Psychology and Neuroscience Post Docs Database
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Psychology and Neuroscience Postdocs: Publications since January 2023

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%% Bourassa, Kyle   
@article{fds366153,
   Author = {Bourassa, KJ and Moffitt, TE and Harrington, H and Houts, R and Poulton,
             R and Ramrakha, S and Rasmussen, LJH and Wertz, J and Caspi,
             A},
   Title = {Childhood Adversity and Midlife Health: Shining a Light on
             the Black Box of Psychosocial Mechanisms.},
   Journal = {Prevention Science : the Official Journal of the Society for
             Prevention Research},
   Volume = {24},
   Number = {5},
   Pages = {817-828},
   Year = {2023},
   Month = {July},
   url = {http://dx.doi.org/10.1007/s11121-022-01431-y},
   Abstract = {Adverse childhood experiences (ACEs) are associated with
             poorer health, which has spurred public health efforts to
             reduce the number of adverse events children experience.
             Unfortunately, it is unlikely that all ACEs can be
             prevented. For adults who already experienced ACEs in
             childhood, what psychological, social, and behavioral
             intervention targets might reduce risk for negative health
             outcomes? To provide insight into the "black box" of
             psychosocial mechanisms linking ACEs to poor health, our
             study used data from the Dunedin Study, a longitudinal
             cohort assessed from birth to age 45. Mediation models
             (N = 859) were used to examine whether candidate
             psychosocial variables in adulthood explained the
             association between childhood ACEs and health in midlife.
             Potential psychosocial mediators included stressful life
             events, perceived stress, negative emotionality, and health
             behaviors. Children who experienced more ACEs had poorer
             health in midlife. They also had significantly more
             stressful life events, more perceived stress, more negative
             emotionality, and unhealthier behaviors as adults. These
             mediators were each independently associated with poorer
             health in midlife and statistically mediated the association
             between ACEs and midlife health. Health behaviors evidenced
             the strongest indirect effect from ACEs to midlife health.
             Together, these psychosocial mediators accounted for the
             association between ACEs in childhood and health three
             decades later. Public health efforts to mitigate the health
             consequences of ACEs could aim to reduce the stressful life
             events people experience, reduce negative emotionality,
             reduce perceived stress, or improve health behaviors among
             adults who experienced childhood adversity.},
   Doi = {10.1007/s11121-022-01431-y},
   Key = {fds366153}
}

@article{fds370508,
   Author = {Bourassa, KJ and Caspi, A and Brennan, GM and Hall, KS and Harrington,
             H and Houts, R and Kimbrel, NA and Poulton, R and Ramrakha, S and Taylor,
             GA and Moffitt, TE},
   Title = {Which Types of Stress Are Associated With Accelerated
             Biological Aging? Comparing Perceived Stress, Stressful Life
             Events, Childhood Adversity, and Posttraumatic Stress
             Disorder.},
   Journal = {Psychosom Med},
   Volume = {85},
   Number = {5},
   Pages = {389-396},
   Year = {2023},
   Month = {June},
   url = {http://dx.doi.org/10.1097/PSY.0000000000001197},
   Abstract = {OBJECTIVE: Stress and stressful events are associated with
             poorer health; however, there are multiple ways to
             conceptualize and measure stress and stress responses. One
             physiological mechanism through which stress could result in
             poorer health is accelerated biological aging. This study
             tested which types of stress were associated with
             accelerated biological aging in adulthood. METHODS: Studying
             955 participants from the Dunedin Longitudinal Study, we
             tested whether four types of stress assessed from ages 32 to
             45 years-perceived stress, number of stressful life events,
             adverse childhood experiences, and posttraumatic stress
             disorder-were associated with accelerated biological aging.
             RESULTS: Higher levels of all four measures of stress were
             significantly associated with accelerated aging in separate
             models. In a combined model, more perceived stress and more
             stressful life events remained associated with faster aging,
             and the stress measures explained 6.9% of the variance in
             aging. The magnitudes of the associations between the four
             measures of stress and biological aging were comparable to
             associations for smoking and low education, two established
             risk factors for accelerated aging. People with high levels
             of perceived stress, numerous adverse childhood experiences
             (4+), high stressful life event counts, or posttraumatic
             stress disorder were aging an additional estimated 2.4
             months, 1.1 additional months, 1.4 months, and 1.4 months
             per year, respectively. CONCLUSIONS: Assessing stress,
             particularly perceived stress, could help identify people at
             risk of accelerated aging. Intervening to treat stress or
             the health-relevant sequelae of stress could potentially
             slow the rate at which people are aging, improving their
             health as they age.},
   Doi = {10.1097/PSY.0000000000001197},
   Key = {fds370508}
}


