Kenneth A. Dodge

Publications of Kenneth A. Dodge    :recent first  alphabetical  combined  by tags listing:

%% Journal Articles   
   Author = {Dodge, KA},
   Title = {Nature Versus Nurture in Childhood Conduct Disorder: It Is
             Time to Ask a Different Question},
   Journal = {Developmental Psychology},
   Volume = {26},
   Number = {5},
   Pages = {698-701},
   Publisher = {American Psychological Association (APA)},
   Year = {1990},
   Month = {January},
   ISSN = {0012-1649},
   url = {},
   Abstract = {Lytton (1990, this issue) offers a lucid review of factors
             in the development of conduct disorder in children that
             focuses on the question of the "relative strength" of child
             effects versus environmental effects. This question ignores
             the fact that such estimates are a function of the
             subpopulation being assessed and the context in which
             measurement occurs. These estimates pit nature versus
             nurture in a way that detracts from an emphasis on the
             interaction of factors that characterizes most human
             behavioral development. This perspective also assumes that
             "child effects," "environmental effects," and "conduct
             disorder" are homogeneous constructs, but these are more
             likely aggregations of heterogeneous phenomena that have
             been grouped together only for heuristic reasons. It is
             recommended that instead of focusing on the relative sizes
             of effects, researchers should focus on the questions of
             which mechanisms operate and how they interact during
             transactional development.},
   Doi = {10.1037/0012-1649.26.5.698},
   Key = {fds272263}

   Author = {Dodge, KA},
   Title = {New wrinkles in the person versus situation
   Journal = {Psychological Inquiry},
   Volume = {4},
   Number = {4},
   Pages = {284-286},
   Year = {1993},
   url = {},
   Doi = {10.1207/s15327965pli0404_6},
   Key = {fds272273}

   Author = {Dodge, KA and Pettit, GS},
   Title = {A biopsychosocial model of the development of chronic
             conduct problems in adolescence.},
   Journal = {Developmental Psychology},
   Volume = {39},
   Number = {2},
   Pages = {349-371},
   Year = {2003},
   Month = {March},
   ISSN = {0012-1649},
   url = {},
   Abstract = {A biopsychosocial model of the development of adolescent
             chronic conduct problems is presented and supported through
             a review of empirical findings. This model posits that
             biological dispositions and sociocultural contexts place
             certain children at risk in early life but that life
             experiences with parents, peers. and social institutions
             increment and mediate this risk. A transactional
             developmental model is best equipped to describe the
             emergence of chronic antisocial behavior across time.
             Reciprocal influences among dispositions, contexts, and life
             experiences lead to recursive iterations across time that
             exacerbate or diminish antisocial development. Cognitive and
             emotional processes within the child, including the
             acquisition of knowledge and social-information-processing
             patterns, mediate the relation between life experiences and
             conduct problem outcomes. Implications for prevention
             research and public policy are noted.},
   Doi = {10.1037//0012-1649.39.2.349},
   Key = {fds272138}

   Author = {Dodge, KA},
   Title = {The Nature-Nurture Debate and Public Policy.},
   Journal = {Merrill Palmer Quarterly},
   Volume = {50},
   Number = {4},
   Pages = {418-427},
   Year = {2004},
   Month = {October},
   ISSN = {0272-930X},
   url = {},
   Abstract = {The contentious nature-nurture debate in developmental
             psychology is poised to reach a rapprochement with
             contemporary concepts of gene-environment interaction,
             transaction, and fit. Discoveries over the past decade have
             revealed how neither genes nor the environment offers a
             sufficient window into human development. Rather, the most
             important discoveries have come from unearthing the manner
             in which the environment alters gene expression (and how
             genes impose limits on environmental effects), how biology
             and the environment influence each other across time, and
             how maximizing gene-environment fit leads to optimal
             outcomes for children. The manner in which these factors
             operate in tandem should direct future scholarship,
             practice, and public policy.},
   Doi = {10.1353/mpq.2004.0028},
   Key = {fds272285}

