and Dawson, G and Kelly, J and Estes, A and Jane Webb,
             S},
   Title = {Parent-delivered early intervention in infants at risk for
             ASD: Effects on electrophysiological and habituation
             measures of social attention.},
   Journal = {Autism Research},
   Volume = {10},
   Number = {5},
   Pages = {961-972},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1002/aur.1754},
   Abstract = {Prospective longitudinal studies of infants with older
             siblings with autism spectrum disorder (ASD) have indicated
             that differences in the neurocognitive systems underlying
             social attention may emerge prior to the child meeting ASD
             diagnostic criteria. Thus, targeting social attention with
             early intervention might have the potential to alter
             developmental trajectories for infants at high risk for ASD.
             Electrophysiological and habituation measures of social
             attention were collected at 6, 12, and 18 months in a group
             of high-risk infant siblings of children with ASD
             (N = 33). Between 9 and 11 months of age, infant
             siblings received a parent-delivered intervention, promoting
             first relationships (PFR), (n = 19) or on-going
             assessment without intervention (n = 14). PFR has been
             previously shown to increase parental responsivity to infant
             social communicative cues and infant contingent responding.
             Compared to infants who only received assessment and
             monitoring, infants who received the intervention showed
             improvements in neurocognitive metrics of social attention,
             as reflected in a greater reduction in habituation times to
             face versus object stimuli between 6 and 12 months,
             maintained at 18 months; a greater increase in frontal EEG
             theta power between 6 and 12 months; and a more comparable
             P400 response to faces and objects at 12 months. The
             high-risk infants who received the intervention showed a
             pattern of responses that appeared closer to the normative
             responses of two groups of age-matched low-risk control
             participants. Though replication is necessary, these results
             suggest that early parent-mediated intervention has the
             potential to impact the brain systems underpinning social
             attention in infants at familial risk for ASD. Autism Res
             2017, 10: 961-972. © 2017 International Society for Autism
             Research, Wiley Periodicals, Inc.},
   Doi = {10.1002/aur.1754},
   Key = {fds325854}
}

@article{fds326349,
   Author = {Dawson, G and Sun, JM and Davlantis, KS and Murias, M and Franz, L and Troy, J and Simmons, R and Sabatos-DeVito, M and Durham, R and Kurtzberg, J},
   Title = {Autologous Cord Blood Infusions Are Safe and Feasible in
             Young Children with Autism Spectrum Disorder: Results of a
             Single-Center Phase I Open-Label Trial.},
   Journal = {Stem Cells Translational Medicine},
   Volume = {6},
   Number = {5},
   Pages = {1332-1339},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1002/sctm.16-0474},
   Abstract = {Despite advances in early diagnosis and behavioral
             therapies, more effective treatments for children with
             autism spectrum disorder (ASD) are needed. We hypothesized
             that umbilical cord blood-derived cell therapies may have
             potential in alleviating ASD symptoms by modulating
             inflammatory processes in the brain. Accordingly, we
             conducted a phase I, open-label trial to assess the safety
             and feasibility of a single intravenous infusion of
             autologous umbilical cord blood, as well as sensitivity to
             change in several ASD assessment tools, to determine
             suitable endpoints for future trials. Twenty-five children,
             median age 4.6 years (range 2.26-5.97), with a confirmed
             diagnosis of ASD and a qualified banked autologous umbilical
             cord blood unit, were enrolled. Children were evaluated with
             a battery of behavioral and functional tests immediately
             prior to cord blood infusion (baseline) and 6 and 12 months
             later. Assessment of adverse events across the 12-month
             period indicated that the treatment was safe and well
             tolerated. Significant improvements in children's behavior
             were observed on parent-report measures of social
             communication skills and autism symptoms, clinician ratings
             of overall autism symptom severity and degree of
             improvement, standardized measures of expressive vocabulary,
             and objective eye-tracking measures of children's attention
             to social stimuli, indicating that these measures may be
             useful endpoints in future studies. Behavioral improvements
             were observed during the first 6 months after infusion and
             were greater in children with higher baseline nonverbal
             intelligence quotients. These data will serve as the basis
             for future studies to determine the efficacy of umbilical
             cord blood infusions in children with ASD. Stem Cells
             Translational Medicine 2017;6:1332-1339.},
   Doi = {10.1002/sctm.16-0474},
   Key = {fds326349}
}

@article{fds324049,
   Author = {Campbell, K and Carpenter, KLH and Espinosa, S and Hashemi, J and Qiu,
             Q and Tepper, M and Calderbank, R and Sapiro, G and Egger, HL and Baker,
             JP and Dawson, G},
   Title = {Use of a Digital Modified Checklist for Autism in Toddlers -
             Revised with Follow-up to Improve Quality of Screening for
             Autism.},
   Journal = {The Journal of Pediatrics},
   Volume = {183},
   Pages = {133-139.e1},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jpeds.2017.01.021},
   Abstract = {To assess changes in quality of care for children at risk
             for autism spectrum disorders (ASD) due to process
             improvement and implementation of a digital screening
             form.The process of screening for ASD was studied in an
             academic primary care pediatrics clinic before and after
             implementation of a digital version of the Modified
             Checklist for Autism in Toddlers - Revised with Follow-up
             with automated risk assessment. Quality metrics included
             accuracy of documentation of screening results and
             appropriate action for positive screens (secondary screening
             or referral). Participating physicians completed pre- and
             postintervention surveys to measure changes in attitudes
             toward feasibility and value of screening for ASD. Evidence
             of change was evaluated with statistical process control
             charts and χ2 tests.Accurate documentation in the
             electronic health record of screening results increased from
             54% to 92% (38% increase, 95% CI 14%-64%) and appropriate
             action for children screening positive increased from 25% to
             85% (60% increase, 95% CI 35%-85%). A total of 90% of
             participating physicians agreed that the transition to a
             digital screening form improved their clinical assessment of
             autism risk.Implementation of a tablet-based digital version
             of the Modified Checklist for Autism in Toddlers - Revised
             with Follow-up led to improved quality of care for children
             at risk for ASD and increased acceptability of screening for
             ASD. Continued efforts towards improving the process of
             screening for ASD could facilitate rapid, early diagnosis of
             ASD and advance the accuracy of studies of the impact of
             screening.},
   Doi = {10.1016/j.jpeds.2017.01.021},
   Key = {fds324049}
}

@article{fds329542,
   Author = {Ness, SL and Manyakov, NV and Bangerter, A and Lewin, D and Jagannatha,
             S and Boice, M and Skalkin, A and Dawson, G and Janvier, YM and Goodwin,
             MS and Hendren, R and Leventhal, B and Shic, F and Cioccia, W and Pandina,
             G},
   Title = {JAKE® Multimodal Data Capture System: Insights from an
             Observational Study of Autism Spectrum Disorder.},
   Journal = {Frontiers in Neuroscience},
   Volume = {11},
   Pages = {517},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fnins.2017.00517},
   Abstract = {Objective: To test usability and optimize the Janssen Autism
             Knowledge Engine (JAKE®) system's components, biosensors,
             and procedures used for objective measurement of core and
             associated symptoms of autism spectrum disorder (ASD) in
             clinical trials. Methods: A prospective, observational study
             of 29 children and adolescents with ASD using the JAKE
             system was conducted at three sites in the United States.
             This study was designed to establish the feasibility of the
             JAKE system and to learn practical aspects of its
             implementation. In addition to information collected by web
             and mobile components, wearable biosensor data were
             collected both continuously in natural settings and
             periodically during a battery of experimental tasks
             administered in laboratory settings. This study is
             registered at clinicaltrials.gov, NCT02299700. Results:
             Feedback collected throughout the study allowed future
             refinements to be planned for all components of the system.
             The Autism Behavior Inventory (ABI), a parent-reported
             measure of ASD core and associated symptoms, performed well.
             Among biosensors studied, the eye-tracker, sleep monitor,
             and electrocardiogram were shown to capture high quality
             data, whereas wireless electroencephalography was difficult
             to use due to its form factor. On an exit survey, the
             majority of parents rated their overall reaction to JAKE as
             positive/very positive. No significant device-related events
             were reported in the study. Conclusion: The results of this
             study, with the described changes, demonstrate that the JAKE
             system is a viable, useful, and safe platform for use in
             clinical trials of ASD, justifying larger validation and
             deployment studies of the optimized system.},
   Doi = {10.3389/fnins.2017.00517},
   Key = {fds329542}
}

@article{fds333740,
   Author = {Li, Y and Murias, M and Major, S and Dawson, G and Dzirasa, K and Carin, L and Carlson, DE},
   Title = {Targeting EEG/LFP Synchrony with Neural Nets.},
   Journal = {NIPS},
   Volume = {2017-December},
   Pages = {4623-4633},
   Editor = {Guyon, I and Luxburg, UV and Bengio, S and Wallach, HM and Fergus, R and Vishwanathan, SVN and Garnett, R},
   Year = {2017},
   Abstract = {© 2017 Neural information processing systems foundation.
             All rights reserved. We consider the analysis of
             Electroencephalography (EEG) and Local Field Potential (LFP)
             datasets, which are "big" in terms of the size of recorded
             data but rarely have sufficient labels required to train
             complex models (e.g., conventional deep learning methods).
             Furthermore, in many scientific applications, the goal is to
             be able to understand the underlying features related to the
             classification, which prohibits the blind application of
             deep networks. This motivates the development of a new model
             based on parameterized convolutional filters guided by
             previous neuroscience research; the filters learn relevant
             frequency bands while targeting synchrony, which are
             frequency-specific power and phase correlations between
             electrodes. This results in a highly expressive
             convolutional neural network with only a few hundred
             parameters, applicable to smaller datasets. The proposed
             approach is demonstrated to yield competitive (often
             state-of-the-art) predictive performance during our
             empirical tests while yielding interpretable features.
             Furthermore, a Gaussian process adapter is developed to
             combine analysis over distinct electrode layouts, allowing
             the joint processing of multiple datasets to address
             overfitting and improve generalizability. Finally, it is
             demonstrated that the proposed framework effectively tracks
             neural dynamics on children in a clinical trial on Autism
             Spectrum Disorder.},
   Key = {fds333740}
}


%% De Bellis, Michael D.   
@article{fds336066,
   Author = {Sun, D and Haswell, CC and Morey, RA and De Bellis,
             MD},
   Title = {Brain structural covariance network centrality in maltreated
             youth with PTSD and in maltreated youth resilient to
             PTSD.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1017/s0954579418000093},
   Abstract = {Child maltreatment is a major cause of pediatric
             posttraumatic stress disorder (PTSD). Previous studies have
             not investigated potential differences in network
             architecture in maltreated youth with PTSD and those
             resilient to PTSD. High-resolution magnetic resonance
             imaging brain scans at 3 T were completed in maltreated
             youth with PTSD (n = 31), without PTSD (n = 32), and
             nonmaltreated controls (n = 57). Structural covariance
             network architecture was derived from between-subject
             intraregional correlations in measures of cortical thickness
             in 148 cortical regions (nodes). Interregional positive
             partial correlations controlling for demographic variables
             were assessed, and those correlations that exceeded
             specified thresholds constituted connections in cortical
             brain networks. Four measures of network centrality
             characterized topology, and the importance of cortical
             regions (nodes) within the network architecture were
             calculated for each group. Permutation testing and principle
             component analysis method were employed to calculate
             between-group differences. Principle component analysis is a
             methodological improvement to methods used in previous brain
             structural covariance network studies. Differences in
             centrality were observed between groups. Larger centrality
             was found in maltreated youth with PTSD in the right
             posterior cingulate cortex; smaller centrality was detected
             in the right inferior frontal cortex compared to youth
             resilient to PTSD and controls, demonstrating network
             characteristics unique to pediatric maltreatment-related
             PTSD. Larger centrality was detected in right frontal pole
             in maltreated youth resilient to PTSD compared to youth with
             PTSD and controls, demonstrating structural covariance
             network differences in youth resilience to PTSD following
             maltreatment. Smaller centrality was found in the left
             posterior cingulate cortex and in the right inferior frontal
             cortex in maltreated youth compared to controls,
             demonstrating attributes of structural covariance network
             topology that is unique to experiencing maltreatment. This
             work is the first to identify cortical thickness-based
             structural covariance network differences between maltreated
             youth with and without PTSD. We demonstrated network
             differences in both networks unique to maltreated youth with
             PTSD and those resilient to PTSD. The networks identified
             are important for the successful attainment of
             age-appropriate social cognition, attention, emotional
             processing, and inhibitory control. Our findings in
             maltreated youth with PTSD versus those without PTSD suggest
             vulnerability mechanisms for developing PTSD.},
   Doi = {10.1017/s0954579418000093},
   Key = {fds336066}
}

@article{fds333815,
   Author = {Pfefferbaum, A and Kwon, D and Brumback, T and Thompson, WK and Cummins,
             K and Tapert, SF and Brown, SA and Colrain, IM and Baker, FC and Prouty, D and De Bellis, MD and Clark, DB and Nagel, BJ and Chu, W and Park, SH and Pohl,
             KM and Sullivan, EV},
   Title = {Altered Brain Developmental Trajectories in Adolescents
             After Initiating Drinking.},
   Journal = {The American Journal of Psychiatry},
   Volume = {175},
   Number = {4},
   Pages = {370-380},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1176/appi.ajp.2017.17040469},
   Abstract = {The authors sought evidence for altered adolescent brain
             growth trajectory associated with moderate and heavy alcohol
             use in a large national, multisite, prospective study of
             adolescents before and after initiation of appreciable
             alcohol use.This study examined 483 adolescents (ages 12-21)
             before initiation of drinking and 1 and 2 years later. At
             the 2-year assessment, 356 participants continued to meet
             the study's no/low alcohol consumption entry criteria, 65
             had initiated moderate drinking, and 62 had initiated heavy
             drinking. MRI was used to quantify regional cortical and
             white matter volumes. Percent change per year (slopes) in
             adolescents who continued to meet no/low criteria served as
             developmental control trajectories against which to compare
             those who initiated moderate or heavy drinking.In no/low
             drinkers, gray matter volume declined throughout adolescence
             and slowed in many regions in later adolescence.
             Complementing gray matter declines, white matter regions
             grew at faster rates at younger ages and slowed toward young
             adulthood. Youths who initiated heavy drinking exhibited an
             accelerated frontal cortical gray matter trajectory,
             divergent from the norm. Although significant effects on
             trajectories were not observed in moderate drinkers, their
             intermediate position between no/low and heavy drinkers
             suggests a dose effect. Neither marijuana co-use nor
             baseline volumes contributed significantly to the alcohol
             effect.Initiation of drinking during adolescence, with or
             without marijuana co-use, disordered normal brain growth
             trajectories. Factors possibly contributing to abnormal
             cortical volume trajectories include peak consumption in the
             past year and family history of alcoholism.},
   Doi = {10.1176/appi.ajp.2017.17040469},
   Key = {fds333815}
}

@article{fds331438,
   Author = {Nooner, KB and Hooper, SR and De Bellis, MD},
   Title = {An Examination of Sex Differences on Neurocognitive
             Functioning and Behavior Problems in Maltreated
             Youth.},
   Journal = {Psychological Trauma : Theory, Research, Practice and
             Policy},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1037/tra0000356},
   Abstract = {In the developmental traumatology model, the biological
             construct of sex is considered a moderator that may
             negatively influence child maltreatment sequelae including
             those pertaining to neurocognitive function.This study
             examined sex-differences in neurocognitive function and
             behavior problems in maltreated boys (n = 42), maltreated
             girls (n = 56) versus nonmaltreated boys (n = 45) and girls
             (n = 59). Maltreated boys were hypothesized to have poorer
             neurocognitive functioning than maltreated girls, and
             nonmaltreated boys and girls, in all neurocognitive domains,
             particularly pertaining to executive function and attention.
             We also examined correlations between cognitive function and
             parent report of child behavior problems for maltreated and
             nonmaltreated children.Maltreated boys performed more poorly
             on measures of intelligence, attention, language, memory,
             executive function, and academic achievement in both reading
             and math than nonmaltreated boys. Maltreated boys did not
             perform more poorly on these cognitive measures or
             behavioral measures than maltreated girls, except for one
             memory measure. Maltreated girls performed more poorly on
             measures of intelligence, language, memory, executive
             function, and academic achievement than nonmaltreated girls.
             Maltreated girls with better visual-spatial skills had more
             internalizing and externalizing problems. Effect sizes for
             these sex differences ranged from small to large.Both
             maltreated boys and girls showed poorer cognitive function
             than their nonmaltreated sex-matched controls. Maltreated
             girls had subtle sparing of attention and short-term memory
             (STM). Understanding sex differences in neurocognitive
             functioning may have implications for designing large
             population studies of maltreated youth. (PsycINFO Database
             Record},
   Doi = {10.1037/tra0000356},
   Key = {fds331438}
}

@misc{fds332915,
   Author = {Tervo-Clemmens, B and Quach, A and Luna, B and Foran, W and Chung, T and De
             Bellis, MD and Clark, DB},
   Title = {Neural correlates of rewarded response inhibition in youth
             at risk for problematic alcohol use},
   Journal = {Frontiers in Behavioral Neuroscience},
   Volume = {11},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.3389/fnbeh.2017.00205},
   Abstract = {© 2017 Tervo-Clemmens, Quach, Luna, Foran, Chung, De Bellis
             and Clark. Risk for substance use disorder (SUD) is
             associated with poor response inhibition and heightened
             reward sensitivity. During adolescence, incentives improve
             performance on response inhibition tasks and increase
             recruitment of cortical control areas (Geier et al., 2010)
             associated with SUD (Chung et al., 2011). However, it is
             unknown whether incentives moderate the relationship between
             response inhibition and trait-level psychopathology and
             personality features of substance use risk. We examined
             these associations in the current project using a rewarded
             antisaccade (AS) task (Geier et al., 2010) in youth at risk
             for substance use. Participants were 116 adolescents and
             young adults (ages 12–21) from the University of
             Pittsburgh site of the National Consortium on Adolescent
             Neurodevelopment and Alcohol [NCANDA] study, with
             neuroimaging data collected at baseline and 1 year follow up
             visits. Building upon previous work using this task in
             normative developmental samples (Geier et al., 2010) and
             adolescents with SUD (Chung et al., 2011), we examined both
             trial-wise BOLD responses and those associated with
             individual task-epochs (cue presentation, response
             preparation, and response) and associated them with multiple
             substance use risk factors (externalizing and internalizing
             psychopathology, family history of substance use, and trait
             impulsivity). Results showed that externalizing
             psychopathology and high levels of trait impulsivity
             (positive urgency, SUPPS-P) were associated with general
             decreases in antisaccade performance. Accompanying this main
             effect of poor performance, positive urgency was associated
             with reduced recruitment of the frontal eye fields (FEF) and
             inferior frontal gyrus (IFG) in both a priori regions of
             interest and at the voxelwise level. Consistent with
             previous work, monetary incentive improved antisaccade
             behavioral performance and was associated with increased
             activation in the striatum and cortical control areas.
             However, incentives did not moderate the association between
             response inhibition behavioral performance and any
             trait-level psychopathology and personality factor of
             substance use risk. Reward interactions were observed for
             BOLD responses at the task-epoch level, however, they were
             inconsistent across substance use risk types. The results
             from this study may suggest poor response inhibition and
             heightened reward sensitivity are not overlapping
             neurocognitive features of substance use risk.
             Alternatively, more subtle, common longitudinal processes
             might jointly explain reward sensitivity and response
             inhibition deficits in substance use risk.},
   Doi = {10.3389/fnbeh.2017.00205},
   Key = {fds332915}
}

@article{fds330475,
   Author = {Sullivan, EV and Lane, B and Kwon, D and Meloy, MJ and Tapert, SF and Brown, SA and Colrain, IM and Baker, FC and De Bellis, MD and Clark, DB and Nagel, BJ and Pohl, KM and Pfefferbaum, A},
   Title = {Structural brain anomalies in healthy adolescents in the
             NCANDA cohort: relation to neuropsychological test
             performance, sex, and ethnicity.},
   Journal = {Brain Imaging and Behavior},
   Volume = {11},
   Number = {5},
   Pages = {1302-1315},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1007/s11682-016-9634-2},
   Abstract = {Structural MRI of volunteers deemed "normal" following
             clinical interview provides a window into normal brain
             developmental morphology but also reveals unexpected
             dysmorphology, commonly known as "incidental findings."
             Although unanticipated, these anatomical findings raise
             questions regarding possible treatment that could even
             ultimately require neurosurgical intervention, which itself
             carries significant risk but may not be indicated if the
             anomaly is nonprogressive or of no functional consequence.
             Neuroradiological readings of 833 structural MRI from the
             National Consortium on Alcohol and NeuroDevelopment in
             Adolescence (NCANDA) cohort found an 11.8 % incidence of
             brain structural anomalies, represented proportionately
             across the five collection sites and ethnic groups.
             Anomalies included 26 mega cisterna magna, 15 subarachnoid
             cysts, 12 pineal cysts, 12 white matter dysmorphologies, 5
             tonsillar ectopias, 5 prominent perivascular spaces, 5 gray
             matter heterotopias, 4 pituitary masses, 4 excessively large
             or asymmetrical ventricles, 4 cavum septum pellucidum, 3
             developmental venous anomalies, 1 exceptionally large
             midsagittal vein, and single cases requiring clinical
             followup: cranio-cervical junction stenosis, parietal
             cortical mass, and Chiari I malformation. A case of possible
             demyelinating disorder (e.g., neuromyelitis optica or
             multiple sclerosis) newly emerged at the 1-year NCANDA
             followup, requiring clinical referral. Comparing test
             performance of the 98 anomalous cases with 619 anomaly-free
             no-to-low alcohol consuming adolescents revealed
             significantly lower scores on speed measures of attention
             and motor functions; these differences were not attributed
             to any one anomaly subgroup. Further, we devised an
             automated approach for quantifying posterior fossa CSF
             volumes for detection of mega cisterna magna, which
             represented 26.5 % of clinically identified anomalies.
             Automated quantification fit a Gaussian distribution with a
             rightward skew. Using a 3SD cut-off, quantification
             identified 22 of the 26 clinically-identified cases,
             indicating that cases with percent of CSF in the
             posterior-inferior-middle aspect of the posterior fossa
             ≥3SD merit further review, and support complementing
             clinical readings with objective quantitative analysis.
             Discovery of asymptomatic brain structural anomalies, even
             when no clinical action is indicated, can be disconcerting
             to the individual and responsible family members, raising a
             disclosure dilemma: refrain from relating the incidental
             findings to avoid unnecessary alarm or anxiety; or
             alternatively, relate the neuroradiological findings as
             "normal variants" to the study volunteers and family,
             thereby equipping them with knowledge for the future should
             they have the occasion for a brain scan following an illness
             or accident that the incidental findings predated the later
             event.},
   Doi = {10.1007/s11682-016-9634-2},
   Key = {fds330475}
}

@article{fds326501,
   Author = {Hasler, BP and Franzen, PL and de Zambotti, M and Prouty, D and Brown,
             SA and Tapert, SF and Pfefferbaum, A and Pohl, KM and Sullivan, EV and De
             Bellis, MD and Nagel, BJ and Baker, FC and Colrain, IM and Clark,
             DB},
   Title = {Eveningness and Later Sleep Timing Are Associated with
             Greater Risk for Alcohol and Marijuana Use in Adolescence:
             Initial Findings from the National Consortium on Alcohol and
             Neurodevelopment in Adolescence Study.},
   Journal = {Alcoholism, Clinical and Experimental Research},
   Volume = {41},
   Number = {6},
   Pages = {1154-1165},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1111/acer.13401},
   Abstract = {Abundant cross-sectional evidence links eveningness (a
             preference for later sleep-wake timing) and increased
             alcohol and drug use among adolescents and young adults.
             However, longitudinal studies are needed to examine whether
             eveningness is a risk factor for subsequent alcohol and drug
             use, particularly during adolescence, which is marked by
             parallel peaks in eveningness and risk for the onset of
             alcohol use disorders. This study examined whether
             eveningness and other sleep characteristics were associated
             with concurrent or subsequent substance involvement in a
             longitudinal study of adolescents.Participants were 729
             adolescents (368 females; age 12 to 21 years) in the
             National Consortium on Alcohol and Neurodevelopment in
             Adolescence study. Associations between the sleep variables
             (circadian preference, sleep quality, daytime sleepiness,
             sleep timing, and sleep duration) and 3 categorical
             substance variables (at-risk alcohol use, alcohol bingeing,
             and past-year marijuana use [y/n]) were examined using
             ordinal and logistic regression with baseline age, sex,
             race, ethnicity, socioeconomic status, and psychiatric
             problems as covariates.At baseline, greater eveningness was
             associated with greater at-risk alcohol use, greater
             bingeing, and past-year use of marijuana. Later weekday and
             weekend bedtimes, but not weekday or weekend sleep duration,
             showed similar associations across the 3 substance outcomes
             at baseline. Greater baseline eveningness was also
             prospectively associated with greater bingeing and past-year
             use of marijuana at the 1-year follow-up, after covarying
             for baseline bingeing and marijuana use. Later baseline
             weekday and weekend bedtimes, and shorter baseline weekday
             sleep duration, were similarly associated with greater
             bingeing and past-year use of marijuana at the 1-year
             follow-up after covarying for baseline values.Findings
             suggest that eveningness and sleep timing may be under
             recognized risk factors and future areas of intervention for
             adolescent involvement in alcohol and marijuana that should
             be considered along with other previously identified sleep
             factors such as insomnia and insufficient
             sleep.},
   Doi = {10.1111/acer.13401},
   Key = {fds326501}
}

@article{fds325128,
   Author = {Sullivan, EV and Brumback, T and Tapert, SF and Prouty, D and Fama, R and Thompson, WK and Brown, SA and Cummins, K and Colrain, IM and Baker, FC and Clark, DB and Chung, T and De Bellis, MD and Hooper, SR and Nagel, BJ and Nichols, BN and Chu, W and Kwon, D and Pohl, KM and Pfefferbaum,
             A},
   Title = {Effects of prior testing lasting a full year in NCANDA
             adolescents: Contributions from age, sex, socioeconomic
             status, ethnicity, site, family history of alcohol or drug
             abuse, and baseline performance.},
   Journal = {Developmental Cognitive Neuroscience},
   Volume = {24},
   Pages = {72-83},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.dcn.2017.01.003},
   Abstract = {Longitudinal study provides a robust method for tracking
             developmental trajectories. Yet inherent problems of
             retesting pose challenges in distinguishing biological
             developmental change from prior testing experience. We
             examined factors potentially influencing change scores on 16
             neuropsychological test composites over 1year in 568
             adolescents in the National Consortium on Alcohol and
             NeuroDevelopment in Adolescence (NCANDA) project. The
             twice-minus-once-tested method revealed that performance
             gain was mainly attributable to testing experience
             (practice) with little contribution from predicted
             developmental effects. Group mean practice slopes for 13
             composites indicated that 60% to ∼100% variance was
             attributable to test experience; General Ability accuracy
             showed the least practice effect (29%). Lower baseline
             performance, especially in younger participants, was a
             strong predictor of greater gain. Contributions from age,
             sex, ethnicity, examination site, socioeconomic status, or
             family history of alcohol/substance abuse were nil to small,
             even where statistically significant. Recognizing that a
             substantial proportion of change in longitudinal testing,
             even over 1-year, is attributable to testing experience
             indicates caution against assuming that performance gain
             observed during periods of maturation necessarily reflects
             development. Estimates of testing experience, a form of
             learning, may be a relevant metric for detecting interim
             influences, such as alcohol use or traumatic episodes, on
             behavior.},
   Doi = {10.1016/j.dcn.2017.01.003},
   Key = {fds325128}
}

@article{fds330476,
   Author = {Stave, EA and De Bellis, MD and Hooper, SR and Woolley, DP and Chang,
             SK and Chen, SD},
   Title = {Dimensions of Attention Associated With the Microstructure
             of Corona Radiata White Matter.},
   Journal = {Journal of Child Neurology},
   Volume = {32},
   Number = {5},
   Pages = {458-466},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1177/0883073816685652},
   Abstract = {Mirsky proposed a model of attention that included these
             dimensions: focus/execute, sustain, stabilize, encode, and
             shift. The neural correlates of these dimensions were
             investigated within corona radiata subregions in healthy
             youth. Diffusion tensor imaging and neuropsychological
             assessments were conducted in 79 healthy, right-handed youth
             aged 4-17 years. Diffusion tensor imaging maps were analyzed
             using standardized parcellation methods. Partial Pearson
             correlations between neuropsychological standardized scores,
             representing these attention dimensions, and diffusion
             tensor imaging measures of corona radiata subregions were
             calculated after adjusting for gender and IQ. Significant
             correlations were found between the focus/execute, sustain,
             stabilize, and shift dimensions and imaging metrics in
             hypothesized corona radiata subregions. Results suggest that
             greater microstructural white matter integrity of the corona
             radiata is partly associated with attention across 4
             attention dimensions. Findings suggest that white matter
             microstructure of the corona radiata is a neural correlate
             of several, but not all, attention dimensions.},
   Doi = {10.1177/0883073816685652},
   Key = {fds330476}
}

@article{fds326856,
   Author = {Sege, RD and Amaya-Jackson, L and AMERICAN ACADEMY OF PEDIATRICS
             Committee on Child Abuse and Neglect, Council on Foster
             Care and Adoption and Kinship Care and AMERICAN ACADEMY OF CHILD
             AND ADOLESCENT PSYCHIATRY Committee on Child Maltreatment and Violence and NATIONAL CENTER FOR CHILD TRAUMATIC
             STRESS},
   Title = {Clinical Considerations Related to the Behavioral
             Manifestations of Child Maltreatment.},
   Journal = {Pediatrics},
   Volume = {139},
   Number = {4},
   Pages = {e20170100-e20170100},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1542/peds.2017-0100},
   Abstract = {Children who have suffered early abuse or neglect may later
             present with significant health and behavior problems that
             may persist long after the abusive or neglectful environment
             has been remediated. Neurobiological research suggests that
             early maltreatment may result in an altered psychological
             and physiologic response to stressful stimuli, a response
             that deleteriously affects the child's subsequent
             development. Pediatricians can assist caregivers by helping
             them recognize the abused or neglected child's emotional and
             behavioral responses associated with child maltreatment and
             guide them in the use of positive parenting strategies,
             referring the children and families to evidence-based
             therapeutic treatment and mobilizing available community
             resources.},
   Doi = {10.1542/peds.2017-0100},
   Key = {fds326856}
}

@article{fds332916,
   Author = {Clark, DB and Chung, T and Martin, CS and Hasler, BP and Fitzgerald, DH and Luna, B and Brown, SA and Tapert, SF and Brumback, T and Cummins, K and Pfefferbaum, A and Sullivan, EV and Pohl, KM and Colrain, IM and Baker,
             FC and De Bellis, MD and Nooner, KB and Nagel, BJ},
   Title = {Adolescent Executive Dysfunction in Daily Life:
             Relationships to Risks, Brain Structure and Substance
             Use.},
   Journal = {Frontiers in Behavioral Neuroscience},
   Volume = {11},
   Pages = {223},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fnbeh.2017.00223},
   Abstract = {During adolescence, problems reflecting cognitive,
             behavioral and affective dysregulation, such as inattention
             and emotional dyscontrol, have been observed to be
             associated with substance use disorder (SUD) risks and
             outcomes. Prior studies have typically been with small
             samples, and have typically not included comprehensive
             measurement of executive dysfunction domains. The
             relationships of executive dysfunction in daily life with
             performance based testing of cognitive skills and structural
             brain characteristics, thought to be the basis for executive
             functioning, have not been definitively determined. The aims
             of this study were to determine the relationships between
             executive dysfunction in daily life, measured by the
             Behavior Rating Inventory of Executive Function (BRIEF),
             cognitive skills and structural brain characteristics, and
             SUD risks, including a global SUD risk indicator, sleep
             quality, and risky alcohol and cannabis use. In addition to
             bivariate relationships, multivariate models were tested.
             The subjects (n = 817; ages 12 through 21) were participants
             in the National Consortium on Alcohol and Neurodevelopment
             in Adolescence (NCANDA) study. The results indicated that
             executive dysfunction was significantly related to SUD
             risks, poor sleep quality, risky alcohol use and cannabis
             use, and was not significantly related to cognitive skills
             or structural brain characteristics. In multivariate models,
             the relationship between poor sleep quality and risky
             substance use was mediated by executive dysfunction. While
             these cross-sectional relationships need to be further
             examined in longitudinal analyses, the results suggest that
             poor sleep quality and executive dysfunction may be viable
             preventive intervention targets to reduce adolescent
             substance use.},
   Doi = {10.3389/fnbeh.2017.00223},
   Key = {fds332916}
}


%% De Brigard, Felipe   
@article{fds335558,
   Author = {Parikh, N and Ruzic, L and Stewart, GW and Spreng, RN and De Brigard,
             F},
   Title = {What if? Neural activity underlying semantic and episodic
             counterfactual thinking},
   Journal = {Neuroimage},
   Volume = {178},
   Pages = {332-345},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2018.05.053},
   Doi = {10.1016/j.neuroimage.2018.05.053},
   Key = {fds335558}
}

@article{fds336415,
   Author = {Stanley, ML and Yang, BW and De Brigard and F},
   Title = {No evidence for unethical amnesia for imagined actions: A
             failed replication and extension},
   Journal = {Memory & Cognition},
   Volume = {46},
   Number = {5},
   Pages = {787-795},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.3758/s13421-018-0803-y},
   Doi = {10.3758/s13421-018-0803-y},
   Key = {fds336415}
}

@article{fds335559,
   Author = {De Brigard and F and Hanna, E and St Jacques and PL and Schacter,
             DL},
   Title = {How thinking about what could have been affects how we feel
             about what was},
   Journal = {Cognition and Emotion},
   Pages = {1-14},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1080/02699931.2018.1478280},
   Doi = {10.1080/02699931.2018.1478280},
   Key = {fds335559}
}

@article{fds335560,
   Author = {De Brigard and F and Brady, WJ},
   Title = {Correction to: The Effect of What We Think may Happen on our
             Judgments of Responsibility},
   Journal = {Review of Philosophy and Psychology},
   Volume = {9},
   Number = {2},
   Pages = {447-447},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s13164-018-0389-0},
   Doi = {10.1007/s13164-018-0389-0},
   Key = {fds335560}
}

@article{fds335561,
   Author = {De Freitas and J and Sarkissian, H and Newman, GE and Grossmann, I and De
             Brigard, F and Luco, A and Knobe, J},
   Title = {Consistent Belief in a Good True Self in Misanthropes and
             Three Interdependent Cultures},
   Journal = {Cognitive Science},
   Volume = {42},
   Pages = {134-160},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1111/cogs.12505},
   Doi = {10.1111/cogs.12505},
   Key = {fds335561}
}

@article{fds332864,
   Author = {Murray, S and Murray, ED and Stewart, G and Sinnott-Armstrong, W and de
             Brigard, F},
   Title = {Responsibility for forgetting},
   Journal = {Philosophical Studies},
   Pages = {1-25},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1007/s11098-018-1053-3},
   Abstract = {© 2018 Springer Science+Business Media B.V., part of
             Springer Nature In this paper, we focus on whether and to
             what extent we judge that people are responsible for the
             consequences of their forgetfulness. We ran a series of
             behavioral studies to measure judgments of responsibility
             for the consequences of forgetfulness. Our results show that
             we are disposed to hold others responsible for some of their
             forgetfulness. The level of stress that the forgetful agent
             is under modulates judgments of responsibility, though the
             level of care that the agent exhibits toward performing the
             forgotten action does not. We argue that this result has
             important implications for a long-running debate about the
             nature of responsible agency.},
   Doi = {10.1007/s11098-018-1053-3},
   Key = {fds332864}
}

@article{fds337053,
   Author = {De Brigard and F},
   Title = {Memory and the intentional stance},
   Pages = {62-91},
   Booktitle = {The Philosophy of Daniel Dennett},
   Year = {2018},
   Month = {February},
   ISBN = {9780199367511},
   url = {http://dx.doi.org/10.1093/oso/9780199367511.003.0005},
   Abstract = {© Oxford University Press 2018. All rights reserved.
             Despite Dennett's vast scholarship, he seemed to only have
             directly addressed the topic of memory in a relatively
             unknown coauthored article published in a somewhat obscure
             volume. The current chapter attempts to reconstruct the
             ideas from this old article, and argues that it offers a
             viable and coherent view of episodic memory with substantial
             empirical support. Specifically, the chapter uncovers three
             empirically supported theses. A functional thesis, according
             to which our memory system not only processes information
             about past events but also uses this information to
             construct useful anticipations of possible future events. A
             computational thesis, according to which statistical
             regularities, along with individual limitations and goals,
             probabilistically constrain the search space examined during
             memory retrieval. And a metaphysical thesis, according to
             which memories do not exist as subpersonal-level brain
             structures encoding particular intentional contents but
             rather as personal-level psychological phenomena only
             accessible from the intentional stance.},
   Doi = {10.1093/oso/9780199367511.003.0005},
   Key = {fds337053}
}

@article{fds335562,
   Author = {De Brigard and F},
   Title = {Memory, attention, and joint reminiscing},
   Pages = {200-220},
   Booktitle = {New Directions in the Philosophy of Memory},
   Year = {2018},
   Month = {January},
   ISBN = {9781351660020},
   url = {http://dx.doi.org/10.4324/9781315159591},
   Abstract = {© 2018 Taylor & Francis. When people jointly reminisce,
             they often talk about past objects, which may or may no
             longer exist. How can two or more people jointly refer to an
             object that is long gone-or at least, that is not present in
             their surrounding? In this chapter, I offer a three-part
             answer to this question. First, I suggest that our capacity
             to remember intentional objects during memory retrieval
             depends on our capacity to direct our attention inwardly
             toward the relevant component of a memorial content-a mental
             act I call mental ostension. Second, I argue that, for us to
             refer to remembered intentional objects, we must possess the
             ability to refer to them indirectly or “deferredly” by
             way of mentally ostending toward a present mental content;
             in short, we must be capable of deferred mental ostension.
             Finally, I claim that to jointly reminisce, we must have the
             capacity to guide someone else’s attention inwardly toward
             the relevant aspect of the mental content we want them to
             focus on so that they become aware of the past object we are
             deferredly ostending; that is, we need concerted deferred
             mental ostension.},
   Doi = {10.4324/9781315159591},
   Key = {fds335562}
}

@article{fds335563,
   Author = {De Brigard and F and Gessell, B},
   Title = {Why episodic memory may not be for communication},
   Journal = {Behavioral and Brain Sciences},
   Volume = {41},
   Year = {2018},
   url = {http://dx.doi.org/10.1017/S0140525X17001303},
   Doi = {10.1017/S0140525X17001303},
   Key = {fds335563}
}

@article{fds329948,
   Author = {De Brigard and F and Parikh, N and Stewart, GW and Szpunar, KK and Schacter, DL},
   Title = {Neural activity associated with repetitive simulation of
             episodic counterfactual thoughts.},
   Journal = {Neuropsychologia},
   Volume = {106},
   Pages = {123-132},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2017.09.022},
   Abstract = {When people revisit past autobiographical events they often
             imagine alternative ways in which such events could have
             occurred. Often these episodic counterfactual thoughts
             (eCFT) are momentary and fleeting, but sometimes they are
             simulated frequently and repeatedly. However, little is
             known about the neural differences between frequently versus
             infrequently repeated eCFT. The current study explores this
             issue. In a three-session study, participants were asked to
             simulate alternative ways positive, negative, and neutral
             autobiographical memories could have occurred. Half of these
             eCFT were repeatedly re-simulated while the other half were
             not. Immediately after, participants were asked to simulate
             all these eCFT again while undergoing fMRI. A partial least
             squares analysis on the resultant fMRI data revealed that
             eCFT that were not frequently repeated preferentially
             engaged brain regions including middle (BA 21) and superior
             temporal gyri (BA 38/39), middle (BA 11) and superior
             frontal gyri (BA 9), and hippocampus. By contrast,
             frequently repeated eCFT preferentially engaged regions
             including medial frontal gyri (BA 10), anterior cingulate
             cortex, insula, and inferior parietal lobule (BA 40). Direct
             contrasts for each type of eCFT were also conducted. The
             results of these analyses suggest differential contributions
             of regions traditionally associated with eCFT, such as BA
             10, anterior cingulate cortex, and hippocampus, as a
             function of kind of eCFT and frequency of repetition.
             Consequences for future research on eCFT and rumination are
             considered.},
   Doi = {10.1016/j.neuropsychologia.2017.09.022},
   Key = {fds329948}
}

@article{fds329104,
   Author = {Stanley, ML and Dougherty, AM and Yang, BW and Henne, P and De Brigard,
             F},
   Title = {Reasons Probably Won't Change Your Mind: The Role of Reasons
             in Revising Moral Decisions},
   Journal = {Journal of Experimental Psychology. General},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1037/xge0000368},
   Abstract = {© 2017 APA, all rights reserved). Although many
             philosophers argue that making and revising moral decisions
             ought to be a matter of deliberating over reasons, the
             extent to which the consideration of reasons informs
             people's moral decisions and prompts them to change their
             decisions remains unclear. Here, after making an initial
             decision in 2-option moral dilemmas, participants examined
             reasons for only the option initially chosen (affirming
             reasons), reasons for only the option not initially chosen
             (opposing reasons), or reasons for both options. Although
             participants were more likely to change their initial
             decisions when presented with only opposing reasons compared
             with only affirming reasons, these effect sizes were
             consistently small. After evaluating reasons, participants
             were significantly more likely not to change their initial
             decisions than to change them, regardless of the set of
             reasons they considered. The initial decision accounted for
             most of the variance in predicting the final decision,
             whereas the reasons evaluated accounted for a relatively
             small proportion of the variance in predicting the final
             decision. This resistance to changing moral decisions is at
             least partly attributable to a biased, motivated evaluation
             of the available reasons: participants rated the reasons
             supporting their initial decisions more favorably than the
             reasons opposing their initial decisions, regardless of the
             reported strategy used to make the initial decision.
             Overall, our results suggest that the consideration of
             reasons rarely induces people to change their initial
             decisions in moral dilemmas. (PsycINFO Database
             Record},
   Doi = {10.1037/xge0000368},
   Key = {fds329104}
}

@article{fds329949,
   Author = {De Brigard and F},
   Title = {Memory and imagination},
   Pages = {127-140},
   Booktitle = {The Routledge Handbook of Philosophy of Memory},
   Year = {2017},
   Month = {June},
   ISBN = {9781315687315},
   url = {http://dx.doi.org/10.4324/9781315687315},
   Doi = {10.4324/9781315687315},
   Key = {fds329949}
}

@article{fds326194,
   Author = {Stanley, ML and Henne, P and Iyengar, V and Sinnott-Armstrong, W and De
             Brigard, F},
   Title = {I'm not the person I used to be: The self and
             autobiographical memories of immoral actions.},
   Journal = {Journal of Experimental Psychology. General},
   Volume = {146},
   Number = {6},
   Pages = {884-895},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1037/xge0000317},
   Abstract = {People maintain a positive identity in at least two ways:
             They evaluate themselves more favorably than other people,
             and they judge themselves to be better now than they were in
             the past. Both strategies rely on autobiographical memories.
             The authors investigate the role of autobiographical
             memories of lying and emotional harm in maintaining a
             positive identity. For memories of lying to or emotionally
             harming others, participants judge their own actions as less
             morally wrong and less negative than those in which other
             people lied to or emotionally harmed them. Furthermore,
             people judge those actions that happened further in the past
             to be more morally wrong than those that happened more
             recently. Finally, for periods of the past when they
             believed that they were very different people than they are
             now, participants judge their actions to be more morally
             wrong and more negative than those actions from periods of
             their pasts when they believed that they were very similar
             to who they are now. The authors discuss these findings in
             relation to theories about the function of autobiographical
             memory and moral cognition in constructing and perceiving
             the self over time. (PsycINFO Database Record},
   Doi = {10.1037/xge0000317},
   Key = {fds326194}
}

@article{fds327002,
   Author = {De Brigard and F},
   Title = {Cognitive systems and the changing brain},
   Journal = {Philosophical Explorations},
   Volume = {20},
   Number = {2},
   Pages = {224-241},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1080/13869795.2017.1312503},
   Doi = {10.1080/13869795.2017.1312503},
   Key = {fds327002}
}

@article{fds327003,
   Author = {De Brigard and F and Rodriguez, DC and Montañés,
             P},
   Title = {Exploring the experience of episodic past, future, and
             counterfactual thinking in younger and older adults: A study
             of a Colombian sample},
   Journal = {Consciousness and Cognition},
   Volume = {51},
   Pages = {258-267},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.concog.2017.04.007},
   Doi = {10.1016/j.concog.2017.04.007},
   Key = {fds327003}
}

@article{fds318357,
   Author = {Henne, P and Pinillos, Á and De Brigard and F},
   Title = {Cause by Omission and Norm: Not Watering
             Plants},
   Journal = {Australasian Journal of Philosophy},
   Volume = {95},
   Number = {2},
   Pages = {270-283},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1080/00048402.2016.1182567},
   Doi = {10.1080/00048402.2016.1182567},
   Key = {fds318357}
}

@article{fds323231,
   Author = {Stanley, ML and Parikh, N and Stewart, GW and De Brigard,
             F},
   Title = {Emotional intensity in episodic autobiographical memory and
             counterfactual thinking},
   Journal = {Consciousness and Cognition},
   Volume = {48},
   Pages = {283-291},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.concog.2016.12.013},
   Abstract = {© 2016Episodic counterfactual thoughts—imagined
             alternative ways in which personal past events might have
             occurred—are frequently accompanied by intense emotions.
             Here, participants recollected positive and negative
             autobiographical memories and then generated better and
             worse episodic counterfactual events from those memories.
             Our results suggest that the projected emotional intensity
             during the simulated remembered/imagined event is
             significantly higher than but typically positively related
             to the emotional intensity while remembering/imagining the
             event. Furthermore, repeatedly simulating counterfactual
             events heightened the emotional intensity felt while
             simulating the counterfactual event. Finally, for both the
             emotional intensity accompanying the experience of
             remembering/imagining and the projected emotional intensity
             during the simulated remembered/imagined event, the
             emotional intensity of negative memories was greater than
             the emotional intensity of upward counterfactuals generated
             from them but lower than the emotional intensity of downward
             counterfactuals generated from them. These findings are
             discussed in relation to clinical work and functional
             theories of counterfactual thinking.},
   Doi = {10.1016/j.concog.2016.12.013},
   Key = {fds323231}
}

@article{fds323662,
   Author = {De Brigard and F and Brady, TF and Ruzic, L and Schacter,
             DL},
   Title = {Tracking the emergence of memories: A category-learning
             paradigm to explore schema-driven recognition.},
   Journal = {Memory & Cognition},
   Volume = {45},
   Number = {1},
   Pages = {105-120},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.3758/s13421-016-0643-6},
   Abstract = {Previous research has shown that prior knowledge structures
             or schemas affect recognition memory. However, since the
             acquisition of schemas occurs over prolonged periods of
             time, few paradigms allow the direct manipulation of schema
             acquisition to study their effect on memory performance.
             Recently, a number of parallelisms in recognition memory
             between studies involving schemas and studies involving
             category learning have been identified. The current paper
             capitalizes on these findings and offers a novel
             experimental paradigm that allows manipulation of category
             learning between individuals to study the effects of schema
             acquisition on recognition. First, participants learn to
             categorize computer-generated items whose category-inclusion
             criteria differ between participants. Next, participants
             study items that belong to either the learned category, the
             non-learned category, both, or neither. Finally,
             participants receive a recognition test that includes old
             and new items, either from the learned, the non-learned, or
             neither category. Using variations on this paradigm, four
             experiments were conducted. The results from the first three
             studies suggest that learning a category increases hit rates
             for old category-consistent items and false alarm rates for
             new category-consistent lures. Absent the category learning,
             no such effects are evident, even when participants are
             exposed to the same learning trials as those who learned the
             categories. The results from the fourth experiment suggest
             that, at least for false alarm rates, the effects of
             category learning are not solely attributable to frequency
             of occurrence of category-consistent items during learning.
             Implications for recognition memory as well as advantages of
             the proposed paradigm are discussed.},
   Doi = {10.3758/s13421-016-0643-6},
   Key = {fds323662}
}

@article{fds331533,
   Author = {De Brigard and F},
   Title = {Responsibility and the relevance of alternative future
             possibilities},
   Journal = {Teoria},
   Volume = {37},
   Number = {2},
   Pages = {25-35},
   Year = {2017},
   Month = {January},
   Abstract = {In the past decade, philosophical and psychological research
             on people's beliefs about free will and responsibility has
             skyrocketed. For the most part, these vignette-based studies
             have exclusively focused on participants' judgments of the
             causal history of the events leading up to an agent's action
             and considerations about what the agent could have done
             differently in the past. However, recent evidence suggests
             that, when judging whether or not an individual is
             responsible for a certain action - even in concrete,
             emotionally laden and fully deterministic scenarios -
             considerations about alternative future possibilities may
             become relevant. This paper reviews this evidence and
             suggests a way of interpreting the nature of these effects
             as well as some consequences for experimental philosophy and
             psychology of free will and responsibility going
             forward.},
   Key = {fds331533}
}


%% Dodge, Kenneth A.   
@article{fds335169,
   Author = {Thartori, E and Zuffianò, A and Pastorelli, C and Di Giunta and L and Lunetti, C and Lansford, JE and Dodge, KA and Favini, A and Gómez
             Plata, M and Caprara, GV},
   Title = {The interactive effects of maternal personality and
             adolescent temperament on externalizing behavior problem
             trajectories from age 12 to 14},
   Journal = {Personality and Individual Differences},
   Volume = {134},
   Pages = {301-307},
   Year = {2018},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.paid.2018.06.021},
   Doi = {10.1016/j.paid.2018.06.021},
   Key = {fds335169}
}

@article{fds337498,
   Author = {Lansford, JE and Rothenberg, WA and Jensen, TM and Lippold, MA and Bacchini, D and Bornstein, MH and Chang, L and Deater-Deckard, K and Di
             Giunta, L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Uribe
             Tirado, LM and Alampay, LP and Al-Hassan, SM},
   Title = {Bidirectional Relations Between Parenting and Behavior
             Problems From Age 8 to 13 in Nine Countries},
   Journal = {Journal of Research on Adolescence},
   Volume = {28},
   Number = {3},
   Pages = {571-590},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1111/jora.12381},
   Abstract = {© 2018 Society for Research on Adolescence This study used
             data from 12 cultural groups in nine countries (China,
             Colombia, Italy, Jordan, Kenya, Philippines, Sweden,
             Thailand, and the United States; N = 1,298) to understand
             the cross-cultural generalizability of how parental warmth
             and control are bidirectionally related to externalizing and
             internalizing behaviors from childhood to early adolescence.
             Mothers, fathers, and children completed measures when
             children were ages 8–13. Multiple-group autoregressive,
             cross-lagged structural equation models revealed that child
             effects rather than parent effects may better characterize
             how warmth and control are related to child externalizing
             and internalizing behaviors over time, and that parent
             effects may be more characteristic of relations between
             parental warmth and control and child externalizing and
             internalizing behavior during childhood than early
             adolescence.},
   Doi = {10.1111/jora.12381},
   Key = {fds337498}
}