%% Brennan, Grace   
@article{fds375249,
   Author = {Brennan, GM and Moffitt, TE and Bourassa, KJ and Harrington, H and Hogan, S and Houts, RM and Poulton, R and Ramrakha, S and Caspi,
             A},
   Title = {The Continuity of Adversity: Negative Emotionality Links
             Early Life Adversity With Adult Stressful Life
             Events},
   Journal = {Clinical Psychological Science : a Journal of the
             Association for Psychological Science},
   Publisher = {SAGE Publications},
   Year = {2024},
   Month = {January},
   url = {http://dx.doi.org/10.1177/21677026231220337},
   Abstract = {<jats:p> Adversity that exhibits continuity across the life
             course has long-term detrimental effects on physical and
             mental health. Using 920 participants from the Dunedin
             Study, we tested the following hypotheses: (a) Children
             (ages 3–15) who experienced adversity would also tend to
             experience adversity in adulthood (ages 32–45), and (2)
             interim personality traits in young adulthood (ages 18–26)
             would help account for this longitudinal association.
             Children who experienced more adversity tended to also
             experience more stressful life events as adults, β = 0.11,
             95% confidence interval [CI] = [0.04, 0.18], p = .002.
             Negative emotionality—particularly its subfacet
             alienation, characterized by mistrust of others—helped
             explain this childhood-to-midlife association (indirect
             effect: β = 0.06, 95% CI = [0.04, 0.09], p &lt; .001).
             Results were robust to adjustment for sex, socioeconomic
             origins, childhood IQ, preschool temperament, and other
             young-adult personality traits. Prevention of early life
             adversity and treatment of young-adult negative emotionality
             may reduce vulnerability to later life stress and thereby
             promote the health of aging adults. </jats:p>},
   Doi = {10.1177/21677026231220337},
   Key = {fds375249}
}

@article{fds373039,
   Author = {Brennan, GM and Moffitt, TE and Ambler, A and Harrington, H and Hogan,
             S and Houts, RM and Mani, R and Poulton, R and Ramrakha, S and Caspi,
             A},
   Title = {Tracing the origins of midlife despair: association of
             psychopathology during adolescence with a syndrome of
             despair-related maladies at midlife.},
   Journal = {Psychological Medicine},
   Volume = {53},
   Number = {16},
   Pages = {7569-7580},
   Year = {2023},
   Month = {December},
   url = {http://dx.doi.org/10.1017/s0033291723001320},
   Abstract = {<h4>Background</h4>Midlife adults are experiencing a crisis
             of deaths of despair (i.e. deaths from suicide, drug
             overdose, and alcohol-related liver disease). We tested the
             hypothesis that a syndrome of despair-related maladies at
             midlife is preceded by psychopathology during
             adolescence.<h4>Methods</h4>Participants are members of a
             representative cohort of 1037 individuals born in Dunedin,
             New Zealand in 1972-73 and followed to age 45 years, with
             94% retention. Adolescent mental disorders were assessed in
             three diagnostic assessments at ages 11, 13, and 15 years.
             Indicators of despair-related maladies across four domains -
             suicidality, substance misuse, sleep problems, and pain -
             were assessed at age 45 using multi-modal measures including
             self-report, informant-report, and national register
             data.<h4>Results</h4>We identified and validated a syndrome
             of despair-related maladies at midlife involving
             suicidality, substance misuse, sleep problems, and pain.
             Adults who exhibited a more severe syndrome of
             despair-related maladies at midlife tended to have had
             early-onset emotional and behavioral disorders [<i>β</i> =
             0.23, 95% CI (0.16-0.30), <i>p</i> < 0.001], even after
             adjusting for sex, childhood SES, and childhood IQ. A more
             pronounced midlife despair syndrome was observed among
             adults who, as adolescents, were diagnosed with a greater
             number of mental disorders [<i>β</i> = 0.26, 95% CI
             (0.19-0.33), <i>p</i> < 0.001]. Tests of diagnostic
             specificity revealed that associations generalized across
             different adolescent mental disorders.<h4>Conclusions</h4>Midlife
             adults who exhibited a more severe syndrome of
             despair-related maladies tended to have had psychopathology
             as adolescents. Prevention and treatment of adolescent
             psychopathology may mitigate despair-related maladies at
             midlife and ultimately reduce deaths of despair.},
   Doi = {10.1017/s0033291723001320},
   Key = {fds373039}
}