   Author = {Jaffee, SR and Caspi, A and Moffitt, TE and Dodge, KA and Rutter, M and Taylor, A and Tully, LA},
   Title = {Nature X nurture: genetic vulnerabilities interact with
             physical maltreatment to promote conduct
   Journal = {Development and Psychopathology},
   Volume = {17},
   Number = {1},
   Pages = {67-84},
   Year = {2005},
   Month = {January},
   ISSN = {0954-5794},
   url = {},
   Abstract = {Maltreatment places children at risk for psychiatric
             morbidity, especially conduct problems. However, not all
             maltreated children develop conduct problems. We tested
             whether the effect of physical maltreatment on risk for
             conduct problems was strongest among those who were at high
             genetic risk for these problems using data from the E-risk
             Study, a representative cohort of 1,116 5-year-old British
             twin pairs and their families. Children's conduct problems
             were ascertained via parent and teacher interviews. Physical
             maltreatment was ascertained via parent report. Children's
             genetic risk for conduct problems was estimated as a
             function of their co-twin's conduct disorder status and the
             pair's zygosity. The effect of maltreatment on risk for
             conduct problems was strongest among those at high genetic
             risk. The experience of maltreatment was associated with an
             increase of 2% in the probability of a conduct disorder
             diagnosis among children at low genetic risk for conduct
             disorder but an increase of 24% among children at high
             genetic risk. Prediction of behavioral pathology can attain
             greater accuracy if both pathogenic environments and genetic
             risk are ascertained. Certain genotypes may promote
             resistance to trauma. Physically maltreated children whose
             first-degree relatives engage in antisocial behavior warrant
             priority for therapeutic intervention.},
   Doi = {10.1017/s0954579405050042},
   Key = {fds272287}

   Author = {Dodge, KA},
   Title = {Mechanisms of Gene-Environment Interaction Effects in the
             Development of Conduct Disorder.},
   Journal = {Perspectives on Psychological Science : a Journal of the
             Association for Psychological Science},
   Volume = {4},
   Number = {4},
   Pages = {408-414},
   Year = {2009},
   Month = {July},
   ISSN = {1745-6916},
   url = {},
   Abstract = {The gene-environment interaction effect in the development
             of conduct disorder is one of the most important discoveries
             of the past decade, but the mechanisms through which this
             effect operates remain elusive. I propose a model of these
             processes that focuses on the individual's response to a
             threatening stimulus in ongoing social interaction. The
             individual's response coordinates three interrelated
             systems: neural, autonomic, and information-processing. In
             each system, adaptive, evolutionarily selected response
             patterns characterize normal responding, but in
             psychopathology these patterns have gone awry. Antecedents
             of individual differences in these response patterns arise
             from genetic polymorphisms, adverse environmental
             experiences early in life, and their interaction. Programs
             of research are proposed to test hypotheses in the model
             through longitudinal, experimental, and clinical
             intervention methods. This model can serve as a template for
             inquiry in other forms of developmental psychopathology.},
   Doi = {10.1111/j.1745-6924.2009.01147.x},
   Key = {fds272061}

   Author = {McMahon, and J, R and Witkiewitz, and K, and Kotler, and S, J and Group,
   Title = {Predictive validity of callous-unemotional traits measured
             in early adolescence with respect to multiple antisocial
   Journal = {Journal of Abnormal Psychology},
   Volume = {119},
   Number = {4},
   Pages = {752-763},
   Publisher = {American Psychological Association (APA)},
   Year = {2010},
   url = {},
   Abstract = {This study investigated the predictive validity of youth
             callous-unemotional (CU) traits, as measured in early
             adolescence (Grade 7) by the Antisocial Process Screening
             Device (APSD; Frick & Hare, 2001), in a longitudinal sample
             (N = 754). Antisocial outcomes, assessed in adolescence and
             early adulthood, included self-reported general delinquency
             from 7th grade through 2 years post-high school,
             self-reported serious crimes through 2 years post-high
             school, juvenile and adult arrest records through 1 year
             post-high school, and antisocial personality disorder
             symptoms and diagnosis at 2 years post-high school. CU
             traits measured in 7th grade were highly predictive of 5 of
             the 6 antisocial outcomes-general delinquency, juvenile and
             adult arrests, and early adult antisocial personality
             disorder criterion count and diagnosis-over and above prior
             and concurrent conduct problem behavior (i.e., criterion
             counts of oppositional defiant disorder and conduct
             disorder) and attention-deficit/hyperactivity disorder
             (criterion count). Incorporating a CU traits specifier for
             those with a diagnosis of conduct disorder improved the
             positive prediction of antisocial outcomes, with a very low
             false-positive rate. There was minimal evidence of
             moderation by sex, race, or urban/rural status. Urban/rural
             status moderated one finding, with being from an urban area
             associated with stronger relations between CU traits and
             adult arrests. Findings clearly support the inclusion of CU
             traits as a specifier for the diagnosis of conduct disorder,
             at least with respect to predictive validity.},
   Doi = {10.1037/a0020796},
   Key = {fds272038}

