Kenneth A. Dodge

Publications of Kenneth A. Dodge    :chronological  alphabetical  combined  by tags listing:

%% Journal Articles   
@article{fds271925,
   Author = {Chan, TWS and Bates, JE and Lansford, JE and Dodge, KA and Pettit, GS and Dick, DM and Latendresse, SJ},
   Title = {Impulsivity and genetic variants in DRD2 and ANKK1 moderate
             longitudinal associations between sleep problems and
             overweight from ages 5 to 11},
   Journal = {International Journal of Obesity},
   Volume = {38},
   Number = {3},
   Pages = {404-410},
   Year = {2014},
   Month = {January},
   ISSN = {0307-0565},
   url = {http://dx.doi.org/10.1038/ijo.2013.123},
   Abstract = {OBJECTIVE:Short sleep duration and sleep problems increase
             risks of overweight and weight gain. Few previous studies
             have examined sleep and weight repeatedly over development.
             This study examined the associations between yearly reports
             of sleep problems and weight status from ages 5 to 11.
             Although, previous studies have shown that inter-individual
             differences moderate the effect of short sleep duration on
             weight, it is not known whether inter-individual differences
             also moderate the effect of sleep problems on weight. We
             tested how the longitudinal associations between sleep
             problems and weight status were moderated by impulsivity and
             genetic variants in DRD2 and ANKK1.DESIGN:Seven-year
             longitudinal study.PARTICIPANTS:A total of 567 children from
             the Child Development Project for the analysis with
             impulsivity and 363 for the analysis with genetic
             variants.MEASUREMENTS and RESULTS:Sleep problems and weight
             status were measured by mothers' reports yearly. Impulsivity
             was measured by teachers' reports yearly. Six
             single-nucleotide polymorphisms located in DRD2 and ANKK1
             were genotyped. Data were analyzed using multilevel
             modeling. Higher average levels of sleep deprivation across
             years were associated with greater increases in overweight
             (P=0.0024). Sleep problems and overweight were associated at
             both within-person across time (P<0.0001) and between-person
             levels (P<0.0001). Impulsivity and two polymorphisms,
             rs1799978 and rs4245149 in DRD2, moderated the association
             between sleep problems and overweight; the association was
             stronger in children who were more impulsive (P=0.0022), in
             G allele carriers for rs1799978 (P=0.0007) and in A allele
             carriers for rs4245149 (P=0.0002).CONCLUSIONS:This study
             provided incremental evidence for the influence of sleep
             problems on weight. Findings of DRD2, ANKK1 and impulsivity
             are novel; they suggest that reward sensitivity and
             self-regulatory abilities might modulate the influences of
             sleep on weight gain. The analysis of polymorphisms was
             restricted to European Americans and hence the results might
             not generalize to other populations. © 2014 Macmillan
             Publishers Limited.},
   Doi = {10.1038/ijo.2013.123},
   Key = {fds271925}
}

@article{fds272028,
   Author = {Latendresse, SJ and Bates, JE and Goodnight, JA and Lansford, JE and Budde, JP and Goate, A and Dodge, KA and Pettit, GS and Dick,
             DM},
   Title = {Differential Susceptibility to Adolescent Externalizing
             Trajectories: Examining the Interplay Between CHRM2 and Peer
             Group Antisocial Behavior},
   Journal = {Child Development},
   Volume = {82},
   Number = {6},
   Pages = {1797-1814},
   Year = {2011},
   ISSN = {0009-3920},
   url = {http://dx.doi.org/10.1111/j.1467-8624.2011.01640.x},
   Abstract = {The present study characterized prototypical patterns of
             development in self-reported externalizing behavior, between
             12 and 22years of age, within a community sample of 452
             genotyped individuals. A Caucasian subset (n=378) was then
             examined to determine whether their probabilities of
             displaying discrete trajectories were differentially
             associated with CHRM2, a gene implicated in self-regulatory
             processes across a range of externalizing behaviors, and if
             affiliating with antisocial peers moderated these
             associations. Findings indicate that relative to a normative
             "lower risk" externalizing trajectory, likelihood of
             membership in two "higher risk" trajectories increased with
             each additional copy of the minor allelic variant at CHRM2,
             and that this association was exacerbated among those
             exposed to higher levels of peer group antisocial behavior.
             © 2011 The Authors. Child Development © 2011 Society for
             Research in Child Development, Inc.},
   Doi = {10.1111/j.1467-8624.2011.01640.x},
   Key = {fds272028}
}

@article{fds272038,
   Author = {McMahon, RJ and Witkiewitz, K and Kotler, JS},
   Title = {Predictive validity of callous–unemotional traits measured
             in early adolescence with respect to multiple antisocial
             outcomes.},
   Journal = {Journal of Abnormal Psychology},
   Volume = {119},
   Number = {4},
   Pages = {752-763},
   Year = {2010},
   url = {http://dx.doi.org/10.1037/a0020796},
   Doi = {10.1037/a0020796},
   Key = {fds272038}
}