@article{fds373038,
   Author = {Bourassa, KJ and Caspi, A and Brennan, GM and Hall, KS and Harrington,
             H and Houts, R and Kimbrel, NA and Poulton, R and Ramrakha, S and Taylor,
             GA and Moffitt, TE},
   Title = {Which Types of Stress Are Associated With Accelerated
             Biological Aging? Comparing Perceived Stress, Stressful Life
             Events, Childhood Adversity, and Posttraumatic Stress
             Disorder.},
   Journal = {Psychosom Med},
   Volume = {85},
   Number = {5},
   Pages = {389-396},
   Year = {2023},
   Month = {June},
   url = {http://dx.doi.org/10.1097/PSY.0000000000001197},
   Abstract = {OBJECTIVE: Stress and stressful events are associated with
             poorer health; however, there are multiple ways to
             conceptualize and measure stress and stress responses. One
             physiological mechanism through which stress could result in
             poorer health is accelerated biological aging. This study
             tested which types of stress were associated with
             accelerated biological aging in adulthood. METHODS: Studying
             955 participants from the Dunedin Longitudinal Study, we
             tested whether four types of stress assessed from ages 32 to
             45 years-perceived stress, number of stressful life events,
             adverse childhood experiences, and posttraumatic stress
             disorder-were associated with accelerated biological aging.
             RESULTS: Higher levels of all four measures of stress were
             significantly associated with accelerated aging in separate
             models. In a combined model, more perceived stress and more
             stressful life events remained associated with faster aging,
             and the stress measures explained 6.9% of the variance in
             aging. The magnitudes of the associations between the four
             measures of stress and biological aging were comparable to
             associations for smoking and low education, two established
             risk factors for accelerated aging. People with high levels
             of perceived stress, numerous adverse childhood experiences
             (4+), high stressful life event counts, or posttraumatic
             stress disorder were aging an additional estimated 2.4
             months, 1.1 additional months, 1.4 months, and 1.4 months
             per year, respectively. CONCLUSIONS: Assessing stress,
             particularly perceived stress, could help identify people at
             risk of accelerated aging. Intervening to treat stress or
             the health-relevant sequelae of stress could potentially
             slow the rate at which people are aging, improving their
             health as they age.},
   Doi = {10.1097/PSY.0000000000001197},
   Key = {fds373038}
}


