Kenneth A. Dodge
%% Journal Articles
@article{fds272028,
Author = {Latendresse, SJ and Bates, JE and Goodnight, JA and Lansford, JE and Budde, JP and Goate, A and Dodge, KA and Pettit, GS and Dick,
DM},
Title = {Differential susceptibility to adolescent externalizing
trajectories: examining the interplay between CHRM2 and peer
group antisocial behavior.},
Journal = {Child development},
Volume = {82},
Number = {6},
Pages = {1797-1814},
Year = {2011},
Month = {November},
ISSN = {0009-3920},
url = {http://dx.doi.org/10.1111/j.1467-8624.2011.01640.x},
Abstract = {The present study characterized prototypical patterns of
development in self-reported externalizing behavior, between
12 and 22 years of age, within a community sample of 452
genotyped individuals. A Caucasian subset (n = 378) was then
examined to determine whether their probabilities of
displaying discrete trajectories were differentially
associated with CHRM2, a gene implicated in self-regulatory
processes across a range of externalizing behaviors, and if
affiliating with antisocial peers moderated these
associations. Findings indicate that relative to a normative
"lower risk" externalizing trajectory, likelihood of
membership in two "higher risk" trajectories increased with
each additional copy of the minor allelic variant at CHRM2,
and that this association was exacerbated among those
exposed to higher levels of peer group antisocial
behavior.},
Doi = {10.1111/j.1467-8624.2011.01640.x},
Key = {fds272028}
}
@article{fds272045,
Author = {Edwards, AC and Dodge, KA and Latendresse, SJ and Lansford, JE and Bates, JE and Pettit, GS and Budde, JP and Goate, AM and Dick,
DM},
Title = {MAOA-uVNTR and early physical discipline interact to
influence delinquent behavior.},
Journal = {Journal of child psychology and psychiatry, and allied
disciplines},
Volume = {51},
Number = {6},
Pages = {679-687},
Year = {2010},
Month = {June},
ISSN = {0021-9630},
url = {http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=000272027300049&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=47d3190e77e5a3a53558812f597b0b92},
Abstract = {<h4>Background</h4>A functional polymorphism in the promoter
region of the monoamine oxidizing gene monoamine oxidase A
(MAOA) has been associated with behavioral sensitivity to
adverse environmental conditions in multiple studies (e.g.,
Caspi et al. 2002; Kim-Cohen et al., 2006). The present
study investigates the effects of genotype and early
physical discipline on externalizing behavior. We expand on
the current literature in our assessment of externalizing,
incorporating information across multiple reporters and over
a broad developmental time period, and in our understanding
of environmental risk.<h4>Method</h4>This study uses data
from the Child Development Project, an ongoing longitudinal
study following a community sample of children beginning at
age 5. Physical discipline before age 6 was quantified using
a subset of questions from the Conflict Tactics Scale
(Straus, 1979). Externalizing behavior was assessed in the
male, European-American sub-sample (N = 250) by parent,
teacher, and self-report using Achenbach's Child Behavior
Checklist, Teacher Report Form, and Youth Self-Report
(Achenbach, 1991), at 17 time points from ages 6 to 22.