@article{fds337499,
   Author = {Saint-Eloi Cadely and H and Pittman, JF and Pettit, GS and Lansford, JE and Bates, JE and Dodge, KA and Holtzworth-Munroe,
             A},
   Title = {Predicting Patterns of Intimate Partner Violence
             Perpetration From Late Adolescence to Young
             Adulthood.},
   Journal = {Journal of Interpersonal Violence},
   Pages = {886260518795173},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1177/0886260518795173},
   Abstract = {Saint-Eloi Cadely et al. found longitudinal patterns for the
             perpetration of both psychological and physical intimate
             partner violence (IPV), including actively and minimally
             aggressive patterns. The current study builds on these
             findings by examining four theory-derived variables
             (interparental aggression, social-information processing
             [SIP] biases, relationship insecurities [preoccupied and
             fearful], and discontinuity in relationship partner over
             time) as predictors of membership within these patterns,
             using multinomial logistic regression. The analysis sample
             consisted of 484 participants who were romantically involved
             at least once during the eight waves of data collection from
             the ages of 18 to 25. In predicting psychological IPV, more
             SIP biases, higher levels of a preoccupied insecurity, and
             less discontinuity in relationship partners over time
             differentiated the actively aggressive patterns from the
             minimally aggressive pattern. In addition, two actively
             aggressive patterns of psychological IPV differed in terms
             of SIP biases and discontinuity in romantic partners.
             Specifically, more SIP biases and less discontinuity in
             romantic partnerships distinguished the extensively
             aggressive pattern from the pattern that mainly consisted of
             minor types of aggression. In predicting physical IPV, the
             aggressive pattern differed from the nonaggressive pattern
             in terms of more interparental aggression, more SIP biases,
             and more relationship insecurities. The findings that
             developmental patterns of IPV can be predicted by social and
             psychological factors may aid both developmental theory and
             practice.},
   Doi = {10.1177/0886260518795173},
   Key = {fds337499}
}

@article{fds337500,
   Author = {Lansford, JE and Godwin, J and Bornstein, MH and Chang, L and Deater-Deckard, K and Di Giunta and L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Uribe Tirado and LM and Alampay, LP and Al-Hassan, SM and Bacchini, D},
   Title = {Parenting, culture, and the development of externalizing
             behaviors from age 7 to 14 in nine countries.},
   Journal = {Development and Psychopathology},
   Pages = {1-22},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1017/s0954579418000925},
   Abstract = {Using multilevel models, we examined mother-, father-, and
             child-reported (N = 1,336 families) externalizing behavior
             problem trajectories from age 7 to 14 in nine countries
             (China, Colombia, Italy, Jordan, Kenya, the Philippines,
             Sweden, Thailand, and the United States). The intercept and
             slope of children's externalizing behavior trajectories
             varied both across individuals within culture and across
             cultures, and the variance was larger at the individual
             level than at the culture level. Mothers' and children's
             endorsement of aggression as well as mothers' authoritarian
             attitudes predicted higher age 8 intercepts of child
             externalizing behaviors. Furthermore, prediction from
             individual-level endorsement of aggression and authoritarian
             attitudes to more child externalizing behaviors was
             augmented by prediction from cultural-level endorsement of
             aggression and authoritarian attitudes, respectively.
             Cultures in which father-reported endorsement of aggression
             was higher and both mother- and father-reported
             authoritarian attitudes were higher also reported more child
             externalizing behavior problems at age 8. Among fathers,
             greater attributions regarding uncontrollable success in
             caregiving situations were associated with steeper declines
             in externalizing over time. Understanding cultural-level as
             well as individual-level correlates of children's
             externalizing behavior offers potential insights into
             prevention and intervention efforts that can be more
             effectively targeted at individual children and parents as
             well as targeted at changing cultural norms that increase
             the risk of children's and adolescents' externalizing
             behavior.},
   Doi = {10.1017/s0954579418000925},
   Key = {fds337500}
}

@article{fds337501,
   Author = {Lansford, JE and Malone, PS and Tapanya, S and Tirado, LMU and Zelli, A and Alampay, LP and Al-Hassan, SM and Bacchini, D and Bornstein, MH and Chang, L and Deater-Deckard, K and Giunta, LD and Dodge, KA and Oburu,
             P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg,
             L},
   Title = {Household income predicts trajectories of child
             internalizing and externalizing behavior in high-, middle-,
             and low-income countries},
   Journal = {International Journal of Behavioral Development},
   Pages = {016502541878327-016502541878327},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1177/0165025418783272},
   Doi = {10.1177/0165025418783272},
   Key = {fds337501}
}

@article{fds335170,
   Author = {Bushman, BJ and Coyne, SM and Anderson, CA and Björkqvist, K and Boxer,
             P and Dodge, KA and Dubow, EF and Farrington, DP and Gentile, DA and Huesmann, LR and Lansford, JE and Novaco, RW and Ostrov, JM and Underwood, MK and Warburton, WA and Ybarra, ML},
   Title = {Risk factors for youth violence: Youth violence commission,
             International Society For Research On Aggression
             (ISRA).},
   Journal = {Aggressive Behavior},
   Volume = {44},
   Number = {4},
   Pages = {331-336},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1002/ab.21766},
   Doi = {10.1002/ab.21766},
   Key = {fds335170}
}

@article{fds333727,
   Author = {Olson, SL and Davis-Kean, P and Chen, M and Lansford, JE and Bates, JE and Pettit, GS and Dodge, KA},
   Title = {Mapping the Growth of Heterogeneous Forms of Externalizing
             Problem Behavior Between Early Childhood and
             Adolescence:A Comparison of Parent and Teacher
             Ratings.},
   Journal = {Journal of Abnormal Child Psychology},
   Volume = {46},
   Number = {5},
   Pages = {935-950},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1007/s10802-018-0407-9},
   Abstract = {We compared long-term growth patterns in teachers' and
             mothers' ratings of Overt Aggression, Covert Aggression,
             Oppositional Defiance, Impulsivity/inattention, and Emotion
             Dysregulation across developmental periods spanning
             kindergarten through grade 8 (ages 5 to 13 years). We also
             determined whether salient background characteristics and
             measures of child temperament and parenting risk
             differentially predicted growth in discrete categories of
             child externalizing symptoms across development.
             Participants were 549 kindergarten-age children (51% male;
             83% European American; 17% African American) whose problem
             behaviors were rated by teachers and parents each successive
             year of development through 8th grade. Latent growth curve
             analyses were performed for each component scale,
             contrasting with an overall index of externalizing, in a
             piecewise fashion encompassing two periods of development:
             K-1and grades 1-8. Our findings showed that there were
             meaningful differences and similarities between informants
             in their levels of concern about specific forms of
             externalizing problems, patterns of change in problem
             behavior reports across development, and in the extent to
             which their ratings of specific problems were associated
             with distal and proximal covariates. Thus, these data
             provided novel information about issues that have received
             scant empirical attention and have important implications
             for understanding the development and prevention of
             children's long-term externalizing problems.},
   Doi = {10.1007/s10802-018-0407-9},
   Key = {fds333727}
}

@article{fds333726,
   Author = {Duell, N and Icenogle, G and Silva, K and Chein, J and Steinberg, L and Banich, MT and Di Guinta and L and Dodge, KA and Fanti, KA and Lansford,
             JE and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Tapanya, S and Uribe Tirado and LM and Alampay, LP and Al-Hassan, SM and Takash, HMS and Bacchini, D and Chang, L and Chaudhary,
             N},
   Title = {A cross-sectional examination of response inhibition and
             working memory on the Stroop task},
   Journal = {Cognitive Development},
   Volume = {47},
   Pages = {19-31},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.cogdev.2018.02.003},
   Abstract = {© 2018 Elsevier Inc. The authors examined the association
             between working memory and response inhibition on the Stroop
             task using a cross-sectional, international sample of 5099
             individuals (49.3% male) ages 10–30 (M = 17.04 years; SD =
             5.9). Response inhibition was measured using a Stroop task
             that included “equal” and “unequal” blocks, during
             which the relative frequency of neutral and incongruent
             trials was manipulated. Competing stimuli in incongruent
             trials evinced inhibitory functioning, and having a lower
             proportion of incongruent trials (as in unequal blocks)
             placed higher demands on working memory. Results for
             accuracy indicated that age and working memory were
             independently associated with response inhibition. Age
             differences in response inhibition followed a curvilinear
             trajectory, with performance improving into early adulthood.
             Response inhibition was greatest among individuals with high
             working memory. For response time, age uniquely predicted
             response inhibition in unequal blocks. In equal blocks, age
             differences in response inhibition varied as a function of
             working memory, with age differences being least pronounced
             among individuals with high working memory. The implications
             of considering the association between response inhibition
             and working memory in the context of development are
             discussed.},
   Doi = {10.1016/j.cogdev.2018.02.003},
   Key = {fds333726}
}

@article{fds335171,
   Author = {Muschkin, CG and Ladd, HF and Dodge, KA and Bai, Y},
   Title = {Gender Differences in the Impact of North Carolina’s Early
             Care and Education Initiatives on Student Outcomes in
             Elementary School},
   Journal = {Educational Policy},
   Pages = {089590481877390-089590481877390},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1177/0895904818773901},
   Doi = {10.1177/0895904818773901},
   Key = {fds335171}
}

@article{fds330212,
   Author = {McQuillan, ME and Kultur, EC and Bates, JE and O'Reilly, LM and Dodge,
             KA and Lansford, JE and Pettit, GS},
   Title = {Dysregulation in children: Origins and implications from age
             5 to age 28.},
   Journal = {Development and Psychopathology},
   Volume = {30},
   Number = {2},
   Pages = {695-713},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1017/s0954579417001572},
   Abstract = {Research shows that childhood dysregulation is associated
             with later psychiatric disorders. It does not yet resolve
             discrepancies in the operationalization of dysregulation. It
             is also far from settled on the origins and implications of
             individual differences in dysregulation. This study tested
             several operational definitions of dysregulation using
             Achenbach attention, anxious/depressed, and aggression
             subscales. Individual growth curves of dysregulation were
             computed, and predictors of growth differences were
             considered. The study also compared the predictive utility
             of the dysregulation indexes to standard externalizing and
             internalizing indexes. Dysregulation was indexed annually
             for 24 years in a community sample (n = 585). Hierarchical
             linear models considered changes in dysregulation in
             relation to possible influences from parenting, family
             stress, child temperament, language, and peer relations. In
             a test of the meaning of dysregulation, it was related to
             functional and psychiatric outcomes in adulthood.
             Dysregulation predictions were further compared to those of
             the more standard internalizing and externalizing indexes.
             Growth curve analyses showed strong stability of
             dysregulation. Initial levels of dysregulation were
             predicted by temperamental resistance to control, and change
             in dysregulation was predicted by poor language ability and
             peer relations. Dysregulation and externalizing problems
             were associated with negative adult outcomes to a similar
             extent.},
   Doi = {10.1017/s0954579417001572},
   Key = {fds330212}
}

@article{fds329395,
   Author = {Duell, N and Steinberg, L and Icenogle, G and Chein, J and Chaudhary, N and Di Giunta and L and Dodge, KA and Fanti, KA and Lansford, JE and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Tapanya, S and Uribe
             Tirado, LM and Alampay, LP and Al-Hassan, SM and Takash, HMS and Bacchini, D and Chang, L},
   Title = {Age Patterns in Risk Taking Across the World.},
   Journal = {Journal of Youth and Adolescence},
   Volume = {47},
   Number = {5},
   Pages = {1052-1072},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1007/s10964-017-0752-y},
   Abstract = {Epidemiological data indicate that risk behaviors are among
             the leading causes of adolescent morbidity and mortality
             worldwide. Consistent with this, laboratory-based studies of
             age differences in risk behavior allude to a peak in
             adolescence, suggesting that adolescents demonstrate a
             heightened propensity, or inherent inclination, to take
             risks. Unlike epidemiological reports, studies of risk
             taking propensity have been limited to Western samples,
             leaving questions about the extent to which heightened risk
             taking propensity is an inherent or culturally constructed
             aspect of adolescence. In the present study, age patterns in
             risk-taking propensity (using two laboratory tasks: the
             Stoplight and the BART) and real-world risk taking (using
             self-reports of health and antisocial risk taking) were
             examined in a sample of 5227 individuals (50.7% female) ages
             10-30 (M = 17.05 years, SD = 5.91) from 11 Western
             and non-Western countries (China, Colombia, Cyprus, India,
             Italy, Jordan, Kenya, the Philippines, Sweden, Thailand, and
             the US). Two hypotheses were tested: (1) risk taking follows
             an inverted-U pattern across age groups, peaking earlier on
             measures of risk taking propensity than on measures of
             real-world risk taking, and (2) age patterns in risk taking
             propensity are more consistent across countries than age
             patterns in real-world risk taking. Overall, risk taking
             followed the hypothesized inverted-U pattern across age
             groups, with health risk taking evincing the latest peak.
             Age patterns in risk taking propensity were more consistent
             across countries than age patterns in real-world risk
             taking. Results suggest that although the association
             between age and risk taking is sensitive to measurement and
             culture, around the world, risk taking is generally highest
             among late adolescents.},
   Doi = {10.1007/s10964-017-0752-y},
   Key = {fds329395}
}

@article{fds332672,
   Author = {Crowley, DM and Dodge, KA and Barnett, WS and Corso, P and Duffy, S and Graham, P and Greenberg, M and Haskins, R and Hill, L and Jones, DE and Karoly, LA and Kuklinski, MR and Plotnick, R},
   Title = {Standards of Evidence for Conducting and Reporting Economic
             Evaluations in Prevention Science.},
   Journal = {Prevention Science : the Official Journal of the Society for
             Prevention Research},
   Volume = {19},
   Number = {3},
   Pages = {366-390},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s11121-017-0858-1},
   Abstract = {Over a decade ago, the Society for Prevention Research
             endorsed the first standards of evidence for research in
             preventive interventions. The growing recognition of the
             need to use limited resources to make sound investments in
             prevention led the Board of Directors to charge a new task
             force to set standards for research in analysis of the
             economic impact of preventive interventions. This article
             reports the findings of this group's deliberations, proposes
             standards for economic analyses, and identifies
             opportunities for future prevention science. Through
             examples, policymakers' need and use of economic analysis
             are described. Standards are proposed for framing economic
             analysis, estimating costs of prevention programs,
             estimating benefits of prevention programs, implementing
             summary metrics, handling uncertainty in estimates, and
             reporting findings. Topics for research in economic analysis
             are identified. The SPR Board of Directors endorses the
             "Standards of Evidence for Conducting and Reporting Economic
             Evaluations in Prevention Science."},
   Doi = {10.1007/s11121-017-0858-1},
   Key = {fds332672}
}

@article{fds323764,
   Author = {Steinberg, L and Icenogle, G and Shulman, EP and Breiner, K and Chein,
             J and Bacchini, D and Chang, L and Chaudhary, N and Giunta, LD and Dodge,
             KA and Fanti, KA and Lansford, JE and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Tapanya, S and Tirado,
             LMU and Alampay, LP and Al-Hassan, SM and Takash,
             HMS},
   Title = {Around the world, adolescence is a time of heightened
             sensation seeking and immature self-regulation.},
   Journal = {Developmental Science},
   Volume = {21},
   Number = {2},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1111/desc.12532},
   Abstract = {The dual systems model of adolescent risk-taking portrays
             the period as one characterized by a combination of
             heightened sensation seeking and still-maturing
             self-regulation, but most tests of this model have been
             conducted in the United States or Western Europe. In the
             present study, these propositions are tested in an
             international sample of more than 5000 individuals between
             ages 10 and 30 years from 11 countries in Africa, Asia,
             Europe and the Americas, using a multi-method test battery
             that includes both self-report and performance-based
             measures of both constructs. Consistent with the dual
             systems model, sensation seeking increased between
             preadolescence and late adolescence, peaked at age 19, and
             declined thereafter, whereas self-regulation increased
             steadily from preadolescence into young adulthood, reaching
             a plateau between ages 23 and 26. Although there were some
             variations in the magnitude of the observed age trends, the
             developmental patterns were largely similar across
             countries.},
   Doi = {10.1111/desc.12532},
   Key = {fds323764}
}

@article{fds330823,
   Author = {Petersen, IT and Lindhiem, O and LeBeau, B and Bates, JE and Pettit, GS and Lansford, JE and Dodge, KA},
   Title = {Development of internalizing problems from adolescence to
             emerging adulthood: Accounting for heterotypic continuity
             with vertical scaling.},
   Journal = {Developmental Psychology},
   Volume = {54},
   Number = {3},
   Pages = {586-599},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1037/dev0000449},
   Abstract = {Manifestations of internalizing problems, such as specific
             symptoms of anxiety and depression, can change across
             development, even if individuals show strong continuity in
             rank-order levels of internalizing problems. This
             illustrates the concept of heterotypic continuity, and
             raises the question of whether common measures might be
             construct-valid for one age but not another. This study
             examines mean-level changes in internalizing problems across
             a long span of development at the same time as accounting
             for heterotypic continuity by using age-appropriate,
             changing measures. Internalizing problems from age 14-24
             were studied longitudinally in a community sample (N = 585),
             using Achenbach's Youth Self-Report (YSR) and Young Adult
             Self-Report (YASR). Heterotypic continuity was evaluated
             with an item response theory (IRT) approach to vertical
             scaling, linking different measures over time to be on the
             same scale, as well as with a Thurstone scaling approach.
             With vertical scaling, internalizing problems peaked in
             mid-to-late adolescence and showed a group-level decrease
             from adolescence to early adulthood, a change that would not
             have been seen with the approach of using only age-common
             items. Individuals' trajectories were sometimes different
             than would have been seen with the common-items approach.
             Findings support the importance of considering heterotypic
             continuity when examining development and vertical scaling
             to account for heterotypic continuity with changing
             measures. (PsycINFO Database Record},
   Doi = {10.1037/dev0000449},
   Key = {fds330823}
}

@article{fds330822,
   Author = {Lansford, JE and Godwin, J and Al-Hassan, SM and Bacchini, D and Bornstein, MH and Chang, L and Chen, B-B and Deater-Deckard, K and Di
             Giunta, L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Alampay,
             LP and Uribe Tirado and LM and Zelli, A},
   Title = {Longitudinal associations between parenting and youth
             adjustment in twelve cultural groups: Cultural normativeness
             of parenting as a moderator.},
   Journal = {Developmental Psychology},
   Volume = {54},
   Number = {2},
   Pages = {362-377},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1037/dev0000416},
   Abstract = {To examine whether the cultural normativeness of parents'
             beliefs and behaviors moderates the links between those
             beliefs and behaviors and youths' adjustment, mothers,
             fathers, and children (N = 1,298 families) from 12 cultural
             groups in 9 countries (China, Colombia, Italy, Jordan,
             Kenya, Philippines, Sweden, Thailand, and the United States)
             were interviewed when children were, on average, 10 years
             old and again when children were 12 years old. Multilevel
             models examined 5 aspects of parenting (expectations
             regarding family obligations, monitoring, psychological
             control, behavioral control, warmth/affection) in relation
             to 5 aspects of youth adjustment (social competence,
             prosocial behavior, academic achievement, externalizing
             behavior, internalizing behavior). Interactions between
             family level and culture-level predictors were tested to
             examine whether cultural normativeness of parenting
             behaviors moderated the link between those behaviors and
             children's adjustment. More evidence was found for within-
             than between-culture differences in parenting predictors of
             youth adjustment. In 7 of the 8 instances in which cultural
             normativeness was found to moderate the link between
             parenting and youth adjustment, the link between a
             particular parenting behavior and youth adjustment was
             magnified in cultural contexts in which the parenting
             behavior was more normative. (PsycINFO Database
             Record},
   Doi = {10.1037/dev0000416},
   Key = {fds330822}
}

@article{fds333728,
   Author = {Miller, AB and Sheridan, MA and Hanson, JL and McLaughlin, KA and Bates,
             JE and Lansford, JE and Pettit, GS and Dodge, KA},
   Title = {Dimensions of deprivation and threat, psychopathology, and
             potential mediators: A multi-year longitudinal
             analysis.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {127},
   Number = {2},
   Pages = {160-170},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1037/abn0000331},
   Abstract = {Prior research demonstrates a link between exposure to
             childhood adversity and psychopathology later in
             development. However, work on mechanisms linking adversity
             to psychopathology fails to account for specificity in these
             pathways across different types of adversity. Here, we test
             a conceptual model that distinguishes deprivation and threat
             as distinct forms of childhood adversity with different
             pathways to psychopathology. Deprivation involves an absence
             of inputs from the environment, such as cognitive and social
             stimulation, that influence psychopathology by altering
             cognitive development, such as verbal abilities. Threat
             includes experiences involving harm or threat of harm that
             increase risk for psychopathology through disruptions in
             social-emotional processing. We test the prediction that
             deprivation, but not threat, increases risk for
             psychopathology through altered verbal abilities. Data were
             drawn from the Child Development Project (N = 585), which
             followed children for over a decade. We analyze data from
             assessment points at age 5, 6, 14, and 17 years. Mothers
             completed interviews at age 5 and 6 on exposure to threat
             and deprivation experiences. Youth verbal abilities were
             assessed at age 14. At age 17, mothers reported on child
             psychopathology. A path analysis model tested longitudinal
             paths to internalizing and externalizing problems from
             experiences of deprivation and threat. Consistent with
             predictions, deprivation was associated with risk for
             externalizing problems via effects on verbal abilities at
             age 14. Threat was associated longitudinally with both
             internalizing and externalizing problems, but these effects
             were not mediated by verbal abilities. Results suggest that
             unique developmental mechanisms link different forms of
             adversity with psychopathology. (PsycINFO Database
             Record},
   Doi = {10.1037/abn0000331},
   Key = {fds333728}
}

@article{fds326573,
   Author = {Wang, FL and Chassin, L and Bates, JE and Dick, D and Lansford, JE and Pettit, GS and Dodge, KA},
   Title = {Serotonin functioning and adolescents' alcohol use: A
             genetically informed study examining mechanisms of
             risk.},
   Journal = {Development and Psychopathology},
   Volume = {30},
   Number = {1},
   Pages = {213-233},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1017/s095457941700058x},
   Abstract = {The current study used data from two longitudinal samples to
             test whether self-regulation, depressive symptoms, and
             aggression/antisociality were mediators in the relation
             between a polygenic score indexing serotonin (5-HT)
             functioning and alcohol use in adolescence. The results from
             an independent genome-wide association study of
             5-hydroxyindoleacetic acid in the cerebrospinal fluid were
             used to create 5-HT polygenic risk scores. Adolescents
             and/or parents reported on adolescents' self-regulation
             (Time 1), depressive symptoms (Time 2), aggression/antisociality
             (Time 2), and alcohol use (Time 3). The results showed that
             5-HT polygenic risk did not predict self-regulation.
             However, adolescents with higher levels of 5-HT polygenic
             risk showed greater depression and aggression/antisociality.
             Adolescents' aggression/antisociality mediated the relation
             between 5-HT polygenic risk and later alcohol use. Deficits
             in self-regulation also predicted depression and
             aggression/antisociality, and indirectly predicted alcohol
             use through aggression/antisociality. Pathways to alcohol
             use were especially salient for males from families with low
             parental education in one of the two samples. The results
             provide insights into the longitudinal mechanisms underlying
             the relation between 5-HT functioning and alcohol use (i.e.,
             earlier aggression/antisociality). There was no evidence
             that genetically based variation in 5-HT functioning
             predisposed individuals to deficits in self-regulation.
             Genetically based variation in 5-HT functioning and
             self-regulation might be separate, transdiagnostic risk
             factors for several types of psychopathology.},
   Doi = {10.1017/s095457941700058x},
   Key = {fds326573}
}

@article{fds330213,
   Author = {Schwartz, D and Lansford, JE and Dodge, KA and Pettit, GS and Bates,
             JE},
   Title = {Peer Victimization during Middle Childhood as a Marker of
             Attenuated Risk for Adult Arrest.},
   Journal = {Journal of Abnormal Child Psychology},
   Volume = {46},
   Number = {1},
   Pages = {57-65},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s10802-017-0354-x},
   Abstract = {This longitudinal investigation examined interactions
             between aggression and peer victimization during middle
             childhood in the prediction of arrest through the adult
             years for 388 (198 boys, 190 girls) study participants. As
             part of an ongoing multisite study (i.e., Child Development
             Project), peer victimization and aggression were assessed
             via a peer nomination inventory in middle childhood, and
             juvenile and adult arrest histories were assessed via a
             self-report questionnaire as well as review of court
             records. Early aggression was linked to later arrest but
             only for those youths who were rarely victimized by peers.
             Although past investigators have viewed youths who are both
             aggressive and victimized as a high-risk subgroup, our
             findings suggest that the psychological and behavioral
             attributes of these children may mitigate trajectories
             toward antisocial problems.},
   Doi = {10.1007/s10802-017-0354-x},
   Key = {fds330213}
}

@article{fds332381,
   Author = {Zheng, Y and Albert, D and McMahon, RJ and Dodge, K and Dick, D and Conduct
             Problems Prevention Research Group},
   Title = {Glucocorticoid Receptor (NR3C1) Gene Polymorphism Moderate
             Intervention Effects on the Developmental Trajectory of
             African-American Adolescent Alcohol Abuse.},
   Journal = {Prevention Science : the Official Journal of the Society for
             Prevention Research},
   Volume = {19},
   Number = {1},
   Pages = {79-89},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1007/s11121-016-0726-4},
   Abstract = {Accumulative evidence from recent genotype × intervention
             studies suggests that individuals carrying susceptible
             genotypes benefit more from intervention and provides one
             avenue to identify subgroups that respond differentially to
             intervention. This study examined the moderation by
             glucocorticoid receptor (NR3C1) gene variants of
             intervention effects on the developmental trajectories of
             alcohol abuse through adolescence. Participants were
             randomized into Fast Track intervention and control groups
             self-reported past-year alcohol abuse annually from grade 7
             through 2 years post-high school and provided genotype data
             at age 21 (69% males; European Americans [EAs] = 270,
             African-Americans [AAs] = 282). Latent growth curve
             models were fit to examine developmental trajectories of
             alcohol abuse. The interactions of 10 single nucleotide
             polymorphisms (SNPs) in NR3C1 with intervention were
             examined separately. Both EAs and AAs showed significant
             increases in past-year alcohol abuse with substantial
             inter-individual differences in rates of linear growth. AAs
             showed lower general levels and slower rates of linear
             growth than EAs. Adjusting for multiple tests, one NR3C1 SNP
             (rs12655166) significantly moderated intervention effects on
             the developmental trajectories of alcohol abuse among AAs.
             Intervention effects on the rates of linear growth were
             stronger among AAs carrying minor alleles than those not
             carrying minor alleles. The findings highlight the
             importance of taking a developmental perspective on
             adolescent alcohol use and have implications for future
             intervention design and evaluation by identifying subgroups
             that could disproportionally benefit from
             intervention.},
   Doi = {10.1007/s11121-016-0726-4},
   Key = {fds332381}
}

@article{fds327155,
   Author = {Deater-Deckard, K and Godwin, J and Lansford, JE and Bacchini, D and Bombi, AS and Bornstein, MH and Chang, L and Di Giunta and L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring,
             E and Steinberg, L and Tapanya, S and Alampay, LP and Uribe Tirado and LM and Zelli, A and Al-Hassan, SM},
   Title = {Within- and between-person and group variance in behavior
             and beliefs in cross-cultural longitudinal
             data.},
   Journal = {Journal of Adolescence},
   Volume = {62},
   Pages = {207-217},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.adolescence.2017.06.002},
   Abstract = {This study grapples with what it means to be part of a
             cultural group, from a statistical modeling perspective. The
             method we present compares within- and between-cultural
             group variability, in behaviors in families. We demonstrate
             the method using a cross-cultural study of adolescent
             development and parenting, involving three biennial waves of
             longitudinal data from 1296 eight-year-olds and their
             parents (multiple cultures in nine countries). Family
             members completed surveys about parental negativity and
             positivity, child academic and social-emotional adjustment,
             and attitudes about parenting and adolescent behavior.
             Variance estimates were computed at the cultural group,
             person, and within-person level using multilevel models. Of
             the longitudinally consistent variance, most was within and
             not between cultural groups-although there was a wide range
             of between-group differences. This approach to quantifying
             cultural group variability may prove valuable when applied
             to quantitative studies of acculturation.},
   Doi = {10.1016/j.adolescence.2017.06.002},
   Key = {fds327155}
}

@article{fds327289,
   Author = {Berlin, LJ and Martoccio, TL and Appleyard Carmody and K and Goodman,
             WB and O'Donnell, K and Williams, J and Murphy, RA and Dodge,
             KA},
   Title = {Can typical US home visits affect infant attachment?
             Preliminary findings from a randomized trial of Healthy
             Families Durham.},
   Journal = {Attachment & Human Development},
   Volume = {19},
   Number = {6},
   Pages = {559-579},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1080/14616734.2017.1339359},
   Abstract = {US government-funded early home visiting services are
             expanding significantly. The most widely implemented home
             visiting models target at-risk new mothers and their
             infants. Such home visiting programs typically aim to
             support infant-parent relationships; yet, such programs'
             effects on infant attachment quality per se are as yet
             untested. Given these programs' aims, and the crucial role
             of early attachments in human development, it is important
             to understand attachment processes in home visited families.
             The current, preliminary study examined 94 high-risk
             mother-infant dyads participating in a randomized evaluation
             of the Healthy Families Durham (HFD) home visiting program.
             We tested (a) infant attachment security and disorganization
             as predictors of toddler behavior problems and (b) program
             effects on attachment security and disorganization. We found
             that (a) infant attachment disorganization (but not
             security) predicted toddler behavior problems and (b)
             participation in HFD did not significantly affect infant
             attachment security or disorganization. Findings are
             discussed in terms of the potential for attachment-specific
             interventions to enhance the typical array of home visiting
             services.},
   Doi = {10.1080/14616734.2017.1339359},
   Key = {fds327289}
}

@article{fds330211,
   Author = {Lansford, JE and Godwin, J and Bornstein, MH and Chang, L and Deater-Deckard, K and Di Giunta and L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Alampay, LP and Uribe Tirado and LM and Al-Hassan, SM and Bacchini, D},
   Title = {Reward sensitivity, impulse control, and social cognition as
             mediators of the link between childhood family adversity and
             externalizing behavior in eight countries.},
   Journal = {Development and Psychopathology},
   Volume = {29},
   Number = {5},
   Pages = {1675-1688},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1017/s0954579417001328},
   Abstract = {Using data from 1,177 families in eight countries (Colombia,
             Italy, Jordan, Kenya, the Philippines, Sweden, Thailand, and
             the United States), we tested a conceptual model of direct
             effects of childhood family adversity on subsequent
             externalizing behaviors as well as indirect effects through
             psychological mediators. When children were 9 years old,
             mothers and fathers reported on financial difficulties and
             their use of corporal punishment, and children reported
             perceptions of their parents' rejection. When children were
             10 years old, they completed a computerized battery of tasks
             assessing reward sensitivity and impulse control and
             responded to questions about hypothetical social
             provocations to assess their hostile attributions and
             proclivity for aggressive responding. When children were 12
             years old, they reported on their externalizing behavior.
             Multigroup structural equation models revealed that across
             all eight countries, childhood family adversity had direct
             effects on externalizing behaviors 3 years later, and
             childhood family adversity had indirect effects on
             externalizing behavior through psychological mediators. The
             findings suggest ways in which family-level adversity poses
             risk for children's subsequent development of problems at
             psychological and behavioral levels, situated within diverse
             cultural contexts.},
   Doi = {10.1017/s0954579417001328},
   Key = {fds330211}
}

@article{fds330210,
   Author = {Sorensen, LC and Cook, PJ and Dodge, KA},
   Title = {From Parents to Peers: Trajectories in Sources of Academic
             Influence Grades 4 to 8},
   Journal = {Educational Evaluation and Policy Analysis},
   Volume = {39},
   Number = {4},
   Pages = {697-711},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.3102/0162373717708335},
   Doi = {10.3102/0162373717708335},
   Key = {fds330210}
}

@article{fds330175,
   Author = {Cook, PJ and Dodge, KA and Gifford, EJ and Schulting,
             AB},
   Title = {A new program to prevent primary school absenteeism: Results
             of a pilot study in five schools},
   Journal = {Children and Youth Services Review},
   Volume = {82},
   Pages = {262-270},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.childyouth.2017.09.017},
   Doi = {10.1016/j.childyouth.2017.09.017},
   Key = {fds330175}
}

@article{fds327351,
   Author = {Goodnight, JA and Bates, JE and Holtzworth-Munroe, A and Pettit, GS and Ballard, RH and Iskander, JM and Swanson, A and Dodge, KA and Lansford,
             JE},
   Title = {Dispositional, demographic, and social predictors of
             trajectories of intimate partner aggression in early
             adulthood.},
   Journal = {Journal of Consulting and Clinical Psychology},
   Volume = {85},
   Number = {10},
   Pages = {950-965},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1037/ccp0000226},
   Abstract = {From a developmental systems perspective, the origins of
             maladjusted behavior are multifaceted, interdependent, and
             may differ at different points in development. Personality
             traits influence developmental outcomes, as do socialization
             environments, but the influence of personality depends on
             the socialization environment, and the influence of the
             socialization environment varies according to personality.
             The present study takes a developmental systems approach to
             investigate pathways through which dispositional traits in
             childhood might act in concert with peer and parental
             socialization contexts to predict trajectories of intimate
             partner aggression (IPA) during emerging adulthood.The study
             included 466 participants (49% male, 81% European American,
             15% African American) from a longitudinal study of social
             development. Measures of demographics, temperament,
             personality, parent-child relations, romantic relationships,
             peer relationships, and IPA were administered between 5 and
             23 years of age. The study used latent growth curve analysis
             to predict variations in trajectories of IPA during early
             adulthood.Numerous variables predicted risk for the
             perpetration of IPA, but different factors were associated
             at the end of adolescence (e.g., psychopathic traits) than
             with changes across early adulthood (e.g., friend
             antisociality). Males and individuals with a history of
             resistance to control temperament showed enhanced
             susceptibility to social risk factors, such as exposure to
             antisocial peers and poor parent-adolescent
             relations.Consistent with a developmental systems
             perspective, multiple factors, including personality traits
             in early childhood and aspects of the social environment in
             adolescence, predict trajectories of IPA during early
             adulthood through additive, mediated, and moderated
             pathways. Knowledge of these risk factors and for whom they
             are most influential could help inform efforts to prevent
             the emergence and persistence of IPA. (PsycINFO Database
             Record},
   Doi = {10.1037/ccp0000226},
   Key = {fds327351}
}

@article{fds322250,
   Author = {Icenogle, G and Steinberg, L and Olino, TM and Shulman, EP and Chein, J and Alampay, LP and Al-Hassan, SM and Takash, HMS and Bacchini, D and Chang,
             L and Chaudhary, N and Di Giunta and L and Dodge, KA and Fanti, KA and Lansford, JE and Malone, PS and Oburu, P and Pastorelli, C and Skinner,
             AT and Sorbring, E and Tapanya, S and Uribe Tirado,
             LM},
   Title = {Puberty Predicts Approach But Not Avoidance on the Iowa
             Gambling Task in a Multinational Sample.},
   Journal = {Child Development},
   Volume = {88},
   Number = {5},
   Pages = {1598-1614},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1111/cdev.12655},
   Abstract = {According to the dual systems model of adolescent risk
             taking, sensation seeking and impulse control follow
             different developmental trajectories across adolescence and
             are governed by two different brain systems. The authors
             tested whether different underlying processes also drive age
             differences in reward approach and cost avoidance. Using a
             modified Iowa Gambling Task in a multinational,
             cross-sectional sample of 3,234 adolescents (ages 9-17;
             M = 12.87, SD = 2.36), pubertal maturation, but not age,
             predicted reward approach, mediated through higher sensation
             seeking. In contrast, age, but not pubertal maturation,
             predicted increased cost avoidance, mediated through greater
             impulse control. These findings add to evidence that
             adolescent behavior is best understood as the product of two
             interacting, but independently developing, brain
             systems.},
   Doi = {10.1111/cdev.12655},
   Key = {fds322250}
}

@article{fds325210,
   Author = {Bornstein, MH and Putnick, DL and Lansford, JE and Al-Hassan, SM and Bacchini, D and Bombi, AS and Chang, L and Deater-Deckard, K and Di
             Giunta, L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring, E and Steinberg, L and Tapanya, S and Tirado,
             LMU and Zelli, A and Alampay, LP},
   Title = {'Mixed blessings': parental religiousness, parenting, and
             child adjustment in global perspective.},
   Journal = {Journal of Child Psychology and Psychiatry, and Allied
             Disciplines},
   Volume = {58},
   Number = {8},
   Pages = {880-892},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/jcpp.12705},
   Abstract = {Most studies of the effects of parental religiousness on
             parenting and child development focus on a particular
             religion or cultural group, which limits generalizations
             that can be made about the effects of parental religiousness
             on family life.We assessed the associations among parental
             religiousness, parenting, and children's adjustment in a
             3-year longitudinal investigation of 1,198 families from
             nine countries. We included four religions (Catholicism,
             Protestantism, Buddhism, and Islam) plus unaffiliated
             parents, two positive (efficacy and warmth) and two negative
             (control and rejection) parenting practices, and two
             positive (social competence and school performance) and two
             negative (internalizing and externalizing) child outcomes.
             Parents and children were informants.Greater parent
             religiousness had both positive and negative associations
             with parenting and child adjustment. Greater parent
             religiousness when children were age 8 was associated with
             higher parental efficacy at age 9 and, in turn, children's
             better social competence and school performance and fewer
             child internalizing and externalizing problems at age 10.
             However, greater parent religiousness at age 8 was also
             associated with more parental control at age 9, which in
             turn was associated with more child internalizing and
             externalizing problems at age 10. Parental warmth and
             rejection had inconsistent relations with parental
             religiousness and child outcomes depending on the informant.
             With a few exceptions, similar patterns of results held for
             all four religions and the unaffiliated, nine sites, mothers
             and fathers, girls and boys, and controlling for demographic
             covariates.Parents and children agree that parental
             religiousness is associated with more controlling parenting
             and, in turn, increased child problem behaviors. However,
             children see religiousness as related to parental rejection,
             whereas parents see religiousness as related to parental
             efficacy and warmth, which have different associations with
             child functioning. Studying both parent and child views of
             religiousness and parenting are important to understand the
             effects of parental religiousness on parents and
             children.},
   Doi = {10.1111/jcpp.12705},
   Key = {fds325210}
}

@article{fds327154,
   Author = {Alampay, LP and Godwin, J and Lansford, JE and Bombi, AS and Bornstein,
             MH and Chang, L and Deater-Deckard, K and Di Giunta and L and Dodge, KA and Malone, PS and Oburu, P and Pastorelli, C and Skinner, AT and Sorbring,
             E and Tapanya, S and Uribe Tirado and LM and Zelli, A and Al-Hassan, S and Bacchini, D},
   Title = {Severity and Justness Do Not Moderate the Relation Between
             Corporal Punishment and Negative Child Outcomes: A
             Multicultural and Longitudinal Study.},
   Journal = {International Journal of Behavioral Development},
   Volume = {41},
   Number = {4},
   Pages = {491-502},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1177/0165025417697852},
   Abstract = {There is strong evidence of a positive association between
             corporal punishment and negative child outcomes, but
             previous studies have suggested that the manner in which
             parents implement corporal punishment moderates the effects
             of its use. This study investigated whether severity and
             justness in the use of corporal punishment moderate the
             associations between frequency of corporal punishment and
             child externalizing and internalizing behaviors. This
             question was examined using a multicultural sample from
             eight countries and two waves of data collected one year
             apart. Interviews were conducted with 998 children aged 7-10
             years, and their mothers and fathers, from China, Colombia,
             Italy, Jordan, Kenya, Philippines, Thailand, and the United
             States. Mothers and fathers responded to questions on the
             frequency, severity, and justness of their use of corporal
             punishment; they also reported on the externalizing and
             internalizing behavior of their child. Children reported on
             their aggression. Multigroup path models revealed that
             across cultural groups, and as reported by mothers and
             fathers, there is a positive relation between the frequency
             of corporal punishment and externalizing child behaviors.
             Mother-reported severity and father-reported justness were
             associated with child-reported aggression. Neither severity
             nor justness moderated the relation between frequency of
             corporal punishment and child problem behavior. The null
             result suggests that more use of corporal punishment is
             harmful to children regardless of how it is implemented, but
             requires further substantiation as the study is unable to
             definitively conclude that there is no true interaction
             effect.},
   Doi = {10.1177/0165025417697852},
   Key = {fds327154}
}

@article{fds332382,
   Author = {Saint-Eloi Cadely and H and Pittman, JF and Pettit, GS and Lansford, JE and Bates, JE and Dodge, KA and Holtzworth-Munroe,
             A},
   Title = {Classes of Intimate Partner Violence From Late Adolescence
             to Young Adulthood.},
   Journal = {Journal of Interpersonal Violence},
   Pages = {886260517715601},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1177/0886260517715601},
   Abstract = {Researchers do not agree on how intimate partner violence
             (IPV) emerges and changes from adolescence to young
             adulthood. This may be because change in these behaviors
             varies across individuals. The present study uses a
             longitudinal, person-centered approach to examine whether
             there are multiple classes or patterns of change in the
             perpetration of IPV during the transitional period from
             adolescence (age 18) to young adulthood (age 25) using data
             collected annually from a community sample of 484
             participants. Latent class analysis was the analytic
             approach used. Results revealed three patterns for
             psychological IPV (Little-to-None, Minor/Increasing, and
             Extensive/Increasing) and two patterns for physical IPV
             (Little-to-None and Extensive). Patterns varied greatly in
             number of representatives, although they were more balanced
             in size for psychological than physical IPV. Variations in
             IPV behaviors were also revealed across classes, although as
             expected in a community sample, minor forms of IPV were more
             common than severe forms. In addition, classes differed in
             demographic and relationship status variables. These
             findings suggest that IPV may occur in multiple distinct
             patterns as opposed to one average pattern across a
             population. This suggests that interventions for IPV may
             need to be geared to differences in patterns to enhance
             their efficacy.},
   Doi = {10.1177/0886260517715601},
   Key = {fds332382}
}

@article{fds315899,
   Author = {Di Giunta and L and Iselin, A-MR and Eisenberg, N and Pastorelli, C and Gerbino, M and Lansford, JE and Dodge, KA and Caprara, GV and Bacchini,
             D and Uribe Tirado and LM and Thartori, E},
   Title = {Measurement Invariance and Convergent Validity of Anger and
             Sadness Self-Regulation Among Youth From Six Cultural
             Groups.},
   Journal = {Assessment},
   Volume = {24},
   Number = {4},
   Pages = {484-502},
   Year = {2017},
   Month = {June},
   ISSN = {1073-1911},
   url = {http://dx.doi.org/10.1177/1073191115615214},
   Abstract = {The present study examined measurement invariance and
             convergent validity of a novel vignette-based measure of
             emotion-specific self-regulation that simultaneously
             assesses attributional bias, emotion-regulation, and
             self-efficacy beliefs about emotion regulation. Participants
             included 541 youth-mother dyads from three countries (Italy,
             the United States, and Colombia) and six ethnic/cultural
             groups. Participants were 12.62 years old ( SD = 0.69). In
             response to vignettes involving ambiguous peer interactions,
             children reported their hostile/depressive attribution bias,
             self-efficacy beliefs about anger and sadness regulation,
             and anger/sadness regulation strategies (i.e., dysregulated
             expression and rumination). Across the six cultural groups,
             anger and sadness self-regulation subscales had full metric
             and partial scalar invariance for a one-factor model, with
             some exceptions. We found support for both a four- and
             three-factor oblique model (dysregulated expression and
             rumination loaded on a second-order factor) for both anger
             and sadness. Anger subscales were related to externalizing
             problems, while sadness subscales were related to
             internalizing symptoms.},
   Doi = {10.1177/1073191115615214},
   Key = {fds315899}
}

@article{fds320116,
   Author = {Dodge, KA and Bai, Y and Ladd, HF and Muschkin, CG},
   Title = {Impact of North Carolina's Early Childhood Programs and
             Policies on Educational Outcomes in Elementary
             School.},
   Journal = {Child Development},
   Volume = {88},
   Number = {3},
   Pages = {996-1014},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1111/cdev.12645},
   Abstract = {North Carolina's Smart Start and More at Four (MAF) early
             childhood programs were evaluated through the end of
             elementary school (age 11) by estimating the impact of state
             funding allocations to programs in each of 100 counties
             across 13 consecutive years on outcomes for all children in
             each county-year group (n = 1,004,571; 49% female; 61%
             non-Latinx White, 30% African American, 4% Latinx, 5%
             other). Student-level regression models with county and year
             fixed effects indicated significant positive impacts of each
             program on reading and math test scores and reductions in
             special education and grade retention in each grade. Effect
             sizes grew or held steady across years. Positive effects
             held for both high- and low-poverty families, suggesting
             spillover of effects to nonparticipating
             peers.},
   Doi = {10.1111/cdev.12645},
   Key = {fds320116}
}

@article{fds324191,
   Author = {Calvert, SL and Appelbaum, M and Dodge, KA and Graham, S and Nagayama
             Hall, GC and Hamby, S and Fasig-Caldwell, LG and Citkowicz, M and Galloway, DP and Hedges, LV},
   Title = {The American Psychological Association Task Force assessment
             of violent video games: Science in the service of public
             interest.},
   Journal = {American Psychologist},
   Volume = {72},
   Number = {2},
   Pages = {126-143},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1037/a0040413},
   Abstract = {A task force of experts was convened by the American
             Psychological Association (APA) to update the knowledge and
             policy about the impact of violent video game use on
             potential adverse outcomes. This APA Task Force on Media
             Violence examined the existing literature, including the
             meta-analyses in the field, since the last APA report on
             media violence in 2005. Because the most recent
             meta-analyses were published in 2010 and reflected work
             through 2009, the task force conducted a search of the
             published studies from 2009-2013. These recently published
             articles were scored and assessed by a systematic
             evidentiary review, followed by a meta-analysis of the high
             utility studies, as documented in the evidentiary review.
             Consistent with the literature that we reviewed, we found
             that violent video game exposure was associated with: an
             increased composite aggression score; increased aggressive
             behavior; increased aggressive cognitions; increased
             aggressive affect, increased desensitization, and decreased
             empathy; and increased physiological arousal. The size of
             the effects was similar to that in prior meta-analyses,
             suggesting a stable result. Our task force concluded that
             violent video game use is a risk factor for adverse
             outcomes, but found insufficient studies to examine any
             potential link between violent video game use and
             delinquency or criminal behavior. Our technical report is
             the basis of this article. (PsycINFO Database
             Record},
   Doi = {10.1037/a0040413},
   Key = {fds324191}
}


%% Duffy, Korrina A   
@article{fds323898,
   Author = {Duffy, KA and Harris, LT and Chartrand, TL and Stanton,
             SJ},
   Title = {Women recovering from social rejection: The effect of the
             person and the situation on a hormonal mechanism of
             affiliation.},
   Journal = {Psychoneuroendocrinology},
   Volume = {76},
   Pages = {174-182},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2016.11.017},
   Abstract = {Rejection can motivate either affiliation or withdrawal. In
             order to study how personality and situational variables
             influence whether women will be motivated to affiliate
             versus withdraw, we manipulate social feedback (rejection
             vs. acceptance) and opportunity for face-to-face interaction
             (blocked vs. face-to-face) and measure the individual
             difference variables rejection sensitivity and social
             anxiety. We test how these variables affect endogenous
             progesterone and cortisol concentrations, which are presumed
             to signal motivational responses to rejection. We find that
             three-way interactions involving social feedback,
             opportunity for face-to-face interactions, and either social
             anxiety or rejection sensitivity significantly predict
             progesterone change, but not cortisol change. Both
             interactions are driven by sharp progesterone decreases for
             women high in social anxiety/rejection sensitivity who have
             been rejected and who have no opportunity to reaffiliate in
             a face-to-face interaction. This progesterone change may be
             a physiological marker of motivation for social avoidance
             following rejection for women who cannot reaffiliate and who
             are particularly socially anxious or sensitive to
             rejection.},
   Doi = {10.1016/j.psyneuen.2016.11.017},
   Key = {fds323898}
}


%% Egger, Helen L.   
@article{fds338017,
   Author = {Hashemi, J and Dawson, G and Carpenter, KLH and Campbell, K and Qiu, Q and Espinosa, S and Marsan, S and Baker, JP and Egger, HL and Sapiro,
             G},
   Title = {Computer Vision Analysis for Quantification of Autism Risk
             Behaviors},
   Journal = {Ieee Transactions on Affective Computing},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1109/TAFFC.2018.2868196},
   Abstract = {IEEE Observational behavior analysis plays a key role for
             the discovery and evaluation of risk markers for many
             neurodevelopmental disorders. Research on autism spectrum
             disorder (ASD) suggests that behavioral risk markers can be
             observed at 12 months of age, with diagnosis possible at 18
             months. To date, studies and evaluations involving
             observational analysis tend to rely heavily on clinical
             practitioners and specialists who have undergone intensive
             training to be able to reliably administer carefully
             designed behavioral-eliciting tasks, code the resulting
             behaviors, and interpret them. These methods are therefore
             extremely expensive, time-intensive, and are not easily
             scalable for large or longitudinal observational analysis.
             We developed a self-contained, closed-loop, mobile
             application with movie stimuli designed to engage the
             child&#x0027;s attention and elicit specific behavioral and
             social responses, which are recorded with the mobile
             device&#x0027;s camera and analyzed via computer vision
             algorithms. Here, in addition to presenting this paradigm,
             we validate the system to measure engagement, name-call, and
             emotional responses of toddlers with and without ASD who
             were presented with the application. Additionally, we
             demonstrate how the proposed framework can further risk
             marker research with fine-grained quantification of
             behaviors. The results suggest these objective and automatic
             methods can be considered to aid behavioral
             analysis.},
   Doi = {10.1109/TAFFC.2018.2868196},
   Key = {fds338017}
}

@article{fds336938,
   Author = {Campbell, K and Carpenter, KL and Hashemi, J and Espinosa, S and Marsan,
             S and Borg, JS and Chang, Z and Qiu, Q and Vermeer, S and Adler, E and Tepper,
             M and Egger, HL and Baker, JP and Sapiro, G and Dawson,
             G},
   Title = {Computer vision analysis captures atypical attention in
             toddlers with autism.},
   Journal = {Autism},
   Pages = {1362361318766247},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1177/1362361318766247},
   Abstract = {To demonstrate the capability of computer vision analysis to
             detect atypical orienting and attention behaviors in
             toddlers with autism spectrum disorder. One hundered and
             four toddlers of 16-31 months old (mean = 22)
             participated in this study. Twenty-two of the toddlers had
             autism spectrum disorder and 82 had typical development or
             developmental delay. Toddlers watched video stimuli on a
             tablet while the built-in camera recorded their head
             movement. Computer vision analysis measured participants'
             attention and orienting in response to name calls.
             Reliability of the computer vision analysis algorithm was
             tested against a human rater. Differences in behavior were
             analyzed between the autism spectrum disorder group and the
             comparison group. Reliability between computer vision
             analysis and human coding for orienting to name was
             excellent (intra-class coefficient 0.84, 95% confidence
             interval 0.67-0.91). Only 8% of toddlers with autism
             spectrum disorder oriented to name calling on >1 trial,
             compared to 63% of toddlers in the comparison group
             (p = 0.002). Mean latency to orient was significantly
             longer for toddlers with autism spectrum disorder (2.02 vs
             1.06 s, p = 0.04). Sensitivity for autism spectrum
             disorder of atypical orienting was 96% and specificity was
             38%. Older toddlers with autism spectrum disorder showed
             less attention to the videos overall (p = 0.03).
             Automated coding offers a reliable, quantitative method for
             detecting atypical social orienting and reduced sustained
             attention in toddlers with autism spectrum
             disorder.},
   Doi = {10.1177/1362361318766247},
   Key = {fds336938}
}