%% Craig, Maureen A.   
@article{fds372821,
   Author = {Wallace, LE and Craig, MA and Wegener, DT},
   Title = {Biased, but expert: Trade-offs in how stigmatized versus
             non-stigmatized advocates are perceived and consequences for
             persuasion},
   Journal = {Journal of Experimental Social Psychology},
   Volume = {110},
   Year = {2024},
   Month = {January},
   url = {http://dx.doi.org/10.1016/j.jesp.2023.104519},
   Abstract = {Stigmatized versus non-stigmatized people advocating on
             behalf of the stigmatized group are perceived as more
             biased, suggesting that they might be less effective
             advocates. Yet, research testing whether stigmatized or
             non-stigmatized advocates are more persuasive has yielded
             mixed results. The current work builds on previous research
             to clarify that this occurs because stigmatized advocates
             are also perceived as more expert on social justice issues.
             Six studies document these trade-offs in perceptions. Three
             studies demonstrate that stigmatized and non-stigmatized
             advocates seem not to differ in their effectiveness because
             while perceived expertise boosts the effectiveness of
             stigmatized advocates, perceived bias undermines it. This
             occurs both when people confront societal inequality and
             interpersonal prejudice. Despite the lack of difference in
             persuasiveness, people predict that the stigmatized advocate
             will be more effective, suggesting that observers may not
             recognize perceived bias's role in undermining
             effectiveness. The present findings differ not only from
             participants' lay theories, but also from conclusions
             commonly reached by reviews of the literature which suggest
             that stigmatized advocates may be less effective than their
             non-stigmatized counterparts. By examining a broader range
             of perceptions and effects on audience members' attitudes
             and intentions to behave consistently with advocacy, we
             provide a more complete view of these effects.},
   Doi = {10.1016/j.jesp.2023.104519},
   Key = {fds372821}
}

@article{fds374969,
   Author = {Brown, RM and Dietze, P and Craig, MA},
   Title = {Highlighting health consequences of racial disparities
             sparks support for action.},
   Journal = {Science (New York, N.Y.)},
   Volume = {382},
   Number = {6677},
   Pages = {1394-1398},
   Year = {2023},
   Month = {December},
   url = {http://dx.doi.org/10.1126/science.adh4262},
   Abstract = {Racial disparities arise across many vital areas of American
             life, including employment, health, and interpersonal
             treatment. For example, one in three Black children lives in
             poverty (versus one in nine white children), and, on
             average, Black Americans live four fewer years compared with
             white Americans. Which disparity is more likely to spark
             reduction efforts? We find that highlighting disparities in
             health-related (versus economic) outcomes spurs greater
             social media engagement and support for disparity-mitigating
             policy. Further, reading about racial health disparities
             elicits greater support for action (e.g., protesting)
             compared with economic- or belonging-based disparities. This
             occurs in part because people view health disparities as
             violating morally sacred values, which enhances perceived
             injustice. This work elucidates which manifestations of
             racial inequality are most likely to prompt Americans to
             action.},
   Doi = {10.1126/science.adh4262},
   Key = {fds374969}
}

@article{fds371153,
   Author = {Brown, RM and Craig, MA},
   Title = {Understanding tactical responses to social problems through
             the lens of regulatory scope},
   Journal = {Nature Reviews Psychology},
   Volume = {2},
   Number = {7},
   Pages = {440-449},
   Publisher = {Springer Science and Business Media LLC},
   Year = {2023},
   Month = {July},
   url = {http://dx.doi.org/10.1038/s44159-023-00184-x},
   Abstract = {People may address societal problems either by engaging in
             collective action, aiming to change underlying structural
             systems, or by engaging in prosocial behaviours, aiming to
             help those affected. In this Perspective, we draw on
             construal level theory and regulatory scope theory to
             understand how people might choose to mitigate social
             problems. Specifically, we propose that people pursue
             solutions that alleviate the suffering of those affected by
             the problem (consequence-focused solutions) when they focus
             on lower-level or more psychologically proximal features and
             that they pursue solutions that address the underlying
             causes of the problem (cause-focused solutions) when they
             focus on higher-level or more psychologically distant
             features. Thus, people’s preferences for different
             solutions might be explained by understanding how people
             view the underlying problem. This framework explains the
             different ways people seek to address perceived social
             problems, providing insights into when and why people devote
             their time and energy to pursuing different forms of social
             action.},
   Doi = {10.1038/s44159-023-00184-x},
   Key = {fds371153}
}


%% Li, Rosa   
@article{fds369953,
   Author = {Li, R},
   Title = {Children are adaptive decision-makers: how environment
             shapes decision preferences.},
   Journal = {Proceedings of the Royal Society B: Biological
             Sciences},
   Volume = {290},
   Number = {1995},
   Pages = {20222117},
   Year = {2023},
   Month = {March},
   url = {http://dx.doi.org/10.1098/rspb.2022.2117},
   Doi = {10.1098/rspb.2022.2117},
   Key = {fds369953}
}