Regression analyses tested the influence of genotype,
physical discipline, and their interaction on externalizing
behavior, and its subscales, delinquency and
aggression.<h4>Results</h4>We found a significant
interaction effect between genotype and physical discipline
on levels of delinquent behavior. Similar trends were
observed for aggression and overall externalizing behavior,
although these did not reach statistical significance. Main
effects of physical discipline held for all outcome
variables, and no main effects held for genotype.<h4>Conclusion</h4>The
adverse consequences of physical discipline on forms of
externalizing behavior are exacerbated by an underlying
biological risk conferred by MAOA genotype.},
Doi = {10.1111/j.1469-7610.2009.02196.x},
Key = {fds272045}
}
@article{fds272038,
Author = {McMahon, and J, R and Witkiewitz, and K, and Kotler, and S, J and Group,
TCPPR},
Title = {Predictive validity of callous-unemotional traits measured
in early adolescence with respect to multiple antisocial
outcomes},
Journal = {Journal of Abnormal Psychology},
Volume = {119},
Number = {4},
Pages = {752-763},
Publisher = {American Psychological Association (APA)},
Year = {2010},
url = {http://dx.doi.org/10.1037/a0020796},
Abstract = {This study investigated the predictive validity of youth
callous-unemotional (CU) traits, as measured in early
adolescence (Grade 7) by the Antisocial Process Screening
Device (APSD; Frick & Hare, 2001), in a longitudinal sample
(N = 754). Antisocial outcomes, assessed in adolescence and
early adulthood, included self-reported general delinquency
from 7th grade through 2 years post-high school,
self-reported serious crimes through 2 years post-high
school, juvenile and adult arrest records through 1 year
post-high school, and antisocial personality disorder
symptoms and diagnosis at 2 years post-high school. CU
traits measured in 7th grade were highly predictive of 5 of
the 6 antisocial outcomes-general delinquency, juvenile and
adult arrests, and early adult antisocial personality
disorder criterion count and diagnosis-over and above prior
and concurrent conduct problem behavior (i.e., criterion
counts of oppositional defiant disorder and conduct
disorder) and attention-deficit/hyperactivity disorder
(criterion count). Incorporating a CU traits specifier for
those with a diagnosis of conduct disorder improved the
positive prediction of antisocial outcomes, with a very low
false-positive rate. There was minimal evidence of
moderation by sex, race, or urban/rural status. Urban/rural
status moderated one finding, with being from an urban area
associated with stronger relations between CU traits and
adult arrests. Findings clearly support the inclusion of CU
traits as a specifier for the diagnosis of conduct disorder,
at least with respect to predictive validity.},
Doi = {10.1037/a0020796},
Key = {fds272038}
}
@article{fds272061,
Author = {Dodge, KA},
Title = {Mechanisms of Gene-Environment Interaction Effects in the
Development of Conduct Disorder.},
Journal = {Perspectives on psychological science : a journal of the
Association for Psychological Science},
Volume = {4},
Number = {4},
Pages = {408-414},
Year = {2009},
Month = {July},
ISSN = {1745-6916},
url = {http://www.ncbi.nlm.nih.gov/pubmed/19779577},
Abstract = {The gene-environment interaction effect in the development
of conduct disorder is one of the most important discoveries
of the past decade, but the mechanisms through which this
effect operates remain elusive. I propose a model of these
processes that focuses on the individual's response to a
threatening stimulus in ongoing social interaction. The
individual's response coordinates three interrelated
systems: neural, autonomic, and information-processing. In
each system, adaptive, evolutionarily selected response
patterns characterize normal responding, but in
psychopathology these patterns have gone awry. Antecedents
of individual differences in these response patterns arise
from genetic polymorphisms, adverse environmental
experiences early in life, and their interaction. Programs
of research are proposed to test hypotheses in the model
through longitudinal, experimental, and clinical
intervention methods. This model can serve as a template for
inquiry in other forms of developmental psychopathology.},
Doi = {10.1111/j.1745-6924.2009.01147.x},
Key = {fds272061}
}
@article{fds272287,
Author = {Jaffee, SR and Caspi, A and Moffitt, TE and Dodge, KA and Rutter, M and Taylor, A and Tully, LA},
Title = {Nature X nurture: genetic vulnerabilities interact with
physical maltreatment to promote conduct
problems.},
Journal = {Development and psychopathology},
Volume = {17},
Number = {1},
Pages = {67-84},
Year = {2005},
Month = {January},
ISSN = {0954-5794},
url = {http://www.ncbi.nlm.nih.gov/pubmed/15971760},
Abstract = {Maltreatment places children at risk for psychiatric
morbidity, especially conduct problems. However, not all
maltreated children develop conduct problems. We tested
whether the effect of physical maltreatment on risk for
conduct problems was strongest among those who were at high
genetic risk for these problems using data from the E-risk
Study, a representative cohort of 1,116 5-year-old British
twin pairs and their families. Children's conduct problems
were ascertained via parent and teacher interviews. Physical
maltreatment was ascertained via parent report. Children's
genetic risk for conduct problems was estimated as a
function of their co-twin's conduct disorder status and the
pair's zygosity. The effect of maltreatment on risk for
conduct problems was strongest among those at high genetic
risk. The experience of maltreatment was associated with an
increase of 2% in the probability of a conduct disorder
diagnosis among children at low genetic risk for conduct
disorder but an increase of 24% among children at high
genetic risk. Prediction of behavioral pathology can attain
greater accuracy if both pathogenic environments and genetic
risk are ascertained. Certain genotypes may promote
resistance to trauma. Physically maltreated children whose
first-degree relatives engage in antisocial behavior warrant
priority for therapeutic intervention.},
Doi = {10.1017/s0954579405050042},
Key = {fds272287}
}
@article{fds272285,
Author = {Dodge, KA},
Title = {The Nature-Nurture Debate and Public Policy.},
Journal = {Merrill-Palmer quarterly (Wayne State University.