@article{fds328293,
   Author = {Zucker, N and Mauro, C and Craske, M and Wagner, HR and Datta, N and Hopkins, H and Caldwell, K and Kiridly, A and Marsan, S and Maslow, G and Mayer, E and Egger, H},
   Title = {Acceptance-based interoceptive exposure for young children
             with functional abdominal pain.},
   Journal = {Behaviour Research and Therapy},
   Volume = {97},
   Pages = {200-212},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.brat.2017.07.009},
   Abstract = {Functional abdominal pain (FAP) is a common childhood
             somatic complaint that contributes to impairment in daily
             functioning (e.g., school absences) and increases risk for
             chronic pain and psychiatric illness. Cognitive behavioral
             treatments for FAP target primarily older children
             (9 + years) and employ strategies to reduce a focus on
             pain. The experience of pain may be an opportunity to teach
             viscerally hypersensitive children to interpret the function
             of a variety of bodily signals (including those of hunger,
             emotions) thereby reducing fear of bodily sensations and
             facilitating emotion awareness and self-regulation. We
             designed and tested an interoceptive exposure treatment for
             younger children (5-9 years) with FAP. Assessments included
             diagnostic interviews, 14 days of daily pain monitoring, and
             questionnaires. Treatment involved 10 weekly appointments.
             Using cartoon characters to represent bodily sensations
             (e.g., Gassy Gus), children were trained to be "FBI agents"
             - Feeling and Body Investigators - who investigated
             sensations through exercises that provoked somatic
             experience. 24 parent-child dyads are reported. Pain
             (experience, distress, and interference) and negative affect
             demonstrated clinically meaningful and statistically
             significant change with effect sizes ranging from 0.48 to 71
             for pain and from 0.38 to 0.61 for pain distress, total
             pain: X2 (1, n = 24) = 13.14, p < 0.0003. An
             intervention that helps children adopt a curious stance and
             focus on somatic symptoms reduces pain and may help lessen
             somatic fear generally.NCT02075437.},
   Doi = {10.1016/j.brat.2017.07.009},
   Key = {fds328293}
}

@article{fds323858,
   Author = {Campbell, K and Carpenter, KLH and Espinosa, S and Hashemi, J and Qiu,
             Q and Tepper, M and Calderbank, R and Sapiro, G and Egger, HL and Baker,
             JP and Dawson, G},
   Title = {Use of a Digital Modified Checklist for Autism in Toddlers -
             Revised with Follow-up to Improve Quality of Screening for
             Autism.},
   Journal = {The Journal of Pediatrics},
   Volume = {183},
   Pages = {133-139.e1},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jpeds.2017.01.021},
   Abstract = {To assess changes in quality of care for children at risk
             for autism spectrum disorders (ASD) due to process
             improvement and implementation of a digital screening
             form.The process of screening for ASD was studied in an
             academic primary care pediatrics clinic before and after
             implementation of a digital version of the Modified
             Checklist for Autism in Toddlers - Revised with Follow-up
             with automated risk assessment. Quality metrics included
             accuracy of documentation of screening results and
             appropriate action for positive screens (secondary screening
             or referral). Participating physicians completed pre- and
             postintervention surveys to measure changes in attitudes
             toward feasibility and value of screening for ASD. Evidence
             of change was evaluated with statistical process control
             charts and χ2 tests.Accurate documentation in the
             electronic health record of screening results increased from
             54% to 92% (38% increase, 95% CI 14%-64%) and appropriate
             action for children screening positive increased from 25% to
             85% (60% increase, 95% CI 35%-85%). A total of 90% of
             participating physicians agreed that the transition to a
             digital screening form improved their clinical assessment of
             autism risk.Implementation of a tablet-based digital version
             of the Modified Checklist for Autism in Toddlers - Revised
             with Follow-up led to improved quality of care for children
             at risk for ASD and increased acceptability of screening for
             ASD. Continued efforts towards improving the process of
             screening for ASD could facilitate rapid, early diagnosis of
             ASD and advance the accuracy of studies of the impact of
             screening.},
   Doi = {10.1016/j.jpeds.2017.01.021},
   Key = {fds323858}
}


%% Egner, Tobias   
@article{fds337438,
   Author = {Jiang, J and Wagner, AD and Egner, T},
   Title = {Integrated externally and internally generated task
             predictions jointly guide cognitive control in prefrontal
             cortex.},
   Journal = {Elife},
   Volume = {7},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.7554/eLife.39497},
   Abstract = {Cognitive control proactively configures information
             processing to suit expected task demands. Predictions of
             forthcoming demand can be driven by explicit external cues
             or be generated internally, based on past experience
             (cognitive history). However, it is not known whether and
             how the brain reconciles these two sources of information to
             guide control. Pairing a probabilistic task-switching
             paradigm with computational modeling, we found that external
             and internally generated predictions jointly guide task
             preparation, with a bias for internal predictions. Using
             model-based neuroimaging, we then show that the two sources
             of task prediction are integrated in dorsolateral prefrontal
             cortex, and jointly inform a representation of the
             likelihood of a change in task demand, encoded in
             frontoparietal cortex. Upon task-stimulus onset, dorsomedial
             prefrontal cortex encoded the need for reactive task-set
             adjustment. These data reveal how the human brain integrates
             external cues and cognitive history to prepare for an
             upcoming task.},
   Doi = {10.7554/eLife.39497},
   Key = {fds337438}
}

@article{fds335644,
   Author = {Tan, J and Yin, S and Wang, L and Chen, A and Egner,
             T},
   Title = {Processing overlap-dependent distractor dilution rather than
             perceptual target load determines attentional
             selectivity.},
   Journal = {Attention, Perception & Psychophysics},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.3758/s13414-018-1545-4},
   Abstract = {The perceptual load theory of attentional selection argues
             that the degree to which distractors interfere with target
             processing is determined by the "perceptual load" (or
             discrimination difficulty) of target processing: when
             perceptual load is low, distractors interfere to a greater
             extent than when it is high. A well-known exception is
             load-independent interference effects from face distractors
             during processing of name targets. This finding was
             reconciled with load theory by proposing distinct processing
             resources for faces versus names. In the present study, we
             revisit this effect to test (a) whether increasing the
             processing overlap (perceptual, lexical, conceptual) between
             potential targets and distractors would reinstate the
             classic load effect, and (b) whether this data pattern could
             be better explained by load theory or by a rival account
             that argues that distractor dilution rather than target load
             determines the degree of distractor interference. Over four
             experiments, we first replicate the original finding and
             then show that load effects grow with increasing processing
             overlap between potential targets and distractors. However,
             by adding dilution conditions, we also show that these
             processing overlap dependent modulations of distractor
             interference can be explained by the distractor dilution
             perspective but not by perceptual load theory. Thus, our
             findings support a processing overlap dilution account of
             attentional selection.},
   Doi = {10.3758/s13414-018-1545-4},
   Key = {fds335644}
}

@article{fds332979,
   Author = {Bejjani, C and Zhang, Z and Egner, T},
   Title = {Control by association: Transfer of implicitly primed
             attentional states across linked stimuli.},
   Journal = {Psychonomic Bulletin & Review},
   Volume = {25},
   Number = {2},
   Pages = {617-626},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.3758/s13423-018-1445-6},
   Abstract = {Although cognitive control has traditionally been viewed in
             opposition to associative learning, recent studies show that
             people can learn to link particular stimuli with specific
             cognitive control states (e.g., high attentional
             selectivity). Here, we tested whether such learned
             stimulus-control associations can transfer across
             paired-associates. In the Stimulus-Stimulus (S-S)
             Association phase, specific face or house images repeatedly
             preceded the presentation of particular scene stimuli,
             creating paired face/house-scene associates in memory. The
             Stimulus-Control (S-C) Association phase then associated
             these scenes with different attentional control states by
             probabilistically biasing specific scenes to mostly precede
             either congruent or incongruent trials in a Stroop task.
             Finally, in the Stimulus-Control Transfer (S-CT) phase, the
             faces and houses from the S-S phase preceded Stroop trials
             but were not predictive of congruency, testing whether
             stimulus-control associations would transfer from scenes to
             their associated face/house stimuli. In Experiments 1 and 3,
             we found that learned implicit stimulus-control associations
             could transfer across closely linked cues, and in Experiment
             2, we showed that this transfer depended on the memory
             associations formed in the S-S phase. While this form of
             transfer learning has previously been demonstrated for
             stimulus-reward associations, the present study provides the
             first evidence for the associative transfer of
             stimulus-control associations across arbitrarily linked
             stimuli. This work demonstrates how people can learn to
             implicitly adapt their processing strategies in a flexible
             context-dependent manner and establishes a novel learning
             mechanism supporting the generalization of cognitive
             control.},
   Doi = {10.3758/s13423-018-1445-6},
   Key = {fds332979}
}

@article{fds331649,
   Author = {Muhle-Karbe, PS and Jiang, J and Egner, T},
   Title = {Causal Evidence for Learning-Dependent Frontal Lobe
             Contributions to Cognitive Control.},
   Journal = {The Journal of Neuroscience : the Official Journal of the
             Society for Neuroscience},
   Volume = {38},
   Number = {4},
   Pages = {962-973},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1523/JNEUROSCI.1467-17.2017},
   Abstract = {The lateral prefrontal cortex (LPFC) plays a central role in
             the prioritization of sensory input based on task relevance.
             Such top-down control of perception is of fundamental
             importance in goal-directed behavior, but can also be costly
             when deployed excessively, necessitating a mechanism that
             regulates control engagement to align it with changing
             environmental demands. We have recently introduced the
             "flexible control model" (FCM), which explains this
             regulation as resulting from a self-adjusting
             reinforcement-learning mechanism that infers latent
             statistical structure in dynamic task environments to
             predict forthcoming states. From this perspective,
             LPFC-based control is engaged as a function of anticipated
             cognitive demand, a notion for which we previously obtained
             correlative neuroimaging evidence. Here, we put this
             hypothesis to a rigorous, causal test by combining the FCM
             with a transcranial magnetic stimulation (TMS) intervention
             that transiently perturbed the LPFC. Human participants
             (male and female) completed a nonstationary version of the
             Stroop task with dynamically changing probabilities of
             conflict between task-relevant and task-irrelevant stimulus
             features. TMS was given on each trial before stimulus onset
             either over the LPFC or over a control site. In the control
             condition, we observed adaptive performance fluctuations
             consistent with demand predictions that were inferred from
             recent and remote trial history and effectively captured by
             our model. Critically, TMS over the LPFC eliminated these
             fluctuations while leaving basic cognitive and motor
             functions intact. These results provide causal evidence for
             a learning-based account of cognitive control and delineate
             the nature of the signals that regulate top-down biases over
             stimulus processing.SIGNIFICANCE STATEMENT A core function
             of the human prefrontal cortex is to control the signal flow
             in sensory brain regions to prioritize processing of
             task-relevant information. Abundant work suggests that such
             control is flexibly recruited to accommodate dynamically
             changing environmental demands, yet the nature of the
             signals that serve to engage control remains unknown. Here,
             we combined computational modeling with noninvasive brain
             stimulation to show that changes in control engagement are
             captured by a self-adjusting reinforcement-learning
             mechanism that tracks changing environmental statistics to
             predict forthcoming processing demands and that transient
             perturbation of the prefrontal cortex abolishes these
             adjustments. These findings delineate the learning signals
             that underpin adaptive engagement of prefrontal control
             functions and provide causal evidence for their relevance in
             behavioral control.},
   Doi = {10.1523/JNEUROSCI.1467-17.2017},
   Key = {fds331649}
}

@article{fds328427,
   Author = {Kiyonaga, A and Dowd, EW and Egner, T},
   Title = {Neural Representation of Working Memory Content Is Modulated
             by Visual Attentional Demand.},
   Journal = {Journal of Cognitive Neuroscience},
   Volume = {29},
   Number = {12},
   Pages = {2011-2024},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1162/jocn_a_01174},
   Abstract = {Recent theories assert that visual working memory (WM)
             relies on the same attentional resources and sensory
             substrates as visual attention to external stimuli.
             Behavioral studies have observed competitive tradeoffs
             between internal (i.e., WM) and external (i.e., visual)
             attentional demands, and neuroimaging studies have revealed
             representations of WM content as distributed patterns of
             activity within the same cortical regions engaged by
             perception of that content. Although a key function of WM is
             to protect memoranda from competing input, it remains
             unknown how neural representations of WM content are
             impacted by incoming sensory stimuli and concurrent
             attentional demands. Here, we investigated how neural
             evidence for WM information is affected when attention is
             occupied by visual search-at varying levels of
             difficulty-during the delay interval of a WM match-to-sample
             task. Behavioral and fMRI analyses suggested that WM
             maintenance was impacted by the difficulty of a concurrent
             visual task. Critically, multivariate classification
             analyses of category-specific ventral visual areas revealed
             a reduction in decodable WM-related information when
             attention was diverted to a visual search task, especially
             when the search was more difficult. This study suggests that
             the amount of available attention during WM maintenance
             influences the detection of sensory WM representations.},
   Doi = {10.1162/jocn_a_01174},
   Key = {fds328427}
}

@article{fds326206,
   Author = {Chiu, Y-C and Egner, T},
   Title = {Cueing cognitive flexibility: Item-specific learning of
             switch readiness.},
   Journal = {Journal of Experimental Psychology. Human Perception and
             Performance},
   Volume = {43},
   Number = {12},
   Pages = {1950-1960},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1037/xhp0000420},
   Abstract = {The rich behavioral repertoire of the human species derives
             from our ability to flexibly reconfigure processing
             strategies (task sets) in response to changing requirements.
             This updating of task sets is effortful, as reflected by
             longer response times when switching a task than repeating
             it (switch costs). However, some recent data suggest that
             switch costs can be reduced by cueing switch readiness
             bottom-up, by associating particular stimuli with frequent
             switch requirements. This type of "stimulus-control (S-C)
             learning" would be highly adaptive, as it combines the speed
             of automatic (bottom-up) processing with the flexibility and
             generalizability of controlled (top-down) processing.
             However, it is unclear whether S-C learning of switch
             readiness is truly possible, and what the underlying
             mechanisms are. Here we address these questions by pairing
             specific stimuli with a need to update task-sets either
             frequently or rarely. In all 3 experiments, we observe
             robust item-specific switch probability (ISSP) effects as
             revealed by smaller switch costs for frequent switch items
             than for rare switch items. By including a neutral
             condition, we also show that the ISSP effect is primarily
             driven by S-C learning reducing switch costs in frequent
             switch items. Furthermore, by employing 3 tasks in
             Experiment 3, we establish that the ISSP effect reflects an
             enhancement of general switch readiness, rather than of the
             readiness to switch to a specific alternate task. These
             results firmly establish that switch readiness is malleable
             by item-specific S-C learning processes, documenting that a
             generalizable state of cognitive flexibility can be primed
             by a bottom-up stimulus. (PsycINFO Database
             Record},
   Doi = {10.1037/xhp0000420},
   Key = {fds326206}
}

@article{fds331138,
   Author = {Whitehead, PS and Egner, T},
   Title = {Cognitive Control Over Prospective Task-set
             Interference},
   Journal = {Journal of Experimental Psychology. Human Perception and
             Performance},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1037/xhp0000493},
   Abstract = {© 2017 APA, all rights reserved). Recent studies have
             demonstrated that maintaining task-sets in working memory
             (WM) for prospective implementation can interfere with
             performance on an intervening task when the same stimulus
             requires incompatible responses in the ongoing versus the
             prospective task. This prospective task-set interference
             effect has previously been conceptualized as an obligatory
             process, resulting from instruction-based reflexivity (IBR).
             However, the extent to which strategic control can be
             exerted over interference in ongoing behavior from
             prospective task-sets held in WM has heretofore not been
             tested directly. To probe for strategic control over this
             effect, the authors conducted 3 experiments using a common
             inducer-diagnostic task design that manipulated the
             proportion compatibility of trials in the ongoing task. They
             hypothesized that if prospective task-set interference were
             malleable by control, participants would suppress the
             influence of the prospective set on ongoing processing when
             incompatible trials are frequent. Consistent with this
             prediction, the results show that prospective task-set
             interference is subject to modulation by strategic control
             such that the magnitude of interference is reduced,
             eliminated, or reversed in the presence of frequent
             incompatible trials. Thus, the influence on ongoing behavior
             of a prospective task-set held in WM is not obligatory, but
             subject to strategic control. (PsycINFO Database
             Record},
   Doi = {10.1037/xhp0000493},
   Key = {fds331138}
}

@article{fds329983,
   Author = {Qiao, L and Zhang, L and Chen, A and Egner, T},
   Title = {Dynamic Trial-by-Trial Recoding of Task-Set Representations
             in the Frontoparietal Cortex Mediates Behavioral
             Flexibility.},
   Journal = {The Journal of Neuroscience : the Official Journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {45},
   Pages = {11037-11050},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1523/JNEUROSCI.0935-17.2017},
   Abstract = {Cognitive flexibility forms the core of the extraordinary
             ability of humans to adapt, but the precise neural
             mechanisms underlying our ability to nimbly shift between
             task sets remain poorly understood. Recent functional
             magnetic resonance imaging (fMRI) studies employing
             multivoxel pattern analysis (MVPA) have shown that a
             currently relevant task set can be decoded from activity
             patterns in the frontoparietal cortex, but whether these
             regions support the dynamic transformation of task sets from
             trial to trial is not clear. Here, we combined a cued
             task-switching protocol with human (both sexes) fMRI, and
             harnessed representational similarity analysis (RSA) to
             facilitate a novel assessment of trial-by-trial changes in
             neural task-set representations. We first used MVPA to
             define task-sensitive frontoparietal and visual regions and
             found that neural task-set representations on switch trials
             are less stably encoded than on repeat trials. We then
             exploited RSA to show that the neural representational
             pattern dissimilarity across consecutive trials is greater
             for switch trials than for repeat trials, and that the
             degree of this pattern dissimilarity predicts behavior.
             Moreover, the overall neural pattern of representational
             dissimilarities followed from the assumption that repeating
             sets, compared with switching sets, results in stronger
             neural task representations. Finally, when moving from cue
             to target phase within a trial, pattern dissimilarities
             tracked the transformation from previous-trial task
             representations to the currently relevant set. These results
             provide neural evidence for the longstanding assumptions of
             an effortful task-set reconfiguration process hampered by
             task-set inertia, and they demonstrate that frontoparietal
             and stimulus processing regions support "dynamic adaptive
             coding," flexibly representing changing task sets in a
             trial-by-trial fashion.SIGNIFICANCE STATEMENT Humans can
             fluently switch between different tasks, reflecting an
             ability to dynamically configure "task sets," rule
             representations that link stimuli to appropriate responses.
             Recent studies show that neural signals in frontal and
             parietal brain regions can tell us which of two tasks a
             person is currently performing. However, it is not known
             whether these regions are also involved in dynamically
             reconfiguring task-set representations when switching
             between tasks. Here we measured human brain activity during
             task switching and tracked the similarity of neural task-set
             representations from trial to trial. We show that frontal
             and parietal brain regions flexibly recode changing task
             sets in a trial-by-trial fashion, and that task-set
             similarity over consecutive trials predicts
             behavior.},
   Doi = {10.1523/JNEUROSCI.0935-17.2017},
   Key = {fds329983}
}

@article{fds328321,
   Author = {Oh-Descher, H and Beck, JM and Ferrari, S and Sommer, MA and Egner,
             T},
   Title = {Probabilistic inference under time pressure leads to a
             cortical-to-subcortical shift in decision evidence
             integration.},
   Journal = {Neuroimage},
   Volume = {162},
   Pages = {138-150},
   Publisher = {Elsevier},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2017.08.069},
   Abstract = {Real-life decision-making often involves combining multiple
             probabilistic sources of information under finite time and
             cognitive resources. To mitigate these pressures, people
             "satisfice", foregoing a full evaluation of all available
             evidence to focus on a subset of cues that allow for fast
             and "good-enough" decisions. Although this form of
             decision-making likely mediates many of our everyday
             choices, very little is known about the way in which the
             neural encoding of cue information changes when we satisfice
             under time pressure. Here, we combined human functional
             magnetic resonance imaging (fMRI) with a probabilistic
             classification task to characterize neural substrates of
             multi-cue decision-making under low (1500 ms) and high
             (500 ms) time pressure. Using variational Bayesian
             inference, we analyzed participants' choices to track and
             quantify cue usage under each experimental condition, which
             was then applied to model the fMRI data. Under low time
             pressure, participants performed near-optimally,
             appropriately integrating all available cues to guide
             choices. Both cortical (prefrontal and parietal cortex) and
             subcortical (hippocampal and striatal) regions encoded
             individual cue weights, and activity linearly tracked
             trial-by-trial variations in the amount of evidence and
             decision uncertainty. Under increased time pressure,
             participants adaptively shifted to using a satisficing
             strategy by discounting the least informative cue in their
             decision process. This strategic change in decision-making
             was associated with an increased involvement of the
             dopaminergic midbrain, striatum, thalamus, and cerebellum in
             representing and integrating cue values. We conclude that
             satisficing the probabilistic inference process under time
             pressure leads to a cortical-to-subcortical shift in the
             neural drivers of decisions.},
   Doi = {10.1016/j.neuroimage.2017.08.069},
   Key = {fds328321}
}

@article{fds333713,
   Author = {Oh-Descher, H and Beck, J and Ferrari, S and Sommer, MA and Egner,
             T},
   Title = {Probabilistic inference under time pressure leads to a
             cortical-to-subcortical shift in decision evidence
             integration},
   Publisher = {Elsevier},
   Year = {2017},
   Month = {August},
   Abstract = {Real-life decision-making often involves combining multiple
             probabilistic sources of information under finite time and
             cognitive resources. To mitigate these pressures, people
             “satisfice”, foregoing a full evaluation of all
             available evidence to focus on a subset of cues that allow
             for fast and “good-enough” decisions. Although this form
             of decision-making likely mediates many of our everyday
             choices, very little is known about the way in which the
             neural encoding of cue information changes when we satisfice
             under time pressure. Here, we combined human functional
             magnetic resonance imaging (fMRI) with a probabilistic
             classification task to characterize neural substrates of
             multi-cue decision-making under low (1500 ms) and high (500
             ms) time pressure. Using variational Bayesian inference, we
             analyzed participants’ choices to track and quantify cue
             usage under each experimental condition, which was then
             applied to model the fMRI data. Under low time pressure,
             participants performed near-optimally, appropriately
             integrating all available cues to guide choices. Both
             cortical (prefrontal and parietal cortex) and subcortical
             (hippocampal and striatal) regions encoded individual cue
             weights, and activity linearly tracked trial-by-trial
             variations in amount of evidence and decision uncertainty.
             Under increased time pressure, participants adaptively
             shifted to using a satisficing strategy by discounting the
             least informative cue in their decision process. This
             strategic change in decision-making was associated with an
             increased involvement of the dopaminergic midbrain,
             striatum, thalamus, and cerebellum in representing and
             integrating cue values. We conclude that satisficing the
             probabilistic inference process under time pressure leads to
             a cortical-to-subcortical shift in the neural drivers of
             decisions.},
   Key = {fds333713}
}

@article{fds328117,
   Author = {Korb, FM and Jiang, J and King, JA and Egner, T},
   Title = {Hierarchically Organized Medial Frontal Cortex-Basal Ganglia
             Loops Selectively Control Task- and Response-Selection.},
   Journal = {The Journal of Neuroscience : the Official Journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {33},
   Pages = {7893-7905},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1523/JNEUROSCI.3289-16.2017},
   Abstract = {Adaptive behavior requires context-sensitive configuration
             of task-sets that specify time-varying stimulus-response
             mappings. Intriguingly, response time costs associated with
             changing task-sets and motor responses are known to be
             strongly interactive: switch costs at the task level are
             small in the presence of a response-switch but large when
             accompanied by a response-repetition, and vice versa for
             response-switch costs. The reasons behind this well known
             interdependence between task- and response-level control
             processes are currently not well understood. Here, we
             formalized and tested a model assuming a hierarchical
             organization of superordinate task-set and subordinate
             response-set selection processes to account for this effect.
             The model was found to successfully explain the full range
             of behavioral task- and response-switch costs across first
             and second order trial transitions. Using functional
             magnetic resonance imaging (fMRI) in healthy humans, we then
             characterized the neural circuitry mediating these effects.
             We found that presupplementary motor area (preSMA) activity
             tracked task-set control costs, SMA activity tracked
             response-set control costs, and basal ganglia (BG) activity
             mirrored the interaction between task- and response-set
             regulation processes that characterized participants'
             response times. A subsequent fMRI-guided transcranial
             magnetic stimulation experiment confirmed dissociable roles
             of the preSMA and SMA in determining response costs.
             Together, these data provide evidence for a hierarchical
             organization of posterior medial frontal cortex and its
             interaction with the BG, where a superordinate preSMA-BG
             loop establishes task-set selection, which imposes a
             (unidirectional) constraint on a subordinate SMA-BG loop
             that determines response-selection, resulting in the
             characteristic interdependence in task- and response-switch
             costs in behavior.SIGNIFICANCE STATEMENT The ability to use
             context-sensitive task-sets to guide our responses is
             central to human adaptive behavior. Task and response
             selection are strongly interactive: it is more difficult to
             repeat a response in the context of a changing task-set, and
             vice versa. However, the neurocognitive architecture giving
             rise to this interdependence is currently not understood.
             Here we use modeling, neuroimaging, and noninvasive
             neurostimulation to show that this phenomenon derives from a
             hierarchical organization of posterior medial frontal cortex
             and its interaction with the basal ganglia, where a more
             anterior corticostriatal loop establishes task-set
             selection, which constrains a more posterior loop
             responsible for response-selection. These data provide a
             neural explanation for a key behavioral signature of human
             cognitive control.},
   Doi = {10.1523/JNEUROSCI.3289-16.2017},
   Key = {fds328117}
}

@article{fds324879,
   Author = {Dowd, EW and Pearson, JM and Egner, T},
   Title = {Decoding working memory content from attentional
             biases.},
   Journal = {Psychonomic Bulletin & Review},
   Volume = {24},
   Number = {4},
   Pages = {1252-1260},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.3758/s13423-016-1204-5},
   Abstract = {What we are currently thinking influences where we attend.
             The finding that active maintenance of visual items in
             working memory (WM) biases attention toward memory-matching
             objects-even when WM content is irrelevant for attentional
             goals-suggests a tight link between WM and attention. To
             test whether this link is reliable enough to infer specific
             WM content from measures of attentional bias, we applied
             multivariate pattern classification techniques to response
             times from an unrelated visual search task during a WM
             delay. Single-trial WM content was successfully decoded from
             incidental attentional bias within an individual,
             highlighting the specificity and reliability of the
             WM-attention link. Furthermore, classifiers trained on a
             group of individuals predicted WM content in another,
             completely independent individual-implying a shared
             cognitive mechanism of memory-driven attentional bias. The
             existence of such classifiers demonstrates that memory-based
             attentional bias is both a robust and generalizable probe of
             WM.},
   Doi = {10.3758/s13423-016-1204-5},
   Key = {fds324879}
}

@book{fds328428,
   Author = {Egner, T},
   Title = {The Wiley Handbook of Cognitive Control},
   Pages = {656 pages},
   Publisher = {WILEY-BLACKWELL},
   Year = {2017},
   Month = {March},
   ISBN = {1118920546},
   Abstract = {Covering basic theory, new research, and intersections with
             adjacent fields, this is the first comprehensive reference
             work on cognitive control – our ability to use internal
             goals to guide thought and behavior.},
   Key = {fds328428}
}

@article{fds318684,
   Author = {Braem, S and King, JA and Korb, FM and Krebs, RM and Notebaert, W and Egner, T},
   Title = {The Role of Anterior Cingulate Cortex in the Affective
             Evaluation of Conflict.},
   Journal = {Journal of Cognitive Neuroscience},
   Volume = {29},
   Number = {1},
   Pages = {137-149},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1162/jocn_a_01023},
   Abstract = {An influential theory of ACC function argues that this brain
             region plays a crucial role in the affective evaluation of
             performance monitoring and control demands. Specifically,
             control-demanding processes such as response conflict are
             thought to be registered as aversive signals by ACC, which
             in turn triggers processing adjustments to support avoidance
             learning. In support of conflict being treated as an
             aversive event, recent behavioral studies demonstrated that
             incongruent (i.e., conflict inducing), relative to
             congruent, stimuli can speed up subsequent negative,
             relative to positive, affective picture processing. Here, we
             used fMRI to investigate directly whether ACC activity in
             response to negative versus positive pictures is modulated
             by preceding control demands, consisting of conflict and
             task-switching conditions. The results show that negative,
             relative to positive, pictures elicited higher ACC
             activation after congruent, relative to incongruent, trials,
             suggesting that ACC's response to negative (positive)
             pictures was indeed affectively primed by incongruent
             (congruent) trials. Interestingly, this pattern of results
             was observed on task repetitions but disappeared on task
             alternations. This study supports the proposal that conflict
             induces negative affect and is the first to show that this
             affective signal is reflected in ACC activation.},
   Doi = {10.1162/jocn_a_01023},
   Key = {fds318684}
}

@article{fds323840,
   Author = {Chiu, Y-C and Jiang, J and Egner, T},
   Title = {The Caudate Nucleus Mediates Learning of Stimulus-Control
             State Associations.},
   Journal = {The Journal of Neuroscience : the Official Journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {4},
   Pages = {1028-1038},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1523/JNEUROSCI.0778-16.2016},
   Abstract = {A longstanding dichotomy in cognitive psychology and
             neuroscience pits controlled, top-down driven behavior
             against associative, bottom-up driven behavior, where
             cognitive control processes allow us to override
             well-learned stimulus-response (S-R) associations. By
             contrast, some previous studies have raised the intriguing
             possibility of an integration between associative and
             controlled processing in the form of stimulus-control state
             (S-C) associations, the learned linkage of specific stimuli
             to particular control states, such as high attentional
             selectivity. The neural machinery mediating S-C learning
             remains poorly understood, however. Here, we combined human
             functional magnetic resonance imaging (fMRI) with a
             previously developed Stroop protocol that allowed us to
             dissociate reductions in Stroop interference based on S-R
             learning from those based on S-C learning. We modeled
             subjects' acquisition of S-C and S-R associations using an
             associative learning model and then used trial-by-trial S-C
             and S-R prediction error (PE) estimates in model-based
             behavioral and fMRI analyses. We found that PE estimates
             derived from S-C and S-R associations accounted for the
             reductions in behavioral Stroop interference effects in the
             S-C and S-R learning conditions, respectively. Moreover,
             model-based fMRI analyses identified the caudate nucleus as
             the key structure involved in selectively updating
             stimulus-control state associations. Complementary analyses
             also revealed a greater reliance on parietal cortex when
             using the learned S-R versus S-C associations to minimize
             Stroop interference. These results support the emerging view
             that generalizable control states can become associated with
             specific bottom-up cues, and they place the caudate nucleus
             of the dorsal striatum at the center of the neural
             stimulus-control learning machinery.Previous behavioral
             studies have demonstrated that control states, for instance,
             heightened attentional selectivity, can become directly
             associated with, and subsequently retrieved by, particular
             stimuli, thus breaking down the traditional dichotomy
             between top-down and bottom-up driven behavior. However, the
             neural mechanisms underlying this type of stimulus-control
             learning remain poorly understood. We therefore combined
             noninvasive human neuroimaging with a task that allowed us
             to dissociate the acquisition of stimulus-control
             associations from that of stimulus-response associations.
             The results revealed the caudate nucleus as the key brain
             structure involved in selectively driving stimulus-control
             learning. These data represent the first identification of
             the neural mechanisms of stimulus-specific control
             associations, and they significantly extend current
             conceptions of the type of learning processes mediated by
             the caudate.},
   Doi = {10.1523/JNEUROSCI.0778-16.2016},
   Key = {fds323840}
}

@article{fds323485,
   Author = {Mansouri, FA and Egner, T and Buckley, MJ},
   Title = {Monitoring Demands for Executive Control: Shared Functions
             between Human and Nonhuman Primates.},
   Journal = {Trends in Neurosciences},
   Volume = {40},
   Number = {1},
   Pages = {15-27},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.tins.2016.11.001},
   Abstract = {Fifteen years ago, an influential model proposed that the
             human dorsal anterior cingulate cortex (dACC) detects
             conflict and induces adaptive control of behavior. Over the
             years support for this model has been mixed, in particular
             due to divergent findings in human versus nonhuman primates.
             We here review recent findings that suggest greater
             commonalities across species. These include equivalent
             behavioral consequences of conflict and similar neuronal
             signals in the dACC, but also a common failure of dACC
             lesions to reliably abolish conflict-driven behavior. We
             conclude that conflict might be one among many drivers of
             adjustments in executive control and that the ACC might be
             just one component of overlapping distributed systems
             involved in context-dependent learning and behavioral
             control.},
   Doi = {10.1016/j.tins.2016.11.001},
   Key = {fds323485}
}


%% Ehrensaft, Miriam K   
@article{fds329143,
   Author = {Ehrensaft, MK and Westfall, HK and Niolon, PH and Lopez, T and Kamboukos, D and Huang, K-Y and Brotman, LM},
   Title = {Can a Parenting Intervention to Prevent Early Conduct
             Problems Interrupt Girls' Risk for Intimate Partner Violence
             10 Years Later?},
   Journal = {Prevention Science : the Official Journal of the Society for
             Prevention Research},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1007/s11121-017-0831-z},
   Abstract = {This study tests whether a parenting intervention for
             families of preschoolers at risk for conduct problems can
             prevent later risk for intimate partner violence (IPV).
             Ninety-nine preschoolers at familial risk for conduct
             problems were randomly assigned to intervention or control
             conditions. Ten years later, 45 preschoolers and 43 of their
             siblings completed an assessment of their romantic
             relationships, including measures of physical and
             psychological IPV. The study focuses on the 54 females,
             including targets (n = 27) and siblings (n = 27) who
             participated in a 10-year follow-up (M age = 16.5,
             SD = 5.2, range = 10-28). Using an intent-to-treat (ITT)
             design, multivariate regressions suggest that females from
             families randomly assigned to intervention in early
             childhood scored lower than those in the control condition
             on perceptions of dating violence as normative, beliefs
             about IPV prevalence, exposure to IPV in their own peer
             group, and expected sanction behaviors for IPV perpetration
             and victimization. Findings suggest that early parenting
             intervention may reduce association of high-risk females
             with aggressive peers and partners in adolescence.},
   Doi = {10.1007/s11121-017-0831-z},
   Key = {fds329143}
}

@article{fds319604,
   Author = {Ehrensaft, MK and Knous-Westfall, H and Cohen,
             P},
   Title = {Long-term influence of intimate partner violence and
             parenting practices on offspring trauma symptoms.},
   Journal = {Psychology of Violence},
   Volume = {7},
   Number = {2},
   Pages = {296-305},
   Year = {2017},
   url = {http://dx.doi.org/10.1037/a0040168},
   Doi = {10.1037/a0040168},
   Key = {fds319604}
}


%% Fairbank, John A.   
@article{fds336068,
   Author = {Steenkamp, MM and Corry, NH and Qian, M and Li, M and McMaster, HS and Fairbank, JA and Stander, VA and Hollahan, L and Marmar,
             CR},
   Title = {Prevalence of psychiatric morbidity in United States
             military spouses: The Millennium Cohort Family
             Study.},
   Journal = {Depression and Anxiety},
   Volume = {35},
   Number = {9},
   Pages = {815-829},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1002/da.22768},
   Abstract = {Approximately half of US service members are married,
             equating to 1.1 million military spouses, yet the prevalence
             of psychiatric morbidity among military spouses remains
             understudied. We assessed the prevalence and correlates of
             eight mental health conditions in spouses of service members
             with 2-5 years of service.We employed baseline data from the
             Millennium Cohort Family Study, a 21-year longitudinal
             survey following 9,872 military-affiliated married couples
             representing all US service branches and active duty,
             Reserve, and National Guard components. Couples were
             surveyed between 2011 and 2013, a period of high military
             operational activity associated with Operation Iraqi Freedom
             and Operation Enduring Freedom. Primary outcomes included
             depression, anxiety, posttraumatic stress disorder (PTSD),
             panic, alcohol misuse, insomnia, somatization, and binge
             eating, all assessed with validated self-report
             questionnaires.A total of 35.90% of military spouses met
             criteria for at least one psychiatric condition. The most
             commonly endorsed conditions were moderate-to-severe
             somatization symptoms (17.63%) and moderate-to-severe
             insomnia (15.65%). PTSD, anxiety, depression, panic, alcohol
             misuse, and binge eating were endorsed by 9.20%, 6.65%,
             6.05%, 7.07%, 8.16%, and 5.23% of spouses, respectively.
             Having a partner who deployed with combat resulted in higher
             prevalence of anxiety, insomnia, and somatization. Spouses
             had lower prevalence of PTSD, alcohol misuse, and insomnia
             but higher rates of panic and binge eating than service
             members. Both members of a couple rarely endorsed having the
             same psychiatric problem.One third of junior military
             spouses screened positive for one or more psychiatric
             conditions, underscoring the need for high-quality
             prevention and treatment services.},
   Doi = {10.1002/da.22768},
   Key = {fds336068}
}

@article{fds337455,
   Author = {Fairbank, JA and Briggs, EC and Lee, RC and Corry, NH and Pflieger, JC and Gerrity, ET and Amaya-Jackson, LM and Stander, VA and Murphy,
             RA},
   Title = {Mental Health of Children of Deployed and Nondeployed US
             Military Service Members: The Millennium Cohort Family
             Study.},
   Journal = {Journal of Developmental and Behavioral Pediatrics :
             Jdbp},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1097/dbp.0000000000000606},
   Abstract = {Families experience multiple stressors as a result of
             military service. The purpose of this study was to
             investigate the associations among service member deployment
             experiences, family and military factors, and children's
             mental health using baseline data from the Millennium Cohort
             Family Study, a study designed to evaluate the health and
             mental health effects of military service on families,
             including children.This study examined administrative data
             on deployment status (combat, noncombat, and no
             deployments), as well as service member- and spouse-reported
             data on deployment experiences and family functioning in
             relation to the mental health of children in the family who
             were aged 9 to 17 years.Most children were not reported to
             have mental health, emotional, or behavioral difficulties
             regardless of parental deployment status. For an important
             minority of children, however, parental deployments with
             combat, compared with those with no deployment, were
             associated with a parental report of attention-deficit
             disorder/attention-deficit hyperactivity disorder and
             depression as diagnosed by a clinical provider, after
             accounting for demographics, psychosocial context, and
             military factors. Children's odds of a parental report of
             depression were significantly higher in both the combat and
             the noncombat deployment groups than in the no deployment
             group.These findings extend our understanding of the
             association between parental deployments and children's
             mental health, with implications for services and training
             mental health providers serving military
             families.},
   Doi = {10.1097/dbp.0000000000000606},
   Key = {fds337455}
}

@article{fds336067,
   Author = {Swanson, J and Easter, M and Brancu, M and VA Mid-Atlantic MIRECC
             Workgroup, and Fairbank, JA},
   Title = {Informing Federal Policy on Firearm Restrictions for
             Veterans with Fiduciaries: Risk Indicators in the
             Post-Deployment Mental Health Study.},
   Journal = {Administration and Policy in Mental Health},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1007/s10488-018-0881-y},
   Abstract = {This article examines the public safety rationale for a
             federal policy of prohibiting gun sales to veterans with
             psychiatric disabilities who are assigned a fiduciary to
             manage their benefits from the Department of Veterans
             Affairs. The policy was evaluated using data on 3200
             post-deployment veterans from the Iraq and Afghanistan war
             era. Three proxy measures of fiduciary need-based on
             intellectual disability, drug abuse, or acute
             psychopathology-were associated in bivariate analysis with
             interpersonal violence and suicidality. In multivariate
             analysis, statistical significance remained only for the
             measure based on acute psychopathology. Implications for
             reforms to the fiduciary firearm restriction policy are
             discussed.},
   Doi = {10.1007/s10488-018-0881-y},
   Key = {fds336067}
}

@article{fds336069,
   Author = {Martindale, SL and Epstein, EL and Taber, KH and Rowland, JA and Brancu,
             M and Beckham, JC and Calhoun, PS and Dedert, E and Elbogen, EB and Fairbank, JA and Green, KT and Hurley, RA and Kilts, JD and Kimbrel, N and Kirby, A and Marx, CE and McCarthy, G and McDonald, SD and Miller-Mumford, M and Moore, SD and Morey, RA and Naylor, JC and Pickett, TC and Runnals, JJ and Swinkels, C and Szabo, ST and Tupler,
             LA and Van Voorhees and EE and Wagner, HR and Weiner, RD and Yoash-Gantz,
             RE},
   Title = {Behavioral and Health Outcomes Associated With Deployment
             and Nondeployment Acquisition of Traumatic Brain Injury in
             Iraq and Afghanistan Veterans},
   Journal = {Archives of Physical Medicine and Rehabilitation},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.apmr.2018.04.029},
   Doi = {10.1016/j.apmr.2018.04.029},
   Key = {fds336069}
}

@article{fds336070,
   Author = {Elbogen, EB and Wagner, HR and Brancu, M and Kimbrel, NA and Naylor, JC and Swinkels, CM and VA Mid-Atlantic MIRECC Workgroup, and Fairbank,
             JA},
   Title = {Psychosocial Risk Factors and Other Than Honorable Military
             Discharge: Providing Healthcare to Previously Ineligible
             Veterans.},
   Journal = {Military Medicine},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1093/milmed/usx128},
   Abstract = {In response to a strong focus on suicide prevention for all
             veterans, the Department of Veterans Affairs (VA) recently
             revised policy to provide emergency mental healthcare for
             veterans who received Other Than Honorable (OTH) discharges
             from the military. This current study takes a preliminary
             step toward identifying demographic, historic, military,
             clinical, and social characteristics of veterans with OTH
             discharges.N = 1,172 Iraq/Afghanistan-era veterans were
             evaluated between 2005 and 2016 in the multi-site VA
             Mid-Atlantic Mental Illness, Research, Education and
             Clinical Center (MIRECC) Study of Post-Deployment Mental
             Health (PDMH Study).Veterans with OTH discharges constituted
             2.7% of our sample, approximating the estimated rate in the
             overall U.S. veteran population. Compared to veterans
             discharged under honorable conditions, veterans with OTH
             discharges were more likely to be younger and have greater
             odds of reporting family history of drug abuse and
             depression. Further, veterans with OTH discharges reported a
             lower level of social support and were more likely to be
             single, endorse more sleep problems, score higher on
             measures of drug misuse, have a history of incarceration,
             and meet diagnostic criteria for major depressive disorder.
             A subsequent matching analysis provided further evidence of
             the association between OTH discharge and two risk factors:
             drug misuse and incarceration.These findings elucidate
             potential factors associated with veterans with OTH
             discharges, particularly substance abuse and criminal
             justice involvement. Results also indicate higher incidence
             of risk factors that often accompany suicidal ideation and
             should be a highlighted component of healthcare delivery to
             this vulnerable cohort of veterans.},
   Doi = {10.1093/milmed/usx128},
   Key = {fds336070}
}

@article{fds336071,
   Author = {Sun, D and Davis, SL and Haswell, CC and Swanson, CA and Mid-Atlantic
             MIRECC Workgroup, and LaBar, KS and Fairbank, JA and Morey,
             RA},
   Title = {Brain Structural Covariance Network Topology in Remitted
             Posttraumatic Stress Disorder.},
   Journal = {Frontiers in Psychiatry},
   Volume = {9},
   Pages = {90},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.3389/fpsyt.2018.00090},
   Abstract = {Posttraumatic stress disorder (PTSD) is a prevalent, chronic
             disorder with high psychiatric morbidity; however, a
             substantial portion of affected individuals experience
             remission after onset. Alterations in brain network topology
             derived from cortical thickness correlations are associated
             with PTSD, but the effects of remitted symptoms on network
             topology remain essentially unexplored. In this
             cross-sectional study, US military veterans (N = 317)
             were partitioned into three diagnostic groups, current PTSD
             (CURR-PTSD, N = 101), remitted PTSD with lifetime but no
             current PTSD (REMIT-PTSD, N = 35), and trauma-exposed
             controls (CONTROL, n = 181). Cortical thickness was
             assessed for 148 cortical regions (nodes) and suprathreshold
             interregional partial correlations across subjects
             constituted connections (edges) in each group. Four
             centrality measures were compared with characterize
             between-group differences. The REMIT-PTSD and CONTROL groups
             showed greater centrality in left frontal pole than the
             CURR-PTSD group. The REMIT-PTSD group showed greater
             centrality in right subcallosal gyrus than the other two
             groups. Both REMIT-PTSD and CURR-PTSD groups showed greater
             centrality in right superior frontal sulcus than CONTROL
             group. The centrality in right subcallosal gyrus, left
             frontal pole, and right superior frontal sulcus may play a
             role in remission, current symptoms, and PTSD history,
             respectively. The network centrality changes in critical
             brain regions and structural networks are associated with
             remitted PTSD, which typically coincides with enhanced
             functional behaviors, better emotion regulation, and
             improved cognitive processing. These brain regions and
             associated networks may be candidates for developing novel
             therapies for PTSD. Longitudinal work is needed to
             characterize vulnerability to chronic PTSD, and resilience
             to unremitting PTSD.},
   Doi = {10.3389/fpsyt.2018.00090},
   Key = {fds336071}
}

@article{fds331314,
   Author = {Martindale, SL and Farrell-Carnahan, LV and Ulmer, CS and Kimbrel,
             NA and McDonald, SD and Rowland, JA and VA Mid-Atlantic MIRECC
             Registry Workgroup},
   Title = {Sleep quality in returning veterans: The influence of mild
             traumatic brain injury.},
   Journal = {Rehabilitation Psychology},
   Volume = {62},
   Number = {4},
   Pages = {563-570},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1037/rep0000159},
   Abstract = {Sleep disturbance is a key behavioral health concern among
             Iraq and Afghanistan era veterans and is a frequent
             complaint among veterans with a history of mild traumatic
             brain injury (mTBI). Currently, it is unclear whether sleep
             disturbance is a core sequelae of mTBI or if it may be
             related to other behavioral health conditions that are
             commonly present in postdeployment veterans. The purpose of
             this study was to determine if history of mTBI is associated
             with poor sleep quality beyond combat exposure and
             behavioral health concerns, including posttraumatic stress
             disorder (PTSD), mood disorders, anxiety disorders, and
             substance use disorders.Participants included 527 veterans
             who deployed after September 11, 2001, in support of the
             wars in Iraq and Afghanistan. Participants completed the
             Structured Clinical Interview for DSM-IV Disorders, Combat
             Exposure Scale, a structured clinical TBI interview, and the
             Pittsburgh Sleep Quality Index.Deployment-related mTBI was
             associated with poor sleep quality independent of combat
             exposure, PTSD, mood disorders, anxiety disorders, and
             substance use disorders (B = 1.84, p = .001) an average of 6
             years after the injury event. No interaction effects between
             deployment-related mTBI and combat exposure, PTSD, mood
             disorders, anxiety disorders, or substance use disorders on
             sleep quality were detected.Veterans with a history of mTBI
             sustained during deployment reported significantly poorer
             sleep quality than veterans without history of mTBI, even
             when considering combat exposure and behavioral health
             issues. Clinicians should consider mTBI as a potential
             contributor to sleep problems, even years after an injury
             event. (PsycINFO Database Record},
   Doi = {10.1037/rep0000159},
   Key = {fds331314}
}

@article{fds331181,
   Author = {Naylor, JC and Ryan Wagner and H and Brancu, M and Shepherd-Banigan, M and Elbogen, E and Kelley, M and Fecteau, T and Goldstein, K and Kimbrel,
             NA and Marx, CE and VA Mid-Atlantic MIRECC Work Group, and VA
             Mid-Atlantic MIRECC Women Veterans Work Group, and Strauss,
             JL},
   Title = {Self-Reported Pain in Male and Female Iraq/Afghanistan-Era
             Veterans: Associations with Psychiatric Symptoms and
             Functioning.},
   Journal = {Pain Medicine (Malden, Mass.)},
   Volume = {18},
   Number = {9},
   Pages = {1658-1667},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1093/pm/pnw308},
   Abstract = {To examine pain symptoms and co-occurring psychiatric and
             functional indices in male and female Iraq/Afghanistan-era
             veterans.Self-reported data collection and interviews of
             Iraq/Afghanistan-era veterans who participated in a
             multisite study of postdeployment mental health.Veterans
             were enrolled at one of four participating VA sites.Two
             thousand five hundred eighty-seven male and 662 female
             Iraq/Afghanistan-era veterans.Nonparametric Wilcoxon rank
             tests examined differences in pain scores between male and
             female veterans. Chi-square tests assessed differences
             between male and female veterans in the proportion of
             respondents endorsing moderate to high levels of pain vs no
             pain. Multilevel regression analyses evaluated the effect of
             pain on a variety of psychiatric and functional
             measures.Compared with males, female veterans reported
             significantly higher mean levels of headache ( P
              < 0.0001), muscle soreness ( P  < 0.008), and total
             pain ( P  < 0.0001), and were more likely to report the
             highest levels of headache ( P  < 0.0001) and muscle
             soreness ( P  < 0.0039). The presence of pain symptoms
             in Iraq/Afghanistan-era veterans was positively associated
             with psychiatric comorbidity and negatively associated with
             psychosocial functioning. There were no observed gender
             differences in psychiatric and functional indices when
             levels of pain were equated.Although female
             Iraq/Afghanistan-era veterans reported higher levels of pain
             than male veterans overall, male and female veterans
             experienced similar levels of psychiatric and functional
             problems at equivalent levels of reported pain. These
             findings suggest that pain-associated psychological and
             functional impacts are comparable and consequential for both
             male and female veterans.},
   Doi = {10.1093/pm/pnw308},
   Key = {fds331181}
}

@article{fds327056,
   Author = {Brancu, M and Wagner, HR and Morey, RA and Beckham, JC and Calhoun, PS and Tupler, LA and Marx, CE and Taber, KH and Hurley, RA and Rowland, J and McDonald, SD and Hoerle, JM and Moore, SD and Kudler, HS and Weiner, RD and VA Mid-Atlantic MIRECC Workgroup, and Fairbank,
             JA},
   Title = {The Post-Deployment Mental Health (PDMH) study and
             repository: A multi-site study of US Afghanistan and Iraq
             era veterans.},
   Journal = {International Journal of Methods in Psychiatric
             Research},
   Volume = {26},
   Number = {3},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1002/mpr.1570},
   Abstract = {The United States (US) Department of Veterans Affairs (VA)
             Mid-Atlantic Mental Illness Research, Education, and
             Clinical Center (MIRECC) Post-Deployment Mental Health
             (PDMH) multi-site study examines post-deployment mental
             health in US military Afghanistan/Iraq-era veterans. The
             study includes the comprehensive behavioral health
             characterization of over 3600 study participants and the
             genetic, metabolomic, neurocognitive, and neuroimaging data
             for many of the participants. The study design also
             incorporates an infrastructure for a data repository to
             re-contact participants for follow-up studies. The
             overwhelming majority (94%) of participants consented to be
             re-contacted for future studies, and our recently completed
             feasibility study indicates that 73-83% of these
             participants could be reached successfully for enrollment
             into longitudinal follow-up investigations. Longitudinal
             concurrent cohort follow-up studies will be conducted (5-10+
             years post-baseline) to examine predictors of illness
             chronicity, resilience, recovery, functional outcome, and
             other variables, and will include neuroimaging,
             genetic/epigenetic, serum biomarker, and neurocognitive
             studies, among others. To date, the PDMH study has generated
             more than 35 publications from the baseline data and the
             repository has been leveraged in over 20 publications from
             follow-up studies drawing from this cohort. Limitations that
             may affect data collection for a longitudinal follow-up
             study are also presented.},
   Doi = {10.1002/mpr.1570},
   Key = {fds327056}
}