@article{fds272045,
   Author = {Edwards, AC and Dodge, KA and Latendresse, SJ and Lansford, JE and Bates, JE and Pettit, GS and Budde, JP and Goate, AM and Dick,
             DM},
   Title = {MAOA-uVNTR and early physical discipline interact to
             influence delinquent behavior},
   Journal = {The Journal of Child Psychology and Psychiatry and Allied
             Disciplines},
   Volume = {51},
   Number = {6},
   Pages = {679-687},
   Year = {2010},
   ISSN = {0021-9630},
   url = {http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=000272027300049&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=47d3190e77e5a3a53558812f597b0b92},
   Abstract = {Background: A functional polymorphism in the promoter region
             of the monoamine oxidizing gene monoamine oxidase A (MAOA)
             has been associated with behavioral sensitivity to adverse
             environmental conditions in multiple studies (e.g., Caspi et
             al. 2002; Kim-Cohen et al., 2006). The present study
             investigates the effects of genotype and early physical
             discipline on externalizing behavior. We expand on the
             current literature in our assessment of externalizing,
             incorporating information across multiple reporters and over
             a broad developmental time period, and in our understanding
             of environmental risk. Method: This study uses data from the
             Child Development Project, an ongoing longitudinal study
             following a community sample of children beginning at age 5.
             Physical discipline before age 6 was quantified using a
             subset of questions from the Conflict Tactics Scale (Straus,
             1979). Externalizing behavior was assessed in the male,
             European-American sub-sample (N = 250) by parent, teacher,
             and self-report using Achenbach's Child Behavior Checklist,
             Teacher Report Form, and Youth Self-Report (Achenbach,
             1991), at 17 time points from ages 6 to 22. Regression
             analyses tested the influence of genotype, physical
             discipline, and their interaction on externalizing behavior,
             and its subscales, delinquency and aggression. Results: We
             found a significant interaction effect between genotype and
             physical discipline on levels of delinquent behavior.
             Similar trends were observed for aggression and overall
             externalizing behavior, although these did not reach
             statistical significance. Main effects of physical
             discipline held for all outcome variables, and no main
             effects held for genotype. Conclusion: The adverse
             consequences of physical discipline on forms of
             externalizing behavior are exacerbated by an underlying
             biological risk conferred by MAOA genotype. © 2009
             Association for Child and Adolescent Mental
             Health.},
   Doi = {10.1111/j.1469-7610.2009.02196.x},
   Key = {fds272045}
}

@article{fds272061,
   Author = {Dodge, KA},
   Title = {Mechanisms of Gene-Environment Interaction Effects in the
             Development of Conduct Disorder.},
   Journal = {Perspectives on Psychological Science},
   Volume = {4},
   Number = {4},
   Pages = {408-414},
   Year = {2009},
   Month = {July},
   ISSN = {1745-6916},
   url = {http://www.ncbi.nlm.nih.gov/pubmed/19779577},
   Abstract = {The gene-environment interaction effect in the development
             of conduct disorder is one of the most important discoveries
             of the past decade, but the mechanisms through which this
             effect operates remain elusive. I propose a model of these
             processes that focuses on the individual's response to a
             threatening stimulus in ongoing social interaction. The
             individual's response coordinates three interrelated
             systems: neural, autonomic, and information-processing. In
             each system, adaptive, evolutionarily selected response
             patterns characterize normal responding, but in
             psychopathology these patterns have gone awry. Antecedents
             of individual differences in these response patterns arise
             from genetic polymorphisms, adverse environmental
             experiences early in life, and their interaction. Programs
             of research are proposed to test hypotheses in the model
             through longitudinal, experimental, and clinical
             intervention methods. This model can serve as a template for
             inquiry in other forms of developmental psychopathology.},
   Doi = {10.1111/j.1745-6924.2009.01147.x},
   Key = {fds272061}
}

@article{fds272287,
   Author = {Jaffee, SR and Caspi, A and Moffitt, TE and Dodge, KA and Rutter, M and Taylor, A and Tully, LA},
   Title = {Nature X nurture: genetic vulnerabilities interact with
             physical maltreatment to promote conduct
             problems.},
   Journal = {Development and Psychopathology},
   Volume = {17},
   Number = {1},
   Pages = {67-84},
   Year = {2005},
   ISSN = {0954-5794},
   url = {http://www.ncbi.nlm.nih.gov/pubmed/15971760},
   Abstract = {Maltreatment places children at risk for psychiatric
             morbidity, especially conduct problems. However, not all
             maltreated children develop conduct problems. We tested
             whether the effect of physical maltreatment on risk for
             conduct problems was strongest among those who were at high
             genetic risk for these problems using data from the E-risk
             Study, a representative cohort of 1,116 5-year-old British
             twin pairs and their families. Children's conduct problems
             were ascertained via parent and teacher interviews. Physical
             maltreatment was ascertained via parent report. Children's
             genetic risk for conduct problems was estimated as a
             function of their co-twin's conduct disorder status and the
             pair's zygosity. The effect of maltreatment on risk for
             conduct problems was strongest among those at high genetic
             risk. The experience of maltreatment was associated with an
             increase of 2% in the probability of a conduct disorder
             diagnosis among children at low genetic risk for conduct
             disorder but an increase of 24% among children at high
             genetic risk. Prediction of behavioral pathology can attain
             greater accuracy if both pathogenic environments and genetic
             risk are ascertained. Certain genotypes may promote
             resistance to trauma. Physically maltreated children whose
             first-degree relatives engage in antisocial behavior warrant
             priority for therapeutic intervention.},
   Doi = {10.1017/S0954579405050042},
   Key = {fds272287}
}