Press)},
Volume = {50},
Number = {4},
Pages = {418-427},
Year = {2004},
Month = {October},
ISSN = {0272-930X},
url = {http://www.ncbi.nlm.nih.gov/pubmed/20011615},
Abstract = {The contentious nature-nurture debate in developmental
psychology is poised to reach a rapprochement with
contemporary concepts of gene-environment interaction,
transaction, and fit. Discoveries over the past decade have
revealed how neither genes nor the environment offers a
sufficient window into human development. Rather, the most
important discoveries have come from unearthing the manner
in which the environment alters gene expression (and how
genes impose limits on environmental effects), how biology
and the environment influence each other across time, and
how maximizing gene-environment fit leads to optimal
outcomes for children. The manner in which these factors
operate in tandem should direct future scholarship,
practice, and public policy.},
Doi = {10.1353/mpq.2004.0028},
Key = {fds272285}
}
@article{fds272138,
Author = {Dodge, KA and Pettit, GS},
Title = {A biopsychosocial model of the development of chronic
conduct problems in adolescence.},
Journal = {Developmental psychology},
Volume = {39},
Number = {2},
Pages = {349-371},
Year = {2003},
Month = {March},
ISSN = {0012-1649},
url = {http://www.ncbi.nlm.nih.gov/pubmed/12661890},
Abstract = {A biopsychosocial model of the development of adolescent
chronic conduct problems is presented and supported through
a review of empirical findings. This model posits that
biological dispositions and sociocultural contexts place
certain children at risk in early life but that life
experiences with parents, peers. and social institutions
increment and mediate this risk. A transactional
developmental model is best equipped to describe the
emergence of chronic antisocial behavior across time.
Reciprocal influences among dispositions, contexts, and life
experiences lead to recursive iterations across time that
exacerbate or diminish antisocial development. Cognitive and
emotional processes within the child, including the
acquisition of knowledge and social-information-processing
patterns, mediate the relation between life experiences and
conduct problem outcomes. Implications for prevention
research and public policy are noted.},
Doi = {10.1037//0012-1649.39.2.349},
Key = {fds272138}
}
@article{fds272273,
Author = {Dodge, KA},
Title = {New wrinkles in the person versus situation
debate},
Journal = {Psychological Inquiry},
Volume = {4},
Number = {4},
Pages = {284-286},
Year = {1993},
url = {http://dx.doi.org/10.1207/s15327965pli0404_6},
Doi = {10.1207/s15327965pli0404_6},
Key = {fds272273}
}
@article{fds272263,
Author = {Dodge, KA},
Title = {Nature Versus Nurture in Childhood Conduct Disorder: It Is
Time to Ask a Different Question},
Journal = {Developmental Psychology},
Volume = {26},
Number = {5},
Pages = {698-701},
Publisher = {American Psychological Association (APA)},
Year = {1990},
Month = {January},
ISSN = {0012-1649},
url = {http://dx.doi.org/10.1037/0012-1649.26.5.698},
Abstract = {Lytton (1990, this issue) offers a lucid review of factors
in the development of conduct disorder in children that
focuses on the question of the "relative strength" of child
effects versus environmental effects. This question ignores
the fact that such estimates are a function of the
subpopulation being assessed and the context in which
measurement occurs. These estimates pit nature versus
nurture in a way that detracts from an emphasis on the
interaction of factors that characterizes most human
behavioral development. This perspective also assumes that
"child effects," "environmental effects," and "conduct
disorder" are homogeneous constructs, but these are more
likely aggregations of heterogeneous phenomena that have
been grouped together only for heuristic reasons. It is
recommended that instead of focusing on the relative sizes
of effects, researchers should focus on the questions of
which mechanisms operate and how they interact during
transactional development.},
Doi = {10.1037/0012-1649.26.5.698},
Key = {fds272263}
}
%% Book Reviews
@article{fds219663,
Author = {Dodge, K.A.},
Title = {Book review: The Handbook of Clinical Child Neuropsychology,
3rd edition},
Journal = {Journal of Clinical Psychiatry},
Volume = {72},
Number = {5},
Pages = {726},
Editor = {Edited by Cecil R. Reynolds and Elaine Fletcher-Janzen},
Year = {2010},
Key = {fds219663}
}