%% Rendina, Danielle   
@article{fds371131,
   Author = {Amaral, WZ and Lubach, GR and Rendina, DN and Phillips, GJ and Lyte, M and Coe, CL},
   Title = {Significant Microbial Changes Are Evident in the
             Reproductive Tract of Pregnant Rhesus Monkeys at
             Mid-Gestation but Their Gut Microbiome Does Not Shift until
             Late Gestation.},
   Journal = {Microorganisms},
   Volume = {11},
   Number = {6},
   Pages = {1481},
   Publisher = {MDPI AG},
   Year = {2023},
   Month = {June},
   url = {http://dx.doi.org/10.3390/microorganisms11061481},
   Abstract = {Vaginal and rectal specimens were obtained from cycling,
             pregnant, and nursing rhesus monkeys to assess
             pregnancy-related changes in the commensal bacteria in their
             reproductive and intestinal tracts. Using 16S rRNA gene
             amplicon sequencing, significant differences were found only
             in the vagina at mid-gestation, not in the hindgut. To
             verify the apparent stability in gut bacterial composition
             at mid-gestation, the experiment was repeated with
             additional monkeys, and similar results were found with both
             16S rRNA gene amplicon and metagenomic sequencing. A
             follow-up study investigated if bacterial changes in the
             hindgut might occur later in pregnancy. Gravid females were
             assessed closer to term and compared to nonpregnant females.
             By late pregnancy, significant differences in bacterial
             composition, including an increased abundance of 4 species
             of <i>Lactobacillus</i> and <i>Bifidobacterium
             adolescentis</i>, were detected, but without a shift in the
             overall community structure. Progesterone levels were
             assessed as a possible hormone mediator of bacterial change.
             The relative abundance of only some taxa (e.g.,
             <i>Bifidobacteriaceae</i>) were specifically associated with
             progesterone. In summary, pregnancy changes the microbial
             profiles in monkeys, but the bacterial diversity in their
             lower reproductive tract is different from women, and the
             composition of their intestinal symbionts remains stable
             until late gestation when several Firmicutes become more
             prominent.},
   Doi = {10.3390/microorganisms11061481},
   Key = {fds371131}
}

@article{fds370894,
   Author = {Smith, CJ and Rendina, DN and Kingsbury, MA and Malacon, KE and Nguyen,
             DM and Tran, JJ and Devlin, BA and Raju, RM and Clark, MJ and Burgett, L and Zhang, JH and Cetinbas, M and Sadreyev, RI and Chen, K and Iyer, MS and Bilbo, SD},
   Title = {Microbial modulation via cross-fostering prevents the
             effects of pervasive environmental stressors on microglia
             and social behavior, but not the dopamine
             system.},
   Journal = {Molecular Psychiatry},
   Year = {2023},
   Month = {May},
   url = {http://dx.doi.org/10.1038/s41380-023-02108-w},
   Abstract = {Environmental toxicant exposure, including air pollution, is
             increasing worldwide. However, toxicant exposures are not
             equitably distributed. Rather, low-income and minority
             communities bear the greatest burden, along with higher
             levels of psychosocial stress. Both air pollution and
             maternal stress during pregnancy have been linked to
             neurodevelopmental disorders such as autism, but biological
             mechanisms and targets for therapeutic intervention remain
             poorly understood. We demonstrate that combined prenatal
             exposure to air pollution (diesel exhaust particles, DEP)
             and maternal stress (MS) in mice induces social behavior
             deficits only in male offspring, in line with the male bias
             in autism. These behavioral deficits are accompanied by
             changes in microglial morphology and gene expression as well
             as decreased dopamine receptor expression and dopaminergic
             fiber input in the nucleus accumbens (NAc). Importantly, the
             gut-brain axis has been implicated in ASD, and both
             microglia and the dopamine system are sensitive to the
             composition of the gut microbiome. In line with this, we
             find that the composition of the gut microbiome and the
             structure of the intestinal epithelium are significantly
             shifted in DEP/MS-exposed males. Excitingly, both the
             DEP/MS-induced social deficits and microglial alterations in
             males are prevented by shifting the gut microbiome at birth
             via a cross-fostering procedure. However, while social
             deficits in DEP/MS males can be reversed by chemogenetic
             activation of dopamine neurons in the ventral tegmental
             area, modulation of the gut microbiome does not impact
             dopamine endpoints. These findings demonstrate male-specific
             changes in the gut-brain axis following DEP/MS and suggest
             that the gut microbiome is an important modulator of both
             social behavior and microglia.},
   Doi = {10.1038/s41380-023-02108-w},
   Key = {fds370894}
}