   Author = {Edwards, AC and Dodge, KA and Latendresse, SJ and Lansford, JE and Bates, JE and Pettit, GS and Budde, JP and Goate, AM and Dick,
   Title = {MAOA-uVNTR and early physical discipline interact to
             influence delinquent behavior.},
   Journal = {Journal of Child Psychology and Psychiatry, and Allied
   Volume = {51},
   Number = {6},
   Pages = {679-687},
   Year = {2010},
   Month = {June},
   ISSN = {0021-9630},
   url = {},
   Abstract = {<h4>Background</h4>A functional polymorphism in the promoter
             region of the monoamine oxidizing gene monoamine oxidase A
             (MAOA) has been associated with behavioral sensitivity to
             adverse environmental conditions in multiple studies (e.g.,
             Caspi et al. 2002; Kim-Cohen et al., 2006). The present
             study investigates the effects of genotype and early
             physical discipline on externalizing behavior. We expand on
             the current literature in our assessment of externalizing,
             incorporating information across multiple reporters and over
             a broad developmental time period, and in our understanding
             of environmental risk.<h4>Method</h4>This study uses data
             from the Child Development Project, an ongoing longitudinal
             study following a community sample of children beginning at
             age 5. Physical discipline before age 6 was quantified using
             a subset of questions from the Conflict Tactics Scale
             (Straus, 1979). Externalizing behavior was assessed in the
             male, European-American sub-sample (N = 250) by parent,
             teacher, and self-report using Achenbach's Child Behavior
             Checklist, Teacher Report Form, and Youth Self-Report
             (Achenbach, 1991), at 17 time points from ages 6 to 22.
             Regression analyses tested the influence of genotype,
             physical discipline, and their interaction on externalizing
             behavior, and its subscales, delinquency and
             aggression.<h4>Results</h4>We found a significant
             interaction effect between genotype and physical discipline
             on levels of delinquent behavior. Similar trends were
             observed for aggression and overall externalizing behavior,
             although these did not reach statistical significance. Main
             effects of physical discipline held for all outcome
             variables, and no main effects held for genotype.<h4>Conclusion</h4>The
             adverse consequences of physical discipline on forms of
             externalizing behavior are exacerbated by an underlying
             biological risk conferred by MAOA genotype.},
   Doi = {10.1111/j.1469-7610.2009.02196.x},
   Key = {fds272045}

   Author = {Latendresse, SJ and Bates, JE and Goodnight, JA and Lansford, JE and Budde, JP and Goate, A and Dodge, KA and Pettit, GS and Dick,
   Title = {Differential susceptibility to adolescent externalizing
             trajectories: examining the interplay between CHRM2 and peer
             group antisocial behavior.},
   Journal = {Child Development},
   Volume = {82},
   Number = {6},
   Pages = {1797-1814},
   Year = {2011},
   Month = {November},
   ISSN = {0009-3920},
   url = {},
   Abstract = {The present study characterized prototypical patterns of
             development in self-reported externalizing behavior, between
             12 and 22 years of age, within a community sample of 452
             genotyped individuals. A Caucasian subset (n = 378) was then
             examined to determine whether their probabilities of
             displaying discrete trajectories were differentially
             associated with CHRM2, a gene implicated in self-regulatory
             processes across a range of externalizing behaviors, and if
             affiliating with antisocial peers moderated these
             associations. Findings indicate that relative to a normative
             "lower risk" externalizing trajectory, likelihood of
             membership in two "higher risk" trajectories increased with
             each additional copy of the minor allelic variant at CHRM2,
             and that this association was exacerbated among those
             exposed to higher levels of peer group antisocial
   Doi = {10.1111/j.1467-8624.2011.01640.x},
   Key = {fds272028}

%% Book Reviews   
   Author = {Dodge, K.A.},
   Title = {Book review: The Handbook of Clinical Child Neuropsychology,
             3rd edition},
   Journal = {Journal of Clinical Psychiatry},
   Volume = {72},
   Number = {5},
   Pages = {726},
   Editor = {Edited by Cecil R. Reynolds and Elaine Fletcher-Janzen},
   Year = {2010},
   Key = {fds219663}