@article{fds336072,
   Author = {Elbogen, EB and Wagner, HR and Kimbrel, NA and Brancu, M and Naylor, J and Graziano, R and Crawford, E and VA Mid-Atlantic MIRECC
             Workgroup},
   Title = {Risk Factors for Concurrent Suicidal Ideation and Violent
             Impulses in Military Veterans.},
   Journal = {Psychological Assessment},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1037/pas0000490},
   Abstract = {Suicide and violence are significant problems in a subset of
             Iraq/Afghanistan-era veterans. This study investigates how
             posttraumatic stress disorder (PTSD) and resilience in
             veterans are associated with suicidal ideation and violent
             impulses while controlling for known covariates of both
             adverse outcomes. Structured clinical interviews were
             conducted of N = 2,543 Iraq/Afghanistan-era U.S. veterans.
             Compared with veterans denying suicidal ideation or violent
             impulses (n = 1,927), veterans endorsing both (n = 171) were
             more likely to meet diagnostic criteria for PTSD, report
             childhood abuse, combat exposure, physical pain symptoms,
             and drug misuse, and less likely to endorse
             self-direction/life purpose. Veterans reporting concurrent
             suicidal ideation and violent impulses had higher odds of
             misusing drugs and reporting pain symptoms relative to
             veterans reporting suicidal ideation only (n = 186) and had
             lower odds of endorsing self-direction/life purpose compared
             with veterans reporting violent impulses only (n = 259). The
             findings underscore the importance of examining drug abuse,
             physical pain symptoms, and self-direction/life purpose, as
             well as PTSD and history of trauma, in the context of
             clinical assessment and empirical research aimed at
             optimizing risk management of suicide and violence in
             military veterans. (PsycINFO Database Record},
   Doi = {10.1037/pas0000490},
   Key = {fds336072}
}

@article{fds326331,
   Author = {McMaster, HS and LeardMann, CA and Speigle, S and Dillman, DA and Millennium Cohort Family Study Team},
   Title = {An experimental comparison of web-push vs. paper-only survey
             procedures for conducting an in-depth health survey of
             military spouses.},
   Journal = {Bmc Medical Research Methodology},
   Volume = {17},
   Number = {1},
   Pages = {73},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1186/s12874-017-0337-1},
   Abstract = {Previous research has found that a "web-push" approach to
             data collection, which involves contacting people by mail to
             request an Internet survey response while withholding a
             paper response option until later in the contact process,
             consistently achieves lower response rates than a
             "paper-only" approach, whereby all respondents are contacted
             and requested to respond by mail.An experiment was designed,
             as part of the Millennium Cohort Family Study, to compare
             response rates, sample representativeness, and cost between
             a web-push and a paper-only approach; each approach
             comprised 3 stages of mail contacts. The invited sample
             (n = 4,935) consisted of spouses married to U.S. Service
             members, who had been serving in the military between 2 and
             5 years as of October, 2011.The web-push methodology
             produced a significantly higher response rate, 32.8%
             compared to 27.8%. Each of the 3 stages of postal contact
             significantly contributed to response for both treatments
             with 87.1% of the web-push responses received over the
             Internet. The per-respondent cost of the paper-only
             treatment was almost 40% higher than the web-push treatment
             group. Analyses revealed no meaningfully significant
             differences between treatment groups in representation.These
             results provide evidence that a web-push methodology is more
             effective and less expensive than a paper-only approach
             among young military spouses, perhaps due to their heavy
             reliance on the internet, and we suggest that this approach
             may be more effective with the general population as they
             become more uniformly internet savvy.},
   Doi = {10.1186/s12874-017-0337-1},
   Key = {fds326331}
}

@article{fds326857,
   Author = {Sege, RD and Amaya-Jackson, L and AMERICAN ACADEMY OF PEDIATRICS
             Committee on Child Abuse and Neglect, Council on Foster
             Care and Adoption and Kinship Care and AMERICAN ACADEMY OF CHILD
             AND ADOLESCENT PSYCHIATRY Committee on Child Maltreatment and Violence and NATIONAL CENTER FOR CHILD TRAUMATIC
             STRESS},
   Title = {Clinical Considerations Related to the Behavioral
             Manifestations of Child Maltreatment.},
   Journal = {Pediatrics},
   Volume = {139},
   Number = {4},
   Pages = {e20170100-e20170100},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1542/peds.2017-0100},
   Abstract = {Children who have suffered early abuse or neglect may later
             present with significant health and behavior problems that
             may persist long after the abusive or neglectful environment
             has been remediated. Neurobiological research suggests that
             early maltreatment may result in an altered psychological
             and physiologic response to stressful stimuli, a response
             that deleteriously affects the child's subsequent
             development. Pediatricians can assist caregivers by helping
             them recognize the abused or neglected child's emotional and
             behavioral responses associated with child maltreatment and
             guide them in the use of positive parenting strategies,
             referring the children and families to evidence-based
             therapeutic treatment and mobilizing available community
             resources.},
   Doi = {10.1542/peds.2017-0100},
   Key = {fds326857}
}


%% Fitzsimons, Gavan J.   
@article{fds332784,
   Title = {Erratum},
   Journal = {Journal of Consumer Research},
   Volume = {44},
   Number = {5},
   Pages = {1174-1174},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/jcr/ucx095},
   Doi = {10.1093/jcr/ucx095},
   Key = {fds332784}
}

@article{fds333551,
   Author = {Acikalin, MY and Watson, KK and Fitzsimons, GJ and Platt,
             ML},
   Title = {Rhesus macaques form preferences for brand logos through sex
             and social status based advertising.},
   Journal = {Plos One},
   Volume = {13},
   Number = {2},
   Pages = {e0193055},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1371/journal.pone.0193055},
   Abstract = {Like humans, monkeys value information about sex and status,
             inviting the hypothesis that our susceptibility to these
             factors in advertising arises from shared, ancestral
             biological mechanisms that prioritize social information. To
             test this idea, we asked whether rhesus macaques (Macaca
             mulatta) show choice behavior that is similar to humans in
             response to sex and social status in advertising. Our
             results show that monkeys form preferences for brand logos
             repeatedly paired with images of macaque genitals and high
             status monkeys. Moreover, monkeys sustain preferences for
             these brand logos even though choosing them provided no
             tangible rewards, a finding that cannot be explained by a
             decision mechanism operating solely on material outcomes.
             Together, our results endorse the hypothesis that the power
             of sex and status in advertising emerges from the
             spontaneous engagement of shared, ancestral neural circuits
             that prioritize information useful for navigating the social
             environment. Finally, our results show that simple
             associative conditioning is sufficient to explain the
             formation of preferences for brand logos paired with sexual
             or status-based images.},
   Doi = {10.1371/journal.pone.0193055},
   Key = {fds333551}
}

@article{fds337739,
   Author = {Gaustad, T and Samuelsen, BM and Warlop, L and Fitzsimons,
             GJ},
   Title = {The perils of self-brand connections: Consumer response to
             changes in brand meaning},
   Journal = {Psychology and Marketing},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1002/mar.21137},
   Abstract = {© 2018 Wiley Periodicals, Inc. Companies commit
             considerable resources to building brand associations that
             resonate with consumers’ identities and facilitate strong
             consumer–brand bonds. The current research investigates a
             potential disadvantage of this popular strategy. The results
             from three studies show that consumers with a high degree of
             self-brand connection respond negatively to brand
             developments (e.g., brand acquisitions and repositioning)
             that change brand meaning. The authors show that this effect
             is due to a change in the identity signaled by the brand.
             The results contrast with existing research, which has
             consistently found that brand connections promote probrand
             behavior and serve as a buffer against negative brand
             information.},
   Doi = {10.1002/mar.21137},
   Key = {fds337739}
}

@article{fds329373,
   Author = {Wu, F and Samper, A and Morales, AC and Fitzsimons,
             GJ},
   Title = {It’s Too Pretty to Use! When and How Enhanced Product
             Aesthetics Discourage Usage and Lower Consumption
             Enjoyment},
   Journal = {Journal of Consumer Research},
   Volume = {44},
   Number = {3},
   Pages = {651-672},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1093/jcr/ucx057},
   Doi = {10.1093/jcr/ucx057},
   Key = {fds329373}
}

@article{fds324822,
   Author = {Zemack-Rugar, Y and Moore, SG and Fitzsimons, GJ},
   Title = {Just do it! Why committed consumers react negatively to
             assertive ads},
   Journal = {Journal of Consumer Psychology},
   Volume = {27},
   Number = {3},
   Pages = {287-301},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.jcps.2017.01.002},
   Doi = {10.1016/j.jcps.2017.01.002},
   Key = {fds324822}
}

@article{fds332785,
   Author = {Brick, DJ and Fitzsimons, GM and Chartrand, TL and Fitzsimons,
             GJ},
   Title = {Coke vs. Pepsi: Brand Compatibility, Relationship Power, and
             Life Satisfaction},
   Journal = {Journal of Consumer Research},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1093/jcr/ucx079},
   Doi = {10.1093/jcr/ucx079},
   Key = {fds332785}
}

@article{fds322756,
   Author = {Brick, DJ and Fitzsimons, GJ},
   Title = {Oppositional brand choice: Using brands to respond to
             relationship frustration},
   Journal = {Journal of Consumer Psychology},
   Volume = {27},
   Number = {2},
   Pages = {257-263},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.jcps.2016.10.002},
   Doi = {10.1016/j.jcps.2016.10.002},
   Key = {fds322756}
}


%% Fitzsimons, Gráinne M   
@article{fds336939,
   Title = {Erratum},
   Journal = {Journal of Consumer Research},
   Volume = {44},
   Number = {5},
   Pages = {1174-1174},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1093/jcr/ucx095},
   Doi = {10.1093/jcr/ucx095},
   Key = {fds336939}
}

@article{fds337738,
   Author = {Fitzsimons, GM and Finkel, EJ},
   Title = {Transactive-Goal-Dynamics Theory: A Discipline-Wide
             Perspective},
   Journal = {Current Directions in Psychological Science},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1177/0963721417754199},
   Abstract = {© The Author(s) 2018. Theories of goal pursuit typically
             conceptualize goal pursuers as isolated actors; in contrast,
             empirical research from diverse areas of psychology has
             demonstrated that goal setting, pursuit, and achievement are
             deeply embedded within social relationships. Because much of
             this emerging literature is developing within subfields with
             minimal cross talk, the potential for integration and
             advances to basic theory has not been realized. The present
             article leverages transactive-goal-dynamics theory in an
             effort to bring these literatures together. In doing so, it
             distills a common set of primary research questions toward
             the goal of promoting a cumulative, integrative,
             interdisciplinary field of research on the ways in which
             goal pursuit is socially embedded.},
   Doi = {10.1177/0963721417754199},
   Key = {fds337738}
}

@article{fds332998,
   Author = {Briskin, JL and Kopetz, CE and Fitzsimons, GM and Slatcher,
             RB},
   Title = {For Better or for Worse? Outsourcing Self-Regulation and
             Goal Pursuit},
   Journal = {Social Psychological and Personality Science},
   Pages = {194855061773611-194855061773611},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1177/1948550617736112},
   Abstract = {© 2017, The Author(s) 2017. In recent years, there has been
             increasing interest in how close relationships can impact
             personal goal pursuit. Extensive research on social support
             has shown that support often facilitates goal pursuit.
             However, Fitzsimons and Finkel found that perceived partner
             support may actually undermine motivation and decrease goal
             pursuit intentions. In this article, we report three
             well-powered studies (N = 850) that investigated the
             conditions under which romantic partners may bolster or
             undermine goal pursuit. In contrast with the original
             Fitzsimons and Finkel’s findings, the results of these
             studies consistently showed that perceived partner support
             bolsters goal pursuit intentions by increasing goal
             commitment. Implications for successful goal pursuit in the
             context of relationships are discussed.},
   Doi = {10.1177/1948550617736112},
   Key = {fds332998}
}

@article{fds332786,
   Author = {Brick, DJ and Fitzsimons, GM and Chartrand, TL and Fitzsimons,
             GJ},
   Title = {Coke vs. Pepsi: Brand Compatibility, Relationship Power, and
             Life Satisfaction},
   Journal = {Journal of Consumer Research},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1093/jcr/ucx079},
   Doi = {10.1093/jcr/ucx079},
   Key = {fds332786}
}

@article{fds330732,
   Author = {Green, PI and Finkel, EJ and Fitzsimons, GM and Gino,
             F},
   Title = {The energizing nature of work engagement: Toward a new
             need-based theory of work motivation},
   Journal = {Research in Organizational Behavior},
   Volume = {37},
   Pages = {1-18},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.riob.2017.10.007},
   Abstract = {© 2017 Elsevier Ltd We present theory suggesting that
             experiences at work that meet employees’ expectations of
             need fulfillment drive work engagement. Employees have needs
             (e.g., a desire to be authentic) and they also have
             expectations for how their job or their organization will
             fulfill them. We argue that experiences at work that confirm
             employees’ need fulfillment expectations yield a positive
             emotional state that is energizing, and that this energy is
             manifested in employees’ behaviors at work. Our theorizing
             draws on a review of the work engagement literature, in
             which we identify three core characteristics of work
             engagement: (a) a positive emotional state that (b) yields a
             feeling of energy and (c) leads to positive work-oriented
             behaviors. These key themes provide the foundation for
             further theorizing suggesting that interactions at work
             confirm or disconfirm employees’ need fulfillment
             expectations, leading to different levels of engagement. We
             extend our theorizing to argue that confirmation, or
             disconfirmation, of different need expectations will yield
             emotional experience of varying magnitudes, with
             confirmation of approach-oriented need expectations exerting
             stronger effects than the confirmation of avoidance-oriented
             need expectations. We close with a review suggesting that
             organizational contextual features influence the expression
             of these needs, sustaining or undermining the positive
             emotional experiences that fuel work engagement.},
   Doi = {10.1016/j.riob.2017.10.007},
   Key = {fds330732}
}


%% Flanagan, Owen   
@article{fds335564,
   Author = {Flanagan, O and Zhao, W},
   Title = {The self and its good vary cross-culturally: A dozen
             self-variations and Chinese familial selves},
   Pages = {287-301},
   Booktitle = {Self, Culture and Consciousness: Interdisciplinary
             Convergences on Knowing and Being},
   Year = {2018},
   Month = {February},
   ISBN = {9789811057779},
   url = {http://dx.doi.org/10.1007/978-981-10-5777-9_17},
   Doi = {10.1007/978-981-10-5777-9_17},
   Key = {fds335564}
}

@book{fds335566,
   Author = {Caruso, G and Flanagan, O},
   Title = {Neuroexistentialism: Meaning, morals, and purpose in the age
             of neuroscience},
   Pages = {1-374},
   Year = {2018},
   Month = {January},
   ISBN = {9780190460723},
   url = {http://dx.doi.org/10.1093/oso/9780190460723.001.0001},
   Abstract = {© Oxford University Press 2018. Existentialism is a concern
             about the foundation of meaning, morals, and purpose.
             Existentialisms arise when some foundation for these
             elements of being is under assault. In the past, first-wave
             existentialism concerned the increasingly apparent inability
             of religion and religious tradition to provide such a
             foundation, as typified in the writings of Kierkegaard,
             Dostoevsky, and Nietzsche. Second-wave existentialism,
             personified philosophically by Sartre, Camus, and de
             Beauvoir, developed in response to the inability of an
             overly optimistic Enlightenment vision of reason and the
             common good to provide such a foundation. There is a
             third-wave existentialism, a new existentialism, developing
             in response to advances in the neurosciences that threaten
             the last vestiges of an immaterial soul or self. With the
             increasing explanatory and therapeutic power of
             neuroscience, the mind no longer stands apart from the world
             to serve as a foundation of meaning. This produces
             foundational anxiety. This collection of new essays explores
             the anxiety caused by this third-wave existentialism and
             some responses to it. It brings together some of the
             world℉s leading philosophers, neuroscientists, cognitive
             scientists, and legal scholars to tackle our
             neuroexistentialist predicament and explore what the mind
             sciences can tell us about morality, love, emotion,
             autonomy, consciousness, selfhood, free will, moral
             responsibility, law, the nature of criminal punishment,
             meaning in life, and purpose.},
   Doi = {10.1093/oso/9780190460723.001.0001},
   Key = {fds335566}
}

@article{fds335565,
   Author = {Flanagan, O and Caruso, G},
   Title = {Neuroexistentialism: Third-wave existentialism},
   Pages = {1-22},
   Booktitle = {Neuroexistentialism: Meaning, Morals, and Purpose in the Age
             of Neuroscience},
   Year = {2018},
   Month = {January},
   ISBN = {9780190460723},
   url = {http://dx.doi.org/10.1093/oso/9780190460723.003.0001},
   Abstract = {© Oxford University Press 2018. Neuroexistentialism is a
             recent expression of existential anxiety over the nature of
             persons. Unlike previous existentialisms,
             neuroexistentialism is not caused by a problem with
             ecclesiastical authority, as was the existentialism
             represented by Kierkegaard, Dostoevsky, and Nietzsche, nor
             by the shock of coming face to face with the moral horror of
             nation state actors and their citizens, as in the
             mid-century existentialism of Sartre and Camus. Rather,
             neuroexistentialism is caused by the rise of the scientific
             authority of the human sciences and a resultant clash
             between the scientific and the humanistic image of persons.
             Flanagan and Caruso explain what neuroexistentialism is and
             how it is related to two earlier existentialisms and they
             spell out how neuroexistentialism makes particularly vivid
             the clash between the humanistic and the scientific image of
             persons. They conclude by providing a brief summary of the
             chapters to follow.},
   Doi = {10.1093/oso/9780190460723.003.0001},
   Key = {fds335565}
}

@article{fds329381,
   Author = {Gyal, P and Flanagan, O},
   Title = {The role of pain in buddhism: The conquest of
             suffering},
   Pages = {288-296},
   Booktitle = {The Routledge Handbook of Philosophy of Pain},
   Year = {2017},
   Month = {June},
   ISBN = {9781315742205},
   url = {http://dx.doi.org/10.4324/9781315742205},
   Doi = {10.4324/9781315742205},
   Key = {fds329381}
}

@article{fds327006,
   Author = {Flanagan, O},
   Title = {Addiction Doesn’t Exist, But it is Bad for
             You},
   Journal = {Neuroethics},
   Volume = {10},
   Number = {1},
   Pages = {91-98},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s12152-016-9298-z},
   Doi = {10.1007/s12152-016-9298-z},
   Key = {fds327006}
}


%% Fuemmeler, Bernard F.   
@article{fds336074,
   Author = {Do, EK and Prom-Wormley, EC and Fuemmeler, BF and Dick, DM and Kendler,
             KS and Maes, HH},
   Title = {Associations Between Initial Subjective Experiences with
             Tobacco and Self-Reported Recent Use in Young
             Adulthood.},
   Journal = {Substance Use & Misuse},
   Volume = {53},
   Number = {14},
   Pages = {2291-2298},
   Year = {2018},
   Month = {December},
   url = {http://dx.doi.org/10.1080/10826084.2018.1473435},
   Abstract = {Youth tobacco use behaviors are predictive of patterns in
             adulthood and effect long-term health outcomes. Yet, few
             studies have examined the effect of initial subjective
             experiences (ISEs) during first tobacco use, which has been
             found to be an indicator of individuals. sensitivity to
             nicotine and vulnerability to dependence.The present study
             aimed to determine the prevalence of ISEs across a variety
             of tobacco products, evaluate the factor structure of ISEs
             by first tobacco product used, and examine the relationship
             between ISEs and recent (30-day) use of tobacco products
             across time, using a university sample.Exploratory factor
             analyses were conducted to identify latent factors present
             with respect to items measuring ISEs with tobacco,
             separately by tobacco product (e.g. cigarettes, cigars,
             hookah, e-cigarettes). Factor scores for positive and
             negative ISEs were calculated. Multiple logistic regression
             analyses were conducted to examine associations between ISEs
             and recent use of each tobacco product, adjusted for age at
             first use, sex, race/ethnicity, and cohort.ISEs differ by
             the first tobacco product used. Associations between factor
             scores for positive and negative ISEs and recent use were
             found across a variety of tobacco products. Overall,
             positive ISEs were more strongly associated with recent use,
             relative to negative ISEs.Further research is needed to
             identify genetic and biological pathways and social contexts
             influencing initial subjective experiences with tobacco use,
             in efforts to delay the initiation for tobacco use and
             reduce risk for continued use among young
             adults.},
   Doi = {10.1080/10826084.2018.1473435},
   Key = {fds336074}
}

@article{fds337133,
   Author = {Gao, L and Liu, X and Millstein, J and Siegmund, KD and Dubeau, L and Maguire, RL and Jim Zhang and J and Fuemmeler, BF and Kollins, SH and Hoyo,
             C and Murphy, SK and Breton, CV},
   Title = {Self-reported prenatal tobacco smoke exposure, AXL gene-body
             methylation, and childhood asthma phenotypes.},
   Journal = {Clinical Epigenetics},
   Volume = {10},
   Number = {1},
   Pages = {98},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1186/s13148-018-0532-x},
   Abstract = {Epigenetic modifications, including DNA methylation, act as
             one potential mechanism underlying the detrimental effects
             associated with prenatal tobacco smoke (PTS) exposure.
             Methylation in a gene called AXL was previously reported to
             differ in response to PTS.We investigated the association
             between PTS and epigenetic changes in AXL and how this was
             related to childhood asthma phenotypes. We tested the
             association between PTS and DNA methylation at multiple CpG
             loci of AXL at birth using Pyrosequencing in two separate
             study populations, the Children's Health Study (CHS, n =
             799) and the Newborn Epigenetic Study (NEST, n = 592).
             Plasma cotinine concentration was used to validate findings
             with self-reported smoking status. The inter-relationships
             among AXL mRNA and miR-199a1 expression, PTS, and AXL
             methylation were examined. Lastly, we evaluated the joint
             effects of AXL methylation and PTS on the risk of asthma and
             related symptoms at age 10 years old.PTS was associated with
             higher methylation level in the AXL gene body in both CHS
             and NEST subjects. In the pooled analysis, exposed subjects
             had a 0.51% higher methylation level in this region compared
             to unexposed subjects (95% CI 0.29, 0.74; p < 0.0001).
             PTS was also associated with 21.2% lower expression of
             miR-199a1 (95% CI - 37.9, - 0.1; p = 0.05), a
             microRNA known to regulate AXL expression. Furthermore, the
             combination of higher AXL methylation and PTS exposure at
             birth increased the risk of recent episodes of bronchitic
             symptoms in childhood.PTS was associated with methylation
             level of AXL and the combination altered the risk of
             childhood bronchitic symptoms.},
   Doi = {10.1186/s13148-018-0532-x},
   Key = {fds337133}
}

@article{fds330367,
   Author = {Sanchez, CE and Barry, C and Sabhlok, A and Russell, K and Majors, A and Kollins, SH and Fuemmeler, BF},
   Title = {Maternal pre-pregnancy obesity and child neurodevelopmental
             outcomes: a meta-analysis.},
   Journal = {Obesity Reviews : an Official Journal of the International
             Association for the Study of Obesity},
   Volume = {19},
   Number = {4},
   Pages = {464-484},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1111/obr.12643},
   Abstract = {This review examined evidence of the association between
             maternal pre-pregnancy overweight/obesity status and child
             neurodevelopmental outcomes. PubMed and PsycINFO databases
             were systematically searched for empirical studies published
             before April 2017 using keywords related to prenatal obesity
             and children's neurodevelopment. Of 1483 identified papers,
             41 were included in the systematic review, and 32 articles
             representing 36 cohorts were included in the meta-analysis.
             Findings indicated that compared with children of normal
             weight mothers, children whose mothers were overweight or
             obese prior to pregnancy were at increased risk for
             compromised neurodevelopmental outcomes (overweight:
             OR = 1.17, 95% CI [1.11, 1.24], I2  = 65.51; obese:
             OR = 1.51; 95% CI [1.35, 1.69], I2  = 79.63).
             Pre-pregnancy obesity increased the risk of attention
             deficit-hyperactivity disorder (OR = 1.62; 95% CI [1.23,
             2.14], I2  = 70.15), autism spectrum disorder
             (OR = 1.36; 95% CI [1.08, 1.70], I2  = 60.52),
             developmental delay (OR = 1.58; 95% CI [1.39, 1.79], I2
              = 75.77) and emotional/behavioural problems (OR = 1.42;
             95% CI [1.26, 1.59], I2  = 87.74). Given the current
             obesity prevalence among young adults and women of
             childbearing age, this association between maternal obesity
             during pregnancy and atypical child neurodevelopment
             represents a potentially high public health
             burden.},
   Doi = {10.1111/obr.12643},
   Key = {fds330367}
}

@article{fds336075,
   Author = {Dozmorov, MG and Bilbo, SD and Kollins, SH and Zucker, N and Do, EK and Schechter, JC and Zhang, JJ and Murphy, SK and Hoyo, C and Fuemmeler,
             BF},
   Title = {Associations between maternal cytokine levels during
             gestation and measures of child cognitive abilities and
             executive functioning.},
   Journal = {Brain, Behavior, and Immunity},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.bbi.2018.03.029},
   Abstract = {Preclinical studies demonstrate that environmentally-induced
             alterations in inflammatory cytokines generated by the
             maternal and fetal immune system can significantly impact
             fetal brain development. Yet, the relationship between
             maternal cytokines during gestation and later cognitive
             ability and executive function remains understudied.
             Children (n = 246) were born of mothers enrolled in the
             Newborn Epigenetic Study - a prospective pre-birth cohort in
             the Southeastern US. We characterized seven cytokines
             [IL-1β, IL-4,IL-6, IL-12p70, IL-17A, tumor necrosis
             factor-α (TNFα), and interferon-γ (IFNγ)] and one
             chemokine (IL-8) from maternal plasma collected during
             pregnancy. We assessed children's cognitive abilities and
             executive functioning at a mean age of 4.5 (SD = 1.1)
             years. Children's DAS-II and NIH toolbox scores were
             regressed on cytokines and the chemokine, controlling for
             maternal age, race, education, body mass index, IQ, parity,
             smoking status, delivery type, gestational weeks, and child
             birth weight and sex. Higher IL-12p70 (βIL-12p70 = 4.26,
             p = 0.023) and IL-17A (βIL-17A = 3.70, p = 0.042)
             levels were related to higher DAS-II GCA score, whereas
             higher IL-1β (βIL-1B = -6.07, p = 0.003) was related
             to lower GCA score. Higher IL-12p70 was related to higher
             performance on NIH toolbox measures of executive functions
             related to inhibitory control and attention (βIL-12p70 =
             5.20, p = 0.046) and cognitive flexibility (βIL-12p70 =
             5.10, p = 0.047). Results suggest that dysregulation in
             gestational immune activity are associated with child
             cognitive ability and executive functioning.},
   Doi = {10.1016/j.bbi.2018.03.029},
   Key = {fds336075}
}

@article{fds336076,
   Author = {Lee, C-T and McClernon, FJ and Kollins, SH and Fuemmeler,
             BF},
   Title = {Childhood ADHD Symptoms and Future Illicit Drug Use: The
             Role of Adolescent Cigarette Use.},
   Journal = {Journal of Pediatric Psychology},
   Volume = {43},
   Number = {2},
   Pages = {162-171},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1093/jpepsy/jsx098},
   Abstract = {The aim of this study is to understand how early cigarette
             use might predict subsequent illicit drug use, especially
             among individuals with attention-deficit hyperactivity
             disorder (ADHD) symptoms during childhood.Data were drawn
             from the National Longitudinal Study of Adolescent Health
             (Waves I-IV). The analysis sample involves participants who
             had not used illicit drugs at Wave I, with no missing
             responses for studied predictors (N = 7,332).Smoking
             status at Wave I (ever regular vs. never regular) and
             childhood ADHD symptoms predicted subsequent illicit drug
             use at Waves II to IV. No interaction effect of smoking
             status at Wave I and childhood ADHD symptoms was found.
             However, an indirect effect from childhood ADHD symptoms on
             illicit drug use was identified, through smoking status at
             Wave I. Similar results were observed for predicting illicit
             drug dependence.The findings support the notion that smoking
             status during early adolescence may mediate the association
             between childhood ADHD symptoms and risk of later adult drug
             use. Interventions to prevent smoking among adolescents may
             be particularly effective at decreasing subsequent drug use,
             especially among children with ADHD symptoms.},
   Doi = {10.1093/jpepsy/jsx098},
   Key = {fds336076}
}

@article{fds333723,
   Author = {Van Voorhees and EE and Dennis, PA and Elbogen, EB and Fuemmeler, B and Neal, LC and Calhoun, PS and Beckham, JC},
   Title = {Characterizing anger-related affect in individuals with
             posttraumatic stress disorder using ecological momentary
             assessment.},
   Journal = {Psychiatry Research},
   Volume = {261},
   Pages = {274-280},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.psychres.2017.12.080},
   Abstract = {This study employed secondary analyses of existing
             ecological momentary assessment (EMA) data to characterize
             hostile and irritable affect in the day-to-day experience of
             52 smokers with, and 65 smokers without, posttraumatic
             stress disorder (PTSD). EMA monitoring occurred over a mean
             of 8.2 days, and participants responded to an average of 2.8
             random prompts/day. Analyses included Wilcoxon rank sum
             tests of group differences, and path analyses of
             cross-lagged multilevel models. Participants with PTSD
             endorsed a significantly higher proportion of total EMA
             entries indicating hostile affect and irritable affect than
             did individuals without PTSD. Cross-lagged analyses
             indicated that over a period of hours, PTSD symptoms
             significantly predicted subsequent hostile and irritable
             affect, but hostile and irritable affect did not predict
             subsequent PTSD symptoms. Findings suggest that day-to-day
             exposure to PTSD-related trauma cues may contribute to
             chronically elevated levels of anger-related affect. Such
             heightened affective arousal may, in turn, underlie an
             increased risk for verbal or physical aggression, as well as
             other health and quality-of-life related impairments
             associated with PTSD. Clinical implications include
             conceptualizing anger treatment in the broader context of
             trauma history and symptoms, and specifically targeting
             physiological arousal and maladaptive hostile cognitions
             triggered by trauma reminders in patients with
             PTSD.},
   Doi = {10.1016/j.psychres.2017.12.080},
   Key = {fds333723}
}

@article{fds333243,
   Author = {Cates, JR and Fuemmeler, BF and Diehl, SJ and Stockton, LL and Porter,
             J and Ihekweazu, C and Gurbani, AS and Coyne-Beasley,
             T},
   Title = {Developing a Serious Videogame for Preteens to Motivate HPV
             Vaccination Decision Making: Land of Secret
             Gardens.},
   Journal = {Games for health journal},
   Volume = {7},
   Number = {1},
   Pages = {51-66},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1089/g4h.2017.0002},
   Abstract = {Vaccination against human papillomavirus (HPV) is routinely
             recommended for ages 11-12, yet in 2016 only 49.5% of women
             and 37.5% of men had completed the three-dose series in the
             United States. Offering information and cues to action
             through a serious videogame for preteens may foster HPV
             vaccination awareness, information seeking, and
             communication.An iterative process was used to develop an
             interactive videogame, Land of Secret Gardens. Three focus
             groups were conducted with 16 boys and girls, ages 11-12,
             for input on game design, acceptability, and functioning.
             Two parallel focus groups explored parents' (n = 9)
             perspectives on the game concept. Three researchers
             identified key themes.Preteens wanted a game that is both
             entertaining and instructional. Some parents were skeptical
             that games could be motivational. A back-story about a
             secret garden was developed as a metaphor for a preteen's
             body and keeping it healthy. The goal is to plant a lush
             secret garden and protect the seedlings by treating them
             with a potion when they sprout to keep them healthy as they
             mature. Points to buy seeds and create the potion are earned
             by playing mini-games. Throughout play, players are exposed
             to messaging about HPV and the benefits of the vaccine. Both
             boys and girls liked the garden concept and getting facts
             about HPV. Parents were encouraged to discuss the game with
             their preteens.Within a larger communication strategy,
             serious games could be useful for engaging preteens in
             health decision making about HPV vaccination.},
   Doi = {10.1089/g4h.2017.0002},
   Key = {fds333243}
}

@article{fds331558,
   Author = {Schechter, JC and Fuemmeler, BF and Hoyo, C and Murphy, SK and Zhang,
             JJ and Kollins, SH},
   Title = {Impact of Smoking Ban on Passive Smoke Exposure in Pregnant
             Non-Smokers in the Southeastern United States.},
   Journal = {International Journal of Environmental Research and Public
             Health},
   Volume = {15},
   Number = {1},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.3390/ijerph15010083},
   Abstract = {Prenatal passive smoke exposure raises risk for negative
             birth outcomes. Legislation regulating public smoking has
             been shown to impact exposure levels, though fewer studies
             involving pregnant women have been conducted within the U.S.
             where bans are inconsistent across regions. This study
             examined the effect of a ban enacted in the southeastern
             U.S. on pregnant women's cotinine levels. Additional
             analyses compared self-reported exposure to cotinine and
             identified characteristics associated with passive exposure.
             Pregnant women (N = 851) were recruited prospectively
             between 2005 and 2011 in North Carolina. Sociodemographic
             and health data were collected via surveys; maternal blood
             samples were assayed for cotinine. Among non-active smokers
             who provided self-report data regarding passive exposure (N
             = 503), 20% were inconsistent with corresponding cotinine.
             Among all non-smokers (N = 668), being unmarried, African
             American, and less educated were each associated with
             greater passive exposure. Controlling for covariates, mean
             cotinine was higher prior to the ban compared to after, F(1,
             640) = 24.65, p < 0.001. Results suggest that banning
             smoking in public spaces may reduce passive smoke exposure
             for non-smoking pregnant women. These data are some of the
             first to examine the impact of legislation on passive smoke
             exposure in pregnant women within the U.S. using a biomarker
             and can inform policy in regions lacking comprehensive
             smoke-free legislation.},
   Doi = {10.3390/ijerph15010083},
   Key = {fds331558}
}

@article{fds331494,
   Author = {Chawla, D and Fuemmeler, B and Benjamin-Neelon, SE and Hoyo, C and Murphy, S and Daniels, JL},
   Title = {Early prenatal vitamin D concentrations and social-emotional
             development in infants.},
   Journal = {The Journal of Maternal Fetal & Neonatal Medicine : the
             Official Journal of the European Association of Perinatal
             Medicine, the Federation of Asia and Oceania Perinatal
             Societies, the International Society of Perinatal
             Obstetricians},
   Pages = {1-8},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1080/14767058.2017.1408065},
   Abstract = {Many pregnant women in the United States have suboptimal
             vitamin D, but the impact on infant development is unclear.
             Moreover, no pregnancy-specific vitamin D recommendations
             have been widely accepted.Given the ubiquitous expression of
             vitamin D receptors in the brain, we investigated the
             association between early prenatal plasma 25-hydroxyvitamin
             D (25(OH)D) concentrations and children's social and
             emotional development in the Newborn Epigenetic Study, a
             prospective study of pregnancies from 2009 to 2011 in
             Durham, North Carolina.We measured 25(OH)D concentrations in
             first or second trimester plasma samples and categorized
             25(OH)D concentrations into quartiles. Covariates were
             derived from maternal questionnaires. Mothers completed the
             Infant Toddler Social-Emotional Development Assessment when
             children were 12-24 months of age. We used multivariable
             linear regression to evaluate associations between 25(OH)D
             and specific behavior scores, adjusted for season of blood
             draw, maternal age, education, parity, smoking, marital
             status, prepregnancy BMI, and infant gender. We investigated
             effect-measure modification by race/ethnicity.Of the 218
             mother-infant pairs with complete data, Black mothers had
             much lower 25(OH)D concentrations as compared to White and
             Hispanic mothers. After adjustment, lower prenatal 25(OH)D
             was associated with slightly higher (less favorable)
             Internalizing scores among White children, but lower (more
             favorable) Internalizing scores among Black and Hispanic
             children. Lower prenatal 25(OH)D also appears to be
             associated with higher (less favorable) dysregulation
             scores, though only among White and Hispanic children.Though
             imprecise, preliminary results warrant further investigation
             regarding a role for prenatal vitamin D on children's early
             social and emotional development.},
   Doi = {10.1080/14767058.2017.1408065},
   Key = {fds331494}
}

@article{fds329030,
   Author = {McCullough, LE and Miller, EE and Calderwood, LE and Shivappa, N and Steck, SE and Forman, MR and A Mendez and M and Maguire, R and Fuemmeler,
             BF and Kollins, SH and D Bilbo and S and Huang, Z and Murtha, AP and Murphy,
             SK and Hébert, JR and Hoyo, C},
   Title = {Maternal inflammatory diet and adverse pregnancy outcomes:
             Circulating cytokines and genomic imprinting as potential
             regulators?},
   Journal = {Epigenetics},
   Volume = {12},
   Number = {8},
   Pages = {688-697},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1080/15592294.2017.1347241},
   Abstract = {Excessive inflammation during pregnancy alters homeostatic
             mechanisms of the developing fetus and has been linked to
             adverse pregnancy outcomes. An anti-inflammatory diet could
             be a promising avenue to combat the pro-inflammatory state
             of pregnancy, particularly in obese women, but we lack
             mechanistic data linking this dietary pattern during
             pregnancy to inflammation and birth outcomes. In an
             ethnically diverse cohort of 1057 mother-child pairs, we
             estimated the relationships between dietary inflammatory
             potential [measured via the energy-adjusted dietary
             inflammatory index (E-DII™)] and birth outcomes overall,
             as well as by offspring sex and maternal pre-pregnancy body
             mass index (BMI). In a subset of women, we also explored
             associations between E-DII, circulating cytokines (n = 105),
             and offspring methylation (n = 338) as potential modulators
             of these relationships using linear regression. Adjusted
             regression models revealed that women with pro-inflammatory
             diets had elevated rates of preterm birth among female
             offspring [β = -0.22, standard error (SE) = 0.07, P<0.01],
             but not male offspring (β=0.09, SE = 0.06, P<0.12)
             (Pinteraction = 0.003). Similarly, we observed
             pro-inflammatory diets were associated with higher rates of
             caesarean delivery among obese women (β = 0.17, SE = 0.08,
             P = 0.03), but not among women with BMI <25 kg/m2
             (Pinteraction = 0.02). We observed consistent inverse
             associations between maternal inflammatory cytokine
             concentrations (IL-12, IL-17, IL-4, IL-6, and TNFα) and
             lower methylation at the MEG3 regulatory sequence (P<0.05);
             however, results did not support the link between maternal
             E-DII and circulating cytokines. We replicate work by others
             on the association between maternal inflammatory diet and
             adverse pregnancy outcomes and provide the first empirical
             evidence supporting the inverse association between
             circulating cytokine concentrations and offspring
             methylation.},
   Doi = {10.1080/15592294.2017.1347241},
   Key = {fds329030}
}

@article{fds326256,
   Author = {Fuemmeler, BF and Behrman, P and Taylor, M and Sokol, R and Rothman, E and Jacobson, LT and Wischenka, D and Tercyak, KP},
   Title = {Child and family health in the era of prevention: new
             opportunities and challenges.},
   Journal = {Journal of Behavioral Medicine},
   Volume = {40},
   Number = {1},
   Pages = {159-174},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1007/s10865-016-9791-1},
   Abstract = {To maintain positive health outcomes over the life course,
             prevention efforts should begin early in childhood. Two
             research domains that significantly impact the trajectory of
             health over the life course are childhood obesity and early
             trauma and violence. Prevention strategies addressing
             multiple levels of influence are being adopted in these
             fields. Childhood obesity prevention efforts no longer focus
             solely on individuals, but embrace multiple ecological
             levels, such as family, school, and community. Similarly,
             research on early trauma and violence has broadened to
             consider risk and protective factors across domains of
             influence. Although we have improved our understanding and
             prevention of these two issues, gaps remain in research,
             practice, and policy. The purpose of this review is to relay
             relevant findings that could enhance prevention strategies.
             We describe early life and multilevel risk factors relevant
             to these areas of research. We also provide recommendations
             for future efforts to better ensure good health for
             generations to come.},
   Doi = {10.1007/s10865-016-9791-1},
   Key = {fds326256}
}

@article{fds72465,
   Title = {Fuemmeler BF, Agurs-Collins T, McClernon FJ, Kollins SH,
             Kail ME, Bergen AW, Ashley-Koch AE.  Genes implicated in
             serotonergic and dopamine functioning interact with gender
             to predict BMI categories: Findings from a nationally
             representative sample of young adults.  Obesity (in
             press) },
   Key = {fds72465}
}

@article{fds72466,
   Title = {McClernon FJ, Fuemmeler BF, Kollins SH, Kail ME, Ashley-Koch
             AE. Interactions between genotype and retrospective ADHD
             symptoms predict lifetime smoking risk in a sample of young
             adults. Nicotine & Tobacco Research (in press)
               },
   Key = {fds72466}
}

@article{fds202522,
   Title = {Fuemmeler, B. F., Yang, C., Costanzo, P., Hoyle, R.H.,
             Siegler, I.C., Williams, R.B., Ostbye, T. (in press).
             Parenting styles and BMI trajectories from adolescence into
             adulthood: A longitudinal study of the Add Health cohort.
             Health Psychology},
   Key = {fds202522}
}

@article{fds202523,
   Title = {Ranby, K.W., Boynton, M.H., Kollins, S. H., McClernon, J.,
             Yang, C., & Fuemmeler, B.F. (in press). Understanding the
             phenotypic structure of ADHD in a US population-based
             sample. Journal of Clinical Child and Adolescent Psychology.
              },
   Key = {fds202523}
}

@article{fds211227,
   Title = {Fuemmeler, B.F., Lovelady, C.A., Zucker, N.L., Ostbye, T.
             (in press). Parental obesity moderates relationship between
             childhood appetitive traits and weight. Obesity},
   Key = {fds211227}
}

@article{fds211230,
   Title = {Fuemmeler, B.F., Pendzich, M.K., Clark, K., Rossoff, P.,
             Blatt, J., Lovelady , C., Demark-Wahnefried, W. (in press).
             Changes in weight, body composition, diet, and physical
             activity during the first year of treatment for childhood
             acute leukemia and lymphoma. Journal of Pediatric Hematology
             and Oncology },
   Key = {fds211230}
}

@article{fds215514,
   Title = {Fuemmeler, B.F., Lovelady, C.A., Zucker, N.L., Ostbye, T.
             (in press). Parental obesity moderates relationship between
             childhood eating behaviors and weight. Obesity },
   Key = {fds215514}
}


%% Gaffrey, Michael S.   
@article{fds337154,
   Author = {Gaffrey, MS and Tillman, R and Barch, DM and Luby,
             JL},
   Title = {Continuity and stability of preschool depression from
             childhood through adolescence and following the onset of
             puberty.},
   Journal = {Comprehensive Psychiatry},
   Volume = {86},
   Pages = {39-46},
   Year = {2018},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.comppsych.2018.07.010},
   Abstract = {A growing body of research now supports the validity,
             clinical significance, and long-term negative impact of
             depression occurring during the preschool period. However,
             the prospective continuity of depressive symptoms and risk
             for major depressive disorder (MDD) from childhood through
             adolescence for preschoolers experiencing this highly
             impairing disorder remains unexplored. Such information is
             likely to be critical for understanding the developmental
             continuity of preschool depression and whether it continues
             to be a salient risk factor for an MDD diagnosis following
             the transition into adolescence and the onset of biological
             changes associated with it (i.e., puberty).Subjects were
             participants in the Preschool Depression Study conducted at
             the Early Emotional Development Program at Washington
             University School of Medicine in St. Louis. Subjects and
             their parents completed baseline assessments that included
             comprehensive measures of psychopathology and development at
             baseline and up to 9 follow-up assessments between 2003 and
             2017. N = 279 subjects had diagnostic and clinical data
             available for the preschool period and the early pubertal
             and/or later pubertal periods and were included in the
             analyses. There were N = 275 subjects assessed during
             the early pubertal period and N = 184 subjects assessed
             during the later pubertal period.Preschool depression was a
             highly salient predictor of prepubertal and mid-to-post
             pubertal MDD. Across all modeled time points children with a
             history of preschool depression continued to demonstrate
             elevated levels of depressive symptoms from childhood
             through adolescence, suggesting a heightened trajectory of
             depressive symptoms relative to their same age
             peers.Findings from the current study suggest that children
             with a history of preschool depression follow a trajectory
             of depression severity elevated relative to their same age
             peers from childhood through adolescence but with a similar
             shape over time. They also support the homotypic continuity
             of preschool depression into adolescence and the onset of
             puberty.},
   Doi = {10.1016/j.comppsych.2018.07.010},
   Key = {fds337154}
}

@article{fds337475,
   Author = {Somerville, LH and Bookheimer, SY and Buckner, RL and Burgess, GC and Curtiss, SW and Dapretto, M and Elam, JS and Gaffrey, MS and Harms, MP and Hodge, C and Kandala, S and Kastman, EK and Nichols, TE and Schlaggar,
             BL and Smith, SM and Thomas, KM and Yacoub, E and Van Essen and DC and Barch,
             DM},
   Title = {The Lifespan Human Connectome Project in Development: A
             large-scale study of brain connectivity development in 5-21
             year olds.},
   Journal = {Neuroimage},
   Volume = {183},
   Pages = {456-468},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2018.08.050},
   Abstract = {Recent technological and analytical progress in brain
             imaging has enabled the examination of brain organization
             and connectivity at unprecedented levels of detail. The
             Human Connectome Project in Development (HCP-D) is
             exploiting these tools to chart developmental changes in
             brain connectivity. When complete, the HCP-D will comprise
             approximately ∼1750 open access datasets from
             1300 + healthy human participants, ages 5-21 years,
             acquired at four sites across the USA. The participants are
             from diverse geographical, ethnic, and socioeconomic
             backgrounds. While most participants are tested once, others
             take part in a three-wave longitudinal component focused on
             the pubertal period (ages 9-17 years). Brain imaging
             sessions are acquired on a 3 T Siemens Prisma platform and
             include structural, functional (resting state and
             task-based), diffusion, and perfusion imaging, physiological
             monitoring, and a battery of cognitive tasks and
             self-reports. For minors, parents additionally complete a
             battery of instruments to characterize cognitive and
             emotional development, and environmental variables relevant
             to development. Participants provide biological samples of
             blood, saliva, and hair, enabling assays of pubertal
             hormones, health markers, and banked DNA samples. This paper
             outlines the overarching aims of the project, the approach
             taken to acquire maximally informative data while minimizing
             participant burden, preliminary analyses, and discussion of
             the intended uses and limitations of the
             dataset.},
   Doi = {10.1016/j.neuroimage.2018.08.050},
   Key = {fds337475}
}

@article{fds335645,
   Author = {Gaffrey, MS and Barch, DM and Bogdan, R and Farris, K and Petersen, SE and Luby, JL},
   Title = {Amygdala Reward Reactivity Mediates the Association Between
             Preschool Stress Response and Depression
             Severity.},
   Journal = {Biological Psychiatry},
   Volume = {83},
   Number = {2},
   Pages = {128-136},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.biopsych.2017.08.020},
   Abstract = {Research in adolescents and adults has suggested that
             altered neural processing of reward following early life
             adversity is a highly promising depressive intermediate
             phenotype. However, very little is known about how stress
             response, neural processing of reward, and depression are
             related in very young children. The present study examined
             the concurrent associations between cortisol response
             following a stressor, functional brain activity to reward,
             and depression severity in children 4 to 6 years
             old.Medication-naïve children 4 to 6 years old (N = 52)
             participated in a study using functional magnetic resonance
             imaging to assess neural reactivity to reward, including
             gain, loss, and neutral outcomes. Parent-reported child
             depression severity and child cortisol response following
             stress were also measured.Greater caudate and medial
             prefrontal cortex reactivity to gain outcomes and increased
             amygdala reactivity to salient (i.e., both gain and loss)
             outcomes were observed. Higher total cortisol output
             following a stressor was associated with increased
             depression severity and reduced amygdala reactivity to
             salient outcomes. Amygdala reactivity was also inversely
             associated with depression severity and was found to mediate
             the relationship between cortisol output and depression
             severity.Results suggest that altered neural processing of
             reward is already related to increased cortisol output and
             depression severity in preschoolers. These results also
             demonstrate an important role for amygdala function as a
             mediator of this relationship at a very early age. Our
             results further underscore early childhood as an important
             developmental period for understanding the neurobiological
             correlates of early stress and increased risk for
             depression.},
   Doi = {10.1016/j.biopsych.2017.08.020},
   Key = {fds335645}
}


%% Gaither, Sarah E.   
@article{fds335674,
   Author = {Chen, JM and Pauker, K and Gaither, SE and Hamilton, DL and Sherman,
             JW},
   Title = {Black + White = Not White: A minority bias in
             categorizations of Black-White multiracials},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {78},
   Pages = {43-54},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.jesp.2018.05.002},
   Doi = {10.1016/j.jesp.2018.05.002},
   Key = {fds335674}
}

@article{fds337470,
   Author = {Albuja, AF and Sanchez, DT and Gaither, SE},
   Title = {Identity Denied: Comparing American or White Identity Denial
             and Psychological Health Outcomes Among Bicultural and
             Biracial People.},
   Journal = {Personality & Social Psychology Bulletin},
   Pages = {146167218788553},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1177/0146167218788553},
   Abstract = {Because bicultural and biracial people have two identities
             within one social domain (culture or race), their
             identification is often challenged by others. Although it is
             established that identity denial is associated with poor
             psychological health, the processes through which this
             occurs are less understood. Across two high-powered studies,
             we tested identity autonomy, the perceived compatibility of
             identities, and social belonging as mediators of the
             relationship between identity denial and well-being among
             bicultural and biracial individuals. Bicultural and biracial
             participants who experienced challenges to their American or
             White identities felt less freedom in choosing an identity
             and perceived their identities as less compatible, which was
             ultimately associated with greater reports of depressive
             symptoms and stress. Study 2 replicated these results and
             measured social belonging, which also accounted for
             significant variance in well-being. The results suggest the
             processes were similar across populations, highlighting
             important implications for the generalizability to other
             dual-identity populations.},
   Doi = {10.1177/0146167218788553},
   Key = {fds337470}
}

@article{fds335675,
   Author = {Gaither, SE},
   Title = {The multiplicity of belonging: Pushing identity research
             beyond binary thinking},
   Journal = {Self and Identity},
   Volume = {17},
   Number = {4},
   Pages = {443-454},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1080/15298868.2017.1412343},
   Doi = {10.1080/15298868.2017.1412343},
   Key = {fds335675}
}

@article{fds335676,
   Author = {Albuja, AF and Sanchez, DT and Gaither, SE},
   Title = {Fluid racial presentation: Perceptions of contextual
             “passing” among biracial people},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {77},
   Pages = {132-142},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.jesp.2018.04.010},
   Doi = {10.1016/j.jesp.2018.04.010},
   Key = {fds335676}
}

@article{fds335677,
   Author = {Gaither, SE and Toosi, NR and Babbitt, LG and Sommers,
             SR},
   Title = {Exposure to Biracial Faces Reduces Colorblindness.},
   Journal = {Personality & Social Psychology Bulletin},
   Pages = {146167218778012},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1177/0146167218778012},
   Abstract = {Across six studies, we demonstrate that exposure to biracial
             individuals significantly reduces endorsement of
             colorblindness as a racial ideology among White individuals.
             Real-world exposure to biracial individuals predicts lower
             levels of colorblindness compared with White and Black
             exposure (Study 1). Brief manipulated exposure to images of
             biracial faces reduces colorblindness compared with exposure
             to White faces, Black faces, a set of diverse monoracial
             faces, or abstract images (Studies 2-5). In addition, these
             effects occur only when a biracial label is paired with the
             face rather than resulting from the novelty of the
             mixed-race faces themselves (Study 4). Finally, we show that
             the shift in White participants' colorblindness attitudes is
             driven by social tuning, based on participants' expectations
             that biracial individuals are lower in colorblindness than
             monoracial individuals (Studies 5-6). These studies suggest
             that the multiracial population's increasing size and
             visibility has the potential to positively shift racial
             attitudes.},
   Doi = {10.1177/0146167218778012},
   Key = {fds335677}
}

@article{fds335678,
   Author = {Pauker, K and Meyers, C and Sanchez, DT and Gaither, SE and Young,
             DM},
   Title = {A review of multiracial malleability: Identity,
             categorization, and shifting racial attitudes},
   Journal = {Social and Personality Psychology Compass},
   Volume = {12},
   Number = {6},
   Pages = {e12392-e12392},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1111/spc3.12392},
   Doi = {10.1111/spc3.12392},
   Key = {fds335678}
}