%% Smith, Caroline   
@article{fds370893,
   Author = {Smith, CJ and Rendina, DN and Kingsbury, MA and Malacon, KE and Nguyen,
             DM and Tran, JJ and Devlin, BA and Raju, RM and Clark, MJ and Burgett, L and Zhang, JH and Cetinbas, M and Sadreyev, RI and Chen, K and Iyer, MS and Bilbo, SD},
   Title = {Microbial modulation via cross-fostering prevents the
             effects of pervasive environmental stressors on microglia
             and social behavior, but not the dopamine
             system.},
   Journal = {Molecular Psychiatry},
   Year = {2023},
   Month = {May},
   url = {http://dx.doi.org/10.1038/s41380-023-02108-w},
   Abstract = {Environmental toxicant exposure, including air pollution, is
             increasing worldwide. However, toxicant exposures are not
             equitably distributed. Rather, low-income and minority
             communities bear the greatest burden, along with higher
             levels of psychosocial stress. Both air pollution and
             maternal stress during pregnancy have been linked to
             neurodevelopmental disorders such as autism, but biological
             mechanisms and targets for therapeutic intervention remain
             poorly understood. We demonstrate that combined prenatal
             exposure to air pollution (diesel exhaust particles, DEP)
             and maternal stress (MS) in mice induces social behavior
             deficits only in male offspring, in line with the male bias
             in autism. These behavioral deficits are accompanied by
             changes in microglial morphology and gene expression as well
             as decreased dopamine receptor expression and dopaminergic
             fiber input in the nucleus accumbens (NAc). Importantly, the
             gut-brain axis has been implicated in ASD, and both
             microglia and the dopamine system are sensitive to the
             composition of the gut microbiome. In line with this, we
             find that the composition of the gut microbiome and the
             structure of the intestinal epithelium are significantly
             shifted in DEP/MS-exposed males. Excitingly, both the
             DEP/MS-induced social deficits and microglial alterations in
             males are prevented by shifting the gut microbiome at birth
             via a cross-fostering procedure. However, while social
             deficits in DEP/MS males can be reversed by chemogenetic
             activation of dopamine neurons in the ventral tegmental
             area, modulation of the gut microbiome does not impact
             dopamine endpoints. These findings demonstrate male-specific
             changes in the gut-brain axis following DEP/MS and suggest
             that the gut microbiome is an important modulator of both
             social behavior and microglia.},
   Doi = {10.1038/s41380-023-02108-w},
   Key = {fds370893}
}