@article{fds272285,
   Author = {Dodge, KA},
   Title = {The Nature-Nurture Debate and Public Policy.},
   Journal = {Merrill-Palmer quarterly (Wayne State University.
             Press)},
   Volume = {50},
   Number = {4},
   Pages = {418-427},
   Year = {2004},
   Month = {October},
   ISSN = {0272-930X},
   url = {http://www.ncbi.nlm.nih.gov/pubmed/20011615},
   Abstract = {The contentious nature-nurture debate in developmental
             psychology is poised to reach a rapprochement with
             contemporary concepts of gene-environment interaction,
             transaction, and fit. Discoveries over the past decade have
             revealed how neither genes nor the environment offers a
             sufficient window into human development. Rather, the most
             important discoveries have come from unearthing the manner
             in which the environment alters gene expression (and how
             genes impose limits on environmental effects), how biology
             and the environment influence each other across time, and
             how maximizing gene-environment fit leads to optimal
             outcomes for children. The manner in which these factors
             operate in tandem should direct future scholarship,
             practice, and public policy.},
   Doi = {10.1353/mpq.2004.0028},
   Key = {fds272285}
}

@article{fds272138,
   Author = {Dodge, KA and Pettit, GS},
   Title = {A biopsychosocial model of the development of chronic
             conduct problems in adolescence.},
   Journal = {Developmental Psychology},
   Volume = {39},
   Number = {2},
   Pages = {349-371},
   Year = {2003},
   Month = {March},
   ISSN = {0012-1649},
   url = {http://www.ncbi.nlm.nih.gov/pubmed/12661890},
   Abstract = {A biopsychosocial model of the development of adolescent
             chronic conduct problems is presented and supported through
             a review of empirical findings. This model posits that
             biological dispositions and sociocultural contexts place
             certain children at risk in early life but that life
             experiences with parents, peers. and social institutions
             increment and mediate this risk. A transactional
             developmental model is best equipped to describe the
             emergence of chronic antisocial behavior across time.
             Reciprocal influences among dispositions, contexts, and life
             experiences lead to recursive iterations across time that
             exacerbate or diminish antisocial development. Cognitive and
             emotional processes within the child, including the
             acquisition of knowledge and social-information-processing
             patterns, mediate the relation between life experiences and
             conduct problem outcomes. Implications for prevention
             research and public policy are noted.},
   Doi = {10.1037//0012-1649.39.2.349},
   Key = {fds272138}
}

@article{fds272273,
   Author = {Dodge, KA},
   Title = {New wrinkles in the person versus situation
             debate},
   Journal = {Psychological Inquiry},
   Volume = {4},
   Number = {4},
   Pages = {284-286},
   Year = {1993},
   url = {http://dx.doi.org/10.1207/s15327965pli0404_6},
   Doi = {10.1207/s15327965pli0404_6},
   Key = {fds272273}
}

@article{fds272263,
   Author = {Dodge, KA},
   Title = {Nature Versus Nurture in Childhood Conduct Disorder: It Is
             Time to Ask a Different Question},
   Journal = {Developmental Psychology},
   Volume = {26},
   Number = {5},
   Pages = {698-701},
   Year = {1990},
   ISSN = {0012-1649},
   Abstract = {Lytton (1990, this issue) offers a lucid review of factors
             in the development of conduct disorder in children that
             focuses on the question of the "relative strength" of child
             effects versus environmental effects. This question ignores
             the fact that such estimates are a function of the
             subpopulation being assessed and the context in which
             measurement occurs. These estimates pit nature versus
             nurture in a way that detracts from an emphasis on the
             interaction of factors that characterizes most human
             behavioral development. This perspective also assumes that
             "child effects," "environmental effects," and "conduct
             disorder" are homogeneous constructs, but these are more
             likely aggregations of heterogeneous phenomena that have
             been grouped together only for heuristic reasons. It is
             recommended that instead of focusing on the relative sizes
             of effects, researchers should focus on the questions of
             which mechanisms operate and how they interact during
             transactional development.},
   Key = {fds272263}
}


%% Book Reviews   
@article{fds219663,
   Author = {Dodge, K.A.},
   Title = {Book review: The Handbook of Clinical Child Neuropsychology,
             3rd edition},
   Journal = {Journal of Clinical Psychiatry},
   Volume = {72},
   Number = {5},
   Pages = {726},
   Editor = {Edited by Cecil R. Reynolds and Elaine Fletcher-Janzen},
   Year = {2010},
   Key = {fds219663}
}