@article{fds335681,
   Author = {Loyd, AB and Gaither, SE},
   Title = {Racial/ethnic socialization for White youth: What we know
             and future directions},
   Journal = {Journal of Applied Developmental Psychology},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.appdev.2018.05.004},
   Abstract = {© 2018 Elsevier Inc. Teaching and talking about race and
             ethnicity with children and adults is especially important
             in racially diverse societies. This process has been coined
             racial/ethnic socialization (RES). Despite the importance of
             RES, we still know very little about how this process
             unfolds in the lives of White youth. Thus, from a social,
             cognitive, and developmental perspective, the authors
             summarize findings from empirical research and theory on RES
             for White youth across stages of development—early
             childhood through young adulthood. Since RES is linked with
             cross-group attitudes (e.g., less bias, prejudice,
             stereotyping) and behaviors (e.g., inclusion), we highlight
             future directions for research and discuss applications for
             existing findings for an increasingly diverse
             society.},
   Doi = {10.1016/j.appdev.2018.05.004},
   Key = {fds335681}
}

@article{fds335679,
   Author = {Gaither, SE and Babbitt, LG and Sommers, SR},
   Title = {Resolving racial ambiguity in social interactions},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {76},
   Pages = {259-269},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.jesp.2018.03.003},
   Doi = {10.1016/j.jesp.2018.03.003},
   Key = {fds335679}
}

@article{fds332755,
   Author = {Gaither, SE and Apfelbaum, EP and Birnbaum, HJ and Babbitt, LG and Sommers, SR},
   Title = {Mere Membership in Racially Diverse Groups Reduces
             Conformity},
   Journal = {Social Psychological and Personality Science},
   Volume = {9},
   Number = {4},
   Pages = {402-410},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1177/1948550617708013},
   Doi = {10.1177/1948550617708013},
   Key = {fds332755}
}

@article{fds335680,
   Author = {Chen, EE and Corriveau, KH and Lai, VKW and Poon, SL and Gaither,
             SE},
   Title = {Learning and Socializing Preferences in Hong Kong Chinese
             Children.},
   Journal = {Child Development},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1111/cdev.13083},
   Abstract = {The impact of social group information on the learning and
             socializing preferences of Hong Kong Chinese children were
             examined. Specifically, the degree to which variability in
             racial out-group exposure affects children's use of race to
             make decisions about unfamiliar individuals (Chinese, White,
             Southeast Asian) was investigated. Participants (N = 212;
             Mage  = 60.51 months) chose functions for novel objects
             after informants demonstrated their use; indicated with
             which peer group member to socialize; and were measured on
             racial group recognition, preference, and identification.
             Overall, children preferred in-group members, though
             out-group exposure and the relative social status of
             out-groups mattered as well. At a young age, children's
             specific experiences with different races influence how they
             learn and befriend others across racial group
             lines.},
   Doi = {10.1111/cdev.13083},
   Key = {fds335680}
}

@article{fds330902,
   Author = {Dukes, KN and Gaither, SE},
   Title = {Black Racial Stereotypes and Victim Blaming: Implications
             for Media Coverage and Criminal Proceedings in Cases of
             Police Violence against Racial and Ethnic
             Minorities},
   Journal = {Journal of Social Issues},
   Volume = {73},
   Number = {4},
   Pages = {789-807},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1111/josi.12248},
   Abstract = {© 2017 The Society for the Psychological Study of Social
             Issues Posthumous stereotypical media portrayals of Michael
             Brown and other racial and ethnic minority victims of police
             violence have sparked questions about the influence of
             racial stereotypes on public opinions about their deaths and
             criminal proceedings for their killers. However, few studies
             have empirically investigated how the specific type of
             information released about a victim impacts opinions
             surrounding such incidents. Participants (N = 453) read
             about an altercation that resulted in a shooting death where
             the race of the victim and shooter (Black vs. White) was
             randomly assigned. Participants learned either negative,
             Black male stereotypic or positive, Black male
             counterstereotypic information about the victim. Next,
             participants appraised levels of fault and blame, sympathy
             and empathy for the victim and shooter, and indictment
             recommendations for the shooter. Findings suggest that the
             type of information released about a victim can
             significantly sway attitudes toward the victim and the
             shooter. Implications for media portrayals of racial/ethnic
             minority victims of police violence and its impact on
             criminal sentencing are discussed.},
   Doi = {10.1111/josi.12248},
   Key = {fds330902}
}

@article{fds330903,
   Author = {Gomez, EM and Young, DM and Preston, AG and Wilton, LS and Gaither, SE and Kaiser, CR},
   Title = {Loss and loyalty: Change in political and gender identity
             among Clinton supporters after the 2016 U.S. presidential
             election},
   Journal = {Self and Identity},
   Pages = {1-23},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1080/15298868.2017.1391873},
   Doi = {10.1080/15298868.2017.1391873},
   Key = {fds330903}
}


%% Gassman-Pines, Anna   
@article{fds337288,
   Author = {Gassman-Pines, A and Bellows, L},
   Title = {Food Instability and Academic Achievement: A
             Quasi-Experiment Using SNAP Benefit Timing},
   Journal = {American Educational Research Journal},
   Pages = {000283121876133-000283121876133},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.3102/0002831218761337},
   Doi = {10.3102/0002831218761337},
   Key = {fds337288}
}

@article{fds333175,
   Author = {Gassman-Pines, A and Skinner, AT},
   Title = {Psychological Acculturation and Parenting Behaviors in
             Mexican Immigrant Families.},
   Journal = {Journal of Family Issues},
   Volume = {39},
   Number = {5},
   Pages = {1139-1164},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1177/0192513x16687001},
   Abstract = {This study examined the relation between mothers' and
             fathers' psychological acculturation and parenting behaviors
             in two samples of Mexican immigrant families. The middle
             childhood sample included 47 mothers, 38 fathers and 46
             children in families with children age 9 - 12, and the early
             childhood sample included 185 mothers and 155 fathers in
             families with children age 2 - 6. In both samples, compared
             to families in which fathers reported feeling connected only
             to Latino culture, fathers who reported feeling connected to
             both Latinos and Americans engaged in fewer aversive and
             withdrawn interactions and more warm interactions with
             children. In families where fathers reported feeling
             connected to both Latinos and Americans, mothers also
             engaged in fewer aversive and withdrawn interactions and
             more warm interactions with children. Results were
             consistent across the two samples and across different
             family member reports of parent-child interactions.},
   Doi = {10.1177/0192513x16687001},
   Key = {fds333175}
}

@article{fds328959,
   Author = {Schenck-Fontaine, A and Gassman-Pines, A and Hill,
             Z},
   Title = {Use of Informal Safety Nets during the Supplemental
             Nutrition Assistance Program Benefit Cycle: How Poor
             Families Cope with Within-Month Economic
             Instability},
   Journal = {Social Service Review},
   Volume = {91},
   Number = {3},
   Pages = {456-487},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1086/694091},
   Doi = {10.1086/694091},
   Key = {fds328959}
}

@article{fds326690,
   Author = {Ananat, EO and Gassman-Pines, A and Francis, DV and Gibson-Davis,
             CM},
   Title = {Linking job loss, inequality, mental health, and
             education.},
   Journal = {Science (New York, N.Y.)},
   Volume = {356},
   Number = {6343},
   Pages = {1127-1128},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1126/science.aam5347},
   Doi = {10.1126/science.aam5347},
   Key = {fds326690}
}

@article{fds326510,
   Author = {Schenck-Fontaine, A and Gassman-Pines, A and Gibson-Davis, CM and Ananat, EO},
   Title = {Local Job Losses and Child Maltreatment: The Importance of
             Community Context},
   Journal = {Social Service Review},
   Volume = {91},
   Number = {2},
   Pages = {233-263},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1086/692075},
   Abstract = {© 2017 by The University of Chicago. All rights reserved.A
             growing body of literature suggests that economic downturns
             predict an increase in child maltreatment. However, to
             inform policies and practices to prevent and intervene in
             child maltreatment, it is necessary to identify how, when,
             and under what conditions community-level economic
             conditions affect child maltreatment. In this study, we use
             North Carolina administrative data from 2006 to 2011 on
             child maltreatment reports and job losses to distinguish
             effects on maltreatment frequency from effects on severity,
             identify the timing of these effects, and test whether
             community characteristics moderate these effects. To isolate
             effects of unanticipated job losses and to control for
             potential confounding factors, we use a fixed effects
             regression approach. We find that, though job losses did not
             affect the frequency of reports, job losses increased the
             share of reports that were relatively severe. This effect
             endured for 9 months following job losses and was only
             evident in economically disadvantaged communities.},
   Doi = {10.1086/692075},
   Key = {fds326510}
}

@article{fds321769,
   Author = {Gassman-Pines, A and Gibson-Davis, CM and Vernot, C and Butler, M and Hall, N and Taylor, L and Eastwood, K and Zhang, X},
   Title = {They Should Say “I Don't”: Norms About Midpregnancy
             Marriage and Job Loss},
   Journal = {Journal of Marriage and the Family},
   Volume = {79},
   Number = {2},
   Pages = {405-418},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1111/jomf.12379},
   Doi = {10.1111/jomf.12379},
   Key = {fds321769}
}


%% George, Linda K.   
@article{fds256055,
   Author = {Saltz, CC and Eisenberg, M and Fillenbaum, G and George,
             LK},
   Title = {Functional Status and Service Use Among Community-based
             Spinal Cord-injured Male Veterans12},
   Journal = {Neurorehabilitation},
   Volume = {1},
   Number = {4},
   Pages = {25-35},
   Editor = {Frank, RG},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.3233/NRE-1991-1405},
   Doi = {10.3233/NRE-1991-1405},
   Key = {fds256055}
}


%% Gibson-Davis, Christina M.   
@article{fds337711,
   Author = {Rackin, HM and Gibson-Davis, CM},
   Title = {Social Class Divergence in Family Transitions: The
             Importance of Cohabitation},
   Journal = {Journal of Marriage and the Family},
   Volume = {80},
   Number = {5},
   Pages = {1271-1286},
   Year = {2018},
   Month = {October},
   url = {http://dx.doi.org/10.1111/jomf.12522},
   Abstract = {© 2018 National Council on Family Relations Objective: This
             study examined trends in familial transitions by maternal
             education and whether transitions rose because of changes in
             prevalence (the share of children exposed to a relationship
             state, either marriage or cohabitation) or churning (the
             number of entrances and exits conditional on being exposed
             to a relationship state). Background: Children's experiences
             of transitions, an important predictor of well-being, have
             leveled off in recent decades. Plateauing in transitions may
             reflect heterogeneity by socioeconomic status. Method: Data
             came from the National Survey of Family Growth on firstborn
             children observed from ages 0 to 5 among mothers aged 15 to
             34 at the time of the child's birth (N = 7,265). Kitagawa
             methods decomposed changes in transitions into those
             attributable to changes in prevalence and churning. Analyses
             were conducted separately by maternal education. Results:
             Children born to lower and moderately educated women
             experienced an increase in transitions because cohabitation
             increased in prevalence rather than a change in the number
             of exits and entrances from cohabiting unions. Among this
             disadvantaged group, children exposed to cohabitation
             experienced much more churning than children exposed to
             marriage. Children born to mothers with a 4-year degree did
             not experience an increase in transitions and predominantly
             experienced stable parental marriages. Conclusion:
             Transitions only plateaued for children born to highly
             educated mothers, whereas transitions rose for
             less-advantaged children. Transitions appear to be another
             aspect of early family life experiences that contributes to
             diverging destinies.},
   Doi = {10.1111/jomf.12522},
   Key = {fds337711}
}

@article{fds335175,
   Author = {Gibson-Davis, CM and Percheski, C},
   Title = {Children and the Elderly: Wealth Inequality Among America's
             Dependents.},
   Journal = {Demography},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1007/s13524-018-0676-5},
   Abstract = {Life cycle theory predicts that elderly households have
             higher levels of wealth than households with children, but
             these wealth gaps are likely dynamic, responding to changes
             in labor market conditions, patterns of debt accumulation,
             and the overall economic context. Using Survey of Consumer
             Finances data from 1989 through 2013, we compare wealth
             levels between and within the two groups that make up
             America's dependents: the elderly and child households
             (households with a resident child aged 18 or younger). Over
             the observed period, the absolute wealth gap between elderly
             and child households in the United States increased
             substantially, and diverging trends in wealth accumulation
             exacerbated preexisting between-group disparities. Widening
             gaps were particularly pronounced among the least-wealthy
             elderly and child households. Differential demographic
             change in marital status and racial composition by subgroup
             do not explain the widening gap. We also find increasing
             wealth inequality within child households and the rise of a
             "parental 1 %." During a time of overall economic growth,
             the elderly have been able to maintain or increase their
             wealth, whereas many of the least-wealthy child households
             saw precipitous declines. Our findings suggest that many
             child households may lack sufficient assets to promote the
             successful flourishing of the next generation.},
   Doi = {10.1007/s13524-018-0676-5},
   Key = {fds335175}
}

@article{fds326123,
   Author = {Rackin, HM and Gibson-Davis, CM},
   Title = {Low-Income Childless Young Adults' Marriage and Fertility
             Frameworks},
   Journal = {Journal of Marriage and the Family},
   Volume = {79},
   Number = {4},
   Pages = {1096-1110},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/jomf.12405},
   Abstract = {© National Council on Family Relations, 2017.The authors
             investigated how low-income young adults without children
             understand marriage and fertility. Data come from the
             Becoming Partners and Parents Study (N=69), a qualitative
             study of African American adults aged 18 to 22 in a mid-size
             southern city. This is the first study to analyze young,
             low-income, childless, and unmarried Black respondents'
             frameworks (i.e., internal understandings of the world) of
             marriage and fertility. In contrast to research conducted on
             parents, this research on childless adults indicated a
             narrative in which there were close connections between
             marriage and fertility and an economic bar adhered to both
             marriage and childbearing. Respondents also believed that
             childbearing was meaningful and provided purpose, but that
             it was morally questionable if the parent was not
             financially stable. The results suggest that prior findings
             related to meanings of family formation and childbearing for
             low-income parents may not extend to those without
             children.},
   Doi = {10.1111/jomf.12405},
   Key = {fds326123}
}

@article{fds324192,
   Author = {Holbein, JB and Hillygus, DS and Gibson-Davis, CM and Hill, D and Lenard, MA},
   Title = {The Development of Students’ Engagement in School,
             Community, and Democracy},
   Year = {2017},
   Month = {June},
   Key = {fds324192}
}

@article{fds326472,
   Author = {Schenck-Fontaine, A and Gassman-Pines, A and Gibson-Davis, CM and Ananat, EO},
   Title = {Local Job Losses and Child Maltreatment: The Importance of
             Community Context},
   Journal = {Social Service Review},
   Volume = {91},
   Number = {2},
   Pages = {233-263},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1086/692075},
   Abstract = {© 2017 by The University of Chicago. All rights reserved.A
             growing body of literature suggests that economic downturns
             predict an increase in child maltreatment. However, to
             inform policies and practices to prevent and intervene in
             child maltreatment, it is necessary to identify how, when,
             and under what conditions community-level economic
             conditions affect child maltreatment. In this study, we use
             North Carolina administrative data from 2006 to 2011 on
             child maltreatment reports and job losses to distinguish
             effects on maltreatment frequency from effects on severity,
             identify the timing of these effects, and test whether
             community characteristics moderate these effects. To isolate
             effects of unanticipated job losses and to control for
             potential confounding factors, we use a fixed effects
             regression approach. We find that, though job losses did not
             affect the frequency of reports, job losses increased the
             share of reports that were relatively severe. This effect
             endured for 9 months following job losses and was only
             evident in economically disadvantaged communities.},
   Doi = {10.1086/692075},
   Key = {fds326472}
}

@article{fds326877,
   Author = {Ananat, EO and Gassman-Pines, A and Francis, DV and Gibson-Davis,
             CM},
   Title = {Linking job loss, inequality, mental health, and
             education.},
   Journal = {Science (New York, N.Y.)},
   Volume = {356},
   Number = {6343},
   Pages = {1127-1128},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1126/science.aam5347},
   Doi = {10.1126/science.aam5347},
   Key = {fds326877}
}

@article{fds320823,
   Author = {Gassman-Pines, A and Gibson-Davis, CM and Vernot, C and Butler, M and Hall, N and Taylor, L and Eastwood, K and Zhang, X},
   Title = {They Should Say “I Don't”: Norms About Midpregnancy
             Marriage and Job Loss},
   Journal = {Journal of Marriage and the Family},
   Volume = {79},
   Number = {2},
   Pages = {405-418},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1111/jomf.12379},
   Doi = {10.1111/jomf.12379},
   Key = {fds320823}
}


%% Giusto, Ali   
@article{fds337471,
   Author = {Giusto, A and Puffer, E},
   Title = {A systematic review of interventions targeting men's alcohol
             use and family relationships in low- and middle-income
             countries},
   Journal = {Global Mental Health (Cambridge, England)},
   Volume = {5},
   Year = {2018},
   url = {http://dx.doi.org/10.1017/gmh.2017.32},
   Doi = {10.1017/gmh.2017.32},
   Key = {fds337471}
}

@article{fds337472,
   Author = {Giusto, A and Friis, E and Sim, AL and Chase, RM and Zayzay, JO and Green,
             E and Puffer, E},
   Title = {A Qualitative Study of Mechanisms Underlying Effects of a
             Parenting Intervention in Rural Liberia},
   Journal = {The European Journal of Development Research},
   Volume = {29},
   Number = {5},
   Pages = {964-982},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1057/s41287-017-0101-8},
   Doi = {10.1057/s41287-017-0101-8},
   Key = {fds337472}
}

@article{fds326207,
   Author = {Giusto, AM and Green, EP and Puffer, ES},
   Title = {Associations Between Fathers' and Sons' Sexual Risk in Rural
             Kenya: The Potential for Intergenerational
             Transmission.},
   Journal = {Journal of Adolescent Health},
   Volume = {61},
   Number = {2},
   Pages = {219-225},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.jadohealth.2017.02.007},
   Abstract = {Despite high rates of HIV in areas of Sub-Saharan Africa and
             men's role in driving the epidemic, little is known about
             whether or how sexual risk-both behaviors and beliefs-may be
             passed down through generations of males. This study
             examined associations between sexual risk behaviors and
             sex-related beliefs of adolescent males and those of their
             male caregivers in Kenya, as well as the potential
             moderating effects of parenting characteristics and
             father-son relationship quality.Cross-sectional linear
             regression analysis was applied to baseline data from a
             trial of a family- and church-based intervention for
             families in rural Kenya that followed a stepped-wedge
             cluster randomized design. Our subsample consisted of 79
             male caregiver and son (aged 10-16 years) dyads.Results
             demonstrated a direct relationship between fathers' and
             sons' sex-related beliefs that was not moderated by
             parenting or quality of father-son relationship.
             Parenting/relationship characteristics did moderate the
             relationship between fathers' and sons' sexual behavior; if
             fathers did not engage in high-risk sex and exhibited more
             positive parenting/higher relationship quality, their sons
             were less likely to be sexually active. Among fathers having
             high-risk sex, parenting was unrelated to sons' behavior
             except at very high levels of positive parenting/relationship
             quality; at these levels, sons were actually more likely to
             have had sex.Findings support recommendations to include
             male caregivers in youth HIV prevention efforts, potentially
             by targeting fathers' parenting strategies and their
             individual risk.},
   Doi = {10.1016/j.jadohealth.2017.02.007},
   Key = {fds326207}
}


%% Gold, Deborah T.   
@article{fds337474,
   Author = {Silverman, SL and Siris, E and Belazi, D and Recknor, C and Papaioannou,
             A and Brown, JP and Gold, DT and Lewiecki, EM and Quinn, G and Balasubramanian, A and Yue, S and Stolshek, B and Kendler,
             DL},
   Title = {Persistence at 24 months with denosumab among
             postmenopausal women with osteoporosis: results of a
             prospective cohort study.},
   Journal = {Archives of Osteoporosis},
   Volume = {13},
   Number = {1},
   Pages = {85},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1007/s11657-018-0491-z},
   Abstract = {Persistence with prescribed medications for chronic diseases
             is important; however, persistence with osteoporosis
             treatments is historically poor. In this prospective cohort
             study of postmenopausal women treated for osteoporosis in
             real-world clinical practice settings in the USA and Canada,
             24-month persistence with denosumab was 58%.Patients who
             persist with their prescribed osteoporosis treatment have
             increased bone mineral density (BMD) and reduced risk of
             fracture. Twelve-month persistence with denosumab in routine
             clinical practice is as high as 95%, but there are limited
             data on longer-term persistence with denosumab in this
             setting.This single-arm, prospective, cohort study evaluated
             24-month persistence with denosumab administered every
             6 months in postmenopausal women receiving treatment for
             osteoporosis in real-world clinical practice in the USA and
             Canada. Endpoints and analyses included the percentage of
             patients who persist with denosumab at 24 months (greater
             than or equal to four injections with a gap between
             injections of no more than 6 months plus 8 weeks), the
             total number of injections received by each patient, changes
             in BMD in persistent patients, and the incidence of serious
             adverse events (SAEs) and fractures.Among 935 enrolled
             patients, 24-month persistence was 58% (50% in US patients
             and 75% in Canadian patients). A majority of patients
             received at least four injections over the observation
             period (62% of US patients and 81% of Canadian patients).
             Among patients who were persistent at 24 months and who had
             a baseline, 12-month, and 24-month DXA scan, mean BMD
             increased from baseline to 24 months by 7.8% at the lumbar
             spine and 2.1% at the femoral neck. SAEs and fractures were
             reported for 122 (13.0%) patients and 54 (5.8%) patients,
             respectively.Persistence with denosumab for 24 months
             yields improvement in BMD among postmenopausal women with
             osteoporosis treated in routine clinical practice in the USA
             and Canada.},
   Doi = {10.1007/s11657-018-0491-z},
   Key = {fds337474}
}

@article{fds326232,
   Author = {Kerr, C and Bottomley, C and Shingler, S and Giangregorio, L and de
             Freitas, HM and Patel, C and Randall, S and Gold,
             DT},
   Title = {The importance of physical function to people with
             osteoporosis.},
   Journal = {Osteoporosis International : a Journal Established as Result
             of Cooperation Between the European Foundation for
             Osteoporosis and the National Osteoporosis Foundation of the
             Usa},
   Volume = {28},
   Number = {5},
   Pages = {1597-1607},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1007/s00198-017-3911-9},
   Abstract = {There is increasing need to understand patient outcomes in
             osteoporosis. This article discusses that fracture in
             osteoporosis can lead to a cycle of impairment, driven by
             complex psychosocial factors, having a profound impact on
             physical function/activity which accumulates over time. More
             information is required on how treatments impact physical
             function.There is increasing need to understand
             patient-centred outcomes in osteoporosis (OP) clinical
             research and management. This multi-method paper provides
             insight on the effect of OP on patients' physical function
             and everyday activity.Data were collected from three
             sources: (1) targeted literature review on OP and physical
             function, conducted in MEDLINE, Embase and PsycINFO; (2)
             secondary thematic analysis of transcripts from patient
             interviews, conducted to develop a patient-reported outcome
             instrument. Transcripts were re-coded to focus on OP impact
             on daily activities and physical function for those with and
             without fracture history; and (3) discussions of the
             literature review and secondary qualitative analysis results
             with three clinical experts to review and interpret the
             importance and implications of the findings.Results suggest
             that OP, particularly with fracture, can have profound
             impacts on physical function/activity. These impacts
             accumulate over time through a cycle of impairment, as
             fracture leads to longer term detriments in physical
             function, including loss of muscle, activity avoidance and
             reduced physical capacity, which in turn leads to greater
             risk of fracture and potential for further physical
             restrictions. The cycle of impairment is complex, as other
             physical, psychosocial and treatment-related factors, such
             as comorbidities, fears and beliefs about physical activity
             and fracture risk influence physical function and everyday
             activity.More information on how treatments impact physical
             function would benefit healthcare professionals and persons
             with OP in making treatment decisions and improving
             treatment compliance/persistence, as these impacts may be
             more salient to patients than fracture incidence.},
   Doi = {10.1007/s00198-017-3911-9},
   Key = {fds326232}
}

@article{fds326233,
   Author = {Diez-Perez, A and Naylor, KE and Abrahamsen, B and Agnusdei, D and Brandi, ML and Cooper, C and Dennison, E and Eriksen, EF and Gold, DT and Guañabens, N and Hadji, P and Hiligsmann, M and Horne, R and Josse, R and Kanis, JA and Obermayer-Pietsch, B and Prieto-Alhambra, D and Reginster, J-Y and Rizzoli, R and Silverman, S and Zillikens, MC and Eastell, R and Adherence Working Group of the International
             Osteoporosis Foundation and the European Calcified Tissue
             Society},
   Title = {International Osteoporosis Foundation and European Calcified
             Tissue Society Working Group. Recommendations for the
             screening of adherence to oral bisphosphonates.},
   Journal = {Osteoporosis International : a Journal Established as Result
             of Cooperation Between the European Foundation for
             Osteoporosis and the National Osteoporosis Foundation of the
             Usa},
   Volume = {28},
   Number = {3},
   Pages = {767-774},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1007/s00198-017-3906-6},
   Abstract = {Adherence to oral bisphosphonates is low. A screening
             strategy is proposed based on the response of biochemical
             markers of bone turnover after 3 months of therapy. If no
             change is observed, the clinician should reassess the
             adherence to the treatment and also other potential issues
             with the drug.Low adherence to oral bisphosphonates is a
             common problem that jeopardizes the efficacy of treatment of
             osteoporosis. No clear screening strategy for the assessment
             of compliance is widely accepted in these patients.The
             International Osteoporosis Foundation and the European
             Calcified Tissue Society have convened a working group to
             propose a screening strategy to detect a lack of adherence
             to these drugs. The question to answer was whether the bone
             turnover markers (BTMs) PINP and CTX can be used to identify
             low adherence in patients with postmenopausal osteoporosis
             initiating oral bisphosphonates for osteoporosis. The
             findings of the TRIO study specifically address this
             question and were used as the basis for testing the
             hypothesis.Based on the findings of the TRIO study,
             specifically addressing this question, the working group
             recommends measuring PINP and CTX at baseline and 3 months
             after starting therapy to check for a decrease above the
             least significant change (decrease of more than 38% for PINP
             and 56% for CTX). Detection rate for the measurement of PINP
             is 84%, for CTX 87% and, if variation in at least one is
             considered when measuring both, the level of detection is
             94.5%.If a significant decrease is observed, the treatment
             can continue, but if no decrease occurs, the clinician
             should reassess to identify problems with the treatment,
             mainly low adherence.},
   Doi = {10.1007/s00198-017-3906-6},
   Key = {fds326233}
}


%% Goldston, David B.   
@article{fds337179,
   Author = {Ribeiro, SP and LaCroix, JM and De Oliveira and F and Novak, LA and Lee-Tauler, SY and Darmour, CA and Perera, KU and Goldston, DB and Weaver, J and Soumoff, A and Ghahramanlou-Holloway,
             M},
   Title = {The Link between Posttraumatic Stress Disorder and
             Functionality among United States Military Service Members
             Psychiatrically Hospitalized Following a Suicide
             Crisis.},
   Journal = {Healthcare (Basel, Switzerland)},
   Volume = {6},
   Number = {3},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.3390/healthcare6030095},
   Abstract = {Posttraumatic stress disorder (PTSD) is one of the most
             commonly diagnosed psychiatric disorders in the United
             States and has been linked to suicidal thoughts and
             behaviors, yet the role of a PTSD diagnosis on functional
             impairment among suicidal individuals remains unknown. This
             study examined the association between PTSD status and
             functional impairment among military psychiatric inpatients
             admitted for acute suicide risk (N = 166) with a lifetime
             history of at least one suicide attempt. Measures of
             functionality included: (1) alcohol use; (2) sleep quality;
             (3) social problem-solving; and (4) work and social
             adjustment. Thirty-eight percent of the sample met criteria
             for PTSD. Women were more likely than men to meet criteria
             for PTSD (p = 0.007), and participants who met PTSD criteria
             had significantly more psychiatric diagnoses (p < 0.001).
             Service members who met PTSD criteria reported more
             disturbed sleep (p = 0.003) and greater difficulties with
             work and social adjustment (p = 0.004) than those who did
             not meet PTSD criteria. However, functionality measures were
             not significantly associated with PTSD status after
             controlling for gender and psychiatric comorbidity. Gender
             and number of psychiatric comorbidities other than PTSD were
             significant predictors of PTSD in logistic regression models
             across four functionality measures. Future studies should
             assess the additive or mediating effect of psychiatric
             comorbidities in the association between impaired
             functioning and PTSD. Clinicians are encouraged to assess
             and address functionality during treatment with suicidal
             individuals, paying particular attention to individuals with
             multiple psychiatric diagnoses.},
   Doi = {10.3390/healthcare6030095},
   Key = {fds337179}
}

@article{fds330045,
   Author = {Copeland, WE and Goldston, DB and Costello, EJ},
   Title = {Adult Associations of Childhood Suicidal Thoughts and
             Behaviors: A Prospective, Longitudinal Analysis.},
   Journal = {Journal of the American Academy of Child and Adolescent
             Psychiatry},
   Volume = {56},
   Number = {11},
   Pages = {958-965.e4},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jaac.2017.08.015},
   Abstract = {Suicidal thoughts and behavior (STBs) have their peak period
             of onset in adolescence, but little is known about how such
             behavior is associated with later functioning. The aim of
             this study is to test whether childhood STBs are related to
             adult psychiatric, suicidal, and functional outcomes.This is
             a prospective, population-based community study of 1,420
             participants assessed with structured interviews up to 7
             times in childhood/adolescence (ages 9-16 years; 6,674
             observations) for STBs including passive and active
             ideation, plans, and attempts. Participants were then
             assessed 4 times in young adulthood (ages 19, 21, 24, and 30
             years; 4,556 observations of 1,273 participants) for
             psychiatric diagnoses, STBs, and functional outcomes.By age
             16 years, 7.0% of participants had reported some type of
             STBs, with 3.9% reporting an attempt. Both ideation only and
             suicide attempts were associated with higher levels of
             anxiety disorders and STBs in adulthood, as well as poor
             functioning across financial, health, risky/illegal, and
             social domains. These observed effects generally were
             attenuated after adjusting for other psychiatric and
             psychosocial factors that predict childhood STBs
             (particularly maltreatment, depression, and disruptive
             behavior disorders). The exception was adult suicidal
             behavior, which was predicted by both childhood ideation and
             attempts, even in the fully adjusted model. Children and
             adolescents with STBs were more likely to have had a
             disrupted transition to adulthood.Childhood STBs are a
             marker for a multitude of poor psychiatric and functional
             outcomes in adulthood, but these effects are largely
             accounted for by other factors. In contrast, childhood STBs
             are a robust risk factor for adult suicidal thoughts and
             behavior.},
   Doi = {10.1016/j.jaac.2017.08.015},
   Key = {fds330045}
}

@article{fds322758,
   Author = {Daniel, SS and Goldston, DB and Erkanli, A and Heilbron, N and Franklin,
             JC},
   Title = {Prospective Study of Major Loss Life Events and Risk for
             Suicidal Thoughts and Behaviors Among Adolescents and Young
             Adults.},
   Journal = {Suicide & Life Threatening Behavior},
   Volume = {47},
   Number = {4},
   Pages = {436-449},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1111/sltb.12305},
   Abstract = {This prospective, naturalistic study examined the
             association between major loss life experiences, other
             psychiatric risk factors (depression, hopelessness, and
             anxiety), and suicidal thoughts and behaviors (STBs) among
             adolescents followed through young adulthood for up to
             14 years. Major loss life events were related to subsequent
             increases in STBs. Major loss life events were primarily
             related to increases in suicide ideation in the presence of
             lower levels of other risk factors. There was a
             bidirectional relationship between major losses and other
             risk factors. Implications for the association between loss
             experiences, other risk factors, and future STBs are
             discussed.},
   Doi = {10.1111/sltb.12305},
   Key = {fds322758}
}


%% Grimes, Christina   
@article{fds326698,
   Author = {Golonka, MM and Peairs, KF and Malone, PS and Grimes, CL and Costanzo,
             PR},
   Title = {Natural Peer Leaders as Substance Use Prevention Agents: the
             Teens' Life Choice Project.},
   Journal = {Prevention Science : the Official Journal of the Society for
             Prevention Research},
   Volume = {18},
   Number = {5},
   Pages = {555-566},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1007/s11121-017-0790-4},
   Abstract = {In adolescent social groups, natural peer leaders have been
             found to engage in more frequent experimentation with
             substance use and to possess disproportionate power to
             affect the behavior and social choices of their associated
             peer followers. In the current exploratory study, we used
             sociometrics and social cognitive mapping to identify
             natural leaders of cliques in a seventh grade population and
             invited the leaders to develop anti-drug presentations for
             an audience of younger peers. The program employed
             social-psychological approaches directed at having leaders
             proceed from extrinsic inducements to intrinsic
             identification with their persuasive products in the context
             of the group intervention process. The goals of the
             intervention were to induce substance resistant
             self-persuasion in the leaders and to produce a spread of
             this resistance effect to their peer followers. To test the
             intervention, we compared the substance use behaviors of the
             selected leaders and their peers to a control cohort. The
             study found preliminary support that the intervention
             produced changes in the substance use behavior among the
             leaders who participated in the intervention, but did not
             detect a spread to non-leader peers in the short term. This
             descriptive study speaks to the plausibility of employing
             self-persuasion paradigms to bring about change in high-risk
             behaviors among highly central adolescents. In addition, it
             highlights the viability of applying social psychological
             principles to prevention work and calls for more research in
             this area.},
   Doi = {10.1007/s11121-017-0790-4},
   Key = {fds326698}
}


%% Groh, Jennifer M.   
@article{fds336520,
   Author = {Caruso, VC and Mohl, JT and Glynn, C and Lee, J and Willett, SM and Zaman,
             A and Ebihara, AF and Estrada, R and Freiwald, WA and Tokdar, ST and Groh,
             JM},
   Title = {Single neurons may encode simultaneous stimuli by switching
             between activity patterns.},
   Journal = {Nature Communications},
   Volume = {9},
   Number = {1},
   Pages = {2715},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1038/s41467-018-05121-8},
   Abstract = {How the brain preserves information about multiple
             simultaneous items is poorly understood. We report that
             single neurons can represent multiple stimuli by
             interleaving signals across time. We record single units in
             an auditory region, the inferior colliculus, while monkeys
             localize 1 or 2 simultaneous sounds. During dual-sound
             trials, we find that some neurons fluctuate between firing
             rates observed for each single sound, either on a
             whole-trial or on a sub-trial timescale. These fluctuations
             are correlated in pairs of neurons, can be predicted by the
             state of local field potentials prior to sound onset, and,
             in one monkey, can predict which sound will be reported
             first. We find corroborating evidence of fluctuating
             activity patterns in a separate dataset involving responses
             of inferotemporal cortex neurons to multiple visual stimuli.
             Alternation between activity patterns corresponding to each
             of multiple items may therefore be a general strategy to
             enhance the brain processing capacity, potentially linking
             such disparate phenomena as variable neural firing, neural
             oscillations, and limits in attentional/memory
             capacity.},
   Doi = {10.1038/s41467-018-05121-8},
   Key = {fds336520}
}

@article{fds332221,
   Author = {Caruso, VC and Pages, DS and Sommer, MA and Groh,
             JM},
   Title = {Beyond the labeled line: variation in visual reference
             frames from intraparietal cortex to frontal eye fields and
             the superior colliculus.},
   Journal = {Journal of Neurophysiology},
   Volume = {119},
   Number = {4},
   Pages = {1411-1421},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1152/jn.00584.2017},
   Abstract = {We accurately perceive the visual scene despite moving our
             eyes ~3 times per second, an ability that requires
             incorporation of eye position and retinal information. In
             this study, we assessed how this neural computation unfolds
             across three interconnected structures: frontal eye fields
             (FEF), intraparietal cortex (LIP/MIP), and the superior
             colliculus (SC). Single-unit activity was assessed in
             head-restrained monkeys performing visually guided saccades
             from different initial fixations. As previously shown, the
             receptive fields of most LIP/MIP neurons shifted to novel
             positions on the retina for each eye position, and these
             locations were not clearly related to each other in either
             eye- or head-centered coordinates (defined as hybrid
             coordinates). In contrast, the receptive fields of most SC
             neurons were stable in eye-centered coordinates. In FEF,
             visual signals were intermediate between those patterns:
             around 60% were eye-centered, whereas the remainder showed
             changes in receptive field location, boundaries, or
             responsiveness that rendered the response patterns hybrid or
             occasionally head-centered. These results suggest that FEF
             may act as a transitional step in an evolution of
             coordinates between LIP/MIP and SC. The persistence across
             cortical areas of mixed representations that do not provide
             unequivocal location labels in a consistent reference frame
             has implications for how these representations must be read
             out. NEW & NOTEWORTHY How we perceive the world as stable
             using mobile retinas is poorly understood. We compared the
             stability of visual receptive fields across different
             fixation positions in three visuomotor regions. Irregular
             changes in receptive field position were ubiquitous in
             intraparietal cortex, evident but less common in the frontal
             eye fields, and negligible in the superior colliculus (SC),
             where receptive fields shifted reliably across fixations.
             Only the SC provides a stable labeled-line code for stimuli
             across saccades.},
   Doi = {10.1152/jn.00584.2017},
   Key = {fds332221}
}

@article{fds332360,
   Author = {Gruters, KG and Murphy, DLK and Jenson, CD and Smith, DW and Shera, CA and Groh, JM},
   Title = {The eardrums move when the eyes move: A multisensory effect
             on the mechanics of hearing.},
   Journal = {Proceedings of the National Academy of Sciences of the
             United States of America},
   Volume = {115},
   Number = {6},
   Pages = {E1309-E1318},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1073/pnas.1717948115},
   Abstract = {Interactions between sensory pathways such as the visual and
             auditory systems are known to occur in the brain, but where
             they first occur is uncertain. Here, we show a multimodal
             interaction evident at the eardrum. Ear canal microphone
             measurements in humans (n = 19 ears in 16 subjects) and
             monkeys (n = 5 ears in three subjects) performing a saccadic
             eye movement task to visual targets indicated that the
             eardrum moves in conjunction with the eye movement. The
             eardrum motion was oscillatory and began as early as 10 ms
             before saccade onset in humans or with saccade onset in
             monkeys. These eardrum movements, which we dub eye
             movement-related eardrum oscillations (EMREOs), occurred in
             the absence of a sound stimulus. The amplitude and phase of
             the EMREOs depended on the direction and horizontal
             amplitude of the saccade. They lasted throughout the saccade
             and well into subsequent periods of steady fixation. We
             discuss the possibility that the mechanisms underlying
             EMREOs create eye movement-related binaural cues that may
             aid the brain in evaluating the relationship between visual
             and auditory stimulus locations as the eyes
             move.},
   Doi = {10.1073/pnas.1717948115},
   Key = {fds332360}
}


%% Hard, Bridgette M.   
@article{fds336521,
   Author = {Brady, ST and Hard, BM and Gross, JJ},
   Title = {Reappraising test anxiety increases academic performance of
             first-year college students},
   Journal = {Journal of Educational Psychology},
   Volume = {110},
   Number = {3},
   Pages = {395-406},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1037/edu0000219},
   Abstract = {© 2017 American Psychological Association. The idea that
             test anxiety hurts performance is deeply ingrained in
             American culture and schools. However, researchers have
             found that it is actually worry about performance and
             anxiety-not bodily feelings of anxiety (emotionality)-that
             impairs performance. Drawing on this insight, anxiety
             reappraisal interventions encourage the view that anxiety
             can be neutral or even helpful. Initial evidence-largely
             from laboratory studies-suggests that these kinds of
             reappraisal interventions can improve student performance in
             mathematics. But can they do so in other domains and within
             the constraints of everyday classroom activities? If so, for
             whom and how? In an intervention study, we tested whether a
             minimal reappraisal message embedded in an email from course
             instructors could improve students' academic experience and
             performance in an introductory college course. The night
             before their first exam, students received an e-mail that
             either did or did not include a paragraph designed to lead
             them to interpret exam anxiety as beneficial or at least
             neutral. First-year students, who experience greater test
             anxiety and are less certain about how to perform well,
             benefited from the reappraisal message, showing decreased
             worry and increased performance on the exam the next day as
             well as increased performance in the course overall.
             Mediation analyses revealed that the effect on overall
             course performance for first-year students was partially
             mediated by reduced exam worry and enhanced performance on
             the first exam. The message did not affect the performance
             of upper year students.},
   Doi = {10.1037/edu0000219},
   Key = {fds336521}
}


%% Hariri, Ahmad   
@article{fds332756,
   Author = {Nikolova, YS and Misquitta, KA and Rocco, BR and Prevot, TD and Knodt,
             AR and Ellegood, J and Voineskos, AN and Lerch, JP and Hariri, AR and Sibille, E and Banasr, M},
   Title = {Shifting priorities: highly conserved behavioral and brain
             network adaptations to chronic stress across
             species},
   Journal = {Translational Psychiatry},
   Volume = {8},
   Number = {1},
   Year = {2018},
   Month = {December},
   url = {http://dx.doi.org/10.1038/s41398-017-0083-5},
   Abstract = {© 2018 The Author(s). Parallel clinical and preclinical
             research have begun to illuminate the biological basis of
             stress-related disorders, including major depression, but
             translational bridges informing discrete mechanistic targets
             for intervention are missing. To address this critical need,
             we used structural MRI in a mouse model and in a large human
             sample to examine stress effects on brain structure that may
             be conserved across species. Specifically, we focused on a
             previously unexplored approach, whole-brain structural
             covariance, as it reflects synchronized changes in
             neuroanatomy, potentially due to mutual trophic influences
             or shared plasticity across regions. Using the unpredictable
             chronic mild stress (UCMS) paradigm in mouse we first
             demonstrate that UCMS-induced elevated behavioral
             emotionality correlates with increased size of the amygdala
             and other corticolimbic regions. We further identify focal
             increases in the amygdala's 'hubness' (degree and strength)
             set against the background of a global stress-related loss
             of network clustering and modularity. These macroscopic
             changes are supported on the molecular level by increased
             postsynaptic density-95 protein in the amygdala, consistent
             with stress-induced plastic changes and synaptic
             strengthening. Finally, we provide clinical evidence that
             strikingly similar structural network reorganization
             patterns exist in young adults reporting high childhood
             trauma and increased mood symptoms. Collectively, we provide
             initial translational evidence for a conserved
             stress-related increase in amygdala-centered structural
             synchrony, as measured by enhanced structural covariance,
             which is paralleled by a decrease in global structural
             synchrony. This putative trade-off reflected in increased
             amygdala-centered plastic changes at the expense of global
             structural dedifferentiation may represent a mechanistic
             pathway for depression and related psychopathology.},
   Doi = {10.1038/s41398-017-0083-5},
   Key = {fds332756}
}

@article{fds335682,
   Author = {Lam, M and Trampush, JW and Yu, J and Knowles, E and Djurovic, S and Melle,
             I and Sundet, K and Christoforou, A and Reinvang, I and DeRosse, P and Lundervold, AJ and Steen, VM and Espeseth, T and Räikkönen, K and Widen, E and Palotie, A and Eriksson, JG and Giegling, I and Konte, B and Roussos, P and Giakoumaki, S and Burdick, KE and Payton, A and Ollier,
             W and Chiba-Falek, O and Attix, DK and Need, AC and Cirulli, ET and Voineskos, AN and Stefanis, NC and Avramopoulos, D and Hatzimanolis,
             A and Arking, DE and Smyrnis, N and Bilder, RM and Freimer, NA and Cannon,
             TD and London, E and Poldrack, RA and Sabb, FW and Congdon, E and Conley,
             ED and Scult, MA and Dickinson, D and Straub, RE and Donohoe, G and Morris,
             D and Corvin, A and Gill, M and Hariri, AR and Weinberger, DR and Pendleton, N and Bitsios, P and Rujescu, D and Lahti, J and Hellard, SL and Keller, MC and Andreassen, OA and Glahn, DC and Malhotra, AK and Lencz,
             T},
   Title = {Multi-Trait Analysis of GWAS and Biological Insights Into
             Cognition: A Response to Hill (2018).},
   Journal = {Twin Research and Human Genetics : the Official Journal of
             the International Society for Twin Studies},
   Pages = {1-4},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1017/thg.2018.46},
   Abstract = {Hill (Twin Research and Human Genetics, Vol. 21, 2018,
             84-88) presented a critique of our recently published paper
             in Cell Reports entitled 'Large-Scale Cognitive GWAS
             Meta-Analysis Reveals Tissue-Specific Neural Expression and
             Potential Nootropic Drug Targets' (Lam et al., Cell Reports,
             Vol. 21, 2017, 2597-2613). Specifically, Hill offered
             several interrelated comments suggesting potential problems
             with our use of a new analytic method called Multi-Trait
             Analysis of GWAS (MTAG) (Turley et al., Nature Genetics,
             Vol. 50, 2018, 229-237). In this brief article, we respond
             to each of these concerns. Using empirical data, we conclude
             that our MTAG results do not suffer from 'inflation in the
             FDR [false discovery rate]', as suggested by Hill (Twin
             Research and Human Genetics, Vol. 21, 2018, 84-88), and are
             not 'more relevant to the genetic contributions to education
             than they are to the genetic contributions to
             intelligence'.},
   Doi = {10.1017/thg.2018.46},
   Key = {fds335682}
}

@article{fds335683,
   Author = {Kim, MJ and Scult, MA and Knodt, AR and Radtke, SR and d'Arbeloff, TC and Brigidi, BD and Hariri, AR},
   Title = {A Link Between Childhood Adversity and Trait Anger Reflects
             Relative Activity of the Amygdala and Dorsolateral
             Prefrontal Cortex.},
   Journal = {Biological Psychiatry. Cognitive Neuroscience and
             Neuroimaging},
   Volume = {3},
   Number = {7},
   Pages = {644-649},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.bpsc.2018.03.006},
   Abstract = {Trait anger, or the dispositional tendency to experience a
             wide range of situations as annoying or frustrating, is
             associated with negative mental and physical health
             outcomes. The experience of adversity during childhood is
             one risk factor for the later emergence of high trait anger.
             This association has been hypothesized to reflect
             alterations in neural circuits supporting bottom-up threat
             processing and top-down executive control.Here, using
             functional magnetic resonance imaging and self-report
             questionnaire data from 220 volunteers, we examined how
             individual differences in top-down prefrontal executive
             control and bottom-up amygdala threat activity modulate the
             association between childhood adversity and trait anger
             during young adulthood.We report that the association
             between childhood adversity and trait anger is attenuated
             specifically in young adults who have both relatively low
             threat-related amygdala activity and high executive
             control-related dorsolateral prefrontal cortex
             activity.These brain activity patterns suggest that
             simultaneous consideration of their underlying cognitive
             processes-namely, threat processing and executive
             control-may be useful in strategies designed to mitigate the
             negative mental health consequences of childhood
             adversity.},
   Doi = {10.1016/j.bpsc.2018.03.006},
   Key = {fds335683}
}

@article{fds335684,
   Author = {Savage, JE and Jansen, PR and Stringer, S and Watanabe, K and Bryois, J and de Leeuw, CA and Nagel, M and Awasthi, S and Barr, PB and Coleman, JRI and Grasby, KL and Hammerschlag, AR and Kaminski, JA and Karlsson, R and Krapohl, E and Lam, M and Nygaard, M and Reynolds, CA and Trampush, JW and Young, H and Zabaneh, D and Hägg, S and Hansell, NK and Karlsson, IK and Linnarsson, S and Montgomery, GW and Muñoz-Manchado, AB and Quinlan,
             EB and Schumann, G and Skene, NG and Webb, BT and White, T and Arking, DE and Avramopoulos, D and Bilder, RM and Bitsios, P and Burdick, KE and Cannon, TD and Chiba-Falek, O and Christoforou, A and Cirulli, ET and Congdon, E and Corvin, A and Davies, G and Deary, IJ and DeRosse, P and Dickinson, D and Djurovic, S and Donohoe, G and Conley, ED and Eriksson,
             JG and Espeseth, T and Freimer, NA and Giakoumaki, S and Giegling, I and Gill, M and Glahn, DC and Hariri, AR and Hatzimanolis, A and Keller, MC and Knowles, E and Koltai, D and Konte, B and Lahti, J and Le Hellard and S and Lencz, T and Liewald, DC and London, E and Lundervold, AJ and Malhotra,
             AK and Melle, I and Morris, D and Need, AC and Ollier, W and Palotie, A and Payton, A and Pendleton, N and Poldrack, RA and Räikkönen, K and Reinvang, I and Roussos, P and Rujescu, D and Sabb, FW and Scult, MA and Smeland, OB and Smyrnis, N and Starr, JM and Steen, VM and Stefanis, NC and Straub, RE and Sundet, K and Tiemeier, H and Voineskos, AN and Weinberger, DR and Widen, E and Yu, J and Abecasis, G and Andreassen,
             OA and Breen, G and Christiansen, L and Debrabant, B and Dick, DM and Heinz, A and Hjerling-Leffler, J and Ikram, MA and Kendler, KS and Martin, NG and Medland, SE and Pedersen, NL and Plomin, R and Polderman,
             TJC and Ripke, S and van der Sluis, S and Sullivan, PF and Vrieze, SI and Wright, MJ and Posthuma, D},
   Title = {Genome-wide association meta-analysis in 269,867 individuals
             identifies new genetic and functional links to
             intelligence.},
   Journal = {Nature Genetics},
   Volume = {50},
   Number = {7},
   Pages = {912-919},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1038/s41588-018-0152-6},
   Abstract = {Intelligence is highly heritable1 and a major determinant of
             human health and well-being2. Recent genome-wide
             meta-analyses have identified 24 genomic loci linked to
             variation in intelligence3-7, but much about its genetic
             underpinnings remains to be discovered. Here, we present a
             large-scale genetic association study of intelligence
             (n = 269,867), identifying 205 associated genomic loci
             (190 new) and 1,016 genes (939 new) via positional mapping,
             expression quantitative trait locus (eQTL) mapping,
             chromatin interaction mapping, and gene-based association
             analysis. We find enrichment of genetic effects in conserved
             and coding regions and associations with 146 nonsynonymous
             exonic variants. Associated genes are strongly expressed in
             the brain, specifically in striatal medium spiny neurons and
             hippocampal pyramidal neurons. Gene set analyses implicate
             pathways related to nervous system development and synaptic
             structure. We confirm previous strong genetic correlations
             with multiple health-related outcomes, and Mendelian
             randomization analysis results suggest protective effects of
             intelligence for Alzheimer's disease and ADHD and
             bidirectional causation with pleiotropic effects for
             schizophrenia. These results are a major step forward in
             understanding the neurobiology of cognitive function as well
             as genetically related neurological and psychiatric
             disorders.},
   Doi = {10.1038/s41588-018-0152-6},
   Key = {fds335684}
}

@article{fds331566,
   Author = {Avinun, R and Nevo, A and Knodt, AR and Elliott, ML and Hariri,
             AR},
   Title = {Replication in Imaging Genetics: The Case of Threat-Related
             Amygdala Reactivity},
   Journal = {Biological Psychiatry},
   Volume = {84},
   Number = {2},
   Pages = {148-159},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.biopsych.2017.11.010},
   Abstract = {© 2017 Society of Biological Psychiatry. Background: Low
             replication rates are a concern in most, if not all,
             scientific disciplines. In psychiatric genetics
             specifically, targeting intermediate brain phenotypes, which
             are more closely associated with putative genetic effects,
             was touted as a strategy leading to increased power and
             replicability. In the current study, we attempted to
             replicate previously published associations between single
             nucleotide polymorphisms and threat-related amygdala
             reactivity, which represents a robust brain phenotype not
             only implicated in the pathophysiology of multiple
             disorders, but also used as a biomarker of future risk.
             Methods: We conducted a literature search for published
             associations between single nucleotide polymorphisms and
             threat-related amygdala reactivity and found 37 unique
             findings. Our replication sample consisted of 1117 young
             adult volunteers (629 women, mean age 19.72 ± 1.25 years)
             for whom both genetic and functional magnetic resonance
             imaging data were available. Results: Of the 37 unique
             associations identified, only three replicated as previously
             reported. When exploratory analyses were conducted with
             different model parameters compared to the original
             findings, significant associations were identified for 28
             additional studies: eight of these were for a different
             contrast/laterality; five for a different gender and/or
             race/ethnicity; and 15 in the opposite direction and for a
             different contrast, laterality, gender, and/or
             race/ethnicity. No significant associations, regardless of
             model parameters, were detected for six studies. Notably,
             none of the significant associations survived correction for
             multiple comparisons. Conclusions: We discuss these patterns
             of poor replication with regard to the general strategy of
             targeting intermediate brain phenotypes in genetic
             association studies and the growing importance of advancing
             the replicability of imaging genetics findings.},
   Doi = {10.1016/j.biopsych.2017.11.010},
   Key = {fds331566}
}