@article{fds369663,
   Author = {Bilbo, S and Smith, C and Rendina, D and Kingsbury, M and Malacon, K and Nguyen, D and Tran, J and Devlin, B and Raju, R and Clark, M and Burgett,
             L and Zhang, J and Cetinbas, M and Sadreyev, R and Chen, K and Iyer,
             M},
   Title = {Microbial modulation prevents the effects of pervasive
             environmental stressors on microglia and social behavior,
             but not the dopamine system.},
   Journal = {Res Sq},
   Year = {2023},
   Month = {February},
   url = {http://dx.doi.org/10.21203/rs.3.rs-2548369/v1},
   Abstract = {Environmental toxicant exposure, including air pollution, is
             increasing worldwide. However, toxicant exposures are not
             equitably distributed. Rather, low-income and minority
             communities bear the greatest burden, along with higher
             levels of psychosocial stress. Both air pollution and
             maternal stress during pregnancy have been linked to
             neurodevelopmental disorders such as autism, but biological
             mechanisms and targets for therapeutic intervention remain
             poorly understood. We demonstrate that combined prenatal
             exposure to air pollution (diesel exhaust particles, DEP)
             and maternal stress (MS) in mice induces social behavior
             deficits only in male offspring, in line with the male bias
             in autism. These behavioral deficits are accompanied by
             changes in microglial morphology and gene expression as well
             as decreased dopamine receptor expression and dopaminergic
             fiber input in the nucleus accumbens (NAc). Importantly, the
             gut-brain axis has been implicated in ASD, and both
             microglia and the dopamine system are sensitive to the
             composition of the gut microbiome. In line with this, we
             find that the composition of the gut microbiome and the
             structure of the intestinal epithelium are significantly
             shifted in DEP/MS-exposed males. Excitingly, both the
             DEP/MS-induced social deficits and microglial alterations in
             males are prevented by shifting the gut microbiome at birth
             via a cross-fostering procedure. However, while social
             deficits in DEP/MS males can be reversed by chemogenetic
             activation of dopamine neurons in the ventral tegmental
             area, modulation of the gut microbiome does not impact
             dopamine endpoints. These findings demonstrate male-specific
             changes in the gut-brain axis following DEP/MS and suggest
             that the gut microbiome is an important modulator of both
             social behavior and microglia.},
   Doi = {10.21203/rs.3.rs-2548369/v1},
   Key = {fds369663}
}


%% Van Cappellen, Patty   
@article{fds372239,
   Author = {Reece, GA and Van Tongeren and DR and Van Cappellen,
             P},
   Title = {Eternal outgroups: Afterlife beliefs predict
             prejudice},
   Journal = {Personality and Individual Differences},
   Volume = {214},
   Pages = {112352-112352},
   Publisher = {Elsevier BV},
   Year = {2023},
   Month = {November},
   url = {http://dx.doi.org/10.1016/j.paid.2023.112352},
   Abstract = {People express prejudice toward ideologically dissimilar
             groups. However, little research has explored (a) how
             specific beliefs, such as afterlife beliefs, relate to
             prejudice, (b) whether such beliefs can explain the known
             association between religiosity and prejudice toward
             value-threatening groups, and (c) the mechanisms through
             which afterlife beliefs predict prejudice. We addressed
             these questions by assessing individual differences in
             religious, spiritual, and secular afterlife beliefs and
             attitudes toward religious and political groups. Across four
             studies (N = 3996 U.S. adults), we find evidence supporting
             a group exclusion hypothesis: religious and secular
             afterlife beliefs predict prejudice toward value-incongruent
             groups (even after controlling for religiosity). We also
             find some evidence that religious afterlife beliefs explain
             the association between religiosity and prejudice against
             value-threatening groups. Finally, we find moderate support
             for an existential anxiety buffer hypothesis, wherein some
             afterlife beliefs predict increased peace of mind, reduced
             death anxiety, and in turn, reduced prejudice.},
   Doi = {10.1016/j.paid.2023.112352},
   Key = {fds372239}
}

@article{fds365925,
   Author = {Van Cappellen and P and Edwards, ME and Shiota, MN},
   Title = {Shades of expansiveness: Postural expression of dominance,
             high-arousal positive affect, and warmth.},
   Journal = {Emotion},
   Volume = {23},
   Number = {4},
   Pages = {973-985},
   Year = {2023},
   Month = {June},
   url = {http://dx.doi.org/10.1037/emo0001146},
   Abstract = {In addition to the face, bodily posture plays an important
             role in communicating affective states. Postural
             expansion-how much space the body takes up-has been much
             studied as expressing and signaling dominance and pride. The
             present research aimed to expand research on the range of
             affect dimensions and affect-laden personality
             characteristics that are expressed via expansiveness,
             investigating specific forms of expansiveness and their
             interactions with other postural elements (e.g., arm
             position). Using an innovative expression-production method,
             Study 1 (<i>N</i> = 146) characterized full-body expressions
             of dominance, joy, hope, and awe; results indicated joy is
             communicated most expansively and suggested a signature arm
             position for most feelings. Studies 2 and 3 (<i>N</i>s = 352
             and 183) revealed that other postural features interact with
             expansiveness to signal dominance (arms akimbo, head raised,
             stability), as distinct from high-arousal positive affect
             (arms high up, head raised) and warmth (arms high up, head
             raised, instability). Together, this research adds needed
             data on full-body expressions of positive affect states and
             provides systematic analysis of different affective messages
             and varieties of postural expansiveness. (PsycInfo Database
             Record (c) 2023 APA, all rights reserved).},
   Doi = {10.1037/emo0001146},
   Key = {fds365925}
}