@article{fds335691,
   Author = {Miller, JA and Scult, MA and Conley, ED and Chen, Q and Weinberger, DR and Hariri, AR},
   Title = {Effects of Schizophrenia Polygenic Risk Scores on Brain
             Activity and Performance During Working Memory Subprocesses
             in Healthy Young Adults.},
   Journal = {Schizophrenia Bulletin},
   Volume = {44},
   Number = {4},
   Pages = {844-853},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1093/schbul/sbx140},
   Abstract = {Recent work has begun to shed light on the neural correlates
             and possible mechanisms of polygenic risk for schizophrenia.
             Here, we map a schizophrenia polygenic risk profile score
             (PRS) based on genome-wide association study significant
             loci onto variability in the activity and functional
             connectivity of a frontoparietal network supporting the
             manipulation versus maintenance of information during a
             numerical working memory (WM) task in healthy young adults
             (n = 99, mean age = 19.8). Our analyses revealed that higher
             PRS was associated with hypoactivity of the dorsolateral
             prefrontal cortex (dlPFC) during the manipulation but not
             maintenance of information in WM (r2 = .0576, P = .018).
             Post hoc analyses revealed that PRS-modulated dlPFC
             hypoactivity correlated with faster reaction times during WM
             manipulation (r2 = .0967, P = .002), and faster processing
             speed (r2 = .0967, P = .003) on a separate behavioral task.
             These PRS-associated patterns recapitulate dlPFC
             hypoactivity observed in patients with schizophrenia during
             central executive manipulation of information in WM on this
             task.},
   Doi = {10.1093/schbul/sbx140},
   Key = {fds335691}
}

@article{fds335685,
   Author = {Davies, G and Lam, M and Harris, SE and Trampush, JW and Luciano, M and Hill, WD and Hagenaars, SP and Ritchie, SJ and Marioni, RE and Fawns-Ritchie, C and Liewald, DCM and Okely, JA and Ahola-Olli, AV and Barnes, CLK and Bertram, L and Bis, JC and Burdick, KE and Christoforou,
             A and DeRosse, P and Djurovic, S and Espeseth, T and Giakoumaki, S and Giddaluru, S and Gustavson, DE and Hayward, C and Hofer, E and Ikram,
             MA and Karlsson, R and Knowles, E and Lahti, J and Leber, M and Li, S and Mather, KA and Melle, I and Morris, D and Oldmeadow, C and Palviainen,
             T and Payton, A and Pazoki, R and Petrovic, K and Reynolds, CA and Sargurupremraj, M and Scholz, M and Smith, JA and Smith, AV and Terzikhan, N and Thalamuthu, A and Trompet, S and van der Lee, SJ and Ware, EB and Windham, BG and Wright, MJ and Yang, J and Yu, J and Ames, D and Amin, N and Amouyel, P and Andreassen, OA and Armstrong, NJ and Assareh,
             AA and Attia, JR and Attix, D and Avramopoulos, D and Bennett, DA and Böhmer, AC and Boyle, PA and Brodaty, H and Campbell, H and Cannon, TD and Cirulli, ET and Congdon, E and Conley, ED and Corley, J and Cox, SR and Dale, AM and Dehghan, A and Dick, D and Dickinson, D and Eriksson, JG and Evangelou, E and Faul, JD and Ford, I and Freimer, NA and Gao, H and Giegling, I and Gillespie, NA and Gordon, SD and Gottesman, RF and Griswold, ME and Gudnason, V and Harris, TB and Hartmann, AM and Hatzimanolis, A and Heiss, G and Holliday, EG and Joshi, PK and Kähönen, M and Kardia, SLR and Karlsson, I and Kleineidam, L and Knopman, DS and Kochan, NA and Konte, B and Kwok, JB and Le Hellard and S and Lee, T and Lehtimäki, T and Li, S-C and Liu, T and Koini, M and London, E and Longstreth, WT and Lopez, OL and Loukola, A and Luck, T and Lundervold,
             AJ and Lundquist, A and Lyytikäinen, L-P and Martin, NG and Montgomery,
             GW and Murray, AD and Need, AC and Noordam, R and Nyberg, L and Ollier, W and Papenberg, G and Pattie, A and Polasek, O and Poldrack, RA and Psaty,
             BM and Reppermund, S and Riedel-Heller, SG and Rose, RJ and Rotter, JI and Roussos, P and Rovio, SP and Saba, Y and Sabb, FW and Sachdev, PS and Satizabal, CL and Schmid, M and Scott, RJ and Scult, MA and Simino, J and Slagboom, PE and Smyrnis, N and Soumaré, A and Stefanis, NC and Stott,
             DJ and Straub, RE and Sundet, K and Taylor, AM and Taylor, KD and Tzoulaki,
             I and Tzourio, C and Uitterlinden, A and Vitart, V and Voineskos, AN and Kaprio, J and Wagner, M and Wagner, H and Weinhold, L and Wen, KH and Widen, E and Yang, Q and Zhao, W and Adams, HHH and Arking, DE and Bilder,
             RM and Bitsios, P and Boerwinkle, E and Chiba-Falek, O and Corvin, A and De
             Jager, PL and Debette, S and Donohoe, G and Elliott, P and Fitzpatrick,
             AL and Gill, M and Glahn, DC and Hägg, S and Hansell, NK and Hariri, AR and Ikram, MK and Jukema, JW and Vuoksimaa, E and Keller, MC and Kremen, WS and Launer, L and Lindenberger, U and Palotie, A and Pedersen, NL and Pendleton, N and Porteous, DJ and Räikkönen, K and Raitakari, OT and Ramirez, A and Reinvang, I and Rudan, I and Dan Rujescu, and Schmidt,
             R and Schmidt, H and Schofield, PW and Schofield, PR and Starr, JM and Steen, VM and Trollor, JN and Turner, ST and Van Duijn and CM and Villringer, A and Weinberger, DR and Weir, DR and Wilson, JF and Malhotra, A and McIntosh, AM and Gale, CR and Seshadri, S and Mosley,
             TH and Bressler, J and Lencz, T and Deary, IJ},
   Title = {Study of 300,486 individuals identifies 148 independent
             genetic loci influencing general cognitive
             function.},
   Journal = {Nature Communications},
   Volume = {9},
   Number = {1},
   Pages = {2098},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1038/s41467-018-04362-x},
   Abstract = {General cognitive function is a prominent and relatively
             stable human trait that is associated with many important
             life outcomes. We combine cognitive and genetic data from
             the CHARGE and COGENT consortia, and UK Biobank (total
             N = 300,486; age 16-102) and find 148 genome-wide
             significant independent loci (P < 5 × 10-8)
             associated with general cognitive function. Within the novel
             genetic loci are variants associated with neurodegenerative
             and neurodevelopmental disorders, physical and psychiatric
             illnesses, and brain structure. Gene-based analyses find 709
             genes associated with general cognitive function. Expression
             levels across the cortex are associated with general
             cognitive function. Using polygenic scores, up to 4.3% of
             variance in general cognitive function is predicted in
             independent samples. We detect significant genetic overlap
             between general cognitive function, reaction time, and many
             health variables including eyesight, hypertension, and
             longevity. In conclusion we identify novel genetic loci and
             pathways contributing to the heritability of general
             cognitive function.},
   Doi = {10.1038/s41467-018-04362-x},
   Key = {fds335685}
}

@article{fds335686,
   Author = {d'Arbeloff, TC and Kim, MJ and Knodt, AR and Radtke, SR and Brigidi, BD and Hariri, AR},
   Title = {Microstructural Integrity of a Pathway Connecting the
             Prefrontal Cortex and Amygdala Moderates the Association
             Between Cognitive Reappraisal and Negative
             Emotions},
   Journal = {Emotion},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1037/emo0000447},
   Abstract = {© 2018 American Psychological Association. Cognitive
             reappraisal is a commonly used form of emotion regulation
             that utilizes frontal-executive control to reframe an
             approaching emotional event to moderate its potential
             psychological impact. Use of cognitive reappraisal has been
             associated with diminished experience of anxiety and
             depressive symptoms, as well as greater overall well-being.
             Using data from a study of 647 healthy young adults, we
             provide initial evidence that an association between typical
             use of cognitive reappraisal in daily life and the
             experience of anxiety and depressive symptoms is moderated
             by the microstructural integrity of the uncinate fasciculus,
             which provides a major anatomical link between the amygdala
             and prefrontal cortex. Our findings are consistent with the
             nature of top-down regulation of bottom-up negative emotions
             and suggest the uncinate fasciculus may be a useful target
             in the search for biomarkers predicting not only disorder
             risk but also response to psychotherapy utilizing cognitive
             reappraisal.},
   Doi = {10.1037/emo0000447},
   Key = {fds335686}
}

@article{fds335687,
   Author = {Agrawal, A and Chou, Y-L and Carey, CE and Baranger, DAA and Zhang, B and Sherva, R and Wetherill, L and Kapoor, M and Wang, J-C and Bertelsen, S and Anokhin, AP and Hesselbrock, V and Kramer, J and Lynskey, MT and Meyers,
             JL and Nurnberger, JI and Rice, JP and Tischfield, J and Bierut, LJ and Degenhardt, L and Farrer, LA and Gelernter, J and Hariri, AR and Heath,
             AC and Kranzler, HR and Madden, PAF and Martin, NG and Montgomery, GW and Porjesz, B and Wang, T and Whitfield, JB and Edenberg, HJ and Foroud, T and Goate, AM and Bogdan, R and Nelson, EC},
   Title = {Genome-wide association study identifies a novel locus for
             cannabis dependence.},
   Journal = {Molecular Psychiatry},
   Volume = {23},
   Number = {5},
   Pages = {1293-1302},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1038/mp.2017.200},
   Abstract = {Despite moderate heritability, only one study has identified
             genome-wide significant loci for cannabis-related
             phenotypes. We conducted meta-analyses of genome-wide
             association study data on 2080 cannabis-dependent cases and
             6435 cannabis-exposed controls of European descent. A
             cluster of correlated single-nucleotide polymorphisms (SNPs)
             in a novel region on chromosome 10 was genome-wide
             significant (lowest P=1.3E-8). Among the SNPs, rs1409568
             showed enrichment for H3K4me1 and H3K427ac marks, suggesting
             its role as an enhancer in addiction-relevant brain regions,
             such as the dorsolateral prefrontal cortex and the angular
             and cingulate gyri. This SNP is also predicted to modify
             binding scores for several transcription factors. We found
             modest evidence for replication for rs1409568 in an
             independent cohort of African American (896 cases and 1591
             controls; P=0.03) but not European American (EA; 781 cases
             and 1905 controls) participants. The combined meta-analysis
             (3757 cases and 9931 controls) indicated trend-level
             significance for rs1409568 (P=2.85E-7). No genome-wide
             significant loci emerged for cannabis dependence criterion
             count (n=8050). There was also evidence that the minor
             allele of rs1409568 was associated with a 2.1% increase in
             right hippocampal volume in an independent sample of 430 EA
             college students (fwe-P=0.008). The identification and
             characterization of genome-wide significant loci for
             cannabis dependence is among the first steps toward
             understanding the biological contributions to the etiology
             of this psychiatric disorder, which appears to be rising in
             some developed nations.},
   Doi = {10.1038/mp.2017.200},
   Key = {fds335687}
}

@article{fds335688,
   Author = {Chen, Q and Ursini, G and Romer, AL and Knodt, AR and Mezeivtch, K and Xiao, E and Pergola, G and Blasi, G and Straub, RE and Callicott, JH and Berman, KF and Hariri, AR and Bertolino, A and Mattay, VS and Weinberger, DR},
   Title = {Schizophrenia polygenic risk score predicts mnemonic
             hippocampal activity},
   Journal = {Brain},
   Volume = {141},
   Number = {4},
   Pages = {1218-1228},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1093/brain/awy004},
   Abstract = {© The Author(s) (2018). The use of polygenic risk scores
             has become a practical translational approach to
             investigating the complex genetic architecture of
             schizophrenia, but the link between polygenic risk scores
             and pathophysiological components of this disorder has been
             the subject of limited research. We investigated in healthy
             volunteers whether schizophrenia polygenic risk score
             predicts hippocampal activity during simple memory encoding,
             which has been proposed as a risk-associated intermediate
             phenotype of schizophrenia. We analysed the relationship
             between polygenic risk scores and hippocampal activity in a
             discovery sample of 191 unrelated healthy volunteers from
             the USA and in two independent replication samples of 76 and
             137 healthy unrelated participants from Europe and the USA,
             respectively. Polygenic risk scores for each individual were
             calculated as the sum of the imputation probability of
             reference alleles weighted by the natural log of odds ratio
             from the recent schizophrenia genome-wide association study.
             We examined hippocampal activity during simple memory
             encoding of novel visual stimuli assessed using blood oxygen
             level-dependent functional MRI. Polygenic risk scores were
             significantly associated with hippocampal activity in the
             discovery sample [P = 0.016, family-wise error (FWE)
             corrected within Anatomical Automatic Labeling (AAL)
             bilateral hippocampal-parahippocampal mask] and in both
             replication samples (P = 0.033, FWE corrected within AAL
             right posterior hippocampal-parahippocampal mask in Bari
             sample, and P = 0.002 uncorrected in the Duke Neurogenetics
             Study sample). The relationship between polygenic risk
             scores and hippocampal activity was consistently negative,
             i.e. lower hippocampal activity in individuals with higher
             polygenic risk scores, consistent with previous studies
             reporting decreased hippocampal-parahippocampal activity
             during declarative memory tasks in patients with
             schizophrenia and in their healthy siblings. Polygenic risk
             scores accounted for more than 8% of variance in hippocampal
             activity during memory encoding in discovery sample. We
             conclude that polygenic risk scores derived from the most
             recent schizophrenia genome-wide association study predict
             significant variability in hippocampal activity during
             memory encoding in healthy participants. Our findings
             validate mnemonic hippocampal activity as a genetic risk
             associated intermediate phenotype of schizophrenia,
             indicating that the aggregate neurobiological effect of
             schizophrenia risk alleles converges on this pattern of
             neural activity.},
   Doi = {10.1093/brain/awy004},
   Key = {fds335688}
}

@article{fds335689,
   Author = {Sethi, A and McCrory, E and Puetz, V and Hoffmann, F and Knodt, AR and Radtke, SR and Brigidi, BD and Hariri, AR and Viding,
             E},
   Title = {Primary and Secondary Variants of Psychopathy in a Volunteer
             Sample Are Associated With Different Neurocognitive
             Mechanisms},
   Journal = {Biological Psychiatry: Cognitive Neuroscience and
             Neuroimaging},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.bpsc.2018.04.002},
   Doi = {10.1016/j.bpsc.2018.04.002},
   Key = {fds335689}
}

@article{fds335690,
   Author = {Elliott, ML and Romer, A and Knodt, AR and Hariri,
             AR},
   Title = {A Connectome-wide Functional Signature of Transdiagnostic
             Risk for Mental Illness},
   Journal = {Biological Psychiatry},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.biopsych.2018.03.012},
   Abstract = {© 2018 Society of Biological Psychiatry Background: High
             rates of comorbidity, shared risk, and overlapping
             therapeutic mechanisms have led psychopathology research
             toward transdiagnostic dimensional investigations of
             clustered symptoms. One influential framework accounts for
             these transdiagnostic phenomena through a single general
             factor, sometimes referred to as the “p” factor,
             associated with risk for all common forms of mental illness.
             Methods: We build on previous research identifying unique
             structural neural correlates of the p factor by conducting a
             data-driven analysis of connectome-wide intrinsic functional
             connectivity (n = 605). Results: We demonstrate that higher
             p factor scores and associated risk for common mental
             illness maps onto hyperconnectivity between visual
             association cortex and both frontoparietal and default mode
             networks. Conclusions: These results provide initial
             evidence that the transdiagnostic risk for common forms of
             mental illness is associated with patterns of inefficient
             connectome-wide intrinsic connectivity between visual
             association cortex and networks supporting executive control
             and self-referential processes, networks that are often
             impaired across categorical disorders.},
   Doi = {10.1016/j.biopsych.2018.03.012},
   Key = {fds335690}
}

@article{fds326208,
   Author = {Romer, AL and Knodt, AR and Houts, R and Brigidi, BD and Moffitt, TE and Caspi, A and Hariri, AR},
   Title = {Structural alterations within cerebellar circuitry are
             associated with general liability for common mental
             disorders.},
   Journal = {Molecular Psychiatry},
   Volume = {23},
   Number = {4},
   Pages = {1084-1090},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1038/mp.2017.57},
   Abstract = {Accumulating mental-health research encourages a shift in
             focus toward transdiagnostic dimensional features that are
             shared across categorical disorders. In support of this
             shift, recent studies have identified a general liability
             factor for psychopathology-sometimes called the 'p factor'-
             that underlies shared risk for a wide range of mental
             disorders. Identifying neural correlates of this general
             liability would substantiate its importance in
             characterizing the shared origins of mental disorders and
             help us begin to understand the mechanisms through which the
             'p factor' contributes to risk. Here we believe we first
             replicate the 'p factor' using cross-sectional data from a
             volunteer sample of 1246 university students, and then using
             high-resolution multimodal structural neuroimaging, we
             demonstrate that individuals with higher 'p factor' scores
             show reduced structural integrity of white matter pathways,
             as indexed by lower fractional anisotropy values, uniquely
             within the pons. Whole-brain analyses further revealed that
             higher 'p factor' scores are associated with reduced gray
             matter volume in the occipital lobe and left cerebellar
             lobule VIIb, which is functionally connected with prefrontal
             regions supporting cognitive control. Consistent with the
             preponderance of cerebellar afferents within the pons, we
             observed a significant positive correlation between the
             white matter integrity of the pons and cerebellar gray
             matter volume associated with higher 'p factor' scores. The
             results of our analyses provide initial evidence that
             structural alterations in corticocerebellar circuitry
             supporting core functions related to the basic integration,
             coordination and monitoring of information may contribute to
             a general liability for common mental disorders.},
   Doi = {10.1038/mp.2017.57},
   Key = {fds326208}
}

@article{fds332044,
   Author = {Scult, MA and Hariri, AR},
   Title = {A BRIEF INTRODUCTION TO THE NEUROGENETICS OF
             COGNITION-EMOTION INTERACTIONS.},
   Journal = {Current Opinion in Behavioral Sciences},
   Volume = {19},
   Pages = {50-54},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.cobeha.2017.09.014},
   Abstract = {Neuroscience research has demonstrated that cognition,
             emotion, and their dynamic interactions emerge from complex
             and flexible patterns of activity across distributed neural
             circuits. A parallel branch of research in genetics has
             begun to identify common variation in the human DNA sequence
             (i.e., genome) that may shape individual differences in
             cognition-emotion interactions by altering molecular and
             cellular pathways that modulate the activity of these neural
             circuits. Here we provide a brief introduction to such
             neurogenetics research and how it may usefully inform our
             understanding of the biological mechanisms through which
             dynamic cognition-emotion interactions emerge and,
             subsequently, help shape normal and abnormal
             behavior.},
   Doi = {10.1016/j.cobeha.2017.09.014},
   Key = {fds332044}
}

@article{fds331564,
   Author = {Swartz, JR and Knodt, AR and Radtke, SR and Hariri,
             AR},
   Title = {Post-secondary maternal education buffers against neural
             risk for psychological vulnerability to future life
             stress},
   Journal = {Neuropsychologia},
   Volume = {109},
   Pages = {134-139},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.neuropsychologia.2017.12.019},
   Abstract = {© 2017 Elsevier Ltd We have previously reported that
             threat-related amygdala activity measured during a baseline
             fMRI scan predicts the experience of depression and anxiety
             associated with stressful life events years later. Here, we
             examine whether two broad measures of childhood
             environmental enrichment, namely parental educational
             achievement and subjective parental socioeconomic status,
             buffer against the effects of amygdala activity on future
             vulnerability to stress. Analyses of data available from 579
             young adults revealed that maternal, but not paternal,
             educational achievement moderates the association between
             amygdala activity, recent life stress, and changes in mood
             and anxiety symptoms, even when controlling for
             participants’ current subjective socioeconomic status.
             Specifically, only participants reporting lower maternal
             educational achievement exhibited our previously observed
             interaction between amygdala activity and future life stress
             predicting increases in depression and anxiety. These
             results suggest that higher maternal educational achievement
             may help buffer stress sensitivity associated with
             heightened threat-related amygdala activity.},
   Doi = {10.1016/j.neuropsychologia.2017.12.019},
   Key = {fds331564}
}

@article{fds332980,
   Author = {Hanson, JL and Knodt, AR and Brigidi, BD and Hariri,
             AR},
   Title = {Heightened connectivity between the ventral striatum and
             medial prefrontal cortex as a biomarker for stress-related
             psychopathology: understanding interactive effects of early
             and more recent stress},
   Journal = {Psychological Medicine},
   Pages = {1-9},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1017/S0033291717003348},
   Abstract = {Copyright © Cambridge University Press 2017 Background: The
             experience of childhood maltreatment is a significant risk
             factor for the development of depression. This risk is
             particularly heightened after exposure to additional, more
             contemporaneous stress. While behavioral evidence exists for
             this relation, little is known about biological correlates
             of these stress interactions. Identifying such correlates
             may provide biomarkers of risk for later depression.
             Methods: Here, we leverage behavioral, experiential, and
             neuroimaging data from the Duke Neurogenetics Study to
             identify potential biomarkers of stress exposure. Based on
             the past research, we were specifically interested in
             reward-related connectivity and the interaction of early and
             more recent stress. We examined psychophysiological
             interactions between the ventral striatum and other brain
             regions in relation to these stress variables, as well as
             measures of internalizing symptomatology (n = 926,
             participant age range = 18–22 years of age). Results: We
             found relatively increased reward-related functional
             connectivity between the left ventral striatum and the
             medial prefrontal cortex in individuals exposed to greater
             levels of childhood maltreatment who also experienced
             greater levels of recent life stress (β = 0.199, p <
             0.005). This pattern of functional connectivity was further
             associated with elevated symptoms of depression (β = 0.089,
             p = 0.006). Furthermore, using a moderated mediation
             framework, we demonstrate that this functional connectivity
             provides a biological link between cumulative stress
             exposure and internalizing symptomatology. Conclusions:
             These findings suggest a novel biomarker linking cumulative
             stress exposure with the later experience of depressive
             symptoms. Our results are discussed in the context of past
             research examining stress exposure in relation to
             depression.},
   Doi = {10.1017/S0033291717003348},
   Key = {fds332980}
}

@article{fds329769,
   Author = {Dotterer, HL and Waller, R and Neumann, CS and Shaw, DS and Forbes, EE and Hariri, AR and Hyde, LW},
   Title = {Examining the Factor Structure of the Self-Report of
             Psychopathy Short-Form Across Four Young Adult
             Samples.},
   Journal = {Assessment},
   Volume = {24},
   Number = {8},
   Pages = {1062-1079},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1177/1073191116640355},
   Abstract = {Psychopathy refers to a range of complex behaviors and
             personality traits, including callousness and antisocial
             behavior, typically studied in criminal populations. Recent
             studies have used self-reports to examine psychopathic
             traits among noncriminal samples. The goal of the current
             study was to examine the underlying factor structure of the
             Self-Report of Psychopathy Scale-Short Form (SRP-SF) across
             complementary samples and examine the impact of gender on
             factor structure. We examined the structure of the SRP-SF
             among 2,554 young adults from three undergraduate samples
             and a high-risk young adult sample. Using confirmatory
             factor analysis, a four-correlated factor model and a
             four-bifactor model showed good fit to the data. Evidence of
             weak invariance was found for both models across gender.
             These findings highlight that the SRP-SF is a useful measure
             of low-level psychopathic traits in noncriminal samples,
             although the underlying factor structure may not fully
             translate across men and women.},
   Doi = {10.1177/1073191116640355},
   Key = {fds329769}
}

@article{fds329035,
   Author = {Kim, MJ and Avinun, R and Knodt, AR and Radtke, SR and Hariri,
             AR},
   Title = {Neurogenetic plasticity and sex influence the link between
             corticolimbic structural connectivity and trait
             anxiety},
   Journal = {Scientific Reports},
   Volume = {7},
   Number = {1},
   Year = {2017},
   Month = {December},
   url = {http://dx.doi.org/10.1038/s41598-017-11497-2},
   Doi = {10.1038/s41598-017-11497-2},
   Key = {fds329035}
}

@article{fds330404,
   Author = {Schaefer, JD and Scult, MA and Caspi, A and Arseneault, L and Belsky,
             DW and Hariri, AR and Harrington, H and Houts, R and Ramrakha, S and Poulton, R and Moffitt, TE},
   Title = {Is low cognitive functioning a predictor or consequence of
             major depressive disorder? A test in two longitudinal birth
             cohorts.},
   Journal = {Development and Psychopathology},
   Pages = {1-15},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1017/s095457941700164x},
   Abstract = {Cognitive impairment has been identified as an important
             aspect of major depressive disorder (MDD). We tested two
             theories regarding the association between MDD and cognitive
             functioning using data from longitudinal cohort studies. One
             theory, the cognitive reserve hypothesis, suggests that
             higher cognitive ability in childhood decreases risk of
             later MDD. The second, the scarring hypothesis, instead
             suggests that MDD leads to persistent cognitive deficits
             following disorder onset. We tested both theories in the
             Dunedin Study, a population-representative cohort followed
             from birth to midlife and assessed repeatedly for both
             cognitive functioning and psychopathology. We also used data
             from the Environmental Risk Longitudinal Twin Study to test
             whether childhood cognitive functioning predicts future MDD
             risk independent of family-wide and genetic risk using a
             discordant twin design. Contrary to both hypotheses, we
             found that childhood cognitive functioning did not predict
             future risk of MDD, nor did study members with a past
             history of MDD show evidence of greater cognitive decline
             unless MDD was accompanied by other comorbid psychiatric
             conditions. Our results thus suggest that low cognitive
             functioning is related to comorbidity, but is neither an
             antecedent nor an enduring consequence of MDD. Future
             research may benefit from considering cognitive deficits
             that occur during depressive episodes from a transdiagnostic
             perspective.},
   Doi = {10.1017/s095457941700164x},
   Key = {fds330404}
}

@article{fds330405,
   Author = {Lam, M and Trampush, JW and Yu, J and Knowles, E and Davies, G and Liewald,
             DC and Starr, JM and Djurovic, S and Melle, I and Sundet, K and Christoforou, A and Reinvang, I and DeRosse, P and Lundervold, AJ and Steen, VM and Espeseth, T and Räikkönen, K and Widen, E and Palotie,
             A and Eriksson, JG and Giegling, I and Konte, B and Roussos, P and Giakoumaki, S and Burdick, KE and Payton, A and Ollier, W and Chiba-Falek, O and Attix, DK and Need, AC and Cirulli, ET and Voineskos,
             AN and Stefanis, NC and Avramopoulos, D and Hatzimanolis, A and Arking,
             DE and Smyrnis, N and Bilder, RM and Freimer, NA and Cannon, TD and London,
             E and Poldrack, RA and Sabb, FW and Congdon, E and Conley, ED and Scult,
             MA and Dickinson, D and Straub, RE and Donohoe, G and Morris, D and Corvin,
             A and Gill, M and Hariri, AR and Weinberger, DR and Pendleton, N and Bitsios, P and Rujescu, D and Lahti, J and Le Hellard and S and Keller, MC and Andreassen, OA and Deary, IJ and Glahn, DC and Malhotra, AK and Lencz,
             T},
   Title = {Large-Scale Cognitive GWAS Meta-Analysis Reveals
             Tissue-Specific Neural Expression and Potential Nootropic
             Drug Targets.},
   Journal = {Cell Reports},
   Volume = {21},
   Number = {9},
   Pages = {2597-2613},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.celrep.2017.11.028},
   Abstract = {Here, we present a large (n = 107,207) genome-wide
             association study (GWAS) of general cognitive ability ("g"),
             further enhanced by combining results with a large-scale
             GWAS of educational attainment. We identified 70 independent
             genomic loci associated with general cognitive ability.
             Results showed significant enrichment for genes causing
             Mendelian disorders with an intellectual disability
             phenotype. Competitive pathway analysis implicated the
             biological processes of neurogenesis and synaptic
             regulation, as well as the gene targets of two pharmacologic
             agents: cinnarizine, a T-type calcium channel blocker, and
             LY97241, a potassium channel inhibitor. Transcriptome-wide
             and epigenome-wide analysis revealed that the implicated
             loci were enriched for genes expressed across all brain
             regions (most strongly in the cerebellum). Enrichment was
             exclusive to genes expressed in neurons but not
             oligodendrocytes or astrocytes. Finally, we report genetic
             correlations between cognitive ability and disparate
             phenotypes including psychiatric disorders, several
             autoimmune disorders, longevity, and maternal age at first
             birth.},
   Doi = {10.1016/j.celrep.2017.11.028},
   Key = {fds330405}
}

@article{fds332981,
   Author = {Lissek, T and Adams, M and Adelman, J and Ahissar, E and Akaaboune, M and Akil, H and al'Absi, M and Arain, F and Arango-Lasprilla, JC and Atasoy,
             D and Avila, J and Badawi, A and Bading, H and Baig, AM and Baleriola, J and Belmonte, C and Bertocchi, I and Betz, H and Blakemore, C and Blanke, O and Boehm-Sturm, P and Bonhoeffer, T and Bonifazi, P and Brose, N and Campolongo, P and Celikel, T and Chang, CC and Chang, T-Y and Citri, A and Cline, HT and Cortes, JM and Cullen, K and Dean, K and Delgado-Garcia,
             JM and Desroches, M and Disterhoft, JF and Dowling, JE and Draguhn, A and El-Khamisy, SF and El Manira and A and Enam, SA and Encinas, JM and Erramuzpe, A and Esteban, JA and Fariñas, I and Fischer, E and Fukunaga, I and Gabilondo, I and Ganten, D and Gidon, A and Gomez-Esteban, JC and Greengard, P and Grinevich, V and Gruart, A and Guillemin, R and Hariri, AR and Hassan, B and Häusser, M and Hayashi,
             Y and Hussain, NK and Jabbar, AA and Jaber, M and Jahn, R and Janahi, EM and Kabbaj, M and Kettenmann, H and Kindt, M and Knafo, S and Köhr, G and Komai, S and Krugers, H and Kuhn, B and Ghazal, NL and Larkum, ME and London, M and Lutz, B and Matute, C and Martinez-Millan, L and Maroun,
             M and McGaugh, J and Moustafa, AA and Nasim, A and Nave, K-A and Neher, E and Nikolich, K and Outeiro, T and Palmer, LM and Penagarikano, O and Perez-Otano, I and Pfaff, DW and Poucet, B and Rahman, A-U and Ramos-Cabrer, P and Rashidy-Pour, A and Roberts, RJ and Rodrigues, S and Sanes, JR and Schaefer, AT and Segal, M and Segev, I and Shafqat, S and Siddiqui, NA and Soreq, H and Soriano-García, E and Spanagel, R and Sprengel, R and Stuart, G and Südhof, TC and Tønnesen, J and Treviño,
             M and Uthman, BM and Venter, JC and Verkhratsky, A and Weiss, C and Wiesel,
             TN and Yaksi, E and Yizhar, O and Young, LJ and Young, P and Zawia, NH and Zugaza, JL and Hasan, MT},
   Title = {Building Bridges through Science.},
   Journal = {Neuron},
   Volume = {96},
   Number = {4},
   Pages = {730-735},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.neuron.2017.09.028},
   Abstract = {Science is ideally suited to connect people from different
             cultures and thereby foster mutual understanding. To promote
             international life science collaboration, we have launched
             "The Science Bridge" initiative. Our current project focuses
             on partnership between Western and Middle Eastern
             neuroscience communities.},
   Doi = {10.1016/j.neuron.2017.09.028},
   Key = {fds332981}
}

@article{fds328846,
   Author = {Gard, AM and Waller, R and Shaw, DS and Forbes, EE and Hariri, AR and Hyde,
             LW},
   Title = {The Long Reach of Early Adversity: Parenting, Stress, and
             Neural Pathways to Antisocial Behavior in
             Adulthood},
   Journal = {Biological Psychiatry: Cognitive Neuroscience and
             Neuroimaging},
   Volume = {2},
   Number = {7},
   Pages = {582-590},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1016/j.bpsc.2017.06.005},
   Doi = {10.1016/j.bpsc.2017.06.005},
   Key = {fds328846}
}

@article{fds329468,
   Author = {Avinun, R and Nevo, A and Knodt, AR and Elliott, ML and Radtke, SR and Brigidi, BD and Hariri, AR},
   Title = {Reward-Related Ventral Striatum Activity Buffers against the
             Experience of Depressive Symptoms Associated with Sleep
             Disturbances.},
   Journal = {The Journal of Neuroscience : the Official Journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {40},
   Pages = {9724-9729},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1523/JNEUROSCI.1734-17.2017},
   Abstract = {Sleep disturbances represent one risk factor for depression.
             Reward-related brain function, particularly the activity of
             the ventral striatum (VS), has been identified as a
             potential buffer against stress-related depression. We were
             therefore interested in testing whether reward-related VS
             activity would moderate the effect of sleep disturbances on
             depression in a large cohort of young adults. Data were
             available from 1129 university students (mean age 19.71 ±
             1.25 years; 637 women) who completed a reward-related
             functional MRI task to assay VS activity and provided
             self-reports of sleep using the Pittsburgh Sleep Quality
             Index and symptoms of depression using a summation of the
             General Distress/Depression and Anhedonic Depression
             subscales of the Mood and Anxiety Symptoms
             Questionnaire-short form. Analyses revealed that as VS
             activity increased the association between sleep
             disturbances and depressive symptoms decreased. The
             interaction between sleep disturbances and VS activity was
             robust to the inclusion of sex, age, race/ethnicity, past or
             present clinical disorder, early and recent life stress, and
             anxiety symptoms, as well as the interactions between VS
             activity and early or recent life stress as covariates. We
             provide initial evidence that high reward-related VS
             activity may buffer against depressive symptoms associated
             with poor sleep. Our analyses help advance an emerging
             literature supporting the importance of individual
             differences in reward-related brain function as a potential
             biomarker of relative risk for depression.SIGNIFICANCE
             STATEMENT Sleep disturbances are a common risk factor for
             depression. An emerging literature suggests that
             reward-related activity of the ventral striatum (VS), a
             brain region critical for motivation and goal-directed
             behavior, may buffer against the effect of negative
             experiences on the development of depression. Using data
             from a large sample of 1129 university students we
             demonstrate that as reward-related VS activity increases,
             the link between sleep disturbances and depression
             decreases. This finding contributes to accumulating research
             demonstrating that reward-related brain function may be a
             useful biomarker of relative risk for depression in the
             context of negative experiences.},
   Doi = {10.1523/JNEUROSCI.1734-17.2017},
   Key = {fds329468}
}

@article{fds331565,
   Author = {Baranger, DAA and Margolis, S and Hariri, AR and Bogdan,
             R},
   Title = {An earlier time of scan is associated with greater
             threat-related amygdala reactivity.},
   Journal = {Social Cognitive and Affective Neuroscience},
   Volume = {12},
   Number = {8},
   Pages = {1272-1283},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1093/scan/nsx057},
   Abstract = {Time-dependent variability in mood and anxiety suggest that
             related neural phenotypes, such as threat-related amygdala
             reactivity, may also follow a diurnal pattern. Here, using
             data from 1,043 young adult volunteers, we found that
             threat-related amygdala reactivity was negatively coupled
             with time of day, an effect which was stronger in the left
             hemisphere (β = -0.1083, p-fdr = 0.0012). This
             effect was moderated by subjective sleep quality
             (β = -0.0715, p-fdr = 0.0387); participants who
             reported average and poor sleep quality had relatively
             increased left amygdala reactivity in the morning.
             Bootstrapped simulations suggest that similar
             cross-sectional samples with at least 300 participants would
             be able to detect associations between amygdala reactivity
             and time of scan. In control analyses, we found no
             associations between time and V1 activation. Our results
             provide initial evidence that threat-related amygdala
             reactivity may vary diurnally, and that this effect is
             potentiated among individuals with average to low sleep
             quality. More broadly, our results suggest that considering
             time of scan in study design or modeling time of scan in
             analyses, as well as collecting additional measures of
             circadian variation, may be useful for understanding
             threat-related neural phenotypes and their associations with
             behavior, such as fear conditioning, mood and anxiety
             symptoms, and related phenotypes.},
   Doi = {10.1093/scan/nsx057},
   Key = {fds331565}
}

@article{fds325037,
   Author = {Bogdan, R and Salmeron, BJ and Carey, CE and Agrawal, A and Calhoun, VD and Garavan, H and Hariri, AR and Heinz, A and Hill, MN and Holmes, A and Kalin, NH and Goldman, D},
   Title = {Imaging Genetics and Genomics in Psychiatry: A Critical
             Review of Progress and Potential.},
   Journal = {Biological Psychiatry},
   Volume = {82},
   Number = {3},
   Pages = {165-175},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.biopsych.2016.12.030},
   Abstract = {Imaging genetics and genomics research has begun to provide
             insight into the molecular and genetic architecture of
             neural phenotypes and the neural mechanisms through which
             genetic risk for psychopathology may emerge. As it
             approaches its third decade, imaging genetics is confronted
             by many challenges, including the proliferation of studies
             using small sample sizes and diverse designs, limited
             replication, problems with harmonization of neural
             phenotypes for meta-analysis, unclear mechanisms, and
             evidence that effect sizes may be more modest than
             originally posited, with increasing evidence of
             polygenicity. These concerns have encouraged the field to
             grow in many new directions, including the development of
             consortia and large-scale data collection projects and the
             use of novel methods (e.g., polygenic approaches, machine
             learning) that enhance the quality of imaging genetic
             studies but also introduce new challenges. We critically
             review progress in imaging genetics and offer suggestions
             and highlight potential pitfalls of novel approaches.
             Ultimately, the strength of imaging genetics and genomics
             lies in their translational and integrative potential with
             other research approaches (e.g., nonhuman animal models,
             psychiatric genetics, pharmacologic challenge) to elucidate
             brain-based pathways that give rise to the vast individual
             differences in behavior as well as risk for
             psychopathology.},
   Doi = {10.1016/j.biopsych.2016.12.030},
   Key = {fds325037}
}

@article{fds315805,
   Author = {Scult, MA and Knodt, AR and Hanson, JL and Ryoo, M and Adcock, RA and Hariri, AR and Strauman, TJ},
   Title = {Individual differences in regulatory focus predict neural
             response to reward.},
   Journal = {Social Neuroscience},
   Volume = {12},
   Number = {4},
   Pages = {419-429},
   Year = {2017},
   Month = {August},
   ISSN = {1747-0919},
   url = {http://dx.doi.org/10.1080/17470919.2016.1178170},
   Abstract = {Although goal pursuit is related to both functioning of the
             brain's reward circuits and psychological factors, the
             literatures surrounding these concepts have often been
             separate. Here, we use the psychological construct of
             regulatory focus to investigate individual differences in
             neural response to reward. Regulatory focus theory proposes
             two motivational orientations for personal goal pursuit: (1)
             promotion, associated with sensitivity to potential gain,
             and (2) prevention, associated with sensitivity to potential
             loss. The monetary incentive delay task was used to
             manipulate reward circuit function, along with instructional
             framing corresponding to promotion and prevention in a
             within-subject design. We observed that the more promotion
             oriented an individual was, the lower their ventral striatum
             response to gain cues. Follow-up analyses revealed that
             greater promotion orientation was associated with decreased
             ventral striatum response even to no-value cues, suggesting
             that promotion orientation may be associated with relatively
             hypoactive reward system function. The findings are also
             likely to represent an interaction between the cognitive and
             motivational characteristics of the promotion system with
             the task demands. Prevention orientation did not correlate
             with ventral striatum response to gain cues, supporting the
             discriminant validity of regulatory focus theory. The
             results highlight a dynamic association between individual
             differences in self-regulation and reward system
             function.},
   Doi = {10.1080/17470919.2016.1178170},
   Key = {fds315805}
}

@article{fds325971,
   Author = {Di Iorio and CR and Carey, CE and Michalski, LJ and Corral-Frias, NS and Conley, ED and Hariri, AR and Bogdan, R},
   Title = {Hypothalamic-pituitary-adrenal axis genetic variation and
             early stress moderates amygdala function.},
   Journal = {Psychoneuroendocrinology},
   Volume = {80},
   Pages = {170-178},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2017.03.016},
   Abstract = {Early life stress may precipitate psychopathology, at least
             in part, by influencing amygdala function. Converging
             evidence across species suggests that links between
             childhood stress and amygdala function may be dependent upon
             hypothalamic-pituitary-adrenal (HPA) axis function. Using
             data from college-attending non-Hispanic European-Americans
             (n=308) who completed the Duke Neurogenetics Study, we
             examined whether early life stress (ELS) and HPA axis
             genetic variation interact to predict threat-related
             amygdala function as well as psychopathology symptoms. A
             biologically-informed multilocus profile score (BIMPS)
             captured HPA axis genetic variation (FKBP5 rs1360780, CRHR1
             rs110402; NR3C2 rs5522/rs4635799) previously associated with
             its function (higher BIMPS are reflective of higher HPA axis
             activity). BOLD fMRI data were acquired while participants
             completed an emotional face matching task. ELS and
             depression and anxiety symptoms were measured using the
             childhood trauma questionnaire and the mood and anxiety
             symptom questionnaire, respectively. The interaction between
             HPA axis BIMPS and ELS was associated with right amygdala
             reactivity to threat-related stimuli, after accounting for
             multiple testing (empirical-p=0.016). Among individuals with
             higher BIMPS (i.e., the upper 21.4%), ELS was positively
             coupled with threat-related amygdala reactivity, which was
             absent among those with average or low BIMPS. Further,
             higher BIMPS were associated with greater self-reported
             anxious arousal, though there was no evidence that amygdala
             function mediated this relationship. Polygenic variation
             linked to HPA axis function may moderate the effects of
             early life stress on threat-related amygdala function and
             confer risk for anxiety symptomatology. However, what, if
             any, neural mechanisms may mediate the relationship between
             HPA axis BIMPS and anxiety symptomatology remains
             unclear.},
   Doi = {10.1016/j.psyneuen.2017.03.016},
   Key = {fds325971}
}

@article{fds325036,
   Author = {Dotterer, HL and Hyde, LW and Swartz, JR and Hariri, AR and Williamson,
             DE},
   Title = {Amygdala reactivity predicts adolescent antisocial behavior
             but not callous-unemotional traits.},
   Journal = {Developmental Cognitive Neuroscience},
   Volume = {24},
   Pages = {84-92},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.dcn.2017.02.008},
   Abstract = {Recent neuroimaging studies have suggested divergent
             relationships between antisocial behavior (AB) and
             callous-unemotional (CU) traits and amygdala reactivity to
             fearful and angry facial expressions in adolescents.
             However, little work has examined if these findings extend
             to dimensional measures of behavior in ethnically diverse,
             non-clinical samples, or if participant sex, ethnicity,
             pubertal stage, and age moderate associations. We examined
             links between amygdala reactivity and dimensions of AB and
             CU traits in 220 Hispanic and non-Hispanic Caucasian
             adolescents (age 11-15; 49.5% female; 38.2% Hispanic), half
             of whom had a family history for depression and thus were at
             relatively elevated risk for late starting, emotionally
             dysregulated AB. We found that AB was significantly related
             to increased right amygdala reactivity to angry facial
             expressions independent of sex, ethnicity, pubertal stage,
             age, and familial risk status for depression. CU traits were
             not related to fear- or anger-related amygdala reactivity.
             The present study further demonstrates that AB is related to
             increased amygdala reactivity to interpersonal threat cues
             in adolescents, and that this relationship generalizes
             across sex, ethnicity, pubertal stage, age, and familial
             risk status for depression.},
   Doi = {10.1016/j.dcn.2017.02.008},
   Key = {fds325036}
}

@article{fds325035,
   Author = {Swartz, JR and Prather, AA and Hariri, AR},
   Title = {Threat-related amygdala activity is associated with
             peripheral CRP concentrations in men but not
             women.},
   Journal = {Psychoneuroendocrinology},
   Volume = {78},
   Pages = {93-96},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.psyneuen.2017.01.024},
   Abstract = {Increased levels of peripheral inflammatory markers,
             including C-Reactive Protein (CRP), are associated with
             increased risk for depression, anxiety, and suicidality. The
             brain mechanisms that may underlie the association between
             peripheral inflammation and internalizing problems remain to
             be determined. The present study examines associations
             between peripheral CRP concentrations and threat-related
             amygdala activity, a neural biomarker of depression and
             anxiety risk, in a sample of 172 young adult undergraduate
             students. Participants underwent functional MRI scanning
             while performing an emotional face matching task to obtain a
             measure of threat-related amygdala activity to angry and
             fearful faces; CRP concentrations were assayed from dried
             blood spots. Results indicated a significant interaction
             between CRP and sex: in men, but not women, higher CRP was
             associated with higher threat-related amygdala activity.
             These results add to the literature finding associations
             between systemic levels of inflammation and brain function
             and suggest that threat-related amygdala activity may serve
             as a potential pathway through which heightened chronic
             inflammation may increase risk for mood and anxiety
             problems.},
   Doi = {10.1016/j.psyneuen.2017.01.024},
   Key = {fds325035}
}

@article{fds323253,
   Author = {Carey, CE and Knodt, AR and Conley, ED and Hariri, AR and Bogdan,
             R},
   Title = {Reward-Related Ventral Striatum Activity Links Polygenic
             Risk for Attention-Deficit/Hyperactivity Disorder to
             Problematic Alcohol Use in Young Adulthood},
   Journal = {Biological Psychiatry: Cognitive Neuroscience and
             Neuroimaging},
   Volume = {2},
   Number = {2},
   Pages = {180-187},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.bpsc.2016.10.003},
   Doi = {10.1016/j.bpsc.2016.10.003},
   Key = {fds323253}
}

@article{fds323689,
   Author = {Trampush, JW and Yang, MLZ and Yu, J and Knowles, E and Davies, G and Liewald, DC and Starr, JM and Djurovic, S and Melle, I and Sundet, K and Christoforou, A and Reinvang, I and DeRosse, P and Lundervold, AJ and Steen, VM and Espeseth, T and Räikkönen, K and Widen, E and Palotie,
             A and Eriksson, JG and Giegling, I and Konte, B and Roussos, P and Giakoumaki, S and Burdick, KE and Payton, A and Ollier, W and Horan, M and Chiba-Falek, O and Attix, DK and Need, AC and Cirulli, ET and Voineskos,
             AN and Stefanis, NC and Avramopoulos, D and Hatzimanolis, A and Arking,
             DE and Smyrnis, N and Bilder, RM and Freimer, NA and Cannon, TD and London,
             E and Poldrack, RA and Sabb, FW and Congdon, E and Conley, ED and Scult,
             MA and Dickinson, D and Straub, RE and Donohoe, G and Morris, D and Corvin,
             A and Gill, M and Hariri, AR and Weinberger, DR and Pendleton, N and Bitsios, P and Rujescu, D and Lahti, J and Le Hellard and S and Keller, MC and Andreassen, OA and Deary, IJ and Glahn, DC and Malhotra, AK and Lencz,
             T},
   Title = {GWAS meta-analysis reveals novel loci and genetic correlates
             for general cognitive function: a report from the COGENT
             consortium.},
   Journal = {Molecular Psychiatry},
   Volume = {22},
   Number = {3},
   Pages = {336-345},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1038/mp.2016.244},
   Abstract = {The complex nature of human cognition has resulted in
             cognitive genomics lagging behind many other fields in terms
             of gene discovery using genome-wide association study (GWAS)
             methods. In an attempt to overcome these barriers, the
             current study utilized GWAS meta-analysis to examine the
             association of common genetic variation (~8M
             single-nucleotide polymorphisms (SNP) with minor allele
             frequency ⩾1%) to general cognitive function in a sample
             of 35 298 healthy individuals of European ancestry across
             24 cohorts in the Cognitive Genomics Consortium (COGENT). In
             addition, we utilized individual SNP lookups and polygenic
             score analyses to identify genetic overlap with other
             relevant neurobehavioral phenotypes. Our primary GWAS
             meta-analysis identified two novel SNP loci (top SNPs:
             rs76114856 in the CENPO gene on chromosome 2 and rs6669072
             near LOC105378853 on chromosome 1) associated with cognitive
             performance at the genome-wide significance level (P<5 ×
             10-8). Gene-based analysis identified an additional three
             Bonferroni-corrected significant loci at chromosomes
             17q21.31, 17p13.1 and 1p13.3. Altogether, common variation
             across the genome resulted in a conservatively estimated SNP
             heritability of 21.5% (s.e.=0.01%) for general cognitive
             function. Integration with prior GWAS of cognitive
             performance and educational attainment yielded several
             additional significant loci. Finally, we found robust
             polygenic correlations between cognitive performance and
             educational attainment, several psychiatric disorders, birth
             length/weight and smoking behavior, as well as a novel
             genetic association to the personality trait of openness.
             These data provide new insight into the genetics of
             neurocognitive function with relevance to understanding the
             pathophysiology of neuropsychiatric illness.},
   Doi = {10.1038/mp.2016.244},
   Key = {fds323689}
}

@article{fds330406,
   Author = {Swartz, JR and Waller, R and Bogdan, R and Knodt, AR and Sabhlok, A and Hyde, LW and Hariri, AR},
   Title = {A Common Polymorphism in a Williams Syndrome Gene Predicts
             Amygdala Reactivity and Extraversion in Healthy
             Adults.},
   Journal = {Biological Psychiatry},
   Volume = {81},
   Number = {3},
   Pages = {203-210},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.biopsych.2015.12.007},
   Abstract = {Williams syndrome (WS), a genetic disorder resulting from
             hemizygous microdeletion of chromosome 7q11.23, has emerged
             as a model for identifying the genetic architecture of
             socioemotional behavior. Common polymorphisms in GTF2I,
             which is found within the WS microdeletion, have been
             associated with reduced social anxiety in the general
             population. Identifying neural phenotypes affected by these
             polymorphisms would help advance our understanding not only
             of this specific genetic association but also of the broader
             neurogenetic mechanisms of variability in socioemotional
             behavior.Through an ongoing parent protocol, the Duke
             Neurogenetics Study, we measured threat-related amygdala
             reactivity to fearful and angry facial expressions using
             functional magnetic resonance imaging, assessed trait
             personality using the Revised NEO Personality Inventory, and
             imputed GTF2I rs13227433 from saliva-derived DNA using
             custom Illumina arrays. Participants included 808
             non-Hispanic Caucasian, African American, and Asian
             university students.The GTF2I rs13227433 AA genotype,
             previously associated with lower social anxiety, predicted
             decreased threat-related amygdala reactivity. An indirect
             effect of GTF2I genotype on the warmth facet of extraversion
             was mediated by decreased threat-related amygdala reactivity
             in women but not men.A common polymorphism in the WS gene
             GTF2I associated with reduced social anxiety predicts
             decreased threat-related amygdala reactivity, which mediates
             an association between genotype and increased warmth in
             women. These results are consistent with reduced
             threat-related amygdala reactivity in WS and suggest that
             common variation in GTF2I contributes to broader variability
             in socioemotional brain function and behavior, with
             implications for understanding the neurogenetic bases of WS
             as well as social anxiety.},
   Doi = {10.1016/j.biopsych.2015.12.007},
   Key = {fds330406}
}

@article{fds322497,
   Author = {Swartz, JR and Knodt, AR and Radtke, SR and Hariri,
             AR},
   Title = {Peering into the brain to predict behavior: Peer-reported,
             but not self-reported, conscientiousness links
             threat-related amygdala activity to future problem
             drinking.},
   Journal = {Neuroimage},
   Volume = {146},
   Pages = {894-903},
   Year = {2017},
   Month = {February},
   url = {http://dx.doi.org/10.1016/j.neuroimage.2016.10.003},
   Abstract = {Personality traits such as conscientiousness as
             self-reported by individuals can help predict a range of
             outcomes, from job performance to longevity. Asking others
             to rate the personality of their acquaintances often
             provides even better predictive power than using
             self-report. Here, we examine whether peer-reported
             personality can provide a better link between brain
             function, namely threat-related amygdala activity, and
             future health-related behavior, namely problem drinking,
             than self-reported personality. Using data from a sample of
             377 young adult university students who were rated on five
             personality traits by peers, we find that higher
             threat-related amygdala activity to fearful facial
             expressions is associated with higher peer-reported, but not
             self-reported, conscientiousness. Moreover, higher
             peer-reported, but not self-reported, conscientiousness
             predicts lower future problem drinking more than one year
             later, an effect specific to men. Remarkably, relatively
             higher amygdala activity has an indirect effect on future
             drinking behavior in men, linked by peer-reported
             conscientiousness to lower future problem drinking. Our
             results provide initial evidence that the perceived
             conscientiousness of an individual by their peers uniquely
             reflects variability in a core neural mechanism supporting
             threat responsiveness. These novel patterns further suggest
             that incorporating peer-reported measures of personality
             into individual differences research can reveal novel
             predictive pathways of risk and protection for problem
             behaviors.},
   Doi = {10.1016/j.neuroimage.2016.10.003},
   Key = {fds322497}
}

@article{fds316880,
   Author = {Swartz, JR and Hariri, AR and Williamson, DE},
   Title = {An epigenetic mechanism links socioeconomic status to
             changes in depression-related brain function in high-risk
             adolescents.},
   Journal = {Molecular Psychiatry},
   Volume = {22},
   Number = {2},
   Pages = {209-214},
   Year = {2017},
   Month = {February},
   ISSN = {1359-4184},
   url = {http://dx.doi.org/10.1038/mp.2016.82},
   Abstract = {Identifying biological mechanisms through which the
             experience of adversity emerges as individual risk for
             mental illness is an important step toward developing
             strategies for personalized treatment and, ultimately,
             prevention. Preclinical studies have identified epigenetic
             modification of gene expression as one such mechanism.
             Recent clinical studies have suggested that epigenetic
             modification, particularly methylation of gene regulatory
             regions, also acts to shape human brain function associated
             with risk for mental illness. However, it is not yet clear
             whether differential gene methylation as a function of
             adversity contributes to the emergence of individual risk
             for mental illness. Using prospective longitudinal
             epigenetic, neuroimaging and behavioral data from 132
             adolescents, we demonstrate that changes in gene methylation
             associated with lower socioeconomic status (SES) predict
             changes in risk-related brain function. Specifically, we
             find that lower SES during adolescence is associated with an
             increase in methylation of the proximal promoter of the
             serotonin transporter gene, which predicts greater increases
             in threat-related amygdala reactivity. We subsequently
             demonstrate that greater increases in amygdala reactivity
             moderate the association between a positive family history
             for depression and the later manifestation of depressive
             symptoms. These initial results suggest a specific
             biological mechanism through which adversity contributes to
             altered brain function, which in turn moderates the
             emergence of general liability as individual risk for mental
             illness. If replicated, this prospective pathway may
             represent a novel target biomarker for intervention and
             prevention among high-risk individuals.},
   Doi = {10.1038/mp.2016.82},
   Key = {fds316880}
}

@article{fds318715,
   Author = {Swartz, JR and Prather, AA and Di Iorio and CR and Bogdan, R and Hariri,
             AR},
   Title = {A Functional Interleukin-18 Haplotype Predicts Depression
             and Anxiety through Increased Threat-Related Amygdala
             Reactivity in Women but Not Men.},
   Journal = {Neuropsychopharmacology},
   Volume = {42},
   Number = {2},
   Pages = {419-426},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1038/npp.2016.129},
   Abstract = {Common functional polymorphisms in the gene encoding
             interleukin-18 (IL18), a cytokine belonging to the IL-1
             superfamily that can induce synthesis of several other
             cytokines, have been associated with major depressive
             episodes following the experience of stressful life events.
             The neural mechanisms underlying these associations remain
             unexamined. Here we use an imaging genetics strategy to
             examine the effects of risk-related IL18 haplotypes
             comprising rs187238 and rs1946518 on threat-related amygdala
             reactivity and, through an indirect effect, stress-related
             symptoms of depression and anxiety in 448 non-Hispanic
             Caucasian university students. Analyses indicated that women
             but not men possessing an IL18 haplotype comprising both
             risk-related alleles evidenced increased threat-related left
             centromedial amygdala reactivity relative to other haplotype
             groups. Moreover, in women only, increased threat-related
             left centromedial amygdala reactivity predicted increased
             symptoms of depression and anxiety in individuals also
             reporting higher levels of life stress. Path analyses
             revealed a significant indirect effect of IL18 risk
             haplotype on symptoms of depression and anxiety through
             increased threat-related amygdala reactivity. These results
             suggest that a common functional IL18 haplotype associated
             with heightened proinflammatory responses confers
             susceptibility to stress-related depression and anxiety
             through effects on threat-related amygdala function, a risk
             pathway specific to women. If replicated, these patterns can
             inform the search for personalized interventions targeting
             neurobiological pathways of risk associated with
             inflammation.},
   Doi = {10.1038/npp.2016.129},
   Key = {fds318715}
}

@article{fds323776,
   Author = {Scult, MA and Knodt, AR and Swartz, JR and Brigidi, BD and Hariri,
             AR},
   Title = {Thinking and Feeling: Individual Differences in Habitual
             Emotion Regulation and Stress-Related Mood are Associated
             with Prefrontal Executive Control.},
   Journal = {Clinical Psychological Science : a Journal of the
             Association for Psychological Science},
   Volume = {5},
   Number = {1},
   Pages = {150-157},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1177/2167702616654688},
   Abstract = {Calculating math problems from memory may seem unrelated to
             everyday processing of emotions, but they have more in
             common than one might think. Prior research highlights the
             importance of the dorsolateral prefrontal cortex (dlPFC) in
             executive control, intentional emotion regulation, and
             experience of dysfunctional mood and anxiety. While it has
             been hypothesized that emotion regulation may be related to
             'cold' (ie. not emotion-related) executive control, this
             assertion has not been tested. We address this gap by
             providing evidence that greater dlPFC activity during 'cold'
             executive control is associated with increased use of
             cognitive reappraisal to regulate emotions in everyday life.
             We then demonstrate that in the presence of increased life
             stress, increased dlPFC activity is associated with lower
             mood and anxiety symptoms and clinical diagnoses.
             Collectively, our results encourage ongoing efforts to
             understand prefrontal executive control as a possible
             intervention target for improving emotion regulation in mood
             and anxiety disorders.},
   Doi = {10.1177/2167702616654688},
   Key = {fds323776}
}

@article{fds318712,
   Author = {Scult, MA and Paulli, AR and Mazure, ES and Moffitt, TE and Hariri, AR and Strauman, TJ},
   Title = {The association between cognitive function and subsequent
             depression: a systematic review and meta-analysis.},
   Journal = {Psychological Medicine},
   Volume = {47},
   Number = {1},
   Pages = {1-17},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1017/S0033291716002075},
   Abstract = {Despite a growing interest in understanding the cognitive
             deficits associated with major depressive disorder (MDD), it
             is largely unknown whether such deficits exist before
             disorder onset or how they might influence the severity of
             subsequent illness. The purpose of the present study was to
             conduct a systematic review and meta-analysis of
             longitudinal datasets to determine whether cognitive
             function acts as a predictor of later MDD diagnosis or
             change in depression symptoms. Eligible studies included
             longitudinal designs with baseline measures of cognitive
             functioning, and later unipolar MDD diagnosis or symptom
             assessment. The systematic review identified 29
             publications, representing 34 unique samples, and 121 749
             participants, that met the inclusion/exclusion criteria.
             Quantitative meta-analysis demonstrated that higher
             cognitive function was associated with decreased levels of
             subsequent depression (r = -0.088, 95% confidence interval.
             -0.121 to -0.054, p < 0.001). However, sensitivity analyses
             revealed that this association is likely driven by
             concurrent depression symptoms at the time of cognitive
             assessment. Our review and meta-analysis indicate that the
             association between lower cognitive function and later
             depression is confounded by the presence of contemporaneous
             depression symptoms at the time of cognitive assessment.
             Thus, cognitive deficits predicting MDD likely represent
             deleterious effects of subclinical depression symptoms on
             performance rather than premorbid risk factors for
             disorder.},
   Doi = {10.1017/S0033291716002075},
   Key = {fds318712}
}


%% Hopkins, Christina M.   
@article{fds338001,
   Author = {Hopkins, CM and Bennett, GG},
   Title = {Weight-Related Terms Differentially Affect Self-Efficacy and
             Perception of Obesity.},
   Journal = {Obesity (Silver Spring, Md.)},
   Volume = {26},
   Number = {9},
   Pages = {1405-1411},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1002/oby.22255},
   Abstract = {Little work has explored the effect of weight-related terms
             on treatment initiation; only one study has investigated
             weight-related terms and the psychological constructs
             associated with treatment uptake. The present study examines
             the effects of four common weight-related terms on treatment
             initiation and the moderating effect of weight bias
             internalization.Adult participants with overweight and
             obesity (n = 436) were recruited online and asked to
             read three vignettes describing clinical encounters; the
             weight-related term (i.e., "weight," "BMI," "obesity," or
             "fat") was varied randomly. Participants then reported
             self-efficacy, cognitive and emotional illness beliefs about
             obesity (i.e., illness perception), and interest in a weight
             loss program.The term "obesity" resulted in the greatest
             self-efficacy and perceived control over obesity. "Fat"
             resulted in the least illness coherence (i.e., understanding
             of obesity). Weight bias internalization did not moderate
             the effect of term on self-efficacy, nor did it moderate
             illness perception. No differences in weight loss program
             enrollment were observed.Use of the term "obesity" may
             promote patients' perceived control and self-efficacy. Use
             of "fat" should be avoided. Results suggest that, despite
             patient and clinician preference for euphemistic weight
             terms, use of clinical language such as "obesity" may
             perform better in provider intervention.},
   Doi = {10.1002/oby.22255},
   Key = {fds338001}
}

@article{fds335692,
   Author = {Faulconbridge, LF and Driscoll, CFB and Hopkins, CM and Bailer
             Benforado, B and Bishop-Gilyard, C and Carvajal, R and Berkowitz, RI and DeRubeis, R and Wadden, TA},
   Title = {Combined Treatment for Obesity and Depression: A Pilot
             Study.},
   Journal = {Obesity (Silver Spring, Md.)},
   Volume = {26},
   Number = {7},
   Pages = {1144-1152},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1002/oby.22209},
   Abstract = {Obesity and depression frequently co-occur, and each
             increases risk for cardiovascular disease (CVD). This study
             tested whether a combined treatment, targeting obesity and
             depression simultaneously, would yield greater improvements
             in weight, mood, and CVD risk factors than treatments that
             targeted each disease individually.Seventy-six participants
             with obesity and major depression were randomly assigned to
             (1) behavioral weight control (BWC), (2) cognitive
             behavioral therapy for depression (CBT-D), or (3) BWC
             combined with CBT-D. Participants were provided 18 group
             treatment sessions over 20 weeks. Mood, weight, and CVD risk
             were assessed at baseline and weeks 8 and 20, with a
             follow-up visit at week 46.At week 20, participants in
             combined treatment lost significantly (P < 0.02) more
             weight (5.2% ± 1.2%) than those assigned to CBT-D
             (0.8% ± 1.3%) and comparable amounts as those in BWC
             (3.5% ± 1.3%). Depression scores decreased
             significantly from baseline levels in each group, with no
             significant differences between groups. All three groups
             showed significant improvements in 10-year CVD risk, with no
             significant differences between groups. Groups did not
             differ significantly on any of these measures at week 46.BWC
             yielded short-term improvements in weight, mood, and CVD
             risk, comparable to a combined treatment that incorporated
             CBT-D. Results require replication with a larger sample
             size.},
   Doi = {10.1002/oby.22209},
   Key = {fds335692}
}


%% Hoyle, Rick   
@article{fds337342,
   Author = {Gehrt, TB and Berntsen, D and Hoyle, RH and Rubin,
             DC},
   Title = {Psychological and clinical correlates of the Centrality of
             Event Scale: A systematic review.},
   Journal = {Clinical Psychology Review},
   Volume = {65},
   Pages = {57-80},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.cpr.2018.07.006},
   Abstract = {The Centrality of Event Scale (CES) was introduced to
             examine the extent to which a traumatic or stressful event
             is perceived as central to an individual's identity and life
             story, and how this relates to Posttraumatic Stress Disorder
             (PTSD) symptoms. In addition, the CES has been examined in
             relation to a range of other conditions and dispositions. We
             present a systematic review of the correlates of the CES.
             Results from 92 publications resulted in 25 measurement
             categories in the six theoretical domains of trauma,
             negative affect and distress, autobiographical memory,
             personality, positive affect, and gender. The mean weighted
             correlations of the 25 measurement categories ranged from
             -.17 to .55, with standard errors from .01 to .02, allowing
             us to distinguish empirically among effects. Consistent with
             the theoretical motivation for the CES and predictions
             predating the review, the CES correlated positively with a
             range of measures, correlating most highly with measures
             related to trauma, PTSD, grief, and autobiographical memory.
             The findings show that the CES probes aspects of
             autobiographical memory of broad relevance to clinical
             disorders, and with specific implications for theories of
             PTSD.},
   Doi = {10.1016/j.cpr.2018.07.006},
   Key = {fds337342}
}

@article{fds332881,
   Author = {Arco-Tirado, JL and Fernández-Martín, FD and Hoyle,
             RH},
   Title = {Development and Validation of a Spanish Version of the
             Grit-S Scale},
   Journal = {Frontiers in Psychology},
   Volume = {9},
   Number = {FEB},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.3389/fpsyg.2018.00096},
   Abstract = {© 2018 Arco-Tirado, Fernández-Martín and Hoyle. This
             paper describes the development and initial validation of a
             Spanish version of the Short Grit (Grit-S) Scale. The Grit-S
             Scale was adapted and translated into Spanish using the
             Translation, Review, Adjudication, Pre-testing, and
             Documentation model and responses to a preliminary set of
             items from a large sample of university students (N =
             1,129). The resultant measure was validated using data from
             a large stratified random sample of young adults (N =
             1,826). Initial validation involved evaluating the internal
             consistency of the adapted scale and its subscales and
             comparing the factor structure of the adapted version to
             that of the original scale. The results were comparable to
             results from similar analyses of the English version of the
             scale. Although the internal consistency of the subscales
             was low, the internal consistency of the full scale was
             well-within the acceptable range. A two-factor model offered
             an acceptable account of the data; however, when a single
             correlated error involving two highly similar items was
             included, a single factor model fit the data very well. The
             results support the use of overall scores from the Spanish
             Grit-S Scale in future research.},
   Doi = {10.3389/fpsyg.2018.00096},
   Key = {fds332881}
}

@article{fds336522,
   Author = {Duffy, KA and Helzer, EG and Hoyle, RH and Fukukura Helzer and J and Chartrand, TL},
   Title = {Pessimistic expectations and poorer experiences: The role of
             (low) extraversion in anticipated and experienced enjoyment
             of social interaction.},
   Journal = {Plos One},
   Volume = {13},
   Number = {7},
   Pages = {e0199146},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1371/journal.pone.0199146},
   Abstract = {Given research suggesting that social interactions are
             beneficial, it is unclear why individuals lower in
             extraversion engage less in social interactions. In this
             study, we test whether individuals lower in extraversion
             reap fewer hedonic rewards from social interactions and
             explore social psychological processes that explain their
             experiences. Before participants socialized, we measured
             extraversion, state positive affect, cognitive capacity, and
             expectations about the social interactions. After
             participants socialized with one another, we measured state
             positive affect and cognitive capacity again as well as fear
             of negative evaluation and belief in limited cognitive
             capacity. Participants also rated the social skillfulness of
             each interaction partner. We found that less extraverted
             individuals expect to feel worse after socializing. However,
             all but those extremely low in extraversion (17% of sample)
             actually experience an increase in positive affect after
             socializing. Surprisingly, those low in extraversion did not
             show reduced cognitive capacity after socializing. Although
             they are more likely to believe that cognitive capacity is
             limited and to be fearful of negative evaluation, these
             characteristics did not explain the social experience of
             those low in extraversion.},
   Doi = {10.1371/journal.pone.0199146},
   Key = {fds336522}
}

@article{fds327866,
   Author = {Piontak, JR and Russell, MA and Danese, A and Copeland, WE and Hoyle,
             RH and Odgers, CL},
   Title = {Violence exposure and adolescents' same-day obesogenic
             behaviors: New findings and a replication.},
   Journal = {Social Science & Medicine},
   Volume = {189},
   Pages = {145-151},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.socscimed.2017.07.004},
   Abstract = {To test whether exposure to violence is associated with
             same-day increases in obesogenic behaviors among young
             adolescents, including unhealthy food and beverage
             consumption, poor quality sleep, and lack of physical
             activity.Young at-risk adolescents between 12 and 15 years
             of age were recruited via telephone screening from
             low-income neighborhoods. Adolescents and their parents
             completed in-person assessments, followed by Ecological
             Momentary Assessment (EMA) delivered to 151 adolescents'
             mobile phones three times a day for 30 days (4329 person
             days). Three obesogenic behaviors - unhealthy food
             consumption, poor sleep quality, and lack of physical
             activity - and violence exposure were assessed daily.
             Adolescents' body mass index (BMI) was assessed prior to the
             EMA and 18 months later. A replication was performed among
             395 adolescents from a population-representative sample
             (with 5276 EMA person days).On days that at-risk adolescents
             were exposed versus not exposed to violence, they were more
             likely to consume unhealthy foods and beverages (b = 0.12,
             p = 0.01), report feeling tired the next morning
             (OR = 1.58, p < 0.01), and to be active (OR = 1.61,
             p < 0.01). At-risk adolescents who reported higher
             consumption of soda and caffeinated beverages during the
             30-day EMA were more likely to experience increases in BMI
             in later adolescence. Findings related to sleep and activity
             were supported in the population-based replication sample;
             however, no significant same-day associations were found
             between violence exposure and unhealthy dietary
             consumption.This study provides evidence that exposure to
             violence is associated with same-day unhealthy dietary
             consumption among at-risk adolescents and next-day tiredness
             related to sleep quality among adolescents from both at-risk
             and normative populations. Findings also point to unhealthy
             soda consumption during early adolescence as an important
             predictor of weight gain among at-risk adolescents.},
   Doi = {10.1016/j.socscimed.2017.07.004},
   Key = {fds327866}
}

@article{fds326828,
   Author = {Leary, MR and Diebels, KJ and Davisson, EK and Jongman-Sereno, KP and Isherwood, JC and Raimi, KT and Deffler, SA and Hoyle,
             RH},
   Title = {Cognitive and Interpersonal Features of Intellectual
             Humility.},
   Journal = {Personality & Social Psychology Bulletin},
   Volume = {43},
   Number = {6},
   Pages = {793-813},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1177/0146167217697695},
   Abstract = {Four studies examined intellectual humility-the degree to
             which people recognize that their beliefs might be wrong.
             Using a new Intellectual Humility (IH) Scale, Study 1 showed
             that intellectual humility was associated with variables
             related to openness, curiosity, tolerance of ambiguity, and
             low dogmatism. Study 2 revealed that participants high in
             intellectual humility were less certain that their beliefs
             about religion were correct and judged people less on the
             basis of their religious opinions. In Study 3, participants
             high in intellectual humility were less inclined to think
             that politicians who changed their attitudes were
             "flip-flopping," and Study 4 showed that people high in
             intellectual humility were more attuned to the strength of
             persuasive arguments than those who were low. In addition to
             extending our understanding of intellectual humility, this
             research demonstrates that the IH Scale is a valid measure
             of the degree to which people recognize that their beliefs
             are fallible.},
   Doi = {10.1177/0146167217697695},
   Key = {fds326828}
}


%% Huettel, Scott   
@article{fds332798,
   Author = {Wing, EA and Iyengar, V and Hess, TM and LaBar, KS and Huettel, SA and Cabeza, R},
   Title = {Neural mechanisms underlying subsequent memory for personal
             beliefs:An fMRI study.},
   Journal = {Cognitive, Affective & Behavioral Neuroscience},
   Volume = {18},
   Number = {2},
   Pages = {216-231},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.3758/s13415-018-0563-y},
   Abstract = {Many fMRI studies have examined the neural mechanisms
             supporting emotional memory for stimuli that generate
             emotion rather automatically (e.g., a picture of a dangerous
             animal or of appetizing food). However, far fewer studies
             have examined how memory is influenced by emotion related to
             social and political issues (e.g., a proposal for large
             changes in taxation policy), which clearly vary across
             individuals. In order to investigate the neural substrates
             of affective and mnemonic processes associated with personal
             opinions, we employed an fMRI task wherein participants
             rated the intensity of agreement/disagreement to
             sociopolitical belief statements paired with neural face
             pictures. Following the rating phase, participants performed
             an associative recognition test in which they distinguished
             identical versus recombined face-statement pairs. The study
             yielded three main findings: behaviorally, the intensity of
             agreement ratings was linked to greater subjective emotional
             arousal as well as enhanced high-confidence subsequent
             memory. Neurally, statements that elicited strong (vs. weak)
             agreement or disagreement were associated with greater
             activation of the amygdala. Finally, a subsequent memory
             analysis showed that the behavioral memory advantage for
             statements generating stronger ratings was dependent on the
             medial prefrontal cortex (mPFC). Together, these results
             both underscore consistencies in neural systems supporting
             emotional arousal and suggest a modulation of
             arousal-related encoding mechanisms when emotion is
             contingent on referencing personal beliefs.},
   Doi = {10.3758/s13415-018-0563-y},
   Key = {fds332798}
}

@article{fds327384,
   Author = {Meade, CS and Addicott, M and Hobkirk, AL and Towe, SL and Chen, N-K and Sridharan, S and Huettel, SA},
   Title = {Cocaine and HIV are independently associated with neural
             activation in response to gain and loss valuation during
             economic risky choice.},
   Journal = {Addiction Biology},
   Volume = {23},
   Number = {2},
   Pages = {796-809},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1111/adb.12529},
   Abstract = {Stimulant abuse is disproportionately common in HIV-positive
             persons. Both HIV and stimulants are independently
             associated with deficits in reward-based decision making,
             but their interactive and/or additive effects are poorly
             understood despite their prevalent co-morbidity. Here, we
             examined the effects of cocaine dependence and HIV infection
             in 69 adults who underwent functional magnetic resonance
             imaging while completing an economic loss aversion task. We
             identified two neural networks that correlated with the
             evaluation of the favorable characteristics of the gamble
             (i.e. higher gains/lower losses: ventromedial prefrontal
             cortex, anterior cingulate, anterior and posterior precuneus
             and visual cortex) versus unfavorable characteristics of the
             gamble (i.e. lower gains/higher losses: dorsal prefrontal,
             lateral orbitofrontal, posterior parietal cortex, anterior
             insula and dorsal caudate). Behaviorally, cocaine and HIV
             had additive effects on loss aversion scores, with
             HIV-positive cocaine users being the least loss averse.
             Cocaine users had greater activation in brain regions that
             tracked the favorability of gamble characteristics (i.e.
             increased activation to gains, but decreased activation to
             losses). In contrast, HIV infection was independently
             associated with lesser activation in regions that tracked
             the unfavorability of gamble characteristics. These results
             suggest that cocaine is associated with an overactive
             reward-seeking system, while HIV is associated with an
             underactive cognitive control system. Together, these
             alterations may leave HIV-positive cocaine users
             particularly vulnerable to making unfavorable decisions when
             outcomes are uncertain.},
   Doi = {10.1111/adb.12529},
   Key = {fds327384}
}

@article{fds330849,
   Author = {Kwak, Y and Huettel, S},
   Title = {The order of information processing alters economic
             gain-loss framing effects.},
   Journal = {Acta Psychologica},
   Volume = {182},
   Pages = {46-54},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.actpsy.2017.11.013},
   Abstract = {Adaptive decision making requires analysis of available
             information during the process of choice. In many decisions
             that information is presented visually - which means that
             variations in visual properties (e.g., salience, complexity)
             can potentially influence the process of choice. In the
             current study, we demonstrate that variation in the
             left-right positioning of risky and safe decision options
             can influence the canonical gain-loss framing effect. Two
             experiments were conducted using an economic framing task in
             which participants chose between gambles and certain
             outcomes. The first experiment demonstrated that the
             magnitude of the gain-loss framing effect was greater when
             the certain option signaling the current frame was presented
             on the left side of the visual display. Eye-tracking data
             during task performance showed a left-gaze bias for initial
             fixations, suggesting that the option presented on the left
             side was processed first. Combination of eye-tracking and
             choice data revealed that there was a significant effect of
             direction of first gaze (i.e. left vs. right) as well as an
             interaction between gaze direction and identity of the first
             fixated information (i.e. certain vs. gamble) regardless of
             frame. A second experiment presented the gamble and certain
             options in a random order, with a temporal delay between
             their presentations. We found that the magnitude of
             gain-loss framing was larger when the certain option was
             presented first, regardless of left and right positioning,
             only in individuals with lower risk-taking tendencies. The
             effect of presentation order on framing was not present in
             high risk-takers. These results suggest that the sequence of
             visual information processing as well as their left-right
             positioning can bias choices by changing the impact of the
             presented information during risky decision
             making.},
   Doi = {10.1016/j.actpsy.2017.11.013},
   Key = {fds330849}
}

@article{fds329283,
   Author = {Stanton, SJ and Sinnott-Armstrong, W and Huettel,
             SA},
   Title = {Neuromarketing: Ethical Implications of its Use and
             Potential Misuse},
   Journal = {Journal of Business Ethics},
   Volume = {144},
   Number = {4},
   Pages = {799-811},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1007/s10551-016-3059-0},
   Doi = {10.1007/s10551-016-3059-0},
   Key = {fds329283}
}

@article{fds329566,
   Author = {Utevsky, AV and Smith, DV and Young, JS and Huettel,
             SA},
   Title = {Large-Scale Network Coupling with the Fusiform Cortex
             Facilitates Future Social Motivation.},
   Journal = {Eneuro},
   Volume = {4},
   Number = {5},
   Pages = {ENEURO.0084-17.2017-ENEURO.0084-17.2017},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1523/ENEURO.0084-17.2017},
   Abstract = {Large-scale functional networks, as identified through the
             coordinated activity of spatially distributed brain regions,
             have become central objects of study in neuroscience because
             of their contributions to many processing domains. Yet, it
             remains unclear how these domain-general networks interact
             with focal brain regions to coordinate thought and action.
             Here, we investigated how the default-mode network (DMN) and
             executive control network (ECN), two networks associated
             with goal-directed behavior, shape task performance through
             their coupling with other cortical regions several seconds
             in advance of behavior. We measured these networks'
             connectivity during an adaptation of the monetary incentive
             delay (MID) response-time task in which human participants
             viewed social and nonsocial images (i.e., pictures of faces
             and landscapes, respectively) while brain activity was
             measured using fMRI. We found that participants displayed
             slower reaction times (RTs) subsequent to social trials
             relative to nonsocial trials. To examine the neural
             mechanisms driving this subsequent-RT effect, we integrated
             independent components analysis (ICA) and a network-based
             psychophysiological interaction (nPPI) analysis; this
             allowed us to investigate task-related changes in network
             coupling that preceded the observed trial-to-trial variation
             in RT. Strikingly, when subjects viewed social rewards, an
             area of the fusiform gyrus (FG) consistent with the
             functionally-defined fusiform face area (FFA) exhibited
             increased coupling with the ECN (relative to the DMN), and
             the relative magnitude of coupling tracked the slowing of RT
             on the following trial. These results demonstrate how
             large-scale, domain-general networks can interact with
             focal, domain-specific cortical regions to orchestrate
             subsequent behavior.},
   Doi = {10.1523/ENEURO.0084-17.2017},
   Key = {fds329566}
}

@article{fds327383,
   Author = {Meade, CS and Hobkirk, AL and Towe, SL and Chen, N-K and Bell, RP and Huettel, SA},
   Title = {Cocaine dependence modulates the effect of HIV infection on
             brain activation during intertemporal decision
             making.},
   Journal = {Drug and Alcohol Dependence},
   Volume = {178},
   Pages = {443-451},
   Year = {2017},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.drugalcdep.2017.05.043},
   Abstract = {Both HIV infection and chronic cocaine use alter the neural
             circuitry of decision making, but the interactive effects of
             these commonly comorbid conditions have not been adequately
             examined. This study tested how cocaine moderates
             HIV-related neural activation during an intertemporal
             decision-making task.The sample included 73 participants who
             differed on cocaine and HIV status (18 COC+/HIV+, 19
             COC+/HIV-, 19 COC-/HIV+, 17 COC-/HIV-). Participants made
             choices between smaller, sooner and larger, delayed rewards
             while undergoing functional MRI. Choices varied in
             difficulty based on subjective value: hard (equivalently
             valued), easy (disparately valued), and control choices. A
             mixed-effects model controlling for education and smoking
             identified main and interactive effects of HIV and COC
             during hard relative to easy choices (difficulty
             contrast).COC+ status was associated with lower activation
             in bilateral frontal gyri and right insular and posterior
             parietal cortices. HIV+ status was associated with higher
             activation in the visual cortex, but lower activation in
             bilateral prefrontal cortices and cerebellum and left
             posterior parietal cortex. COC moderated the effects of HIV
             in several clusters centered in the bilateral prefrontal
             cortices and cerebellum. In post-hoc analyses, there were
             significant effects of HIV status on activation for COC+,
             but not COC-, participants; interaction effects remained
             after controlling for polysubstance use.Cocaine use may
             diminish the compensatory neural activation often seen among
             HIV+ samples during decision making. Our results highlight
             the importance of examining the neuropsychiatric effects of
             comorbid medical conditions to identify potential neural
             targets for cognitive remediation interventions.},
   Doi = {10.1016/j.drugalcdep.2017.05.043},
   Key = {fds327383}
}

@article{fds330179,
   Author = {Kelly, M and Ngo, L and Chituc, V and Huettel, S and Sinnott-Armstrong,
             W},
   Title = {Moral conformity in online interactions: rational
             justifications increase influence of peer opinions on moral
             judgments},
   Journal = {Social Influence},
   Volume = {12},
   Number = {2-3},
   Pages = {57-68},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1080/15534510.2017.1323007},
   Doi = {10.1080/15534510.2017.1323007},
   Key = {fds330179}
}

@article{fds325992,
   Author = {Li, R and Roberts, RC and Huettel, SA and Brannon,
             EM},
   Title = {Five-year-olds do not show ambiguity aversion in a risk and
             ambiguity task with physical objects.},
   Journal = {Journal of Experimental Child Psychology},
   Volume = {159},
   Pages = {319-326},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1016/j.jecp.2017.02.013},
   Abstract = {Ambiguity aversion arises when a decision maker prefers
             risky gambles with known probabilities over equivalent
             ambiguous gambles with unknown probabilities. This
             phenomenon has been consistently observed in adults across a
             large body of empirical work. Evaluating ambiguity aversion
             in young children, however, has posed methodological
             challenges because probabilistic representations appropriate
             for adults might not be understood by young children. Here,
             we established a novel method for representing risk and
             ambiguity with physical objects that overcomes previous
             methodological limitations and allows us to measure
             ambiguity aversion in young children. We found that
             individual 5-year-olds exhibited consistent choice
             preferences and, as a group, exhibited no ambiguity aversion
             in a task that evokes ambiguity aversion in adults. Across
             individuals, 5-year-olds exhibited greater variance in
             ambiguity preferences compared with adults tested under
             similar conditions. This suggests that ambiguity aversion is
             absent during early childhood and emerges over the course of
             development.},
   Doi = {10.1016/j.jecp.2017.02.013},
   Key = {fds325992}
}

@article{fds324858,
   Author = {Cordero, DM and Towe, SL and Chen, N-K and Robertson, KR and Madden, DJ and Huettel, SA and Meade, CS},
   Title = {Cocaine dependence does not contribute substantially to
             white matter abnormalities in HIV infection.},
   Journal = {Journal of Neurovirology},
   Volume = {23},
   Number = {3},
   Pages = {441-450},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s13365-017-0512-5},
   Abstract = {This study investigated the association of HIV infection and
             cocaine dependence with cerebral white matter integrity
             using diffusion tensor imaging (DTI). One hundred
             thirty-five participants stratified by HIV and cocaine
             status (26 HIV+/COC+, 37 HIV+/COC-, 37 HIV-/COC+, and 35
             HIV-/COC-) completed a comprehensive substance abuse
             assessment, neuropsychological testing, and MRI with DTI.
             Among HIV+ participants, all were receiving HIV care and 46%
             had an AIDS diagnosis. All COC+ participants were current
             users and met criteria for cocaine use disorder. We used
             tract-based spatial statistics (TBSS) to assess the relation
             of HIV and cocaine to fractional anisotropy (FA) and mean
             diffusivity (MD). In whole-brain analyses, HIV+ participants
             had significantly reduced FA and increased MD compared to
             HIV- participants. The relation of HIV and FA was widespread
             throughout the brain, whereas the HIV-related MD effects
             were restricted to the corpus callosum and thalamus. There
             were no significant cocaine or HIV-by-cocaine effects. These
             DTI metrics correlated significantly with duration of HIV
             disease, nadir CD4+ cell count, and AIDS diagnosis, as well
             as some measures of neuropsychological functioning. These
             results suggest that HIV is related to white matter
             integrity throughout the brain, and that HIV-related effects
             are more pronounced with increasing duration of infection
             and greater immune compromise. We found no evidence for
             independent effects of cocaine dependence on white matter
             integrity, and cocaine dependence did not appear to
             exacerbate the effects of HIV.},
   Doi = {10.1007/s13365-017-0512-5},
   Key = {fds324858}
}

@article{fds325991,
   Author = {Li, R and Smith, DV and Clithero, JA and Venkatraman, V and Carter, RM and Huettel, SA},
   Title = {Reason's Enemy Is Not Emotion: Engagement of Cognitive
             Control Networks Explains Biases in Gain/Loss
             Framing.},
   Journal = {The Journal of Neuroscience : the Official Journal of the
             Society for Neuroscience},
   Volume = {37},
   Number = {13},
   Pages = {3588-3598},
   Year = {2017},
   Month = {March},
   url = {http://dx.doi.org/10.1523/jneurosci.3486-16.2017},
   Abstract = {In the classic gain/loss framing effect, describing a gamble
             as a potential gain or loss biases people to make
             risk-averse or risk-seeking decisions, respectively. The
             canonical explanation for this effect is that frames
             differentially modulate emotional processes, which in turn
             leads to irrational choice behavior. Here, we evaluate the
             source of framing biases by integrating functional magnetic
             resonance imaging data from 143 human participants
             performing a gain/loss framing task with meta-analytic data
             from >8000 neuroimaging studies. We found that activation
             during choices consistent with the framing effect were most
             correlated with activation associated with the resting or
             default brain, while activation during choices inconsistent
             with the framing effect was most correlated with the
             task-engaged brain. Our findings argue against the common
             interpretation of gain/loss framing as a competition between
             emotion and control. Instead, our study indicates that this
             effect results from differential cognitive engagement across
             decision frames.SIGNIFICANCE STATEMENT The biases frequently
             exhibited by human decision makers have often been
             attributed to the presence of emotion. Using a large fMRI
             sample and analysis of whole-brain networks defined with the
             meta-analytic tool Neurosynth, we find that neural activity
             during frame-biased decisions was more significantly
             associated with default behaviors (and the absence of
             executive control) than with emotion. These findings point
             to a role for neuroscience in shaping long-standing
             psychological theories in decision science.},
   Doi = {10.1523/jneurosci.3486-16.2017},
   Key = {fds325991}
}

@misc{fds330540,
   Author = {Jack, J and Appelbaum, LG and Beam, E and Moody, J and Huettel,
             SA},
   Title = {Mapping rhetorical topologies in cognitive
             neuroscience},
   Pages = {125-150},
   Booktitle = {Topologies as Techniques for a Post-Critical
             Rhetoric},
   Year = {2017},
   Month = {January},
   ISBN = {9783319512686},
   url = {http://dx.doi.org/10.1007/978-3-319-51268-6_7},
   Abstract = {© The Author(s) 2017. Many tools that neuroscientists use
             to trace the complex topography of the human brain draw on
             the neuroscience literature to yield “metanalyses” or
             “syntheses of data.” These approaches conflate
             rhetorical connections in the literature with physical
             connections in the brain. By contrast, the model presented
             in this chapter seeks not a topography of the brain but a
             topology of neuroscience. A social network analysis of
             titles and abstracts for cognitive neuroscience articles
             yields a topology of brain regions and functions. This map
             can help researchers identify underresearched areas (e.g.,
             the thalamus) or areas that are oversaturated (e.g., the
             amygdala). The map also helps researchers identify
             subdisciplines, such as “neuroeconomics,” that have not
             yet integrated with the broader field–“islands” where
             rhetorical work could yield benefits.},
   Doi = {10.1007/978-3-319-51268-6_7},
   Key = {fds330540}
}


%% Kay, Aaron C.   
@article{fds333552,
   Author = {Proudfoot, D and Kay, AC},
   Title = {How perceptions of one's organization can affect perceptions
             of the self: Membership in a stable organization can sustain
             individuals' sense of control},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {76},
   Pages = {104-115},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.jesp.2018.01.004},
   Abstract = {© 2018 Elsevier Inc. Building on contemporary perspectives
             regarding the role that group identification can play in
             sustaining control motives, we propose that being a member
             of a stable organization—one experienced as predictable
             and consistent rather than changing and in flux—can
             maintain individuals' sense of control. Four studies test
             this prediction. We observe that higher social
             identification as an organizational member (as compared to
             lower identification) is associated with an increased
             generalized sense of personal efficacy in life specifically
             when one's organization is experienced as relatively stable
             (Study 1 and Study 2). Further, the perceived stability of
             one's organization moderates the extent to which those who
             recently experienced a threat to personal control—and are
             thereby motivated to reestablish feelings of control—seek
             increased social identification as an organizational member
             (Study 3 and Study 4). Results suggest that membership in a
             stable organization can provide a psychological buffer
             against threats to personal control encountered in daily
             life outside work. Contributions to understanding the ways
             in which people maintain feelings of personal control in the
             social world are discussed.},
   Doi = {10.1016/j.jesp.2018.01.004},
   Key = {fds333552}
}

@article{fds332999,
   Author = {Landau, MJ and Khenfer, J and Keefer, LA and Swanson, TJ and Kay,
             AC},
   Title = {When and why does belief in a controlling God strengthen
             goal commitment?},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {75},
   Pages = {71-82},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.11.012},
   Abstract = {© 2017 Elsevier Inc. The perception that God controls one's
             life can bolster motivation to pursue personal goals, but it
             can also have no impact and even squelch motivation. To
             better understand how religious beliefs impact
             self-regulation, the current research built on Compensatory
             Control Theory's claim that perceiving the environment as
             predictable (vs. unpredictable) strengthens commitment to
             long-term goals. Perceiving God's intervention as following
             an understandable logic, which implies a predictable
             environment, increased self-reported and behavioral
             commitment to save money (Studies 1–3), excel academically
             (Study 4), and improve physical health (Study 5). In
             contrast, perceiving God as intervening in mysterious ways,
             which implies that worldly affairs are under control yet
             unpredictable, did not increase goal commitment. Exploratory
             mediational analyses focused on self-efficacy, response
             efficacy, and confidence in God's control. A meta-analysis
             (Study 6) yielded a reliable effect whereby belief in divine
             control supports goal pursuit specifically when it signals
             the predictability of one's environment.},
   Doi = {10.1016/j.jesp.2017.11.012},
   Key = {fds332999}
}

@article{fds333000,
   Author = {Tang, S and King, M and Kay, AC},
   Title = {Fate as a motivated (and de-motivating) belief: Evidence for
             a link from task importance to belief in fate to
             effort},
   Journal = {Organizational Behavior and Human Decision
             Processes},
   Volume = {144},
   Pages = {74-84},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.obhdp.2017.08.003},
   Doi = {10.1016/j.obhdp.2017.08.003},
   Key = {fds333000}
}

@article{fds329823,
   Author = {Khenfer, J and Laurin, K and Tafani, E and Roux, E and Kay,
             AC},
   Title = {Interventionist external agents make specific advice less
             demotivating},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {73},
   Pages = {189-196},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.07.003},
   Doi = {10.1016/j.jesp.2017.07.003},
   Key = {fds329823}
}

@article{fds333001,
   Author = {Fath, S and Proudfoot, D and Kay, AC},
   Title = {Effective to a fault: Organizational structure predicts
             attitudes toward minority organizations},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {73},
   Pages = {290-297},
   Year = {2017},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.jesp.2017.10.003},
   Doi = {10.1016/j.jesp.2017.10.003},
   Key = {fds333001}
}

@article{fds323320,
   Author = {Shepherd, S and Eibach, RP and Kay, AC},
   Title = {“One Nation Under God”: The System-Justifying Function
             of Symbolically Aligning God and Government},
   Journal = {Political Psychology},
   Volume = {38},
   Number = {5},
   Pages = {703-720},
   Year = {2017},
   Month = {October},
   url = {http://dx.doi.org/10.1111/pops.12353},
   Abstract = {© 2016 International Society of Political Psychology. Do
             references to God in political discourse increase confidence
             in the U.S. sociopolitical system? Using a system
             justification framework (Jost & Banaji, ), five studies
             provide evidence that, (1) increasingly governments
             symbolically associate the nation with God when public
             confidence in the social system may be threatened and (2)
             associating the nation with God serves a system-justifying
             function by increasing public confidence in the system. In
             an analysis of U.S. presidential speeches, presidents were
             more likely to symbolically associate the nation with God
             during threatening times (Study 1). Among religious
             individuals, referencing God in political rhetoric increased
             the perceived trustworthiness of politicians, compared to
             patriotic secular rhetoric (Study 2) or simply priming the
             concept of God (Study 3). These effects were also unique to
             politicians from one's own sociopolitical system (Study 4).
             Finally, believing God has a plan for the United States
             attenuates the deleterious effect that perceptions of
             national decline have on system confidence (Study 5).
             Implications for the system-justifying function of religion
             are discussed.},
   Doi = {10.1111/pops.12353},
   Key = {fds323320}
}

@article{fds328086,
   Author = {Ma, A and Landau, MJ and Narayanan, J and Kay, AC},
   Title = {Thought-control difficulty motivates structure
             seeking.},
   Journal = {Journal of Experimental Psychology. General},
   Volume = {146},
   Number = {8},
   Pages = {1067-1072},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1037/xge0000282},
   Abstract = {Struggling to control one's mind can change how the world
             appears. In prior studies testing the compensatory control
             theory, reduced control over the external environment
             motivated the search for perceptual patterns and other forms
             of structured knowledge, even in remote domains. Going
             further, the current studies test whether difficulty
             controlling thoughts similarly predicts structure seeking.
             As hypothesized, thought-control difficulty positively
             predicted perceptions of causal connections between remote
             events (Study 1a) and nonexistent objects in visual noise
             (Study 1b). This effect was mediated by aversive arousal
             (Study 2) and caused specifically by thought-control
             difficulty as distinct from general difficulty (Study 3).
             Study 4 replicated the effect with a sample of meditators
             learning to control their thoughts, showing that
             thought-control difficulty was a powerful predictor of
             structure seeking. These findings reveal a novel form of
             motivated perception. (PsycINFO Database
             Record},
   Doi = {10.1037/xge0000282},
   Key = {fds328086}
}

@article{fds326633,
   Author = {Ma, A and Kay, AC},
   Title = {Compensatory control and ambiguity intolerance},
   Journal = {Organizational Behavior and Human Decision
             Processes},
   Volume = {140},
   Pages = {46-61},
   Year = {2017},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.obhdp.2017.04.001},
   Doi = {10.1016/j.obhdp.2017.04.001},
   Key = {fds326633}
}

@article{fds327057,
   Author = {Leander, NP and Kay, AC and Chartrand, TL and Payne,
             BK},
   Title = {An Affect Misattribution Pathway to Perceptions of Intrinsic
             Reward},
   Journal = {Social Cognition},
   Volume = {35},
   Number = {2},
   Pages = {163-180},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1521/soco.2017.35.2.163},
   Abstract = {© 2017 Guilford Publications, Inc. Intrinsic rewards are
             typically thought to stem from an activity's inherent
             properties and not from separable rewards one receives from
             it. Yet, people may not consciously notice or remember all
             the subtle external rewards that correspond with an activity
             and may misattribute some directly to the activity itself.
             We propose that perceptions of intrinsic reward can often be
             byproducts of misattributed causal inference, and present
             some initial evidence that perceptions of intrinsic reward
             can in fact increase when words pertaining to an activity
             are subtly paired with pleasant context cues. Importantly,
             these effects follow classic boundary conditions of both
             misattribution and intrinsic motivation, insofar as they
             were extinguished when participants could make a proper
             source attribution and/or when the activity became
             associated with a blatant external reward. We further
             propose a distinction can be made between authentically
             "intrinsic" rewards and the illusion of intrinsic rewards
             caused by misattributed positive affect.},
   Doi = {10.1521/soco.2017.35.2.163},
   Key = {fds327057}
}

@article{fds326064,
   Author = {Laurin, K and Kay, AC},
   Title = {The Motivational Underpinnings of Belief in
             God},
   Journal = {Advances in Experimental Social Psychology},
   Volume = {56},
   Pages = {201-257},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1016/bs.aesp.2017.02.004},
   Abstract = {© 2017 Elsevier Inc. Beliefs in powerful Gods are prevalent
             across time and across societies. In this chapter, we
             explore the motivated underpinnings of this phenomenon.
             After describing two popular theories that help account for
             some of this prevalence—one focused on byproducts of
             normal human cognition and the other focused on the cultural
             benefit conferred by shared belief in powerful Gods—we
             propose that a third perspective may be needed to fully
             explain why so many people believe: that believing in God is
             one mechanism through which people fulfill their need to
             perceive the world as structured, orderly, and nonrandom. We
             then describe a model that outlines the causes and
             consequences of perceptions of structure, and leverage this
             model to organize the evidence connecting belief in God to
             people's need for structure. We then note the ways in which
             belief in a powerful God, though not the only form of belief
             that can satisfy the need for structure, may hold an
             advantage over most alternatives. Finally, we conclude by
             discussing the implications of this perspective for
             understanding the ongoing evolution of religious
             belief.},
   Doi = {10.1016/bs.aesp.2017.02.004},
   Key = {fds326064}
}


%% Keefe, Francis J.   
@article{fds336943,
   Author = {Van Denburg and AN and Shelby, RA and Caldwell, DS and O'Sullivan, ML and Keefe, FJ},
   Title = {Self-Efficacy for Pain Communication Moderates the Relation
             Between Ambivalence Over Emotional Expression and Pain
             Catastrophizing Among Patients With Osteoarthritis.},
   Journal = {The Journal of Pain : Official Journal of the American Pain
             Society},
   Volume = {19},
   Number = {9},
   Pages = {1006-1014},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1016/j.jpain.2018.04.001},
   Abstract = {Pain catastrophizing (ie, the tendency to focus on and
             magnify pain sensations and feel helpless in the face of
             pain) is one of the most important and consistent
             psychological predictors of the pain experience. The present
             study examined, in 60 patients with osteoarthritis pain who
             were married or partnered: 1) the degree to which
             ambivalence over emotional expression and negative network
             orientation were associated with pain catastrophizing, and
             2) whether self-efficacy for pain communication moderated
             these relations. Hierarchical multiple linear regression
             analyses revealed a significant main effect for the
             association between ambivalence over emotional expression
             and pain catastrophizing; as ambivalence over emotional
             expression increased, the degree of pain catastrophizing
             increased. In addition, the interaction between ambivalence
             over emotional expression and self-efficacy for pain
             communication was significant, such that as self-efficacy
             for pain communication increased, the association between
             ambivalence over emotional expression and pain
             catastrophizing became weaker. Negative network orientation
             was not significantly associated with pain catastrophizing.
             Findings suggest that higher levels of self-efficacy for
             pain communication may help weaken the effects of
             ambivalence over emotional expression on pain
             catastrophizing. In light of these results, patients may
             benefit from interventions that target pain communication
             processes and emotion regulation.This article examines
             interpersonal processes involved in pain catastrophizing.
             This study has the potential to lead to better understanding
             of maladaptive pain coping strategies and possibly better
             prevention and treatment strategies.},
   Doi = {10.1016/j.jpain.2018.04.001},
   Key = {fds336943}
}

@article{fds336940,
   Author = {Cook, CE and George, SZ and Keefe, F},
   Title = {Different interventions, same outcomes? Here are four good
             reasons.},
   Journal = {British Journal of Sports Medicine},
   Volume = {52},
   Number = {15},
   Pages = {951-952},
   Year = {2018},
   Month = {August},
   url = {http://dx.doi.org/10.1136/bjsports-2017-098978},
   Doi = {10.1136/bjsports-2017-098978},
   Key = {fds336940}
}

@article{fds337107,
   Author = {Parmelee, PA and Scicolone, MA and Cox, BS and DeCaro, JA and Keefe, FJ and Smith, DM},
   Title = {Global Versus Momentary Osteoarthritis Pain and Emotional
             Distress: Emotional Intelligence as Moderator.},
   Journal = {Annals of Behavioral Medicine},
   Volume = {52},
   Number = {8},
   Pages = {713-723},
   Year = {2018},
   Month = {July},
   url = {http://dx.doi.org/10.1093/abm/kax044},
   Abstract = {Pain and emotional well-being are complexly associated both
             globally and in the moment. Emotional regulation strategies
             may contribute to that complexity by shaping the
             pain-well-being association.Using emotional intelligence
             (EI) as an integrative conceptual framework, this study
             probed the role of emotional regulation in the associations
             of osteoarthritis pain with emotional well-being in varying
             time frames. Perceived attention to, clarity, and regulation
             of emotions were examined as predictors of well-being, and
             as moderators of the well-being-pain association, at global
             and momentary (within-day) levels.In a microlongitudinal
             study, 218 older adults with physician-diagnosed knee
             osteoarthritis self-reported global pain, depressive
             symptoms, and EI (mood attention, clarity, and repair).
             Momentary pain and positive and negative affect were then
             assessed four times daily for 7 days. EI subscales were
             examined as moderators of the pain-well-being association at
             global and momentary levels, controlling demographics and
             general health.Global and momentary pain were positively
             associated with mood clarity and negatively with attention,
             but not with repair. Clarity and repair negatively predicted
             depression, and buffered effects of pain on depression.
             Momentary negative affect was negatively predicted by mood
             clarity and repair; again, clarity and mood repair buffered
             effects of momentary pain on negative affect. Only mood
             repair predicted positive affect, with no interactions
             emerging.Attention to mood states exacerbates the experience
             of pain in both short and long terms. In contrast, both mood
             clarity and ability to repair moods appear important to both
             momentary and longer-term emotional well-being.},
   Doi = {10.1093/abm/kax044},
   Key = {fds337107}
}