%% Weeks, Molly   
@misc{fds376036,
   Author = {Cerda-Smith, J and Yust, PKS and Weeks, MS and Asher, SR and Mulvey,
             KL},
   Title = {A Novel Approach for Evaluating a Schoolwide Antiracist
             Curriculum Intervention},
   Journal = {AERA Open},
   Volume = {10},
   Publisher = {SAGE Publications},
   Year = {2024},
   Month = {January},
   url = {http://dx.doi.org/10.1177/23328584231223476},
   Abstract = {This manuscript describes our effort to apply a novel
             approach to understanding student outcomes associated with a
             schoolwide antiracist intervention. We report a multimethod
             quantitative approach to evaluate a 10-week antiracist
             intervention designed and implemented by school staff by
             examining patterns of student intervention engagement and
             measures of key constructs that connect to antiracism,
             psychological well-being, and school connectedness. Our
             novel approach combines schoolwide surveys with smaller
             samples of daily diary participants, documenting variation
             in intervention engagement and examining postintervention
             outcomes. Our findings are limited by high attrition rates,
             small sample size, and data collection during the COVID-19
             pandemic; however, our methods offer a promising
             transferable approach to evaluate school-based antiracist
             interventions by examining patterns and predictors of
             intervention engagement, as well as daily fluctuations in
             student experience throughout the intervention
             period.},
   Doi = {10.1177/23328584231223476},
   Key = {fds376036}
}

@misc{fds371265,
   Author = {Yust, PKS and Weeks, MS and Williams, GA and Asher,
             SR},
   Title = {Social relationship provisions and loneliness in school:
             Child- and classroom-level effects.},
   Journal = {Journal of school psychology},
   Volume = {99},
   Pages = {101218},
   Publisher = {Elsevier BV},
   Year = {2023},
   Month = {August},
   url = {http://dx.doi.org/10.1016/j.jsp.2023.05.001},
   Abstract = {Building on social needs theory (Weiss, 1974), this study
             introduces the construct of classroom provision richness and
             examines the association between the exchange of social
             provisions among children in classrooms and children's
             feelings of loneliness in school. We examined the receipt of
             provisions from reciprocally nominated friends versus
             unilateral (one-sided) and non-friend classmates and
             examined associations between social provisions and
             loneliness at the child and classroom levels. Participants
             were 998 third- through fifth-grade children (468 girls, 530
             boys; 88.5% White) in 38 classrooms who indicated which
             classmates they played with, helped, validated, and provided
             opportunities for self-disclosure. In addition to the social
             provisions nomination measure, children responded to (a) a
             measure of loneliness that avoided content overlapping with
             social provisions, (b) a rating-scale sociometric measure of
             peer acceptance, and (c) a measure that asked them to
             indicate which classmates engaged in prosocial, aggressive,
             or withdrawn-type behaviors. Multilevel analyses indicated
             that social provisions received from reciprocal friends and
             from unilateral-received friends were associated with
             children's feelings of loneliness in school. Furthermore, a
             measure of the provision richness of classrooms moderated
             the association between child-level provisions received and
             feelings of loneliness, such that children who received
             fewer provisions were less lonely in classrooms that were
             more provision-rich. Classroom provision richness was also
             associated with the general level of prosocial behavior and
             peer acceptance in the classroom. Together, findings suggest
             that efforts to foster the exchange of social provisions in
             classrooms could reduce loneliness and facilitate a more
             caring classroom environment.},
   Doi = {10.1016/j.jsp.2023.05.001},
   Key = {fds371265}
}


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