@article{fds337361,
   Author = {Burns, JW and Gerhart, J and Post, KM and Smith, DA and Porter, LS and Buvanendran, A and Fras, AM and Keefe, FJ},
   Title = {Spouse Criticism/Hostility Toward Partners With Chronic
             Pain: The Role of Spouse Attributions for Patient Control
             Over Pain Behaviors.},
   Journal = {The Journal of Pain : Official Journal of the American Pain
             Society},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.jpain.2018.05.007},
   Abstract = {Spouse attributions regarding displays of pain behaviors by
             their partners with chronic pain may account for subsequent
             increases in spouse critical/hostile responses toward their
             partners. People with chronic low back pain (n = 105)
             and their pain-free spouses (n = 105) completed
             electronic diary measures 5 times per day for 14 consecutive
             days. Key items assessed spouse observations of patient pain
             behavior, attributions regarding these behaviors, and spouse
             critical/hostile responses toward patients. Results were 1)
             spouse observations of patient pain behavior at time 1
             predicted high levels of spouse critical/hostile responses
             toward the patient at time 2. 2) "Internal" attributions
             (eg, the patient was attempting to influence spouse's
             feelings) at time 1 predicted high levels of spouse
             critical/hostile responses toward the patient at time 2. 3)
             Internal attributions mediated links between spouse-observed
             pain behaviors at time 1 and levels of spouse
             critical/hostile responses at time 2. Spouse observations of
             patient pain behavior was also related to an "external"
             attribution (ie, patient pain behavior was due to pain
             condition), but this attribution was not a significant
             mediator. A vital factor linking spouse scrutiny to spouse
             critical/hostile responses may be the spouse's ascribed
             reasons for the patient's grimacing, bracing, complaining,
             and so forth.Results indicate that spouse internal and
             negative attributions for pain behaviors of their partners
             with chronic pain may influence subsequent spouse
             critical/hostile reactions to them. Findings suggest that
             replacing spouse internal and negative attributions with
             external, compassionate, and accepting explanations may be
             useful therapeutic targets for couples coping with chronic
             pain.},
   Doi = {10.1016/j.jpain.2018.05.007},
   Key = {fds337361}
}

@article{fds331439,
   Author = {Porter, LS and Keefe, FJ},
   Title = {Couple-based communication interventions for cancer: moving
             beyond a 'one size fits all' approach.},
   Journal = {Acta Oncologica (Stockholm, Sweden)},
   Volume = {57},
   Number = {5},
   Pages = {693-695},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1080/0284186x.2017.1400687},
   Doi = {10.1080/0284186x.2017.1400687},
   Key = {fds331439}
}

@article{fds337030,
   Author = {Keefe, FJ and Main, CJ and George, SZ},
   Title = {Advancing Psychologically Informed Practice for Patients
             With Persistent Musculoskeletal Pain: Promise, Pitfalls, and
             Solutions.},
   Journal = {Physical Therapy},
   Volume = {98},
   Number = {5},
   Pages = {398-407},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1093/ptj/pzy024},
   Abstract = {There has been growing interest in psychologically oriented
             pain management over the past 3 to 4 decades, including a
             2011 description of psychologically informed practice (PIP)
             for low back pain. PIP requires a broader focus than
             traditional biomechanical and pathology-based approaches
             that have been traditionally used to manage musculoskeletal
             pain. A major focus of PIP is addressing the behavioral
             aspects of pain (ie, peoples' responses to pain) by
             identifying individual expectations, beliefs, and feelings
             as prognostic factors for clinical and occupational outcomes
             indicating progression to chronicity. Since 2011, the
             interest in PIP seems to be growing, as evidenced by its use
             in large trials, inclusion in scientific conferences,
             increasing evidence base, and expansion to other
             musculoskeletal pain conditions. Primary care physicians and
             physical therapists have delivered PIP as part of a
             stratified care approach involving screening and targeting
             of treatment for people at high risk for continued
             pain-associated disability. Furthermore, PIP is consistent
             with recent national priorities emphasizing
             nonpharmacological pain management options. In this
             perspective, PIP techniques that range in complexity are
             described, considerations for implementation in clinical
             practice are offered, and future directions that will
             advance the understanding of PIP are outlined.},
   Doi = {10.1093/ptj/pzy024},
   Key = {fds337030}
}

@article{fds336941,
   Author = {Jensen, MP and Thorn, BE and Carmody, J and Keefe, FJ and Burns,
             JW},
   Title = {The Role of Cognitive Content and Cognitive Processes in
             Chronic Pain: An Important Distinction?},
   Journal = {The Clinical Journal of Pain},
   Volume = {34},
   Number = {5},
   Pages = {391-401},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1097/ajp.0000000000000559},
   Abstract = {Pain-related cognitive content (what people think about
             pain) and cognitive processes (how people think about pain;
             what they do with their pain-related thoughts) and their
             interaction are hypothesized to play distinct roles in
             patient function. However, questions have been raised
             regarding whether it is possible or practical to assess
             cognitive content and cognitive process as distinct domains.
             The aim of this study was to determine the extent to which
             measures that seem to assess mostly pain-related cognitive
             content, cognitive processes, and content and process, are
             relatively independent from each other and contribute unique
             variance to the prediction of patient function.Individuals
             with chronic low back pain (N=165) participating in an
             ongoing RCT were administered measures of cognitions, pain,
             and function (depressive symptoms and pain interference)
             pretreatment.Analyses provided support for the hypothesis
             that cognitive content and cognitive process, while related,
             can be assessed as distinct components. However, the measure
             assessing a cognitive process-mindfulness-evidenced
             relatively weak associations with function, especially
             compared with the stronger and more consistent findings for
             the measures of content (catastrophizing and
             self-efficacy).The results provide preliminary evidence for
             the possibility that mindfulness could have both benefits
             and costs. Research to evaluate this possibility is
             warranted.},
   Doi = {10.1097/ajp.0000000000000559},
   Key = {fds336941}
}

@article{fds336942,
   Author = {Whitney, CA and Dorfman, CS and Shelby, RA and Keefe, FJ and Gandhi, V and Somers, TJ},
   Title = {Reminders of cancer risk and pain catastrophizing:
             relationships with cancer worry and perceived risk in women
             with a first-degree relative with breast
             cancer.},
   Journal = {Familial Cancer},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s10689-018-0082-6},
   Abstract = {First-degree relatives of women with breast cancer may
             experience increased worry or perceived risk when faced with
             reminders of their own cancer risk. Worry and risk reminders
             may include physical symptoms (e.g., persistent breast pain)
             and caregiving experiences. Women who engage in pain
             catastrophizing may be particularly likely to experience
             increased distress when risk reminders are present. We
             examined the degree to which persistent breast pain and
             experience as a cancer caregiver were related to cancer
             worry and perceived risk in first-degree relatives of women
             with breast cancer (N = 85) and how catastrophic
             thoughts about breast pain could impact these relationships.
             There was a significant interaction between persistent
             breast pain and pain catastrophizing in predicting cancer
             worry (p = .03); among women who engaged in pain
             catastrophizing, cancer worry remained high even in the
             absence of breast pain. Pain catastrophizing also moderated
             the relationships between caregiving involvement and cancer
             worry (p = .003) and perceived risk (p = .03). As
             the degree of caregiving responsibility increased, cancer
             worry and perceived risk increased for women who engaged in
             pain catastrophizing; levels of cancer worry and perceived
             risk remained low and stable for women who did not engage in
             pain catastrophizing regardless of caregiving experience.
             The results suggest that first-degree relatives of breast
             cancer survivors who engage in pain catastrophizing may
             experience greater cancer worry and perceived risk and may
             benefit from interventions aimed at reducing catastrophic
             thoughts about pain.},
   Doi = {10.1007/s10689-018-0082-6},
   Key = {fds336942}
}

@article{fds330575,
   Author = {O'Sullivan, ML and Shelby, RA and Dorfman, CS and Kelleher, SA and Fisher, HM and Rowe Nichols and KA and Keefe, FJ and Sung, AD and Somers,
             TJ},
   Title = {The effect of pre-transplant pain and chronic disease
             self-efficacy on quality of life domains in the year
             following hematopoietic stem cell transplantation.},
   Journal = {Supportive Care in Cancer : Official Journal of the
             Multinational Association of Supportive Care in
             Cancer},
   Volume = {26},
   Number = {4},
   Pages = {1243-1252},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1007/s00520-017-3947-6},
   Abstract = {Pain is common for hematopoietic stem cell transplant (HSCT)
             patients and may be experienced pre-transplant, acutely
             post-transplant, and for months or years following
             transplant. HSCT patients with persistent pain may be at
             risk for poor quality of life following transplant; however,
             the impact of pre-transplant pain on quality of life
             post-transplant is not well understood. Self-efficacy for
             chronic disease management is associated with quality of
             life among cancer patients and may impact quality of life
             for HSCT patients. The primary aim was to examine the effect
             of pre-transplant pain and self-efficacy on quality of life
             domains in the year following transplant.One hundred
             sixty-six HSCT patients completed questionnaires providing
             information on pain, self-efficacy, and quality of life
             prior to transplant, at discharge, and 3-, 6-, and 12-months
             post-transplant as part of a longitudinal, observational
             study. Linear mixed modeling examined the trajectories of
             these variables and the effect of pre-transplant pain and
             self-efficacy on post-transplant quality of life.Pain and
             social and emotional quality of life remained stable in the
             year following transplant while self-efficacy and physical
             and functional quality of life improved. Pre-transplant pain
             was significantly related to lower physical well-being
             post-transplant. Lower pre-transplant self-efficacy was
             related to lower quality of life across all domains
             post-transplant.Above and beyond the effect of
             pre-transplant pain, self-efficacy for managing chronic
             disease is important in understanding quality of life
             following transplant. Identifying patients with pain and/or
             low self-efficacy pre-transplant may allow for early
             intervention with self-management strategies.},
   Doi = {10.1007/s00520-017-3947-6},
   Key = {fds330575}
}

@article{fds330868,
   Author = {Rini, C and Vu, MB and Lerner, H and Bloom, C and Carda-Auten, J and Wood,
             WA and Basch, EM and Voorhees, PM and Reeder-Hayes, KE and Keefe,
             FJ},
   Title = {A qualitative study of patient and provider perspectives on
             using web-based pain coping skills training to treat
             persistent cancer pain.},
   Journal = {Palliative & Supportive Care},
   Volume = {16},
   Number = {2},
   Pages = {155-169},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1017/s1478951517000086},
   Abstract = {Persistent pain is common and inadequately treated in cancer
             patients. Behavioral pain interventions are a recommended
             part of multimodal pain treatments, but they are underused
             in clinical care due to barriers such as a lack of the
             resources needed to deliver them in person and difficulties
             coordinating their use with clinical care. Pain coping
             skills training (PCST) is an evidence-based behavioral pain
             intervention traditionally delivered in person. Delivering
             this training via the web would increase access to it by
             addressing barriers that currently limit its use. We
             conducted a patient pilot study of an 8-week web-based PCST
             program to determine the acceptability of this approach to
             patients and the program features needed to meet their
             needs. Focus groups with healthcare providers identified
             strategies for coordinating the use of web-based PCST in
             clinical care.Participants included 7 adults with bone pain
             due to multiple myeloma or metastasized breast or prostate
             cancer and 12 healthcare providers (4 physicians and 8
             advanced practice providers) who treat cancer-related bone
             pain. Patients completed web-based PCST at home and then
             took part in an in-depth qualitative interview. Providers
             attended focus groups led by a trained moderator.
             Qualitative analyses identified themes in the patient and
             provider data.Patients reported strongly favorable responses
             to web-based PCST and described emotional and physical
             benefits. They offered suggestions for adapting the approach
             to better fit their needs and to overcome barriers to
             completion. Focus groups indicated a need to familiarize
             healthcare providers with PCST and to address concerns about
             overburdening patients. Providers would recommend the
             program to patients they felt could benefit. They suggested
             applying a broad definition of cancer pain and having
             various types of providers help coordinate program its use
             with clinical care.Web-based PCST was acceptable to patients
             and providers. Our findings suggest that patients could
             benefit from this approach, especially if patient and
             provider barriers are addressed.},
   Doi = {10.1017/s1478951517000086},
   Key = {fds330868}
}

@article{fds333734,
   Author = {DeBar, L and Benes, L and Bonifay, A and Deyo, RA and Elder, CR and Keefe,
             FJ and Leo, MC and McMullen, C and Mayhew, M and Owen-Smith, A and Smith,
             DH and Trinacty, CM and Vollmer, WM},
   Title = {Interdisciplinary team-based care for patients with chronic
             pain on long-term opioid treatment in primary care (PPACT) -
             Protocol for a pragmatic cluster randomized
             trial.},
   Journal = {Contemporary Clinical Trials},
   Volume = {67},
   Pages = {91-99},
   Year = {2018},
   Month = {April},
   url = {http://dx.doi.org/10.1016/j.cct.2018.02.015},
   Abstract = {Chronic pain is one of the most common, disabling, and
             expensive public health problems in the United States.
             Interdisciplinary pain management treatments that employ
             behavioral approaches have been successful in helping
             patients with chronic pain reduce symptoms and regain
             functioning. However, most patients lack access to such
             treatments. We are conducting a pragmatic clinical trial to
             test the hypothesis that patients who receive an
             interdisciplinary biopsychosocial intervention, the Pain
             Program for Active Coping and Training (PPACT), at their
             primary care clinic will have a greater reduction in pain
             impact in the year following than patients receiving usual
             care.This is an effectiveness-implementation hybrid
             pragmatic clinical trial in which we randomize clusters of
             primary care providers and their patients with chronic pain
             who are on long-term opioid therapy to 1) receive an
             interdisciplinary behavioral intervention in conjunction
             with their current health care or 2) continue with current
             health care services. Our primary outcome is pain impact (a
             composite of pain intensity and pain-related interference)
             measured using the PEG, a validated three-item assessment.
             Secondary outcomes include pain-related disability, patient
             satisfaction, opioids dispensed and health care utilization.
             An economic evaluation assesses the resources and costs
             necessary to deliver the intervention and its
             cost-effectiveness compared with usual care. A formative
             evaluation employs mixed methods to understand the context
             for implementation in the participating health care
             systems.This trial will inform the feasibility of
             implementing interdisciplinary behavioral approaches to pain
             management in the primary care setting, potentially
             providing a more effective, safer, and more satisfactory
             alternative to opioid-based chronic pain treatment. Clinical
             Trials Registration Number: NCT02113592.},
   Doi = {10.1016/j.cct.2018.02.015},
   Key = {fds333734}
}

@article{fds336944,
   Author = {Somers, TJ and Kelleher, SA and Dorfman, CS and Shelby, RA and Fisher,
             HM and Rowe Nichols and K and Sullivan, KM and Chao, NJ and Samsa, GP and Abernethy, AP and Keefe, FJ},
   Title = {An mHealth Pain Coping Skills Training Intervention for
             Hematopoietic Stem Cell Transplantation Patients:
             Development and Pilot Randomized Controlled
             Trial.},
   Journal = {Jmir Mhealth and Uhealth},
   Volume = {6},
   Number = {3},
   Pages = {e66},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.2196/mhealth.8565},
   Abstract = {Pain is a challenge for patients following hematopoietic
             stem cell transplantation (HCT).This study aimed to develop
             and test the feasibility, acceptability, and initial
             efficacy of a Web-based mobile pain coping skills training
             (mPCST) protocol designed to address the needs of HCT
             patients.Participants had undergone HCT and reported pain
             following transplant (N=68). To guide intervention
             development, qualitative data were collected from focus
             group participants (n=25) and participants who completed
             user testing (n=7). After their input was integrated into
             the mPCST intervention, a pilot randomized controlled trial
             (RCT, n=36) was conducted to examine the feasibility,
             acceptability, and initial efficacy of the intervention.
             Measures of acceptability, pain severity, pain disability,
             pain self-efficacy, fatigue, and physical disability
             (self-report and 2-min walk test [2MWT]) were
             collected.Participants in the focus groups and user testing
             provided qualitative data that were used to iteratively
             refine the mPCST protocol. Focus group qualitative data
             included participants' experiences with pain following
             transplant, perspectives on ways to cope with pain, and
             suggestions for pain management for other HCT patients. User
             testing participants provided feedback on the HCT protocol
             and information on the use of videoconferencing. The final
             version of the mPCST intervention was designed to bridge the
             intensive outpatient (1 in-person session) and home settings
             (5 videoconferencing sessions). A key component of the
             intervention was a website that provided personalized
             messages based on daily assessments of pain and activity.
             The website also provided intervention materials (ie,
             electronic handouts, short videos, and audio files). The
             intervention content included pain coping advice from other
             transplant patients and instructions on how to apply pain
             coping skills while engaging in meaningful and leisure
             activities. In the RCT phase of this research, HCT patients
             (n=36) were randomized to receive the mPCST intervention or
             to proceed with the treatment as usual. Results revealed
             that the mPCST participants completed an average of 5 out of
             6 sessions. The participants reported that the intervention
             was highly acceptable (mean 3/4), and they found the
             sessions to be helpful (mean 8/10) and easy to understand
             (mean 7/7). The mPCST participants demonstrated significant
             improvements in pre- to post-treatment pain, self-efficacy
             (P=.03, d=0.61), and on the 2MWT (P=.03, d=0.66), whereas
             the patients in the treatment-as-usual group did not report
             any such improvements. Significant changes in pain
             disability and fatigue were found in both groups (multiple
             P<.02); the magnitudes of the effect sizes were larger for
             the mPCST group than for the control group (pain disability:
             d=0.79 vs 0.69; fatigue: d=0.94 vs 0.81). There were no
             significant changes in pain severity in either group.Using
             focus groups and user testing, we developed an mPCST
             protocol that was feasible, acceptable, and beneficial for
             HCT patients with pain.ClinicalTrials.gov NCT01984671;
             https://clinicaltrials.gov/ct2/show/NCT01984671 (Archived by
             WebCite at http://www.webcitation.org/6xbpx3clZ).},
   Doi = {10.2196/mhealth.8565},
   Key = {fds336944}
}

@article{fds337362,
   Author = {Janke, EA and Cheatle, M and Keefe, FJ and Dhingra, L and Society of
             Behavioral Medicine Health Policy Committee},
   Title = {Society of Behavioral Medicine (SBM) position statement:
             improving access to psychosocial care for individuals with
             persistent pain: supporting the National Pain Strategy's
             call for interdisciplinary pain care.},
   Journal = {Translational Behavioral Medicine},
   Volume = {8},
   Number = {2},
   Pages = {305-308},
   Year = {2018},
   Month = {March},
   url = {http://dx.doi.org/10.1093/tbm/ibx043},
   Abstract = {Policy makers have articulated a need for clear,
             evidence-based guidance to help inform pain policy.
             Persistent pain is common, expensive, and debilitating, and
             requires comprehensive assessment and treatment planning.
             Recently released opioid prescribing guidelines by the CDC
             (2016) emphasize the importance of using nonopioid therapies
             before considering opioid treatment for those without a
             malignant illness. The National Pain Strategy (2016)
             underscores the importance of comprehensive,
             interdisciplinary pain care. Unfortunately, despite
             persuasive evidence supporting the efficacy of psychosocial
             approaches, these interventions are inaccessible to the
             majority of Americans. Psychosocial approaches to pain
             management should be available for all individuals with
             persistent pain and in all health care settings and contexts
             as part of the comprehensive, interdisciplinary approach to
             pain care as outlined in the National Pain Strategy. To
             achieve this, we must prioritize reimbursement of
             evidence-based psychosocial approaches for pain assessment
             and management and improve provider training and
             competencies to implement these approaches.},
   Doi = {10.1093/tbm/ibx043},
   Key = {fds337362}
}

@article{fds333735,
   Author = {Gerhart, JI and Burns, JW and Bruehl, S and Smith, DA and Post, KM and Porter, LS and Schuster, E and Buvanendran, A and Fras, AM and Keefe,
             FJ},
   Title = {Variability in negative emotions among individuals with
             chronic low back pain: relationships with pain and
             function.},
   Journal = {Pain},
   Volume = {159},
   Number = {2},
   Pages = {342-350},
   Year = {2018},
   Month = {February},
   url = {http://dx.doi.org/10.1097/j.pain.0000000000001102},
   Abstract = {Chronic pain is associated with elevated negative emotions,
             and resources needed to adaptively regulate these emotions
             can be depleted during prolonged pain. Studies of links
             between pain, function, and negative emotions in people with
             chronic pain, however, have focused almost exclusively on
             relationships among mean levels of these factors. Indexes
             that may reflect aspects of emotion regulation have
             typically not been analyzed. We propose that 1 index of
             emotion regulation is variability in emotion over time as
             opposed to average emotion over time. The sample was 105
             people with chronic low back pain and 105 of their pain-free
             spouses. They completed electronic diary measures 5x/d for
             14 consecutive days, producing 70 observations per person
             from which we derived estimates of within-subject variance
             in negative emotions. Location-scale models were used to
             simultaneously model predictors of both mean level and
             variance in patient negative emotions over time. Patients
             reported significantly more variability in negative emotions
             compared to their spouses. Patients who reported higher
             average levels of pain, pain interference, and downtime
             reported significantly higher levels of variability in
             negative emotions. Spouse-observed pain and pain behaviors
             were also associated with greater variability in patients'
             negative emotions. Test of the inverse associations between
             negative emotion level and variability in pain and function
             were significant but weaker in magnitude. These findings
             support the notion that chronic pain may erode negative
             emotion regulation resources, to the potential detriment of
             intra- and inter-personal function.},
   Doi = {10.1097/j.pain.0000000000001102},
   Key = {fds333735}
}

@article{fds333736,
   Author = {Burns, JW and Post, KM and Smith, DA and Porter, LS and Buvanendran, A and Fras, AM and Keefe, FJ},
   Title = {Spouse criticism and hostility during marital interaction:
             effects on pain intensity and behaviors among individuals
             with chronic low back pain.},
   Journal = {Pain},
   Volume = {159},
   Number = {1},
   Pages = {25-32},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1097/j.pain.0000000000001037},
   Abstract = {Individuals with chronic pain may experience negative
             responses from spouse, family, and friends. Responses such
             as overt criticism and hostility may be associated with
             worsening pain and function for chronic pain sufferers. We
             used a laboratory procedure to evaluate whether variability
             in spouse criticism/hostility exhibited toward chronic low
             back pain (CLBP) patients during a conflictual discussion
             predicted variability in patient pain and function during a
             subsequent pain-induction task. Chronic low back pain
             patients (n = 71) and their spouses (n = 71) participated in
             a 10-minute discussion followed by the patient undergoing a
             10-minute structured pain behavior task (SPBT). Spouse
             criticism/hostility perceived by patients and patient Beck
             Depression Inventory-II (BDI) scores correlated
             significantly and positively with pain intensity during the
             SPBT, whereas perceived spouse hostility, patient BDI
             scores, and spouse trait hostility correlated significantly
             and positively with observed pain behaviors during the SPBT.
             Spouse criticism/hostility coded by raters from video
             recordings interacted significantly with patient BDI scores,
             such that observed spouse criticism/hostility was related
             significantly and positively with pain behaviors only for
             patients with high BDI scores. Patient sex interacted
             significantly with observed spouse criticism/hostility, such
             that observed spouse criticism/hostility was related
             significantly and positively with pain behaviors only for
             female patients. Results support the hypothesis that spouse
             criticism and hostility-actually expressed or perceived-may
             worsen CLBP patient symptoms. Further, women patients and
             patients high in depressive symptoms appeared most
             vulnerable to spouse criticism/hostility. Thus, negative
             marital communication patterns may be appropriate targets
             for intervention, especially among these 2 at risk
             groups.},
   Doi = {10.1097/j.pain.0000000000001037},
   Key = {fds333736}
}

@article{fds332333,
   Author = {Lumley, MA and Anderson, T and Ankawi, B and Goldman, G and Perri, LM and Bianco, JA and Keefe, FJ},
   Title = {The working alliance and Clinician-assisted Emotional
             Disclosure for rheumatoid arthritis.},
   Journal = {Journal of Psychosomatic Research},
   Volume = {104},
   Pages = {9-15},
   Year = {2018},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jpsychores.2017.11.004},
   Abstract = {The working alliance predicts improvement following general
             psychotherapy, but how it operates in brief interventions
             conducted with medically ill patients is unknown. Also, the
             role of the working alliance may differ in emotion-focused
             versus educational interventions.We report secondary
             analyses of a randomized clinical trial (Keefe et al.) [35],
             in which patients with rheumatoid arthritis (RA) received
             four nurse-provided sessions of either a) Clinician-assisted
             Emotional Disclosure (CAED), which emphasized the
             disclosure, expression, and processing of emotions related
             to stressful events; or b) Arthritis Education (AE), which
             provided basic education about RA. The Working Alliance
             Inventory was completed by both patient and nurse after each
             session. Patients were evaluated on multiple health measures
             at baseline and 1, 3, and 12months post-treatment.Analyses
             compared the alliance between interventions and related the
             alliance to outcomes within interventions. Patients in CAED
             reported a lower alliance than patients in AE.
             Interestingly, in CAED, lower alliance ratings predicted
             better outcomes (improved functioning, lower pain behaviors,
             lower inflammation, lower daily stress), whereas in AE, the
             working alliance was largely not predictive of
             outcomes.Having nurses encourage emotional disclosure among
             patients with RA reduced the patients' working alliance, but
             a lower alliance nonetheless predicted better patient
             outcomes, perhaps reflecting successful engagement in an
             intervention that is emotionally and relationally
             challenging. The level and predictive validity of the
             working alliance likely depends on patient, provider, and
             intervention factors, and further study of the working
             alliance in psychosocial interventions in the medical
             context is needed.},
   Doi = {10.1016/j.jpsychores.2017.11.004},
   Key = {fds332333}
}

@article{fds336945,
   Author = {Lawford, BJ and Hinman, RS and Kasza, J and Nelligan, R and Keefe, F and Rini, C and Bennell, KL},
   Title = {Moderators of Effects of Internet-Delivered Exercise and
             Pain Coping Skills Training for People With Knee
             Osteoarthritis: Exploratory Analysis of the IMPACT
             Randomized Controlled Trial},
   Journal = {Journal of Medical Internet Research},
   Volume = {20},
   Number = {5},
   Pages = {e10021-e10021},
   Year = {2018},
   url = {http://dx.doi.org/10.2196/10021},
   Doi = {10.2196/10021},
   Key = {fds336945}
}

@article{fds330576,
   Author = {Lefebvre, JC and Jensen, MP and Waters, SJ and Molton, IR and Keefe, FJ and Caldwell, DS},
   Title = {The development and assessment of the Worry About Pain
             Questionnaire.},
   Journal = {European Journal of Pain (London, England)},
   Volume = {21},
   Number = {7},
   Pages = {1154-1164},
   Year = {2017},
   Month = {August},
   url = {http://dx.doi.org/10.1002/ejp.1015},
   Abstract = {Worry can be conceptualized as a cognitive-affective
             automatic process initiated in order to address uncertainty
             and potential personal inadequacies that could result in
             negative outcomes. The purpose of the current study was to
             develop a measure of pain-specific worry - the Worry About
             Pain Questionnaire (WAPQ).In study 1, responses of 335
             pain-free participants were used to complete an item
             analysis and exploratory factors analysis to develop and
             assess the internal structure of the WAPQ. Study 2 included
             224 pain-free participants who completed the WAPQ in order
             to confirm its factor structure, and to examine its relation
             to the experience of acute experimental pain. In study 3,
             137 individuals with persistent pain were asked to complete
             the WAPQ as well as measures of pain and depressive
             symptoms.The resulting 15-item measure assesses
             uncertainties and potential negative outcomes related to the
             experience of pain. The results of the exploratory and
             confirmatory factor analyses showed a two-factor structure.
             Across all studies, the WAPQ was found to be related to
             measures of pain in clinical and non-clinical samples, acute
             experimental pain stimuli, as well as pain anxiety, pain
             catastrophizing, fear of pain, rumination and depressive
             symptomatology.The results suggest that the WAPQ is a
             reliable and valid measure for the assessment of worry about
             pain that can be used to understand how pain-specific
             worries are related to the experience and impact of pain
             across different populations.Worry has been assessed in pain
             populations using measures that assess worry in general. The
             current study shows a relationship between pain-specific
             worry and the experience of pain. Further, worry about pain
             is related to but not synonymous with pain
             catastrophizing.},
   Doi = {10.1002/ejp.1015},
   Key = {fds330576}
}

@article{fds319635,
   Author = {Porter, LS and Keefe, FJ and Baucom, DH and Olsen, M and Zafar, SY and Uronis, H},
   Title = {A randomized pilot trial of a videoconference couples
             communication intervention for advanced GI
             cancer.},
   Journal = {Psycho Oncology},
   Volume = {26},
   Number = {7},
   Pages = {1027-1035},
   Year = {2017},
   Month = {July},
   url = {http://dx.doi.org/10.1002/pon.4121},
   Abstract = {This study aims to test the feasibility and preliminary
             efficacy of a couple-based communication intervention for
             advanced GI cancer delivered via videoconference.Thirty-two
             couples were randomly assigned to either couples
             communication skills training (CCST) or an education
             comparison intervention, both delivered via videoconference.
             Participation was limited to couples who reported
             communication difficulties at screening. Patients and
             partners completed measures of relationship functioning and
             individual functioning at baseline and post-intervention.Eighty-eight
             percent of randomized dyads completed all six sessions and
             reported high levels of satisfaction with the intervention.
             Between-group effect sizes suggested that the CCST
             intervention led to improvements in relationship
             satisfaction for patients and partners and to improvements
             in intimacy and communication for patients.A couples-based
             communication intervention delivered via videoconference is
             feasible and acceptable in the context of advanced cancer.
             Preliminary findings suggest that the intervention shows
             promise in contributing to enhanced relationship
             functioning. Copyright © 2016 John Wiley & Sons,
             Ltd.},
   Doi = {10.1002/pon.4121},
   Key = {fds319635}
}

@article{fds330867,
   Author = {Parmelee, PA and Cox, BS and DeCaro, JA and Keefe, FJ and Smith,
             DM},
   Title = {Racial/ethnic differences in sleep quality among older
             adults with osteoarthritis.},
   Journal = {Sleep Health},
   Volume = {3},
   Number = {3},
   Pages = {163-169},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.sleh.2017.03.010},
   Abstract = {To examine racial/ethnic differences in sleep quality and
             the pain-sleep association among older adults with
             osteoarthritis of the knee.Baseline interview followed by a
             7-day microlongitudinal study using accelerometry and
             self-reports.Participants were community residents in
             western Alabama and Long Island, NY.Ninety-six African
             Americans (AAs) and 128 non-Hispanic whites (NHWs) with
             physician-diagnosed knee osteoarthritis, recruited from a
             variety of clinical and community settings.Self-reports
             yielded demographics, body mass index, physical health
             problems, and depressive symptoms. Sleep quality was
             measured for 3 to 7 nights using wrist-worn accelerometers;
             pain was self-reported daily over the same period.With
             demographics and health controlled, AAs displayed poorer
             sleep efficiency, greater time awake after sleep onset and
             sleep fragmentation, and marginally more awakenings during
             the night, but no differences in total sleep time. AAs also
             showed greater night-to-night variability in number of
             awakenings and sleep fragmentation, and marginally greater
             variability in total sleep time and sleep efficiency. Sleep
             quality was not associated with pain either the day before
             sleep or the day after. Average daily pain interacted with
             race, whereas AAs displayed no effect of pain on sleep
             efficiency, NHWs exhibited better sleep efficiency at higher
             levels of average pain.These data corroborate previous
             studies documenting poorer sleep among AAs vs NHWs. The
             findings of greater night-to-night variability in sleep
             among AAs, as well as a negative association of pain with
             sleep quality among NHWs, are unique. Further study is
             needed to elucidate these findings.},
   Doi = {10.1016/j.sleh.2017.03.010},
   Key = {fds330867}
}

@article{fds330577,
   Author = {Gerhart, JI and Burns, JW and Post, KM and Smith, DA and Porter, LS and Burgess, HJ and Schuster, E and Buvanendran, A and Fras, AM and Keefe,
             FJ},
   Title = {Relationships Between Sleep Quality and Pain-Related Factors
             for People with Chronic Low Back Pain: Tests of Reciprocal
             and Time of Day Effects.},
   Journal = {Annals of Behavioral Medicine},
   Volume = {51},
   Number = {3},
   Pages = {365-375},
   Year = {2017},
   Month = {June},
   url = {http://dx.doi.org/10.1007/s12160-016-9860-2},
   Abstract = {Poor sleep quality among people with chronic low back pain
             appears to be related to worse pain, affect, poor physical
             function, and pain catastrophizing. The causal direction
             between poor sleep and pain remains an open question,
             however, as does whether sleep quality exerts effects on low
             back pain differently across the course of the day.This
             daily diary study examined lagged temporal associations
             between prior night sleep quality and subsequent day pain,
             affect, physical function and pain catastrophizing, the
             reverse lagged temporal associations between prior day
             pain-related factors and subsequent night sleep quality, and
             whether the time of day during which an assessment was made
             moderated these temporal associations.Chronic low back pain
             patients (n = 105) completed structured electronic diary
             assessments five times per day for 14 days. Items included
             patient ratings of their pain, affect, physical function,
             and pain catastrophizing.Collapsed across all observations,
             poorer sleep quality was significantly related to higher
             pain ratings, higher negative affect, lower positive affect,
             poorer physical function, and higher pain catastrophizing.
             Lagged analyses averaged across the day revealed that poorer
             prior night sleep quality significantly predicted greater
             next day patient ratings of pain, and poorer physical
             function and higher pain catastrophizing. Prior poorer night
             sleep quality significantly predicted greater reports of
             pain, and poorer physical function, and higher pain
             catastrophizing, especially during the early part of the
             day. Sleep quality × time of day interactions showed that
             poor sleepers reported high pain, and negative mood and low
             function uniformly across the day, whereas good sleepers
             reported relatively good mornings, but showed pain, affect
             and function levels comparable to poor sleepers by the end
             of the day. Analyses of the reverse causal pathway were
             mostly nonsignificant.Sleep quality appears related not only
             to pain intensity but also to a wide range of patient mood
             and function factors. A good night's sleep also appears to
             offer only temporary respite, suggesting that comprehensive
             interventions for chronic low back pain not only should
             include attention to sleep problems but also focus on
             problems with pain appraisals and coping.},
   Doi = {10.1007/s12160-016-9860-2},
   Key = {fds330577}
}

@article{fds331440,
   Title = {Internet-Delivered Exercise and Pain-Coping Skills Training
             for Chronic Knee Pain},
   Journal = {Annals of Internal Medicine},
   Volume = {166},
   Number = {7},
   Pages = {I-13-I-13},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.7326/P17-9031},
   Doi = {10.7326/P17-9031},
   Key = {fds331440}
}

@article{fds333023,
   Author = {Keefe, FJ and Sommer, C},
   Title = {An introduction to the Biennial Review of
             Pain.},
   Journal = {Pain},
   Volume = {158 Suppl 1},
   Pages = {S1},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.1097/j.pain.0000000000000877},
   Doi = {10.1097/j.pain.0000000000000877},
   Key = {fds333023}
}

@article{fds333024,
   Author = {Bennell, KL and Nelligan, R and Dobson, F and Rini, C and Keefe, F and Kasza, J and French, S and Bryant, C and Dalwood, A and Abbott, JH and Hinman, RS},
   Title = {Effectiveness of an Internet-Delivered Exercise and
             Pain-Coping Skills Training Intervention for Persons With
             Chronic Knee Pain: A Randomized Trial.},
   Journal = {Annals of Internal Medicine},
   Volume = {166},
   Number = {7},
   Pages = {453-462},
   Year = {2017},
   Month = {April},
   url = {http://dx.doi.org/10.7326/m16-1714},
   Abstract = {Effective, accessible biopsychosocial treatments are needed
             to manage chronic knee pain on a population level.To
             evaluate the effectiveness of Internet-delivered,
             physiotherapist-prescribed home exercise and pain-coping
             skills training (PCST).Pragmatic parallel-group randomized,
             controlled trial. (Australian New Zealand Clinical Trials
             Registry: ACTRN12614000243617).Community (Australia).148
             persons aged 50 years or older with chronic knee pain.The
             intervention was delivered via the Internet and included
             educational material, 7 videoconferencing (Skype
             [Microsoft]) sessions with a physiotherapist for home
             exercise, and a PCST program over 3 months. The control was
             Internet-based educational material.Primary outcomes were
             pain during walking (11-point numerical rating scale) and
             physical function (Western Ontario and McMaster Universities
             Osteoarthritis Index) at 3 months. Secondary outcomes were
             knee pain, quality of life, global change (overall, pain,
             and functional status), arthritis self-efficacy, coping, and
             pain catastrophizing. Outcomes were also measured at 9
             months.Of participants enrolled, 139 (94%) completed primary
             outcome measures at 3 months and 133 (90%) completed
             secondary outcome measures at 9 months; multiple imputation
             was used for missing data. The intervention group reported
             significantly more improvement in pain (mean difference, 1.6
             units [95% CI, 0.9 to 2.3 units]) and physical function
             (mean difference, 9.3 units [CI, 5.9 to 12.7 units]) than
             the control group at 3 months, and improvements were
             sustained at 9 months (mean differences, 1.1 units [CI, 0.4
             to 1.8 units] and 7.0 units [CI, 3.4 to 10.5 units],
             respectively). Intervention participants showed
             significantly more improvement in most secondary outcomes
             than control participants. At both time points,
             significantly more intervention participants reported global
             improvements.Participants were unblinded.For persons with
             chronic knee pain, Internet-delivered, physiotherapist-prescribed
             exercise and PCST provide clinically meaningful improvements
             in pain and function that are sustained for at least 6
             months.National Health and Medical Research
             Council.},
   Doi = {10.7326/m16-1714},
   Key = {fds333024}
}

@article{fds330906,
   Author = {Edmond, SN and Shelby, RA and Keefe, FJ and Fisher, HM and Schmidt, JE and Soo, MS and Skinner, CS and Ahrendt, GM and Manculich, J and Sumkin, JH and Zuley, ML and Bovbjerg, DH},
   Title = {Persistent Breast Pain Among Women With Histories of
             Breast-conserving Surgery for Breast Cancer Compared With
             Women Without Histories of Breast Surgery or
             Cancer.},
   Journal = {The Clinical Journal of Pain},
   Volume = {33},
   Number = {1},
   Pages = {51-56},
   Year = {2017},
   Month = {January},
   url = {http://dx.doi.org/10.1097/AJP.0000000000000377},
   Abstract = {This study compared persistent breast pain among women who
             received breast-conserving surgery for breast cancer and
             women without a history of breast cancer.Breast cancer
             survivors (n=200) were recruited at their first postsurgical
             surveillance mammogram (6 to 15 mo postsurgery). Women
             without a breast cancer history (n=150) were recruited at
             the time of a routine screening mammogram. All women
             completed measures of breast pain, pain interference with
             daily activities and intimacy, worry about breast pain,
             anxiety symptoms, and depression symptoms. Demographic and
             medical information were also collected.Persistent breast
             pain (duration ≥6 mo) was reported by 46.5% of breast
             cancer survivors and 12.7% of women without a breast cancer
             history (P<0.05). Breast cancer survivors also had
             significantly higher rates of clinically significant
             persistent breast pain (pain intensity score ≥3/10), as
             well as higher average breast pain intensity and
             unpleasantness scores. Breast cancer survivors with
             persistent breast pain had significantly higher levels of
             depressive symptoms, as well as pain worry and interference,
             compared with survivors without persistent breast pain or
             women without a breast cancer history. Anxiety symptoms were
             significantly higher in breast cancer survivors with
             persistent breast pain compared with women without a breast
             cancer history.Results indicate that persistent breast pain
             negatively impacts women with a history of breast-conserving
             cancer surgery compared with women without that history.
             Strategies to ameliorate persistent breast pain and to
             improve adjustment among women with persistent breast pain
             should be explored for incorporation into standard care for
             breast cancer survivors.},
   Doi = {10.1097/AJP.0000000000000377},
   Key = {fds330906}
}


%% Keefe, Richard S.   
@article{fds337700,
   Author = {Lam, M and Lee, J and Rapisarda, A and See, YM and Yang, Z and Lee, S-A and Abdul-Rashid, NA and Kraus, M and Subramaniam, M and Chong, S-A and Keefe, RSE},
   Title = {Longitudinal Cognitive Changes in Young Individuals at
             Ultrahigh Risk for Psychosis.},
   Journal = {Jama Psychiatry},
   Volume = {75},
   Number = {9},
   Pages = {929-939},
   Year = {2018},
   Month = {September},
   url = {http://dx.doi.org/10.1001/jamapsychiatry.2018.1668},
   Abstract = {Cognitive deficits are a key feature of risk for psychosis.
             Longitudinal changes in cognitive architecture may be
             associated with the social and occupational functioning in
             young people.To examine longitudinal profiles of cognition
             in individuals at ultrahigh risk (UHR) for psychosis,
             compared with healthy controls, and to investigate the
             association of cognition with functioning.This study has a
             multiple-group prospective design completed in 24 months and
             was conducted from January 1, 2009, to November 11, 2012, as
             part of the Longitudinal Youth at-Risk Study conducted in
             Singapore. Participants either were recruited from
             psychiatric outpatient clinics, educational institutions,
             and community mental health agencies or self-referred.
             Follow-up assessments were performed every 6 months for 2
             years or until conversion to psychosis. Individuals with
             medical causes for psychosis, current illicit substance use,
             or color blindness were excluded. Data analysis was
             conducted from June 2014 to May 2018.Neuropsychological,
             perceptual, and social cognitive tasks; semi-structured
             interviews, and the Structured Clinical Interview for DSM-IV
             Axis I disorders were administered every 6 months. The UHR
             status of nonconverters, converters, remitters, and
             nonremitters was monitored. Cognitive domain scores and
             functioning were investigated longitudinally.In total, 384
             healthy controls and 173 UHR individuals between ages 14 and
             29 years were evaluated prospectively. Of the 384 healthy
             controls, 153 (39.8%) were female and 231 (60.2%) were male
             with a mean (SD) age of 21.69 (3.26) years. Of the 173
             individuals at UHR for psychosis, 56 (32.4%) were female and
             117 (67.6%) were male with a mean (SD) age of 21.27 (3.52)
             years). After 24 months of follow-up, 383 healthy controls
             (99.7%) and 122 individuals at UHR for psychosis (70.5%)
             remained. Baseline cognitive deficits were associated with
             psychosis conversion later (mean odds ratio [OR], 1.66;
             combined 95% CI, 1.08-2.83; P = .04) and nonremission of
             UHR status (mean OR, 1.67; combined 95% CI, 1.09-2.95;
             P = .04). Five cognitive components-social cognition,
             attention, verbal fluency, general cognitive function, and
             perception-were obtained from principal components analysis.
             Longitudinal component structure change was observed in
             general cognitive function (maximum vertical
             deviation = 0.59; χ2 = 8.03; P = .01).
             Group-by-time interaction on general cognitive function
             (F = 12.23; η2 = 0.047; P < .001) and
             perception (F = 8.33; η2 = 0.032; P < .001) was
             present. Changes in attention (F = 5.65;
             η2 = 0.013; P = .02) and general cognitive function
             (F = 7.18; η2 = 0.014; P = .01) accounted for
             longitudinal changes in social and occupational
             functioning.Individuals in this study who met the UHR
             criteria appeared to demonstrate cognitive deficits, and
             those whose UHR status remitted were seen to recover
             cognitively. Cognition appeared as poor in nonremitters and
             appeared to be associated with poor functional outcome. This
             study suggests that cognitive dimensions are sensitive to
             the identification of young individuals at risk for
             psychosis and to the longitudinal course of those at highest
             risk.},
   Doi = {10.1001/jamapsychiatry.2018.1668},
   Key = {fds337700}
}

@article{fds332787,
   Author = {Xavier, RM and Dungan, JR and Keefe, RSE and Vorderstrasse,
             A},
   Title = {Polygenic signal for symptom dimensions and cognitive
             performance in patients with chronic schizophrenia.},
   Journal = {Schizophrenia Research. Cognition},
   Volume = {12},
   Pages = {11-19},
   Year = {2018},
   Month = {June},
   url = {http://dx.doi.org/10.1016/j.scog.2018.01.001},
   Abstract = {Genetic etiology of psychopathology symptoms and cognitive
             performance in schizophrenia is supported by candidate gene
             and polygenic risk score (PRS) association studies. Such
             associations are reported to be dependent on several factors
             - sample characteristics, illness phase, illness severity
             etc. We aimed to examine if schizophrenia PRS predicted
             psychopathology symptoms and cognitive performance in
             patients with chronic schizophrenia. We also examined if
             schizophrenia associated autosomal loci were associated with
             specific symptoms or cognitive domains. Case-only analysis
             using data from the Clinical Antipsychotics Trials of
             Intervention Effectiveness-Schizophrenia trials
             (n = 730). PRS was constructed using Psychiatric
             Genomics Consortium (PGC) leave one out genome wide
             association analysis as the discovery data set. For
             candidate region analysis, we selected 105-schizophrenia
             associated autosomal loci from the PGC study. We found a
             significant effect of PRS on positive symptoms at
             p-threshold (PT ) of 0.5 (R2 = 0.007, p = 0.029,
             empirical p = 0.029) and negative symptoms at PT of
             1e-07 (R2 = 0.005, p = 0.047, empirical
             p = 0.048). For models that additionally controlled for
             neurocognition, best fit PRS predicted positive (p-threshold
             0.01, R2 = 0.007, p = 0.013, empirical
             p = 0.167) and negative symptoms (p-threshold 0.1,
             R2 = 0.012, p = 0.004, empirical p = 0.329). No
             associations were seen for overall neurocognitive and social
             cognitive performance tests. Post-hoc analyses revealed that
             PRS predicted working memory and vigilance performance but
             did not survive correction. No candidate regions that
             survived multiple testing corrections were associated with
             either symptoms or cognitive performance. Our findings point
             to potentially distinct pathogenic mechanisms for
             schizophrenia symptoms.},
   Doi = {10.1016/j.scog.2018.01.001},
   Key = {fds332787}
}

@article{fds336077,
   Author = {Yang, Z and Lim, K and Lam, M and Keefe, R and Lee, J},
   Title = {Factor structure of the positive and negative syndrome scale
             (PANSS) in people at ultra high risk (UHR) for
             psychosis.},
   Journal = {Schizophrenia Research},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.schres.2018.05.024},
   Abstract = {The Positive and Negative Syndrome Scale (PANSS), a
             comprehensive psychopathology assessment scale used in the
             evaluation of psychopathology in schizophrenia, is also
             often used in the Ultra-High-Risk (UHR) population. This
             paper examined the dimensional structure of the PANSS in a
             UHR sample.A total of 168 individuals assessed to be at UHR
             for psychosis on the Comprehensive Assessment of At-Risk
             Mental States (CAARMS) were evaluated on the PANSS, Calgary
             Depression Scale for Schizophrenia (CDSS), Beck Anxiety
             Inventory (BAI), Brief Assessment of Cognition in
             Schizophrenia (BACS), and Global Assessment of Functioning
             (GAF). Exploratory factor analysis (EFA) of the PANSS was
             performed to identify the factorial structure. Convergent
             validity was explored with the CAARMS, CDSS, BAI and
             BACS.EFA of the PANSS yielded five symptom factors -
             Positive, Negative, Cognition/Disorganization,
             Anxiety/Depression, and Hostility. This 5-factor solution
             showed good convergent validity with the CAARMS composite
             score, CDSS, BAI, and BACS. Positive, Negative and
             Anxiety/Depression factors were associated with
             functioning.The reported PANSS factor structure may serve to
             improve the understanding and measurement of clinical
             symptom dimensions manifested in people with UHR for future
             research and clinical setting.},
   Doi = {10.1016/j.schres.2018.05.024},
   Key = {fds336077}
}

@article{fds329375,
   Author = {Blokland, GAM and Del Re and EC and Mesholam-Gately, RI and Jovicich, J and Trampush, JW and Keshavan, MS and DeLisi, LE and Walters, JTR and Turner, JA and Malhotra, AK and Lencz, T and Shenton, ME and Voineskos,
             AN and Rujescu, D and Giegling, I and Kahn, RS and Roffman, JL and Holt,
             DJ and Ehrlich, S and Kikinis, Z and Dazzan, P and Murray, RM and Di Forti,
             M and Lee, J and Sim, K and Lam, M and Wolthusen, RPF and de Zwarte, SMC and Walton, E and Cosgrove, D and Kelly, S and Maleki, N and Osiecki, L and Picchioni, MM and Bramon, E and Russo, M and David, AS and Mondelli, V and Reinders, AATS and Falcone, MA and Hartmann, AM and Konte, B and Morris,
             DW and Gill, M and Corvin, AP and Cahn, W and Ho, NF and Liu, JJ and Keefe,
             RSE and Gollub, RL and Manoach, DS and Calhoun, VD and Schulz, SC and Sponheim, SR and Goff, DC and Buka, SL and Cherkerzian, S and Thermenos,
             HW and Kubicki, M and Nestor, PG and Dickie, EW and Vassos, E and Ciufolini, S and Reis Marques and T and Crossley, NA and Purcell, SM and Smoller, JW and van Haren, NEM and Toulopoulou, T and Donohoe, G and Goldstein, JM and Seidman, LJ and McCarley, RW and Petryshen,
             TL},
   Title = {The Genetics of Endophenotypes of Neurofunction to
             Understand Schizophrenia (GENUS) consortium: A collaborative
             cognitive and neuroimaging genetics project.},
   Journal = {Schizophrenia Research},
   Volume = {195},
   Pages = {306-317},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1016/j.schres.2017.09.024},
   Abstract = {Schizophrenia has a large genetic component, and the
             pathways from genes to illness manifestation are beginning
             to be identified. The Genetics of Endophenotypes of
             Neurofunction to Understand Schizophrenia (GENUS) Consortium
             aims to clarify the role of genetic variation in brain
             abnormalities underlying schizophrenia. This article
             describes the GENUS Consortium sample collection.We
             identified existing samples collected for schizophrenia
             studies consisting of patients, controls, and/or individuals
             at familial high-risk (FHR) for schizophrenia. Samples had
             single nucleotide polymorphism (SNP) array data or genomic
             DNA, clinical and demographic data, and neuropsychological
             and/or brain magnetic resonance imaging (MRI) data. Data
             were subjected to quality control procedures at a central
             site.Sixteen research groups contributed data from 5199
             psychosis patients, 4877 controls, and 725 FHR individuals.
             All participants have relevant demographic data and all
             patients have relevant clinical data. The sex ratio is 56.5%
             male and 43.5% female. Significant differences exist between
             diagnostic groups for premorbid and current IQ (both
             p<1×10-10). Data from a diversity of neuropsychological
             tests are available for 92% of participants, and 30% have
             structural MRI scans (half also have diffusion-weighted MRI
             scans). SNP data are available for 76% of participants. The
             ancestry composition is 70% European, 20% East Asian, 7%
             African, and 3% other.The Consortium is investigating the
             genetic contribution to brain phenotypes in a schizophrenia
             sample collection of >10,000 participants. The breadth of
             data across clinical, genetic, neuropsychological, and MRI
             modalities provides an important opportunity for elucidating
             the genetic basis of neural processes underlying
             schizophrenia.},
   Doi = {10.1016/j.schres.2017.09.024},
   Key = {fds329375}
}

@article{fds336078,
   Author = {Keefe, RSE and Nomikos, G and Zhong, W and Christensen, MC and Jacobson,
             W},
   Title = {A Subgroup Analysis of the Impact of Vortioxetine on
             Functional Capacity, as Measured by UPSA, in Patients with
             Major Depressive Disorder and Subjective Cognitive
             Dysfunction.},
   Journal = {The International Journal of Neuropsychopharmacology},
   Volume = {21},
   Number = {5},
   Pages = {442-447},
   Year = {2018},
   Month = {May},
   url = {http://dx.doi.org/10.1093/ijnp/pyy020},
   Abstract = {We evaluated vortioxetine's effects on functional capacity
             in demographic and clinical subgroups of patients with major
             depressive disorder.This was an explorat